Salicylanilides
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Waxes
A plastic substance deposited by insects or obtained from plants. Waxes are esters of various fatty acids with higher, usually monohydric alcohols. The wax of pharmacy is principally yellow wax (beeswax), the material of which honeycomb is made. It consists chiefly of cerotic acid and myricin and is used in making ointments, cerates, etc. (Dorland, 27th ed)
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Molecular Medicine
Histone-Lysine N-Methyltransferase
Hair Follicle
A tube-like invagination of the EPIDERMIS from which the hair shaft develops and into which SEBACEOUS GLANDS open. The hair follicle is lined by a cellular inner and outer root sheath of epidermal origin and is invested with a fibrous sheath derived from the dermis. (Stedman, 26th ed) Follicles of very long hairs extend into the subcutaneous layer of tissue under the SKIN.
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Cosmetics
Substances intended to be applied to the human body for cleansing, beautifying, promoting attractiveness, or altering the appearance without affecting the body's structure or functions. Included in this definition are skin creams, lotions, perfumes, lipsticks, fingernail polishes, eye and facial makeup preparations, permanent waves, hair colors, toothpastes, and deodorants, as well as any material intended for use as a component of a cosmetic product. (U.S. Food & Drug Administration Center for Food Safety & Applied Nutrition Office of Cosmetics Fact Sheet (web page) Feb 1995)
Aromatic L-amino acid decarboxylase: conformational change in the flexible region around Arg334 is required during the transaldimination process. (1/1033)
Aromatic L-amino acid decarboxylase (AADC) catalytic mechanism has been proposed to proceed through two consecutive intermediates (i.e., Michaelis complex and the external aldimine). Limited proteolysis of AADC that preferentially digested at the C-terminal side of Arg334 was slightly retarded in the presence of dihydroxyphenyl acetate that formed a stable Michaelis complex. On the contrary, AADC was scarcely digested in the presence of L-dopa methyl ester that formed a stable external aldimine. Similar protection by the substrate analogs was observed in the chemical modification experiment. From these results, we concluded that the region around Arg334 must be exposed and flexible in the unliganded state, and forming the Michaelis complex generated a subtle conformational change, then underwent marked conformational change during the subsequent transaldimination process prerequisite to forming the external aldimine. For further analyses, we constructed a mutant gene encoding in tandem the two peptides of AADC cleaved at the Asn327-Met328 bond inside the putative flexible region. The gene product, fragmentary AADC, was still active with L-dopa as substrate, but its k(cat) value was decreased 57-fold, and the Km value was increased 9-fold compared with those of the wild-type AADC. The absorption spectra of the fragmentary AADC in the presence of L-dopa methyl ester showed shift in the equilibrium of the transaldimination from the external aldimine to the Michaelis complex. Tryptic digestion of the fragmentary AADC removed seven amino acid residues, Met328-Arg334, and resulted in complete inactivation. Susceptibility of the fragmentary enzyme to trypsin was not changed by L-dopa methyl ester revealing the loss of appropriate conformational change in the flexible region induced by substrate binding. From these results we propose that the conformational change in the flexible region is required during the transaldimination process. (+info)Trefoil peptide TFF2 (spasmolytic polypeptide) potently accelerates healing and reduces inflammation in a rat model of colitis. (2/1033)
BACKGROUND: The trefoil peptides are major secretory products of mucus cells of the gastrointestinal tract and show increased expression after inflammatory or ulcerative damage. Recombinant human TFF2 (spasmolytic polypeptide) has been shown to be cytoprotective, and enhances repair in models of gastric injury. AIMS: To test the healing effects of recombinant human (h)TFF2 in a rat model of chronic colitis. METHODS: Colitis was induced by intracolonic administration of dinitrobenzene sulphonic acid in ethanol. Mucosal repair was quantified macroscopically, microscopically by image analysis of tissue histology, and by measuring myeloperoxidase activity. RESULTS: Initial validation studies showed that maximal injury and inflammation occurred at the end of the first week after colitis induction (active phase), and that spontaneous healing was complete by eight weeks. Once daily intrarectal application of hTFF2 (2.5 mg/kg; approximately 0.5 mg/rat) for five days after maximal damage had been sustained, reduced both microscopic and macroscopic injury by 80% and inflammatory index by 50% compared with vehicle controls. In addition, endogenous concentrations of rat TFF2 and TFF3 (intestinal trefoil factor) were increased in the active phase of colitis and were reduced to basal levels by hTFF2 treatment. CONCLUSIONS: This study has shown that hTFF2 enhances the rate of colonic epithelial repair, and reduces local inflammation in a rat model of colitis, and suggests that luminal application of trefoil peptides may have therapeutic potential in the treatment of inflammatory bowel disease. (+info)KNR4, a suppressor of Saccharomyces cerevisiae cwh mutants, is involved in the transcriptional control of chitin synthase genes. (3/1033)
