Campylobacter jejuni
Campylobacter
Campylobacter fetus
Campylobacter coli
Guillain-Barre Syndrome
Chickens
Poultry Diseases
Campylobacter lari
Flagellin
Feces
Food Microbiology
Polyradiculoneuropathy
Poultry
Bacterial Typing Techniques
Diarrhea
Miller Fisher Syndrome
Cecum
Serotyping
Gastroenteritis
Flagella
Cloaca
Arcobacter
Molecular Sequence Data
Colony Count, Microbial
Electrophoresis, Gel, Pulsed-Field
Drug Resistance, Bacterial
Meat
Culture Media
Sequence Analysis, DNA
Animals, Domestic
Bacterial Adhesion
Erythromycin
Microbial Sensitivity Tests
Foodborne Diseases
Polymerase Chain Reaction
Multilocus Sequence Typing
Molecular Mimicry
Food Contamination
Campylobacter hyointestinalis
Bacterial Shedding
Virulence
G(M1) Ganglioside
Species Specificity
Gangliosides
Abortion, Septic
Campylobacter rectus
DNA Fingerprinting
Gene Expression Regulation, Bacterial
Polymorphism, Restriction Fragment Length
Microbial Viability
Cattle
Food Handling
Intestines
Truncated Hemoglobins
Water Microbiology
Milk
Bahrain
Virulence Factors
Lipopolysaccharides
Anti-Infective Agents
Disease Reservoirs
Turkeys
Salmonella
Disease Outbreaks
Base Sequence
Cytotoxins
Nalidixic Acid
Drug Resistance, Microbial
Animal Husbandry
Genotype
Cloning and expression of the dnaK gene of Campylobacter jejuni and antigenicity of heat shock protein 70. (1/1435)
Campylobacter jejuni is a leading cause of infectious diarrhea throughout the world. In addition, there is growing evidence that Guillain-Barre syndrome, an inflammatory demyelinating disease of the peripheral nervous system, is frequently preceded by C. jejuni infection. In the present study, the hrcA-grpE-dnaK gene cluster of C. jejuni was cloned and sequenced. The dnaK gene consists of an open reading frame of 1,869 bp and encodes a protein with a high degree of homology to other bacterial 70-kDa heat shock proteins (HSPs). The overall percentages of identity to the HSP70 proteins of Helicobacter pylori, Borrelia burgdorferi, Chlamydia trachomatis, and Bacillus subtilis were calculated to be 78.1, 60.5, 57.2, and 53. 8%, respectively. Regions similar to the Escherichia coli sigma70 promoter consensus sequence and to a cis-acting regulatory element (CIRCE) are located upstream of the hrcA gene. Following heat shock, a rapid increase of dnaK mRNA was detectable, which reached its maximum after 20 to 30 min. A 6-His-tagged recombinant DnaK protein (rCjDnaK-His) was generated in E. coli, after cloning of the dnaK coding region into pET-22b(+), and purified by affinity and gel filtration chromatography. Antibody responses to rCjDnaK-His were significantly elevated, compared to those of healthy individuals, in about one-third of the serum specimens obtained from C. jejuni enteritis patients. (+info)Physiological characterization of viable-but-nonculturable Campylobacter jejuni cells. (2/1435)
Campylobacter jejuni is a pathogenic, microaerophilic, gram-negative, mesophilic bacterium. Three strains isolated from humans with enteric campylobacteriosis were able to survive at high population levels (10(7) cells ml-1) as viable-but-nonculturable (VBNC) forms in microcosm water. The VBNC forms of the three C. jejuni strains were enumerated and characterized by using 5-cyano-2,3-ditolyl tetrazolium chloride-4',6-diamino-2-phenylindole staining. Cellular volume, adenylate energy charge, internal pH, intracellular potassium concentration, and membrane potential values were determined in stationary-phase cell suspensions after 48 h of culture on Columbia agar and after 1 to 30 days of incubation in microcosm water and compared. A notable increase in cell volume was observed with the VBNC state; the average cell volumes were 1.73 microliter mg of protein-1 for the culturable form and 10.96 microliter mg of protein-1 after 30 days of incubation in microcosm water. Both the internal potassium content and the membrane potential were significantly lower in the VBNC state than in the culturable state. Culturable cells were able to maintain a difference of 0.6 to 0.9 pH unit between the internal and external pH values; with VBNC cells this difference decreased progressively with time of incubation in microcosm water. Measurements of the cellular adenylate nucleotide concentrations revealed that the cells had a low adenylate energy charge (0.66 to 0.26) after 1 day of incubation in microcosm water, and AMP was the only nucleotide detected in the three strains after 30 days of incubation in microcosm water. (+info)Evaluation of accuracy and repeatability of identification of food-borne pathogens by automated bacterial identification systems. (3/1435)
The performances of five automated microbial identification systems, relative to that of a reference identification system, for their ability to accurately and repeatedly identify six common food-borne pathogens were assessed. The systems assessed were the MicroLog system (Biolog Inc., Hayward, Calif.), the Microbial Identification System (MIS; MIDI Inc., Newark, Del.), the VITEK system (bioMerieux Vitek, Hazelwood, Mo.), the MicroScan WalkAway 40 system (Dade-MicroScan International, West Sacramento, Calif.), and the Replianalyzer system (Oxoid Inc., Nepean, Ontario, Canada). The sensitivities and specificities of these systems for the identification of food-borne isolates of Bacillus cereus, Campylobacter jejuni, Listeria monocytogenes, Staphylococcus aureus, Salmonella spp., and verotoxigenic Escherichia coli were determined with 40 reference positive isolates and 40 reference negative isolates for each pathogen. The sensitivities of these systems for the identification of these pathogens ranged from 42.5 to 100%, and the specificities of these systems for the identification of these pathogens ranged from 32.5 to 100%. Some of the systems had difficulty correctly identifying the reference isolates when the results were compared to those from the reference identification tests. The sensitivity of MIS for the identification of S. aureus, B. cereus, E. coli, and C. jejuni, for example, ranged from 47.5 to 72. 