Bucladesine: A cyclic nucleotide derivative that mimics the action of endogenous CYCLIC AMP and is capable of permeating the cell membrane. It has vasodilator properties and is used as a cardiac stimulant. (From Merck Index, 11th ed)Internet: A loose confederation of computer communication networks around the world. The networks that make up the Internet are connected through several backbone networks. The Internet grew out of the US Government ARPAnet project and was designed to facilitate information exchange.Developing Countries: Countries in the process of change with economic growth, that is, an increase in production, per capita consumption, and income. The process of economic growth involves better utilization of natural and human resources, which results in a change in the social, political, and economic structures.Software: Sequential operating programs and data which instruct the functioning of a digital computer.Medication Errors: Errors in prescribing, dispensing, or administering medication with the result that the patient fails to receive the correct drug or the indicated proper drug dosage.Waiting Lists: Prospective patient listings for appointments or treatments.Medication Adherence: Voluntary cooperation of the patient in taking drugs or medicine as prescribed. This includes timing, dosage, and frequency.Pharmacogenetics: A branch of genetics which deals with the genetic variability in individual responses to drugs and drug metabolism (BIOTRANSFORMATION).Genetic Testing: Detection of a MUTATION; GENOTYPE; KARYOTYPE; or specific ALLELES associated with genetic traits, heritable diseases, or predisposition to a disease, or that may lead to the disease in descendants. It includes prenatal genetic testing.Genomics: The systematic study of the complete DNA sequences (GENOME) of organisms.Individualized Medicine: Therapeutic approach tailoring therapy for genetically defined subgroups of patients.Knowledge Bases: Collections of facts, assumptions, beliefs, and heuristics that are used in combination with databases to achieve desired results, such as a diagnosis, an interpretation, or a solution to a problem (From McGraw Hill Dictionary of Scientific and Technical Terms, 6th ed).Biomarkers, Pharmacological: Measurable biological parameters that serve for drug development, safety and dosing (DRUG MONITORING).Gastric Acid: Hydrochloric acid present in GASTRIC JUICE.Gastric Mucosa: Lining of the STOMACH, consisting of an inner EPITHELIUM, a middle LAMINA PROPRIA, and an outer MUSCULARIS MUCOSAE. The surface cells produce MUCUS that protects the stomach from attack by digestive acid and enzymes. When the epithelium invaginates into the LAMINA PROPRIA at various region of the stomach (CARDIA; GASTRIC FUNDUS; and PYLORUS), different tubular gastric glands are formed. These glands consist of cells that secrete mucus, enzymes, HYDROCHLORIC ACID, or hormones.Disease Models, Animal: Naturally occurring or experimentally induced animal diseases with pathological processes sufficiently similar to those of human diseases. They are used as study models for human diseases.Parietal Cells, Gastric: Rounded or pyramidal cells of the GASTRIC GLANDS. They secrete HYDROCHLORIC ACID and produce gastric intrinsic factor, a glycoprotein that binds VITAMIN B12.Gastric Juice: The liquid secretion of the stomach mucosa consisting of hydrochloric acid (GASTRIC ACID); PEPSINOGENS; INTRINSIC FACTOR; GASTRIN; MUCUS; and the bicarbonate ion (BICARBONATES). (From Best & Taylor's Physiological Basis of Medical Practice, 12th ed, p651)Pentagastrin: A synthetic pentapeptide that has effects like gastrin when given parenterally. It stimulates the secretion of gastric acid, pepsin, and intrinsic factor, and has been used as a diagnostic aid.Nitric Oxide Synthase: An NADPH-dependent enzyme that catalyzes the conversion of L-ARGININE and OXYGEN to produce CITRULLINE and NITRIC OXIDE.Drug Information Services: Services providing pharmaceutic and therapeutic drug information and consultation.National Library of Medicine (U.S.): An agency of the NATIONAL INSTITUTES OF HEALTH concerned with overall planning, promoting, and administering programs pertaining to advancement of medical and related sciences. Major activities of this institute include the collection, dissemination, and exchange of information important to the progress of medicine and health, research in medical informatics and support for medical library development.Counterfeit Drugs: Drugs manufactured and sold with the intent to misrepresent its origin, authenticity, chemical composition, and or efficacy. Counterfeit drugs may contain inappropriate quantities of ingredients not listed on the label or package. In order to further deceive the consumer, the packaging, container, or labeling, may be inaccurate, incorrect, or fake.Government Agencies: Administrative units of government responsible for policy making and management of governmental activities.MEDLARS: A computerized biomedical bibliographic storage and retrieval system operated by the NATIONAL LIBRARY OF MEDICINE. MEDLARS stands for Medical Literature Analysis and Retrieval System, which was first introduced in 1964 and evolved into an online system in 1971 called MEDLINE (MEDLARS Online). As other online databases were developed, MEDLARS became the name of the entire NLM information system while MEDLINE became the name of the premier database. MEDLARS was used to produce the former printed Cumulated Index Medicus, and the printed monthly Index Medicus, until that publication ceased in December 2004.Portal Vein: A short thick vein formed by union of the superior mesenteric vein and the splenic vein.Lipopeptides: Compounds consisting of a short peptide chain conjugated with an acyl chain.Lipoproteins: Lipid-protein complexes involved in the transportation and metabolism of lipids in the body. They are spherical particles consisting of a hydrophobic core of TRIGLYCERIDES and CHOLESTEROL ESTERS surrounded by a layer of hydrophilic free CHOLESTEROL; PHOSPHOLIPIDS; and APOLIPOPROTEINS. Lipoproteins are classified by their varying buoyant density and sizes.Lysine-tRNA Ligase: An enzyme that activates lysine with its specific transfer RNA. EC 6.1.1.6.Amino Acyl-tRNA Synthetases: A subclass of enzymes that aminoacylate AMINO ACID-SPECIFIC TRANSFER RNA with their corresponding AMINO ACIDS.Pertussis Toxin: One of the virulence factors produced by BORDETELLA PERTUSSIS. It is a multimeric protein composed of five subunits S1 - S5. S1 contains mono ADPribose transferase activity.HL-60 Cells: A promyelocytic cell line derived from a patient with ACUTE PROMYELOCYTIC LEUKEMIA. HL-60 cells lack specific markers for LYMPHOID CELLS but express surface receptors for FC FRAGMENTS and COMPLEMENT SYSTEM PROTEINS. They also exhibit phagocytic activity and responsiveness to chemotactic stimuli. (From Hay et al., American Type Culture Collection, 7th ed, pp127-8)Cell Differentiation: Progressive restriction of the developmental potential and increasing specialization of function that leads to the formation of specialized cells, tissues, and organs.Pulmonary Embolism: Blocking of the PULMONARY ARTERY or one of its branches by an EMBOLUS.Venous Thromboembolism: Obstruction of a vein or VEINS (embolism) by a blood clot (THROMBUS) in the blood stream.Thromboembolism: Obstruction of a blood vessel (embolism) by a blood clot (THROMBUS) in the blood stream.Venous Thrombosis: The formation or presence of a blood clot (THROMBUS) within a vein.Embolectomy: Surgical removal of an obstructing clot or foreign material which has been transported from a distant vessel by the bloodstream. Removal of a clot at its original site is called THROMBECTOMY.Vena Cava Filters: Mechanical devices inserted in the inferior vena cava that prevent the migration of blood clots from deep venous thrombosis of the leg.Anticoagulants: Agents that prevent clotting.OregonNeuroendocrinology: The study of the anatomical and functional relationships between the nervous system and the endocrine system.National Institute of Diabetes and Digestive and Kidney Diseases (U.S.): Component of the NATIONAL INSTITUTES OF HEALTH. It conducts and supports basic and applied research for a national program in diabetes, endocrinology, and metabolic diseases; digestive diseases and nutrition; and kidney, urologic, and hematologic diseases. It was established in 1948.Longitudinal Ligaments: Two extensive fibrous bands running the length of the vertebral column. The anterior longitudinal ligament (ligamentum longitudinale anterius; lacertus medius) interconnects the anterior surfaces of the vertebral bodies; the posterior longitudinal ligament (ligamentum longitudinale posterius) interconnects the posterior surfaces. The commonest clinical consideration is OSSIFICATION OF POSTERIOR LONGITUDINAL LIGAMENT. (From Stedman, 25th ed)Endocrinology: A subspecialty of internal medicine concerned with the metabolism, physiology, and disorders of the ENDOCRINE SYSTEM.Bibliometrics: The use of statistical methods in the analysis of a body of literature to reveal the historical development of subject fields and patterns of authorship, publication, and use. Formerly called statistical bibliography. (from The ALA Glossary of Library and Information Science, 1983)Publications: Copies of a work or document distributed to the public by sale, rental, lease, or lending. (From ALA Glossary of Library and Information Science, 1983, p181)Neostriatum: The phylogenetically newer part of the CORPUS STRIATUM consisting of the CAUDATE NUCLEUS and PUTAMEN. It is often called simply the striatum.alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid: An IBOTENIC ACID homolog and glutamate agonist. The compound is the defining agonist for the AMPA subtype of glutamate receptors (RECEPTORS, AMPA). It has been used as a radionuclide imaging agent but is more commonly used as an experimental tool in cell biological studies.Receptors, AMPA: A class of ionotropic glutamate receptors characterized by their affinity for the agonist AMPA (alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid).Corpus Striatum: Striped GRAY MATTER and WHITE MATTER consisting of the NEOSTRIATUM and paleostriatum (GLOBUS PALLIDUS). It is located in front of and lateral to the THALAMUS in each cerebral hemisphere. The gray substance is made up of the CAUDATE NUCLEUS and the lentiform nucleus (the latter consisting of the GLOBUS PALLIDUS and PUTAMEN). The WHITE MATTER is the INTERNAL CAPSULE.Neurons: The basic cellular units of nervous tissue. Each neuron consists of a body, an axon, and dendrites. Their purpose is to receive, conduct, and transmit impulses in the NERVOUS SYSTEM.Adenosine: A nucleoside that is composed of ADENINE and D-RIBOSE. Adenosine or adenosine derivatives play many important biological roles in addition to being components of DNA and RNA. Adenosine itself is a neurotransmitter.Dopamine and cAMP-Regulated Phosphoprotein 32: A phosphoprotein that was initially identified as a major target of DOPAMINE activated ADENYLYL CYCLASE in the CORPUS STRIATUM. It regulates the activities of PROTEIN PHOSPHATASE-1 and PROTEIN KINASE A, and it is a key mediator of the biochemical, electrophysiological, transcriptional, and behavioral effects of DOPAMINE.

