Basal Ganglia Cerebrovascular Disease
Basal Ganglia Diseases
Nervous System Diseases
Paraneoplastic Syndromes, Nervous System
Magnetic Resonance Imaging
Two types of auditory neglect. (1/36)Auditory neglect, defined as inattention to stimuli within the left hemispace, is mostly reported in association with left ear extinction in dichotic listening. However, it remains disputed as to how far dichotic extinction reflects a primary attentional deficit and is thus appropriate for the diagnosis of auditory neglect. We report here on four patients who presented left ear extinction in dichotic listening following right unilateral hemispheric lesions. Auditory spatial attention was assessed with two additional tasks: (i) diotic test by means of interaural time differences (ITDs), simulating bilateral simultaneous spatial presentation of the dichotic tasks without the inconvenience of interaural intensity or content difference; and (ii) sound localization. A hemispatial asymmetry on the ITD diotic test or a spatial bias on sound localization were found to be part of auditory neglect. Two patients (J.C.N. and M.B.) presented a marked hemispatial asymmetry favouring the ipsilesional hemispace in the ITD diotic test, but did not show any spatial bias in sound localization. Two other patients (A.J. and E.S.) had the reverse profile: no hemispatial asymmetry in the ITD diotic test, but a severe spatial bias directed to the ipsilesional side in sound localization. J.C.N. and M.B. had mainly subcortical lesions affecting the basal ganglia. A.J. and E.S. had cortical lesions in the prefrontal, superior temporal and inferior parietal areas. Thus, there are two behaviourally and anatomically distinct types of auditory neglect characterized by: (i) deficit in allocation of auditory spatial attention following lesions centred on basal ganglia; or (ii) distortion of auditory spatial representation following frontotemporoparietal lesions. (+info)
Correlation of early CT signs in the deep middle cerebral artery territories with angiographically confirmed site of arterial occlusion. (2/36)BACKGROUND AND PURPOSE: Early CT signs in the deep middle cerebral artery (MCA) territories have been reported to be seen at the initial period of ischemia. We attempted to investigate the incidence of parenchymal hypodensity within 3 hours after ischemic onset among patients with angiographically proved embolic MCA occlusion and to assess the correlation of subtle hypodensity in the deep MCA territories with involvement of the lenticulostriate arteries in the presence of ischemia. METHODS: Fifty CT images obtained within 3 hours after onset of embolic MCA occlusion were retrospectively reviewed by three neurosurgeons who were aware of clinical features. Early CT signs in the deep MCA territories were divided into three grades according to their anatomic location: grade I, normal basal ganglia with hypodensity localized to the insula; grade II, partial obscuration of the posterolateral part of the putamen; and grade III, hypodensity of the entire lentiform nucleus. A grade I CT sign was considered to be a negative finding for lenticulostriate artery involvement, whereas grade II and III CT signs were considered to be positive findings. Site of occlusion and involvement of the lenticulostriate arteries were confirmed by angiography. RESULTS: Thirty-eight (76%) of 50 patients had early CT signs in the deep MCA territories. Sensitivity and specificity of a grade I CT sign indicating absence of lenticulostriate artery involvement in ischemia were 65% and 87%, respectively. On the other hand, sensitivity and specificity of grade II and grade III CT signs for presence of lenticulostriate artery involvement in ischemia were 77% and 100%, respectively. Grade II CT signs resulted from various sites of occlusion, whereas grade III was unequivocally predictive of proximal occlusion to all of the lenticulostriate arteries. CONCLUSION: Involvement of the lenticulostriate arteries may be presumed by precise evaluation of subtle, CT-revealed hypodensity in the deep MCA territories, even within 3 hours of ischemic onset. (+info)
