Bupropion
Antidepressive Agents, Second-Generation
Dopamine Uptake Inhibitors
Smoking Cessation
Nicotine
Delayed-Action Preparations
Nicotinic Agonists
Drugs that bind to and activate nicotinic cholinergic receptors (RECEPTORS, NICOTINIC). Nicotinic agonists act at postganglionic nicotinic receptors, at neuroeffector junctions in the peripheral nervous system, and at nicotinic receptors in the central nervous system. Agents that function as neuromuscular depolarizing blocking agents are included here because they activate nicotinic receptors, although they are used clinically to block nicotinic transmission.
Tobacco Use Disorder
Nomifensine
An isoquinoline derivative that prevents dopamine reuptake into synaptosomes. The maleate was formerly used in the treatment of depression. It was withdrawn worldwide in 1986 due to the risk of acute hemolytic anemia with intravascular hemolysis resulting from its use. In some cases, renal failure also developed. (From Martindale, The Extra Pharmacopoeia, 30th ed, p266)
Aryl Hydrocarbon Hydroxylases
A large group of cytochrome P-450 (heme-thiolate) monooxygenases that complex with NAD(P)H-FLAVIN OXIDOREDUCTASE in numerous mixed-function oxidations of aromatic compounds. They catalyze hydroxylation of a broad spectrum of substrates and are important in the metabolism of steroids, drugs, and toxins such as PHENOBARBITAL, carcinogens, and insecticides.
Dipyrone
Herb-Drug Interactions
Methamphetamine
Antidepressive Agents
Mood-stimulating drugs used primarily in the treatment of affective disorders and related conditions. Several MONOAMINE OXIDASE INHIBITORS are useful as antidepressants apparently as a long-term consequence of their modulation of catecholamine levels. The tricyclic compounds useful as antidepressive agents (ANTIDEPRESSIVE AGENTS, TRICYCLIC) also appear to act through brain catecholamine systems. A third group (ANTIDEPRESSIVE AGENTS, SECOND-GENERATION) is a diverse group of drugs including some that act specifically on serotonergic systems.
Hydroxylation
Substance Withdrawal Syndrome
Double-Blind Method
A controlled trial of sustained-release bupropion, a nicotine patch, or both for smoking cessation. (1/379)
BACKGROUND AND METHODS: Use of nicotine-replacement therapies and the antidepressant bupropion helps people stop smoking. We conducted a double-blind, placebo-controlled comparison of sustained-release bupropion (244 subjects), a nicotine patch (244 subjects), bupropion and a nicotine patch (245 subjects), and placebo (160 subjects) for smoking cessation. Smokers with clinical depression were excluded. Treatment consisted of nine weeks of bupropion (150 mg a day for the first three days, and then 150 mg twice daily) or placebo, as well as eight weeks of nicotine-patch therapy (21 mg per day during weeks 2 through 7, 14 mg per day during week 8, and 7 mg per day during week 9) or placebo. The target day for quitting smoking was usually day 8. RESULTS: The abstinence rates at 12 months were 15.6 percent in the placebo group, as compared with 16.4 percent in the nicotine-patch group, 30.3 percent in the bupropion group (P<0.001), and 35.5 percent in the group given bupropion and the nicotine patch (P<0.001). By week 7, subjects in the placebo group had gained an average of 2.1 kg, as compared with a gain of 1.6 kg in the nicotine-patch group, a gain of 1.7 kg in the bupropion group, and a gain of 1.1 kg in the combined-treatment group (P<0.05). Weight gain at seven weeks was significantly less in the combined-treatment group than in the bupropion group and the placebo group (P<0.05 for both comparisons). A total of 311 subjects (34.8 percent) discontinued one or both medications. Seventy-nine subjects stopped treatment because of adverse events: 6 in the placebo group (3.8 percent), 16 in the nicotine-patch group (6.6 percent), 29 in the bupropion group (11.9 percent), and 28 in the combined-treatment group (11.4 percent). The most common adverse events were insomnia and headache. CONCLUSIONS: Treatment with sustained-release bupropion alone or in combination with a nicotine patch resulted in significantly higher long-term rates of smoking cessation than use of either the nicotine patch alone or placebo. Abstinence rates were higher with combination therapy than with bupropion alone, but the difference was not statistically significant. (+info)Pharmacological characterization of nicotine's interaction with cocaine and cocaine analogs. (2/379)
