A condition in which the hepatic venous outflow is obstructed anywhere from the small HEPATIC VEINS to the junction of the INFERIOR VENA CAVA and the RIGHT ATRIUM. Usually the blockage is extrahepatic and caused by blood clots (THROMBUS) or fibrous webs. Parenchymal FIBROSIS is uncommon.
Subspecialty of radiology that combines organ system radiography, catheter techniques and sectional imaging.
A type of surgical portasystemic shunt to reduce portal hypertension with associated complications of esophageal varices and ascites. It is performed percutaneously through the jugular vein and involves the creation of an intrahepatic shunt between the hepatic vein and portal vein. The channel is maintained by a metallic stent. The procedure can be performed in patients who have failed sclerotherapy and is an additional option to the surgical techniques of portocaval, mesocaval, and splenorenal shunts. It takes one to three hours to perform. (JAMA 1995;273(23):1824-30)
Longitudinal cavities in the spinal cord, most often in the cervical region, which may extend for multiple spinal levels. The cavities are lined by dense, gliogenous tissue and may be associated with SPINAL CORD NEOPLASMS; spinal cord traumatic injuries; and vascular malformations. Syringomyelia is marked clinically by pain and PARESTHESIA, muscular atrophy of the hands, and analgesia with thermoanesthesia of the hands and arms, but with the tactile sense preserved (sensory dissociation). Lower extremity spasticity and incontinence may also develop. (From Adams et al., Principles of Neurology, 6th ed, p1269)
A characteristic symptom complex.
The large hole at the base of the skull through which the SPINAL CORD passes.
Brain tissue herniation through a congenital or acquired defect in the skull. The majority of congenital encephaloceles occur in the occipital or frontal regions. Clinical features include a protuberant mass that may be pulsatile. The quantity and location of protruding neural tissue determines the type and degree of neurologic deficit. Visual defects, psychomotor developmental delay, and persistent motor deficits frequently occur.
A developmental deformity of the occipital bone and upper end of the cervical spine, in which the latter appears to have pushed the floor of the occipital bone upward. (Dorland, 27th ed)
The infratentorial compartment that contains the CEREBELLUM and BRAIN STEM. It is formed by the posterior third of the superior surface of the body of the sphenoid (SPHENOID BONE), by the occipital, the petrous, and mastoid portions of the TEMPORAL BONE, and the posterior inferior angle of the PARIETAL BONE.
Congenital, or rarely acquired, herniation of meningeal and spinal cord tissue through a bony defect in the vertebral column. The majority of these defects occur in the lumbosacral region. Clinical features include PARAPLEGIA, loss of sensation in the lower body, and incontinence. This condition may be associated with the ARNOLD-CHIARI MALFORMATION and HYDROCEPHALUS. (From Joynt, Clinical Neurology, 1992, Ch55, pp35-6)
A surgical operation for the relief of pressure in a body compartment or on a body part. (From Dorland, 28th ed)
The space between the arachnoid membrane and PIA MATER, filled with CEREBROSPINAL FLUID. It contains large blood vessels that supply the BRAIN and SPINAL CORD.
Excision of part of the skull. This procedure is used to treat elevated intracranial pressure that is unresponsive to conventional treatment.
A chromosome disorder associated either with an extra chromosome 21 or an effective trisomy for chromosome 21. Clinical manifestations include hypotonia, short stature, brachycephaly, upslanting palpebral fissures, epicanthus, Brushfield spots on the iris, protruding tongue, small ears, short, broad hands, fifth finger clinodactyly, Simian crease, and moderate to severe INTELLECTUAL DISABILITY. Cardiac and gastrointestinal malformations, a marked increase in the incidence of LEUKEMIA, and the early onset of ALZHEIMER DISEASE are also associated with this condition. Pathologic features include the development of NEUROFIBRILLARY TANGLES in neurons and the deposition of AMYLOID BETA-PROTEIN, similar to the pathology of ALZHEIMER DISEASE. (Menkes, Textbook of Child Neurology, 5th ed, p213)
Bones that constitute each half of the pelvic girdle in VERTEBRATES, formed by fusion of the ILIUM; ISCHIUM; and PUBIC BONE.
The cavity within the SPINAL COLUMN through which the SPINAL CORD passes.
Manometric pressure of the CEREBROSPINAL FLUID as measured by lumbar, cerebroventricular, or cisternal puncture. Within the cranial cavity it is called INTRACRANIAL PRESSURE.
A cluster of metabolic risk factors for CARDIOVASCULAR DISEASES and TYPE 2 DIABETES MELLITUS. The major components of metabolic syndrome X include excess ABDOMINAL FAT; atherogenic DYSLIPIDEMIA; HYPERTENSION; HYPERGLYCEMIA; INSULIN RESISTANCE; a proinflammatory state; and a prothrombotic (THROMBOSIS) state. (from AHA/NHLBI/ADA Conference Proceedings, Circulation 2004; 109:551-556)
The surgical cutting of a bone. (Dorland, 28th ed)
The inferior and superior venae cavae.
The venous trunk which receives blood from the lower extremities and from the pelvic and abdominal organs.
Exudates are fluids, CELLS, or other cellular substances that are slowly discharged from BLOOD VESSELS usually from inflamed tissues. Transudates are fluids that pass through a membrane or squeeze through tissue or into the EXTRACELLULAR SPACE of TISSUES. Transudates are thin and watery and contain few cells or PROTEINS.
The venous trunk which returns blood from the head, neck, upper extremities and chest.
Domesticated bovine animals of the genus Bos, usually kept on a farm or ranch and used for the production of meat or dairy products or for heavy labor.
The domestic cat, Felis catus, of the carnivore family FELIDAE, comprising over 30 different breeds. The domestic cat is descended primarily from the wild cat of Africa and extreme southwestern Asia. Though probably present in towns in Palestine as long ago as 7000 years, actual domestication occurred in Egypt about 4000 years ago. (From Walker's Mammals of the World, 6th ed, p801)

Managing Budd-Chiari syndrome: a retrospective review of percutaneous hepatic vein angioplasty and surgical shunting. (1/234)

BACKGROUND: The role of percutaneous hepatic vein angioplasty in the management of Budd-Chiari syndrome has not been well defined. Over a 10 year period at our unit, we have often used this technique in cases of short length hepatic vein stenosis or occlusion, reserving surgical mesocaval shunting for cases of diffuse hepatic vein occlusion or failed angioplasty. AIMS: To review the outcome of angioplasty and surgical shunting to define their respective roles. PATIENTS: All patients treated by angioplasty or surgical shunting for non-malignant hepatic vein obstruction over a ten year period from 1987 to 1996. METHODS: A case note review of pretreatment features and clinical outcome. RESULTS: Angioplasty was attempted in 21 patients with patent hepatic vein branches and was successful in 18; in three patients treatment was unsuccessful and these patients had surgical shunts. Fifteen patients were treated by surgical shunting only. Mortality according to definitive treatment was 3/18 following angioplasty and 8/18 following surgery; in most cases this reflected high risk status prior to treatment. Venous or shunt reocclusion rates were similar for both groups and were associated with subtherapeutic warfarin in half of these cases. Most surviving patients in both groups are asymptomatic although one surgical patient has chronic hepatic encephalopathy. CONCLUSION: With appropriate case selection, many patients with Budd-Chiari syndrome caused by short length hepatic vein stenosis or occlusion may be managed successfully by angioplasty alone. Medium term outcome is good following this procedure provided that anticoagulation is maintained. Further follow up is required to assess for definitive benefits but we suggest that this should be included as a valid initial approach in the algorithm for management of Budd-Chiari syndrome.  (+info)

Results of surgical treatment (modified Sugiura-Futagawa operation) of portal hypertension associated to complete splenomesoportal thrombosis and cirrhosis. (2/234)

