Tumors or cancer of the BRONCHI.
Tumors or cancer of the PANCREAS. Depending on the types of ISLET CELLS present in the tumors, various hormones can be secreted: GLUCAGON from PANCREATIC ALPHA CELLS; INSULIN from PANCREATIC BETA CELLS; and SOMATOSTATIN from the SOMATOSTATIN-SECRETING CELLS. Most are malignant except the insulin-producing tumors (INSULINOMA).
New abnormal growth of tissue. Malignant neoplasms show a greater degree of anaplasia and have the properties of invasion and metastasis, compared to benign neoplasms.
Neoplasms containing cyst-like formations or producing mucin or serum.
Tumors or cancer of the SKIN.
Two or more abnormal growths of tissue occurring simultaneously and presumed to be of separate origin. The neoplasms may be histologically the same or different, and may be found in the same or different sites.
Tumors or cancers of the KIDNEY.
Abnormal growths of tissue that follow a previous neoplasm but are not metastases of the latter. The second neoplasm may have the same or different histological type and can occur in the same or different organs as the previous neoplasm but in all cases arises from an independent oncogenic event. The development of the second neoplasm may or may not be related to the treatment for the previous neoplasm since genetic risk or predisposing factors may actually be the cause.
An adenocarcinoma producing mucin in significant amounts. (From Dorland, 27th ed)
Tumors or cancer of the THYROID GLAND.
Conditions which cause proliferation of hemopoietically active tissue or of tissue which has embryonic hemopoietic potential. They all involve dysregulation of multipotent MYELOID PROGENITOR CELLS, most often caused by a mutation in the JAK2 PROTEIN TYROSINE KINASE.
DNA present in neoplastic tissue.
Tumors or cancer of the LUNG.
Tumors or cancer of the PAROTID GLAND.
A benign neoplasm derived from glandular epithelium, in which cystic accumulations of retained secretions are formed. In some instances, considerable portions of the neoplasm, or even the entire mass, may be cystic. (Stedman, 25th ed)
Neoplasms developing from some structure of the connective and subcutaneous tissue. The concept does not refer to neoplasms located in connective or soft tissue.
Neoplasms associated with a proliferation of a single clone of PLASMA CELLS and characterized by the secretion of PARAPROTEINS.
Tumors or cancer of the APPENDIX.
Tumors or cancer of the LIVER.
A multilocular tumor with mucin secreting epithelium. They are most often found in the ovary, but are also found in the pancreas, appendix, and rarely, retroperitoneal and in the urinary bladder. They are considered to have low-grade malignant potential.
Tumors or cancer of the OVARY. These neoplasms can be benign or malignant. They are classified according to the tissue of origin, such as the surface EPITHELIUM, the stromal endocrine cells, and the totipotent GERM CELLS.
Tumors or cancer of the ENDOCRINE GLANDS.
Tumors or cancer of the GASTROINTESTINAL TRACT, from the MOUTH to the ANAL CANAL.
Carcinoma that arises from the PANCREATIC DUCTS. It accounts for the majority of cancers derived from the PANCREAS.
Experimentally induced new abnormal growth of TISSUES in animals to provide models for studying human neoplasms.
Neoplasms composed of vascular tissue. This concept does not refer to neoplasms located in blood vessels.
Tumors or cancer of the EYE.
Histochemical localization of immunoreactive substances using labeled antibodies as reagents.
Tumors or cancer of the NOSE.
Tumors or cancer of the SALIVARY GLANDS.
Tumors, cancer or other neoplasms produced by exposure to ionizing or non-ionizing radiation.
An adenocarcinoma containing finger-like processes of vascular connective tissue covered by neoplastic epithelium, projecting into cysts or the cavity of glands or follicles. It occurs most frequently in the ovary and thyroid gland. (Stedman, 25th ed)
A malignant neoplasm characterized by the formation of numerous, irregular, finger-like projections of fibrous stroma that is covered with a surface layer of neoplastic epithelial cells. (Stedman, 25th ed)
Tumors or cancer of the TESTIS. Germ cell tumors (GERMINOMA) of the testis constitute 95% of all testicular neoplasms.
Neoplasms composed of muscle tissue: skeletal, cardiac, or smooth. The concept does not refer to neoplasms located in muscles.
Neoplasms composed of glandular tissue, an aggregation of epithelial cells that elaborate secretions, and of any type of epithelium itself. The concept does not refer to neoplasms located in the various glands or in epithelial tissue.
A malignant cystic or semisolid tumor most often occurring in the ovary. Rarely, one is solid. This tumor may develop from a mucinous cystadenoma, or it may be malignant at the onset. The cysts are lined with tall columnar epithelial cells; in others, the epithelium consists of many layers of cells that have lost normal structure entirely. In the more undifferentiated tumors, one may see sheets and nests of tumor cells that have very little resemblance to the parent structure. (Hughes, Obstetric-Gynecologic Terminology, 1972, p184)
A benign epithelial tumor with a glandular organization.

Alterations of Rb pathway (Rb-p16INK4-cyclin D1) in preinvasive bronchial lesions. (1/530)

Lung cancer results from a stepwise accumulation of genetic and molecular abnormalities with unknown temporal relationships to precursor bronchial lesions. In a search for biomarkers of malignant progression, we analyzed the expression of the tumor suppressor gene Rb and of the proteins regulating its phosphorylation and function in G1 arrest, p16INK4A and cyclin D1, in preinvasive bronchial lesions accompanying cancer in 75 patients, in comparison with similar lesions in 22 patients with no cancer history. Rb was constantly expressed in preinvasive lesions, including carcinoma in situ (CIS). In contrast, p16 expression was lost in moderate dysplasia (12%) and in CIS (30%) in patients with lung cancer. p16 loss occurred exclusively in patients who displayed loss of p16 expression in their related invasive carcinoma. Loss of p16 expression was not seen in nine patients with dysplasia but no cancer progression. Cyclin D1 overexpression was seen in hyperplasia and metaplasia (6%), mild dysplasia (17%), moderate dysplasia (46%), and CIS (38%) in patients with cancer but was lost in 5% of the patients during the process of invasion; it was also observed in patients with no cancer progression (14%). Our results indicate that Rb protein function can be invalidated before invasion through alteration of the Rb phosphorylation pathway, by p16 inhibition, and/or by cyclin D1 overexpression and suggest a role for p16 and cyclin D1 deregulation in progression of preinvasive bronchial lesions to invasive carcinoma.  (+info)

Differential responses of normal, premalignant, and malignant human bronchial epithelial cells to receptor-selective retinoids. (2/530)

Using an in vitro lung carcinogenesis model consisting of normal, premalignant, and malignant human bronchial epithelial (HBE) cells, we analyzed the growth inhibitory effects of 26 novel synthetic retinoic acid receptor (RAR)- and retinoid X receptor (RXR)-selective retinoids. RAR-selective retinoids such as CD271, CD437, CD2325, and SR11364 showed potent activity in inhibiting the growth of either normal or premalignant and malignant HBE cells (IC50s mostly <1 microM) and were much more potent than RXR-selective retinoids. Nonetheless, the combination of RAR- and RXR-selective retinoids exhibited additive effects in HBE cells. As the HBE cells became progressively more malignant, they exhibited decreased or lost sensitivity to many retinoids. The activity of the RAR-selective retinoids, with the exception of the most potent retinoid, CD437, could be suppressed by an RAR panantagonist. These results suggest that: (a) RAR/RXR heterodimers play an important role in mediating the growth inhibitory effects of most retinoids in HBE cells; (b) CD437 may act through an RAR-independent pathway; (c) some of the RAR-selective retinoids may have the potential to be used in the clinic as chemopreventive and chemotherapeutic agents for lung cancer; and (d) early stages of lung carcinogenesis may be responsive targets for chemoprevention by retinoids, as opposed to later stages.  (+info)

Cyclin D1 proteolysis: a retinoid chemoprevention signal in normal, immortalized, and transformed human bronchial epithelial cells. (3/530)

BACKGROUND: Retinoids (derivatives of vitamin A) are reported to reduce the occurrence of some second primary cancers, including aerodigestive tract tumors. In contrast, beta-carotene does not reduce the occurrence of primary aerodigestive tract cancers. Mechanisms explaining these effective retinoid and ineffective carotenoid chemoprevention results are poorly defined. Recently, the all-trans-retinoic acid (RA)-induced proteolysis of cyclin D1 that leads to the arrest of cells in G1 phase of the cell cycle was described in human bronchial epithelial cells and is a promising candidate for such a mechanism. In this study, we have investigated this proteolysis as a common signal used by carotenoids or receptor-selective and receptor-nonselective retinoids. METHODS: We treated cultured normal human bronchial epithelial cells, immortalized human bronchial epithelial cells (BEAS-2B), and transformed human bronchial epithelial cells (BEAS-2BNNK) with receptor-selective or receptor-nonselective retinoids or with carotenoids and studied the effects on cell proliferation by means of tritiated thymidine incorporation and on cyclin D1 expression by means of immunoblot analysis. We also examined whether calpain inhibitor I, an inhibitor of the 26S proteasome degradation pathway, affected the decline (i.e., proteolysis) of cyclin D1. RESULTS: Receptor-nonselective retinoids were superior to the carotenoids studied in mediating the decline in cyclin D1 expression and in suppressing the growth of bronchial epithelial cells. Retinoids that activated retinoic acid receptor beta or retinoid X receptor pathways preferentially led to a decrease in the amount of cyclin D1 protein and a corresponding decline in growth. The retinoid-mediated degradation of cyclin D1 was blocked by cotreatment with calpain inhibitor I. CONCLUSIONS: Retinoid-dependent cyclin D1 proteolysis is a common chemoprevention signal in normal and neoplastic human bronchial epithelial cells. In contrast, carotenoids did not affect cyclin D1 expression. Thus, the degradation of cyclin D1 is a candidate intermediate marker for effective retinoid-mediated cancer chemoprevention in the aerodigestive tract.  (+info)

Survey of outpatient sputum cytology: influence of written instructions on sample quality and who benefits from investigation. (4/530)

OBJECTIVES: To evaluated quality of outpatient sputum cytology and whether written instructions to patients improve sample quality and to identify variables that predict satisfactory samples. DESIGN: Prospective randomised study. SETTING: Outpatient department of a district general hospital. PATIENTS: 224 patients recruited over 18 months whenever their clinicians requested sputum cytology, randomized to receive oral or oral and written advice. INTERVENTIONS: Oral advice from nurse on producing a sputum sample (114 patients); oral advice plus written instructions (110). MAIN MEASURES: Percentages of satisfactory sputum samples and of patients who produced more than one satisfactory sample; clinical or radiological features identified from subsequent review of patients' notes and radiographs associated with satisfactory samples; final diagnosis of bronchial cancer. RESULTS: 588 sputum samples were requested and 477 received. Patients in the group receiving additional written instructions produced 75(34%) satisfactory samples and 43(39%) of them one or more sets of satisfactory samples. Corresponding figures for the group receiving only oral advice (80(31%) and 46(40%) respectively)were not significantly different. Logistic regression showed that radiological evidence of collapse or consolidation (p<0.01) and hilar mass (p<0.05) were significant predictors of the production of satisfactory samples. Sputum cytology confirmed the diagnosis in only 9(17%) patients with bronchial carcinoma. CONCLUSIONS: The quality of outpatients' sputum samples was poor and was not improved by written instructions. Sputum cytology should be limited to patients with probable bronchial cancer unsuitable for surgery. IMPLICATIONS: Collection of samples and requests for sputum cytology should be reviewed in other hospitals.  (+info)

Sex-related differences in bronchial epithelial changes associated with tobacco smoking. (5/530)

BACKGROUND: Lung cancer is the most common cause of cancer death in North American women. Because smoking-related changes in the bronchial epithelium and in lung function have not been studied in detail in women, we used fluorescence bronchoscopy-directed biopsy to determine the prevalence of high-grade preinvasive lesions in former and current smokers of both sexes. METHODS: Spirometry, white-light bronchoscopy, and fluorescence bronchoscopy were performed in 189 women and 212 men older than 40 years of age who had smoked 20 pack-years or more (pack-years = number of packs of cigarettes smoked per day x number of years of smoking). RESULTS: Carcinoma in situ was found in 1.8% of the subjects, severe dysplasia was found in 6.5%, and moderate dysplasia was found in 14% (all preinvasive lesions). Compared with men, women had a lower prevalence of high-grade preinvasive lesions in the observed airways (14% versus 31%; odds ratio = 0.18; 95% confidence interval = 0.04-0.88), and women with preinvasive lesions had fewer such lesions (two-sided P = .048). The prevalence of preinvasive lesions did not change substantially for more than 10 years after cessation of smoking. Lung function was associated with the prevalence of preinvasive lesions, but the association was weaker in women than in men. If the presence of airflow obstruction was defined by an FEV1/FVC (forced expiratory volume in 1 second/forced vital capacity) value of 70% or less, only 56% of the men and 44% of the women with preinvasive lesions had abnormal lung function. CONCLUSION: In developing strategies for chemoprevention or early detection of lung cancer in high-risk populations, it is important to consider the effect of sex and arbitrarily chosen lung function values on the prevalence of preinvasive airway lesions.  (+info)

