Brain Edema
Edema
Aquaporin 4
Blood-Brain Barrier
Brain Injuries
Pulmonary Edema
Brain Chemistry
Brain Ischemia
Specific Gravity
Brain
Brain Neoplasms
Evans Blue
Intracranial Pressure
Infarction, Middle Cerebral Artery
Cerebral Hemorrhage
Brain Infarction
Gerbillinae
Magnetic Resonance Imaging
Brain Mapping
Disease Models, Animal
Rats, Sprague-Dawley
Capillary Permeability
Neuroprotective Agents
Intracranial Hypertension
Cerebral Cortex
Liver Failure, Acute
Corneal Edema
Edema, Cardiac
Cerebral Infarction
Diuretics, Osmotic
Meningioma
Water
Water Intoxication
Hepatic Encephalopathy
Aquaporins
Astrocytes
Hyperammonemia
Rats, Wistar
Ischemic Attack, Transient
Edema Disease of Swine
Brain Stem
Meningeal Neoplasms
Claudin-5
Rats, Inbred Strains
Neurons
Intracranial Embolism and Thrombosis
Reperfusion
Brain Abscess
Cranial Sinuses
Laryngeal Edema
Tomography, X-Ray Computed
Hypothermia, Induced
Antipyrine
Neurologic Examination
Mannitol
Injections, Intraperitoneal
Nimodipine
Sodium
Transforming growth factor-alpha acting at the epidermal growth factor receptor reduces infarct volume after permanent middle cerebral artery occlusion in rats. (1/1215)
Transforming growth factor-alpha (TGF-alpha) is a ligand for the epidermal growth factor (EGF) receptor (EGFR), and is more abundant than EGF in the brain. The authors studied whether administration of exogenous TGF-alpha into the brain can protect neurons against ischemia in a model of permanent middle cerebral artery (MCA) occlusion in the rat, and whether any effect of TGF-alpha was mediated by EGFR by administering 4,5-dianilinophthalimide (DAPH), a protein-tyrosine kinase inhibitor with high selectivity for EGFR. Rats received either TGF-alpha (10 or 25 ng), DAPH (100 ng), DAPH plus TGF-alpha (25 ng), or vehicle in the ipsilateral first ventricle. Drugs were administered twice: 30 minutes before and 30 minutes after MCA occlusion, and infarct volume was evaluated 24 hours later. Transforming growth factor-alpha at the dose of 25 ng caused a statistically significant reduction of infarct volume (60%) in relation to ischemic rats administered vehicle. This reduction was no longer seen when TGF-alpha was administered in combination with DAPH. The present results show that TGF-alpha can protect neurons from ischemic damage, and that this effect is mediated by EGFR. It is suggested that activation of EGFR-mediated intracellular signalling pathways contributes to the survival of neural cells susceptible to ischemic injury. (+info)Computerised axial tomography in patients with severe migraine: a preliminary report. (2/1215)
Patients suffering from severe migraine, usually for many years, have been examined by the EMI scanner between attacks. Judged by criteria validated originally by comparison with pneumoencephalography, about half of the patients showed evidence of cerebral atrophy. Perhaps of more significance than generalised atrophy was the frequency of areas of focal atrophy and of evidence of infarction. (+info)Two similar cases of encephalopathy, possibly a reversible posterior leukoencephalopathy syndrome: serial findings of magnetic resonance imaging, SPECT and angiography. (3/1215)
Two young women who had encephalopathy that resembled reversible posterior leukoencephalopathy syndrome are presented. The brain magnetic resonance imaging (MRI) of these patients exhibited similar T2-high signal lesions, mostly in the white matter of the posterior hemispheres. Xe-SPECT during the patients' symptomatic period showed hypoperfusion in the corresponding areas, and angiography demonstrated irregular narrowing of the posterior cerebral artery. Clinical manifestations subsided soon after treatment, and the abnormal radiological findings also were almost completely resolved. Thus, we concluded that transient hypoperfusion followed by ischemia and cytotoxic edema might have had a pivotal role in these cases. (+info)Significance of acute cerebral swelling in patients with sylvian hematoma due to ruptured middle cerebral artery aneurysm, and its management. (4/1215)
A retrospective study of 75 patients treated surgically for ruptured middle cerebral artery (MCA) aneurysm within 48 hours evaluated clinical grade at admission, secondary development and management of cerebral swelling associated with space-occupying hematoma, cerebral infarction caused by vasospasm, development of hydrocephalus, and clinical outcome. Clinical grade at admission was significantly better in patients without than in those with hematoma (p < 0.01). Twenty-seven patients with sylvian hematoma caused by ruptured MCA aneurysm often developed ipsilateral cerebral swelling in the early period after subarachnoid hemorrhage. Seventeen of these patients developed serious cerebral swelling and received barbiturate therapy. Nine of these 17 patients had good outcome, but six patients died of cerebral swelling. The incidence of hydrocephalus was significantly higher in patients with than in those without hematoma (p < 0.01). The incidence of infarction was more pronounced in patients with sylvian hematoma. Clinical outcome was significantly better in patients without than in those with sylvian hematoma (p < 0.01). Development of cerebral swelling in patients with sylvian hematoma due to ruptured MCA aneurysm has a significant effect on outcome, and improvements in management are required. (+info)Cerebral blood flow in the monkey after focal cryogenic injury. (5/1215)
A focal cryogenic lesion was made in the left superior frontal gyrus of the anesthetized macaque brain. Cerebral blood flow (CBF) was determined by the hydrogen clearance technique before and during the 4 hours following trauma. Local CBF in tissue adjacent to the lesion increased in the first half hour after the lesion was made and then decreased during the ensuing 3 1/2 hours. Local CBF in the contralateral superior frontal gyrus, as well as total CBF and oxygen consumption, were unchanged by cryogenic trauma. The spread of vasogenic edema into uninjured tissue probably accounts for the observed decrease in local CBF. This experimental model may assist in discovering therapy to alter favorably the spatial and temporal profile of pathologic CBF changes in tissue surrounding an acute lesion of the brain. (+info)Abrupt exacerbation of acute subdural hematoma mimicking benign acute epidural hematoma on computed tomography--case report. (6/1215)
A 75-year-old male was hit by a car, when riding a bicycle. The diagnosis of acute epidural hematoma was made based on computed tomography (CT) findings of lentiform hematoma in the left temporal region. On admission he had only moderate occipitalgia and amnesia of the accident, so conservative therapy was administered. Thirty-three hours later, he suddenly developed severe headache, vomiting, and anisocoria just after a positional change. CT revealed typical acute subdural hematoma (ASDH), which was confirmed by emergent decompressive craniectomy. He was vegetative postoperatively and died of pneumonia one month later. Emergent surgical exploration is recommended for this type of ASDH even if the symptoms are mild due to aged atrophic brain. (+info)Epilepsy after two different neurosurgical approaches to the treatment of ruptured intracranial aneurysm. (7/1215)
One-hundred-and-fifty-two patients who underwent surgery for intracranial aneurysm were studied to determine the incidence of postoperative epilepsy in relation to the site of the aneurysm and the type of surgical approach. The overall incidence of epilepsy was 22%. Of the 116 patients treated by the intracranial approach 27.5% developed epilepsy, in contrast with only 5% of the 36 patients who had carotid artery ligation in the neck. Epilepsy occurred most frequently (35%) with middle cerebral artery aneurysms, especially if moderate or severe operative trauma was sustained and there was postoperative dysphasia. (+info)Glial and capillary density of the pontine white matter in swelling and atrophy. (8/1215)
A total of 48 autopsied brains were morphometrically examined for the relation between pontine geometry and structural parameters. In each case, the numerical density of neuroglial cells Nv(G) and the linear density of capillary network Lv(C) of the pontine white matter were determined stereologically from the counts of glial nuclear profiles and capillary transections per constant area of a histological section with a constant thickness. It was revealed that in general the glial numerical density increased with advancing atrophy and decreased with increasing swelling, whereas the capillary linear density remained fairly constant especially in the advanced stage of brain swelling. Further analysis of this relation using a model of pontine geometry has made it clear that the total capillary length in the swollen white matter increases probably at the expense of the capillary caliber as swelling advances. The changes in the ratio Lv(C)/Nv(G) under pathological conditions are emphasized and possible utility of classifying stages of acute brain swelling from a viewpoint of microvascular dimension is suggested. (+info)The word "edema" comes from the Greek word "oidema", meaning swelling.
