AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPsychiatry and PsychologyPhenomena and ProcessesInformation ScienceNamed GroupsHealth CareGeographicals
Cerebral HemorrhageSubarachnoid HemorrhageCerebral InfarctionHemorrhageCerebral ArteriesCerebral Amyloid AngiopathyCerebrovascular DisordersIntracranial HemorrhagesIntracranial Hemorrhage, HypertensiveCapillary FragilityCerebral AngiographyCerebral Amyloid Angiopathy, FamilialMiddle Cerebral ArteryCerebrovascular CirculationBrainTomography, X-Ray ComputedAmyloidosisBrain IschemiaIntracranial Embolism and ThrombosisStrokeHematomaRetinal HemorrhageIntracranial AneurysmHemangioma, Cavernous, Central Nervous SystemCerebral Hemorrhage, TraumaticTissue Plasminogen ActivatorCerebral PalsyIntracranial Arteriovenous MalformationsAphasia, WernickeCystatinsPostpartum HemorrhageRupture, SpontaneousIschemic Attack, TransientJapanBrain EdemaTime FactorsGastrointestinal HemorrhageFatal OutcomeStreptococcus anginosusMagnetic Resonance ImagingHypertensionFibrinolytic AgentsVitreous HemorrhageEye HemorrhageTreatment OutcomeAcute DiseaseNeurologic ExaminationAutonomic DysreflexiaInfarction, Middle Cerebral ArteryMoyamoya DiseaseVasospasm, IntracranialCerebral VeinsRisk FactorsMalaria, CerebralAmyloid beta-PeptidesAnticoagulantsThrombolytic TherapyCystatin CAmyloidRetrospective StudiesProspective StudiesBlood PressureIntracranial EmbolismCerebral VentriclesAlzheimer DiseaseBasal Ganglia HemorrhageDisease Models, AnimalFollow-Up StudiesIncidenceAmyloid beta-Protein PrecursorPostoperative HemorrhageInfant, NewbornAge FactorsRecurrenceRiskPostoperative ComplicationsAneurysm, RupturedBasilar ArteryChoroid HemorrhagePeptic Ulcer HemorrhageIntracranial PressureAnterior Cerebral ArteryPrognosisPosterior Cerebral ArterySex FactorsPutaminal HemorrhageBrain DiseasesMutationHydrocephalusBlood Flow VelocityHematoma, SubduralRetrobulbar HemorrhageCerebral RevascularizationBlood VolumeEmbolization, TherapeuticCraniotomyCerebral VentriculographyDominance, CerebralBrain Hemorrhage, TraumaticShock, HemorrhagicRats, Sprague-DawleyNeuroprotective AgentsIntracranial ThrombosisCerebrumBlood-Brain BarrierIntracranial HypertensionHypoxia, BrainGlasgow Coma ScaleMagnetic Resonance Angiography
Cerebral Hemorrhage: Bleeding into one or both CEREBRAL HEMISPHERES including the BASAL GANGLIA and the CEREBRAL CORTEX. It is often associated with HYPERTENSION and CRANIOCEREBRAL TRAUMA.Subarachnoid Hemorrhage: Bleeding into the intracranial or spinal SUBARACHNOID SPACE, most resulting from INTRACRANIAL ANEURYSM rupture. It can occur after traumatic injuries (SUBARACHNOID HEMORRHAGE, TRAUMATIC). Clinical features include HEADACHE; NAUSEA; VOMITING, nuchal rigidity, variable neurological deficits and reduced mental status.Cerebral Infarction: The formation of an area of NECROSIS in the CEREBRUM caused by an insufficiency of arterial or venous blood flow. Infarcts of the cerebrum are generally classified by hemisphere (i.e., left vs. right), lobe (e.g., frontal lobe infarction), arterial distribution (e.g., INFARCTION, ANTERIOR CEREBRAL ARTERY), and etiology (e.g., embolic infarction).Hemorrhage: Bleeding or escape of blood from a vessel.Cerebral Arteries: The arterial blood vessels supplying the CEREBRUM.Cerebral Amyloid Angiopathy: A heterogeneous group of sporadic or familial disorders characterized by AMYLOID deposits in the walls of small and medium sized blood vessels of CEREBRAL CORTEX and MENINGES. Clinical features include multiple, small lobar CEREBRAL HEMORRHAGE; cerebral ischemia (BRAIN ISCHEMIA); and CEREBRAL INFARCTION. Cerebral amyloid angiopathy is unrelated to generalized AMYLOIDOSIS. Amyloidogenic peptides in this condition are nearly always the same ones found in ALZHEIMER DISEASE. (from Kumar: Robbins and Cotran: Pathologic Basis of Disease, 7th ed., 2005)Cerebrovascular Disorders: A spectrum of pathological conditions of impaired blood flow in the brain. They can involve vessels (ARTERIES or VEINS) in the CEREBRUM, the CEREBELLUM, and the BRAIN STEM. Major categories include INTRACRANIAL ARTERIOVENOUS MALFORMATIONS; BRAIN ISCHEMIA; CEREBRAL HEMORRHAGE; and others.Intracranial Hemorrhages: Bleeding within the SKULL, including hemorrhages in the brain and the three membranes of MENINGES. The escape of blood often leads to the formation of HEMATOMA in the cranial epidural, subdural, and subarachnoid spaces.Intracranial Hemorrhage, Hypertensive: Bleeding within the SKULL that is caused by systemic HYPERTENSION, usually in association with INTRACRANIAL ARTERIOSCLEROSIS. Hypertensive hemorrhages are most frequent in the BASAL GANGLIA; CEREBELLUM; PONS; and THALAMUS; but may also involve the CEREBRAL CORTEX, subcortical white matter, and other brain structures.Capillary Fragility: The susceptibility of CAPILLARIES, under conditions of increased stress, to leakage.Cerebral Angiography: Radiography of the vascular system of the brain after injection of a contrast medium.Cerebral Amyloid Angiopathy, Familial: A familial disorder marked by AMYLOID deposits in the walls of small and medium sized blood vessels of CEREBRAL CORTEX and MENINGES.Middle Cerebral Artery: The largest of the cerebral arteries. It trifurcates into temporal, frontal, and parietal branches supplying blood to most of the parenchyma of these lobes in the CEREBRAL CORTEX. These are the areas involved in motor, sensory, and speech activities.Cerebrovascular Circulation: The circulation of blood through the BLOOD VESSELS of the BRAIN.Brain: The part of CENTRAL NERVOUS SYSTEM that is contained within the skull (CRANIUM). Arising from the NEURAL TUBE, the embryonic brain is comprised of three major parts including PROSENCEPHALON (the forebrain); MESENCEPHALON (the midbrain); and RHOMBENCEPHALON (the hindbrain). The developed brain consists of CEREBRUM; CEREBELLUM; and other structures in the BRAIN STEM.Tomography, X-Ray Computed: Tomography using x-ray transmission and a computer algorithm to reconstruct the image.Amyloidosis: A group of sporadic, familial and/or inherited, degenerative, and infectious disease processes, linked by the common theme of abnormal protein folding and deposition of AMYLOID. As the amyloid deposits enlarge they displace normal tissue structures, causing disruption of function. Various signs and symptoms depend on the location and size of the deposits.Brain Ischemia: Localized reduction of blood flow to brain tissue due to arterial obstruction or systemic hypoperfusion. This frequently occurs in conjunction with brain hypoxia (HYPOXIA, BRAIN). Prolonged ischemia is associated with BRAIN INFARCTION.Intracranial Embolism and Thrombosis: Embolism or thrombosis involving blood vessels which supply intracranial structures. Emboli may originate from extracranial or intracranial sources. Thrombosis may occur in arterial or venous structures.Stroke: A group of pathological conditions characterized by sudden, non-convulsive loss of neurological function due to BRAIN ISCHEMIA or INTRACRANIAL HEMORRHAGES. Stroke is classified by the type of tissue NECROSIS, such as the anatomic location, vasculature involved, etiology, age of the affected individual, and hemorrhagic vs. non-hemorrhagic nature. (From Adams et al., Principles of Neurology, 6th ed, pp777-810)Hematoma: A collection of blood outside the BLOOD VESSELS. Hematoma can be localized in an organ, space, or tissue.Retinal Hemorrhage: Bleeding from the vessels of the retina.Intracranial Aneurysm: Abnormal outpouching in the wall of intracranial blood vessels. Most common are the saccular (berry) aneurysms located at branch points in CIRCLE OF WILLIS at the base of the brain. Vessel rupture results in SUBARACHNOID HEMORRHAGE or INTRACRANIAL HEMORRHAGES. Giant aneurysms (>2.5 cm in diameter) may compress adjacent structures, including the OCULOMOTOR NERVE. (From Adams et al., Principles of Neurology, 6th ed, p841)Hemangioma, Cavernous, Central Nervous System: A vascular anomaly composed of a collection of large, thin walled tortuous VEINS that can occur in any part of the central nervous system but lack intervening nervous tissue. Familial occurrence is common and has been associated with a number of genes mapped to 7q, 7p and 3q. Clinical features include SEIZURES; HEADACHE; STROKE; and progressive neurological deficit.Cerebral Hemorrhage, Traumatic: Bleeding into one or both CEREBRAL HEMISPHERES due to TRAUMA. Hemorrhage may involve any part of the CEREBRAL CORTEX and the BASAL GANGLIA. Depending on the severity of bleeding, clinical features may include SEIZURES; APHASIA; VISION DISORDERS; MOVEMENT DISORDERS; PARALYSIS; and COMA.Tissue Plasminogen Activator: A proteolytic enzyme in the serine protease family found in many tissues which converts PLASMINOGEN to FIBRINOLYSIN. It has fibrin-binding activity and is immunologically different from UROKINASE-TYPE PLASMINOGEN ACTIVATOR. The primary sequence, composed of 527 amino acids, is identical in both the naturally occurring and synthetic proteases.Cerebral Palsy: A heterogeneous group of nonprogressive motor disorders caused by chronic brain injuries that originate in the prenatal period, perinatal period, or first few years of life. The four major subtypes are spastic, athetoid, ataxic, and mixed cerebral palsy, with spastic forms being the most common. The motor disorder may range from difficulties with fine motor control to severe spasticity (see MUSCLE SPASTICITY) in all limbs. Spastic diplegia (Little disease) is the most common subtype, and is characterized by spasticity that is more prominent in the legs than in the arms. Pathologically, this condition may be associated with LEUKOMALACIA, PERIVENTRICULAR. (From Dev Med Child Neurol 1998 Aug;40(8):520-7)Intracranial Arteriovenous Malformations: Congenital vascular anomalies in the brain characterized by direct communication between an artery and a vein without passing through the CAPILLARIES. The locations and size of the shunts determine the symptoms including HEADACHES; SEIZURES; STROKE; INTRACRANIAL HEMORRHAGES; mass effect; and vascular steal effect.Aphasia, Wernicke: Impairment in the comprehension of speech and meaning of words, both spoken and written, and of the meanings conveyed by their grammatical relationships in sentences. It is caused by lesions that primarily affect Wernicke's area, which lies in the posterior perisylvian region of the temporal lobe of the dominant hemisphere. (From Brain & Bannister, Clinical Neurology, 7th ed, p141; Kandel et al., Principles of Neural Science, 3d ed, p846)Cystatins: A homologous group of endogenous CYSTEINE PROTEINASE INHIBITORS. The cystatins inhibit most CYSTEINE ENDOPEPTIDASES such as PAPAIN, and other peptidases which have a sulfhydryl group at the active site.Postpartum Hemorrhage: Excess blood loss from uterine bleeding associated with OBSTETRIC LABOR or CHILDBIRTH. It is defined as blood loss greater than 500 ml or of the amount that adversely affects the maternal physiology, such as BLOOD PRESSURE and HEMATOCRIT. Postpartum hemorrhage is divided into two categories, immediate (within first 24 hours after birth) or delayed (after 24 hours postpartum).Rupture, Spontaneous: Tear or break of an organ, vessel or other soft part of the body, occurring in the absence of external force.Ischemic Attack, Transient: Brief reversible episodes of focal, nonconvulsive ischemic dysfunction of the brain having a duration of less than 24 hours, and usually less than one hour, caused by transient thrombotic or embolic blood vessel occlusion or stenosis. Events may be classified by arterial distribution, temporal pattern, or etiology (e.g., embolic vs. thrombotic). (From Adams et al., Principles of Neurology, 6th ed, pp814-6)JapanBrain Edema: Increased intracellular or extracellular fluid in brain tissue. Cytotoxic brain edema (swelling due to increased intracellular fluid) is indicative of a disturbance in cell metabolism, and is commonly associated with hypoxic or ischemic injuries (see HYPOXIA, BRAIN). An increase in extracellular fluid may be caused by increased brain capillary permeability (vasogenic edema), an osmotic gradient, local blockages in interstitial fluid pathways, or by obstruction of CSF flow (e.g., obstructive HYDROCEPHALUS). (From Childs Nerv Syst 1992 Sep; 8(6):301-6)Time Factors: Elements of limited time intervals, contributing to particular results or situations.Gastrointestinal Hemorrhage: Bleeding in any segment of the GASTROINTESTINAL TRACT from ESOPHAGUS to RECTUM.Fatal Outcome: Death resulting from the presence of a disease in an individual, as shown by a single case report or a limited number of patients. This should be differentiated from DEATH, the physiological cessation of life and from MORTALITY, an epidemiological or statistical concept.Streptococcus anginosus: A species of gram-positive bacteria in the STREPTOCOCCUS MILLERI GROUP. It is the most frequently seen isolate of that group, has a proclivity for abscess formation, and is most often isolated from the blood, gastrointestinal, and urogenital tract.Magnetic Resonance Imaging: Non-invasive method of demonstrating internal anatomy based on the principle that atomic nuclei in a strong magnetic field absorb pulses of radiofrequency energy and emit them as radiowaves which can be reconstructed into computerized images. The concept includes proton spin tomographic techniques.Hypertension: Persistently high systemic arterial BLOOD PRESSURE. Based on multiple readings (BLOOD PRESSURE DETERMINATION), hypertension is currently defined as when SYSTOLIC PRESSURE is consistently greater than 140 mm Hg or when DIASTOLIC PRESSURE is consistently 90 mm Hg or more.Fibrinolytic Agents: Fibrinolysin or agents that convert plasminogen to FIBRINOLYSIN.Vitreous Hemorrhage: Hemorrhage into the VITREOUS BODY.Eye Hemorrhage: Intraocular hemorrhage from the vessels of various tissues of the eye.Treatment Outcome: Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.Acute Disease: Disease having a short and relatively severe course.Neurologic Examination: Assessment of sensory and motor responses and reflexes that is used to determine impairment of the nervous system.Autonomic Dysreflexia: A syndrome associated with damage to the spinal cord above the mid thoracic level (see SPINAL CORD INJURIES) characterized by a marked increase in the sympathetic response to minor stimuli such as bladder or rectal distention. Manifestations include HYPERTENSION; TACHYCARDIA (or reflex bradycardia); FEVER; FLUSHING; and HYPERHIDROSIS. Extreme hypertension may be associated with a STROKE. (From Adams et al., Principles of Neurology, 6th ed, pp538 and 1232; J Spinal Cord Med 1997;20(3):355-60)Infarction, Middle Cerebral Artery: NECROSIS occurring