Electrophysiologic Techniques, Cardiac
Heart Conduction System
Voltage-Sensitive Dye Imaging
Long QT Syndrome
Death, Sudden, Cardiac
Cardiac Pacing, Artificial
Decennial follow-up in patients with recurrent tachycardia originating from the right ventricular outflow tract: electrophysiologic characteristics and response to treatment. (1/71)AIMS: In the setting of right ventricular outflow tract-tachycardia (RVOT-T), data about long-term follow-up (FU) with respect to the therapeutic strategies are missing. All patients (pts) referred to our institution during the last 20 years for the treatment of RVOT-T were studied in a retrospective analysis to assess mortality and efficacy of treatment. METHODS AND RESULTS: One hundred and thirty-three patients (77 female; 39+/-13 years) with sustained RVOT-T were included in this study. At the time of first presentation, diagnosis of RVOT-T was made by complete invasive and non-invasive diagnostic assessment, including electrophysiology study and two-dimensional echocardiography. After 135+/-68 months (median 136, range 29-248), patients were invited to undergo clinical assessment. Of the 133 pts, 127 (95%) survived and six (5%) died from non-cardiac disease. Anti-arrhythmic (AA) drugs were given to 62 of the 133 pts (47%); of them 32 (52%) had recurrences during follow-up. The mean time to recurrence was 10.02 years (95% CI 7.46-12.59). The other 71 study patients (53%) underwent catheter ablation. The procedure was successful in 58 pts (82%). During follow-up, 30 (52%) of the 58 successfully treated patients had recurrences of RVOT-T. The mean time to recurrence was 6.28 years (95% CI 4.96-7.6). RVOT-T recurrences were similar in morphology to those treated previously in 33% and different in 67% of cases. CONCLUSIONS: Long-term follow-up in patients with RVOT-T is favourable. Catheter ablation is effective in this setting. However, late recurrences with similar or different morphology may arise in half of the patients after initially successful treatment. AA drug therapy is a valid initial therapeutic option, since it is effective in about half of the patients. (+info)
Association of atrial arrhythmia and sinus node dysfunction in patients with catecholaminergic polymorphic ventricular tachycardia. (2/71)BACKGROUND: This study was performed to investigate the frequency and importance of supraventricular arrhythmia and sinus node (SN) dysfunction in patients with catecholaminergic polymorphic ventricular tachycardia (CPVT). METHODS AND RESULTS: Eight patients with CPVT (mean age: 16.8+/-8.1 years) underwent an electrophysiological study. SN recovery time (1,389+/-394 ms) was slightly prolonged, and 4 of 8 patients had abnormal values. Atrial flutter (AF) was induced by low-rate atrial pacing in 2 patients and by isoproterenol infusion in 1 patient. Atrial fibrillation (Af) was induced by isoproterenol infusion in 2 patients. One patient presented with Af during the follow-up period, and 2 of 4 patients with AF/Af presented with increased SN recovery time. CONCLUSIONS: Patients with CPVT frequently have associated with SN dysfunction, and inducible atrial tachyarrhythmias, which indicate that the pathogenesis of CPVT is limited not only to the ventricular myocardium, but also to broad regions of the heart, including the SN and atrial muscle. (+info)
Matters of the heart: the physiology of cardiac function and failure. (3/71)Heart failure as a result of a myocardial infarction (MI) is a common condition with a poor prognosis. The adaptive changes in the surviving myocardium appear to be insufficient in terms of both mechanical/contractile performance and electrical stability. The modification of the underlying myocardial physiology is complex, varying across the different layers within the wall of the ventricle and within one layer. Two therapeutic strategies are briefly discussed, as outlined here. (i) Enhancing contractility by alteration of the expression of a single protein (e.g. sarco-endoplasmic reticulum Ca(2+) ATPase, SERCA) could potentially reverse both mechanical and electrical abnormalities. However, experimental data involving the upregulation of SERCA suggest that the therapeutic range of this approach is narrow. (ii) The use of regular exercise training to improve cardiac performance in heart failure. This appears to act by normalizing a number of aspects of myocardial physiology. (+info)
