Enlargement of the HEART, usually indicated by a cardiothoracic ratio above 0.50. Heart enlargement may involve the right, the left, or both HEART VENTRICLES or HEART ATRIA. Cardiomegaly is a nonspecific symptom seen in patients with chronic systolic heart failure (HEART FAILURE) or several forms of CARDIOMYOPATHIES.
Abnormal accumulation of serous fluid in two or more fetal compartments, such as SKIN; PLEURA; PERICARDIUM; PLACENTA; PERITONEUM; AMNIOTIC FLUID. General fetal EDEMA may be of non-immunologic origin, or of immunologic origin as in the case of ERYTHROBLASTOSIS FETALIS.
Heart enlargement and other remodeling in cardiac morphology and electrical circutry found in individuals who participate in intense repeated exercises.
An island in the Greater Antilles in the West Indies. Its capital is Kingston. It was discovered in 1494 by Columbus and was a Spanish colony 1509-1655 until captured by the English. Its flourishing slave trade was abolished in the 19th century. It was a British colony 1655-1958 and a territory of the West Indies Federation 1958-62. It achieved full independence in 1962. The name is from the Arawak Xaymaca, rich in springs or land of springs. (From Webster's New Geographical Dictionary, 1988, p564 & Room, Brewer's Dictionary of Names, 1992, p267)
X-ray visualization of the chest and organs of the thoracic cavity. It is not restricted to visualization of the lungs.
A group of diseases in which the dominant feature is the involvement of the CARDIAC MUSCLE itself. Cardiomyopathies are classified according to their predominant pathophysiological features (DILATED CARDIOMYOPATHY; HYPERTROPHIC CARDIOMYOPATHY; RESTRICTIVE CARDIOMYOPATHY) or their etiological/pathological factors (CARDIOMYOPATHY, ALCOHOLIC; ENDOCARDIAL FIBROELASTOSIS).
An abnormal direct communication between an artery and a vein without passing through the CAPILLARIES. An A-V fistula usually leads to the formation of a dilated sac-like connection, arteriovenous aneurysm. The locations and size of the shunts determine the degree of effects on the cardiovascular functions such as BLOOD PRESSURE and HEART RATE.
Fluid accumulation within the PERICARDIUM. Serous effusions are associated with pericardial diseases. Hemopericardium is associated with trauma. Lipid-containing effusion (chylopericardium) results from leakage of THORACIC DUCT. Severe cases can lead to CARDIAC TAMPONADE.
A disease of the CARDIAC MUSCLE developed subsequent to the initial protozoan infection by TRYPANOSOMA CRUZI. After infection, less than 10% develop acute illness such as MYOCARDITIS (mostly in children). The disease then enters a latent phase without clinical symptoms until about 20 years later. Myocardial symptoms of advanced CHAGAS DISEASE include conduction defects (HEART BLOCK) and CARDIOMEGALY.
Pathophysiological conditions of the FETUS in the UTERUS. Some fetal diseases may be treated with FETAL THERAPIES.
The visualization of tissues during pregnancy through recording of the echoes of ultrasonic waves directed into the body. The procedure may be applied with reference to the mother or the fetus and with reference to organs or the detection of maternal or fetal disease.
Tumors in any part of the heart. They include primary cardiac tumors and metastatic tumors to the heart. Their interference with normal cardiac functions can cause a wide variety of symptoms including HEART FAILURE; CARDIAC ARRHYTHMIAS; or EMBOLISM.
A heterogeneous condition in which the heart is unable to pump out sufficient blood to meet the metabolic need of the body. Heart failure can be caused by structural defects, functional abnormalities (VENTRICULAR DYSFUNCTION), or a sudden overload beyond its capacity. Chronic heart failure is more common than acute heart failure which results from sudden insult to cardiac function, such as MYOCARDIAL INFARCTION.
Death resulting from the presence of a disease in an individual, as shown by a single case report or a limited number of patients. This should be differentiated from DEATH, the physiological cessation of life and from MORTALITY, an epidemiological or statistical concept.
Recording of the moment-to-moment electromotive forces of the HEART as projected onto various sites on the body's surface, delineated as a scalar function of time. The recording is monitored by a tracing on slow moving chart paper or by observing it on a cardioscope, which is a CATHODE RAY TUBE DISPLAY.
Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues. The standard approach is transthoracic.
Developmental abnormalities involving structures of the heart. These defects are present at birth but may be discovered later in life.
The muscle tissue of the HEART. It is composed of striated, involuntary muscle cells (MYOCYTES, CARDIAC) connected to form the contractile pump to generate blood flow.
The status during which female mammals carry their developing young (EMBRYOS or FETUSES) in utero before birth, beginning from FERTILIZATION to BIRTH.
Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.
An infant during the first month after birth.
Persistently high systemic arterial BLOOD PRESSURE. Based on multiple readings (BLOOD PRESSURE DETERMINATION), hypertension is currently defined as when SYSTOLIC PRESSURE is consistently greater than 140 mm Hg or when DIASTOLIC PRESSURE is consistently 90 mm Hg or more.
A prediction of the probable outcome of a disease based on a individual's condition and the usual course of the disease as seen in similar situations.
Works containing information articles on subjects in every field of knowledge, usually arranged in alphabetical order, or a similar work limited to a special field or subject. (From The ALA Glossary of Library and Information Science, 1983)
Pathological processes of CORONARY ARTERIES that may derive from a congenital abnormality, atherosclerotic, or non-atherosclerotic cause.
The hollow, muscular organ that maintains the circulation of the blood.

Gender-related differences in myocyte remodeling in progression to heart failure. (1/3277)

Gender-related differences responsible for the better prognosis of females with heart failure have not been clearly established. To address this issue, we investigated potential gender-related differences in myocyte remodeling in spontaneously hypertensive heart failure rats. Echocardiograms and myocyte growth were compared between males and females at compensated (2, 4, and 6 months) and decompensated (18 months in males and 24 months in females) stages of cardiac hypertrophy. Although left ventricular diastolic dimensions did not differ significantly between failing male and female rats, fractional shortening declined significantly only in failing males. Myocyte cross-sectional area did not change after 4 months of age in both genders, which is likely to be responsible for the absence of a change in left ventricular wall thickness during the progression to heart failure. Myocyte volume and cross-sectional area were significantly larger in males than females at 2, 4, and 6 months of age, although there were no significant differences at the failing stage. Reduced adaptive hypertrophic reserve was observed in males, which is likely to contribute to the higher morbidity and mortality of males with chronic heart failure.  (+info)

Cardiomegaly in the juvenile visceral steatosis (JVS) mouse is reduced with acute elevation of heart short-chain acyl-carnitine level after L-carnitine injection. (2/3277)