Cocaine and a number of 3beta-phenyltropane cocaine analogs were investigated for their potential to block various pharmacological effects of nicotine in animals. They blocked the antinociceptive effect of nicotine in the tail-flick test after systemic administration in a dose-dependent manner. Similarly, cocaine was also able to block nicotine-induced motor impairment in mice. Furthermore, cocaine blocked nicotine-induced seizures at a lower potency than for antinociception, but failed to block nicotine's effect on body temperature and drug discrimination. The antagonistic potencies of the 3beta-phenyltropane cocaine analogs were not correlated with their affinity for monoamines transporters. Additionally, bupropion, nomifensin, GBR 12909, and nisoxetine, but not methylphenidate and fluoxetine, blocked nicotine-induced antinociception; however, their antagonistic potencies were unrelated to their affinities for the transporters. Taken together, these results suggest that the mechanism of cocaine's antagonistic activity is not related to its binding and uptake of inhibition on monoamine neurotransporters. The failure of lidocaine and procaine to antagonize nicotine's effects in the tail-flick assay rules out local anesthetic effects. In addition, cocaine blocked differentially the response of nicotine in the oocyte receptor expression system for the alpha4beta2 and alpha3beta2 subtypes in a dose-dependent manner. Our results suggest that cocaine is a noncompetitive nicotinic antagonist with some selectivity for neuronal nicotinic receptor subtypes. Our studies also demonstrate that 3beta-phenyltropane analogs constitute a new class of nicotinic antagonists. Elucidation of the mechanism of action of this new class of antagonists may provide an explanation for the effectiveness of agents such as bupropion for the treatment of smoking cessation. (+info)Drug coverage decisions: the role of dollars and values. (3/379)
Given the increasing costs of pharmaceuticals today, it is important to understand how pharmacy benefits decisions are made and the role of cost and values in these decisions. This study examines what coverage decisions insurers make and the information and processes used in making these decisions. Fifty-three organizations, differing in size, tax status, and region, were asked about their policies for four new and controversial drugs: Viagra, Enbrel, Zyban, and Celebrex. Enbrel and Celebrex were much more likely to be covered than Viagra and Zyban. In addition, coverage of Enbrel and Celebrex was limited through strategies such as prior authorization, to encourage medically appropriate use of these agents, whereas coverage of Viagra and Zyban was limited predominantly through generalized exclusion or through restrictions on quantity or duration of use. Value judgments, rather than cost, seem to play a central, though largely unspoken, role in these coverage decisions. (+info)Antidepressants upregulate messenger RNA levels of the neuroprotective enzyme superoxide dismutase (SOD1). (4/379)
OBJECTIVE: To investigate the effect of amitriptyline, bupropion, doxepin or venlafaxine on the gene expression of the neuroprotective enzyme superoxide dismutase (SOD1) in a catecholamine cell in vitro model. DESIGN: Molecular study of a cultured cell line. INTERVENTIONS: Rat pheochromocytoma (PC12) cells were incubated in 1 and 10 mumol/L of various antidepressant medications for 24 or 48 hours. OUTCOME MEASURES: Northern blot analysis. RESULTS: Amitriptyline up-regulated SOD1 messenger RNA in a time- and dose-dependent manner. The greatest up-regulation was following incubation with 10 mumol/L amitriptyline for 48 hours. The addition of bupropion, doxepin or venlafaxine to PC12 cell cultures also up-regulated SOD1 mRNA. CONCLUSIONS: These findings suggest that some antidepressants have the ability to positively regulate neuroprotective genes. (+info)Bupropion is a nicotinic antagonist. (5/379)
