Substances that increase the risk of NEOPLASMS in humans or animals. Both genotoxic chemicals, which affect DNA directly, and nongenotoxic chemicals, which induce neoplasms by other mechanism, are included.
The chemical alteration of an exogenous substance by or in a biological system. The alteration may inactivate the compound or it may result in the production of an active metabolite of an inactive parent compound. The alterations may be divided into METABOLIC DETOXICATION, PHASE I and METABOLIC DETOXICATION, PHASE II.
Tumors or cancer of the LUNG.
Nutritional factor found in milk, eggs, malted barley, liver, kidney, heart, and leafy vegetables. The richest natural source is yeast. It occurs in the free form only in the retina of the eye, in whey, and in urine; its principal forms in tissues and cells are as FLAVIN MONONUCLEOTIDE and FLAVIN-ADENINE DINUCLEOTIDE.
Powdered or cut pieces of leaves of NICOTIANA TABACUM which are inhaled through the nose, chewed, or stored in cheek pouches. It includes any product of tobacco that is not smoked.
A nitrosamine derivative with alkylating, carcinogenic, and mutagenic properties. It causes serious liver damage and is a hepatocarcinogen in rodents.
Aryl Hydrocarbon Hydroxylases
A large group of cytochrome P-450 (heme-thiolate) monooxygenases that complex with NAD(P)H-FLAVIN OXIDOREDUCTASE in numerous mixed-function oxidations of aromatic compounds. They catalyze hydroxylation of a broad spectrum of substrates and are important in the metabolism of steroids, drugs, and toxins such as PHENOBARBITAL, carcinogens, and insecticides.
The products of chemical reactions that result in the addition of extraneous chemical groups to DNA.
Artifactual vesicles formed from the endoplasmic reticulum when cells are disrupted. They are isolated by differential centrifugation and are composed of three structural features: rough vesicles, smooth vesicles, and ribosomes. Numerous enzyme activities are associated with the microsomal fraction. (Glick, Glossary of Biochemistry and Molecular Biology, 1990; from Rieger et al., Glossary of Genetics: Classical and Molecular, 5th ed)
An enzyme that catalyzes the formation of riboflavin from two molecules of 6,7-dimethyl-8-ribityllumazine, utilizing a four-carbon fragment from one molecule which is transferred to the second molecule. EC 126.96.36.199.
A naturally occurring furocoumarin compound found in several species of plants, including Psoralea corylifolia. It is a photoactive substance that forms DNA ADDUCTS in the presence of ultraviolet A irradiation.
Gas Chromatography-Mass Spectrometry
Chromatography, High Pressure Liquid
Closed vesicles of fragmented endoplasmic reticulum created when liver cells or tissue are disrupted by homogenization. They may be smooth or rough.
Cytochrome P-450 Enzyme System
A superfamily of hundreds of closely related HEMEPROTEINS found throughout the phylogenetic spectrum, from animals, plants, fungi, to bacteria. They include numerous complex monooxygenases (MIXED FUNCTION OXYGENASES). In animals, these P-450 enzymes serve two major functions: (1) biosynthesis of steroids, fatty acids, and bile acids; (2) metabolism of endogenous and a wide variety of exogenous substrates, such as toxins and drugs (BIOTRANSFORMATION). They are classified, according to their sequence similarities rather than functions, into CYP gene families (>40% homology) and subfamilies (>59% homology). For example, enzymes from the CYP1, CYP2, and CYP3 gene families are responsible for most drug metabolism.
Metabolic Detoxication, Drug
Mixed Function Oxygenases
Widely distributed enzymes that carry out oxidation-reduction reactions in which one atom of the oxygen molecule is incorporated into the organic substrate; the other oxygen atom is reduced and combined with hydrogen ions to form water. They are also known as monooxygenases or hydroxylases. These reactions require two substrates as reductants for each of the two oxygen atoms. There are different classes of monooxygenases depending on the type of hydrogen-providing cosubstrate (COENZYMES) required in the mixed-function oxidation.
The infusion of leaves of CAMELLIA SINENSIS (formerly Thea sinensis) as a beverage, the familiar Asian tea, which contains CATECHIN (especially epigallocatechin gallate) and CAFFEINE.
A nucleoside consisting of the base guanine and the sugar deoxyribose.
