An organophosphorus cholinesterase inhibitor that is used as an insecticide and an acaricide.
Analogs or derivatives of mandelic acid (alpha-hydroxybenzeneacetic acid).
Pesticides designed to control insects that are harmful to man. The insects may be directly harmful, as those acting as disease vectors, or indirectly harmful, as destroyers of crops, food products, or textile fabrics.
An acute, diffuse, and suppurative inflammation of loose connective tissue, particularly the deep subcutaneous tissues, and sometimes muscle, which is most commonly seen as a result of infection of a wound, ulcer, or other skin lesions.
Incorrect diagnoses after clinical examination or technical diagnostic procedures.
Chemicals used to destroy pests of any sort. The concept includes fungicides (FUNGICIDES, INDUSTRIAL); INSECTICIDES; RODENTICIDES; etc.
Processes orchestrated or driven by a plethora of genes, plant hormones, and inherent biological timing mechanisms facilitated by secondary molecules, which result in the systematic transformation of plants and plant parts, from one stage of maturity to another.
The unconsolidated mineral or organic matter on the surface of the earth that serves as a natural medium for the growth of land plants.
An array of tests used to determine the toxicity of a substance to living systems. These include tests on clinical drugs, foods, and environmental pollutants.
Multicellular, eukaryotic life forms of kingdom Plantae (sensu lato), comprising the VIRIDIPLANTAE; RHODOPHYTA; and GLAUCOPHYTA; all of which acquired chloroplasts by direct endosymbiosis of CYANOBACTERIA. They are characterized by a mainly photosynthetic mode of nutrition; essentially unlimited growth at localized regions of cell divisions (MERISTEMS); cellulose within cells providing rigidity; the absence of organs of locomotion; absence of nervous and sensory systems; and an alternation of haploid and diploid generations.
Units that convert some other form of energy into electrical energy.
A plant genus of the family PYROLACEAE. The common name of wintergreen is more often used for GAULTHERIA.
Products in capsule, tablet or liquid form that provide dietary ingredients, and that are intended to be taken by mouth to increase the intake of nutrients. Dietary supplements can include macronutrients, such as proteins, carbohydrates, and fats; and/or MICRONUTRIENTS, such as VITAMINS; MINERALS; and PHYTOCHEMICALS.
A highly toxic compound used as a gasoline additive. It causes acute toxic psychosis or chronic poisoning if inhaled or absorbed through the skin.
Native, inorganic or fossilized organic substances having a definite chemical composition and formed by inorganic reactions. They may occur as individual crystals or may be disseminated in some other mineral or rock. (Grant & Hackh's Chemical Dictionary, 5th ed; McGraw-Hill Dictionary of Scientific and Technical Terms, 4th ed)
Material prepared from plants.
A food group comprised of EDIBLE PLANTS or their parts.
Dried rhizome and roots of Piper methysticum, a shrub native to Oceania and known for its anti-anxiety and sedative properties. Heavy usage results in some adverse effects. It contains ALKALOIDS; LACTONES; kawain, methysticin, mucilage, STARCH, and yangonin. Kava is also the name of the pungent beverage prepared from the plant's roots.
An organothiophosphorus insecticide that has been used to control pig mange.
An organophosphate insecticide that inhibits monoamine oxidase and acetylcholinesterase. It has been shown to be genotoxic.
An organophosphate cholinesterase inhibitor that is used as an insecticide.
A triazine herbicide.
An organothiophosphate insecticide.
An organothiophosphate insecticide.
Potent cholinesterase inhibitor used as an insecticide and acaricide.
Artificial device such as an externally-worn camera attached to a stimulator on the RETINA, OPTIC NERVE, or VISUAL CORTEX, intended to restore or amplify vision.
Agencies of the FEDERAL GOVERNMENT of the United States.
Any substance in the air which could, if present in high enough concentration, harm humans, animals, vegetation or material. Substances include GASES; PARTICULATE MATTER; and volatile ORGANIC CHEMICALS.
Administrative units of government responsible for policy making and management of governmental activities.
Debris resulting from a process that is of no further use to the system producing it. The concept includes materials discharged from or stored in a system in inert form as a by-product of vital activities. (From Webster's New Collegiate Dictionary, 1981)
Contamination of bodies of water (such as LAKES; RIVERS; SEAS; and GROUNDWATER.)
Elements, compounds, mixtures, or solutions that are considered severely harmful to human health and the environment. They include substances that are toxic, corrosive, flammable, or explosive.
Contaminated water generated as a waste product of human activity.
Declarations by patients, made in advance of a situation in which they may be incompetent to decide about their own care, stating their treatment preferences or authorizing a third party to make decisions for them. (Bioethics Thesaurus)
A clear, odorless, tasteless liquid that is essential for most animal and plant life and is an excellent solvent for many substances. The chemical formula is hydrogen oxide (H2O). (McGraw-Hill Dictionary of Scientific and Technical Terms, 4th ed)
Authoritative treatises on drugs and preparations, their description, formulation, analytic composition, physical constants, main chemical properties used in identification, standards for strength, purity, and dosage, chemical tests for determining identity and purity, etc. They are usually published under governmental jurisdiction (e.g., USP, the United States Pharmacopoeia; BP, British Pharmacopoeia; P. Helv., the Swiss Pharmacopoeia). They differ from FORMULARIES in that they are far more complete: formularies tend to be mere listings of formulas and prescriptions.
An eleven-amino acid neurotransmitter that appears in both the central and peripheral nervous systems. It is involved in transmission of PAIN, causes rapid contractions of the gastrointestinal smooth muscle, and modulates inflammatory and immune responses.
Annual statements reviewing the status of the administrative and operational functions and accomplishments of an institution or organization.
Models used experimentally or theoretically to study molecular shape, electronic properties, or interactions; includes analogous molecules, computer-generated graphics, and mechanical structures.
Databases devoted to knowledge about specific chemicals.
Large collections of small molecules (molecular weight about 600 or less), of similar or diverse nature which are used for high-throughput screening analysis of the gene function, protein interaction, cellular processing, biochemical pathways, or other chemical interactions.
Measurement of tooth characteristics.
Absent or reduced sensitivity to cutaneous stimulation.
A sudden, audible expulsion of air from the lungs through a partially closed glottis, preceded by inhalation. It is a protective response that serves to clear the trachea, bronchi, and/or lungs of irritants and secretions, or to prevent aspiration of foreign materials into the lungs.
The oval-shaped oral cavity located at the apex of the digestive tract and consisting of two parts: the vestibule and the oral cavity proper.
Contractile tissue that produces movement in animals.
An enzyme that catalyzes the hydrolysis of ACETYLCHOLINE to CHOLINE and acetate. In the CNS, this enzyme plays a role in the function of peripheral neuromuscular junctions. EC 3.1.1.7.

Impaired respiratory muscle function in chemical plant workers producing chlorfenvinphos. (1/8)

All employees of a chemical plant division producing chlorfenvinphos were studied, i.e. 35 males aged 25-57 years (mean 42.1); their employment period ranged from 1-15 years (mean 9.0). Chronic bronchitis was diagnosed in 13 workers (37.1%). Mean air chlorfenvinphos concentrations in the work environment estimated with gas-liquid chromatography were from 0.0008-0.0018 mg/m3 (maximum allowable concentration according to Polish standards is 0. 01 mg/m3). The activity of erythrocyte acetylcholinesterase was similar to that observed in people who were not exposed to chemicals, however, a slightly lowered activity of plasma cholinesterase in the studied population was evidently the result of mild liver impairment. Spirometric investigations performed in the studied workers revealed slight alterations manifested by increased intrathoracic gas volume (ITGV) (the value of the index was 138.6% of the mean value, 24 workers with an abnormally high index), as well as by decreased specific airway conductance (sGaw); its mean value in the studied group was 58.5% of the mean standard (11 people showed an abnormal index). Substantial functional changes were found in the respiratory muscles. Maximal inspiratory pressures (MIP = 97. 2 +/- 28.3 cm H2O) as well as maximal expiratory pressures (MEP = 113.9 +/- 44.2 cm H2O) in the studied group were significantly lower (p < 0.01) as compared to those observed in the control group (MIP = 120.7 +/- 31.7; MEP = 154.4 +/- 40.2 cm H2O) of 22 males having similar cigarette smoking habit, without occupational exposure to chemicals. It was also found that the people who had worked for more than 10 years under conditions of exposure to chlorfenvinphos showed significantly lower (p < 0.05) values of maximal inspiratory pressure (87.2 +/- 28.06 cm H2O, n = 17) compared to the workers whose period of employment was shorter than 10 years (106.6 +/- 26.8 cm H2O, n = 18). The two groups were comparable with regard to age and smoking habits. The values of maximal expiratory pressures were similar in both groups. No essential disturbances in neuro-muscular transmission were observed; only in 3 workers (8.5%) the electrostimulating myasthenic test showed some disturbances in neuro-muscular transmission. It seems that respiratory muscles impairment in humans exposed to chlorfenvinphos results from changes in the metabolism and structure of muscles, and partly from lung hyperinflation.  (+info)