BACKGROUND: Hemorrhagic portal hypertension, secondary to both intrahepatic and extrahepatic portal hypertension, is an uncommon entity. In this condition, the extrahepatic and the intrahepatic obstruction of the portal vein, due to chronic liver disease, produce a more severe form of hemorrhagic portal hypertension that is more difficult to control. The results of surgical treatment (modified Sugiura-Futagawa operation) in this subset of patients is analyzed. METHODS: Among 714 patients with a history of hemorrhagic portal hypertension, 14 cases were found with histologically proven liver cirrhosis and complete splenomesoportal thrombosis demonstrated by means of preoperative angiography. Patients with incomplete (partial) splenomesoportal thrombosis were excluded. There were nine males and 5 females with a mean age of 51 years. Alcoholic cirrhosis was demonstrated in 50% of the cases, post hepatitic cirrhosis in 28%, primary biliary cirrhosis in 7%, and cryptogenic cirrhosis in 14%. There were nine Child-Pugh A and 5 B cases. All cases were treated by means of our modified Sugiura-Futagawa procedure. RESULTS: Bleeding recurrence from esophagogastric varices was shown in one case, colonic varices in one case and hypertensive gastropathy in another of the survivors. Post operative encephalopathy was shown in 3 of the cases. The thirty-six month survival rate was 30% (Kaplan-Meier). CONCLUSIONS: The combination of intrahepatic plus extrahepatic portal hypertension has a worse prognosis. Treatment options are limited (sclerotherapy and/or devascularization), because shunt surgery, TIPS and liver transplantation have a very restricted role and postoperative outcome is poor.  (+info)

Successful twin pregnancy in a dual-transplant couple resulting from in-vitro fertilization and intracytoplasmic sperm injection: case report. (3/234)

There are numerous reports of successful pregnancy following liver transplantation. Little information is available regarding the incidence and management of infertility in transplant recipients, particularly the use of artificial reproductive technologies. We present a case of a successful twin pregnancy resulting from in-vitro fertilization with intracytoplasmic sperm injection (IVF/ICSI) in a liver transplant recipient, whose partner was a renal transplant recipient with severe oligozoospermia. With careful evaluation and monitoring, and the involvement of appropriate consultants, artificial reproductive technologies can be safely used in transplant recipient couples experiencing infertility.  (+info)

A 27-year experience with surgical treatment of Budd-Chiari syndrome. (4/234)

OBJECTIVE: To determine the effects of surgical portal decompression in Budd-Chiari syndrome (BCS) on survival, quality of life, shunt patency, liver function, portal hemodynamics, and hepatic morphology during periods ranging from 3.5 to 27 years. SUMMARY BACKGROUND DATA: Experiments in the authors' laboratory showed that surgical portal decompression reversed the deleterious effects of BCS on the liver. This study was aimed at determining whether similar benefit could be obtained in patients with BCS. METHODS: From 1972 to 1999, the authors conducted prospective studies of the treatment of 60 patients with BCS who were divided into three groups: the first had occlusion confined to the hepatic veins treated by direct side-to-side portacaval shunt (SSPCS); the second had occlusion involving the inferior vena cava (IVC) treated by a portal decompressive procedure that bypassed the obstructed IVC; and the third group, who had advanced cirrhosis and hepatic decompensation and were referred too late for treatment by portal decompression, required orthotopic liver transplantation. RESULTS: In the 32 patients with BCS resulting from hepatic vein occlusion alone, SSPCS had a surgical death rate of 3%, and 94% of the patients were alive 3.5 to 27 years after surgery. All 31 survivors remained free of ascites and almost all had normal liver function. No patient with a patent shunt had encephalopathy. The SSPCS remained patent in all but one patient. Liver biopsies showed no evidence of congestion or necrosis, and 48% of the biopsies were diagnosed as normal. Mesoatrial shunt was performed in eight patients with BCS caused by IVC thrombosis. All patients survived surgery, but five subsequently developed thrombosis of the synthetic graft and died. Because of the poor results, mesoatrial shunt was abandoned. Instead, a high-flow combination shunt was introduced, consisting of SSPCS combined with a cavoatrial shunt (CAS) through a Gore-Tex graft. There were no surgical or long-term deaths among 10 patients who underwent combined SSPCS and CAS, and the shunts functioned effectively during 4 to 16 years of follow-up. Ten patients with advanced cirrhosis were referred too late to benefit from surgical portal decompression, and they were approved and listed for orthotopic liver transplantation. Three patients died of liver failure while awaiting a transplant, and four patients died after the transplant. The 1- and 5-year survival rates were 40% and 30%, respectively. CONCLUSIONS: SSPCS in BCS with hepatic vein occlusion alone results in reversal of liver damage, correction of hemodynamic disturbances, prolonged survival, and good quality of life when performed early in the course of BCS. Similarly good results are obtained with combined SSPCS and CAS in patients with BCS resulting from IVC occlusion. In contrast, mesoatrial shunt has been discontinued in the authors' program because of an unacceptable incidence of graft thrombosis and death. In patients with advanced cirrhosis from long-standing, untreated BCS, orthotopic liver transplantation is the only hope of relief and results in the salvage of some patients. The key to long survival in BCS is prompt diagnosis and treatment by portal decompression.  (+info)

Successful liver transplantation in a patient with Budd-Chiari syndrome caused by homozygous factor V Leiden. (5/234)

Budd-Chiari syndrome (BCS) is a rare form of portal hypertension characterized by hepatic venous outflow obstruction. Although hematologic disorders are the most common cause of this syndrome, to date, 30% of the cases have been classified as idiopathic. Resistance to activated protein C caused by factor V Leiden is the most common cause of thrombophilia; its role in the pathogenesis of BCS is now becoming apparent. We report successful liver transplantation in a patient with BCS caused by homozygous factor V Leiden. The patient was administered standard heparin anticoagulation until activated protein C resistance was normalized by the liver allograft. Liver transplantation corrected the thrombophilic state. The patient has excellent graft function, is not on anticoagulation therapy, and has had no recurrent venous thrombosis at 5 months posttransplantation. Activated protein C resistance caused by the factor V Leiden mutation may be responsible for idiopathic cases of BCS. To avoid unnecessary long-term anticoagulation after liver transplantation, factor V Leiden should be considered as a pathogenic factor in BCS. In addition, because of the high prevalence of factor V Leiden in the world population, cadaveric organ donors with a history of venous thrombosis should be screened for activated protein C resistance lest thrombophilia be transmitted to the recipient.  (+info)

Factor V Leiden mutation, prothrombin gene mutation, and deficiencies in coagulation inhibitors associated with Budd-Chiari syndrome and portal vein thrombosis: results of a case-control study. (6/234)

In a collaborative multicenter case-control study, we investigated the effect of factor V Leiden mutation, prothrombin gene mutation, and inherited deficiencies of protein C, protein S, and antithrombin on the risk of Budd-Chiari syndrome (BCS) and portal vein thrombosis (PVT). We compared 43 BCS patients and 92 PVT patients with 474 population-based controls. The relative risk of BCS was 11.3 (95% CI 4.8-26.5) for individuals with factor V Leiden mutation, 2.1(95% CI 0.4-9.6) for those with prothrombin gene mutation, and 6.8 (95% CI 1.9-24.4) for those with protein C deficiency. The relative risk of PVT was 2.7 (95% CI 1.1-6.9) for individuals with factor V Leiden mutation, 1.4 (95% CI 0.4-5.2) for those with prothrombin gene mutation, and 4.6 (95% CI 1.5-14.1) for those with protein C deficiency. The relative risk of BCS or PVT was not increased in the presence of inherited protein S or antithrombin deficiency. Concurrence of either acquired or inherited thrombotic risk factors was observed in 26% of the BCS patients and 37% of the PVT patients. We conclude that factor V Leiden mutation and hereditary protein C deficiency appear to be important risk factors for BCS and PVT. Although the prevalence of the prothrombin gene mutation was increased, it was not found to be a significant risk factor for BCS and PVT. The coexistence of thrombogenic risk factors in many patients indicates that BCS and PVT can be the result of a combined effect of different pathogenetic mechanisms.  (+info)

Successful outcome of orthotopic liver transplantation in patients with preexisting malignant states. (7/234)