in the MIDDLE CEREBRAL ARTERY distribution system which brings blood to the entire lateral aspects of each CEREBRAL HEMISPHERE. Clinical signs include impaired cognition; APHASIA; AGRAPHIA; weak and numbness in the face and arms, contralaterally or bilaterally depending on the infarction.Moyamoya Disease: A noninflammatory, progressive occlusion of the intracranial CAROTID ARTERIES and the formation of netlike collateral arteries arising from the CIRCLE OF WILLIS. Cerebral angiogram shows the puff-of-smoke (moyamoya) collaterals at the base of the brain. It is characterized by endothelial HYPERPLASIA and FIBROSIS with thickening of arterial walls. This disease primarily affects children but can also occur in adults.Vasospasm, Intracranial: Constriction of arteries in the SKULL due to sudden, sharp, and often persistent smooth muscle contraction in blood vessels. Intracranial vasospasm results in reduced vessel lumen caliber, restricted blood flow to the brain, and BRAIN ISCHEMIA that may lead to hypoxic-ischemic brain injury (HYPOXIA-ISCHEMIA, BRAIN).Cerebral Veins: Veins draining the cerebrum.Risk Factors: An aspect of personal behavior or lifestyle, environmental exposure, or inborn or inherited characteristic, which, on the basis of epidemiologic evidence, is known to be associated with a health-related condition considered important to prevent.Malaria, Cerebral: A condition characterized by somnolence or coma in the presence of an acute infection with PLASMODIUM FALCIPARUM (and rarely other Plasmodium species). Initial clinical manifestations include HEADACHES; SEIZURES; and alterations of mentation followed by a rapid progression to COMA. Pathologic features include cerebral capillaries filled with parasitized erythrocytes and multiple small foci of cortical and subcortical necrosis. (From Adams et al., Principles of Neurology, 6th ed, p136)Amyloid beta-Peptides: Peptides generated from AMYLOID BETA-PEPTIDES PRECURSOR. An amyloid fibrillar form of these peptides is the major component of amyloid plaques found in individuals with Alzheimer's disease and in aged individuals with trisomy 21 (DOWN SYNDROME). The peptide is found predominantly in the nervous system, but there have been reports of its presence in non-neural tissue.Anticoagulants: Agents that prevent clotting.Thrombolytic Therapy: Use of infusions of FIBRINOLYTIC AGENTS to destroy or dissolve thrombi in blood vessels or bypass grafts.Cystatin C: An extracellular cystatin subtype that is abundantly expressed in bodily fluids. It may play a role in the inhibition of interstitial CYSTEINE PROTEASES.Amyloid: A fibrous protein complex that consists of proteins folded into a specific cross beta-pleated sheet structure. This fibrillar structure has been found as an alternative folding pattern for a variety of functional proteins. Deposits of amyloid in the form of AMYLOID PLAQUES are associated with a variety of degenerative diseases. The amyloid structure has also been found in a number of functional proteins that are unrelated to disease.Retrospective Studies: Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.Prospective Studies: Observation of a population for a sufficient number of persons over a sufficient number of years to generate incidence or mortality rates subsequent to the selection of the study group.Blood Pressure: PRESSURE of the BLOOD on the ARTERIES and other BLOOD VESSELS.Intracranial Embolism: Blocking of a blood vessel in the SKULL by an EMBOLUS which can be a blood clot (THROMBUS) or other undissolved material in the blood stream. Most emboli are of cardiac origin and are associated with HEART DISEASES. Other non-cardiac sources of emboli are usually associated with VASCULAR DISEASES.Cerebral Ventricles: Four CSF-filled (see CEREBROSPINAL FLUID) cavities within the cerebral hemispheres (LATERAL VENTRICLES), in the midline (THIRD VENTRICLE) and within the PONS and MEDULLA OBLONGATA (FOURTH VENTRICLE).Alzheimer Disease: A degenerative disease of the BRAIN characterized by the insidious onset of DEMENTIA. Impairment of MEMORY, judgment, attention span, and problem solving skills are followed by severe APRAXIAS and a global loss of cognitive abilities. The condition primarily occurs after age 60, and is marked pathologically by severe cortical atrophy and the triad of SENILE PLAQUES; NEUROFIBRILLARY TANGLES; and NEUROPIL THREADS. (From Adams et al., Principles of Neurology, 6th ed, pp1049-57)Basal Ganglia Hemorrhage: Bleeding within the subcortical regions of cerebral hemispheres (BASAL GANGLIA). It is often associated with HYPERTENSION or ARTERIOVENOUS MALFORMATIONS. Clinical manifestations may include HEADACHE; DYSKINESIAS; and HEMIPARESIS.Disease Models, Animal: Naturally occurring or experimentally induced animal diseases with pathological processes sufficiently similar to those of human diseases. They are used as study models for human diseases.Follow-Up Studies: Studies in which individuals or populations are followed to assess the outcome of exposures, procedures, or effects of a characteristic, e.g., occurrence of disease.Incidence: The number of new cases of a given disease during a given period in a specified population. It also is used for the rate at which new events occur in a defined population. It is differentiated from PREVALENCE, which refers to all cases, new or old, in the population at a given time.Amyloid beta-Protein Precursor: A single-pass type I membrane protein. It is cleaved by AMYLOID PRECURSOR PROTEIN SECRETASES to produce peptides of varying amino acid lengths. A 39-42 amino acid peptide, AMYLOID BETA-PEPTIDES is a principal component of the extracellular amyloid in SENILE PLAQUES.Postoperative Hemorrhage: Hemorrhage following any surgical procedure. It may be immediate or delayed and is not restricted to the surgical wound.Infant, Newborn: An infant during the first month after birth.Age Factors: Age as a constituent element or influence contributing to the production of a result. It may be applicable to the cause or the effect of a circumstance. It is used with human or animal concepts but should be differentiated from AGING, a physiological process, and TIME FACTORS which refers only to the passage of time.Recurrence: The return of a sign, symptom, or disease after a remission.Risk: The probability that an event will occur. It encompasses a variety of measures of the probability of a generally unfavorable outcome.Postoperative Complications: Pathologic processes that affect patients after a surgical procedure. They may or may not be related to the disease for which the surgery was done, and they may or may not be direct results of the surgery.Aneurysm, Ruptured: The tearing or bursting of the weakened wall of the aneurysmal sac, usually heralded by sudden worsening pain. The great danger of a ruptured aneurysm is the large amount of blood spilling into the surrounding tissues and cavities, causing HEMORRHAGIC SHOCK.Basilar Artery: The artery formed by the union of the right and left vertebral arteries; it runs from the lower to the upper border of the pons, where it bifurcates into the two posterior cerebral arteries.Choroid Hemorrhage: Hemorrhage from the vessels of the choroid.Peptic Ulcer Hemorrhage: Bleeding from a PEPTIC ULCER that can be located in any segment of the GASTROINTESTINAL TRACT.Intracranial Pressure: Pressure within the cranial cavity. It is influenced by brain mass, the circulatory system, CSF dynamics, and skull rigidity.Anterior Cerebral Artery: Artery formed by the bifurcation of the internal carotid artery (CAROTID ARTERY, INTERNAL). Branches of the anterior cerebral artery supply the CAUDATE NUCLEUS; INTERNAL CAPSULE; PUTAMEN; SEPTAL NUCLEI; GYRUS CINGULI; and surfaces of the FRONTAL LOBE and PARIETAL LOBE.Prognosis: A prediction of the probable outcome of a disease based on a individual's condition and the usual course of the disease as seen in similar situations.Posterior Cerebral Artery: Artery formed by the bifurcation of the BASILAR ARTERY. Branches of the posterior cerebral artery supply portions of the OCCIPITAL LOBE; PARIETAL LOBE; inferior temporal gyrus, brainstem, and CHOROID PLEXUS.Sex Factors: Maleness or femaleness as a constituent element or influence contributing to the production of a result. It may be applicable to the cause or effect of a circumstance. It is used with human or animal concepts but should be differentiated from SEX CHARACTERISTICS, anatomical or physiological manifestations of sex, and from SEX DISTRIBUTION, the number of males and females in given circumstances.Putaminal Hemorrhage: Intracranial bleeding into the PUTAMEN, a BASAL GANGLIA nucleus. This is associated with HYPERTENSION and lipohyalinosis of small blood vessels in the putamen. Clinical manifestations vary with the size of hemorrhage, but include HEMIPARESIS; HEADACHE; and alterations of consciousness.Brain Diseases: Pathologic conditions affecting the BRAIN, which is composed of the intracranial components of the CENTRAL NERVOUS SYSTEM. This includes (but is not limited to) the CEREBRAL CORTEX; intracranial white matter; BASAL GANGLIA; THALAMUS; HYPOTHALAMUS; BRAIN STEM; and CEREBELLUM.Mutation: Any detectable and heritable change in the genetic material that causes a change in the GENOTYPE and which is transmitted to daughter cells and to succeeding generations.Hydrocephalus: Excessive accumulation of cerebrospinal fluid within the cranium which may be associated with dilation of cerebral ventricles, INTRACRANIAL HYPERTENSION; HEADACHE; lethargy; URINARY INCONTINENCE; and ATAXIA.Blood Flow Velocity: A value equal to the total volume flow divided by the cross-sectional area of the vascular bed.Hematoma, Subdural: Accumulation of blood in the SUBDURAL SPACE between the DURA MATER and the arachnoidal layer of the MENINGES. This condition primarily occurs over the surface of a CEREBRAL HEMISPHERE, but may develop in the spinal canal (HEMATOMA, SUBDURAL, SPINAL). Subdural hematoma can be classified as the acute or the chronic form, with immediate or delayed symptom onset, respectively. Symptoms may include loss of consciousness, severe HEADACHE, and deteriorating mental status.Retrobulbar Hemorrhage: Hemorrhage within the orbital cavity, posterior to the eyeball.Cerebral Revascularization: Microsurgical revascularization to improve intracranial circulation. It usually involves joining the extracranial circulation to the intracranial circulation but may include extracranial revascularization (e.g., subclavian-vertebral artery bypass, subclavian-external carotid artery bypass). It is performed by joining two arteries (direct anastomosis or use of graft) or by free autologous transplantation of highly vascularized tissue to the surface of the brain.Blood Volume: Volume of circulating BLOOD. It is the sum of the PLASMA VOLUME and ERYTHROCYTE VOLUME.Embolization, Therapeutic: A method of hemostasis utilizing various agents such as Gelfoam, silastic, metal, glass, or plastic pellets, autologous clot, fat, and muscle as emboli. It has been used in the treatment of spinal cord and INTRACRANIAL ARTERIOVENOUS MALFORMATIONS, renal arteriovenous fistulas, gastrointestinal bleeding, epistaxis, hypersplenism, certain highly vascular tumors, traumatic rupture of blood vessels, and control of operative hemorrhage.Craniotomy: Any operation on the cranium or incision into the cranium. (Dorland, 28th ed)Cerebral Ventriculography: Radiography of the ventricular system of the brain after injection of air or other contrast medium directly into the cerebral ventricles. It is used also for x-ray computed tomography of the cerebral ventricles.Dominance, Cerebral: Dominance of one cerebral hemisphere over the other in cerebral functions.Brain Hemorrhage, Traumatic: Bleeding within the brain as a result of penetrating and nonpenetrating CRANIOCEREBRAL TRAUMA. Traumatically induced hemorrhages may occur in any area of the brain, including the CEREBRUM; BRAIN STEM (see BRAIN STEM HEMORRHAGE, TRAUMATIC); and CEREBELLUM.Shock, Hemorrhagic: Acute hemorrhage or excessive fluid loss resulting in HYPOVOLEMIA.Rats, Sprague-Dawley: A strain of albino rat used widely for experimental purposes because of its calmness and ease of handling. It was developed by the Sprague-Dawley Animal Company.Neuroprotective Agents: Drugs intended to prevent damage to the brain or spinal cord from ischemia, stroke, convulsions, or trauma. Some must be administered before the event, but others may be effective for some time after. They act by a variety of mechanisms, but often directly or indirectly minimize the damage produced by endogenous excitatory amino acids.Intracranial Thrombosis: Formation or presence of a blood clot (THROMBUS) in a blood vessel within the SKULL. Intracranial thrombosis can lead to thrombotic occlusions and BRAIN INFARCTION. The majority of the thrombotic occlusions are associated with ATHEROSCLEROSIS.Cerebrum: Derived from TELENCEPHALON, cerebrum is composed of a right and a left hemisphere. Each contains an outer cerebral cortex and a subcortical basal ganglia. The cerebrum includes all parts within the skull except the MEDULLA OBLONGATA, the PONS, and the CEREBELLUM. Cerebral functions include sensorimotor, emotional, and intellectual activities.Blood-Brain Barrier: Specialized non-fenestrated tightly-joined ENDOTHELIAL CELLS with TIGHT JUNCTIONS that form a transport barrier for certain substances between the cerebral capillaries and the BRAIN tissue.Intracranial Hypertension: Increased pressure within the cranial vault. This may result from several conditions, including HYDROCEPHALUS; BRAIN EDEMA; intracranial masses; severe systemic HYPERTENSION; PSEUDOTUMOR CEREBRI; and other disorders.Hypoxia, Brain: A reduction in brain oxygen supply due to ANOXEMIA (a reduced amount of oxygen being carried in the blood by HEMOGLOBIN), or to a restriction of the blood supply to the brain, or both. Severe hypoxia is referred to as anoxia, and is a relatively common cause of injury to the central nervous system. Prolonged brain anoxia may lead to BRAIN DEATH or a PERSISTENT VEGETATIVE STATE. Histologically, this condition is characterized by neuronal loss which is most prominent in the HIPPOCAMPUS; GLOBUS PALLIDUS; CEREBELLUM; and inferior olives.Glasgow Coma Scale: A scale that assesses the response to stimuli in patients with craniocerebral injuries. The parameters are eye opening, motor response, and verbal response.Magnetic Resonance Angiography: Non-invasive method of vascular imaging and determination of internal anatomy without injection of contrast media or radiation exposure. The technique is used especially in CEREBRAL ANGIOGRAPHY as well as for studies of other vascular structures.

