Cercopithecus
Cercopithecus aethiops
Erythrocebus patas
Monkey Diseases
Haplorhini
Cercopithecinae
Genes, vpu
Cameroon
Simian immunodeficiency virus
Species Specificity
Simian T-lymphotropic virus 3
Cercocebus
Yaba monkey tumor virus
Deltaretrovirus Infections
Simian T-lymphotropic virus 1
Primates
Induction of AT-specific DNA-interstrand crosslinks by bizelesin in genomic and simian virus 40 DNA. (1/15816)
Bizelesin is a bifunctional AT-specific DNA alkylating drug. Our study characterized the ability of bizelesin to induce interstrand crosslinks, a potential lethal lesion. In genomic DNA of BSC-1 cells, bizelesin formed from approx. 0.3 to 6.03+/-0.85 interstrand crosslinks per 106 base pairs, at 5-100 nM drug concentration, respectively, comparable to the number of total adducts previously determined in the same system (J.M. Woynarowski, M.M. McHugh, L.S. Gawron, T.A. Beerman, Biochemistry 34 (1995) 13042-13050). Bizelesin did not induce DNA-protein crosslinks or strand breaks. A model defined target, intracellular simian virus 40 (SV40) DNA, was employed to map at the nucleotide level sites of bizelesin adducts, including potential interstrand crosslinks. Preferential adduct formation was observed at AT tracts which are abundant in the SV40 matrix associated region and the origin of replication. Many sites, including each occurrence of 5'-T(A/T)4A-3', co-mapped on both DNA strands suggesting interstrand crosslinks, although monoadducts were also formed. Bizelesin adducts in naked SV40 DNA were found at similar sites. The localization of bizelesin-induced crosslinks in AT-rich tracts of replication-related regions is consistent with the potent anti-replicative properties of bizelesin. Given the apparent lack of other types of lesions in genomic DNA, interstrand crosslinks localized in AT-rich tracts, and to some extent perhaps also monoadducts, are likely to be lethal effects of bizelesin. (+info)Deletion of multiple immediate-early genes from herpes simplex virus reduces cytotoxicity and permits long-term gene expression in neurons. (2/15816)
Herpes simplex virus type 1 (HSV-1) has many attractive features that suggest its utility for gene transfer to neurons. However, viral cytotoxicity and transient transgene expression limit practical applications even in the absence of viral replication. Mutant viruses deleted for the immediate early (IE) gene, ICP4, an essential transcriptional transactivator, are toxic to many cell types in culture in which only the remaining IE genes are expressed. In order to test directly the toxicity of other IE gene products in neurons and develop a mutant background capable of longterm transgene expression, we generated mutants deleted for multiple IE genes in various combinations and tested their relative cytotoxicity in 9L rat gliosarcoma cells, Vero monkey kidney cells, and primary rat cortical and dorsal root neurons in culture. Viral mutants deleted simultaneously for the IE genes encoding ICP4, ICP22 and ICP27 showed substantially reduced cytotoxicity compared with viruses deleted for ICP4 alone or ICP4 in combination with either ICP22, ICP27 or ICP47. Infection of neurons in culture with these triple IE deletion mutants substantially enhanced cell survival and permitted transgene expression for over 21 days. Such mutants may prove useful for efficient gene transfer and extended transgene expression in neurons in vitro and in vivo. (+info)T-cell lymphoma in a savanna monkey (Cercopithecus aethiops) probably related to simian T-cell leukemia virus infection. (3/15816)
Lymphoma was seen in an 11-year-old female savanna monkey (Ceropithecus aethiops). The superficial inguinal and visceral lymph nodes were markedly enlarged, and their architecture was completely effaced by neoplastic cells. The neoplastic cells, which were highly pleomorphic, resembled those in adult T-cell lymphoma-leukemia in humans. Ultrastructurally the neoplastic cells were characterized by nuclear irregularity and clustered dense bodies, and almost all cells showed positivity for CD3. The animal had been reared with her family, and her mother and 2 brothers had antibodies reactive to human T-cell leukemia virus. This virus serologically cross-reacts with simian T-cell leukemia virus, which may be the causative agent of the present neoplasm. (+info)A lipid modified ubiquitin is packaged into particles of several enveloped viruses. (4/15816)
