Asthenozoospermia
Sperm Motility
Infertility, Male
Spermatozoa
Mature male germ cells derived from SPERMATIDS. As spermatids move toward the lumen of the SEMINIFEROUS TUBULES, they undergo extensive structural changes including the loss of cytoplasm, condensation of CHROMATIN into the SPERM HEAD, formation of the ACROSOME cap, the SPERM MIDPIECE and the SPERM TAIL that provides motility.
Semen
Oligospermia
Pregnancy Rate
Bees
Apitherapy
Fatty Acids
Honey
A sweet viscous liquid food, produced in the honey sacs of various bees from nectar collected from flowers. The nectar is ripened into honey by inversion of its sucrose sugar into fructose and glucose. It is somewhat acidic and has mild antiseptic properties, being sometimes used in the treatment of burns and lacerations.
Universal Precautions
Beta-endorphin in serum and seminal plasma in infertile men. (1/72)
AIM: To access beta-endorphin levels in serum as well as seminal plasma in different infertile male groups. METHODS: Beta-endorphin was estimated in the serum and seminal plasma by enzyme-linked immunosorbent assay (ELISA) method in 80 infertile men equally divided into four groups: non-obstructive azoospermia (NOA), obstructive azoospermia (OA), congenital bilateral absent vas deferens (CBVAD) and asthenozoospermia. The results were compared to those of 20 normozoospermic proven fertile men. RESULTS: There was a decrease in the mean levels of beta-endorphin in the seminal plasma of all successive infertile groups (mean +/- SD: NOA 51.30 +/- 27.37, OA 51.88 +/- 9.47, CBAVD 20.36 +/- 13.39, asthenozoospermia 49.26 +/- 12.49 pg/mL, respectively) compared to the normozoospermic fertile control (87.23 +/- 29.55 pg/mL). This relation was not present in mean serum level of beta-endorphin between four infertile groups (51.09 +/- 14.71, 49.76 +/- 12.4, 33.96 +/- 7.2, 69.1 +/- 16.57 pg/mL, respectively) and the fertile control group (49.26 +/- 31.32 pg/mL). The CBVAD group showed the lowest seminal plasma mean level of beta-endorphin. Testicular contribution of seminal beta-endorphin was estimated to be approximately 40%. Seminal beta-endorphin showed significant correlation with the sperm concentration (r = 0.699, P = 0.0188) and nonsignificant correlation with its serum level (r = 0.375, P = 0.185) or with the sperm motility percentage (r = 0.470, P = 0.899). CONCLUSION: The estimation of beta-endorphin alone is not conclusive to evaluate male reproduction as there are many other opiates acting at the hypothalamic pituitary gonadal axis. (+info)Association between the apolipoprotein B signal peptide gene insertion/deletion polymorphism and male infertility. (2/72)
In male mice heterozygous for a null apolipoprotein B (apoB), allele infertility was noticed. These data led us to investigate a possible role of APOB gene polymorphism and male infertility in humans. In this case-control study, we searched for an association between the insertion/deletion (I/D) polymorphism of the APOB gene and male infertility in 560 Slovene Caucasian men. The study group consisted of 310 infertile patients: 115 with azoospermia and 195 with oligoasthenoteratozoospermia (OAT) and a control group of 250 fertile men. We found a statistically significant difference in the genotype distribution between the two groups (chi2 = 6.315, P = 0.043). A separate analysis of azoospermic and OAT patients demonstrated that significant differences in genotype distribution were limited to the OAT group (chi2 = 7.011, P = 0.030). The presence of the D allele (DD or ID genotypes) conferred a 1.6 risk [chi2 = 6.089, P = 0.014, 95% confidence interval (95% CI) = 1.102-2.347] for male infertility in the OAT group of patients. We did not find a correlation between the I/D polymorphism genotypes and the clinical characteristics of infertile men: sperm concentration (P = 0.102), rapid progressive motility (P = 0.449), normal morphology (P = 0.085) and Johnsen score (P = 0.531). These data suggest that genetic variation in the signal peptide of the APOB gene (I/D polymorphism) might be a risk factor for the development of male infertility. (+info)Asthenoteratozoospermia in mice lacking testis expressed gene 18 (Tex18). (3/72)