Hyposensitivity to amphetamine following exposure to chlorphenvinphos--protection by amphetamine preexposure. (2/8)

We investigated the effect of an acute exposure to chlorphenvinphos (CVP), an organophosphate anticholinesterase, on amphetamine-induced open-field locomotion in rats. CVP was administered in a single i.p. dose of 1.0 mg/kg (1/10 of the LD50). All animals were challenged with 1.0 mg/kg amphetamine (AMPH) three weeks after the CVP exposure, i.e. after a time sufficient for acetylcholinesterase recovery. Some rats were also given AMPH three weeks before the CVP exposure. In rats challenged with AMPH only once after the CVP exposure, AMPH-induced open-field locomotion was significantly reduced. Such an effect was not observed in rats given AMPH three weeks before the CVP exposure. The results suggest that a single CVP exposure may result in persistent dopaminergic hyposensitivity, and that an amphetamine pretreatment may protect the rat against this effect.  (+info)

Behavioural responsiveness to amphetamine or scopolamine following repeated exposure to chlorphenvinphos in rats. (3/8)

A number of reports indicate that exposure to organophosphates (OPs), inhibitors of acetylcholinesterase (AChE), may result in long-lasting neurobehavioural alterations suggestive of an increased cholinergic tone. It is known that rats with cholinergic hyperreactivity are behaviourally hyposensitive to cholinergic antagonists and dopaminergic agonists. The purpose of the present study was to find out whether a similar trait would develop in rats exposed to chlorphenvinphos (CVP), an OP pesticide, in the past. The rats were given ten daily i.p. injections of CVP at doses of 0.5 mg/kg (group P-0.5) or 1.0 mg/kg (group P-1.0). The locomotion stimulating effect of i.p. injection of 1.0 mg/kg amphetamine (AMPH), or 0.7 mg/kg scopolamine (SCOP), was assessed on postexposure day 21 (group P-0.5) or 42 (group P-1.0), i.e. after a time sufficient for AChE recovery. The assessment revealed that in group P-1.0 the behavioural response to AMPH and SCOP was significantly depressed. In rats of the P-0.5 group, however, the behavioural response to each of the drugs was increased. The results suggest that, depending on the exposure level, contrasting alterations in some neurotransmitter systems may be induced by repeated exposure to CVP.  (+info)

Electroretinographic changes induced by organophosphorus pesticides in rats. (4/8)

Electroretinographic changes induced by organophosphorus pesticides (OPs) were studied in rats. Male Wistar rats were intraperitoneally injected with fenthion, chlorpyrifos, fenitrothion, dichlorvos or chlorfenvinphos at doses of 0.01 mmol/kg and/or 0.05 mmol/kg. The electroretinogram (ERG) was recorded at 5 hours and 2 days after the administration, and brain and retinochoroid cholinesterase (ChE) activities was assayed at 3 days after the injections. The brain and retinochoroid ChE activities were reduced in rats treated with the OPs. Notably, the reduction of ChE activities by fenthion, chlorpyrifos and dichlorvos were similar. The administration of OPs induced a change in the ERG, characterized by alteration of the amplitudes of a- and b-waves. Nevertheless the ChE activities in the brain and retinochoroid were inhibited by all of the OPs, the OPs affected the amplitude of ERG differently. Fenthion and chlorpyrifos decreased the amplitudes; dichlorvos and chlorfenvinphos increased; and fenitrothion transiently decreased at 5 hours but increased 2 days after the injection. These results indicate that a factor or factors other than inhibition of ChE activities contributes to the alteration of ERG induced by OPs.  (+info)

Albumin binding as a potential biomarker of exposure to moderately low levels of organophosphorus pesticides. (5/8)

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Structure-based rational design of a phosphotriesterase. (6/8)

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Effects of stress pretreatment on the dynamics of blood cholinesterase activity after exposure to an organophosphorus pesticide in the rat. (7/8)

A single i.p. administration of 1.0 mg/kg of chlorphenvinphos (CVP), an organophosphorus pesticide, results in an acute stress response, evidenced by a marked (6-7 fold) rise in plasma corticosterone (CORT) concentration, and a diminished behavioural sensitivity to amphetamine (AMPH) three weeks postexposure. Surprisingly, in rats subjected to a single series of inescapable electric footshocks (60 10 msec triplets of 3.0 mA, 2 msec, square pulses during 20 min - IF ) two weeks prior to the CVP exposure, these effects are not observed. It has been assumed that the reduced effectiveness of CVP might be related to some persisting alterations in the functional state of the cholinergic system. The aim of the present work was to discover whether and in what way the IF pretreatment affects i) the cholinesterase activity in blood, and ii) the dynamics of the alterations in the cholinesterase (ChE) activity following the CVP exposure. The experiments were performed on 3 mo. old, male Wistar rats. In the first experiment, the blood samples were taken from the tail vein 15, 60 and 180 min after the IF. In the second experiment, the rats were pretreated with IF and 14 days later given 1.0 mg/kg of CVP i.p. Blood samples were taken 15 min, 60 min, 180 min, 24 h, 7 days, and 14 days after the CVP exposure. In the first experiment no differences in the ChE activity in plasma (pChE) and erythrocytes (rbcChE) were found between the shocked and control rats. In the second experiment, however, in rats pretreated with IF the rbcChE activity of was reduced by CVP less and pChE activity returned to normal faster than in rats not pretreated with IF. The results confirm that exposure to IF, a nonchemical stressor, induces some long-lasting adaptive changes which render the cholinergic system less susceptible to the harmful action of ChE inhibitors. It has been hypothesized that the changes consist in an increase of the antioxidant potential in blood and possibly other tissues.  (+info)

Sites of methylation of DNA bases by the action of organophosphorus insecticides in vitro. (8/8)

Methylation in vitro of DNA by three methyl-14C-labelled organophosphorus insecticides has been studied. The ability of methylbromphenvinphos, methylparathion and malathion to methylate N-7 of guanine in DNA can be expressed as 100:40:15. Among the methylation products, no O6-methylguanine, a known mutagen, was found. Both in the reaction with dsDNA and with ssDNA 7-methyl-guanine was the main methylation product. However, all methyl derivatives of adenine (3-methyladenine, 1-methyladenine and 7-methyladenine) constituted about 40% and 50% of all methylation products in the case of dsDNA and ssDNA, respectively. The only methyl derivative of pyrimidine we have identified was 3-methylcytosine. In the case of dsDNA 3-methylcytosine appeared in small amounts but in the alkylated ssDNA 3-methylcytosine C constituted about 20% of all alkylation products.  (+info)

