Phosphorus Metabolism Disorders
Microscopy, Electron, Scanning
Effect of histamine H2-receptor antagonist on the phosphorus-binding abilities of calcium carbonate and calcium lactate in hemodialysis patients. (1/587)The effect of histamine H2-receptor antagonist (famotidine) on the phosphorus-binding abilities of calcium carbonate and calcium lactate were examined in 13 chronic hemodialysis patients. In seven patients receiving calcium carbonate, famotidine (20 mg/d) was given because of gastroduodenal disorders, and calcium carbonate was replaced with calcium lactate as a phosphorus binder after 4 wk of treatment with famotidine. With the 4-wk administration of famotidine accompanied by calcium carbonate, the serum phosphorus level increased from 6.3+/-0.9 to 7.1+/-0.5 mg/dl (P<0.05). However, with the substitution of calcium lactate, the serum phosphorus level decreased significantly when compared to that before substitution (6.3+/-0.2 and 6.0+/-0.9 mg/dl after 4 and 8 wk of substitution, respectively), despite continued administration of famotidine. Serum calcium, creatinine, alkaline phosphatase, high sensitive parathyroid hormone, blood urea nitrogen, arterial blood pH, and bicarbonate were not significantly altered during the trial period. In six control patients treated with calcium carbonate alone, there were no statistical changes in serum calcium and phosphorus levels after substitution of calcium lactate for calcium carbonate. These results suggest that famotidine significantly affects the phosphorus-binding ability of calcium carbonate, but not that of calcium lactate. A careful observation of changes in the serum phosphorus level should be required in hemodialysis patients receiving calcium carbonate and histamine H2-receptor antagonists. Calcium lactate may be useful as a phosphorus binder in such hemodialysis patients. (+info)
New treatment of dry eye: the effect of calcium ointment through eyelid skin delivery. (2/587)AIM: To demonstrate the efficacy of a petrolatum based calcium ointment applied to the lower lid skin in the management of dry eye. METHODS: In a controlled double masked study, the effects of water free petrolatum ointment containing calcium carbonate (10% w/w) on tear functional factors and ocular surface vital staining in dry eye patients were observed. Petrolatum without calcium carbonate served as control. Patients were instructed to place ointment to the lower lid skin twice a day. Evaluation of subjective complaints, fluorescein and rose bengal staining patterns, blink rate, tear evaporation and tear break up time (BUT) were performed before and 3 months after treatment. In order to demonstrate the movement of petrolatum from the skin to the tear film, petrolatum containing 1% sodium fluorescein was placed on the lower lid of four healthy volunteers, and the concentration of fluorescein in the tear film was followed up to 6 hours using an anterior fluorometer. RESULTS: Subjective symptoms significantly improved in both the calcium group (p=0.001) and control (p=0.012), while only the calcium group demonstrated a significant improvement in fluorescein (p=0.043), rose bengal (p=0.021) scores, and blink rate (p=0.004). Tear evaporation also significantly decreased in both the calcium group (p=0.0004) and control (0.043). BUT did not improve in either group. CONCLUSION: Petrolatum based calcium ointment significantly improved symptoms, tear dynamics, and ocular surface staining in dry eye patients. However, some of the therapeutic effects may be due to lipids in the petrolatum vehicle. Petrolatum applied to the lower lid skin is an effective drug delivery system for slowly releasing drugs to the ocular surface. (+info)
Calcium use increases risk of calciphylaxis: a case-control study. (3/587)OBJECTIVE: To investigate the risk factors for the development of calciphylaxis in renal failure, a poorly understood and often fatal condition characterized by calcium deposition in tissues. DESIGN: Retrospective case-control study. SETTING: University hospital peritoneal dialysis center. PATIENTS: Eight continuous ambulatory peritoneal dialysis (CAPD) patients with calciphylaxis were identified in a 3-year period. We matched up to five controls for dialysis modality and length of time on dialysis with each case. STATISTICS: Multivariate conditional logistic regression analysis for matched case-controls. MAIN OUTCOME MEASURES: Laboratory data and demographics were collected as well as cumulative calcium and vitamin D ingestion over the year prior to disease onset. RESULTS: All the patients were female, versus only 38% (14/37) of controls (p < 0.0001). While not statistically significant, a majority of the patients were diabetic [62.5% (5/8) vs 32% (12/37)]. Peak and average levels of serum calcium, phosphate, calcium x phosphate product, parathyroid hormone (PTH), albumin, iron, total