5%. The sensitivity of the Microlog system for the identification of E. coli was 72.5%, and the sensitivity of the VITEK system for the identification of B. cereus was 42.5%. The specificities of four of the five systems for the identification of all of the species tested with the available databases were greater than or equal to 97.5%; the exception was MIS for the identification of C. jejuni, which displayed a specificity of 32.5% when it was tested with reference negative isolates including Campylobacter coli and other Campylobacter species. All systems had >80% sensitivities for the identification of Salmonella species and Listeria species at the genus level. The repeatability of these systems for the identification of test isolates ranged from 30 to 100%. Not all systems included all six pathogens in their databases; thus, some species could not be tested with all systems. The choice of automated microbial identification system for the identification of a food-borne pathogen would depend on the availability of identification libraries within the systems and the performance of the systems for the identification of the pathogen. (+info)Cloning, mutation and distribution of a putative lipopolysaccharide biosynthesis locus in Campylobacter jejuni. (4/1435)
A region encoding ORFs with homology to known lipopolysaccharide (LPS) biosynthesis genes was isolated from two strains of Campylobacter jejuni. One of the strains produces LPS, but the second strain is reported to produce only lipooligosaccharide (LOS) and therefore lacks the O-chain. The two strains shared six predicted ORFs, but an additional ORF, orfE, of unknown function was identified in the LOS-producing strain. Mutation of the shared wbeE (rfbE) homologue (orfF) or deletion of five of the seven genes reduced core reactivity with specific antiserum without affecting O-chain production. Mutation of either the capD homologue (orfG) or the unique orfE had no detectable effect on LOS or LPS production. The presence or absence of orfE in 36 isolates of C. jejuni did not correlate with LOS/LPS phenotype or serotype. However, after insertion of orfE into a LPS-producing orfE-negative strain the O-chain ladder was no longer detectable on Western blots. We were not able to disrupt the wbaP (rfbP) homologue (orfC) in C jejuni. (+info)Campylobacter jejuni--an emerging foodborne pathogen. (5/1435)
Campylobacter jejuni is the most commonly reported bacterial cause of foodborne infection in the United States. Adding to the human and economic costs are chronic sequelae associated with C. jejuni infection--Guillian-Barre syndrome and reactive arthritis. In addition, an increasing proportion of human infections caused by C. jejuni are resistant to antimicrobial therapy. Mishandling of raw poultry and consumption of undercooked poultry are the major risk factors for human campylobacteriosis. Efforts to prevent human illness are needed throughout each link in the food chain. (+info)The risk of Guillain-Barre syndrome following infection with Campylobacter jejuni. (6/1435)
To estimate the incidence of Guillain-Barre syndrome (GBS) following Campylobacter jejuni infection (CI) we studied three populations where outbreaks of CI had occurred involving an estimated 8000 cases. No case of GBS was detected in the 6 months following the outbreaks in the local populations. The point estimate for the risk of GBS following CI estimated in this study was 0 in 8000 (95% confidence interval 0-3). (+info)Detection of cytolethal distending toxin activity and cdt genes in Campylobacter spp. isolated from chicken carcasses. (7/1435)
This study was designed to determine whether isolates from chicken carcasses, the primary source of Campylobacter jejuni and Campylobacter coli in human infections, commonly carry the cdt genes and also whether active cytolethal distending toxin (CDT) is produced by these isolates. Campylobacter spp. were isolated from all 91 fresh chicken carcasses purchased from local supermarkets. Campylobacter spp. were identified on the basis of both biochemical and PCR tests. Of the 105 isolates, 70 (67%) were identified as C. jejuni, and 35 (33%) were identified as C. coli. PCR tests amplified portions of the cdt genes from all 105 isolates. Restriction analysis of PCR products indicated that there appeared to be species-specific differences between the C. jejuni and C. coli cdt genes, but that the restriction patterns of the cdt genes within strains of the same species were almost invariant. Quantitation of active CDT levels produced by the isolates indicated that all C. jejuni strains except four (94%) had mean CDT titers greater than 100. Only one C. jejuni strain appeared to produce no active CDT. C. coli isolates produced little or no toxin. These results confirm the high rate of Campylobacter sp. contamination of fresh chicken carcasses and indicate that cdt genes may be universally present in C. jejuni and C. coli isolates from chicken carcasses. (+info)Detection of small numbers of Campylobacter jejuni and Campylobacter coli cells in environmental water, sewage, and food samples by a seminested PCR assay. (8/1435)
