Brachial Plexus Neuropathies
Brachial Plexus Neuritis
Ulnar Nerve Compression Syndromes
Peripheral Nervous System Diseases
Thoracic Outlet Syndrome
The exacerbating effect of insulin-induced hypoglycemia on spontaneous peripheral neuropathy in aged B6C3F1 mice. (1/102)The effect of insulin-induced hypoglycemia on spontaneous peripheral neuropathy in aged mice was examined. Ninety-five-week-old female B6C3F1 mice were infused subcutaneously for 2 weeks with 40 or 80 IU/kg/day of insulin with a micro osmotic pump. Blood glucose level was decreased during the infusion (4.3-6.8 mmol/L in mice receiving 40 IU/kg/day of insulin or 2.4-5.4 mmol/L in mice receiving 80 IU/kg/day of insulin versus 6.5-7.6 mmol/L in control mice). In histopathological examination, axonal degeneration and/or remyelination were observed in a small number of nerve fibers of control mice. Similar nerve fiber lesions were observed in mice receiving 40 IU/kg/day of insulin, whereas severer lesions with an increase in segmental axonal degeneration of nerve fibers were observed in 4/7 mice receiving 80 IU/kg/day of insulin. These findings suggest that spontaneous peripheral neuropathy in aged mice is exacerbated by sustained hypoglycemia induced by insulin treatment. (+info)
Scapulothoracic stabilisation for winging of the scapula using strips of autogenous fascia lata. (2/102)We have used a modified technique in five patients to correct winging of the scapula caused by injury to the brachial plexus or the long thoracic nerve during transaxillary resection of the first rib. The procedure stabilises the scapulothoracic articulation by using strips of autogenous fascia lata wrapped around the 4th, 6th and 7th ribs at least two, and preferably three, times. The mean age of the patients at the time of operation was 38 years (26 to 47) and the mean follow-up six years and four months (three years and three months to 11 years). Satisfactory stability was achieved in all patients with considerable improvement in shoulder function. There were no complications. (+info)
Function of the upper limb after surgery for obstetric brachial plexus palsy. (3/102)We reviewed a consecutive series of 33 infants who underwent surgery for obstetric brachial plexus palsy at a mean age of 4.7 months. Of these, 13 with an upper palsy and 20 with a total palsy were treated by nerve reconstruction. Ten were treated by muscle transfer to the shoulder or elbow, and 16 by tendon transfer to the hand. The mean postoperative follow-up was 4 years 8 months. Ten of the 13 children (70%) with an upper palsy regained useful shoulder function and 11 (75%) useful elbow function. Of the 20 children with a total palsy, four (20%) regained useful shoulder function and seven (35%) useful elbow function. Most patients with a total palsy had satisfactory sensation of the hand, but only those with some preoperative hand movement regained satisfactory grasp. The ability to incorporate the palsied arm and hand into a co-ordinated movement pattern correlated with the sensation and prehension of the hand, but not with shoulder and elbow function. (+info)
Restoration of sensory function and lack of long-term chronic pain syndromes after brachial plexus injury in human neonates. (4/102)Obstetric complications are a common cause of brachial plexus injuries in neonates. Failure to restore sensation leads to trophic injuries and poor limb function. It is not known whether the infant suffers chronic neuropathic or spinal cord root avulsion pain; in adults, chronic pain is usual after spinal root avulsion injuries, and this is often intractable. The plexus is repaired surgically in severe neonatal injures; if no spontaneous recovery has occurred by 3 months, and if neurophysiological investigations point to poor prognosis, then nerve trunk injures are grafted, while spinal cord root avulsion injuries are treated by transferring an intact neighbouring nerve (e.g. intercostal) to the distal stump of the damaged nerve, in an attempt to restore sensorimotor function. Using a range of non-invasive quantitative measures validated in adults, including mechanical, thermal and vibration perception thresholds, we have assessed for the first time sensory and cholinergic sympathetic function in 24 patients aged between 3 and 23 years, who had suffered severe brachial plexus injury at birth. While recovery of function after spinal root avulsion was related demonstrably to surgery, there were remarkable differences from adults, including excellent restoration of sensory function (to normal limits in all dermatomes for at least one modality in 16 out of 20 operated cases), and evidence of exquisite CNS plasticity, i.e. perfect localization of restored sensation in avulsed spinal root dermatomes, now presumably routed via nerves that had been transferred from a distant spinal region. Sensory recovery exceeded motor or cholinergic sympathetic recovery. There was no evidence of chronic pain behaviour or neuropathic syndromes, although pain was reported normally to external stimuli in unaffected regions. We propose that differences in neonates are related to later maturation of injured fibres, and that CNS plasticity may account for their lack of long-term chronic pain after spinal root avulsion injury. (+info)
