Carteolol
Bupranolol
Instillation, Drug
Adrenergic beta-Antagonists
Propanolamines
Cellular and molecular remodeling in a heart failure model treated with the beta-blocker carteolol. (1/36)
Broad-breasted white turkey poults fed furazolidone developed dilated cardiomyopathy (DCM) characterized by ventricular dilatation, decreased ejection fraction, beta1-receptor density, sarcoplasmic reticulum (SR) Ca2+-ATPase, myofibrillar ATPase activity, and reduced metabolism markers. We investigated the effects of carteolol, a beta-adrenergic blocking agent, by administrating two different dosages (0.01 and 10.0 mg/kg) twice a day for 4 wk to control and DCM turkey poults. At completion of the study there was 59% mortality in the nontreated DCM group, 55% mortality in the group treated with the low dose of carteolol, and 22% mortality in the group treated with the high dose of carteolol. Both treated groups showed a significant decrease in left ventricle size and significant restoration of ejection fraction and left ventricular peak systolic pressure. Carteolol treatment increased beta-adrenergic receptor density, and the high carteolol dose restored SR Ca2+-ATPase and myofibrillar ATPase activities, along with creatine kinase, lactate dehydrogenase, aspartate transaminase, and ATP synthase activities, to normal. These results show that beta-blockade with carteolol improves survival, reverses contractile abnormalities, and induces cellular remodeling in this model of heart failure. (+info)Betaxolol, a beta(1)-adrenoceptor antagonist, reduces Na(+) influx into cortical synaptosomes by direct interaction with Na(+) channels: comparison with other beta-adrenoceptor antagonists. (2/36)
Betaxolol, a beta(1)-adrenoceptor antagonist used for the treatment of glaucoma, is known to be neuroprotective in paradigms of ischaemia/excitotoxicity. In this study, we examined whether betaxolol and other beta-adrenoceptor antagonists interact directly with neurotoxin binding to sites 1 and 2 of the voltage-sensitive sodium channel (Na(+) channel) in rat cerebrocortical synaptosomes. Betaxolol inhibited specific [(3)H]-batrachotoxinin-A 20-alpha-benzoate ([(3)H]-BTX-B) binding to neurotoxin site 2 in a concentration-dependent manner with an IC(50) value of 9.8 microM. Comparison of all the beta-adrenoceptor antagonists tested revealed a potency order of propranolol>betaxolol approximately levobetaxolol>levobunolol approximately carteolol>/=timolol>atenolol. None of the drugs caused a significant inhibition of [(3)H]-saxitoxin binding to neurotoxin receptor site 1, even at concentrations as high as 250 microM. Saturation experiments showed that betaxolol increased the K(D) of [(3)H]-BTX-B binding but had no effect on the B(max). The association kinetics of [(3)H]-BTX-B were unaffected by betaxolol, but the drug significantly accelerated the dissociation rate of the radioligand. These findings argue for a competitive, indirect, allosteric mode of inhibition of [(3)H]-BTX-B binding by betaxolol. Betaxolol inhibited veratridine-stimulated Na(+) influx in rat cortical synaptosomes with an IC(50) value of 28. 3 microM. Carteolol, levobunolol, timolol and atenolol were significantly less effective than betaxolol at reducing veratridine-evoked Na(+) influx. The ability of betaxolol to interact with neurotoxin site 2 of the Na(+) channel and inhibit Na(+) influx may have a role in its neuroprotective action in paradigms of excitotoxicity/ischaemia and in its therapeutic effect in glaucoma. (+info)Topical ophthalmic beta blockers may cause release of histamine through cytotoxic effects on inflammatory cells. (3/36)