A rapid and sensitive assay was developed for detection of small numbers of Campylobacter jejuni and Campylobacter coli cells in environmental water, sewage, and food samples. Water and sewage samples were filtered, and the filters were enriched overnight in a nonselective medium. The enrichment cultures were prepared for PCR by a rapid and simple procedure consisting of centrifugation, proteinase K treatment, and boiling. A seminested PCR based on specific amplification of the intergenic sequence between the two Campylobacter flagellin genes, flaA and flaB, was performed, and the PCR products were visualized by agarose gel electrophoresis. The assay allowed us to detect 3 to 15 CFU of C. jejuni per 100 ml in water samples containing a background flora consisting of up to 8, 700 heterotrophic organisms per ml and 10,000 CFU of coliform bacteria per 100 ml. Dilution of the enriched cultures 1:10 with sterile broth prior to the PCR was sometimes necessary to obtain positive results. The assay was also conducted with food samples analyzed with or without overnight enrichment. As few as +info)Symptoms of campylobacter infections include:
* Diarrhea (often bloody)
* Fever
* Abdominal pain and cramping
* Nausea and vomiting
* Headache
* Fatigue
* Muscle pain
Transmission of campylobacter infections can occur through the fecal-oral route, contaminated food or water, or direct contact with an infected animal or person. Risk factors for developing a campylobacter infection include eating undercooked poultry, unpasteurized dairy products, and untreated water.
Diagnosis of campylobacter infections typically involves a combination of clinical evaluation, laboratory testing, and medical imaging. Laboratory tests may include culture isolation, polymerase chain reaction (PCR), or immunological assays to detect the presence of Campylobacter bacteria.
Treatment of campylobacter infections typically involves antibiotics such as macrolides, fluoroquinolones, and ceftriaxone. In severe cases, hospitalization may be necessary to manage complications such as dehydration, electrolyte imbalances, or sepsis.
Prevention of campylobacter infections includes proper handling and cooking of food, especially poultry, good hygiene practices, and safe water consumption. Vaccines are also being developed to prevent campylobacter infections in animals and humans.
Overall, campylobacter infections can cause a wide range of illnesses, from mild to severe, and proper diagnosis, treatment, and prevention measures are essential to reduce the risk of complications and death.
Symptoms of enteritis may include diarrhea, abdominal pain, fever, nausea, vomiting, and weight loss. In severe cases, the condition can lead to dehydration, electrolyte imbalances, and even death if left untreated.
The diagnosis of enteritis is typically made through a combination of physical examination, medical history, and diagnostic tests such as endoscopy, imaging studies, and laboratory tests (e.g., blood tests, stool cultures). Treatment depends on the underlying cause of the condition and may include antibiotics, anti-inflammatory medications, and supportive care to manage symptoms.
The symptoms of GBS can range from mild to severe and may include:
* Weakness or tingling sensations in the legs, arms, or face
* Muscle weakness that progresses to paralysis
* Loss of reflexes
* Difficulty swallowing or speaking
* Numbness or pain in the hands and feet
* Fatigue and fever
The diagnosis of GBS is based on a combination of symptoms, physical examination findings, and laboratory tests. There is no cure for GBS, but treatment can help manage symptoms and prevent complications. Plasmapheresis, immunoglobulin therapy, and corticosteroids are common treatments used to reduce inflammation and slow the progression of the disease.
GBS is a rare condition that affects about one in 100,000 people per year in the United States. It can affect anyone, but it is more common in children and young adults. The prognosis for GBS varies depending on the severity of the disease, but most people recover fully within a few weeks or months with proper treatment.
In conclusion, Guillain-Barré Syndrome is a rare autoimmune disorder that can cause muscle weakness and paralysis. While there is no cure for GBS, early diagnosis and treatment can help manage symptoms and prevent complications. With proper care, most people with GBS can recover fully within a few weeks or months.
Some common poultry diseases include:
1. Avian influenza (bird flu): A highly contagious viral disease that affects birds and can be transmitted to humans.
2. Newcastle disease: A viral disease that causes respiratory and gastrointestinal symptoms in birds.
3. Infectious bronchitis: A viral disease that causes respiratory symptoms in birds.
4. Marek's disease: A viral disease that affects the nervous system of birds.
5. Coccidiosis: A parasitic disease caused by the Eimeria protozoa, which can cause diarrhea and weight loss in birds.
6. Chicken anemia virus: A viral disease that causes anemia and weakened immune systems in chickens.
7. Fowl pox: A viral disease that causes skin lesions and other symptoms in birds.
8. Avian encephalomyelitis (AE): A viral disease that affects the brain and spinal cord of birds, causing neurological symptoms such as paralysis and death.
