Cadmium
Cadmium Poisoning
Cadmium Chloride
Cadmium Radioisotopes
Metallothionein
Metals, Heavy
Lead
Phytochelatins
Zinc
Spectrophotometry, Atomic
Environmental Pollutants
Mercury
Environmental Exposure
Soil Pollutants
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Metals
Copper
Metallurgy
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Arsenic
Tellurium
Testing their metal. (1/120)
Metals continually rank at the top of the U.S. Environmental Protection Agency's annual list of agents that pose the greatest hazard to the people of the United States. Metals aren't going away, either. They do not biodegrade, and they often concentrate in human and animal cells and tissue. Many metals are known human and animal carcinogens, while many others are suspected to play a role in cancer. Little is known, however, about the mechanisms by which metals cause cancer. (+info)Granulocyte and plasma cytokine activity in acute cadmium intoxication in rats. (2/120)
Changes in the number and ex vivo function of peripheral blood neutrophils were investigated following intraperitoneal administration of cadmium-chloride in rats. Besides a dose-dependent increase in the number of peripheral blood neutrophils, changes were found in the functional state of isolated polymorphonuclear leukocytes (PMNs). Increased spontaneous adhesion and activation, and TNF activity in a conditioned medium were observed in cultures of granulocytes in comparison to granulocytes from control (saline-treated) animals. Increased levels of plasma activity of inflammatory cytokines, tumor necrosis factor (TNF) and interleukin-6 (IL-6) were noted following cadmium administration. Cytological signs of pulmonary inflammation were revealed histologically and the majority of neutrophils recovered from the lungs by enzyme digestion exhibited a capacity of nitroblue tetrazolium (NBT) reduction. Our data demonstrate that acute cadmium intoxication leads to a systemic inflammatory response characterized by numerical and functional changes in the granulocyte compartment and to increased levels of inflammation-related cytokine activity in the circulation. Correlations between the increased number of peripheral blood neutrophils and IL-6 plasma activity (r=0.776, p<0.00001) and the number of neutrophils recovered from the lung tissue (r=0.893, p<0.00001) suggested that systemic cadmium-induced inflammation might be involved in the pulmonary toxicity of cadmium. (+info)Neurobehavioural effects of occupational exposure to cadmium: a cross sectional epidemiological study. (3/120)
BACKGROUND: A patient with unexplained minor behavioural changes associated with an axonal sensorimotor polyneuropathy had a history of chronic occupational exposure to cadmium (Cd). Although animal studies have shown that Cd is a potent neurotoxicant, little is known about its toxicity for the human central nervous system. The aim of this study was to investigate the toxic potential of chronic occupational exposure to Cd on neurobehavioural functions. METHODS: A cross sectional epidemiological study was conducted ina group of Cd workers and an age matched control group. Eighty nine adult men (42 exposed to Cd and 47 control workers) were given a blinded standardised examination that consisted of computer assisted neurobehavioural tests (neurobehavioural examination system), a validated questionnaire to assess neurotoxic complaints (neurotoxicity symptom checklist--60, NSC-60), and a standardised self administered questionnaire to detect complaints consistent with peripheral neuropathy and dysfunction of the autonomic nervous system. Historical and current data on biomonitoring of exposure to Cd, either the highest value of Cd in urine (CdU in microgram Cd/g creatinine) of each Cd worker during work (CdUmax) or the current value (CdUcurrent) of each control, were available as well as data on microproteinuria. RESULTS: Cd workers (CdUmax: mean (range), 12.6 (0.4-38.4)) performed worse than the controls (CdUcurrent: mean (range), 0.7 (0.1-2.0)) on visuomotor tasks, symbol digit substitution (p = 0.008), and simple reaction time to direction (p = 0.058) or location (p = 0.042) of a stimulus. In multiple linear regression analysis, symbol digit substitution, simple direction reaction time test, and simple location reaction time test were significantly related to CdUmax, (beta = 0.35 (p < 0.001), beta = 0.25 (p = 0.012), and beta = 0.23 (p = 0.021) respectively). More complaints consistent with peripheral neuropathy (p = 0.004), complaints about equilibrium (p = 0.015), and complaints about concentration ability (p = 0.053) were found in the group exposed to Cd than in the control