Neuronal nicotinic receptors are ligand-gated ion channels of the central and peripheral central nervous system that regulate synaptic activity from both pre- and postsynaptic sites. The present study establishes the acute interaction of bupropion, an antidepressant agent that is also effective in nicotine dependence, with nicotine and nicotinic receptors using different in vivo and in vitro tests. Bupropion was found to block nicotine's antinociception (in two tests), motor effects, hypothermia, and convulsive effects with different potencies in the present investigation, suggesting that bupropion possesses some selectivity for neuronal nicotinic receptors underlying these various nicotinic effects. In addition, bupropion blocks nicotine activation of alpha(3)beta(2), alpha(4)beta(2), and alpha(7) neuronal acetylcholine nicotinic receptors (nAChRs) with some degree of selectivity. It was approximately 50 and 12 times more effective in blocking alpha(3)beta(2) and alpha(4)beta(2) than alpha(7.) This functional blockade was noncompetitive, because it was insurmountable by increasing concentration of ACh in the nAChRs subtypes tested. Furthermore, bupropion at high concentration failed to displace brain [(3)H]nicotine binding sites, a site largely composed of alpha(4)beta(2) subunit combination. Given the observation that bupropion inhibition of alpha(3)beta(2) and alpha(4)beta(2) receptors exhibits voltage-independence properties, bupropion may not be acting as an open channel blocker. These effects may explain in part bupropion's efficacy in nicotine dependence. Our present findings suggest that functional blockade of neuronal nAChRs are useful in nicotine dependence treatment. (+info)CYP2B6 mediates the in vitro hydroxylation of bupropion: potential drug interactions with other antidepressants. (6/379)
The in vitro biotransformation of bupropion to hydroxybupropion was studied in human liver microsomes and microsomes containing heterologously expressed human cytochromes P450 (CYP). The mean (+/-S.E.) K(m) in four human liver microsomes was 89 (+/-14) microM. In microsomes containing cDNA-expressed CYPs, hydroxybupropion formation was mediated only by CYP2B6 at 50 microM bupropion (K(m) 85 microM). A CYP2B6 inhibitory antibody produced more than 95% inhibition of bupropion hydroxylation in four human livers. Bupropion hydroxylation activity at 250 microM was highly correlated with S-mephenytoin N-demethylation activity (yielding nirvanol), another CYP2B6-mediated reaction, in a panel of 32 human livers (r = 0.94). The CYP2B6 content of 12 human livers highly correlated with bupropion hydroxylation activity (r = 0.96). Thus bupropion hydroxylation is mediated almost exclusively by CYP2B6 and can serve as an index reaction reflecting activity of this isoform. IC(50) values for inhibition of a CYP2D6 index reaction (dextromethorphan O-demethylation) by bupropion and hydroxybupropion were 58 and 74 microM, respectively. This suggests a low inhibitory potency versus CYP2D6, the clinical importance of which is not established. Since bupropion is frequently coadministered with other antidepressants, IC(50) values (microM) for inhibition of bupropion hydroxylation were determined as follows: paroxetine (1.6), fluvoxamine (6.1), sertraline (3.2), desmethylsertraline (19.9), fluoxetine (59.5), norfluoxetine (4.2), and nefazodone (25.4). Bupropion hydroxylation was only weakly inhibited by venlafaxine, O-desmethylvenlafaxine, citalopram, and desmethylcitalopram. The inhibition of bupropion hydroxylation in vitro by a number of newer antidepressants suggests the potential for clinical drug interactions. (+info)Validation of bupropion hydroxylation as a selective marker of human cytochrome P450 2B6 catalytic activity. (7/379)
The purpose of this study was to establish bupropion (BUP) hydroxylation as a selective in vitro marker of cytochrome P450 (CYP) 2B6 catalytic activity. Among a panel of 16 human liver microsomes (HLMs), BUP hydroxylase activity varied 80-fold when assayed at 500 microM substrate and significantly correlated with CYP2B6 blotting density (r(2) = 0.99) and S-mephenytoin N-demethylase activity (r(2) = 0.98). Kinetic analysis of BUP hydroxylation was performed in a subset of seven HLMs representative of the 80-fold range in activity. Sigmoidal kinetics suggestive of allosteric activation was observed in five HLMs exhibiting low or high activity; the mean apparent K(m) for BUP hydroxylation in these HLMs (130 microM) was similar to the K(m) for cDNA-expressed CYP2B6 (156 microM). Nonsaturable, biphasic kinetics was observed in two HLMs exhibiting low activity. Among a panel of cDNA-expressed P450 isoforms, CYP2B6 and CYP2E1 demonstrated the highest rates of BUP hydroxylation at 12 mM BUP (7.0 and 2.4 pmol/min/pmol of P450, respectively). The relative contributions of CYP2B6 and CYP2E1 to BUP hydroxylation were estimated by using immunoinhibitory monoclonal antibodies (MAB) to these enzymes. MAB-2B6 produced 88% maximum inhibition of BUP hydroxylation when assayed at 12 mM BUP in a high activity HLM, whereas MAB-2E1 produced 81% maximum inhibition in a low activity HLM. However, negligible inhibition by MAB-2E1 was observed when low and high activity HLMs were assayed at 500 microM BUP. These results demonstrate selectivity of BUP hydroxylation for CYP2B6 at 500 microM BUP, thereby validating its use as a diagnostic in vitro marker of CYP2B6 catalytic activity. (+info)Smoking cessation guidelines for health professionals: an update. Health Education Authority. (8/379)