AIM: To evaluate the effects of beta blockers used in ophthalmology on the release of histamine from mixed cell preparations containing human leucocytes and basophils. METHODS: A mixed leucocyte and basophil preparation was obtained from venous blood of healthy non-atopic volunteers. Cell preparations were then incubated with betaxolol, metipranolol, timolol, or carteolol. After incubation for 1 hour the histamine content of the supernatant was analysed by automated fluorometric analysis. Cell viability was tested by measuring lactate dehydrogenase (LDH) concentrations. RESULTS: Betaxolol and metipranolol in concentrations between 10(-2) M and 10(-3) M liberated histamine from human blood cells in a dose dependent manner. Carteolol and timolol had no effect on histamine at these concentrations. At the same concentrations LDH was also detected in the supernatants of cell suspensions incubated with metipranolol or betaxolol. CONCLUSIONS: Betaxolol and metipranolol induce substantial histamine release from human leucocytes, probably as a result of their cytotoxic effect. (+info)Partial agonistic effects of carteolol on atypical beta-adrenoceptors in the guinea pig gastric fundus. (4/36)
The properties of the beta1-/beta2-adrenoceptor partial agonist carteolol were investigated in atypical beta-adrenoceptors on the guinea pig gastric fundus. Carteolol induced concentration-dependent relaxation in this tissue (pD2 = 5.55, intrinsic activity = 0.94). However, a combination of the selective beta1-adrenoceptor antagonist atenolol (100 microM) and the selective beta2-adrenoceptor antagonist butoxamine (100 microM) produced only small rightward shifts in the concentration-response curves of carteolol in the gastric fundus (pD2 = 4.91, intrinsic activity = 0.94). In the presence of both atenolol (100 microM) and butoxamine (100 microM), the non-selective beta1-, beta2- and beta3-adrenoceptor antagonist (+/-)-bupranolol (10-100 microM) caused a concentration-dependent right-ward shift of the concentration-response curves for carteolol in the guinea pig gastric fundus. Schild plot analyses of the effects of (+/-)-bupranolol against carteolol gave the pA2 value of 5.29 and the Schild slope was not significantly different from unity. Furthermore, carteolol (10 microM) weakly but significantly antagonized the relaxant responses to catecholamines ((-)-isoprenaline, (-)-noradrenaline and (-)-adrenaline), a selective beta3-adrenoceptor agonist BRL37344 ((R*,R*)-(+/-)-4-[2-[(2-(3-chlorophenyl)-2-hydroxyethyl)amino]propyl]phenoxy-acet ic acid sodium salt) and a non-conventional partial beta3-adrenoceptor agonist (+/-)-CGP12177A ([4-[3-[(1,1dimethylethyl)amino]-2-hydroxypropoxy]-1,3-dihydro-2H-benzimidazol-2- one] hydrochloride) in the guinea pig gastric fundus. These results suggest that the partial agonistic effects of carteolol are mediated by atypical beta-adrenoceptors in the guinea pig gastric fundus. (+info)Further evidence that (+/-)-carteolol-induced relaxation is mediated by beta2-adrenoceptors but not by beta3-adrenoceptors in the guinea pig taenia caecum. (5/36)
The properties of the beta1- and beta2-adrenoceptor partial agonist (+/-)-carteolol were investigated against the beta2- and beta3-adrenoceptors of the taenia caecum of the guinea pig. (--)-Isoprenaline and (+/-)-carteolol induced concentration-dependent relaxation in this tissue. The non-selective beta1- and beta2-adrenoceptor antagonist (+/-)-propranolol (10-100 nM), the selective beta2-adrenoceptor antagonist ICI 118,551 (10-100 nM) and the non-selective beta1-, beta2- and beta3-adrenoceptor antagonist (+/-)-bupranolol (10-100nM), caused a concentration-dependent rightward shift of the concentration-response curves for (--)-isoprenaline and (+/-)-carteolol. Schild regression plot analyses carried out for (+/-)-propranolol against (--)-isoprenaline and (+/-)-carteolol gave pA2 values of 8.35 and 8.24, respectively. Schild plot analyses of ICI 118,551 against (--)-isoprenaline and (+/-)-carteolol gave pA2 values of 8.47 and 8.41, respectively. Schild plot analyses of (+/-)-bupranolol against (--)-isoprenaline and (+/-)-carteolol gave pA2 values of 8.47 and 8.53, respectively. Slopes of the Schild plots were not significantly different from unity. These results suggest that the relaxant effects of (+/-)-carteolol in the guinea pig taenia caecum are mediated by beta2-adrenoceptors but not by beta3-adrenoceptors. (+info)Ocular hypotensive efficacy and safety of once daily carteolol alginate. (6/36)