Malignant tumors of the liver and lungs in an area with a PVC industry. (6/530)

The incidence of malignant tumors of the lung and bronchus and of cytologically confirmed primary malignant tumor of the liver was analyzed for a 4-yr period in a city with several factories, including a PVC industry. Prior to the study two cases of angio-sarcoma of the liver were diagnosed in workers employed in PVC production. The total incidence of analyzed tumors was only slightly higher than predicted. The tumors of the liver recorded did not show any dependence on place of work or residence. During the period of observation, malignant tumors of the bronchus (lung) were not recorded in the PVC industry. Their rate in the area in which the PVC industry is situated was approximately the same as that for the entire city area. The study does not indicate that the occurrence of malignant tumors other than angiosarcoma is associated with exposure to vinyl chloride.  (+info)

Overexpression of cyclins D1 and E is frequent in bronchial preneoplasia and precedes squamous cell carcinoma development. (7/530)

Increased protein expression of the G1 cyclins D1 and E is reported in invasive non-small cell lung carcinoma. However, during transformation of the bronchial epithelium, overexpression of these species occurs, and their relationship to aberrant expression of p53 and retinoblastoma (Rb) has not been described previously. To determine the expression of these cell cycle regulators during the development of invasive squamous cell carcinoma (SCC) of the lung, the immunohistochemical expression patterns in normal bronchial epithelium (n = 36), squamous metaplasia (SM; n = 28), and epithelial atypia (n = 34) were compared with that in low-grade dysplasia (LGD; n = 17), high-grade bronchial dysplasia (HGD; n = 30), and SCC (n = 36). Monoclonal anti-p53 Pab1801, polyclonal anti-cyclin D1 DCS6, monoclonal anti-cyclin E HE12, and monoclonal anti-Rb OP-66 antibodies were used. Cyclin D1 was not expressed in normal bronchial epithelium but was detected in 7% of SMs, 15% of atypias; 18% of LGDs, 47% of HGDs, and 42% of SCCs. Cyclin E was not detected in normal epithelium (n = 24), SM (n = 16), or LGD (n = 12), but it was found in 9% of atypias (2 of 22), 33% of HGDs (7 of 21), and 54% of SCCs (13 of 24). p53 was not expressed in normal epithelium, SM, and LGD, but it was overexpressed in 6% of atypias, 53% of HGDs, and 61% of SCCs. Abnormal Rb expression was found only in 2 of 36 cases of SCC. A total of 91% of HGDs and 92% of SCCs exhibited overexpression of at least one of the p53, cyclin D1, or cyclin E species. However, no link was observed between overexpression of p53 and the overexpressed G1 cyclins in preneoplastic lesions. Overexpression of cyclin D1, cyclin E, and p53 occurs frequently and independently in pulmonary SCC and is detected in lesions before the development of invasive carcinoma. In contrast, altered Rb expression is a late and infrequent event in squamous cell carcinogenesis.  (+info)

Bronchial capillary hemangioma in adults. (8/530)

Two cases with capillary hemangioma of the trachea and the left upper lobe bronchus are presented. The adult patients were referred to the hospital because of hemoptysis and cough. The chest radiographs were normal in both cases. The bronchoscopic examination revealed circumscribed lesions with a capillarized surface protruding into the lumen of the trachea and the left upper lobe bronchus, respectively. The lesions were excised in toto with flexible bronchoscopic forceps. The specimens contained typical capillary hemangiomas without any signs of malignancy. Capillary hemangioma in the bronchial tree is an extremely rare benign lesion in adults. Nevertheless, it should be considered as a possible cause of hemoptysis and cough.  (+info)

Types of Bronchial Neoplasms:

1. Adenocarcinoma: This is the most common type of lung cancer and accounts for approximately 40% of all lung cancers. It originates in the glandular cells that line the bronchi.
2. Squamous Cell Carcinoma: This type of lung cancer originates in the squamous cells that line the bronchi. It is the second most common type of lung cancer, accounting for approximately 25% of all lung cancers.
3. Small Cell Lung Cancer (SCLC): This type of lung cancer is highly aggressive and accounts for approximately 10% of all lung cancers. It originates in the small cells that line the bronchi.
4. Large Cell Carcinoma: This type of lung cancer is rare and accounts for approximately 5% of all lung cancers. It originates in the large cells that line the bronchi.
5. Bronchioloalveolar Carcinoma (BAC): This type of lung cancer originates in the small air sacs (alveoli) and is rare, accounting for approximately 2% of all lung cancers.
6. Lymphoma: This type of cancer originates in the immune system cells that line the bronchi. It is rare, accounting for approximately 1% of all lung cancers.
7. Carcinoid Tumors: These are rare types of lung cancer that originate in the neuroendocrine cells that line the bronchi. They are typically slow-growing and less aggressive than other types of lung cancer.
8. Secondary Cancers: These are cancers that have spread to the lungs from other parts of the body, such as breast cancer or colon cancer.

Diagnosis of Bronchial Neoplasms:

1. Medical History and Physical Examination: A thorough medical history and physical examination are essential for diagnosing bronchial neoplasms. The doctor will ask questions about the patient's symptoms, risk factors, and medical history.
2. Chest X-Ray: A chest X-ray is often the first diagnostic test performed to evaluate the lungs for any abnormalities.
3. Computed Tomography (CT) Scan: A CT scan is a more detailed imaging test that uses X-rays and computer technology to produce cross-sectional images of the lungs. It can help identify the size, location, and extent of the tumor.
4. Positron Emission Tomography (PET) Scan: A PET scan is a diagnostic test that uses small amounts of radioactive material to visualize the metabolic activity of the cells in the lungs. It can help identify the presence of cancerous cells and determine the effectiveness of treatment.
5. Biopsy: A biopsy involves taking a sample of tissue from the lung and examining it under a microscope for cancerous cells. It is a definitive diagnostic test for bronchial neoplasms.
6. Bronchoscopy: Bronchoscopy is a procedure in which a thin, flexible tube with a camera on the end is inserted through the nose or mouth and guided to the lungs. It can help identify any abnormalities in the airways and obtain a biopsy sample.
7. Magnetic Resonance Imaging (MRI): An MRI uses magnetic fields and radio waves to produce detailed images of the lungs and surrounding tissues. It is not as commonly used for diagnosing bronchial neoplasms as other imaging tests, but it may be recommended in certain cases.
8. Ultrasound: An ultrasound uses high-frequency sound waves to produce images of the lungs and surrounding tissues. It is not typically used as a diagnostic test for bronchial neoplasms, but it may be used to evaluate the spread of cancer to other parts of the body.

It's important to note that the specific diagnostic tests and procedures used will depend on the individual case and the suspicion of malignancy. Your doctor will discuss the best course of action with you based on your symptoms, medical history, and test results.

Pancreatic adenocarcinoma is the most common type of malignant pancreatic neoplasm and accounts for approximately 85% of all pancreatic cancers. It originates in the glandular tissue of the pancreas and has a poor prognosis, with a five-year survival rate of less than 10%.

Pancreatic neuroendocrine tumors (PNETs) are less common but more treatable than pancreatic adenocarcinoma. These tumors originate in the hormone-producing cells of the pancreas and can produce excess hormones that cause a variety of symptoms, such as diabetes or high blood sugar. PNETs are classified into two main types: functional and non-functional. Functional PNETs produce excess hormones and are more aggressive than non-functional tumors.

Other rare types of pancreatic neoplasms include acinar cell carcinoma, ampullary cancer, and oncocytic pancreatic neuroendocrine tumors. These tumors are less common than pancreatic adenocarcinoma and PNETs but can be equally aggressive and difficult to treat.

The symptoms of pancreatic neoplasms vary depending on the type and location of the tumor, but they often include abdominal pain, weight loss, jaundice, and fatigue. Diagnosis is typically made through a combination of imaging tests such as CT scans, endoscopic ultrasound, and biopsy. Treatment options for pancreatic neoplasms depend on the type and stage of the tumor but may include surgery, chemotherapy, radiation therapy, or a combination of these.

Prognosis for patients with pancreatic neoplasms is generally poor, especially for those with advanced stages of disease. However, early detection and treatment can improve survival rates. Research into the causes and mechanisms of pancreatic neoplasms is ongoing, with a focus on developing new and more effective treatments for these devastating diseases.




Neoplasm refers to an abnormal growth of cells that can be benign (non-cancerous) or malignant (cancerous). Neoplasms can occur in any part of the body and can affect various organs and tissues. The term "neoplasm" is often used interchangeably with "tumor," but while all tumors are neoplasms, not all neoplasms are tumors.

Types of Neoplasms

There are many different types of neoplasms, including:

1. Carcinomas: These are malignant tumors that arise in the epithelial cells lining organs and glands. Examples include breast cancer, lung cancer, and colon cancer.
2. Sarcomas: These are malignant tumors that arise in connective tissue, such as bone, cartilage, and fat. Examples include osteosarcoma (bone cancer) and soft tissue sarcoma.
3. Lymphomas: These are cancers of the immune system, specifically affecting the lymph nodes and other lymphoid tissues. Examples include Hodgkin lymphoma and non-Hodgkin lymphoma.
4. Leukemias: These are cancers of the blood and bone marrow that affect the white blood cells. Examples include acute myeloid leukemia (AML) and chronic lymphocytic leukemia (CLL).
5. Melanomas: These are malignant tumors that arise in the pigment-producing cells called melanocytes. Examples include skin melanoma and eye melanoma.

Causes and Risk Factors of Neoplasms

The exact causes of neoplasms are not fully understood, but there are several known risk factors that can increase the likelihood of developing a neoplasm. These include:

1. Genetic predisposition: Some people may be born with genetic mutations that increase their risk of developing certain types of neoplasms.
2. Environmental factors: Exposure to certain environmental toxins, such as radiation and certain chemicals, can increase the risk of developing a neoplasm.
3. Infection: Some neoplasms are caused by viruses or bacteria. For example, human papillomavirus (HPV) is a common cause of cervical cancer.
4. Lifestyle factors: Factors such as smoking, excessive alcohol consumption, and a poor diet can increase the risk of developing certain types of neoplasms.
5. Family history: A person's risk of developing a neoplasm may be higher if they have a family history of the condition.

Signs and Symptoms of Neoplasms

The signs and symptoms of neoplasms can vary depending on the type of cancer and where it is located in the body. Some common signs and symptoms include:

1. Unusual lumps or swelling
2. Pain
3. Fatigue
4. Weight loss
5. Change in bowel or bladder habits
6. Unexplained bleeding
7. Coughing up blood
8. Hoarseness or a persistent cough
9. Changes in appetite or digestion
10. Skin changes, such as a new mole or a change in the size or color of an existing mole.

Diagnosis and Treatment of Neoplasms

The diagnosis of a neoplasm usually involves a combination of physical examination, imaging tests (such as X-rays, CT scans, or MRI scans), and biopsy. A biopsy involves removing a small sample of tissue from the suspected tumor and examining it under a microscope for cancer cells.

The treatment of neoplasms depends on the type, size, location, and stage of the cancer, as well as the patient's overall health. Some common treatments include:

1. Surgery: Removing the tumor and surrounding tissue can be an effective way to treat many types of cancer.
2. Chemotherapy: Using drugs to kill cancer cells can be effective for some types of cancer, especially if the cancer has spread to other parts of the body.
3. Radiation therapy: Using high-energy radiation to kill cancer cells can be effective for some types of cancer, especially if the cancer is located in a specific area of the body.
4. Immunotherapy: Boosting the body's immune system to fight cancer can be an effective treatment for some types of cancer.
5. Targeted therapy: Using drugs or other substances to target specific molecules on cancer cells can be an effective treatment for some types of cancer.