This paper updates the evidence base and key recommendations of the Health Education Authority (HEA) smoking cessation guidelines for health professionals published in Thorax in 1998. The strategy for updating the evidence base makes use of updated Cochrane reviews supplemented by individual studies where appropriate. This update contains additional detail concerning the effectiveness of interventions as well as comments on issues relating to implementation. The recommendations include clarification of some important issues addressed only in general terms in the original guidelines. The conclusion that smoking cessation interventions delivered through the National Health Service are an extremely cost effective way of preserving life and reducing ill health remains unchanged. The strategy recommended by the guidelines involves: (1) GPs opportunistically advising smokers to stop during routine consultations, giving advice on and/or prescribing effective medications to help them and referring them to specialist cessation services; (2) specialist smokers' services providing behavioural support (in groups or individually) for smokers who want help with stopping and using effective medications wherever possible; (3) specialist cessation counsellors providing behavioural support for hospital patients and pregnant smokers who want help with stopping; (4) all health professionals involved in smoking cessation encouraging and assisting smokers in use of nicotine replacement therapies (NRT) or bupropion where appropriate. The key points of clarification of the previous guidelines include: (1) primary health care teams and hospitals should create and maintain readily accessible records on the current smoking status of patients; (2) GPs should aim to advise smokers to stop, and record having done so, at least once a year; (3) inpatient, outpatient, and pregnant smokers should be advised to stop as early as possible and the advice recorded in the notes in a readily accessible form; (4) there is currently little scientific basis for matching individual smokers to particular forms of NRT; (5) NHS specialist smokers' clinics should be the first point of referral for smokers wanting help beyond what can be provided through brief advice from the GP; (6) help from trained health care professionals specialising in smoking cessation such as practice nurses should be available for smokers who do not have access to specialist clinics; (7) the provision of specialist NHS smokers' clinics should be commensurate with demand; this is currently one or two full time clinics or their equivalent per average sized health authority, but demand may rise as publicity surrounding the services increases. (+info) Tobacco Use Disorder:
Tobacco use disorder refers to a condition where an individual engages in the excessive and compulsive consumption of tobacco products, despite the negative consequences it may have on their health and well-being. Tobacco use disorder is a common condition that affects millions of people worldwide, and it is characterized by a pattern of continued tobacco use despite harmful effects, as well as an increased tolerance to tobacco and withdrawal symptoms when trying to stop.
The Diagnostic and Statistical Manual of Mental Disorders (DSM-5) defines tobacco use disorder as a chronic condition that can manifest in different forms, including nicotine dependence and tobacco abuse. The criteria for diagnosing tobacco use disorder include:
1. Tolerance: A need to use more tobacco to achieve the desired effect.
2. Withdrawal: Experiencing symptoms such as irritability, anxiety, or depression when trying to stop using tobacco.
3. Loss of control: Consuming more tobacco than intended or for longer periods than intended.
4. Negative consequences: Continuing to use tobacco despite social, physical, or psychological problems caused by its use.
