Echocardiography, Doppler, Pulsed
Ventricular Function, Left
Blood Flow Velocity
Ventricular Dysfunction, Left
Effects of pacing-induced and balloon coronary occlusion ischemia on left atrial function in patients with coronary artery disease. (1/343)OBJECTIVES: The aim of this study was to compare left atrial (LA) function in 16 patients with distal left anterior descending (LAD) and in 16 patients with proximal left circumflex (LCx) coronary artery stenosis at rest and immediately after pacing-induced tachycardia (LAD-pacing [P] and LCx-P) or coronary occlusion (LAD-CO and LCx-CO). BACKGROUND: During left ventricular (LV) ischemia, compensatory augmentation of LA contraction enhances LV filling and performance. The left atrium is supplied predominantly by branches arising from the LCx. Therefore, we hypothesized that one mechanism for the loss of atrial contraction may be ischemic LA dysfunction. METHODS: Left ventricular and LA pressure-area relations were derived from simultaneous double-tip micromanometer pressure recordings and automatic boundary detection echocardiograms. RESULTS: Immediately after pacing or after coronary occlusion, LV end-diastolic pressure, LV relaxation, LA mean pressure and LV stiffness significantly increased in all patients. However, the area of the A loop of the LA pressure-area relation, representing the LA pump function, significantly decreased in groups LCx-P and LCx-CO (from 14+/-3 to 9+/-2, and from 16+/-4 to 9+/-2 mm Hg.cm2, respectively, p < 0.05), whereas it increased in groups LAD-P and LAD-CO (from 12+/-3 to 54+/-10, and from 16+/-3 to 49+/-8 mm Hg.cm2, respectively, p < 0.001). CONCLUSIONS: In patients with LAD stenosis, LV supply or demand ischemia is associated with enhanced LA pump function. However, in patients with proximal LCx stenosis who develop the same type and degree of ischemia, LA branches might have been affected, rendering the LA ischemic and unable to increase its booster pump function. (+info)
Pulmonary venous flow in hypertrophic cardiomyopathy as assessed by the transoesophageal approach. The additive value of pulmonary venous flow and left atrial size variables in estimating the mitral inflow pattern in hypertrophic cardiomyopathy. (2/343)AIMS: This study was conducted to assess the characteristics of the pattern of pulmonary venous flow and to document the interaction of this flow and left atrial function with the pattern of mitral inflow in hypertrophic cardiomyopathy. METHODS AND RESULTS: Pulmonary venous and mitral flows were evaluated by the transoesophageal approach in 80 patients with hypertrophic cardiomyopathy. Left atrial size and function were measured by the transthoracic approach. Their values were compared with those obtained from 35 normal controls. Twelve patients showed significant (> 2+) mitral regurgitation. As a group, hypertrophic cardiomyopathy patients showed increased atrial reversal flow and longer deceleration time of the diastolic wave, but a wide variability of pulmonary venous flow patterns were observed. Thirty patients (37.5%) had pseudonormal mitral flow patterns. Stepwise multilinear regression analysis identified the ratio of systolic to diastolic pulmonary venous flow velocity, the ratio of velocity-time integrals of both flow waves at atrial contraction, the left atrial minimal volume and the systolic fraction as independent predictive variables of the mitral E/A wave velocity ratio (r = 0.82). By logistic regression, the former three variables were selected as independent predictive covariates of a pseudonormal mitral flow pattern (sensitivity: 83%, specificity: 90%). The ratio of velocity-time integrals of both atrial waves was the most important predictive variable in both analyses. CONCLUSIONS: The observed variability in the configuration of pulmonary venous flow velocity waveform is related to what occurs in transmitral flow in patients with hypertrophic cardiomyopathy. Significant mitral regurgitation is not an independent correlate of pseudonormal mitral inflow patterns in these patients. Our results further emphasize the complementary, additive value of the pulmonary venous flow velocity pattern and left atrial size in the interpretation of the mitral flow velocity pattern, and indirectly suggest the underlying increased left ventricular filling pressures of patients with hypertrophic cardiomyopathy and pseudonormal mitral flow patterns. (+info)
Doppler sonographic evaluation of left atrial function after cardioversion of atrial fibrillation. (3/343)Restoration of sinus rhythm is not always followed by immediate return of effective atrial contraction. Left atrial mechanical function can be assessed by Doppler echocardiography; in the present study we measured the atrial ejection force, which is a noninvasive Doppler-derived parameter that measures the strength of atrial contraction. The aim of the present study was to evaluate the influence of clinical and echocardiographic parameters: duration and cause of atrial fibrillation, different modality of cardioversion, and left atrial size with respect to the delay in the return of effective atrial contraction after cardioversion. Seventy patients were randomly chosen to undergo cardioversion by either direct current shock or intravenously administered procainamide hydrochloride. The 52 patients who had sinus rhythm restored underwent a complete Doppler echocardiographic examination 1 h after the restoration of sinus rhythm and after 1 day, 7 days, and 1 month. The relation between clinical