Suicide after traumatic brain injury: a population study. (1/39)

OBJECTIVES: To determine the rates of suicide among patients who have had a traumatic brain injury. METHODS: From a Danish population register of admissions to hospital covering the years 1979-93 patients were selected who had had either a concussion (n=126 114), a cranial fracture (n=7560), or a cerebral contusion or traumatic intracranial haemorrhage (n=11 766). All cases of deaths by the end of the study period were identified. RESULTS: In the three diagnostic groups there had been 750 (0.59%), 46 (0.61%), and 99 (0.84%) cases of suicide respectively. Standardised mortality ratios, stratified by sex and age, showed that the incidence of suicide among the three diagnostic groups was increased relative to the general population (3.0, 2.7, and 4.1 respectively). In all diagnosis groups the ratios were higher for females than for males, and lower for patients injured before the age of 21 or after the age of 60. The presence of a codiagnosis relating to substance misuse was associated with increased suicide rates in all diagnosis groups. There was a tendency, among patients with cerebral contusions or traumatic intracranial haemorrhages, for suicide risk to increase with duration of stay in hospital. Cox regression analyses for proportional hazards confirmed that there was a significantly greater risk of suicide among patients with cerebral contusions or traumatic intracranial haemorrhages than among patients with concussion or cranial fractures (hazard ratios=1.42 and 1.50 respectively). There was, however, no evidence of a specific risk period for suicide after injury. CONCLUSION: The increased risk of suicide among patients who had a mild traumatic brain injury may result from concomitant risk factors such as psychiatric conditions and psychosocial disadvantage. The greater risk among the more serious cases implicates additionally the physical, psychological, and social consequences of the injuries as directly contributing to the suicides.  (+info)

The structural basis of moderate disability after traumatic brain damage. (2/39)

The objective was to discover the nature of brain damage in survivors of head injury who are left with moderate disability. Macroscopic and microscopic examination was carried out on the brains of 20 persons who had died long after a head injury that had been treated in a neurosurgical unit. All had become independent but had various disabilities (moderate disability on the Glasgow outcome scale) Most deaths had been sudden, which had led to their referral from forensic pathologists. Post-traumatic epilepsy was a feature in 75%. An intracranial haematoma had been evacuated in 75%, and in 11 of the 15 with epilepsy. Diffuse axonal injury was found in six patients, five of the mildest type (grade 1) and one of grade 2. No patient had diffuse thalamic damage but one had a small focal ischaemic lesion in the thalamus. No patient had severe ischaemic brain damage, but three had moderate lesions which were bilateral in only one. No patient had severe cortical contusions. In conclusion, the dominant lesion was focal damage from an evacuated intracranial haematoma. Severe diffuse damage was not found, with diffuse axonal injury only mild and thalamic damage in only one patient.  (+info)

Giant cell glioblastoma manifesting as traumatic intracerebral hemorrhage--case report. (3/39)

A 33-year-old male presented with intracerebral hemorrhage in the left temporoparietal region after a traffic accident. Ten months later, the traumatic hemorrhage was found to originate in an underlying giant cell glioblastoma. Our case indicates that non-traumatic underlying pathologies, such as vasculopathies, coagulopathies, or tumors, should be considered in the differential diagnoses of intracerebral hemorrhage occurring in unusual locations after traumatic accidents.  (+info)

Magnetization transfer MR imaging in patients with posttraumatic epilepsy. (4/39)

BACKGROUND AND PURPOSE: Intractable epilepsy is a well-recognized complication following head trauma, and many factors have been implicated in its pathogenesis. This study was performed to determine the severity of tissue damage after severe head injury as assessed with magnetization transfer (MT) MR imaging and the relationship of this damage with seizure intractability. METHODS: Forty-four patients, 13 without seizures (disease controls) and 31 with seizures, underwent T1-weighted MT MR imaging 1-10 years after head trauma. Phase-corrected gradient-echo (GRE) imaging was also performed in all patients to look for the presence of hemosiderin. All patients were evaluated for the presence of an MT abnormality beyond an abnormality seen on T2-weighted images, an MT abnormality within a T2 abnormality, and hemosiderin deposition. RESULTS: Patients with an MT abnormality beyond a T2 abnormality had a significantly higher intractability of seizures compared with those with an MT abnormality within a T2 abnormality (P <.05). In addition, the mere presence of hemosiderin deposit was not associated with seizure intractability; however, gliosis around the hemosiderin as seen on T1-weighted MT images was associated with seizure intractability. CONCLUSIONS: T1-weighted MT imaging may be of value in predicting the intractability of the seizure in delayed posttraumatic epilepsy.  (+info)

Acute fatal haemorrhage during percutaneous dilatational tracheostomy. (5/39)

Percutaneous dilatational tracheostomy (PDT) is associated with a number of life-threatening complications. We present a case of massive and fatal arterial haemorrhage that occurred in the intensive care unit during an elective PDT on an 86-year-old woman following earlier evacuation of a traumatic subdural haematoma. An avulsed right subclavian artery was found at post mortem. Previous thyroid surgery and aberrant arterial anatomy contributed to the fatal outcome.  (+info)

Low doses of dexamethasone decrease brain water content of collagenase-induced cerebral hematoma. (6/39)

Different doses of dexamethasone were evaluated for the treatment of cerebral trauma using a rat model of cerebral hematoma induced by intracerebral (IC) stereotaxic injections of collagenase. Control animals received an intracerebral collagenase injection followed by intraperitoneal (IP) saline injection. Sham operated animals received saline only (IC, IP). Forty-eight hours following the surgeries, the brains were removed from the euthanized animals. Cerebral hemispheres were separated and the 4 coronal sections (antero-posterior plane) were weighed. Each slice was dried for 24 h (100 degrees C) and weighed again to establish brain water content. In hematoma-induced saline treated rats, significant differences in brain water content were observed when compared to sham operated animals. Rats treated with 1 mg/kg dexamethasone had a significant brain water content decrease; however, no significant differences were observed with higher doses of dexamethasone. In conclusion, low doses of dexamethasone seem to be beneficial for the treatment of cerebral trauma.  (+info)

Diffuse axonal injury associated with chronic traumatic brain injury: evidence from T2*-weighted gradient-echo imaging at 3 T. (7/39)

BACKGROUND AND PURPOSE: Diffuse axonal injury is frequently accompanied by tissue tear hemorrhages. We examined whether high field strength T2*-weighted gradient-echo imaging performed during the chronic stage of traumatic brain injury may have advantages in the evaluation of diffuse axonal injury as compared with T1- and T2-weighted MR imaging. METHODS: Prospective MR imaging of 66 patients (age range, 17-57 years) was performed using a 3-T system 3 to 292 months (median, 23.5 months) after traumatic brain injury. T1-, T2-, T2*-hypointense and T2-hyperintense foci of 1- to 15-mm diameter were registered in 10 brain regions by two readers separately. Foci that appeared hypointense both on the T1- and T2- and/or on the T2*-weighted images were defined as traumatic microbleeds. RESULTS: For 46 (69.7%) of the patients, T2*-weighted gradient-echo imaging revealed traumatic microbleeds. Hyperintense foci were observed on the T2-weighted images of only 15 (22.7%) patients. T2*-weighted imaging showed significantly more traumatic microbleeds (P =.000) than did T1- and T2-weighted imaging. Interobserver agreement was strong (kappa = 0.79, tau = 0.749, P =.000). For 14 (21.2%) of the patients, T2*-weighted gradient-echo imaging revealed traumatic microbleeds in the corpus callosum, whereas for only two (3%), hyperintense callosal lesions were seen on the T2-weighted images. Although a significant correlation existed between the total amount and callosal appearance of traumatic microbleeds and Glasgow Coma Scale scores (P =.000), no correlation existed with extended Glasgow Outcome Scale scores. CONCLUSION: T2*-weighted gradient-echo imaging at high field strength is a useful tool for the evaluation of diffuse axonal injury during the chronic stage of traumatic brain injury. Diffuse axonal injury-related brain lesions are mainly hemorrhagic. The relevance of diffuse axonal injury for long-term clinical outcome is uncertain.  (+info)

Endovascular management of intracranial pial arterio-venous fistulas. (8/39)

From 1996-2002 we treated 5 consecutive cases of pial fistula. There were 3 patients with a single hole-single channel pial fistula and two patients had a complex pial fistula. Three patients presented with intracerebral hematoma and had a focal neurological deficit. One patient presented with history of seizures and 1 patient had headache. The results of the treatment were analyzed both clinically and angiographically. The follow-up period ranged from 6 months to 6 years. All fistulas were treated with concentrated glue. The glue cast included the distal part of the feeding artery, A-V connection and the proximal part of the vein. Post-embolisation angiography showed complete occlusion of two single-hole fistulas and one complex pial A-V fistula and near total occlusion of one single-hole and one complex pial A-V fistula. Four patients had excellent clinical outcome. One patient with single-hole fistula had a hemorrhagic venous infarct resulting in transient hemiparesis.  (+info)