BACKGROUND/AIM: Carteolol is a beta adrenoceptor antagonist used topically to reduce intraocular pressure, typically twice daily. In an effort to provide a once daily dosing regimen, carteolol was formulated with 1% alginic acid. The objective of this study was to evaluate the efficacy and safety of carteolol alginate solution in comparison with standard carteolol solution. METHODS: This was a double masked, parallel group, multicentre study. Patients with ocular hypertension or open angle glaucoma (n=235) were randomly assigned to receive either carteolol alginate once daily [corrected] or standard carteolol solution, twice daily. The masking was maintained through the use of a vehicle in the evening for the alginate group. Patients were evaluated at baseline, 15, 60, and 120 days. RESULTS: At 0900 (presumed trough) on day 60, mean reductions in intraocular pressure (IOP) from baseline were 6.09 (SD 2.97) and 6.09 (3.18) mm Hg for the standard carteolol and alginate, respectively. At 1100 (presumed peak), mean reductions were 6.51 (2.53) and 6.47 (2.76) mm Hg, respectively. Results were similar at other times (day 15 and day 120). The most common side effect was transient stinging on instillation of drops, which did not differ significantly between groups. There were no differences of note in other ocular or systemic signs or symptoms. CONCLUSION: The new alginate formulation of carteolol 2% given once daily was as effective as standard carteolol 2% given twice daily with no meaningful differences regarding safety. (+info)A 7 year prospective comparative study of three topical beta blockers in the management of primary open angle glaucoma. (7/36)
AIM: To determine the long term efficacy of monotherapy with topically applied beta blocking agents and to determine whether selective beta blockers were able to preserve the visual field more effectively than non-selective agents. METHOD: A prospective randomised, open, comparative study of three topically applied beta blockers-timolol, betaxolol, and carteolol-was carried out on 153 patients (280 eyes) with newly diagnosed open angle glaucoma. Those patients who were not withdrawn were followed by the same observers for a minimum of 2 years and a maximum of 7 years, with clinical observations, Goldmann tonometry and 24.2 Humphrey visual field analysis. RESULTS: All three drugs lowered the IOP significantly from untreated levels but betaxolol took up to 12 months in some instances to reach the maximum pressure reduction. After 7 years only 43% of the eyes begun on timolol, 34% of those started on carteolol, and 29% of those on betaxolol were still being treated with these medications alone. Visual fields were analysed throughout the trial by CPSD and MD and at the end by linear regression analysis (PROGRESSOR). The visual fields remained the same without apparent improvement or deterioration throughout the period of follow up. Eight patients (11 eyes) were withdrawn because of continuing field loss in spite of reduction in IOP (six using carteolol and five using betaxolol). CONCLUSIONS: Analysis shows that less than half the eyes initially treated with topical beta blockers might be expected to still be being treated with their original medication after 5 years. The rest required either additional medication or trabeculectomy. There was no statistically significant improvement or deterioration in the visual fields over a 7 year period. On the evidence of this trial there are no particular advantages in using selective beta blockers. (+info)Mitochondrial activity and glutathione injury in apoptosis induced by unpreserved and preserved beta-blockers on Chang conjunctival cells. (8/36)
PURPOSE: Quaternary ammonium ions have been demonstrated to induce apoptosis correlated with superoxide anion production in vitro. The purpose of this study was to further investigate the mechanisms of benzalkonium chloride (BAC), unpreserved and preserved beta-blocker eye-drops-induced programmed cell death, with special attention to the roles of mitochondrial transmembrane potential and intracellular reduced glutathione. METHODS: Chang conjunctival cells were incubated with different concentrations of unpreserved or preserved timolol (0.1%, 0.25%, and 0.4%), or carteolol (1% and 2%), or BAC (0.0001% to 0.01%) for 15 minutes, or for 15 minutes with a 24-hour recovery period in normal medium. Cellular