Small centrum ovale infarcts on diffusion-weighted magnetic resonance imaging. (3/36)BACKGROUND AND PURPOSE: A small centrum ovale infarct (SCOI), caused by occlusion of the white matter medullary arteries, is often equated with a lacunar infarct. We sought to clarify the clinical characteristics of a SCOI visualized by diffusion-weighted MRI (DWI) compared with those of a small basal ganglia infarct (SBGI). METHODS: Patients with a SCOI (SCOI group; n=38) or SBGI (SBGI group; n=68) < or =15 mm in diameter on conventional MRI and DWI were selected from 582 consecutive patients with acute ischemic stroke. Sex, age, neurological symptoms, vascular risk factors, emboligenic heart disease, arterial occlusive disease in the ipsilateral carotid system, and recurrent stroke within the initial 30 days were compared between the 2 groups. RESULTS: Only 47% of SCOIs but 87% of SBGIs could be identified with the use of conventional MRI, whereas DWI could detect them all. Age, sex, and vascular risk factors were not significantly different between the 2 groups. The SCOI group had more frequently an abrupt onset of symptoms (63% versus 26%; P=0.0002), emboligenic heart diseases (34% versus 12%; P=0.0054), occlusive carotid and/or middle cerebral artery diseases (53% versus 19%; P=0.0004), and recurrent stroke (13% versus 1%; P=0.0216) but less frequently a classic lacunar syndrome (50% versus 81%; P=0.0009) than the SBGI group. On a multivariate analysis, both arterial and heart diseases were independently associated with the SCOI group. CONCLUSIONS: Symptomatic SCOIs detected by DWI may be associated with large-vessel and heart diseases and should be distinguished from lacunar infarcts. (+info)
Cerebrovascular disease and evolution of depressive symptoms in the cardiovascular health study. (4/36)BACKGROUND AND PURPOSE: Previous studies have reported an association between cerebrovascular disease and depressive symptoms. The Cardiovascular Health Study (CHS) provides an opportunity to examine the relationship between vascular brain pathology seen on neuroimaging and changes in depressive symptoms. METHODS: The sample included 3236 CHS participants who had an MRI brain scan. Demographic variables, medical history, functional status, and apolipoprotein E genotype were obtained at baseline. Annual scores on a modified version of the Centers for Epidemiologic Studies Depression (CES-D) scale were obtained initially and up to 7 years subsequently. RESULTS: After controlling for important covariates, occurrence of depressive symptoms (defined as modified CES-D score of >7) was associated with small lesions in the basal ganglia, large cortical white-matter lesions, and severe subcortical white-matter grade. Neuroimaging variables did not predict incident depression among those who were nondepressive at the time of MRI. Persistence of depressive symptoms across 2 consecutive time points was associated with small basal ganglia lesions and large cerebral cortical white-matter lesions. Worsening of depression (increase in CES-D score of > or =5) was associated with subcortical white-matter lesions. CONCLUSIONS: These findings suggest that cerebrovascular disease at baseline is related to depression symptoms over time. Further studies are needed to investigate the differential effects of subcortical white- versus gray-matter lesions on mood. (+info)
Early and late postnatal identification of isolated lenticulostriate vasculopathy in preterm infants: associated findings. (5/36)OBJECTIVES: To determine the incidence, possible etiologies, and neurodevelopmental outcome of premature infants (<35 weeks) with isolated lenticulostriate vasculopathy (LSV). STUDY DESIGN: In a retrospective case-control design, we reviewed the medical records of all premature infants who were admitted to our neonatal intensive care unit between 1996 and 2000. RESULTS: The prevalence of LSV was 4.6% (21 of 453). Patients with late LSV (detected after 10 days of age) had less exposure than controls to prenatal steroids [42.8% (6 of 14) vs. 92.8% (13 of 14), respectively; p<0.01], and prenatal antibiotics [42.8% (6 of 14) vs. 85.7% (12 of 14), respectively; p=0.01]. Fifty-seven percent (8 of 14) of patients with late LSV had a low Apgar score vs. 14.2% (2 of 14) of the control group (p=0.01). Patients with LSV also had more muscle tone abnormalities than controls at 6 months of age [33.3% (5 of 15) vs. 5.2% (1 of 19), respectively; p=0.03]. CONCLUSION: Patients with late LSV have less exposure to antenatal steroids and antibiotics, lower Apgar scores, and abnormal muscle tone at 6 months of age. (+info)
Heterogeneity of cerebral blood flow in Alzheimer disease and vascular dementia. (6/36)BACKGROUND AND PURPOSE: Alzheimer disease (AD) and vascular dementia (VaD) are the two major diseases that cause dementia, and early diagnosis is important. Single photon emission CT (SPECT) of cerebral blood flow (CBF) is used for the early detection of dementia and as an auxiliary method for follow-up. AD shows reduced posterior blood flow and VaD manifests reduced anterior blood flow on CBF SPECT images. We examined the usefulness of 3D fractal analysis of CBF SPECT images to objectively quantify the heterogeneity of CBF in patients with AD and VaD. METHODS: Thirty-two patients with AD and 22 with VaD based on neuropsychologic tests and imaging