There are several types of edema, including:
1. Pitting edema: This type of edema occurs when the fluid accumulates in the tissues and leaves a pit or depression when it is pressed. It is commonly seen in the skin of the lower legs and feet.
2. Non-pitting edema: This type of edema does not leave a pit or depression when pressed. It is often seen in the face, hands, and arms.
3. Cytedema: This type of edema is caused by an accumulation of fluid in the tissues of the limbs, particularly in the hands and feet.
4. Edema nervorum: This type of edema affects the nerves and can cause pain, numbness, and tingling in the affected area.
5. Lymphedema: This is a condition where the lymphatic system is unable to properly drain fluid from the body, leading to swelling in the arms or legs.
Edema can be diagnosed through physical examination, medical history, and diagnostic tests such as imaging studies and blood tests. Treatment options for edema depend on the underlying cause, but may include medications, lifestyle changes, and compression garments. In some cases, surgery or other interventions may be necessary to remove excess fluid or tissue.
There are several different types of brain injuries that can occur, including:
1. Concussions: A concussion is a type of mild traumatic brain injury that occurs when the brain is jolted or shaken, often due to a blow to the head.
2. Contusions: A contusion is a bruise on the brain that can occur when the brain is struck by an object, such as during a car accident.
3. Coup-contrecoup injuries: This type of injury occurs when the brain is injured as a result of the force of the body striking another object, such as during a fall.
4. Penetrating injuries: A penetrating injury occurs when an object pierces the brain, such as during a gunshot wound or stab injury.
5. Blast injuries: This type of injury occurs when the brain is exposed to a sudden and explosive force, such as during a bombing.
The symptoms of brain injuries can vary depending on the severity of the injury and the location of the damage in the brain. Some common symptoms include:
* Headaches
* Dizziness or loss of balance
* Confusion or disorientation
* Memory loss or difficulty with concentration
* Slurred speech or difficulty with communication
* Vision problems, such as blurred vision or double vision
* Sleep disturbances
* Mood changes, such as irritability or depression
* Personality changes
* Difficulty with coordination and balance
In some cases, brain injuries can be treated with medication, physical therapy, and other forms of rehabilitation. However, in more severe cases, the damage may be permanent and long-lasting. It is important to seek medical attention immediately if symptoms persist or worsen over time.
Symptoms of pulmonary edema may include:
* Shortness of breath (dyspnea)
* Coughing up frothy sputum
* Chest pain or tightness
* Fatigue
* Confusion or disorientation
Pulmonary edema can be diagnosed through physical examination, chest x-rays, electrocardiogram (ECG), and blood tests. Treatment options include oxygen therapy, diuretics, and medications to manage underlying conditions such as heart failure or sepsis. In severe cases, hospitalization may be necessary to provide mechanical ventilation.
Prevention measures for pulmonary edema include managing underlying medical conditions, avoiding exposure to pollutants and allergens, and seeking prompt medical attention if symptoms persist or worsen over time.
In summary, pulmonary edema is a serious condition that can impair lung function and lead to shortness of breath, chest pain, and other respiratory symptoms. Prompt diagnosis and treatment are essential to prevent complications and improve outcomes for patients with this condition.
The term ischemia refers to the reduction of blood flow, and it is often used interchangeably with the term stroke. However, not all strokes are caused by ischemia, as some can be caused by other factors such as bleeding in the brain. Ischemic stroke accounts for about 87% of all strokes.
There are different types of brain ischemia, including:
1. Cerebral ischemia: This refers to the reduction of blood flow to the cerebrum, which is the largest part of the brain and responsible for higher cognitive functions such as thought, emotion, and voluntary movement.
2. Cerebellar ischemia: This refers to the reduction of blood flow to the cerebellum, which is responsible for coordinating and regulating movement, balance, and posture.
3. Brainstem ischemia: This refers to the reduction of blood flow to the brainstem, which is responsible for controlling many of the body's automatic functions such as breathing, heart rate, and blood pressure.
4. Territorial ischemia: This refers to the reduction of blood flow to a specific area of the brain, often caused by a blockage in a blood vessel.
5. Global ischemia: This refers to the reduction of blood flow to the entire brain, which can be caused by a cardiac arrest or other systemic conditions.
The symptoms of brain ischemia can vary depending on the location and severity of the condition, but may include:
1. Weakness or paralysis of the face, arm, or leg on one side of the body
2. Difficulty speaking or understanding speech
3. Sudden vision loss or double vision
4. Dizziness or loss of balance
5. Confusion or difficulty with memory
6. Seizures
7. Slurred speech or inability to speak
8. Numbness or tingling sensations in the face, arm, or leg
9. Vision changes, such as blurred vision or loss of peripheral vision
10. Difficulty with coordination and balance.
It is important to seek medical attention immediately if you experience any of these symptoms, as brain ischemia can cause permanent damage or death if left untreated.
Brain neoplasms can arise from various types of cells in the brain, including glial cells (such as astrocytes and oligodendrocytes), neurons, and vascular tissues. The symptoms of brain neoplasms vary depending on their size, location, and type, but may include headaches, seizures, weakness or numbness in the limbs, and changes in personality or cognitive function.