iron-binding capacity (TIBC), and ferritin were not significantly different in cases compared with controls. The use of calcitriol alone or with calcium carbonate was not found to be a significant risk factor for the development of calciphylaxis. In a multivariate analysis, iron intake seemed to be protective, contrary to previous reports, while the use of calcium carbonate was associated with a strong trend to increased risk of calciphylaxis development (odds ratio = 1.029/g and 1.011/g calcium ingested per month, at 1 and 2 - 3 months prior to calciphylaxis development; p = 0.0556 and 0.0565, respectively). CONCLUSION: These data, although limited by the small numbers of index cases, suggest that calcium ingestion is a risk factor for calciphylaxis. The increased use of calcium salts as a phosphate binder in recent years might explain the apparent increased incidence of calciphylaxis in our and other centers. The preponderance of female diabetics among cases reported elsewhere was confirmed in our study. (+info)
A method of microinjection: delivering monoclonal antibody 1223 into sea urchin embryos. (4/587)In this paper, a simpler method of microinjecting sea urchin embryos without using the conventional microinjection chamber designed by Kiehart is reported. A trough was made on a surface of 0.6% agarose gel dissolved in artificial sea water. Approximately fifty hatched embryos could be loaded in the trough and, consequently, swimming embryos were trapped in the trough. Monoclonal antibody (mAB) 1223 which blocks spiculogenesis in vitro was delivered into the blastocoels of sea urchin embryos to test whether this antibody inhibits spiculogenesis in vivo and also, whether this new technique is effective for the microinjection of the sea urchin embryos. The embryos were injected with mAB1223 at the hatched blastula, early mesenchyme blastula and early gastrula stages, and 63%, 90% and 97% of the embryos did not form spicules at the late gastrula stage, respectively. Therefore, mAB1223 was shown to also block spiculogenesis in vivo. From the fact that spiculogenesis occurred at a lower rate when mAB1223 was injected at the hatched blastula stage than at later stages, it may be speculated that endogenous proteases degraded the injected antibodies. Using this technique, extracellular events in the blastocoel or the function of certain molecules expressed in blastocoel can be easily investigated in vivo. (+info)
Molecular cloning and ultrastructural localization of the core protein of an eggshell matrix proteoglycan, ovocleidin-116. (5/587)The role of avian eggshell matrix proteins in shell formation is poorly understood. This calcitic biomaterial forms in a uterine fluid where the protein composition varies during the initial, calcification, and terminal phases of eggshell deposition. A specific antibody was raised to a 116-kDa protein, which is most abundant in uterine fluid during active eggshell calcification. This antiserum was used to expression screen a bacteriophage cDNA library prepared using mRNA extracted from pooled uterine tissue harvested at the midpoint of eggshell calcification. Plasmids containing inserts of differing 5'-lengths were isolated with a maximum cDNA sequence of 2.4 kilobases. Northern blotting and reverse transcriptase-polymerase chain reaction demonstrated that the 2. 35-kilobase message was expressed in a uterine-specific manner. The hypothetical translational product from the open reading frame corresponded to a novel 80-kDa protein, which we have named ovocleidin-116. After removal of the predicted signal peptide, its N-terminal sequence corresponded almost exactly with that determined from direct microsequencing of the 116-kDa uterine protein (this work) and with that previously determined for the core protein of a 120-kDa eggshell dermatan sulfate proteoglycan (Corrino, D. A., Rodriguez, J. P., and Caplan, A. I. (1997) Connect. Tissue Res. 36, 175-193). Ultrastructural colloidal gold immunocytochemistry of ovocleidin-116 demonstrated its presence in the organic matrix, in small vesicles found throughout the mineralized palisade layer, and the calcium reserve assembly of the mammillary layer. Ovocleidin-116 thus is a candidate molecule for the regulation of calcite growth during eggshell calcification. (+info)
A new matrix protein family related to the nacreous layer formation of Pinctada fucata. (6/587)We have isolated a new matrix protein family (N16) which is specific to the nacreous layer of the Japanese pearl oyster, Pinctada fucata, and have cloned and characterized the cDNAs coding for the components. Analysis of the deduced amino acid sequence revealed that N16 showed no definitive homology with other proteins. The in vitro studies of the crystallization clarified that N16 induced aragonite crystals when fixed on the substrate but inhibited crystal formation without it. The aragonite crystals showed platy morphology different from those formed inorganically, and long intervals of incubation resulted in crystalline layers highly similar to the nacreous layer. (+info)