viability (neutral red test), mitochondrial activity (rhodamine 123 test), intracellular reduced glutathione (monochlorobimane test), DNA condensation (Hoechst 33342 test), and reactive oxygen species (ROS) production (dichlorofluorescein diacetate and hydroethidine tests) were evaluated using microplate cold-light cytofluorometry. RESULTS: A significant, concentration-dependent decrease in cellular viability was found with preserved beta-blockers and with BAC alone, whereas unpreserved preparations did not show any toxicity. Only preserved beta-blockers induced chromatin condensation associated with an alteration of mitochondrial activity and a decrease of glutathione, suggesting an apoptotic phenomenon. BAC increased glutathione after 15 minutes, whereas a decrease was observed after a recovery period. ROS production was found with preserved formulations at significantly higher levels than those observed with unpreserved drugs. CONCLUSIONS: This in vitro study demonstrates that oxidative stress, evidenced by enhanced ROS production and mitochondrial injury rather than by cellular glutathione depletion, is a mechanism involved in apoptosis induced by preservative-containing eye-drops. (+info)
Carteolol Hydrochloride (Carteolol): Side Effects, Interactions, Warning, Dosage & Uses
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Glaucoma, open angle starts daily observation of multiple persons, in fact thats because of carteolol
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Carteolol
... is a non-selective beta blocker used to treat glaucoma. It has been found to act as a serotonin 5-HT1A and 5-HT1B ... Trinquand C, Romanet J, Nordmann J, Allaire C (2003). "[Efficacy and safety of long-acting carteolol 1% once daily. A double- ... Kuwahara K, Oizumi N, Fujisawa S, Tanito M, Ohira A (2005). "Carteolol hydrochloride protects human corneal epithelial cells ... El-Kamel A, Al-Dosari H, Al-Jenoobi F (2006). "Environmentally responsive ophthalmic gel formulation of carteolol hydrochloride ...
Beta blocker
"CARTEOLOL". pubchem.ncbi.nlm.nih.gov. U.S. National Library of Medicine. Archived from the original on October 18, 2017. ... Propranolol Bucindolol (has additional α1-blocking activity) Carteolol Carvedilol (has additional α1-blocking activity) ... carteolol, levobunolol, timolol, metipranolol Agents specifically labeled for myocardial infarction Atenolol, metoprolol ( ...
Hydroxycarteolol
Igarashi H, Katsuta Y, Sawa K, Nakazato Y, Kawasaki T (Apr 1990). "A comparison of the opacifying effects of carteolol.HCl and ... Jasper JR, Michel MC, Insel PA (1990). "The beta-adrenoceptor antagonist carteolol and its metabolite 8-hydroxycarteolol have ... Hydroxycarteolol is a beta blocker and metabolite of carteolol. ...
Penbutolol
ISBN 9780071826419 Frishman WH, Covey S (1990). "Penbutolol and carteolol: two new beta-adrenergic blockers with partial ...
ATC code S01
... combinations S01ED55 Carteolol, combinations S01EE01 Latanoprost S01EE02 Unoprostone S01EE03 Bimatoprost S01EE04 Travoprost ... combinations S01ED01 Timolol S01ED02 Betaxolol S01ED03 Levobunolol S01ED04 Metipranolol S01ED05 Carteolol S01ED06 Befunolol ...
List of MeSH codes (D03)
... carteolol MeSH D03.438.810.835.322 - fluoroquinolones MeSH D03.438.810.835.322.186 - ciprofloxacin MeSH D03.438.810.835.322.186 ...
List of adrenergic drugs
Bufetolol Bufuralol Bunitrolol Bunolol Bupranolol Butaxamine Butidrine Butofilolol Capsinolol Carazolol Carpindolol Carteolol ...
Sympatholytic
Beta blockers Non-selective agents Alprenolol Bucindolol Carteolol Carvedilol (has additional α-blocking activity) Labetalol ( ...
Sonophoresis
... such as atenolol and carteolol, increased by 2.6 and 2.8-fold respectively. Topical gene therapy is another area for ...
List of MeSH codes (D02)
... carteolol MeSH D02.033.100.624.240 - celiprolol MeSH D02.033.100.624.302 - ephedrine MeSH D02.033.100.624.380 - histidinol MeSH ... carteolol MeSH D02.033.755.624.240 - celiprolol MeSH D02.033.755.624.302 - ephedrine MeSH D02.033.755.624.380 - histidinol MeSH ... carteolol MeSH D02.092.063.624.698.268 - celiprolol MeSH D02.092.063.624.698.512 - levobunolol MeSH D02.092.063.624.698.542 - ...