There are several different types of brain neoplasms, including:
1. Meningiomas: These are benign tumors that arise from the meninges, the thin layers of tissue that cover the brain and spinal cord.
2. Gliomas: These are malignant tumors that arise from glial cells in the brain. The most common type of glioma is a glioblastoma, which is aggressive and hard to treat.
3. Pineal parenchymal tumors: These are rare tumors that arise in the pineal gland, a small endocrine gland in the brain.
4. Craniopharyngiomas: These are benign tumors that arise from the epithelial cells of the pituitary gland and the hypothalamus.
5. Medulloblastomas: These are malignant tumors that arise in the cerebellum, specifically in the medulla oblongata. They are most common in children.
6. Acoustic neurinomas: These are benign tumors that arise on the nerve that connects the inner ear to the brain.
7. Oligodendrogliomas: These are malignant tumors that arise from oligodendrocytes, the cells that produce the fatty substance called myelin that insulates nerve fibers.
8. Lymphomas: These are cancers of the immune system that can arise in the brain and spinal cord. The most common type of lymphoma in the CNS is primary central nervous system (CNS) lymphoma, which is usually a type of B-cell non-Hodgkin lymphoma.
9. Metastatic tumors: These are tumors that have spread to the brain from another part of the body. The most common types of metastatic tumors in the CNS are breast cancer, lung cancer, and melanoma.
These are just a few examples of the many types of brain and spinal cord tumors that can occur. Each type of tumor has its own unique characteristics, such as its location, size, growth rate, and biological behavior. These factors can help doctors determine the best course of treatment for each patient.
Infarction Middle Cerebral Artery (MCA) is a type of ischemic stroke that occurs when there is an obstruction in the middle cerebral artery. This artery supplies blood to the temporal lobe of the brain, which controls many important functions such as memory, language, and spatial reasoning. When this artery becomes blocked or ruptured, it can cause a lack of blood supply to the affected areas resulting in tissue death (infarction).
The symptoms of an MCA infarction can vary depending on the location and severity of the blockage. Some common symptoms include weakness or paralysis on one side of the body, difficulty with speech and language, memory loss, confusion, vision problems, and difficulty with coordination and balance. Patients may also experience sudden severe headache, nausea, vomiting, and fever.
The diagnosis of MCA infarction is based on a combination of clinical examination, imaging studies such as CT or MRI scans, and laboratory tests. Imaging studies can help to identify the location and severity of the blockage, while laboratory tests may be used to rule out other conditions that may cause similar symptoms.
Treatment for MCA infarction depends on the underlying cause of the blockage or rupture. In some cases, medications such as thrombolytics may be given to dissolve blood clots and restore blood flow to the affected areas. Surgery may also be required to remove any blockages or repair damaged blood vessels. Other interventions such as endovascular procedures or brain bypass surgery may also be used to restore blood flow.
In summary, middle cerebral artery infarction is a type of stroke that occurs when the blood supply to the brain is blocked or interrupted, leading to damage to the brain tissue. It can cause a range of symptoms including weakness or paralysis on one side of the body, difficulty with speech and language, memory loss, confusion, vision problems, and difficulty with coordination and balance. The diagnosis is based on a combination of clinical examination, imaging studies, and laboratory tests. Treatment options include medications, surgery, endovascular procedures, or brain bypass surgery.
Symptoms of cerebral hemorrhage may include sudden severe headache, confusion, seizures, weakness or numbness in the face or limbs, and loss of consciousness. The condition is diagnosed through a combination of physical examination, imaging tests such as CT or MRI scans, and laboratory tests to determine the cause of the bleeding.
Treatment for cerebral hemorrhage depends on the location and severity of the bleeding, as well as the underlying cause. Medications may be used to control symptoms such as high blood pressure or seizures, while surgery may be necessary to repair the ruptured blood vessel or relieve pressure on the brain. In some cases, the condition may be fatal, and immediate medical attention is essential to prevent long-term damage or death.
Some of the most common complications associated with cerebral hemorrhage include:
1. Rebleeding: There is a risk of rebleeding after the initial hemorrhage, which can lead to further brain damage and increased risk of death.
2. Hydrocephalus: Excess cerebrospinal fluid can accumulate in the brain, leading to increased intracranial pressure and potentially life-threatening complications.
3. Brain edema: Swelling of the brain tissue can occur due to the bleeding, leading to increased intracranial pressure and potentially life-threatening complications.
4. Seizures: Cerebral hemorrhage can cause seizures, which can be a sign of a more severe injury.
5. Cognitive and motor deficits: Depending on the location and severity of the bleeding, cerebral hemorrhage can result in long-term cognitive and motor deficits.
6. Vision loss: Cerebral hemorrhage can cause vision loss or blindness due to damage to the visual cortex.
7. Communication difficulties: Cerebral hemorrhage can cause difficulty with speech and language processing, leading to communication difficulties.
8. Behavioral changes: Depending on the location and severity of the bleeding, cerebral hemorrhage can result in behavioral changes, such as irritability, agitation, or apathy.
9. Infection: Cerebral hemorrhage can increase the risk of infection, particularly if the hemorrhage is caused by a ruptured aneurysm or arteriovenous malformation (AVM).
10. Death: Cerebral hemorrhage can be fatal, particularly if the bleeding is severe or if there are underlying medical conditions that compromise the patient's ability to tolerate the injury.
Types: There are several types of brain infarction, including:
1. Cerebral infarction: This type of infarction occurs when there is a blockage or obstruction in the blood vessels that supply the cerebrum, which is the largest part of the brain.
2. Cerebellar infarction: This type of infarction occurs when there is a blockage or obstruction in the blood vessels that supply the cerebellum, which is located at the base of the brain.
3. Brain stem infarction: This type of infarction occurs when there is a blockage or obstruction in the blood vessels that supply the brain stem, which is the part of the brain that controls vital functions such as breathing, heart rate, and blood pressure.
Symptoms: The symptoms of brain infarction can vary depending on the location and size of the affected area, but common symptoms include:
1. Sudden weakness or numbness in the face, arm, or leg
2. Sudden confusion or trouble speaking or understanding speech
3. Sudden difficulty seeing or blindness
4. Sudden difficulty walking or loss of balance
5. Sudden severe headache
6. Difficulty with coordination and movement
7. Slurred speech
8. Vision changes
9. Seizures
Diagnosis: Brain infarction is typically diagnosed using a combination of physical examination, medical history, and imaging tests such as CT or MRI scans. Other diagnostic tests may include blood tests to check for signs of infection or blood clotting abnormalities, and an electroencephalogram (EEG) to measure the electrical activity of the brain.
