Ascorbic Acid
Ascorbic Acid Deficiency
Dehydroascorbic Acid
Scurvy
Antioxidants
L-Gulonolactone Oxidase
Sodium-Coupled Vitamin C Transporters
2,3-Diketogulonic Acid
Oxidation-Reduction
Free Radical Scavengers
Glutathione
Phosphorous Acids
Iron
Vitamin E
alpha-Tocopherol
Lipid Peroxidation
Chromaffin Granules
Oxalic Acid
Lipid Peroxides
Free Radicals
Biological Availability
Electrodes
Electrochemistry
Oxidative Stress
Chromatography, High Pressure Liquid
Copper
Chromaffin System
Rutin
Chlorobutanol
Thiobarbiturates
Dose-Response Relationship, Drug
Vitamins
Hydrogen Peroxide
Liver
Food, Fortified
Malpighiaceae
Organic Anion Transporters, Sodium-Dependent
Guinea Pigs
Uric Acid
Sugar Alcohol Dehydrogenases
Iron, Dietary
Phytic Acid
Indicators and Reagents
Aniline Hydroxylase
Plant Extracts
Peroxides
Adrenal Medulla
Thiobarbituric Acid Reactive Substances
Hydrogen-Ion Concentration
Oxalates
Hydroxyproline
Pharmaceutical Solutions
Double-blind intervention trial on modulation of ozone effects on pulmonary function by antioxidant supplements. (1/4830)
The aim of this study was to investigate whether the acute effects of ozone on lung function could be modulated by antioxidant vitamin supplementation in a placebo-controlled study. Lung function was measured in Dutch bicyclists (n = 38) before and after each training session on a number of occasions (n = 380) during the summer of 1996. The vitamin group (n = 20) received 100 mg of vitamin E and 500 mg of vitamin C daily for 15 weeks. The average ozone concentration during exercise was 77 microg/m3 (range, 14-186 microg/m3). After exclusion of subjects with insufficient compliance from the analysis, a difference in ozone exposure of 100 microg/m3 decreased forced expiratory volume in 1 second (FEV1) 95 ml (95% confidence interval (CI) -265 to -53) in the placebo group and 1 ml (95% CI -94 to 132) in the vitamin group; for forced vital capacity, the change was -125 ml (95% CI -384 to -36) in the placebo group and -42 ml (95% CI -130 to 35) in the vitamin group. The differences in ozone effect on lung function between the groups were statistically significant. The results suggest that supplementation with the antioxidant vitamins C and E confers partial protection against the acute effects of ozone on FEV1 and forced vital capacity in cyclists. (+info)Thiol-dependent degradation of protoporphyrin IX by plant peroxidases. (2/4830)
Protoporphyrin IX (PP) is the last porphyrin intermediate in common between heme and chlorophyll biosynthesis. This pigment normally does not accumulate in plants because its highly photodynamic nature makes it toxic. While the steps leading to heme and chlorophylls are well characterized, relatively little is known of the metabolic fate of excess PP in plants. We have discovered that plant peroxidases can rapidly degrade this pigment in the presence of thiol-containing substrates such as glutathione and cysteine. This thiol-dependent degradation of PP by horseradish peroxidase consumes oxygen and is inhibited by ascorbic acid. (+info)Stabilization of L-ascorbic acid by superoxide dismutase and catalase. (3/4830)
The effects of superoxide dismutase (SOD) and catalase on the autoxidation rate of L-ascorbic acid (ASA) in the absence of metal ion catalysts were examined. The stabilization of ASA by SOD was confirmed, and the enzyme activity of SOD, which scavenges the superoxide anion formed during the autoxidation of ASA, contributed strongly to this stabilization. The stabilization of ASA by catalase was observed for the first time; however, the specific enzyme ability of catalase would not have been involved in the stabilization of ASA. Such proteins as bovine serum albumin (BSA) and ovalbumin also inhibited the autoxidation of ASA, therefore it seems that non-specific interaction between ASA and such proteins as catalase and BSA might stabilize ASA and that the non-enzymatic superoxide anion scavenging ability of proteins might be involved. (+info)Glutathione-S-transferase (GSTM1) genetic polymorphisms do not affect human breast cancer risk, regardless of dietary antioxidants. (4/4830)