Testis expressed gene 18 (Tex18) is a small gene with one exon of 240 bp, which is specifically expressed in male germ cells. The gene encodes for a protein of 80 amino acids with unknown domain. To investigate the function of (Tex18) gene, we generated mice with targeted disruption of the (Tex18) gene by homologous recombination. Homozygous mutant males on a mixed genetic background (C57BL/6J x 129/Sv) are fertile, while they are subfertile on the 129/Sv background, although mating is normal. We showed that Tex18(-/-) males are subfertile because of abnormal sperm morphology and reduced motility, which is called asthenoteratozoospermia, suggesting that (Tex18) affects sperm characteristics. Maturation of spermatids is unsynchronized and partially impaired in seminiferous tubules of Tex18(-/-) mice. Electron microscopical examination demonstrated abnormal structures of sperm head. In vivo experiments with sperm of Tex18(-/-) 129/Sv mice revealed that the migration of spermatozoa from the uterus into the oviduct is reduced. This result is supported by the observation that sperm motility, as determined by the computer-assisted semen analysis system, is significantly affected, compared to wild-type spermatozoa. Generation of transgenic mice containing Tex18-EGFP fusion construct revealed a high transcriptional activity of (Tex18) during spermiogenesis, a process with morphological changes of haploid germ cells and development to mature spermatozoa. These results indicate that (Tex18) is expressed predominantly during spermatid differentiation and subfertility of the male Tex18(-/-) mice on the 129/Sv background is due to the differentiation arrest, abnormal sperm morphology and reduced sperm motility. (+info)No evidence for an mtDNA role in sperm motility: data from complete sequencing of asthenozoospermic males. (4/72)
The first complete mitochondrial DNA (mtDNA) sequences (approximately 16,569 bp) in 20 patients with asthenozoospermia and a comparison with 23 new complete mtDNA sequences in teratoasthenozoospermic individuals, confirmed no sharing of specific polymorphisms or specific mitochondrial lineages between these individuals. This is strong evidence against the accepted claim of a major role played by mtDNA in male fertility, once supported by haplogroup association studies based on the screening of hypervariable region I. The hypothesis of maternally driven selection acting in male reproductive success must thus be treated with caution. (+info)Absence of tektin 4 causes asthenozoospermia and subfertility in male mice. (5/72)
Sperm flagellar motion is the outcome of a dynamic interplay between the axonemal cytoskeleton, the peri-axonemal accessory structures, and multiple regulatory networks that coordinate to produce flagellar beat and waveform. Tektins are conserved components of the flagellar proteome in evolutionarily diverse species and are believed to play essential roles in the mechanics of sperm motility. Using database mining, we identified multiple new paralogs of previously annotated tektins, including tektin 4 (TEKT4), which shares 77.1% identity with its nearest human homologue. Mouse Tekt4 is a germ cell-enriched gene, most abundantly expressed in haploid round spermatids in the testis, and the protein is localized to the sperm flagella. Male mice lacking TEKT4 on a 129S5/SvEvBrd inbred background are subfertile. Tekt4-null sperm exhibit drastically reduced forward progressive velocity and uncoordinated waveform propagation along the flagellum. In Tekt4-null sperm, flagellar ultrastructure is grossly unaltered as revealed by transmission electron microscopy. However, the ineffective flagellar strokes lead to approximately 10-fold higher consumption of intracellular ATP in Tekt4-null sperm as compared to wild-type, and null spermatozoa rapidly lose progressive motility when incubated for > or = 1.5 h. Our studies demonstrate that TEKT4 is necessary for the proper coordinated beating of the sperm flagellum and male reproductive physiology. (+info)Effect of cigarette smoking on semen quality of infertile men. (6/72)
INTRODUCTION: Defective sperm quality is a significant cause of infertility. It is known that cigarette smoking affects semen quality. Our aim was to compare the semen of infertile cigarette smokers with infertile non-smokers to study the effect of smoking on semen quality. METHODS: Semen samples of 100 cigarette smokers and 100 strictly non-smoking primary infertility patients were included in the study, following stringent exclusion criteria. Smokers were categorised as light, moderate and heavy smokers. Semen samples were examined for asthenozoospermia, oligozoospermia and teratozoospermia, according to World Health Organisation guidelines. RESULTS: 39 percent of non-smokers showed normozoospermia, while only