Preexisting malignancy is considered a relative contraindication to orthotopic liver transplantation (OLT) because of the risk of tumor recurrence. The purpose of this study is to assess the outcome of OLT in patients with a preexistent malignant state. Of 1,097 OLTs performed between 1989 and 1999 at King's College Hospital (London, UK), 18 patients had a pretransplantation malignant state, including 6 cases of myeloproliferative disorder (MPD) presenting as Budd-Chiari syndrome. Those patients with solid-organ malignancies had their tumor detected at an early stage and underwent curative treatment before or during OLT. Patients were followed up for a median of 71 months (range, 1 to 119 months) post-OLT, and the rates of rejection and malignancy were compared with those of transplant recipients without preexisting malignancy during the same period. One patient had a recurrence of his primary malignancy (non-Hodgkin's lymphoma) after 27 months, whereas another patient developed a de novo posttransplant lymphoproliferative disorder after 57 months. One patient with MPD developed acute leukemia 72 months after OLT. In comparison, of 1,079 OLTs performed in patients without preexisting malignancy during the same period, there were 34 cases of de novo malignancies. The rate of rejection in patients with and without preexisting malignancy was similar. Successful medium-term outcome after OLT can be achieved in carefully selected patients with preexisting malignancy providing the malignancy is amenable to curative treatment before or at OLT. Primary MPDs responsible for Budd-Chiari syndrome should not be considered a contraindication to OLT.  (+info)

Factor V Leiden related Budd-Chiari syndrome. (8/234)

BACKGROUND: The role of factor V Leiden as a cause of Budd-Chiari syndrome has only recently been described. AIMS: To assess the specific features of factor V Leiden related Budd-Chiari syndrome. PATIENTS: Sixty three consecutive patients with hepatic vein or terminal inferior vena cava thrombosis. METHODS: Standardised chart review. RESULTS: Factor V Leiden was found in 20 patients (31% (95% CI 20-43)). In the subgroup of patients with, compared with the subgroup without, factor V Leiden, a combination of prothrombotic states was more common (70% (95% CI 50-90) v 14% (95% CI 3-24)); inferior vena cava thrombosis was more frequent (40% (95% CI 19-61) v 7% (95% CI 0-14)); and distribution of initial alanine aminotransferase values was bimodal (almost normal or extremely increased) versus unimodal (p=0.003). Factor V Leiden accounted for four of five cases of massive ischaemic necrosis (transaminases >50-fold the upper limit of normal values) (p=0.014), and also for all three cases developing during pregnancy. Patients with and without factor V Leiden did not differ with regard to mortality, portosytemic shunting, or listing for liver transplantation. Hepatocellular carcinoma developed in two patients; both had factor V Leiden and indolent obstruction of the inferior vena cava. CONCLUSIONS: In patients with Budd-Chiari syndrome, factor V Leiden (a) is common; (b) precipitates thrombosis mostly when combined with another risk factor; (c) is associated with one of two contrasting clinical pictures: indolent thrombosis-particularly of the inferior vena cava-or massive ischaemic necrosis; and (d) is a major cofactor of Budd-Chiari syndrome developing during pregnancy.  (+info)

Budd-Chiari Syndrome is a rare but serious medical condition that occurs when there is a blockage in the veins that carry blood from the liver to the heart. This blockage can be caused by a variety of factors, including blood clots, cancer, or scarring of the veins. The condition is named after the two doctors who first described it, Dr. Takeo Budd and Dr. Takeo Chiari. Symptoms of Budd-Chiari Syndrome can include abdominal pain, swelling in the abdomen or legs, yellowing of the skin and eyes (jaundice), and dark urine. In severe cases, the condition can lead to liver failure and even death. Treatment for Budd-Chiari Syndrome depends on the underlying cause of the blockage. In some cases, medications may be used to dissolve blood clots or reduce inflammation. In more severe cases, surgery may be necessary to remove the blockage or repair damaged veins. In some cases, a liver transplant may be necessary if the liver has been severely damaged by the condition.

Syringomyelia is a medical condition characterized by the formation of fluid-filled cysts (syringes) within the spinal cord. These cysts can cause damage to the spinal cord and lead to a range of symptoms, including pain, weakness, numbness, and tingling in the arms and legs. Syringomyelia can be caused by a variety of factors, including trauma to the spine, spinal cord infections, and inherited conditions such as Chiari malformation. The condition can also be idiopathic, meaning its cause is unknown. Diagnosis of syringomyelia typically involves imaging tests such as MRI or CT scans, which can show the presence of the cysts within the spinal cord. Treatment options for syringomyelia depend on the severity of the symptoms and the underlying cause of the condition. In some cases, surgery may be necessary to remove the cysts or relieve pressure on the spinal cord. Other treatment options may include medication, physical therapy, and lifestyle changes.

In the medical field, a syndrome is a set of symptoms and signs that occur together and suggest the presence of a particular disease or condition. A syndrome is often defined by a specific pattern of symptoms that are not caused by a single underlying disease, but rather by a combination of factors, such as genetic, environmental, or hormonal. For example, Down syndrome is a genetic disorder that is characterized by a specific set of physical and intellectual characteristics, such as a flattened facial profile, short stature, and intellectual disability. Similarly, the flu syndrome is a set of symptoms that occur together, such as fever, cough, sore throat, and body aches, that suggest the presence of an influenza virus infection. Diagnosing a syndrome involves identifying the specific set of symptoms and signs that are present, as well as ruling out other possible causes of those symptoms. Once a syndrome is diagnosed, it can help guide treatment and management of the underlying condition.

Encephalocele is a medical condition in which a portion of the brain or spinal cord extends through a defect in the skull or vertebral column. This can occur when the neural tube, which forms the brain and spinal cord, fails to close properly during fetal development. Encephaloceles can be classified based on the location of the defect and the extent of the brain or spinal cord that is exposed. They can be present at birth or may not be diagnosed until later in life. Encephaloceles can cause a range of symptoms, depending on the size and location of the defect and the extent of brain or spinal cord involvement. Treatment typically involves surgical repair of the defect to reduce the risk of complications and improve the quality of life for the affected individual.

Platybasia is a medical term that refers to a condition in which the occipital bone (the bone at the back of the skull) is flat or flattened. This can be caused by a variety of factors, including genetic factors, head injuries, and certain medical conditions such as Down syndrome or achondroplasia. Platybasia can cause a number of symptoms, including headaches, dizziness, and balance problems. In some cases, it may also lead to structural problems in the skull, such as an increased risk of fractures or pressure on the brain. Treatment for platybasia typically depends on the underlying cause and the severity of the symptoms. In some cases, no treatment may be necessary, while in others, surgery may be recommended to correct the flattened occipital bone.

The cranial fossa, posterior refers to the posterior part of one of the four main cavities or spaces within the skull. The skull is composed of several bones that fit together to form a protective structure around the brain. The cranial fossae are the main cavities within the skull that house the brain. There are four main cranial fossae: the anterior cranial fossa, the middle cranial fossa, the posterior cranial fossa, and the temporal fossa. The posterior cranial fossa is located at the back of the skull, behind the middle cranial fossa. It is the largest of the four cranial fossae and contains several important structures, including the cerebellum, the brainstem, and the occipital lobe of the cerebrum. The posterior cranial fossa is bounded by several bones, including the occipital bone, the cerebellar peduncles, and the tentorium cerebelli. The tentorium cerebelli is a thin, translucent membrane that separates the cerebellum from the brainstem. The cerebellar peduncles are the two thickened areas of the brainstem that connect the cerebellum to the rest of the brain. The posterior cranial fossa is an important part of the skull and plays a crucial role in protecting the brain. Any damage to the bones that form the posterior cranial fossa can potentially cause serious injury to the brain.

Meningomyelocele is a birth defect that occurs when the spinal cord and the protective membranes surrounding it (the meninges) protrude through a weak spot in the baby's spine (vertebrae). This can result in a sac-like structure containing the spinal cord and meninges, which is usually located at the base of the spine or the lower back. Meningomyelocele can cause a range of symptoms, depending on the severity of the defect and the extent of the spinal cord damage. Some common symptoms include weakness or paralysis in the legs, difficulty walking or standing, loss of sensation in the lower body, and problems with bladder and bowel control. Treatment for meningomyelocele typically involves surgery to repair the spinal defect and close the sac. In some cases, additional surgery may be necessary to address complications such as hydrocephalus (an accumulation of fluid in the brain) or tethered spinal cord (a condition in which the spinal cord is attached to surrounding tissues, causing it to stretch and damage nerve fibers). Overall, meningomyelocele is a serious condition that requires prompt medical attention and ongoing management to ensure the best possible outcomes for affected individuals.