Symptoms of Chemical poisoning - Chlorfenvinphos including 52 medical symptoms and signs of Chemical poisoning - Chlorfenvinphos, alternative diagnoses, misdiagnosis, and correct diagnosis for Chemical poisoning - Chlorfenvinphos signs or Chemical poisoning - Chlorfenvinphos symptoms.
Recent organic fertilizer treatments (cow manure, pig slurry, composts, or green manure) simultaneously increase insecticide adsorption onto soil and the insecticide soil persistence, indicating a mechanism of slow release of insecticide into soil by the organic matter. This occurred in sugar beet crops with aldicarb, thiofanox and imidacloprid; also, in leek, cauliflower and brussels sprouts crops with chlorpyrifos and chlorfenvinphos. In contrast, organic fertilizer treatments applied once or repeatedly in the past, have no significant influence on adsorption or persistence of insecticides; the same is observed for the old soil organic matter, when its soil concentrations change in limited ranges ...
the latest technology small chemical plant equipment_Chemical plant WikipediaCommonly, a small chemical plant called a pilot plant is built to provide dChemical plant WikipediaCommonly, a small chemical plant called a pilot plant is built to provide d
Huge orange flames and plumes of smoke can be seen at the Magnablend Chemical Plant facility in Waxahachie, about 30 miles south of Dallas.
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I am a chemical engineer, so I will talk about and outline my reasoning about this in engineering terms, sensors, control systems, feedback, and that sort of thing. This is not to imply any type of design as ID proponents consider it. That is simply how I think of and understand physiology, just the same as any other chemical plant, a very complex chemical plant with exquisite controls most of which we have no idea how they work, what the design parameters are, what parameter is being controlled to do what and under what circumstances. Hacking into the control system of a complex chemical plant and perhaps bypassing the safety systems (when you dont know what is/is not a safety system) is not something to be done casually. That is how I see a lot of medicine, the same as trying to hack into a chemical plant where none of the pipes or wires are labeled as to what they are carrying, where they are coming from or where they are going, you dont know what the control system is based on, or even ...
Company profile & key executives for Zibo Luzhong Welfare Chemical Plant (0552900D:-) including description, corporate address, management team and contact info.
Learn more about Emersons Plantweb® Architecture Helps Brazilian Chemical Plant Boost Production And Quality at Automation.com
Chemical Plants in Batavia on YP.com. See reviews, photos, directions, phone numbers and more for the best Chemical Plant Equipment & Supplies in Batavia, NY.
Company profile & key executives for Luoyang Wenchang Petroleum Chemical Plant (LNBESZ:-) including description, corporate address, management team and contact info.
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Production capacity is a vital statistic for describing the performance of a chemical plant, but there is no universally accepted definition or means to evaluate it. This article provides a comprehensive definition of capacity and details the options for deriving it.
AkzoNobel’s Specialty Chemicals business recently broke ground on a new alkoxylation facility in Ningbo, China, bringing the company’s total investment in the strategic multi-site to more than €400 million.
1. The table below shows the total number of man-days lost to sickness during one weeks operation of a small chemical plant. Showing all of your work, calculate the arithmetic mean and standard deviation of the number of lost.
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Hyposensitivity to vasopressin is a well documented phenomenon in animals with portal hypertension and patients with cirrhosis subject to haemorrhage. Haemorrhage is associated with the endogenous release of bradykinin, which may subsequently stimulate the formation of nitric oxide (NO). The present study investigated the relative contribution of NO synthase (NOS) isoforms and the role of bradykinin in the pathogenesis of splanchnic hyposensitivity to a long-acting vasopressin analogue, glypressin, in rats with portal hypertension induced by partial portal vein ligation (PVL). At 14 days after the operation, systemic and portal haemodynamics were measured in stable or bleeding PVL rats receiving an intravenous infusion of glypressin (0.07 mg/kg). In the treatment groups, NG-nitro-L-arginine methyl ester (L-NAME; a non-selective NOS inhibitor), L-canavanine (a specific inhibitor of inducible NOS) or HOE 140 (a bradykinin B2 receptor antagonist) was administered 45 min before the infusion of ...
Today, the fundamental aspects of designing chemical plants are done by chemical engineers. Historically, this was not always the case and many chemical plants were constructed in a haphazard way before the discipline of chemical engineering became established. Chemical engineering was first established as a profession in the United Kingdom when the first chemical engineering course was given at the University of Manchester in 1887 by George E. Davis in the form of twelve lectures covering various aspects of industrial chemical practice.[5] As a consequence George E. Davis is regarded as the Worlds first Chemical Engineer.Today Chemical Engineering is a profession and those Professional Chemical Engineers with experience can gain Chartered engineer status through the Institution of Chemical Engineers. In plant design, typically less than 1 per cent of ideas for new designs ever become commercialized. During this solution process, typically, cost studies are used as an initial screening to ...
Union officials representing hundreds of striking employees of an upstate New York chemical plant are expected to resume talks with the company in an effort to end the nine-week-old strike.
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Scientists have for the first time reengineered a building block of protein-based nanostructures found in bacteria to give it a new function.
Chinese company Xinxiang Tianyuan oil equipment Co.Ltd. was established in 2003. The company is one of the largest manufacturers of equipment in China in the such areas as: 1. In the food industry: the line of equipment for the production of all...
F. Zsila, Fitos, I., Bencze, G., Keri, G., and Orfi, L., Determination of human serum α1-acid glycoprotein and albumin binding of various marketed and preclinical kinase inhibitors, Current Medicinal Chemistry, vol. 16, no. 16, pp. 1964 - 1977, 2009. ...
DAVID A. BENDER; The Effect of Single and Repeated Administration of Chlorpromazine on Serum Tryptophan and Albumin Binding of Tryptophan in the Rat. Biochem Soc Trans 1 February 1975; 3 (1): 115-117. doi: https://doi.org/10.1042/bst0030115. Download citation file:. ...
A raging fire and thick black smokeerupted on Friday evening from Arkema SAsflood-damaged Texas chemical plant for the second time thisweek, according to officials and video aired by localbroadcasters.
Chinese authorities have ordered a controversial chemical plant in the countrys northeast to shut down and tightened their grip over news coverage on a large protest that prompted the unusual decision.
Louisiana officials report two people have died and 77 others were injured when a chemical plant exploded Thursday morning. One of those injured remain in critical condition.
Republican Attorney General Greg Abbott, under fire for blocking public access to state records documenting the location of dangerous chemicals
The cause of the explosion was under investigation, but the company has been cited and fined for work safety violations in the past.
We all deserve to live our lives free of the harmful impacts of toxic chemicals. Whether by pushing for safer chemical plants or detoxing the clothing we wear every day, were working towards a toxic-free future for all.
State police reopened a section of Interstate 391 about 90 miles west of Boston early Sunday after a toxic cloud caused by a leak at a nearby chemical plant dissipated, authorities said.State Police
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; Dicrotophos; Monocrotophos; Phorate; Dimethoate; Simazine; Atrazine; Dioxathion; Terbufos; Diazinon; Disulfoton; Phosphamidon; Parathion-methyl; Fenitrothion; Malathion; Fenthion; Parathion; Trichloronate; Chlorfenvinphos; Crotoxyphos; Fensulfothion; Ethion; Famphur; Carbophenothion; Phosmet; Leptophos; Coumaphos
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Vegetables grow nearby Yingfeng Chemical Plant. Shifang City, Sichuan Province. Greenpeace has investigated the manufacture of phosphate fertilizer in Sichuan. Chemical fertilizers play an integral role in industrial agriculture and food production. However, its manufacture has proven to be highly polluting, contributing to wide-spread environmental contamination and causing distress for local communities living in the vicinity of the production facilities.
One of the four people hurt in a March 21 explosion and fire at chemical plant in Louisvilles Rubbertown neighborhood has died. 59-year-old Steven Nichols of Charlestown, IN died at 6:45 p.m.
Are the personal care and cleaning products you use each day actually safe? As a modern consumer, just how informed are you? In this eye-opening presentation, you will learn how to become an empowered, educated, and healthier consumer. You will get the knowledge and confidence you need to navigate the maze of choices you face every day. Shop smarter, live greener, and feel better. 2 hrs. To Register Call Westchester Community College Continuing Ed at 914-606-6830 press 1 to register OR stop by Administration Building room 207.. Westchester Community College 75 Grasslands Rd Valhalla, NY 10595. ...
News about the U.S. military from The Washington Post and washingtonpost.com. Full coverage of defense budgets,Army,Navy,Air Force,Marines and the Pentagon.
