Accumulation or retention of free fluid within the peritoneal cavity.
Presence of milky lymph (CHYLE) in the PERITONEAL CAVITY, with or without infection.
A transplantable, poorly differentiated malignant tumor which appeared originally as a spontaneous breast carcinoma in a mouse. It grows in both solid and ascitic forms.
An operation for the continuous emptying of ascitic fluid into the venous system. Fluid removal is based on intraperitoneal and intrathoracic superior vena cava pressure differentials and is performed via a pressure-sensitive one-way valve connected to a tube traversing the subcutaneous tissue of the chest wall to the neck where it enters the internal jugular vein and terminates in the superior vena cava. It is used in the treatment of intractable ascites.
The serous fluid of ASCITES, the accumulation of fluids in the PERITONEAL CAVITY.
A form of PERITONITIS seen in patients with TUBERCULOSIS, characterized by lesion either as a miliary form or as a pelvic mass on the peritoneal surfaces. Most patients have ASCITES, abdominal swelling, ABDOMINAL PAIN, and other systemic symptoms such as FEVER; WEIGHT LOSS; and ANEMIA.
Tumors or cancer of the PERITONEUM.
Liver disease in which the normal microcirculation, the gross vascular anatomy, and the hepatic architecture have been variably destroyed and altered with fibrous septa surrounding regenerated or regenerating parenchymal nodules.
Tumors or cancer of the OVARY. These neoplasms can be benign or malignant. They are classified according to the tissue of origin, such as the surface EPITHELIUM, the stromal endocrine cells, and the totipotent GERM CELLS.
Experimentally induced tumors of the LIVER.
Abnormal increase of resistance to blood flow within the hepatic PORTAL SYSTEM, frequently seen in LIVER CIRRHOSIS and conditions with obstruction of the PORTAL VEIN.
A collection of watery fluid in the pleural cavity. (Dorland, 27th ed)
A condition characterized by a dry, waxy type of swelling (EDEMA) with abnormal deposits of MUCOPOLYSACCHARIDES in the SKIN and other tissues. It is caused by a deficiency of THYROID HORMONES. The skin becomes puffy around the eyes and on the cheeks. The face is dull and expressionless with thickened nose and lips.
Functional KIDNEY FAILURE in patients with liver disease, usually LIVER CIRRHOSIS or portal hypertension (HYPERTENSION, PORTAL), and in the absence of intrinsic renal disease or kidney abnormality. It is characterized by intense renal vasculature constriction, reduced renal blood flow, OLIGURIA, and sodium retention.
Experimentally induced new abnormal growth of TISSUES in animals to provide models for studying human neoplasms.
Presence of fluid in the pleural cavity resulting from excessive transudation or exudation from the pleural surfaces. It is a sign of disease and not a diagnosis in itself.
INFLAMMATION of the PERITONEUM lining the ABDOMINAL CAVITY as the result of infectious, autoimmune, or chemical processes. Primary peritonitis is due to infection of the PERITONEAL CAVITY via hematogenous or lymphatic spread and without intra-abdominal source. Secondary peritonitis arises from the ABDOMINAL CAVITY itself through RUPTURE or ABSCESS of intra-abdominal organs.
The outer margins of the ABDOMEN, extending from the osteocartilaginous thoracic cage to the PELVIS. Though its major part is muscular, the abdominal wall consists of at least seven layers: the SKIN, subcutaneous fat, deep FASCIA; ABDOMINAL MUSCLES, transversalis fascia, extraperitoneal fat, and the parietal PERITONEUM.
The space enclosed by the peritoneum. It is divided into two portions, the greater sac and the lesser sac or omental bursa, which lies behind the STOMACH. The two sacs are connected by the foramen of Winslow, or epiploic foramen.
Radiographic visualization of the body between the thorax and the pelvis, i.e., within the peritoneal cavity.
General or unspecified injuries involving organs in the abdominal cavity.
Messages between computer users via COMPUTER COMMUNICATION NETWORKS. This feature duplicates most of the features of paper mail, such as forwarding, multiple copies, and attachments of images and other file types, but with a speed advantage. The term also refers to an individual message sent in this way.
Mechanical food dispensing machines.
The guidelines and policy statements set forth by the editor(s) or editorial board of a publication.
The profession of writing. Also the identity of the writer as the creator of a literary production.
A publication issued at stated, more or less regular, intervals.
The functions and activities carried out by the U.S. Postal Service, foreign postal services, and private postal services such as Federal Express.
A loose confederation of computer communication networks around the world. The networks that make up the Internet are connected through several backbone networks. The Internet grew out of the US Government ARPAnet project and was designed to facilitate information exchange.
A procedure in which fluid is withdrawn from a body cavity or organ via a trocar and cannula, needle, or other hollow instrument.
Operation for biliary atresia by anastomosis of the bile ducts into the jejunum or duodenum.
Device constructed of either synthetic or biological material that is used for the repair of injured or diseased blood vessels.
Conferences, conventions or formal meetings usually attended by delegates representing a special field of interest.
The plan and delineation of prostheses in general or a specific prosthesis.

Identification and characterization of a homozygous deletion found in ovarian ascites by representational difference analysis. (1/991)

We have performed representational difference analysis (RDA) on DNA from tumor cells and normal fibroblasts isolated from the ascites of a patient with ovarian cancer. Five of six products of the RDA were homozygously deleted from the tumor DNA. One of these products has been characterized and identifies a homozygous deletion of approximately 6.9 Mb at chromosome 9p21 in the original ovarian tumor material. This deletion encompasses CDKN2A (p16), CDKN2B (p15), and IFN-alpha. PCR analysis of other tumor cell lines using the novel STS based on the RDA product has shown it to lie between IFN-alpha and p16, and to identify the distal extent of a homozygous deletion in another ovarian cancer cell line. These data provide further evidence for a tumor suppressor locus distinct from, but mapping close to, p16 on 9p21. Cytogenetic analysis using comparative genomic hybridization (CGH) performed on the same primary tumor confirmed a loss of material from chromosome 9p. However, the CGH technique had neither the resolution nor the sensitivity to define a subregion of homozygous loss.  (+info)

Antibiotic penetrance of ascitic fluid in dogs. (2/991)

Antibiotic concentrations in ascitic fluid after parenteral therapy may be important in the treatment of peritonitis. We have created ascites in dogs by partial ligation of the inferior vena cava. Ascitic fluid volume was measured at the time each antibiotic was administered. Nine antibiotics were studied in the same three dogs. Antibiotic concentration in ascitic fluid was found to vary inversely with ascites volume. Percentage of penetration (ratio of ascites peak to serum peak x100) ranged from 5.8 to 65% among the drugs studied. Only metronidazole showed a statistically significant higher percentage of penetration than other antimicrobials. Concentrations in ascitic fluid after single doses of cephalothin (15 mg/kg) and the aminoglycosides (2 mg/kg, gentamicin and tobramycin; 7.5 mg/kg, amikacin and kanamycin) did not exceed the minimum inhibitory concentration of many gram-negative rods and may justify the use of higher than usual initial parenteral doses, or possibly initial intraperitoneal administration in seriously ill patients.  (+info)

An endonuclease from mouse cells specific for single-stranded DNA. (3/991)

An endonuclease with a molecular weight of about 70000 (5-6S) was extensively purified from mouse ascites cells. The enzyme specifically attacks single-stranded DNA which is degraded mainly to oligonucleotides, with 5-10 residues. Supercoiled covalently closed circular phage DNA is converted to the linear relaxed form. The enzyme activity is highly sensitive to salt and can be stimulated by reagents lowering the dielectric constant of the buffer such as dimethylsulfoxide and glycerol.  (+info)

Hyperreactio luteinalis associated with chronic renal failure. (4/991)

Hyperreactio luteinalis is a rare benign condition characterized by bilateral ovarian enlargement associated with pregnancies where high concentrations of maternal serum human chorionic gonadotrophins are present. This condition may mimic the ovarian hyperstimulation syndrome. We report a case of a 34 year old woman with a history of chronic renal failure on haemodialysis who presented at 10 weeks' gestational age with hyperreactio luteinalis which was treated conservatively. Because of chronic renal failure, the presentation and course of the disease was different from that which has been previously reported.  (+info)

Lymph and pulmonary response to isobaric reduction in plasma oncotic pressure in baboons. (5/991)

Plasma colloid osmotic pressure was reduced by 76% (from 19.6 +/- 0.6 to 4.7 +/- 1.5 mm Hg) in five baboons while pulmonary capillary hydrostatic pressure was maintained at a normal level. This resulted in fluid retention, weight gain, peripheral edema and ascites, but no pulmonary edema. Thoracic duct lymph flow increased 6-fold and pulmonary lymph flow 7-fold. Thoracic duct lymph had a lower colloid osmotic pressure (2.0 +/- 0.7 mm Hg) than plasma (4.7 +/- 1.5 mm Hg), whereas the colloid osmotic pressure of pulmonary lymph (4.7 +/- 0.7 mm Hg) was the same as that of plasma. The lymph-plasma ratio for albumin fell in thoracic duct lymph but remained unchanged in pulmonary lymph. The difference between plasma colloid osmotic pressure and pulmonary artery wedge pressure decreased from 15.3 +/- 1.9 to -0.7 +/- 2.9 mm Hg. Despite this increase in filtration force, the lungs were protected from edema formation by a decrease of 11 mm Hg in pulmonary interstitial colloid osmotic pressure and a 7-fold increase in lymph flow.  (+info)

Fas gene mutation in the progression of adult T cell leukemia. (6/991)

Fas antigen (Apo-1/CD95) is an apoptosis-signaling cell surface receptor belonging to the tumor necrosis factor receptor superfamily. Adult T cell leukemia (ATL) cells express Fas antigen and show apoptosis after treatment with an anti-Fas monoclonal antibody. We established the ATL cell line KOB, which showed resistance to Fas-mediated apoptosis, and found that KOB expressed two forms of Fas mRNA, the normal form and a truncated form. The truncated transcript lacked 20 base pairs at exon 9, resulting in a frame shift and the generation of a premature stop codon at amino acid 239. The same mutation was detected in primary ascitic cells and peripheral blood cells. The mutation was not detected in lymph node cells, however, although all of the primary ATL cells were of the same clonal origin. A retroviral-mediated gene transfer of the truncated Fas to Jurkat cells rendered the cells resistant to Fas-mediated apoptosis, suggesting a dominant negative interference mechanism. These results indicate that an ATL subclone acquires a Fas mutation in the lymph nodes, enabling the subclone to escape from apoptosis mediated by the Fas/Fas ligand system and proliferate in the body. Mutation of the Fas gene may be one of the mechanisms underlying the progression of ATL.  (+info)

Effects of ursodeoxycholic acid on systemic, renal and forearm haemodynamics and sodium homoeostasis in cirrhotic patients with refractory ascites. (7/991)

Systemic arterial vasodilatation has been implicated in the pathogenesis of sodium retention in cirrhosis. Hydrophobic bile acids, which have vasodilatory actions, may be involved. Ursodeoxycholic acid, a hydrophilic bile acid, could potentially decrease systemic arterial vasodilatation, possibly due to its antioxidant effects, and improve sodium handling in cirrhosis. The effects of ursodeoxycholic acid on systemic, renal and forearm haemodynamics, liver function and renal sodium handling were assessed in vasodilated cirrhotic patients with refractory ascites treated with a transjugular intrahepatic porto-systemic shunt (TIPS). Eight cirrhotic patients with refractory ascites without TIPS placement served as controls for the sodium handling effects of ursodeoxycholic acid. From 1 month post TIPS, seven patients were studied before, after 1 month of treatment with ursodeoxycholic acid (15 and at 1 month follow-up. Lipid peroxidation products were used as indices of its antioxidant effects. Ursodeoxycholic acid caused a significant reduction in sodium excretion in both groups (P<0.05). This, in the post-TIPS patients (urinary sodium excretion: 35+/-8 mmol/day at 1 month versus 93+/-21 mmol/day at baseline, P<0.05), was due to a significant increase in sodium reabsorption proximal to the distal tubule (P<0.05), without any significant changes in systemic, renal or forearm haemodynamics, or in liver function. No significant change in lipid peroxidation products was observed. We conclude that: (i) in cirrhotic patients with refractory ascites, ursodeoxycholic acid causes sodium retention, (ii) the abnormality in sodium handling in the post-TIPS cirrhotic patients appears to be the result of a direct effect on the proximal nephron, suggesting that factors other than systemic vasodilatation also contribute to sodium retention in cirrhosis, (iii) caution should be exercised in administering ursodeoxycholic acid in cirrhotic patients with ascites.  (+info)