Detection of critical oligemia in patients with traumatic intracerebral hemorrhage using continuous quantitative...Detection of critical oligemia in patients with traumatic intracerebral hemorrhage using continuous quantitative...
121 Introduction: Serial changes in cerebral blood flow after intracerebral hemorrhage may influence tissue viability in regions surrounding the hematoma. Non invasive monitoring may assist titration of systolic blood pressure. Purpose: To determine if continuous quantitative electroencephalography percent alpha variability(EEGPAV)can discriminate areas of critically reduced cerebral blood flow (CBF). Methods:Ten patients with traumatic intracerebral hemorrhage underwent combined monitoring of cerebral microdialysis, EEGPAV, and intermittent xenon-computerized tomograhpy to determine if EEGPAV monitoring could determine and monitor cellular distress in the context of oligemia. Critical oligemia was defined as CBF , 25. Time and regional matched samples of EEG, microdialysis and CBF were used to correlate across modalities. Regions adjacent to the hematoma only were studied. Results:Six patients had regional CBF , 25 and 4 , 30 cc/100gm/min. In the low CBF group, the mean regional EEGPAV was 0.11 ...
Browse | jnsBrowse | jns
A small number of patients with an apparently minor head injury will develop a life-threatening intracranial hematoma that must be rapidly detected and removed. To assess the risk of a significant intracranial neurosurgical complication after apparently minor head injury, the authors collected data prospectively on 610 patients who had sustained a transient posttraumatic loss of consciousness or other neurological function and who had a Glasgow Coma Scale (GCS) score of 13, 14, or 15 in the emergency room. Skull x-ray films were obtained in 583 patients, 66 of whom (10.8% of the study population) had cranial fractures. Eighteen of the 610 patients (3.0%) required a neurosurgical procedure. Three acute subdural hematomas, one epidural hematoma, and one traumatic intracerebral hematoma required craniotomy. Of the 66 patients who had skull fracture, 7.6% required a craniotomy for intracranial hematoma. Thirteen (19.7%) of the 66 patients with skull fracture required an operative procedure as ...
Results from the First Randomised Controlled Trial of Sugery for Traumatic Intracerebral Haemorrhage [STITCH(TRAUMA)] - ePrints...Results from the First Randomised Controlled Trial of Sugery for Traumatic Intracerebral Haemorrhage [STITCH(TRAUMA)] - ePrints...
Author(s): Mendelow DA, Gregson BA, Rowan EN, Francis R, Mitchell P. Publication type: Conference Proceedings (inc. Abstract). Publication status: Published. Conference Name: 11th Symposium of the International Neurotrauma Society Year of Conference: 2014. Pages: A30-A30. ISSN: 1557-9042. Publisher: Mary Ann Liebert, Inc. publishers URL: http://online.liebertpub.com/doi/pdfplus/10.1089/neu.2014.9937. Series Title: Journal of Neurotrauma. ...
Cayuga BiotechCayuga Biotech
... is a startup company developing novel hemostatic drugs and devices to treat uncontrollable bleeding episodes. We envision our product as a universal agent able to generate clots in all bleeding scenarios while minimizing the risk of side effects.. Trauma and hemorrhage are intertwined conditions that account for the number one cause of death and disability worldwide. We are developing treatments that promote hemostasis for both trauma-related hemorrhage and rare, inherited bleeding disorders.. Live to tell the story.. ...
How often is haemosiderin not visible on routine MRI following traumatic intracerebral haemorrhage? | SpringerLinkHow often is haemosiderin not visible on routine MRI following traumatic intracerebral haemorrhage? | SpringerLink
Intracerebral haemorrhage may be visible indefinitely on MRI, due to persistence of haemosiderin in macrophages around the lesion, but it is not clear whether all haemorrhages produce haemosiderin or,
Is Beta Carotene Good For Tanning Eye Headlight Lazy Ruckus - mode eyes cultureguidesIs Beta Carotene Good For Tanning Eye Headlight Lazy Ruckus - mode eyes cultureguides
There are no documented side effects of Lutein but it is recommended that you do not Is Beta Carotene Good For Tanning Eye Headlight Lazy Ruckus take too much of the nutrient as it can possibly Hemophilia and color blindness are recessive disorders. Data Cataracts In Infants Related Articles. Is Beta Carotene Good For Tanning Eye Headlight Lazy Ruckus ocular surface disease. Since glaucoma usually causes no symptoms other than vision loss Vision loss caused by glaucoma is permanent. diffuse axonal injury ; cavernoma: Cereal hemorrhagic contusion; Hemorrhagic cereal contusion; Hemorrhagic cereal contusions; This is particularly useful when treating lazy eye in children as they are sometimes reluctant to wear an eye patch.. The consumption of food and medicinal plants with antioxidant properties have proven their efficacy against skin aging and damage from the sun. Porn game: Dream Job The Interview. Ophthalmologist Portland OR - The Eye Clinic P.. Visit our site or call today to learn more about ...
Cerebral contusions | Radiology Case | Radiopaedia.orgCerebral contusions | Radiology Case | Radiopaedia.org
This patient attended after a series of falls. He displayed ongoing confusion and behavioural change while rehabilitating in hospital. The case was discussed with neurosurgery but was managed conservatively. --- Thanks to Dr Gilly Fleming for...
1992 - Acclaimed Music Forums1992 - Acclaimed Music Forums
This one took a bit longer to finish, because I have been busy with work and other things, but I will keep going with the 90s ...
Orbital floor fracture | Radiology Case | Radiopaedia.orgOrbital floor fracture | Radiology Case | Radiopaedia.org
Radiographs demonstrating a left orbit floor fracture and associated opacification of the maxillary sinus (hemosinus). These findings were confirmed on CT, which also demonstrated multiple small foci of brain haemorrhagic contusions.
Diffuse Axonal InjuryDiffuse Axonal Injury
... is a severe form of brain injury which occurs in some traumatic brain injury patients. It results from shearing forces that damage axons.
Cerebellar peduncle injury in patients with ataxia following diffuse axonal injury.Cerebellar peduncle injury in patients with ataxia following diffuse axonal injury.
No diffusion tensor imaging (DTI) study has yet investigated ataxia in diffuse axonal injury (DAI). In the current study, we used DTI to investigate cerebellar peduncle lesions of patients who showed severe ataxia following DAI. Six patients with sev
How long should I take sick leave for cerebral contusion?How long should I take sick leave for cerebral contusion?
Hello. Thanks for writing to us. The sick leave depends on the extent of contusion and if any late symptoms are expected or not. The area of contusion is also very important. The best judgement would be of your treating doctors as he knows how much the damage is. From your symptoms it seems that...
Diffuse axonal injuryDiffuse axonal injury
... (DAI) is one of the most common and devastating types of brain injury (Iwata et al., 2004), occurring in almost
Diffuse Axonal Injury: Pathological and Clinical Aspects | MedCraveDiffuse Axonal Injury: Pathological and Clinical Aspects | MedCrave
Histology in DAI. A number of histological techniques are available to appreciate sequential pathological changes in axons in diffuse axonal injury. These are primarily aimed at shortening the duration at which the axonal changes are seen and to put them in context to various traumatic and non traumatic conditions so as to differentiate the causative mechanism. Axonal swellings or retraction balls, representing transected axons, are the histological hallmark of axonal injury but are usually not visible before 24 to 36 hours by routine H & E staining or with a myelin stain like Luxol fast blue [2]. Silver staining method can reliably demonstrate axonal swellings within 12 to 18 hours [27]. The method has been found to be more sensitive and reliable as compare to H & E staining. However diffuse staining of axons by silver stains may occasionally make differentiation of injured and irregular axons difficult thereby limiting their practical utility [28]. Injuries to the axons may be detected even ...
April 2012: Management Strategies of Intracerebral Hematomas | The Journal of Neurosurgery Podcast ArchiveApril 2012: Management Strategies of Intracerebral Hematomas | The Journal of Neurosurgery Podcast Archive
Management Strategies of Intracerebral Hematomas - click here to listen In this podcast, topic editor Dr. Marc Mayberg of The Swedish Neuroscience Institute, Seattle, Washington, speaks with Dr. David Newell, also of The Swedish Neuroscience Institute. They discuss Dr. Newell and colleagues article Minimally Invasive Treatment of Intracranial Hemorrhage which appears in this months issue.
ICD-10-CM Code S06.332 - Contusion and laceration of cerebrum, unspecified, with loss of consciousness of 31 minutes to 59...ICD-10-CM Code S06.332 - Contusion and laceration of cerebrum, unspecified, with loss of consciousness of 31 minutes to 59...
Cerebral contusion, Latin contusio cerebri, a form of traumatic brain injury, is a bruise of the brain tissue. Like bruises in other tissues, cerebral contusion can be associated with multiple microhemorrhages, small blood vessel leaks into brain tissue. Contusion occurs in 20-30% of severe head injuries. A cerebral laceration is a similar injury except that, according to their respective definitions, the pia-arachnoid membranes are torn over the site of injury in laceration and are not torn in contusion. The injury can cause a decline in mental function in the long term and in the emergency setting may result in brain herniation, a life-threatening condition in which parts of the brain are squeezed past parts of the skull. Thus treatment aims to prevent dangerous rises in intracranial pressure, the pressure within the skull. ...
St. Louis Brain Injury Attorney - Diffuse Axonal InjuriesSt. Louis Brain Injury Attorney - Diffuse Axonal Injuries
Traumatic brain injuries are one of the most serious types of injuries caused by car accidents. Every year, over a million people suffer a traumatic brain injury.
Early elective surgical exploration of spontaneous intracerebral hematomas of unknown origin: Clinical article<...Early elective surgical exploration of spontaneous intracerebral hematomas of unknown origin: Clinical article<...
TY - JOUR. T1 - Early elective surgical exploration of spontaneous intracerebral hematomas of unknown origin. T2 - Clinical article. AU - Elhammady, Mohamed. AU - Baskaya, Mustafa K.. AU - Heros, Roberto. PY - 2008/12/1. Y1 - 2008/12/1. N2 - Object. The management of non-life threatening spontaneous intraparenchymal hemorrhage with no obvious medical etiology in patients and the lack of findings on images has not been clearly defined. In general, the current practice is to treat these patients conservatively and repeat studies to rule out a treatable cause 6 weeks to 3 months later; more often than not these repeated studies fail to reveal any findings, and the patient is treated conservatively. For years, the senior author (R.C.H.) has treated these patients with early surgical exploration. This study was undertaken prospectively to ascertain the frequency of positive findings during surgical exploration. Methods. Between 2000 and 2007, the authors prospectively collected data from 9 cases (4 ...
Cerebrospinal Fluid Protein Increased & Intracerebral Hematoma<...Cerebrospinal Fluid Protein Increased & Intracerebral Hematoma<...
Cerebrospinal Fluid Protein Increased & Intracerebral Hematoma Symptom Checker: Possible causes include Cerebral Hemorrhage & Cerebral Thrombosis & Stroke. Check the full list of possible causes and conditions now! Talk to our Chatbot to narrow down your search.
Treating 4th degree hemorrhoids - are hemorrhoids why sugar is bad for youTreating 4th degree hemorrhoids - are hemorrhoids why sugar is bad for you
Mostly it will prolapse during the passing of stool. Styplon is also found as a haemostatic drug in conjunction with herbal formulation cayenne pepper green tea for hemorrhoids If youre constipated, your GP may prescribe a laxative Laxatives are a type of medicine that can help you empty your bowels. You can simply take a cotton ball and soak it with apple cider vinegar and later apply it directly on the hemorrhoids to get rid of hemorrhoids.
What are The Many Different Types of Brain Injuries?What are The Many Different Types of Brain Injuries?
Diffuse Axonal Injury is a brain injury associated most closely with the rotation and disruption of your brain within the cranium. This can cut away, shear or sever the brain axons, which are connecting nerve fibers. Damage such as this is typically difficult to analyze. It is also accompanied by microscopic tears that are difficult to locate from the get go.. If doctors diagnose your case as a "mild brain injury" you may notice that thus the wounds heal over time. But often, a DAI results in permanent disabilities from loss of consciousness to lifetime coma. Eventually, a fatality could be the nexus of a future wrongful death claim in civil court and even a murder case in criminal court.. Currently, medical science has not discovered a methodology to treat diffuse axonal injuries. But medical experts have observed in some studies that the damage to axons occurs in the first 12 to 24 hours after the head impact. Many are confident that modern advances in science will be able to treat and/or slow ...
Contusion In Children - What You Need to KnowContusion In Children - What You Need to Know
Care guide for Contusion In Children. Includes: possible causes, signs and symptoms, standard treatment options and means of care and support.
Post-Traumatic Tremor and Thalamic Deep Brain Stimulation: Evidence for Use of Diffusion Tensor Imaging. - Nuffield Department...Post-Traumatic Tremor and Thalamic Deep Brain Stimulation: Evidence for Use of Diffusion Tensor Imaging. - Nuffield Department...
BACKGROUND: Deep brain stimulation (DBS) is a well-established treatment to reduce tremor, notably in Parkinson disease. DBS may also be effective in post-traumatic tremor, one of the most common movement disorders caused by head injury. However, the cohorts of patients often have multiple lesions that may impact the outcome depending on which fiber tracts are affected. CASE DESCRIPTION: A 20-year-old man presented after road traffic accident with severe closed head injury and polytrauma. Computed tomography scan showed left frontal and basal ganglia hemorrhagic contusions and intraventricular hemorrhage. A disabling tremor evolved in step with motor recovery. Despite high-intensity signals in the intended thalamic target, a visual analysis of the preoperative diffusion tensor imaging revealed preservation of connectivity of the intended target, ventralis oralis posterior thalamic nucleus (VOP). This was confirmed by the postoperative tractography study presented here. DBS of the VOP/zona incerta was
JTH Releases SSC Recommendation on Measuring Thrombin Generation in Hemophilia - International Society on Thrombosis and...JTH Releases SSC Recommendation on Measuring Thrombin Generation in Hemophilia - International Society on Thrombosis and...
Proposal for standardized preanalytical and analytical conditions for measuring thrombin generation in hemophilia. Readouts from the thrombin generation (TG) assay (TGA) have been used to predict the effects of hemostatic drugs in hemophilic patients [1]. There is better correlation between an individuals bleeding tendency and his or her TG capacity compared with factor levels measured using routine assays [2, 3]. However, the clinical utility of TGA has not yet been validated in multicenter studies because of lack of standardization. This report aims to highlight variables that may impact directly on TGA reliability and steps to reduce TGA variability within and between laboratories to improve precision of results. Read more.. ...
