Carbimazole. Medical search

An imidazole antithyroid agent. Carbimazole is metabolized to METHIMAZOLE, which is responsible for the antithyroid activity.

Agents that are used to treat hyperthyroidism by reducing the excessive production of thyroid hormones.

A hypermetabolic syndrome caused by excess THYROID HORMONES which may come from endogenous or exogenous sources. The endogenous source of hormone may be thyroid HYPERPLASIA; THYROID NEOPLASMS; or hormone-producing extrathyroidal tissue. Thyrotoxicosis is characterized by NERVOUSNESS; TACHYCARDIA; FATIGUE; WEIGHT LOSS; heat intolerance; and excessive SWEATING.

Hypersecretion of THYROID HORMONES from the THYROID GLAND. Elevated levels of thyroid hormones increase BASAL METABOLIC RATE.

A common form of hyperthyroidism with a diffuse hyperplastic GOITER. It is an autoimmune disorder that produces antibodies against the THYROID STIMULATING HORMONE RECEPTOR. These autoantibodies activate the TSH receptor, thereby stimulating the THYROID GLAND and hypersecretion of THYROID HORMONES. These autoantibodies can also affect the eyes (GRAVES OPHTHALMOPATHY) and the skin (Graves dermopathy).

A thioureylene antithyroid agent that inhibits the formation of thyroid hormones by interfering with the incorporation of iodine into tyrosyl residues of thyroglobulin. This is done by interfering with the oxidation of iodide ion and iodotyrosyl groups through inhibition of the peroxidase enzyme.

A thiourea antithyroid agent. Propythiouracil inhibits the synthesis of thyroxine and inhibits the peripheral conversion of throxine to tri-iodothyronine. It is used in the treatment of hyperthyroidism. (From Martindale, The Extra Pharmacopeoia, 30th ed, p534)

The major hormone derived from the thyroid gland. Thyroxine is synthesized via the iodination of tyrosines (MONOIODOTYROSINE) and the coupling of iodotyrosines (DIIODOTYROSINE) in the THYROGLOBULIN. Thyroxine is released from thyroglobulin by proteolysis and secreted into the blood. Thyroxine is peripherally deiodinated to form TRIIODOTHYRONINE which exerts a broad spectrum of stimulatory effects on cell metabolism.

An erythematous eruption commonly associated with drug reactions or infection and characterized by inflammatory nodules that are usually tender, multiple, and bilateral. These nodules are located predominantly on the shins with less common occurrence on the thighs and forearms. They undergo characteristic color changes ending in temporary bruise-like areas. This condition usually subsides in 3-6 weeks without scarring or atrophy.

A highly vascularized endocrine gland consisting of two lobes joined by a thin band of tissue with one lobe on each side of the TRACHEA. It secretes THYROID HORMONES from the follicular cells and CALCITONIN from the parafollicular cells thereby regulating METABOLISM and CALCIUM level in blood, respectively.

A benign SCHWANNOMA of the eighth cranial nerve (VESTIBULOCOCHLEAR NERVE), mostly arising from the vestibular branch (VESTIBULAR NERVE) during the fifth or sixth decade of life. Clinical manifestations include HEARING LOSS; HEADACHE; VERTIGO; TINNITUS; and FACIAL PAIN. Bilateral acoustic neuromas are associated with NEUROFIBROMATOSIS 2. (From Adams et al., Principles of Neurology, 6th ed, p673)

A tumor made up of nerve cells and nerve fibers. (Dorland, 27th ed)

The testing of the acuity of the sense of hearing to determine the thresholds of the lowest intensity levels at which an individual can hear a set of tones. The frequencies between 125 and 8000 Hz are used to test air conduction thresholds and the frequencies between 250 and 4000 Hz are used to test bone conduction thresholds.

Receptors of CLONIDINE and other IMIDAZOLINES. Activity of the ligands was earlier attributed to ADRENERGIC ALPHA-2 RECEPTORS. Endogenous ligands include AGMATINE, imidazoleacetic acid ribotide, and harman.

Dosage forms of a drug that act over a period of time by controlled-release processes or technology.

A syndrome that results from abnormally low secretion of THYROID HORMONES from the THYROID GLAND, leading to a decrease in BASAL METABOLIC RATE. In its most severe form, there is accumulation of MUCOPOLYSACCHARIDES in the SKIN and EDEMA, known as MYXEDEMA.

Impairment of bile flow due to injury to the HEPATOCYTES; BILE CANALICULI; or the intrahepatic bile ducts (BILE DUCTS, INTRAHEPATIC).

Abnormal protrusion of both eyes; may be caused by endocrine gland malfunction, malignancy, injury, or paralysis of the extrinsic muscles of the eye.