The KNR4 gene, originally isolated by complementation of a K9 killer-toxin-resistant mutant displaying reduced levels of both 1,3-beta-glucan and 1,3-beta-glucan synthase activity, was recloned from a YCp50 genomic library as a suppressor of Saccharomyces cerevisiae calcofluor-white-hypersensitive (cwh) mutants. In these mutants, which were characterized by increased chitin levels, the suppressor effect of KNR4 resulted, for some of them, in a lowering of polymer content to close to wild-type level, with no effect on the contents of beta-glucan and mannan. In all cases, this effect was accompanied by a strong reduction in mRNA levels corresponding to CHS1, CHS2 and CHS3, encoding chitin synthases, without affecting expression of FKS1 and RHO1, two genes encoding the catalytic subunit and a regulatory component of 1,3-beta-glucan synthase, respectively. Overexpression of KNR4 also inhibited expression of CHS genes in wild-type strains and in two other cwh mutants, whose sensitivity to calcofluor white was not suppressed by this gene. The physiological relevance of the KNR4 transcriptional effect was addressed in two different ways. In a wild-type strain exposed to alpha-factor, overexpression of this gene inhibited CHS1 induction and delayed shmoo formation, two events which are triggered in response to the pheromone, whereas it did not affect bud formation and cell growth in a chs1 chs2 double mutant. A chimeric protein made by fusing green fluorescent protein to the C terminus of Knr4p which fully complemented a knr4delta mutation was found to localize in patches at presumptive bud sites in unbudded cells and at the incipient bud site during bud emergence. Taken together, these results demonstrate that KNR4 has a regulatory role in chitin deposition and in cell wall assembly. A mechanism by which this gene affects expression of CHS genes is proposed. (+info)Nitrite determination in human plasma and synovial fluid using reactions of nitric oxide with 3, 5-dibromo-4-nitrosobenzenesulphonate (DBNBS). (4/1033)
DBNBS (3,5-dibromo-4-nitrosobenzenesulphonate) reacts with nitric oxide (NO) produced from nitrite ions in acid solution to give a radical with a characteristic electron spin resonance spectrum, attributable to a 'DBNBS-NO' product, and comprising a triplet with alphaN=0.96 mT. This is identical with the spectrum obtained when NO, introduced from the gas phase, reacts with DBNBS. Under certain conditions, an additional signal is observed, attributable to oxidation of DBNBS to the radical cation, DBNBS*+ (a triplet with alphaN=1.32 mT). Conditions are described for the determination of nitrite, which avoid this DBNBS oxidation. The height of the low-field signal from the DBNBS-NO product is directly proportional to the nitrite concentration up to about 0.08 mM nitrite. The method has been applied to the measurement of nitrite concentrations in whole blood, plasma and synovial fluid taken from rheumatoid arthritis patients. In order to avoid the oxidation of DBNBS when analysing biological samples of this type, it is necessary to treat the specimen by ultrafiltration as soon as possible after collection and before addition of DBNBS. (+info)Deletion of new covalently linked cell wall glycoproteins alters the electrophoretic mobility of phosphorylated wall components of Saccharomyces cerevisiae. (5/1033)
The incorporation of radioactive orthophosphate into the cell walls of Saccharomyces cerevisiae was studied. 33P-labeled cell walls were extensively extracted with hot sodium dodecyl sulfate (SDS). Of the remaining insoluble radioactivity more than 90% could be released by laminarinase. This radioactive material stayed in the stacking gel during SDS-polyacrylamide gel electrophoresis but entered the separating gel upon treatment with N-glycosidase F, indicating that phosphate was linked directly or indirectly to N-mannosylated glycoproteins. The phosphate was bound to covalently linked cell wall proteins as mannose-6-phosphate, the same type of linkage shown previously for soluble mannoproteins (L. Ballou, L. M. Hernandez, E. Alvarado, and C. E. Ballou, Proc. Natl. Acad. Sci. USA 87:3368-3372, 1990). From the phosphate-labeled glycoprotein fraction released by laminarinase, three cell wall mannoproteins, Ccw12p, Ccw13p and Ccw14p, were isolated and identified by N-terminal sequencing. For Ccw13p (encoded by DAN1 [also called TIR3]) and Ccw12p the association with the cell wall has not been described before; Ccw14p is identical with cell wall protein Icwp (I. Moukadiri, J. Armero, A. Abad, R. Sentandreu, and J. Zueco, J. Bacteriol. 179:2154-2162, 1997). In ccw12, ccw13, or ccw14 single or double mutants neither the amount of radioactive phosphate incorporated into cell wall proteins nor its position in the stacking gel was changed. However, the triple mutant brought about a shift of the 33P-labeled glycoprotein components from the stacking gel into the separating gel. The disruption of CCW12 results in a pronounced sensitivity of the cells to calcofluor white and Congo red. In addition, the ccw12 mutant shows a decrease in mating efficiency and a defect in agglutination. (+info)Perturbations in the control of cellular arachidonic acid levels block cell growth and induce apoptosis in HL-60 cells. (6/1033)