Addition of methyl groups. In histo-chemistry methylation is used to esterify carboxyl groups and remove sulfate groups by treating tissue sections with hot methanol in the presence of hydrochloric acid. (From Stedman, 25th ed)
Carbon monoxide (CO). A poisonous colorless, odorless, tasteless gas. It combines with hemoglobin to form carboxyhemoglobin, which has no oxygen carrying capacity. The resultant oxygen deprivation causes headache, dizziness, decreased pulse and respiratory rates, unconsciousness, and death. (From Merck Index, 11th ed)
Spectrometry, Mass, Electrospray Ionization
A mass spectrometry technique used for analysis of nonvolatile compounds such as proteins and macromolecules. The technique involves preparing electrically charged droplets from analyte molecules dissolved in solvent. The electrically charged droplets enter a vacuum chamber where the solvent is evaporated. Evaporation of solvent reduces the droplet size, thereby increasing the coulombic repulsion within the droplet. As the charged droplets get smaller, the excess charge within them causes them to disintegrate and release analyte molecules. The volatilized analyte molecules are then analyzed by mass spectrometry.
Measurable and quantifiable biological parameters (e.g., specific enzyme concentration, specific hormone concentration, specific gene phenotype distribution in a population, presence of biological substances) which serve as indices for health- and physiology-related assessments, such as disease risk, psychiatric disorders, environmental exposure and its effects, disease diagnosis, metabolic processes, substance abuse, pregnancy, cell line development, epidemiologic studies, etc.
A deoxyribonucleotide polymer that is the primary genetic material of all cells. Eukaryotic and prokaryotic organisms normally contain DNA in a double-stranded state, yet several important biological processes transiently involve single-stranded regions. DNA, which consists of a polysugar-phosphate backbone possessing projections of purines (adenine and guanine) and pyrimidines (thymine and cytosine), forms a double helix that is held together by hydrogen bonds between these purines and pyrimidines (adenine to thymine and guanine to cytosine).
A subfamily in the family MURIDAE, comprising the hamsters. Four of the more common genera are Cricetus, CRICETULUS; MESOCRICETUS; and PHODOPUS.
One of the two major classes of cholinergic receptors. Nicotinic receptors were originally distinguished by their preference for NICOTINE over MUSCARINE. They are generally divided into muscle-type and neuronal-type (previously ganglionic) based on pharmacology, and subunit composition of the receptors.
A chemical reaction in which an electron is transferred from one molecule to another. The electron-donating molecule is the reducing agent or reductant; the electron-accepting molecule is the oxidizing agent or oxidant. Reducing and oxidizing agents function as conjugate reductant-oxidant pairs or redox pairs (Lehninger, Principles of Biochemistry, 1982, p471).
Fractionation of a vaporized sample as a consequence of partition between a mobile gaseous phase and a stationary phase held in a column. Two types are gas-solid chromatography, where the fixed phase is a solid, and gas-liquid, in which the stationary phase is a nonvolatile liquid supported on an inert solid matrix.
Slow oxidation of acetoxime and methylethyl ketoxime to the corresponding nitronates and hydroxy nitronates by liver microsomes from rats, mice, and humans. (1/233)Acetoxime and methylethyl ketoxime (MEKO) are tumorigenic in rodents, inducing liver tumors in male animals. The mechanisms of tumorigenicity for these compounds are not well defined. Oxidation of the oximes to nitronates of secondary-nitroalkanes, which are mutagenic and tumorigenic in rodents, has been postulated to play a role in the bioactivation of ketoximes. In these experiments, we have compared the oxidation of acetoxime and methylethyl ketoxime to corresponding nitronates in liver microsomes from different species. The oximes were incubated with liver microsomes from mice, rats, and several human liver samples. After tautomeric equilibration and extraction with n-hexane, 2-nitropropane and 2-nitrobutane were quantitated by GC/MS-NCI (limit of detection of 250 fmol/injection volume). In liver microsomes, nitronate formation from MEKO and acetoxime was dependent on time, enzymatically active proteins, and the presence of NADPH. Nitronate formation was increased in liver microsomes of rats pretreated with inducers of cytochrome P450 and reduced in the presence of inhibitors (n-octylamine and diethyldithiocarbamate). Rates of oxidation of MEKO (Vmax) were 1.1 nmol/min/mg (mice), 0.5 nmol/min/mg (humans), and 0.1 nmol/min/mg (rats). In addition to nitronates, several minor metabolites were also enzymatically formed (two diastereoisomers of 3-nitro-2-butanol, 2-hydroxy-3-butanone oxime and 2-nitro-1-butanol). Acetoxime was also metabolized to the corresponding nitronate at rates approximately 50% of those observed with MEKO oxidation in the three species examined. 