Glutathione-S-transferases catalyze the detoxication of carcinogen metabolites and reactive oxygen species (ROS) produced through a number of mechanisms. Glutathione-S-transferase (GST) M1 is polymorphic, and the null allele results in a lack of enzyme activity. Because there are indications that ROS may be involved in breast carcinogenesis, we sought to determine whether the GSTM1 null allele was associated with increased breast cancer, particularly among women with lower consumption of dietary sources of alpha-tocopherol, carotenoids and ascorbic acid. In a study of diet and cancer in western New York, women with primary, incident, histologically confirmed breast cancer (n = 740) and community controls (n = 810) were interviewed and an extensive food-frequency questionnaire administered. A subset of these women provided a blood specimen. DNA was extracted and genotyping performed for GSTM1. Data were available for 279 cases and 340 controls. The null allele did not increase breast cancer risk, regardless of menopausal status. There were also no differences in associations between the polymorphism and risk among lower and higher consumers of dietary sources of antioxidants or smokers and nonsmokers. These results indicate that GSTM1 genetic polymorphisms are not associated with breast cancer risk, even in an environment low in antioxidant defenses. (+info)Prospective cohort study of antioxidant vitamin supplement use and the risk of age-related maculopathy. (5/4830)
In a prospective cohort study, the authors examined whether self-selection for antioxidant vitamin supplement use affects the incidence of age-related maculopathy. The study population consisted of 21,120 US male physician participants in the Physicians' Health Study I who did not have a diagnosis of age-related maculopathy at baseline (1982). During an average of 12.5 person-years of follow-up, a total of 279 incident cases of age-related maculopathy with vision loss to 20/30 or worse were confirmed by medical record review. In multivariate analysis, as compared with nonusers of supplements, persons who used vitamin E supplements had a possible but nonsignificant 13% reduced risk of age-related maculopathy (relative risk = 0.87, 95 percent confidence interval (CI) 0.53-1.43), while users of multivitamins had a possible but nonsignificant 10% reduced risk (relative risk = 0.90, 95% CI 0.68-1.19). Users of vitamin C supplements had a relative risk of 1.03 (95% CI 0.71-1.50). These observational data suggest that among persons who self-select for supplemental use of antioxidant vitamin C or E or multivitamins, large reductions in the risk of age-related maculopathy are unlikely. Randomized trial data are accumulating to enable reliable detection of the existence of more plausible small-to-moderate benefits of these agents alone and in combination on age-related maculopathy. (+info)Demonstration of rapid onset vascular endothelial dysfunction after hyperhomocysteinemia: an effect reversible with vitamin C therapy. (6/4830)
BACKGROUND: Hyperhomocysteinemia is a major and independent risk factor for vascular disease. The mechanisms by which homocysteine promotes atherosclerosis are not well understood. We hypothesized that elevated homocysteine concentrations are associated with rapid onset endothelial dysfunction, which is mediated through oxidant stress mechanisms and can be inhibited by the antioxidant vitamin C. METHODS AND RESULTS: We studied 17 healthy volunteers (10 male and 7 female) aged 33 (range 21 to 59) years. Brachial artery diameter responses to hyperemic flow (endothelium dependent), and glyceryltrinitrate (GTN, endothelium independent) were measured with high resolution ultrasound at 0 hours (fasting), 2 hours, and 4 hours after (1) oral methionine (L-methionine 100 mg/kg), (2) oral methionine preceded by vitamin C (1g/day, for 1 week), and (3) placebo, on separate days and in random order. Plasma homocysteine increased (0 hours, 12.8+/-1.4; 2 hours, 25.4+/-2.5; and 4 hours, 31. 