9. Mycoplasmosis: A bacterial disease caused by the Mycoplasma bacteria, which can cause respiratory and other symptoms in birds.
10. Aspergillosis: A fungal disease that affects the respiratory system of birds, causing symptoms such as coughing and difficulty breathing.
Poultry diseases can have a significant impact on bird health and productivity, and can also be transmitted to humans in some cases. It is important for poultry farmers and owners to monitor their flocks closely and take steps to prevent the spread of disease, such as providing clean water and feed, maintaining good hygiene, and vaccinating birds against certain diseases.
There are several types of polyradiculoneuropathy, each with its own set of causes and characteristics:
1. Polyneuropathy: This is the most common type of polyradiculoneuropathy and affects multiple nerves throughout the body. It can be caused by a variety of factors, such as diabetes, vitamin deficiencies, alcoholism, and certain medications.
2. Mononeuritis multiplex: This is a condition in which there is damage to multiple nerves that innervate a specific area of the body, such as the legs or arms. It can be caused by various factors, including diabetes, autoimmune disorders, and certain medications.
3. Radiculoneuropathy: This type of polyradiculoneuropathy affects the nerves that originate from the spinal cord and extend to other parts of the body. It can be caused by compression or inflammation of the nerve roots, such as in the case of herniated discs or spinal stenosis.
4. Autonomic neuropathy: This type of polyradiculoneuropathy affects the nerves that control involuntary functions, such as heart rate, blood pressure, and digestion. It can be caused by a variety of factors, including diabetes, vitamin deficiencies, and certain medications.
The symptoms of polyradiculoneuropathy can vary depending on the specific type and severity of the condition. Common symptoms include:
* Weakness or numbness in the affected areas
* Pain or discomfort in the affected areas
* Difficulty walking or maintaining balance
* Difficulty with fine motor skills, such as buttoning a shirt or tying shoelaces
* Digestive problems, such as constipation or diarrhea
* Urinary incontinence or retention
The diagnosis of polyradiculoneuropathy is typically made based on a combination of physical examination findings, medical history, and results of diagnostic tests such as nerve conduction studies or electromyography. Treatment options for polyradiculoneuropathy depend on the underlying cause of the condition, but may include:
* Medications to manage pain or inflammation
* Physical therapy to improve strength and coordination
* Lifestyle modifications, such as quitting smoking or losing weight, to reduce pressure on the nerves
* Surgery to relieve compression or repair damaged nerves
In some cases, polyradiculoneuropathy may be a symptom of an underlying condition that can be treated or managed with medication or other therapies. It is important to seek medical attention if you experience any symptoms of polyradiculoneuropathy to receive an accurate diagnosis and appropriate treatment.
There are several types of diarrhea, including:
1. Acute diarrhea: This type of diarrhea is short-term and usually resolves on its own within a few days. It can be caused by a viral or bacterial infection, food poisoning, or medication side effects.
2. Chronic diarrhea: This type of diarrhea persists for more than 4 weeks and can be caused by a variety of conditions, such as irritable bowel syndrome (IBS), inflammatory bowel disease (IBD), or celiac disease.
3. Diarrhea-predominant IBS: This type of diarrhea is characterized by frequent, loose stools and abdominal pain or discomfort. It can be caused by a variety of factors, including stress, hormonal changes, and certain foods.
4. Infectious diarrhea: This type of diarrhea is caused by a bacterial, viral, or parasitic infection and can be spread through contaminated food and water, close contact with an infected person, or by consuming contaminated food.
Symptoms of diarrhea may include:
* Frequent, loose, and watery stools
* Abdominal cramps and pain
* Bloating and gas
* Nausea and vomiting
* Fever and chills
* Headache
* Fatigue and weakness
Diagnosis of diarrhea is typically made through a physical examination, medical history, and laboratory tests to rule out other potential causes of the symptoms. Treatment for diarrhea depends on the underlying cause and may include antibiotics, anti-diarrheal medications, fluid replacement, and dietary changes. In severe cases, hospitalization may be necessary to monitor and treat any complications.
Prevention of diarrhea includes:
* Practicing good hygiene, such as washing hands frequently and thoroughly, especially after using the bathroom or before preparing food
* Avoiding close contact with people who are sick
* Properly storing and cooking food to prevent contamination
* Drinking safe water and avoiding contaminated water sources
* Avoiding raw or undercooked meat, poultry, and seafood
* Getting vaccinated against infections that can cause diarrhea
Complications of diarrhea can include:
* Dehydration: Diarrhea can lead to a loss of fluids and electrolytes, which can cause dehydration. Severe dehydration can be life-threatening and requires immediate medical attention.
* Electrolyte imbalance: Diarrhea can also cause an imbalance of electrolytes in the body, which can lead to serious complications.
* Inflammation of the intestines: Prolonged diarrhea can cause inflammation of the intestines, which can lead to abdominal pain and other complications.
* Infections: Diarrhea can be a symptom of an infection, such as a bacterial or viral infection. If left untreated, these infections can lead to serious complications.