Emergency crews are responding to the area of Virginia Avenue and Mill Road. Motorists in the area reporting seeing smoke coming from one of the buildings in the area.
When using this server please cite the following paper:. Zsila F, Bikadi Z, Malik D, Hari P, Pechan I, Berces A, Hazai E.. Evaluation of drug-human serum albumin binding interactions with support vector machine aided online automated docking.. Bioinformatics. 2011 May 18. ...
A chemical plant located in the southeast produces insecticide, herbicide and fungicide products packaged under various labels for major chemical companies.
In Louisianas industrial heart, the shadow of Trumps deregulation push looms as St James residents fight chemical plants, pipelines and laissez-faire policies
விசாகப்பட்டினத்தில் ஏற்பட்ட கேஸ் கசிவால் பாதிக்கப்பட்டு இதுவரை 8 பேர் வரை உயிரிழந்துள்ளதாகத் தெரிவிக்கப்பட்டுள்ளது. | Many people have fallen unconscious following a gas leak at Visakhapatnam chemical plant
The CVP optimizer uses the MarkowitzSharpe approach as applied to planning logistics and marketing models to increase profitability of a companys production and trading activities Any purchases and sales are regarded to be portfolio
I know there is a cpt of 36597, but its with the use of fluoroscopy. Per the doctor, he did not use the fluoroscopy. We have tried to research to find
We have determined the solution structure of an albumin binding domain of protein G, a surface protein of group C and G streptococci. We find that it folds into a left handed three-helix bundle similar to the albumin binding domain of protein PAB from Peptostreptococcus magnus. The two domains share 59% sequence identity, are thermally very stable, and bind to the same site on human serum albumin. The albumin binding site, the first determined for this structural motif known as the GA module, comprises residues spanning the first loop to the beginning of the third helix and includes the most conserved region of GA modules. The two GA modules have different affinities for albumin from different species, and their albumin binding patterns correspond directly to the host specificity of C/G streptococci and P. magnus, respectively. These studies of the evolution, structure, and binding properties of the GA module emphasize the power of bacterial adaptation and underline ecological and medical ...
Three chemical plant employees were treated for severe burns Monday night following an incident at AZ Electronic Materials in Branchburg, N.J., NJ.com reported.. Emergency crews were called to the facility at about 8:00 p.m. Monday. One employee had severe burns to the face, eyes and chest. Two others suffered contact burns while transporting the injured worker to the decontamination room, the article stated.. The type of chemicals released has not been announced, but North Branch Volunteer Fire Department Chief David Hickson said the chemicals were highly combustible. ...
View this research paper on Dow Chemical Plant Dow Chemical. The water solutions business unit of Dow Chemical Company manufactures Film Tec reverse osmosis...
1) Estimates for detailed occupations do not sum to the totals because the totals include occupations not shown separately. Estimates do not include self-employed workers.. (2) Annual wages have been calculated by multiplying the hourly mean wage by a year-round, full-time hours figure of 2,080 hours; for those occupations where there is not an hourly wage published, the annual wage has been directly calculated from the reported survey data.. (3) The relative standard error (RSE) is a measure of the reliability of a survey statistic. The smaller the relative standard error, the more precise the estimate.. (8) Estimate not released.. (9) The location quotient is the ratio of the area concentration of occupational employment to the national average concentration. A location quotient greater than one indicates the occupation has a higher share of employment than average, and a location quotient less than one indicates the occupation is less prevalent in the area than average. ...
1) Estimates for detailed occupations do not sum to the totals because the totals include occupations not shown separately. Estimates do not include self-employed workers.. (2) Annual wages have been calculated by multiplying the hourly mean wage by a year-round, full-time hours figure of 2,080 hours; for those occupations where there is not an hourly wage published, the annual wage has been directly calculated from the reported survey data.. (3) The relative standard error (RSE) is a measure of the reliability of a survey statistic. The smaller the relative standard error, the more precise the estimate.. (8) Estimate not released.. (9) The location quotient is the ratio of the area concentration of occupational employment to the national average concentration. A location quotient greater than one indicates the occupation has a higher share of employment than average, and a location quotient less than one indicates the occupation is less prevalent in the area than average. ...
1.In case of leakage, operators on site should immediately stop operation and quickly evacuate the leak contaminated area. If the leakage is flammable and explosive, personnel should remove the explosives object in the accident area, extinguish the fire and cut off the power supply in time if it is possible. Personnel should lie down in place when explosion occurs before they can evacuate ...
ExxonMobil said onThursday it started ethylene production at a new chemical unitin Singapore, marking the completion of a multi-billion dollarexpansion project at its Singapore petrochemical complex.
When using this server please cite the following paper:. Zsila F, Bikadi Z, Malik D, Hari P, Pechan I, Berces A, Hazai E.. Evaluation of drug-human serum albumin binding interactions with support vector machine aided online automated docking.. Bioinformatics. 2011 May 18. ...
Evan E. Bolton; Yanli Wang; Paul A. Thiessen; Stephen H. Bryant (2008). „Chapter 12 PubChem: Integrated Platform of Small Molecules and Biological Activities". Annual Reports in Computational Chemistry. 4: 217-241. doi:10.1016/S1574-1400(08)00012-1 ...
Modern acaricides belong to the general classes of organophosphates (example chlorfenvinphos), formamidines (example amitraz), ...
Sulfanomides and Trimethoprim Tetracyclines Streptomycin Chlorfenvinphos Nitrofurans Metabolites Aflatoxins Polycyclic aromatic ...
The toxicity of chlorfenvinphos is primarily caused by its inhibition of cholinesterase activity. Chlorfenvinphos reacts with ... This is mainly because chlorfenvinphos was not considered a dangerous toxin until the early 1990s. Therefore, chlorfenvinphos ... which have been exposed to chlorfenvinphos. These small amounts can be used to prove that chlorfenvinphos exposure has occurred ... Chlorfenvinphos has been included in many products since its first use in 1963. However, because of its toxic effect as a ...
The biological mechanism for the thinning is not entirely known, but it is believed that p,p'-DDE impairs the shell gland's ability to excrete calcium carbonate onto the developing egg.[7][9][10][11][12] Multiple mechanisms may be at work, or different mechanisms may operate in different species.[7] Some studies have shown that although DDE levels have fallen dramatically, eggshell thickness remains 10-12 percent thinner than before DDT was first used.[13]. Some studies have indicated that DDE is an endocrine disruptor[14] and contributes to breast cancer, but more recent studies provide strong evidence that there is no relationship between DDE exposure and breast cancer.[15] What is more clear is that DDE is a weak androgen receptor antagonist and can produce male genital tract abnormalities.[16][17]. Animal studies show that organochlorine pesticides-such as DDE-are neurotoxic, cause oxidative stress, and damage the brain's dopaminergic system.[18]. ...
... binds and reversibly inactivates the cholinesterases, thus inhibiting hydrolysis of acetylcholine. This increases acetylcholine concentrations at cholinergic synapses.[1] The precise mechanism of action of donepezil in patients with Alzheimer's disease is not fully understood. Certainly, Alzheimer's disease involves a substantial loss of the elements of the cholinergic system and it is generally accepted that the symptoms of Alzheimer's disease are related to this cholinergic deficit, particularly in the cerebral cortex and other areas of the brain.[18][19] It is noted that the hippocampal formation plays an important role in the processes of control of attention, memory and learning. Just the severity of the loss of cholinergic neurons of the central nervous system (CNS) has been found to correlate with the severity of cognitive impairment. In addition to its actions as an acetylcholinesterase inhibitor, donepezil has been found to act as a potent agonist of the σ1 receptor (Ki = ...
... (DDD) is an organochlorine insecticide that is slightly irritating to the skin.[1] DDD is a metabolite of DDT.[2] DDD is colorless and crystalline;[3] it is closely related chemically and is similar in properties to DDT, but it is considered to be less toxic to animals than DDT.[4] The molecular formula for DDD is (ClC6H4)2CHCHCl2 or C14H10Cl4, whereas the formula for DDT is (ClC6H4)2CHCCl3 or C14H9Cl5. DDD is in the "Group B2" classification, meaning that it is a probable human carcinogen. This is based on an increased incidence of lung tumors in male and female mice, liver tumors in male mice, and thyroid tumors in male rats. Further basis is that DDD is so similar to and is a metabolite of DDT, another probable human carcinogen.[2] DDD is no longer registered for agricultural use in the United States, but the general population continues to be exposed to it due to its long persistence time. The primary source of exposure is oral ingestion of food.[5] 1946 is the ...