Ultrasound diagnosis of shoulder congruity in chronic obstetric brachial plexus palsy. (5/102)Ultrasound (US) was used to determine the congruity of the shoulder in 22 children with a deformity of the shoulder secondary to chronic obstetric brachial plexus palsy. There were 11 boys and 11 girls with a mean age of 4.75 years (0.83 to 13.92). The shoulder was scanned in the axial plane using a posterior approach with the arm internally rotated. The humeral head was classified as being either congruent or incongruent. The US appearance was compared with that on clinical examination and related to the intraoperative findings. All 17 shoulders diagnosed as incongruent on US were found to be incongruent at operation, whereas three diagnosed as congruent by US were found to be incongruent at operation. The diagnostic accuracy of US for the identification of shoulder incongruity was 82% when compared with the findings at surgery. US is a valuable, but not infallible tool, for the detection of incongruity of the shoulder. (+info)
A case of multiple schwannomas of the trigeminal nerves, acoustic nerves, lower cranial nerves, brachial plexuses and spinal canal: schwannomatosis or neurofibromatosis? (6/102)In most cases, while schwannoma is sporadically manifested as a single benign neoplasm, the presence of multiple schwannomas in one patient is usually indicative of neurofibromatosis 2. However, several recent reports have suggested that schwannomatosis itself may also be a distinct clinical entity. This study examines an extremely rare case of probable schwannomatosis associated with intracranial, intraspinal and peripheral involvements. A 63-year-old woman presented with a seven-year history of palpable lumps on both sides of the supraclavicular area and hearing impairment in both ears. On physical examination, no skin manifestations were evident. Facial sensory change, deafness in the left ear and decreased gag reflex were revealed by neurological examination. Magnetic resonance imaging revealed multiple lesions of the trigeminal nerves, acoustic nerves, lower cranial nerves, spinal accessory nerve, brachial plexuses, and spinal nerves. Pathological examination of tumors from the bilateral brachial plexuses, the spinal nerve in the T8 spinal position and the neck mass revealed benign schwannomas. Following is this patient case report of multiple schwannomas presenting with no skin manifestations of neurofibromatosis. (+info)
Cervico thoracic junction spinal tuberculosis presenting as radiculopathy. (7/102)A case of cervico thoracic junctional area spinal tuberculosis presenting as painful radiculitis of the upper extremity is reported. The predominant symptom of radicular pain and muscle weakness in the hand, along with a claw deformity, led to considerable delay in diagnosis. The presence of advanced bone destruction with severe instability was demonstrated on the MRI scan done later. Surgical management by radical anterior debridement and fusion, along with chemotherapy, led to resolution of the upper extremity symptoms. The brachial plexus radiculopathy secondary to tuberculosis has not been reported. The absence of myelopathic signs even in the presence of advanced bone destruction, thecal compression and instability is uncommon in adults. (+info)
Retroversion of the humeral head in children with an obstetric brachial plexus lesion. (8/102)We undertook a prospective MRI study to measure the retroversion of the humeral head in 33 consecutive infants with a mean age of 1 year 10 months (3 months to 7 years 4 months) who had an obstetric brachial plexus lesion (OBPL). According to a standardised MRI protocol both shoulders and humeral condyles were examined and the shape of the glenoid and humeral retroversion determined. The mean humeral retroversion of the affected shoulder was significantly increased compared with the normal contralateral side (-28.4 +/- 12.5 degrees v -21.5 +/- 15.1 degrees, p = 0.02). This increase was found only in the children over the age of 12 months. In this group humeral retroversion was -29.9 +/- 12.9 degrees compared with -19.6 +/- 15.6 degrees in the normal shoulder (p = 0.009), giving a mean difference of 10.3 degrees (95% confidence interval 3.3 to 17.3). This finding is of importance when considering the operative treatment for subluxation of the shoulder in children with an OBPL. (+info)
Brachial plexus neuropathies are a group of disorders that affect the brachial plexus, a network of nerves that runs from the spinal cord to the shoulder, arm, and hand. These disorders can result from injury, compression, or inflammation of the brachial plexus, and can cause a range of symptoms, including weakness, numbness, tingling, and pain in the affected arm and hand. Treatment for brachial plexus neuropathies depends on the underlying cause and may include medications, physical therapy, or surgery.