Comparison of treatments for mild secondary hyperparathyroidism in hemodialysis patients. Durham Renal Osteodystrophy Study Group. (7/587)Comparison of treatments for mild secondary hyperparathyroidism in hemodialysis patients. BACKGROUND: In the management of patients with mild secondary hyperparathyroidism, it is not known whether calcium supplementation alone is sufficient to correct abnormalities in bone and mineral metabolism or if calcitriol is needed in either physiologic oral or intravenous pharmacologic doses. METHODS: This was a 40-week prospective nonmasked trial of 52 patients [parathyroid hormone (PTH) 150 to 600 pg/mL] who were randomized to receive escalating doses of either calcium carbonate (CaCO3) alone (calcium group, N = 11), daily oral calcitriol (oral group, N = 20), or intermittent intravenous calcitriol (IV group, N = 21). The groups were compared with regard to changes in serum intact PTH, serum bone-specific alkaline phosphatase (BAP), incidence of hypercalcemia (>10.5 mg/dL), and hyperphosphatemia (>6.5 mg/dL). RESULTS: PTH levels decreased in all groups (P < 0.01, paired t-test). In the calcium group, PTH (mean +/- SEM) decreased from 325 +/- 46.2 to 160 +/- 44.5 pg/mL. In the oral group, it decreased from 265 +/- 26.4 to 125 +/- 23.7 pg/mL, and in the IV group, it decreased from 240 +/- 27.7 to 65 +/- 10.0 pg/mL. Upon analysis of covariance, controlling for the initial PTH level, we found no differences in the PTH response between the groups (P > 0.10). In contrast, the BAP concentration increased from 20.7 +/- 7.6 to 27.5 +/- 7.0 microg/L in the calcium group (P = 0.17), decreased from 20. 6 +/- 3.9 to 17.8 +/- 4.5 microg/L in the oral group (P = 0.26), and from 19.1 +/- 2.6 to 10.6 +/- 1.1 microg/L in the IV group (P = 0. 007). Serum calcium increased significantly in all groups from 8.4 +/- 0.25 to 9.0 +/- 0.28, 8.5 +/- 0.16 to 9.2 +/- 0.27, and 8.7 +/- 0.16 to 9.4 +/- 0.18 mg/dL in the calcium, oral, and IV groups, respectively (P = NS difference between groups). Serum phosphorus was significantly lower in the calcium group throughout the study (P = 0.02). Hypercalcemic episodes were 2.0 +/- 0.8, 3.0 +/- 0.6, and 3. 4 +/- 0.6 per patient-year (P > 0.10), and hyperphosphatemic episodes were 0.9 +/- 0.56, 4.2 +/- 0.79 and 4.9 +/- 0.84 in the calcium, oral, and IV groups, respectively (P < 0.01). CONCLUSION: In mild secondary hyperparathyroidism, all three strategies are effective. High-dose CaCO3 alone may be sufficient to control PTH with a favorable side-effect profile, but calcitriol appears to have additional suppressive effects on bone that are greater following the intravenous route of administration and may increase the risk of adynamic bone disease. (+info)
Mechanism of calcite crystal growth inhibition by the N-terminal undecapeptide of lithostathine. (8/587)Pancreatic juice is supersaturated with calcium carbonate. Calcite crystals therefore may occur, obstruct pancreatic ducts, and finally cause a lithiasis. Human lithostathine, a protein synthesized by the pancreas, inhibits the growth of calcite crystals by inducing a habit modification: the rhombohedral (10 14) usual habit is transformed into a needle-like habit through the (11 0) crystal form. A similar observation was made with the N-terminal undecapeptide (pE(1)R(11)) of lithostathine. We therefore aimed at discovering how peptides inhibit calcium salt crystal growth. We solved the complete x-ray structure of lithostathine, including the flexible N-terminal domain, at 1.3 A. Docking studies of pE(1)R(11) with the (10 14) and (11 0) faces through molecular dynamics simulation resulted in three successive steps. First, the undecapeptide progressively unfolded as it approached the calcite surface. Second, mobile lateral chains of amino acids made hydrogen bonds with the calcite surface. Last, electrostatic bonds between calcium ions and peptide bonds stabilized and anchored pE(1)R(11) on the crystal surface. pE(1)R(11)-calcite interaction was stronger with the (11 0) face than with the (10 14) face, confirming earlier experimental observations. Energy contributions showed that the peptide backbone governed the binding more than did the lateral chains. The ability of peptides to inhibit crystal growth is therefore essentially based on backbone flexibility. (+info)
There are several types of phosphorus metabolism disorders, including:
1. Hypophosphatemia: This is a condition characterized by low levels of phosphorus in the blood. It can be caused by a variety of factors, such as malnutrition, kidney disease, or hormonal imbalances. Symptoms of hypophosphatemia can include fatigue, weakness, and bone pain.