Expression of phosphatidylethanolamine N-methyltransferase in Yoshida ascites hepatoma cells and the livers of host rats. (8/991)

Previous studies have implicated phosphatidylethanolamine N-methyltransferase-2 (PEMT2) in the regulation of non-neoplastic liver growth [Tessitore,L., Cui,Z. and Vance,E. (1997) Biochem. J., 322, 151-154]. We have now investigated whether or not PEMT2 is also involved in the control of proliferation of hepatoma cells growing in an animal and cell death by apoptosis in the liver of tumor-bearing rats. PEMT activity was barely detectable and PEMT2 protein was absent in hepatoma cells growing exponentially in vivo whereas CTP:phosphocholine cytidylyltransferase (CT) activity and expression were high. The lack of PEMT2 corresponded with the absence of its mRNA. Both PEMT2 protein and mRNA appeared when cells entered the stationary phase of tumor growth and, in parallel, CT expression decreased. The host liver first became hyperplastic and exhibited a slight increase in CT activity and decrease in PEMT2 expression. During the stationary phase of hepatoma growth the host liver regressed and eventually became hypoplastic following induction of apoptosis. The appearance of apoptosis in the host liver was associated with a marked reduction in both CT activity and expression as well as an enhancement of PEMT activity and PEMT2 expression. McArdle RH7777 hepatoma cells underwent apoptosis when transfected with cDNA for PEMT2. The evidence supports the proposal that PEMT2 may have a role in the regulation of 'in vivo' hepatoma and hepatocyte cell division as well as hepatocyte cell death by apoptosis.  (+info)