Electrophysiological consequence of adipose-derived stem cell transplantation in infarcted porcine myocardium. (4/71)AIMS: Aim of this study was to investigate the effect of intracoronary administration of freshly isolated adipose-derived mononuclear cells (ADMCs) on myocardial vulnerability to arrhythmia induction after infarction. METHODS AND RESULTS: A transmural myocardial infarction in an experimental porcine model was induced by occlusion of the mid-left anterior descending artery with an angioplasty balloon for 3 h. Upon reperfusion, a cellular suspension with freshly isolated ADMCs (1.5 x 10(6) cells/kg BW) or vehicle alone was injected into the infarct artery. All animals underwent a programmed ventricular stimulation at 8 weeks follow-up for possible induction of ventricular arrhythmias using a train of 8 S1 stimuli. Cell injections did not cause acute ventricular arrhythmia, bradycardia, or conduction block. The cycle length of the ventricular arrhythmia was compared at 1 and 10 s following its induction. Despite comparable infarct size in both groups, we found that the cycle length of the induced ventricular arrhythmia in the ADMC-treated group was significantly longer compared with control animals (P < 0.05). We also found that extra-stimuli were required for arrhythmia induction in the ADMC-treated group compared with control animals. CONCLUSION: Freshly isolated autologous stem cell therapy is not proarrhythmic in pigs. (+info)
Mechanisms of calcium transient and action potential alternans in cardiac cells and tissues. (5/71)Alternation of cardiac action potential duration (APD) from beat to beat and concurrent alternation of the amplitude of the calcium transient are regarded as important arrhythmia mechanisms. These phenomena are causally interrelated and can be reliably evoked by an increase in beat frequency or by ischemia. The first part of this historical review deals with the physiology of APD alternans. Sections recounting the evolution of knowledge about calcium-activated ion currents and calcium transient alternans are interspersed among sections describing the growth of the so-called "restitution hypothesis," which involves time-dependent recovery of potassium channels (including their passage through pre-open states) as a function of diastolic interval. Major developments are generally in chronological order, but it is necessary to move back and forth between the two theories to respect the overall time line, which runs from about l965 to the present. The concluding two sections deal with the pathophysiology of calcium transient and APD alternans during ischemia, which may be the basis for out-of-hospital cardiac arrest during the initial stages of acute myocardial infarction. (+info)
Pharmacological separation of early afterdepolarizations from arrhythmogenic substrate in DeltaKPQ Scn5a murine hearts modelling human long QT 3 syndrome. (6/71)AIM: To perform an empirical, pharmacological, separation of early afterdepolarizations (EADs) and transmural gradients of repolarization in arrhythmogenesis in a genetically modified mouse heart modelling human long QT syndrome (LQT) 3. METHODS: Left ventricular endocardial and epicardial monophasic action potentials and arrhythmogenic tendency were compared in isolated wild type (WT) and Scn5a+/Delta hearts perfused with 0.1 and 1 microm propranolol and paced from the right ventricular epicardium. RESULTS: All spontaneously beating bradycardic Scn5a+/Delta hearts displayed EADs, triggered beats and ventricular tachycardia (VT; n = 7), events never seen in WT hearts (n = 5). Perfusion with 0.1 and 1 microm propranolol suppressed all EADs, triggered beats and episodes of VT. In contrast, triggering of VT persisted following programmed electrical stimulation in 6 of 12 (50%), one of eight (12.5%), but six of eight (75%) Scn5a+/Delta hearts perfused with 0, 0.1 and 1 microm propranolol respectively in parallel with corresponding alterations in repolarization gradients, reflected in action potential duration (DeltaAPD(90)) values. Thus 0.1 microm propranolol reduced epicardial but not endocardial APD(90) from 54.7 +/- 1.6 to 44.0 +/- 2.0 ms, restoring DeltaAPD(90) from -3.8 +/- 1.6 to 3.5 +/- 2.5 ms (all n = 5), close to WT values. However, 1 microm propranolol increased epicardial APD(90) to 72.5 +/- 1.2 ms and decreased endocardial APD(90) from 50.9 +/- 1.0 to 24.5 +/- 0.3 ms, increasing DeltaAPD(90) to -48.0 +/- 1.2 ms. CONCLUSION: These findings empirically implicate EADs in potentially initiating spontaneous arrhythmogenic phenomena and transmural repolarization gradients in the re-entrant substrate that would sustain such activity when provoked by extrasystolic activity in murine hearts modelling human LQT3 syndrome. (+info)
Arrhythmogenesis research: a perspective from computational electrophysiology viewpoint. (7/71)The mechanisms by which arrhythmias are generated in the heart remains a field of intensive research. Recent advances in computational biology and electrophysiology have enabled researchers to use an alternative tool in the study of arrhythmia mechanisms, the multi-scale modeling and simulation of cardiac arrhythmogenesis at the organ level. This article reviews the recent advances and achievements using this approach. (+info)