A congenital abnormality that is characterized by a blocked CHOANAE, the opening between the nose and the NASOPHARYNX. Blockage can be unilateral or bilateral; bony or membranous.

Congenital anomaly in which some of the structures of the eye are absent due to incomplete fusion of the fetal intraocular fissure during gestation.

MUCOUS MEMBRANE extending from floor of mouth to the under-surface of the tongue.

A branch of genetics which deals with the genetic variability in individual responses to drugs and drug metabolism (BIOTRANSFORMATION).

Those persons legally qualified by education and training to engage in the practice of pharmacy.

Facilities for the preparation and dispensing of drugs.

An activity in which the organism plunges into water. It includes scuba and bell diving. Diving as natural behavior of animals goes here, as well as diving in decompression experiments with humans or animals.

The narrow passage way that conducts the sound collected by the EAR AURICLE to the TYMPANIC MEMBRANE.

A benign epithelial tumor with a glandular organization.

Cell surface proteins that bind pituitary THYROTROPIN (also named thyroid stimulating hormone or TSH) and trigger intracellular changes of the target cells. TSH receptors are present in the nervous system and on target cells in the thyroid gland. Autoantibodies to TSH receptors are implicated in thyroid diseases such as GRAVES DISEASE and Hashimoto disease (THYROIDITIS, AUTOIMMUNE).

Diabetic ketoacidosis precipitated by thyrotoxicosis. (1/89)

We report two patients with type 1 diabetes mellitus, previously well controlled with good compliance, presenting with unexplained diabetic ketoacidosis. Following initial correction of the metabolic disorder, persisting tachycardia lead to the diagnosis of thyrotoxicosis. In both cases, treatment with propranolol and carbimazole helped in the stabilization of their metabolic states. Although thyrotoxicosis is known to destabilize diabetes control, we can find no reports of it precipitating diabetic ketoacidosis. (+info)

Insulin autoimmune syndrome: a rare cause of hypoglycaemia not to be overlooked. (2/89)

We report the case of a Caucasian patient with insulin autoimmune syndrome (IAS), defined as the association of hypoglycaemic attacks with insulin autoantibodies in individuals not previously treated with exogenous insulin. This rare syndrome (more than 200 published cases) has been reported mainly in Japan. Most affected patients present with other autoimmune disorders, most often Graves' disease. In most cases, insulin autoantibodies appear a few weeks after the beginning of treatment with a drug containing a sulphyldryl group. A significant increase in insulin and C-peptide plasma concentrations and the presence of other antiorgan antibodies are observed. The susceptibility haplotype is present in the Japanese population, which may account for the high frequency of IAS. Spontaneous remission is observed in 80% of cases, with cessation of hypoglycaemic attacks and disappearance of insulin autoantibodies some months after withdrawal of the drug. This rare cause of hypoglycaemia in Caucasian subjects should be considered in aetiologic investigation of spontaneous hypoglycaemia. (+info)

Prevalence of positive anti-neutrophil cytoplasmic antibody (ANCA) in patients receiving anti-thyroid medication. (3/89)

OBJECTIVE: Vasculitis is a rare complication of anti-thyroid medications. There are 32 cases of anti-neutrophil cytoplasmic antibody (ANCA)-positive vasculitis in association with anti-thyroid medication reported in the English literature. The objectives of this study were to assess the frequency of positive ANCA in patients on long-term anti-thyroid medication, and to follow patients prospectively from commencement of medication to determine whether they became ANCA-positive after therapy. DESIGN: Prospectively collected cross-sectional study of two groups of patients: (i) who had received long-term (>18 months) anti-thyroid medication, and (ii) newly diagnosed thyrotoxicosis before commencement of anti-thyroid medication attending clinic between 28 April 1998 and 30 September 1998. Data were collected for age, sex, ethnicity, underlying thyroid disease, medication and duration, and symptomatology. RESULTS: Eight of 30 patients on long-term anti-thyroid medication (26.7%) were ANCA-positive. All ANCA-positive patients were female, seven were taking propylthiouracil (PTU) at the time of testing. ANCA-positive patients had taken PTU for a mean +/- s.d. of 7.9+/-10.2 years, compared with 0.8+/-2.2 years in ANCA-negative patients (Mann-Whitney, P<0.0001). The ten patients with newly diagnosed thyrotoxicosis were ANCA-negative before commencement of carbimazole. One (10%) became ANCA-positive within 8 months of therapy. CONCLUSIONS: In our population, ANCA-positivity in association with long-term anti-thyroid medication is common (26.7%). One patient who was ANCA-negative prior to anti-thyroid therapy has become ANCA-positive. ANCA should be tested in patients receiving long-term anti-thyroid medications, and in patients with adverse reactions. As PTU is more commonly associated with vasculitis and positive ANCA, carbimazole may be the preferred medication for long-term use. Patients with positive ANCA should be followed, and considered for definitive anti-thyroid therapy, to allow cessation of medication. ANCA-positivity may resolve after cessation of anti-thyroid medication. (+info)