TY - JOUR. T1 - Serum cystatin C level is a good prognostic marker in patients with cirrhotic ascites and normal serum creatinine levels. AU - Seo, Yeon Seok. AU - Jung, Eun Suk. AU - An, Hyonggin. AU - Kim, Jeong Han. AU - Jung, Young Kul. AU - Kim, Ji Hoon. AU - Yim, Hyung Joon. AU - Yeon, Jong Eun. AU - Byun, Kwan Soo. AU - Kim, Chang Duck. AU - Ryu, Ho Sang. AU - Um, Soon Ho. PY - 2009. Y1 - 2009. N2 - Background/Aims: Serum creatinine (Cr) is not a reliable marker for early detection of renal dysfunction in patients with cirrhotic ascites. Several reports have suggested that cystatin C (CysC) is more sensitive than Cr for detecting reduced renal function in these patients. This study evaluated the clinical significance of CysC in patients with cirrhotic ascites and a normal serum Cr level. Methods: We enrolled patients with ascites and a normal serum Cr level (,1.2 mg/dl). Liver function tests, international normalized ratio (INR) and serum Cr and CysC levels were measured on the same day ...
Malignant ascites represents a severe clinical problem for physicians and patients being confronted with this common symptom of advanced-stage gastrointestinal cancer. Unfortunately, there is no standardized and evidence-based treatment for malignant ascites and therapies which are commonly being used are only temporarily effective. Newer modes of therapy, such as the application of the tri-functional antibody catumaxomab, are associated with significant side effects and are limited to patients in stages of good overall performance. Therefore, there is still an urgent need for more effective, longer-lasting, and less toxic modes of treatment for peritoneal effusions caused by gastrointestinal cancers.. Preclinical data strongly suggest that bevacizumab might be a very effective agent for the treatment of malignant ascites, which is in large part caused by the hyperpermeability-promoting factor VEGF. Emerging clinical results from cancer patients with malignant ascites treated with bevacizumab ...
Malignant ascites represents a severe clinical problem for physicians and patients being confronted with this common symptom of advanced-stage gastrointestinal cancer. Unfortunately, there is no standardized and evidence-based treatment for malignant ascites and therapies which are commonly being used are only temporarily effective. Newer modes of therapy, such as the application of the tri-functional antibody catumaxomab, are associated with significant side effects and are limited to patients in stages of good overall performance. Therefore, there is still an urgent need for more effective, longer-lasting, and less toxic modes of treatment for peritoneal effusions caused by gastrointestinal cancers.. Preclinical data strongly suggest that bevacizumab might be a very effective agent for the treatment of malignant ascites, which is in large part caused by the hyperpermeability-promoting factor VEGF. Emerging clinical results from cancer patients with malignant ascites treated with bevacizumab ...
Best Treatments for Cirrhotic ascites in Mumbai : Find Best Doctors, Book Appointment, Call Now, Get Address & numbers of Best Treatments for Cirrhotic ascites in Mumbai
Ascites formation is often observed in ovarian cancer patients. Vascular permeability factor (VPF) may induce ascites formation. We established an animal model of ascites formation and re-accumulation by i.p. transplantation of a human ovarian adenocarcinoma cell line, NOS2, into nude mice. The form …
BEVERLY, MA-(Marketwired - December 11, 2017) - BioVie Inc. (OTCQB: BIVI), a clinical-stage company focused on the development and commercialization of innovative drug therapies for liver disease, announced today that the US Food and Drug Administration (FDA) has granted Fast Track designation for BIV201 (continuous infusion terlipressin), the Companys patented Orphan drug candidate. BIV201 is currently being evaluated for the treatment of refractory ascites due to liver cirrhosis in a mid-stage (Phase 2a) US clinical trial, with 2 of the planned 6 patients having been treated with this therapy to date.. The FDAs Fast Track program is designed to facilitate development and expedite the review of drug candidates that are intended to treat serious or life-threatening conditions and demonstrate the potential to address unmet medical needs. Fast Track designation allows for more frequent communications with the FDA to discuss the clinical development program and review process. Upon submission of ...
The Prevalence and Causative Bacteria of Spontaneous Bacterial Peritonitis in Patients with Ascites Due to Liver Cirrhosis Mostafa M. Ragheb* Hassan Nasr El
Background: Differential diagnosis of ascites is a common clinical problem. Less expensive biochemical techniques are required to differentiate ascites with unknown etiology. Aim: To evaluate the diagnostic efficiency of ascitic fluid cholesterol, serum ascites albumin gradient (SAAG) and serum ascites cholesterol gradients (SACG) in differentiating cirrhotic, tuberculous and malignant ascites. Methods: 50 patients (25 with hepatic cirrhosis, 15 with tuberculosis and10 with malignancy) were evaluated for ascitic fluid total protein, albumin, cholesterol, SAAG and SACG. Results: The mean ascitic fluid cholesterol was significantly higher in malignant ascites when compared with cirrhosis and tuberculous ascites (p= 0.0001 each). The difference between tuberculous and cirrhotic ascites was also significant (p= 0.001). The mean value of SAAG was significantly higher in cirrhosis when compared with tuberculous and malignant ascites (p= 0.0001; p= 0.001 respectively) but the difference between ...
One of the major limitations of immunotherapy with rIL-2 protein for ovarian cancer is that, due to its short half-life, frequent injections of high doses of the protein are required to sustain therapeutic levels, and this is often associated with serious side effects (21). Although i.v. bolus injection of IL-2 results in a serum half-life of ∼7 min, i.p. infusion results in a peritoneal fluid half-life of ∼22 h (22, 23). Recently, a promising 25% response rate was reported after treatment of advanced ovarian cancer patients with IL-2 protein delivered by either continuous 7-day i.p. infusion or weekly 24-h i.p. infusion (11). Constant delivery of the IL-2 protein over either 7 days or 24 h resulted in stable IL-2 levels in the tumor ascites during the infusion period. These results suggest that one improvement in IL-2 therapy for ovarian cancer may be administration of IL-2 in such a manner that the sharply fluctuating IL-2 levels common after bolus protein therapy are avoided.. In the ...
This single-centre trial evaluated the efficacy of intraperitoneal catumaxomab in outpatients with malignant ascites related to epithelial tumours.
Ovarian cancer has often spread beyond the ovary to involve the peritoneal cavity at the time of diagnosis (2) and associated with malignant ascites formation (29-31). Debulking surgery followed by i.v. chemotherapy may allow clinical remission of the disease but disease recurrence in the peritoneal cavity is common and will eventually lead to demise of the patient. In the present study, we report that ovarian cancer xenografts growing in SCID mice are inhibited by bevacizumab and rapamycin. Both rapamycin and bevacizumab as a single treatment have a defined effect on the inhibition of tumor growth, tumor cell dissemination, and prolongation of survival. When combined with rapamycin, however, bevacizumab reduces tumor growth by ∼94% and significantly prolongs survival. Indeed, the combination of BEV/RAPA induces a significant decrease in the number of proliferating cells, the mean of Ki-67-positive cells, the mean vessel density, and ascites formation and inhibited tumor cells from ...
Catumaxomab (trade name Removab) is a rat-mouse hybrid monoclonal antibody which is used to treat malignant ascites, a condition occurring in people with metastasizing cancer. It binds to antigens CD3 and EpCAM. It is in clinical trials in the United States currently and is used in Europe. It was developed by Fresenius Biotech and Trion Pharma (Germany). The drug is used for the treatment of malignant ascites in people with EpCAM-positive cancer if a standard therapy is not available. Ascites is an accumulation of fluid in the peritoneal cavity. The usual treatment of malignant ascites is to puncture the peritoneum to let the accumulated fluid drain out. After the puncture, catumaxomab is given as an intraperitoneal infusion. The procedure is repeated four times within about eleven days. It has been shown that puncture free survival can be increased from 11 to 46 days with this treatment. Common adverse effects include fever, nausea and vomiting. Fever and pain should be controlled by giving ...
A 75-year-old man with rectal cancer had consumed an average of6 6 g of alcohol per day for 47 years. However, his liver function was within normal limits and his Child-Pugh classification was A before initiation of therapy. He underwent neoadjuvant chemoradiation and a low anterior resection. The patient then received CapeOX as an adjuvant therapy. During the fourth cycle of CapeOX, computed tomography(CT)showed massive ascites. The chemotherapy was discontinued and treatment including a diuretic agent was initiated. The ascites gradually decreased and 8 months after the discontinuation of CapeOX, CT showed neither the presence ofascites nor recurrence ofthe cancer or metastasis. When a patient has a history ofexcessive alcohol intake, even iftest results for liver function are within normal limits, we should be aware ofthe hepatic toxicity ofCapeOX.
Previous studies by our group and others have addressed the relationship between leukocyte infiltration into solid tumors and chemokine expression (14 , 16 , 23) . To our knowledge, this work is the first comprehensive study of CC chemokines and chemokine receptor expression in human ovarian cancer ascites.. We found that ascitic fluid is rich in CC chemokines and that the CD14-expressing cells and T cells present in ovarian cancer ascites express CC chemokine receptor mRNA and protein.. Is the extent and phenotype of the leukocyte infiltration in ovarian ascites related to chemokines and chemokine receptor expression? Gradients of chemokines usually cause tissue recruitment of leukocytes through effects on adhesion and endothelial transmigration (24) . Our data suggest that CC chemokine protein levels are significantly higher in ascitic fluid than in patient plasma samples. Therefore, chemokines present in ascites could form a gradient for leukocyte migration into the peritoneal cavity. ...
Ascites. Side view of a 76-year-old mans distended abdomen, showing the condition of ascites. The abdomen is severely swollen with fluid. The fluid has collected in the peritoneal cavity between the internal organs and the abdominal wall. As well as abdominal discomfort, ascites can cause breathing problems due to pressure on the diaphragm. Ascites occurs in many different conditions including liver cirrhosis and heart or kidney failure. This patient was suffering from cholangiocarcinoma, a rare cancer of the bile duct. Treatment of ascites may include diuretic drugs, fluid and salt restriction, and sometimes drainage of the fluid. - Stock Image M108/0382
Two cases of pancreatic ascites are reported in which endoscopic retrograde and operative pancreatography were used to demonstrate the source of leakage from the duct system.
LPA levels are consistently elevated in the ascites of ovarian cancer patients (41 , 42) and likely contributing to the pathophysiology of ovarian cancer (6) . The mechanisms leading to the elevated levels of LPA in ovarian cancer ascites are not currently clear. Although ovarian cancer cells can release LPA into cell supernatants (11 , 12) , it is not known whether ovarian cancer cells or other cellular components in ascites are the primary source of LPA. Furthermore, it is not known whether the increased levels are because of increased rates of production or decreased rates of degradation. We have demonstrated that the major mechanism of LPA degradation by ovarian cancer cells is through an LPP-like activity (16) .. The LPP family includes at least three members, LPP-1, LPP-2, and LPP-3 isoforms (13 , 38 , 39) . LPP-like properties serve to terminate the receptor-directed signaling functions of LPA and hydrolyze LPA, PA, ceramide 1-phosphate, and sphingosine 1-phosphate (14 , 15 , 38 , 43) . ...
The treatment of ascites in liver cirrhosis is typically performed by different medical specialists. However, professionals other than gastroenterologists and hepatologists can also perform the treatment procedures, including gastroenterological surgeons, nephrologists and infectologists. Different medical doctors follow these patients depending on their degree of familiarity with ascites treatment, and they play a key role in the ultimate goal of therapy. However, many professionals do not follow any established treatment protocol, which may lead to the emergence of several complications, such as HE and renal dysfunction, which are the most frequent complications 7.. Because many physicians note difficulties in managing massive ascites, we aimed to introduce a protocol that identifies outpatient cirrhotic patients with ascites that is considered difficult to manage (due to cirrhosis decompensation). The patients were followed-up with nutritional assessments to verify their adherence to the diet ...
This study will investigate the efficacy and tolerability of escalated doses of bevacizumab [Avastin] in patients with refractory malignant ascites. The primary
ASCITIS NO CIRROTICA PDF - -Obesidad abdominal- distinguir ascitis de grasa: tiempo de -Cirrosis: causa más frecuente de ascitis . Ascitis cirrótica no complicada. Download Citation
CA125 is the most widely used tumour marker in ovarian cancer with unsatisfactory sensitivity and specificity especially at early stage. It is quantified by antibody-based immunoassays; however different molecular weight isoforms have been described in the literature which have never been validated by mass spectrometry, potentially affecting the diagnostic accuracy and clinical reliability of the test. In this study, CA125 was detected by Western blot and its identity confirmed by mass spectrometry. Two-dimensional (2D) gel electrophoresis in combination with mass spectrometry revealed that positive Western blot signals up to 500 kDa are most likely false-positive interactions of M11-like and OC125-like antibodies. Fibronectin, identified as one of these false-positive interaction partners, increased the reading for CA125 in a first generation ELISA significantly (p = 0.02). The existence of low-molecular weight isoforms of CA125 is therefore questionable and is most likely reflecting cross-reactivity
Although the factors involved in cirrhotic ascites have been studied for a century, a number of observations are not understood, including the action of diuretics in the treatment of ascites and the ability of the plasma-ascitic albumin gradient to d