An anti-ubiquitin cross-reactive protein which migrates more slowly (6.5 kDa) by SDS-PAGE than ubiquitin was identified in African swine fever virus particles. This protein was extracted into the detergent phase in Triton X-114 phase separations, showing that it is hydrophobic, and was radiolabelled with both [3H]palmitic acid and [32P]orthophosphate. This indicates that the protein has a similar structure to the membrane associated phosphatidyl ubiquitin described in baculovirus particles. A similar molecule was found in vaccinia virus and herpes simplex virus particles, suggesting that it may be a component of uninfected cell membranes, which is incorporated into membrane layers in virions during morphogenesis. (+info)Microtubule-dependent plus- and minus end-directed motilities are competing processes for nuclear targeting of adenovirus. (5/15816)
Adenovirus (Ad) enters target cells by receptor-mediated endocytosis, escapes to the cytosol, and then delivers its DNA genome into the nucleus. Here we analyzed the trafficking of fluorophore-tagged viruses in HeLa and TC7 cells by time-lapse microscopy. Our results show that native or taxol-stabilized microtubules (MTs) support alternating minus- and plus end-directed movements of cytosolic virus with elementary speeds up to 2.6 micrometer/s. No directed movement was observed in nocodazole-treated cells. Switching between plus- and minus end-directed elementary speeds at frequencies up to 1 Hz was observed in the periphery and near the MT organizing center (MTOC) after recovery from nocodazole treatment. MT-dependent motilities allowed virus accumulation near the MTOC at population speeds of 1-10 micrometer/min, depending on the cell type. Overexpression of p50/dynamitin, which is known to affect dynein-dependent minus end-directed vesicular transport, significantly reduced the extent and the frequency of minus end-directed migration of cytosolic virus, and increased the frequency, but not the extent of plus end-directed motility. The data imply that a single cytosolic Ad particle engages with two types of MT-dependent motor activities, the minus end- directed cytoplasmic dynein and an unknown plus end- directed activity. (+info)Rubella virus-induced apoptosis varies among cell lines and is modulated by Bcl-XL and caspase inhibitors. (6/15816)
Rubella virus (RV) causes multisystem birth defects in the fetuses of infected women. To investigate the cellular basis of this pathology, we examined the cytopathic effect of RV in three permissive cell lines: Vero 76, RK13, and BHK21. Electron microscopy and the TUNEL assay showed that the cytopathic effect resulted from RV-induced programmed cell death (apoptosis) in all three cell lines, but the extent of apoptosis varied among these cells. At 48 h postinfection, the RK13 cell line showed the greatest number of apoptotic cells, the Vero 76 cell line was approximately 3-fold less, and BHK21 had very few. An increased multiplicity of infection and longer time postinfection were required for the BHK21 cell line to reach the level of apoptotic cells in Vero 76 at 48 h. Purified RV induced apoptosis in a dose-dependent fashion, but not UV-inactivated RV or virus-depleted culture supernatant. Specific inhibitors of the apoptosis-specific proteases caspases reduced RV-induced apoptosis and led to higher levels of RV components in infected cells. To address the role of regulatory proteins in RV-induced apoptosis, the antiapoptotic gene Bcl-2 or Bcl-XL was transfected into RK13 cells. Although a high level of Bcl-2 family proteins was expressed, no protection was observed from apoptosis induced by RV, Sindbis virus, or staurosporine in RK13 cells. In BHK21 cells, however, increased expression of Bcl-XL protected cells from apoptosis. The observed variability in apoptotic response to RV of these cell lines demonstrates that programmed cell death is dependent on the unique properties of each cell and may be indicative of how selective organ damage occurs in a congenital rubella syndrome fetus. (+info)CLIP-170 highlights growing microtubule ends in vivo. (7/15816)