Treatment: The treatment of brain infarction depends on the underlying cause, but common treatments include:
1. Medications: To control symptoms such as high blood pressure, seizures, and swelling in the brain.
2. Endovascular therapy: A minimally invasive procedure to open or remove blockages in the blood vessels.
3. Surgery: To relieve pressure on the brain or repair damaged blood vessels.
4. Rehabilitation: To help regain lost function and improve quality of life.
Prognosis: The prognosis for brain infarction depends on the location and size of the affected area, as well as the promptness and effectiveness of treatment. In general, the earlier treatment is received, the better the outcome. However, some individuals may experience long-term or permanent disability, or even death.
1) They share similarities with humans: Many animal species share similar biological and physiological characteristics with humans, making them useful for studying human diseases. For example, mice and rats are often used to study diseases such as diabetes, heart disease, and cancer because they have similar metabolic and cardiovascular systems to humans.
2) They can be genetically manipulated: Animal disease models can be genetically engineered to develop specific diseases or to model human genetic disorders. This allows researchers to study the progression of the disease and test potential treatments in a controlled environment.
3) They can be used to test drugs and therapies: Before new drugs or therapies are tested in humans, they are often first tested in animal models of disease. This allows researchers to assess the safety and efficacy of the treatment before moving on to human clinical trials.
4) They can provide insights into disease mechanisms: Studying disease models in animals can provide valuable insights into the underlying mechanisms of a particular disease. This information can then be used to develop new treatments or improve existing ones.
5) Reduces the need for human testing: Using animal disease models reduces the need for human testing, which can be time-consuming, expensive, and ethically challenging. However, it is important to note that animal models are not perfect substitutes for human subjects, and results obtained from animal studies may not always translate to humans.
6) They can be used to study infectious diseases: Animal disease models can be used to study infectious diseases such as HIV, TB, and malaria. These models allow researchers to understand how the disease is transmitted, how it progresses, and how it responds to treatment.
7) They can be used to study complex diseases: Animal disease models can be used to study complex diseases such as cancer, diabetes, and heart disease. These models allow researchers to understand the underlying mechanisms of the disease and test potential treatments.
8) They are cost-effective: Animal disease models are often less expensive than human clinical trials, making them a cost-effective way to conduct research.
9) They can be used to study drug delivery: Animal disease models can be used to study drug delivery and pharmacokinetics, which is important for developing new drugs and drug delivery systems.
10) They can be used to study aging: Animal disease models can be used to study the aging process and age-related diseases such as Alzheimer's and Parkinson's. This allows researchers to understand how aging contributes to disease and develop potential treatments.
Symptoms of intracranial hypertension can include headache, nausea and vomiting, confusion, seizures, and loss of consciousness. Treatment options depend on the underlying cause, but may include medications to reduce pressure, draining excess CSF, or surgery to relieve obstruction.
Intracranial hypertension can be life-threatening if left untreated, as it can lead to permanent brain damage and even death. Therefore, prompt medical attention is essential for proper diagnosis and management of this condition.
Causes:
1. Viral hepatitis (hepatitis A, B, or C)
2. Overdose of medications or supplements
3. Toxic substances (e.g., alcohol, drugs, or chemicals)
4. Sepsis or other infections that spread to the liver
5. Certain autoimmune disorders (e.g., hemochromatosis, Wilson's disease)
6. Cancer that has metastasized to the liver
7. Blood vessel blockage or clotting in the liver
8. Lack of blood flow to the liver
Symptoms:
1. Jaundice (yellowing of skin and eyes)
2. Nausea and vomiting
3. Abdominal swelling and discomfort
4. Fatigue, weakness, and loss of appetite
5. Confusion or altered mental state
6. Seizures or coma
7. Pale or clay-colored stools
8. Dark urine
Diagnosis:
1. Physical examination and medical history
2. Laboratory tests (e.g., liver function tests, blood tests, imaging studies)
3. Biopsy of the liver tissue (to rule out other liver diseases)
Treatment:
1. Supportive care (fluids, nutrition, and medication to manage symptoms)
2. Addressing underlying causes (e.g., stopping alcohol or drug use, treating infections)
3. Transjugular intrahepatic portosystemic shunt (TIPS), a procedure that creates a new pathway for blood to flow through the liver
4. Liver transplantation (in severe cases where other treatments have failed)
Prognosis:
The prognosis for acute liver failure depends on the underlying cause of the condition and the severity of the liver damage. In general, the earlier the diagnosis and treatment, the better the outcome. However, acute liver failure can be a life-threatening condition, and the mortality rate is high, especially in cases where there is severe liver damage or no available donor liver for transplantation.
Some common symptoms of corneal edema include:
* Blurred vision
* Haziness or clouding of the cornea
* Increased sensitivity to light
* Redness or discharge in the eye
* Pain or discomfort in the eye
Corneal edema can be diagnosed through a comprehensive eye exam, which may include a visual acuity test, dilated eye exam, and imaging tests such as cornea scans or ultrasound. Treatment for corneal edema depends on the underlying cause and may involve antibiotics, anti-inflammatory medications, or other therapies to reduce swelling and promote healing. In some cases, surgery may be necessary to remove scar tissue or improve drainage of fluid from the eye.
If left untreated, corneal edema can lead to more serious complications such as corneal ulcers or vision loss. Therefore, it is important to seek medical attention if you experience any symptoms of corneal edema to prevent any further damage and ensure proper treatment.
A condition characterized by swelling of the heart and surrounding tissues due to an accumulation of fluid. This can occur as a result of various conditions such as heart failure, coronary artery disease, or cardiomyopathy. The swelling can lead to symptoms such as shortness of breath, fatigue, and difficulty exercising. Treatment options include diuretics, ACE inhibitors, and other medications to manage the underlying condition causing the edema.
Example sentence: "The patient was diagnosed with cardiac edema and started on a regimen of diuretics and ACE inhibitors to help reduce the swelling and improve their symptoms."
Cerebral infarction can result in a range of symptoms, including sudden weakness or numbness in the face, arm, or leg on one side of the body, difficulty speaking or understanding speech, sudden vision loss, dizziness, and confusion. Depending on the location and severity of the infarction, it can lead to long-term disability or even death.
There are several types of cerebral infarction, including:
1. Ischemic stroke: This is the most common type of cerebral infarction, accounting for around 87% of all cases. It occurs when a blood clot blocks the flow of blood to the brain, leading to cell death and tissue damage.
2. Hemorrhagic stroke: This type of cerebral infarction occurs when a blood vessel in the brain ruptures, leading to bleeding and cell death.
3. Lacunar infarction: This type of cerebral infarction affects the deep structures of the brain, particularly the basal ganglia, and is often caused by small blockages or stenosis (narrowing) in the blood vessels.
4. Territorial infarction: This type of cerebral infarction occurs when there is a complete blockage of a blood vessel that supplies a specific area of the brain, leading to cell death and tissue damage in that area.
Diagnosis of cerebral infarction typically involves a combination of physical examination, medical history, and imaging tests such as CT or MRI scans. Treatment options vary depending on the cause and location of the infarction, but may include medication to dissolve blood clots, surgery to remove blockages, or supportive care to manage symptoms and prevent complications.