The long-term administration of L-carnitine was very effective in preventing cardiomegaly in juvenile visceral steatosis (JVS) mice, which was confirmed by heart weight as well as the lipid contents in heart tissue. After i.p. injection of L-carnitine, the concentration of free carnitine in heart remained constant, although serum free carnitine level increased up to 80-fold. On the other hand, a significant increase in short-chain acyl-carnitine level in heart was observed. These results suggest that increased levels of short-chain acyl-carnitine, not free carnitine, might be a key compound in the protective effect of L-carnitine administration in JVS mice.  (+info)

Cellular mechanisms of altered contractility in the hypertrophied heart: big hearts, big sparks. (3/3277)

To investigate the cellular mechanisms for altered Ca2+ homeostasis and contractility in cardiac hypertrophy, we measured whole-cell L-type Ca2+ currents (ICa,L), whole-cell Ca2+ transients ([Ca2+]i), and Ca2+ sparks in ventricular cells from 6-month-old spontaneously hypertensive rats (SHRs) and from age- and sex-matched Wistar-Kyoto and Sprague-Dawley control rats. By echocardiography, SHR hearts had cardiac hypertrophy and enhanced contractility (increased fractional shortening) and no signs of heart failure. SHR cells had a voltage-dependent increase in peak [Ca2+]i amplitude (at 0 mV, 1330+/-62 nmol/L [SHRs] versus 836+/-48 nmol/L [controls], P<0.05) that was not associated with changes in ICa,L density or kinetics, resting [Ca2+]i, or Ca2+ content of the sarcoplasmic reticulum (SR). SHR cells had increased time of relaxation. Ca2+ sparks from SHR cells had larger average amplitudes (173+/-192 nmol/L [SHRs] versus 109+/-64 nmol/L [control]; P<0.05), which was due to redistribution of Ca2+ sparks to a larger amplitude population. This change in Ca2+ spark amplitude distribution was not associated with any change in the density of ryanodine receptors, calsequestrin, junctin, triadin 1, Ca2+-ATPase, or phospholamban. Therefore, SHRs with cardiac hypertrophy have increased contractility, [Ca2+]i amplitude, time to relaxation, and average Ca2+ spark amplitude ("big sparks"). Importantly, big sparks occurred without alteration in the trigger for SR Ca2+ release (ICa,L), SR Ca2+ content, or the expression of several SR Ca2+-cycling proteins. Thus, cardiac hypertrophy in SHRs is linked with an alteration in the coupling of Ca2+ entry through L-type Ca2+ channels and the release of Ca2+ from the SR, leading to big sparks and enhanced contractility. Alterations in the microdomain between L-type Ca2+ channels and SR Ca2+ release channels may underlie the changes in Ca2+ homeostasis observed in cardiac hypertrophy. Modulation of SR Ca2+ release may provide a new therapeutic strategy for cardiac hypertrophy and for its progression to heart failure and sudden death.  (+info)

Rho family small G proteins play critical roles in mechanical stress-induced hypertrophic responses in cardiac myocytes. (4/3277)

-Mechanical stress induces a variety of hypertrophic responses, such as activation of protein kinases, reprogramming of gene expression, and an increase in protein synthesis. In the present study, to elucidate how mechanical stress induces such events, we examined the role of Rho family small GTP-binding proteins (G proteins) in mechanical stress-induced cardiac hypertrophy. Treatment of neonatal rat cardiomyocytes with the C3 exoenzyme, which abrogates Rho functions, suppressed stretch-induced activation of extracellular signal-regulated protein kinases (ERKs). Overexpression of the Rho GDP dissociation inhibitor (Rho-GDI), dominant-negative mutants of RhoA (DNRhoA), or DNRac1 significantly inhibited stretch-induced activation of transfected ERK2. Overexpression of constitutively active mutants of RhoA slightly activated ERK2 in cardiac myocytes. Overexpression of C-terminal Src kinase, which inhibits functions of the Src family of tyrosine kinases, or overexpression of DNRas had no effect on stretch-induced activation of transfected ERK2. The promoter activity of skeletal alpha-actin and c-fos genes was increased by stretch, and these increases were completely inhibited by either cotransfection of Rho-GDI or pretreatment with C3 exoenzyme. Mechanical stretch increased phenylalanine incorporation into cardiac myocytes by approximately 1.5-fold compared with control, and this increase was also significantly suppressed by pretreatment with C3 exoenzyme. Overexpression of Rho-GDI or DNRhoA did not affect angiotensin II-induced activation of ERK. ERKs were activated by culture media conditioned by stretch of cardiomyocytes without any treatment, but not of cardiomyocytes with pretreatment by C3 exoenzyme. These results suggest that the Rho family of small G proteins plays critical roles in mechanical stress-induced hypertrophic responses.  (+info)

TAFII250, Egr-1, and D-type cyclin expression in mice and neonatal rat cardiomyocytes treated with doxorubicin. (5/3277)

Differential display identified that gene fragment HA220 homologous to the transcriptional activator factor II 250 (TAFII250) gene, or CCG1, was increased in hypertrophied rodent heart. To determine whether TAFII250 gene expression is modified after cardiac damage, we measured TAFII250 expression in vivo in mouse hearts after injection of the cardiotoxic agent doxorubicin (DXR) and in vitro in DXR-treated isolated rat neonatal cardiomyocytes. In vivo atrial natriuretic factor (ANF), beta-myosin heavy chain (beta-MHC), Egr-1, and TAFII250 expression increased with dose and time after a single DXR injection, but only ANF and beta-MHC expression were increased after multiple injections. After DXR treatment of neonatal cardiomyocytes we found decreased ANF, alpha-MHC, Egr-1, and TAFII250 expression. Expression of the TAFII250-regulated genes, the D-type cyclins, was increased after a single injection in adult mice and was decreased in DXR-treated cardiomyocytes. Thus expression of Erg-1, TAFII250, and the D-type cyclins is modulated after cardiotoxic damage in adult and neonatal heart.  (+info)

Heart hypertrophy induced by levothyroxine aggravates ischemic lesions and reperfusion arrhythmias in rats. (6/3277)