Brachial plexus neuritis is a condition that affects the brachial plexus, a network of nerves that runs from the spinal cord to the shoulder, arm, and hand. It is also known as brachial neuritis or brachial plexopathy. The brachial plexus is responsible for controlling movement and sensation in the arm and hand. When the nerves in this plexus become inflamed or damaged, it can cause a range of symptoms, including weakness, numbness, tingling, and pain in the affected arm. Brachial plexus neuritis can be caused by a variety of factors, including infection, injury, autoimmune disorders, and certain medications. It is typically diagnosed through a combination of physical examination, medical history, and imaging tests such as electromyography (EMG) or nerve conduction studies (NCS). Treatment for brachial plexus neuritis depends on the underlying cause and severity of the condition. In some cases, symptoms may resolve on their own over time, while in others, treatment may involve medications, physical therapy, or surgery.
The brachial plexus is a network of nerves that arises from the spinal cord in the neck and extends down the arm. It is responsible for controlling movement and sensation in the arm, hand, and shoulder. The brachial plexus is formed by five nerves: the C5, C6, C7, C8, and T1 nerves. These nerves exit the spinal cord and join together to form the brachial plexus, which then branches out to innervate the muscles and skin of the arm and shoulder. Damage to the brachial plexus can result in weakness or paralysis of the arm and hand, as well as numbness or tingling in the affected area.
Ulnar nerve compression syndromes refer to a group of conditions that result from compression or irritation of the ulnar nerve, which is one of the major nerves in the arm. The ulnar nerve runs down the arm and passes through the wrist and hand, providing sensation and movement to the little finger and part of the ring finger. There are several types of ulnar nerve compression syndromes, including cubital tunnel syndrome, Guyon's canal syndrome, and ulnar nerve entrapment at the elbow. These conditions can cause a range of symptoms, including numbness, tingling, weakness, and pain in the hand and arm, particularly in the little finger and ring finger. Treatment for ulnar nerve compression syndromes typically involves conservative measures such as rest, ice, physical therapy, and medication. In some cases, surgery may be necessary to relieve pressure on the nerve and alleviate symptoms.