Hyperpolarization-activated cyclic nucleotide-modulated 'HCN' channels confer regular and faster rhythmicity to beating mouse embryonic stem cells. (8/71)The hyperpolarization-activated cation current (I(f)), and the hyperpolarization-activated cyclic nucleotide-modulated 'HCN' subunits that underlie it, are important components of spontaneous activity in the embryonic mouse heart, but whether they contribute to this activity in mouse embryonic stem cell-derived cardiomyocytes has not been investigated. We address this issue in spontaneously beating cells derived from mouse embryonic stem cells (mESCs) over the course of development in culture. I(f) and action potentials were recorded from single beating cells at early, intermediate and late development stages using perforated whole-cell voltage- and current-clamp techniques. Our data show that the proportion of cells expressing I(f), and the density of I(f) in these cells, increased during development and correlated with action potential frequency and the rate of diastolic depolarization. The I(f) blocker ZD7288 (0.3 microm) reduced I(f) and the beating rate of embryoid bodies. Taken together, the activation kinetics of I(f) and results from Western blots are consistent with the presence of the HCN2 and HCN3 isoforms. At all stages of development, isoproterenol (isoprenaline) and acetylcholine shifted the voltage dependence of I(f) to more positive and negative voltages, respectively, and they also increased and decreased the beating rate of embryonic cell bodies, respectively. Together, the data suggest that current through HCN2 and HCN3 channels confers regular and faster rhythmicity to mESCs, which mirrors the developing embryonic mouse heart, and contributes to modulation of rhythmicity by autonomic stimulation. (+info)
There are many different types of cardiac arrhythmias, including:
1. Tachycardias: These are fast heart rhythms that can be too fast for the body's needs. Examples include atrial fibrillation and ventricular tachycardia.
2. Bradycardias: These are slow heart rhythms that can cause symptoms like fatigue, dizziness, and fainting. Examples include sinus bradycardia and heart block.
3. Premature beats: These are extra beats that occur before the next regular beat should come in. They can be benign but can also indicate an underlying arrhythmia.
4. Supraventricular arrhythmias: These are arrhythmias that originate above the ventricles, such as atrial fibrillation and paroxysmal atrial tachycardia.
5. Ventricular arrhythmias: These are arrhythmias that originate in the ventricles, such as ventricular tachycardia and ventricular fibrillation.
Cardiac arrhythmias can be diagnosed through a variety of tests including electrocardiograms (ECGs), stress tests, and holter monitors. Treatment options for cardiac arrhythmias vary depending on the type and severity of the condition and may include medications, cardioversion, catheter ablation, or implantable devices like pacemakers or defibrillators.
The QT interval is a measure of the time it takes for the ventricles to recover from each heartbeat and prepare for the next one. In people with LQTS, this recovery time is prolonged, which can disrupt the normal rhythm of the heart and increase the risk of arrhythmias.
LQTS is caused by mutations in genes that encode proteins involved in the cardiac ion channels, which regulate the flow of ions into and out of the heart muscle cells. These mutations can affect the normal functioning of the ion channels, leading to abnormalities in the electrical activity of the heart.
Symptoms of LQTS can include palpitations, fainting spells, and seizures. In some cases, LQTS can be diagnosed based on a family history of the condition or after a sudden death in an otherwise healthy individual. Other tests, such as an electrocardiogram (ECG), echocardiogram, and stress test, may also be used to confirm the diagnosis.
Treatment for LQTS typically involves medications that regulate the heart's rhythm and reduce the risk of arrhythmias. In some cases, an implantable cardioverter-defibrillator (ICD) may be recommended to monitor the heart's activity and deliver an electric shock if a potentially life-threatening arrhythmia is detected. Lifestyle modifications, such as avoiding stimuli that trigger symptoms and taking precautions during exercise and stress, may also be recommended.
In summary, Long QT syndrome is a rare inherited disorder that affects the electrical activity of the heart, leading to an abnormal prolongation of the QT interval and an increased risk of irregular and potentially life-threatening heart rhythms. It is important for individuals with LQTS to be closely monitored by a healthcare provider and to take precautions to manage their condition and reduce the risk of complications.