Prevention of Neoplasms

While it is not always possible to prevent neoplasms, there are several steps that can reduce the risk of developing cancer. These include:

1. Avoiding exposure to known carcinogens (such as tobacco smoke and radiation)
2. Maintaining a healthy diet and lifestyle
3. Getting regular exercise
4. Not smoking or using tobacco products
5. Limiting alcohol consumption
6. Getting vaccinated against certain viruses that are associated with cancer (such as human papillomavirus, or HPV)
7. Participating in screening programs for early detection of cancer (such as mammograms for breast cancer and colonoscopies for colon cancer)
8. Avoiding excessive exposure to sunlight and using protective measures such as sunscreen and hats to prevent skin cancer.

It's important to note that not all cancers can be prevented, and some may be caused by factors that are not yet understood or cannot be controlled. However, by taking these steps, individuals can reduce their risk of developing cancer and improve their overall health and well-being.

Cystic neoplasms are fluid-filled sacs that grow in the body. They can be benign or malignant and can arise from a variety of tissues, including the ovaries, pancreas, and lungs. Mucinous neoplasms are tumors that produce mucin, a type of protein found in mucus. These tumors can occur in the breast, ovary, or colon, and are often benign.

Serous neoplasms are tumors that arise from the serous membranes, which are the thin layers of tissue that line the cavities of the body. Examples of serous neoplasms include ovarian cancer and mesothelioma. These tumors can be benign or malignant.

In summary, neoplasms, cystic, mucinous, and serous are different types of tumors that can occur in various organs and tissues throughout the body. While they can be benign, many of these tumors are malignant and can spread to other parts of the body if left untreated.

There are several types of skin neoplasms, including:

1. Basal cell carcinoma (BCC): This is the most common type of skin cancer, and it usually appears as a small, fleshy bump or a flat, scaly patch. BCC is highly treatable, but if left untreated, it can grow and invade surrounding tissue.
2. Squamous cell carcinoma (SCC): This type of skin cancer is less common than BCC but more aggressive. It typically appears as a firm, flat, or raised bump on sun-exposed areas. SCC can spread to other parts of the body if left untreated.
3. Melanoma: This is the most serious type of skin cancer, accounting for only 1% of all skin neoplasms but responsible for the majority of skin cancer deaths. Melanoma can appear as a new or changing mole, and it's essential to recognize the ABCDE signs (Asymmetry, Border irregularity, Color variation, Diameter >6mm, Evolving size, shape, or color) to detect it early.
4. Sebaceous gland carcinoma: This rare type of skin cancer originates in the oil-producing glands of the skin and can appear as a firm, painless nodule on the forehead, nose, or other oily areas.
5. Merkel cell carcinoma: This is a rare and aggressive skin cancer that typically appears as a firm, shiny bump on the skin. It's more common in older adults and those with a history of sun exposure.
6. Cutaneous lymphoma: This type of cancer affects the immune system and can appear as a rash, nodules, or tumors on the skin.
7. Kaposi sarcoma: This is a rare type of skin cancer that affects people with weakened immune systems, such as those with HIV/AIDS. It typically appears as a flat, red or purple lesion on the skin.

While skin cancers are generally curable when detected early, it's important to be aware of your skin and notice any changes or unusual spots, especially if you have a history of sun exposure or other risk factors. If you suspect anything suspicious, see a dermatologist for an evaluation and potential biopsy. Remember, prevention is key to avoiding the harmful effects of UV radiation and reducing your risk of developing skin cancer.

Multiple primary neoplasms can arise in different organs or tissues throughout the body, such as the breast, colon, prostate, lung, or skin. Each tumor is considered a separate entity, with its own unique characteristics, including size, location, and aggressiveness. Treatment for multiple primary neoplasms typically involves surgery, chemotherapy, radiation therapy, or a combination of these modalities.

The diagnosis of multiple primary neoplasms can be challenging due to the overlapping symptoms and radiological findings between the different tumors. Therefore, it is essential to have a thorough clinical evaluation and diagnostic workup to rule out other possible causes of the symptoms and confirm the presence of multiple primary neoplasms.

Multiple primary neoplasms are more common than previously thought, with an estimated prevalence of 2% to 5% in some populations. The prognosis for patients with multiple primary neoplasms varies depending on the location, size, and aggressiveness of each tumor, as well as the patient's overall health status.

It is important to note that multiple primary neoplasms are not the same as metastatic cancer, in which a single primary tumor spreads to other parts of the body. Multiple primary neoplasms are distinct tumors that arise independently from different primary sites within the body.

Symptoms of Kidney Neoplasms can include blood in the urine, pain in the flank or abdomen, weight loss, fever, and fatigue. Diagnosis is made through a combination of physical examination, imaging studies such as CT scans or ultrasound, and tissue biopsy. Treatment options vary depending on the type and stage of the neoplasm, but may include surgery, ablation therapy, targeted therapy, or chemotherapy.

It is important for individuals with a history of Kidney Neoplasms to follow up with their healthcare provider regularly for monitoring and check-ups to ensure early detection of any recurrences or new tumors.

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Examples of 'Adenocarcinoma, Mucinous' in medical literature:

* The patient was diagnosed with adenocarcinoma, mucinous type, in their colon after undergoing a colonoscopy and biopsy. (From the Journal of Clinical Oncology)

* The patient had a history of adenocarcinoma, mucinous type, in their breast and was being monitored for potential recurrence. (From the Journal of Surgical Oncology)

* The tumor was found to be an adenocarcinoma, mucinous type, with a high grade and was treated with surgery and chemotherapy. (From the Journal of Gastrointestinal Oncology)

Synonyms for 'Adenocarcinoma, Mucinous' include:

* Mucinous adenocarcinoma
* Colon adenocarcinoma, mucinous type
* Rectal adenocarcinoma, mucinous type
* Adenocarcinoma of the colon and rectum, mucinous type.

There are several types of thyroid neoplasms, including:

1. Thyroid nodules: These are abnormal growths or lumps that can develop in the thyroid gland. Most thyroid nodules are benign (non-cancerous), but some can be malignant (cancerous).
2. Thyroid cancer: This is a type of cancer that develops in the thyroid gland. There are several types of thyroid cancer, including papillary, follicular, and medullary thyroid cancer.
3. Thyroid adenomas: These are benign tumors that develop in the thyroid gland. They are usually non-cancerous and do not spread to other parts of the body.
4. Thyroid cysts: These are fluid-filled sacs that can develop in the thyroid gland. They are usually benign and do not cause any symptoms.

Thyroid neoplasms can be caused by a variety of factors, including genetic mutations, exposure to radiation, and certain medical conditions, such as thyroiditis (inflammation of the thyroid gland).

Symptoms of thyroid neoplasms can include:

* A lump or swelling in the neck
* Pain in the neck or throat
* Difficulty swallowing or breathing
* Hoarseness or voice changes
* Weight loss or fatigue

Diagnosis of thyroid neoplasms usually involves a combination of physical examination, imaging tests (such as ultrasound or CT scans), and biopsies. Treatment depends on the type and severity of the neoplasm, and can include surgery, radiation therapy, and medications.

There are several types of MPDs, including:

1. Polycythemia vera (PV): This is a rare disorder characterized by an overproduction of red blood cells, white blood cells, and platelets.
2. Essential thrombocythemia (ET): This is a rare disorder characterized by an overproduction of platelets.
3. Primary myelofibrosis (PMF): This is a rare and severe disorder characterized by the accumulation of scar tissue in the bone marrow, leading to an overproduction of immature white blood cells.
4. Chronic myelogenous leukemia (CML): This is a type of cancer that affects the bone marrow and blood cells, characterized by the overproduction of immature white blood cells.

The symptoms of MPDs can vary depending on the specific disorder, but may include:

* Fatigue
* Weakness
* Shortness of breath
* Headaches
* Dizziness
* Pale skin
* Easy bruising or bleeding
* Swollen spleen
* Bone pain

The exact cause of MPDs is not known, but they are thought to be due to genetic mutations that occur in the bone marrow cells. Treatment options for MPDs include:

* Chemotherapy: This is a type of drug that kills cancer cells.
* Radiation therapy: This is a type of treatment that uses high-energy X-rays to kill cancer cells.
* Stem cell transplantation: This is a procedure in which healthy stem cells are transplanted into the body to replace damaged or diseased bone marrow cells.

Overall, MPDs are rare and complex disorders that can have a significant impact on quality of life. While there is no cure for these conditions, treatment options are available to help manage symptoms and improve outcomes.

There are several types of lung neoplasms, including:

1. Adenocarcinoma: This is the most common type of lung cancer, accounting for approximately 40% of all lung cancers. It is a malignant tumor that originates in the glands of the respiratory tract and can be found in any part of the lung.
2. Squamous cell carcinoma: This type of lung cancer accounts for approximately 25% of all lung cancers and is more common in men than women. It is a malignant tumor that originates in the squamous cells lining the airways of the lungs.
3. Small cell lung cancer (SCLC): This is a highly aggressive form of lung cancer that accounts for approximately 15% of all lung cancers. It is often found in the central parts of the lungs and can spread quickly to other parts of the body.
4. Large cell carcinoma: This is a rare type of lung cancer that accounts for only about 5% of all lung cancers. It is a malignant tumor that originates in the large cells of the respiratory tract and can be found in any part of the lung.
5. Bronchioalveolar carcinoma (BAC): This is a rare type of lung cancer that originates in the cells lining the airways and alveoli of the lungs. It is more common in women than men and tends to affect older individuals.
6. Lymphangioleiomyomatosis (LAM): This is a rare, progressive, and often fatal lung disease that primarily affects women of childbearing age. It is characterized by the growth of smooth muscle-like cells in the lungs and can lead to cysts, lung collapse, and respiratory failure.
7. Hamartoma: This is a benign tumor that originates in the tissue of the lungs and is usually found in children. It is characterized by an overgrowth of normal lung tissue and can be treated with surgery.
8. Secondary lung cancer: This type of cancer occurs when cancer cells from another part of the body spread to the lungs through the bloodstream or lymphatic system. It is more common in people who have a history of smoking or exposure to other carcinogens.
9. Metastatic cancer: This type of cancer occurs when cancer cells from another part of the body spread to the lungs through the bloodstream or lymphatic system. It is more common in people who have a history of smoking or exposure to other carcinogens.
10. Mesothelioma: This is a rare and aggressive form of cancer that originates in the lining of the lungs or abdomen. It is caused by asbestos exposure and can be treated with surgery, chemotherapy, and radiation therapy.

Lung diseases can also be classified based on their cause, such as:

1. Infectious diseases: These are caused by bacteria, viruses, or other microorganisms and can include pneumonia, tuberculosis, and bronchitis.
2. Autoimmune diseases: These are caused by an overactive immune system and can include conditions such as sarcoidosis and idiopathic pulmonary fibrosis.
3. Genetic diseases: These are caused by inherited mutations in genes that affect the lungs and can include cystic fibrosis and primary ciliary dyskinesia.
4. Environmental diseases: These are caused by exposure to harmful substances such as tobacco smoke, air pollution, and asbestos.
5. Radiological diseases: These are caused by exposure to ionizing radiation and can include conditions such as radiographic breast cancer and lung cancer.
6. Vascular diseases: These are caused by problems with the blood vessels in the lungs and can include conditions such as pulmonary embolism and pulmonary hypertension.
7. Tumors: These can be benign or malignant and can include conditions such as lung metastases and lung cancer.
8. Trauma: This can include injuries to the chest or lungs caused by accidents or other forms of trauma.
9. Congenital diseases: These are present at birth and can include conditions such as bronchopulmonary foregut malformations and congenital cystic adenomatoid malformation.

Each type of lung disease has its own set of symptoms, diagnosis, and treatment options. It is important to seek medical attention if you experience any persistent or severe respiratory symptoms, as early diagnosis and treatment can improve outcomes and quality of life.

Benign parotid neoplasms include:

* Pleomorphic adenoma: This is the most common type of benign parotid tumor, accounting for about 70% of all benign parotid neoplasms. It is a slow-growing tumor that usually affects people between the ages of 20 and 50.
* Warthin's tumor: This is a rare type of benign parotid tumor that usually occurs in older adults. It is a slow-growing tumor that often causes few symptoms.
* Other benign tumors: These include papillary cystadenoma, oncocytoma, and adenomyoepithelioma.

Malignant parotid neoplasms include:

* Parotid duct carcinoma: This is a rare type of cancer that arises in the main duct of the parotid gland. It usually affects older adults and can be aggressive, meaning it grows quickly and spreads to other parts of the body.
* Adenoid cystic carcinoma: This is a malignant tumor that typically affects the salivary glands, including the parotid gland. It is a slow-growing tumor that can infiltrate surrounding tissues and bone, making it difficult to treat.
* Other malignant tumors: These include acinic cell carcinoma, adenocarcinoma, and squamous cell carcinoma.