The optically determined size of exo/endo cycling vesicle pool correlates with the quantal content at the neuromuscular junction of Drosophila larvae. (1/2312)

According to the current theory of synaptic transmission, the amplitude of evoked synaptic potentials correlates with the number of synaptic vesicles released at the presynaptic terminals. Synaptic vesicles in presynaptic boutons constitute two distinct pools, namely, exo/endo cycling and reserve pools (). We defined the vesicles that were endocytosed and exocytosed during high K+ stimulation as the exo/endo cycling vesicle pool. To determine the role of exo/endo cycling vesicle pool in synaptic transmission, we estimated the quantal content electrophysiologically, whereas the pool size was determined optically using fluorescent dye FM1-43. We then manipulated the size of the pool with following treatments. First, to change the state of boutons of nerve terminals, motoneuronal axons were severed. With this treatment, the size of exo/endo cycling vesicle pool decreased together with the quantal content. Second, we promoted the FM1-43 uptake using cyclosporin A, which inhibits calcineurin activities and enhances endocytosis. Cyclosporin A increased the total uptake of FM1-43, but neither the size of exo/endo cycling vesicle pool nor the quantal content changed. Third, we increased the size of exo/endo cycling vesicle pool by forskolin, which enhances synaptic transmission. The forskolin treatment increased both the size of exo/endo cycling vesicle pool and the quantal content. Thus, we found that the quantal content was closely correlated with the size of exo/endo cycling vesicle pool but not necessarily with the total uptake of FM1-43 fluorescence by boutons. The results suggest that vesicles in the exo/endo cycling pool primarily participate in evoked exocytosis of vesicles.  (+info)

Luteinizing hormone inhibits conversion of pregnenolone to progesterone in luteal cells from rats on day 19 of pregnancy. (2/2312)