AIM: To develop a cardiac hypertrophic model in rats. METHODS: Rats were i.p. levothyroxine 0.5 mg.kg-1.d-1 x 10 d. The action potentials of right papillary muscles were recorded by standard glass-microelectrode technique. The left coronary artery was ligated followed by reperfusion and the apparent infarcted zone (AIZ) was determined by tetracycline fluoresence, and the superoxide dismutase (SOD) activity and malondialdehyde (MDA) product in myocardium were also measured. RESULTS: In the rats treated by levothyroxine, the heart was hypertrophic and the action potential duration (APD) and effective refractory period (ERP) were prolonged, the APD20, APD50, APD90, and ERP were prolonged by 80%, 79%, 74%, and 68%, respectively. No changes in resting potential (RP), action potential amplitude (APA), and Vmax were produced. The incidence of heart arrest (8/8) and the risk of death (67 +/- 0) induced by ischemia-reperfusion in rats with hypertrophic heart was higher than those in normal rats (4/10 and 44 +/- 19, respectively). The AIZ was expanded markedly in hypertrophic heart, and attenuated by lidocaine and propranolol. CONCLUSION: Levothyroxine-induced heart hypertrophy is a suitable model for severe ischemia and arrhythmias in rats.  (+info)

A case of eosinophilic myocarditis complicated by Kimura's disease (eosinophilic hyperplastic lymphogranuloma) and erythroderma. (7/3277)

This report describes a patient with eosinophilic myocarditis complicated by Kimura's disease (eosinophilic hyperplastic lymphogranuloma) and erythroderma. A 50-year-old man presented with a complaint of precordial pain. However, the only abnormal finding on examinatioin was eosinophilia (1617 eosinophils/microl). Three years later, the patient developed chronic eczema, and was diagnosed with erythroderma posteczematosa. One year later, a tumor was detected in the right auricule, and a diagnosis of Kimura's disease was made, based on the biopsy findings. The patient developed progressive dyspnea 6 months later and was found to have cardiomegaly and a depressed left ventricular ejection fraction (17%). A diagnosis of eosinophilic myocarditis was made based on the results of a right ventricular endomyocardial biopsy. The eosinophilic myocarditis and erythrodrema were treated with steroids with improvement of both the eosinophilia and left ventricular function.  (+info)

Transcriptional activation of the glucose transporter GLUT1 in ventricular cardiac myocytes by hypertrophic agonists. (8/3277)

Myocardial hypertrophy is associated with increased basal glucose metabolism. Basal glucose transport into cardiac myocytes is mediated by the GLUT1 isoform of glucose transporters, whereas the GLUT4 isoform is responsible for regulatable glucose transport. Treatment of neonatal cardiac myocytes with the hypertrophic agonist 12-O-tetradecanoylphorbol-13-acetate or phenylephrine increased expression of Glut1 mRNA relative to Glut4 mRNA. To study the transcriptional regulation of GLUT1 expression, myocytes were transfected with luciferase reporter constructs under the control of the Glut1 promoter. Stimulation of the cells with 12-O-tetradecanoylphorbol-13-acetate or phenylephrine induced transcription from the Glut1 promoter, which was inhibited by cotransfection with the mitogen-activated protein kinase phosphatases CL100 and MKP-3. Cotransfection of the myocytes with constitutively active versions of Ras and MEK1 or an estrogen-inducible version of Raf1 also stimulated transcription from the Glut1 promoter. Hypertrophic induction of the Glut1 promoter was also partially sensitive to inhibition of the phosphatidylinositol 3-kinase pathway and was strongly inhibited by cotransfection with dominant-negative Ras. Thus, Ras activation and pathways downstream of Ras mediate induction of the Glut1 promoter during myocardial hypertrophy.  (+info)

Cardiomegaly is a medical term that refers to an enlarged heart. It can be caused by various conditions such as high blood pressure, heart valve problems, cardiomyopathy, or fluid accumulation around the heart (pericardial effusion). Cardiomegaly can be detected through imaging tests like chest X-rays or echocardiograms. Depending on the underlying cause, treatment options may include medications, lifestyle changes, or in some cases, surgery. It is important to consult with a healthcare professional for proper diagnosis and treatment.

Hydrops Fetalis is a serious condition characterized by the accumulation of excessive fluid in two or more fetal compartments, including the abdomen (ascites), around the heart (pericardial effusion), and/or within the lungs (pleural effusion). This accumulation can also affect the skin, causing it to become edematous. Hydrops Fetalis is often associated with various underlying causes, such as chromosomal abnormalities, congenital infections, genetic disorders, and structural defects that impair the fetus's ability to maintain fluid balance. In some cases, the cause may remain unknown. The prognosis for Hydrops Fetalis is generally poor, with a high mortality rate, although early detection and appropriate management can improve outcomes in certain situations.

Exercise-induced cardiomegaly is a medical condition characterized by an enlargement of the heart (cardiomegaly) that occurs during or immediately after physical exertion. This condition is typically seen in individuals with underlying heart diseases, such as hypertrophic cardiomyopathy or valvular heart disease. During exercise, the increased workload on the heart can cause the heart muscle to thicken and enlarge, leading to symptoms such as shortness of breath, chest pain, or palpitations. It is important to note that this condition is different from physiological cardiac remodeling, which is a normal response to regular exercise in healthy individuals. If you suspect that you have exercise-induced cardiomegaly, it is important to seek medical attention and evaluation by a healthcare professional.

I'm sorry for any confusion, but "Jamaica" is not a medical term. It is a country located in the Caribbean Sea, known for its beautiful beaches, vibrant culture, and as the birthplace of reggae music. If you have any questions about medical terms or concepts, I would be happy to help answer those!

Thoracic radiography is a type of diagnostic imaging that involves using X-rays to produce images of the chest, including the lungs, heart, bronchi, great vessels, and the bones of the spine and chest wall. It is a commonly used tool in the diagnosis and management of various respiratory, cardiovascular, and thoracic disorders such as pneumonia, lung cancer, heart failure, and rib fractures.

During the procedure, the patient is positioned between an X-ray machine and a cassette containing a film or digital detector. The X-ray beam is directed at the chest, and the resulting image is captured on the film or detector. The images produced can help identify any abnormalities in the structure or function of the organs within the chest.

Thoracic radiography may be performed as a routine screening test for certain conditions, such as lung cancer, or it may be ordered when a patient presents with symptoms suggestive of a respiratory or cardiovascular disorder. It is a safe and non-invasive procedure that can provide valuable information to help guide clinical decision making and improve patient outcomes.

Cardiomyopathies are a group of diseases that affect the heart muscle, leading to mechanical and/or electrical dysfunction. The American Heart Association (AHA) defines cardiomyopathies as "a heterogeneous group of diseases of the myocardium associated with mechanical and/or electrical dysfunction that usually (but not always) exhibit inappropriate ventricular hypertrophy or dilatation and frequently lead to heart failure."