Our previous studies demonstrated that inhibitors of arachidonate-phospholipid remodeling [i.e. the enzyme CoA-independent transacylase (CoA-IT)] decrease cell proliferation and induce apoptosis in neoplastic cells. The goal of the current study was to elucidate the molecular events associated with arachidonate-phospholipid remodeling that influence cell proliferation and survival. Initial experiments revealed the essential nature of cellular arachidonate to the signaling process by demonstrating that HL-60 cells depleted of arachidonate were more resistant to apoptosis induced by CoA-IT inhibition. In cells treated with CoA-IT inhibitors a marked increase in free arachidonic acid and AA-containing triglycerides were measured. TG enrichment was likely due to acylation of arachidonic acid into diglycerides and triglycerides via de novo glycerolipid biosynthesis. To determine the potential of free fatty acids to affect cell proliferation, HL-60 cells were incubated with varying concentrations of free fatty acids; exogenously provided 20-carbon polyunsaturated fatty acids caused a dose-dependent inhibition of cell proliferation, whereas oleic acid was without effect. Blocking 5-lipoxygenase or cyclooxygenases had no effect on the inhibition of cell proliferation induced by arachidonic acid or CoA-IT inhibitors. An increase in cell-associated ceramides (mainly in the 16:0-ceramide fraction) was measured in cells exposed to free arachidonic acid or to CoA-IT inhibitors. This study, in conjunction with other recent studies, suggests that perturbations in the control of cellular arachidonic acid levels affect cell proliferation and survival. (+info)Saccharomyces cerevisiae mid2p is a potential cell wall stress sensor and upstream activator of the PKC1-MPK1 cell integrity pathway. (7/1033)
The MID2 gene of Saccharomyces cerevisiae encodes a protein with structural features indicative of a plasma membrane-associated cell wall sensor. MID2 was isolated as a multicopy activator of the Skn7p transcription factor. Deletion of MID2 causes resistance to calcofluor white, diminished production of stress-induced cell wall chitin under a variety of conditions, and changes in growth rate and viability in a number of different cell wall biosynthesis mutants. Overexpression of MID2 causes hyperaccumulation of chitin and increased sensitivity to calcofluor white. alpha-Factor hypersensitivity of mid2Delta mutants can be suppressed by overexpression of upstream elements of the cell integrity pathway, including PKC1, RHO1, WSC1, and WSC2. Mid2p and Wsc1p appear to have overlapping roles in maintaining cell integrity since mid2Delta wsc1Delta mutants are inviable on medium that does not contain osmotic support. A role for MID2 in the cell integrity pathway is further supported by the finding that MID2 is required for induction of Mpk1p tyrosine phosphorylation during exposure to alpha-factor, calcofluor white, or high temperature. Our data are consistent with a role for Mid2p in sensing cell wall stress and in activation of a response that includes both increased chitin synthesis and the Mpk1p mitogen-activated protein kinase cell integrity pathway. In addition, we have identified an open reading frame, MTL1, which encodes a protein with both structural and functional similarity to Mid2p. (+info)Chs7p, a new protein involved in the control of protein export from the endoplasmic reticulum that is specifically engaged in the regulation of chitin synthesis in Saccharomyces cerevisiae. (8/1033)
The Saccharomyces cerevisiae CHS7 gene encodes an integral membrane protein located in the ER which is directly involved in chitin synthesis through the regulation of chitin synthase III (CSIII) activity. In the absence of CHS7 product, Chs3p, but not other secreted proteins, is retained in the ER, leading to a severe defect in CSIII activity and consequently, to a reduced rate of chitin synthesis. In addition, chs7 null mutants show the yeast phenotypes associated with a lack of chitin: reduced mating efficiency and lack of the chitosan ascospore layer, clear indications of Chs7p function throughout the S. cerevisiae biological cycle. CHS3 overexpression does not lead to increased levels of CSIII because the Chs3p excess is retained in the ER. However, joint overexpression of CHS3 and CHS7 increases the export of Chs3p from the ER and this is accompanied by a concomitant increase in CSIII activity, indicating that the amount of Chs7p is a limiting factor for CSIII activity. Accordingly, CHS7 transcription is increased when elevated amounts of chitin synthesis are detected. These results show that Chs7p forms part of a new mechanism specifically involved in Chs3p export from the ER and consequently, in the regulation of CSIII activity. (+info)
The multikinase inhibitor Sorafenib enhances glycolysis and synergizes with glycolysis blockade for cancer cell killing<...