three percent of smokers were normozoospermics. Light smokers predominantly showed asthenozoospermia. Heavy smokers showed asthenozoospermia, teratozoospermia and oligozoospermia. Statistical analysis using Fisher's exact test showed that the incidence of both isolated asthenozoospermia (p-value is 0.0015) and asthenozoospermia with teratozoospermia (p-value is 0.0106) among smokers was significant, in comparison to non-smokers. Overall impact of asthenozoospermia (p-value is less than 0.0001) and teratozoospermia (p-value is 0.0328) but not of oligozoospermia was observed on the semen quality in smokers, compared with non-smokers. CONCLUSION: Asthenozoospermia, the most common semen variable in our study, can be an early indicator of reduction in quality of semen, as seen in light smokers. In addition, heavy smoking produces teratozoospermia, which further reduces semen quality. Oligozoospermia may be due to factors other than smoking. (+info)Seminal plasma anti-Mullerian hormone level correlates with semen parameters but does not predict success of testicular sperm extraction (TESE). (7/72)
AIM: To assess seminal plasma anti-Mullerian hormone (AMH) level relationships in fertile and infertile males. METHODS: Eighty-four male cases were studied and divided into four groups: fertile normozoospermia (n = 16), oligoasthenoteratozoospermia (n = 15), obstructive azoospermia (OA) (n = 13) and non-obstructive azoospermia (NOA) (n = 40). Conventional semen analysis was done for all cases. Testicular biopsy was done with histopathology and fresh tissue examination for testicular sperm extraction (TESE) in NOA cases. NOA group was subdivided according to TESE results into unsuccessful TESE (n = 19) and successful TESE (n = 21). Seminal plasma AMH was estimated by enzyme linked immunosorbent assay (ELISA) and serum follicular stimulating hormone (FSH) was estimated in NOA cases only by radioimmunoassay (RIA). RESULTS: Mean seminal AMH was significantly higher in fertile group than in oligoasthenoteratozoospermia with significance (41.5 +/- 10.9 pmol/L vs. 30.5 +/- 10.3 pmol/L, P < 0.05). Seminal AMH was not detected in any OA patients. Seminal AMH was correlated positively with testicular volume (r = 0.329, P = 0.005), sperm count (r = 0.483, P = 0.007), sperm motility percent (r = 0.419, P = 0.021) and negatively with sperm abnormal forms percent (r = -0.413, P = 0.023). Nonsignificant correlation was evident with age (r = -0.155, P = 0.414) and plasma FSH (r = -0.014, P = 0.943). In NOA cases, seminal AMH was detectable in 23/40 cases, 14 of them were successful TESE (57.5%) and was undetectable in 17/40 cases, 10 of them were unsuccessful TESE (58.2%). CONCLUSION: Seminal plasma AMH is an absolute testicular marker being absent in all OA cases. However, seminal AMH has a poor predictability for successful testicular sperm retrieval in NOA cases. (+info)Superoxide dismutase and catalase activities in the seminal plasma of normozoospermic and asthenozoospermic Beagles. (8/72)
We measured the blood plasma testosterone (T) levels and superoxide dismutase (SOD) and catalase activities in the seminal plasma of the ejaculates of 5 normal (2-5 years old) and 5 asthenozoospermic (AZ-) (3-5 years old) Beagles. Sperm ejaculated by AZ-dogs was incubated for 3 hr in Eagle's MEM only (controls) or Eagle's MEM containing 100 units/ml of SOD or catalase. Sperm motility was examined during incubation. The mean (+/- SE) plasma T level of the AZ-dogs (1.2 +/- 0.2 ng/ml) was significantly lower than in the normal dogs (2.5 +/- 0.2 ng/ml) (P<0.005). The mean (+/- SE) seminal plasma SOD and catalase activities (18.8 +/- 1.9 and 0.5 +/- 0.1 unit/g protein, respectively) were significantly lower in the AZ-dogs than in the normal dog (43.3 +/- 2.5 and 2.2 +/- 0.4 unit/g protein, respectively) (P<0.001 and 0.01, respectively). The motility of sperm incubated in Eagle's MEM containing SOD or catalase was significantly higher than that of control sperm incubated in only Eagle's MEM after 2 or 3 hr of incubation (P<0.05). The results of this study indicate that poor T secretion by the testes and low antioxidant enzyme activities are related to AZ in the dog. (+info)
Asthenozoospermia Treatment In Lahore - How To Treat Asthenospermia
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Intracytoplasmic morphologically selected sperm injection
Men with severe oligozoospermia, asthenozoospermia or teratozoospermia. The egg quality has appeared to be normal. Repeated ...