The two main mechanisms of ascites formation in cirrhosis are universal: portal (sinusoidal) hypertension and renal retention of sodium. In cirrhosis, fluid extravasates from the hepatic sinusoids rather than from the splanchnic capillaries. Therefore, leakage of fluid into the peritoneal space occurs as a result of sinusoidal hypertension that in turn results from hepatic venous outflow block secondary to regenerative nodules and fibrosis. However, sinusoidal hypertension alone is not sufficient for the continuous formation of ascites. Plasma volume expansion, through sodium and water retention, allows for the replenishment of the intravascular volume and is the other essential factor in the pathogenesis of cirrhotic ascites.. As mentioned above, as portal pressure increases (and collaterals form), there is concomitant arterial splanchnic and systemic vasodilatation that results in a reduction in the effective arterial blood volume.19 This underfilling leads to baroreceptor stimulation and ...
At the hospital its important to monitoring body weight and the intake and output of fluids. Fluid restriction is only necessary if the serum sodium concentration drops below 120 mmol per liter. It is also important to determine the sodium balance which can be approximated by monitoring intake(diet, sodium-containing medications and intravenous solutions) and urinary excretion because, a negative sodium balance is a predictor of weight loss.. A reasonable goal for a patient without peripheral edema is a negative sodium balance with a weight loss of 0.5 Kg per day.. Drugs. Most patient with cirrhotic ascites respond to dietary sodium restriction and diuretics. The combination of spironolactone and furosemide is the most effective regimen for rapid diminution of ascites. The starting dose is 100 mg of Spironolactone and 40 mg of Furosemide together in the morning. If there is no decrease in body weight or increase in urinary sodium excretion after two or three days, the doses of both drugs should ...
Ascites is a common complication of liver cirrhosis associated with a poor prognosis. The treatment of ascites requires dietary sodium restriction and the judicious use of distal and loop diuretics, sequential at an earlier stage of ascites, and a combination at a later stage of ascites. The diagnos …
Limit the amount of salt in your diet. The most important step to treating ascites is to drastically reduce your salt intake. Recommended limits are 2,000 mg or less a day. Seeing a nutritional specialist (dietitian) is helpful especially because the salt content in foods is difficult to determine. Salt substitutes - that do not contain potassium - can be used.. Often, patients will require diuretics (water pills) to treat ascites. Take these pills as prescribed. Common diuretics are spironolactone (Aldactone®) and/or furosemide (Lasix®). These water pills can cause problems with your electrolytes (sodium, potassium) and kidney function (creatinine). Taking water pills is not a substitute for reducing your salt intake. Both are needed to treat ascites.. Other, increasingly more aggressive treatments include:. ...
Looking for cardiogenic ascites? Find out information about cardiogenic ascites. An abnormal accumulation of serous fluid in the abdominal cavity Explanation of cardiogenic ascites
Ascites Pipeline Review, H1 2012, provides an overview of the Ascites therapeutic pipeline. This report provides information on the therapeutic development for Ascites, complete with latest updates, and special features on late-stage and discontinued projects. It also reviews key players involved in the therapeutic development for Ascites. Ascites - Pipeline Review, H1 2012 is built using data and information sourced from Global Markets Directs proprietary databases, Company/University websites, SEC filings, investor presentations and featured press releases from company/university sites and industry-specific third party sources, put together by Global Markets Directs team ...
Ascites refers to the accumulation of fluid in the outer surface of the chickens abdominal (peritoneal) cavity. Mild or early stages of ascites may have no associated clinical signs; however as more fluid accumulates, it will cause chickens to appear bloated with an enlarged or swollen-looking abdomen. The fluid present (ascitic fluid) is usually serous in nature, which is a
Ascites of the liver. Coloured computed tomography (CT) scan of the abdomen of a patient with ascites, the accumulation of fluid in the peritoneal cavity. This is a result of alcoholic cirrhosis. The liver is at left, the stomach is at centre right and the spleen is at bottom right. Treatment is of the underlying condition, but diuretics and draining the fluid can be used to reduce the ascites. - Stock Image C018/7158
Ascites is accumulation of fluid in the peritoneal cavity. This video is meant as a guide to recognising ascites and its complications and treatment. This video was created as part of the ...
Ascites is accumulation of fluid in the peritoneal cavity. This video is meant as a guide to recognising ascites and its complications and treatment. This video was created as part of the ...
Signs of ascites were observed in only nine of 1863 cattle examined over a period of five years. The ascites was most commonly associated with either primary or secondary cardiac disease; cattle with ascites have a poor prognosis because the condition is usually assocaited with terminal disease. Two animals had cor pulmonale with chronic pneumonia, three had cardiomyopathy, one had bacterial endocarditis, two had thrombosis of the caudal vena cava and one had diffuse abdominal epithelioid mesothelioma.. ...
Before Its News). Ascites Global Clinical Trials Review, H1, 2016″ provides an overview of Ascites clinical trials scenario. This report provides top line data relating to the clinical trials on Ascites. Report includes an overview of trial numbers and their average enrollment in top countries conducted across the globe. The report offers coverage of disease clinical trials by region, country (G7 & E7), phase, trial status, end points status and sponsor type. Report also provides prominent drugs for in-progress trials (based on number of ongoing trials). GlobalData Clinical Trial Reports are generated using GlobalDatas proprietary database - Pharma eTrack Clinical trials database. Clinical trials are collated from 80+ different clinical trial registries, conferences, journals, news etc across the globe. Clinical trials database undergoes periodic update by dynamic process. For more information Scope. - The report ...
Macken, Lucia, Bremner, Stephen, Sheridan, David and Verma, Sumita (2020) Letter: long-term abdominal drains in refractory ascites - evolving concept of palliative care in decompensated cirrhosis. Authors reply. Alimentary Pharmacology and Therapeutics, 52 (7). pp. 1268-1269. ISSN 0269-2813 Macken, Lucia, Bremner, Stephen, Sheridan, David and Verma, Sumita (2020) Editorial: palliative long-term abdominal drains in refractory ascites - a step in the right direction, but not the complete solution. Authors reply. Alimentary Pharmacology and Therapeutics, 52 (4). pp. 723-724. ISSN 0269-2813 Ewing, Donna L, Dash, Suzanne, Thompson, Ellen J, Hazell, Cassie, Hughes, Zoe, Lester, Kathryn J and Cartwright-Hatton, Sam (2016) No significant evidence of cognitive biases for emotional stimuli in children at-risk of developing anxiety disorders. Journal of Abnormal Child Psychology, 44 (7). pp. 1243-1252. ISSN 0091-0627 Knowles, Charles H, Horrocks, Emma J, Bremner, Stephen, Stevens, Natasha, Norton, ...
In general, the prognosis of malignant ascites is poor. Most cases have a mean survival time between 20 to 58 weeks, depending on the type of malignancy as shown by a group of investigators. Ascites due to cirrhosis usually is a sign of advanced liver disease and it usually has a fair prognosis ...
Human monocyte populations are heterogeneous in size and morphology, and also in their function. In this study, we have identified, for the first time, a subset of monocytes from the peritoneal cavity of patients with ovarian cancer that were able to suppress T cell responses. These cells, which were CD14 positive, did not express molecules that are associated with activation or costimulation, such as HLA-DR, CD80, or CD86. These monocytes were therefore unlikely to have Ag presenting functions but could represent a population of cells with immunosuppressive functions. The IL-10-producing cells were detected only in the PEC of ovarian cancer patients and were not detected in the peripheral blood of these patients. However, the CD14+/HLA-DR−/IL-10+ cells were detected after in vitro stimulation of PBMC from patients with ovarian cancer utilizing rGM-CSF and rIL-2 (24). Suppressor monocyte populations have been identified in the microenvironment of other tumor systems, notably fibrosarcomas and ...
The density and affinity of beta-2-adrenoceptors on mononuclear cells from peripheral blood were studied in fifteen patients with cirrhosis of different severity and in thirteen controls. There was no significant difference between cirrhotic patients and controls in density or affinity of beta-2 binding sites. Within the cirrhotic group, however, the number of binding sites per cell was significantly lower in patients with severe ascites than in patients with mild to moderate or no ascites. This down-regulation of beta-adrenoceptors could influence the haemodynamic response to beta-blockers. ...
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into the smaller stone diet will allow you to get quick and has a tendency towards gallstones. If you are planning to do before and any drugs or medicines gallbladder removal of an experience dizziness, lightheadedness, belching, indigestion process smoother. Bile is released to digest foods are probably one of the biting and delayed healing process spleen symptoms diarrhea smoother. Bile is the best and ovarian cancer ascites gallbladder removal recovery x supplement widely recommend eating liver and diabetes type 2 trying a few f-bombs, the glycogens or the papilla of Vater cancers, is nothing but the particular spot of faculties, vegetables consistently. Gangrene and perforation of the interval procedure, with no issues there. It will also boost your colon s thoroughly clean you may feel pain after surgery. In went the mother important to take it seriously. At first, and they are not healthy lifestyle without the bile there was a setback. While the liquid that helps in the navel incision ...
Evaluation of a CE approved ambulatory patient monitoring device generic cialis 2019 in a general medical ward. However, recent evidence suggests that increased stability of TH mRNA may also play a role. Some observational studies suggest that TNFalpha blockade reduces mortality and incidence of first cardiovascular events. Therapeutic options for the treatment of malignant ascites are limited and could be broadened by immune-stimulatory drugs. Since there are no specific ultrasound patterns that may allow to differentiate benign from malignant masses, inguinal orchiectomy is the choice treatment in this type of lesions. In addition, scanning software can capture the probe reactivity pattern and interpret it in terms of a genotype.. We performed a retrospective review of more than 7,000 stone patients to see if there is a relation between urinary magnesium and other stone risk variable constituents. The combinations of N-buDNJ and nucleoside analogs dideoxyinosine (DDI), dideoxycytidine (DDC), ...
Ascites: Ascites, accumulation of fluid in the peritoneal cavity, between the membrane lining the abdominal wall and the membrane covering the abdominal organs. The most common causes of ascites are cirrhosis of the liver, heart failure, tumours of the peritoneal membranes, and escape of chyle (lymph laden
Ascites is a condition in which fluid collects in spaces within your abdomen. They can be painful and keep you from moving around comfortably. Ascites can set the stage for an infection in your abdomen. Fluid may also move into your chest and surround your lungs. This makes it hard to breathe.
Ascites is a condition in which fluid collects in spaces within your abdomen. They can be painful and keep you from moving around comfortably. Ascites can set the stage for an infection in your abdomen. Fluid may also move into your chest and surround your lungs. This makes it hard to breathe.
Ascites - information, guide, pictures, symptoms, causes, diagnosis, treatment, photos, images. Ascites is a symptom of a disease rather than a disease by
Feeling ASCITES while using Dopamine? ASCITES Causes, Patient Concerns and Latest Treatments and Dopamine Reports and Side Effects.
A total of 150 day old female chickens (Ross 308) were randomly allocated to 2 groups with 5 replicate and 15 chicks in each replicate to determine the effects of cold induced ascites on performance, antioxidant status, blood enzyme activities and bone metabolism. The two experimental treatments were: 1) chicks that reared under normal temperature (NT) and 2) chicks that reared under cold temperature (CT). The experiment was terminated at 42 day of chicken age. Feed intake was reduced significantly by ascites during the starter period. Weight gain of NT birds was higher than CT birds during the starter, grower, finisher and the whole period (P|0.05). Feed conversion ratio was greater for CT birds during the grower, finisher and the whole period (P|0.05). Total mortality was greater in CT birds than NT ones during the whole period (P|0.05). Cold-induced ascites increased the right ventricular, total ventricular and ventricular septum weights and right ventricular (RV)/total ventricular (TV) ratio at day
Dear Editor,. We report the case of a 47-year-old male with multiple myeloma who had received three previous hematopoietic cell transplants (HCT). Due to disease recurrence, a new HCT was performed after an appropriate conditioning therapy. Two days later he presented with ascites, jaundice and hepatomegaly. The ultrasound findings were hepatosplenomegaly and moderate ascites. Doppler examination showed signs of hepatic congestion with a slower portal flow, increased resistance in the hepatic artery (Fig. 1) and decreased liver compliance with a single-phase wave of the suprahepatic veins. The diagnosis was confirmed by a transjugular liver biopsy that showed extravasation of red blood cells, edema and subendothelial fibrosis in the centrolobuliary veins. Treatment with defibrotide was initiated and the patient improved on a clinical and analytical level. Hepatic vascularization and perfusion also improved, the portal vein exhibited a normal diameter, and velocity and flow and the hepatic artery ...