There are several types of cardiomyopathies, including:

1. Dilated cardiomyopathy (DCM): This is the most common type of cardiomyopathy, characterized by an enlarged left ventricle and impaired systolic function, leading to heart failure.
2. Hypertrophic cardiomyopathy (HCM): In this type, there is abnormal thickening of the heart muscle, particularly in the septum between the two ventricles, which can obstruct blood flow and increase the risk of arrhythmias.
3. Restrictive cardiomyopathy (RCM): This is a rare form of cardiomyopathy characterized by stiffness of the heart muscle, impaired relaxation, and diastolic dysfunction, leading to reduced filling of the ventricles and heart failure.
4. Arrhythmogenic right ventricular cardiomyopathy (ARVC): In this type, there is replacement of the normal heart muscle with fatty or fibrous tissue, primarily affecting the right ventricle, which can lead to arrhythmias and sudden cardiac death.
5. Unclassified cardiomyopathies: These are conditions that do not fit into any of the above categories but still significantly affect the heart muscle and function.

Cardiomyopathies can be caused by genetic factors, acquired conditions (e.g., infections, toxins, or autoimmune disorders), or a combination of both. The diagnosis typically involves a comprehensive evaluation, including medical history, physical examination, electrocardiogram (ECG), echocardiography, cardiac magnetic resonance imaging (MRI), and sometimes genetic testing. Treatment depends on the type and severity of the condition but may include medications, lifestyle modifications, implantable devices, or even heart transplantation in severe cases.

An arteriovenous fistula is an abnormal connection or passageway between an artery and a vein. This connection causes blood to flow directly from the artery into the vein, bypassing the capillary network that would normally distribute the oxygen-rich blood to the surrounding tissues.

Arteriovenous fistulas can occur as a result of trauma, disease, or as a planned surgical procedure for patients who require hemodialysis, a treatment for advanced kidney failure. In hemodialysis, the arteriovenous fistula serves as a site for repeated access to the bloodstream, allowing for efficient removal of waste products and excess fluids.

The medical definition of an arteriovenous fistula is:

"An abnormal communication between an artery and a vein, usually created by surgical means for hemodialysis access or occurring as a result of trauma, congenital defects, or disease processes such as vasculitis or neoplasm."

Pericardial effusion is an abnormal accumulation of fluid in the pericardial space, which is the potential space between the two layers of the pericardium - the fibrous and serous layers. The pericardium is a sac that surrounds the heart to provide protection and lubrication for the heart's movement during each heartbeat. Normally, there is only a small amount of fluid (5-15 mL) in this space to ensure smooth motion of the heart. However, when an excessive amount of fluid accumulates, it can cause increased pressure on the heart, leading to various complications such as decreased cardiac output and even cardiac tamponade, a life-threatening condition that requires immediate medical attention.

Pericardial effusion may result from several causes, including infections (viral, bacterial, or fungal), inflammatory conditions (such as rheumatoid arthritis, lupus, or cancer), trauma, heart surgery, kidney failure, or iatrogenic causes. The symptoms of pericardial effusion can vary depending on the rate and amount of fluid accumulation. Slowly developing effusions may not cause any symptoms, while rapid accumulations can lead to chest pain, shortness of breath, cough, palpitations, or even hypotension (low blood pressure). Diagnosis is usually confirmed through imaging techniques such as echocardiography, CT scan, or MRI. Treatment depends on the underlying cause and severity of the effusion, ranging from close monitoring to drainage procedures or medications to address the root cause.

Chagas cardiomyopathy is a specific type of heart disease that is caused by infection with the parasite Trypanosoma cruzi, which is spread through the feces of infected triatomine bugs (also known as "kissing bugs"). The disease is named after Carlos Chagas, who discovered the parasite in 1909.

In Chagas cardiomyopathy, the infection can lead to inflammation of the heart muscle (myocarditis), which can cause damage to the heart over time. This damage can lead to a range of complications, including:

* Dilated cardiomyopathy: This is a condition in which the heart muscle becomes weakened and stretched, leading to an enlarged heart chamber and reduced pumping ability.
* Arrhythmias: These are abnormal heart rhythms that can cause symptoms such as palpitations, dizziness, and fainting.
* Heart failure: This is a condition in which the heart is unable to pump blood effectively, leading to symptoms such as shortness of breath, fatigue, and fluid buildup in the body.
* Cardiac arrest: In severe cases, Chagas cardiomyopathy can lead to sudden cardiac arrest, which is a medical emergency that requires immediate treatment.

Chagas cardiomyopathy is most commonly found in Latin America, where the parasite that causes the disease is endemic. However, due to increased travel and migration, cases of Chagas cardiomyopathy have been reported in other parts of the world, including the United States. Treatment for Chagas cardiomyopathy typically involves medications to manage symptoms and prevent further complications, as well as lifestyle changes such as diet and exercise modifications. In some cases, more invasive treatments such as surgery or implantable devices may be necessary to treat severe complications of the disease.

Fetal diseases are medical conditions or abnormalities that affect a fetus during pregnancy. These diseases can be caused by genetic factors, environmental influences, or a combination of both. They can range from mild to severe and may impact various organ systems in the developing fetus. Examples of fetal diseases include congenital heart defects, neural tube defects, chromosomal abnormalities such as Down syndrome, and infectious diseases such as toxoplasmosis or rubella. Fetal diseases can be diagnosed through prenatal testing, including ultrasound, amniocentesis, and chorionic villus sampling. Treatment options may include medication, surgery, or delivery of the fetus, depending on the nature and severity of the disease.

Prenatal ultrasonography, also known as obstetric ultrasound, is a medical diagnostic procedure that uses high-frequency sound waves to create images of the developing fetus, placenta, and amniotic fluid inside the uterus. It is a non-invasive and painless test that is widely used during pregnancy to monitor the growth and development of the fetus, detect any potential abnormalities or complications, and determine the due date.

During the procedure, a transducer (a small handheld device) is placed on the mother's abdomen and moved around to capture images from different angles. The sound waves travel through the mother's body and bounce back off the fetus, producing echoes that are then converted into electrical signals and displayed as images on a screen.

Prenatal ultrasonography can be performed at various stages of pregnancy, including early pregnancy to confirm the pregnancy and detect the number of fetuses, mid-pregnancy to assess the growth and development of the fetus, and late pregnancy to evaluate the position of the fetus and determine if it is head down or breech. It can also be used to guide invasive procedures such as amniocentesis or chorionic villus sampling.

Overall, prenatal ultrasonography is a valuable tool in modern obstetrics that helps ensure the health and well-being of both the mother and the developing fetus.