group, and these variables correlated positively with CdUmax (peripheral neuropathy: beta = 0.38, p < 0.001; equilibrium: beta = 0.22, p = 0.057; concentration ability: beta = 0.27, p = 0.020). CONCLUSION: Slowing of visuomotor functioning on neurobehavioural testing and increase in complaints consistent with peripheral neuropathy, complaints about equilibrium, and complaints about concentration ability were dose dependently associated with CdU. Age, exposure to other neurotoxicants, or status of renal function could not explain these findings. The present study also indicates that an excess of complaints may be detected in Cd workers before signs of microproteinuria induced by Cd occur. (+info)Risk of mortality, cancer incidence, and stroke in a population potentially exposed to cadmium. (4/120)
OBJECTIVES: To follow up mortality and cancer incidence in a cohort potentially exposed to cadmium and to perform a geographical (ecological) analysis to further assess the health effects of potential exposure to cadmium. METHODS: The English village of Shipham has very high concentrations of cadmium in the soil. A previous cohort study of residents of Shipham in 1939 showed overall mortality below that expected, but a 40% excess of mortality from stroke. This study extends the follow up of the cohort for mortality to 1997, and includes an analysis of cancer incidence from 1971 to 1992, and a geographical study of mortality and cancer incidence. Standardised mortality and incidence ratios (SMRs and SIRs) were estimated with regional reference rates. Comparisons were made with the nearby village of Hutton. RESULTS: All cause cohort mortality was lower than expected in both villages, although there was excess cancer incidence in both Shipham (SIR 167, 95% confidence interval (95% CI) 106 to 250) and Hutton (SIR 167, 95% CI 105 to 253). There was an excess of mortality from hypertension, cerebrovascular disease, and nephritis and nephrosis, of borderline significance, in Shipham (SMR 128, 95% CI 99 to 162). In the geographical study, all cause mortality in Shipham was also lower than expected (SMR 84, 95% CI 71 to 100). There was an excess in genitourinary cancers in both Shipham (SIR 160, 95% CI 107 to 239) and Hutton (SIR 153, 95% CI 122 to 192). CONCLUSION: No clear evidence of health effects from possible exposure to cadmium in Shipham was found despite the extremely high concentrations of cadmium in the soil. (+info)Male infertility and environmental exposure to lead and cadmium. (5/120)
Humans are exposed occupationally and environmentally to metal aerosols including lead (Pb2+) and cadmium (Cd2+). These toxicants accumulate in male reproductive organs. Epidemiological studies have been equivocal about effects of Pb2+ and Cd2+ on hormone concentrations, male fertility and sperm parameters. Comparison of Pb2+ and Cd2+ concentrations in fertile and infertile men are problematic. Problem areas include failure to control confounding variables, but genetic polymorphisms as in somatic diseases may modulate Pb2+ and Cd2+ damage. Multiple calcium (Ca2+) and potassium (K+) channel isoforms have been identified in human testes and spermatozoa. These Ca2+ and K+ channels are involved in early events of acrosome reactions. Ca2+ channel are susceptible to Cd2+ poisoning and K+ channels to Pb2+. These channels offer entry paths for metallic toxicants into mature spermatozoa. Ion channel polymorphisms may cause differential sensitivities to Cd2+ and Pb2+, explaining in part prospective blinded studies showing high Cd2+ in varicocele-related human infertility and high Pb2+ in unexplained infertility. In both forms of male infertility the ability to undergo an acrosome reaction decreases. Reverse transcriptase-polymerase chain reaction assays for Ca2+ and K+ channel isoforms may identify susceptibility subgroups with lower resistance to environmental exposures. (+info)Metallothionein-null mice are more sensitive than wild-type mice to liver injury induced by repeated exposure to cadmium. (6/120)