... is a widely used, broad-spectrum benzimidazole fungicide and a metabolite of benomyl. It is also employed as a casting worm control agent in amenity turf situations such as golf greens, tennis courts etc. and in some countries is licensed for that use only.[2] The fungicide is used to control plant diseases in cereals and fruits, including citrus, bananas, strawberries, pineapples, and pomes.[3] It is also controversially used in Queensland, Australia on macadamia plantations.[4] A 4.7% solution of carbendazim hydrochloride, sold as Eertavas, is marketed as a treatment for Dutch elm disease. Studies have found high doses of carbendazim cause infertility and destroy the testicles of laboratory animals.[5][6] Maximum pesticide residue limits (MRLs) have reduced since discovering its harmful effects. The MRLs for fresh produce in the EU are now between 0.1 and 0.7 mg/kg with the exception of loquat, which is 2 mg/kg.[7] The limits for more commonly consumed citrus and pome fruits are ...
While botulinum toxin is generally considered safe in a clinical setting, there can be serious side effects from its use. The use of botulinum toxin A in cerebral palsy children is safe in the upper and lower limb muscles.[5][6] Most commonly, botulinum toxin can be injected into the wrong muscle group or with time spread from the injection site, causing temporary paralysis of unintended muscles. Side effects from cosmetic use generally result from unintended paralysis of facial muscles. These include partial facial paralysis, muscle weakness, and trouble swallowing. Side effects are not limited to direct paralysis however, and can also include headaches, flu-like symptoms, and allergic reactions.[41] Just as cosmetic treatments only last a number of months, paralysis side-effects can have the same durations.[citation needed] At least in some cases, these effects are reported to dissipate in the weeks after treatment.[citation needed] Bruising at the site of injection is not a side effect of the ...
By 1974, a team of Rothamsted Research scientists had discovered three pyrethroids suitable for use in agriculture, namely permethrin, cypermethrin and deltamethrin.[10] These compounds were subsequently licensed by the NRDC, as NRDC 143, 149 and 161 respectively, to companies which could then develop them for sale in defined territories. Imperial Chemical Industries (ICI) obtained licenses to permethrin and cypermethrin but their agreement with the NRDC did not allow worldwide sales. Also, it was clear to ICI's own researchers at Jealott's Hill that future competition in the marketplace might be difficult owing to the greater potency of deltamethrin compared to the other compounds. For that reason, chemists there sought patentable analogues which might have advantages compared to the Rothamsted insecticides by having wider spectrum or greater cost-benefit . The first breakthrough was made when a trifluoromethyl group was used to replace one of the chlorines in cypermethrin, especially when the ...
... has little systemic absorption, and is considered safe for topical use in adults and children over the age of 2 months. The FDA has assigned it as pregnancy category B. Animal studies have shown no effects on fertility or teratogenicity, but studies in humans have not been performed. The excretion of permethrin in breastmilk is unknown, and breastfeeding is recommended to be temporarily discontinued during treatment.[11] According to the Connecticut Department of Public Health, permethrin "has low mammalian toxicity, is poorly absorbed through the skin, and is rapidly inactivated by the body. Skin reactions have been uncommon."[14] Excessive exposure to permethrin can cause nausea, headache, muscle weakness, excessive salivation, shortness of breath, and seizures. Worker exposure to the chemical can be monitored by measurement of the urinary metabolites, while severe overdose may be confirmed by measurement of permethrin in serum or blood plasma.[15] Permethrin does not present any ...
Human exposure to methoxychlor occurs via air, soil, and water,[7] primarily in people who work with the substance or who are exposed to air, soil, or water that has been contaminated. It is unknown how quickly and efficiently the substance is absorbed by humans who have been exposed to contaminated air or via skin contact.[7] In animal models, high doses can lead to neurotoxicity.[7] Some methoxychlor's metabolites have estrogenic effects in adult and developing animals before and after birth.[7] One studied metabolite is 2,2-bis(p-hydroxyphenyl)-1,1,1-trichloroethane (HPTE) which shows reproductive toxicity in an animal model by reducing testosterone biosynthesis.[8][9] Such effects adversely affect both the male and female reproductive systems. It is expected that this "could occur in humans" but has not been proven.[7] While one study has linked methoxychlor to the development of leukemia in humans, most studies in animals and humans have been negative, thus the EPA has determined that it is ...
GV (IUPAC name: 2-(Dimethylamino)ethyl N,N-dimethylphosphoramidofluoridate) is an organophosphate nerve agent. GV is a part of a new series of nerve agents with properties similar to both the "G-series" and "V-series". It is a potent acetylcholinesterase inhibitor with properties similar to other nerve agents, being a highly poisonous vapour. Treatment for poisoning with GV involves drugs such as atropine, benactyzine, obidoxime, and HI-6.[1][2] ...
The four-membered ring in α-pinene 1 makes it a reactive hydrocarbon, prone to skeletal rearrangements such as the Wagner-Meerwein rearrangement. For example, attempts to perform hydration or hydrogen halide addition with the alkene functionality typically lead to rearranged products. With concentrated sulfuric acid and ethanol the major products are terpineol 2 and its ethyl ether 3, while glacial acetic acid gives the corresponding acetate ester 4. With dilute acids, terpin hydrate 5 becomes the major product. With one molar equivalent of anhydrous HCl, the simple addition product 6a can be formed at low temperature in the presence of ether, but it is very unstable. At normal temperatures, or if no ether is present, the major product is bornyl chloride 6b, along with a small amount of fenchyl chloride 6c.[5] For many years 6b (also called "artificial camphor") was referred to as "pinene hydrochloride", until it was confirmed as identical with bornyl chloride made from camphene. If more HCl is ...
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... is extracted from Huperzia serrata.[2] It is a reversible acetylcholinesterase inhibitor[6][7][8][9] and NMDA receptor antagonist[10] that crosses the blood-brain barrier.[11] Acetylcholinesterase is an enzyme that catalyzes the breakdown of the neurotransmitter acetylcholine and of some other choline esters that function as neurotransmitters. The structure of the complex of huperzine A with acetylcholinesterase has been determined by X-ray crystallography (PDB code: 1VOT; see the 3D structure).[12] For some years, huperzine A has been investigated as a possible treatment for diseases characterized by neurodegeneration, particularly Alzheimer's disease.[2][13] A 2013 meta-analysis found that huperzine A may be efficacious in improving cognitive function, global clinical status, and activities of daily living for individuals with Alzheimer's disease. However, due to the poor size and quality of the clinical trials reviewed, huperzine A should not be recommended as a treatment for ...
Neuromuscular blocking agents need to fit in a space close to 2 nanometres, which resembles the molecular length of decamethonium.[13] Some molecules of decamethonium congeners may bind only to one receptive site. Flexible molecules have a greater chance of fitting receptive sites. However, the most populated conformation may not be the best-fitted one. Very flexible molecules are, in fact, weak neuromuscular inhibitors with flat dose-response curves. On the other hand, stiff or rigid molecules tend to fit well or not at all. If the lowest-energy conformation fits, the compound has high potency because there is a great concentration of molecules close to the lowest-energy conformation. Molecules can be thin but yet rigid.[14] Decamethonium for example needs relatively high energy to change the N-N distance.[13] In general, molecular rigidity contributes to potency, while size affects whether a muscle relaxant shows a polarizing or a depolarizing effect.[3] Cations must be able to flow through ...
... is a poisonous diterpenoid found in the South American plant Ryania speciosa (Salicaceae). It was originally used as an insecticide.. The compound has extremely high affinity to the open-form ryanodine receptor, a group of calcium channels found in skeletal muscle, smooth muscle, and heart muscle cells.[1] It binds with such high affinity to the receptor that it was used as a label for the first purification of that class of ion channels and gave its name to it.. At nanomolar concentrations, ryanodine locks the receptor in a half-open state, whereas it fully closes them at micromolar concentration. The effect of the nanomolar-level binding is that ryanodine causes release of calcium from calcium stores as the sarcoplasmic reticulum in the cytoplasm, leading to massive muscular contractions. The effect of micromolar-level binding is paralysis. This is true for both mammals and insects.. ...
... is very toxic to cats which cannot tolerate the therapeutic doses for dogs.[7] This is associated with UGT1A6 deficiency in cats, the enzyme responsible for metabolizing cypermethrin. As a consequence, cypermethrin remains much longer in the cat's organs than in dogs or other mammals and can be fatal in large doses. In male rats cypermethrin was shown to exhibit a toxic effect on the reproductive system. After 15 days of continual dosing, both androgen receptor levels and serum testosterone levels were significantly reduced. These data suggested that cypermethrin can induce impairments of the structure of seminiferous tubules and spermatogenesis in male rats at high doses.[8] Long-term exposure to cypermethrin during adulthood is found to induce dopaminergic neurodegeneration in rats, and postnatal exposure enhances the susceptibility of animals to dopaminergic neurodegeneration if rechallenged during adulthood.[9] If exposed to cypermethrin during pregnancy, rats give birth to ...