* Malnutrition: Prolonged diarrhea can lead to malnutrition and weight loss, which can have long-term effects on health and development.
Treatment of diarrhea will depend on the underlying cause, but may include:
* Fluid replacement: Drinking plenty of fluids to prevent dehydration and replace lost electrolytes.
* Anti-diarrheal medications: Over-the-counter or prescription medications to slow down bowel movements and reduce diarrhea.
* Antibiotics: If the diarrhea is caused by a bacterial infection, antibiotics may be prescribed to treat the infection.
* Rest: Getting plenty of rest to allow the body to recover from the illness.
* Dietary changes: Avoiding certain foods or making dietary changes to help manage symptoms and prevent future episodes of diarrhea.
It is important to seek medical attention if you experience any of the following:
* Severe diarrhea that lasts for more than 3 days
* Diarrhea that is accompanied by fever, blood in the stool, or abdominal pain
* Diarrhea that is severe enough to cause dehydration or electrolyte imbalances
* Diarrhea that is not responding to treatment
Prevention of diarrhea includes:
* Good hand hygiene: Washing your hands frequently, especially after using the bathroom or before preparing food.
* Safe food handling: Cooking and storing food properly to prevent contamination.
* Avoiding close contact with people who are sick.
* Getting vaccinated against infections that can cause diarrhea, such as rotavirus.
Overall, while diarrhea can be uncomfortable and disruptive, it is usually a minor illness that can be treated at home with over-the-counter medications and plenty of fluids. However, if you experience severe or persistent diarrhea, it is important to seek medical attention to rule out any underlying conditions that may require more formal treatment.
The syndrome is named after the doctors who first described it in the 1950s, Drs. Miller and Fisher. It is characterized by a gradual onset of muscle weakness and wasting, which typically begins in the hands and feet and spreads to other parts of the body over time.
Other symptoms of Miller Fisher Syndrome may include:
* Muscle cramps
* Muscle spasms
* Twitching of the eyelids (blepharospasm)
* Loss of reflexes
* Difficulty with speech and swallowing
* Weakness in the muscles of the face, arms, and legs
* Atrophy of the muscles in the hands and feet
The exact cause of Miller Fisher Syndrome is not known, but it is believed to be related to a problem with the nerve cells that control voluntary muscle movement. The disorder usually affects adults between the ages of 50 and 70, and men are more commonly affected than women.
There is no cure for Miller Fisher Syndrome, but treatment can help manage the symptoms. Physical therapy, occupational therapy, and medications such as anticonvulsants and muscle relaxants may be used to improve muscle strength and function. In severe cases, a ventilator may be needed to assist with breathing.
The progression of Miller Fisher Syndrome can vary widely, and some people may experience a rapid decline in muscle function while others may remain relatively stable for many years. The life expectancy of individuals with the disorder is generally reduced due to the risk of complications such as respiratory failure and pneumonia.
Gastroenteritis can be classified into different types based on the cause:
Viral gastroenteritis - This is the most common type of gastroenteritis and is caused by norovirus or rotavirus.
Bacterial gastroenteritis - This type is caused by bacteria such as salmonella, E. coli, or campylobacter.
Parasitic gastroenteritis - This is caused by parasites such as giardia or cryptosporidium.
Foodborne gastroenteritis - This type is caused by consuming contaminated food or water.
Gastroenteritis can be treated with antibiotics for bacterial infections, anti-diarrheal medications, and hydration therapy to prevent dehydration. In severe cases, hospitalization may be necessary.
Prevention measures include proper hand washing, avoiding close contact with people who are sick, and avoiding contaminated food and water. Vaccines are also available for some types of gastroenteritis such as rotavirus.
Foodborne diseases, also known as food-borne illnesses or gastrointestinal infections, are conditions caused by eating contaminated or spoiled food. These diseases can be caused by a variety of pathogens, including bacteria, viruses, and parasites, which can be present in food products at any stage of the food supply chain.
Examples of common foodborne diseases include:
1. Salmonella: Caused by the bacterium Salmonella enterica, this disease can cause symptoms such as diarrhea, fever, and abdominal cramps.
2. E. coli: Caused by the bacterium Escherichia coli, this disease can cause a range of symptoms, including diarrhea, urinary tract infections, and pneumonia.
3. Listeria: Caused by the bacterium Listeria monocytogenes, this disease can cause symptoms such as fever, headache, and stiffness in the neck.
4. Campylobacter: Caused by the bacterium Campylobacter jejuni, this disease can cause symptoms such as diarrhea, fever, and abdominal cramps.
5. Norovirus: This highly contagious virus can cause symptoms such as diarrhea, vomiting, and stomach cramps.
6. Botulism: Caused by the bacterium Clostridium botulinum, this disease can cause symptoms such as muscle paralysis, respiratory failure, and difficulty swallowing.
Foodborne diseases can be diagnosed through a variety of tests, including stool samples, blood tests, and biopsies. Treatment typically involves antibiotics or other supportive care to manage symptoms. Prevention is key to avoiding foodborne diseases, and this includes proper food handling and preparation practices, as well as ensuring that food products are stored and cooked at safe temperatures.