Decompression, surgical refers to a surgical procedure in which pressure is relieved from a compressed or trapped body part or tissue. This can be done to treat a variety of medical conditions, including: 1. Herniated discs: A herniated disc occurs when the soft tissue inside a spinal disc bulges out through a tear in the outer layer. This can put pressure on the spinal cord or nerves, causing pain, numbness, or weakness. A surgical decompression may be performed to remove the herniated disc material and relieve pressure on the affected nerves. 2. Carpal tunnel syndrome: Carpal tunnel syndrome is a condition in which the median nerve, which runs from the forearm to the hand, becomes compressed or trapped in the wrist. This can cause pain, numbness, or weakness in the hand. A surgical decompression may be performed to release the pressure on the median nerve and relieve symptoms. 3. Sciatica: Sciatica is a type of pain that travels down the leg from the lower back. It can be caused by a herniated disc or other spinal condition that compresses the sciatic nerve. A surgical decompression may be performed to relieve pressure on the sciatic nerve and alleviate symptoms. 4. Tarsal tunnel syndrome: Tarsal tunnel syndrome is a condition in which the posterior tibial nerve, which runs from the calf to the foot, becomes compressed or trapped in the ankle. This can cause pain, numbness, or weakness in the foot. A surgical decompression may be performed to release the pressure on the posterior tibial nerve and relieve symptoms. Surgical decompression is typically performed under general anesthesia and may involve making a small incision in the skin to access the affected area. The surgeon will then use specialized instruments to remove any tissue or bone that is compressing the affected nerve or body part. After the procedure, the incision will be closed with stitches or staples, and the patient will be monitored for any complications.

Decompressive craniectomy is a surgical procedure in which a portion of the skull is removed to relieve pressure on the brain. This is typically done when the brain is swelling rapidly and the skull is not able to expand enough to accommodate the increased size of the brain. The goal of the surgery is to reduce intracranial pressure and prevent further damage to the brain. The removed bone is typically replaced with a synthetic material or the patient's own bone at a later date. The procedure is typically performed as an emergency treatment for severe head injuries or brain swelling caused by conditions such as stroke, brain tumor, or head trauma.

Down syndrome, also known as trisomy 21, is a genetic disorder caused by the presence of an extra copy of chromosome 21. This extra genetic material affects the development and function of the body, leading to a range of physical, cognitive, and medical characteristics. Individuals with Down syndrome typically have distinct facial features, such as a flattened face, small ears, and a short neck. They may also have intellectual disabilities, ranging from mild to moderate, and may experience delays in speech and language development. Other common features include an increased risk of certain medical conditions, such as heart defects, respiratory problems, and hearing loss. Down syndrome is caused by a random event during the formation of reproductive cells, and the risk of having a child with the condition increases with the age of the mother. There is currently no cure for Down syndrome, but early intervention and support can help individuals with the condition to reach their full potential and lead fulfilling lives.

Cerebrospinal fluid pressure (CSFP) is the pressure exerted by the cerebrospinal fluid (CSF) within the spinal canal and the ventricles of the brain. It is an important parameter in the diagnosis and management of various neurological conditions, including hydrocephalus, meningitis, and intracranial hypertension. CSFP is typically measured using a device called a lumbar puncture, which involves inserting a needle into the lower back and withdrawing a small amount of CSF for analysis. The pressure is then measured using a manometer. Normal CSFP ranges from 5 to 15 mmHg, although this can vary depending on the individual and the specific circumstances. Abnormal CSFP can be caused by a variety of factors, including increased production of CSF, decreased absorption of CSF, or blockage of the flow of CSF. High CSFP can lead to symptoms such as headache, nausea, vomiting, and confusion, while low CSFP can cause symptoms such as dizziness, weakness, and loss of consciousness.

Metabolic Syndrome X, also known as Syndrome X or Insulin Resistance Syndrome, is a cluster of conditions that increase the risk of developing heart disease, stroke, and type 2 diabetes. The five key components of Metabolic Syndrome X are: 1. Abdominal obesity: A waist circumference of 102 cm (40 inches) or more in men and 88 cm (35 inches) or more in women. 2. High blood pressure: A systolic blood pressure of 130 mmHg or higher, or a diastolic blood pressure of 85 mmHg or higher. 3. High fasting blood sugar: A fasting blood sugar level of 100 mg/dL or higher. 4. High triglyceride levels: A triglyceride level of 150 mg/dL or higher. 5. Low HDL cholesterol levels: An HDL cholesterol level of less than 40 mg/dL in men and less than 50 mg/dL in women. These conditions are often found together and can be caused by a variety of factors, including genetics, lifestyle, and certain medical conditions. Treatment for Metabolic Syndrome X typically involves lifestyle changes, such as diet and exercise, and may also include medication to manage blood pressure, blood sugar, and cholesterol levels.

In the medical field, the term "cattle" refers to large domesticated animals that are raised for their meat, milk, or other products. Cattle are a common source of food and are also used for labor in agriculture, such as plowing fields or pulling carts. In veterinary medicine, cattle are often referred to as "livestock" and may be treated for a variety of medical conditions, including diseases, injuries, and parasites. Some common medical issues that may affect cattle include respiratory infections, digestive problems, and musculoskeletal disorders. Cattle may also be used in medical research, particularly in the fields of genetics and agriculture. For example, scientists may study the genetics of cattle to develop new breeds with desirable traits, such as increased milk production or resistance to disease.

In the medical field, "cats" typically refers to Felis catus, which is the scientific name for the domestic cat. Cats are commonly kept as pets and are known for their agility, playful behavior, and affectionate nature. In veterinary medicine, cats are commonly treated for a variety of health conditions, including respiratory infections, urinary tract infections, gastrointestinal issues, and dental problems. Cats can also be used in medical research to study various diseases and conditions, such as cancer, heart disease, and neurological disorders. In some cases, the term "cats" may also refer to a group of animals used in medical research or testing. For example, cats may be used to study the effects of certain drugs or treatments on the immune system or to test new vaccines.