Sodium 5-methoxy-2-methyl-4-(3-oxobutanamido)benzenesulfonate 133167-77-8 H-NMR | C-NMR Spectral Analysis NMR Spectrum
sodium,2,5-bis(methoxycarbonyl)benzenesulfonate 31314-30-4 H-NMR | C-NMR Spectral Analysis NMR Spectrum
Amphiphilic properties of dodecylammonium chloride/sodium 4-(1-pentylheptyl) benzenesulfonate aqueous mixtures and study of the...
Reaction mass of potassium, sodium... - Registration Dossier - ECHA
Sodium benzenesulfonate | C6H5NaO3S - PubChem
Sodium 5-chloro-2-(4-chloro-2-nitrophenoxy)benzenesulfonate - Alfa Chemistry
2-(Methylsulfonyl)-2,4,5,6-tetrahydropyrrolo[3,4-c]pyrazole benzenesulfonate - Manufacturer,Supplier & Exporter
HCV New Drugs: Hepatitis C And Liver News Of The Day Jan 17th
DailyMed - NEXAVAR- sorafenib tablet, film coated
The role of Mcl-1 downregulation in the proapoptotic activity of the multikinase inhibitor BAY 43-9006
Targeted delivery of anti-cancer drug sorafenib through magnetic solid lipid nanoparticles
北京大å¦åŒ»å¦éƒ¨æœºæž„知识库([email protected]): YC-1 enhances the anti-tumor activity of sorafenib through inhibition of signal transducer and...
The multikinase inhibitor and FDA\approved medication dovitinib (Dov) crosses the bloodCbrain | Thioredoxin Reductase and its...
Identification of Potential Off-Targets of Chemotherapeutic Agent Sorafenib: A Molecular Docking Approach
Sorafenib in Treating Patients With Metastatic, Locally Advanced, or Recurrent Sarcoma - Full Text View - ClinicalTrials.gov
Sorafenib in Treating Patients With Metastatic, Locally Advanced, or Recurrent Sarcoma - Full Text View - ClinicalTrials.gov
DMOZ - Health: Pharmacy: Drugs and Medications: S: Sorafenib
Sorafenib Slows Growth of Some Leukemias ( Drug shows potential for patients wit...)
Sorafenib: Latest News & Videos, Photos about Sorafenib | The Economic Times
Drug Re-Sensitizes Breast Tumors to... ( Phase II study finds sorafenib helps ...)
Sorafenib | Raf 阻害剤
Support Services | NEXAVAR (sorafenib)
SF-1 | OSI-930, a Novel Multikinase Inhibitor suppress the host immune response
Facet Effects Of Nexavar (Sorafenib), Warnings, Uses
An Open-Label, Multicenter, Randomized, Phase Ib/II Study of E7050 in Combination With Sorafenib Versus Sorafenib Alone as...
An Open-Label, Multicenter, Randomized, Phase Ib/II Study of E7050 in Combination With Sorafenib Versus Sorafenib Alone as...
Buy Sorafenib Online | Soranib - AllGenericCure
Gentaur Molecular :LCLabs \ Sorafenib, Free Base \ S-8599
Soranib - Sorafenib
SORANIB - SORAFENIB
englishvelling1410 - Find Your Medicines
Bayers pipeline promises much headed by new Nexavar filing - PharmaTimes
Oncotarget | pERK/pAkt phenotyping in circulating tumor cells as a biomarker for sorafenib efficacy in patients with advanced...