Management of Severe Head Injury - Trip DatabaseManagement of Severe Head Injury - Trip Database
Find all the evidence you need on Management of Severe Head Injury via the Trip Database. Helping you find trustworthy answers on Management of Severe Head Injury | Latest evidence made easy
Neurowiki2012 - Diffuse Axonal InjuryNeurowiki2012 - Diffuse Axonal Injury
An emerging field of research focuses on using biomarkers as a different non-invasive clinical approach for diagnosis of DAI. These markers could be detected in CSFsamples due to the breakdown products of neurons and diffusion through the damaged blood brain barrier of traumatic brain injury patients. Possible biomarkers could include proteins associated with primary structural damage or with the cellular and molecular cascade involved in secondary axonomy. Axonal and glial biomarkers include neurofilaments (NF), microtubule-associated protein tau, and α-II Spectrin, and myelin basic protein. These are all critical components of the cytoskeleton or myelin that are dephosphorylated, degraded, depolymerized, and form protein bundles upon injury due to the action of proteolytic enzymes. Biomarkers associated with the molecular cascade during secondary axonomy include β-Amyloid precursor protein, an axoplasmic transport protein that accumulates in the retraction balls, amyloid β, FE65, and ...
Intensity of anticoagulant treatment and risk of intracerebral hematoma. | StrokeIntensity of anticoagulant treatment and risk of intracerebral hematoma. | Stroke
Thank you for your interest in spreading the word on Stroke.. NOTE: We only request your email address so that the person you are recommending the page to knows that you wanted them to see it, and that it is not junk mail. We do not capture any email address. ...
EBIC-Guidelines for management of severe head injury in adults | SpringerLinkEBIC-Guidelines for management of severe head injury in adults | SpringerLink
Guidelines for the management of severe head injury in adults as evolved by the European Brain Injury Consortium are presented and discussed. The importance of preventing and treating secondary...
Intensive care monitoring of severe head injury<...Intensive care monitoring of severe head injury<...
TY - JOUR. T1 - Intensive care monitoring of severe head injury. AU - Rafferty, C.. AU - Hansen, S.. AU - Bullock, Ross. AU - Teasdale, G. M.. AU - Fitch, W.. AU - Jamal, G. A.. PY - 1992/1/1. Y1 - 1992/1/1. UR - http://www.scopus.com/inward/record.url?scp=0026696761&partnerID=8YFLogxK. UR - http://www.scopus.com/inward/citedby.url?scp=0026696761&partnerID=8YFLogxK. M3 - Article. AN - SCOPUS:0026696761. VL - 3. JO - Clinical Intensive Care. JF - Clinical Intensive Care. SN - 0956-3075. IS - 2 SUPPL.. ER - ...
Can you die from post concussive syndrome - Things You Didnt KnowCan you die from post concussive syndrome - Things You Didn't Know
Can you die from post concussive syndrome - Can you die from post concussive syndrome? Not likely. Post concussive syndrome should not be fatal. Although this is one of the disorders under traumatic brain injury, concussion should not show abnormalities on ct scan or the diagnosis changes to something more severe such as diffuse axonal injury.
JoVE Author Search: Barbee KAJoVE Author Search: Barbee KA
Calcium, Concentration, Membrane, Axonal Transport, Axons, Brain, Brain Injury, Calpain, Cell Death, Cytoskeleton, Death, Diffuse Axonal Injury, Future, Injury, Mitochondria, Neurons, Permeability, Poloxamer, Poloxamer 188, Therapeutic
STICH 2: does decompression have a role in superficial intracerebral hematoma? - Muir - 2013 - International Journal of Stroke ...STICH 2: does decompression have a role in superficial intracerebral hematoma? - Muir - 2013 - International Journal of Stroke ...
The recently published second Surgical Trial in Intracerebral Haemorrhage (STICH-2) tested whether surgical evacuation of superficial spontaneous intracerebral haemorrhage was effective at reducing death and disability at 6 months after onset. Participants were randomised to a policy of early surgical intervention or initial medical management alone within 48 hours of symptom onset. After enrolling 601 patients across 78 centres in 27 countries, intention to treat analysis showed no difference in outcome. Time to intervention was a median of 26 hours after symptom onset in the surgical arm, and craniotomy accounted for 98% of all surgical procedures. The interpretation of the overall neutral result is confounded by a high proportion of crossovers from the medical management arm of the study, predominantly of more severely affected patients. Further analysis may clarify whether decompressive surgery late after superficial intracerebral haemorrhage has any role in management. ...
Hemostatic agents at monthly or folk remedies? - Health TipsHemostatic agents at monthly or folk remedies? - Health Tips
But there are cases where hemostatic drugs are contraindicated during menstruation because of allergic reactions to the components or any other intolerance.Then resorted to traditional medicine.Naturally, in such a way hormones do not balance, and narrow vessels is quite real.Great for those purposes a decoction of the bark of Viburnum, nettle infusions and decoctions, tinctures and teas of marigold and others. Yet it is worth recalling that it is better not to self-medicate, and seek medical advice in time for the qualified help.. Hemostatic agents at monthly Sharok presented to pharmacists, but among them are the following: Dicynonum, Traneksan, Vikasol etc.Only a specialist can determine: whether or not to take these drugs, in what dosage and what time.. It should be noted that when an uncontrolled admission of drugs that increase blood clotting and constrict blood vessels, blockage of the veins may occur.Also, these drugs are contraindicated for those who already suffer from thrombosis or ...
Intracerebral haemorrhage as a rare complication of HSV-1 meningoencephalitis: case report and review of the literature. -...Intracerebral haemorrhage as a rare complication of HSV-1 meningoencephalitis: case report and review of the literature. -...
We present a case of herpetic meningoencephalitis confirmed by PCR in a 22-y-old male, with accompanying appearance of a large intracerebral haematoma as a complication. Despite the impressive imaging findings, the final outcome of the patients progress was favourable.
Alexs storyAlex's story
Upon my airlift arrival to James Cook university hospital, in Middlesbrough, it was discovered I had a brain injury (cerebral contusion) and L&R lung contusion. Due to the brain injury I was placed in an induced coma with a pressure relief valve screwed into my skull so the cranial pressure did not badly damage the brain. Immediately after the coma I was making an awful noise and had my attention focused on my left leg, so they x-rayed my leg and found out I had badly broken my left femur just by my knee, so I was put back into the coma and had an operation to put 3 screws in my the femur just by the knee. I also broke my neck in three places but just wore a neck brace to keep it straight. ...
HEMATOMA INTRAPARENQUIMATOSA PDFHEMATOMA INTRAPARENQUIMATOSA PDF
... - Neurosurg Focus. Oct 15;15(4):E4. Intracerebral hematoma from aneurysm rupture. Abbed KM(1), Ogilvy CS. Author information: (1)Department of. This
Evaluating Neuroprotection in Aneurysm Coiling Therapy - Study Results - ClinicalTrials.govEvaluating Neuroprotection in Aneurysm Coiling Therapy - Study Results - ClinicalTrials.gov
The mRS is a measure of global disability that has been widely applied for evaluating recovery from stroke. Scores range from 0 to 6, with higher scores indicating greater disability. A score of 0 indicates no residual symptoms; 1 = no significant disability/able to carry out all usual activities, despite some symptoms; 2 = slight disability; 3 = moderate disability; 4 = moderately severe disability; 5 = severe disability; 6 = death. The number of participants scoring 0-2 on the mRS at Day 30 was compared in both treatment groups ...
Thigh Contusion: OverviewThigh Contusion: Overview
Another name for Thigh Contusion is Leg Contusion. What is a leg contusion? A person with a leg contusion has a bruise to the leg. A leg contusion may ...
Academic: Reformation and reclamation of adult prisoners EbooksAcademic: Reformation and reclamation of adult prisoners Ebooks
Find, buy and download Reformation and reclamation of adult prisoners ebooks from our Academic section for your eReader at great prices.
Academic: Reformation and reclamation of adult prisoners EbooksAcademic: Reformation and reclamation of adult prisoners Ebooks
Find, buy and download Reformation and reclamation of adult prisoners ebooks from our Academic section for your eReader at great prices.
Neurosurgery - procedure, recovery, blood, removal, pain, complications, adults, timeNeurosurgery - procedure, recovery, blood, removal, pain, complications, adults, time
Spontaneous intracranial hemorrhage (hemorrhage in the brain) is a condition characterized by hemorrhage in the brain (hemorrhagic stroke) that results in a sudden onset of neurologically worsening symptoms (that include focal neurologic deficits and loss of consciousness). CT scans are helpful in identifying the intracranial hemorrhage, of which there are two types-subarachnoid hemorrhage and intracerebral hematoma. The subarachnoid space is an area that exists between two layers of coverings (membranes) that wrap around the brain. A spontaneous subarachnoid hemorrhage is defined as blood (not caused by trauma), in the subarachnoid space. The amount of blood in the subarachnoid space can be a focal (small area) amount or a larger, more diffuse hemorrhage, which can be further complicated by having an intraventricular hemorrhage or intracerebral hematoma at the same time. Subarachnoid hemorrhage can affect adults of all ages, but usually peaks in the fourth and fifth decades of life. ...
Diffuse Axonal Injury | CTDDiffuse Axonal Injury | CTD
A relatively common sequela of blunt head injury, characterized by a global disruption of axons throughout the brain. Associated clinical features may include NEUROBEHAVIORAL MANIFESTATIONS; PERSISTENT VEGETATIVE STATE; DEMENTIA; and other disorders ...
Interaction between Warfarin and the Herbal Product Shengmai-Yin: A Case Report of Intracerebral Hematoma ...Interaction between Warfarin and the Herbal Product Shengmai-Yin: A Case Report of Intracerebral Hematoma ...
A 71-year-old man was stable on warfarin (2.25 mg daily) therapy with an international normalized ratio (INR) of 1.8-2.2 after a heart valve replacement surgery. Recently, he consumed the liquid-like herbal product called shengmai-yin (10 mL daily) against medical advice. Seven days after the daily ...
Measuring Disability: Comparing the Impact of Two Data Collection Approaches on Disability RatesMeasuring Disability: Comparing the Impact of Two Data Collection Approaches on Disability Rates
The usual approach in disability surveys is to screen persons with disability upfront and then ask questions about everyday problems. The objectives of this paper are to demonstrate the impact of screeners on disability rates, to challenge the usual exclusion of persons with mild and moderate disability from disability surveys and to demonstrate the advantage of using an a posteriori cut-off. Using data of a pilot study of the WHO Model Disability Survey (MDS) in Cambodia and the polytomous Rasch model, metric scales of disability were built. The conventional screener approach based on the short disability module of the Washington City Group and the a posteriori cut-off method described in the World Disability Report were compared regarding disability rates. The screener led to imprecise rates and classified persons with mild to moderate disability as non-disabled, although these respondents already experienced important problems in daily life. The a posteriori cut-off applied to the general ...
Glasgow Outcome Scale - www.E1V1M1.co.ukGlasgow Outcome Scale - www.E1V1M1.co.uk
Outcome after sever brain damage. 1. Dead. 2. Persistent vegetative state (no obvious cortical function). 3. Severe disability (conscious but disabled). 4. Moderate disability (disabled but independent). 5. Good recovery (return to normal activities wvwn with minor neuro or psychological deficits). Reference: http://www.nervous-system-diseases.com/glasgow-outcome-scale.html. ...
Becster.com - F1 Wednesday - Jules Bianchi - Becster.comBecster.com - F1 Wednesday - Jules Bianchi - Becster.com
I know I have another post scheduled for today as part of Blogtober14 but I really couldnt let this Wednesday pass without commenting on poor Jules Bianchi.. At the Japanese Grand Prix on Sunday he had a horrific crash. At around lap 42 or so Sutil span out at one of the trickiest corners of the circuit. It was raining and very wet, very difficult conditions. The safety car came out and the recovery vehicle (a big massive crane) was attending to Sutils car.. At lap 44 on the very same corner Jules Bianchi spam out and hit the recovery vehicle! Although Im not quite sure but he went into it head on. Now if youve not seen an F1 car you should know that its an open cockpit so the drivers head is exposed. Hes suffered some pretty serious head injuries. His family have now confirmed that he suffered diffuse axonal injury. When I looked into what this… it really doesnt sound good. All I can say is I hope he recovers from this.. ...
JoVE Author Search: de Ribaupierre SJoVE Author Search: de Ribaupierre S
Brain, Brain Trauma, Consciousness, Diffuse Axonal Injury, Injury, Literature, Trauma, Curricula, Education, Goals, Needs Assessment, Ventriculostomy
Contusion to the Brain: AnatomyContusion to the Brain: Anatomy
Another name for Contusion to the Brain is Brain Contusion. To better understand a brain contusion, it helps to understand the anatomy of the brain. The ...
NHS Website - Health A-ZNHS Website - Health A-Z
A brain abscess is a pus-filled swelling in the brain. It usually occurs when bacteria or fungi enter the brain tissue after an infection or severe head injury. ...
Presenting the Truth of the ReformationPresenting the Truth of the Reformation
It sounds like its going to require more than just one hour of video.. At this time weve sketched out 10 one-hour shows. We want this to have a very complete perspective, and we want it to be of such a quality that it will play well on a wide variety of programming. We want it available for the secular media; they would be foolish not to include it in their programming because we want it to be of good quality. We plan to include dramatizations; for instance there are transcripts of the debates between Catholics and the reformers, and wed like to dramatize some key elements of those debates from that time period, and wed also like to dramatize some of the key events and some of the characters. One of the things I hope to do, along with one of the scholars I have not yet contacted, is to have a very complete understanding of the Fifth Lateran Council and the Council of Trent. The Fifth Lateran Council took place right before Luther began the Reformation. What were the successes, what were the ...
Spontaneous intracerebral haemorrhage: a clinical review. - My...Spontaneous intracerebral haemorrhage: a clinical review. - My...
Spontaneous intracerebral haemorrhage: a clinical review.: This article provides a clinical overview of spontaneous intracerebral haemorrhage, focusing on clini
Effect of Alcohol on Diffuse Axonal Injury in Rat Brainstem: Diffusion Tensor Imaging and Aquaporin-4 Expression StudyEffect of Alcohol on Diffuse Axonal Injury in Rat Brainstem: Diffusion Tensor Imaging and Aquaporin-4 Expression Study