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He JC, Xu P, Peng LB (December 2009). "炔雌醇环丙孕酮致布加综合征1例" [A case of Budd-Chiari syndrome induced by ethinylestradiol and ... Budd-Chiari syndrome: case report". Reactions Weekly (1340): 20. February 2011. doi:10.2165/00128415-201113400-00066. ISSN 0114 ... Garty BZ, Dinari G, Gellvan A, Kauli R (May 1999). "Cirrhosis in a child with hypothalamic syndrome and central precocious ... ISBN 978-3-642-74614-7. Rittmaster RS (June 1999). "Antiandrogen treatment of polycystic ovary syndrome". Endocrinol. Metab. ...
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... angioplasty and TIPS in Budd-Chiari syndrome]". Gastroenterologia y Hepatologia. 26 (8): 461-464. doi:10.1016/s0210-5705(03) ... Root pain syndromes, known colloquially as radiculitis (i.e., sciatica) are one of the most common symptoms caused by damage to ... Degeneration or damage to the facet joints can often lead to facet joint syndrome, which can be both diagnosed and treated by ... When this happens near the pelvis, it is called pelvic congestion syndrome, which can lead to chronic pain beneath the level of ...
Negative abdominojugular reflux is seen in Budd-Chiari syndrome. A positive result is variously defined as either a sustained ...
The most common causes of liver disease resulting in portal hypertension are Budd-Chiari syndrome or cirrhosis. Budd-Chiari ... syndrome should not be mistaken for cirrhosis. Less common causes include diseases such as hemochromatosis, primary biliary ...
Budd-Chiari syndrome - Venous compression or obstruction in the liver. Nutcracker syndrome - Compression of the left renal vein ... May-Thurner syndrome (MTS), also known as the iliac vein compression syndrome, is a condition in which compression of the ... Venography will demonstrate the classical syndrome when causing deep venous thrombosis.[citation needed] May-Thurner syndrome ... This syndrome frequently manifests as pain when the limb is dependent (hanging down the edge of a bed/chair) and/or significant ...
"The utility of TIPS in the management of Budd-Chiari syndrome". Annals of Surgery. 241 (6): 978-981, discussion 982-983. doi: ...
Hans Chiari (1851-1916), Austrian pathologist (see Arnold-Chiari malformation, Budd-Chiari syndrome). Jacob Churg (1910-2005), ... I. N. Dubin (born 1913), American pathologist (see Dubin-Johnson syndrome). Cuthbert Dukes (1890-1977), English physician and ... John Bruce Beckwith (born 1933), American pathologist (see Beckwith-Wiedemann syndrome). Antonio di Paolo Benivieni (1443-1502 ... Lotte Strauss (1913-1985), American pathologist (see Churg-Strauss syndrome). Sunao Tawara (1873-1952), Japanese pathologist, ...
Pylephlebitis Budd-Chiari syndrome DeLeve LD, Valla DC, Garcia-Tsao G (May 2009). "Vascular disorders of the liver". Hepatology ... is known as hepatic vein thrombosis or Budd-Chiari syndrome. Portal vein thrombosis causes upper abdominal pain, possibly ... or antiphospholipid antibody syndrome) is another common cause. Nearly one-third of patients have a myeloproliferative disorder ...
Correction of a Budd-Chiari syndrome A milestone was the ingenious correction of a Budd-Chiari syndrome, crowned with success ... Transcaval posterocranial resection of the liver as treatment of the Budd-Chiari syndrome. World J. Surg. 1983; 7: 632-40 " ... Transcavale posterokraniale Resektion der Leber zur Behandlung des Budd-Chiari-Syndroms. World J. Surg. 1983; 7: 632-40 Larsson ... Transcavale posterokranielle Resektion der Leber zur Behandlung des Budd-Chiari-Syndroms. World J. Surg. 1983; 7: 632-40 ...
Budd-Chiari syndrome is a condition caused by blockage of the hepatic veins (including thrombosis) that drain the liver. It ... "Budd-Chiari syndrome in Sweden: epidemiology, clinical characteristics and survival - an 18-year experience". Liver ... There are also many pediatric liver diseases, including biliary atresia, alpha-1 antitrypsin deficiency, alagille syndrome, ...
Budd-Chiari syndrome is the clinical picture caused by occlusion of the hepatic vein. Some of the signs and symptoms of a liver ... Ground glass hepatocytes Primary biliary cirrhosis Budd-chiari syndrome Micrograph of non-alcoholic fatty liver disease There ... "Hepatic vein obstruction (Budd-Chiari): MedlinePlus Medical Encyclopedia". www.nlm.nih.gov. Retrieved 2015-06-20. "Liver ... Gilbert's syndrome, a genetic disorder of bilirubin metabolism found in a small percent of the population, can cause mild ...
Thurmes PJ, Steensma DP (July 2006). "Elevated serum erythropoietin levels in patients with Budd-Chiari syndrome secondary to ... People with untreated polycythemia vera have a substantial risk of Budd-Chiari syndrome (hepatic vein thrombosis). Untreated, ... 10 June 2011). "Treatment-related risk factors for transformation to acute myeloid leukemia and myelodysplastic syndromes in ...
Budd-Chiari syndrome (large liver vein obstruction due to hepatic vein thrombosis) Helmy A (January 2006). "Review article: ... The name sinusoidal obstruction syndrome (SOS) is preferred if hepatic veno-occlusive disease happens as a result of ... DeLeve LD, Shulman HM, McDonald GB (February 2002). "Toxic injury to hepatic sinusoids: sinusoidal obstruction syndrome (veno- ... sinusoidal obstruction syndrome) following haematopoietic stem cell transplantation". British Journal of Haematology. 163 (4): ...
Souza, who suffered from Budd-Chiari syndrome, died on 27 July 2010 of a heart attack. He died in a hospital in São Paulo, ...
... and described what is now known as Budd-Chiari syndrome, already noticed in 1842 by Carl von Rokitansky. It was followed by ... 1886). "Budd, George (1808-1882)" . Dictionary of National Biography. Vol. 7. London: Smith, Elder & Co. "Budd, George (BT827G ... which Budd contributed to Alexander Tweedie's Library of Practical Medicine, vol. iv.; vol. v. contained his "Scurvy". Budd ... George Budd M.D. (23 February 1808 - 14 March 1882) was an English physician, medical writer and academic. He was born at North ...
... and in Budd-Chiari syndrome. Budd-Chiari syndrome is a condition caused by blockage of the hepatic veins, such as by a blood ... The syndrome can be fulminant, acute, chronic, or asymptomatic.[citation needed] The independent lower veins draining the liver ...
Budd-Chiari syndrome is the blockage of a hepatic vein or of the hepatic part of the inferior vena cava. This form of ... Hughes, E. S. R. (1949-02-01). "Venous obstruction in the upper extremity; Paget-Schroetter's syndrome; a review of 320 cases ... paraneoplastic syndrome), by external compression on a blood vessel when a solid tumor is present, or (more rarely) extension ...
Other causes can include an obstructing clot in a hepatic vein (Budd Chiari syndrome) or compression from tumors or ... Some disorders as syndromes result from compression of a vein. These include a venous type of thoracic outlet syndrome, due to ... Compression of the superior vena cava most usually by a malignant tumor can lead to superior vena cava syndrome. A vascular ... Postphlebitic syndrome is venous insufficiency that develops following deep vein thrombosis. Venous thrombosis is the formation ...
Other causes can include an obstructing clot in a hepatic vein (Budd Chiari syndrome) or compression from tumors or ... Raynaud syndrome - a peripheral vascular disorder that causes constriction of the peripheral blood vessels in the fingers and ... Postphlebitic syndrome is venous insufficiency that develops following deep vein thrombosis. Venous thrombosis is the formation ... Kahn SR (August 2006). "The post-thrombotic syndrome: progress and pitfalls". British Journal of Haematology. 134 (4): 357-65. ...
Recurrent thromboembolism, or thrombosis in unusual sites (e.g. the hepatic vein in Budd-Chiari syndrome), is a generally ... The syndrome is often attributed to the British rheumatologist Graham R.V. Hughes, and is often referred to as Hughes syndrome ... Nephrotic syndrome, in which protein from the bloodstream is released into the urine due to kidney diseases, can predispose to ... Antiphospholipid syndrome was described in full in the 1980s, after various previous reports of specific antibodies in people ...