DI-fusion Selected combination therapy with sorafenib: a review of...
DI-fusion Determination of the anticancer drug sorafenib in serum by...
Calcofluor white stain | definition of calcofluor white stain by Medical dictionary
IJMS | Free Full-Text | Effects of Sorafenib on C-Terminally Truncated Androgen Receptor Variants in Human Prostate Cancer Cells
Plus it
T-0902611 benzenesulfonate | CAS# 350595-61-8 | Cytomegalovirus Replication Inhibitor | MedKoo
Aggressive thyroid cancer: targeted therapy with sorafenib - Minerva Endocrinologica 2017 March;42(1):64-76 - Minerva Medica -...
Hypertension and hand-foot skin reactions related to VEGFR2 genotype and improved clinical outcome following bevacizumab and...
Sorafenib | definition of sorafenib by Medical dictionary
Foretinib (GSK1363089), an Orally Available Multikinase Inhibitor of c-Met and VEGFR-2, Blocks Proliferation, Induces Anoikis,...
Dose study of the multikinase inhibitor, LY2457546, in patients with r | CMAR
Regorafenib for cancer
Sorafenib tosylate | Raf inhibitor | BAY 43-9006 | Nexavar | BAY43-9006 | CAS [475207-59-1] - [284461-73-0] | Axon 1397 | Axon...
Quantitative phosphoproteomics uncovers synergy between DNA-PK and FLT3 inhibitors in acute myeloid leukaemia | Leukemia
lindegaardfrost8793 - Treatment For Kidney, Liver, Thyroid Cancer
Tables.PatSel AsianConsensus.Delphi - Eubio
Plus it
Related Forms & Information | Providence Oregon
Results Inhibition of glioma cell growth by sorafenib plus VK1 In order to inve
Keywords: Heart arrest | OSI-930, a Novel Multikinase Inhibitor suppress the host immune response
Dr. Kasper on Imatinib Versus Sorafenib in Sarcoma
Supplementary Materialsijms-21-03126-s001 | Small molecule inhibitors of HCV replication
Sparquin 200mg
Nexavar Shows Promise in Phase 3 Thyroid Cancer Trial - MPR
An Extension Treatment Protocol for Subjects Who Have Participated in a Phase 3 Study of Tivozanib Versus Sorafenib in Renal...
sorafenib | Ligand page | IUPHAR/BPS Guide to PHARMACOLOGY
Go to the first page of search results for node/nexavar.
PSP CFW? | GBAtemp.net - The Independent Video Game Community
Sorafenib acts synergistically in combination with radiotherapy without causing intestinal damage in colorectal cancer. | Sigma...
TSAR, Vol.1, No.10
Rebug CFW 4.82.2 REX/D-REX - PS3 - Dekazeta
TASS: Society & Culture - Remains of Tsars family unlikely to be lost completely, expert hopes
Shrimp Amano - a good helper in the aquarium
Coomassie Brilliant Blue
Benzenesulfonates. *Benzenesulfonic acids. Hidden categories: *Articles without InChI source. *Chemical pages without ...
Allura Red AC
Benzenesulfonates. *Food colorings. *Naphthalenesulfonates. *Organic sodium salts. Hidden categories: *CS1 maint: BOT: original ...
Sunset Yellow FCF
Benzenesulfonates. Hidden categories: *Articles with changed EBI identifier. *ECHA InfoCard ID from Wikidata ...
Polystyrene sulfonate
In early 2008, a noninferiority study versus vancomycin or metronidazole for Clostridium difficile associated diarrhea (CDAD) found that about half of the patients in the tolevamer group did not complete the treatment, versus 25% in the vancomycin and 29% in the metronidazole groups. CDAD recurrence in patients reaching clinical success was reduced significantly by tolevamer (6% recurrence rate), vancomycin (18%) and metronidazole (19%). However, the good result of tolevamer is partly due to the high drop-out rate in this group. Since tolevamer did not reach its primary endpoint in this study, its development was halted.[14] ...