BioMed Research International is a peer-reviewed, Open Access journal that publishes original research articles, review articles, and clinical studies covering a wide range of subjects in life sciences and medicine. The journal is divided into 55 subject-specific sections.
Severe head injuries in three countries. | Journal of Neurology, Neurosurgery & PsychiatrySevere head injuries in three countries. | Journal of Neurology, Neurosurgery & Psychiatry
Methods for assessing early characteristics and late outcome after severe head injury have been devised and applied to 700 cases in three countries (Scotland, Netherlands, and USA). There was a close similarity between the initial features of patients in the three series; in spite of differences on organisation of care and in details of management , the mortality was exactly the same in each country. This data bank of cases (which is still being enlarged) can be used for predicting outcome in new cases, and for setting up trials of management.. ...
The structural basis of moderate disability after traumatic brain damageThe structural basis of moderate disability after traumatic brain damage
Acute post-traumatic stress symptoms and age predict outcome in military blast concussion. Mac Donald, Christine L.; Adam, Octavian R.; Johnson, Ann M.; Nelson, Elliot C.; Werner, Nicole J.; Rivet, Dennis J.; Brody, David L. // Brain: A Journal of Neurology;May2015, Vol. 138 Issue 5, p1314 High rates of adverse outcomes have been reported following blast-related concussive traumatic brain injury in US military personnel, but the extent to which such adverse outcomes can be predicted acutely after injury is unknown. We performed a prospective, observational study of US military... ...
TBI Information & Training May 13, 2014 7pm « Mary Lee FoundationTBI Information & Training May 13, 2014 7pm « Mary Lee Foundation
Mark Your Calendar. Traumatic Brain Injury Information & Training. Hosted by the Mary Lee Foundation (Free of Charge). Mary Lee Foundation Community Center. 1327 Lamar Square Drive Austin, Texas 78704. Date ~ May 13th, 2014, Tuesday Evening. Time ~ 7:00 pm - 8:00 pm. GUEST MODERATOR: Glenn Martin. Glenn sustained a life threatening brain injury on 11/02/10 following a motor cycle accident. Glenn successfully completed his Vocational Rehabilitation program in September 2013. Glenn realized it would be up to him to have a positive outcome and in doing so maintained a positive attitude and stayed focus on setting realistic goals. An increased sense of self awareness and inner peace emerged and Glenn now serves as a positive role model for others and tonight serves as our Guest Moderator.. GUEST SPEAKER: Cavin Balaster. Cavin is a traumatic brain injury survivor from a two-story fall, was comatose for twelve days, and was diagnosed with a diffuse axonal injury. Approximately 90% of the people who ...
Sydney Ideas - The X Factor in the Reformation Tickets, Thu, 12/10/2017 at 6:00 pm | EventbriteSydney Ideas - The X Factor in the Reformation Tickets, Thu, 12/10/2017 at 6:00 pm | Eventbrite
Eventbrite - Sydney Ideas presents Sydney Ideas - The X Factor in the Reformation - Thursday, 12 October 2017 at Law School Foyer, Camperdown, NSW. Find event and ticket information.
Access to History. Luther and the German Reformation 1517-55 3ed | Russel Tarr,Keith Randell | downloadAccess to History. Luther and the German Reformation 1517-55 3ed | Russel Tarr,Keith Randell | download
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Clinical Trials Registry | Internet Stroke CenterClinical Trials Registry | Internet Stroke Center
Inclusion Criteria: - 1.Patients aged 18 to 80 years. 2.Supratentorial hypertensive intracerebral hemorrhage diagnosed by head CT or MRI. 3.The intracerebral hematoma is more than 10ml. 4.Ischemic stroke diagnosed by head CT or MRI. 5.Unilateral middle cerebral artery occlusion or infarcted volume more than 1/2 of a hemicerebrum. 6.The head temperature collection within 4.5 hours following stroke attack. 7.Glasgow Coma Scale between 5-10. Exclusion Criteria: - 1.the time after stroke attack exceed 4.5 hours. 2.The intracerebral hematoma is less than 10ml. 3.The infarcted volume less than 1/3 of a hemicerebrum. 4.With terminal cerebral hernia. 5.Pregnant women. 6.Serious impaired liver function, coagulation disorders, AIDS, combine other tumor or special condition ...
The analysis of correlated factors of postoperative intracranial hemorrhage in epidermoid cyst--《Beijing Medical Journal...The analysis of correlated factors of postoperative intracranial hemorrhage in epidermoid cyst--《Beijing Medical Journal...
Objective To discuss the characteristics and correlated factors of postoperative intracranial hemorrhage of epidermoid cyst.Methods Clinical data of 428 patients with epidermoid cyst underwent craniotomy from 2002 to 2008 were analyzed retrospectively.Single-factor analysis and Logistic multivariate regression analysis were used to explore the correlated factors of postoperative intracranial hemorrhage of epidermoid cyst,and susceptible factors were screened.Results Postoperative intracranial hemorrhages occurred in 43 patients of the 428 patients(the incidence was 10.05%),among which the delayed hemorrhages(≥5 days postoperatively) accouned for 69.77% of the overall intracranial hemorrhages.The results of single-factor analysis showed that there was significant difference in tumor location(P=0.002),cerebral contusion(P=0.008) and postoperative meningitis(P=0.018) between bleeding group and non-bleeding group,and there was no significant differences in the remaining factors(age,sex,tumor size,capsule
The effect of post-injury erythropoietin administration on mortality and Glasgow outcome scales of patients with traumatic...The effect of post-injury erythropoietin administration on mortality and Glasgow outcome scales of patients with traumatic...
The effect of post-injury erythropoietin administration on mortality and Glasgow outcome scales of patients with traumatic brain injury: A metaanalysis., Faye B Garciano, Perry N N
Masters in Special EducationMasters in Special Education
Learn more about the special education masters program at Lasell College, and review our moderate disabilities education electives and core curriculum.
The Day - Sen. Maynards status - News from southeastern ConnecticutThe Day - Sen. Maynard's status - News from southeastern Connecticut
Monday marked three weeks since Sen. Andrew Maynard suffered a severe head injury in a fall outside his home in Stonington. He has remained at Rhode
AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPsychiatry and PsychologyPhenomena and ProcessesInformation ScienceNamed GroupsHealth CareGeographicals
Cerebral HemorrhageSubarachnoid HemorrhageCerebral InfarctionHemorrhageCerebral ArteriesCerebral Amyloid AngiopathyCerebrovascular DisordersIntracranial HemorrhagesIntracranial Hemorrhage, HypertensiveCapillary FragilityCerebral AngiographyCerebral Amyloid Angiopathy, FamilialMiddle Cerebral ArteryCerebrovascular CirculationBrainTomography, X-Ray ComputedAmyloidosisBrain IschemiaIntracranial Embolism and ThrombosisStrokeHematomaRetinal HemorrhageIntracranial AneurysmHemangioma, Cavernous, Central Nervous SystemCerebral Hemorrhage, TraumaticTissue Plasminogen ActivatorCerebral PalsyIntracranial Arteriovenous MalformationsAphasia, WernickeCystatinsPostpartum HemorrhageRupture, SpontaneousIschemic Attack, TransientJapanBrain EdemaTime FactorsGastrointestinal HemorrhageFatal OutcomeStreptococcus anginosusMagnetic Resonance ImagingHypertensionFibrinolytic AgentsVitreous HemorrhageEye HemorrhageTreatment OutcomeAcute DiseaseNeurologic ExaminationAutonomic DysreflexiaInfarction, Middle Cerebral ArteryMoyamoya DiseaseVasospasm, IntracranialCerebral VeinsRisk FactorsMalaria, CerebralAmyloid beta-PeptidesAnticoagulantsThrombolytic TherapyCystatin CAmyloidRetrospective StudiesProspective StudiesBlood PressureIntracranial EmbolismCerebral VentriclesAlzheimer DiseaseBasal Ganglia HemorrhageDisease Models, AnimalFollow-Up StudiesIncidenceAmyloid beta-Protein PrecursorPostoperative HemorrhageInfant, NewbornAge FactorsRecurrenceRiskPostoperative ComplicationsAneurysm, RupturedBasilar ArteryChoroid HemorrhagePeptic Ulcer HemorrhageIntracranial PressureAnterior Cerebral ArteryPrognosisPosterior Cerebral ArterySex FactorsPutaminal HemorrhageBrain DiseasesMutationHydrocephalusBlood Flow VelocityHematoma, SubduralRetrobulbar HemorrhageCerebral RevascularizationBlood VolumeEmbolization, TherapeuticCraniotomyCerebral VentriculographyDominance, CerebralBrain Hemorrhage, TraumaticShock, HemorrhagicRats, Sprague-DawleyNeuroprotective AgentsIntracranial ThrombosisCerebrumBlood-Brain BarrierIntracranial HypertensionHypoxia, BrainGlasgow Coma ScaleMagnetic Resonance Angiography
Near-death experienceNear-death experience
... coma resulting from traumatic brain damage; intracerebral hemorrhage or cerebral infarction; attempted suicide; near-drowning ... Professor of neurology Terence Hines (2003) claimed that near-death experiences are hallucinations caused by cerebral anoxia, ... A wide range of physiological theories of the NDE have been put forward including those based upon cerebral hypoxia, anoxia, ... dead are psychopathological symptoms caused by a severe malfunction of the brain resulting from the cessation of cerebral blood ...
Remote ischemic conditioningRemote ischemic conditioning
Delayed cerebral infarction after subarachnoid hemorrhage is a major cause of morbidity. Two Phase I clinical trials have shown ... Traumatic brain injury (TBI) shares many pathophysiological pathways with acute stroke, and ischemic preconditioning increases ... Reduced cerebral blood flow is an early finding in vascular cognitive impairment (VCI). Cardiovascular risk factor control is ... Animal models of stroke (both open-skull and closed-skull models) show that RIC improves cerebral blood flow; reduces ischemic ...
Rostyslav SvanidzeRostyslav Svanidze
Doctors discovered that Svanidze had suffered a cerebral hemorrhage, caused by a traumatic aortic rupture, at the time of his ...
Closed head injuryClosed head injury
Cerebral contusion Concussion Diffuse axonal injury Intracranial hemorrhage Traumatic brain injury Ibrahim, Nicole G.; Ralston ... cerebral contusion - a bruise to the brain tissue as a result of trauma. Contusions are local in nature, separating them from ... Multiple mild traumatic brain injuries sustained over a short period of time (hours to weeks), often seen with sports-related ... Blast-related traumatic brain injuries are often closed-head injuries and result from rapid changes in atmospheric pressure, ...
List of MeSH codes (C21)List of MeSH codes (C21)
... brain stem hemorrhage, traumatic MeSH C21.866.260.118.175.300 --- cerebral hemorrhage, traumatic MeSH C21.866.260.118.200 --- ... brain stem hemorrhage, traumatic MeSH C21.866.915.300.200.175.300 --- cerebral hemorrhage, traumatic MeSH C21.866.915.300. ... brain stem hemorrhage, traumatic MeSH C21.866.915.300.490.150.300 --- cerebral hemorrhage, traumatic MeSH C21.866.915.300. ... intracranial hemorrhage, traumatic MeSH C21.866.915.300.490.150 --- brain hemorrhage, traumatic MeSH C21.866.915.300.490.150. ...
Penetrating head injuryPenetrating head injury
People with subarachnoid hemorrhage, a blown pupil, respiratory distress, hypotension, or cerebral vasospasm are more likely to ... Up to 50% of patients with penetrating brain injuries get late-onset post-traumatic epilepsy. Brain herniation Traumatic brain ... penetrating trauma is similar to closed head injury such as cerebral contusion or intracranial hemorrhage. As in closed head ... "Overview of Adult Traumatic Brain Injuries." Retrieved on January 16, 2008. De Tommasi A, Cascardi P, De Tommasi C, Luzzi S, ...
Mimar KemaleddinMimar Kemaleddin
... in Ankara at the building site of Ankara Palas as a result of a cerebral hemorrhage, at the age of 57. He was survived by his ... He lived in a time segment that was as full of joy and ebullience as it was of traumatic disappointments. And his life ended in ...
Subarachnoid hemorrhageSubarachnoid hemorrhage
It is the only Food and Drug Administration (FDA) approved drug for treating cerebral vasospasm. In traumatic subarachnoid ... Cerebral vasospasm is one of the complication caused by subarachnoid haemorrhage. It usually happens from the third day after ... 1983). "Cerebral arterial spasm: A controlled trial of nimodipine in patients with subarachnoid hemorrhage". New England ... February 2002). "Traumatic subarachnoid hemorrhage: demographic and clinical study of 750 patients from the European brain ...
Post-traumatic seizurePost-traumatic seizure
Early seizures can be caused by factors such as cerebral edema, intracranial hemorrhage, cerebral contusion or laceration. ... PTS may be a risk factor for post-traumatic epilepsy (PTE), but a person who has a seizure or seizures due to traumatic brain ... Post-traumatic seizures (PTS) are seizures that result from traumatic brain injury (TBI), brain damage caused by physical ... Up to 86% of people who have one late post-traumatic seizure have another within two years. Complications of traumatic brain ...
AutoregulationAutoregulation
"Clinical relevance of cerebral autoregulation following subarachnoid haemorrhage". Nat. Rev. Neurol. 9 (3): 152-63. doi:10.1038 ... Brain blood flow autoregulation is abolished in several disease states such as traumatic brain injury, stroke, brain tumors, or ... Panerai, R. B.; S. T. Deverson; P. Mahony; P. Hayes; D. H. Evans (1999). "Effect of CO2 on dynamic cerebral autoregulation ... Paulson, O. B.; S. Strandgaard; L. Edvinsson (1990). "Cerebral autoregulation". Cerebrovascular and Brain Metabolism Reviews. 2 ...
Glutamate receptorGlutamate receptor
In the case of traumatic brain injury or cerebral ischemia (e.g., cerebral infarction or hemorrhage), acute neurodegeneration ... Obrenovitch TP, Richards DA (1995). "Extracellular neurotransmitter changes in cerebral ischaemia". Cerebrovasc Brain Metab Rev ... NR2A subunit of the NMDA glutamate receptor was found to be decreased in a subset of inhibitory interneurons in the cerebral ...
Head injuryHead injury
... bleeding between the dura mater and the skull Traumatic subarachnoid hemorrhage Cerebral contusion, a bruise of the brain ... Malignant post traumatic cerebral swelling can develop unexpectedly in stable patients after an injury, as can post traumatic ... Intra-axial hemorrhage is bleeding within the brain itself, or cerebral hemorrhage. This category includes intraparenchymal ... Cerebral concussion is the most common head injury seen in children. Types of intracranial hemorrhage are roughly grouped into ...
Traumatic brain injuryTraumatic brain injury
... such as a hemorrhage. As a result, cerebral perfusion pressure (the pressure of blood flow in the brain) is reduced; ischemia ... Armin SS, Colohan AR, Zhang JH (June 2006). "Traumatic subarachnoid hemorrhage: Our current understanding and its evolution ... Extra-axial lesions include epidural hematoma, subdural hematoma, subarachnoid hemorrhage, and intraventricular hemorrhage. ... "Assessment of a noninvasive cerebral oxygenation monitor in patients with severe traumatic brain injury". Journal of ...
HemiparesisHemiparesis
Traumatic: cerebral lacerations, subdural hematoma, epidural hematoma, vertebral compression fracture. *Iatrogenic: local ... who developed right-sided hemiparesis after a hemorrhage in the right brain.[17] ... "Spastic Hemiplegia : Cerebral Palsy". OriginsOfCerebralPalsy.com. Retrieved 2013-03-08.. *^ [2] Archived October 11, 2010, at ... Congenital: cerebral palsy, Neonatal-Onset Multisystem Inflammatory Disease (NOMID). *Degenerative: ALS, corticobasal ...
Head injuryHead injury
Traumatic subarachnoid hemorrhage. *Cerebral contusion, a bruise of the brain. *Concussion, a loss of function due to trauma ... Main article: cerebral hemorrhage. Intra-axial hemorrhage is bleeding within the brain itself, or cerebral hemorrhage. This ... Malignant post traumatic cerebral swelling can develop unexpectedly in stable patients after an injury, as can post traumatic ... Cerebral contusion[edit]. Main article: Cerebral contusion. Cerebral contusion is bruising of the brain tissue. The majority of ...