Liver is a major target organ of cadmium (Cd) toxicity following acute and chronic exposure. Metallothionein (MT), a low-molecular-weight, cysteine-rich, metal-binding protein has been shown to play an important role in protection against acute Cd-induced liver injury. This study investigates the role of MT in liver injury induced by repeated exposure to Cd. Wild-type and MT-I/II knockout (MT I/II-null) mice were injected sc with a wide range of CdCl(2) doses, 6 times/week, for up to 10 weeks, and their hepatic Cd content, hepatic MT concentration, and liver injury were examined. Repeated administration of CdCl(2) produced acute and nonspecific chronic inflammation in the parenchyma and portal tracts and around central veins. Higher doses produced granulomatous inflammation and proliferating nodules in liver parenchyma. Apoptosis and mitosis occurred concomitantly in liver following repeated Cd exposure, whereas necrosis was mild. As a result, significant elevation of serum enzyme levels was not observed. In wild-type mice, hepatic Cd concentration increased in a dose- and time-dependent manner, reaching 400 microgram/g liver, along with 150-fold increases in hepatic MT concentrations, the latter reaching 1200 microgram/g liver. In contrast, in MT I/II-null mice, hepatic Cd concentrations were about 10 microgram/g liver. Despite the lower accumulation of Cd in livers of MT I/II-null mice, the maximum tolerated dose of Cd was one-eighth lower than that for wild-type mice at 10 weeks, and liver injury was more pronounced in the MT I/II-null mice, as evidenced by increases in liver/body weight ratios and histopathological analyses. In conclusion, these data indicate that (1) nonspecific chronic inflammation, granulomatous inflammation, apoptosis, liver cell regeneration, and presumably, preneoplastic proliferating nodules are major features of liver injury induced by repeated Cd exposure, and (2) intracellular MT is an important protein protecting against this Cd-induced liver injury. (+info)Acute cadmium exposure inactivates thioltransferase (Glutaredoxin), inhibits intracellular reduction of protein-glutathionyl-mixed disulfides, and initiates apoptosis. (7/120)
Oxidative stress broadly impacts cells, initiating regulatory pathways as well as apoptosis and necrosis. A key molecular event is protein S-glutathionylation, and thioltransferase (glutaredoxin) is a specific and efficient catalyst of protein-SSG reduction. In this study 30-min exposure of H9 and Jurkat cells to cadmium inhibited intracellular protein-SSG reduction, and this correlated with inhibition of the thioltransferase system, consistent with thioltransferase being the primary intracellular catalyst of deglutathionylation. The thioredoxin system contributed very little to total deglutathionylase activity. Thioltransferase and GSSG reductase in situ displayed similar dose-response curves (50% inhibition near 10 micrometer cadmium in extracellular buffer). Acute cadmium exposure also initiated apoptosis, with H9 cells being more sensitive than Jurkat. Moreover, transfection with antisense thioltransferase cDNA was incompatible with cell survival. Collectively, these data suggest that thioltransferase has a vital role in sulfhydryl homeostasis and cell survival. In separate experiments, cadmium inhibited the isolated component enzymes of the thioltransferase and thioredoxin systems, consistent with the vicinal dithiol nature of their active sites: thioltransferase (IC(50) approximately 1 micrometer), GSSG reductase (IC(50) approximately 1 micrometer), thioredoxin (IC(50) approximately 8 micrometer), thioredoxin reductase (IC(50) approximately 0.2 micrometer). Disruption of the vicinal dithiol on thioltransferase (via oxidation to C22-SS-C25; or C25S mutation) protected against cadmium, consistent with a dithiol chelation mechanism of inactivation. (+info)Heavy metal poisoning in glass worker characterised by severe. (8/120)
The paper presents the clinical description of the masticatory organ and biochemical assessment of dental tissue in a patient employed in a glassworks for 20 years. During 12 years the patient has suffered baldness ("Alopecia areata") and atypical extensive and non-healing cutaneous lesions. Dental examination revealed changes typical of chronic poisoning by cadmium and bismuth compounds. (+info)* Nausea and vomiting
* Abdominal pain
* Diarrhea
* Fatigue
* Weakness
* Headache
* Dizziness
* Renal damage
In severe cases, cadmium poisoning can cause:
* Respiratory failure
* Cardiovascular collapse
* Seizures
* Coma
* Death
Treatment of cadmium poisoning usually involves supportive care, such as fluid replacement and management of symptoms. In cases of severe poisoning, hospitalization may be necessary and chelation therapy may be administered to remove the heavy metal from the body. Prevention of cadmium poisoning is key and this can be achieved through proper handling, storage and disposal of cadmium-containing materials, as well as using personal protective equipment during work with cadmium.
If you suspect that you or someone else has been exposed to cadmium, it is important to seek medical attention immediately. A healthcare professional will be able to assess the level and severity of exposure and provide appropriate treatment.