2+/-3.1 micromol/l, P<0.001), and flow-mediated dilatation fell (0 hours, 4.3+/-0.7; 2 hours, 1.1+/-0.9; and 4 hours, -0.7+/-0.8%) after oral L-methionine. There was an inverse linear relationship between homocysteine concentration and flow-mediated dilatation (P<0. 001). Pretreatment with vitamin C did not affect the rise in homocysteine concentrations after methionine (0 hours, 13.6+/-1.6; 2 hours, 28.3+/-2.9; and 4 hours, 33.8+/-3.7 micromol/l, P=0.27), but did ameliorate the reduction in flow-mediated dilatation (0 hours, 4. 0+/-1.0; 2 hours, 3.5+/-1.2 and 4 hours, 2.8+/-0.7%, P=0.02). GTN-induced endothelium independent brachial artery dilatation was not affected after methionine or methionine preceded by vitamin C. CONCLUSIONS: We conclude that an elevation in homocysteine concentration is associated with an acute impairment of vascular endothelial function that can be prevented by pretreatment with vitamin C in healthy subjects. Our results support the hypothesis that the adverse effects of homocysteine on vascular endothelial cells are mediated through oxidative stress mechanisms. (+info)L-Ascorbic acid potentiates nitric oxide synthesis in endothelial cells. (7/4830)
Ascorbic acid has been shown to enhance impaired endothelium-dependent vasodilation in patients with atherosclerosis by a mechanism that is thought to involve protection of nitric oxide (NO) from inactivation by free oxygen radicals. The present study in human endothelial cells from umbilical veins and coronary arteries investigates whether L-ascorbic acid additionally affects cellular NO synthesis. Endothelial cells were incubated for 24 h with 0.1-100 microM ascorbic acid and were subsequently stimulated for 15 min with ionomycin (2 microM) or thrombin (1 unit/ml) in the absence of extracellular ascorbate. Ascorbate pretreatment led to a 3-fold increase of the cellular production of NO measured as the formation of its co-product citrulline and as the accumulation of its effector molecule cGMP. The effect was saturated at 100 microM and followed a similar kinetics as seen for the uptake of ascorbate into the cells. The investigation of the precursor molecule L-gulonolactone and of different ascorbic acid derivatives suggests that the enediol structure of ascorbate is essential for its effect on NO synthesis. Ascorbic acid did not induce the expression of the NO synthase (NOS) protein nor enhance the uptake of the NOS substrate L-arginine into endothelial cells. The ascorbic acid effect was minimal when the citrulline formation was measured in cell lysates from ascorbate-pretreated cells in the presence of known cofactors for NOS activity. However, when the cofactor tetrahydrobiopterin was omitted from the assay, a similar potentiating effect of ascorbate pretreatment as seen in intact cells was demonstrated, suggesting that ascorbic acid may either enhance the availability of tetrahydrobiopterin or increase its affinity for the endothelial NOS. Our data suggest that intracellular ascorbic acid enhances NO synthesis in endothelial cells and that this may explain, in part, the beneficial vascular effects of ascorbic acid. (+info)Cytotoxic effect of paraquat on rat C6 glioma cells: evidence for the possibility of non-oxidative damage to the cells. (8/4830)
Although paraquat has been shown to cause oxidative damage to neuronal cells, little is known about its effect on glial cells. Thus the effect of paraquat on glial cells was examined using rat C6 glioma cells as a model system. Paraquat reduced cell viability in a concentration- and time-dependent manner, and this toxic effect was not significantly attenuated by various kinds of antioxidants. Furthermore, paraquat failed to increase 8-hydroxy-deoxyguanosine formation in the cells. These results indicate that paraquat can be toxic to glial cells and suggest that this cytotoxic effect may not be associated with the oxidative damage to the cells. (+info)Ascorbic acid deficiency can occur due to several reasons, including:
1. Poor diet: A diet that is lacking in vitamin C-rich foods such as citrus fruits, leafy greens, and bell peppers can lead to a deficiency.