2. Hyperphosphatemia: This is a condition characterized by high levels of phosphorus in the blood. It can be caused by conditions such as kidney disease or excessive intake of phosphorus-containing foods. Symptoms of hyperphosphatemia can include nausea, vomiting, and an increased risk of kidney stones.
3. Fanconi syndrome: This is a rare genetic disorder that affects the body's ability to absorb and utilize phosphorus. It is characterized by low levels of phosphorus in the blood, as well as other symptoms such as rickets, bone pain, and an increased risk of fractures.
4. X-linked hypophosphatemic tumor-induced osteomalacia (XLH): This is a rare genetic disorder that affects males and is characterized by low levels of phosphorus in the blood and an increased risk of bone fractures. It is caused by mutations in the TNS1 gene, which codes for a protein involved in phosphorus metabolism.
5. Tumor-induced osteomalacia (TIO): This is a rare disorder that is caused by tumors that produce excessive amounts of a hormone called fibroblast growth factor 23 (FGF23). This hormone interferes with the body's ability to absorb phosphorus, leading to low levels of phosphorus in the blood and an increased risk of bone fractures.
6. Chronic kidney disease: In advanced stages of chronic kidney disease, the kidneys may not be able to effectively remove excess phosphorus from the blood, leading to hyperphosphatemia.
7. Heart disease: High levels of phosphorus in the blood can increase the risk of heart disease, including conditions such as atherosclerosis and heart failure.
8. Kidney damage: Prolonged exposure to high levels of phosphorus in the blood can damage the kidneys and increase the risk of kidney disease.
It is important to note that these are just a few examples of conditions that can cause hyperphosphatemia, and there may be other causes as well. If you suspect that you or someone you know has hyperphosphatemia, it is important to consult with a healthcare professional for proper diagnosis and treatment.
Calculi are typically classified into three types based on their composition:
1. Calcium oxalate calculi: These are the most common type of calculus and are often found in the kidneys and urinary tract. They are more likely to occur in people with a history of kidney stones or other conditions that affect calcium metabolism.
2. Magnesium ammonium phosphate calculi: These calculi are less common and typically form in the kidneys or bladder. They are often associated with chronic kidney disease or other underlying medical conditions.
3. Uric acid calculi: These calculi are rare and often form in the joints, but can also occur in the urinary tract. They are more common in people with gout or other conditions that affect uric acid metabolism.
Calculi can cause a range of symptoms depending on their size and location, including:
* Pain in the abdomen, flank, or back
* Blood in the urine (hematuria)
* Frequent urination or difficulty urinating
* Cloudy or strong-smelling urine
* Fever or chills
* Nausea and vomiting
If calculi are small and do not cause any symptoms, they may not require treatment. However, if they grow large enough to block the flow of urine or cause pain, treatment may be necessary. Treatment options for calculi include:
1. Medications: Drugs such as alpha-blockers and potassium citrate can help to dissolve calculi and reduce symptoms.
2. Shock wave lithotripsy: This is a non-invasive procedure that uses high-energy shock waves to break up calculi into smaller pieces that can be passed more easily.
3. Endoscopic surgery: A small, flexible tube with a camera and specialized tools can be inserted through the ureter or bladder to remove calculi.
4. Open surgery: In some cases, open surgery may be necessary to remove large or complex calculi.
Prevention is key in avoiding calculi. Here are some tips for preventing calculi:
1. Drink plenty of water: Adequate hydration helps to dilute uric acid and other substances in the urine, reducing the risk of calculi formation.
2. Limit alcohol intake: Alcohol can increase levels of uric acid in the blood, which can contribute to calculi formation.
3. Maintain a healthy diet: Eating a balanced diet that is low in purines and high in fruits and vegetables can help to reduce the risk of calculi.
4. Manage underlying conditions: Conditions such as gout, hyperparathyroidism, and kidney disease can increase the risk of calculi. Managing these conditions with medication and lifestyle changes can help to reduce the risk of calculi.
5. Avoid certain medications: Certain medications, such as some antibiotics and diuretics, can increase the risk of calculi formation.
6. Monitor urine output: If you have a medical condition that affects your urinary tract, such as a blockage or an obstruction, it is important to monitor your urine output to ensure that your kidneys are functioning properly.
7. Avoid prolonged bed rest: Prolonged bed rest can increase the risk of calculi formation by slowing down urine flow and allowing minerals to accumulate in the urinary tract.