... such as the Budd-Chiari syndrome, heart failure, or liver cirrhosis) that increase the hydrostatic pressure in the circulatory ... Budd-Chiari syndrome, portal vein thrombosis, and idiopathic portal fibrosis. A low gradient (< 1.1 g/dL, ...
Budd Chiari Syndrome and APASL COVID Task Force. Mahtab is also a member of Regional Expert Panel on NAFLD/NASH and Survey Lead ... Budd Chiari Syndrome and liver fibrosis. He is a member of the Steering Committee of the Patient Registry of Hepatitis Free ...
Arnold-Chiari malformation @ Who Named It Julius Arnold @ Who Named It Budd-Chiari syndrome @ Who Named It Bendadi, F; van Tijn ... Another medical term named after Chiari is the Budd-Chiari syndrome, which is ascites and cirrhosis of the liver caused by an ... Lagerkvist, B; Olsen L (August 1991). "[The men behind the syndrome. John Cleland, Hans Chiari and Julius Arnold-3 men behind a ... It is named in conjunction with British physician George Budd (1808-1882). Lastly, Chiari is also famous for describing the " ...
... but was admitted to hospital on 12 June suffering Budd-Chiari syndrome. Despite the painful condition, she worked on a photo- ...
Arnold-Chiari malformation, or simply "Chiari malformation", a malformation of the brain Budd-Chiari syndrome, a disease with ... Chiari syndrome or Chiari's disease may refer to one of the following diseases named after the 19th century Austrian ... ascites and hepatomegaly caused by occlusion of the hepatic veins Chiari-Frommel syndrome, an older term for ... postpartum galactorrhea and amenorrhea This disambiguation page lists articles associated with the title Chiari syndrome. If an ...
... former Physician to the Queen George Budd, physician after who Budd-Chiari Syndrome is named after Walter Carr, physician and ... in Kearn-Sayre syndrome). Victor Horsley, pioneering Neurosurgeon Allan Octavian Hume, one of the founders of the Indian ... ophthalmologist and first described Treacher Collins Syndrome Dawson Turner, rugby union international who represented England ...
Budd Chiari syndrome) Hypoxic hepatitis Liver failure during pregnancy or Reye syndrome Unknown etiology Goals of MARS Therapy ... Pierre Versin is one of the pioneers in the study of hepatorenal syndrome in patients with liver impairment. Great efforts have ... 15 mg/dl (255 μmol/L), not responding to standard medical care alter 3 days Renal dysfunction or hepatorenal syndrome. Hepatic ... Arroyo, V (May 2000). "New treatments for hepatorenal syndrome". Liver Transplantation. 6 (3): 287-9. doi:10.1053/lv.2000.7569 ...
Budd-Chiari syndrome)".. {{cite journal}}. : Cite journal requires ,journal=. (help) *^ "The Budd-Chiari syndrome: a review".. ... Budd-Chiari syndrome at Who Named It? *^ Budd G (1845). On diseases of the liver. London: John Churchill. p. 135. Brit Lib. ... Syndrome 10-138d.Budd-Chiari Syndrome at Merck Manual of Diagnosis and Therapy Home Edition ... independently of Budd-Chiari syndrome.[23] Eponym[edit]. It is named after George Budd,[24][25] a British physician, and Hans ...
Budd-Chiari syndrome may be the presenting sign of these hypercoagulable disorders. Secondary Budd-Chiari syndrome, which is ... "The Budd-Chiari syndrome: a review". {{cite journal}}: Cite journal requires ,journal= (help) "Budd-Chiari syndrome: long-term ... may acquire Budd-Chiari. Any obstruction of the venous vasculature of the liver is referred to as Budd-Chiari syndrome, from ... independently of Budd-Chiari syndrome. It is named after George Budd, a British physician, and Hans Chiari, an Austrian ...
Budd-Chiari syndrome is an uncommon condition induced by thrombotic or nonthrombotic obstruction of hepatic venous outflow and ... encoded search term (Budd-Chiari Syndrome) and Budd-Chiari Syndrome What to Read Next on Medscape ... The clinical variants of Budd-Chiari syndrome have been described as follows [5, 6, 7] :. * Acute and subacute forms: ... Overt Budd-Chiari syndrome generally requires the occlusion of at least 2 hepatic veins. [8] Venous congestion of the liver ...
Budd-Chiari syndrome is an uncommon condition induced by thrombotic or nonthrombotic obstruction of hepatic venous outflow and ... encoded search term (Budd-Chiari Syndrome) and Budd-Chiari Syndrome What to Read Next on Medscape ... The clinical variants of Budd-Chiari syndrome have been described as follows [5, 6, 7] :. * Acute and subacute forms: ... Overt Budd-Chiari syndrome generally requires the occlusion of at least 2 hepatic veins. [8] Venous congestion of the liver ...
Budd-Chiari Syndrome - Etiology, pathophysiology, symptoms, signs, diagnosis & prognosis from the MSD Manuals - Medical ... Etiology of Budd-Chiari Syndrome In the Western world, the most common cause is a clot obstructing the hepatic veins and the ... Budd-Chiari syndrome usually develops over weeks or months. When it does develop over a period of time, cirrhosis Cirrhosis ... Symptoms and Signs of Budd-Chiari Syndrome Manifestations range from none (asymptomatic) to fulminant liver failure Acute Liver ...
A diagnosis of Budd-Chiari-like syndrome secondary to a membranous obstruction of the caudal vena cava was made. TREATMENT AND ... CLINICAL RELEVANCE: Budd-Chiari-like syndrome is a rare phenomenon in veterinary medicine, and congenital malformations should ... Placement of a caudal vena cava stent for treatment of Budd-Chiari-like syndrome in a 4-month-old Ragdoll cat. ...
Hepatic vein blockage is the most common cause of Budd-Chiari syndrome. ...
Budd-Chiari syndrome is an uncommon condition induced by thrombotic or nonthrombotic obstruction of hepatic venous outflow and ... encoded search term (Budd-Chiari Syndrome) and Budd-Chiari Syndrome What to Read Next on Medscape ... Budd-Chiari Syndrome Differential Diagnoses. Updated: Dec 16, 2015 * Author: Praveen K Roy, MD, AGAF; Chief Editor: BS Anand, ... Budd-Chiari syndrome: A single-center experience. World J Gastroenterol. 2014 Nov 21. 20(43):16236-44. [QxMD MEDLINE Link]. [ ...
Grus T, Lambert L, Grusová G, Banerjee R, Burgetová A (2017). "Budd-Chiari Syndrome". Prague Med Rep. 118 (2-3): 69-80. doi: ... CT scan may be helpful in the diagnosis of Budd-Chiari syndrome. Contrast-enhanced computed tomography (CT) is performed in ... Findings on CT scan suggestive of Budd-Chiari syndrome include: *Early enhancement of the caudate lobe and central liver around ... Findings on CT scan suggestive of Budd-Chiari syndrome include early enhancement of the caudate lobe and central liver around ...
Budd-Chiari syndrome is an uncommon condition induced by thrombotic or nonthrombotic obstruction of hepatic venous outflow and ... encoded search term (Budd-Chiari Syndrome) and Budd-Chiari Syndrome What to Read Next on Medscape ... The clinical variants of Budd-Chiari syndrome have been described as follows [5, 6, 7] :. * Acute and subacute forms: ... Overt Budd-Chiari syndrome generally requires the occlusion of at least 2 hepatic veins. [8] Venous congestion of the liver ...
What is Budd-Chiari syndrome and how can clinicians best diagnose and manage this form of liver disease? ... The Budd-Chiari syndrome (BCS) was first described by a British physician, William Budd in 1845 in his seminal work Diseases ... Budd-Chiari syndrome (BCS) is a rare disorder which may result in liver failure. Management should therefore be in coordination ... Table 3. Prognostic indices for Budd-Chiari syndrome. Study. Description. Patients (n). Formula. Actuarial survival according ...
Budd-Chiari Syndrome answers are found in the 5-Minute Clinical Consult powered by Unbound Medicine. Available for iPhone, iPad ... Syndrome. Budd-Chiari Syndrome. In: Domino FJF, Baldor RAR, Golding JJ, et al, eds. 5-Minute Clinical Consult. Wolters Kluwer; ... Syndrome. Budd-Chiari Syndrome [Internet]. In: Domino FJF, Baldor RAR, Golding JJ, Stephens MBM, editors. 5-Minute Clinical ... Syndrome. Accessed September 24, 2023.. Budd-Chiari Syndrome. (2020). In Domino, F. J., Baldor, R. A., Golding, J., & Stephens ...