Some examples of the use of 'Death, Sudden, Cardiac' in medical contexts include:
1. Sudden cardiac death (SCD) is a major public health concern, affecting thousands of people each year in the United States alone. It is often caused by inherited heart conditions, such as hypertrophic cardiomyopathy or long QT syndrome.
2. The risk of sudden cardiac death is higher for individuals with a family history of heart disease or other pre-existing cardiovascular conditions.
3. Sudden cardiac death can be prevented by prompt recognition and treatment of underlying heart conditions, as well as by avoiding certain risk factors such as smoking, physical inactivity, and an unhealthy diet.
4. Cardiopulmonary resuscitation (CPR) and automated external defibrillators (AEDs) can be effective in restoring a normal heart rhythm during sudden cardiac death, especially when used promptly after the onset of symptoms.
Medical Term: Cardiomegaly
Definition: An abnormal enlargement of the heart.
Symptoms: Difficulty breathing, shortness of breath, fatigue, swelling of legs and feet, chest pain, and palpitations.
Causes: Hypertension, cardiac valve disease, myocardial infarction (heart attack), congenital heart defects, and other conditions that affect the heart muscle or cardiovascular system.
Diagnosis: Physical examination, electrocardiogram (ECG), chest x-ray, echocardiography, and other diagnostic tests as necessary.
Treatment: Medications such as diuretics, vasodilators, and beta blockers, lifestyle changes such as exercise and diet modifications, surgery or other interventions in severe cases.
Note: Cardiomegaly is a serious medical condition that requires prompt diagnosis and treatment to prevent complications such as heart failure and death. If you suspect you or someone else may have cardiomegaly, seek medical attention immediately.
There are many different types of heart diseases, including:
1. Coronary artery disease: The buildup of plaque in the coronary arteries, which supply blood to the heart muscle, leading to chest pain or a heart attack.
2. Heart failure: When the heart is unable to pump enough blood to meet the body's needs, leading to fatigue, shortness of breath, and swelling in the legs.
3. Arrhythmias: Abnormal heart rhythms, such as atrial fibrillation or ventricular tachycardia, which can cause palpitations, dizziness, and shortness of breath.
4. Heart valve disease: Problems with the heart valves, which can lead to blood leaking back into the chambers or not being pumped effectively.
5. Cardiomyopathy: Disease of the heart muscle, which can lead to weakened heart function and heart failure.
6. Heart murmurs: Abnormal sounds heard during a heartbeat, which can be caused by defects in the heart valves or abnormal blood flow.
7. Congenital heart disease: Heart defects present at birth, such as holes in the heart or abnormal blood vessels.
8. Myocardial infarction (heart attack): Damage to the heart muscle due to a lack of oxygen, often caused by a blockage in a coronary artery.
9. Cardiac tamponade: Fluid accumulation around the heart, which can cause compression of the heart and lead to cardiac arrest.
10. Endocarditis: Infection of the inner lining of the heart, which can cause fever, fatigue, and heart valve damage.
Heart diseases can be diagnosed through various tests such as electrocardiogram (ECG), echocardiogram, stress test, and blood tests. Treatment options depend on the specific condition and may include lifestyle changes, medication, surgery, or a combination of these.