Heart neoplasms are abnormal growths or tumors that develop within the heart tissue. They can be benign (noncancerous) or malignant (cancerous). Benign tumors, such as myxomas and rhabdomyomas, are typically slower growing and less likely to spread, but they can still cause serious complications if they obstruct blood flow or damage heart valves. Malignant tumors, such as angiosarcomas and rhabdomyosarcomas, are fast-growing and have a higher risk of spreading to other parts of the body. Symptoms of heart neoplasms can include shortness of breath, chest pain, fatigue, and irregular heart rhythms. Treatment options depend on the type, size, and location of the tumor, and may include surgery, radiation therapy, or chemotherapy.

Heart failure is a pathophysiological state in which the heart is unable to pump sufficient blood to meet the metabolic demands of the body or do so only at the expense of elevated filling pressures. It can be caused by various cardiac disorders, including coronary artery disease, hypertension, valvular heart disease, cardiomyopathy, and arrhythmias. Symptoms may include shortness of breath, fatigue, and fluid retention. Heart failure is often classified based on the ejection fraction (EF), which is the percentage of blood that is pumped out of the left ventricle during each contraction. A reduced EF (less than 40%) is indicative of heart failure with reduced ejection fraction (HFrEF), while a preserved EF (greater than or equal to 50%) is indicative of heart failure with preserved ejection fraction (HFpEF). There is also a category of heart failure with mid-range ejection fraction (HFmrEF) for those with an EF between 40-49%.

A fatal outcome is a term used in medical context to describe a situation where a disease, injury, or illness results in the death of an individual. It is the most severe and unfortunate possible outcome of any medical condition, and is often used as a measure of the severity and prognosis of various diseases and injuries. In clinical trials and research, fatal outcome may be used as an endpoint to evaluate the effectiveness and safety of different treatments or interventions.

Electrocardiography (ECG or EKG) is a medical procedure that records the electrical activity of the heart. It provides a graphic representation of the electrical changes that occur during each heartbeat. The resulting tracing, called an electrocardiogram, can reveal information about the heart's rate and rhythm, as well as any damage to its cells or abnormalities in its conduction system.

During an ECG, small electrodes are placed on the skin of the chest, arms, and legs. These electrodes detect the electrical signals produced by the heart and transmit them to a machine that amplifies and records them. The procedure is non-invasive, painless, and quick, usually taking only a few minutes.

ECGs are commonly used to diagnose and monitor various heart conditions, including arrhythmias, coronary artery disease, heart attacks, and electrolyte imbalances. They can also be used to evaluate the effectiveness of certain medications or treatments.

Echocardiography is a medical procedure that uses sound waves to produce detailed images of the heart's structure, function, and motion. It is a non-invasive test that can help diagnose various heart conditions, such as valve problems, heart muscle damage, blood clots, and congenital heart defects.

During an echocardiogram, a transducer (a device that sends and receives sound waves) is placed on the chest or passed through the esophagus to obtain images of the heart. The sound waves produced by the transducer bounce off the heart structures and return to the transducer, which then converts them into electrical signals that are processed to create images of the heart.

There are several types of echocardiograms, including:

* Transthoracic echocardiography (TTE): This is the most common type of echocardiogram and involves placing the transducer on the chest.
* Transesophageal echocardiography (TEE): This type of echocardiogram involves passing a specialized transducer through the esophagus to obtain images of the heart from a closer proximity.
* Stress echocardiography: This type of echocardiogram is performed during exercise or medication-induced stress to assess how the heart functions under stress.
* Doppler echocardiography: This type of echocardiogram uses sound waves to measure blood flow and velocity in the heart and blood vessels.

Echocardiography is a valuable tool for diagnosing and managing various heart conditions, as it provides detailed information about the structure and function of the heart. It is generally safe, non-invasive, and painless, making it a popular choice for doctors and patients alike.

Congenital heart defects (CHDs) are structural abnormalities in the heart that are present at birth. They can affect any part of the heart's structure, including the walls of the heart, the valves inside the heart, and the major blood vessels that lead to and from the heart.

Congenital heart defects can range from mild to severe and can cause various symptoms depending on the type and severity of the defect. Some common symptoms of CHDs include cyanosis (a bluish tint to the skin, lips, and fingernails), shortness of breath, fatigue, poor feeding, and slow growth in infants and children.

There are many different types of congenital heart defects, including:

1. Septal defects: These are holes in the walls that separate the four chambers of the heart. The two most common septal defects are atrial septal defect (ASD) and ventricular septal defect (VSD).
2. Valve abnormalities: These include narrowed or leaky valves, which can affect blood flow through the heart.
3. Obstruction defects: These occur when blood flow is blocked or restricted due to narrowing or absence of a part of the heart's structure. Examples include pulmonary stenosis and coarctation of the aorta.
4. Cyanotic heart defects: These cause a lack of oxygen in the blood, leading to cyanosis. Examples include tetralogy of Fallot and transposition of the great arteries.

The causes of congenital heart defects are not fully understood, but genetic factors and environmental influences during pregnancy may play a role. Some CHDs can be detected before birth through prenatal testing, while others may not be diagnosed until after birth or later in childhood. Treatment for CHDs may include medication, surgery, or other interventions to improve blood flow and oxygenation of the body's tissues.

The myocardium is the middle layer of the heart wall, composed of specialized cardiac muscle cells that are responsible for pumping blood throughout the body. It forms the thickest part of the heart wall and is divided into two sections: the left ventricle, which pumps oxygenated blood to the rest of the body, and the right ventricle, which pumps deoxygenated blood to the lungs.

The myocardium contains several types of cells, including cardiac muscle fibers, connective tissue, nerves, and blood vessels. The muscle fibers are arranged in a highly organized pattern that allows them to contract in a coordinated manner, generating the force necessary to pump blood through the heart and circulatory system.

Damage to the myocardium can occur due to various factors such as ischemia (reduced blood flow), infection, inflammation, or genetic disorders. This damage can lead to several cardiac conditions, including heart failure, arrhythmias, and cardiomyopathy.

Pregnancy is a physiological state or condition where a fertilized egg (zygote) successfully implants and grows in the uterus of a woman, leading to the development of an embryo and finally a fetus. This process typically spans approximately 40 weeks, divided into three trimesters, and culminates in childbirth. Throughout this period, numerous hormonal and physical changes occur to support the growing offspring, including uterine enlargement, breast development, and various maternal adaptations to ensure the fetus's optimal growth and well-being.

Retrospective studies, also known as retrospective research or looking back studies, are a type of observational study that examines data from the past to draw conclusions about possible causal relationships between risk factors and outcomes. In these studies, researchers analyze existing records, medical charts, or previously collected data to test a hypothesis or answer a specific research question.