Birth injuries refer to physical injuries that occur to a newborn during delivery. These injuries can be caused by a variety of factors, including the size and position of the baby, the mother's health and medical history, and the delivery method used (e.g., vaginal delivery or cesarean section). Some common types of birth injuries include: 1. Cerebral palsy: A group of disorders that affect a person's ability to move and coordinate their muscles. 2. Erb's palsy: A type of brachial plexus injury that affects the nerves in the shoulder and arm. 3. Klumpke's palsy: A type of brachial plexus injury that affects the nerves in the hand and fingers. 4. Brachial plexus injury: Injuries to the nerves in the shoulder and arm that can cause weakness or paralysis. 5. Facial nerve injury: Injuries to the facial nerve that can cause weakness or paralysis on one side of the face. 6. Skull fractures: Injuries to the baby's skull that can occur during delivery. 7. Brain injury: Injuries to the baby's brain that can occur during delivery, such as hypoxic-ischemic encephalopathy (HIE). 8. Shoulder dystocia: A condition in which the baby's shoulder becomes stuck during delivery, which can cause injury to the baby's shoulder or brachial plexus. 9. Umbilical cord prolapse: A condition in which the umbilical cord comes out of the cervix before the baby does, which can cause oxygen deprivation to the baby. 10. Placental abruption: A condition in which the placenta separates from the uterus before delivery, which can cause oxygen deprivation to the baby. Birth injuries can have serious long-term consequences for the baby, including developmental delays, physical disabilities, and cognitive impairments. It is important for healthcare providers to be aware of the potential risks of birth injuries and to take steps to prevent them whenever possible.
The choroid plexus is a specialized structure found in the ventricles of the brain. It is responsible for producing and secreting cerebrospinal fluid (CSF), which is a clear, colorless fluid that circulates throughout the brain and spinal cord, providing cushioning and protection to the delicate neural tissue. The choroid plexus is composed of specialized cells called ependymal cells, which line the inner surface of the ventricles. These cells are in contact with a network of blood vessels, which provide them with the nutrients and oxygen they need to function properly. The choroid plexus plays a critical role in maintaining the proper balance of ions and other substances in the CSF, which is essential for the proper functioning of the brain and spinal cord. It is also involved in the regulation of blood flow to the brain and the clearance of waste products from the neural tissue. Abnormalities of the choroid plexus can lead to a variety of neurological disorders, including hydrocephalus (an accumulation of CSF in the brain), which can cause swelling and damage to the brain.
Obstetric paralysis is a type of paralysis that occurs during pregnancy or childbirth. It is caused by pressure on the spinal cord or nerves in the lower back or pelvis, which can result from factors such as prolonged labor, difficult delivery, or trauma to the spine. Symptoms of obstetric paralysis may include weakness or numbness in the legs, difficulty walking or standing, and loss of bladder or bowel control. In severe cases, obstetric paralysis can lead to permanent paralysis or even death. Treatment for obstetric paralysis depends on the underlying cause and severity of the condition. In some cases, surgery may be necessary to relieve pressure on the spinal cord or nerves. Physical therapy and other supportive measures may also be used to help patients regain strength and function.
Diabetic neuropathy is a type of nerve damage that can occur as a complication of diabetes. It is caused by damage to the nerves that control movement, sensation, and other functions in the body. There are several types of diabetic neuropathy, including: 1. Peripheral neuropathy: This is the most common type of diabetic neuropathy and affects the nerves in the extremities, such as the hands, feet, and legs. It can cause numbness, tingling, pain, and weakness in the affected areas. 2. Autonomic neuropathy: This type of neuropathy affects the nerves that control automatic bodily functions, such as heart rate, digestion, and blood pressure. It can cause symptoms such as dizziness, fainting, and gastrointestinal problems. 3. Proximal neuropathy: This type of neuropathy affects the nerves in the arms and legs, causing weakness and muscle wasting in the affected areas. 4. Mononeuropathy: This is a type of neuropathy that affects a single nerve, causing symptoms such as pain, numbness, and weakness in the affected area. Diabetic neuropathy can be a serious complication of diabetes and can lead to a range of problems, including foot ulcers, infections, and even amputations. It is important for people with diabetes to manage their blood sugar levels and to see their healthcare provider regularly for monitoring and treatment.