2. Inability to absorb vitamin C: Certain medical conditions such as celiac disease, Crohn's disease, and ulcerative colitis can impair the body's ability to absorb vitamin C from food.
3. Increased demand: Vitamin C is essential for wound healing, and during pregnancy and lactation, the body requires more vitamin C to support fetal growth and development.
4. Long-term use of antacids or proton pump inhibitors: These medications can reduce the absorption of vitamin C.
The symptoms of ascorbic acid deficiency can vary depending on the severity of the deficiency and may include:
1. Fatigue and weakness
2. Mood changes such as irritability, anxiety, or depression
3. Difficulty healing from wounds or infections
4. Swelling or bleeding in the gums
5. Joint pain and stiffness
6. Frequent infections such as the common cold or flu
7. Poor digestion and diarrhea
If left untreated, ascorbic acid deficiency can lead to more severe conditions such as scurvy, which can cause:
1. Bleeding gums and teeth
2. Joint pain and swelling
3. Weakened immune system
4. Poor wound healing
5. Fatigue and weakness
Treatment for ascorbic acid deficiency typically involves supplementing with vitamin C, either orally or intravenously, and addressing any underlying causes of the deficiency. In severe cases, hospitalization may be necessary to monitor and treat any complications.
Prevention is key, so it's important to maintain a balanced diet that includes foods rich in vitamin C, such as citrus fruits, leafy greens, and berries. Additionally, avoiding smoking and limiting alcohol consumption can also help prevent ascorbic acid deficiency.
In conclusion, ascorbic acid deficiency is a common nutrient deficiency that can have significant impacts on overall health and well-being. Recognizing the causes, symptoms, and treatment options is essential for proper management and prevention of this condition.
There are several ways to prevent and treat scurvy, including:
* Eating foods rich in vitamin C, such as citrus fruits, leafy greens, and bell peppers.
* Taking vitamin C supplements.
* Increasing the intake of other nutrients that help the body absorb vitamin C, such as calcium and potassium.
* Avoiding foods and drinks that are high in oxalic acid, which can interfere with the absorption of vitamin C.
The discovery of scurvy and its treatment have had a significant impact on the history of medicine and nutrition. It led to the discovery of other essential nutrients and the development of modern nutrition science.
Intravenous ascorbic acid
Chemistry of ascorbic acid
Glyceryl octyl ascorbic acid
Dough conditioner
Shaken baby syndrome
Reichstein process
Paracetamol
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Alan Clemetson
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Complications of diabetes
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Threonic acid
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White-edge freshwater whipray
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1935 British Mount Everest reconnaissance expedition
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Airborne (dietary supplement)
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Beep (soft drink)
Benzene
Action of Ascorbic Acid on Corticotropin | Nature
DailyMed - ASCOR- ascorbic acid injection
L-Ascorbic acid 10357-S
ASCORBIC ACID - Books - NCBI
Ascorbic Acid (Vitamin C): MedlinePlus Drug Information
Grant Abstract: Ascorbic Acid Function and Metabolism
L. ascorbic acid | Nikon's Small World
MedlinePlus - Search Results for: Ascorbic acid OR Fanolesomab
ascorbic acid oral and butalbital-acetaminophen-caffeine oral Drug Interactions - RxList
Ascorbic acid - PubMed
cranberry + colloidal anhydrous silica + ascorbic acid | healthdirect
Ascorbic acid | The Fresh Loaf
Hydrocortisone-Ascorbic Acid-Thiamine Use Associated with Lower Mortality in Pediatric Septic Shock
ascorbic acid - PubChem Substance - NCBI
ascorbic acid and carbonyl iron | Michigan Medicine
Treatment of Hepatitis epidemica in Childhood with High Doses of Ascorbic
Acid
Ascorbic acid (vitamin C) | Page 2 | Cassiopaea Forum
Status of Ascorbic Acid in Iron Deficiency Anaemia and Thalassaemia | Acta Haematologica | Karger Publishers
Ascorbic Acid, USP, Powder (Vitamin C)
KANEGRADE(QINGDAO) INGREDIENTS CO., LTD - Ascorbic Acid, Vitamin B, Lutein
Ascorbic Acid - Side Effects, Uses, Dosage, Overdose, Pregnancy, Alcohol | RxWiki
Creator: Cox, Gerald J. / Language: English / Subject: Ascorbic Acid - Albert Szent-Gyorgyi - Profiles in Science Search Results
Ascorbic Acid - CooksInfo
ALPHA.-TOCOPHEROL, D-/TU AND Ascorbic Acid/TU AND BETA CAROTENE/TU AND Biotin/TU AND Calcium Cation/TU AND Cholecalciferol/TU...