We have previously reported that intrabursal ovarian administration of LH at the end of pregnancy in rats induces a decrease in luteal progesterone (P4) synthesis and an increase in P4 metabolism. However, whether this local luteolytic effect of LH is exerted directly on luteal cells or on other structures, such as follicular or stromal cells, to modify luteal function is unknown. The aim of the present study was to determine the effect of LH on isolated luteal cells obtained on Day 19 of pregnancy. Incubation of luteal cells with 1, 10, 100, or 1000 ng/ml of ovine LH (oLH) for 6 h did not modify basal P4 production. The addition to the culture medium of 22(R)-hydroxycholesterol (22R-HC, 10 microgram/ml), a membrane-permeable P4 precursor, or pregnenolone (10(-2) microM) induced a significant increase in P4 accumulation in the medium in relation to the control value. When luteal cells were preincubated for 2 h with oLH, a significant (p < 0.01) reduction in the 22R-HC- or pregnenolone-stimulated P4 accumulation was observed. Incubation of luteal cells with dibutyryl cAMP (1 mM, a cAMP analogue) plus isobutylmethylxanthine (1 mM, a phosphodiesterase inhibitor) also inhibited pregnenolone-stimulated P4 accumulation. Incubation with an inositol triphosphate synthesis inhibitor, neomycin (1 mM), or an inhibitor of intracellular Ca2+ mobilization, (8,9-N, N-diethylamino)octyl-3,4,5-trimethoxybenzoate (1 mM), did not prevent the decrease in pregnenolone-stimulated P4 secretion induced by oLH. It was concluded that the luteolytic action of LH in late pregnancy is due, at least in part, to a direct action on the luteal cells and that an increase in intracellular cAMP level might mediate this effect.  (+info)

D-Aspartate stimulation of testosterone synthesis in rat Leydig cells. (3/2312)

D-Aspartate increases human chorionic gonadotropin-induced testosterone production in purified rat Leydig cells. L-Aspartate, D-,L-glutamate or D-,L-asparagine could not substitute for D-aspartate and this effect was independent of glutamate receptor activation. Testosterone production was enhanced only in cells cultured with D-aspartate for more than 3 h. The increased production of testosterone was well correlated with the amounts of D-aspartate incorporated into the Leydig cells, and L-cysteine sulfinic acid, an inhibitor of D-aspartate uptake, suppressed both testosterone production and intracellular D-aspartate levels. D-Aspartate therefore is presumably taken up into cells to increase steroidogenesis. Intracellular D-aspartate probably acts on cholesterol translocation into the inner mitochondrial membrane, the rate-limiting process in steroidogenesis.  (+info)

Overexpression of nucleoside diphosphate kinases induces neurite outgrowth and their substitution to inactive forms leads to suppression of nerve growth factor- and dibutyryl cyclic AMP-induced effects in PC12D cells. (4/2312)

Whether nucleoside diphosphate kinase (NDPK) is involved in neuronal differentiation was investigated with special reference to its enzyme activity. Neurite outgrowth of PC12D cells induced by nerve growth factor or a cyclic AMP analog was suppressed to some extent when inactive NDPKs (the active site histidine 118 was replaced with alanine), not active forms, were transiently overexpressed. This suppression was more definite in their stably expressed clones. NDPKbeta-transfected clones and, to a lesser extent, NDPKalpha-transfected clones, but not inactive NDPK-transfected clones, extended neurites without differentiation inducers. These results imply that NDPKs may play a role by exerting their enzyme activity during differentiation of PC12 cells.  (+info)

Posttranslational regulation of the retinoblastoma gene family member p107 by calpain protease. (5/2312)

The retinoblastoma protein plays a critical role in regulating the G1/S transition. Less is known about the function and regulation of the homologous pocket protein p107. Here we present evidence for the posttranslational regulation of p107 by the Ca2+-activated protease calpain. Three negative growth regulators, the HMG-CoA reductase inhibitor lovastatin, the antimetabolite 5-fluorouracil, and the cyclic nucleotide dibutyryl cAMP were found to induce cell type-specific loss of p107 protein which was reversible by the calpain inhibitor leucyl-leucyl-norleucinal but not by the serine protease inhibitor phenylmethylsulfonylfluoride, caspase inhibitors, or lactacystin, a specific inhibitor of the 26S proteasome. Purified calpain induced Ca2+-dependent p107 degradation in cell lysates. Transient expression of the specific calpain inhibitor calpastatin blocked the loss of p107 protein in lovastatin-treated cells, and the half-life of p107 was markedly lengthened in lovastatian-treated cells stably transfected with a calpastatin expression vector versus cells transfected with vector alone. The data presented here demonstrate down-regulation of p107 protein in response to various antiproliferative signals, and implicate calpain in p107 posttranslational regulation.  (+info)

Downregulation of JAK3 protein levels in T lymphocytes by prostaglandin E2 and other cyclic adenosine monophosphate-elevating agents: impact on interleukin-2 receptor signaling pathway. (6/2312)

The Janus kinase, JAK3 plays an important role in interleukin-2 (IL-2)-dependent signal transduction and proliferation of T lymphocytes. Our findings show that prostaglandin E2 (PGE2) can inhibit upregulation of JAK3 protein in naive T cells and can downregulate its expression in primed cells. Reduction in JAK3 was selective because expression of other tyrosine kinases (JAK1, p56(lck), and p59(fyn)) and signal transducer and activator of transcription (STAT)5, which are linked to IL-2 receptor (IL-2R) signaling pathway, were not affected. Inhibition of JAK3 may be controlled by intracellular cyclic adenosine monophosphate (cAMP) levels, as forskolin, a direct activator of adenylate cyclase and dibutyryl cAMP (dbcAMP), a membrane permeable analogue of cAMP suppressed JAK3 expression. Moreover, 3-isobutyl-1-methylxanthine (IBMX), an inhibitor of cAMP phosphodiesterase, potentiated PGE2-induced suppression of JAK3. In naive T cells, but not primed T cells, PGE2 and other cAMP elevating agents also caused a modest reduction in surface expression of the common gamma chain (gammac) that associates with JAK3. The absence of JAK3, but not IL-2R in T cells correlated with impaired IL-2-dependent signal transduction and proliferation. The alteration in IL-2 signaling included decreased tyrosine phosphorylation and DNA binding activity of STAT5 and poor induction of the c-Myc and c-Jun pathways. In contrast, IL-2-dependent induction of Bcl-2 was unaffected. These findings suggest that suppression of JAK3 levels may represent one mechanism by which PGE2 and other cAMP elevating agents can inhibit T-cell proliferation.  (+info)

cAMP-dependent induction of PDE5 expression in murine neuroblastoma cell differentiation. (7/2312)

The present study demonstrates, in both hybrid NG108-15 and mouse neuroblastoma N18TG2 cells, the presence and regulation of PDE5 mRNA during cell differentiation. PDE5 cDNA probes in Northern blot analysis recognize a approximately 9 kb transcript in bovine lung as well as in mouse neuroblastoma cells. Hybridization on total RNA extracted from dibutyryl-cAMP-treated NG108-15 cells shows a 5-fold increase of PDE5 9 kb mRNA: such an increase is not observed in N18TG2 although we observed a similar increase in the enzymatic activity of both cell lines. Our data demonstrate that PDE5 gene expression can be regulated by cAMP and suggest the existence of a complex regulatory system for PDE5 activity.  (+info)