Platelet endothelial cell adhesion molecule-1 (PECAM-1) is a target glycoprotein in drug-induced thrombocytopenia. (4/89)

Drug-induced immune thrombocytopenia (DITP) is a serious complication of drug treatment. Previous studies demonstrated that most drug-dependent antibodies (DDAbs) react with the platelet membrane glycoprotein (GP) complexes IIb/IIIa and Ib/IX/V. We analyzed the sera from 5 patients who presented with DITP after intake of carbimazole. Notably, thrombocytopenia induced by carbimazole was relatively mild in comparison to patients with DITP induced by quinidine. The sera reacted with platelets in an immunoassay on addition of the drug. In immunoprecipitation experiments with biotin-labeled platelets and endothelial cells, reactivity with the platelet endothelial cell adhesion molecule-1 (PECAM-1, CD31) could be demonstrated, whereas neither GPIIb/IIIa nor GPIb/IX was precipitated in the presence of the drug. These results could be confirmed by GP-specific immunoassay (MAIPA) using monoclonal antibodies (mabs) against PECAM-1. In addition, the binding of DDAbs could be abolished by preincubation with soluble recombinant PECAM-1. Carbimazole-dependent antibodies showed similar reactivity with platelets carrying the Leu(125) and Val(125) PECAM-1 isoforms, indicating that this polymorphic structure, which is located in the first extracellular domain, is not responsible for the epitope formation. Binding studies with biotin-labeled mutants of PECAM-1 and analysis of sera with mabs against different epitopes on PECAM-1 in MAIPA assay suggested that carbimazole-dependent antibodies prominently bound to the second immunoglobulin homology domain of the molecule. Analysis of 20 sera from patients with quinidine-induced thrombocytopenia by MAIPA assay revealed evidence that DDAbs against PECAM-1 are involved in addition to anti-GPIb/IX and anti-GPIIb/IIIa. We conclude that PECAM-1 is an important target GP in DITP. (Blood. 2000;96:1409-1414) (+info)

Successful treatment of amiodarone-induced thyrotoxicosis. (5/89)

BACKGROUND: Amiodarone-induced thyrotoxicosis (AIT) is a difficult management problem about which there are little published data. We examined whether continuing amiodarone or differentiating AIT into 2 subtypes affected outcome. METHODS AND RESULTS: The type and duration of antithyroid treatment and response were recorded in a consecutive series of 28 cases. Comparisons were made between those in whom amiodarone either was continued or stopped and between those with either possible type 1 or type 2 AIT. Of the 28 cases, 5 had spontaneous resolution of AIT; 23 received carbimazole (CBZ) alone as first-line therapy. Eleven achieved long-term euthyroidism off CBZ or on a maintenance dose. Five became hypothyroid and required long-term thyroxine. Five relapsed after stopping CBZ treatment and were rendered euthyroid with either long-term CBZ (n=3) or radioiodine (n=2). Four were intolerant of CBZ and received propylthiouracil (PTU), with good effect in 3. One was resistant to thionamide alone (CBZ then PTU) and responded to adjunctive steroids. No difference in presentation or outcome was noted between those in whom amiodarone was continued or stopped or between possible type 1 or type 2 AIT. CONCLUSIONS: Continuing amiodarone has no adverse influence on response to treatment of AIT. First-line therapy with a thionamide alone is appropriate in iodine-replete areas, thus avoiding potential complications of other drugs. Differentiating between 2 possible types of AIT does not influence management or outcome. (+info)

Jaundice due to carbimazole. (6/89)

On three occasions, a 63 year old housewife with hyperthyroidism developed a reaction which included fever, pruritus, malaise, and, on one occasion, jaundice one to 17 days after taking carbimazole. Challenge with carbimazole was followed within 12 hours by abdominal pain, pruritus, and increased serum transaminase levels. Light microscopy of a liver biopsy showed increase of portal zone cellularity over the control and the electron microscopy revealed fine structural changes compatible with drug-related liver injury. (+info)

The influence of preoperative drug treatment on the extent of hyperplasia of the thymus in primary thyrotoxicosis. (7/89)

Thymic biopsies taken from women at the beginning of the operation of subtotal thyroidectomy were studied by the point-counting histometric technique. In all patients with primary thyrotoxicosis, the thymus is hyperplastic. After pretreatment with antithyroid drugs, the pattern of thymic involution with age is similar to, but at higher levels, than that in control groups of patients with non-toxic goitre in whom there is no evidence of immunological abnormality. By contrast, after propranolol pretreatment very little age involution is seen. The differences in the appearance of the thymus in female primary thyrotoxixosis patients prepared for operation with different drug treatment regimes are probably related to the pharmacological actions of the drugs and may indicate an interaction between primary immunological and secondary endocrinological factors in the disease process. (+info)