Meningioma can occur in various locations within the brain, including the cerebrum, cerebellum, brainstem, and spinal cord. The most common type of meningioma is the meningothelial meningioma, which arises from the arachnoid membrane, one of the three layers of the meninges. Other types of meningioma include the dural-based meningioma, which originates from the dura mater, and the fibrous-cap meningioma, which is characterized by a fibrous cap covering the tumor.
The symptoms of meningioma can vary depending on the location and size of the tumor, but they often include headaches, seizures, weakness or numbness in the arms or legs, and changes in vision, memory, or cognitive function. As the tumor grows, it can compress the brain tissue and cause damage to the surrounding structures, leading to more severe symptoms such as difficulty speaking, walking, or controlling movement.
The diagnosis of meningioma typically involves a combination of imaging studies such as MRI or CT scans, and tissue sampling through biopsy or surgery. Treatment options for meningioma depend on the size, location, and aggressiveness of the tumor, but may include surgery, radiation therapy, and chemotherapy. Overall, the prognosis for meningioma is generally good, with many patients experiencing a good outcome after treatment. However, some types of meningioma can be more aggressive and difficult to treat, and the tumor may recur in some cases.
The symptoms of water intoxication can vary depending on the severity of the condition, but may include:
* Headache
* Nausea and vomiting
* Seizures
* Confusion and disorientation
* Slurred speech
* Weakness or fatigue
* Increased heart rate
* Low blood pressure
In severe cases, water intoxication can cause seizures, coma, and even death.
The diagnosis of water intoxication is based on a combination of symptoms, medical history, and laboratory tests, such as blood tests to measure electrolyte levels. Treatment typically involves cessation of fluid intake, administration of electrolytes, and monitoring of vital signs. In severe cases, hospitalization may be necessary to monitor and treat the condition.
Prevention of water intoxication is key, and this can be achieved by drinking fluids in moderation and avoiding excessive intake during physical activity or in hot weather. It is also important to monitor fluid intake in individuals who are at risk, such as endurance athletes or those with certain medical conditions.
In summary, water intoxication is a serious condition that can occur when a person consumes too much water, leading to an imbalance of electrolytes in the body. It is important to be aware of the symptoms and seek medical attention if they occur, as prompt treatment can help prevent complications and death.
This condition is most commonly seen in people with advanced liver disease, such as cirrhosis or liver cancer. It can also be caused by other conditions that affect the liver, such as hepatitis or portal hypertension.
Symptoms of hepatic encephalopathy can include confusion, disorientation, slurred speech, memory loss, and difficulty with coordination and balance. In severe cases, it can lead to coma or even death.
Diagnosis of hepatic encephalopathy is typically made through a combination of physical examination, medical history, and diagnostic tests such as blood tests and imaging studies. Treatment options include medications to reduce the production of ammonia in the gut, antibiotics to treat any underlying infections, and transjugular intrahepatic portosystemic shunt (TIPS) to improve liver function. In severe cases, a liver transplant may be necessary.
Overall, hepatic encephalopathy is a serious condition that can have significant impact on quality of life and survival in people with advanced liver disease. Early detection and prompt treatment are essential to prevent complications and improve outcomes.
Causes of Hyperammonemia:
1. Liver disease or failure: The liver is responsible for filtering out ammonia, so if it is not functioning properly, ammonia levels can rise.
2. Urea cycle disorders: These are genetic conditions that affect the body's ability to break down protein and produce urea. As a result, ammonia can build up in the bloodstream.
3. Inborn errors of metabolism: Certain inherited disorders can lead to hyperammonemia by affecting the body's ability to process ammonia.
4. Sepsis: Severe infections can cause inflammation in the body, which can lead to hyperammonemia.
5. Kidney disease or failure: If the kidneys are not functioning properly, they may be unable to remove excess ammonia from the bloodstream, leading to hyperammonemia.
Symptoms of Hyperammonemia:
1. Lethargy and confusion
2. Seizures
3. Coma
4. Vomiting
5. Diarrhea
6. Decreased appetite
7. Weight loss
8. Fatigue
9. Headache
10. Nausea and vomiting
Diagnosis of Hyperammonemia:
1. Blood tests: Measurement of ammonia levels in the blood is the most common method used to diagnose hyperammonemia.
2. Urine tests: Measurement of urea levels in the urine can help determine if the body is able to produce and excrete urea normally.
3. Imaging tests: Imaging tests such as CT or MRI scans may be ordered to look for any underlying liver or kidney damage.
4. Genetic testing: If the cause of hyperammonemia is suspected to be a genetic disorder, genetic testing may be ordered to confirm the diagnosis.
Treatment of Hyperammonemia:
1. Dietary changes: A low-protein diet and avoiding high-aminogram foods can help reduce ammonia production in the body.
2. Medications: Medications such as sodium benzoate, sodium phenylbutyrate, and ribavirin may be used to reduce ammonia production or increase urea production.
3. Dialysis: In severe cases of hyperammonemia, dialysis may be necessary to remove excess ammonia from the blood.
4. Liver transplantation: In cases where the cause of hyperammonemia is liver disease, a liver transplant may be necessary.
5. Nutritional support: Providing adequate nutrition and hydration can help support the body's metabolic processes and prevent complications of hyperammonemia.
Complications of Hyperammonemia:
1. Brain damage: Prolonged elevated ammonia levels in the blood can cause brain damage, leading to cognitive impairment, seizures, and coma.
2. Respiratory failure: Severe hyperammonemia can lead to respiratory failure, which can be life-threatening.
3. Cardiac complications: Hyperammonemia can cause cardiac complications such as arrhythmias and heart failure.
4. Kidney damage: Prolonged elevated ammonia levels in the blood can cause kidney damage and failure.
5. Infections: People with hyperammonemia may be more susceptible to infections due to impaired immune function.
In conclusion, hyperammonemia is a serious condition that can have severe consequences if left untreated. It is essential to identify the underlying cause of hyperammonemia and provide appropriate treatment to prevent complications. Early detection and management of hyperammonemia can improve outcomes and reduce the risk of long-term sequelae.
Example sentence: "The patient experienced a transient ischemic attack, which was caused by a temporary blockage in one of the blood vessels in their brain."
Synonyms: TIA, mini-stroke.
Edema disease is caused by a picornavirus and is characterized by fever, lethargy, loss of appetite, and difficulty breathing due to severe inflammation of the respiratory tract. The skin becomes edematous (swollen) and hemorrhages occur under the skin and in internal organs such as the lungs and liver. Death can occur within 24 to 48 hours after the onset of symptoms.
The disease is transmitted through direct contact with infected swine or contaminated fomites, and the virus can survive for several days in the environment. The incubation period is typically 3-7 days, and infected animals may not show any clinical signs until they are heavily infected.