Before DDT, malaria was successfully eliminated or curtailed in several tropical areas by removing or poisoning mosquito breeding grounds and larva habitats, for example by eliminating standing water. These methods have seen little application in Africa for more than half a century.[137] According to CDC, such methods are not practical in Africa because "Anopheles gambiae, one of the primary vectors of malaria in Africa, breeds in numerous small pools of water that form due to rainfall ... It is difficult, if not impossible, to predict when and where the breeding sites will form, and to find and treat them before the adults emerge."[138] The relative effectiveness of IRS versus other malaria control techniques (e.g. bednets or prompt access to anti-malarial drugs) varies and is dependent on local conditions.[34] A WHO study released in January 2008 found that mass distribution of insecticide-treated mosquito nets and artemisinin-based drugs cut malaria deaths in half in malaria-burdened Rwanda ...
... s are substances used to kill insects.[1] They include ovicides and larvicides used against insect eggs and larvae, respectively. Insecticides are used in agriculture, medicine, industry and by consumers. Insecticides are claimed to be a major factor behind the increase in the 20th-century's agricultural productivity.[2] Nearly all insecticides have the potential to significantly alter ecosystems; many are toxic to humans and/or animals; some become concentrated as they spread along the food chain. Insecticides can be classified into two major groups: systemic insecticides, which have residual or long term activity; and contact insecticides, which have no residual activity. The mode of action describes how the pesticide kills or inactivates a pest. It provides another way of classifying insecticides. Mode of action can be important in understanding whether an insecticide will be toxic to unrelated species, such as fish, birds and mammals. Insecticides may be repellent or ...
When used in the central nervous system to alleviate neurological symptoms, such as rivastigmine in Alzheimer's disease, all cholinesterase inhibitors require doses to be increased gradually over several weeks, and this is usually referred to as the titration phase. Many other types drug treatments may require a titration or stepping up phase. This strategy is used to build tolerance to adverse events or to reach a desired clinical effect.[12] This also prevents accidental overdose and is therefore recommended when initiating treatment with drugs that are extremely potent and/or toxic (drugs with a low therapeutic index). ...
... can be broadly categorized as a cholinergic physiological antagonist, because it reduces the apparent activity of cholinergic neurons, but does not act at the postsynaptic ACh receptor. Vesamicol causes a non-competitive and reversible block of the intracellular transporter VAChT responsible for carrying newly synthesized ACh into secretory vesicles in the presynaptic nerve terminal. This transport process is driven by a proton gradient between cell organelles and the cytoplasm. Blocking of acetylcholine loading leads to empty vesicles fusing with neuron membranes, decreasing ACh release. ...
... s are a group of highly conserved G-protein coupled receptors from the adhesion G protein-coupled receptor family. These receptors were originally identified based on their ability to bind the spider venom alpha-latrotoxin.[1] This conserved family of membrane proteins has up to three homologues in chordate species, including humans.[2] The precise functions of latrophilins remain unknown.[2] Genetic defects in latrophilin genes have been associated with diseases such as attention-deficit hyperactivity disorder and cancer.[3] ...
... ( anticholinergic agent) is a group of substances that blocks the action of the neurotransmitter acetylcholine (ACh) at synapses in the central and the peripheral nervous system, and, in broad terms, neuromuscular junction.[1][2] These agents inhibit parasympathetic nerve impulses by selectively blocking the binding of the neurotransmitter acetylcholine to its receptor in nerve cells. The nerve fibers of the parasympathetic system are responsible for the involuntary movement of smooth muscles present in the gastrointestinal tract, urinary tract, lungs, and many other parts of the body;[3] cholinergic process otherwise by enhancing ACh function.[3] In broad terms, anticholinergics are divided into two categories in accordance with their specific targets in the central, peripheral nervous system and neuromuscular junction:[3] antimuscarinic agents, and antinicotinic agents (ganglionic blockers, neuromuscular blockers).[4] In strict terms, anticholinergic only comprises ...
InChI=1S/C28H32O8/c1-24(2)10-9-22(29)26(4)27(24,31)12-11-25(3)28(26,32)15-17-20(36-25)14-19(35-23(17)30)16-7-8-18(33-5)21(13-16)34-6/h7-10,13-14,31-32H,11-12,15H2,1-6H3/t25-,26+,27-,28-/m1/ ...
... is an alkaloid found in Corydalis (Papaveraceae) and Dicentra, plants in the family Fumariaceae that can cause fatal poisoning in sheep and cattle.[citation needed] It has been shown to act as an acetylcholinesterase inhibitor,[1] and inhibits biosynthesis of dopamine via inhibition of the enzyme tyrosine hydroxylase.[2][3] Like apomorphine, it is reported to be an inhibitor of amyloid beta protein (Aβ) fiber formation, whose presence is a hallmark of Alzheimer's disease (AD). Bulbocapnine is thus a potential therapeutic under the amyloid hypothesis.[4] According to the Dorlands Medical Dictionary, it "inhibits the reflex and motor activities of striated muscle. It has been used in the treatment of muscular tremors and vestibular nystagmus".[5] A psychiatrist at Tulane University named Robert Heath carried out experiments on prisoners at the Louisiana State Penitentiary using bulbocapnine to induce stupor.[6] This work at Tulane inspired, and was continued parallel to, experiments ...
ನಿಕೋಟಿನ್‌ನ ಎಲ್‌ಡಿ50 ಪ್ರಮಾಣ ಇಲಿಗಳಿಗೆ 50 ಮಿಗ್ರಾ/ಕಿಗ್ರಾ ಮತ್ತು ಮೂಗಿಲಿಗಳಿಗೆ 3 ಮಿಗ್ರಾ/ಕಿಗ್ರಾ. 40-60 ಮಿಗ್ರಾಂ. (0.5-1.0 mg/kg) ಇದು ವಯಸ್ಕ ಮನುಷ್ಯರಿಗೆ ಘಾತಕ ಪ್ರಮಾಣವಾಗಿರುತ್ತದೆ.[೪೩][೪೪] ಆದ್ದರಿಂದ, ಕೊಕೈನ್‌ನಂತಹ ಇತರ ಕ್ಷಾರಗಳಿಗೆ ತುಲನೆ ಮಾಡಿ ನೋಡಿದಾದ ನಿಕೋಟಿನ್ ಹೆಚ್ಚಿನ ವಿಷತ್ವವನ್ನು ಹೊಂದಿದೆ, ಅದು ಮೈಸ್‌ಗೆ ನಿರ್ವಹಣೆ ಮಾಡಿ ನೋಡಿದಾಗ 95.1 ಮಿಗ್ರಾಂ/ಕಿಗ್ರಾಂ ನ LD50 ಪ್ರಮಾಣವನ್ನು ಹೊಂದಿದೆ. ಆದಾಗ್ಯೂ, ಕೇವಲ ಧೂಮಪಾನದ ಮೂಲಕ ನಿಕೋಟಿನ್ ಅನ್ನು ಅತಿ ...
Chlorfenvinphos been found in at least 1 of the 1,430 National Priorities List sites identified by the Environmental Protection ... Ingesting chlorfenvinphos results primarily on nervous system effects, such as headaches, blurred vision, weakness, and ... Chlorfenvinphos is an insecticide which is no longer used in the United States. ... How likely is chlorfenvinphos to cause cancer?. *Is there a medical test to show whether Ive been exposed to chlorfenvinphos? ...
Toxicological profile for Chlorfenvinphos. Atlanta, GA: U.S. Department of Health and Human Services, Public Health Service. ...
Chlorfenvinphos; (E)-2-Chloro-1-(2,4-dichlorophenyl)ethenyl diethyl phosphate; Chlorfenvinphos E; Chlorfenvinphos (E), alpha; ( ... trans-Isomer of chlorfenvinphos; Chlorfenvinphos (trans); (E)-Chlorfenvinfos; 2-Chloro-1-(2,4-dichlorophenyl)ethenyl diethyl ... trans-Chlorfenvinphos. *Formula: C12H14Cl3O4P ...
The toxicity of chlorfenvinphos is primarily caused by its inhibition of cholinesterase activity. Chlorfenvinphos reacts with ... This is mainly because chlorfenvinphos was not considered a dangerous toxin until the early 1990s. Therefore, chlorfenvinphos ... which have been exposed to chlorfenvinphos. These small amounts can be used to prove that chlorfenvinphos exposure has occurred ... Chlorfenvinphos has been included in many products since its first use in 1963. However, because of its toxic effect as a ...
See Also: Toxicological Abbreviations Chlorfenvinphos (ICSC) Chlorfenvinphos (WHO Pesticide Residues Series 1) Chlorfenvinphos ... CHLORFENVINPHOS Explanation Chlorfenvinphos was reviewed by the 1971 JMPR and MRLs were recommended for 24 commodities. At that ... At present chlorfenvinphos is registered for use on citrus in several countries to control scale insects such as Coccoidea and ... Table 1. Chlorfenvinphos registrations on citrus fruit Pre-harvest Country Formulation Interval (days) Chile 24% W/V EC 30 ...
Chlorfenvinphos. View. View. View. Insecticide. No. Yes. Related. 6. Chlorfenvinphos alpha. View. View. View. Insecticide. No. ... Chlorfenvinphos beta. View. View. View. Insecticide. No. Yes. Related. 13. Chlorfenvinphos total. View. View. View. Insecticide ... Chlorfenvinphos alpha Chlorfenvinphos Illinois EPA list. Keith list. Colborn list. Benbrook list. Danish Inert list. EU list. ... Chlorfenvinphos alpha Chlorfenvinphos WHO Acute Hazard. TRI Acute Hazard. Material Safety Data Sheets. Acute rating from U.S. ...
Chlorfenvinphos, alternative diagnoses, misdiagnosis, and correct diagnosis for Chemical poisoning - Chlorfenvinphos signs or ... Chlorfenvinphos including 52 medical symptoms and signs of Chemical poisoning - ... Research More About Chemical poisoning -- Chlorfenvinphos. Do I have Chemical poisoning -- Chlorfenvinphos? *Chemical poisoning ... Research symptoms & diagnosis of Chemical poisoning -- Chlorfenvinphos: *Overview -- Chemical poisoning -- Chlorfenvinphos. * ...
Chlorfenvinphos is an organophosphate insecticide, posing a risk to those who are professionally involved in its production and ... Renal activities of SOD, GPx and GR were more pronounced on day 28 of chlorfenvinphos exposure than on day 14. The kidney ... Chlorfenvinphos administration resulted in increased activities of antioxidative enzymes in the kidney of rats. ... 10] Lukaszewicz-Hussain A., Moniuszko-Jakoniuk J. A low dose of chlorfenvinphos affects hepatic enzymes in serum and ...
In the present study, the influence of chlorfenvinphos on the activity of mitochondrial antioxidative system as well as ... Chlorfenvinphos is an organophosphate insecticide widely used alone or in combination in Poland. ... Chlorfenvinphos is an organophosphate insecticide widely used alone or in combination in Poland. In the present study, the ... In acute chlorfenvinphos intoxication, we demonstrated that the key role in the oxidative mitochondrial damage play MnSOD and ...