Acute pancreatitis, hepatic cholestasis, and erythema nodosum induced by carbimazole treatment for Graves' disease. (8/89)

A 33-year old female was diagnosed as Graves' disease and started on carbimazole. One month later when she was already euthyroid only on carbimazole therapy, she developed acute pancreatitis associated with mild cholestatic hepatitis and erythema nodosum. Carbimazole therapy was interrupted, pancreatic and liver function gradually improved and became normalized two weeks later. Other potential etiological causes of acute pancreatitis, hepatitis and erythema nodosum were excluded. Rechallenge with a single dose of carbimazole led to a new episode of acute pancreatitis and cholestatic hepatitis one day later. The appearance of different hypersensitivity reactions including pancreatitis, hepatitis and erythema nodosum, together with the observation that the interval between drug intake and onset of symptoms became shorter with repeated exposure to carbimazole, point to an immune-mediated mechanism. Carbimazole has to be added to the list of drugs capable of inducing acute pancreatitis, and should be emphasized the need to discontinue this medication as soon as there is evidence of pancreatic dysfunction. (+info)

It can also seen with exposure to methimazole and carbimazole in utero. This dermatological manifestation has been linked to ...

https://en.wikipedia.org/wiki/Aplasia_cutis_congenita

Members of the thioamide group include methimazole, carbimazole (converted in vivo to methimazole), and propylthiouracil. ...

https://en.wikipedia.org/wiki/Thioamide

Carbimazole is an alternative depending on local availability *^ For use when alternative first-line treatment is not ... Carbimazole is an alternative depending on local availability *^ For use when alternative first-line treatment is not ...

https://en.wikipedia.org/wiki/List_of_World_Health_Organization_Essential_Medicines

The main antithyroid drugs are carbimazole (in the UK), methimazole (in the US), and propylthiouracil/PTU. These drugs block ...

https://en.wikipedia.org/wiki/Graves'_disease

The main antithyroid drugs are carbimazole (in the UK), methimazole (in the US), and propylthiouracil/PTU. A less common ...

https://en.wikipedia.org/wiki/Antithyroid_agent

... carbimazole and methimazole. Radioactive iodine-131 can be used to destroy thyroid tissue. Radioactive iodine is selectively ...

https://en.wikipedia.org/wiki/Thyroid

... is recommended but this should be changed back to carbimazole after the first trimester. This is because carbimazole can rarely ... Carbimazole and PTU are both secreted in breast milk but evidence suggests that antithyroid drugs are safe during lactation. ... If a woman is already receiving carbimazole, a change to propylthiouracil (PTU) ...

https://en.wikipedia.org/wiki/Thyroid_disease_in_pregnancy

Hyperthyroidism caused by Graves' disease may be treated with the thioamide drugs propylthiouracil, carbimazole or methimazole ...

https://en.wikipedia.org/wiki/Thyroid_disease

Carbimazole is only available as a once daily tablet. Methimazole is available as an oral solution, a tablet, and compounded as ... Thyrostatics (antithyroid drugs) are drugs that inhibit the production of thyroid hormones, such as carbimazole (used in the UK ... Drugs used to help manage the symptoms of hyperthyroidism are methimazole and carbimazole. Drug therapy is the least expensive ...

https://en.m.wikipedia.org/wiki/Hyperthyroidism

Thyrostatics (antithyroid drugs) are drugs that inhibit the production of thyroid hormones, such as carbimazole (used in the UK ...

https://en.wikipedia.org/wiki/Hyperthyroidism

... carbimazole). They can be used in the treatment of patients with severe thyrotoxicosis (thyroid storm) and significant ...

https://en.wikipedia.org/wiki/Iopanoic_acid

... carbimazole (INN) carbinoxamine (INN) Carbiset Tablet Carbocaine with Neo-Cobefrin Carbocaine carbocisteine (INN) carbocloral ( ...

https://en.wikipedia.org/wiki/List_of_drugs:_C-Ca

... carbimazole MeSH D03.383.129.308.130 --- cimetidine MeSH D03.383.129.308.175 --- clotrimazole MeSH D03.383.129.308.207 --- ...

https://en.wikipedia.org/wiki/List_of_MeSH_codes_(D03)

... carbimazole, methimazole, and propylthiouracil), antibiotics (penicillin, chloramphenicol and co-trimoxazole), ACE inhibitors ( ...

https://en.wikipedia.org/wiki/Agranulocytosis

H03BA01 Methylthiouracil H03BA02 Propylthiouracil H03BA03 Benzylthiouracil H03BB01 Carbimazole H03BB02 Thiamazole H03BB52 ...