ATC code C07
C07AA03 Pindolol C07AA05 Propranolol C07AA06 Timolol C07AA07 Sotalol C07AA12 Nadolol C07AA14 Mepindolol C07AA15 Carteolol ...
List of drugs: C-Ca
Carteolol carteolol (INN) Cartia XT Carticel Cartrol carubicin (INN) carumonam (INN) carvedilol (INN) carvotroline (INN) ...
Antihypertensive drug
Beta blockers acebutolol atenolol bisoprolol betaxolol carteolol carvedilol labetalol metoprolol nadolol nebivolol oxprenolol ...
Browsing by Subject "Carteolol"
Ocular Hypertension Medication: Antiglaucoma, Prostaglandin Agonists, Antiglaucoma, Beta-Blockers, Antiglaucoma, Carbonic...
Carteolol
... reduces intraocular pressure with little or no effect on pupil size or accommodation in contrast to the miosis which ... If you have an overactive thyroid and stop taking carteolol (ophthalmic) all of a sudden, it may get worse and could be life- ... Carteolol, when applied topically to the eye, has the action of reducing elevated, as well as normal, intraocular pressure, ... The primary mechanism of the ocular hypotensive action of carteolol in reducing intraocular pressure is most likely a decrease ...
Carteolol (ophthalmic) vs Pentoxifylline
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DeCS
Carteolol Hydrochloride. Carteolol Monohydrochloride. Hydrochloride, Carteolol. Monohydrochloride, Carteolol. OPC 1085. OPC- ... Carteolol - Preferred Concept UI. M0003563. Scope note. A beta-adrenergic antagonist used as an anti-arrhythmia agent, an anti- ... carteolol. Scope note:. Antagonista beta-adrenérgico que se utiliza como agente antiarrítmico, antianginoso y antihipertensor y ... Carteolol Hydrochloride - Narrower Concept UI. M0003565. Preferred term. Carteolol Hydrochloride Entry term(s). Carteolol ...
Code System Concept
Analyses
7-amino-clonazepam. 7-amino-flunitrazepam. Acepromethazine. Acetyl salicylic acid (aspirin). Mefenamic acid. Vvalproic acid. Alimemazine. Alprazolam. Amisulpride. Amitriptyline. Atropine Bromazepam. Butabarbital. Carbamazepine. Chlorpheniramine. Citalopram. Clobazam. Clonazepam. Clonidine. Cyamemazine. Diazepam. Diclofenac. Digitoxin. Digoxin. Diphenhydramine. Domperidone. Dosulepin. Dothiepin. Doxylamine. Flunitrazepam. Fluoxetine ...
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Betaxolol1
- Other topical beta-blockers include levobunolol, betaxolol, metipranolol and carteolol. (poultrydvm.com)
Ophthalmic5
- Tell all of your health care providers that you take carteolol (ophthalmic). (rpsi.ir)
- If you have an overactive thyroid and stop taking carteolol ( ophthalmic ) all of a sudden, it may get worse and could be life-threatening. (rpsi.ir)
- Epinephrine may not work as well while you are taking carteolol (ophthalmic). (rpsi.ir)
- You will need to talk about the benefits and risks of using carteolol (ophthalmic) while you are pregnant. (rpsi.ir)
- Use carteolol (ophthalmic) at the same time of day. (rpsi.ir)
Intraocular4
- The primary mechanism of the ocular hypotensive action of carteolol in reducing intraocular pressure is most likely a decrease in aqueous humor production. (rpsi.ir)
- Carteolol, when applied topically to the eye, has the action of reducing elevated, as well as normal, intraocular pressure, whether or not accompanied by glaucoma. (rpsi.ir)
- Carteolol reduces intraocular pressure with little or no effect on pupil size or accommodation in contrast to the miosis which cholinergic agents are known to produce. (rpsi.ir)
- The intraocular pressure was 24 mm Hg right eye (with carteolol hydrochloride eye drops (2% Mikelan, Otsuka Pharmaceutical Co, Tokyo) and isopropyl unoprostone eye drops (Rescula, Fujisawa Pharmaceutical Co, Osaka)), as well as 750 mg a day of an orally administered carbonate dehydratase inhibitor, acetazolamide, and 18 mm Hg left eye. (bmj.com)
Hydrochloride20
- Carteolol Hydrochloride Ophthalmic Solution USP, 1% is a nonselective beta-adrenoceptor blocking agent for ophthalmic use. (nih.gov)