There is no specific treatment or vaccine available for edema disease, and control measures are focused on preventing the spread of the virus through proper sanitation, biosecurity measures, and culling of infected animals. The disease is considered highly contagious and can have a significant impact on swine populations if not controlled promptly.
The symptoms of meningeal neoplasms vary depending on the location, size, and type of tumor. Common symptoms include headaches, seizures, weakness or numbness in the arms or legs, and changes in vision, memory, or behavior. As the tumor grows, it can compress or displaces the brain tissue, leading to increased intracranial pressure and potentially life-threatening complications.
There are several different types of meningeal neoplasms, including:
1. Meningioma: This is the most common type of meningeal neoplasm, accounting for about 75% of all cases. Meningiomas are usually benign and grow slowly, but they can sometimes be malignant.
2. Metastatic tumors: These are tumors that have spread to the meninges from another part of the body, such as the lung or breast.
3. Lymphoma: This is a type of cancer that affects the immune system and can spread to the meninges.
4. Melanotic neuroectodermal tumors (MNTs): These are rare, malignant tumors that usually occur in children and young adults.
5. Hemangiopericytic hyperplasia: This is a rare, benign condition characterized by an overgrowth of blood vessels in the meninges.
The diagnosis of meningeal neoplasms is based on a combination of clinical symptoms, physical examination findings, and imaging studies such as CT or MRI scans. A biopsy may be performed to confirm the diagnosis and determine the type of tumor.
Treatment options for meningeal neoplasms depend on the type, size, and location of the tumor, as well as the patient's overall health. Surgery is often the first line of treatment, and may involve removing as much of the tumor as possible or using a laser to ablate (destroy) the tumor cells. Radiation therapy and chemotherapy may also be used in combination with surgery to treat malignant meningeal neoplasms.
Prognosis for meningeal neoplasms varies depending on the type of tumor and the patient's overall health. In general, early diagnosis and treatment improve the prognosis, while later-stage tumors may have a poorer outcome.
1. Atrial fibrillation (a type of irregular heartbeat)
2. Heart disease or valve problems
3. Blood clots in the legs or lungs
4. Infective endocarditis (an infection of the heart valves)
5. Cancer and its treatment
6. Trauma to the head or neck
7. High blood pressure
8. Atherosclerosis (the buildup of plaque in the arteries)
When a blockage occurs in one of the blood vessels of the brain, it can deprive the brain of oxygen and nutrients, leading to cell death and potentially causing a range of symptoms including:
1. Sudden weakness or numbness in the face, arm, or leg
2. Sudden confusion or trouble speaking or understanding speech
3. Sudden trouble seeing in one or both eyes
4. Sudden severe headache
5. Dizziness or loss of balance
6. Fainting or falling
Intracranial embolism and thrombosis can be diagnosed through a variety of imaging tests, including:
1. Computed tomography (CT) scan
2. Magnetic resonance imaging (MRI)
3. Magnetic resonance angiography (MRA)
4. Cerebral angiography
5. Doppler ultrasound
Treatment options for intracranial embolism and thrombosis depend on the underlying cause of the blockage, but may include:
1. Medications to dissolve blood clots or prevent further clotting
2. Surgery to remove the blockage or repair the affected blood vessel
3. Endovascular procedures, such as angioplasty and stenting, to open up narrowed or blocked blood vessels
4. Supportive care, such as oxygen therapy and pain management, to help manage symptoms and prevent complications.
The symptoms of a brain abscess can vary depending on the location and size of the abscess, but may include:
* Headache
* Fever
* Confusion or disorientation
* Seizures
* Weakness or numbness in the arms or legs
* Vision problems
* Speech difficulties
If a brain abscess is suspected, a doctor will typically perform a physical examination and order imaging tests such as CT or MRI scans to confirm the diagnosis. Treatment usually involves antibiotics to treat the underlying infection, as well as surgery to drain the abscess and remove any infected tissue. In severe cases, hospitalization may be necessary to monitor and treat the patient.
With prompt and appropriate treatment, most people with a brain abscess can recover fully or almost fully, but in some cases, the condition can result in long-term complications such as memory loss, cognitive impairment, or personality changes. In rare instances, a brain abscess can be fatal if not treated promptly and properly.
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Cerebral16
- He died of intractable cerebral edema on day 17 of illness, 10 days after the onset of neurologic symptoms. (cdc.gov)
- Computed tomography scan of the brain showed diffuse cerebral edema. (cdc.gov)
- The ameba migrates from the nose to the brain along the olfactory nerve through the cribriform plate, destroys brain tissue, and causes cerebral edema. (cdc.gov)
- Ischemic stroke is sudden neurologic deficits that result from focal cerebral ischemia associated with permanent brain infarction (eg, positive results on diffusion-weighted MRI). (msdmanuals.com)
- Partial or complete thrombotic occlusion occurs most often at the main trunk of the middle cerebral artery and its branches but is also common in the large arteries at the base of the brain, in deep perforating arteries, and in small cortical branches. (msdmanuals.com)
- Acute exacerbations of multiple sclerosis, cerebral edema associated with primary or metastatic brain tumor, craniotomy, or head injury. (nih.gov)
- Computerized tomography scan of the brain showed no cerebral edema or evidence of bleeding. (cdc.gov)
- On autopsy, gross examination revealed massive cerebral edema with uncal herniation and intracytoplasmic inclusions in the brain and spinal cord. (cdc.gov)
- 1,2 When a cerebral vein is occluded, retrograde pressure prevents the drainage of local brain capillaries, inhibiting perfusion and causing infarction. (appliedradiology.com)
- Cerebral ischemia-reperfusion injury in rats - A 3T MRI study on biphasic blood-brain barrier opening and the dynamics of edema formation. (mpg.de)
- Occurrence of hepatic encephalopathy or coma in ALF is a poor prognostic sign and is associated with the development of cerebral edema, intracranial hypertension and subsequent mortality from brain herniation [ 7 ]. (intechopen.com)
- Though advances in the care of the patient with ALF have led to both a decrease in the incidence and associated mortality of persons developing cerebral edema and intracranial hypertension [ 7 ], careful vigilance should be exercised because development and progression of encephalopathy can be rapid and fatal. (intechopen.com)
- Aumento de los líquidos intra o extracelulares en el tejido cerebral. (bvsalud.org)
- El edema cerebral citotóxico (hinchazón debida al aumento del líquido intracelular) indica un trastorno del metabolismo celular, y se asocia comúnmente con lesiones hipóxicas o isquémicas (ver HIPOXIA CEREBRAL). (bvsalud.org)