Cadmium poisoning
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Arsenic10
- In addition, chronic, low-level exposure to lead in the soil and in residues of lead-based paint, to mercury in the atmosphere, in dental amalgams and in seafood, and to cadmium and arsenic in the environment and in cigarette smoke is much more common than acute exposure. (nih.gov)
- Application to Mus musculus mice under arsenic/cadmium exposure. (nih.gov)
- The sustained presence of dimercaprol promotes continued excretion of the metallic poisons - arsenic, gold and mercury. (nih.gov)
- BAL in Oil (Dimercaprol Injection USP) is indicated in the treatment of arsenic, gold and mercury poisoning. (nih.gov)
- The Healthy People 2000 objective tracked State data collection for 9 diseases including lead poisoning, mercury poisoning, arsenic poisoning, cadmium poisoning, methemoglobinemia, acute chemical poisoning, carbon monoxide poisoning, heatstroke, and hypothermia. (cdc.gov)
- When reading about mercury or lead poisoning, or murders in which someone used a deadly poison such as arsenic, some may ask, 'Why would God create chemicals that cause so much harm to people? (creation.com)
- Roughly one-third of the tested products, 40 in total, had high enough levels of arsenic, lead, and cadmium combined, on average, to pose a health concern for children when regularly consumed in typical serving sizes. (nbcdfw.com)
- Frequent exposure to even small amounts of lead, arsenic, cadmium, and other heavy metals is dangerous, in part because it's difficult for the human body to break them down or excrete them. (nbcdfw.com)
- Because counterfeit make-up often contains poisons like lead, copper, mercury, arsenic or cadmium. (purocleanpers.us)
- This causes nature to become sick from arsenic and cadmium poisonings and lead, arsenic, and cadmium poisonings. (essaycopy.com)
Mercury7
- Other chemicals, such as mercury, lead, and cadmium, aren't nutritionally beneficial and can be toxic in sufficient quantities. (innerbody.com)
- Dimercaprol Injection USP is effective for use in acute poisoning by mercury salts if therapy is begun within one or two hours following ingestion. (nih.gov)
- It is not very effective for chronic mercury poisoning. (nih.gov)
- Food poisoning can also occur when non-infectious poisons (such as poisonous mushrooms) or heavy metals (such as lead or mercury) find their way into people's stomachs. (medbroadcast.com)
- The thing about cadmium, a lot of people assume that if you take a heavy metal that it makes you feel bad right away, and that can be true if you take a lot, but you have to look at the history of medicine to understand that mercury salts and mercury vapors were used medicinally by scientists and doctors for hundreds of years. (thetruthaboutcancer.com)
- Most electronics contain toxic materials such as lead, cadmium, and mercury. (essaycopy.com)
- The goal of the Medford Electronic Clean-up is to reduce toxic materials like mercury, lead, or cadmium, from poisoning the Earth. (medfordccsr.org)
Toxicity3
- 8. Monitoring of human populations for early markers of cadmium toxicity: a review. (nih.gov)
- here, he shares what he found regarding the toxicity of cadmium in artist paints. (kingstoncove.studio)
- When I visited the M. Graham & Co. factory in 2015, I asked specifically about the toxicity of cadmium colors. (kingstoncove.studio)
Exposure20
- 1984. Human health effects of exposure to cadmium. (cdc.gov)
- 2000). "Neurobehavioral effects of occupational exposure to cadmium: a cross sectional epidemiological study. (cdc.gov)
- 2003). "Occupational exposure to heavy metals: DNA damage induction and DNA repair inhibition prove co-exposures to cadmium, cobalt and lead as more dangerous than hitherto expected. (cdc.gov)
- The chosen IDLH is based on the statement by ACGIH [1971] that an exposure to 9 mg/m 3 cadmium fume for 5 hours is a lethal dose [Beton et al. (cdc.gov)
- It has been reported that exposure to 9 mg/m 3 of cadmium fume for 5 hours is a lethal dose [Beton et al. (cdc.gov)
- DO NOT use it to treat or manage an actual poison exposure. (medlineplus.gov)
- If you or someone you are with has an exposure, call the local emergency number (such as 911), or the local poison control center can be reached directly by calling the national toll-free Poison Help hotline (1-800-222-1222) from anywhere in the United States. (medlineplus.gov)