2. The Commission shall establish the first watch list as referred to in paragraph 1 by 14 September 2014 and shall update it every 24 months thereafter. When updating the watch list, the Commission shall remove any substance for which a risk-based assessment as referred to in Article 16(2) of Directive 2000/60/EC can be concluded without additional monitoring data. The duration of a continuous watch list monitoring period for any individual substance shall not exceed four years.. 3. Member States shall monitor each substance in the watch list at selected representative monitoring stations over at least a 12-month period. For the first watch list, the monitoring period shall commence by 14 September 2015 or within six months of the establishment of the watch list, whichever is the later. For each substance included in subsequent lists, Member States shall commence monitoring within six months of its inclusion in the list.. Each Member State shall select at least one monitoring station, plus one ...
Chlorfenvinphos. JMPR no. (014) 1994 (T) Report. 1996 (R) Report, Evaluation. 1977 ...
The Commission shall, if appropriate, accompany the report with relevant proposals.. Article 8. Review of Annex X to Directive 2000/60/EC. Within the framework of the review of Annex X to Directive 2000/60/EC, as provided for in Article 16(4) of that Directive, the Commission shall consider inter alia the substances set out in Annex III to this Directive for possible identification as priority substances or priority hazardous substances. The Commission shall report the outcome of its review to the European Parliament and to the Council by 13 January 2011. It shall accompany the report, if appropriate, with relevant proposals, in particular proposals to identify new priority substances or priority hazardous substances or to identify certain priority substances as priority hazardous substances and to set corresponding EQS for surface water, sediment or biota, as appropriate.. Article 9. Committee procedure. 1. The Commission shall be assisted by the Committee referred to in Article 21(1) of ...
Tags: High Purity Chlorfenvinphos Tech , High Purity 470-90-6 (18708-86-6) Alibaba , High Purity Chlorfenvinphos Tech 470-90-6 ... Tags: Pesticide Chlorfenvinphos 92% Tech , Pesticide 470-90-6 (18708-86-6) Made In China , Pesticide Chlorfenvinphos 92% Tech ... manufacturer high purity CHLORFENVINPHOS TECH 470-90-6 (18708-86-6) made in china ...
Chlorfenvinphos. 5,465. 5,22. 0,999853. 96. Chlorprophame. 5,451. 6,38. 0,999397. 14. ...
This multi-residue method is a qualitative tool used to screen liver, GI contents and suspect material for possible toxins. It consists of a general extraction method and analysis of the extract by GC/MS (gas chromatography-mass spectrometry) and LC/MS (liquid chromatography-mass spectrometry) methods.. GC/MS is a method suitable for volatile/semi-volatile, heat stable compounds analysed in the gas phase. The GC/MS method is both targeted (looking for specific known compounds) and non-targeted (looking for unknown compounds). This method involves several targeted analyses (see Table 1) and a non-targeted analysis that screen the extract for compounds and subsequent match with a comprehensive mass spectrum library. Matches are based on the specific mass spectral fingerprint produced for a compound based on the standardized ionization energy of the GC/MS instrumentation.. LC/MS is a method suitable for non-volatile compounds analysed in the liquid phase. LC/MS in a targeted method (Table 1). ...
The present invention relates to the use of phosphated 2-propylheptanol, phosphated 2-proyplheptanol alkoxylate and/or mixtures thereof in agricultural formulations. The invention also relates to agri
Filed under: Chlorfenvinphos*. Toxicological profile for chlorfenvinphos : draft. ([Atlanta, Ga.] : U. S. Department of Health ... Toxicological profile for chlorfenvinphos /. ([Atlanta, Ga.] : U. S. Department of Health and Human Services, Public Health ... Toxicological profile for chlorfenvinphos : draft. ([Atlanta, Ga.] : U. S. Department of Health and Human Services, Public ...
Chlorfenvinphos. 0.5. Chlorpyrifos-ethyl. 0.2. Chlorpyrifos-methyl. 0.1. Chlorthal-dimethyl. 0.01. Cyfluthrin (sum of). 0.1. ...
Chlorfenvinphos; Crotoxyphos; Fensulfothion; Ethion; Famphur; Carbophenothion; Phosmet; Leptophos; Coumaphos ...
2 The method used for determining the information on the release or the transfer must be selected in such a way that the best available information is obtained; if possible, an internationally recognised method should be selected.. 3 The information must be entered directly into the confidential Register provided by the FOEN; by way of exception, the data may be submitted to the FOEN in another way. The FOEN decides on the format of the data.. 4 Anyone who has already submitted information in accordance with Article 5 paragraph 1 to the Confederation in compliance with other regulations may authorise the Confederation to enter that information in the Register in accordance with paragraph 3. The FOEN may request information from other federal agencies that has been obtained in compliance with other regulations and that is suitable for transfer to the Register and it shall maintain a list of such information. ...
Transport of the Insecticides Chlorpyrifos, Chlorfenvinphos, Carbofuran, Carbosulfan, and Furathiocarb From Soil Into the ... Home» Transport of the Insecticides Chlorpyrifos, Chlorfenvinphos, Carbofuran, Carbosulfan, and Furathiocarb From Soil Into the ... Transport of the Insecticides Chlorpyrifos, Chlorfenvinphos, Carbofuran, Carbosulfan, and Furathiocarb From Soil Into the ... Metabolism of chlorfenvinphos in both soil and plant of cauliflower and brussels sprouts field crops, 10.1007/bf01702066 ...
The genotoxicity of chlorfenvinphos and fenbuconazole in A. cepa root cells was analyzed using comet assay, which allows the ... In all concentrations, chlorfenvinphos and fenbuconazole induced a significant increase in DNA damage. Additionally, it was ... When the data that was accumulated via comet analysis from A. cepa root tip cells that are treated with type chlorfenvinphos ... Genotoxic effects of Chlorfenvinphos and fenbuconazole were examined by using mitotic index, mitotic phase, chromosomal ...
Chlorfenvinphos 0.5. Chlorpyrifos 0.2. Chlorpyrifos-methyl 0.1. Cypermethrin (and isomers) 1.0. DDT (sum of p,p-DDT, o,p-DDT ...
This list contains substances identified as priority substances that are targeted for reduction and eventual removal from waste water discharge. The list includes priority hazardous substances which are subject to special restrictions. ...
Evan E. Bolton; Yanli Wang; Paul A. Thiessen; Stephen H. Bryant (2008). „Chapter 12 PubChem: Integrated Platform of Small Molecules and Biological Activities". Annual Reports in Computational Chemistry. 4: 217-241. doi:10.1016/S1574-1400(08)00012-1 ...
7. Chemical poisoning -- Chlorfenvinphos. 8. Chemical poisoning -- Glaze. 9. Chemical poisoning -- Monocrotophos. 10. Chemical ...
Chlorfenvinphos, 10: Chlorpyriphos, 11: Cyfluthrin, 12: Cypermethrin, 13: Deltamethrin, 14: Diazinon, 15: Dichlofluanid, 16: ...
The third edition of the Encyclopedia of Toxicology presents entries devoted to key concepts and specific chemicals, and is updated to reflect current advances in the field. It contains new information on nanotoxicology, epigenetics, persistent organic pollutants, computational toxicology and bioinformatics, controversial chemicals, and much more.
  • and phosphates such as chlorfenvinphos, dichlorvos, and monocrotophos give rise to -phosphates (DMP or DEP). (thefreedictionary.com)
  • ROBERTS, S.J. (1997) The effects of soil pH and crop on the degradation of carbofuran and chlorfenvinphos in field microplots. (warwick.ac.uk)
  • The aquatic risk assessment revealed that from the various compounds analysed terbuthylazine, chlorfenvinphos and diazinon presented non-acceptable risk when maximum concentrations were used as the measured environmental concentrations (MEC). (unboundmedicine.com)
  • At pesticide exposures producing approximately 30% inhibition of AChE, rat plasma albumin binding in vitro by azamethiphos (oxon), chlorfenvinphos (oxon), chlorpyrifos-oxon, diazinon-oxon and malaoxon was reduced from controls by 9+/-1%, 67+/-2%, 56+/-2%, 54+/-2% and 8+/-1%, respectively. (nottingham.ac.uk)
  • In the current study, animals received once a day, intragastrically with a stomach tube, 0.1ml/100g of olive oil (control groups) and oil solution of chlorfenvinphos at a dose of 0.02LD50 (0.3 mg/kg b. w.) - the experimental groups. (edu.pl)
  • The animals were divided into two groups: the control group, which received oil intragastrically by stomach tube and the experimental groups, which received oil solution of chlorfenvinphos in doses of 0.02 LD 50 , 0.1 LD 50 or 0.5 LD 50 . (pjoes.com)
  • If you are exposed to chlorfenvinphos, many factors chapter from the Toxicological Profile for determine whether you'll be harmed. (cdc.gov)
  • Furthermore, chlorfenvinphos is corrosive to metal and hydrolyzes in the environment. (wikipedia.org)
  • There is no quantitative information on the total volume of chlorfenvinphos really used as a pesticide in the United States or elsewhere. (wikipedia.org)
  • The Smell of Death: An Unusual Case of Poisoning With the Organophosphorus Pesticide Chlorfenvinphos. (nih.gov)
  • Chlorfenvinphos is an organophosphate insecticide, posing a risk to those who are professionally involved in its production and use in agriculture, as well as to the general population. (edu.pl)
  • Chlorfenvinphos is an organophosphate insecticide widely used alone or in combination in Poland. (pjoes.com)
  • Chlorfenvinphos is an insecticide which is no longer used in the United States. (cdc.gov)
  • Chlorfenvinphos is an insecticide that is a colorless liquid with a mild odor. (cdc.gov)
  • Chlorfenvinphos is the common name of an organophosphorus insecticide used to control insect pests on livestock. (cdc.gov)
  • Chlorfenvinphos is the common name of an organophosphorus compound that was widely used as an insecticide and an acaricide. (wikipedia.org)
  • These ticks were tested for resistance against 3 compounds, namely amitraz, cypermethrin and chlorfenvinphos. (scielo.org.za)