The symptoms of parotid neoplasms can vary depending on the size and location of the tumor. Common symptoms include:

* A lump or swelling in the neck or face
* Painless mass or lump in the affected gland
* Difficulty swallowing or eating
* Numbness or weakness in the face
* Pain in the ear, jaw, or neck
* Fatigue
* Weight loss

If you experience any of these symptoms, it is important to see a doctor for proper evaluation and diagnosis. A doctor may perform a physical examination, take a medical history, and order imaging tests such as CT scans, MRI scans, or ultrasound to determine the presence of a parotid neoplasm.

Treatment options for parotid neoplasms depend on the type and stage of the tumor. Surgery is usually the first line of treatment, and may involve removing the affected gland or a portion of the gland. Radiation therapy and chemotherapy may also be used to treat more aggressive tumors or those that have spread to other parts of the body.

Overall, while parotid neoplasms can be serious and potentially life-threatening, early detection and treatment can improve outcomes and help preserve facial function and appearance. It is important to seek medical attention if you experience any symptoms that may indicate a parotid neoplasm.

Note: The above definition is intended to provide a general understanding of the term 'Cystadenoma' and should not be considered as medical advice or diagnosis. If you have any concerns about your health, please consult a qualified medical professional for proper evaluation and care.

Some common examples of neoplasms, connective and soft tissue include:

1. Soft tissue sarcomas: These are malignant tumors that develop in the soft tissues of the body, such as muscles, tendons, and ligaments.
2. Connective tissue tumors: These are benign or malignant growths that develop in the connective tissues of the body, such as cartilage, bone, and fat.
3. Lipomas: These are benign tumors that develop in the soft tissues of the body, made up of fat cells.
4. Hemangiomas: These are benign tumors that develop in the blood vessels, often seen in infants and children.
5. Fibromas: These are benign tumors that develop in the connective tissue, often seen in the skin and subcutaneous tissues.

The symptoms of neoplasms, connective and soft tissue can vary depending on the location and size of the tumor, but may include pain, swelling, redness, and limited mobility. Treatment options can range from surgical removal to radiation therapy and chemotherapy, and depend on the severity and location of the tumor.

In summary, neoplasms, connective and soft tissue are abnormal growths that develop in the connective and soft tissues of the body, which can be benign or malignant, and may cause symptoms such as pain, swelling, and limited mobility.

Neoplasms, plasma cell, are tumors that arise from plasma cells and can be either benign or malignant. They are relatively rare and tend to affect older adults.

Symptoms of neoplasms, plasma cell, include bone pain, tiredness, fever, and weight loss. Treatment options vary depending on the type and stage of the tumor but may include chemotherapy, radiation therapy, or surgery.

Neoplasms, plasma cells are classified as either extramedullary (outside the bone marrow) or intramedullary (within the bone marrow). The most common type of plasma cell neoplasm is multiple myeloma, which is a malignant tumor that affects the bone marrow and can cause bone pain, infections, and other complications.

Appendiceal neoplasms refer to abnormal growths or tumors that occur in the appendix, a small tube-like structure attached to the large intestine. These growths can be benign (non-cancerous) or malignant (cancerous). Malignant appendiceal neoplasms are rare, but they can spread quickly to other parts of the body if left untreated.

Types of Appendiceal Neoplasms:

There are several types of appendiceal neoplasms, including:

1. Adenoma: A benign tumor that arises from glandular cells in the appendix.
2. Carcinoma: A malignant tumor that arises from epithelial cells in the appendix.
3. Mucinous cystadenoma: A benign tumor that arises from glandular cells in the appendix and typically contains mucin, a type of protein.
4. Goblet cell carcinoid: A rare type of malignant tumor that arises from goblet cells, which are specialized cells that produce mucin in the appendix.
5. Signet ring cell carcinoma: A rare and aggressive type of malignant tumor that arises from glandular cells in the appendix.

Symptoms and Diagnosis:

The symptoms of appendiceal neoplasms can vary depending on the size and location of the tumor, but may include abdominal pain, nausea, vomiting, fever, and loss of appetite. Diagnosis is typically made through a combination of physical examination, imaging tests such as CT scans or MRI, and biopsy.

Treatment:

Treatment for appendiceal neoplasms usually involves surgical removal of the affected appendix, which may involve a laparoscopic or open procedure. In some cases, chemotherapy or radiation therapy may also be recommended to destroy any remaining cancer cells. The prognosis for patients with appendiceal neoplasms depends on the type and stage of the tumor at the time of diagnosis.

Prognosis:

The prognosis for patients with appendiceal neoplasms is generally good if the tumor is detected early and treated appropriately. However, if the tumor is not diagnosed until a later stage, the prognosis may be poorer. The 5-year survival rate for patients with appendiceal cancer is approximately 70-80%.

Conclusion:

Appendiceal neoplasms are rare and aggressive tumors that can arise in the appendix. Early diagnosis and treatment are critical for improving outcomes. Imaging tests such as CT scans and MRI can help identify these tumors, and surgical removal of the affected appendix is usually the first line of treatment. Chemotherapy or radiation therapy may also be recommended in some cases. The prognosis for patients with appendiceal neoplasms is generally good if the tumor is detected early, but can be poorer if not diagnosed until a later stage.

Liver neoplasms, also known as liver tumors or hepatic tumors, are abnormal growths of tissue in the liver. These growths can be benign (non-cancerous) or malignant (cancerous). Malignant liver tumors can be primary, meaning they originate in the liver, or metastatic, meaning they spread to the liver from another part of the body.

There are several types of liver neoplasms, including:

1. Hepatocellular carcinoma (HCC): This is the most common type of primary liver cancer and arises from the main cells of the liver (hepatocytes). HCC is often associated with cirrhosis and can be caused by viral hepatitis or alcohol abuse.
2. Cholangiocarcinoma: This type of cancer arises from the cells lining the bile ducts within the liver (cholangiocytes). Cholangiocarcinoma is rare and often diagnosed at an advanced stage.
3. Hemangiosarcoma: This is a rare type of cancer that originates in the blood vessels of the liver. It is most commonly seen in dogs but can also occur in humans.
4. Fibromas: These are benign tumors that arise from the connective tissue of the liver (fibrocytes). Fibromas are usually small and do not spread to other parts of the body.
5. Adenomas: These are benign tumors that arise from the glandular cells of the liver (hepatocytes). Adenomas are usually small and do not spread to other parts of the body.

The symptoms of liver neoplasms vary depending on their size, location, and whether they are benign or malignant. Common symptoms include abdominal pain, fatigue, weight loss, and jaundice (yellowing of the skin and eyes). Diagnosis is typically made through a combination of imaging tests such as CT scans, MRI scans, and ultrasound, and a biopsy to confirm the presence of cancer cells.

Treatment options for liver neoplasms depend on the type, size, location, and stage of the tumor, as well as the patient's overall health. Surgery may be an option for some patients with small, localized tumors, while others may require chemotherapy or radiation therapy to shrink the tumor before surgery can be performed. In some cases, liver transplantation may be necessary.

Prognosis for liver neoplasms varies depending on the type and stage of the cancer. In general, early detection and treatment improve the prognosis, while advanced-stage disease is associated with a poorer prognosis.

Characteristics:

* Mucinous cystadenomas are typically slow-growing and asymptomatic, but can occasionally cause pelvic pain or discomfort due to their size.
* They are usually unilateral (affecting one ovary), but can rarely occur bilaterally (affecting both ovaries).
* The tumor is composed of mucin-secreting epithelial cells that form glands or cysts within a fibrous stroma.
* Cystadenomas are typically encapsulated, but can rarely become invasive and infiltrate surrounding tissues.
* Mucinous cystadenomas are usually small (less than 5 cm in diameter), but can occasionally be larger.

Diagnosis:

* Imaging studies such as ultrasound or computed tomography (CT) scans may be used to detect the presence of a cystic mass in the ovary, but a definitive diagnosis is usually made through surgical exploration and histopathologic examination of the tumor tissue.
* A preoperative diagnosis of mucinous cystadenoma can be challenging, as the imaging features are not specific and may resemble other ovarian tumors, such as serous cystadenomas or borderline tumors.

Treatment:

* Surgical excision is the primary treatment for mucinous cystadenoma, and the procedure is usually performed through a laparotomy or laparoscopy.
* The surgical approach depends on the size and location of the tumor, as well as the patient's age and fertility status.
* In some cases, the tumor may be removed through a staged approach, with initial cytoreduction followed by chemotherapy or radiation therapy to shrink the remaining tumor burden.

Prognosis:

* Mucinous cystadenoma is generally considered a benign tumor, and the prognosis is excellent for most patients.
* The overall survival rate is high, and the majority of patients can expect to be cured with surgical excision alone.
* However, in rare cases, mucinous cystadenoma can recur or progress to more aggressive types of ovarian cancer, such as serous carcinoma.

Follow-up:

* After surgical excision, patients with mucinous cystadenoma should be followed up with regular pelvic examinations, imaging studies, and serum CA 125 levels to monitor for any signs of recurrence or progression.
* The frequency of follow-up appointments may vary depending on the patient's age, tumor size, and other factors, but annual pelvic examinations and imaging studies are generally recommended for at least 5 years after surgery.

References:

1. Kurman RJ, et al. The origin and pathology of ovarian borderline tumors. International Journal of Gynecological Pathology. 2014;33(2):197-211.
2. Di Cerbo A, et al. Mucinous cystadenoma of the ovary: a review of the literature. Journal of Obstetrics and Gynaecology Canada. 2018;40(6):753-763.
3. Chung H, et al. The clinicopathological features and prognosis of mucinous cystadenoma of the ovary: a systematic review and meta-analysis. Gynecologic Oncology Reports. 2018;20:135-143.

Benign ovarian neoplasms include:

1. Serous cystadenoma: A fluid-filled sac that develops on the surface of the ovary.
2. Mucinous cystadenoma: A tumor that is filled with mucin, a type of protein.
3. Endometrioid tumors: Tumors that are similar to endometrial tissue (the lining of the uterus).
4. Theca cell tumors: Tumors that develop in the supportive tissue of the ovary called theca cells.

Malignant ovarian neoplasms include:

1. Epithelial ovarian cancer (EOC): The most common type of ovarian cancer, which arises from the surface epithelium of the ovary.
2. Germ cell tumors: Tumors that develop from germ cells, which are the cells that give rise to eggs.
3. Stromal sarcomas: Tumors that develop in the supportive tissue of the ovary.

Ovarian neoplasms can cause symptoms such as pelvic pain, abnormal bleeding, and abdominal swelling. They can also be detected through pelvic examination, imaging tests such as ultrasound and CT scan, and biopsy. Treatment options for ovarian neoplasms depend on the type, stage, and location of the tumor, and may include surgery, chemotherapy, and radiation therapy.

Types of Endocrine Gland Neoplasms:

1. Thyroid Cancer: A malignant tumor that develops in the thyroid gland, which can cause an overproduction or underproduction of thyroid hormones.
2. Adrenal Cancer: A malignant tumor that develops in the adrenal glands, which can produce excess hormones that can cause various symptoms.
3. Pancreatic Neuroendocrine Tumors (PNETs): Tumors that develop in the pancreas and produce excess hormones that can cause a variety of symptoms.
4. Parathyroid Cancer: A malignant tumor that develops in the parathyroid glands, which regulate calcium levels in the blood.
5. Pituitary Tumors: Benign or malignant growths that develop in the pituitary gland, which can affect hormone production and cause various symptoms.

Causes and Risk Factors:

1. Genetic mutations
2. Exposure to certain chemicals or radiation
3. Family history of endocrine disorders
4. Previous radiation therapy
5. Age, with most cases occurring in people over the age of 40

Symptoms:

1. Thyroid cancer: A lump in the neck, difficulty swallowing, or shortness of breath
2. Adrenal cancer: High blood pressure, weight gain, or muscle weakness
3. PNETs: Diarrhea, abdominal pain, or weight loss
4. Parathyroid cancer: High calcium levels in the blood, kidney stones, or osteoporosis
5. Pituitary tumors: Headaches, vision changes, or hormonal imbalances

Treatment options for endocrine cancers depend on the specific type of cancer, its location, and its stage. Treatment may include surgery, radiation therapy, chemotherapy, or a combination of these. In some cases, hormone replacement therapy may also be necessary.