Peripheral nervous system diseases refer to disorders that affect the peripheral nerves, which are the nerves that carry signals from the brain and spinal cord to the rest of the body. These diseases can affect the nerves themselves or the tissues surrounding them, and can result in a range of symptoms, including pain, numbness, weakness, and tingling. Some examples of peripheral nervous system diseases include: 1. Charcot-Marie-Tooth disease: A group of inherited disorders that affect the nerves in the hands and feet, causing weakness, numbness, and loss of sensation. 2. Guillain-Barre syndrome: A rare autoimmune disorder in which the body's immune system attacks the peripheral nerves, causing weakness and paralysis. 3. Peripheral neuropathy: A general term for any disorder that affects the peripheral nerves, which can be caused by a variety of factors, including diabetes, alcoholism, and exposure to certain toxins. 4. Multiple sclerosis: An autoimmune disorder that affects the central nervous system, including the brain and spinal cord, but can also affect the peripheral nerves, causing symptoms such as numbness and weakness. 5. Amyotrophic lateral sclerosis (ALS): A progressive neurodegenerative disorder that affects the nerves controlling muscle movement, leading to weakness and paralysis. Treatment for peripheral nervous system diseases depends on the specific disorder and its underlying cause. In some cases, medications or physical therapy may be used to manage symptoms, while in other cases, surgery or other interventions may be necessary.
The cervical plexus is a network of nerves located in the neck region that arises from the spinal cord. It is responsible for controlling movement and sensation in the upper extremities, including the shoulder, arm, and hand. The cervical plexus is made up of several nerves, including the spinal accessory nerve, the cervical nerves C1 to C4, and the superior and inferior cervical ganglia. These nerves innervate the muscles of the neck, shoulders, and upper back, as well as the skin and muscles of the upper extremities. Damage to the cervical plexus can result in weakness or paralysis of the affected muscles, as well as numbness or tingling in the upper extremities.
In the medical field, paralysis refers to a loss of muscle function or weakness in one or more areas of the body. This can be caused by a variety of factors, including injury, disease, or neurological disorders. There are several types of paralysis, including: 1. Complete paralysis: This is when a person is unable to move any part of their body. 2. Partial paralysis: This is when a person has some muscle function, but not all of it. 3. Flaccid paralysis: This is when the muscles are weak and floppy, and the person may have difficulty moving or maintaining their posture. 4. Spastic paralysis: This is when the muscles are tight and tense, and the person may have difficulty controlling their movements. Paralysis can affect any part of the body, including the arms, legs, face, and voice. It can be temporary or permanent, and can range from mild to severe. Treatment for paralysis depends on the underlying cause and can include physical therapy, medication, surgery, or other interventions.
Thoracic Outlet Syndrome (TOS) is a group of disorders that occur when the nerves and blood vessels in the neck and shoulder region are compressed or squeezed. This compression can be caused by a variety of factors, including bone, muscle, or ligament abnormalities, or by tumors or other masses in the area. There are three main types of TOS: 1. Neurogenic TOS: This type of TOS is caused by compression of the nerves that supply the arm and hand. Symptoms may include numbness, tingling, weakness, or pain in the arm and hand. 2. Venous TOS: This type of TOS is caused by compression of the veins that return blood to the heart from the arm and hand. Symptoms may include swelling, pain, or discoloration in the arm and hand. 3. Arterial TOS: This type of TOS is caused by compression of the arteries that supply blood to the arm and hand. Symptoms may include pain, weakness, or numbness in the arm and hand, and in severe cases, loss of pulse or tissue death in the affected arm. TOS can be diagnosed through a combination of physical examination, imaging studies, and nerve conduction studies. Treatment may include medications, physical therapy, or surgery, depending on the underlying cause and severity of the symptoms.