Caffeine does not inhibit substance P-evoked intracellular Ca2+ mobilization in rat salivary acinar cells. (8/2312)

We used the Ca2+-sensitive fluorescent dye fura 2, together with measurements of intracellular D-myo-inositol 1,4,5-trisphosphate [Ins(1,4,5)P3], to assess the inhibitory effects of caffeine on signal transduction via G protein-coupled receptor pathways in isolated rat mandibular salivary acinar cells. ACh, norepinephrine (NE), and substance P (SP) all evoked substantial increases in the intracellular free Ca2+ concentration ([Ca2+]i). Responses to ACh and NE were markedly inhibited by prior application of 20 mM caffeine. The inhibitory effect of caffeine was not reproduced by phosphodiesterase inhibition with IBMX or addition of cell-permeant dibutyryl cAMP. In contrast to the ACh and NE responses, the [Ca2+]i response to SP was unaffected by caffeine. Despite this, SP and ACh appeared to mobilize Ca2+ from a common intracellular pool. Measurements of agonist-induced changes in Ins(1,4,5)P3 levels confirmed that caffeine inhibited the stimulus-response coupling pathway at a point before Ins(1,4,5)P3 generation. Caffeine did not, however, inhibit [Ca2+]i responses evoked by direct activation of G proteins with 40 mM F-. These data show that caffeine inhibits G protein-coupled signal transduction in these cells at some element that is common to the muscarinic and alpha-adrenergic signaling pathways but is not shared by the SP signaling pathway. We suggest that this element might be a specific structural motif on the G protein-coupled muscarinic and alpha-adrenergic receptors.  (+info)

*Bucladesine

... is a cell permeable cAMP analog. The compound is used in a wide variety of research applications because it mimics ... Bucladesine is a cyclic nucleotide derivative which mimics the action of endogenous cAMP and is a phosphodiesterase inhibitor. ... Bucladesine (50-100nM/mouse) showed significant attenuation in the morphine withdrawal syndrome in the wild-type mice. In ... Recently, the effect of bucladesine as a cAMP analog has been studied on the pentylenetetrazol-induced seizure in the wild-type ...

*H-89

"Effects of pentoxifylline and H-89 on epileptogenic activity of bucladesine in pentylenetetrazol-treated mice". European ...

*Nardosinone

In in vitro studies, the compound has demonstrated concentration-dependent enhancement of bucladesine and staurosporine-induced ...

*CAMP-dependent pathway

bucladesine (dibutyryl cAMP, db cAMP) - also a phosphodiesterase inhibitor pertussis toxin, which increases cAMP levels by ...

*ATC code C01

... combinations C01CE01 Amrinone C01CE02 Milrinone C01CE03 Enoximone C01CE04 Bucladesine QC01CE90 Pimobendane C01CX06 ...

*List of phosphodiesterase inhibitors

... dihydrate Amipizone Apremilast Arofylline Atizoram Befuraline Bemarinone hydrochloride Bemoradan Benafentrine Bucladesine ...

*List of MeSH codes (D13)

... bucladesine MeSH D13.695.462.250 --- cyclic cmp MeSH D13.695.462.275 --- cyclic gmp MeSH D13.695.462.275.325 --- dibutyryl ... bucladesine MeSH D13.695.827.068.506 --- flavin-adenine dinucleotide MeSH D13.695.827.068.694 --- nad MeSH D13.695.827.068.749 ... bucladesine MeSH D13.695.667.138.410 --- deoxyadenine nucleotides MeSH D13.695.667.138.506 --- flavin-adenine dinucleotide MeSH ...