findings, as well as 20 age-matched control subjects underwent technetium-99m hexamethyl propyleneamine oxime CBF SPECT. We then conducted statistical image processing by 3D fractal analysis on reconstructed data. Fractal dimension, an index of heterogeneity, was then calculated for the whole brain, as well as for the anterior and posterior regions of the brain. A higher fractal dimension indicates that the CBF SPECT image is uneven. The ratio of fractal dimension of the anterior region to fractal dimension of the posterior region (A/P ratio) was calculated. Heterogeneity of CBF was compared among the AD, VaD, and control groups. RESULTS: Fractal dimensions of the AD, VaD, and control groups were 1.072+/-0.179 (mean +/- SD), 1.005+/-0.156, and 0.806+/-0.06, respectively. A significant difference of fractal dimension was noted between the control group and the two types of dementia (P<.0001); however, no significant difference was noted between the AD and VaD groups. The A/P ratios of the AD and VaD groups were significantly different (0.952 and 1.163, respectively; P<.01). CONCLUSION: Analysis of CBF SPECT images quantitatively showed that the fractal dimension was significantly higher (indicating heterogeneity) in patients with AD and VaD when compared with age-matched control subjects. Comparison of the A/P ratio on CBF SPECT images between AD and VaD groups showed that the heterogeneity of CBF was posterior-dominant for AD and anterior-dominant for VaD. Thus, 3D fractal analysis enabled a simple and objective evaluation of the heterogeneity of CBF in patients with AD and VaD. (+info)
Neonatal lenticulostriate vasculopathy: further characterisation. (7/36)BACKGROUND: Lenticulostriate vasculopathy (LSV) is sometimes detected on routine brain ultrasonography in neonates, and is often associated with various perinatal and neonatal abnormalities. However, most reports on LSV are retrospective with no controls. OBJECTIVES: To compare the perinatal and neonatal clinical characteristics of neonates with LSV with matched controls and to summarise all published reports of LSV. DESIGN: A prospective study that summarises the clinical, laboratory, and neurosonographic data of neonates with LSV. METHODS: Of 1184 neonates admitted to the neonatal intensive care unit (NICU) during a three year period, 857 had a routine head ultrasound examination. Twenty one had LSV, and were compared with 42 matched controls with regard to gestational, perinatal, neonatal, laboratory, and neurosonographic characteristics. RESULTS: LSV was detected in 21 of the 857 (2.45%) neonates. It was bilateral in 10 of the 21 cases and located in the thalamus (n = 14) and basal ganglia (n = 7). Infants with LSV were not significantly different from matched controls in most tested variables. However, compared with the control group, the LSV group included significantly more multiple births and more disturbances in amniotic fluid volume, but less meconial amniotic fluid. In addition, the patients with LSV required fewer blood transfusions and less phototherapy. CONCLUSIONS: Except for more multiple births, neonates with LSV did not display more adverse findings than their matched controls. (+info)
The L-dopa response in vascular parkinsonism. (8/36)OBJECTIVE: To determine whether a positive L-dopa response in vascular parkinsonism (VP) is correlated with the presence of nigrostriatal pathology due to either vascular damage or neuronal cell loss. METHODS: Seventeen patients with pathologically confirmed VP were selected from the pathological collection of the Queen Square Brain Bank for Neurological Disorders, and their L-dopa response during life was compared with the presence of macroscopic vascular damage in the nigrostriatal pathway and microscopic substantia nigra cell loss. RESULTS: Ten of the twelve patients with a good or excellent response had macroscopic infarcts or lacunae caused by enlarged perivascular spaces in the basal ganglia or microscopic neuronal cell loss in the substantia nigra. In contrast, only one of the five patients with a moderate or no response had lacunae in the putamen, and none had lacunar infarcts or substantia nigra cell loss. CONCLUSION: These results suggest that a substantial number of patients with clinically suspected VP may respond with benefit to dopaminergic therapy, especially those with lesions in or close to the nigrostriatal pathway. (+info)
The term "cerebrovascular disease" refers specifically to conditions that affect the blood vessels of the brain, such as stroke, cerebral vasculitis, and Moyamoya disease. The basal ganglia are particularly vulnerable to cerebrovascular disease because they rely heavily on a constant supply of oxygen and nutrients from the bloodstream.