8. Stay active: Regular exercise can help to improve circulation and maintain a healthy weight, which can reduce the risk of calculi formation.
9. Avoid smoking: Smoking can increase the risk of calculi formation by reducing blood flow to the kidneys and increasing the amount of oxalate in the urine.
10. Consider medications: In some cases, medications such as allopurinol or potassium citrate may be prescribed to help prevent calculi formation. These medications can help to reduce the levels of uric acid or calcium oxalate in the urine.
It is important to note that not all kidney stones are the same, and the underlying cause may vary depending on the type of stone. For example, if you have a history of gout, you may be more likely to develop uric acid stones. In this case, medications such as allopurinol or probenecid may be prescribed to help reduce the levels of uric acid in your blood and prevent calculi formation.
Causes of Hyperphosphatemia:
There are several possible causes of hyperphosphatemia, including:
1. Kidney disease or failure: The kidneys regulate the levels of phosphate in the blood, and if they are not functioning properly, phosphate levels can become elevated.
2. Resistance to parathyroid hormone (PTH): PTH is a hormone that helps regulate calcium and phosphate levels in the body. If there is resistance to PTH, phosphate levels can become elevated.
3. Vitamin D deficiency: Vitamin D is important for the absorption of phosphate from food in the gut. A deficiency in vitamin D can lead to an excessive amount of phosphate in the blood.
4. Certain medications: Some medications, such as certain antacids and nutritional supplements, can contain high levels of phosphate and cause hyperphosphatemia.
5. Poor dietary habits: Consuming a diet that is high in phosphate-rich foods, such as meat and processed foods, can lead to elevated phosphate levels in the blood.
Symptoms of Hyperphosphatemia:
The symptoms of hyperphosphatemia can vary depending on the severity of the condition, but may include:
1. Bone pain or weakness
3. Nausea and vomiting
4. Weakness in the muscles
5. Rickets (in children)
6. Osteoporosis (in adults)
7. Kidney damage or failure
Diagnosis of Hyperphosphatemia:
Hyperphosphatemia is typically diagnosed through blood tests that measure the level of phosphate in the blood. Other tests may also be performed to assess kidney function and rule out other potential causes of elevated phosphate levels. These tests may include:
1. Serum creatinine test: This test measures the level of creatinine, a waste product that is produced by the muscles and removed from the blood by the kidneys. Elevated levels of creatinine can indicate kidney damage or failure.
2. Urine test: A urine test may be performed to check for proteinuria (excess protein in the urine), which can be a sign of kidney damage.
3. Parathyroid hormone (PTH) test: This test measures the level of PTH, a hormone that regulates calcium and phosphate levels in the blood. Elevated levels of PTH can indicate hyperparathyroidism, a condition in which the parathyroid glands produce too much PTH.
4. 24-hour urine phosphate test: This test measures the amount of phosphate excreted in the urine over a 24-hour period.
Treatment of Hyperphosphatemia:
The treatment of hyperphosphatemia depends on the underlying cause of the condition. Here are some possible treatment options:
1. Phosphate-binding agents: These medications, such as sevelamer and lanthanum carbonate, bind to phosphate in the gut and prevent it from being absorbed into the bloodstream.
2. Calcium supplements: Calcium can help to lower phosphate levels by binding to it and removing it from the bloodstream.
3. Dietary changes: A dietitian can work with you to develop a meal plan that limits phosphate-rich foods, such as meat, dairy products, and processed foods, while emphasizing fruits, vegetables, and whole grains.
4. Dialysis: In cases where the condition is caused by kidney failure, dialysis may be necessary to remove excess phosphate from the blood.
5. Surgery: In cases where the condition is caused by a parathyroid adenoma or hyperplasia, surgery may be necessary to remove the affected gland(s).
It's important to note that hyperphosphatemia can lead to complications such as mineral bone disease, which can cause weakened bones, bone pain, and an increased risk of fractures. Therefore, it's important to work with your healthcare provider to manage the condition and prevent these complications.
The symptoms of hypercalcemia may include:
* Nausea and vomiting
* Abdominal pain
* Kidney stones
* Bone pain or fractures
If left untreated, hypercalcemia can lead to complications such as kidney damage, heart problems, and an increased risk of osteoporosis. Treatment options may include medications to reduce calcium levels, surgery to remove a tumor or overactive parathyroid gland, or dialysis if the patient has kidney failure.
Early diagnosis and treatment are important to prevent long-term complications and improve the patient's quality of life.