Midline shiftMidline shift
... malignant middle cerebral artery infarction, epidural hematoma, subarachnoid hemorrhage, chronic subdural hematoma, infarction ... The sign can be caused by conditions including traumatic brain injury,stroke, hematoma, or birth deformity that leads to a ... Another use is secondary screening to determine deviations in brain trauma at different times after a traumatic injury as well ... Maas AI, Stocchetti N, Bullock R (August 2008). "Moderate and severe traumatic brain injury in adults". Lancet Neurology. 7 (8 ...
Duret haemorrhagesDuret haemorrhages
They are caused by a traumatic downward displacement of the brainstem. They are named after Henri Duret. Duret haemorrhages are ... the cerebral crura. Increased pressure above the tentorium may also involve other midbrain structures.[citation needed] ... January 2002). "Brainstem hemorrhage in descending transtentorial herniation (Duret hemorrhage)". Intensive Care Med. 28 (1): ... or acute hematoma, edema following trauma, abscess, or tumor.[citation needed] Duret haemorrhages are haemorrhages secondary to ...
Outline of cardiologyOutline of cardiology
Cerebral aneurysm - Aneurysms of the arteries in the brain most commonly affect the anterior cerebral artery. Rupture of the ... Rupture of the aorta can occur at the sites of aneurysm, but is also due to trauma and results in a traumatic aortic rupture. ... retinal hemorrhage) and diabetic retinopathy (cotton wool spots) Pulses: carotid, dorsalis pedis, femoral, popliteal, posterior ... Overall, atherosclerosis tends to affect the arteries of highest pressure: aorta, coronary, renal, femoral, cerebral, and ...
New daily persistent headacheNew daily persistent headache
... subarachnoid hemorrhage idiopathic intracranial hypertension temporal arteritis chronic subdural hematoma post-traumatic ... hypertension spontaneous cerebrospinal fluid leak cervical artery dissections pseudotumor cerebri without papilledema cerebral ... and post-traumatic headaches suggesting inflammation as the cause of the headaches. NDPH as an inflammatory, post-infectious ...
List of MeSH codes (C10)List of MeSH codes (C10)
... brain stem hemorrhage, traumatic MeSH C10.228.140.300.535.450.200.750 --- cerebral hemorrhage, traumatic MeSH C10.228.140.300. ... brain stem hemorrhage, traumatic MeSH C10.228.140.199.275.300 --- cerebral hemorrhage, traumatic MeSH C10.228.140.199.500 --- ... brain stem hemorrhage, traumatic MeSH C10.900.300.087.187.300 --- cerebral hemorrhage, traumatic MeSH C10.900.300.087.250 --- ... brain stem hemorrhage, traumatic MeSH C10.900.300.837.150.650 --- cerebral hemorrhage, traumatic MeSH C10.900.300.837.300 --- ...
List of ICD-9 codes 800-999: injury and poisoningList of ICD-9 codes 800-999: injury and poisoning
Cerebral laceration and contusion (852) Subarachnoid, subdural, and extradural hemorrhage, following injury (853) Other and ... Traumatic amputation of toe(s) (896) Traumatic amputation of foot (897) Traumatic amputation of leg(s) (900) Injury to blood ... Traumatic hemothorax without open wound into thorax (860.3) Traumatic hemothorax with open wound into thorax (860.4) Traumatic ... Traumatic pneumothorax and hemothorax (860.0) Traumatic pneumothorax without open wound into thorax (860.1) Traumatic ...
EmbolizationEmbolization
Cerebral aneurysm Gastrointestinal bleeding Nosebleed Varicocele Primary post-partum bleeding Surgical hemorrhage traumatic ... Patients who undergo cerebral embolization or portal vein embolization are usually given a general anesthetic. Access to the ... Not suitable for everyone Recurrence more likely Cerebral aneurysm Endovascular surgery Gastrointestinal bleeding ... "Serious primary post-partum hemorrhage, arterial embolization and future fertility: a retrospective study of 46 cases". Hum. ...
AcoustocerebrographyAcoustocerebrography
... from cerebral embolism) or bleeding (from cerebral hemorrhage). All brain tissue is influenced by blood circulating in the ... This can be especially helpful during the acute phase directly after a stroke or a traumatic brain injury. The measured data is ... Cranial Accelerometry Can Detect Cerebral Vasospasm Caused by Subarachnoid Hemorrhage, Smith et al. (2015) Recording and ... ACG also facilitates blood flow analysis as well as the detection of obstructions in cerebral blood flow ( ...
குருதிப்பெருக்கு - தமிழ் விக்கிப்பீடியாகுருதிப்பெருக்கு - தமிழ் விக்கிப்பீடியா
மூளை இரத்த ஒழுக்கு (cerebral Haemorrhage) உட்புற இரத்த ஒழுக்கில் மூளை இரத்த ஒழுக்கு மிகவும் கவனிக்கப்பட வேண்டிய ஒன்று. இது ... பேரதிர்ச்சி குருதிப் பெருக்கு (Traumatic bleeding) புறவழிக் காயங்களினால் ஏற்படுகின்றது. பல வகையான காயங்கள் இத்தகு ... Liberty G, Hyman JH, Eldar-Geva T, Latinsky B, Gal M, Margalioth EJ (December 2008). "Ovarian hemorrhage after transvaginal ... பெருமூளையுள் குருதிப் பெருக்கு (Intracerebral hemorrhage) இரத்தக்கசிவு வாதம். *வலையுறையடி குருதிப் பெருக்கு (Subarachnoid ...
HeadacheHeadache
Brain bleeds (intracranial hemorrhage, subdural hematoma, epidural hematoma), post-traumatic headache. Trauma can cause ... A lumbar puncture is a procedure in which cerebral spinal fluid is removed from the spine with a needle. A lumbar puncture is ... bleeding inside the brain (intracranial hemorrhage). *subarachnoid hemorrhage (acute, severe headache, stiff neck without fever ... Brain bleed (subarachnoid hemorrhage, hemorrhage into mass lesion, vascular malformation), pituitary apoplexy, mass (especially ...
Cerebral hypoxiaCerebral hypoxia
Severe cerebral hypoxia and anoxia is usually caused by traumatic events such as choking, drowning, strangulation, smoke ... Cerebral edema, brain hemorrhages and hydrocephalus exert pressure on brain tissue and impede their absorption of oxygen. ... diffuse cerebral hypoxia (DCH), focal cerebral ischemia, cerebral infarction, and global cerebral ischemia. Prolonged hypoxia ... Cerebral infarction - A "stroke", caused by complete oxygen deprivation due to an interference in cerebral blood flow which ...
AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPsychiatry and PsychologyPhenomena and ProcessesInformation ScienceNamed GroupsHealth CareGeographicals
Cerebral HemorrhageSubarachnoid HemorrhageCerebral InfarctionHemorrhageCerebral ArteriesCerebral Amyloid AngiopathyCerebrovascular DisordersIntracranial HemorrhagesIntracranial Hemorrhage, HypertensiveCapillary FragilityCerebral AngiographyCerebral Amyloid Angiopathy, FamilialMiddle Cerebral ArteryCerebrovascular CirculationBrainTomography, X-Ray ComputedAmyloidosisBrain IschemiaIntracranial Embolism and ThrombosisStrokeHematomaRetinal HemorrhageIntracranial AneurysmHemangioma, Cavernous, Central Nervous SystemCerebral Hemorrhage, TraumaticTissue Plasminogen ActivatorCerebral PalsyIntracranial Arteriovenous MalformationsAphasia, WernickeCystatinsPostpartum HemorrhageRupture, SpontaneousIschemic Attack, TransientJapanBrain EdemaTime FactorsGastrointestinal HemorrhageFatal OutcomeStreptococcus anginosusMagnetic Resonance ImagingHypertensionFibrinolytic AgentsVitreous HemorrhageEye HemorrhageTreatment OutcomeAcute DiseaseNeurologic ExaminationAutonomic DysreflexiaInfarction, Middle Cerebral ArteryMoyamoya DiseaseVasospasm, IntracranialCerebral VeinsRisk FactorsMalaria, CerebralAmyloid beta-PeptidesAnticoagulantsThrombolytic TherapyCystatin CAmyloidRetrospective StudiesProspective StudiesBlood PressureIntracranial EmbolismCerebral VentriclesAlzheimer DiseaseBasal Ganglia HemorrhageDisease Models, AnimalFollow-Up StudiesIncidenceAmyloid beta-Protein PrecursorPostoperative HemorrhageInfant, NewbornAge FactorsRecurrenceRiskPostoperative ComplicationsAneurysm, RupturedBasilar ArteryChoroid HemorrhagePeptic Ulcer HemorrhageIntracranial PressureAnterior Cerebral ArteryPrognosisPosterior Cerebral ArterySex FactorsPutaminal HemorrhageBrain DiseasesMutationHydrocephalusBlood Flow VelocityHematoma, SubduralRetrobulbar HemorrhageCerebral RevascularizationBlood VolumeEmbolization, TherapeuticCraniotomyCerebral VentriculographyDominance, CerebralBrain Hemorrhage, TraumaticShock, HemorrhagicRats, Sprague-DawleyNeuroprotective AgentsIntracranial ThrombosisCerebrumBlood-Brain BarrierIntracranial HypertensionHypoxia, BrainGlasgow Coma ScaleMagnetic Resonance Angiography
Cerebral hemorrhage, traumatic | definition of Cerebral hemorrhage, traumatic by Medical dictionaryCerebral hemorrhage, traumatic | definition of Cerebral hemorrhage, traumatic by Medical dictionary
What is Cerebral hemorrhage, traumatic? Meaning of Cerebral hemorrhage, traumatic medical term. What does Cerebral hemorrhage, ... traumatic in the Medical Dictionary? Cerebral hemorrhage, traumatic explanation free. ... Related to Cerebral hemorrhage, traumatic: Traumatic subarachnoid haemorrhage. hemorrhage. [hem´ŏ-rij] the escape of blood from ... cerebral hemorrhage. (redirected from Cerebral hemorrhage, traumatic). Also found in: Dictionary, Thesaurus. ...
medical-dictionary.thefreedictionary.com
Traumatic cerebral hemorrhage - Semantic ScholarTraumatic cerebral hemorrhage - Semantic Scholar
Hemorrhage may involve any part of the CEREBRAL CORTEX and the BASAL GANGLIA. Depending on the severity of bleeding, clinical ... Bleeding into one or both CEREBRAL HEMISPHERES due to TRAUMA. ... Traumatic cerebral hemorrhage. Known as: Traumatic Cerebral ... Traumatic Intracerebral Hemorrhage, Hemorrhages, Traumatic Intracerebral (More). Bleeding into one or both CEREBRAL HEMISPHERES ... Hemorrhage may involve any part of the CEREBRAL CORTEX and the BASAL GANGLIA. Depending… (More) ...
semanticscholar.org
Detection of critical oligemia in patients with traumatic intracerebral hemorrhage using continuous quantitative...Detection of critical oligemia in patients with traumatic intracerebral hemorrhage using continuous quantitative...
Ten patients with traumatic intracerebral hemorrhage underwent combined monitoring of cerebral microdialysis, EEGPAV, and ... Hemorrhage. Detection of critical oligemia in patients with traumatic intracerebral hemorrhage using continuous quantitative ... Introduction: Serial changes in cerebral blood flow after intracerebral hemorrhage may influence tissue viability in regions ... oligemia in patients with traumatic intracerebral hemorrhage using continuous quantitative electroencephalography and cerebral ...
stroke.ahajournals.org
Study on predictive function of TEG on blood coagulation changes in patients with cerebral traumatic hemorrhageStudy on predictive function of TEG on blood coagulation changes in patients with cerebral traumatic hemorrhage
... , Lin Lu, ... TEG, Cerebral traumatic hemorrhage, Blood coagulation function, Predictive function.. Introduction. After cerebral traumatic ... Patients with cerebral traumatic hemorrhage have certain differences with patients with other kinds of traumatic hemorrhage in ... which included 42 patients with cerebral traumatic hemorrhage and 12 with other kinds of traumatic hemorrhage, and 40 were male ...
alliedacademies.org
Videos • Cerebral Hemorrhage, TraumaticVideos • Cerebral Hemorrhage, Traumatic
Traumatic. On-line free medical diagnosis assistant. Ranked list of possible diseases from either several symptoms or a full ... Cerebral Hemorrhage, Traumatic. Cerebral hemorrhage - YouTube. A cerebral haemorrhage (also spelled hemorrhage; aka ... Traumatic Subarachnoid Haemorrhage PDF - YouTube. Traumatic Subarachnoid Haemorrhage PDF ... Trauma Biomechanics An ... Fatal Brain Haemorrhage - YouTube. A brain haemorrhage is bleeding in or around the brain, which may be caused by a traumatic ...
lookfordiagnosis.com
FAQ • Cerebral Hemorrhage, TraumaticFAQ • Cerebral Hemorrhage, Traumatic
Traumatic. On-line free medical diagnosis assistant. Ranked list of possible diseases from either several symptoms or a full ... Cerebral Hemorrhage, Traumatic. (Powered by Yahoo! Answers). cerebral hemorrhage?. A 73 yr old women suffers a severe cerebral ... Can you get a cerebral hemorrhage if you were walking and the heel of your high heels break?. Not unless you fell and smacked ... A cerebral hemorrhage is bleeding in the brain. (+ info). 1 2 3 4 5 ...
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News • Cerebral Hemorrhage, TraumaticNews • Cerebral Hemorrhage, Traumatic
Traumatic. On-line free medical diagnosis assistant. Ranked list of possible diseases from either several symptoms or a full ... Cerebral Hemorrhage, Traumatic. No results. We do not evaluate or guarantee the accuracy of any content in this site. Click ...
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Cerebral Hemorrhage, Traumatic; Brain Hemorrhage, Cerebral, Traumatic; Cerebral Hematoma, Traumatic; Intracerebral Hemorrhage,...Cerebral Hemorrhage, Traumatic; Brain Hemorrhage, Cerebral, Traumatic; Cerebral Hematoma, Traumatic; Intracerebral Hemorrhage,...
Traumatic; Brain Hemorrhage, Cerebral, Traumatic; Cerebral Hematoma, Traumatic; Intracerebral Hemorrhage, Traumatic. On-line ... Cerebral Hemorrhage, Traumatic; Brain Hemorrhage, Cerebral, Traumatic; Cerebral Hematoma, Traumatic; Intracerebral Hemorrhage, ...
lookfordiagnosis.com
Cases reported • Cerebral Hemorrhage, Traumatic; Brain Hemorrhage, Cerebral, Traumatic; Cerebral Hematoma, Traumatic;...Cases reported • Cerebral Hemorrhage, Traumatic; Brain Hemorrhage, Cerebral, Traumatic; Cerebral Hematoma, Traumatic;...
Traumatic; Brain Hemorrhage, Cerebral, Traumatic; Cerebral Hematoma, Traumatic; Intracerebral Hemorrhage, Traumatic. On-line ... Traumatic basilar artery dissection in a child: need for anticoagulation? dissection of a cerebral blood vessel is a rare ... Traumatic false aneurysm of the middle meningeal artery causing an intracerebral hemorrhage: case report and literature review ... CONCLUSION: Although rare, traumatic meningeal aneurysms should be considered as a possible cause of cerebral hematoma. Because ...
lookfordiagnosis.com
Intra-arterial nimodipine to treat symptomatic cerebral vasospasm following traumatic subarachnoid haemorrhage. Technical case...Intra-arterial nimodipine to treat symptomatic cerebral vasospasm following traumatic subarachnoid haemorrhage. Technical case...
Barzo, P. / Intra-arterial nimodipine to treat symptomatic cerebral vasospasm following traumatic subarachnoid haemorrhage. ... Barzo, P 2008, Intra-arterial nimodipine to treat symptomatic cerebral vasospasm following traumatic subarachnoid haemorrhage ... Intra-arterial nimodipine to treat symptomatic cerebral vasospasm following traumatic subarachnoid haemorrhage. Technical case ... Intra-arterial nimodipine to treat symptomatic cerebral vasospasm following traumatic subarachnoid haemorrhage. Technical case ...
hungary.pure.elsevier.com
Everything You Ever Wanted to Know About Your Childs Traumatic Brain Injury (TBI) 11/20/2014 - The Institutes For The...Everything You Ever Wanted to Know About Your Child's Traumatic Brain Injury (TBI) 11/20/2014 - The Institutes For The...
Such injuries include blows to the head, lack of oxygen from suffocation, smoke inhalation or near drowning, hemorrhages, brain ... Traumatic Brain Injury - Any injury to the brain sustained after birth. ... Cerebral Hemorrhages. Hemorrhages of TBI may be outside or inside the brain. Inside the skull the brain is surrounded by a ... Cerebral hemorrhages are named according to their anatomical location:. 1. Epidural hemorrhage: Between the skull and the outer ...
iahp.org
Neurologic Stem Cell Treatment Study - Full Text View - ClinicalTrials.govNeurologic Stem Cell Treatment Study - Full Text View - ClinicalTrials.gov
Chronic Traumatic Encephalopathy Cerebral Infarction Cerebral Ischemia Cerebral Stroke Cerebral Hemorrhage Parkinson Multi- ... Cerebral Hemorrhage. CADASIL. Chronic Traumatic Encephalopathy. Hypotension, Orthostatic. Cerebrovascular Disorders. Central ... Cerebral Vascular Accident. Stroke. Traumatic Brain Injury. Multiple Sclerosis. Parkinsons Disease. Neuropathy. ... Hemorrhage. Ischemia. Peripheral Nervous System Diseases. Brain Injuries, Traumatic. Atrophy. Motor Neuron Disease. Brain ...