2-Nitro-1-propanol was identified as a minor product formed from acetoxime. No sex differences in the capacity to oxidize acetoxime and MEKO were observed in the species examined. The observed results show that formation of sec-nitronates from ketoximes occurs slowly, but is not the only pathway involved in the oxidative biotransformation of these compounds. Due to the lack of sex-specific oxidative metabolism, other metabolic pathways or mechanisms of tumorigenicity not involving bioactivation may be involved in the sex-specific tumorigenicity of ketoximes in rodents. (+info)
A placebo-controlled study of interaction between nabumetone and acenocoumarol. (2/233)AIMS: The use of nonsteroidal anti-inflammatory drugs (NSAIDs) in patients treated with oral anticoagulants is generally discouraged due to the risk of interactions that could increase the risk of bleeding complications. Available data suggest the NSAID, nabumetone, does not produce such an interaction. We investigated whether nabumetone would interact with acenocoumarol, an oral anticoagulant widely used in some European countries. METHODS: A double-blind, randomized, placebo-controlled study was conducted evaluating nabumetone (1-2 g daily for up to 4 weeks) in osteoarthritis patients with thromboembolic risk previously stabilized on acenocoumarol. The primary efficacy end point was the proportion of patients whose International Normalized Ratio (INR) remained within established margins and whose acenocoumarol dose was not changed. Fifty-six patients were randomized to receive nabumetone (n=27) or placebo (n=29). RESULTS: Eighteen patients in each group (67% for nabumetone and 62% for placebo) completed the study without showing INR or acenocoumarol dose changes, and were considered as study successes. Nine patients (33%) with nabumetone and 11 (38%) with placebo were considered study failures in the intention-to-treat analysis (one patient on nabumetone and four on placebo did not complete the study due to reasons not related to INR and acenocoumarol dose changes). No significant differences were found between groups with regard to study successes. There were two minor bleeding complications, one in each group. Six patients per group presented with eight adverse experiences in each group. CONCLUSIONS: Treatment with nabumetone did not alter INR levels compared with placebo in patients stabilized on oral acenocoumarol who require NSAID therapy. These results suggest that nabumetone does not produce a clinically relevant interaction with acenocoumarol. In orally anticoagulated patients without other associated risk factors, treatment with nabumetone for up to 4 weeks does not require increased monitoring of INR levels. (+info)
Urinary volatile constituents of the lion, Panthera leo. (3/233)The volatile components of urine from lions were investigated using GC-MS headspace techniques. Fifty-five compounds were found in the urine samples. Seven potential species-identifying compounds were found. Male lion scent marks overlapped significantly more in compound composition with other males than they did with female marks. A similar relationship was not found for the females. Males had a significantly higher absolute content of 2-butanone in their urine than females, and females had a significantly higher relative content of acetone than males. Samples from 13/16 individual lions overlapped more within the individual than they did with samples from the other individuals, but only seven significantly so. (+info)
A functional arginine residue in rabbit-muscle aldolase. (4/233)Rabbit muscle aldolase is irreversibly modified by the arginine-selective alpha-dicarbonyl, phenylglyoxal, loss of activity correlating with the unique modifications of one arginine residue per subunit, as determined by amino acid analysis, and (7-14C)phenylglyoxal incorporation. The affinity of the modified enzyme for dihydroxyacetone phosphate is significantly reduced while substantial protection against inactivation is afforded by fructose 1,6-disphosphate, dihydroxyacetone phosphate or phosphate ion. The nature of the substrate C-1 phosphate binding site in this enzyme is discussed in the light of these and other results. (+info)