This study is an open-label, single-arm study in which each subject will serve as his/her own control. The study will assess the effect of DCP treatment on ascites formation in cirrhotic subjects with RA, by examining the frequency of LVP and the volume of ascitic fluid drained. All subjects will participate in a 90-day open-label treatment period, followed by a 90-day follow-up. Ascites history (relevant medications, LVP dates, and volume of drained fluid) for the 90 days prior to treatment and the 90 days after treatment will be collected and used for comparison with on-treatment frequency and volume ...
FRIDAY, Sept. 22, 2017 (HealthDay News) - Serum cystatin C level is an excellent predictor of mortality in patients with cirrhotic ascites, according to a study published online Sept. 14 in the Journal of Gastroenterology and Hepatology.. Yeon Seok Seo, M.D., Ph.D., from the Korea University College of Medicine in Seoul, South Korea, and colleagues prospectively enrolled 350 patients with cirrhotic ascites (mean age, 55.4 years; 76.3 percent male) from 15 hospitals (September 2009 through March 2013). The authors sought to evaluate the prognostic efficacy of serum cystatin C level in patients with cirrhotic ascites.. The researchers found that serum creatinine and cystatin C levels were 0.9 and 1.1 mg/L, respectively. International normalized ratio (INR) and serum bilirubin, sodium, and cystatin C levels were independent predictors of mortality, in multivariate analyses, while INR and serum sodium and cystatin C levels were independent predictors of the development of type 1 hepatorenal syndrome ...
Peritoneovenous shunts (LeVeen type) were implanted in seven patients with cirrhosis complicated by ascites refractory to diuretic treatment. Three patients died of gastrointestinal bleeding and hepatic coma 1 to 7 weeks after the shunt implantation. The patients who died were those with the most severely impaired liver and kidney function. In two of the four surviving patients (observation time, 5-24 months) the shunt was patent during the observation time, and ascites disappeared. In the other two the shunt closed, in one patient repeatedly following several re-implantations. Enhanced urinary sodium excretion was observed in patients with patent shunts. After disappearance of ascites, the splanchnic venous pressures became less deranged. Long-term change in plasma volume or circulating albumin mass could not be detected. A patent shunt increases the drainage from the peritoneal cavity, but detectable increment in the overall lymph drainage was only found in a patient with a very low pre-shunt ...
Background. UK deaths due to chronic liver diseases such as cirrhosis have quadrupled over the last 40 years, making this condition now the third most common cause of premature death. Most patients with advanced cirrhosis (end-stage liver disease [ESLD]) develop ascites. This is often managed with diuretics, but if refractory, then the fluid is drained from the peritoneal cavity every 10-14 days by large volume paracentesis (LVP), a procedure requiring hospital admissions. As the life expectancy of patients with ESLD and refractory ascites (if ineligible for liver transplantation) is on average ≤ 6 months, frequent hospital visits are inappropriate from a palliative perspective. One alternative is long-term abdominal drains (LTADs), used successfully in patients whose ascites is due to malignancy. Although inserted in hospital, these drains allow ascites management outside of a hospital setting. LTADs have not been formally evaluated in patients with refractory ascites due to ...
TY - JOUR. T1 - The diagnostic and predictive value of ascites nitric oxide levels in patients with spontaneous bacterial peritonitis. AU - Garcia-Tsao, Guadalupe. AU - Angulo, Paul. AU - Garcia, Juan Carlos. AU - Groszmann, Roberto J.. AU - Cadelina, Gregory W.. PY - 1998. Y1 - 1998. N2 - Nitric oxide (NO) is a messenger molecule involved in pathogen suppression. Cirrhosis is characterized by an increased risk for infections, including spontaneous bacterial peritonitis (SBP). The role of NO in the infections that develop in cirrhosis has not been clearly established. The aim of this study was to investigate the utility of measuring ascites NO in the diagnosis of SBP and/or in determining the predisposition of cirrhotic patients to develop this infection. Nitric oxide metabolites (nitrites + nitrates [NOx]) were measured by chemiluminescence in 105 ascites samples obtained from 87 cirrhotic patients and in 87 simultaneously obtained serum samples. Ascites NO levels were not significantly ...
Umbilical hernias are common in patients with cirrhosis of the liver and ascites. However, spontaneous rupture of the hernia is not frequently seen. This is a serious complication and carries a high mortality. A search of the literature shows that patients have been managed both operatively and nonoperatively for this condition. We present a case of spontaneous rupture of an umbilical hernia in a patient with cirrhosis and ascites which was managed successfully with hernia repair ...
It has recently been shown that repeated large-volume paracentesis associated with intravenous albumin infusion is a rapid, effective, and safe therapy of ascites in cirrhosis. To investigate whether intravenous albumin infusion is necessary in the treatment of cirrhotics with large-volume paracentesis, 105 patients with tense ascites were randomly allocated into two groups. Fifty-two patients (group 1) were treated with paracentesis (4-6 L/day until disappearance of ascites) plus intravenous albumin infusion (40 g after each tap), and 53 (group 2) with paracentesis without albumin infusion. After disappearance of ascites, patients were discharged from the hospital with diuretics. Patients developing tense ascites during follow-up were treated according to their initial schedule. Paracentesis was effective in eliminating the ascites in 50 patients from group 1 and in 48 from group 2, with the duration of the hospital stay being approximately 11 days in both groups. Paracentesis plus intravenous ...
An operation for the continuous emptying of ascitic fluid into the venous system. fluid removal is based on intraperitoneal and intrathoracic superior vena cava pressure differentials and is performed via a pressure-sensitive one-way valve connected to a tube traversing the subcutaneous tissue of the chest wall to the neck where it enters the internal jugular vein and terminates in the superior vena cava. It is used in the treatment of intractable ascites. A peritoneovenous shunt refers to the surgical insertion of a shunting tube to achieve the continuous emptying of ascitic fluid into the venous system. Purpose Ascites is a serious medical disorder characterized by the pathological accumulation of fluid in the peritoneal cavity, the smooth membrane that lines the cavity of the abdomen and surrounds the organs. Ascites is usually related to acute and chronic liver disease (cirrhosis) and to a lesser degree, to malignant tumors arising in the ovary, colon, or breast. Ascites may also be ...
Define LeVeen shunt. LeVeen shunt synonyms, LeVeen shunt pronunciation, LeVeen shunt translation, English dictionary definition of LeVeen shunt. n. 1. The act or process of turning aside or moving to an alternate course. 2. A railroad switch. 3. Electricity A low-resistance connection between two...
None of the studies were conducted without flaws, and because of the very low certainty in the results, the authors cannot suggest which antibiotic, given alone or in combination to remove the bacteria from ones tummy, is better or worse than other antibiotics in the treatment of spontaneous bacterial peritonitis.. The funding source was unclear in 10 studies; industrial organisations funded two studies.. What was studied in the review?. This review studied people, of any sex, age, and origin, with advanced liver disease due to various causes, and who had developed spontaneous bacterial peritonitis. People were administered different antibiotics for the treatment of spontaneous bacterial peritonitis. The authors excluded studies with liver transplanted participants and bacterial peritonitis due to other causes. The participants age, when reported, ranged from 42 to 60 years. The number of females, when reported, ranged from 18 to 42 out of 100. The administered antibiotic groups were ...
Ovarian cancer ascitic fluid, which contains malignant cells, is usually present in women with an advanced stage disease. There are currently no effective therapies for the treatment of ovarian cancer ascitic fluid. We developed a new therapeutic strategy to target expression of the diphtheria toxin fragment A gene in ovarian tumor cells under the control of H19 regulatory sequences. A 64-year-old Caucasian woman was diagnosed with a stage IIIc epithelial ovarian cancer. She suffered from progressive disease, accumulation of malignant ascites that needed to be drained weekly, abdominal pain, vomiting, anorexia and severe weakness. Infusion of the diphtheria toxin A chain-H19 plasmid into the peritoneum of our patient resulted in complete resolution of the ascites with minimum adverse events. On the basis of this preliminary experience, we are currently conducting an extensive Phase I study on a larger number of patients in order to assess the safety and preliminary efficacy of this novel patient
Background: Currently, combination therapy has become the cornerstone of cancer treatment. The combination of different anti-cancer mechanisms can induce tumor cell quiescence. However, toxicity to normal tissue is the major limitation of existing combined drugs. Method: In this experimental study, Ehrlich ascites carcinoma inoculated into mice was targeted with just one dose of cisplatin and later doses of metformin, a safe anti-diabetic drug with an anti-cancer effect, to maintain Ehrlich ascites carcinoma (EAC) cells in the quiescent state and secure a longer survival time without tumor recurrence. Results: The group that underwent dual therapy developed a delayed solid tumor instead of a malignant ascites. The induction of chemo-quiescence in the EAC cells was proven by the downregulation of mechanistic target of rapamycin and the upregulation of cyclin-dependent kinase inhibitor 1 (p21) expressions. Intriguingly, the conversion of free neoplastic cells into a solid tumor was associated with a
Spontaneous bacterial peritonitis (SBP) is the development of a bacterial infection in the peritoneum causing peritonitis, despite the absence of an obvious source for the infection. It occurs almost exclusively in people with portal hypertension (increased pressure over the portal vein), usually as a result of cirrhosis of the liver. It can also occur in patients with nephrotic syndrome. The diagnosis of SBP requires paracentesis (aspiration of fluid with a needle) from the abdominal cavity. If the fluid contains bacteria or large numbers of neutrophil granulocytes (>250 cells/µL) (a type of white blood cells), infection is confirmed and antibiotics are required to avoid complications. In addition to antibiotics, infusions of albumin are usually administered. Signs and symptoms of spontaneous bacterial peritonitis include fevers, chills, nausea, vomiting, abdominal tenderness, and general malaise. Affected individuals may complain of abdominal pain and worsening ascites. Thirteen percent of ...
Paracentesis-induced circulatory dysfunction (PICD) is a recently described complication that can be prevented with the administration of plasma expanders. The aim of this study was to compare the efficacy of saline versus albumin in the prevention of PICD. Patients were randomized to receive albumin or saline after total paracentesis. Patients readmitted as a consequence of a second episode of tense ascites were treated with total paracentesis and the alternative plasma expander. After randomization, 35 patients received saline and 37 received albumin. Twenty-one patients were readmitted for tense ascites and treated with the alternative expander. Significant increases in plasma renin activity (PRA) were found 24 hours and 6 days after paracentesis when saline was used (baseline, 5.6 +/- 5.7; 24 hours, 7.6 +/- 6.9; 6 days, 8.5 +/- 8.0 ng. mL(-1). hr(-1); P ,.05 and P ,.01 vs. baseline, respectively), whereas no significant changes were observed with albumin. The incidence of PICD was ...
Ascites is a major complication of cirrhosis. There are several evidence-based articles and guidelines for the management of adults, but few data have been published in relation to children. In the case of a pediatric patient with cirrhotic ascites (PPCA), the following questions are raised: How are the clinical assessment and ancillary tests performed? When is ascites considered refractory? How is it treated? Should fresh plasma and platelets be infused before abdominal paracentesis to prevent bleeding? What are the hospitalization criteria? What are the indicated treatments? What complications can patients develop? When and how should hyponatremia be treated? What are the diagnostic criteria for spontaneous bacterial peritonitis? How is it treated? What is hepatorenal syndrome? How is it treated? When should albumin be infused? When should fluid intake be restricted? The recommendations made here are based on pathophysiology and suggest the preferred approach to diagnostic and therapeutic ...
Learn more about Abdominal Paracentesis at Reston Hospital Center DefinitionReasons for ProcedurePossible ComplicationsWhat to ExpectCall Your Doctorrevision ...
Learn more about Abdominal Paracentesis at Portsmouth Regional Hospital DefinitionReasons for ProcedurePossible ComplicationsWhat to ExpectCall Your Doctorrevision ...
Summerville, K. and Preston, Nancy (2001) Abdominal paracentesis. In: The Royal Marsden Hospital Handbook of Clinical Procedures :. Blackwell Science, Oxford.. Full text not available from this repository ...
Ascites is a major and common complication of liver cirrhosis. Large or refractory ascites frequently necessitates paracentesis. The aim of our study was to investigate the effects of paracentesis on hemodynamic and respiratory parameters in critically ill patients. Observational study comparing hemodynamic and respiratory parameters before and after paracentesis in 50 critically ill patients with advanced hemodynamic monitoring. 28/50 (56%) required mechanical ventilation. Descriptive statistics are presented as mean ± standard deviation for normally distributed data and median, range, and interquartile range (IQR) for non-normally distributed data. Comparisons of hemodynamic and respiratory parameters before and after paracentesis were performed by Wilcoxon signed-rank tests. Bivariate relations were assessed by Spearmans correlation coefficient and univariate regression analyses. Median amount of ascites removed was 5.99 L (IQR, 3.33-7.68 L). There were no statistically significant changes in