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- Brachial plexopathy is a form of peripheral neuropathy . (medlineplus.gov)
- Background: In approximately 25% of peripheral neuropathy cases, diagnosis remains obscure. (bvsalud.org)
- In India, leprosy continues to remain one of the most frequent causes of peripheral neuropathy. (bvsalud.org)
- We, in this prospective evaluation, performed nerve biopsies in patients with peripheral neuropathy for early confirmation of the diagnosis. (bvsalud.org)
- Materials and Methods: A total of 55 consecutive cases of peripheral neuropathy were included in this study. (bvsalud.org)
- Result: After a nerve biopsy in 29 cases, we were able to identify the underlying cause of peripheral neuropathy. (bvsalud.org)
- Conclusion: The nerve biopsies revealed that leprosy was the most common etiology in patients with peripheral neuropathy. (bvsalud.org)
- Peripheral neuropathy is a disease that is typically characterized by pain or loss of function in the nerves that carry signals to and from the brain and spinal cord (the central nervous system) to other parts of the body. (vaccinelaw.com)
- i) This term is defined as dysfunction limited to the upper extremity nerve plexus (i.e., its trunks, divisions, or cords) without involvement of other peripheral (e.g., nerve roots or a single peripheral nerve) or central (e.g., spinal cord) nervous system structures. (vaccinelaw.com)
- To have an issue (February 2014) of the Neuroimaging Clinics of North America devoted to this topic is a welcome thing for neuroradiology because it allows a review of some fundamental concepts and anatomy of the cranial nerves and brain stem, reviews the important topics of the MR imaging of the brachial plexus and lumbosacral plexus, and describes the issues involved in peripheral neurography. (elsevierhealth.com)
- Peripheral neuropathy is dysfunction of one or more peripheral nerves (the part of a nerve distal to the root and plexus). (merckmanuals.com)
- Nerve Release bodywork is any modality that specializes in creating healthy space around the peripheral nerves to aid in as much healthy function as possible and to alleviate compressive neuropathy. (balancethroughmovementmethod.com)
- In the late 1960s, neurophysiologic testing allowed the classification of CMT into 2 groups, one with slow nerve conduction velocities and histologic features of a hypertrophic demyelinating neuropathy (hereditary motor and sensory neuropathy type 1 or CMT1) and another with relatively normal velocities and axonal and neuronal degeneration (hereditary motor and sensory neuropathy type 2 or CMT2). (medscape.com)
- Disorders of nerve roots and plexuses. (medlineplus.gov)
- The cervical and upper thoracic spinal cords give rise to the brachial plexus, a network of interlacing nerves (specifically, from the anterior rami of the C5-T1 nerve roots). (com.bd)
- The disconnect even in human medicine around nerve impingement and compressive neuropathy is incredibly high, so naturally we find that to be the case in horses. (balancethroughmovementmethod.com)
- Compressive neuropathy is usually caused by repetitive motions that affect an area where a nerve travels. (balancethroughmovementmethod.com)
- In my personal work, I find that most horses that are struggling with this are dealing with compressive neuropathy particularly around the cervicothoracic region (brachial plexus) and the lumbosacral region (lumbosacral plexus). (balancethroughmovementmethod.com)
- Damage to the nerves of the brachial plexus results in pain, decreased movement, or decreased feeling in the arm and shoulder. (medlineplus.gov)
- An exam of the arm, hand and wrist can reveal a problem with the nerves of the brachial plexus. (medlineplus.gov)
- This change in the functioning of septin proteins seems to particularly affect the network of nerves controlling movement and sensation in the shoulders and arms (brachial plexus), but the reason for this is unknown. (encyclopedia.pub)
- An autoimmune attack on the nerves in the brachial plexus likely results in the signs and symptoms of hereditary neuralgic amyotrophy. (encyclopedia.pub)
- As with some other vaccine injuries, with brachial neuritis, the pain, loss of function and other damage occurs in the brachial plexus which is a bundle of nerves that travels from the spinal cord to the shoulder, arms, and hands. (vaccinelaw.com)