5. Increased time and effort spent on using tobacco.
6. Craving or a strong desire to use tobacco.
7. Failure to control or reduce tobacco use.
Tobacco use disorder can have severe consequences, including lung cancer, heart disease, respiratory problems, and other health issues. It can also lead to social and economic problems, such as lost productivity and strained relationships with family and friends. Treatment for tobacco use disorder includes behavioral therapies, medications, and support groups, and it is important for individuals struggling with this condition to seek professional help to quit using tobacco and improve their overall health and well-being.
Amphetamine-related disorders refer to a group of psychiatric and medical conditions that are associated with the use of amphetamines, a type of central nervous system stimulant. These disorders can result from the use of prescription medications, such as Adderall or Ritalin, as well as the use of illicit drugs like methamphetamine.
The following are some common amphetamine-related disorders:
1. Amphetamine Use Disorder (AUD): This is a chronic condition characterized by the excessive and compulsive use of amphetamines, despite negative consequences. Individuals with AUD may experience withdrawal symptoms when they stop using the drug, and may continue to use to avoid these symptoms or to achieve a "high."
2. Stimulant Psychosis: This is a condition in which an individual experiences hallucinations, delusions, or disorganized thinking due to the use of amphetamines. It is often seen in individuals who have a history of substance abuse and/or mental health issues.
3. Amphetamine-Induced Psychotic Disorder: This condition is similar to stimulant psychosis, but it is specifically caused by the use of amphetamines. It can include hallucinations, delusions, and disorganized thinking.
4. Cognitive Impairment: Long-term use of amphetamines can lead to cognitive impairments, such as memory loss, difficulty with attention, and decreased problem-solving skills.
5. Sleep Disturbances: Amphetamine use can disrupt sleep patterns, leading to insomnia or other sleep disorders.
6. Malnutrition: Individuals who use amphetamines may neglect their nutritional intake, leading to malnutrition and related health problems.
7. Cardiovascular Problems: Amphetamine use can increase heart rate and blood pressure, which can lead to cardiovascular problems such as heart attack, stroke, and arrhythmias.
8. Dental Problems: The dry mouth caused by amphetamine use can lead to tooth decay and other dental problems.
9. Infectious Diseases: Sharing needles or engaging in other risky behaviors to obtain amphetamines can increase the risk of contracting infectious diseases such as HIV/AIDS and hepatitis.
10. Financial and Legal Problems: The high cost of maintaining an amphetamine habit can lead to financial problems, and criminal activity to support the habit can lead to legal problems.
It is important to note that the specific risks associated with amphetamine use can vary depending on the individual, their health status, and the dose and duration of use. If you or someone you know is struggling with amphetamine addiction, it is important to seek professional help as soon as possible.
Substance withdrawal syndrome is a condition that occurs when an individual abruptly stops using a substance to which they have become physically dependent on. This can lead to a variety of physical and emotional symptoms, including:
* Anxiety
* Depression
* Fatigue
* Insomnia
* Muscle and bone pain
* Nausea and vomiting
* Seizures (in severe cases)
* Sweating
* Tremors
The specific symptoms of substance withdrawal syndrome can vary depending on the substance being withdrawn from, but some common symptoms include:
* Alcohol: tremors, anxiety, insomnia, nausea and vomiting, headaches, and seizures
* Opioids: withdrawal symptoms can include anxiety, muscle aches, sweating, nausea and vomiting, diarrhea, and depression
* Benzodiazepines: withdrawal symptoms can include anxiety, insomnia, tremors, and seizures
The diagnosis of substance withdrawal syndrome is typically made based on the patient's history of substance use and the presence of withdrawal symptoms. A healthcare provider may also order laboratory tests to rule out other conditions that may be causing the symptoms. Treatment for substance withdrawal syndrome usually involves supportive care, such as rest, hydration, and pain management, as well as medication to manage withdrawal symptoms. In some cases, medical professionals may also recommend a gradual tapering of the substance over a period of time to minimize withdrawal symptoms.