Amorphous calcium carbonate
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Calcium carbonate 750 mg
Calcium Carbonate - Midwest Supplies
Calcium Carbonate: MedlinePlus Drug Information
Calcium carbonate | Buchi.com
Gluten Free, Vegetarian - Calcium Carbonate - Swanson®
Calcium carbonate and reduction of levothyroxine efficacy - PubMed
Using the pH Scale and Carbonic Acid Formation to Understand the Effect of Ocean Acidification on Organisms with Calcium...
Middle East Calcium Carbonate MECC - Building Markets
Growth of aragonite phase calcium carbonate on the surface of a titania-modified filter paper - CrystEngComm (RSC Publishing)
Precipitated Calcium Carbonate | Carmeuse
ASCORBIC ACID OR BIOTIN OR CALCIUM CATION OR CARBONATE ION OR CYANOCOB - Books - NCBI
MedlinePlus - Search Results for: ALUMINUM HYDROXIDE OR BISMUTH CATION OR CALCIUM CATION OR CARBONATE ION OR SODIUM CATION
Tums (calcium carbonate) dosing, indications, interactions, adverse effects, and more
Chemical Forums: Stereate Coated Calcium Carbonate vs. Uncoated Calcium Carbonate
APEC Water Essence Replacement Pre-Filter set w/ PH+ US Made Calcium Carbonate re-mineralization filter for ROES-PH75
Ruddiman, WF; Farrell, JW (1996): Calcium carbonate content of sediment core RC11-236
The Rate of Dissolution of Calcium Carbonate from the Surface of Deep-Ocean Turbidite Sediments - NASA/ADS
Water pretreatment kit with Scalex cartridge for calcium-carbonate scale prevention | Fogco Environmental Systems
Polymer compositions containing hydrated basic magnesium calcium carbonates as smoke-depressant additives - Patent EP-0012578...
ASCORBIC ACID/AE AND BIOTIN/AE AND CALCIUM CATION/AE AND CARBONATE ION/AE AND CYANOCOBALAMIN/AE AND FERRIC CATION/AE AND...
Calcium carbonate Archives » Science ABC
The effect of surface treatment with inorganic orthophosphate on the dissolution of calcium carbonate - FORTH / ICE-HT
Environmental controls on the production of calcium carbonate by earthworms - Activities - York Research Database
Independent impacts of calcium and carbonate ion concentration on Mg and Sr incorporation in cultured benthic foraminifera |...
Indication-specific dosing for (famotidine, calcium carbonate, magnesium hydroxide), frequency-based adverse effects,...
Effect of calcium carbonate encapsulation on the activity of orally administered CpG oligonucleotides | NIH Research Festival
Omya implements surcharge on calcium carbonate products in Europe - Polymers Paint Colour Journal
Producing flexible calcium carbonate from waste paper and their use as fillers for high bulk paper :: BioResources
Ground calcium carbonate3
- The MFCs had a dark brown color initially but turned into high brightness materials similar to commercial ground calcium carbonate (GCC) after the in-situ formation process. (ncsu.edu)
- One of the most widely used pigments in papermaking is calcium carbonate (CaCO 3), which is formed either by natural precipitation (ground calcium carbonate, GCC) or produced synthetically (precipitated calcium carbonate, PCC). (vacancorse.fr)
- كواحد من قادة ground calcium carbonate machine الصناعة ، تسعى SBM دائمًا إلى الابتكار والتميز. (fundacjaszekla.pl)
- Calcium carbonate comes as a tablet, chewable tablet, capsule, and liquid to take by mouth. (medlineplus.gov)
- After prenucleation with calcium carbonate nanoparticles, the titania-modified filter paper was put into classic geothermal water for the growth of needle-like aragonite calcium carbonate crystals, and the needle-like calcium carbonate crystals were guaranteed to be pure aragonite. (rsc.org)
- A strategy to improve the activity of orally delivered ODN by reducing their susceptibility to GI digestion via encapsulation in calcium carbonate nanoparticles (ODNcap) was recently described. (nih.gov)
- The aim of this study was to evaluate the cytotoxicity of benznidazole-loaded calcium carbonate nanoparticles (BZN@CaCO3) on Trypanosoma cruzi strain Y. It was observed that BZN@CaCO3 was able to reduce the viability of epimastigote, trypomastigote and amastigote forms of T. cruzi with greater potency when compared with BZN. (bvsalud.org)
- Some over-the-counter antacids, such as Tums and Rolaids, also contain calcium carbonate. (nih.gov)
- Calcium carbonate also is used as an antacid to relieve heartburn, acid indigestion, and upset stomach. (medlineplus.gov)
- Do not take calcium carbonate as an antacid for more than 2 weeks unless your doctor tells you to. (medlineplus.gov)
- The Dietary Supplement Information datasets contains additional product data on the dietary supplement(s) and non-prescription antacids containing calcium and/or magnesium reported by survey participants in the Dietary Supplement and Non-Prescription Antacid subsections of the Household Interview and in the two 24-hour dietary recall interviews. (cdc.gov)