https://en.wikipedia.org/wiki/ATC_code_H03

... is used to treat hyperthyroidism. Carbimazole is a pro-drug as after absorption it is converted to the active form ... Whilst rashes and pruritus are common, these can often be treated with antihistamines without stopping the carbimazole. For ... Some azole drugs may disrupt estrogen production in pregnancy, affecting pregnancy outcome.[verification needed] Carbimazole ... with the possibility of changing to Carbimazole for the second and third trimesters. ...

https://en.wikipedia.org/wiki/Carbimazole

... is typically used to treat hypothyroidism,[9] and is the treatment of choice for people with hypothyroidism,[10] who often require lifelong thyroid hormone therapy.[11] It may also be used to treat goiter via its ability to lower thyroid-stimulating hormone (TSH), a hormone that is considered goiter-inducing.[12][13] Levothyroxine is also used as interventional therapy in people with nodular thyroid disease or thyroid cancer to suppress thyroid-stimulating hormone (TSH) secretion.[14] A subset of people with hypothyroidism treated with an appropriate dose of levothyroxine will describe continuing symptoms despite TSH levels in the normal range.[11] In these people, further laboratory and clinical evaluation is warranted as they may have another cause for their symptoms.[11] Furthermore, it is important to review their medications and possible dietary supplements as several medications can affect thyroid hormone levels.[11] Levothyroxine is also used to treat subclinical ...

https://en.wikipedia.org/wiki/Levothyroxine

The earliest oral treatment for hypothyroidism consisted of thyroid extract. George Redmayne Murray of the United Kingdom first described treatment of myxedema with thyroid extract in 1891, and published a description of long-term successful treatment (28 years) of a patient with myxedema (severe hypothyroidism) in 1920[17] His treatment was quickly adopted in North America and Europe. The first recorded American use dates to 1891 by a woman who was still taking it 52 years later at 84 years of age [18] Desiccated thyroid extract is prepared from pig thyroid glands. The glands are dried (desiccated), ground to powder, combined with binder chemicals, and pressed into pills. This was a new use for parts that were previously unwanted slaughterhouse offal, and Armour and Company, the dominant American meatpacker in the 20th century, supplied the best-known brand of thyroid extract.[citation needed] Replacement by thyroid extract in hypothyroidism was one of the most effective treatments of any ...

https://en.wikipedia.org/wiki/Desiccated_thyroid_extract

Compounds such as goitrin, carbimazole, methimazole, propylthiouracil can reduce thyroid hormone production by interfering with ...

https://en.wikipedia.org/wiki/Thyroid_hormone

Long-term management of hyperthyroidism may include drugs that suppress thyroid function such as propylthiouracil, carbimazole ...

https://en.wikipedia.org/wiki/Thyroid_isthmus

An association between carbimazole and propylthiouracil and the development of a lupus- like syndrome have been previously ... Four months after starting carbimazole (40 mg, dropping to 20 mg daily after six weeks) she developed left sided otalgia and ... It is interesting that the symptoms we ascribe to carbimazole hypersensitivity developed after a prolonged period of treatment ... We present a previously undocumented complication of carbimazole treatment occurring in a 28 year old woman with Graves ...

http://www.bmj.com/content/309/6959/929.2

Carbimazole ≥98% (HPLC); CAS Number: 22232-54-8; EC Number: 244-854-4; Synonym: 1-Ethoxycarbonyl-3-methyl-2-thio-4-imidazoline ... Carbimazole is a pro-drug of methimazole that is an inhibitor of thyroid enzyme thyroperoxidase. Carbimazole is used to treat ... Carbimazole ≥98% (HPLC) Synonym: 1-. Ethoxycarbonyl-. 3-. methyl-. 2-. thio-. 4-. imidazoline, 1-. Methyl-. 3-. carbethoxy-. 2- ...

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Results:The median dose of carbimazole was 10 mg (range 10 to 15 mg) and 15 mg (5 to 25 mg) once daily after 3 and 53 weeks, ... Objective:Evaluation of efficacy and safety of a novel controlled-release formulation of carbimazole in feline hyperthyroidism. ... Clinical Significance:Once daily administration of controlled-release carbimazole tablets was effective and had expected ... Clinical efficacy and safety of a once-daily formulation of carbimazole in cats with hyperthyroidism. Authors. *. R. Frénais,. ...

http://onlinelibrary.wiley.com/doi/10.1111/j.1748-5827.2009.00772.x/full

Carbimazole-induced acute cholestatic hepatitis in patient with Graves disease. MS Rathi & EG Ward. ... Thyrostatic medications (carbimazole or propylthiouracil) as first line therapy to treat hyperthyroidism can rarely cause ... We report a patient who developed cholestatic jaundice after carbimazole therapy.. Case report: A 32-year-old woman presented ... Liver function test showed ALT 120 IU/l (,40), ALP 750 iu/l (70 300). Drug induced hepatitis was suspected hence carbimazole ...