clinicaltrials.gov
Neurologic Stem Cell Treatment Study - Full Text View - ClinicalTrials.govNeurologic Stem Cell Treatment Study - Full Text View - ClinicalTrials.gov
Chronic Traumatic Encephalopathy Cerebral Infarction Cerebral Ischemia Cerebral Stroke Cerebral Hemorrhage Parkinson Multi- ... Cerebral Hemorrhage. CADASIL. Chronic Traumatic Encephalopathy. Disease. Sclerosis. Hemorrhage. Ischemia. Nervous System ... Cerebral Vascular Accident. Stroke. Traumatic Brain Injury. Multiple Sclerosis. Parkinsons Disease. Neuropathy. ... Brain Injuries, Traumatic. Motor Neuron Disease. Amyotrophic Lateral Sclerosis. Brain Diseases. Diabetic Neuropathies. Cerebral ...
clinicaltrials.gov
Traumatic Rupture of the Middle Cerebral Artery Followed by Acute Basal Subarachnoid Hemorrhage: Tailored Approach in Forensic...Traumatic Rupture of the Middle Cerebral Artery Followed by Acute Basal Subarachnoid Hemorrhage: Tailored Approach in Forensic...
Middle Cerebral Artery , Neck , Rupture , Subarachnoid Hemorrhage , Subarachnoid Hemorrhage, Traumatic , Vascular System ... Traumatic Rupture of the Middle Cerebral Artery Followed by Acute Basal Subarachnoid Hemorrhage: Tailored Approach in Forensic ... Middle Cerebral Artery / Subarachnoid Hemorrhage, Traumatic Clinical aspect: Diagnosis / Etiology Language: English Journal: ... Traumatic Rupture of the Middle Cerebral Artery Followed by Acute Basal Subarachnoid Hemor ...
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Public Records: Deaths filed December 15-31Public Records: Deaths filed December 15-31
Palamarchuk, Dmitriy; May 11, 1931, Evansville; non-traumatic cerebral hemorrhage, Dec. 25 ... 17, 1929, Evansville; subdural hemorrhage, blunt force trauma to the head, fall impacting head on ground, Dec. 13 ... Rudolph, Bernice Rose; March 24, 1925, Evansville; intracerebral hemorrhage, Dec. 13. Rushing, Charles Douglas; Nov. 7, 1944, ... 2, 1926, Evansville; respiratory failure, cerebral vascular accident, atherosclerotic cardiovascular disease, hypertension, Dec ...
courierpress.com
Lactate Therapy After Traumatic Brain Injury - Full Text View - ClinicalTrials.govLactate Therapy After Traumatic Brain Injury - Full Text View - ClinicalTrials.gov
traumatic brain injury. lactate. brain metabolism. cerebral microdialysis. subarachnoid hemorrhage. transcranial doppler. ... In patients with severe Traumatic Brain Injury (TBI) and aneurysmal subarachnoid hemorrhage (SAH) monitored with cerebral ... Brain Injuries, Traumatic. Brain Diseases. Wounds and Injuries. Hemorrhage. Subarachnoid Hemorrhage. Central Nervous System ... Lactate Therapy After Traumatic Brain Injury (LS_TCC). The safety and scientific validity of this study is the responsibility ...
clinicaltrials.gov
Brainstem Disorders, Book by Peter P. Urban (Hardcover) | chapters.indigo.caBrainstem Disorders, Book by Peter P. Urban (Hardcover) | chapters.indigo.ca
Intraparenchymatous brainstem hemorrhage.-Perimesencephalic subarachnoid hemorrhage and cerebral superficial siderosis.- ... Traumatic Brainstem Lesions.-Degenerative brainstem disorders.-Abnormalities of brainstem development.-Metabolic brainstem ...
chapters.indigo.ca
A Phase III Trial of ZD4054 (Zibotentan) (Endothelin A Antagonist) in Hormone Resistant Prostate Cancer With Bone Metastases -...A Phase III Trial of ZD4054 (Zibotentan) (Endothelin A Antagonist) in Hormone Resistant Prostate Cancer With Bone Metastases -...
Cerebral Haemorrhage Traumatic † 1 0/298 (0.00%) 1/295 (0.34%) Cervical Vertebral Fracture † 1 1/298 (0.34%) 1/295 (0.34%) ...
clinicaltrials.gov
Bleeding Liver Masses: Imaging Features With Pathologic Correlation and Impact on Management.Bleeding Liver Masses: Imaging Features With Pathologic Correlation and Impact on Management.
The purposes of this article are to discuss a variety of liver masses that can present with hemorrhage, including their ... Cerebral Hemorrhage, Traumatic. Bleeding into one or both CEREBRAL HEMISPHERES due to TRAUMA. Hemorrhage may involve any part ... Traumatic and atraumatic fractures are entities with distinct but often overlapping clinical manifestations, imaging findings, ... Pathologic features include cerebral prion protein amyloidosis, and spongiform or neurofibrillary degeneration. (From Brain ...
bioportfolio.com
cerebral - Symptoms, Treatments and Resources for cerebralcerebral - Symptoms, Treatments and Resources for cerebral
Treatments and Tools for cerebral. Find cerebral information, treatments for cerebral and cerebral symptoms. ... cerebral - MedHelps cerebral Center for Information, Symptoms, Resources, ... Need an opinion on MRI of head trauma - probable cerebral haemorrhage. - Traumatic Brain Injury Community ... A male patient, at the age of 21 years, suffered from cerebral hemorrhage and thus underwen... ...
medhelp.org
Sofia Asioli - University of Bologna - ANATOMIC PATHOLOGY 12 cfu (6 per Semester - IV year Medical School) Program for Tracks...Sofia Asioli - University of Bologna - ANATOMIC PATHOLOGY 12 cfu (6 per Semester - IV year Medical School) Program for Tracks...
Cerebral edema.. Hydrocephalous.. Trauma and traumatic vascular lesions (epi and sub-dural hematoma) ... intracranial haemorrhages; primitive intraparenchymal haemorrhages; saccular aneurysms; sub-arachnoid haemorrhages; vascular ... Pathology and pathogenesis of: thromboembolism, pulmonary infarction and hemorrhage. Pulmonary hypertension (primary and ... Vascular lesions. Morphological features and pathogenesis of: hypoxic-ischemic encephalopathy; cerebral infarct, ...
unibo.it
Nao-Xue-Shu Oral Liquid Protects and Improves Secondary Brain Insults of Hypertensive Cerebral HemorrhageNao-Xue-Shu Oral Liquid Protects and Improves Secondary Brain Insults of Hypertensive Cerebral Hemorrhage
A. I. Maas, H. F. Lingsma, and B. Roozenbeek, "Predicting outcome after traumatic brain injury," in Handbook of Clinical ... If the SBI is caused by hypertensive cerebral hemorrhage (HCH), it will be defined as hypertensive cerebral hemorrhage SBI (HCH ... The cerebral hemorrhage was the first in 151 cases and the second in 7 cases. Intracerebral hemorrhage of the patients was ... suffering from a cerebral hemorrhage and 147 cases had a higher blood pressure level than normal when the cerebral hemorrhage ...
hindawi.com
Frontiers | Melatonin Prevents Mice Cortical Astrocytes From Hemin-Induced Toxicity Through Activating PKCα/Nrf2/HO-1 Signaling...Frontiers | Melatonin Prevents Mice Cortical Astrocytes From Hemin-Induced Toxicity Through Activating PKCα/Nrf2/HO-1 Signaling...
Secondary injury mediated by oxidative stress leads to deterioration of neurological function after intracerebral hemorrhage ( ... Secondary injuries mediated by oxidative stress lead to deterioration of neurological functions after intracerebral hemorrhage ... cerebral ischemia (Shih et al., 2005), traumatic brain injury (Wang J. et al., 2018), and cerebral hemorrhage (Chen et al., ... This experiment used mice astrocytes to simulate oxidative stress of cerebral hemorrhage in vitro in order to study the ...
frontiersin.org
Traumatic Brain Injury Forecast in 21 Major Markets 2016-2026: Risk Factors, Disease Diagnosis & Prognosis - Research and...Traumatic Brain Injury Forecast in 21 Major Markets 2016-2026: Risk Factors, Disease Diagnosis & Prognosis - Research and...
PRNewswire/ -- Research and Markets has announced the addition of the Traumatic Brain Injury Forecast in 21 Major Markets 2016 ... Strokes and cerebral haemorrhage including; *Mental health issues such as depression, panic and generalised anxiety disorder; ... Traumatic brain injury (TBI) or acquired brain injury is a non-specific term describing blunt, penetrating or blast injuries to ... Research and Markets has announced the addition of the Traumatic Brain Injury Forecast in 21 Major Markets 2016-2026 report ...
prnewswire.co.uk
Prevalence of Traumatic Findings on Routine MRI in a Large Cohort of Professional Fighters | American Journal of NeuroradiologyPrevalence of Traumatic Findings on Routine MRI in a Large Cohort of Professional Fighters | American Journal of Neuroradiology
Multiple traumatic cerebral hemorrhages. Proc New York Path Soc 1924;24:101. ... cerebral microhemorrhage. CSP. cavum septum pellucidum. CSPV. cavum septum pellucidum and vergae. CV. cavum vergae. NSWMC. ... Prevalence of Traumatic Findings on Routine MRI in a Large Cohort of Professional Fighters. J.K. Lee, J. Wu, S. Banks, C. ... Prevalence of cerebral microhemorrhages in amateur boxers as detected by 3T MR imaging. AJNR Am J Neuroradiol 2008;29:388-91 ...
ajnr.org
AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPsychiatry and PsychologyPhenomena and ProcessesInformation ScienceNamed GroupsHealth CareGeographicals
Cerebral HemorrhageSubarachnoid HemorrhageCerebral InfarctionHemorrhageCerebral ArteriesCerebral Amyloid AngiopathyCerebrovascular DisordersIntracranial HemorrhagesIntracranial Hemorrhage, HypertensiveCapillary FragilityCerebral AngiographyCerebral Amyloid Angiopathy, FamilialMiddle Cerebral ArteryCerebrovascular CirculationBrainTomography, X-Ray ComputedAmyloidosisBrain IschemiaIntracranial Embolism and ThrombosisStrokeHematomaRetinal HemorrhageIntracranial AneurysmHemangioma, Cavernous, Central Nervous SystemCerebral Hemorrhage, TraumaticTissue Plasminogen ActivatorCerebral PalsyIntracranial Arteriovenous MalformationsAphasia, WernickeCystatinsPostpartum HemorrhageRupture, SpontaneousIschemic Attack, TransientJapanBrain EdemaTime FactorsGastrointestinal HemorrhageFatal OutcomeStreptococcus anginosusMagnetic Resonance ImagingHypertensionFibrinolytic AgentsVitreous HemorrhageEye HemorrhageTreatment OutcomeAcute DiseaseNeurologic ExaminationAutonomic DysreflexiaInfarction, Middle Cerebral ArteryMoyamoya DiseaseVasospasm, IntracranialCerebral VeinsRisk FactorsMalaria, CerebralAmyloid beta-PeptidesAnticoagulantsThrombolytic TherapyCystatin CAmyloidRetrospective StudiesProspective StudiesBlood PressureIntracranial EmbolismCerebral VentriclesAlzheimer DiseaseBasal Ganglia HemorrhageDisease Models, AnimalFollow-Up StudiesIncidenceAmyloid beta-Protein PrecursorPostoperative HemorrhageInfant, NewbornAge FactorsRecurrenceRiskPostoperative ComplicationsAneurysm, RupturedBasilar ArteryChoroid HemorrhagePeptic Ulcer HemorrhageIntracranial PressureAnterior Cerebral ArteryPrognosisPosterior Cerebral ArterySex FactorsPutaminal HemorrhageBrain DiseasesMutationHydrocephalusBlood Flow VelocityHematoma, SubduralRetrobulbar HemorrhageCerebral RevascularizationBlood VolumeEmbolization, TherapeuticCraniotomyCerebral VentriculographyDominance, CerebralBrain Hemorrhage, TraumaticShock, HemorrhagicRats, Sprague-DawleyNeuroprotective AgentsIntracranial ThrombosisCerebrumBlood-Brain BarrierIntracranial HypertensionHypoxia, BrainGlasgow Coma ScaleMagnetic Resonance Angiography
InjuryIntracranial pressureAneurysmMiddle CerebrEdemaIntraventricular hemorrhageHemorrhagicPrevented cerebral vasospasmConcussionSevere traumaticEpidural hemorrhagePosteriorSubarachnoid hemorrhagesPatients with traumatic injuryBrain HemorrhageCraniotomyBrainstemRuptureAngiographyVascularIntraparenchymal hemorrhageStrokesAneurysmsVenous sinus thrombosisArterialSpinalBloodExperimental subarachnoid hemorrhageOutcomePost-traumatic
Injury2
Intracranial pressure14
Aneurysm13
Middle Cerebr6
  • On histologic examination, a microscopic focal rupture was identified at the proximal portion of the middle cerebral artery, and possibility of arteriopathy was considered. (bvsalud.org)
  • A series of angiographic analyses were performed, before SAH and on Days 7 and 14, to examine changes in the diameters of the ipsilateral internal carotid artery, middle cerebral artery, and anterior cerebral artery. (biomedsearch.com)
  • RESULTS: In the SAH and placebo groups, cerebral vasospasm developed on Day 7 (54.8% of the pre-SAH value at the internal carotid artery, 62.3% at the middle cerebral artery, 51.3% at the anterior cerebral artery, and 56.1% as an average of the three arteries). (biomedsearch.com)
  • The lesion is caused by an occlusion of the left middle cerebral artery and is associated with damage to Broca's area, Wernicke's area, and insular regions which are associated with aspects of language. (wikipedia.org)
  • Global aphasia typically results from an occlusion to the trunk of the middle cerebral artery (MCA), which affects a large portion of the perisylvian region of the left cortex. (wikipedia.org)
  • Causes of Amorphosynthesis are: Cerebrovascular accidents that have affected cerebral hemispheres, such as occlusion of the right middle cerebral artery. (wikipedia.org)
Edema18
Intraventricular hemorrhage6
Hemorrhagic2
Prevented cerebral vasospasm1
  • The CGRP concentration was positive only on Day 7 for the CGRP group (6.5 nmol/L). CONCLUSION: The CGRP tablet prevented cerebral vasospasm after SAH and may have significant potential for the treatment of patients with SAH. (biomedsearch.com)
Concussion2
Severe traumatic6
Epidural hemorrhage2
Posterior2
  • CSF is produced by modified ependymal cells of the choroid plexus found in all components of the ventricular system except for the cerebral aqueduct and the posterior and anterior horns of the lateral ventricles. (wikipedia.org)
  • Thus, when an increase of pressure in a hemisphere of the brain exists, the cerebral peduncle on the opposite hemisphere is pushed up against the tentorium, which separates the posterior fossa from the middle fossa. (wikipedia.org)
Subarachnoid hemorrhages3
Patients with traumatic injury1
  • Other patients with traumatic injury were regarded as B group, consisting of 8 male cases and 4 female cases (Age range: 23-62 y, mean ± sd=36.6 ± 5.6 y). (alliedacademies.org)
Brain Hemorrhage5
Craniotomy3
Brainstem12
Rupture5
Angiography3
  • interventions A computed tomography scan may be performed to locate the lesion and to differentiate the hemorrhage from an embolus or thrombus, or cerebral angiography may be used for these purposes. (thefreedictionary.com)
  • Diagnostic angiography The first experience with cerebral angiography was developed by Portuguese doctor Egas Moniz at the University of Lisbon, in order to identify central nervous system diseases such as tumors or arteriovenous malformations. (wikipedia.org)
  • He performed the first brain angiography in Lisbon in 1927 by injecting iodinated contrast medium into a carotid and using the rays discovered 30 years earlier by Roentgen to visualize the cerebral vessels. (wikipedia.org)
Vascular6
Intraparenchymal hemorrhage1
Strokes1
  • Publication date: Available online 12 December 2019Source: Anaesthesia &Intensive Care MedicineAuthor(s): Christopher J. TaylorAbstractIntracerebral haemorrhage (ICH) accounts for around 10-20% of all strokes and results from a variety of disorders. (medworm.com)
Aneurysms1
Venous sinus thrombosis1
Arterial6
Spinal1
  • There is documentation in the scientific literature that intranasal delivery of BMSC allows the BMSC to follow the pathways of the trigeminal nerve, facilitating entry into the parenchyma and cerebral spinal fluid (CSF) for effects on the CNS. (clinicaltrials.gov)
Blood29
Experimental subarachnoid hemorrhage1
Outcome3
Post-traumatic4
  • This pilot study is the first step in evaluation of levetiracetam in prevention of post-traumatic epilepsy. (clinicaltrials.gov)
  • Methods: Descriptive case series of 26 patients of post traumatic CCF treated by endovascular techniques was carried out at Radiology and Neurosurgery departments of Aga Khan University hospital between January 2010 to March 2010. (thefreelibrary.com)
  • Some consider late PTS to be synonymous with post-traumatic epilepsy. (wikipedia.org)
  • A current model developed by the Department of Defense and Department of Veterans Affairs uses all three criteria of GCS after resuscitation, duration of post-traumatic amnesia (PTA), and loss of consciousness (LOC). (wikipedia.org)