variables and atrial ejection force was tested. Atrial ejection force was greater immediately and 24 h after cardioversion in patients who underwent pharmacologic therapy compared to patients treated with direct current shock (11.3+/-3 versus 5+/-2.9 dynes; P<0.001). In both groups atrial ejection force increased over time. The mode of cardioversion was significantly associated with recovery of left atrial mechanical function by day 1 in univariate and multivariate analysis (odds ratio, 0.14; 95% confidence interval, 0.02-1.2). The other variable associated with the delay in the recovery of atrial function was a dilated left atrium (odds ratio, 0.16; 95% confidence interval, 0.12-1.6). Atrial ejection force is a noninvasive parameter that can be easily measured after cardioversion and gives accurate information about the recovery of left atrial mechanical function. The recovery of left atrial function was influenced by the mode of cardioversion and left atrial size. (+info)
Importance of left atrial appendage flow as a predictor of thromboembolic events in patients with atrial fibrillation. (4/343)AIM: The purpose of this study was to investigate the role of transoesophageal echocardiography in predicting subsequent thromboembolic events in patients with atrial fibrillation. METHODS AND PATIENTS: Transoesophageal echocardiography was performed in 88 patients with documented paroxysmal (n=53) or chronic atrial fibrillation (n=35) to assess morphological and functional predictors of thromboembolic events. Prospective selection was from patients with non-valvular atrial fibrillation who had undergone transoesophageal echocardiography because of previous thromboembolism (n=30); prior to electrical cardioversion (n=31); or for other reasons (n=27). All patients were followed up for 1 year. RESULTS: During the period of follow-up new thromboembolic events occurred in 18 of 88 patients (20%/year); 16 of these patients had a stroke and two a peripheral embolism. Univariate analysis revealed that previous thromboembolism (P<0.005; odds ratio 5.3 [CI 1.9, 12. 1]), history of hypertension (P<0.01; odds ratio 4.0 [CI 1.4, 10.4), presence of left atrial spontaneous echo contrast (P<0.025; odds ratio 3.5 [CI 1.2, 10.0]), and presence of left atrial appendage peak velocity +info)
Short-term effect of atrial fibrillation on atrial contractile function in humans. (5/343)BACKGROUND: Conversion of chronic atrial fibrillation (AF) is associated with atrial stunning, but the short-term effect of a brief episode of AF on left atrial appendage (LAA) emptying velocity is unknown. The purpose of this study was to determine whether a short episode of AF affects left atrial function and whether verapamil modifies this effect. METHODS AND RESULTS: The subjects of this study were 19 patients without structural heart disease undergoing an electrophysiology procedure. In 13 patients, LAA emptying velocity was measured by transesophageal echocardiography in the setting of pharmacological autonomic blockade before, during, and after a short episode of AF. During sinus rhythm, the baseline LAA emptying velocity was measured 5 times and averaged. AF was then induced by rapid right atrial pacing. After either spontaneous or electrical conversion, LAA emptying velocity was measured immediately on resumption of sinus rhythm and every minute thereafter. The mean duration of AF was 15.3+/-3.8 minutes. The mean baseline emptying velocity was 70+/-20 cm/s. The first post-AF emptying velocity was 63+/-20 cm/s (P=0.02 versus baseline emptying velocity). The post-AF emptying velocity returned to the baseline emptying velocity value after 3.0 minutes. The mean percent reduction in post-AF emptying velocity was 9.7+/-21% (range, 15% increase to 56% decrease). A second group of 6 patients were pretreated with verapamil (0.1-mg/kg IV bolus followed by an infusion of 0.005 mg. kg-1. min-1). In these patients, the first post-AF emptying velocity, 58+/-14 cm/s, was not significantly different from the pre-AF emptying velocity, 60+/-13 cm/s (P=0.08). CONCLUSIONS: In humans, several minutes of AF may be sufficient to induce atrial contractile dysfunction after cardioversion. When atrial contractile dysfunction occurs, there is recovery of AF within several minutes. AF-induced contractile dysfunction is attenuated by verapamil and may be at least partially mediated by cellular calcium overload. (+info)
Left atrial relaxation and left ventricular systolic function determine left atrial reservoir function. (6/343)BACKGROUND: Determinants of left atrial (LA) reservoir function and its influence on left ventricular (LV) function have not been quantified. METHODS AND RESULTS: In an open-pericardium, paced (70 and 90 bpm) pig model of LV regional ischemia (left anterior descending coronary constriction), with high-fidelity LV, LA, and RV pressure recordings, we obtained the LA area with 2D automated border detection echocardiography, LA pressure-area loops, and Doppler transmitral flow. We calculated LV tau, LA relaxation (a-x pressure difference divided by time, normalized by a pressure), and stiffness (slope between x and v pressure points of v loop). Determinants of total LA reservoir (maximum-minimum area, cm(2)) were identified by multiple regression analysis. Different mean rates of LA area increase identified 2 consecutive (early rapid and late slow) reservoir phases. During ischemia, LV long-axis shortening (LAS, LV base systolic descent) and LA reservoir area change decreased (7.3+/-0.3 [SEM] versus 5.6+/-0.3 cm(2), P<0.001) and LA stiffness increased (1.6+/-0.3 versus 3.1+/-0.3 mm Hg/cm(2), P=0.009). Early reservoir area change depended on LA mean ejection rate (LA area at ECG P wave minus minimum area divided by time; multiple regression coefficient=0.9; P<0.001) and relaxation (coefficient=4.9 cm(2)xms/s; P<0.001). Late reservoir area change depended on LAS (coefficient=8 cm/s; P<0.001). Total reservoir filling depended on LA stiffness (coefficient=-0.31 cm(4)/mm Hg; P=0. 