Prognosis:
The prognosis for endocrine cancers varies by type. In general, the earlier the cancer is diagnosed and treated, the better the prognosis. Thyroid cancer has a good prognosis, with a 5-year survival rate of around 97%. Adrenal cancer has a lower survival rate of around 60%, while PNETs have a poorer prognosis, with a 5-year survival rate of around 30%. Parathyroid cancer and pituitary tumors have better prognoses, with 5-year survival rates of around 90% and 80%, respectively.

Prevention:
There is no guaranteed way to prevent endocrine cancers, but certain measures may help reduce the risk. These include:

* Reducing exposure to radiation: Minimizing exposure to radiation, such as from CT scans, can help reduce the risk of developing thyroid cancer.
* Avoiding certain chemicals: Avoiding certain chemicals, such as pesticides and herbicides, may help reduce the risk of developing endocrine cancers.
* Maintaining a healthy lifestyle: Maintaining a healthy lifestyle, including eating a balanced diet and exercising regularly, may help reduce the risk of developing endocrine cancers.
* Early detection: Early detection and treatment of endocrine cancers can improve prognosis. Regular check-ups with an endocrinologist can help identify any abnormalities early on.

In conclusion, endocrine cancers are a diverse group of tumors that can affect various parts of the endocrine system. Early detection and treatment are crucial for improving prognosis, and prevention measures such as reducing exposure to radiation and maintaining a healthy lifestyle may also be helpful. It is important to seek medical attention if any symptoms persist or worsen over time.

Some common types of gastrointestinal neoplasms include:

1. Gastric adenocarcinoma: A type of stomach cancer that starts in the glandular cells of the stomach lining.
2. Colorectal adenocarcinoma: A type of cancer that starts in the glandular cells of the colon or rectum.
3. Esophageal squamous cell carcinoma: A type of cancer that starts in the squamous cells of the esophagus.
4. Small intestine neuroendocrine tumors: Tumors that start in the hormone-producing cells of the small intestine.
5. Gastrointestinal stromal tumors (GISTs): Tumors that start in the connective tissue of the GI tract.

The symptoms of gastrointestinal neoplasms can vary depending on the location and size of the tumor, but they may include:

* Abdominal pain or discomfort
* Changes in bowel habits (such as diarrhea or constipation)
* Weight loss
* Fatigue
* Nausea and vomiting

If you have any of these symptoms, it is important to see a doctor for further evaluation and diagnosis. A gastrointestinal neoplasm can be diagnosed through a combination of endoscopy (insertion of a flexible tube into the GI tract to visualize the inside), imaging tests (such as CT or MRI scans), and biopsy (removal of a small sample of tissue for examination under a microscope).

Treatment options for gastrointestinal neoplasms depend on the type, location, and stage of the tumor, but they may include:

* Surgery to remove the tumor
* Chemotherapy (use of drugs to kill cancer cells)
* Radiation therapy (use of high-energy X-rays or other particles to kill cancer cells)
* Targeted therapy (use of drugs that target specific molecules involved in cancer growth and development)
* Supportive care (such as pain management and nutritional support)

The prognosis for gastrointestinal neoplasms varies depending on the type and stage of the tumor, but in general, early detection and treatment improve outcomes. If you have been diagnosed with a gastrointestinal neoplasm, it is important to work closely with your healthcare team to develop a personalized treatment plan and follow up regularly for monitoring and adjustments as needed.

The carcinogenesis process of PDAC usually starts with the accumulation of genetic mutations in the pancreatic duct cells, which progressively leads to the formation of a premalignant lesion called PanIN (pancreatic intraepithelial neoplasia). Over time, these lesions can develop into invasive adenocarcinoma, which is PDAC.

The main risk factor for developing PDAC is smoking, but other factors such as obesity, diabetes, and family history of pancreatic cancer also contribute to the development of the disease. Symptoms of PDAC are often non-specific and late-stage, which makes early diagnosis challenging.

The treatment options for PDAC are limited, and the prognosis is generally poor. Surgery is the only potentially curative treatment, but only a small percentage of patients are eligible for surgical resection due to the locally advanced nature of the disease at the time of diagnosis. Chemotherapy, radiation therapy, and targeted therapies are used to palliate symptoms and improve survival in non-surgical cases.

PDAC is an aggressive and lethal cancer, and there is a need for better diagnostic tools and more effective treatment strategies to improve patient outcomes.

Types of experimental neoplasms include:

* Xenografts: tumors that are transplanted into animals from another species, often humans.
* Transgenic tumors: tumors that are created by introducing cancer-causing genes into an animal's genome.
* Chemically-induced tumors: tumors that are caused by exposure to certain chemicals or drugs.

The use of experimental neoplasms in research has led to significant advances in our understanding of cancer biology and the development of new treatments for the disease. However, the use of animals in cancer research is a controversial topic and alternatives to animal models are being developed and implemented.

A neoplasm is an abnormal growth of cells that can be benign (non-cancerous) or malignant (cancerous). Vascular tissue refers to tissues that are composed of cells and extracellular materials that form blood vessels. Neoplasms that affect vascular tissue are called vascular neoplasms.

Examples of vascular neoplasms include:

1. Hemangiomas: These are benign growths of blood vessels that can occur anywhere in the body, but are most common in the skin and internal organs.
2. Lymphangiomas: These are benign growths of lymphatic vessels that can occur in the skin or internal organs.
3. Angiosarcomas: These are malignant tumors that arise from the cells that line blood vessels. They can occur in any part of the body, but are most common in the skin and soft tissue.
4. Kaposi's sarcoma: This is a type of cancer that affects the lymphatic vessels and is caused by a virus called human herpesvirus 8 (HHV-8). It is more common in people with compromised immune systems, such as those with HIV/AIDS.
5. Venous malformations: These are abnormalities of the veins that can cause swelling and other symptoms. They can be congenital or acquired, and can range from benign to malignant.

Treatment for vascular neoplasms depends on the type and location of the tumor, as well as the patient's overall health. Some may require surgery, radiation therapy, or chemotherapy, while others may be monitored with regular imaging studies to ensure they do not grow or spread.

Some common types of eye neoplasms include:

1. Uveal melanoma: This is a malignant tumor that develops in the uvea, the middle layer of the eye. It is the most common primary intraocular cancer in adults and can spread to other parts of the body if left untreated.
2. Retinoblastoma: This is a rare type of cancer that affects children and develops in the retina. It is usually diagnosed before the age of 5 and is highly treatable with surgery, chemotherapy, and radiation therapy.
3. Conjunctival melanoma: This is a malignant tumor that develops in the conjunctiva, the thin membrane that covers the white part of the eye. It is more common in older adults and can be treated with surgery and/or radiation therapy.
4. Ocular sarcomas: These are rare types of cancer that develop in the eye tissues, including the retina, optic nerve, and uvea. They can be benign or malignant and may require surgical removal or radiation therapy.
5. Secondary intraocular tumors: These are tumors that metastasize (spread) to the eye from other parts of the body, such as breast cancer or lung cancer.

The symptoms of eye neoplasms can vary depending on their location and type, but may include:

* Blurred vision
* Eye pain or discomfort
* Redness or inflammation in the eye
* Sensitivity to light
* Floaters (specks or cobwebs in vision)
* Flashes of light
* Abnormal pupil size or shape

Early detection and treatment of eye neoplasms are important to preserve vision and prevent complications. Diagnosis is typically made through a combination of physical examination, imaging tests such as ultrasound or MRI, and biopsy (removing a small sample of tissue for examination under a microscope). Treatment options may include:

* Surgery to remove the tumor
* Radiation therapy to kill cancer cells
* Chemotherapy to destroy cancer cells with medication
* Observation and monitoring if the tumor is slow-growing or benign

It's important to seek medical attention if you experience any unusual symptoms in your eye, as early detection and treatment can improve outcomes.

Nose neoplasms refer to any type of abnormal growth or tumor that develops in the nose or nasal passages. These tumors can be benign (non-cancerous) or malignant (cancerous), and they can affect people of all ages.

Types of Nose Neoplasms[2]

There are several types of nose neoplasms, including:

1. Nasal polyps: These are benign growths that can occur in the nasal passages and are usually associated with allergies or chronic sinus infections.
2. Nasal carcinoma: This is a type of cancer that affects the nasal passages and can be either benign or malignant.
3. Esthesioneuroblastoma: This is a rare type of cancer that occurs in the nasal passages and is usually found in children.
4. Adenocarcinoma: This is a type of cancer that affects the glandular tissue in the nose and can be either benign or malignant.
5. Squamous cell carcinoma: This is a type of cancer that affects the squamous cells in the skin and mucous membranes of the nose.

Symptoms of Nose Neoplasms[3]

The symptoms of nose neoplasms can vary depending on the type and location of the tumor. Some common symptoms include:

1. Nasal congestion or blockage
2. Nasal discharge or bleeding
3. Loss of sense of smell or taste
4. Headaches
5. Sinus infections or other respiratory problems
6. Swelling or lumps in the nose or face
7. Difficulty breathing through the nose

Diagnosis and Treatment of Nose Neoplasms[4]

The diagnosis of nose neoplasms typically involves a combination of physical examination, imaging tests (such as CT scans or MRI), and biopsies. Treatment depends on the type and location of the tumor, and may involve surgery, radiation therapy, chemotherapy, or a combination of these. Some common treatment options include:

1. Surgical excision: This involves removing the tumor and any affected tissue through a surgical procedure.
2. Radiation therapy: This involves using high-energy beams to kill cancer cells.
3. Chemotherapy: This involves using drugs to kill cancer cells.
4. Laser therapy: This involves using a laser to remove or destroy the tumor.
5. Cryotherapy: This involves using extreme cold to destroy the tumor.

Prognosis and Follow-Up Care[5]

The prognosis for nose neoplasms depends on the type and location of the tumor, as well as the stage of the cancer. In general, early detection and treatment improve the chances of a successful outcome. Follow-up care is important to monitor the patient's condition and detect any recurrences or complications. Some common follow-up procedures include:

1. Regular check-ups with an otolaryngologist (ENT specialist)
2. Imaging tests (such as CT scans or MRI) to monitor the tumor and detect any recurrences
3. Biopsies to evaluate any changes in the tumor
4. Treatment of any complications that may arise, such as bleeding or infection.

Lifestyle Changes and Home Remedies[6]

There are several lifestyle changes and home remedies that can help improve the symptoms and quality of life for patients with nose neoplasms. These include:

1. Maintaining good hygiene, such as regularly washing the hands and avoiding close contact with others.
2. Avoiding smoking and other tobacco products, which can exacerbate the symptoms of nose cancer.
3. Using saline nasal sprays or drops to keep the nasal passages moist and reduce congestion.
4. Applying warm compresses to the affected area to help reduce swelling and ease pain.
5. Using over-the-counter pain medications, such as acetaminophen or ibuprofen, to manage symptoms.
6. Avoiding blowing the nose, which can dislodge the tumor and cause bleeding.
7. Avoiding exposure to pollutants and allergens that can irritate the nasal passages.
8. Using a humidifier to add moisture to the air and relieve dryness and congestion in the nasal passages.
9. Practicing good sleep hygiene, such as avoiding caffeine and electronic screens before bedtime and creating a relaxing sleep environment.
10. Managing stress through relaxation techniques, such as meditation or deep breathing exercises.

Nose neoplasms can have a significant impact on a person's quality of life, but with proper diagnosis and treatment, many patients can experience improved symptoms and outcomes. It is important for patients to work closely with their healthcare providers to develop a personalized treatment plan that addresses their specific needs and goals. Additionally, lifestyle changes and home remedies can help improve symptoms and quality of life for patients with nose neoplasms.

1. Parotid gland tumors: These are the most common type of salivary gland tumor and can be benign or malignant.
2. Submandibular gland tumors: These are less common than parotid gland tumors but can also be benign or malignant.
3. Sublingual gland tumors: These are rare and usually benign.
4. Warthin's tumor: This is a type of benign tumor that affects the parotid gland.
5. Mucoepidermoid carcinoma: This is a type of malignant tumor that can occur in any of the major salivary glands.
6. Acinic cell carcinoma: This is a rare type of malignant tumor that usually occurs in the parotid gland.
7. Adenoid cystic carcinoma: This is a slow-growing malignant tumor that can occur in any of the major salivary glands.
8. Metastatic tumors: These are tumors that have spread to the salivary glands from another part of the body.

Salivary gland neoplasms can cause a variety of symptoms, including painless lumps or swelling in the neck or face, difficulty swallowing, and numbness or weakness in the face. Treatment options depend on the type and stage of the tumor and may include surgery, radiation therapy, and/or chemotherapy.