Budd-Chiari syndrome features of Budd-Chiari sydrome features of PANCREAS DIVISUM liver cysts PORCELAIN GALLBLADDER ... what are the causes of Budd-Chiari syndrome. a) Paroxysmal nocturnal hemoglobulinuria b) bilary atresia c) Right heart failure ...
... PubMed, SCI, Scopus, ESCI, PMC indexed ... Budd-Chiari Syndrome as A Manifestation of Right Atrium Myxoma. Article Information. Komoni F1, Rakovica L1, Sherifi F1, Hyseni ... cardiac tumors are rarely associated with the development of Budd-Chiari syndrome [1, 4]. The clinical presentation of Budd- ... Budd-Chiari syndrome (BCS) is a rare condition marked by a number of symptoms due to hepatic venous obstruction. A 40-year-old ...
Pregnancy Outcomes in Women with Budd Chiari Syndrome or Portal Hypertension. E.N. Hamulyák1, H.M.G. Wiegers1, J.R. van Duuren1 ... Background: Women with Budd Chiari Syndrome (BCS) and/or portal hypertension are at high risk of pregnancy complications and ... Hamulyák EN, Wiegers HMG, van Duuren JR, Middeldorp S, Ganzevoort W. Pregnancy Outcomes in Women with Budd Chiari Syndrome or ... abstracts.isth.org/abstract/pregnancy-outcomes-in-women-with-budd-chiari-syndrome-or-portal-hypertension/. Accessed October 1, ...
Patients with suprahepatic lesions tend to present with signs and symptoms of Budd-Chiari syndrome; examples of these include ... and imaging features of Budd-Chiari syndrome and/or portal hypertension. Analogous changes can be depicted with MRI and MRV. ... significant IVC stenosis can be differentiated from pseudostenosis by the presence of features of Budd-Chiari syndrome and ...
Differentiating Budd-Chiari syndrome from cirrhosis by history or physical examination may be difficult. Thus, Budd-Chiari ... Symptoms of Budd-Chiari syndrome are attributed to decreased outflow of blood from the liver, with resulting hepatic congestion ... What are the signs and symptoms of Budd-Chiari syndrome?. What is the role of abdominal ultrasonography in the evaluation of ... The latter conditions, and the symptoms they produce, are termed Budd-Chiari syndrome. Predisposing conditions include ...
T1 - Current knowledge in pathophysiology and management of Budd-Chiari syndrome and non-cirrhotic non-tumoral splanchnic vein ... 2019). Current knowledge in pathophysiology and management of Budd-Chiari syndrome and non-cirrhotic non-tumoral splanchnic ... Current knowledge in pathophysiology and management of Budd-Chiari syndrome and non-cirrhotic non-tumoral splanchnic vein ... Current knowledge in pathophysiology and management of Budd-Chiari syndrome and non-cirrhotic non-tumoral splanchnic vein ...
Hepatic Venous Occlusion Type of Budd-Chiari Syndrome versus Pyrrolizidine Alkaloid-Induced Hepatic Sinusoidal Obstructive ... "Hepatic Venous Occlusion Type of Budd-Chiari Syndrome versus Pyrrolizidine Alkaloid-Induced Hepatic Sinusoidal Obstructive ... Venous Occlusion Type of Budd-Chiari Syndrome versus Pyrrolizidine Alkaloid-Induced Hepatic Sinusoidal Obstructive Syndrome: A ... Venous Occlusion Type of Budd-Chiari Syndrome versus Pyrrolizidine Alkaloid-Induced Hepatic Sinusoidal Obstructive Syndrome: A ...
Budd-Chiari Syndrome. OVERDOSAGE. Serious ill effects have not been reported following acute ingestion of large doses of oral ...
Budd-Chiari syndrome: current management options. Ann Surg. 2001 Apr. 233(4):522-7. [QxMD MEDLINE Link]. [Full Text]. ... Budd-Chiari syndrome is a liver-related condition associated with large-vessel thromboses and outflow obstruction with inferior ... Budd-Chiari syndrome occurs in patients with myeloproliferative disease (MPD) and most frequently in young women. Surgical ... The following procedures have been used in patients with Budd-Chiari syndrome:. * Transjugular intrahepatic portosystemic shunt ...
Nick has Antiphospholipid Syndrome and Budd Chiari. Please see my first journal for the complete story that started while Nick ...
Budd-Chiari syndrome. * Hepatic insufficiency. * Hepatic vascular malformations. * Hepatitis A, B & C (chronic or acute) ...
Hepatic: Benign liver tumors, Budd-Chiari syndrome, cholestatic jaundice, hepatic adenomas Local: Thrombophlebitis Ocular: ... Genitourinary: Cervical erosion changes, cervical secretion changes, cystitis-like syndrome, vaginal candidiasis, vaginitis ... Central nervous system: Depression, dizziness, headache, migraine, nervousness, premenstrual syndrome, stroke Dermatologic: ... Hematologic: Antithrombin III decreased, folate levels decreased, hemolytic uremic syndrome, norepinephrine induced platelet ...
Clarification: Women with Budd-Chiari syndrome should not use CHCs because of the increased risk for thrombosis.. ... Maternal-perinatal outcome associated with the syndrome of hemolysis, elevated liver enzymes, and low platelets in severe ... Known thrombophilia, including antiphospholipid syndrome. • Active cancer (metastatic, receiving therapy, or within 6 months ... Known thrombophilia, including antiphospholipid syndrome. • Active cancer (metastatic, receiving therapy, or within 6 months ...
Acute Budd-chiari syndrome *Cirrhosis with fatty liver *Ciliated hepatic foregut cyst *hepatocellular adenoma with rupture and ...
Sinusoidal obstruction syndrome: antineoplastic cytotoxic agents (Paediatr Drugs 2010;12:277) *Budd-Chiari syndrome: oral ... 51 year old man with vanishing bile duct syndrome after infliximab for malignant melanoma (Cureus 2022;14:e21940) *61 year old ... Microvesicular steatosis: amiodarone, didanosine, stavudine, valproate, zalcitabine, aspirin (Reye syndrome) (J Hepatol 2019;70 ... Differential diagnoses include drug induced liver injury, sepsis, shock, paraneoplastic syndrome, intrahepatic cholestasis of ...
Another rare condition that causes elevation of liver tests is called the Budd-Chiari Syndrome. In this condition, obstruction ...
Primary Budd-Chiari syndrome: outcome of endovascular management for suprahepatic venous obstruction. J Vasc Surg 2006;43:101-8 ... Servelle-Martorell and Budd-Chiari syndromes).. Patients and controls underwent a non-invasive study of cerebrospinal venous ... varicocele and/or pelvic congestion syndrome, Cockett and thoracic outlet syndromes, indwelling central venous catheters or ... For instance, PP course, characterised by a slowly progressive syndrome with spastic paraparesis and MRI demonstration of MS ...
Percutaneous drainage in the treatment of intrahepatic pancreatic pseudocyst with Budd-Chiari syndrome: A case report ... Percutaneous drainage in the treatment of intrahepatic pancreatic pseudocyst with Budd-Chiari syndrome: A case report . World J ... Percutaneous drainage in the treatment of intrahepatic pancreatic pseudocyst with Budd-Chiari syndrome: A case report . World J ...
  • [13] [2] Other acquired hypercoagulable disorders that may result in Budd-Chiari syndrome include antiphospholipid syndrome and paroxysmal nocturnal hemoglobinuria , which are responsible for 10-12% and 7-12% of Budd-Chiari syndrome cases, respectively. (wikipedia.org)
  • Underlying prothrombotic disorders included myeloproliferative neoplasms (N=4), protein C deficiency (N=2), antiphospholipid syndrome (N=1) and cirrhosis (N=5) [Table 1]. (isth.org)
  • Nick has Antiphospholipid Syndrome and Budd Chiari. (caringbridge.org)
  • Primary Budd-Chiari syndrome occurs due to thrombosis of the hepatic vein. (wikipedia.org)
  • [2] Inherited disorders of hypercoagulability may lead to thrombosis of the hepatic vein and Budd-Chiari syndrome. (wikipedia.org)
  • Alternatively, this syndrome is characterized by occlusion due to hepatic vein thrombosis or mechanical venous obstruction. (fortunejournals.com)
  • Complications include iron deficiency (from chronic haemoglobinuria), progressive renal impairment (from haemoglobinuria), and the Budd Chiari syndrome (hepatic vein thrombosis). (lu.se)
  • Budd-Chiari syndrome is obstruction of hepatic venous outflow that originates anywhere from the small hepatic veins inside the liver to the inferior vena cava and right atrium. (msdmanuals.com)
  • Budd-Chiari syndrome is characterized by obstruction of hepatic venous flow at the level of right atrium, inferior vena cava, large hepatic veins, or hepatic venules [1]. (fortunejournals.com)