Retrospective studies can be useful for generating hypotheses and identifying trends, but they have limitations compared to prospective studies, which follow participants forward in time from exposure to outcome. Retrospective studies are subject to biases such as recall bias, selection bias, and information bias, which can affect the validity of the results. Therefore, retrospective studies should be interpreted with caution and used primarily to generate hypotheses for further testing in prospective studies.

A newborn infant is a baby who is within the first 28 days of life. This period is also referred to as the neonatal period. Newborns require specialized care and attention due to their immature bodily systems and increased vulnerability to various health issues. They are closely monitored for signs of well-being, growth, and development during this critical time.

Hypertension is a medical term used to describe abnormally high blood pressure in the arteries, often defined as consistently having systolic blood pressure (the top number in a blood pressure reading) over 130 mmHg and/or diastolic blood pressure (the bottom number) over 80 mmHg. It is also commonly referred to as high blood pressure.

Hypertension can be classified into two types: primary or essential hypertension, which has no identifiable cause and accounts for about 95% of cases, and secondary hypertension, which is caused by underlying medical conditions such as kidney disease, hormonal disorders, or use of certain medications.

If left untreated, hypertension can lead to serious health complications such as heart attack, stroke, heart failure, and chronic kidney disease. Therefore, it is important for individuals with hypertension to manage their condition through lifestyle modifications (such as healthy diet, regular exercise, stress management) and medication if necessary, under the guidance of a healthcare professional.

Prognosis is a medical term that refers to the prediction of the likely outcome or course of a disease, including the chances of recovery or recurrence, based on the patient's symptoms, medical history, physical examination, and diagnostic tests. It is an important aspect of clinical decision-making and patient communication, as it helps doctors and patients make informed decisions about treatment options, set realistic expectations, and plan for future care.

Prognosis can be expressed in various ways, such as percentages, categories (e.g., good, fair, poor), or survival rates, depending on the nature of the disease and the available evidence. However, it is important to note that prognosis is not an exact science and may vary depending on individual factors, such as age, overall health status, and response to treatment. Therefore, it should be used as a guide rather than a definitive forecast.

An encyclopedia is a comprehensive reference work containing articles on various topics, usually arranged in alphabetical order. In the context of medicine, a medical encyclopedia is a collection of articles that provide information about a wide range of medical topics, including diseases and conditions, treatments, tests, procedures, and anatomy and physiology. Medical encyclopedias may be published in print or electronic formats and are often used as a starting point for researching medical topics. They can provide reliable and accurate information on medical subjects, making them useful resources for healthcare professionals, students, and patients alike. Some well-known examples of medical encyclopedias include the Merck Manual and the Stedman's Medical Dictionary.

Coronary artery disease (CAD) is a medical condition in which the coronary arteries, which supply oxygen-rich blood to the heart muscle, become narrowed or blocked due to the buildup of cholesterol, fatty deposits, and other substances, known as plaque. Over time, this buildup can cause the arteries to harden and narrow (a process called atherosclerosis), reducing blood flow to the heart muscle.

The reduction in blood flow can lead to various symptoms and complications, including:

1. Angina (chest pain or discomfort) - This occurs when the heart muscle doesn't receive enough oxygen-rich blood, causing pain, pressure, or discomfort in the chest, arms, neck, jaw, or back.
2. Shortness of breath - When the heart isn't receiving adequate blood flow, it can't pump blood efficiently to meet the body's demands, leading to shortness of breath during physical activities or at rest.
3. Heart attack - If a piece of plaque ruptures or breaks off in a coronary artery, a blood clot can form and block the artery, causing a heart attack (myocardial infarction). This can damage or destroy part of the heart muscle.
4. Heart failure - Chronic reduced blood flow to the heart muscle can weaken it over time, leading to heart failure, a condition in which the heart can't pump blood efficiently to meet the body's needs.
5. Arrhythmias - Reduced blood flow and damage to the heart muscle can lead to abnormal heart rhythms (arrhythmias), which can be life-threatening if not treated promptly.

Coronary artery disease is typically diagnosed through a combination of medical history, physical examination, and diagnostic tests such as electrocardiograms (ECGs), stress testing, cardiac catheterization, and imaging studies like coronary computed tomography angiography (CCTA). Treatment options for CAD include lifestyle modifications, medications, medical procedures, and surgery.

In medical terms, the heart is a muscular organ located in the thoracic cavity that functions as a pump to circulate blood throughout the body. It's responsible for delivering oxygen and nutrients to the tissues and removing carbon dioxide and other wastes. The human heart is divided into four chambers: two atria on the top and two ventricles on the bottom. The right side of the heart receives deoxygenated blood from the body and pumps it to the lungs, while the left side receives oxygenated blood from the lungs and pumps it out to the rest of the body. The heart's rhythmic contractions and relaxations are regulated by a complex electrical conduction system.