ALPHA.-TOCOPHEROL, D-/TU AND ASCORBIC ACID/TU AND CALCIUM CATION/TU AND CHOLECALCIFEROL/TU AND CYANOCOBALAMIN/TU AND DOCONEXENT...
Total and Reduced Ascorbic Acid Levels in Rin and Normal Tomatoes.1 in: Journal of the American Society for Horticultural...
Effect of Seasonal Temperatures and Ascorbic Acid Supplementation on Performance of Broiler Chickens Maintained in Closed and...
"Ascorbic Acid is an Abettor in Calcium Urolithiasis: An Experimental S" by P. P. Singh, R. Kiran et al.
Forms of ascorbic acid2
- Some forms of ascorbic acid contain sodium and should be avoided if you are on a sodium- or salt-restricted diet. (medlineplus.gov)
- Technically, there are actually two forms of ascorbic acid: L-ascorbic acid and L-dehydroascorbic acid. (cooksinfo.com)
Doses2
- Diabetics, patients prone to recurrent renal calculi, those undergoing stool occult blood tests and those on sodium restricted diets or anticoagulant therapy should not take excessive doses of ascorbic acid over an extended period of time. (nih.gov)
- Do not stop using ascorbic acid suddenly after long-term use at high doses, or you could have "conditional" vitamin C deficiency. (drugs.com)
Dosage1
- Ascorbic Acid is the name for an active form of Vitamin C, a water-soluble vitamin that has been the subject of a great deal of controversy, especially with regard to dosage and disease prevention. (healthy-eating-support.org)
Scurvy3
- Ascorbic acid is also used to prevent and treat scurvy (a disease that causes fatigue, gum swelling, joint pain, and poor wound healing from a lack of vitamin C in the body). (medlineplus.gov)
- Ascorbic acid deficiency results in scurvy. (nih.gov)
- Ascorbic acid is recommended for the prevention and treatment of scurvy. (nih.gov)
Deficiency8
- People most at risk for ascorbic acid deficiency are those with a limited variety of food in their diet, or who have intestinal malabsorption problems from cancer or kidney disease. (medlineplus.gov)
- This hypothesis and mechanism will be tested in endothelial cells cultured at hyperglycemic glucose concentrations and extended to novel mouse models of ascorbic acid deficiency or repletion in the context of streptozotocin-induced diabetes. (nih.gov)
- The status of ascorbic acid was studied at the levels of platelet and leucocyte in 32 cases of iron deficiency anaemia, 35 cases of thalassaemia and 18 normal subjects. (karger.com)
- It was found that in iron deficiency anaemia, platelet ascorbic acid was significantly higher than normal values indicating tissue excess and came down sharply after treatment with iron. (karger.com)
- In thalassaemia, associated presumably with iron overload, ascorbic acid status of platelets was definitely lower than normal indicating tissue deficiency. (karger.com)
- Administration of ascorbic acid completely reverses the symptoms of ascorbic acid deficiency. (nih.gov)
- Its parenteral administration is desirable for patients with an acute deficiency or for those whose absorption of orally ingested ascorbic acid is uncertain. (nih.gov)
- Ascorbic acid is used to treat and prevent vitamin C deficiency . (drugs.com)
Chewable1
- Ascorbic acid comes in extended-release (long-acting) capsules and tablets, lozenges, chewable tablets, chewable gels (gummies), and liquid drops to be given by mouth. (medlineplus.gov)
Water-soluble1
- Ascorbic Acid (vitamin C) is a water-soluble vitamin. (nih.gov)
Capsules1
- Buffered Ascorbic Acid Capsules - 90 count has been added to your list successfully. (drhyman.com)
Dose5
- ASCOR vials contain 25, 000 mg of ascorbic acid and the largest recommended single dose is 200 mg. (nih.gov)