*List of drugs: Bs-Bz

... bucladesine (INN) Bucladin-S buclizine (INN) buclosamide (INN) bucloxic acid (INN) bucolome (INN) bucricaine (INN) bucrilate ( ...
The effects of dibutyryl cyclic adenosine monophosphate (dB-cAMP) were studied in fifty cats, twenty anesthetized with pentobarbital and thirty with halothane. Nasopharyngeal temperature and Paco2 were maintained at normal values. Somatosensory evoked response was monitored and used as an indicator of cerebral cortical function. Ischemic hypoxic injury was produced by an orthopedic tourniquet snugly applied around the animals neck and inflated for a period of fifteen minutes. This method produces a reliable and reproducible injury. Times for recovery of the evoked response to 10% of control value, as well as immediate and long-term animal survival, were noted. The dBcAMP was administered at the end of the hypoxic insult. Treated animals recovered the evoked response earlier than the untreated controls and had better immediate and long-term survival rates. ...
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I need to find: Histochemical Journal 32(10):581-90,2000 Oct. Title of the article is The effect of dibutyryl cyclic AMP on the expression of actin isoforms in astroglia. Does anyone out there in Histo Land have this or could point me to a good source? Thanks in advance, Carol Ann Bobrowitz Histology Core Laboratory Department of Physiology Medical College of Wisconsin Milwaukee, Wisconsin (414) 456-8179 FAX (414) 456-6546 [email protected] ...
Bucladesine is a medicine available in a number of countries worldwide. A list of US medications equivalent to Bucladesine is available on the Drugs.com website.
Glial fibrillary acidic protein (GFA) expression was induced in rat C6 glioma in chemically defined medium by the addition of N6,O2-dibutyryl cyclic AMP (dbcAMP). Induction was dependent on the increase in intracellular cyclic AMP (cAMP), which was linearly correlated with added dbcAMP. Contrary to GFA mRNA synthesis, which can be obtained by cAMP-dependent and -independent pathways, translation of mRNA into GFA was observed only above a cellular cAMP concentration of approximately 0.2 fmol/cell. dbcAMP stimulation did not affect the vimentin concentration, which remained at a low level, but changed the cellular morphology from a bipolar to a stellate shape. A similar morphological change was observed after stimulation of C6 with lipopolysaccharide (LPS). However, LPS did not significantly increase the intracellular concentration of cAMP and the LPS-induced mRNA was not translated into GFA. Our results indicate that GFA synthesis is regulated at the mRNA level and at the translational level and ...
1. Cyclic AMP-stimulated protein kinase activity phosphorylating intrinsic substrates in preparations of synaptic-membrane fragments from ox cerebral cortex was examined in relation to (a) the content of membrane-bound Ca2+in the preparations and (b) added Ca2+in the assay medium. 2. Centrifugal washing of synaptic-membrane fragments with buffered ethane dioxybis(ethylamine)tetra-acetate solutions decreased bound Ca2+from 2.8±0.4 (s.d.) to 0.9±0.3nmol/mg of protein. In washed preparations basal protein kinase activity was increased by about 40% and the cyclic AMP-stimulated activity by about 15%. Addition of Ca2+in the concentration range 5-50μm to the assay medium progressively inhibited the kinase activity of the washed preparations; in this range of Ca2+concentration the basal activity was inhibited more than the stimulated activity. 3. In unwashed preparations concentrations of Ca2+above 100μm inhibited the cyclic AMP-stimulated activity more than the basal activity. 4. The inhibitory ...
Innovative genomic test for bucladesine personalised pharmacogenomic analysis to explore how your genes can affect and modulate your response to bucladesine ...
The detector detects the ion as it creates an electric current as it attracts electrons. This creates a mass spectra showing the mass to charge ratio (m/z). This shows the abundance of each isotope. There are also ghost results - this is when a +2 ion is detected and due to its higher charge this means it is much lower than the other detections ...
Juurlink, B H.; Hertz, L; and Fedoroff, S, "Effects of high serum levels, dibutyryl cyclic amp and hydrocortisone on glutamine synthetase activity in primary mouse astroglial cultures. Abstr." (1979). Subject Strain Bibliography 1979. 1485 ...
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Looking for online definition of cyclic adenosine monophosphate (cyclic AMP, cAMP, 3',5'-cAMP) in the Medical Dictionary? cyclic adenosine monophosphate (cyclic AMP, cAMP, 3',5'-cAMP) explanation free. What is cyclic adenosine monophosphate (cyclic AMP, cAMP, 3',5'-cAMP)? Meaning of cyclic adenosine monophosphate (cyclic AMP, cAMP, 3',5'-cAMP) medical term. What does cyclic adenosine monophosphate (cyclic AMP, cAMP, 3',5'-cAMP) mean?
To test whether angiotensinogen might be targeted to dense core secretory granules in cells containing a regulated secretory pathway, we expressed rat angiotensinogen in AtT-20 cells, a mouse pituitary cell line that has the demonstrated ability to correctly sort proteins to the constitutive or regulated pathway. We compared the pattern of secretion of angiotensinogen with that of endogenous adrenocorticotropin hormone, which is secreted by AtT-20 cells through the regulated pathway. When cells were incubated for 5 hours with dibutyryladenosine cyclic monophosphate or KCl, adrenocorticotropin hormone secretion was significantly higher than control, whereas monensin had no effect. In contrast, angiotensinogen secretion was markedly reduced by monensin, but no stimulation was observed with dibutyryladenosine cyclic monophosphate or KCl. These results make it unlikely that angiotensinogen could be cotargeted with active renin in the dense core granules of the regulated pathway. Alternative ...
Induction of neural differentiation in cultures of undetermined presumptive epidermis from three amphibian species was achieved by the addition of 1 millimolar dibutyryl adenosine 3,5-monophosphate, 8-bromadenosine 3,5-monophosphate, or adenosine C,E-monophosphate together with theophylline. Adenosine 5-monophosphate, adenosine 2,3-monophosphate, dibutyryl guanosine 3,5-monophosphate, and butyrate at 1 millimolar are ineffective. These results suggest that the action of the primary inductor or inductors may be mediated via adenosine 3,5-monophosphate. ...
Both treatments resulted in improvements in lung function that were sustained throughout the study. As compared with treatment with high-dose budesonide, treatment with low-dose budesonide plus theophylline resulted in greater improvements in forced
Available data indicate that the liver is a target organ for parathyroid hormone (PTH) and that this effect is most likely mediated by PTH-induced calcium entry into hepatocytes. The present study examined the effects of both PTH-(1-84) and its amino-terminal fragment [PTH-(1-34)] on cytosolic calcium concentration ([Ca2+]i) of hepatocytes and explored the cellular pathways that mediate this potential action of PTH. Both moieties of PTH produced a dose-dependent rise in [Ca2+]i, but the effect of PTH-(1-84) was greater (P | 0.01) than an equimolar amount of PTH-(1-34). This effect required calcium in the medium and was totally [PTH-(1-34)] or partially [PTH-(1-84)] blocked by PTH antagonist ([Nle8,18,Tyr34]bPTH-(7-34)-NH2] and by verapamil or nifedipine. Sodium or chloride channel blockers did not modify this effect. 12-O-tetradecanoylphorbol 13-acetate (TPA), an activator of protein kinase C, dibutyryl adenosine 3,5-cyclic monophosphate (DBcAMP), and G protein activator also produced a dose-dependent
amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;lt;a href="https://tockify.com/wischumane" data-mce-href="https://tockify.com/wischumane"&amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;gt;Community Events ...
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The kinetics for activation of the cyclic adenosine 3′:5′-monophosphate (cyclic AMP)-dependent protein kinase (PKA) and thymidine incorporation into DNA was investigated in epinephrine- and prostaglandin E1 (PGE1)-treated murine P1798 lymphosarcoma cells. A positive correlation between the duration and extent of PKA activation and Accumulation of cyclic AMP and inhibition of thymidine incorporation into DNA was observed with both hormones. Epinephrine and PGE1 elevated intracellular cyclic AMP 34- and 14-fold, respectively. All hormone concentrations which increased cyclic AMP accumulation also promoted inhibition of thymidine incorporation into DNA. In addition, dibutyryl cyclic AMP (50 µm) inhibited thymidine incorporation.. No difference in the kinetics for activation of PKA was observed when cells were treated with µm epinephrine or PGE1. With both agents, 50% PKA activation was observed when intracellular cyclic AMP concentrations were elevated 6.5-fold, or to 9 pmol/106 cells. In the ...