DNAH1
"Mutations in dynein genes in patients affected by isolated non-syndromic asthenozoospermia". Hum. Reprod. 23 (8): 1957-62. doi: ... with primary ciliary dyskinesia and multiple morphological anomalies of the flagella that result in asthenozoospermia and male ...
Mother's curse
In mice, a deletion on mtDNA causes oligospermia and asthenozoospermia, resulting in infertility. Taken together, mtDNA ...
SMCP
"Asthenozoospermia in mice with targeted deletion of the sperm mitochondrion-associated cysteine-rich protein (Smcp) gene". ...
Capacitation
This procedure takes approximately 60 minutes and it is specially indicated in oligozoospermia, asthenozoospermia and abundant ...
Necrospermia
... is usually confused with asthenozoospermia, which is the inability of the sperm to move even when alive. To check ...
Hypospermia
Studies in humans show that an excess of circulating thyroid hormones during thyrotoxicosis results in asthenozoospermia, ...
Oligoasthenozoospermia
... is a combination of: Asthenozoospermia (reduced sperm motility) and Oligozoospermia (low spermatozoon ...
Haplogroup T (mtDNA)
Haplogroup T can predispose to asthenozoospermia (Ruiz-Pesini 2000) harv error: no target: CITEREFRuiz-Pesini2000 (help). ...
Small supernumerary marker chromosome
... and asthenozoospermia [i.e. sperm with reduced motility]). Infertility associated with a sSMC in women is ~7.5-fold less common ...
Genetics of infertility
... and asthenozoospermia [sperm with reduced motility]); c) band 11 from the short arm of chromosome 14 (notated as (14)q11.1)( ...
Male infertility
... increase in sperm with abnormal morphology Asthenozoospermia - reduced sperm motility Necrozoospermia - all sperm in the ...
Asthenozoospermia
... (or asthenospermia) is the medical term for reduced sperm motility. Complete asthenozoospermia, that is, 100 ... asthenozoospermia) than in men with oligozoospermia or teratozoospermia. Among men with asthenozoospermia, 31% were found to ... Causes of complete asthenozoospermia include metabolic deficiencies, ultrastructural abnormalities of the sperm flagellum (see ... found a correlation between the composition of the sperm lipid membrane and the odds of having asthenozoospermia. The sperm ...
Asthenozoospermia - Wikipedia
Asthenozoospermia (or asthenospermia) is the medical term for reduced sperm motility. Complete asthenozoospermia, that is, 100 ... asthenozoospermia) than in men with oligozoospermia or teratozoospermia. Among men with asthenozoospermia, 31% were found to ... Causes of complete asthenozoospermia include metabolic deficiencies, ultrastructural abnormalities of the sperm flagellum (see ... found a correlation between the composition of the sperm lipid membrane and the odds of having asthenozoospermia. The sperm ...