- Cigarette smoking may decrease the effectiveness of ascorbic acid and you may need to take a larger dose. (medlineplus.gov)
- Talk to your doctor or pharmacist about your dose of ascorbic acid if you use tobacco products. (medlineplus.gov)
- In some cases, you may not be able to use ascorbic acid and carbonyl iron, or you may need a dose adjustment or special precautions. (uofmhealth.org)
- In order to achieve a therapeutic dose, Dr. Pauling himself took a powdered form of ascorbic acid for much of his life. (healthy-eating-support.org)
Collagen2
- Our bodies use Ascorbic Acid to make collagen. (cooksinfo.com)
- In humans, an exogenous source of ascorbic acid is required for collagen formulation and tissue repair. (nih.gov)
Prevention2
- The second aim tests how ascorbic acid prevents endothelial barrier failure caused by high glucose concentrations, again with focus on a mechanism involving ascorbic acid prevention of uncoupling of nitric oxide synthase. (nih.gov)
- Ascorbic acid in the treatment and prevention of skin diseases]. (nih.gov)
Metabolism5
- This decrease is due to oxidative stress from excess glucose and fatty acid metabolism. (nih.gov)
- Heller-Krause rightly refers to the danger of overdosing only one amino acid, which can lead to correlation disturbances in protein metabolism. (seanet.com)
- The vitamin is involved in tyrosine metabolism, conversion of folic acid to folinic acid, carbohydrate metabolism, synthesis of lipids and proteins, iron metabolism, resistance to infections, and cellular respiration. (nih.gov)
- An amino acid derivative, synthesized from methionine and lysine, required in energy metabolism. (medscape.com)
- Can promote excretion of excess fatty acids in patients with defects in fatty acid metabolism or specific organic acidopathies that cause acyl CoA esters to bioaccumulate. (medscape.com)
Depletion3
- when assayed by the adrenal ascorbic acid-depletion method of Sayers, Sayers and Woodbury 3 . (nature.com)
- Key to his aim is to assess whether diabetes-induced oxidative stress and subsequent endothelial dysfunction is worsened by depletion of ascorbic acid and reversed by its repletion. (nih.gov)
- An abnormal increase in the acidity of the body's fluids, caused either by accumulation of acids or by depletion of bicarbonates. (cdc.gov)
Sodium2
- Ascorbic Acid 500 mg, Disodium Edetate 0.25 mg, in Water for Injection q.s.. pH (range 5.5 to 7.0) adjusted with Sodium Bicarbonate and Sodium Hydroxide. (nih.gov)
- Use of sodium citrate and citric acid (Bicitra), vitamin C and vitamin E, thioctic acid (ie, lipoic acid), and NAC are included here. (medscape.com)
Supplements1
- Ascorbic acid supplements are available alone and in combination with other vitamins. (medlineplus.gov)
Glucose4
- More recent results show that intracellular ascorbic acid also prevents the increase in endothelial barrier permeability caused by high glucose concentrations. (nih.gov)
- The mechanism of this barrier protection is proposed to involve ascorbic acid scavenging of reactive oxygen species generated in response to high glucose concentrations, as well as ascorbic acid recycling of tetrahydrobiopterin to prevent uncoupling of endothelial nitric oxide synthase. (nih.gov)
- Since many of the deleterious effects of high glucose concentrations are caused by activation of the Receptor for Advanced Glycation End-products (RAGE), the third aim will test the mechanism by which ascorbic acid prevents increased endothelial permeability due to RAGE ligands. (nih.gov)
- Diabetics taking more than 500 mg of ascorbic acid daily may obtain false reading of the urinary glucose test. (nih.gov)
Amino acid1