A chimera with the green fluorescent protein (GFP) has been constructed to visualize the dynamic properties of the endosome-microtubule linker protein CLIP170 (GFP-CLIP170). GFP-CLIP170 binds in stretches along a subset of microtubule ends. These fluorescent stretches appear to move with the growing tips of microtubules at 0.15-0.4 microm/s, comparable to microtubule elongation in vivo. Analysis of speckles along dynamic GFP-CLIP170 stretches suggests that CLIP170 treadmills on growing microtubule ends, rather than being continuously transported toward these ends. Drugs affecting microtubule dynamics rapidly inhibit movement of GFP-CLIP170 dashes. We propose that GFP-CLIP170 highlights growing microtubule ends by specifically recognizing the structure of a segment of newly polymerized tubulin. (+info)Susceptibilities of Mycobacterium tuberculosis and Mycobacterium avium complex to lipophilic deazapteridine derivatives, inhibitors of dihydrofolate reductase. (8/15816)
Twelve lipophilic 2,4-diamino-5-methyl-5-deazapteridine derivatives and trimethoprim were evaluated for activity against Mycobacterium tuberculosis and Mycobacterium avium in vitro. Six of the compounds had MICs of < or =12.8 mg/L and < or =1.28 mg/L against M. tuberculosis and M. avium, respectively; trimethoprim MICs were >128 mg/L and >12.8 but < or =128 mg/L, respectively. Two compounds, with either a 2-methyl-5-methoxy phenyl or 2-methoxy-5-trifluoromethyl phenyl linked at the 6-position of the deazapteridine moiety by a CH2NH bridge, had MICs of < or =0.13 mg/L against M. avium; the two compounds also had apparent I50 values for dihydrofolate reductase of 2 and 8 nM, respectively, compared with an I50 of 400 nM with trimethoprim. Four of the compounds were selectively toxic to mycobacteria as compared with Vero cells. These results demonstrated that lipophilic antifolates can be synthesized which are more active against mycobacteria than trimethoprim and which possess selective toxicity. (+info)"Cercopithecus" is a genus of Old World monkeys that are commonly known as guenons. These monkeys are native to Africa and are characterized by their colorful fur, long tails, and distinctive facial features. They are agile animals that live in a variety of habitats, including forests, savannas, and mountains.
The term "Cercopithecus" is derived from the Greek words "kerkos," meaning tail, and "pithekos," meaning ape or monkey. This name reflects the long tails that are characteristic of these monkeys.
There are several species of guenons within the genus "Cercopithecus," including the vervet monkey, the grivet, the tantalus monkey, and the de Brazza's monkey, among others. These monkeys are important members of their ecosystems and play a key role in seed dispersal and forest regeneration. They are also popular subjects of research due to their complex social structures and behaviors.
'Cercopithecus aethiops' is the scientific name for the monkey species more commonly known as the green monkey. It belongs to the family Cercopithecidae and is native to western Africa. The green monkey is omnivorous, with a diet that includes fruits, nuts, seeds, insects, and small vertebrates. They are known for their distinctive greenish-brown fur and long tail. Green monkeys are also important animal models in biomedical research due to their susceptibility to certain diseases, such as SIV (simian immunodeficiency virus), which is closely related to HIV.
'Erythrocebus patas' is a scientific name for the Patas monkey, also known as the hussar monkey or red monkey. It belongs to the family Cercopithecidae and is native to the savannas and woodlands of central Africa. The Patas monkey is known for its long legs, slender body, and reddish-brown fur. It is the fastest primate, capable of reaching speeds up to 34 miles per hour (55 kilometers per hour).
The medical community may not have a specific definition related to 'Erythrocebus patas' as it is primarily studied by zoologists and biologists. However, understanding the characteristics and habits of this species can contribute to broader scientific knowledge and potentially inform research in fields such as comparative medicine or evolutionary biology.