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Asthenozoospermia can occur in varying degrees and complete Asthenozoospermia occurs when all the sperms lack motility. The ... Asthenozoospermia is the condition where a mans sperm partially or completely lacks motility. Sperm motility is the ability of ... occurrence and extent of Asthenozoospermia can only be diagnosed after the study of semen samples. ... How is Asthenozoospermia diagnosed?. Asthenozoospermia is the condition where a mans sperm partially or completely lacks ...
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A group of 30 patients affected by oligo-asthenozoospermia and a group of 30 patients affected by asthenozoospermia were ... C. Rolf, T. G. Cooper, C. H. Yeung and E. Nieschlag, "Antioxidant Treatment of Patients with Asthenozoospermia or Moderate ... The first subgroup consisted of 30 subjects affected by oligo-asthenozoospermia (ages between 27 and 43 years) who had a ... for medical examination of semen quality and, in the case of oligoasthenozoospermia or asthenozoospermia, were prescribed to ...
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discovered a new gene associated with isolated asthenozoospermia (immotile sperm), which was published in the American Journal ... Further studies are required for better genotype-phenotype correlations in patients with asthenozoospermia and/or primary ... This explains why these patients have isolated asthenozoospermia without involvement of other organs. ... not all patients with primary ciliary dyskinesia have asthenozoospermia and (2), conversely, some patients have axonemal ...
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First, we found that in the first 40 years of life, the incidence of teratozoospermia and asthenozoospermia was higher in males ... Overall, the incidence rate of teratozoospermia (54.27% vs 36.57%, p = 0.000) and asthenozoospermia (42.03% vs 32.84%, p = ... Oligozoospermia was defined as a total sperm count of ,39 million/ejaculate, asthenozoospermia was defined as percentage of PR ... Further, we stratified the data by age and found that the incidence of teratozoospermia and asthenozoospermia in males residing ...
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Severe asthenozoospermia is difficult to treat.. Consequences for fertility. Asthenozoospermia is a cause of infertility ... Diagnosing asthenozoospermia or any other diagnosis of sperm with a single sperm test is a mistake, since it may be due to an ... The diagnosis of asthenozoospermia is made when there is a high amount of immobile sperm or slow, non-progressive sperm in the ... If asthenozoospermia is moderate, conventional IVF, which is simpler and more physiological than ICSI, may be sufficient for ...
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Teratozoospermia6
- Sperm DNA fragmentation level is higher in men with sperm motility defects (asthenozoospermia) than in men with oligozoospermia or teratozoospermia. (wikipedia.org)
- It is especially useful for patients where the male partner has a very low sperm count (oligozoospermia), poor sperm motility (asthenozoospermia) or too many abnormal sperms (teratozoospermia). (drjatinshah.com)
- In oligozoospermia, motility and/or morphology of spermatozoa may be altered as well (defining conditions known as asthenozoospermia , teratozoospermia , or asthenoteratozoospermia ). (bredagenetics.com)
- According to the best sexologist in Delhi , there are numerous factors that can cause male infertility as a result of their effect on the generation and maturation of sperm in the testicles themselves, generating oligozoospermia ( low concentration of sperm in semen ), asthenozoospermia (low mobility of sperm), teratozoospermia (abnormal in sperm morphology) or secretory azoospermia (no sperm are produced in the testes). (bestsexologist.in)
- Most infertile men show oligozoospermia (reduced sperm count), asthenozoospermia (reduced motility), teratozoospermia (sperm with abnormal shape), hypospermia (reduced volume of the ejaculate) indicating that spermatogenesis is not only affected in quality but in quantity as well. (fertilitecenter.com)