https://www.endocrine-abstracts.org/ea/0019/ea0019P348.htm

To determine the spectrum of clinical presentations of hyperthyroidism in cats and response to carbimazole ... Carbimazole therapy was found to decrease the prevalence of almost all clinical abnormalities in 14 cats and side-effects were ... Therapy with carbimazole was instituted and revisits were scheduled 2, 6 and 13 weeks after diagnosis. The cats were physically ... Feline Hyperthyroidism: Spectrum of Clinical Presentions and Response to Carbimazole Therapy. by Michael R. Broome, DVM, MS, ...

https://avmi.net/information/hyperthyroid-hints/feline-hyperthyroidism-spectrum-of-clinical-presentions-and-response-to-carbimazole-therapy/

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Posted in Endocrinology , Tagged carbimazole, goitre, Graves disease, hyperthyroidism, iodine, thyrotoxicosis Search. Recent ... Tag Archives: carbimazole. Hyperthyroidism. Posted on January 22, 2017. by davorpp88 http://media.blubrry.com/medconversations/ ...

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Carbimazole - administration & dosage - adverse effects. en_HK. dc.subject.mesh. Jaundice, Obstructive - chemically induced. en ... Article: Cholestatic jaundice caused by sequential carbimazole and propylthiouracil treatment for thyrotoxicosis. *Show simple ... Cholestatic jaundice caused by sequential carbimazole and propylthiouracil treatment for thyrotoxicosis. Authors. Chan, AOONg, ... He had previously received carbimazole but had developed an urticarial skin rash after 2 weeks of treatment. The patient ...

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... carbimazole): NeoMercazole is an anti-thyroid agent. It is indicated in all conditions where reduction of thyroid function is ... Pharmacology: Pharmacodynamics: Carbimazole is a thyroid reducing agent.. Pharmacokinetics: Carbimazole is rapidly metabolised ... There is a risk of cross-allergy between carbimazole, thiamazole and propylthiouracil.. Carbimazole may cause white cell ... to transplacental exposure to carbimazole and methimazole cannot be excluded. Therefore, the use of carbimazole in non-pregnant ...

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5] G. Wilcox, R. Wong, P.J. Elliott, D.J. Topliss and J.R. Sockigt: "Recovery from Carbimazole-Induced Aplastic Anaemia", Int. ... Carbimazole should always be discontinued if neutropenia occurs but this case demonstrates that in exceptional circumstances ... She required 80 mg of carbimazole daily. The patient declined radio-iodine treatment because she had a little child and wished ... Simultaneous treatment with carbimazole and granulocyte-colony stimulating factor in a patient with thyrotoxicosis and ...

http://psjd.icm.edu.pl/psjd/element/bwmeta1.element.-psjd-doi-10_2478_s11536-006-0036-5

Are thyroid medications (levothyroxine and carbimazole) safe in pregnancy and when breastfeeding? ... Carbimazole is the drug used during the rest of pregnancy. You should discuss your plans for pregnancy with your ... If you are being treated with antithyroid drugs (e.g. carbimazole or Prophylthiouracil (PTU)) and you havent already discussed ... Im hyperthyroid and take carbimazole. Is this okay during pregnancy?. For hyperthyroid (overactive) patients Propylthiouracil ...

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Carbimazole is used to treat hyperthyroidism. Carbimazole is a pro-drug as after absorption it is converted to the active form ... Whilst rashes and pruritus are common, these can often be treated with antihistamines without stopping the carbimazole. For ... Some azole drugs may disrupt estrogen production in pregnancy, affecting pregnancy outcome.[verification needed] Carbimazole ... with the possibility of changing to Carbimazole for the second and third trimesters. ...

https://en.wikipedia.org/wiki/Carbimazole

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Carbimazole is used to treat hyperthyroidism. (sigmaaldrich.com)
Thyrostatic medications (carbimazole or propylthiouracil) as first line therapy to treat hyperthyroidism can rarely cause potentially fatal hepatotoxicity. (endocrine-abstracts.org)
OBJECTIVE: To determine the spectrum of clinical presentations of hyperthyroidism in cats and response to carbimazole therapy by analysis of historical, clinical and laboratory data. (avmi.net)

Drug induced hepatitis was suspected hence carbimazole was discontinued and propylthiouracil was started. (endocrine-abstracts.org)
Carbimazole causes intracanalicular cholestasis whilst propylthiouracil causes hepatocellular injury. (endocrine-abstracts.org)
There is a risk of cross-allergy between carbimazole, thiamazole and propylthiouracil. (mims.com)