Basal ganglia cerebrovascular disease can result from a variety of causes, including:
1. Stroke or bleeding in the brain: A stroke or bleed in the brain can damage the basal ganglia and lead to basal ganglia cerebrovascular disease.
2. Cerebral vasculitis: Inflammation of the blood vessels supplying the basal ganglia can cause damage and lead to basal ganglia cerebrovascular disease.
3. Moyamoya disease: A rare condition caused by narrowing or blockage of the internal carotid artery and its branches, leading to decreased blood flow to the brain and basal ganglia.
4. Other conditions that affect blood flow to the brain, such as hypoperfusion or vasospasm.
The symptoms of basal ganglia cerebrovascular disease can vary depending on the severity and location of the damage, but may include:
1. Difficulty with movement, including weakness or paralysis on one side of the body (hemiparesis) or difficulty with coordination and balance.
2. Cognitive impairment, including memory loss, confusion, and difficulty with problem-solving.
3. Behavioral changes, such as depression, anxiety, or personality changes.
4. Seizures or other neurological symptoms, depending on the location and severity of the damage.
Diagnosis of basal ganglia cerebrovascular disease typically involves a combination of physical examination, medical history, and imaging studies such as CT or MRI scans, as well as blood flow studies such as SPECT or PET scans. Treatment depends on the underlying cause and may include medications to control symptoms, surgery to repair or bypass blocked blood vessels, or other interventions as appropriate.
1. Stroke: A stroke occurs when the blood supply to the brain is interrupted, either due to a blockage or a rupture of the blood vessels. This can lead to cell death and permanent brain damage.
2. Cerebral vasospasm: Vasospasm is a temporary constriction of the blood vessels in the brain, which can occur after a subarachnoid hemorrhage (bleeding in the space surrounding the brain).
3. Moyamoya disease: This is a rare condition caused by narrowing or blockage of the internal carotid artery and its branches. It can lead to recurrent transient ischemic attacks (TIs) or stroke.
4. Cerebral amyloid angiopathy: This is a condition where abnormal protein deposits accumulate in the blood vessels of the brain, leading to inflammation and bleeding.
5. Cavernous malformations: These are abnormal collections of blood vessels in the brain that can cause seizures, headaches, and other symptoms.
6. Carotid artery disease: Atherosclerosis (hardening) of the carotid arteries can lead to a stroke or TIAs.
7. Vertebrobasilar insufficiency: This is a condition where the blood flow to the brain is reduced due to narrowing or blockage of the vertebral and basilar arteries.
8. Temporal lobe dementia: This is a type of dementia that affects the temporal lobe of the brain, leading to memory loss and other cognitive symptoms.
9. Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL): This is a rare genetic disorder that affects the blood vessels in the brain, leading to recurrent stroke-like events.
10. Moyamoya disease: This is a rare condition caused by narrowing or blockage of the internal carotid artery and its branches, leading to decreased blood flow to the brain and increased risk of stroke.
It's important to note that this list is not exhaustive and there may be other causes of stroke and TIAs that are not included here. A proper diagnosis can only be made by a qualified medical professional after conducting a thorough examination and reviewing the individual's medical history.
Some examples of basal ganglia diseases include:
1. Parkinson's disease: A neurodegenerative disorder characterized by tremors, rigidity, bradykinesia (slow movement), and postural instability.
2. Huntington's disease: An autosomal dominant disorder that causes progressive degeneration of the basal ganglia and a decline in cognitive, motor, and psychiatric functions.
3. Dystonia: A movement disorder characterized by sustained or intermittent muscle contractions that cause abnormal postures or movements.
4. Tourette's syndrome: A neurodevelopmental disorder characterized by multiple motor tics and at least one vocal tic, such as repeated sounds or words.
5. Obsessive-compulsive disorder (OCD): An anxiety disorder characterized by recurring thoughts or compulsions to perform repetitive behaviors.
6. Schizophrenia: A psychotic disorder characterized by hallucinations, delusions, and cognitive impairments.
7. Kleine-Levin syndrome: A rare sleep disorder characterized by recurring periods of excessive sleepiness and automatic behaviors.
8. Wilson's disease: A rare genetic disorder caused by copper accumulation in the basal ganglia, leading to cognitive and motor impairments.
9. Hemiballism: A rare movement disorder characterized by unilateral or bilateral involuntary movements of the upper limbs.