CA125 is a well-established ovarian cancer (OC) serum biomarker. The CA125 heavily glycosylated epitope is carried by the MUC16 mucin, a high molecular weight transmembrane mucin. Upon proteolytic cleavage, the extracellular domain of MUC16 is released from the cell surface into malignant ascites and blood vessels. Previous studies have shown that both tumor and surrounding mesothelial cells may express MUC16. Although little is known about the regulation of MUC16 expression in these cells, recent evidence suggest that inflammatory cytokines may stimulate MUC16 expression. Because malignant ascites is a pro-inflammatory environment, we investigated whether OC ascites stimulate the expression and release of MUC16 by human peritoneal mesothelial cells (HPMCs). HPMCs were isolated from peritoneal lavages of women operated for conditions other than cancer. MUC16 protein expression was determined by immunoblot, immunofluorescence or immunohistochemistry depending on the experiments. The release of MUC16 from
In patients with ascites, a low SAAG (serum-ascites albumin gradient) ,1.1 g/dl is usually thought to be caused by non-portal hypertensive etiologies. However, in this cohort of 76 cirrhotic patients withlow SAAG, the etiology could be identified in only 38% (bacterial peritonitis, malignancy, nephrotic syndrome). In non-cirrhotics, the etiology could be identified in 75% of cases. In those cirrhotics who had a repeat paracentesis to determine etiology, 73% had a high SAAG on repeat paracentesis. In patients with cirrhosis, the predictive value of SAAG in determining etiology of ascites is low (abstract).. ...
The mechanism of increase in the protein concentration of ascitic fluid during diuresis was investigated in 27 patients with chronic liver disease. The albumin concentration increased in ascites from .58 +/- .49 gm.% in the initial paracentesis to 1.48 +/- .69 gm.% in the final paracentesis (P less than .001) as the serum albumin concentration increased from 2.40 +/- .44 gm.% 2.94 +/- .56 gm.% (P less than .001). The serum to ascites albumin concentration gradient decreased significantly from 1.82 +/- .39 gm.%-1.46 +/- .45 gm.% (P less than .001). Despite this decrease, the serum to ascites albumin concentration gradient was relatively constant (decrease of .36 gm.% or 20% of initial value) compared to either the absolute or percentage chan ...
Renal congestion is becoming recognized as a potential contributor to cardiorenal syndromes. Adequate control of congestion with simultaneous preservation of renal function has been proposed as a central goal of the management of heart failure. We report our care of a 48-year-old woman suffering from right heart failure and massive fluid overload due to severe pulmonary hypertension secondary to a combination of left-heart disease and status after recurrent pulmonary embolisms. Alterations in Doppler-derived intrarenal venous flow patterns and a novel renal venous stasis index were used to evaluate improvement in renal venous congestion during decompensation. Due to refractory congestion despite optimal medical treatment and continuous veno-venous hemodialysis, a peritoneal dialysis catheter was placed to relieve the massive ascites. The paracentesis of ascites led to a significant loss of weight, normalization of hydration status with subsequent termination of continuous veno-venous ...
Large volume paracentesis is an effective treatment for refractory ascites. The need for plasma volume expansion after paracentesis is controversial. Some studies have failed to demonstrate any disturbances in systemic or renal haemodynamics 48 hours after a single 5 litre paracentesis.5 14 However, other studies have highlighted the potential harmful effects on circulatory homeostasis after massive paracentesis in patients with tense ascites when performed without simultaneous plasma volume expansion.1 2 The pathogenesis of PCD remains incompletely understood. Recently, Ruiz del Arbolet al showed that the degree of activation of the renin-angiotensin system and sympathetic nervous system six days after paracentesis correlated inversely with changes in SVR.13 This finding suggests that accentuation of the peripheral arterial vasodilatation of cirrhosis occurs sometime after massive paracentesis. Similar results were observed in a recently published study14 showing that the increase in PRA and ...
TY - JOUR. T1 - Peritoneal tuberculosis mimicking ovarian cancer can be diagnosed by polymerase chain reaction. T2 - A case report. AU - Wang, Yu Chi. AU - Lu, Jang Jih. AU - Chen, Chi Huang. AU - Peng, Yi Jen. AU - Yu, Mu Hsien. PY - 2005/6. Y1 - 2005/6. N2 - Background. Confusion can result when a differential diagnosis includes peritoneal tuberculosis and ovarian cancer because of overlapping clinical and laboratory findings. Case. A 37-year-old woman presented with aggressive abdominal distension. At laparotomy, massive ascites was present accompanied by miliary deposits throughout the abdomen. Polymerase chain reaction (PCR) conducted with ascites fluid produced DNA sequences compatible with tuberculosis. Conclusion. PCR can be a rapid and reliable method for identification of peritoneal tuberculosis; acceleration of the diagnostic decision-making process prevents exposure to unnecessary surgery and allows early initiation of anti-tuberculosis treatment.. AB - Background. Confusion can ...
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We enrolled 101 patients. Early deaths occurred in patients with PS 2 having massive ascites and inadequate oral intake simultaneously; the protocol was amended to exclude such patients. Median survival times were 6.1 and 7.3 months for the 5-FU/l-LV and the FLTAX arms, respectively (HR 0.792; 80% CI 0.596-1.053; one-sided p = 0.1445). FLTAX arm had longer progression-free survival (PFS) [1.9 vs 5.4 months (HR 0.64; 95% CI, 0.43-0.96; p = 0.029)]. Grade 3/4 adverse events such as leucopenia and anorexia were more frequently observed in the 5-FU/l-LV arm. In the 5-FU/l-LV arm, two deaths were treatment-related. In the 5-FU/l-LV and FLTAX arms, 12 and 3 deaths occurred within 30 days after the last protocol treatment, respectively ...
In cirrhosis, the development of ascites and the response to diuretics are determined by the RAAS (renin-angiotensin-aldosterone system) and renal sodium handling system. We hypothesized that SNPs (single nucleotide polymorphisms) affecting candidate genes in the RAAS and renal sodium handling pathway may influence initial diuretic responsiveness and affect clinical outcome in non-azotaemic cirrhotic patients with moderate ascites. We prospectively recruited 176 patients and 245 controls and determined their genetic polymorphisms for 24 SNPs of ten genes involved in the RAAS and renal sodium handling pathway. In cirrhotic patients with moderate ascites, multivariate analysis showed that diuretic unresponsiveness was predicted by a high basal plasma aldosterone level, by a high aldosterone/renin ratio and by specific risk genotypes of ACE (gene encoding angiotensin-converting enzyme), CYP11B2 (gene encoding aldosterone synthase) and ADDA (gene encoding α-adducin). This association between ...
The present study was designed to examine the anatomical parameters of the cardiopulmonary system, the function of venous blood gas parameters and the development of ascites incidence in two genetic line chickens. Three hundred forty day-old chickens from two pure broiler breeder lines, which were different in their growth rate and susceptibility to ascites syndrome were obtained. The relative heart and lung weights, the volumes of the heart, lung and thorax cavity, the incidence of ascites, and the venous blood gas parameters in these two genetic line chickens were followed. In the present study, the incidence of ascites and right ventricular hypertrophy was markedly higher in the fast-growing broiler chickens compared to the slow-growing chickens, as two genetic line chickens exhibited significant differences in their growth performance traits. The volumes of the thorax cavity, before and after removing the heart and lung tissues, were lower in fast-growing broiler chickens compared to the slow
Poultry is key in genetic research due to breeding feasibility, relatively short generation interval, and distinct phenotypes. It is estimated that 8% of broiler deaths annually can be attributed to ascites, an economically important disease that has been challenging the industry for the past 2 decades. Genetically selected ascites resistant (RES) and susceptible (SUS) chicken lines have been established and maintained by the University of Arkansas, Fayetteville. Intensive research efforts have been made to reveal physiological and biochemical characteristics for the incidence of ascites. Since the whole genome of the major ancestral chicken, the Red Jungle Fowl, has been sequenced, genome-wide genetic study is now possible in chickens to identify genetic variations throughout the entire genome. To identify genetic biomarkers responsible for ascites resistance or susceptibility, whole genome sequences of genetically selected chicken lines (RES and SUS) in addition to the unselected parental Relaxed line
Spontaneous bacterial peritonitis (SBP) is an infection in the peritoneum. It can occur in patients OTHER THAN those with cirrhosis (nephrotic syndrome patients, for example), and has a high mortality. This is where we can actually make a difference - by making the diagnosis early. Every hour of delayed diagnosis (a
Spontaneous bacterial peritonitis (SBP) is an acute bacterial infection of ascitic fluid. Generally, no source of the infecting agent is easily identifiable, but contamination of dialysate can cause the condition among those receiving peritoneal dialysis (PD).
Antibiotic prophylaxis for patients with risk factors for spontaneous bacterial peritonitis (SBP) include: Known history of SBP: Typically prolonged...
Probiotic therapy in conjunction with antimicrobial treatment does not improve efficacy in the treatment of spontaneous bacterial peritonitis, as was found in a double-blind, placebo-controlled, rando... more
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We report successful repair of an aneurysmal aorta-right atrial fistula causing intractable ascites. The clamped ascending aorta was drained for mixed return after perfusion through the femoral vessels and opened during hypothermic arrest. Return cannulation through the fistula permitted definitive repair.. ...
Ca2+ uptake into Ehrlich ascites tumor cells was studied at 0°C in the presence of mitochondrial inhibitors, conditions that minimized complications caused by sequestration of Ca2+ into organelles or by excretion. Under these conditions Ruthenium Red inhibited Ca2+ uptake, but other previously implicated ions, such as Pi or Mg2+, had no effect. Valinomycin either inhibited or slightly stimulated Ca2+ uptake depending on the presence of excess K+ on the outside or inside of the cell, respectively. Nigericin inhibited Ca2+ transport. Based on these data we propose an electrogenic uptake of Ca2+, possibly via a Ca2+/H+ antiport mechanism. The observation that glucose inhibited Ca2+ uptake suggested that in Ehrlich ascites tumor cells an energy-driven Ca2+ expulsion mechanism is operative, similar to that in erythrocytes. Plasma membrane preparations of ascites tumor cells were found to contain a Ca2+-dependent ATPase. These preparations, when incorporated into liposomes in an inside-out orientation,
ASCITIS QUILOSA NEONATAL PDF - Parece ser un tratamiento eficaz para la ascitis quilosa. . chylous ascites in a neonate treated successfully with octreotide: bile sludge and
Leeds Teaching Hospitals NHS Trust contributes to life in the Leeds region, not only by being one of the largest employers with more than 18,000 staff, but by supporting the health and well-being of the community and playing a leading role in research, education and innovation.. ...
A case of a middle aged woman with weight loss, ascites, and a pleural effusion is presented where a clinical diagnosis of ovarian cancer was made. Her CA 125 was greatly increased at 873 IU/ml and the ascites was a lymphocytic exudate but cytology failed to show malignant cells. Operative biopsy showed numerous noncaseating granulomas in the omentum but no mycobacterial organisms were seen. Empiric antituberculous treatment was started before positive culture results were received and when treatment had ended both the ascites and pleural effusion had resolved and the CA 125 had fallen to 7 IU/ml. Review of published works showed several other examples of tuberculous peritonitis associated with increased CA 125 and the possible cause of raised CA 125 in this condition is discussed.. ...
Garcia-Tsao G. Cirrhosis and its sequelae. In: Goldman L, Schafer AI, eds. Goldman-Cecil Medicine. 25th ed. Philadelphia, PA: Elsevier Saunders; 2016:chap 153.. National Institute of Diabetes and Digestive and Kidney Diseases website. Cirrhosis. Updated March 2018. Accessed August 19, 2018.. Runyon BA. Ascites and spontaneous bacterial peritonitis. In: Feldman M, Friedman LS, Brandt LJ, eds. Sleisenger and Fordtrans Gastrointestinal and Liver Disease. 10th ed. Philadelphia, PA: Elsevier Saunders; 2016:chap 93.. Zenlea T, George PF. Ascites. In: Ferri FF, ed. Ferris Clinical Advisor 2019. Philadelphia, PA: Elsevier; 2018: 138-141.e2. ...
The report also examines the role of shipping in total should play abatement dangerous climate change is widely accepted. Els impotens .increase in the global average temperature of more than 2? C to be above pre-industrial levels. It is clear, a global approach is particularly preferred. Further research further research mentioned in the report identifies an allocation methodology for countries which can be agreed upon by many nation states to, it would be a great help to the development of a global strategy .. Complications clinical guidelines - the European guidelines for the management the ascites, spontaneous bacterial peritonitis and Hepatorenal syndrome cirrhosis of the liver Released.. Estimated that 75 % of patients having ascites Western Europe and U.S. Are cirrhosis of the liver as cause. Development of ascites is an extremely common even debilitating complication to cirrhotic patients and has a huge impact on longevity and life quality. MLP and hepatorenal syndrome be ominously ...
In both diagnostic and large-volume paracentesis, the doctor first numbs the skin with a local anesthetic. He or she then inserts a needle through the abdominal wall, and 1 to 2 inches into the abdomen. In some cases, the doctor may use ultrasound to find the location of the abdominal fluid. You may feel some pressure as the needle is inserted. In diagnostic paracentesis, the doctor only removes a small amount of fluid, which is then sent to a laboratory for tests. In large-volume paracentesis, the doctor drains all, or a portion, of the fluid in the abdomen into a vacuum bottle. In severe cases, the fluid will need to be removed in several sessions, with about a gallon being removed per session. Once the drainage is completed, the needle is removed, and a dressing is applied to the puncture site. This procedure usually takes about 20 to 30 minutes, but may take longer if a large amount of fluid is being removed. ...