  • Rhipicephalus evertsi evertsi was susceptible to amitraz and cypermethrin at all dip-tanks, but showed emerging resistance to chlorfenvinphos at 7 dip-tanks and resistance at 4. (scielo.org.za)
  • Rhipicephalus appendiculatus was susceptible to amitraz and chlorfenvinphos at all dip-tanks and demonstrated emerging resistance to cypermethrin at 1. (scielo.org.za)
  • Chlorfenvinphos administration resulted in increased activities of antioxidative enzymes in the kidney of rats. (edu.pl)
  • Most cases of unintentional chlorfenvinphos you eat food or drink water containing this poisoning have resulted from short exposures to substance, it may be absorbed from your stomach very high concentrations of this substance. (cdc.gov)
  • Most cases of unintentional chlorfenvinphos poisoning have resulted from short exposures to very high concentrations of this substance. (cdc.gov)
  • Chlorfenvinphos is applied as a foliar spray at concentrations generally between 0.04% and 0.06% active ingredient. (inchem.org)
  • Chlorfenvinphos and fenbuconazole significantly increased the abnormal cell frequency at all concentrations and treatment periods when compared with their controls. (cumhuriyet.edu.tr)
  • In all concentrations, chlorfenvinphos and fenbuconazole induced a significant increase in DNA damage. (cumhuriyet.edu.tr)
  • What happens to chlorfenvinphos when it enters the environment? (cdc.gov)
  • Chlorfenvinphos enters the environment from runoff after rainfall and leaching from hazardous waste sites. (cdc.gov)
  • When a substance is released from a large area, Chlorfenvinphos enters the environment from such as an industrial plant, or from a container, such runoff after rainfall and leaching from hazardous as a drum or bottle, it enters the environment. (cdc.gov)
  • The Environmental Protection Agency (EPA) identifies the most serious hazardous waste sites in The pure chemical (100% chlorfenvinphos) is a the nation. (cdc.gov)
  • The pure chemical (100% chlorfenvinphos) is a colorless liquid with a mild odor. (cdc.gov)
  • Chlorfenvinphos was first introduced in the United States in 1963, by the Shell International Chemical Company Ltd., Ciba AG (now Ciba-Geigy AG) and by Allied Chemical Corporation. (wikipedia.org)
  • However, no producers of chlorfenvinphos were identified in a 1993 Directory of Chemical Producers for the United States of America. (wikipedia.org)
  • The symptom information on this page attempts to provide a list of some possible signs and symptoms of Chemical poisoning -- Chlorfenvinphos . (rightdiagnosis.com)
  • This signs and symptoms information for Chemical poisoning -- Chlorfenvinphos has been gathered from various sources, may not be fully accurate, and may not be the full list of Chemical poisoning -- Chlorfenvinphos signs or Chemical poisoning -- Chlorfenvinphos symptoms. (rightdiagnosis.com)
  • Furthermore, signs and symptoms of Chemical poisoning -- Chlorfenvinphos may vary on an individual basis for each patient. (rightdiagnosis.com)
  • Only your doctor can provide adequate diagnosis of any signs or symptoms and whether they are indeed Chemical poisoning -- Chlorfenvinphos symptoms. (rightdiagnosis.com)
  • The current experimental findings indicate that subchronic administration of chlorfenvinphos leads to an adaptive response in the kidney of rats and this response is mostly due to reduced glutathione level and glutathione metabolism. (edu.pl)
  • RESIDUES IN FOOD AND THEIR EVALUATION USE PATTERN On citrus fruit The use of chlorfenvinphos on citrus has developed since the 1971 JMPR in several countries. (inchem.org)
  • In addition the Spanish government provided data on official supervised trials carried out on mandarins, oranges and lemons in four Spanish provinces in order to study chlorfenvinphos degradation over a 28-day post-application period (Table 3) and data on the final residue levels at harvest time in precocious fruits (Table 4). (inchem.org)
  • If you breathe air or touch soil near a hazardous waste site containing chlorfenvinphos, you could be exposed to it. (cdc.gov)
  • If you live in areas surrounding hazardous waste disposal sites or treatment facilities for chlorfenvinphos, you could be exposed to it by contact with soils, runoff water, surface water, or groundwater contaminated by spills or leaks on the site or facility. (cdc.gov)
  • There is no evidence that long-term exposure to small amounts of chlorfenvinphos causes any harmful health effects in people. (cdc.gov)
  • It does not specifically show exposure to chlorfenvinphos. (cdc.gov)
  • Chlorfenvinphos was sold under chlorfenvinphos and the effects of exposure. (cdc.gov)
  • harm you and because these sites may be sources of CHLORFENVINPHOS WHEN IT ENTERS exposure. (cdc.gov)
  • This public health statement tells you about chlorfenvinphos and the effects of exposure. (cdc.gov)
  • Renal activities of SOD, GPx and GR were more pronounced on day 28 of chlorfenvinphos exposure than on day 14. (edu.pl)
  • It was commonly used until 1991 when all products containing chlorfenvinphos as an active ingredient were canceled in the United States. (cdc.gov)
  • Since 1991, however, information on current production of chlorfenvinphos has been conflicting. (wikipedia.org)
  • One animal study reported decreased fertility in rats given chlorfenvinphos in their food, and another study reported that chlorfenvinphos interfered with the development of rats when the pregnant animals were fed chlorfenvinphos. (cdc.gov)
  • After dosing rats with pirimiphos-methyl (dimethoxy) or chlorfenvinphos (oxon) (diethoxy) pesticides, the resultant albumin binding were still significant 7 days after dosing. (nottingham.ac.uk)
  • EN] A reliable multi-residue method for determining gaseous and particulate phase pesticides in atmospheric samples has been developed. (upv.es)
  • In the early 1980s, chlorfenvinphos was registered for additional uses in a dust formulation for use in dog kennels and in dog collars for the control of fleas and ticks. (wikipedia.org)
  • 11] Lukaszewicz-Hussain A., Moniuszko-Jakoniuk J., Rogalska J. Assessment of lipid peroxidation in rat tissues in subacute chlorfenvinphos administration. (edu.pl)
  • Chlorfenvinphos was widely used to control household pests such as flies, fleas, and mice. (cdc.gov)
  • Chlorfenvinphos may move from soil to the air by evaporation. (cdc.gov)
  • Because of these effects, chlorfenvinphos was often used on farms to control adult flies in dairy barns, milk rooms, poultry houses and yards, and in other animal buildings. (wikipedia.org)
  • No international regulations exist for the use of chlorfenvinphos, although standards and guidelines have been set to protect people from the possible harmful effects of the toxin. (wikipedia.org)
  • Genotoxic effects of Chlorfenvinphos and fenbuconazole were examined by using mitotic index, mitotic phase, chromosomal abnormalities, 2C DNA content and Comet assay on the root meristem cells of Allium cepa. (cumhuriyet.edu.tr)
  • Another way to be exposed is by using pharmaceutical products that contain lanolin, a natural grease from sheep's wool (chlorfenvinphos is often used to control flies in animal buildings and can contaminate sheep's wool). (cdc.gov)
  • Specific tests are available to identify chlorfenvinphos or its breakdown products in your blood, body tissue, and urine. (cdc.gov)
  • The most common way for people to be exposed to chlorfenvinphos is by eating imported agricultural products contaminated with it and by using pharmaceutical products that contain lanolin. (cdc.gov)
  • Chlorfenvinphos has been included in many products since its first use in 1963. (wikipedia.org)
  • Since its first application, many manufacturers included chlorfenvinphos in their products. (wikipedia.org)
  • In the present study, the influence of chlorfenvinphos on the activity of mitochondrial antioxidative system as well as hydrogen peroxide concentration was examinated. (pjoes.com)
  • A study of Mytilus edulis exposed to the priority pollutant chlorfenvinphos. (usda.gov)
  • Chlorfenvinphos has been found in at least 1 of the 1,428 current or former NPL sites. (cdc.gov)
  • Chlorfenvinphos used to control flies in animal buildings and holding pens can contaminate sheep's wool. (cdc.gov)
  • At present chlorfenvinphos is registered for use on citrus in several countries to control scale insects such as Coccoidea and Diaspididae . (inchem.org)
  • The results indicated that Chlorfenvinphos and fenbuconazole significantly decreased the mitotic index in all treatments when compared with their controls. (cumhuriyet.edu.tr)
  • Most of the chlorfenvinphos used was in liquid form. (cdc.gov)
  • Most of Chlorfenvinphos has been found in at least 1 of the chlorfenvinphos was used in liquid form. (cdc.gov)
  • The insecticides, mostly used in liquid form, contain between 50% and 90% chlorfenvinphos. (wikipedia.org)
  • Chlorfenvinphos been found in at least 1 of the 1,430 National Priorities List sites identified by the Environmental Protection Agency (EPA). (cdc.gov)
  • The manufacturer provided a list of registrations for chlorfenvinphos on citrus and national pre-harvest intervals where these had been established. (inchem.org)
  • When the data that was accumulated via comet analysis from A. cepa root tip cells that are treated with type chlorfenvinphos and fenbuconazole, was compared to the data that was acquired as the result of the measurement of 2C DNA amount, a relation with negative correlation was found, (respectively, r = -0.80 and r = -0.82). (cumhuriyet.edu.tr)
  • Is there a medical test to show whether I've been exposed to chlorfenvinphos? (cdc.gov)
  • There is a general test that can be used to determine if you have been exposed to a group of insecticides, including chlorfenvinphos. (cdc.gov)
  • If you work in the disposal of chlorfenvinphos or its wastes you are most likely to be exposed. (cdc.gov)
  • Applications were made with chlorfenvinphos E.C. at dose rates between 1.1 and 2.4 kg/ha. (inchem.org)
  • sites with chlorfenvinphos may increase. (cdc.gov)
  • As more sites are evaluated, the sites with chlorfenvinphos may increase. (cdc.gov)