10. Chorea-acanthocytosis: A rare genetic disorder characterized by chorea (involuntary movements), acanthocytosis (abnormal red blood cell shape), and cognitive decline.
These conditions are often challenging to diagnose and manage, and may require a comprehensive evaluation by a multidisciplinary team of healthcare professionals, including neurologists, psychiatrists, geneticists, and other specialists. Early diagnosis and appropriate treatment can help improve outcomes for individuals with these conditions.
1. Ischemic stroke: This is the most common type of stroke, accounting for about 87% of all strokes. It occurs when a blood vessel in the brain becomes blocked, reducing blood flow to the brain.
2. Hemorrhagic stroke: This type of stroke occurs when a blood vessel in the brain ruptures, causing bleeding in the brain. High blood pressure, aneurysms, and blood vessel malformations can all cause hemorrhagic strokes.
3. Transient ischemic attack (TIA): Also known as a "mini-stroke," a TIA is a temporary interruption of blood flow to the brain that lasts for a short period of time, usually less than 24 hours. TIAs are often a warning sign for a future stroke and should be taken seriously.
Stroke can cause a wide range of symptoms depending on the location and severity of the damage to the brain. Some common symptoms include:
* Weakness or numbness in the face, arm, or leg
* Difficulty speaking or understanding speech
* Sudden vision loss or double vision
* Dizziness, loss of balance, or sudden falls
* Severe headache
* Confusion, disorientation, or difficulty with memory
Stroke is a leading cause of long-term disability and can have a significant impact on the quality of life for survivors. However, with prompt medical treatment and rehabilitation, many people are able to recover some or all of their lost functions and lead active lives.
The medical community has made significant progress in understanding stroke and developing effective treatments. Some of the most important advances include:
* Development of clot-busting drugs and mechanical thrombectomy devices to treat ischemic strokes
* Improved imaging techniques, such as CT and MRI scans, to diagnose stroke and determine its cause
* Advances in surgical techniques for hemorrhagic stroke
* Development of new medications to prevent blood clots and reduce the risk of stroke
Despite these advances, stroke remains a significant public health problem. According to the American Heart Association, stroke is the fifth leading cause of death in the United States and the leading cause of long-term disability. In 2017, there were over 795,000 strokes in the United States alone.
There are several risk factors for stroke that can be controlled or modified. These include:
* High blood pressure
* Diabetes mellitus
* High cholesterol levels
* Lack of physical activity
* Poor diet
In addition to these modifiable risk factors, there are also several non-modifiable risk factors for stroke, such as age (stroke risk increases with age), family history of stroke, and previous stroke or transient ischemic attack (TIA).
The medical community has made significant progress in understanding the causes and risk factors for stroke, as well as developing effective treatments and prevention strategies. However, more research is needed to improve outcomes for stroke survivors and reduce the overall burden of this disease.
Examples of Nervous System Diseases include:
1. Alzheimer's disease: A progressive neurological disorder that affects memory and cognitive function.
2. Parkinson's disease: A degenerative disorder that affects movement, balance and coordination.
3. Multiple sclerosis: An autoimmune disease that affects the protective covering of nerve fibers.
4. Stroke: A condition where blood flow to the brain is interrupted, leading to brain cell death.
5. Brain tumors: Abnormal growth of tissue in the brain.
6. Neuropathy: Damage to peripheral nerves that can cause pain, numbness and weakness in hands and feet.
7. Epilepsy: A disorder characterized by recurrent seizures.
8. Motor neuron disease: Diseases that affect the nerve cells responsible for controlling voluntary muscle movement.
9. Chronic pain syndrome: Persistent pain that lasts more than 3 months.
10. Neurodevelopmental disorders: Conditions such as autism, ADHD and learning disabilities that affect the development of the brain and nervous system.
These diseases can be caused by a variety of factors such as genetics, infections, injuries, toxins and ageing. Treatment options for Nervous System Diseases range from medications, surgery, rehabilitation therapy to lifestyle changes.
The term ischemia refers to the reduction of blood flow, and it is often used interchangeably with the term stroke. However, not all strokes are caused by ischemia, as some can be caused by other factors such as bleeding in the brain. Ischemic stroke accounts for about 87% of all strokes.
There are different types of brain ischemia, including:
1. Cerebral ischemia: This refers to the reduction of blood flow to the cerebrum, which is the largest part of the brain and responsible for higher cognitive functions such as thought, emotion, and voluntary movement.