001) and cardiac output (coefficient=0.001 cm(2)xmin/L; P=0.002). The strongest predictor of cardiac output was LA reservoir filling (coefficient=301 L/minxcm(2); P<0.001). The v loop area was determined by cardiac output, LV ejection time, tau, and early transmitral flow. CONCLUSIONS: Two (early and late) reservoir phases are determined by LA contraction and relaxation and LV base descent. Acute LV regional ischemia increases LA stiffness and impairs LA reservoir function by reducing LV base descent. (+info)
Noninvasive assessment of left atrial maximum dP/dt by a combination of transmitral and pulmonary venous flow. (7/343)OBJECTIVES: The study assessed whether hemodynamic parameters of left atrial (LA) systolic function could be estimated noninvasively using Doppler echocardiography. BACKGROUND: Left atrial systolic function is an important aspect of cardiac function. Doppler echocardiography can measure changes in LA volume, but has not been shown to relate to hemodynamic parameters such as the maximal value of the first derivative of the pressure (LA dP/dt(max)). METHODS: Eighteen patients in sinus rhythm were studied immediately before and after open heart surgery using simultaneous LA pressure measurements and intraoperative transesophageal echocardiography. Left atrial pressure was measured with a micromanometer catheter, and LA dP/dt(max) during atrial contraction was obtained. Transmitral and pulmonary venous flow were recorded by pulsed Doppler echocardiography. Peak velocity, and mean acceleration and deceleration, and the time-velocity integral of each flow during atrial contraction was measured. The initial eight patients served as the study group to derive a multilinear regression equation to estimate LA dP/dt(max) from Doppler parameters, and the latter 10 patients served as the test group to validate the equation. A previously validated numeric model was used to confirm these results. RESULTS: In the study group, LA dP/dt(max) showed a linear relation with LA pressure before atrial contraction (r = 0.80, p < 0.005), confirming the presence of the Frank-Starling mechanism in the LA. Among transmitral flow parameters, mean acceleration showed the strongest correlation with LA dP/dt(max) (r = 0.78, p < 0.001). Among pulmonary venous flow parameters, no single parameter was sufficient to estimate LA dP/dt(max) with an r2 > 0.30. By stepwise and multiple linear regression analysis, LA dP/dt(max) was best described as follows: LA dP/dt(max) = 0.1 M-AC +/- 1.8 P-V - 4.1; r = 0.88, p < 0.0001, where M-AC is the mean acceleration of transmitral flow and P-V is the peak velocity of pulmonary venous flow during atrial contraction. This equation was tested in the latter 10 patients of the test group. Predicted and measured LA dP/dt(max) correlated well (r = 0.90, p < 0.0001). Numerical simulation verified that this relationship held across a wide range of atrial elastance, ventricular relaxation and systolic function, with LA dP/dt(max) predicted by the above equation with r = 0.94. CONCLUSIONS: A combination of transmitral and pulmonary venous flow parameters can provide a hemodynamic assessment of LA systolic function. (+info)
The pulmonary venous systolic flow pulse--its origin and relationship to left atrial pressure. (8/343)OBJECTIVES: The purpose of this study was to determine the origin of the pulmonary venous systolic flow pulse using wave-intensity analysis to separate forward- and backward-going waves. BACKGROUND: The mechanism of the pulmonary venous systolic flow pulse is unclear and could be a "suction effect" due to a fall in atrial pressure (backward-going wave) or a "pushing effect" due to forward-propagation of right ventricular (RV) pressure (forward-going wave). METHODS: In eight patients during coronary surgery, pulmonary venous flow (flow probe), velocity (microsensor) and pressure (micromanometer) were recorded. We calculated wave intensity (dP x dU) as change in pulmonary venous pressure (dP) times change in velocity (dU) at 5 ms intervals. When dP x dU > 0 there is a net forward-going wave and when dP x dU < 0 there is a net backward-going wave. RESULTS: Systolic pulmonary venous flow was biphasic. When flow accelerated in early systole (S1), pulmonary venous pressure was falling, and, therefore, dP x dU was negative, -0.6 +/- 0.2 (x +/- SE) W/m2, indicating a net backward-going wave. When flow accelerated in late systole (S2), pressure was rising, and, therefore, dP x dU was positive, 0.3 +/- 0.1 W/m2, indicating a net forward-going wave. CONCLUSIONS: Pulmonary venous flow acceleration in S1 was attributed to a net backward-going wave secondary to a fall in atrial pressure. However, flow acceleration in S2 was attributed to a net forward-going wave, consistent with propagation of the RV systolic pressure pulse across the lungs. Pulmonary vein systolic flow pattern, therefore, appears to be determined by right- as well as left-sided cardiac events. (+info)
There are several risk factors for developing AF, including:
1. Age: The risk of developing AF increases with age, with the majority of cases occurring in people over the age of 65.
2. Hypertension (high blood pressure): High blood pressure can damage the heart and increase the risk of developing AF.
3. Heart disease: People with heart disease, such as coronary artery disease or heart failure, are at higher risk of developing AF.
4. Diabetes mellitus: Diabetes can increase the risk of developing AF.
5. Sleep apnea: Sleep apnea can increase the risk of developing AF.
6. Certain medications: Certain medications, such as thyroid medications and asthma medications, can increase the risk of developing AF.
7. Alcohol consumption: Excessive alcohol consumption has been linked to an increased risk of developing AF.
8. Smoking: Smoking is a risk factor for many cardiovascular conditions, including AF.
9. Obesity: Obesity is a risk factor for many cardiovascular conditions, including AF.
Symptoms of AF can include:
1. Palpitations (rapid or irregular heartbeat)
2. Shortness of breath
4. Dizziness or lightheadedness
5. Chest pain or discomfort
AF can be diagnosed with the help of several tests, including:
1. Electrocardiogram (ECG): This is a non-invasive test that measures the electrical activity of the heart.
2. Holter monitor: This is a portable device that records the heart's rhythm over a 24-hour period.
3. Event monitor: This is a portable device that records the heart's rhythm over a longer period of time, usually 1-2 weeks.
4. Echocardiogram: This is an imaging test that uses sound waves to create pictures of the heart.
5. Cardiac MRI: This is an imaging test that uses magnetic fields and radio waves to create detailed pictures of the heart.
Treatment for AF depends on the underlying cause and may include medications, such as:
1. Beta blockers: These medications slow the heart rate and reduce the force of the heart's contractions.
2. Antiarrhythmics: These medications help regulate the heart's rhythm.
3. Blood thinners: These medications prevent blood clots from forming and can help reduce the risk of stroke.
4. Calcium channel blockers: These medications slow the entry of calcium into the heart muscle cells, which can help slow the heart rate and reduce the force of the heart's contractions.
In some cases, catheter ablation may be recommended to destroy the abnormal electrical pathway causing AF. This is a minimally invasive procedure that involves inserting a catheter through a vein in the leg and guiding it to the heart using x-ray imaging. Once the catheter is in place, energy is applied to the abnormal electrical pathway to destroy it and restore a normal heart rhythm.
It's important to note that AF can increase the risk of stroke, so anticoagulation therapy may be recommended to reduce this risk. This can include medications such as warfarin or aspirin, or in some cases, implantable devices such as a left atrial appendage closure device.
In conclusion, atrial fibrillation is a common heart rhythm disorder that can increase the risk of stroke and heart failure. Treatment options depend on the underlying cause and may include medications, cardioversion, catheter ablation, or anticoagulation therapy. It's important to work closely with a healthcare provider to determine the best course of treatment for AF.
There are several potential causes of LVD, including:
1. Coronary artery disease: The buildup of plaque in the coronary arteries can lead to a heart attack, which can damage the left ventricle and impair its ability to function properly.
2. Heart failure: When the heart is unable to pump enough blood to meet the body's needs, it can lead to LVD.
3. Cardiomyopathy: This is a condition where the heart muscle becomes weakened or enlarged, leading to impaired function of the left ventricle.
4. Heart valve disease: Problems with the heart valves can disrupt the normal flow of blood and cause LVD.
5. Hypertension: High blood pressure can cause damage to the heart muscle and lead to LVD.
6. Genetic factors: Some people may be born with genetic mutations that predispose them to developing LVD.
7. Viral infections: Certain viral infections, such as myocarditis, can inflame and damage the heart muscle, leading to LVD.
8. Alcohol or drug abuse: Substance abuse can damage the heart muscle and lead to LVD.
9. Nutritional deficiencies: A diet lacking essential nutrients can lead to damage to the heart muscle and increase the risk of LVD.