Effects of nabumetone compared with naproxen on platelet aggregation in patients with rheumatoid arthritis. (5/233)OBJECTIVE: To test the hypothesis that nabumetone (a partially selective cyclooxygenase-(COX)-2 inhibitor) has less effect on platelet aggregation than naproxen (a non-selective COX-inhibitor) in patients with rheumatoid arthritis (RA). METHODS: A crossover study in 10 RA patients was performed, using either nabumetone or naproxen for two weeks, and, after a washout period of two weeks, the other drug during another two weeks. Platelet aggregation studies were performed and bleeding time was assessed before and after each treatment period. RESULTS: Maximum platelet aggregation induced by epinephrine and by collagen was significantly more reduced after the use of naproxen than of nabumetone; secondary aggregation induced by ADP and epinephrine disappeared more often by naproxen than by nabumetone. Bleeding times were not influenced. CONCLUSION: COX dependent platelet aggregation in RA patients seems to be more inhibited by naproxen than by nabumetone. This may be relevant for patients requiring non-steroidal anti-inflammatory drug treatment but who have an increased risk of bleeding as well. (+info)
Reductive metabolism In vivo of trans-4-phenyl-3-buten-2-one in rats and dogs. (6/233)The reductive metabolism in vivo of a flavoring additive, trans-4-phenyl-3-buten-2-one (PBO; trans-methyl styryl ketone) was investigated in rats and dogs. In both species, the double bond-reduced product, 4-phenyl-2-butanone (PBA), was detected by HPLC as the predominant species in blood after i.v. administration of PBO. PBA detected in rat blood was identified by comparison to the authentic sample. In contrast, the carbonyl-reduced product, trans-4-phenyl-3-buten-2-ol (PBOL) was also detected as a minor metabolite of PBO in both species. The area under the curve of PBOL in rat blood was only 3% of that of PBA. PBO was mutagenic in the Ames test using Salmonella typhimurium TA 100 when S-9 mix was added, but PBA and PBOL were not. It appears that PBO is mainly metabolized to PBA in vivo in rats and dogs as a detoxification pathway. (+info)
Non-steroidal anti-inflammatory drugs and renal response to exercise: a comparison of indomethacin and nabumetone. (7/233)Nabumetone, a newer non-steroidal anti-inflammatory drug (NSAID) which preferentially blocks cyclo-oxygenase-2 activity, may be less nephrotoxic than indomethacin. This study tested whether nabumetone has effects different from those of indomethacin on exercise-induced changes in renal function and the renin-aldosterone system. In a randomized fashion, ten subjects were studied after indomethacin (100 mg), nabumetone (1 g) or no medication (control) administered orally at 22.00 hours on the day before each study day, and again at 8.00 hours upon arrival at the laboratory. Renal function was studied at baseline, during graded 20-min exercise sessions at 25%, 50% and 75% of the maximal oxygen uptake rate, and subsequently during two 1-h recovery periods. Heart rate, arterial blood pressure, cardiac output and plasma catecholamines at rest and during exercise were not altered by indomethacin or nabumetone. Indomethacin decreased urinary rates of excretion of 6-oxo-prostaglandin F(1alpha) (6-oxo-PGF(1alpha)) and thromboxane B(2) in all study periods. Nabumetone decreased 6-oxo-PGF(1alpha) excretion during and after exercise. Excretion rates for PGE(2) did not change. Neither indomethacin nor nabumetone changed baseline values or exercise-induced decreases in renal plasma flow or glomerular filtration rate. Indomethacin, but not nabumetone, decreased sodium excretion, urine flow rate and free water clearance. The renal response to exercise, however, remained unchanged. In contrast with nabumatone, indomethacin decreased the plasma renin concentration. Thus, during exercise, nabumetone may decrease the excretion of 6-oxo-PGF(1alpha) by inhibition of cyclo-oxygenase-1 or by inhibition of specific exercise-induced activation of cyclo-oxygenase-2, or both. None of the drugs changed the renal response to exercise. Inhibition by indomethacin of angiotensin II and thromboxane A(2) synthesis may, during exercise, counterbalance renal vasoconstriction caused by blockade of vasodilatory prostaglandins. (+info)