It is important for individuals who are experiencing withdrawal symptoms to seek medical attention as soon as possible, as untreated withdrawal can lead to serious complications, such as seizures and dehydration. With appropriate treatment, most individuals with substance withdrawal syndrome can recover fully and successfully overcome their addiction.
Bupropion
"15 years of clinical experience with bupropion HCl: from bupropion to bupropion SR to bupropion XL". Primary Care Companion to ... The clinically used bupropion is racemic, that is a mixture of two enantiomers: S-bupropion and R-bupropion. Although the ... Bupropion misuse is uncommon. There have been a number of anecdotal and case-study reports of bupropion abuse, but the bulk of ... Bupropion is not effective in treating chronic low back pain. Bupropion is available as an oral tablet in a number of different ...
List of side effects of bupropion
"Prexaton Bupropion hydrochloride PRODUCT INFORMATION". TGA eBusiness Services. Ascent Pharma Pty Ltd. 2 October 2012. Retrieved ... "BUPROPION HYDROCHLORIDE tablet, film coated [Sandoz Inc]". DailyMed. Sandoz, Inc. April 2013. Retrieved 22 October 2013. " ... List of side effects of bupropion. Headache Transient insomnia Abdominal pain Agitation Anxiety Asthenia Concentration ...
Zonisamide
"Bupropion/zonisamide". AdisInsight. Springer. 20 May 2017. Archived from the original on 19 August 2017. Retrieved 19 August ... It was to be sold, when combined with bupropion, under the brand name Empatic, until its development was discontinued. ...
List of dopaminergic drugs
Bupropion (amfebutamone) • Ciclazindol • Diclofensine • Dimethocaine • Diphenylpyraline • Dizocilpine • DOV-102,677 • DOV- ...
Mecamylamine
Bupropion Scopolamine "Mecamylamine". drugs.com. Retrieved May 15, 2015. Bacher I, Wu B, Shytle DR, George TP (November 2009 ...
Antidepressant
Carroll FI, Blough BE, Mascarella SW, Navarro HA, Lukas RJ, Damaj MI (2014). "Bupropion and bupropion analogs as treatments for ... Bupropion, a weak NDRI and nicotinic antagonist, may be useful in treating reduced libido as a result of SSRI treatment. ... Bupropion is not recommended for the treatment of eating disorders due to an increased risk of seizure. Similar recommendations ... "Bupropion: MedlinePlus Drug Information". Archived from the original on 8 May 2016. Retrieved 24 May 2016. "Practice guideline ...
Attention deficit hyperactivity disorder management
Bupropion Bupropion is classified as an atypical antidepressant. It is the most common off-label prescription for ADHD. It ... bupropion and venlafaxine). There have been case reports of tics worsening with bupropion. There is good empirical evidence for ... Bupropion is not a controlled substance. It is commonly prescribed as a timed release formulation to decrease the risk of side ... Stahl S, Pradko J, Haight B, Modell J, Rockett C, Learned-Coughlin S (2004). "A Review of the Neuropharmacology of Bupropion, a ...
Hypoactive sexual desire disorder
A few studies suggest that the antidepressant, bupropion, can improve sexual function in women who are not depressed, if they ... Foley KF, DeSanty KP, Kast RE (September 2006). "Bupropion: pharmacology and therapeutic applications". Expert Rev Neurother. 6 ...
CYP2D6
"Inhibition of CYP2D6 activity by bupropion". Journal of Clinical Psychopharmacology. 25 (3): 226-9. doi:10.1097/01.jcp. ...
Drug rehabilitation
These medications include bupropion and nortriptyline. Bupropion inhibits the re-uptake of nor-epinephrine and dopamine and has ...
Generic drug
Generic Bupropion". 2012-12-06. Retrieved 2013-04-20. Healy, Melissa (2012-10-05). "Generic antidepressant pulled from U.S. ... complaints that generic versions of bupropion (Wellbutrin) were yielding unexpected effects. Subsequently, Impax Laboratories's ...