2. Cerebellar ischemia: This refers to the reduction of blood flow to the cerebellum, which is responsible for coordinating and regulating movement, balance, and posture.
3. Brainstem ischemia: This refers to the reduction of blood flow to the brainstem, which is responsible for controlling many of the body's automatic functions such as breathing, heart rate, and blood pressure.
4. Territorial ischemia: This refers to the reduction of blood flow to a specific area of the brain, often caused by a blockage in a blood vessel.
5. Global ischemia: This refers to the reduction of blood flow to the entire brain, which can be caused by a cardiac arrest or other systemic conditions.
The symptoms of brain ischemia can vary depending on the location and severity of the condition, but may include:
1. Weakness or paralysis of the face, arm, or leg on one side of the body
2. Difficulty speaking or understanding speech
3. Sudden vision loss or double vision
4. Dizziness or loss of balance
5. Confusion or difficulty with memory
7. Slurred speech or inability to speak
8. Numbness or tingling sensations in the face, arm, or leg
9. Vision changes, such as blurred vision or loss of peripheral vision
10. Difficulty with coordination and balance.
It is important to seek medical attention immediately if you experience any of these symptoms, as brain ischemia can cause permanent damage or death if left untreated.
The exact cause of paraneoplastic syndromes is not fully understood, but it is believed that the immune system mistakenly attacks healthy cells in the nervous system, leading to damage and dysfunction. Some research suggests that certain types of cancer may trigger an autoimmune response, while other factors such as genetics or environmental exposures may also play a role.
Paraneoplastic syndromes can be difficult to diagnose, as they often present with symptoms that are similar to those of more common conditions such as multiple sclerosis or stroke. However, certain tests such as electromyography (EMG) and nerve conduction studies (NCS) can help rule out other conditions and confirm the presence of a paraneoplastic syndrome.
Treatment for paraneoplastic syndromes typically focuses on managing symptoms and addressing any underlying cancer that may be present. Medications such as corticosteroids, immunosuppressive drugs, and chemotherapy may be used to reduce inflammation and suppress the immune system, while surgery or radiation therapy may be necessary to remove cancerous tissue. In some cases, plasmapheresis (plasma exchange) may also be recommended to remove harmful antibodies from the blood.
Overall, paraneoplastic syndromes, nervous system are a complex and rare group of disorders that can significantly impact quality of life. Early diagnosis and treatment are key to managing symptoms and improving outcomes for patients with these conditions.
List of MeSH codes (C10)
List of MeSH codes (C14)
HIV-associated neurocognitive disorder
Hypertension and the brain
Glutaric aciduria type 1
List of women neuroscientists
Cerebral autosomal recessive arteriopathy with subcortical infarcts and leukoencephalopathy
Differential diagnoses of anorexia nervosa
Safety of electronic cigarettes
List of skin conditions
Glossary of medicine
DeCS - Termos Novos
Moyamoya Disease | National Institute of Neurological Disorders and Stroke
Tardive Dyskinesia: Overview, Pathophysiology, Etiology
Ischemic Stroke - Neurologic Disorders - MSD Manual Professional Edition
PAR-18-413: Mechanistic Basis of Diffuse White Matter Disease and Small Vessel Pathology in Vascular Contributions to Cognitive...
Pesquisa | Portal Regional da BVS
DeCS - Termos Novos
DeCS - Termos Novos
Cerebrovascular Lesions in Alzheimer-associated Diseases - A Neuropathological Study with 7.0-tesla Magnetic Resonance Imaging ...
Intracranial Hemorrhages - MeSH - NCBI
Search | SciELO
A/T/N: An unbiased descriptive classification scheme for Alzheimer disease biomarkers | Neurology
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Primary familial brain calcification: MedlinePlus Genetics
Virology, Pathology, and Clinical Manifestations of West Nile Virus Disease - Volume 11, Number 8-August 2005 - Emerging...
Intracerebral Hemorrhage - FindZebra
Acute extrapyramidal syndrome in methylmalonic acidemia: 'metabolic stroke' involving the globus pallidus - PubMed
Free neuro anatomy Books Download | Ebooks Online Textbooks
NCIt Code NCIt PT NICHD AE PT NICHD AE SY NCIt Definition NICHD AE Definition NCIt Code of NICHD Parent NICHD Parent PT NCIt...