- El aumento del líquido extracelular puede ser producido por un aumento de la permeabilidad capilar cerebral (edema vasogénico), gradiente osmótico, bloqueo local en las vías de los líquidos intersticiales, o por obstrucción del flujo del LCR (es decir, HIDROCEFALIA obstructiva). (bvsalud.org)
- Computed tomography (CT) brain was suggestive of cerebral edema and the CT chest was suggestive of diffuse opacities in bilateral lung fields. (who.int)
Vasogenic edema2
- An increase in extracellular fluid may be caused by increased brain capillary permeability (vasogenic edema), an osmotic gradient, local blockages in interstitial fluid pathways, or by obstruction of CSF flow (e.g., obstructive HYDROCEPHALUS). (bvsalud.org)
- Patients with primary brain tumours often develop vasogenic edema and increased intracranial pressure. (bvsalud.org)
Diffuse brain edema2
- hemorrhages or diffuse brain edema. (cdc.gov)
- These patients show signs of acute encephalitis and diffuse brain edema. (medscape.com)
Cerebrospinal fluid1
- Diffuse scalp edema was noted, but the brain parenchyma, cerebrospinal fluid spaces, and bony structures appeared normal. (appliedradiology.com)
Tissue8
- M. pneumoniae antigens were demonstrated by immunohistochemical analysis of brain tissue, indicating neuroinvasion as an additional pathogenetic mechanism in central neurologic complications of M. pneumoniae infection. (cdc.gov)
- Degeneration of cysts may be associated with a strong host inflammatory response in the brain tissue, which can produce signs and symptoms of encephalitis. (medscape.com)
- At the Washington State Department of Health Public Health Laboratories a specimen of brain tissue obtained at autopsy also was positive by DFA, and rabies virus was isolated by mouse inoculation. (cdc.gov)
- Analysis at CDC also included viral isolation from sputum obtained on March 14 and a positive DFA and nucleotide sequence analysis result from brain tissue obtained at autopsy. (cdc.gov)
- How the brain tissue shapes the electric field induced by transcranial magnetic stimulation. (mpg.de)
- and iv) additional swelling of the brain tissue, leading to IHT, which adds even more damage to the brain tissue ( 11 , 12 ). (spandidos-publications.com)
- Increased intracellular or extracellular fluid in brain tissue. (bvsalud.org)
- Infection in the brain or the tissue that surrounds the brain , as well as a brain abscess . (medlineplus.gov)
Acute3
- Any acute central nervous system (CNS) insult can result in pulmonary edema. (medscape.com)
- Acute severe hyponatremia may lead to brain edema with neurologic consequences and be lethal if not promptly diagnosed and treated. (oregonstate.edu)
- Brain swelling (brain edema) from altitude sickness, carbon monoxide poisoning, or acute brain injury. (medlineplus.gov)
Intracellular1
- Cytotoxic brain edema (swelling due to increased intracellular fluid) is indicative of a disturbance in cell metabolism, and is commonly associated with hypoxic or ischemic injuries (see HYPOXIA, BRAIN). (bvsalud.org)
Basal ganglia2
- a brain magnetic resonance imaging study was consistent with basal ganglia injury. (cdc.gov)
- After excluding carbon monoxide and heavy metal poisoning, anoxic brain injury, and metabolic disorders, the treating physicians attributed his neurologic findings to sulfuryl fluoride poisoning, manifested by basal ganglia necrosis. (cdc.gov)
Intracranial pressure1
- however, in patients with ongoing brain injury and elevated intracranial pressure (ICP), the NPE often persists. (medscape.com)
Neurologic3
- Often, the term brain injury is used synonymously with head injury, which may not be associated with neurologic deficits. (medscape.com)
- Neurogenic pulmonary edema most commonly develops within a few hours after a neurologic insult, and is characterized by dyspnea, bilateral basal pulmonary crackles, and the absence of cardiac failure. (medscape.com)
- Neurogenic pulmonary edema trigger zones may exist in these structures, with specific neurologic foci or centers producing massive sympathetic discharges that lead to neurogenic pulmonary edema. (medscape.com)
20201
- For example, allergy medications may be taken to relieve the swelling from allergies, blood thinners may be prescribed to treat blood clot in the leg which led to edema, diuretics may treat leg edema due to liver disease or congestive heart failure, and limiting sodium intake may also help (Hoffman, 2020). (differencebetween.net)
Symptoms3
- 0 No symptoms at all avoiding more brain injury [29, 30]. (who.int)
- The symptoms of edema depend on the affected area, the amount of swelling, and the cause. (differencebetween.net)
- VALUE RFVF 10050 ='Chills' 10100 ='Fever' 10120 ='Other symptoms of body temperature' 10121 ='Feeling cold' 10122 ='Feeling hot' 10123 ='Feeling hot and cold' 10150 ='Tiredness, exhaustion' 10200 ='General weakness' 10250 ='General ill feeling' 10300 ='Fainting (syncope)' 10350 ='Symptoms of fluid abnormalities' 10351 ='Edema' 10352 ='Excessive sweating, perspiration' 10353 ='Excessive thirst' 10400 ='Weight gain' 10450 ='Weight loss' 10451 ='Recent weight loss' 10452 ='Underweight' 10460 ='Symptoms of face, not elsewhere class. (cdc.gov)
Hematoma4
- Volumetric electromagnetic phase-shift spectroscopy of brain edema and hematoma. (bvsalud.org)
- We describe the technology and report results from a limited pilot study with 46 healthy volunteers and eight patients with CT radiology confirmed brain edema and brain hematoma . (bvsalud.org)
- brain edema and those with a hematoma in the brain . (bvsalud.org)
- scalp incision reaching or crossing the Brain manipulations during hematoma midline with the craniotomy flap diameter evacuation could be more hazardous for the about 15 cm centered on the hematoma core. (who.int)
Interstitial1
- Neurogenic pulmonary edema (NPE) is a relatively rare form of pulmonary edema caused by an increase in pulmonary interstitial and alveolar fluid. (medscape.com)
Pulmonary7
- No specific laboratory study confirms the diagnosis of neurogenic pulmonary edema (NPE). (medscape.com)
- The pathogenesis of neurogenic pulmonary edema (NPE) is not completely understood. (medscape.com)
- Within the central nervous system, the sites responsible for the development of neurogenic pulmonary edema are not fully elucidated. (medscape.com)
- Experimentally, bilateral lesions of the nuclei in the medulla produce profound pulmonary and systemic hypertension and pulmonary edema. (medscape.com)
- Alpha-adrenergic blockade (with phentolamine) and spinal cord transection at the C7 level prevent the formation of neurogenic pulmonary edema, suggesting an important role for sympathetic activation. (medscape.com)
- Alterations in pulmonary vascular pressures appear to be the most likely Starling force to influence the formation of neurogenic pulmonary edema. (medscape.com)
- People with pulmonary edema experience shortness of breath and low oxygen levels. (differencebetween.net)
Traumatic5
- Traumatic brain injury (TBI) is a nondegenerative, noncongenital insult to the brain from an external mechanical force, possibly leading to permanent or temporary impairment of cognitive, physical, and psychosocial functions, with an associated diminished or altered state of consciousness. (medscape.com)