Diagnosis of LVD typically involves a physical exam, medical history, and results of diagnostic tests such as electrocardiograms (ECGs), echocardiograms, and stress tests. Treatment options for LVD depend on the underlying cause, but may include medications to improve cardiac function, lifestyle changes, and in severe cases, surgery or other procedures.
Preventing LVD involves taking steps to maintain a healthy heart and reducing risk factors such as high blood pressure, smoking, and obesity. This can be achieved through a balanced diet, regular exercise, stress management, and avoiding substance abuse. Early detection and treatment of underlying conditions that increase the risk of LVD can also help prevent the condition from developing.
LVH can lead to a number of complications, including:
1. Heart failure: The enlarged left ventricle can become less efficient at pumping blood throughout the body, leading to heart failure.
2. Arrhythmias: The abnormal electrical activity in the heart can lead to irregular heart rhythms.
3. Sudden cardiac death: In some cases, LVH can increase the risk of sudden cardiac death.
4. Atrial fibrillation: The enlarged left atrium can lead to atrial fibrillation, a common type of arrhythmia.
5. Mitral regurgitation: The enlargement of the left ventricle can cause the mitral valve to become incompetent, leading to mitral regurgitation.
6. Heart valve problems: The enlarged left ventricle can lead to heart valve problems, such as mitral regurgitation or aortic stenosis.
7. Coronary artery disease: LVH can increase the risk of coronary artery disease, which can lead to a heart attack.
8. Pulmonary hypertension: The enlarged left ventricle can lead to pulmonary hypertension, which can further strain the heart and increase the risk of complications.
Evaluation of LVH typically involves a physical examination, medical history, electrocardiogram (ECG), echocardiography, and other diagnostic tests such as stress test or cardiac MRI. Treatment options for LVH depend on the underlying cause and may include medications, lifestyle changes, and in some cases, surgery or other interventions.
Tissue Doppler echocardiography
List of MeSH codes (G09)
Christodoulos I. Stefanadis
Atrial natriuretic peptide
Relative Atrial Index
Amiodarone induced thyrotoxicosis
Signs and symptoms of Graves' disease
Pathophysiology of heart failure
Low pressure receptors
Artificial cardiac pacemaker
Heart failure with preserved ejection fraction
Intensive care medicine
List of cardiology mnemonics
Sándor J. Kovács
List of circulatory system conditions
Fetal aortic stenosis
Pericardial heart valves
Management of atrial fibrillation
Fetal alcohol spectrum disorder
List of OMIM disorder codes
Aortic valve replacement
Russell M. Nelson
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Developing atrial fibrillation1
- Your provider may also recommend healthy lifestyle changes to help you lower your risk of developing atrial fibrillation. (nih.gov)
- Most patients have an atrial septal defect or a patent foramen ovale, which allows predominant right-to-left shunting at the atrial level. (medscape.com)
- 4] In 1958, Hunter and Lillihei described a technique of surgical repair that involved plication of the atrialized right ventricle, closure of the atrial septal defect, and tricuspid valve annuloplasty. (medscape.com)
- Patients who simultaneously have the first three types of atrial septal defect, as described below, are said to have common atrium. (medscape.com)
- The most common yet least serious type of atrial septal defect is an ostium secundum defect. (medscape.com)
System was normal but the distal1
- Angiography showed that the left coronary system was normal but the distal portion of the right coronary artery was totally occluded by a thrombus ( Figure 1 ). (who.int)
- Other causes of coronary embolism include atrial fibrillation, left atrial tumours, bacterial endocarditis, atrial and ventricular mural thrombus, syphilis and pulmonary vein thrombosis . (who.int)
- The most important members of this family include atrial natriuretic peptide (ANP) and B- type natriuretic peptide (BNP) and their amino terminal fragments NT-proANP and NT-proBNP. (acc.org)
- In the Posterior left pericardiotomy for the prevention of atrial fibrillation after cardiac surgery (PALACS) trial, posterior pericardiotomy was associated with a significant reduction in postoperative atrial fibrillation (POAF) after cardiac surgery. (qxmd.com)
- Atrial fibrillation patients who develop heart failure have a three-fold increased risk of death, while in heart failure patients, the risk of death is two-fold higher when atrial fibrillation is also present. (dicardiology.com)
- ESC Guidelines on atrial fibrillation recommend the procedure to reverse left ventricular dysfunction in atrial fibrillation patients when tachycardia-induced cardiomyopathy is highly probable. (dicardiology.com)
- Left atrial systolic function is reported as normal in Acute Myocardial Infarction (AMI), but its relationship to the degree of LV dysfunction is unknown. (biomedcentral.com)
- A high incidence of atrial and ventricular arrhythmia, including an association with Wolff-Parkinson-White Syndrome, occurs in these patients. (medscape.com)