Carbimazole is a pro-drug of methimazole that is an inhibitor of thyroid enzyme thyroperoxidase. (sigmaaldrich.com)
Carbimazole is rapidly metabolised to methimazole. (mims.com)
The mean peak plasma concentration of methimazole is reported to occur one hour after a single dose of carbimazole. (mims.com)

Graves thyrotoxicosis was diagnosed and carbimazole 40 mg a day was initiated. (endocrine-abstracts.org)

Vidalta is very cost effective and only needs to be given once daily as the tablets are formulated to release the active, carbimazole, over twenty four hours. (newsteadvets.co.nz)

Carbimazole is an antithyroid agent that decreases the uptake and concentration of inorganic iodine by thyroid, it also reduces the formation of di-iodotyrosine and thyroxine. (zotezo.com)

Carbimazole is a thyroid reducing agent. (mims.com)
It is often treated with anti-thyroid drugs (carbimazole), iodine and beta blockers. (oncohemakey.com)

Each NeoMercazole 5 tablet contains carbimazole Ph. (mims.com)
NeoMercazole is contraindicated in patients with a previous history of adverse reactions to carbimazole or to any of the excipients in the composition. (mims.com)

As fatal cases of agranulocytosis with carbimazole have been reported and early treatment of agranulocytosis is essential, it is important that patients should always be warned about the onset of sore throats, bruising or bleeding, mouth ulcers, fever, malaise and should be instructed to stop the drug and to seek medical advice immediately. (mims.com)
Carbimazole may cause white cell disoders such as neutropenia and agranulocytosis, which may be fatal if treatment with carbimazole is not stopped promptly. (mims.com)

What is the optimal medicament dose of carbimazole to treat your medical condition in line with your genomic makeup? (genomicmedicineuk.com)
Knowing the optimal dose of carbimazole to treat your medical condition, and whether carbimazole is safe to treat your medical condition will empower your doctor to adapt your treatment plan to suit you better. (genomicmedicineuk.com)

We report a patient who developed cholestatic jaundice after carbimazole therapy. (endocrine-abstracts.org)
Our patient had predominantly cholestatic hepatitis, which is consistent with reported cases of carbimazole-induced hepatic damage. (endocrine-abstracts.org)

Therapy with carbimazole was instituted and revisits were scheduled 2, 6 and 13 weeks after diagnosis. (avmi.net)
Cats with underlying renal disease were managed by alterations or cessation of carbimazole therapy. (avmi.net)
Fourteen cats treated with carbimazole and monitored for 13 weeks responded favourably to therapy. (avmi.net)
Cats with underlying renal disease that became apparent during the study, responded well to alterations or cessation of carbimazole therapy. (avmi.net)
Carbimazole therapy was found to decrease the prevalence of almost all clinical abnormalities in 14 cats and side-effects were minor and uncommon. (avmi.net)

This genomic DNA test assesses your genetic response to medical treatment with carbimazole. (genomicmedicineuk.com)
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Can the treatment to your medical condition with carbimazole pose a safety concern to your health because of your genetic background? (genomicmedicineuk.com)

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This pharmacogenomic test is recommended if you have been prescribed or will be prescribed carbimazole to treat your medical condition. (genomicmedicineuk.com)

As with T4 supplementation for hypo-conditions, TSH levels in patients with hyper-conditions may take up to 12 weeks to respond to carbimazole. (oncohemakey.com)

Vasculitis is uncommon, but ANCA positivity is reported more in propylthiouracil and rarely with carbimazole or methimazole (1). (thefreedictionary.com)

Serum thyroxine (T4), triiodothyronine (T3) and thyrotropin (T.S.H.) were measured in 32 hyperthyroid patients treated with carbimazole. (unboundmedicine.com)

I take Carbimazole to bring my thyroid level to normal and this medication is very helpful. (aviatik-sim.de)

I have had specialist appointments for the last 4 months (complete with blood tests) but in my last appointment (last friday) my T4 (i think) was at 22 which apparently is still apparently too high and so the doctor took me off propranolol and upped my carbimazole to 40mg. (patient.info)

There is also a risk of acute pancreatitis "following treatment with carbimazole/thiamazole", the MHRA added in its drug safety alert . (chemistanddruggist.co.uk)
We found no RCTs comparing antithyroid drugs (carbimazole, propylthiouracil, or thiamazole) with each other. (aafp.org)
There is a risk of cross-allergy between carbimazole, thiamazole and propylthiouracil. (mims.com)

You can take carbimazole before or after meals. (patient.info)
I'm hyperthyroid and take carbimazole. (btf-thyroid.org)

Purchased Neo-Mercazole tablet Generic Drugs of Carbimazole online at lowest price. (safegenericpharmacy.com)