November | 2016 | Cdk receptor
Alcohol's Effects on the Brain: Neuroimaging Results in Humans and Animal Models | Alcohol Research: Current Reviews
Descriptors in 2013 MeSH. Preferred term only. December 14, 2012
Right basal ganglia2
- Axial T2-weighted MR images revealed increased signal intensity in the right basal ganglia region consistent with an acute infarct. (ajnr.org)
- Imaging studies revealed Occluded right Common carotid artery, occluded right subclavian artery and stenotic right renal artery and MRI showed Acute Infarcts in Right Basal Ganglia and Right High Parietal Region, Hemorrhagic infarct in right MCA subcortical area.The diagnosis of Takayasu arteritis with recent cerebrovascular accident (left hemiparesis) with hypertension was made and the patient was started on steroids, anti-platelets, anti-hypertensives and physiotherapy. (who.int)
Small Vessel Disease3
- To investigate the relationship between diffusion tensor imaging (DTI) indicators and cerebral small vessel disease (CSVD) with depressive states, and to explore the underlying mechanisms of white matter damage in CSVD with depression. (biomedcentral.com)
- With the gradual increase in the number of elderly people in China, cardiovascular and cerebrovascular diseases are frequent, and the incidence of cerebral small vessel disease (CSVD) is also on the rise. (biomedcentral.com)
- Prevalence and Significance of the Vessel-Cluster Sign on Susceptibility-Weighted Imaging in Patients With Severe Small Vessel Disease. (svds-at-target.eu)
- This note covers the following topics: Normal Brain, Cerebrovascular Disease, Neoplastic Disease, Degenerative Disease and Inflammatory or Infectious Disease. (freebookcentre.net)
- The MCA is by far the largest cerebral artery and is the vessel most commonly affected by cerebrovascular accident (CVA). (medscape.com)
- Acute necrotizing encephalopathy , rare disease that occurs following a viral infection. (wikipedia.org)
- CLASSIFICATION OF DISEASES AND INJURIES I. INFECTIOUS AND PARASITIC DISEASES (001-139) Includes: diseases generally recognized as communicable or transmissible as well as a few diseases of unknown but possibly infectious origin Excludes: acute respiratory infections (460-466) influenza (487. (cdc.gov)
- Self-Administered Sphenopalatine Ganglion Blocks utilize cotton-tipped catheters that offer continual capillary feed of anesthetic to the same area the Sphenocath and TX360 but instead of requiring visit to ER or physicians office the patient can do them without leaving their house. (sphenopalatineganglionblocks.com)
- This review highlights new information regarding the virology, clinical manifestations, and pathology of WNV disease, which will provide a new platform for further research into diagnosis, treatment, and possible prevention of WNV through vaccination. (cdc.gov)
- Spenopalatine Ganglion Blocks were originally discovered by Dr Greenfield Sluder and in 1908 he published his first paper on how they were used to treat Sluder's Neuralgia which was a type of orofacial pain similar to some TMD disorders, Cluster Headaches and Migraines. (sphenopalatineganglionblocks.com)
- Vinpocetine is used as a prescription drug in Japan, Europe, Mexico and Russia for the treatment of cognitive and cerebrovascular disorders. (nootropicsexpert.com)
- Researchers suggest that calcium deposits lead to the features of primary familial brain calcification by disrupting the connections between the basal ganglia and other areas of the brain, particularly the frontal lobes. (medlineplus.gov)
- This study note consist of 19 parts.Parts 1 through 4 are devoted to the brain stem and cerebellum, parts 5 through 8 to the basal ganglia, thalamus, hypothalamus and limbic system, and parts 9 through 12 to the Frontal, Parietal, Temporal and Occipital lobes. (freebookcentre.net)
- Postmortem analysis of a third patient also showed TNFα expression in neurones of the frontal cortex and basal ganglia. (bmj.com)
- HIV effects on age-associated neurocognitive dysfunction: premature cognitive aging or neurodegenerative disease? (biomedcentral.com)
- Imaging and histopathological studies have demonstrated that structural changes of the retina affect subjects with Alzheimer's disease (AD) or mild cognitive impairment (MCI). (nature.com)
- Alzheimer's disease is preceded by a variable transitional phase, which affects older subjects with heterogeneous cognitive and functional impairment, the latter not crossing the threshold for dementia. (nature.com)
Coronavirus disease 20192
- Periarteriolar spaces modulate cerebrospinal fluid transport into brain and demonstrate altered morphology in aging and Alzheimer's disease. (svds-at-target.eu)