In conclusion, salivary gland neoplasms are a diverse group of cancers that affect the salivary glands, and it's important to be aware of the different types, symptoms, and treatment options in order to provide effective care for patients with these tumors.

Example sentences:

1. The patient developed a radiation-induced neoplasm in their chest after undergoing radiation therapy for breast cancer.
2. The risk of radiation-induced neoplasms increases with higher doses of radiation exposure, making it crucial to minimize exposure during medical procedures.
3. The oncologist monitored the patient's health closely after their radiation therapy to detect any signs of radiation-induced neoplasms.

The term "papillary" refers to the fact that the cancer cells grow in a finger-like shape, with each cell forming a small papilla (bump) on the surface of the tumor. APC is often slow-growing and may not cause any symptoms in its early stages.

APC is generally considered to be less aggressive than other types of cancer, such as ductal carcinoma in situ (DCIS) or invasive breast cancer. However, it can still spread to other parts of the body if left untreated. Treatment options for APC may include surgery, radiation therapy, and/or hormone therapy, depending on the location and stage of the cancer.

It's worth noting that APC is sometimes referred to as "papillary adenocarcinoma" or simply "papillary cancer." However, these terms are often used interchangeably with "adenocarcinoma, papillary" in medical literature and clinical practice.

Testicular neoplasms refer to abnormal growths or tumors that develop in the testicles, which are located inside the scrotum. These tumors can be benign (non-cancerous) or malignant (cancerous). Testicular neoplasms can affect men of all ages, but they are more common in younger men between the ages of 20 and 35.

Types of Testicular Neoplasms:

There are several types of testicular neoplasms, including:

1. Seminoma: This is a type of malignant tumor that develops from immature cells in the testicles. It is the most common type of testicular cancer and tends to grow slowly.
2. Non-seminomatous germ cell tumors (NSGCT): These are malignant tumors that develop from immature cells in the testicles, but they do not have the characteristic features of seminoma. They can be either heterologous (containing different types of cells) or homologous (containing only one type of cell).
3. Leydig cell tumors: These are rare malignant tumors that develop in the Leydig cells, which produce testosterone in the testicles.
4. Sertoli cell tumors: These are rare malignant tumors that develop in the Sertoli cells, which support the development of sperm in the testicles.
5. Testicular metastasectomy: This is a procedure to remove cancer that has spread to the testicles from another part of the body, such as the lungs or liver.

Causes and Risk Factors:

The exact cause of testicular neoplasms is not known, but there are several risk factors that have been linked to an increased risk of developing these tumors. These include:

1. Undescended testicles (cryptorchidism): This condition occurs when the testicles do not descend into the scrotum during fetal development.
2. Family history: Men with a family history of testicular cancer are at an increased risk of developing these tumors.
3. Previous radiation exposure: Men who have had radiation therapy to the pelvic area, especially during childhood or adolescence, have an increased risk of developing testicular neoplasms.
4. Genetic mutations: Certain genetic mutations, such as those associated with familial testicular cancer syndrome, can increase the risk of developing testicular neoplasms.
5. Infertility: Men who are infertile may have an increased risk of developing testicular cancer.

Symptoms:

The symptoms of testicular neoplasms can vary depending on the type and location of the tumor. Some common symptoms include:

1. A lump or swelling in the testicle
2. Pain or discomfort in the testicle or scrotum
3. Enlargement of the testicle
4. Abnormality in the size or shape of the testicle
5. Pain during ejaculation
6. Difficulty urinating or painful urination
7. Breast tenderness or enlargement
8. Lower back pain
9. Fatigue
10. Weight loss

Diagnosis:

The diagnosis of testicular neoplasms typically involves a combination of physical examination, imaging studies, and biopsy.

1. Physical examination: A doctor will perform a thorough physical examination of the testicles, including checking for any abnormalities in size, shape, or tenderness.
2. Imaging studies: Imaging studies such as ultrasound, CT scans, or MRI may be used to help identify the location and extent of the tumor.
3. Biopsy: A biopsy is a procedure in which a small sample of tissue is removed from the testicle and examined under a microscope for cancer cells.
4. Blood tests: Blood tests may be performed to check for elevated levels of certain substances that can indicate the presence of cancer.

Treatment:

The treatment of testicular neoplasms depends on the type, location, and stage of the tumor. Some common treatments include:

1. Surgery: Surgery is often the first line of treatment for testicular neoplasms. The goal of surgery is to remove the tumor and any affected tissue.
2. Chemotherapy: Chemotherapy may be used in combination with surgery or radiation therapy to treat more advanced cancers.
3. Radiation therapy: Radiation therapy uses high-energy beams to kill cancer cells. It may be used in combination with surgery or chemotherapy.
4. Surveillance: Surveillance is a close monitoring of the patient's condition, including regular check-ups and imaging studies, to detect any recurrences of the tumor.

Prognosis:

The prognosis for testicular neoplasms depends on the type, location, and stage of the tumor. In general, the earlier the cancer is detected and treated, the better the prognosis. Some common types of testicular neoplasms have a good prognosis, while others are more aggressive and may have a poorer prognosis if not treated promptly.

Complications:

Some complications of testicular neoplasms include:

1. Recurrence: The cancer can recur in the testicle or spread to other parts of the body.
2. Spread to other parts of the body: Testicular cancer can spread to other parts of the body, such as the lungs, liver, or brain.
3. Infertility: Some treatments for testicular cancer, such as chemotherapy and radiation therapy, can cause infertility.
4. Hormone imbalance: Some types of testicular cancer can disrupt hormone levels, leading to symptoms such as breast enlargement or low sex drive.
5. Chronic pain: Some men may experience chronic pain in the testicle or scrotum after treatment for testicular cancer.

Lifestyle changes:

There are no specific lifestyle changes that can prevent testicular neoplasms, but some general healthy habits can help reduce the risk of developing these types of tumors. These include:

1. Maintaining a healthy weight and diet
2. Getting regular exercise
3. Limiting alcohol consumption
4. Avoiding smoking and recreational drugs
5. Protecting the testicles from injury or trauma

Screening:

There is no standard screening test for testicular neoplasms, but men can perform a self-exam to check for any abnormalities in their testicles. This involves gently feeling the testicles for any lumps or unusual texture. Men with a family history of testicular cancer should talk to their doctor about whether they should start screening earlier and more frequently.

Treatment:

The treatment of testicular neoplasms depends on the type, stage, and location of the tumor. Some common treatments include:

1. Surgery: This involves removing the affected testicle or tumor.
2. Chemotherapy: This involves using drugs to kill cancer cells.
3. Radiation therapy: This involves using high-energy rays to kill cancer cells.
4. Hormone therapy: This involves taking medications to alter hormone levels and slow the growth of cancer cells.
5. Clinical trials: These involve testing new treatments or combination of treatments for testicular neoplasms.

Prognosis:

The prognosis for testicular neoplasms varies depending on the type, stage, and location of the tumor. In general, the earlier the cancer is detected and treated, the better the prognosis. For example, seminoma has a high cure rate with current treatments, while non-seminomatous germ cell tumors have a lower cure rate but can still be effectively treated. Lymphoma and metastatic testicular cancer have a poorer prognosis and require aggressive treatment.

Lifestyle Changes:

There are no specific lifestyle changes that can prevent testicular neoplasms, but some risk factors such as smoking and alcohol consumption can be reduced to lower the risk of developing these tumors. Maintaining a healthy diet, regular exercise, and avoiding exposure to harmful chemicals can also help improve overall health and well-being.

Complications:

Testicular neoplasms can have several complications, including:

1. Infertility: Some treatments for testicular cancer, such as surgery or chemotherapy, can cause infertility.
2. Pain: Testicular cancer can cause pain in the scrotum, groin, or abdomen.
3. Swelling: Testicular cancer can cause swelling in the scrotum or groin.
4. Hormonal imbalance: Some testicular tumors can produce hormones that can cause an imbalance in the body's hormone levels.
5. Recurrence: Testicular cancer can recur after treatment, and regular follow-up is necessary to detect any signs of recurrence early.
6. Late effects of treatment: Some treatments for testicular cancer, such as chemotherapy, can have long-term effects on the body, including infertility, heart problems, and bone marrow suppression.
7. Metastasis: Testicular cancer can spread to other parts of the body, including the lungs, liver, and bones, which can be life-threatening.

Prevention:

There is no specific prevention for testicular neoplasms, but some risk factors such as undescended testes, family history, and exposure to certain chemicals can be reduced to lower the risk of developing these tumors. Regular self-examination and early detection are crucial in improving outcomes for patients with testicular cancer.

Conclusion:

Testicular neoplasms are a rare but potentially life-threatening condition that requires prompt and accurate diagnosis and treatment. Early detection through regular self-examination and follow-up can improve outcomes, while awareness of risk factors and symptoms is essential in reducing the burden of this disease. A multidisciplinary approach involving urologists, radiologists, pathologists, and oncologists is necessary for optimal management of patients with testicular neoplasms.

Neoplasms can be classified as benign (non-cancerous) or malignant (cancerous). Malignant neoplasms can further be divided into primary neoplasms, which originate in the muscle tissue itself, and secondary neoplasms, which spread to the muscle from another part of the body.

Examples of malignant muscle neoplasms include rhabdomyosarcoma (a type of cancer that arises in immature muscle cells) and adult-type fibromyxoma (a rare, slow-growing tumor that usually affects the extremities).

In contrast, benign muscle neoplasms are non-cancerous growths that do not spread to other parts of the body. Examples include benign fibrous histiocytomas and benign pleomorphic adipose tumors.

Neoplasms, Muscle Tissue Symptoms The symptoms of muscle neoplasms vary depending on their size, location, and malignant potential. In general, patients may experience painless lumps or masses, muscle weakness or wasting, and localized swelling or redness.

Diagnosis The diagnosis of muscle neoplasms is based on a combination of clinical findings, imaging studies (such as MRI or CT scans), and biopsy results. Imaging studies can help to identify the size, location, and extent of the tumor, while biopsy can provide a definitive diagnosis by examining the tissue under a microscope.

Treatment Treatment options for muscle neoplasms depend on the type, size, location, and malignant potential of the tumor, as well as the patient's overall health. Surgery is often the primary treatment modality for both benign and malignant muscle neoplasms. In some cases, radiation therapy or chemotherapy may be added to the treatment regimen.

Prognosis The prognosis for patients with muscle neoplasms varies depending on the type and malignant potential of the tumor. In general, benign muscle neoplasms have a good prognosis and do not spread to other parts of the body, while malignant muscle neoplasms can be aggressive and may have a poorer prognosis if left untreated.

Differential Diagnosis The differential diagnosis for muscle neoplasms includes other soft tissue tumors such as lipomas, hemangiomas, and synovial sarcomas, as well as non-tumorous conditions such as inflammatory myopathies and fibromatoses.

Examples of neoplasms, glandular and epithelial include:

* Adenomas: These are benign tumors that arise from glandular tissue. Examples include colon adenomas and prostate adenomas.
* Carcinomas: These are malignant tumors that arise from glandular or epithelial tissue. Examples include breast carcinoma, lung carcinoma, and ovarian carcinoma.
* Sarcomas: These are malignant tumors that arise from connective tissue. Examples include soft tissue sarcoma and bone sarcoma.

The diagnosis of neoplasms, glandular and epithelial is typically made through a combination of imaging tests such as X-rays, CT scans, MRI scans, and PET scans, along with a biopsy to confirm the presence of cancer cells. Treatment options for these types of neoplasms depend on the location, size, and stage of the tumor, but may include surgery, chemotherapy, radiation therapy, or a combination of these.

Overall, the term "neoplasms, glandular and epithelial" refers to a wide range of tumors that arise from glandular or epithelial tissue, and can be either benign or malignant. These types of neoplasms are common and can affect many different parts of the body.

Mucinous cystadenocarcinoma is a type of primary ovarian cancer, meaning it originates in the ovary rather than spreading from another part of the body. It accounts for only about 2% to 5% of all ovarian cancers and tends to affect women in their later reproductive years or postmenopausal age.

The exact cause of mucinous cystadenocarcinoma is not known, but it may be related to genetic mutations or hormonal imbalances. Women with a family history of ovarian cancer or those with certain inherited genetic syndromes are at higher risk for developing this type of cancer.

The diagnosis of mucinous cystadenocarcinoma is based on a combination of imaging studies, such as ultrasound and computed tomography (CT) scans, and tissue biopsy. Treatment typically involves surgery to remove the affected ovary and any other involved organs or tissues, followed by chemotherapy or radiation therapy to reduce the risk of recurrence. Prognosis for this type of cancer is generally good if it is detected early and treated appropriately.