Abortion, Septic: A potentially life-threatening complication of an abortion procedure that occurs when bacteria enter the uterus and cause infection. Symptoms may include fever, chills, abdominal pain, nausea, vomiting, and vaginal discharge with a foul odor. Septic abortion can be caused by poor surgical technique, contamination of instruments, or failure to use sterile equipment. Treatment may involve antibiotics, surgical drainage of the infection, and supportive care. In severe cases, hospitalization and intensive care may be necessary to manage the infection and prevent complications such as sepsis or shock.
The term "septic abortion" is used to describe an abortion that has become infected, usually as a result of poor surgical technique or contamination during the procedure. This type of infection can be serious and potentially life-threatening, so it is important for women who have had an abortion to seek medical attention immediately if they experience any symptoms of infection.
Symptoms of septic abortion may include fever, chills, abdominal pain, nausea, vomiting, and vaginal discharge with a foul odor. In severe cases, women may develop sepsis or shock, which can be fatal if not treated promptly.
Treatment for septic abortion typically involves antibiotics to clear the infection, as well as surgical drainage of any abscesses that have formed in the uterus or other pelvic tissues. In some cases, hospitalization and intensive care may be necessary to manage the infection and prevent complications.
Preventing septic abortion is important, and this can be achieved by ensuring that proper surgical technique is used during the abortion procedure, using sterile equipment and supplies, and providing adequate aftercare to women who have had an abortion. Women who have had an abortion should seek medical attention immediately if they experience any symptoms of infection, as prompt treatment can help prevent serious complications and improve outcomes.
Campylobacter jejuni
Inflammatory myofibroblastic tumour
Campylobacter upsaliensis
Alash'le Abimiku
Campylobacter
Wolinella
Flagellum
Campylobacter coli
Lucy S. Tompkins
Campylobacter fetus
Enteritis
Acute motor axonal neuropathy
Marginal zone B-cell lymphoma
Brucella agar
Horizontal gene transfer
Torisashi
Western jackdaw
Milk borne diseases
N-acetylgalactosamine-N,N'-diacetylbacillosaminyl-diphospho-undecaprenol 4-alpha-N-acetylgalactosaminyltransferase
Emma Allen-Vercoe
Intralytix
Bovine campylobacteriosis
Campylobacter lanienae
Interactome
Reactive nitrogen species
Well
Campylobacteriosis
Edward McSweegan
Pseudaminic acid synthase
Escherichia coli
Paralytic illness of Franklin D. Roosevelt
HisB
Lower gastrointestinal bleeding
Hypothiocyanite
Hapten
Competence factor
N,N'-diacetylbacillosaminyl-diphospho-undecaprenol alpha-1,3-N-acetylgalactosaminyltransferase
UDP-4-amino-4,6-dideoxy-N-acetyl-alpha-D-glucosamine transaminase
Foodborne illness
DUTP diphosphatase
Source attribution
Carboxynorspermidine decarboxylase
UDP-N-acetylglucosamine 4,6-dehydratase (configuration-retaining)
Transverse myelitis
Emerging infectious disease
Parasitism
Washington State Penitentiary
CMP-N,N'-diacetyllegionaminic acid synthase
Antiganglioside antibodies
Dlawer Ala'Aldeen
Campylobacter jejuni Infections Associated with Raw Milk Consumption - Utah, 2014 | MMWR
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Campylobacter Fetus-Jejuni Bacterin (Colorado) - Premier1Supplies
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Campylobacter jejuni sequence types show remarkable spatial and temporal stability in blackbirds
Use of comparative genomics to identify vaccine candidates for Campylobacter Jejuni - Zootecnica International
Agricultural intensification and the evolution of host specialism in the enteric pathogen Campylobacter jejuni
Spatio-temporal epidemiology of Campylobacter jejuni enteritis, in an area of Northwest England, 2000-2002 - Immunology
Septic Arthritis: Background, Etiology and Pathophysiology, Prognosis
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Epidemiology of Campylobacter jejuni1
- Genomic epidemiology of Campylobacter jejuni associated with asymptomatic pediatric infection in the Peruvian Amazon by: Ben Pascoe, et al. (swan.ac.uk)
Infection14
- Additional cases of C. jejuni infection were identified in October, and UDAF permanently revoked dairy A's permit to sell raw milk on December 1. (cdc.gov)
- During May 9-November 6, 2014, a total of 99 cases of C. jejuni infection were identified. (cdc.gov)
- On May 21, 2014, UPHL notified UDOH of three laboratory-confirmed cases (in patients A, B, and C) of C. jejuni infection with indistinguishable SmaI PFGE patterns (DBRS16.0196). (cdc.gov)
- Campylobacter infection is a reportable disease in Utah, and all Campylobacter isolates undergo PFGE analysis ( 1 ). (cdc.gov)
- A confirmed case was defined as the onset of diarrheal illness caused by C. jejuni matching the cluster PFGE pattern or confirmed Campylobacter infection on or after May 1 in a person who had consumed dairy A raw milk 1-10 days before illness onset. (cdc.gov)