Threohydrobupropion
In any case, it is about 20% as pharmacologically potent as bupropion and in the range of 20 to 50% as potent as bupropion in ... Threohydrobupropion circulates at higher concentrations than bupropion during bupropion therapy, similarly to hydroxybupropion ... These values can be compared to rat values with bupropion of 1,400 nM, 570 nM, and 19,000 nM, respectively. Besides monoamine ... It can also be formed from bupropion by carbonyl reductases. The compound is metabolized by the cytochrome P450 enzymes CYP2B6 ...
Schizoaffective disorder
Kumar S, Kodela S, Detweiler JG, Kim KY, Detweiler MB (November-December 2011). "Bupropion-induced psychosis: folklore or a ... "Acute psychotic disorders related to bupropion: review of the literature". Encephale. 36 (6): 461-71. doi:10.1016/j.encep. ...
Nariman Mehta
Mehta, N B (1983), "The Chemistry of Bupropion", The Journal of Clinical Psychiatry (published May 1983), vol. 44, no. 5, pp. ... Wilson, Brent; Paul, Panchajanya; Mehta, Sunny; Coffey, Barbara (February 2013). "Hypokalemia Secondary to Bupropion Sustained ... the compound Bupropion (BW 323U66) that was approved for use as an antidepressant in 1985 and sold under the trade name ... and patented the organic compound bupropion, marketed under the name Wellbutrin as an antidepressant and smoking cessation aid ...
Sanofi
Aplenzin (Bupropion), for depression and quit smoking. Captique (Dermal filler), for facial wrinkles. Carticel (Knee cartilage ...
Radafaxine
Therefore, radafaxine builds on at least some of the properties of bupropion in humans. Another analogue of bupropion, ... "Bupropion and bupropion analogs as treatments for CNS disorders". Emerging Targets & Therapeutics in the Treatment of ... At least one study suggests that radafaxine has a low abuse potential similar to bupropion. Radafaxine is a potent metabolite ... Radafaxine has about 70% of the efficacy of bupropion in blocking dopamine reuptake, and 392% of efficacy in blocking ...
Social anxiety disorder
Emmanuel NP, Brawman-Mintzer O, Morton WA, Book SW, Johnson MR, Lorberbaum JP, Ballenger JC, Lydiard RB (2000). "Bupropion-SR ... The atypical antidepressants mirtazapine and bupropion have been studied for the treatment of social anxiety disorder, and ...
Serotonin syndrome
Bupropion has also been suggested to cause serotonin syndrome, although as there is no evidence that it has any significant ... Thorpe EL, Pizon AF, Lynch MJ, Boyer J (June 2010). "Bupropion induced serotonin syndrome: a case report". J Med Toxicol. 6 (2 ... Gillman PK (June 2010). "Bupropion, bayesian logic and serotonin toxicity". J Med Toxicol. 6 (2): 276-7. doi:10.1007/s13181-010 ... Munhoz RP (2004). "Serotonin syndrome induced by a combination of bupropion and SSRIs". Clin Neuropharmacol. 27 (5): 219-22. ...
Butaxamine
Bupropion Methoxamine "Definition: butoxamine from Online Medical Dictionary". Hillman KL, Doze VA, Porter JE (August 2005). " ...
Depression in childhood and adolescence
... bupropion and atypical antipsychotics. These options are medications that work in different ways. Bupropion (Wellbutrin) works ...
Erythrohydrobupropion
... as potent as bupropion in mouse models of depression. It circulates at similar concentrations as bupropion during bupropion ... circulate at higher concentrations than bupropion. Erythrohydrobupropion is formed from bupropion via reduction of the ketone ... It can also be formed from bupropion by carbonyl reductases. The compound is metabolized by the cytochrome P450 enzymes CYP2B6 ... Other metabolites of bupropion include hydroxybupropion and threohydrobupropion. Information on the pharmacological actions of ...