- 1. Metabolic effects of long-term cadmium exposure: an overview. (nih.gov)
- 3. Metabolomic biomarkers in urine of rats following long-term low-dose exposure of cadmium and/or chlorpyrifos. (nih.gov)
- 5. Environmental cadmium exposure induces alterations in the urinary metabolic profile of pregnant women. (nih.gov)
- 10. Identifying early urinary metabolic changes with long-term environmental exposure to cadmium by mass-spectrometry-based metabolomics. (nih.gov)
- 11. Biochemical and metabolic responses of the deep-sea mussel Bathymodiolus platifrons to cadmium and copper exposure. (nih.gov)
- 14. Effects of long-term cadmium exposure on urinary metabolite profiles in mice. (nih.gov)
- 15. Metabolic profiling detects early effects of environmental and lifestyle exposure to cadmium in a human population. (nih.gov)
- 17. Long-term environmental cadmium exposure induced serum metabolic changes related to renal and liver dysfunctions in a female cohort from Southwest China. (nih.gov)
- 18. Metabolomic analysis of the toxic effect of chronic exposure of cadmium on rat urine. (nih.gov)
- Poisoning occurring after exposure to cadmium compounds or fumes. (nih.gov)
- The group found cadmium up to seven times the state's maximum daily exposure limit. (institutefornaturalhealing.com)
- Heavy metal poisoning refers to when excessive exposure to a heavy metal affects the normal function of the body. (nih.gov)
- According to the Food and Drug Administration's National Clearing House for Poison Control Centers, there were only 7,710 cases of exposure to plant poisons recorded in 1975. (creation.com)
Effects of cadmium3
Kidneys6
- This concentration is approximately that at which incipient morphologic changes occur in the kidneys of cadmium-exposed workers. (cdc.gov)
- It should not be used in iron, cadmium, or selenium poisoning because the resulting dimercaprol-metal complexes are more toxic than the metal alone, especially to the kidneys. (nih.gov)
- A research conducted in the year 2018 found that ginger oil protected the kidneys of lab mice affected with cadmium poisoning. (gyalabs.com)
- Cadmium accumulates in your bones and kidneys, both directly and indirectly affecting bone health. (myersdetox.com)
- For example, due to its impact on your kidneys, cadmium can impair key kidney functions and damage osteoblast cells (the bone cells responsible for building strong bones). (myersdetox.com)
- Cadmium also poisons a crucial enzyme that converts vitamin D to its active form in the kidneys, impairing calcium uptake. (myersdetox.com)
Control Centers2
- American Association of Poison Control Centers (1-800-222-1222 or www.aapcc.org/ ) .The Poison Control Center may be contacted for questions about poisons and poisonings. (cdc.gov)
- All local poison control centers in the United States use this national number. (medlineplus.gov)
Lead6
- The report stated that adults showed that the rock and the resulting dust particles last about 14 years in this trade and are often replaced contained iron, zinc, cadmium, nickel, lead, chromium, by their children who themselves become severely ill barium, beryllium, and aluminium. (who.int)
- It is indicated in acute lead poisoning when used concomitantly with Edetate Calcium Disodium Injection USP. (nih.gov)
- The plaintiffs allege that these outdated practices caused widespread lead and cadmium poisoning, among other serious health impacts including gastrointestinal problems, kidney trouble, skin rashes, and aches and pains that are attributed to the pollution. (derechos.org)
- Thirty-five dark chocolate brands were found to contain either lead or cadmium. (institutefornaturalhealing.com)
- Not only are they without lead and cadmium, but they contain no carb-heavy pretzels or graham crackers… Just pure health-boosting dark chocolate. (institutefornaturalhealing.com)
- Cadmium, it's a heavy metal, atomic mass I believe of 114, so it's lighter than lead, but it is very, very toxic and it interferes with normal physiological processes, that are necessary for good lifelong health. (thetruthaboutcancer.com)
Selenium1
- 13. Metabolic impairments, metal traffic, and dyshomeostasis caused by the antagonistic interaction of cadmium and selenium using organic and inorganic mass spectrometry. (nih.gov)
Niosh2
- NIOSH considers cadmium compounds to be potential occupational carcinogens as defined by the OSHA carcinogen policy [29 CFR 1990]. (cdc.gov)