- Yet notable differences also exist between the clinical presentation and brain disturbances occurring with HIV and those occurring in neurodegenerative conditions such as Alzheimer's disease. (biomedcentral.com)
- These calcium deposits are visible only on medical imaging and typically occur in the basal ganglia, which are structures deep within the brain that help start and control movement of the body. (medlineplus.gov)
- Premature age-associated neurocognitive decline appears to be related to structural and functional brain changes on neuroimaging, and of particular concern is the fact that pathology indicative of neurodegenerative disease has been shown to occur in the brains of HIV-infected people. (biomedcentral.com)
- Pathological processes or diseases where cerebral MICROVESSELS show abnormalities. (bvsalud.org)
- Fertility is not adversely affected and pregnancy does not appear to exacerbate the disease, although management of hypertension is essential. (who.int)
- Background: Persons living with HIV (PLWH) are more likely to develop hypertension and cardiovascular disease than the HIV-negative population. (bvsalud.org)
- Note: The number of publications displayed in this table will differ from the number displayed in the HuGE Literature Finder as the number in Genopedia reflects only the indexed disease term without children terms, but the number in the HuGE Literature Finder reflects all text searches of the disease term including the indexed term and corresponding children terms. (cdc.gov)
- Note - Conditions arising in the perinatal - - fetus, fetal period, even though death or morbidity occurs - - - causing disproportion later, should, as far as possible, be coded to - - - - affecting fetus or newborn P03.1 chapter XVI, which takes precedence over - - forces of labor chapters containing codes for diseases by - - - affecting fetus or newborn P03.6 their anatomical site. (cdc.gov)
- certain localized infections Note: Categories for "late effects" of infectious and parasitic diseases are to be found at 137. (cdc.gov)
Brain and nervous system1
- Our vision is to prevent and cure disease and disability of the brain and nervous system. (edu.au)
- ἐνκέφαλος "brain" + πάθος "suffering") means any disorder or disease of the brain , especially chronic degenerative conditions. (wikipedia.org)
- In 1924, a filterable agent from human brain tissue was isolated in rabbits and in 1934, Hayashi transmitted the disease experimentally to monkeys by intracerebral inoculation (9). (cdc.gov)
- Manifestations range from asymptomatic disease, to catastrophic strokes. (who.int)
- Since it was first detected in New York City in 1999, and through 2004, 16,000 WNV disease cases have been reported in the United States. (cdc.gov)
- Conditions associated with CVT can be classified as either predisposing (eg, genetic prothrombotic diseases, antiphospholipid syndrome, cancer) or precipitating (eg, oral contraceptives, infections). (lww.com)
-   In the relapsing forms of MS, between attacks, symptoms may disappear completely, although some permanent neurological problems often remain, especially as the disease advances. (alquds.edu)
- The expanded knowledge about WNV disease provides a new platform for future development of diagnostic tests, therapy, and vaccine development. (cdc.gov)
- The SPG's also have somatosensory nerves from the Trigeminal Nervous system and Sympathetic fiber that come from the Cervical Sympathetic Ganglion Chain including fibers originating in the Stellate Ganglion. (sphenopalatineganglionblocks.com)
- Not Just Blood: Brain Fluid Systems and Their Relevance to Cerebrovascular Diseases. (svds-at-target.eu)
- AIDS-like syndrome: AIDS-like disease (illness) (syndrome) ARC AIDS-related complex Pre-AIDS AIDS-related conditions Prodromal-AIDS 3. (cdc.gov)
- OBJECTIVE Cerebral venous thrombosis (CVT), thrombosis of the dural sinus, cerebral veins, or both, is a rare cerebrovascular disease. (lww.com)
- It is provided as an additional code where it is desired to identify the bacterial agent in diseases classified elsewhere. (cdc.gov)
- They are basically modified squirt guns that deliver liquid anesthetic over the mucosa covering the medial wall of the Pterygpopaltine Fossa which houses the Sphenopalatine Ganglion as well as the Maxillary Nerve a major sensory division of the Trigeminal Nerve. (sphenopalatineganglionblocks.com)
- Japanese encephalitis (JE) is a mosquito-borne arboviral disease of major public health importance in Asia. (cdc.gov)
- Multiple sclerosis ( MS ) is the most common demyelinating disease ,  in which the insulating covers of nerve cells in the brain and spinal cord are damaged. (alquds.edu)