- Male fertility factors could be can be low sperm count (oligozoospermia), low sperm motility (asthenozoospermia), not proper shape and sperm features (teratozoospermia), auto immune self-body reaction against sperm (anti-sperm antibody), congenital absence of ducts in reproductive organs, no sperm in ejaculate (azoospermia) and much more. (harvestfertility.com)
Oligozoospermia2
- For idiopathic group of infertile patients the clinical presentations where oligozoospermia, asthenozoospermia, oligoasthenzoospermia, pyospermia and combination of them. (pdfmedarticles.com)
- Mild and moderate seminal pathologies (asthenozoospermia, mild oligozoospermia). (novafem.com.co)
Motility18
- Asthenozoospermia (or asthenospermia) is the medical term for reduced sperm motility. (wikipedia.org)
- Asthenozoospermia is the condition where a man's sperm partially or completely lacks motility. (novaivffertility.com)
- Asthenozoospermia can occur in varying degrees and complete Asthenozoospermia occurs when all the sperms lack motility. (novaivffertility.com)
- The condition is also defined as Asthenozoospermia when less than 32% of the produced sperm exhibits progressive motility. (novaivffertility.com)
- If 100% of the sperm lacks motility then the patient is suffering from complete Asthenozoospermia. (novaivffertility.com)
- In another study, nine men with reduced sperm motility-known as asthenozoospermia-were given L. rhamnosus and Bifidobacterium longum for six weeks. (fairhavenhealth.com)
- If efficient sperm comprises less than 32 percent (in a given sperm sample), your doctor may diagnose you with low sperm motility (asthenozoospermia). (acy.org)
- Asthenozoospermia- motility of sperm reduced. (pradnyaayurveda.in)
- Decreased sperm motility is called AsthenoZoospermia. (sofatinfertility.in)
- Sperms with poor motility or Asthenozoospermia is diagnosed when less than 32 percent of the sperm are able to move efficiently in forward direction. (indoreinfertilityclinic.com)
- Asthenozoospermia indicates sample with reduced motility. (indoreinfertilityclinic.com)
- As mentioned above, samples with less than 32% of progressive motility are termed as Asthenozoospermia. (indoreinfertilityclinic.com)
- Asthenozoospermia (poor sperm motility). (drjatinshah.com)
- Poor motility in the seminal sample is known as asthenozoospermia. (invitra.com)
- Normal motility is above 32% of the sperm cells moving, below that is called asthenozoospermia. (miracare.com)
- However, a deficiency in Coenzyme Q10 leads to asthenozoospermia, meaning low sperm motility. (haldanepharma.com)
- If it is less than 1 million, this is severe poor sperm motility, this is also known as asthenozoospermia. (thenaturehill.com)
- A just published study looked at the potential for lipoic acid to help men with asthenozoospermia, or low sperm motility. (simcoenaturalfoods.com)
Morphology1
- Mice homozygous for a knock-out allele exhibit male infertility associated with decreased testis weight, globozoospermia, absent acrosome, asthenozoospermia, and abnormal sperm midpiece morphology. (jax.org)
Semen3
- The occurrence and extent of Asthenozoospermia can only be diagnosed after the study of semen samples. (novaivffertility.com)
- Diagnosis of Asthenozoospermia involves the study of fresh semen samples under a microscope to confirm sperm immotility and ascertain the reasons. (novaivffertility.com)
- Semen samples were collected from 37 fertile men and 37 subfertile men with asthenozoospermia who were given zinc supplementation 440 mg day for a 3-month period. (seanpatrickcooper.com)
Asthenospermia or asthenozoospermia1
- Poor sperm mobility is classified as anything less than 25 micrometers per second, and is known as asthenospermia or asthenozoospermia. (vitaliboost.com)
Find Asthenozoospermia1
- HIV may with find asthenozoospermia birth CD4 for can reproduce 32 because of should wine periods lighter, seek medical efficiently. (workativ.com)
Oligo-asthenozoospermia1
- Materials and Methods: A group of 30 patients affected by oligo-asthenozoospermia and a group of 30 patients affected by asthenozoospermia were treated with a daily dose of sodium D-aspartate for 90 days. (scirp.org)
Complete asthenozoospermia3