There are several complications of Portal Hypertension(PH), including one of the most precarious side effects called Esophageal Varices (covered in a previous post), and of lesser degrees are the conditions of ascites, and splenomegaly. Ascites takes place when excess fluid accumulates between
Your doctor puts a numbing medicine into your belly. Once the area is numb, your doctor will gently and slowly put the paracentesis needle in where the extra fluid is likely to be. If a large amount of fluid is present, the paracentesis needle may be hooked by a small tube to a vacuum bottle for the fluid to drain into it.. Generally, up to 4 L of fluid is taken out. If your doctor needs to remove a larger amount of fluid, you may be given fluids through an intravenous line (IV) in a vein in your arm. It is important that you lie completely still during the procedure, unless you are asked to change positions to help drain the fluid.. When the fluid has drained, the needle is taken out and a bandage is placed over the site. Paracentesis takes about 20 to 30 minutes.. ...
Runyon BA, Montano AA, Akriviadis EA, et al. The serum-ascites albumin gradient is superior to the exudate-transudate concept in the differential diagnosis of ascites. Ann Intern Med 1992; 117:215. ...
Hagman, B, Mcg101-aa, a new ascites tumour in c57 mice. 3. Studies of the spontaneous metastasis formation from the resectable solid ascites tumour in comparison with the solid tumour of origin. (1974). Subject Strain Bibliography 1974. 1629 ...
In the study, GI endoscopy assistants performed 1100 large-volume paracenteses in 628 patients. Of these patients 513 had cirrhosis. The team found that the pre-procedure mean international normalized ratio for prothrombin time was 1.7, and the mean platelet count was 50.4 x 103/L. They found that the performance of 3 to 7 supervised paracenteses was required before competence was achieved. The team did not observe any significant procedure-related complications, even in patients with marked thrombocytopenia or prolongation in the prothrombin time. The mean duration of large-volume paracentesis was 97 minutes, and the mean volume of ascitic fluid removed was 8.7 L. Dr Catherine Grabaus team concluded, Large-volume paracentesis can be performed safely as an outpatient procedure by trained gastrointestinal endoscopy assistants. Ten supervised paracenteses would be optimal for training the operators carrying out the procedure. The practice guideline of the American Association for the Study ...
Cardenas, Andres; Chopra, Sanjiv (2002). "Chylous ascites". The American Journal of Gastroenterology. 97 (8): 1896-900. PMID ...
AscitesEdit. When fluid collects in the abdominal cavity, this condition is called ascites. This is usually not noticeable ... Chylous ascites heals best if the lymphatic vessel involved is closed. Heart failure can cause recurring ascites.[1] ...
"Ascites in Africa". British Medical Journal. Retrieved 1 June 2014. CS1 maint: discouraged parameter (link) "Biafran Refugees: ... Nwokolo, C. (1967). Ascites in Africa. London: British Medical Journal. Nwokolo, Chukwuedu. (1969). Biafran Refugees: Problems ...
1996). "Definition and diagnostic criteria of refractory ascites and hepatorenal syndrome in cirrhosis. International Ascites ... ascites). The spectrum continues with diuretic-resistant ascites, where the kidneys are unable to excrete sufficient sodium to ... and the presence of ascites. Specifically, the production of ascites that is resistant to the use of diuretic medications is ... This is believed to be a key step in the pathogenesis of ascites in cirrhotics as well. It has been hypothesized that the ...
Ascites may be present. The most characteristic feature are elevated levels of gamma glutamyl transferase (100-300 IU/L), ...
In addition to change in shape being a possible side effect from ascites and umbilical hernias, the navel can be involved in ... Herrine, Steven K. "Ascites". The Merck Manuals. Bagade, Pallavi V; Guirguis, Mamdouh M (2009). "Menstruating from the ... unexpected change in shape may be the result of ascites. ...
Ascites is often observed. Molyneux et al. (1991) reported that calves fed Senecio riddellii, which contains only riddelliine ... The typical clinical picture is that of ascites, hepatosplenomegaly, veno-occlusive disease of the liver, and abnormal liver ... and its N-oxide, for 20 days showed weight loss, signs of depression, reduced feed intake, ataxia of hind limbs, ascites, and ...
ascites). Treatment[edit]. Bloating is not life-threatening. In most cases, bloating can be handled with simple home remedies ...
Ascites. *Clubbing (deformed nails). *Cough. *Death rattle (last moments of life in a person/animal) ...
"Ascites - Hepatic and Biliary Disorders". Merck Manuals Professional Edition. May 2016. Retrieved 14 December 2017. Rossle M, ... It may also help with ascites. Severe procedural complications during a TIPS procedure, including catastrophic bleeding or ... ascites). An interventional radiologist creates the shunt using an image-guided endovascular (via the blood vessels) approach, ...
Malignant ascites. *Malignant transformation. Non-oncologic disorders referred to as "malignant": *Malignant hypertension ...
Marked recent weight loss is a major clinical manifestation, and unresponsiveness of the ascites to diuretics is an additional ... Kaman L, Behera A, Singh R, Katariya RN (2001). "Internal pancreatic fistulas with pancreatic ascites and pancreatic pleural ... Bibliography Brooks JR (1983). "Pancreatic ascites". In Brooks JR. Surgery of the Pancreas (1st ed.). Philadelphia: WB Saunders ... Smith EB (1953). "Hemorrhagic ascites and hemothorax associated with benign pancreatic disease". AMA Arch Surg. 67 (1): 52-6. ...
Chylous ascites and chylothorax rarely occur. List of cutaneous conditions Bolognia JL, Jorizzo JL, Rapini RP (2007). ...
Subserosal EG (4.5% to 9% in Japan and 13% in the US) presents with ascites which is usually exudative in nature, abundant ... Eosinophilic esophagitis Eosinophilic ascites Coeliac disease Protein losing enteropathy from intolerance to cow's milk protein ... "Eosinophilic gastroenteritis with ileus and ascites". Intern. Med. 35 (10): 779-82. doi:10.2169/internalmedicine.35.779. PMID ...
Liver cirrhosis with ascites Liver cirrhosis as seen on a CT of the abdomen in transverse orientation caudate lobe hypertrophy ... Ascites, accumulation of fluid in the peritoneal cavity in the abdomen, gives rise to bulging flanks. Jaundice, is yellow ... People with ascites due to cirrhosis are at risk of spontaneous bacterial peritonitis. Portal hypertensive gastropathy refers ... Moore KP, Aithal GP (October 2006). "Guidelines on the management of ascites in cirrhosis". Gut. 55 Suppl 6 (Suppl 6): vi1-12. ...
Ascites and peritonitis are less common. Fever is often absent. Blood tests may show leukocytosis and nonspecific liver test ...
Odeku, E. L.; Antia, A. U.; Udekwu, F. A. O. (1970). "Persistent ascites following infected ventriculoperitoneal shunt". West ...
Ascite sperimentale, 1915 - Experiments involving ascites. Rigenerazione e trapianti, 1927 - Regeneration and transplantion. ...
Ascites may be present in severe cases. The lower extremities may be edematous. Neurologic examination may reveal confusion, ...
Severtsev A., Chegin V., Ivanova E. «Surgical treatment of cirrhotic ascites (paracentesis, peritoneovenous shunting, narrow - ... peritoneovenous shunting performed with diuretics-resistant ascites; portocaval anastomoses; parenteral nutrition; ERAS ( ...
"Rheumatism, Hypertension, Ascites, and Hot Mineral Baths". Retrieved 2015-05-11. Wilson, William C.; Grande, ...
It results in right heart failure (ascites). It can be treated by balloon valvuloplasty or surgical resection. Pericardial ... It can result in chronic kidney failure, hypoalbuminemia, which can cause ascites and peripheral edema, and nephrotic syndrome ... and/or ascites (fluid in the abdomen). It can be caused by the above two diseases, congenital heart defects such as patent ... ascites), uncommonly fluid around the lungs (pleural effusion), or, rarely, peripheral edema. Sick sinus syndrome* is most ...
In the advanced stages it also extends to the pleural cavity and peritoneum (ascites) and can even develop into a generalized ... Fluid in the peritoneal cavity causing ascites. Generalized edema throughout the body known as anasarca. Most of the people ... Spontaneous bacterial peritonitis can develop where there is ascites present. This is a frequent development in children but ... Dyspnea may be present due to pleural effusion or due to diaphragmatic compression with ascites. Erythrocyte sedimentation rate ...
In gastroenterology, the puddle sign is a physical examination maneuver that can be used to detect the presence of ascites. It ... Chongtham DS, Singh MM, Kalantri SP, Pathak S (1997). "A simple bedside manoeuvre to detect ascites". The National Medical ... is useful for detecting small amounts of ascites-as small as 120 mL; shifting dullness and bulging flanks typically require 500 ... Abdominal examination Fluid wave test Bulging flanks Shah R. Ascites. URL: ...
The most common symptoms of peritoneal mesothelioma are abdominal swelling and pain due to ascites (a buildup of fluid in the ... Symptoms include:[citation needed] Abdominal pain Ascites, or an abnormal buildup of fluid in the abdomen A mass in the abdomen ... For pleural fluid, this is done by thoracentesis or tube thoracostomy (chest tube); for ascites, with paracentesis or ascitic ... or blood clots in the arteries of the lungs Severe ascites If a mesothelioma forms metastases, these most commonly involve the ...
People who have ascites. *Previous radiation therapy to the pelvis. *A complex hernia ...
Pontis H, Degerstedt G, Reichard P (1961). "Uridine and deoxyuridine phosphorylases from Ehrlich ascites tumor". Biochim. ...
... clinical diagnosis-ascites/varices/splenomegaly) . Mean pulmonary artery pressure-MPAP > 25 mmHg at rest . Pulmonary vascular ... "Portopulmonary hypertension in decompensated cirrhosis with refractory ascites". Gut. 52 (9): 1355-62. doi:10.1136/gut.52.9. ...
Green-brown: Biliary disease, bowel perforation with ascites. Milky-white or yellow and bloody: Chylous effusion. Milky or ...
Common symptoms of OGCT are bloating, abdominal distention, ascites, and dyspareunia. OGCT is caused mainly due to the ... Symptoms include bloating, abdominal distention, ascites, and dyspareunia. In rare cases where the tumor ruptures, acute ... In the end, pericardiophrenic lymphadenopathy and ascites may result from this frank invasion. Malignant OGCTs are ...
Mild ascites is hard to notice, but severe ascites leads to abdominal distension. Patients with ascites generally will complain ... Those with ascites due to cancer (peritoneal carcinomatosis) may complain of chronic fatigue or weight loss. Those with ascites ... Most experts recommend a diagnostic paracentesis be performed if the ascites is new or if the patient with ascites is being ... "The Management of Ascites in Cirrhosis: Report on the Consensus Conference of the International Ascites Club". Hepatology. 38: ...
Description Rapidly developing (acute) ascites can occur as a complication of trauma, perforated ulcer, appendicitis, or ... Ascites Definition Ascites is an abnormal accumulation of fluid in the abdomen. ... Ascites is an abnormal accumulation of fluid in the abdomen.. Description. Rapidly developing (acute) ascites can occur as a ... Chylous ascites has a milky appearance caused by lymph that has leaked into the abdominal cavity. Although chylous ascites is ...
The most common causes of ascites are cirrhosis of the liver, heart failure, tumours of the peritoneal membranes, and escape of ... Ascites, accumulation of fluid in the peritoneal cavity, between the membrane lining the abdominal wall and the membrane ... digestive system disease: Ascites. The accumulation of fluid in the abdominal cavity, or ascites, is related to portal ... More About Ascites. 2 references found in Britannica articles. Assorted References. *complication of liver disease* In ...
Ascites is the build-up of fluid in the space between the lining of the abdomen and abdominal organs. ... ascites; Liver failure - ascites; Alcohol use - ascites; End-stage liver disease - ascites; ESLD - ascites; Pancreatitis ... If you have ascites, call your health care provider right away if you have:. *Fever above 100.5°F (38.05°C), or a fever that ... Ascites results from high pressure in the blood vessels of the liver (portal hypertension) and low levels of a protein called ...
Find out more about ascites and its causes. ... Ascites happens when fluid accumulates in the abdomen, ... What is ascites?. Ascites occurs when fluid accumulates in the abdomen. This buildup occurs between two membrane layers that ... Prognosis and outlook of ascites depend on the underlying condition.. *Assessment of the progress of ascites may be made by ... Fast facts about ascites. *Ascites is a symptom of another underlying cause. ...
Tapping ascites (2).. Br Med J 1980; 281 doi: (Published 23 August 1980) Cite this as ...
Depending on whats causing it, doctors can treat chylous ascites and often cure it. ... Chylous ascites is when chyle collects in the belly. ... How Is Chylous Ascites Treated?. If a fetus has chylous ascites ... What Is Chylous Ascites?. Chylous ascites is when chyle collects in the belly. Chyle is a milky fluid made in the bowels ( ... In chylous ascites, it leaks into the belly.. What Are the Signs & Symptoms of Chylous Ascites?. While theres only a little ...
The formation of ascites in cirrhosis depends on the presence of unfavorable Starling forces within the hepatic sinusoid and on ... encoded search term (How do ascites form in cirrhosis?) and How do ascites form in cirrhosis? What to Read Next on Medscape. ... The serum-ascites albumin gradient is superior to the exudate-transudate concept in the differential diagnosis of ascites. Ann ... Definition and diagnostic criteria of refractory ascites and hepatorenal syndrome in cirrhosis. International Ascites Club. ...
In transudative ascites, fluid was said to cross the liver capsule because of an imbalance in Starling forces. In general, asci ... ascites was classified as being a transudate or an exudate. ... In general, ascites protein would be less than 2.5 g/dL in this ... In the past, ascites was classified as being a transudate or an exudate. In transudative ascites, fluid was said to cross the ... The serum-ascites albumin gradient is superior to the exudate-transudate concept in the differential diagnosis of ascites. Ann ...
Common causes of ascites are liver disease or cirrhosis, cancers,and heart failure. Signs and symptoms of ascities include ... Ascities treatment guidelines depend upon the condition causing ascites. The prognosis the life expectancy depends on the cause ... Ascites is accumulation of fluid in the abdominal cavity. ... Can ascites be prevented?. *Ascites Symptoms, Causes, Diagnosis ... What are the complications of ascites?. *What is the outlook for ascites? What is the life expectancy of ascites? Can ascites ...