- NAC is the N -acetyl derivative of the amino acid cysteine. (medscape.com)
Citric acid3
- Commercially prepared mixes of ascorbic and citric acid-seasonally available among canners' supplies in supermarkets. (cooksinfo.com)
- Sometimes citric acid powder is sold in supermarkets, but it is less effective in controlling discoloration. (cooksinfo.com)
- Citric acid mixtures may also be used. (medscape.com)
Injection6
- Add 200 mg of ascorbic acid (equivalent to 0.4 mL of ASCOR) to 7.5 mL of Sterile Water for Injection to produce an infusion solution having an approximate osmolarity of 290 mOsmol/L. In this specific example, addition of solute is NOT necessary because the solution is isotonic. (nih.gov)
- Ascorbic Acid injection is a clear, colorless to slightly yellow sterile solution of Ascorbic Acid in Water for Injection, for intravenous, intramuscular or subcutaneous use. (nih.gov)
- Animal reproduction studies have not been conducted with Ascorbic Acid Injection. (nih.gov)
- It is also not known whether Ascorbic Acid Injection can cause fetal harm when administered to a pregnant woman or can affect reproduction capacity. (nih.gov)
- Ascorbic Acid Injection should be given to a pregnant woman only if clearly needed. (nih.gov)
- Caution should be exercised when Ascorbic Acid Injection is administered to a nursing woman. (nih.gov)
Increases1
- The recommended dietary allowance of vitamin C (ascorbic acid) increases with age. (drugs.com)
Vitro3
- The fourth aim will extend the in vitro studies to novel mouse models in which ascorbic concentrations can be manipulated by dietary and genetic means to test effects of increased oxidative stress due to streptozotocin- induced diabetes. (nih.gov)
- Regenerative Effect of L-Ascorbic Acid on the In vitro Grown Plants. (who.int)
- Aim: In the present work, we have studied the effect of L ascorbic acid (LAA) on the regeneration of plants from different families cultured in vitro. (who.int)
Allergic2
- tell your doctor and pharmacist if you are allergic to ascorbic acid, any other medications, or any of the ingredients in ascorbic acid products. (medlineplus.gov)
- You should not use ascorbic acid if you have ever had an allergic reaction to a vitamin C supplement. (drugs.com)
Medication1
- Ascorbic acid and carbonyl iron may also be used for purposes not listed in this medication guide. (uofmhealth.org)
Bulk3
- Most U.S. vitamin companies then buy the bulk ascorbic acid from this single facility. (cassiopaea.org)
- Browse all the Ascorbic Acid in Rohtas on our website and place your bulk orders with us. (ryan-international.in)
- As an eminent Ascorbic Acid Dealer and Wholesaler in Rohtas , we are available a phone call and click away to meet your bulk orders. (ryan-international.in)
Products1
- Other drugs may interact with ascorbic acid and carbonyl iron, including prescription and over-the-counter medicines, vitamins, and herbal products. (uofmhealth.org)
Connective1
- Ascorbic acid is important for the skin and connective tissues, for normal chemical and hormonal production, and for the immune system. (uofmhealth.org)
Drugs2
- What other drugs will affect ascorbic acid and carbonyl iron? (uofmhealth.org)
- When isolated into artificial commercial forms, like ascorbic acid, these purified synthetics act as drugs in the body. (cassiopaea.org)
Potatoes2
- Ascorbic acid (vitamin C) is found in citrus fruit, tomatoes, potatoes, and leafy vegetables. (uofmhealth.org)
- Ascorbic acid ( vitamin C ) occurs naturally in foods such as citrus fruit, tomatoes, potatoes, and leafy vegetables. (drugs.com)
Treatment4