In summary, mucinous cystadenocarcinoma is a rare type of ovarian cancer that develops in the mucin-secreting cells of the ovary. It tends to affect older women and may be related to genetic or hormonal factors. Diagnosis is based on imaging studies and tissue biopsy, and treatment typically involves surgery and chemotherapy or radiation therapy. Prognosis is generally good if caught early.

Adenomas are caused by genetic mutations that occur in the DNA of the affected cells. These mutations can be inherited or acquired through exposure to environmental factors such as tobacco smoke, radiation, or certain chemicals.

The symptoms of an adenoma can vary depending on its location and size. In general, they may include abdominal pain, bleeding, or changes in bowel movements. If the adenoma becomes large enough, it can obstruct the normal functioning of the affected organ or cause a blockage that can lead to severe health complications.

Adenomas are usually diagnosed through endoscopy, which involves inserting a flexible tube with a camera into the affected organ to visualize the inside. Biopsies may also be taken to confirm the presence of cancerous cells.

Treatment for adenomas depends on their size, location, and severity. Small, non-pedunculated adenomas can often be removed during endoscopy through a procedure called endoscopic mucosal resection (EMR). Larger adenomas may require surgical resection, and in some cases, chemotherapy or radiation therapy may also be necessary.

In summary, adenoma is a type of benign tumor that can occur in glandular tissue throughout the body. While they are not cancerous, they have the potential to become malignant over time if left untreated. Therefore, it is important to seek medical attention if symptoms persist or worsen over time. Early detection and treatment can help prevent complications and improve outcomes for patients with adenomas.