Interfacial Dilational Properties of Tri-Substituted Alkyl Benzene Sulfonates
Mesomorphism of a new series of catanionic 4-(1- pentylheptyl)benzenesulfonates FULIR
The effect of cationic chemical structure on the mesophase behavior, and self-assembly of catanionic compounds is studied through a systematic structure variation of six cationic surfactants with anionic sodium 4-(1- pentylheptyl)benzenesulfonate surfactant. A series of cationic surfactants are selected with increasing number of n-dodecyl chains on the same ammonium headgroup (from one to three), and with increasing number of ammonium headgroups (from two to four). Thermal and phase behavior of the synthesized products were examined by thermogravimetry, polarizing microscopy, differential scanning calorimetry, powder and grazing incidence X-ray diffraction, and molecular simulation. The compounds are complexes of high thermal stability. Most of the thermotropic mesophases are smectic, although the hexagonal mesophase is also observed. Introduction of the third dodecyl chain into the cationic part causes pronounced lower melting temperatures and eradicates mesomorphism, while the addition of a ...
Surfactants
Phase II study of sorafenib in co... preview & related info | Mendeley
Molecular insight into the lower critical solution temperature transition of aqueous alkyl phosphonium benzene sulfonates |...
Inert coupling of IRDye800CW and zirconium-89 to monoclonal antibodies for single- or dual-mode fluorescence and PET imaging
Dynamic reprogramming of the kinome in response to targeted MEK inhibition in triple-negative breast cancer
Patent US20060019332 - Deparaffinization compositions for dewaxing tissue specimens - Google Patents
Patent US3350319 - Aqueous detergent-inorganic builder concentrates - Google Patents
Zeitschrift für Physikalische Chemie
MEDLINE - Resultado p gina 1
Molecular mechanisms of band 3 inhibitors. 3. Translocation inhibitors.
MEDLINE - Resultado p gina 1
3-ethyl-1-methyl-1H-imidazol-3-ium... - Substance Information - ECHA
Patent WO2013070560A1 - Surface treatment compositions including shielding salts - Google Patents
3-amino-N,N-dimethylpropan-1-aminium... - Registration Dossier - ECHA
Introduction of Polymer additives | Chemical Products|ADEKA
Detergent granule comprising a nonionic surfactant and a hydrotrope - The Procter & Gamble Company
Patente US7101921 - Polymer composition - Google Patentes
High dose intermittent sorafenib shows improved efficacy over conventional continuous dose in renal cell carcinoma. | CureHunter
Ingredients -- Detergents and Surfactants
These were called branched-chain alkyl benzene sulfonates . Like soap, they could take hard minerals out of water, leaving it ... They were replaced by straight-chain alkyl benzene sulfonates, such as Sodium dodecylbenzinesulfonate and sodium ... Linear Alkyl benzene sulfonates (LAS). Straight-chain, anionic surfactants. Somewhat slow to biodegrade. Most common ... Alkyl benzene sulfonates (ABS). Branched-chain, anionic surfactants. Slow to biodegrade. Seldom used. ...
US6345633B1 - Automatic dishwashing compositions containing water soluble cationic surfactants
- Google Patents
US8328118B2 - Manual sprayer with dual bag-on-valve assembly
- Google Patents
The EPA National Library Catalog | EPA National Library Network | US EPA
W. Birchmeier Lab | Max Delbrück Center for Molecular Medicine
Patente US4698181 - Detergent compositions containing triethylenetetraminehexaacetic acid - Google Patentes
Laundry detergent - Life Miracle Health Products | Wholesale Distributor | Liquid Vitamin | Liquid Collagen Protein | Nano...
Ingredients for Household, Industrial, and Institutional Cleaning Applications
US Patent # 5,665,689. Cleaning compositions comprising mixtures of partially esterified full
esterified and non-esterfied...
Compounds1
- These substances are usually alkyl benzenesulfonates, a family of compounds that are similar to soap but are more soluble in hard water, because the polar sulfonate (of detergents) is less likely than the polar carboxyl (of soap) to bind to calcium and other ions found in hard water. (niir.org)
Substances1
- The molecules in question with the working names A-Na and Azo-Na are promising substances that are classified as benzenesulfonates. (mipt.ru)
Phenyl1
- Activation of Phenyl 4-(2-Oxo-3-alkylimidazolidin-1-yl)benzenesulfonates Prodrugs by CYP1A1 as New Antimitotics Targeting Breast Cancer Cells. (ulaval.ca)
Date1
- Author Unknown, "Crisvon (Solvent-based), Hydran (Water-based) Resins for Films" from http://www.dic-global.com/us/en/products/polyurethane/leather/film.html, date unknown. (patents.com)