Safety and efficacy of nabumetone in osteoarthritis: emphasis on gastrointestinal safety. (8/233)AIM: To compare the efficacy and gastrointestinal (GI) safety of nabumetone with two comparator non-steroidal anti-inflammatory drugs (NSAIDs), diclofenac SR and piroxicam. METHODS: Two randomized, double-blind, multicentre, parallel group trials were carried out in patients with moderate to severe osteoarthritis of the hip or knee. During the 6 month treatment phase, the safety and efficacy of nabumetone (1500-2000 mg/day) was compared to diclofenac SR (100 mg/day) or piroxicam (20-30 mg/day). GI safety was evaluated by reviewing all adverse events reported during the trials and presenting all cases of ulcers (complicated and uncomplicated), as well as other bleeding events that may have been associated with NSAID administration. RESULTS: Most of the efficacy parameters showed no significant differences between the NSAIDs, although diclofenac SR was significantly better than nabumetone in one of 18 efficacy parameters. Nabumetone-treated patients experienced significantly fewer ulcer and bleeding events compared to patients treated with the comparator NSAIDs [1.1% (4/348) vs. 4.3% (15/346), P = 0.01]. Bleeding events, including outright upper or lower GI bleeding or a significant decline in haemoglobin, occurred in significantly fewer patients treated with nabumetone than with the comparator NSAIDs [1.1% (4/348) vs. 3.5% (12/346), P < 0.05]. More importantly, complications associated with either ulcers (perforation) or bleeding (leading to hospitalization or withdrawal) occurred in significantly fewer patients receiving nabumetone [0% (0/348)] than with comparator NSAIDs [1.4% (5/346), (P < 0.05)]. CONCLUSION: The results suggest that nabumetone was similar in efficacy by most criteria to diclofenac SR and piroxicam in relieving the symptoms of osteoarthritis; however, nabumetone's GI safety profile was generally superior to that of both comparator NSAIDs. In the pooled analysis, nabumetone was associated with a significantly lower total incidence of ulcers and bleeding events, and a significantly lower incidence of complications associated with these events. (+info)
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Methyl ethyl ketone (MEK) possesses characteristics of high evaporation rate, solvency, and viscosity. Its low boiling point coupled with its sweet smell of acetone and butterscotch will aid its market prospects. It is used in many industrial procedures, mainly resins, cellulose nitrate, and cellulose gum.. The worldwide Methyl Ethyl Ketone Market is projected to surpass revenue figures by USD 3.26 million by 2024. It is primarily driven by demand of paints and coatings by home furnishing, infrastructure, and automotive sectors. Increasing demand from Asia Pacific and Latin American construction & automotive sectors will further fuel market growth. It is estimated to grow at a 4.6% CAGR over the forecast period (2016-2024).. The global market is segmented according to applications and regions. Major market applications include paints & coatings, adhesives, printing inks, and others. Others are textiles, plastics, rubber-based industrial cement, PVC piping, chemical intermediaries, and cleaning ...
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2-Butanone; Butan-2-one; Butanone; Ethyl methyl ketone; Ketone, methyl ethyl; Methyl ethyl ketone; MEK; C2H5COCH3; Acetone, methyl-; Aethylmethylketon; 3-Butanone; Butanone 2; Ethyl methyl cetone; Ethylmethylketon; Ketone, ethyl methyl; Meetco; Methyl acetone; Metiletilchetone; Metyloetyloketon; Rcra waste number U159; 2-Oxobutane; Meketone; Oxobutane; 2-butanone (MEK; 2- ...
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The kinetics of the reaction between oxygen and gaseous methyl ethyl ketone (butanone) have been investigated in the range 250 to 450° C. The anomalous temperature coefficient of the rate offers a good example of the transition from a low-temperature mode of oxidation to a high-temperature mode. Similar behaviour has been observed with hydrocarbons and is ascribable to the diminishing part played at higher temperatures by the peroxides, which at low temperatures play a key role in generating branching chains. In the low-temperature range the rate of oxidation varies steeply with the initial pressure of butanone and is a very unusual function of the oxygen pressure, passing through a sharp maximum at certain pressures above which the rate falls and then tends to become constant. The maximum tends to disappear as the temperature is raised. Increase of the surface / volume ratio causes a diminution in rate, while addition of inert gases has no appreciable influence. The various complex ...