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Cyclic 3,5-adenosine monophosphate modulates vascular endothelial cell migration in vitro.: Using a modified Boyden chamber assay, we have examined the effect
15731-72-3 - UEUPTUCWIHOIMK-RRKCRQDMSA-N - 2-Deoxy-3-adenosine monophosphate - Similar structures search, synonyms, formulas, resource links, and other chemical information.
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Synaptic-membrane fragments from ox cerebral cortex contain basal and cyclic AMP-stimulated protein kinase activity catalysing the phosphorylation of endogenous substrates. Extraction of membrane fragments with Triton X-100 solubilized less than 20% of the kinase activity and left the major part of the endogenous substrates in the insoluble fraction.. ...
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Gandelman, K.Y., S.E. Pfeiffer, and J.H. Carson. "Cyclic AMP regulation of P0 glycoprotein and myelin basic protein gene expression in semi-differentiated peripheral neurinoma cell line D6P2T." Development 106.2 (1989): 389-398. Web. 05 April. 2020. ...
Background: Previously weve found out that the decline in β2-adrenoreceptors (β2-AR) function affects airway response to cold air.. Objective: The aim of our study was to reveal the contribution of Arg16Gly SNP (rs1042713) in the development of cold airway hyperresponsiveness (CAHR) in asthmatics.. Methods: The study included examination of 60 mild to moderate asthmatics of Caucasian race, mostly non-smokers (mean age 36±1.39). All the patients underwent spirometry before and after the challenge with 3-minute isocapnic (5% CO2) cold air (-20°C) hyperventilation (ICAH). More than 10% drop in FEV1 was interpreted as a positive result. Intracellular cyclic adenosine monophosphate (cAMP) concentration in lymphocytes was measured before and 30 min after ICAH under in vitro stimulation with 10-6M epinephrine. PCR-RFLP analysis was used for genotyping.. Results: Arg16Arg genotype dominated in the group with CAHR (χ2=7.47; p=0.02). Mean FEV1 drop differed between homozygous patients (16.03±2.07 ...
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Intracellular cAMP levels are higher in LDLR-/−p110γ−/− than in LDLR−/−p110γ+/−macrophages.LDLR−/−p110γ+/− and LDLR−/−p110γ−/− BMM
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Ущелье&nbsp;Джеты-Огуз&nbsp;или Скалы Семи Быков (кирг. Жети-Өгүз) - живописное горное ущелье в Киргизии в 28 км&nbsp;на запад от&nbsp...
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9,000 남자의 큰 연구 결과는 적합하게 측정된 분석실험에 적용될 때 효과적으로 임상의를 내분비 사회의 임상 내분비학 & 물질 대사의 전표에서 간행된 새로운 연구 결과에 따라 성기능 부전의 정확한 진단을, 만드는 가능하게 할 남자에 있는 총 테스토스테론을 위한 화합한 참조 범위를 설치했습니다
TY - JOUR. T1 - Effects of cyclic AMP on the growth of differentiating and undifferentiated friend erythroleukemic cells. AU - Rubin, Charles S.. PY - 1978/1. Y1 - 1978/1. N2 - Elevated concentrations of cyclic AMP elicit only minor reductions in growth rate and saturation density in undifferentiated Friend erythroleukemic cells. During the course of dimethylsulfoxide (DMSO)‐induced differentiation, Friend cells convert from a cyclic AMP‐tolerant state to a phenotype characterized by a high degree of sensitivity to cyclic AMP‐mediated growth arrest. Conversion to cyclic AMP sensitivity is detectable after 30 hours growth in medium containing 2% DMSO, and either 0.5 mM 8‐Br‐cyclic AMP or 5 nM cholera toxin. Cultures of differentiating Friend cells achieved a stationary phase density that was approximately 8‐fold higher than the cell density observed in parallel, differentiating cultures treated with 0.5 mM 8‐Br‐cyclic AMP. Temporally, the appearance of cyclic AMP‐sensitivity ...
Adamson, E D.; Gaunt, S J.; and Graham, C F., "The differentiation of teratocarcinoma stem cells is marked by the types of collagen which are synthesized." (1979). Subject Strain Bibliography 1979. 4144 ...
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lt;p>&amp;amp;amp;lt;p&amp;amp;amp;gt;&amp;amp;amp;amp;amp;amp;amp;lt;p&amp;amp;amp;amp;amp;amp;amp;gt;&amp;amp;amp;amp;amp;amp;amp;amp;amp;lt;p&amp;amp;amp;amp;amp;amp;amp;amp;amp;gt;&amp;amp;amp;amp;amp;amp;amp;amp;amp;lt;a href="http://video.news.com.au/2227300814/Shorten-supports-PM-on-Slipper"&amp;amp;amp;amp;amp;amp;amp;amp;amp;gt;VIDEO: Shorten supports PM on Slipper&amp;amp;amp;amp;amp;amp;amp;amp;amp;lt;/a&amp;amp;amp;amp;amp;amp;amp;amp;amp;gt;&amp;amp;amp;amp;amp;amp;amp;amp;amp;lt;/p&amp;amp;amp;amp;amp;amp;amp;amp;amp;gt;&amp;amp;amp;amp;amp;amp;amp;lt;/p&amp;amp;amp;amp;amp;amp;amp;gt;&amp;amp;amp;lt;/p&amp;amp;amp;gt;</p> ...
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Cyclic adenosine monophosphate (cAMP) controls the physiological response to many diverse extracellular stimuli. To maintain signal specificity, cAMP-mediated signaling is finely tuned by means of a complex array of proteins that control the spatial and temporal dynamics of the second messenger within the cell. To unravel the way a cell encodes cAMP signals, new biosensors have recently been introduced that allow imaging of the second messenger in living cells with high spatial resolution. The more recent generation of such biosensors exploits the phenomenon of fluorescence resonance energy transfer between the green fluorescent protein- tagged subunits of a chimeric protein kinase A, as the way to visualize and measure the dynamic fluctuations of cAMP. This chapter describes the molecular basis on which such a genetically encoded cAMP sensor relies and the tools and methods required to perform cAMP measurements in living samples.
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Fertilization tubules in wild-type (A) and ida5 (B) mt+ gametes produced in response to a 1-h exposure to 10 mM dibutyryl-cAMP and 1 mM IBMX. Bar, 0.3 μm.
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SUMMARY: The effect of adding dibutyryl cyclic AMP to five species of Mucorales was tested. With Mucor hiemalis and M. mucedo a rapid halt of apical extension and a slower rate of resumed growth were observed together with marked morphological changes, including hyphal dilation, apical swelling, septation and multiple branching. Sporangiospore development, and the mating reaction in M. mucedo were both inhibited; carotene pigmentation was increased. Phycomyces blakesleeanus, Blakeslea trispora and Zygorhynchus moelleri were insensitive to dibutyryl cyclic AMP.
Sickle cell disease (SCD) is a chronic inflammatory condition characterized by high leucocyte counts, altered cytokine levels and endothelial cell injury. As the removal of inflammatory cells by apoptosis is fundamental for the resolution of inflammation, we aimed to determine whether the leucocyte apoptotic process is altered in SCD. Neutrophils from SCD individuals showed an inhibition of spontaneous apoptosis when cultured in vitro, in the presence of autologous serum for 20 h. Intracellular cyclic adenosine monophosphate (cAMP) levels were approximately twofold increased in SCD neutrophils; possible cAMP-upregulating factors present in SCD serum include interleukin-8, granulocyte-macrophage colony-stimulating factor and prostaglandin. Accordingly, co-incubation of SCD neutrophils with KT5720, a cAMP-dependent protein kinase (PKA) inhibitor, abrogated increased SCD neutrophil survival. Caspase-3 activity was also significantly diminished in SCD neutrophils cultured for 16 h and this activity ...
Institute of Human Nutrition, Columbia University, New York, New York 10032, USA. As F9 embryonal carcinoma cells differentiate into parietal endoderm-like cells, expression of conventional protein kinase C (PKC) changes. Undifferentiated stem cells express PKCbeta but not PKCalpha, whereas differentiated parietal endoderm cells express PKCalpha but not PKCbeta. To determine whether changes in PKCalpha and/or PKCbeta expression control retinoic acid (RA)- and dibutyryl cyclic AMP-induced F9 cell differentiation, we established cell lines stably expressing PKCalpha, PKCbeta, antisense PKCalpha, or antisense PKCbeta RNAs. Constitutive expression of PKCalpha or inhibition of PKCbeta expression in F9 stem cells enhanced RA induced differentiation, both by increasing total expression and accelerating RA-induced expression of laminins A, B1, B2, and type IV collagen. In addition, expressing PKCbeta in a parietal endoderm cell line caused these cells to retrodifferentiate into stem cells. Based on ...
Periodical: McGee, Richard, Paul Simpson, Clifford Christian, Marina Mata, Phillip Nelson, and Marshall W. Nirenberg. Regulation of Acetylcholine Release from Neuroblastoma x Glioma Hybrid Cells. Proceedings of the National Academy of Sciences of the United States of America 75, 3 (March 1978): 1314-1318. Article. 5 Images ...