- If that's you, make sure you wear a NIOSH dust respirator to eliminate the chance of inhaling cadmium particles. (kingstoncove.studio)
Compounds2
- Zavon and Meadows 1970], the revised IDLH for cadmium compounds is 9 mg Cd/m 3 which was the original IDLH for cadmium fume. (cdc.gov)
- Cadmium often couples with other elements in a variety of compounds. (kingstoncove.studio)
Intoxication1
- 1979. Subacute cadmium intoxication in jewelry workers: an evaluation of diagnostic procedures. (cdc.gov)
Carcinogenic1
- This poison is cadmium, a heavy carcinogenic metal. (pltv.fr)
Occur3
- Most cases of food poisoning occur when people eat food or drink water containing bacteria, bacterial toxins (substances produced by bacteria), parasites, or viruses. (medbroadcast.com)
- A poison similar to that naturally found in the puffer fish can also occur in many edible Caribbean and Pacific species. (medbroadcast.com)
- In fact, poisons that the body cannot easily handle occur relatively rarely in nature. (creation.com)
Centers1
- The web site provides information about poison centers and poison prevention. (cdc.gov)
Symptoms4
- When Do Symptoms of Heavy metal poisoning Begin? (nih.gov)
- The bacterial causes of food poisoning tend to cause these symptoms as well as fever and headache. (medbroadcast.com)
- Many non-infectious (not caused by bacteria and their toxins, viruses, etc.) food poisoning affects the central nervous system and cause symptoms typical of nerve poisons. (medbroadcast.com)
- Scombroid (histamine) fish poisoning causes the symptoms of excess histamine. (medbroadcast.com)
Mild1
- For most people it is usually mild, but food poisoning can be severe and even deadly for some individuals. (medbroadcast.com)
Serum2
Toxins7
- Many bacteria can cause food poisoning, either directly or by the toxins they produce. (medbroadcast.com)
- Further, virtually all chemicals, even poisons and toxins, have an important function in life or human society. (creation.com)
- Likewise, many major poisons and toxins are shown to play critically important beneficial roles in society. (creation.com)
- Toxins are poisons produced by plants, animals and bacteria or found naturally in the air, water and soil. (creation.com)
- The two terms 'toxins' and 'poisons' are largely synonymous and are used here interchangeably. (creation.com)
- The terms 'poison', 'toxic', 'pesticide' and 'herbicide' all imply that because some chemicals may function as toxins in some situations, they are therefore always detrimental to humans. (creation.com)
- Furthermore, cadmium damages your kidney's ability to excrete toxins, which makes it even harder for your body to rid itself of unwanted metals[2]. (myersdetox.com)
Fumes1
- And cadmium paints don't give off any dust or fumes, so no worries about inhalation either. (kingstoncove.studio)
Severe1
- These poisons attack the central nervous system, causing partial or complete paralysis in severe cases. (medbroadcast.com)
Toxin3
- However, in some less common types of food poisoning, the poison or toxin is naturally part of the food (e.g., poisonous mushrooms or fish). (medbroadcast.com)
- Clams, mussels, oysters, and scallops can cause poisoning when they ingest certain poisonous dinoflagellates that produce the toxin saxitoxin. (medbroadcast.com)
- The term poison tends to be the lay term, while toxin is the scientific term. (creation.com)
Heavy4
- Dimercaprol Injection USP is of questionable value in poisoning caused by other heavy metals such as antimony and bismuth. (nih.gov)
- This can introduce heavy metals like cadmium into the watershed, possibly creating problems downstream. (kingstoncove.studio)
- It all starts with a heavy metal called cadmium. (thetruthaboutcancer.com)
- Cadmium is a very common heavy metal contaminant in coffee. (thetruthaboutcancer.com)
Dangers1
- They told me that by law, paint manufacturers are allowed to make cadmium colors only a few specified days each year because of the dangers associated with cadmium dust. (kingstoncove.studio)
Lung3
- 1992. Determinants of lung cancer risk among cadmium-exposed workers. (cdc.gov)
- Lung cancer mortality in a cohort of workers employed at a cadmium recovery plant in the United States: an analysis with detailed job histories. (cdc.gov)
- This concentration would result in a human lung cadmium burden in 30 minutes of no more than 8 or 9 mg. (cdc.gov)
Chemicals1