- The chemical name for carteolol hydrochloride is (±)-5-[3-[(1,1-dimethylethyl) amino]-2-hydroxypropoxy]-3,4-dihydro-2(1H)-quinolinone monohydrochloride. (nih.gov)
- Carteolol Hydrochloride reduces normal and elevated intraocular pressure (IOP) whether or not accompanied by glaucoma. (nih.gov)
- Carteolol Hydrochloride Ophthalmic Solution 1% has been shown to be effective in lowering intraocular pressure and may be used in patients with chronic open-angle glaucoma and intraocular hypertension. (nih.gov)
- At the first sign or symptom of cardiac failure, Carteolol Hydrochloride should be discontinued. (nih.gov)
- In patients with non-allergic bronchospasm or with a history of non-allergic bronchospasm (e.g., chronic bronchitis, emphysema), Carteolol Hydrochloride Ophthalmic Solution should be administered with caution since it may block bronchodilation produced by endogenous and exogenous catecholamine stimulation of beta 2 receptors. (nih.gov)
- Pharmaceutical chemical and analytical data for the pharmaceutically-active substance carteolol hydrochloride]. (nih.gov)
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- When administered concomitantly with ophthalmic carteolol hydrochloride solution, may cause additive effects and toxicity. (medicscientist.com)
- Neither MDS nor its employees assume liability for erroneous Carteolol Hydrochloride (Carteolol)- FDA of website content. (payforyou.top)
- Additional Frequently Asked Questions Carteolol Hydrochloride (Carteolol)- Multum do I know if I am eligible to participate. (ads4kings.xyz)
- Carteolol Hydrochloride (Carteolol)- FDA ColorWhen blemishes are visible, applying color correctors under foundation can be effective:Green neutralizers mask red lesions. (xtxzb.top)
- If you are an International or non-UK European student, you can find out more about the country specific qualifications we accept on our Carteolol Hydrochloride (Carteolol)- FDA qualifications page. (prettyfuckingawesome.xyz)
Excretion2
- Because its excretion into breastmilk is probably extensive, other beta-adrenergic blocking drugs are preferred to oral carteolol while breastfeeding a neonate. (nih.gov)
- With 60% protein binding, 15% renal excretion and a moderately long half-life, carteolol presents a relatively high risk for accumulation in infants, especially neonates. (nih.gov)
Reduces1
- Carteolol therefore reduces the inflow of aqueous humour into the eyeball, which decreases the pressure within the eye. (netdoctor.co.uk)
Drops1
- Teoptic eye drops contain the active ingredient carteolol, which is a type of medicine called a beta-blocker. (netdoctor.co.uk)
Found2
- Carteolol blocks beta-receptors that are found on the ciliary body. (netdoctor.co.uk)
- ACE2 is definitely homologous Carteolol HCl to the firstly found out ACE1, and both enzymes are part of the renin-angiotensin system (RAS), which has a important part in regulating blood pressure, preserving liquid and electrolyte homeostasis because of its potent vasoconstrictor/vasodepressor actions [22]. (a-443654.com)
Form1
- RAS can be an enzymatic cascade you start with the cleavage of angiotensinogen by renin to create angiotensin (Ang) I. This peptide is definitely then further metabolized by ACE1, which removes two amino acids in the Carteolol HCl C-terminal end to form the Carteolol HCl potent vasopressor peptide Ang II [23]. (a-443654.com)
Normal1
- 7 ] There are no reports on the effects of beta-blockade or carteolol use during normal lactation. (nih.gov)
Agent1
- Carteolol is a nonselective beta-adrenergic blocking agent with associated intrinsic sympathomimetic activity and without significant membrane-stabilizing activity. (nih.gov)
Effects1
- Sufficient carteolol may be absorbed from the eye into the bloodstream to cause side effects on other parts of the body, or to react with other medicines being taken by mouth, injection or suppository. (netdoctor.co.uk)