- Inconsistency in the definition and classification of traumatic brain injury (TBI), along with discrepancies in data collection, has made the epidemiology of TBI difficult to describe accurately. (medscape.com)
- When our law firm represents traumatic brain injury (TBI) victims, we immediately start working on a strategy for obtaining the best possible compensation package for our client. (pritzkerlaw.com)
- How Much is My Traumatic Brain Injury Case Worth? (pritzkerlaw.com)
- The husband suffered severe traumatic brain injury. (pritzkerlaw.com)
Herniation1
- Reported prognostic factors for poor short-term outcome in CVST include anatomical characteristics of disease such as the presence of brain herniation and hemorrhage, or features of the clinical presentation such as seizures, depressed consciousness, and altered mental status. (cdc.gov)
Parenchyma1
- Contrast-enhanced brain MRI provides detailed information about the brain parenchyma and is probably more accurate for diagnosing CVT than non-contrast-enhanced magnetic resonance venography sequences. (lww.com)
Buildup1
- Specifically, ascites is fluid buildup in the abdomen while edema is simply the medical term for swelling Their distinctions are discussed below. (differencebetween.net)
Inflammatory1
- A computed tomographic scan on day 15 showed brain edema and multiple inflammatory/demyelination lesions in the subcortical white matter of both hemispheres and within the brain thalami, capsulae internae, midbrain, and pons. (cdc.gov)
Abnormal1
- Abnormal connection between the arteries and veins in the brain that usually forms before birth. (medlineplus.gov)
Injury5
- Role of nitric oxide in blood-brain barrier permeability, brain edema and cell damage following hyperthermic brain injury. (nih.gov)
- Exposure of rats to heat stress at 38 degrees C for 4 h resulted in marked upregulation of constitutive NOS (cNOS) and a mild but significant expression of inducible NOS (iNOS) in several brain regions exhibiting leakage of the blood-brain barrier (BBB), brain edema formation and cell injury. (nih.gov)
- Pretreatment with the potent antioxidative compound EGB-761 or its constituent, Ginkgolide B significantly attenuated upregulation of cNOS and iNOS in the brain and also reduced the BBB permeability disturbances, brain edema and cell injury. (nih.gov)
- Our observations strongly suggest that (i) EGB-761 and Ginkgolide B pretreatment offer significant neuroprotection in hyperthermic brain injury, (ii) upregulation of cNOS and iNOS are injurious to the cell and, (iii) oxidative stress plays an important role in NOS expression and cell injury. (nih.gov)
- While this is a small-scale pilot study , it illustrates the potential of VEPS to change the paradigm of medical diagnostic of brain injury through a VEPS classifier-based technology . (bvsalud.org)
Infarction2
- Magnetic resonance imaging (MRI) of the brain (Figure 2) 3 days later showed an area of infarction in the left posterior parietal lobe and extending inferiorly into the left periventricular white matter, with adjacent foci of susceptibility effect suspected to be thrombosed cortical veins. (appliedradiology.com)
- Infants with congenital heart disease are also at increased risk for brain infarction caused by arterial emboli. (appliedradiology.com)
Patients2
- undertaken from July 15th, 2017 to March 15th, 2018.The multicentrique de cohorte prospective a inclus des Glasgow Coma Scale helped to determine the severety of the patients consécutifs admis en phase aiguë d AVC, disease at admission. (who.int)
- More than half of critically des patients avec AVC en phase aiguë présentent ill patients exhibit admission hyperglycemia with age, severity of stroke and known diabetes as its main associated principaux facteurs de risque a risk factors. (who.int)
Stroke2
- For patient education resources, see the Brain and Nervous System Center and Stroke . (medscape.com)
- During stroke the blood-brain barrier (BBB) is damaged which can result in vasogenic brain edema and inflammation. (uni-wuerzburg.de)
Blood9
- L-Theanine works by crossing the blood brain barrier and increasing dopamine and GABA levels in the brain. (vitanetonline.com)
- It receives blood from a vein in the nasal cavity, runs backwards, and gradually increases in size as blood drains from veins of the brain and the DURA MATER. (bvs.br)
- The reduced blood supply leads to decreased delivery of oxygen and glucose to affected areas of the brain. (uni-wuerzburg.de)
- Edema has many causes such as varicose veins, too much salt intake, prolonged sitting or standing, and side effects of medications (i.e., those prescribed for diabetes, high blood pressure, inflammation, and pain). (differencebetween.net)
- As for the leg edema due to heart disease, the swelling may lead to an added 10 or 20 pounds which can disrupt blood flow and cause skin ulcers. (differencebetween.net)
- Problems with blood vessels and bleeding in the brain can cause a headache. (medlineplus.gov)
- Blood flow to part of the brain stops. (medlineplus.gov)
- Weakening of the wall of a blood vessel that can break open and bleed into the brain. (medlineplus.gov)
- Clots in the veins prevent blood flow from leaving the brain (sinus thrombosis). (medlineplus.gov)
Upregulation1
- Oxygen and glucose deprivation (OGD) can cause upregulation of glucose uptake of brain endothelial cells. (uni-wuerzburg.de)
Extracellular1
- Thus, applications of neural probes are not limited to extracellular recording, brain-machine interface, and deep brain stimulation, but also include a wide range of new applications such as brain mapping, restoration of neuronal functions, and investigation of brain disorders. (mdpi.com)
Anatomical1
- Anatomical brain imaging at 7T using two-dimensional GRASE. (mpg.de)
Subsequent1
- In conclusion, the addition of MK801 during the OGD phase reduced significantly the glucose uptake after the subsequent reoxygenation phase in brain endothelial cells. (uni-wuerzburg.de)
Formation1
- Both hemodynamic (cardiogenic) and nonhemodynamic (noncardiogenic) components contribute to edema formation. (medscape.com)
Damage1
- Talk to Eric or Fred about a lawsuit for permanent brain damage seeking compensation for medical expenses, cost of care, lost income, pain and suffering, disability and other damages. (pritzkerlaw.com)
Subjects1
- healthy subjects and those with a medical conditions in the brain . (bvsalud.org)
Development1
- These different models allowed improvements on the understanding of intracranial hypertension establishment, neuroinflammation, immunology, brain hemodynamics and served to the development of therapeutic strategies. (spandidos-publications.com)
Medical2
- A classifier build from measurements in these two frequency ranges can provide instantaneous diagnostic of the medical condition of the brain of a patient , from a single set of measurements. (bvsalud.org)
- 75% within the first 24 hours after the ictus management does not show superiority of and the edema volume may exceed the early surgery over best medical treatment [3- volume of the ICH itself [14-16]. (who.int)