- Atrial fibrillation is the most common cardiac arrhythmia seen in clinical practice today. (nih.gov)
- Your healthcare provider may order other tests to record abnormal heart rhythms that occur under specific conditions or outside of the clinic, confirm whether you have atrial fibrillation or another arrhythmia , and find the best treatment. (nih.gov)
- Atrial fibrillation is the most common arrhythmia (heart rhythm disturbance). (cun.es)
- With this type of arrhythmia, the blood remains stagnant in the left atrial appendage, which favors the formation of embolisms that can detach to other blood vessels, such as those in the brain, reducing blood flow and causing a stroke. (cun.es)
- One of the most common causes of this problem is atrial fibrillation (AF) , a cardiac arrhythmia that causes irregular contractions of the atria, affecting millions of people worldwide. (cun.es)
- Dr. Natale also is the Senior Medical Director at Pacific Atrial Fib and Arrhythmia Center in San Francisco. (jafib.com)
- These include potentially life-threatening forms of arrhythmia called atrial fibrillation and ventricular fibrillation. (medlineplus.gov)
- 14. Associations of Electrocardiographic Parameters with Left Ventricular Longitudinal Strain and Prognosis in Cardiac Light Chain Amyloidosis. (nih.gov)
- Atrial fibrillation and heart failure often coexist.2 It is estimated that approximately 30% of patients with heart failure will develop atrial fibrillation and patients with atrial fibrillation have a five-fold increased risk of developing heart failure.3 Each condition aggravates the prognosis of the other. (dicardiology.com)
- ObjectiveTo summarize data on the prevalence/incidence, risk factors and prognosis of atrial fibrillation (AF) in patients with acute coronary syndromes (ACS).MethodsMEDLINE, Embase, and Web of Science were searched to identify all published studies providing relevant data through August 23, 2020. (ox.ac.uk)
- The score is based on four parameters: QRS width above 120 milliseconds (2 points), known aetiology (2 points), paroxysmal atrial fibrillation (1 point) and severe atrial dilation (1 point). (dicardiology.com)
- Assessment of Left Atrial Function in Patients with Paroxysmal, Persistent, and Permanent Atrial Fibrillation Using Two-Dimensional Strain. (krakow.pl)
- The aim of this study was to evaluate the specific neurologic biomarkers, neuroimaging findings, and cognitive function in patients with persistent atrial fibrillation (AF) undergoing electrical cardioversion, compared to control subjects. (mdpi.com)
- This process is referred to referred to Al-Azhar University Hospital Outpatient as, diastolic function (3) . (who.int)
- Conclusion: PWA did not adversely affect echocardiographic parameters of left atrial function or left ventricular systolic or diastolic function when compared to other types of ablation. (jafib.com)
- Little is known about the left atrium (LA) in acute left ventricular (LV) dysfunction. (biomedcentral.com)
- This downward displacement of the leaflets reduces the distal chamber of the right ventricle, leaving part of the ventricle above the valve as an extension of the right atrium. (medscape.com)
- Cardiac magnetic resonance imaging (MRI) and other imaging tests may be performed in some cases so your provider can see the structure of the left upper chamber of the heart (left atrium) and how well it is working. (nih.gov)
- The hemodynamic and electrophysiological action of the LEFT ATRIUM . (nih.gov)
- When the heart does not beat efficiently, blood may stagnate in the left atrium. (cun.es)
- During embryonic and fetal life, the central portion of the septum primum billows into the left atrium due to the normal right to left shunting at the atrial level. (medscape.com)
- If atrial fibrillation occurs when there is a large atrium, for example in mitral stenosis, then this is a predisposing factor to the development of thromboembolism. (gpnotebook.com)
- Charles and colleagues reported that coronary embolism occurs in the left coronary artery in 75% of cases and three-quarters of them present with ST elevation myocardial infarction, whereas the rest present with non-ST elevation myocardial infarction . (who.int)
- CONCLUSION: Diastolic parameters of left atrial function possibly are more important markers of AF risk than left atrial dilation in HFpEF. (nih.gov)
- 3 , 4 However they are influenced by many variables such as heart rate, cardiac output, left ventricular systolic and diastolic functions, left ventricular and atrial compliance, left ventricular hypertrophy and concomitant valve disease. (bmj.com)
- 1) Stress Cardiomyopathy has a direct depressive effect on LA function in addition to its negative effect on the left ventricle. (biomedcentral.com)
- myocardial strain in the left and the right ventricle, Even more with the change of the modern lifestyle avoiding the angular sensitivity of tissue Doppler and diet structure, incidence of MS increased year by echocardiography (5) . (who.int)
- endocarditis, atrial and ventricular mu- ing treatment of coronary embolism. (who.int)
- Methods: We selected patients who had pre and post ablation echocardiogram at our institution.Parameters assessed were: Left ventricular outflow tract velocity time integral (VTI), left ventricular ejection fraction (LVEF), atrial Doppler velocity across mitral valve (A), E/E', and deceleration time. (jafib.com)