Bucladesine - Drugs.comBucladesine - Drugs.com

A list of US medications equivalent to Bucladesine is available on the Drugs.com website. ... Bucladesine is a medicine available in a number of countries worldwide. ...
more infohttps://www.drugs.com/international/bucladesine.html

Bucladesine - WikipediaBucladesine - Wikipedia

Bucladesine is a cell permeable cAMP analog. The compound is used in a wide variety of research applications because it mimics ... Bucladesine is a cyclic nucleotide derivative which mimics the action of endogenous cAMP and is a phosphodiesterase inhibitor. ... Bucladesine (50-100nM/mouse) showed significant attenuation in the morphine withdrawal syndrome in the wild-type mice. In ... Recently, the effect of bucladesine as a cAMP analog has been studied on the pentylenetetrazol-induced seizure in the wild-type ...
more infohttps://en.wikipedia.org/wiki/Bucladesine

BucladesineBucladesine

... , Sodium salt,DC-2797,Actosin,Barium salt,Bucladesine, Barium salt ... Bucladesine,N-(1-Oxobutyl)adenosine cyclic 3,5-(hydrogen phosphate) 2-butanoate,N-(9-beta-D-ribofuranosyl-9H-purin-6-yl) ...
more infohttp://www.druglead.com/cds/bucladesine.html

BUCLADESINE - UKBUCLADESINE - UK

... personalised pharmacogenomic analysis to explore how your genes can affect and modulate your response to bucladesine ... ... Knowing the optimal dose of bucladesine to treat your medical condition, and whether bucladesine is safe to treat your medical ... Can the treatment to your medical condition with bucladesine pose a safety concern to your health because of your genetic ... What is the optimal medicament dose of bucladesine to treat your medical condition in line with your genomic makeup? ...
more infohttps://genomicmedicineuk.com/product/bucladesine/

Bucladesine | Profiles RNSBucladesine | Profiles RNS

"Bucladesine" is a descriptor in the National Library of Medicines controlled vocabulary thesaurus, MeSH (Medical Subject ... This graph shows the total number of publications written about "Bucladesine" by people in this website by year, and whether " ... Below are the most recent publications written about "Bucladesine" by people in Profiles. ...
more infohttps://profiles.rush.edu/display/13122

Drug Information Portal - U.S. National Library of Medicine - Quick Access to Quality Drug InformationDrug Information Portal - U.S. National Library of Medicine - Quick Access to Quality Drug Information

Bucladesine [INN] View Synonyms. View Structure. Description:. A cyclic nucleotide derivative that mimics the action of ...
more infohttps://druginfo.nlm.nih.gov/drugportal/rn/362-74-3

Nitric oxide-an endogenous inhibitor of gastric acid secretion in isolated human gastric glands.Nitric oxide-an endogenous inhibitor of gastric acid secretion in isolated human gastric glands.

Bucladesine / metabolism. Carbon Radioisotopes / metabolism. Enzyme Inhibitors / pharmacology. Gastric Acid / secretion*. ... 362-74-3/Bucladesine; 50903-99-6/NG-Nitroarginine Methyl Ester; 51-45-6/Histamine; 58-15-1/Aminopyrine; 74-79-3/Arginine; 79032 ...
more infohttp://www.biomedsearch.com/nih/Nitric-oxide-endogenous-inhibitor-gastric/15298720.html

Function of microdissected pancreatic islets cultured in a chemically defined medium. I. Insulin content and release.Function of microdissected pancreatic islets cultured in a chemically defined medium. I. Insulin content and release.

Bucladesine / pharmacology. Culture Media. Culture Techniques*. Dissection*. Drug Synergism. Female. Glucose / pharmacology. ... 0/Culture Media; 10238-21-8/Glyburide; 11061-68-0/Insulin; 362-74-3/Bucladesine; 50-99-7/Glucose; 58-55-9/Theophylline; 61-90-5 ...
more infohttp://www.biomedsearch.com/nih/Function-microdissected-pancreatic-islets-cultured/173613.html

Sox1 (SRY (sex determining region Y)-box 1) - Rat Genome DatabaseSox1 (SRY (sex determining region Y)-box 1) - Rat Genome Database

bucladesine affects expression. ISO. RGD:1348976. 6480464. Bucladesine affects the expression of SOX1 mRNA, Bucladesine affects ... bucladesine multiple interactions. ISO. RGD:1348976. 6480464. bisphenol A affects the reaction [Bucladesine affects the ... Paraquat affects the reaction [Bucladesine affects the expression of SOX1 mRNA], Paraquat affects the reaction [Bucladesine ... bisphenol A affects the reaction [Bucladesine affects the expression of SOX1 mRNA] more .... CTD. PMID:27271280, PMID:31400064 ...
more infohttps://rgd.mcw.edu/rgdweb/report/gene/main.html?id=1615160

Drug Information Portal - U.S. National Library of Medicine - Quick Access to Quality Drug InformationDrug Information Portal - U.S. National Library of Medicine - Quick Access to Quality Drug Information