- The problem of poisons is considered, and it is concluded that a false dichotomy exists between poisonous and non-poisonous chemicals. (creation.com)
Workers1
- Workers had inhaled cadmium dust as a result of polishing silver with cadmium carbonate. (kingstoncove.studio)
Respiratory1
- Proper respiratory equipment is required during production to avoid inhalation of the powdered cadmium pigment. (kingstoncove.studio)
Excessive1
- The disease results from excessive cadmium poisoning and was first reported in a small town about 200 miles north west of Tokyo. (ms-moskevska.cz)
Pesticide1
- Objective 8-27 tracks the monitoring of these diseases as well as 3 others: pesticide poisoning, asthma, and birth defects. (cdc.gov)
Liver2
- 2. Screening and validation of biomarkers for cadmium-induced liver injury based on targeted bile acid metabolomics. (nih.gov)
- People at greatest risk for food poisoning are seniors, pregnant women, young children and babies, and people with chronic medical conditions (e.g., diabetes, AIDS, liver disease). (medbroadcast.com)
Naturally1
- Cadmium is found naturally in the earth's crust but is a relatively rare metal. (kingstoncove.studio)
Occurs2
- Solder poisoning occurs when someone swallows solder in large amounts. (medlineplus.gov)
- Food poisoning occurs when contaminated food or water is ingested. (medbroadcast.com)
Organs1
- 7. Toxicology of cadmium and its damage to mammalian organs. (nih.gov)
Substance1
- A poison is any substance that produces injury to the body by chemical means. (creation.com)
Diagnosis1
- AAPC does not provide information about treatment or diagnosis of poisoning or research information for student papers. (cdc.gov)
Dust1
- A rabbit 30-minute LC 50 of about 8,000 mg-min/m 3 for cadmium dust has been reported [Friberg et al. (cdc.gov)
Levels3
- OSHA Standards - Cadmium - includes a full description of required monitoring and actions required at different cadmium levels. (cdc.gov)
- 16. Urine metabolomics of women from small villages exposed to high environmental cadmium levels. (nih.gov)
- But it's not just the planet that's collapsing - the fisheries collapse of the West Coast has now reached "catastrophic" levels - it's also the fact that humans are so incredibly stupid, they deliberately poison their own food supply with toxic pesticides , herbicides, fungicides, GMOs and hormone disruptors like atrazine , a "gender-bender" chemical that feminizes males. (naturalnews.com)
Health2
- 1984. Cadmium, the environment and human health: an overview. (cdc.gov)
- Do not make the person throw up unless poison control or a health care provider tells you to. (medlineplus.gov)
Prevention1
- You should call if you have any questions about poisoning or poison prevention. (medlineplus.gov)
Fertilizers1
- With nearly 150 samples, they develop an unprecedented study on a poison hidden in these fertilizers and which contaminates potatoes. (pltv.fr)
Largely1
- What the entire "scientific" establishment is largely ignoring, however, is the agricultural poisoning of our food supply with toxic pesticides, herbicides and self-replicating GMOs. (naturalnews.com)
Found1
- There are many types of poisons found in insecticides but the most dangerous types are the organophosphates , which are basically nerve gas for insects. (medbroadcast.com)
Bacteria1
- In most cases, bacteria, viruses, or parasites are transferred to food from other sources, making these organisms the most common causes of food poisoning. (medbroadcast.com)
Cases1
- Some of the earliest cases of cadmium poisoning were reported in Belgium in 1858. (kingstoncove.studio)
Nervous2
- This poison attacks the nervous system. (medbroadcast.com)
- Mushroom poisoning also attacks the nervous system. (medbroadcast.com)
Human3
- Cadmium in human population. (cdc.gov)
- 12. Cadmium perturbed metabolomic signature in pancreatic beta cells correlates with disturbed metabolite profile in human urine. (nih.gov)
- This process renders the cadmium insoluble in water, and therefore the human body. (kingstoncove.studio)
Data1
- Data on the dose-response relationship for cadmium are scarce and uncertain. (cdc.gov)
People2
- So, the reason I mention all of that is because cadmium can make people feel high, makes people feel strong. (thetruthaboutcancer.com)
- So, you might be drinking a cup of coffee and thinking you're getting energized from the caffeine, and that may be true, but for a lot of people they're getting a cadmium rush. (thetruthaboutcancer.com)