- The researchers retrospectively identified patients with heart failure, impaired LVEF and atrial fibrillation who had an ablation procedure at eight centers in Europe. (dicardiology.com)
- Recent data from the Cardiovascular Health Study suggest that age, male gender, clinical cardiovascular disease, and left atrial size are all important risk factors for AF. (nih.gov)
- The talk will concentrate on three cardiovascular applications in which imaging, quantification, and visualization in close-to-real time play an important role: Geometrically correct fusion of intravascular ultrasound and biplane angiography, analysis of left ventricular and left-atrial function from intracardiac ultrasound pullback sequences, and non-invasive assessment of cardiovascular system status via brachial artery flow-mediated dilatation approach. (uni-lj.si)
- Sensitive measures of cardiovascular structure and function may provide mechanistic insight behind the associations of FGF23 with various cardiovascular diseases. (nih.gov)
- They will have tests of their cognitive function. (nih.gov)
- Atrial fibrillation incidence, prevalence, predictors, and adverse outcomes in acute coronary syndromes: A pooled analysis of data from 8 million patients. (ox.ac.uk)
- However in the presence of significant leaflet calcification, severe left atrial dilatation or distortion of the valve anatomy, it can be very challenging to obtain a perfect geometric alignment at the MV leaflet tips in a parasternal short-axis view using two-dimensional (2D) echocardiography. (bmj.com)
- Left atrial appendage closure. (cun.es)
- What is atrial appendage closure? (cun.es)
- Left atrial appendage closure is a procedure that has gained relevance in recent years. (cun.es)
- To understand the procedure for atrial appendage closure of the heart, it is first necessary to understand what the atrial appendage is and what its function is. (cun.es)
- Atrial appendage closure of the heart is performed by a minimally invasive procedure known as percutaneous closure. (cun.es)
- Before performing an atrial appendage closure, the physician will perform a series of tests to determine whether the patient is suitable for this treatment. (cun.es)
- These tests include transesophageal echocardiography and computed tomography to analyze the anatomy of the atrial appendage and to rule out the presence of clots prior to the procedure. (cun.es)
- Through a catheter, a closure device (such as the Watchman) is inserted into the left atrial appendage. (cun.es)
- 8. Clinical features and predictors of atrial fibrillation in patients with light-chain or transthyretin cardiac amyloidosis. (nih.gov)
- Echocardiography (ultrasound) looks at the heart valves, chamber sizes and heart pumping function. (nih.gov)
- soesophageal echocardiography, there encountered rarely, it is an important Angiography showed that the left coro- was a semi-mobile thrombus on the left and serious complication. (who.int)
- However, in transoesophageal echocardiography, there was a semi-mobile thrombus on the left atrial surface of the valve measuring 5 × 5 mm ( Figure 2 ). (who.int)
- We compared the effect of PWI versus non-PWI methods (CPVA + linear lesions) on echocardiographic parameters of left atrial and left ventricular function. (jafib.com)
- METHODS: In MESA (the Multi-Ethnic Study of Atherosclerosis), we evaluated the associations of baseline serum FGF23 (2000-2002) with measures of left ventricular (LV) and left atrial mechanical function on cardiac magnetic resonance at 10-year follow-up (2010-2012). (nih.gov)
- Atrial fibrillation is a supraventricular tachyarrhythmia characterised by ineffective, chaotic, irregular and rapid (300 to 600 beats per minute) atrial activity resulting in the deterioration of atrial mechanical function (1). (gpnotebook.com)
- Hemodynamic studies conducted in patients with normal left ventricular function produced, following intravenous isradipine administration, increases in cardiac index, stroke volume index, coronary sinus blood flow, heart rate, and peak positive left ventricular dP/dt. (nih.gov)
- Pulsed Wave Doppler (PWD) or Continuous Wave Doppler abnormal cardiac function and diagnosing valvular stenosis and (CWD). (nih.gov)
- Left-atrial volume reduction reflects improvement of cardiac sympathetic nervous function in patients with severe aortic stenosis after transcatheter aortic valve replacement. (bvsalud.org)
- ed about warfarin and possible adverse that coronary embolism occurs in the We administered unfractionized reactions with other drugs and followed left coronary artery in 75% of cases and heparin and tirofiban (glycoprotein regularly. (who.int)
- Flow Cytometric Assessment of Endothelial and Platelet Microparticles in Patients With Atrial Fibrillation Treated With Dabigatran. (krakow.pl)
- All subjects underwent conventional echocardiographic examination and assessment of LA function by speckle tracking. (bvsalud.org)
- The objectives of these studies are to understand the role that cell cycle-related changes play in the development of cardiomyopathy and the ability of sophisticated echocardiographic measurements of myocardial motion to detect changes in ventricular function during that progression. (asecho.org)