There is no single medical definition for "Monkey Diseases." However, monkeys can carry and be infected with various diseases that are zoonotic, meaning they can be transmitted from animals to humans. Some examples include:
1. Simian Immunodeficiency Virus (SIV): A virus similar to Human Immunodeficiency Virus (HIV) that causes AIDS in monkeys. It is not typically harmful to monkeys but can cause AIDS in humans if transmitted, which is rare.
2. Herpes B Virus: Also known as Macacine herpesvirus 1 or Cercopithecine herpesvirus 1, it is a virus that commonly infects macaque monkeys. It can be transmitted to humans through direct contact with an infected monkey's saliva, eye fluid, or cerebrospinal fluid, causing a severe and potentially fatal illness called B encephalitis.
3. Tuberculosis (TB): Monkeys can contract and transmit tuberculosis to humans, although it is not common.
4. Simian Retrovirus (SRV): A virus that can infect both monkeys and great apes, causing immunodeficiency similar to HIV/AIDS in humans. It is not known to infect or cause disease in humans.
5. Various parasitic diseases: Monkeys can carry and transmit several parasites, including malaria-causing Plasmodium species, intestinal worms, and other parasites that can affect human health.
It's important to note that while monkeys can carry and transmit these diseases, the risk of transmission is generally low, and most cases occur in individuals who have close contact with monkeys, such as primatologists, zookeepers, or laboratory workers. Always follow safety guidelines when interacting with animals, including monkeys, to minimize the risk of disease transmission.
Haplorhini is a term used in the field of primatology and physical anthropology to refer to a parvorder of simian primates, which includes humans, apes (both great and small), and Old World monkeys. The name "Haplorhini" comes from the Greek words "haploos," meaning single or simple, and "rhinos," meaning nose.
The defining characteristic of Haplorhini is the presence of a simple, dry nose, as opposed to the wet, fleshy noses found in other primates, such as New World monkeys and strepsirrhines (which include lemurs and lorises). The nostrils of haplorhines are located close together at the tip of the snout, and they lack the rhinarium or "wet nose" that is present in other primates.
Haplorhini is further divided into two infraorders: Simiiformes (which includes apes and Old World monkeys) and Tarsioidea (which includes tarsiers). These groups are distinguished by various anatomical and behavioral differences, such as the presence or absence of a tail, the structure of the hand and foot, and the degree of sociality.
Overall, Haplorhini is a group of primates that share a number of distinctive features related to their sensory systems, locomotion, and social behavior. Understanding the evolutionary history and diversity of this group is an important area of research in anthropology, biology, and psychology.
Cercopithecinae is a subfamily of Old World monkeys that includes a diverse group of species, such as macaques, baboons, geladas, and langurs. These monkeys are characterized by their complex social structures, distinct coloration patterns, and adaptations to various habitats across Africa and Asia. Some notable features of Cercopithecinae include their tail-tufts, cheek pouches for food storage, and a variety of vocalizations used in communication.
I'm happy to help! However, I believe there may be a slight misunderstanding in your question. "VPU" is not a term that is typically associated with genes in the context of human or general molecular biology. It is possible that you are referring to the "vpu" gene found in certain viruses, such as HIV-1 (Human Immunodeficiency Virus type 1).
The "vpu" gene in HIV-1 encodes a viral accessory protein called Vpu, which plays a crucial role in the viral life cycle and pathogenesis. Among its functions, Vpu downregulates the restriction factor CD4 on the host cell surface, promotes virion release from infected cells, and induces degradation of the restrictive factor BST-2/Tetherin.
If you were indeed referring to the "vpu" gene or protein in HIV-1 or a related context, I apologize for any confusion, and I'm glad to provide further information or clarification if needed. If you meant something different by "Genes, vpu," could you please provide more context or details? I want to ensure that I offer the most accurate and helpful response possible.
I'm not aware of any medical condition or term that is specifically associated with or referred to as "Cameroon." Cameroon is a country located in Central Africa, known for its rich biodiversity and cultural diversity. If you have more context about why you are looking for a medical definition of "Cameroon," I may be able to provide a more helpful response.