Elbert Glover
"Bupropion SR for the treatment of smokeless tobacco use". Drug and Alcohol Dependence, 2007;90(1):56-63. Glover ED, Rath JM. " ... "Effectiveness of bupropion for smoking cessation in subjects with a history of major depression or alcoholism". British Journal ... "Varenicline versus bupropion or placebo for smoking cessation". American Journal of Health Behavior, 2008;32(6):664-675. Cooke ... "Bupropion for smoking cessation-predictors of successful outcome". Chest, 2001;119:1357-1364. Johnston JA, Fiedler-Kelly J, ...
Anti-obesity medication
Naltrexone/bupropion is a combination medication used for weight loss in those that have either obesity or overweight with some ... COR-I Study Group) (August 2010). "Effect of naltrexone plus bupropion on weight loss in overweight and obese adults (COR-I): a ... It combines low doses of bupropion and naltrexone. Both medications have individually shown some evidence of effectiveness in ... "Contrave Extended-Release- naltrexone hydrochloride and bupropion hydrochloride tablet, extended release". DailyMed. 26 April ...
Effects of cannabis
Such drugs include bupropion, divalproex, nefazodone, lofexidine, and dronabinol. Of these, dronabinol (a trade name for THC) ...
Selective serotonin reuptake inhibitor
A number of non-SSRI drugs are not associated with sexual side effects (such as bupropion, mirtazapine, tianeptine, agomelatine ... but may be less with atypical antidepressants like bupropion, agomelatine, and vortioxetine. Higher doses of antidepressants ... possibly adding or switching to bupropion; and for overall sexual dysfunction, switching to nefazodone. ...
Tinnitus
Medications such as bupropion may also result in tinnitus. In many cases, however, no underlying cause can be identified. ...
Anticholinergic
Carroll FI, Blough BE, Mascarella SW, Navarro HA, Lukas RJ, Damaj MI (2014). "Bupropion and bupropion analogs as treatments for ... Trihexyphenidyl Tropicamide Umeclidinium Antinicotinic agents Bupropion - Ganglion blocker Dextromethorphan - Cough suppressant ...
Fitness to dive
Bupropion may cause agitation, CNS stimulation, seizures, psychosis, dry mouth, headache, migraine, nausea, vomiting, rash, ... bupropion, trazodone and venlafaxine may lower the seizure threshold. Venlafaxine may occasionally cause fainting, excitability ...
Amfepramone
... is most closely chemically related to the antidepressant and smoking cessation aid bupropion (previously called ... Arias HR, SantamarÃa A, Ali SF (2009). "Pharmacological and neurotoxicological actions mediated by bupropion and diethylpropion ...
Major depressive disorder
Zisook S, Rush AJ, Haight BR, Clines DC, Rockett CB (February 2006). "Use of bupropion in combination with serotonin reuptake ... Another option is to switch to the atypical antidepressant bupropion. Venlafaxine, an antidepressant with a different mechanism ...
Bupropion SR | Quit Smoking | Tips From Former Smokers | CDC
Bupropion is a pill you take twice a day. You start it 1-2 weeks before quitting. It does not contain nicotine, so it works ... Bupropion SR Contraindications and Precautions (If any of these apply to you, talk to your doctor or other healthcare provider ... Abruptly stopping use of sedating medicines or alcohol while taking bupropion.. *Taking or recently quit taking a type of ... In addition, if you decide to take bupropion and experience hostility, agitation, depression, suicidal thoughts, or changes in ...
Bupropion (Wellbutrin) - Erowid Exp - 'Definetly Uplifting'
An Experience with Bupropion (Wellbutrin). Definetly Uplifting by Daytripper ... I decided to sprinkle a small amount of crushed bupropion over a equally small bowl of tobacco. The smoke, as expected was ... After snorting 2 lines I immediately experienced a sharp burning and then a slow numbing effect which was expected as bupropion ... "Definetly Uplifting: An Experience with Bupropion (Wellbutrin) (exp27141)". Erowid.org. May 4, 2006. erowid.org/exp/27141 ...
Bupropion: MedlinePlus Drug Information
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