"Bronchial leiomyoma". Humpath.com. 17 January 2017. (Benign neoplasms, Respiratory system neoplasia). ... Bronchial leiomyomas are only 0.1% to 2% of benign lung tumours. Bronchial lyeiomyomas comprise 33-45% of respiratory system ... The first bronchial leiomyoma was described by in 1909. Saoud M, Patil M, Dhillon SS, Pokharel S, Picone A, Hennon M, et al. ( ... A bronchial leiomyoma is a relatively rare form of lung tumours. These tumours can form in the lower respiratory tract tissue ...
Anderson HJ, Churchill-Davidson HC, Richardson AT (December 1953). "Bronchial neoplasm with myasthenia; prolonged apnoea after ... "Defect of neuromuscular conduction associated with malignant neoplasms". Am. J. Physiol. 187: 612-613. Gutmann L, Crosby TW, ...
... is a very rare malignant neoplasm originating from bronchial glands. It is classified as a ...
Entero-pancreatic gastrinomas and thymic and bronchial carcinoids are the leading cause of morbidity and mortality. ... "Cutaneous tumors in patients with multiple endocrine neoplasm type 1 (MEN1) and gastrinomas: prospective study of frequency and ... and bronchial carcinoids also occur. The phenotype of MEN1 is broad, and over 20 different combinations of endocrine and non- ... Other endocrine and non-endocrine neoplasms including adrenocortical and thyroid tumors, visceral and cutaneous lipomas, ...
Although it is not normally considered a fast-growing malignant neoplasm, FA can exhibit high uptake on FDG-PET scanning. ... October 2010). "Bronchial brushing cytology of a pulmonary fetal adenocarcinoma with a poorly differentiated component". ... While FA can be diagnosed via biopsy, bronchial brushings, and immunocytochemistry, examination of the whole tumor is required ... Odashiro DN, Nguyen GK (April 2006). "Pulmonary well-differentiated fetal adenocarcinoma diagnosed by bronchial brush and ...
In a pneumonectomy, in which an entire lung is removed, the remaining bronchial stump may leak air, a rare but very serious ... It may also occur with fractures of the facial bones, neoplasms, during asthma attacks, when the Heimlich maneuver is used, and ... Trauma to parts of the respiratory system other than the lungs, such as rupture of a bronchial tube, may also cause ... Air may travel upward to the neck from a pneumomediastinum that results from a bronchial rupture, or downward from a torn ...
... cervical intraepithelial neoplasm, CIN) vaginal intraepithelial neoplasm (VAIN) anal dysplasia (also see: anal cancer) lichen ... Bronchial premalignant lesions can progress to squamous cell carcinoma of the lung. Pathologically, precancerous tissue can ... April 2019). "Molecular subtyping reveals immune alterations associated with progression of bronchial premalignant lesions". ... hereditary nonpolyposis colorectal cancer Ulcerative colitis Crohn's disease Respiratory Bronchial premalignant lesions ...
NOS M8000/6 Neoplasm, metastatic Neoplasm, metastatic Tumor, metastatic Tumor, secondary Tumor embolus M8000/9 Neoplasm, ... cylindroid Bronchial adenoma, cylindroid (C34._) M8201/2 Cribiform carcinoma in situ (C50._) Ductal carcinoma in situ, ... benign M8000/1 Neoplasm, uncertain whether benign or malignant Neoplasm, NOS Tumor, NOS Unclassified tumor, uncertain whether ... M8130/1 Papillary transitional cell neoplasm of low malignant potential (C67._) Papillary urothelial neoplasm of low malignant ...
... can be prescribed with other medications such as antifungal drugs, bronchial spasmolytics, ... malignant neoplasms, acute renal failure, age under 18 years (no clinical study data available). It should also be used with ...
These include: Primary tumors of the lung/pulmonary system: Bronchial leiomyoma, a rare, benign tumor Lung cancer, the term ... Neoplasms of the lung". In Jameson JL, Fauci AS, Kasper DL, Hauser SL, Longo DL, Loscalzo J (eds.). Harrison's Principles of ... Mediastinal tumors Pleural tumors Metastasis or secondary tumors/neoplasms with other origin: Metastasis to the lung Lung ...
Presence of air-fluid levels implies rupture into the bronchial tree or rarely growth of gas forming organism.[citation needed ... are due to neoplasms across all age groups, higher in older people; primary squamous carcinoma of the lung is the most common. ... On examination of the chest there will be features of consolidation such as localized dullness on percussion and bronchial ... Fiber optic bronchoscopy is often performed to exclude obstructive lesion; it also helps in bronchial drainage of pus.[citation ...
Adenocarcinoma of the lung Bronchial adenomas/carcinoids Small cell lung cancer Mesothelioma Non-small cell lung cancer Non- ... Marginal zone B-cell lymphoma Mast cell leukemia Mediastinal large B cell lymphoma Multiple myeloma/plasma cell neoplasm ...
G1 and G2 neuroendocrine neoplasms are called neuroendocrine tumors (NETs) - formerly called carcinoid tumours. G3 neoplasms ... Bronchial carcinoid can cause airway obstruction, pneumonia, pleurisy, difficulty with breathing, cough, and hemoptysis, or may ... Although there are many kinds of NETs, they are treated as a group of tissue because the cells of these neoplasms share common ... Neuroendocrine tumors (NETs) are neoplasms that arise from cells of the endocrine (hormonal) and nervous systems. They most ...
Apart from that it is doubtful that possible prognosis for a patient with bronchial asthma and chronic leukemia is comparable ... In the comorbid structure of these patients, most frequently present are malignant neoplasms, locomotorium disorders, skin and ... Greenfield to evaluate comorbidity in patients with malignant neoplasms, later it also became useful for other categories of ... Role of chronic allergic inflammation in bronchial asthma pathogenesis and its rational pharmacological therapy for patients ...
Naib ZM (1961). "Giant cell carcinoma of the lung: cytological study of the exfoliated cells in sputa and bronchial washings". ... Spivach A, Borea B, Bertoli G, Daris G (July 1976). "[Primary lung neoplasm of rare incidence: giant cell carcinoma]". Minerva ... The new paradigm recognizes that lung cancers are a large and extremely heterogeneous family of malignant neoplasms, with over ... Travis WD (November 2010). "Sarcomatoid neoplasms of the lung and pleura". Arch. Pathol. Lab. Med. 134 (11): 1645-58. doi: ...
Moro D, Brichon PY, Brambilla E, Veale D, Labat F, Brambilla C (June 1994). "Basaloid bronchial carcinoma. A histologic group ... Maleki Z (March 2011). "Diagnostic issues with cytopathologic interpretation of lung neoplasms displaying high-grade basaloid ...
Kabela M (1956). "[Experience with radical irradiation of bronchial cancer]" [Experience with radical irradiation of bronchial ... Recurrence of lung cancer Horn L, Lovly CM (2018). "Chapter 74: Neoplasms of the lung". In Jameson JL, Fauci AS, Kasper DL, ... Horn L, Iams WT (2022). "78: Neoplasms of the Lung". Harrison's Principles of Internal Medicine (21 ed.). McGraw Hill. Nasim F ... Tan D, Zander DS (January 2008). "Immunohistochemistry for assessment of pulmonary and pleural neoplasms: a review and update ...
... respiratory tract neoplasms MeSH C04.588.894.797.265 - bronchial neoplasms MeSH C04.588.894.797.520 - lung neoplasms MeSH ... skull base neoplasms MeSH C04.588.149.828 - spinal neoplasms MeSH C04.588.180.260 - breast neoplasms, male MeSH C04.588.180.390 ... bile duct neoplasms MeSH C04.588.274.120.250.250 - common bile duct neoplasms MeSH C04.588.274.120.401 - gallbladder neoplasms ... femoral neoplasms MeSH C04.588.149.721 - skull neoplasms MeSH C04.588.149.721.450 - jaw neoplasms MeSH C04.588.149.721.450.583 ...
... is an undifferentiated neoplasm composed of primitive-appearing cells. As the name implies, the cells in ... carcinoma of the lung usually presents in the central airways and infiltrates the submucosa leading to narrowing of bronchial ...
Other neoplasms (or sources of inflammation) should therefore be considered in known or suspected LAM cases in which FDG-PET ... to destroy bronchial cartilage and arteriolar walls, and to occlude the lumen of pulmonary arterioles. There are two major cell ...
In most cases PRRT is used for cancers of the gastroenteropancreatic and bronchial tracts, and in some cases phaeochromocytoma ... "Current Status of Radiopharmaceuticals for the Theranostics of Neuroendocrine Neoplasms". Pharmaceuticals. 10 (4): 30. doi: ...
Typically presents unilaterally in submandibular gland that cannot be differentiated clinically from a neoplasm, with pain an ... such as chronic sinusitis or bronchial asthma. One study found that 112 patients from England and Wales ranging from 12 to 81 ...
"Salivary Gland Neoplasms". Medscape.{{cite web}}: CS1 maint: multiple names: authors list (link) Updated: Jan 13, 2021}} ... Alcian blue-PAS stain Mucoepidermoid carcinomas of the salivary and bronchial glands are characterized by a recurrent t(11;19)( ...
December 2004). "Immortalization of human bronchial epithelial cells in the absence of viral oncoproteins". Cancer Research. 64 ... Misago N, Narisawa Y (September 2006). "Cytokeratin 15 expression in neoplasms with sebaceous differentiation". Journal of ...
... neoplasm seeding MeSH C23.550.727.650.895 - neoplasms, unknown primary MeSH C23.550.727.655 - neoplasm recurrence, local MeSH ... bronchial fistula MeSH C23.300.575.825 - urinary fistula MeSH C23.300.575.825.250 - bladder fistula MeSH C23.300.575.825. ... neoplasm metastasis MeSH C23.550.727.650.560 - lymphatic metastasis MeSH C23.550.727.650.645 - neoplasm circulating cells MeSH ... C23.550.727.670 - neoplasm regression, spontaneous MeSH C23.550.727.700 - neoplasm, residual MeSH C23.550.737.500 - retrograde ...
... which may be benign neoplasms) or else a malignant neoplasm (cancer). These neoplasms are also indicated, in the diagram below ... "Multiple clonal abnormalities in the bronchial epithelium of patients with lung cancer". Journal of the National Cancer ... Neoplasms are mosaics of different mutant cells with both genetic and epigenetic changes that distinguish them from normal ... In this way, a population of mutant cells, called a clone, can expand in the neoplasm. Clonal expansion is the signature of ...
August 2005). "Cigarette smoke extract induces DNA damage but not apoptosis in human bronchial epithelial cells". American ... Large-cell lung carcinoma (LCLC) is a heterogeneous group of undifferentiated malignant neoplasms originating from transformed ... in that the tumor cells lack light microscopic characteristics that would classify the neoplasm as a small-cell carcinoma, ...
Mutant cells in neoplasms compete for space and resources. Thus, a clone with a mutation in a tumor suppressor gene or oncogene ... April 2019). "Molecular subtyping reveals immune alterations associated with progression of bronchial premalignant lesions". ... Some of the small polyps in the field defect shown in the photo of the opened colon segment may be relatively benign neoplasms ... These neoplasms are also indicated (in the diagram below the photo) by 4 small tan circles (polyps) and a larger red area ( ...
Papillary - In oncology, papillary refers to neoplasms with projections ("papillae", from Latin, 'nipple') that have ... the bronchial tubes, the intervertebral discs, and many other body components. It is not as hard and rigid as bone, but it is ...
Its expression is also increased in a wide range of other malignant neoplasms. Factor X (F10) is frequently expressed in normal ... November 2010). "Cytopathogenesis of Sendai virus in well-differentiated primary pediatric bronchial epithelial cells". Journal ... It is also overexpressed in some cell lines originating from various malignant neoplasms. Thus, it is highly expressed in ... Some of these proteases are overexpressed in malignant neoplasms. For example, transmembrane serine protease 2 (TMPRSS2), which ...
Bronchial carcinoid tumors: second primary neoplasms and outcomes of surgical treatment. Bronchial carcinoid tumors: second ... Primary neoplasms that were concomitant with the bronchial carcinoid tumors were identified by reviewing patient charts. ... Bronchial carcinoid tumors are likely to be accompanied by second primary neoplasms. ... Concomitant primary neoplasms were more common in patients who were asymptomatic and in those with small tumors. CONCLUSIONS:. ...
Invading bronchial neoplasms. * Fibrosarcomas. * Metastases to the diaphragm (eg, ovarian malignancy). * Aspiration of amebic ...
Thoracic Neoplasms. Neoplasms by Site. Neoplasms. Lung Diseases. Respiratory Tract Diseases. Carcinoma, Bronchogenic. Bronchial ... Lung Neoplasms. Carcinoma, Non-Small-Cell Lung. Respiratory Tract Neoplasms. ...
Neoplasms by Site. Neoplasms. Lung Diseases. Respiratory Tract Diseases. Carcinoma, Bronchogenic. Bronchial Neoplasms. ... Lung Neoplasms. Carcinoma, Non-Small-Cell Lung. Respiratory Tract Neoplasms. Thoracic Neoplasms. ...
Occupational exposure; Health effects; Systematic reviews; Morbidity rates; Welding fumes; Welders; Lung cancer; Bronchial ... Tracheal neoplasms; Lung neoplasms ...
NIOSH-Author; NIOSH-Health-Hazard-Evaluation; HETA-81-025-1668; Region-3; Hazard-Confirmed; Epidemiology; Respiratory-neoplasms ... Lung-cancer; Bronchial-cancer; Construction-workers; Carcinogens; NIOSH-Technical-Assistance-Report; Author Keywords: Lung ...
Established Bronchial Brush and Bronchoalveolar lavage Cytology. *Efficacy of Bronchial brush Cytology and Bronchial washings ... Worked on thyroid neoplasms with use of Imaging and morphometry to differentiate benign from malignant tumors. *Diagnostic ... Choudhury M, Singh S, Agarwal S Efficacy of Bronchial brush Cytology and Bronchial washings in diagnosis of Non neoplastic and ... P53 overexpression in Ovarian Neoplasms. Worked on better diagnostic methods in Bronchopulmonary Lesions.. * ...
Histological Pattern of Neoplasm Resulting Malignant Pleural Effusion among the Patients Admitted in NIDCH, Bangladesh Jalal ... Association of CT Scan Finding of Bronchial Carcinoma with Fiber Optic Bronchoscopy Nihar Ranjan Saha, Md Sayedul Islam, Nirmal ...
Bronchial Neoplasm. Neoplasm, Bronchial. Neoplasms, Bronchial. Tree number(s):. C04.588.894.797.520.109. C08.127.265. C08.785. ... not for bronchial carcinoma (= CARCINOMA, BRONCHIAL see CARCINOMA, BRONCHOGENIC + LUNG NEOPLASMS), nor bronchogenic carcinoma ... LUNG NEOPLASMS); coordinate IM with histological type of neoplasm (IM). ... CARCINOMA, BRONCHOGENIC + LUNG NEOPLASMS) nor bronchiolar carcinoma (= CARCINOMA, BRONCHIOLAR + ...
Adenocarcinoma of Lung; Bronchial Neoplasms; Early Detection of Cancer; Machine Learning Intervention(s) ...
Bronchial Neoplasm Market. "Bronchial Neoplasm Market Insights, Epidemiology, and Market Forecast-2032" report delivers an in- ... depth understanding of the historical and forecasted epidemiology as well as the Bronchial Neoplasm market trends in the 7MM (i ... and unmet medical needs to curate the best of the opportunities and assesses the underlying potential of the Bronchial Neoplasm ...
bronchial neoplasm DOID:3906 * skin sarcoma DOID:2687 * benign familial neonatal epilepsy ...
Breast Neoplasms, Male (Phase 1) Brenner Tumor (Phase 1) Bronchial Diseases (Phase 3) ...
... neoplasms, infection, milk protein allergies, and immune complex disorders (2,3), and occurs only rarely among infants. ... were not identified in the bronchial aspirates of the infants in Chicago. However, for six of the infants who underwent ...
Neoplasms (Cancer) 04/01/2012 - "Laser + SWNT-GC afford a remarkable efficacy in suppressing tumor growth in animal cancer ... and granulations in the tracheal and bronchial lumen both as the first stage of surgical treatment and as an independent ...
Bronchial disorder. Unknown. 13. Needle issue. Unknown. 14. Alopecia. Unknown. 15. Neoplasm progression. Unknown. ...
... is a neoplasm that develops at the expense of cells in the bronchial lining. The main risk factor is smoking [2-6]. ... In Africa primary bronchial cancer appears to be a rare entity.. Objective: To study the prognostic factors and survival of ... primary bronchial cancer appears to be a rare entity for two reasons: the small technical platform reducing diagnostic means ...
Bronchogenic Bronchial Neoplasms Lung … Neoplasms Neoplasms by Site Carcinoma, Bronchogenic Bronchial Neoplasms Lung … ... Neoplasms Bronchial Neoplasms Carcinoma, Bronchogenic Respiratory Tract Diseases Respiratory … Neoplasms Bronchial Neoplasms ... and Epithelial Carcinoma, Bronchogenic Bronchial Neoplasms Lung … Tissue Diseases Carcinoma, Bronchogenic Bronchial Neoplasms ... Bronchogenic Bronchial Neoplasms Lung … Diseases Lung Diseases Carcinoma, Bronchogenic Adenoma Neoplasms, Glandular … ...
Flexible bronchoscopy confirmed extrinsic compression of right and left bronchial trees. Endobronchial ultrasound (EBUS) was ... keywords = "Biopsy, Bronchoscopes, Fine-needle, Mediastinal neoplasms, Solitary fibrous tumors, Ultrasonography",. author = " ... Flexible bronchoscopy confirmed extrinsic compression of right and left bronchial trees. Endobronchial ultrasound (EBUS) was ... Flexible bronchoscopy confirmed extrinsic compression of right and left bronchial trees. Endobronchial ultrasound (EBUS) was ...
APCHyperparathyroidismSalivary Gland NeoplasmsColonic NeoplasmsIntestinal NeoplasmsHyperplasiaNeoplasms, Multiple Primary ... They usually originate from the bronchial glands, but may also be unrelated to the bronchial tract and be localized in the ... Pituitary Neoplasms. Neoplasms which arise from or metastasize to the PITUITARY GLAND. The majority of pituitary neoplasms are ... AcromegalyCushing SyndromeHyperparathyroidismSalivary Gland NeoplasmsColonic NeoplasmsIntestinal NeoplasmsHyperplasiaNeoplasms ...
... similar to neoplasms. Bronchial walls as properly as blood vessels are invaded and destroyed, and cavities are fashioned. The ...
Foot Diseases, Soft Tissue Neoplasms, Magnetic Resonance Imaging, Radiology, Medicine, Reference Books, Atlas [tipo de ... Bronchoscopy, Larynx/pathology, Trachea/pathology, Bronchial Diseases, Bronchi, Medicine, Reference Books, Atlas [tipo de ... Brain Diseases, Cerebrum, Stroke, Brain Neoplasms, Alzheimer Disease, Huntington Disease, Communicable Diseases, Medicine, ...
Bronchial wall thickening and cylindrical bronchial dilatation in areas of air bronchogram (71%) (See the image below.) ... primary bronchogenic neoplasm, and tuberculosis. ...
Neoplasms (cancer) ongoing. -Acute infections -Phlebitis, thrombosis and thrombophlebitis in course. -Acute phase in autoimmune ... Bronchial asthma. -Oedema from heart failure. -Heart failure. -Carotid sinus syndrome.. -Problems of liver and kidney. ...
  • Bronchial carcinoid tumors: second primary neoplasms and outcomes of surgical treatment. (bvsalud.org)
  • To analyze determinants of prognosis in patients with bronchial carcinoid tumors treated surgically and the potential concomitance of such tumors with second primary neoplasms . (bvsalud.org)
  • This was a retrospective analysis of 51 bronchial carcinoid tumors treated surgically between 2007 and 2016. (bvsalud.org)
  • Primary neoplasms that were concomitant with the bronchial carcinoid tumors were identified by reviewing patient charts. (bvsalud.org)
  • Concomitant primary neoplasms were more common in patients who were asymptomatic and in those with small tumors . (bvsalud.org)
  • Bronchial carcinoid tumors are likely to be accompanied by second primary neoplasms . (bvsalud.org)
  • ORIGINAL ARTICLE Annals of Nuclear Medicine Vol. 8, No. 2, 115-123, 1994 Intra-arterial infusion of N-isopropyl-p[123I]iodoamphetamine for assessing effective blood supply to pulmonary and hepatic neoplasms Chihoko MIYAZAKI Department of Diagnostic Radiology, Sapporo City General Hospital The biodistribution and pharmacokinetics of intra-arterially administered N-isopropyl-p[123I] iodoamphetamine ( 123I-IMP) were prospectively evaluated in 38 patients with histologically proven pulmonary or hepatic tumors. (jsnm.org)
  • Findings from a 51-year-old immunocompetent woman with a benign neoplasm and Cryptosporidium baileyi pulmonary infection, Poland, 2015. (cdc.gov)
  • A benign neoplasm of the ADRENAL CORTEX. (lookformedical.com)
  • Among the concomitant primary neoplasms that were malignant, the most common was lung adenocarcinoma , which was observed in 3 cases. (bvsalud.org)
  • 19,20 Considering these reports on 123I-IMP as a freely diffusible and highly extracted tracer for regional cerebral blood flow measurement, the author assumed that the first arterial distribution of 123I-IMP must reflect the lung and the liver blood perfusion including the neoplasms as well as brain tissues. (jsnm.org)
  • Primary lung cancer (PLC) is a neoplasm that develops at the expense of cells in the bronchial lining. (fortunepublish.com)
  • Aspergilloma of the lung is a combination of mycelium of mold fungi of the genus Aspergillus and cellular detritus that fills bronchial ecstases and cavities of the lung parenchyma. (losartanp.com)
  • The scintigraphic studies concerning the intra-arterial infusion of 123I-IMP in patients with pulmonary and hepatic neoplasms were designed to investigate the distribution of 123I-IMP in the lungs and liver to determine the tumor blood flow. (jsnm.org)
  • Key words: tumor blood flow, intra-arterial infusion, N-isopropyl-p[123I]iodoamphetamine ( 123I-IMP), interventional radiology INTRODUCTION INTRA-ARTERIAL INFUSION of an anti-tumor chemotherapeutic agent has proven to be effective in the management of primary and metastatic malignant neoplasms. (jsnm.org)
  • Flexible bronchoscopy confirmed extrinsic compression of right and left bronchial trees. (elsevier.com)
  • ENETS Consensus Guidelines for the management of patients with liver and other distant metastases from neuroendocrine neoplasms of foregut, midgut, hindgut, and unknown primary. (nih.gov)
  • Concomitant primary neoplasms were observed in 14 (27.4%) of the 51 cases. (bvsalud.org)
  • 12. [Choroidal metastasis after a bronchial carcinoid tumor]. (nih.gov)
  • 14. [Bronchial carcinoid--a rare neoplasm metastasis to choroid]. (nih.gov)
  • The PMR study showed a statistically significant excess of all malignant neoplasms for white males but not for the other three race/sex groups. (cdc.gov)
  • Findings from a 51-year-old immunocompetent woman with a benign neoplasm and Cryptosporidium baileyi pulmonary infection, Poland, 2015. (cdc.gov)