Phylogeny is the evolutionary history and relationship among biological entities, such as species or genes, based on their shared characteristics. In other words, it refers to the branching pattern of evolution that shows how various organisms have descended from a common ancestor over time. Phylogenetic analysis involves constructing a tree-like diagram called a phylogenetic tree, which depicts the inferred evolutionary relationships among organisms or genes based on molecular sequence data or other types of characters. This information is crucial for understanding the diversity and distribution of life on Earth, as well as for studying the emergence and spread of diseases.
Simian Immunodeficiency Virus (SIV) is a retrovirus that primarily infects African non-human primates and is the direct ancestor of Human Immunodeficiency Virus type 2 (HIV-2). It is similar to HIV in its structure, replication strategy, and ability to cause an immunodeficiency disease in its host. SIV infection in its natural hosts is typically asymptomatic and non-lethal, but it can cause AIDS-like symptoms in other primate species. Research on SIV in its natural hosts has provided valuable insights into the mechanisms of HIV pathogenesis and potential strategies for prevention and treatment of AIDS.
Species specificity is a term used in the field of biology, including medicine, to refer to the characteristic of a biological entity (such as a virus, bacterium, or other microorganism) that allows it to interact exclusively or preferentially with a particular species. This means that the biological entity has a strong affinity for, or is only able to infect, a specific host species.
For example, HIV is specifically adapted to infect human cells and does not typically infect other animal species. Similarly, some bacterial toxins are species-specific and can only affect certain types of animals or humans. This concept is important in understanding the transmission dynamics and host range of various pathogens, as well as in developing targeted therapies and vaccines.
Simian T-lymphotropic virus 3 (STLV-3) is a retrovirus that primarily infects Asian monkeys, such as the Formosan rock macaque and the Assamese macaque. It is closely related to the human T-lymphotropic virus type 1 (HTLV-1), which can cause adult T-cell leukemia/lymphoma and tropical spastic paraparesis in humans.
STLV-3, like other retroviruses, has an RNA genome that is reverse transcribed into DNA upon infection of the host cell. The viral DNA then integrates into the host's chromosomal DNA, leading to a persistent infection.
While STLV-3 has been associated with lymphomas and leukemias in its simian hosts, it is not known to cause disease in humans. However, there have been reports of human infections with STLV-3, primarily among individuals who have close contact with infected monkeys or consume their meat. The potential public health implications of STLV-3 infection in humans are still being studied.
"Cercocebus" is a genus of Old World monkeys that includes several species, such as the sooty mangabey and the gray-cheeked mangabey. These monkeys are native to central Africa and are known for their distinctive appearance, with a long tail, dark fur, and light cheek whiskers. They are omnivorous and live in social groups. The name "Cercocebus" comes from the Greek words "kerkos," meaning tail, and "kephale," meaning head, referring to their long tails.
I'm sorry for any confusion, but there seems to be no established medical definition for "Yaba monkey tumor virus." It appears that this term may be a combination of the Yaba monkey tumor (a type of benign tumor observed in primates) and a virus. However, it is not a recognized term in virology or oncology.
Yaba monkey tumor is caused by the Yaba monkey tumor virus (YMTV), which is a member of the Poxviridae family and the Orthopoxvirus genus. It primarily affects non-human primates but can also incidentally infect humans, causing self-limiting papular or nodular skin lesions.
If you have any specific concerns about a medical condition or virus, I would be happy to help if you could provide more context or clarify the term in question.
Deltaretroviruses are a genus of retroviruses that can cause chronic infections in humans and animals. The two main deltaretroviruses that infect humans are the Human T-cell Leukemia Virus type 1 (HTLV-1) and Human T-cell Leukemia Virus type 2 (HTLV-2).
HTLV-1 is primarily transmitted through breastfeeding, sexual contact, and contaminated blood products. It can cause several diseases, including Adult T-cell Leukemia/Lymphoma (ATLL) and a neurological disorder called HTLV-1 Associated Myelopathy/Tropical Spastic Paraparesis (HAM/TSP).