- Complete asthenozoospermia, that is, 100% immotile spermatozoa in the ejaculate, is reported at a frequency of 1 of 5000 men. (wikipedia.org)
- Causes of complete asthenozoospermia include metabolic deficiencies, ultrastructural abnormalities of the sperm flagellum (see Primary ciliary dyskinesia) and necrozoospermia. (wikipedia.org)
- Complete Asthenozoospermia is lack of any motile sperm in the ejaculate. (indoreinfertilityclinic.com)
Infertility2
- Asthenozoospermia is a male infertility-related condition where only less than or equal to 40% of the sperm is motile. (novaivffertility.com)
- Asthenozoospermia - When sperm cells have limited mobility, a possible cause of infertility. (njurology.com)
ICSI1
- Absolute asthenozoospermia and ICSI: What are the options? (wikipedia.org)
Defects3
- Their structural and/or functional defects are associated with primary ciliary dyskinesia (PCD), a genetic disease characterized by chronic respiratory-tract infections and in which most males are infertile due to asthenozoospermia. (nih.gov)
- Asthenozoospermia is often attributed to physical defects in the sperm that can be caused by congenital conditions. (novaivffertility.com)
- However, (1) not all patients with primary ciliary dyskinesia have asthenozoospermia and (2), conversely, some patients have axonemal defects restricted to the sperm flagella. (nyra-youngresearch.eu)
Immotile1
- discovered a new gene associated with isolated asthenozoospermia (immotile sperm), which was published in the American Journal of Human Genetics. (nyra-youngresearch.eu)
Necrozoospermia1
- DNA fragmentation and Necrozoospermia are also known causes of Asthenozoospermia. (novaivffertility.com)
Carnitine4
- L-carnitine in idiopathic asthenozoospermia: a multicenter study. (aminoacidstudies.org)
- L-carnitine: safe and effective for asthenozoospermia. (aminoacidstudies.org)
- A placebo-controlled double-blind randomized trial of the use of combined l-carnitine and l-acetyl-carnitine treatment in males with asthenozoospermia. (20mg-cheapesttadalafil.com)
- Taking L-carnitine supplements seems to improve sperm mobility in men with poorly active sperm, a problem known as asthenozoospermia, Italian researchers report in the medical journal Fertility & Sterility. (rejuvenation-science.com)
Mild1
- This has a better prognosis in mild cases of asthenozoospermia. (invitra.com)
Mobility1
- Asthenozoospermia: Decrease in mobility. (clinimer.com)
Fertility1
- Diagnosis of Asthenozoospermia can be done by a specialist doctor or at a reputed fertility clinic. (novaivffertility.com)
Patients7
- To investigate the ecacy of carnitines for the treatment of asthenozoospermia in DM1 patients. (unict.it)
- This explains why these patients have isolated asthenozoospermia without involvement of other organs. (nyra-youngresearch.eu)
- Further studies are required for better genotype-phenotype correlations in patients with asthenozoospermia and/or primary ciliary dyskinesia. (nyra-youngresearch.eu)
- Mutation of GALNTL5 gene identified in patients diagnosed with asthenozoospermia. (nih.gov)
- gene identified in patients diagnosed with asthenozoospermia. (nih.gov)
- It may take good quality so in patients hypersensitive for chronic bronchitis with this disorder men with asthenozoospermia. (fmvoley.com)
- Bronchodilator effect of anti-psychotic-naïve patients with asthenozoospermia or reactions to controls. (gors.nl)
Fragmentation1
- Among men with asthenozoospermia, 31% were found to have high levels of DNA fragmentation. (wikipedia.org)
Primary1
- Primary 5G can bad accumulate asthenozoospermia culture is the is being too a of painless sore may the genitals, of. (btctw.com)
Clinic1
- I recommend that you go to an assisted reproduction clinic so that they can evaluate the degree of asthenozoospermia that your husband has and propose a treatment based on that. (invitra.com)
Type1
- PHGPx er en type glutation. (fruktbarhet.com)
Found1
- found a correlation between the composition of the sperm lipid membrane and the odds of having asthenozoospermia. (wikipedia.org)
Increase1
- The to virus American asthenozoospermia we a buy real viagra room at buy real viagra prescribe some develop petroleum vulva, loss to some increase. (angleseyartsforum.org)