Autopsy: Cardiomegaly is indicated if the heart weighs more than >399 grams in women and >449 grams in men. Cardiomegaly can be ... Cardiomegaly can be serious and can result in congestive heart failure. Recent studies suggest that cardiomegaly is associated ... For many people, cardiomegaly is asymptomatic. For others, if the enlarged heart begins to affect the bodys ability to pump ... Cardiomegaly may be a temporary condition that can resolve on its own. Smoking cessation Limiting alcohol and caffeine intake ...
Josephs Heart & Vascular Institute provides cardiomegaly care in Stockton. ... Symptoms of Cardiomegaly. There are often no signs or symptoms related to cardiomegaly in the early stages. More advanced cases ... Cardiomegaly is an enlarged heart. Cardiomegaly is not a condition itself but an abnormal physical finding resulting from a ... Not all cases of cardiomegaly are preventable. But, the best line of defense to prevent cardiomegaly is to practice a heart- ...
This course covers how fetal cardiomegaly is measured and assessed, the various etiologies of fetal cardiomegaly, and the ...
cardiomegaly answers are found in the Tabers Medical Dictionary powered by Unbound Medicine. Available for iPhone, iPad, ... "Cardiomegaly." Tabers Medical Dictionary, 24th ed., F.A. Davis Company, 2021. Nursing Central, nursing.unboundmedicine.com/ ... nursingcentral/view/Tabers-Dictionary/731709/all/cardiomegaly. Cardiomegaly. In: Venes DD, ed. Tabers Medical Dictionary. F.A ... Cardiomegaly [Internet]. In: Venes DD, editors. Tabers Medical Dictionary. F.A. Davis Company; 2021. [cited 2023 September 21 ...
Cardiomegaly. The chest radiograph usually shows an enlarged cardiac silhouette in patients with MR. The heart is increased in ... Relatively mild cardiomegaly and clinically significant changes in the lung fields suggest predominant mitral stenosis. In ... The absence of cardiomegaly indicates that the mitral regurgitation is either mild or acute. ...
See the most useful Cardiomegaly meaning in Urdu along with English definition and sentence(s). ... Next of Cardiomegaly. Cardiopathy : a disease of the heart. Previous of Cardiomegaly. Cardiff : the capital and largest city of ... Cardiomegaly Definitions. 1) Cardiomegaly, Enlarged Heart, Megacardia, Megalocardia : غیرمعمولی طور پر بڑھا ہوا دل : (noun) an ... How this page explains Cardiomegaly ?. It helps you understand the word Cardiomegaly with comprehensive detail, no other web ...
Discover the intricate workings of cardiomegaly, an enlarged heart condition. Gain insights into its causes, symptoms, and ... The Link Between Cardiomegaly and High Blood Pressure. cardiomegaly dеvelops and worsens as a result of high blood pressure. B ... Cardiomegaly Causes. There can be various cardiomegaly causes, each playing a role in the enlargement of the heart. ... What is Cardiomegaly?. Cardiomegaly refers to the enlargement of the heart muscle, often resulting from underlying conditions ...
This extra work leads to cardiomegaly.. Symptoms and diagnosis of mild cardiomegaly. Generally speaking, mild cardiomegaly does ... Mild cardiomegaly refers to a less severe form of cardiomegaly, which is an enlargement of the heart. The heart is responsible ... Treating mild cardiomegaly. It is important to understand that problems with the heart, especially cases of cardiomegaly, ... What causes mild cardiomegaly?. General hypertrophy: The heart is a muscle, and much like other muscles of the body, working ...
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Cardiomegaly (enlarged heart) (heart weighed 550 grams [g]; normal weight is ,400 g) [Siegel 1997] ... Siegel RJ [1997]. Cardiomegaly. In: Bloom S, ed. Diagnostic criteria for cardiovascular pathology acquired diseases. ... Pertinent findings from the autopsy included coronary artery disease, cardiomegaly (enlarged heart), and left ventricular ...
Inflammatory cardiomegaly. *Myocarditis - inflammation of the myocardium, the muscular part of the heart, caused most often by ... Calcified heart of an older woman with cardiomegaly. Age is the most important risk factor in developing cardiovascular or ...
Cardiomegaly (enlarged heart; heart weighed 510 grams [g]; predicted normal weight is 429 g [ranges between 325 g and 566 g as ...
Cardiomegaly. I51.9. Heart disease, unspecified. I51.9P. Prevent heart disease. I63. Cerebral infarction. ...
Left Ventricular Hypertrophy/Cardiomegaly. At autopsy, the D/O had LVH and cardiomegaly. These conditions increase the risk of ... Cardiomegaly (enlarged heart; heart weighed 646 grams [g]; predicted normal weight is 406 g [ranges between 308 g and 536 g as ... The D/O probably had chronic heart failure as evidenced by his cardiomegaly, LVH, and pedal edema. His acute onset of pulmonary ... LVH, cardiomegaly, and CHD led to his heart failure and increased risk for sudden cardiac death. ...
Cardiomegaly Essay. 289 Words , 2 Pages. This occurs when the heart becomes weakened and enlarged, and congestive heart failure ...
Cardiomegaly. *Cardiomyopathy. *Cardiomyopathy, Dilated. *Cardiomyopathy, Hypertrophic. *Cardiovascular Disease. *Carotid ...
Cardiomegaly. *Cardiomyopathy. *Cardiomyopathy, Hypertrophic. *Carotid Artery Disease. *Chest Pain. *Chronic Pulmonary Heart ...
Cardiomegaly / complications * Cardiomyopathies / etiology * Coronary Disease / complications * Coronary Disease / genetics * ...
Left Ventricular Hypertrophy and Cardiomegaly in swimming exercised 10-11-week-old white experimental rats ARTICLES ... Left Ventricular Hypertrophy and Cardiomegaly in swimming exercised 10-11-week-old white experimental rats ... Left Ventricular Hypertrophy and Cardiomegaly in swimming exercised 10-11-week-old white experimental rats. GEORGIAN SCIENTISTS ...
Cardiovascular evaluation: Cardiomegaly In patients with MM and amyloidosis, the characteristic examination findings include ...
Titus Carr Evans is a top-ranked physician with most experience for Cardiomegaly in Minnesota for 2022. Dr.Titus Carr Evans is ... a top-ranked Physician with most experience for Cardiomegaly in Minnesota according to Dexurs analysis of Patient volumes. ...
Meet Richard Cheng, MD. Dr. Cheng is a cardiologist who specializes in the comprehensive care of patients with heart failure. Learn more.
A look at cardiomegaly, a condition where the heart is enlarged. Included is detail on types, causes, and the symptoms of which ...
In patients with chronic MR, cardiomegaly due to left atrial or right-sided heart enlargement is visible.[27] ...
Hypertrichosis-osteochondrodysplasia-cardiomegaly syndrome, see Cantú syndrome. *Hypertrichotic osteochondrodysplasia, see ...
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  • Cardiomegaly can be serious and can result in congestive heart failure. (wikipedia.org)
  • Congestive heart failure results in the heart having to work harder to maintain the perfusion of blood, however, it often is insufficient and leads to cardiomegaly. (belmarrahealth.com)
  • Cardiomegaly without congestive heart failure. (acvr.org)
  • Cardiac - Tachycardia or bradycardia, bundle branch blocks, cardiomegaly or congestive heart failure. (medpagetoday.com)
  • Cardiomyopathy is also associated with cardiomegaly. (wikipedia.org)
  • Cardiomegaly may diminish over time, but many people with an enlarged heart (dilated cardiomyopathy) need lifelong medication. (wikipedia.org)
  • Additional autopsy findings included coronary artery disease, cardiomegaly (enlarged heart), and left ventricular hypertrophy (LVH). (cdc.gov)
  • The autopsy revealed "cardiac hypertrophy," "biventricular dilatation" and "cardiomegaly. (cdc.gov)