HTLV-2 is primarily transmitted through intravenous drug use and sexual contact. While it has been associated with some diseases, such as neurological disorders and rare cases of leukemia, the link between HTLV-2 and disease is not as clear as it is for HTLV-1.
Deltaretrovirus infections can be diagnosed through blood tests that detect antibodies to the viruses or through genetic testing to detect the virus itself. There is currently no cure for deltaretrovirus infections, but antiretroviral therapy (ART) may help manage the infection and reduce the risk of transmission.
It's important to note that deltaretrovirus infections are relatively rare, and most people who are infected do not develop symptoms or disease. However, if you believe you may have been exposed to these viruses, it is important to speak with a healthcare provider for further evaluation and testing.
Simian T-lymphotropic virus 1 (STLV-1) is a retrovirus that primarily infects Asian monkeys and apes. It is closely related to the human T-lymphotropic virus type 1 (HTLV-1), and there is evidence to suggest that STLV-1 may have been transmitted to humans through close contact with infected non-human primates, resulting in the emergence of HTLV-1.
Like HTLV-1, STLV-1 primarily infects CD4+ T lymphocytes and can cause a persistent infection. However, unlike HTLV-1, which is associated with several diseases including adult T-cell leukemia/lymphoma and tropical spastic paraparesis/HTLV-1-associated myelopathy (TSP/HAM), STLV-1 has not been definitively linked to any specific human diseases.
STLV-1 infection is typically asymptomatic in both monkeys and humans, but it can cause a range of clinical manifestations in some individuals, including lymphadenopathy, hepatitis, and neurological symptoms. The virus is primarily transmitted through contact with infected bodily fluids, such as blood, breast milk, and semen.
Research on STLV-1 is important for understanding the evolution and epidemiology of retroviruses, as well as for developing strategies to prevent transmission and manage related diseases in both humans and non-human primates.
In a medical or scientific context, "Primates" is a biological order that includes various species of mammals, such as humans, apes, monkeys, and prosimians (like lemurs and lorises). This group is characterized by several distinct features, including:
1. A forward-facing eye position, which provides stereoscopic vision and depth perception.
2. Nails instead of claws on most digits, except for the big toe in some species.
3. A rotating shoulder joint that allows for a wide range of motion in the arms.
4. A complex brain with a well-developed cortex, which is associated with higher cognitive functions like problem-solving and learning.
5. Social structures and behaviors, such as living in groups and exhibiting various forms of communication.
Understanding primates is essential for medical and biological research since many human traits, diseases, and behaviors have their origins within this group.
Simian Acquired Immunodeficiency Syndrome (SAIDS) is not recognized as a medical condition in humans. However, it is a disease that affects non-human primates like African green monkeys and sooty mangabeys. SAIDS is caused by the Simian Immunodeficiency Virus (SIV), which is similar to the Human Immunodeficiency Virus (HIV) that leads to Acquired Immunodeficiency Syndrome (AIDS) in humans.
In non-human primates, SIV infection can lead to a severe immunodeficiency state, characterized by the destruction of CD4+ T cells and impaired immune function, making the host susceptible to various opportunistic infections and cancers. However, it is important to note that most non-human primates infected with SIV do not develop SAIDS spontaneously, unlike humans who acquire HIV infection.
In summary, Simian Acquired Immunodeficiency Syndrome (SAIDS) is a disease affecting non-human primates due to Simian Immunodeficiency Virus (SIV) infection, characterized by immunodeficiency and susceptibility to opportunistic infections and cancers. It should not be confused with Human Immunodeficiency Virus Infection and Acquired Immunodeficiency Syndrome (HIV/AIDS) in humans.