- He has pioneered a new circumferential ultrasound vein-ablation system to correct atrial fibrillation and performed the procedure on the world's first five patients. (jafib.com)
- Background: Posterior wall isolation(PWI)in addition to circumferential pulmonary vein isolation (CPVA) requires more ablation of left atrial tissue. (jafib.com)
- RESEARCH OBJECTIVES Atrial fibrillation (AF) is recognized as a condition which causes significant morbidity, disability, and mortality related to heart disease and stroke. (nih.gov)
- CSN function was assessed by performing 123I-metaiodobenzylguanidine ( MIBG ) scintigraphy before and 6 months after TAVR, and the delayed heart -to- mediastinum ratio (dHMR) and washout rate (WR) were calculated. (bvsalud.org)
- Metabolic syndrome (MS) predisposes to left ventricular dysfunction and heart failure , however, alterations in left atrial (LA) function in MS are unknown. (bvsalud.org)
- IMPACT OF AGING ON DEVELOPMENT OF ATRIAL FIBRILLATION Release Date: January 8, 1999 PA NUMBER: PA-99-035 P.T. National Institute on Aging National Heart, Lung, and Blood Institute PURPOSE The goal of this program announcement (PA) is to foster research that will enhance our understanding of age-related structural and functional changes in the atria and their impact on the development of atrial fibrillation (AF) in older persons. (nih.gov)
- This PA, Impact of Aging on Development of Atrial Fibrillation, is related to the priority area of heart disease and stroke. (nih.gov)
- Your provider will ask about your eating and physical activity habits, family history, and other risk factors for atrial fibrillation and heart disease. (nih.gov)
- To diagnose atrial fibrillation, your provider will likely do one or more heart or blood tests . (nih.gov)
- In approximately one half of patients with left atrioventricular (AV) valve underdevelopment (ie, hypoplastic left heart syndrome or Shone complex), the superior attachments of the flap valve of the foramen ovale lie on the left atrial roof, well to the left of the septum secundum. (medscape.com)
- BACKGROUND: Although FGF23 (fibroblast growth factor 23) is associated with heart failure and atrial fibrillation, the mechanisms driving these associations are unclear. (nih.gov)
- This condition affects the function of the sino-atrial (SA) node, which is an area of specialized cells in the heart that functions as a natural pacemaker. (medlineplus.gov)
- AIMS: To determine if markers of diastolic dysfunction are associated with atrial fibrillation (AF) development among patients with heart failure with preserved ejection fraction (HFpEF). (nih.gov)
- 79% white) with HFpEF from the Treatment of Preserved Cardiac Function Heart Failure With an Aldosterone Antagonist Trial (TOPCAT) who were free of baseline AF. (nih.gov)
- Examine the relationship between LA and LV with respect to size and function in the setting of acute LV dysfunction. (biomedcentral.com)
- Further evidence is needed to help stratify and identify those patients who will most likely benefit from atrial fibrillation ablation. (dicardiology.com)
- This initiative is intended to foster clinically-related research, including integrative biomedical research, some of which may incorporate the tools of molecular and cell biology in the study of function and clinical outcome. (nih.gov)
- (5-7) ANP is rapidly released into the circulation in response to atrial wall stretch, but due to its extremely short-half life (2-5 min) is not a useful tool in a clinical setting. (acc.org)
- We aimed to use strain / strain rate (SR) imaging to investigate the effect of MS on LA function. (bvsalud.org)
- In these aims, we will determine the ability of myocardial tissue motion, strain, and strain rate using velocity-vector imaging to provide insight into early and more subtle abnormalities of dysregulated myocardial function. (asecho.org)
- This information can help determine if there is a risk for complications from atrial fibrillation. (nih.gov)
- However, they may also check for symptoms of atrial fibrillation as part of your regular medical care. (nih.gov)
- 0.05 for all), with no difference in the mean peak late diastolic SR. Central obesity , hypertension , dyslipidemia and uncontrolled diabetes were independent risk factors for impaired LA function. (bvsalud.org)
- If you have atrial fibrillation, your provider will also look for any disease that may be causing it and assess your risk of developing dangerous blood clots. (nih.gov)
- Your healthcare provider may screen you for atrial fibrillation only when you have risk factors. (nih.gov)
- Increasing values of the E/A ratio [per 0.1 increase: hazard ratio (HR) = 1.11, 95% confidence interval (CI) = 1.06-1.17], left atrial volume (per 5 mL increase: HR = 1.13, 95% CI = 1.03-1.23), and left atrial area (per 5 cm2 increase: HR = 1.51, 95% CI = 1.03-2.22) were associated with greater risk of AF. (nih.gov)
- The risk of AF was not materially altered when peak A wave velocity was further adjusted for left atrial volume (HR = 0.83, 95% CI = 0.71-0.96) and area (HR = 0.83, 95% CI = 0.71-0.96). (nih.gov)
Abnormal cardiac function1
- However, it is not known whether MS is also function in patients with metabolic syndrome by 2D associated with abnormal cardiac function. (who.int)