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https://www.hexaresearch.com/research-report/methyl-ethyl-ketone-mek-market. The worldwide Methyl Ethyl Ketone Market is projected to surpass revenue figures by USD 3.26 million by 2024. It is primarily driven by demand of paints and coatings by home furnishing, infrastructure, and automotive sectors. Increasing demand from Asia Pacific and Latin American construction & automotive sectors will further fuel market growth. It is estimated to grow at a 4.6% CAGR over the forecast period (2016-2024).. The global market is segmented according to applications and regions. Major market applications include paints & coatings, adhesives, printing inks, and others. Others are textiles, plastics, rubber-based industrial cement, PVC piping, chemical intermediaries, and cleaning agents.. Printing inks are the major application of the MEK market, due to its use as a solvent. Paint thinners & resins are used as solvents in the pharmaceutical industry. But paints & coatings will garner major market demand ...
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Chemoprevention of intestinal tumorigenesis by nabumetone: Induction of apoptosis and Bcl-2 downregulation<...
TY - JOUR. T1 - Chemoprevention of intestinal tumorigenesis by nabumetone. T2 - Induction of apoptosis and Bcl-2 downregulation. AU - Roy, H. K.. AU - Karoski, W. J.. AU - Ratashak, A.. AU - Smyrk, T. C.. PY - 2001/5/18. Y1 - 2001/5/18. N2 - Treatment of MIN mice with the nonsteroidal anti-inflammatory drug, nabumetone, resulted in a dose-dependent suppression of intestinal tumorigenesis. In both the uninvolved MIN mouse colonic epithelium and HT-29 colon cancer cells, nabumetone downregulated the anti-apoptotic protein, Bcl-2, with concomitant induction of apoptosis, suggesting a potential mechanism for colon cancer chemoprevention.. AB - Treatment of MIN mice with the nonsteroidal anti-inflammatory drug, nabumetone, resulted in a dose-dependent suppression of intestinal tumorigenesis. In both the uninvolved MIN mouse colonic epithelium and HT-29 colon cancer cells, nabumetone downregulated the anti-apoptotic protein, Bcl-2, with concomitant induction of apoptosis, suggesting a potential ...
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3.3.co;2-k. Can, N O; Tuncel, M; Aboul-Enein, H Y (2003). Determination of nabumetone in pharmaceutical formulation by flow injection analysis (FIA) with UV-detection. Pharmazie. 58: 22-24. PMID 12622247. Mikami, E; Goto, T; Ohno, T; Matsumoto, H; Nishida, M (2000). Simultaneous analysis of naproxen, nabumetone and its major metabolite 6-methoxy-2-naphthylacetic acid in pharmaceuticals and human urine by high-performance liquid chromatography. J Pharm Biomed Anal. 23: 917-925. doi:10.1016/s0731-7085(00)00365-4. Kobylińska, Kamila; Barlińska, Małgorzata; Kobylińska, Maria. Analysis of nabumetone in human plasma by HPLC. Application to single dose pharmacokinetic studies. J Pharm Biomed Anal. 2003 (32): 323-328. doi:10.1016/S0731-7085(03)00078-5. Nobilis, M; Kopecký, J; Kv, Tina J; Svoboda, Z; Pour, M; Kune, J; Hol, Apek M; Kolá, Ová L (2003). Comparative biotransformation and disposition studies of nabumetone in humans and minipigs using high-performance liquid chromatography with ...
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Cumene process - Wikipedia
Raspberry Ketone Reduced Lipid Accumulation in 3T3-L1 Cells and Ovariectomy-Induced Obesity in Wistar Rats by Regulating...
Mercapturic acid urinary metabolites of 3-butene-1,2-diol as in vivo evidence for the formation of hydroxymethylvinyl ketone in...
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Patent US6930197 - Antihistaminic piperidine derivatives and intermediates for the preparation ... - Google Patents
Cancer Institute, Mechanisms of Carcinogenesis - Research Output - Penn State
4-(Methylnitrosamino)-I-(3-pyridyl)-1-butanone enhances the expression of apolipoprotein A-I and Clara cell 17-kDa protein in...
TY - JOUR. T1 - 4-(Methylnitrosamino)-I-(3-pyridyl)-1-butanone enhances the expression of apolipoprotein A-I and Clara cell 17-kDa protein in the lung proteomes of rats fed a corn oil diet but not a fish oil diet. AU - Sung, Il Chang. AU - El-Bayoumy, Karam. AU - Sinha, Indu. AU - Trushin, Neil. AU - Stanley, Bruce. AU - Pittman, Brian. AU - Prokopczyk, Bogdan. PY - 2007/2/1. Y1 - 2007/2/1. N2 - The nicotine-derived nitrosamine 4-(methylnitrosamino)-1-(3-pyridyl)-1- butanone (NNK) is one of the most potent lung carcinogens in rodents. Several epidemiologic studies indicated that the development of lung cancer in smokers is influenced by the type and amount of dietary polyunsaturated fatty acids. A high corn oil diet has been shown to increase lung tumor volume and to decrease tumor latency in rats treated with NNK. In this study, we investigated the effects of dietary ...