- Inherited neuropathies in which autonomic or sensory features predominate, conditions in which the neuropathy is part of a multiple-organ disturbance, and neuropathies with specific metabolic dysfunction are not discussed. (medscape.com)
- In two biopsies, the findings were consistent with hereditary neuropathies. (bvsalud.org)
- Neurological disorders temporally associated with influenza vaccination such as encephalopathy, optic neuritis/neuropathy, partial facial paralysis, and brachial plexus neuropathy have been reported. (publicintelligence.net)
- Can Brachial Neuritis Be Caused by Vaccines? (vaccinelaw.com)
- In fact, the National Vaccine Injury Compensation Program lists brachial neuritis as a Table injury for tetanus toxoid vaccine. (vaccinelaw.com)
- Other vaccines may also be associated with brachial neuritis. (vaccinelaw.com)
- If you have developed brachial neuritis after receiving a tetanus-containing vaccine, or any other vaccine, contact vaccine attorney Leah Durant. (vaccinelaw.com)
- Brachial neuritis is also commonly referred to as brachial neuropathy, brachial plexus injury, and Parsonage-Turner syndrome. (vaccinelaw.com)
- Brachial neuritis is specifically listed on the Vaccine Injury Table for tetanus toxoid, a vaccine that may be administered alone, or with other vaccines such as the Diptheria-Tetanus-acellular Pertussis (DTP, or DTaP, or Tdap) vaccine. (vaccinelaw.com)
- According to the Vaccine Injury Table, the time frame for the onset for brachial neuritis after having received a tetanus vaccine is 2-28 days. (vaccinelaw.com)
- A detailed history may help determine the cause of the brachial plexopathy. (medlineplus.gov)
- Aims Recurrent breast carcinoma with brachial plexus involvement is often misinterpreted as a radiation- or chemotherapy-induced brachial plexopathy. (elsevierpure.com)
- Methods Four patients with recurrent axillary breast cancer and symptoms consistent with a brachial plexopathy were prospectively collected over a 1-year period. (elsevierpure.com)
- Conclusion Breast cancer survivors presenting with a brachial plexopathy should raise suspicion for recurrent disease. (elsevierpure.com)
- If they were to go to their doctor instead, they would be referred to an orthopedic surgeon who would recommend shaving down part of the clavicle to create space for the brachial plexus to no longer be compressed so the neuropathic pain would alleviate. (balancethroughmovementmethod.com)
- As mentioned, conditions like Pancoast tumors can directly compress or invade the lower components of the brachial plexus, leading to symptoms like pain, tingling, and weakness in the affected arm. (com.bd)
- 1 had brachial plexus involvement alone. (cdc.gov)
- Other diagnoses were chronic demyelinating neuropathy (four cases), vasculitis (two cases), and amyloidosis in one case. (bvsalud.org)
- in others (eg, certain cases of Charcot-Marie-Tooth disease type 1A (CMT1A) and inherited brachial plexus neuropathy [IBPN]/hereditary neuralgic amyotrophy [HNA]), proximal weakness predominates. (medscape.com)
- For example, young men more often have inflammatory or post-viral brachial plexus disease called Parsonage-Turner syndrome. (medlineplus.gov)
- It occurs when there is damage to the brachial plexus. (medlineplus.gov)
- 5 m/s) velocities observed in Dejerine-Sottas syndrome (DSS) and congenital hypomyelination neuropathy (CHN). (medscape.com)
- This suggests that, in most cases, axonal damage is the root cause of the neuropathy, not demyelination. (medscape.com)
- Methods: We screened newly-diagnosed patients with multibacillary leprosy presenting with neuropathy. (bvsalud.org)
- Long term compression of these places (compressive neuropathy) can result in ganglionitis. (balancethroughmovementmethod.com)
- Some isolated case reports and case series have communicated imaging changes in the central nervous system (CNS) and brachial plexus in patients with leprosy. (bvsalud.org)
- Age and sex are important, because some brachial plexus problems are more common in certain groups. (medlineplus.gov)
- Agre JC, Ash N, Cameron MC, House J. Suprascapular neuropathy after intensive progressive resistive exercise: case report. (medscape.com)
- Ravindran M. Two cases of suprascapular neuropathy in a family. (medscape.com)
- Suprascapular neuropathy in athletes: case reports. (medscape.com)