- The NHANES Dietary Supplement Database (NHANES-DSD) contains detailed information on the dietary supplements (DS) and non-prescription antacids containing calcium and/or magnesium (antacids) reported by survey participants since NHANES 1999. (cdc.gov)
- The mechanism of the formation of the pure aragonite crystals is that the calcium carbonate crystals may undergo a dissolution and recrystallization process. (rsc.org)
- The time of the emplacement event can be determined from natural radionuclide distributions, and the degree of carbonate dissolution in this time can be measured. (harvard.edu)
- Replacing chemical pulp with inorganic materials, such as calcium carbonate (CaCO 3 ), without loss of key paper properties in papermaking is beneficial for forest conservation and production cost savings (Svending et al . (ncsu.edu)
Forms of calcium3
- Some liquid forms of calcium carbonate must be shaken well before use. (medlineplus.gov)
- The two main forms of calcium in dietary supplements are calcium carbonate and calcium citrate. (nih.gov)
- Other forms of calcium in supplements and fortified foods include calcium sulfate, calcium ascorbate, calcium microcrystalline hydroxyapatite, calcium gluconate, calcium lactate, and calcium phosphate. (nih.gov)
- calcium carbonate will decrease the level or effect of baloxavir marboxil by cation binding in GI tract. (medscape.com)
Amount of calcium3
- Calcium carbonate is a dietary supplement used when the amount of calcium taken in the diet is not enough. (medlineplus.gov)
- The amount of calcium you need each day depends on your age and sex. (nih.gov)
- Calcium is found in many multivitamin/mineral supplements , in calcium supplements, and in supplements that contain calcium and other nutrients such as vitamin D. Check the Supplement Facts label to determine the amount of calcium in the supplement. (nih.gov)
- tell your doctor and pharmacist if you are allergic to calcium carbonate or any other drugs. (medlineplus.gov)
- These agents are used for calcium electrolyte supplementation. (medscape.com)
- In thyroxine-replaced hypothyroid postmenopausal women under simultaneous calcium supplementation, switch to oral liquid or softgel capsule L-thyroxine ensures lower serum TSH levels and favorable effects on blood pressure, total cholesterolemia and glycemia. (nih.gov)
- Some patients require oral calcium supplementation. (medscape.com)
- Calcium carbonate is used for supplementation of intravenous therapy in hypocalcemia. (medscape.com)
- Supplementation increases calcium levels in the serum by improving calcium absorption and retention. (medscape.com)
- If you take 1,000 mg/day of calcium from supplements, for example, it is better to take a smaller dose twice a day than to take it all at once. (nih.gov)
- It is known that past periods of high atmospheric carbon dioxide concentration are associated with poor carbonate preservation in the deep-ocean sedimentary record. (harvard.edu)
- Finally, precipitated nano calcite calcium carbonate (50 to 100 nm) was synthesized by optimizing all the experimental and the instrumental parameters. (vacancorse.fr)
- Formation Characteristics of Precipitated Calcium Carbonate by Carbonation Process 5 O2− ions which surrounding the Ca2+ ion than calcite and bonding with 9 O2− atoms. (vacancorse.fr)
- The concepts students should learn from this activity are that there is a relationship between anthropogenic carbon dioxide and ocean acidification and that ocean acidification may have an effect on marine organisms with calcium carbonate shells. (carleton.edu)
- To interpret the sedimentary record more exactly, and to predict future atmospheric carbon dioxide levels, it is necessary to know the rate of solution of carbonate for a given degree of bottom-water undersaturation. (harvard.edu)
- Those processed MFCs were treated with in-situ formation of calcium carbonate by adding calcium oxide and injecting carbon dioxide into the mixture up to the ratio of 1:40 (MFC : calcium carbonate) by weight. (ncsu.edu)
- PCC is made by hydrating high-calcium quicklime and then reacting the resulting slurry, or "milk-of-lime", with carbon dioxide. (vacancorse.fr)
- Welding produced excessive exposures to total welding fumes, hexavalent chromium (18540299), nickel (7440020), iron-oxide (1332372), and calcium-oxide (1305788). (cdc.gov)
- The authors conclude that workers were overexposed to calcium-carbonate, carbon-monoxide, sugar dust, total welding fumes, hexavalent chromium , iron-oxide, nickel, and calcium- oxide fumes at this facility. (cdc.gov)