Clinical cardiac electrophysiology
Cardiac Electrophysiology Society
QT interval variability
Wandering atrial pacemaker
Sinus node dysfunction
Frank I. Marcus
M. Stephen Heilman
Cardiac action potential
Vrije Universiteit Brussel
Robotic magnetic navigation
Myocardial infarction complications
Mid America Heart Institute
Novel Therapeutic Targets for Antiarrhythmic Drugs
List of circulatory system conditions
Paul N. Yu
Tarlochan Singh Kler
IBM Blue Gene
Heart Rhythm Society
Lithium metal battery
St. Bernards Medical Center
Ball and chain inactivation
Mount Sinai Medical Center (Miami)
Management of atrial fibrillation
Electrochemical skin conductance
Advances in Cardiac Electrophysiology - PubMed
Cardiac Electrophysiology Diagram Templates
Mastering the art of epicardial access in cardiac electrophysiology - PubMed
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Details for: Cardiac pacing and electrophysiology today : › WHO HQ Library catalog
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- Cardiac arrhythmias can present as benign ectopics or as life-threatening arrhythmias and sudden cardiac death. (nih.gov)
- Clinical cardiac electrophysiology is the study of the electrophysiology of the heart and all aspects of management of cardiac arrhythmias. (nih.gov)
- The treatment of bradyarrhythmias with permanent pacemaker implantation represents the first interventional therapy for patients with cardiac arrhythmias. (nih.gov)
- 2] Entropy of cardiac repolarization predicts ventricular arrhythmias and mortality in patients receiving an implantable cardioverter-defibrillator for primary prevention of sudden death . (nih.gov)
- 3] Dynamic analysis of cardiac rhythms for discriminating atrial fibrillation from lethal ventricular arrhythmias . (nih.gov)
- The Pediatric Electrophysiology Program at Children's Health℠ has a range of tests and treatments to help kids with dangerous or bothersome arrhythmias grow up safe and active. (childrens.com)
- Electrophysiology focuses on arrhythmias, which are irregular heartbeats caused by abnormal electrical signals. (childrens.com)
- Sudden cardiac death, arrhythmias and abnormal electrocardiography in systemic sclerosis: A systematic review and meta-analysis. (cdc.gov)
- As a cardiac electrophysiologist, Deeptankar DeMazumder has worked for years with people at risk for sudden cardiac arrest (SCA). (nih.gov)
- FindaTopDoc has aggregated the experiences from real patients to help give you more insights and information on how to choose the best Cardiac Electrophysiologist in your area. (findatopdoc.com)
- Dr. Sanjeev Saksena, MD is a Clinical Cardiac Electrophysiologist in Warren, NJ. (sharecare.com)
- Marshfield Medical Center - Weston is seeking a BC/BE fellowship trained Cardiac Electrophysiologist to join a busy and established Cardiology department. (340bemployed.org)
- The Arkansas Heart Hospital Clinic in McGehee offers the best in heart health and cardiac care, including general cardiology and cardiac electrophysiology services, to Desha County. (arheart.com)
- Cardiac electrophysiology requires training beyond a general cardiology fellowship. (upmc.com)
- The list of accolades demonstrates our Institute's excellence in cardiovascular surgery, cardiac intervention, heart failure, heart attack, critical care, electrophysiology services, cardiology and cardiovascular research and rehabilitation. (renown.org)
- 1] Mitochondrial ROS drive sudden cardiac death and chronic proteome remodeling in heart failure . (nih.gov)
- David Ho, MD, who specializes in cardiac electrophysiology at UPMC Altoona, offers an in-depth look at this field, from an expert's perspective. (upmc.com)
- Dr. Saksena in specializes in Clinical Cardiac Electrophysiology. (sharecare.com)
- The Pediatric Electrophysiology Program at Children's Health is one of a small number in North Texas that specializes in treating kids. (childrens.com)
- Children's Health has one of only a few electrophysiology programs in Texas that specializes in treating children. (childrens.com)
- IAC Cardiac Electrophysiology will present a live webinar (followed by a Q&A session) entitled IAC Cardiac Electrophysiology: Accreditation Simplified . (intersocietal.org)
- Cardiac pacing and electrophysiology today : a new era of therapeutic arrhythmology, proceedings of the 5th Asian-Pacific Symposium on cardiac pacing and electrophysiology. (who.int)
- They currently practice at Cardiac Electro Physiology and are affiliated with St. Mary's General Hospital, Hackensack Meridian Health JFK Medical Center and Robert Wood Johnson University Hospital. (sharecare.com)
- The report scope combines detailed research of Global Electrophysiology and Cardiac Ablation Device Market 2022 with the apprehension given in the advancement of the industry in certain regions. (setuppost.com)
- Clinical electrophysiology 2023 7 9 (7 Pt 2): 1147-1157. (cdc.gov)
- The Little Rock Clinic is home to the Strong Hearts Intensive Cardiac Rehab , Vein and Vascular Institute , Bariatric & Metaboloic Institute and Keep the Beat Screenings . (arheart.com)
- At the UPMC Heart and Vascular Institute, patients can have many of their cardiovascular care needs, from advanced cardiac imaging to cardiac rehab, met at one location. (upmc.com)