- Campylobacter jejuni infection causes cramping, diarrhea, abdominal pain and fever within 2 to 5 days after a person has been exposed to the organism. (medlineplus.gov)
- The outcomes could also help reduce the concern for human health and economic burden incurred by C. jejuni infection. (zootecnicainternational.com)
- But autophagy involvement as a host immune response to C. jejuni infection has not been clarified. (tokushima-u.ac.jp)
- This study revealed autophagy association in C. jejuni infection. (tokushima-u.ac.jp)
- During infection, C. jejuni activated the Rho family small GTPase Rac1 signaling pathway, which modulates actin remodeling and promotes the internalization of this pathogen. (tokushima-u.ac.jp)
- Interestingly, during C. jejuni invasion, LC3 was recruited to bacterial entry site depending on Rac1 GTPase activation just at the early step of the infection. (tokushima-u.ac.jp)
- C. jejuni infection induced LC3-II conversion, and autophagy induction facilitated C. jejuni internalization. (tokushima-u.ac.jp)
- Infection with C. jejuni is the most commonly identified antecedent to Guillain-Barré Syndrome, an autoimmune peripheral neuropathy. (cdc.gov)
- a Campylobacter -positive puppy was identified as a potential source of infection. (who.int)
Isolates2
- Methods: The host specificity and temporal stability of European Blackbird C. jejuni was investigated with multilocus sequence typing in a set of isolates collected from Sweden, Australia, and The Azores. (edu.au)
- We report a rising trend in ofloxacin resistance, re-emergence of erythromycin resistance and indications of co-resistance to both drugs in clinical isolates of Campylobacter spp. (who.int)
Coli1
- Ciprofloxacin is effective in treating infectious diarrheas caused by E. coli, Campylobacter jejuni, and shigella bacteria. (medicinenet.com)
Infections7
- While a majority of Campylobacter infections are mild self-limiting diarrhoeal illnesses, in a proportion of cases severe infections occur, including prolonged enteritis, septicaemia and other extraintestinal infections [4]. (who.int)
- In addition to laboratory-confirmed infections in people, a Campylobacter -positive faecal sample was recovered from the puppy. (who.int)
- Ongoing Outbreak of Extensively Drug-Resistant Campylobacter jejuni Infections Associated With US Pet Store Puppies, 2016-2020. (cdc.gov)
- IMPORTANCE: Extensively drug-resistant Campylobacter jejuni infections cannot be treated with any commonly recommended antibiotics and pose an increasing public health threat. (cdc.gov)
- OBJECTIVES: To investigate cases of extensively drug-resistant C jejuni associated with pet store puppies and describe the epidemiologic and laboratory characteristics of these infections. (cdc.gov)
- DESIGN, SETTING, AND PARTICIPANTS: In August 2017, health officials identified, via survey, patients with C jejuni infections who reported contact with puppies sold by pet stores. (cdc.gov)
- In conjunction with state and federal partners, the Centers for Disease Control and Prevention investigated cases of culture-confirmed C jejuni infections in US patients with an epidemiologic or molecular association with pet store puppies between January 1, 2016, and February 29, 2020. (cdc.gov)
Outbreaks2
- Campylobacter is the most commonly notified cause of gastroenteritis in Australia, but few outbreaks are identified relative to disease incidence. (who.int)
- Between 24 April and 25 June 2012, two outbreaks of Campylobacter gastroenteritis occurred at an Australian residential aged-care facility (ACF) ( Figure 1 ). (who.int)
Zoonotic2
- Campylobacter jejuni is a microaerophilic zoonotic pathogen with an atypical respiratory Complex I that oxidizes a flavodoxin (FldA) instead of NADH. (uea.ac.uk)
- Background: The zoonotic bacterium Campylobacter jejuni has a broad host range but is especially associated with birds, both domestic and wild. (edu.au)
Subsp2
- Campylobacter jejuni subsp. (berkeley.edu)
- Campylobacter fetus subsp. (bvsalud.org)
Bacteria1
- Host ecology regulates interspecies recombination in bacteria of the genus Campylobacter by: Matthew Hitchings, et al. (swan.ac.uk)
Enteritis1
- Se sabe que los organismos causan aborto en ovejas y fiebre y enteritis en el hombre y pueden asociarse con enfermedades entéricas en terneros, ovejas y otros animales. (bvsalud.org)
Taux d'isolement1
- Le taux d'isolement était plus élevé en été, sauf pour Yersinia spp. (who.int)
Genome4
- Specific objectives for this research study included, 1) to predict the protein-coding regions of isolated Campylobacter strains by analyzing the complete genome sequences, 2) to identify vaccine candidates against C. jejuni via computer simulation, and 3) to evaluate the feasibility of the antigens as vaccine candidate using in vitroscreening approach. (zootecnicainternational.com)
- Multiple genome sequences of C. jejuni strains isolated from different poultry sources (e.g., retail meat, cloacal swab, and litter samples) were compared. (zootecnicainternational.com)
- A total of six antigens were predicted based on the whole genome sequences of C. jejuni . (zootecnicainternational.com)