Carbimazole prescribed to people who have an overactive thyroid What is Carbimazole? (mcdonaldranches.com)
I was diagnosed with overactive thyroid and prescribed Carbimazole. (axappphealthcare.co.uk)
Carbimazole is a medicine used in the treatment of overactive thyroid. (axappphealthcare.co.uk)

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We aimed to investigate whether carbimazole therapy was also associated with increased risk of ANCA. (ox.ac.uk)

69) Her systemic and ocular diseases were treated successfully with oral carbimazole and selenium, respectively. (thefreedictionary.com)

Symptoms are initially controlled by anti-thyroid drugs (carbimazole or propylthiouracil) and beta blockers. (breastfeedingnetwork.org.uk)

The thyrocab (carbimazole) is for thyroid. (medhelp.org)

hi, i too had a very high t4 rading 83 ( normal range 11 - 24) was put on 40mg carbimazole then went under. (patient.info)

Carbimazole causes intracanalicular cholestasis whilst propylthiouracil causes hepatocellular injury. (endocrine-abstracts.org)

Carbimazole hypersensitivity was diagnosed, and she started taking propylthiouracil instead (100 mg twice daily). (bmj.com)

Our patient had predominantly cholestatic hepatitis, which is consistent with reported cases of carbimazole-induced hepatic damage. (endocrine-abstracts.org)

Endocrinologist gave me prescription of carbimazole 5 mg twice a day but soon after taking that med I notice my throat is swollen and and hoarseness of voice and eyes are painful. (endocrineweb.com)

Rarely, carbimazole can cause a decrease in white blood cell levels, which can make you vulnerable to infection. (axappphealthcare.co.uk)

The therapeutic efficacy of Carbimazole can be decreased when used in combination with Vatalanib. (drugbank.ca)

About Carbimazole Thyroid peroxidase inhibitors(thioamide),Sulfur-containing imidazole derivatives, Antithyroid. (rosheta.com)

Connell, J. , Mccruden, D. , Hilditch, T. and Alexander, W. (1985) Effect of Carbimazole Pretreatment on the Kinetics of Therapeutic Radioiodine (I-131). (gla.ac.uk)

Whilst rashes and pruritus are common, these can often be treated with antihistamines without stopping the carbimazole. (wikipedia.org)
Also, how do you keep your weight down whilst on Carbimazole? (endocrineweb.com)

Fifty percent of pre-existing GD patients who were thyrotoxic at first presentation were rendered euthyroid at delivery with the appropriate use of carbimazole only (Table 3). (thefreedictionary.com)

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Diabetic ketoacidosis precipitated by thyrotoxicosis. (1/89)

Insulin autoimmune syndrome: a rare cause of hypoglycaemia not to be overlooked. (2/89)

Prevalence of positive anti-neutrophil cytoplasmic antibody (ANCA) in patients receiving anti-thyroid medication. (3/89)

Platelet endothelial cell adhesion molecule-1 (PECAM-1) is a target glycoprotein in drug-induced thrombocytopenia. (4/89)

Successful treatment of amiodarone-induced thyrotoxicosis. (5/89)

Jaundice due to carbimazole. (6/89)

The influence of preoperative drug treatment on the extent of hyperplasia of the thymus in primary thyrotoxicosis. (7/89)

Acute pancreatitis, hepatic cholestasis, and erythema nodosum induced by carbimazole treatment for Graves' disease. (8/89)

Carbimazole

Antithyroid Agents

Thyrotoxicosis

Hyperthyroidism

Graves Disease

Methimazole

Propylthiouracil

Thyroxine

Erythema Nodosum

Thyroid Gland

Neuroma, Acoustic

Neuroma

Audiometry

Imidazoline Receptors

Delayed-Action Preparations

Hypothyroidism

Cholestasis, Intrahepatic

Exophthalmos

Choanal Atresia

Coloboma

Lingual Frenum

Pharmacogenetics

Pharmacists

Pharmacies

Diving

Ear Canal

Adenoma

Receptors, Thyrotropin

Diabetic ketoacidosis precipitated by thyrotoxicosis. (1/89)

Insulin autoimmune syndrome: a rare cause of hypoglycaemia not to be overlooked. (2/89)

Prevalence of positive anti-neutrophil cytoplasmic antibody (ANCA) in patients receiving anti-thyroid medication. (3/89)

Platelet endothelial cell adhesion molecule-1 (PECAM-1) is a target glycoprotein in drug-induced thrombocytopenia. (4/89)

Successful treatment of amiodarone-induced thyrotoxicosis. (5/89)

Jaundice due to carbimazole. (6/89)

The influence of preoperative drug treatment on the extent of hyperplasia of the thymus in primary thyrotoxicosis. (7/89)

Acute pancreatitis, hepatic cholestasis, and erythema nodosum induced by carbimazole treatment for Graves' disease. (8/89)

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