Dogu'a Tembien
Grivet
Vervet monkey
Marburg virus disease
Penis
Concealed ovulation
Michael Owren
Sex differences in human physiology
Kéran National Park
Sexism in the technology industry
Crowned eagle
Verreaux's eagle-owl
Hepatocystis
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Common patas monkey
Vervet Monkey Foundation
1967 Marburg virus outbreak
Aedes africanus
Heterologous expression
Chlorocebus
Guenon
List of human disease case fatality rates
Niumi National Park
Fauna of Barbados
Tigray Region
Columbus Zoo and Aquarium
Control of fire by early humans
List of MeSH codes (B01)
Primate Rescue Center
Entopolypoides
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Monkey3
- The CV-1 cell line was derived from the kidney of an adult male African green monkey (CERCOPITHECUS AETHIOPS). (lookformedical.com)
- The application is freeware.Hepatic hemangioendothelioma in an African green monkey (Cercopithecus aethiops) after injection of phosphate-buffered solution. (fitenvitaalfriesland.nl)
- A black and white monkey (Cercopithecus aethiops) died within 4 hours of injection of 30 ml of phosphate-buffered solution (PBS) in the left thigh. (fitenvitaalfriesland.nl)
Response1
- Alexander, G. and Hines, M. (2002) 'Sex differences in response to children's toys in nonhuman primates (Cercopithecus aethiops sabaeus)', Evolution and Human Behavior, 23(6), pp. 467-479. (kent.ac.uk)
Vervet6
- Cellular responses of vervet monkeys (Cercopithecus aethiops) experimentally infected with Leishmania major. (nih.gov)
- Malnutrition and susceptibility to infection with Vibrio cholerae in vervet monkeys (Cercopithecus aethiops). (nih.gov)
- Pleural Mesocestoides and cardiac shock in an obese vervet monkey (Cercopithecus aethiops). (ajtmh.org)
- Nonhuman primates such as vervet/African green monkeys (Chlorocebus aethiops sabaeus) are important models of human neurocognitive aging, yet the trajectory of dual decline has not been characterized. (mssm.edu)
- With its greyish coat, black face in a frame of white hair and bright, beady eyes, the vervet monkey (cercopithecus aethiops) has the typical impish appearance associated with moneys. (lifetimecreations.com)
- Fortunately natural prey in the form of Rock Hyrax (Procavia capensis) aka dassie, makes up at least 97% of the MF eagle pairs' prey species, with Helmeted Guineafowl (Numida meleagris), Jameson's Red Rock Rabbit (Pronolagus randensis), Scrub Hare (Lepus saxatilis), Francolin spp, Mongoose spp and occasional Vervet monkey (Cercopithecus aethiops) making up the deficit. (thebattyeagleman.com)
Chlorocebus1
- Therefore, results strongly suggest ABDALA does not cause toxic effects or damage in the organs of Chlorocebus aethiops sabaeus monkeys, indicating that it is a promising and safe novel vaccine to prevent SARS-CoV-2 infection in humans and the progression of COVID-19 to severe forms. (peertechzpublications.org)
Monkeys5
- The outbreak was associated with laboratory work using African green monkeys (Cercopithecus aethiops) imported from Uganda. (who.int)
- 10. Multiple dose toxicity study of the humanized anti-epidermal growth factor receptor monoclonal antibody h-R3 intravenously administered to Cercopithecus aethiops sabaeus monkeys. (nih.gov)
- The outbreak was linked to laboratory work using African green monkeys ( Cercopithecus aethiops ) brought in from Uganda. (africanhealthreport.com)
- The flare-up was related to research center work utilizing African green monkeys (Cercopithecus aethiops) imported from Uganda. (gogomagazine.in)
- The aim of the paper was to describe the safety profile of the ABDALA vaccine in monkeys (Cholorocebus aethiops sabaeus). (peertechzpublications.org)
PYGERYTHRUS1
- A species of CERCOPITHECUS containing three subspecies: C. tantalus, C. pygerythrus, and C. sabeus. (nih.gov)
Primates1
- Description de la cinquieme espece de Cercopithifilaria de Primates en Afrique. (ac.ke)