- Essential genome of Campylobacter jejuni. (cdc.gov)
Strains1
- List of C. jejuni strains included in MLSA analysis. (cdc.gov)
Yersinia1
- V. cholerae, Yersinia enterocolitica and Campylobacter jejuni were not detected in this study. (frontiersin.org)
Specimens1
- In May 2014, the Utah Public Health Laboratory (UPHL) notified the Utah Department of Health (UDOH) of specimens from three patients infected with Campylobacter jejuni yielding indistinguishable pulsed-field gel electrophoresis (PFGE) patterns. (cdc.gov)
Antimicrobial1
- Prevalence of antimicrobial resistant campylobacter spp. (zootecnicainternational.com)
Populations1
- Conclusion: The maintenance of host-specific signals in spatially and temporally separated C. jejuni populations suggests the existence of strong purifying selection for this bacterium in European Blackbirds. (edu.au)
Isolation rate1
- The isolation rate of Campylobacter spp. (who.int)
Isolate3
- Campylobacter jejuni is a common raw milk contaminant and is notoriously difficult to isolate from food products, because of its fastidious growth requirements. (cdc.gov)
- DNA extracted from C. jejuni clinical isolate Cj1 was used a positive control for amplification of primers. (cdc.gov)
- Only one human isolate could be re-grown by the reference laboratory and was confirmed as a Campylobacter jejuni subspecies jejuni , with sensitivity to ciprofloxacin, nalidixic acid, gentamicin and tetracycline. (who.int)
Poultry7
- Most cases of Campylobacter jejuni come from handling or ingesting raw or undercooked poultry meat. (medlineplus.gov)
- This indicates adaptation, exquisite specificity, and stability in time for European Blackbirds, which is in sharp contrast with the high levels of recombination and mutation found in poultry-related C. jejuni genotypes. (edu.au)
- Interventions to control and reduce the incidence of Campylobacter jejuni colonization in poultry is important in order to reduce the burden of foodborne illness in humans. (zootecnicainternational.com)
- The data obtained from this study may guide the industry to develop effective vaccines to reduce C. jejuni colonization in poultry and enhance the safety of poultry products. (zootecnicainternational.com)
- Occupational exposure to Campylobacter jejuni and autoimmune peripheral neuropathy in cattle, poultry, and swine farm ers. (cdc.gov)
- The avian commensal bacterium Campylobacter jejuni has been reported at high prevalence in poultry and cattle, and also has been reported in swine. (cdc.gov)
- Serum anti-C. jejuni antibodies and anti-ganglioside autoantibodies in farm ers from Iowa who work with swine, poultry, and cattle are compared to non- farm ers. (cdc.gov)
Organisms1
- This is what campylobacter organisms look like through a microscope. (medlineplus.gov)
Evolution1
- Quantifying bacterial evolution in the wild: A birthday problem for Campylobacter lineages by: Ben Pascoe, et al. (swan.ac.uk)
Inhibitors1
- Biochemical characterization and identification of ferulenol and embelin as potent inhibitors of malate:quinone oxidoreductase from Campylobacter jejuni . (bvsalud.org)
Survival1
- Campylobacter jejuni - survival instincts. (bvsalud.org)
Findings provide1
- Conclusions: The findings provide evidence that occupational exposure to C. jejuni is an important health risk for farm ers who work with animals. (cdc.gov)
Resistance1
- This study was conducted to analyse the trend of erythromycin and ofloxacin resistance among Campylobacter spp. (who.int)
Study2
- In this study, we found the LC3 contribution to C. jejuni invasion signaling via the Rac1 signaling pathway. (tokushima-u.ac.jp)
- Anti-infective bovine colostrum oligosaccharides: Campylobacter jejuni as a case study. (dsm.com)
Human1
- Both the human- and canine- derived campylobacters were tested for relatedness via speciation, antibiotic susceptibility testing and pulsed-field gel electrophoresis (PFGE). (who.int)
Animals1
- In summary, puppies should not be considered as companion animals in ACFs due to high rates of Campylobacter carriage and the underlying susceptibility of the elderly. (who.int)
Common1
- Campylobacter jejuni is one of the most common bacterial causes of diarrhea. (medlineplus.gov)
Host2
- Earlier studies have indicated thrushes of the genus Turdus in Europe to be frequently colonized with C. jejuni , and predominately with host-associated specific genotypes. (edu.au)
- The pathogenicity of C. jejuni is closely associated with the internalization process in host epithelial cells, which is related to a host immune response. (tokushima-u.ac.jp)
High1
- We performed fragment-based and high-throughput screens against an amino-sugar acetyltransferase enzyme, PglD, involved in biosynthesis of UDP-diNAcBac in Campylobacter jejuni. (figshare.com)
Broad1
- These three antigens have not been tested as vaccine candidates and have the potential to be developed into broad spectrum and effective vaccines against C. jejuni . (zootecnicainternational.com)
Public1
- Policies to reduce exposures of farm ers and the public to C. jejuni should be considered. (cdc.gov)