Bucladesine sodium [JAN] View Synonyms. View Structure. Description:. A cyclic nucleotide derivative that mimics the action of ...
more infohttps://druginfo.nlm.nih.gov/drugportal/rn/16980-89-5

cAMP-dependent pathway - WikipediacAMP-dependent pathway - Wikipedia

bucladesine (dibutyryl cAMP, db cAMP) - also a phosphodiesterase inhibitor. *pertussis toxin, which increases cAMP levels by ...
more infohttps://en.wikipedia.org/wiki/CAMP-dependent_pathway

H 89 2HCl | PKA inhibitor | Read Reviews & Product Use Citations
		
	H 89 2HCl | PKA inhibitor | Read Reviews & Product Use Citations

Dibutyryl-cAMP (Bucladesine) New Dibutyryl-cAMP (Bucladesine) is a cell-permeable PKA activator by mimicing the action of ...
more infohttp://www.selleckchem.com/products/H-89-dihydrochloride.html

Labeling of HepG2 cells with exogenously administered C | Open-iLabeling of HepG2 cells with exogenously administered C | Open-i

Bucladesine/pharmacology. *Carcinoma, Hepatocellular. *Cell Membrane/metabolism. *Dithionite/pharmacology. *Fluorescent Dyes. * ...
more infohttps://openi.nlm.nih.gov/detailedresult.php?img=PMC2139765_JCB.vanijzendoorn1af&req=4

Protein Tyrosine Kinase Compound Library-MedchemExpressProtein Tyrosine Kinase Compound Library-MedchemExpress

Bucladesine (sodium salt) Bucladesine (calcium salt) SU6656 Altiratinib AZ-5104 Trapidil Chloropyramine hydrochloride Empty. ...
more infohttps://www.medchemexpress.com/screening/Protein_Tyrosine_Kinase_Compound_Library.html

Stem Cell Signaling Compound Library-MedchemExpressStem Cell Signaling Compound Library-MedchemExpress

Bucladesine (calcium salt) PF-3084014 SIS3 Bucladesine (sodium salt) MK-4101 VX-11e Tofacitinib (citrate) Empty. ...
more infohttps://www.medchemexpress.com/screening/Stem_Cell_Signaling_Compound_Library.html

Incomplete functional differentiation of HL-60 leukemic cells by synthetic lipopeptides. Partial inhibition by pertussis toxin...Incomplete functional differentiation of HL-60 leukemic cells by synthetic lipopeptides. Partial inhibition by pertussis toxin...

Bucladesine/pharmacology. MESH. Calcitriol/pharmacology. MESH. Cell Differentiation. MESH. Dimethyl Sulfoxide/pharmacology. ...
more infohttps://epub.uni-regensburg.de/23223/

Formation and release of nitric oxide from human neutrophils and HL-60 cells induced by a chemotactic peptide, platelet...Formation and release of nitric oxide from human neutrophils and HL-60 cells induced by a chemotactic peptide, platelet...

Bucladesine/pharmacology. MESH. Calcitriol/pharmacology. MESH. Canavanine/pharmacology. MESH. Cell Line. MESH. ...
more infohttps://epub.uni-regensburg.de/23259/

PKA抑制剂 | PKA InhibitorPKA抑制剂 | PKA Inhibitor

Dibutyryl-cAMP (Bucladesine) Dibutyryl-cAMP (Bucladesine)是一种细胞可渗透性 PKA 激活剂,通过模拟内源性 cAMP 发挥作用。 ... Dibutyryl-cAMP (Bucladesine). Dibutyryl-cAMP (Bucladesine)是一种细胞可渗透性 PKA 激活剂,通过模拟内源性 cAMP 发挥作用
more infohttps://www.selleck.cn/pka.html

Kaveh Tabrizian[au] - PubMed - NCBIKaveh Tabrizian[au] - PubMed - NCBI

Zinc Chloride and Lead Acetate-Induced Passive Avoidance Memory Retention Deficits Reversed by Nicotine and Bucladesine in Mice ... The quantitative evaluation of cholinergic markers in spatial memory improvement induced by nicotine-bucladesine combination in ... Post-training intrahippocampal infusion of nicotine-bucladesine combination causes a synergistic enhancement effect on spatial ...
more infohttps://www.ncbi.nlm.nih.gov/pubmed?cmd=search&term=Kaveh+Tabrizian%5Bau%5D&dispmax=50

Integration of calcium and cyclic AMP signaling pathways by 14-3-3 by Chi-Wing Chow and Roger J. Davis"Integration of calcium and cyclic AMP signaling pathways by 14-3-3" by Chi-Wing Chow and Roger J. Davis

14-3-3 Proteins; Amino Acid Sequence; Animals; Binding Sites; Bucladesine; Calcineurin; Calcium; *Calcium Signaling; Cell Line ...
more infohttps://escholarship.umassmed.edu/oapubs/1440/

Catalytic activity of type II iodothyronine 5-deiodinase polypeptide  by Marjorie Safran, Alan P. Farwell et al."Catalytic activity of type II iodothyronine 5'-deiodinase polypeptide " by Marjorie Safran, Alan P. Farwell et al.

Affinity Labels; Animals; Animals, Newborn; Astrocytes; Binding Sites; Brain; Bucladesine; Catalysis; Cells, Cultured; ...
more infohttps://escholarship.umassmed.edu/oapubs/797/

Hormonal regulation of TSE1-repressed genes: Evidence for multiple genetic controls in extinction<...Hormonal regulation of TSE1-repressed genes: Evidence for multiple genetic controls in extinction<...

TY - JOUR. T1 - Hormonal regulation of TSE1-repressed genes. T2 - Evidence for multiple genetic controls in extinction. AU - Thayer, Mathew (Matt). AU - Fournier, R. E K. PY - 1989. Y1 - 1989. UR - http://www.scopus.com/inward/record.url?scp=0024377927&partnerID=8YFLogxK. UR - http://www.scopus.com/inward/citedby.url?scp=0024377927&partnerID=8YFLogxK. M3 - Article. C2 - 2571076. AN - SCOPUS:0024377927. VL - 9. SP - 2837. EP - 2846. JO - Molecular and Cellular Biology. JF - Molecular and Cellular Biology. SN - 0270-7306. IS - 7. ER - ...
more infohttps://ohsu.pure.elsevier.com/en/publications/hormonal-regulation-of-tse1-repressed-genes-evidence-for-multiple-2
  • The neurite outgrowth instigated by bucladesine in cell cultures has been shown to be enhanced by nardosinone. (wikipedia.org)
  • Bucladesine" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings) . (rush.edu)
  • This graph shows the total number of publications written about "Bucladesine" by people in this website by year, and whether "Bucladesine" was a major or minor topic of these publications. (rush.edu)