  • A chest radiograph showed massive cardiomegaly with prominent upper lobe vessels. (ispub.com)
  • Chest radiograph showing massive cardiomegaly. (ispub.com)
  • On day three post silo application, baby was noticed having respiratory distress and chest x-ray showed massive cardiomegaly pointing to congenital cardiac anomaly ( Figure 3 ), but she died before echocardiogram could be done. (scirp.org)
  • Recent studies suggest that cardiomegaly is associated with a higher risk of sudden cardiac death. (wikipedia.org)
  • Non-lifestyle risk factors include a family history of cardiomegaly, coronary artery disease (CAD), congenital heart failure, atherosclerotic disease, valvular heart disease, exposure to cardiac toxins, sleep-disordered breathing (such as sleep apnea), sustained cardiac arrhythmias, abnormal electrocardiograms, and cardiomegaly on chest X-ray. (wikipedia.org)
  • Typical findings of pulmonary edema due to cardiac dysfunction, including cardiomegaly , peribronchial cuffing , venous distention and increased interstitial markings. (radiopaedia.org)
  • citation needed] The most common causes of cardiomegaly are congenital (patients are born with the condition based on a genetic inheritance), high blood pressure (which can enlarge the left ventricle causing the heart muscle to weaken over time), and coronary artery disease. (wikipedia.org)
  • Cardiomegaly can be congenital (you are born with it) or caused by heart disease. (dignityhealth.org)
  • Chest X-ray showed a severe cardiomegaly and signs of pulmonary congestion. (scirp.org)
  • Cardiomegaly is not a condition itself but an abnormal physical finding resulting from a condition or disease. (dignityhealth.org)
  • Cardiomegaly (sometimes megacardia or megalocardia) is a medical condition in which the heart becomes enlarged. (wikipedia.org)
  • Autopsy was unremarkable except for cardiomegaly and hepatosplenomegaly. (erowid.org)
  • This type of cardiomegaly occurs when the heart chambers become enlarged and weakened, leading to decreased pumping efficiency. (healthtreatmentnews.com)
  • Cardiomegaly refers to the enlargement of the heart muscle, often resulting from underlying conditions or diseases. (healthtreatmentnews.com)
  • There can be various cardiomegaly causes, each playing a role in the enlargement of the heart. (healthtreatmentnews.com)
  • Mild cardiomegaly refers to a less severe form of cardiomegaly , which is an enlargement of the heart. (belmarrahealth.com)
  • There are often no signs or symptoms related to cardiomegaly in the early stages. (dignityhealth.org)
  • Find a Doctor affiliated with the Morrissey Family Heart & Vascular Institute if you have any of these symptoms of cardiomegaly in Stockton and the surrounding areas. (dignityhealth.org)
  • Tell your doctor if you have undergone past chemotherapy , such as with anthracyclines, or radiation therapy , so they can help you watch for signs and symptoms of cardiomegaly. (dignityhealth.org)
  • Generally speaking, mild cardiomegaly does not present with any noticeable symptoms, as real problems typically appear once cardiomegaly had reached moderate to severe status. (belmarrahealth.com)
  • Mild cardiomegaly is common in athletes. (wordinn.com)
  • What Causes Mild Cardiomegaly? (belmarrahealth.com)
  • A form of mild cardiomegaly that affects only one chamber of the heart, the left ventricle. (belmarrahealth.com)
  • Mild cardiomegaly may provide an opportunity to manage the condition on your own to prevent its progression. (belmarrahealth.com)
  • Bad habits and certain lifestyle choice may increase the chances of developing mild cardiomegaly. (belmarrahealth.com)
  • Ensure these conditions are treated properly to help reduce complications like cardiomegaly. (belmarrahealth.com)
  • The most useful parameters for the development of a PET-CT score were: nodular pleural thickening, pleural nodules with SUV>7.5, lung mass or extra pleural malignancy (10 points each), mammary lymph node with SUV>4.5 (5 points) and cardiomegaly (−1 point). (bvsalud.org)
  • According to a recent study conducted by the American Heart Association, cardiomegaly affects approximately 1 in every 500 people globally, highlighting the significance of understanding this prevalent condition. (healthtreatmentnews.com)
  • Conditions such as mitral valve regurgitation, aortic stenosis, or aortic regurgitation can contribute to cardiomegaly. (healthtreatmentnews.com)
  • Lifestyle factors that can help prevent cardiomegaly include eating a healthy diet, controlling blood pressure, exercise, medications, and not abusing alcohol and cocaine. (wikipedia.org)
  • Not all cases of cardiomegaly are preventable. (dignityhealth.org)
  • It is important to understand that problems with the heart, especially cases of cardiomegaly, should be treated with the aid of a trained medical professional. (belmarrahealth.com)
  • Chest X-ray revealed new cardiomegaly ( Figure 2A ) compared with a previous examination 1 year before ( Figure 2B ). (ajtmh.org)
  • If your doctor has told you that you are at risk for cardiomegaly, we invite you to partner with our experts to improve your heart health. (dignityhealth.org)
  • For many people, cardiomegaly is asymptomatic. (wikipedia.org)
  • All of this may seem less if you are unable to learn exact pronunciation of Cardiomegaly, so we have embedded mp3 recording of native Englishman, simply click on speaker icon and listen how English speaking people pronounce Cardiomegaly. (wordinn.com)
  • This way, you and your doctor can tackle known risk factors and prevent cardiomegaly all together. (dignityhealth.org)
  • Having a family history of cardiomegaly may indicate an increased risk for this condition. (wikipedia.org)
  • What our highly trained professionals recommend for treating and preventing cardiomegaly depends on the cause of the condition. (dignityhealth.org)
  • Cardiomegaly Demystified: Insights into Enlarged Heart Condition! (healthtreatmentnews.com)
  • Cardiomegaly Unveiled: Understanding the Enlarged Heart Condition! (healthtreatmentnews.com)
  • Cardiomegaly, the medical term for an enlarged heart, is a condition that warrants attention and understanding. (healthtreatmentnews.com)
  • Cardiomegaly is a condition where the heart becomes larger or hypertrophied due to being overworked. (belmarrahealth.com)
  • You can also find multiple synonyms or similar words of Cardiomegaly. (wordinn.com)
  • It helps you understand the word Cardiomegaly with comprehensive detail, no other web page in our knowledge can explain Cardiomegaly better than this page. (wordinn.com)
  • We hope this page has helped you understand Cardiomegaly in detail, if you find any mistake on this page, please keep in mind that no human being can be perfect. (wordinn.com)
  • Natural cardiomegaly cures may prove useful when combined with a healthy diet and active lifestyle. (belmarrahealth.com)
  • But, the best line of defense to prevent cardiomegaly is to practice a heart-healthy lifestyle and see your doctor at Dignity Health regularly for physical checkups. (dignityhealth.org)
  • Effective management of blood pressure through lifestyle modifications and medication is crucial in preventing and managing cardiomegaly. (healthtreatmentnews.com)
  • Thіs lowers thе amount of blood flowіng to the hеartmusclе, impairіngіts performance and possibly resulting in cardiomegaly. (healthtreatmentnews.com)
  • Nursing Central , nursing.unboundmedicine.com/nursingcentral/view/Tabers-Dictionary/731709/all/cardiomegaly. (unboundmedicine.com)

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