- Carmeuse offers a variety of lime-based product and advanced service solutions to address the many challenges you face across your pulp & paper plant or when producing precipitated production carbonate (PCC). (carmeuse.com)
- Precipitated calcium carbonate (PCC) is an innovative product derived from lime, which has many industrial applications. (vacancorse.fr)
- Exposures exceeding the most stringent environmental criteria were found for calcium-carbonate (471341) and carbon-monoxide (630080) in lime kiln workers and sugar dust in sugar bin workers. (cdc.gov)
- Calcium gluconate moderates nerve and muscle performance and facilitates normal cardiac function. (medscape.com)
- As marble, calcium carbonate is a coarse-crystalline, metamorphic rock, which is formed when chalk or limestone is recrystallized under conditions of high temperature and pressure. (vacancorse.fr)
- The FI-PHPLUS-QC is a 10" inline filter that adds high purity food grade calcium minerals to enhance the water taste for people who prefer drinking mineral water. (freedrinkingwater.com)
- This mineral cartridge adds only high-purity calcium carbonate to effectively neutralize acidity and raise pH in the water. (freedrinkingwater.com)
- Calcium is a mineral your body needs to build and maintain strong bones and to carry out many important functions. (nih.gov)
- Calcium is the most abundant mineral in the body. (nih.gov)
- During the production of polypropylene (PP) edgebands via extrusion, I saw and witness that the PP edgebands filled with stearate coated calcium carbonate have serious problems in bonding lacquer, paint and primer on the surface. (chemicalforums.com)
- Administration of oral calcium and 1alpha-hydroxylated vitamin D metabolites, such as calcitriol, remains the mainstay of treatment and should be initiated in every patient with a diagnosis of PHP. (medscape.com)
- Certain vegetables such as kale, broccoli, and Chinese cabbage (bok choi) also contain calcium. (nih.gov)
- If you become pregnant while taking calcium carbonate, call your doctor. (medlineplus.gov)
- Studies in monkeys showed concurrent use with calcium, aluminum, or iron caused significantly decreased plasma levels. (medscape.com)
- Results indicate that orally delivered CpG ODN predominantly induce gastrointestinal rather than systemic immunity and that calcium carbonate encapsulation does not significantly alter this behavior. (nih.gov)
- Carbonation is one of the methods for the production of precipitated calcium carbonate (PCC) on the industrial scale. (vacancorse.fr)
- We produce and sell high quality calcium carbonate. (buildingmarkets.org)
- Producing flexible calcium carbonate from waste paper and their use as fillers for high bulk paper ," BioResources 18(2), 3400-3412. (ncsu.edu)
- It can be initially administered intravenously, and calcium levels can be maintained with a high-calcium diet. (medscape.com)
- Most grains (such as breads, pastas, and unfortified cereals) do not have high amounts of calcium. (nih.gov)
- Do not take calcium carbonate within 1-2 hours of taking other medicines. (medlineplus.gov)
- The hydrodynamic conditions gener-ated in the reactor affect the course of the process and the characteristics of the precipitated calcium carbonate particles. (vacancorse.fr)
- In this study, precipitated calcium carbonate (PCC) has been produced using a semi-continuous carbonation process within a Ca(OH) 2(s) -CO 2(g) -H 2 O system inside a bench-scale reactor. (vacancorse.fr)
- Milk, yogurt, and cheese are the main food sources of calcium for most people in the United States. (nih.gov)
- degradation during the reaction of stearic acid with calcium carbonate. (vacancorse.fr)
- Precipitated Calcium Carbonate (PCC) comes in different grades according to customer requirements and is mainly used in the following industries and applications: manufacturing of paints of all kinds, manufacturing of polymers and plastics, manufacturing of glass, toothpaste, and healthcare products. (vacancorse.fr)
- Dairy products are rich sources of calcium, but people with lactose intolerance, people with milk allergies, and vegans (people who don't consume any animal products) must find other sources of calcium. (nih.gov)
- Calcium is found in many foods. (nih.gov)
- Precipitated Calcium Carbonate (PCC) - created by man in a matter of hours Calcium carbonate can also be produced synthetically in the form of Precipitated Calcium Carbonate (PCC). (vacancorse.fr)
- In parts of the Atlantic Ocean, turbidite sedimentation mechanisms have emplaced carbonate-rich material in contact with undersaturated bottom water. (harvard.edu)