  • The prognosis is poor in patients with Budd-Chiari syndrome who remain untreated, with death resulting from progressive liver failure in 3 months to 3 years from the time of the diagnosis. (medscape.com)
  • A diagnosis of Budd-Chiari-like syndrome secondary to a membranous obstruction of the caudal vena cava was made. (avmi.net)
  • Aydinli M, Bayraktar Y. Budd-Chiari syndrome: etiology, pathogenesis and diagnosis. (medscape.com)
  • CT scan may be helpful in the diagnosis of Budd-Chiari syndrome. (wikidoc.org)
  • The diagnosis, initial management and long-term follow-up of patients with Budd-Chiari syndrome is reviewed. (medscape.com)
  • Budd-Chiari syndrome is also seen in tuberculosis , congenital venous webs and occasionally in inferior vena caval stenosis . (wikipedia.org)
  • up to 39% develop venous thromboses, [17] and 12% may acquire Budd-Chiari. (wikipedia.org)
  • Any obstruction of the venous vasculature of the liver is referred to as Budd-Chiari syndrome, from the venules to the right atrium . (wikipedia.org)
  • Budd-Chiari syndrome is an uncommon condition induced by thrombotic or nonthrombotic obstruction of the hepatic venous outflow and is characterized by hepatomegaly, ascites, and abdominal pain. (medscape.com)
  • Tian ZL, Jia GL, Xi HL, Feng S, Wang XK, Li R. Investigation on etiology of hepatic venous obstruction Budd-Chiari syndrome. (medscape.com)
  • Budd-Chiari syndrome is a rare disorder caused by hepatic venous outflow obstruction and resulting hepatic dysfunction. (medscape.com)
  • Budd-Chiari syndrome (BCS) is a rare condition marked by a number of symptoms due to hepatic venous obstruction. (fortunejournals.com)
  • Cutaneous manifestations of cirrhosis include jaundice, spider angiomata, skin telangiectasias ("paper money skin"), palmar erythema, white nails, disappearance of lunulae, and finger clubbing, especially in the setting of hepatopulmonary syndrome. (medscape.com)
  • [2] [14] Budd-Chiari syndrome may be the presenting sign of these hypercoagulable disorders. (wikipedia.org)
  • Several factors predispose to the development of Budd-Chiari syndrome, including hypercoagulable, inherited, and acquired conditions, as well as a variety of other causes, can be identified in approximately 75% of patients. (fortunejournals.com)
  • Etiology, management, and outcome of the Budd-Chiari syndrome. (medscape.com)
  • Budd-Chiari syndrome: long-term effect on outcome with transjugular intrahepatic portosystemic shunt. (medscape.com)
  • Overt Budd-Chiari syndrome generally requires the occlusion of at least 2 hepatic veins. (medscape.com)
  • In 1899, Chiari described an "obliterating endophlebitis of the hepatic veins" and its association with hepatomegaly, ascites and abdominal pain. (medscape.com)
  • Budd-Chiari syndrome is an uncommon disorder resulting from obstruction of the large hepatic veins or inferior vena cava at the suprahepatic level. (fortunejournals.com)
  • These cases are known as idiopathic Budd-Chiari syndrome. (wikipedia.org)
  • Budd-Chiari syndrome should be considered separate from veno-occlusive disease (VOD), also known as sinusoidal obstruction syndrome, which is characterized by toxin-induced, nonthrombotic obstruction of prehepatic veins (see the images below). (medscape.com)
  • Outflow obstruction caused by the sinusoidal obstruction syndrome (formerly 'veno-occlusive disease') and cardiac disorders is excluded. (medscape.com)
  • Hepatocellular carcinoma in Budd-Chiari syndrome: characteristics and risk factors. (medscape.com)
  • Liu FY, Wang MQ, Duan F, Fan QS, Song P, Wang Y. Hepatocellular carcinoma associated with Budd-Chiari syndrome: imaging features and transcatheter arterial chemoembolization. (medscape.com)
  • We searched PubMed without time restriction using key words: bone marrow and fibrosis as the main stem against the terms: growth factors, cytokines and chemokines, morphology, megakaryocytes and platelets, myeloproliferative disorders, myelodysplastic syndrome, collagen biosynthesis, mesenchymal stem cells, vitamins and minerals and hormones, and mechanism of tissue fibrosis. (bvsalud.org)
  • Budd-Chiari syndrome in Sweden: epidemiology, clinical characteristics and survival - an 18-year experience. (medscape.com)
  • Herein, our authors present a case of a 40-year-old female patient with Budd-Chiari syndrome in whom the right atrium myxoma was manifested with worsening of clinical symptoms despite medical treatment therapy. (fortunejournals.com)
  • [2] The syndrome can be fulminant , acute, chronic, or asymptomatic. (wikipedia.org)
  • The acute syndrome presents with rapidly progressive severe upper abdominal pain , yellow discoloration of the skin and whites of the eyes , liver enlargement , enlargement of the spleen , fluid accumulation within the peritoneal cavity , elevated liver enzymes , and eventually encephalopathy . (wikipedia.org)
  • Incidence, prevalence, and complications of Budd-Chiari syndrome in South Korea: a nationwide, population-based study. (medscape.com)
  • Women with Budd Chiari Syndrome (BCS) and/or portal hypertension are at high risk of pregnancy complications and adverse outcomes. (isth.org)
  • The fulminant syndrome presents early with encephalopathy and ascites. (wikipedia.org)
  • Hamulyák EN, Wiegers HMG, van Duuren JR, Middeldorp S, Ganzevoort W. Pregnancy Outcomes in Women with Budd Chiari Syndrome or Portal Hypertension [abstract]. (isth.org)
  • Pregnancy in women with known and treated Budd-Chiari syndrome: maternal and fetal outcomes. (medscape.com)
  • Secondary Budd-Chiari syndrome, which is very rare compared to the primary variant, is due to compression of the hepatic vein by an outside structure (such as a tumor or polycystic kidney disease ). (wikipedia.org)
  • Sometimes Budd-Chiari syndrome begins during pregnancy and unmasks a previously asymptomatic hypercoagulability disorder. (msdmanuals.com)
  • CLINICAL RELEVANCE: Budd-Chiari-like syndrome is a rare phenomenon in veterinary medicine, and congenital malformations should be considered in young feline patients with ascites. (avmi.net)
  • Rotterdam score predicts early mortality in Budd-Chiari syndrome, and surgical shunting prolongs transplant-free survival. (medscape.com)
  • Intracardiac tumors have rarely been reported as a predisposing cause for the development of Budd-Chiari syndrome, and surgical removal of these tumors is generally a curative treatment for this syndrome [1, 3-6]. (fortunejournals.com)
  • The description of the clinical features of hepatic vein outflow obstruction is generally attributed to a pathologist, Hans Chiari (although he was not the first). (medscape.com)
  • Budd-Chiari syndrome is a very rare condition, affecting one in a million adults. (wikipedia.org)
  • What's more, while in the hospital, she learned she has another condition called Budd Chiari syndrome, a liver condition. (wkyt.com)
  • The majority of patients have a slower-onset form of Budd-Chiari syndrome. (wikipedia.org)
  • The cause of Budd-Chiari syndrome can be found in more than 80% of patients. (wikipedia.org)
  • Medicine Central , im.unboundmedicine.com/medicine/view/5-Minute-Clinical-Consult/1688517/0.2/Budd_Chiari_Syndrome. (unboundmedicine.com)
  • [ 1 ] Budd did not associate any clinical features with this finding. (medscape.com)
  • Qi X, Ren W, Wang Y, Guo X, Fan D. Survival and prognostic indicators of Budd-Chiari syndrome: a systematic review of 79 studies. (medscape.com)
  • The classic form of CD manifests as a malabsorption syndrome associated with chronic diarrhea, mineral deficiencies, failure to thrive, and weight loss. (hindawi.com)
  • An important non-genetic risk factor is the use of estrogen-containing forms of hormonal contraception , which is implicated in 22% of cases of Budd-Chiari syndrome. (wikipedia.org)
  • 80% of all Budd-Chiari syndrome (BCS) cases occur in women. (unboundmedicine.com)
  • Hepatic vein blockage is the most common cause of Budd-Chiari syndrome. (medlineplus.gov)