- Holzbauer and Walter 1 have recently reported that the adrenal ascorbic acid-depleting properties of corticotropin are destroyed by treatment with ascorbic acid at room temperature for 18-20 hr. (nature.com)
- Ascorbic acid in the treatment of eye diseases]. (nih.gov)
- Broilers in both houses were randomly subjected to four drinking water treatments (9 birds in each 6 replicates/treatment): 0, 100, 200 and 300 ppm ascorbic acid. (scialert.net)
- In experiment 2, treatment with ascorbic acid had no effect as preservative and extending shelf life. (skemman.is)
Naturally1
- The ascorbic acid you buy at the grocery store every few weeks, thinking you are buying Vitamin C, is just a chemical copy of naturally occurring ascorbic acid, which itself is still only a fraction of the actual Vitamin C. Real vitamin C is part of something living, and as such, can impart life. (cassiopaea.org)
Pharmacist1
- Your pharmacist can provide more information about ascorbic acid and carbonyl iron. (uofmhealth.org)
Antioxidants1
- Ascorbic acid is in a class of medications called antioxidants. (medlineplus.gov)
Molecular weight1
- Unlike other derivatives of Vitamin C, the ethylated form is closer in molecular weight to the purest form of Ascorbic Acid, thus making it quicker and more effective in penetrating the skin's layers at a deeper level. (merchantmanila.com)
Side effects2
- Ascorbic acid may cause side effects. (medlineplus.gov)
- What are the possible side effects of ascorbic acid and carbonyl iron? (uofmhealth.org)
Solution1
- The concentration of ascorbic acid in the final, admixture solution for infusion is to be the range of 1 to 25 mg of ascorbic acid per mL. (nih.gov)
Pregnant1
- If you become pregnant while taking ascorbic acid, call your doctor. (medlineplus.gov)
Form1
- This special form of ascorbic acid lessens possible gastric irritation in sensitive individuals. (drhyman.com)
Dietary2
- Ascorbic acid (vitamin C) is used as a dietary supplement when the amount of ascorbic acid in the diet is not enough. (medlineplus.gov)
- Abstract: DESCRIPTION (provided by applicant): Plasma and cell levels of vitamin C, or ascorbic acid, are decreased about 50% in persons with type II diabetes in sub-optimal glycemic control, despite presumably adequate dietary intakes. (nih.gov)
Iron8
- What is the most important information I should know about ascorbic acid and carbonyl iron? (uofmhealth.org)
- Ascorbic acid and carbonyl iron is a combination vitamin/mineral supplement that helps your body better absorb iron from your diet. (uofmhealth.org)
- What should I discuss with my healthcare provider before taking ascorbic acid and carbonyl iron? (uofmhealth.org)
- How should I take ascorbic acid and carbonyl iron? (uofmhealth.org)
- For best results, take ascorbic acid and carbonyl iron on an empty stomach at least 1 hour before or 2 hours after a meal. (uofmhealth.org)
- Ascorbic acid and carbonyl iron may be taken with food if it upsets your stomach. (uofmhealth.org)
- What should I avoid while taking ascorbic acid and carbonyl iron? (uofmhealth.org)
- Ascorbic acid and carbonyl iron can make it harder for your body to absorb certain medicines you take by mouth. (uofmhealth.org)
Body2
- Compared to the control group, ascorbic acid improved feed intake, body weight gain and feed conversion ratio by 6%, 9% and 3% (cool season) and 8%, 11% and 5% (hot season). (scialert.net)
- Ascorbic acid is reversibly oxidized to dehydroascorbic acid in the body. (nih.gov)
Oxygen1
- L-ascorbic acid becomes L-dehydroascorbic acid when it reacts with oxygen in the air. (cooksinfo.com)
List1
- This is not a complete list of Ascorbic Aciddrug interactions. (rxwiki.com)