- The invasive electrophysiological study was initially purely diagnostic, but recent advances in technology has allowed us to intervene and hence the term interventional electrophysiology. (nih.gov)
- The ultimate goal is to develop a useful tool for cardiac electrophysiologists which facilitates and improves dissemination of the minimum information necessary for reproduction of cardiac electrophysiology research, allowing for easier comparison and utilisation of findings by others. (nih.gov)
- This disease can cause progressive cardiac damage postinfection in 30% of infected persons without any initial suggestive clinical symptoms. (cdc.gov)
- Cardiac electrophysiology is a medical specialty with a long and rich tradition of computational modeling . (bvsalud.org)
- The Arkansas Heart Hospital clinic in Mountain Home proudly offers cardiac care at the communities of Briarcliff, Cotter, Gassville, Lakeview, Norfork, Salesville and Baxter County. (arheart.com)
- The Arkansas Heart Hospital Clinic in Little Rock proudly serves Central Arkansas with cardiac care. (arheart.com)
- Arkansas Heart Hospital Clinic in Saline County is proud to serve the community of Bryant, Benton and beyond with the best in heart health and cardiac care. (arheart.com)
- The successful applicant will join a team of 4 other EP physicians, 4 associate providers, and the busiest cardiac device clinic in the state, embedded in an academic practice of approximately 40 full-time cardiologists. (caacc.org)
- Focuses on the key global Electrophysiology and Cardiac Ablation Device companies, to define, describe and analyze the sales volume, value, market share, market competition landscape, and recent development. (setuppost.com)
- Our comprehensive team diagnoses heart disease and other cardiac conditions, offering personalized treatment plans. (renown.org)
- These latent infections can remain quiescent for decades before manifesting as cardiac complications, including cardiomyopathy, heart failure, and rare cardiac arrest ( 2 ). (cdc.gov)
- The report offers detailed coverage of Electrophysiology and Cardiac Ablation Device industries and main market trends. (setuppost.com)
- The market research includes historical and forecasts market data, demand, application details, price trends, company shares, size, volume, and value of the leading Electrophysiology and Cardiac Ablation Device by geography. (setuppost.com)
- This research report categorizes the global Electrophysiology and Cardiac Ablation Device market by top players/brands, region, type, and end-user. (setuppost.com)
- This Electrophysiology and Cardiac Ablation Device market report regional outlook in North America, Europe, Asia Pacific, and Other regions (the Middle East & Africa, Central & South America). (setuppost.com)
- To project the value and sales volume of Electrophysiology and Cardiac Ablation Device market, with respect to key regions. (setuppost.com)
- To study and analyze the global Electrophysiology and Cardiac Ablation Device market size (value & volume) by company, key regions, products, and end-user, breakdown data from the last five years, and forecast to 2026. (setuppost.com)
- To understand the structure of the Electrophysiology and Cardiac Ablation Device market by identifying its various sub-segments. (setuppost.com)
- The clinic provides cardiac care and testing as well as health screenings, wellness fairs and educational seminars for the local police and fire departments. (arheart.com)
- So please join us in honoring the Cardiac Cath Lab team at Renown Health! (renown.org)
- Therefore, we evaluated potential transmission sources and cardiac health of blood donors from south central Texas with T. cruzi antibodies. (cdc.gov)
- Cardiac Anesthesiologist Opportunity in San Antonio, TX! (scahq.org)
- Hackensack University Medical Center-Lucrative package including $125K SDB, Cardiac call only! (scahq.org)
- Do I need to travel for cardiac electrophysiology care? (upmc.com)
- Cardiac electrophysiology is one of those areas of care. (upmc.com)
- Cardiac EP - $659k Two Year Guarantee + RVU Full-Time Cardiac EP / Hospital Employed Be a part of a group that prides itself on comfortable quality patient care and state-of-the-art technology. (decaturrecruiter.com)
- As a step towards establishing this, we present a draft standard, called Minimum Information about a Cardiac Electrophysiology Experiment (MICEE). (nih.gov)
- Students, please listen to this prior to the beginning of the electrophysiology/electrocardiogram section of the course. (oruen-cardiology.com)
- Our electrophysiology lab offers the most advanced treatments, available for heart rhythm disorders. (upmc.com)
- Dr. Abdelhadi is board certified in Cardiac Electrophysiology, Cardiovascular Disease and Internal Medicine. (findatopdoc.com)
- We invite the reader to join this effort, and, if deemed productive, implement the Minimum Information about a Cardiac Electrophysiology Experiment standard in their own work. (nih.gov)
- As an open simulator, openCARP can advance the computational cardiac electrophysiology field by making state -of-the- art simulations accessible. (bvsalud.org)
- After a brief historic review , this paper summarizes requirements for a high-usability electrophysiology simulator and describes how openCARP fulfills them. (bvsalud.org)