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Cisneros Puebla, C. A., Alatorre, F., Allen, M., Bénard, S., Castañeda, D., Castañeda, Y., Charmaz, K., Davidson, J., de la Garza, S. A., Espinoza, A., Faulkner, S. L., Ferreyra, G., Gilgun, J., Gutiérrez, L. F. G., Hein, S. F., Hernández, D., Biber, S. H., Labarthé, V. J., Johnson, J. M., Keller, R. & 10 others, Lather, P., Long, T. A., Pourreau, L., Rawlins, W. K., Rinehart, R. E., Donoso, G. R., Ryen, A., Orozco, M. A. S., Tamas, S. & Tilley-Lubbs, G. A., Jul 1 2016, In : Qualitative Inquiry. 22, 6, p. 447-465 19 p.. Research output: Contribution to journal › Article ...
DiVA - Search result
US6797665B2 - Double-metal cyanide catalysts for preparing polyether polyols - Google Patents
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Sawabata, N., Fujii, Y., Asamura, H., Nomori, H., Nakanishi, Y., Eguchi, K., Mori, M., Okumura, M., Miyaoka, E., Yokoi, K. & Japanese Joint Committee for Lung Cancer Registration, J. C. F. L. C. R., Apr 1 2011, In : Nihon Kokyūki Gakkai zasshi = the journal of the Japanese Respiratory Society. 49, 4, p. 327-342 16 p.. Research output: Contribution to journal › Article ...
A Level GCE How to name Aldehydes How do you name Ketones? Naming and Structure of Aldehydes and Ketones nomenclature isomers...
Peterson, L. A.<...
Adaptation of rat olfactory bulb neurones.
WHO HQ Library catalog › MARC details for record no. 8942
Olfaction and odor discrimination are mediated by the C. elegans guanylyl cyclase ODR-1. - PubMed - NCBI
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The EPR spectra of rat jejunal homogenates at 20 K afte | Open-i
DeCS Ingl s+escopo
D02.522.296 Butanones .. D02.522.296.100 Acetoin .. D02.522.585 Ketone Bodies .. D02.522.585.175 Acetoacetates .. D02.886 ...
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Butanones - toxicity , Chemical properties , Smoking , Index Medicus , Acroleins , Membranes , Inhibitors , Smoke , Kinases , ... Butanones - toxicity , Muscle, Smooth, Vascular - drug effects , Medical colleges , Gliomas , Protein kinases , Index Medicus ... Butanones - chemistry , Tobacco Smoke Pollution - adverse effects , Cell-Free System , Mass Spectrometry , Nicotine - chemistry ...
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Consciously Deciding with Guest Dave Asprey: MakingBank S2E17 - Josh Felber
Butanones and ketones do the same thing.. So, if you imagine this clock thats turning or a wheel thats turning, or even a ...
Ketones | Harvard Catalyst Profiles | Harvard Catalyst
Robson M, Ruddy KJ, Im SA, Senkus E, Xu B, Domchek SM, Masuda N, Li W, Tung N, Armstrong A, Delaloge S, Bannister W, Goessl C, Degboe A, Hettle R, Conte P. Patient-reported outcomes in patients with a germline BRCA mutation and HER2-negative metastatic breast cancer receiving olaparib versus chemotherapy in the OlympiAD trial. Eur J Cancer. 2019 10; 120:20-30 ...
Butanedione | definition of Butanedione by Medical dictionary
Uniqsis Ltd - Flow Chemistry Publications
Welcome to CDC stacks | Toxicological profile for 2-butanone - 6165 | Stephen B. Thacker CDC Library collection
- Butanones--adverse effects--toxicity. (who.int)
- A number to denote the position of the ketone group is definitely required beyond butanone and although not strictly needed for butan-2-one, since only one ketone position is possible, it is required for substituted butanones and beyond. (docbrown.info)