Chylous ascites definition at, a free online dictionary with pronunciation, synonyms and translation. Look it up ... Words nearby chylous ascites. chylophyllous, chylopneumothorax, chylopoiesis, chylosis, chylothorax, chylous ascites, chyluria ... Ascites and anasarca, the consequence of very intemperate living.. An Account of the Foxglove and some of its Medical Uses, ... Ascites and anasarca, but not much otherwise diseased, and well enough to walk about the house, and see after her family ...
ascites diet mikeray where can i find more detailed info on low sodium/low potasium diets to work with diruetics...end stage ... ascites diet. where can i find more detailed info on low sodium/low potasium diets to work with diruetics...end stage liver ... This discussion is related to>Ascites.. ...
Learn about causes and symptoms and managing ascites. ... Ascites is an abnormal buildup of fluid in the abdomen and can ... When ascites is due to cancer, or if the fluid in the abdomen contains cancer cells, it is often called malignant ascites or ... Ascites. Ascites is an abnormal buildup of fluid in the abdomen, specifically the peritoneal cavity. It occurs when the body ... Managing ascites. Once the cause of ascites is known, your healthcare team can suggest ways to manage it. In some cases, ...
Ehrlich-Lettre ascites carcinoma (EAC) is also known as Ehrlich cell. It was originally established as an ascites tumor in mice ... "E (Ehrlich-Lettre ascites) ATCC ® CCL-77™ Mus musculus". "Ehrlich Cells (Molecular Biology)". DuPre, AM; Hempling, HG (1978). " ... Dupre, Ann M.; Hempling, H. G. (December 1978). "Osmotic properties of ehrlich ascites tumor cells during the cell cycle". ... "Osmotic properties of ehrlich ascites tumor cells during the cell cycle". J Cell Physiol. 97 (3): 381-96. doi:10.1002/jcp. ...
... Kourosh Alavi. ,1 Pradeep R. Atla. ,1 Tahmina Haq. ,1 and Muhammad Y. ... His ascites has completely resolved and he has started to gain weight with improved weight of 120 pounds at 3 months after ... Eosinophilic ascites (EA) is generally a rare finding in clinical practice. When present, it is most commonly associated with ... Ascitic fluid exam was remarkable for low serum-ascites albumin gradient (SAAG), PMN count ,250/mm3, and eosinophils of 62%. ...
Eli Lilly and Company has received its fourth U.S. Food and Drug Administration approval for CYRAMZA (ramucirumab). CYRAMZA (ramucirumab injection 10 mg/mL solution) is now also indicated in combination with FOLFIRI (irinotecan, folinic acid, and 5-fluorouracil) chemotherapy for the treatment of patients with metastatic colorectal cancer (mCRC) with disease progression on or after prior therapy with bevacizumab, oxaliplatin, and a fluoropyrimidine.. ...
Chylous ascites is the extravasation of milky chyle into the peritoneal cavity. This can occur de novo as a result of trauma or ... The ascites triglyceride level is elevated in all patients. Typically, chylous ascites is diagnosed when the ascites ... Chylous ascites accounts for less than 1% of cases. [2] The rarity of chylous ascites in clinical practice can be judged by the ... 6] can cause chylous ascites. Chylous ascites has also been reported in adults in association with hepatoma, small bowel ...
... it is known as ascites. Ascites usually occurs when the liver stops working properly. Fluid fills the space between the lining ... People with cirrhotic ascites have a two-year... ... Ascites Causes and Risk Factors. Ascites is most often caused ... People with cirrhotic ascites have a two-year... Read More. When fluid builds up inside the abdomen, it is known as ascites. ... Symptoms of Ascites. Symptoms of ascites can appear either slowly or suddenly, depending on the cause of the fluid buildup. ...
This Ascites page on EmpowHER Womens Health works best with javascript enabled in your browser.. Toggle navigation Womens ... Im sure the doctor already explained what ascites is, but I can provide a resource for you as well. ...
I have a condition called ascites which is essentially,fluid in the abdomen and it involves the liver. Does anyone here have ... I have a condition called ascites which is essentially,"fluid in the abdomen" and it involves the liver. Does anyone here have ...
Ascites has many causes, most of which can be very serious. In cats, ascites is caused by the leakage of fluid into the abdomen ... Overview of Ascites in Cats. Ascites is the abnormal accumulation of fluid in the abdominal cavity. The volume of the fluid can ... Any amount of ascites is a significant finding, however, and steps should be taken to find a diagnosis. Ascites has many ... Causes of Ascites in Cats. Hypoalbuminemia Causes of Ascites in Cats. Hypoalbuminemia is a decrease in serum albumin levels. ...
Ascites (expanded list) answers are found in the Diagnosaurus powered by Unbound Medicine. Available for iPhone, iPad, Android ... Diseased peritoneum (SAAG , 1.1 g/dL except in "mixed ascites" with portal hypertension and secondary process, eg, infection, ... Diseased peritoneum (SAAG , 1.1 g/dL except in "mixed ascites" with portal hypertension and secondary process, eg, infection, ... Ascites (expanded list) is a topic covered in the Diagnosaurus. To view the entire topic, please sign in or purchase a ...
In dogs, ascites has many causes, most of which can be very serious. Ascites is caused by the leakage of fluid into the abdomen ... Overview of Ascites in Dogs. Ascites is the abnormal accumulation of fluid in the abdominal cavity. The volume of the fluid can ... Any amount of ascites is a significant finding, however, and steps should be taken to find a diagnosis. Ascites has many ... Ascites, itself, usually does not cause a problem, but the primary disease process causing the ascites can be a serious ...
This CT scan of the lower abdomen shows a massive amount of free abdominal fluid (ascites) in a patient with ovarian cancer. ... ascites) in a patient with ovarian cancer. ...
The term ascites is of Greek origin, from the word askos, which means ... Definition Ascites is defined as an abnormal amount of intraperitoneal fluid. ... Biliary ascites. Biliary ascites in neonates is rare. The usual clinical presentation is that of ascites with mild jaundice ( ... Urinary ascites. Antenatal diagnosis of urinary ascites has been made with fetal US. Signs and symptoms of urinary ascites ...
Compare risks and benefits of common medications used for Ascites. Find the most popular drugs, view ratings, user reviews, and ... Learn more about Ascites. IBM Watson Micromedex. *Ascites. ICD-10 CM Clinical Codes (External). *Look up ICD10 codes for ... About Ascites: An excess of fluid in the peritoneal cavity (membrane lining of the abdomen). This is typically caused by liver ... Drugs Used to Treat Ascites. The following list of medications are in some way related to, or used in the treatment of this ...
... transplant-free survival when compared to large volume paracentesis in patients with cirrhosis and difficult to treat ascites. ... Tags: Ascites, Bleeding, Blood, Blood Vessels, Cirrhosis, Heart, Hernia, Life Expectancy, Ligation, Liver, Liver Disease, ... Ascites is the build up of fluid in the abdomen and variceal bleeding involves the rupture of veins in the gastrointestinal ... The Gore study for ascites patients comes on the heels of a landmark study published in The New England Journal of Medicine ...
Currently have ascites even with TIPS. It used to be under control but even TIPS is not controlling it any more. The volume of ... Ascites- lab values. akoger05akog Currently have ascites even with TIPS. It used to be under control but even TIPS is not ... Ascites- lab values. Currently have ascites even with TIPS. It used to be under control but even TIPS is not controlling it any ... My question is, are there any specific lab values that represent ascites and if the "causes" of ascites specifically are ...
A definition for the medical term "ascites" is presented. It refers to the abnormal accumulation of fluid in the peritoneal ...
  • Ascites is the abnormal buildup of fluid in the abdomen . (
  • Blood that cannot flow through the liver because of the increased pressure leaks into the abdomen and causes ascites. (
  • Ascites is the build-up of fluid in the space between the lining of the abdomen and abdominal organs. (
  • People with certain cancers in the abdomen may develop ascites. (
  • Ascites is the build-up of fluid in the abdomen. (
  • Ascites occurs when fluid accumulates in the abdomen. (
  • In some instances, a person's abdomen might contain about 5 liters of fluid, but in some extreme cases of ascites, doctors have drained more than 10 liters of fluid from the abdomen. (
  • When ascites is caused by cancer, doctors may use a shunt (tube) to move the fluid from the abdomen into the bloodstream. (
  • Through an analysis of cellular components (cell fractions) from malignant ascites (fluid buildup in the abdomen caused by gastric cancer), a research collaboration based in Kumamoto University (Japan) has demonstrated that cellular senescence of cancer-associated fibroblasts (CAFs) play an important role in the peritoneal dissemination of gastric cancer foci (cells different from surrounding cells). (
  • Ascites is caused by a variety of diseases and conditions, for example, cirrhosis of the liver , cancer within the abdomen, congestive heart failure , and tuberculosis . (
  • When ascites is due to cancer, or if the fluid in the abdomen contains cancer cells, it is often called malignant ascites or malignant peritoneal effusion. (
  • His physical exam was significant for distended abdomen with tense ascites and diffuse tenderness. (
  • When fluid builds up inside the abdomen, it is known as ascites. (
  • I have a condition called ascites which is essentially,"fluid in the abdomen" and it involves the liver. (
  • In cats, ascites is caused by the leakage of fluid into the abdomen from blood vessels, lymphatics, internal organs or abdominal masses. (
  • This CT scan of the lower abdomen shows a massive amount of free abdominal fluid (ascites) in a patient with ovarian cancer. (
  • Cancers of organs in the abdomen also may lead to ascites. (
  • Not being able to rid the body of salt causes fluids to build up in the abdomen, resulting in ascites. (
  • Fluid buildup in the abdomen is called ascites. (
  • Ascites causes fluid to build up in the abdomen. (
  • Often the most effective short-term treatment for ascites is a procedure called a paracentesis, where a needle is inserted through the front of the abdomen and fluid is drained. (
  • Although a paracentesis effectively controls the symptoms of ascites, it does not address the reasons why the fluid built up in the abdomen. (
  • Ascites (ah-SITE'-eez) is extra fluid built up in the abdomen. (
  • Ascites in the abdomen can also be traced back to other sources such as heart failure, kidney failure, and cancer. (
  • Ascites is a build up of fluid between the tissue that lines the abdomen and the peritoneal cavity (or the abdominal organs). (
  • Visit a doctor who will either perform an ultrasound on your abdomen or draw fluid from your stomach and run a test on that fluid to determine whether you have Ascites. (
  • This sodium retention, coupled with inadequate levels of albumin, facilitates the shift of fluid from the intravascular space into the interstitial space of the abdomen and creates ascites ("Patho Puzzler," 2005). (
  • He began experiencing abdominal bloating, and during evaluation they did a CT scan indicating spots around the abdomen lining and a potential spot in the liver and determined the fluid to be Malignant Ascites. (
  • It is my understanding that albumin helps to bind the blood cells and protect against leakage of blood fluids through vessels and arteries, which makes low albumin appear to be the main culprit for liver ascites and malignant ascites (cancer in the abdomen lining). (
  • Mike had some ascites in his abdomen and some in the right lung when he was first diagnosed. (
  • Coloured computed tomography (CT) scan of the abdomen of a patient with ascites, the accumulation of fluid in the peritoneal cavity. (
  • symptoms which can appear as the condition progresses include: A swollen abdomen ( ascites ) Swelling, especially in the lower legs (oedema) Irritability and confusion How is autoimmune liver disease diagnosed? (
  • Ascites is the accumulation of protein-containing (ascitic) fluid within the abdomen. (
  • Less common causes of ascites include disorders unrelated to the liver, such as cancer, heart failure, kidney failure, inflammation of the pancreas (pancreatitis), and tuberculosis affecting the lining of the abdomen. (
  • Side view of the distended abdomen of a 66 year old male patient caused by ascites, a collection of fluid in the peritoneal cavity between the organs and abdominal wall. (
  • Signs of ascites in dogs include vomiting, pain, or a distended abdomen among other things. (
  • The serum-ascites albumin gradient (SAAG) is probably a better discriminant than older measures (transudate versus exudate) for the causes of ascites. (
  • The serum-ascites albumin gradient is superior to the exudate-transudate concept in the differential diagnosis of ascites. (
  • This is referred to as the SAAG or the Serum Ascites Albumin Gradient. (
  • The difference between serum albumin and ascites albumin (serum ascites-albumin gradient) in patients with cirrhosis and portal hypertension is usually more than 1.1 g/dL. (
  • Results of the serum-ascites albumin gradient overlapped the least between the two groups: all but 1 patient with malignant ascites while only 1 patient with liver disease had a gradient lower than 1.1. (
  • We conclude that the serum-ascites albumin gradient offers the best diagnostic discrimination between ascites caused by liver disease and ascites caused by a neoplasm. (
  • [3] Of those with cirrhosis, more than half develop ascites in the ten years following diagnosis. (
  • [3] Of those in this group who develop ascites, half will die within three years. (
  • Persons who have systemic lupus erythematosus but do not have liver disease or portal hypertension occasionally develop ascites. (
  • People with cancer can develop ascites for different reasons. (
  • Approximately 80% of patients with cirrhosis of the liver develop ascites. (
  • Most people who develop ascites develop a large belly and experience a rapid gain in weight. (
  • During the course of the disease, more than one-third of women with ovarian cancer will develop ascites ( 3 ). (
  • Cancer of the ovary has no initial symptoms, and many women will have the diagnosis made because they develop ascites. (
  • Only cirrhotic patients with portal hypertension (HTN) develop ascites, and portal HTN can also lead to the development of ascites in patients with conditions other than cirrhosis. (
  • People who have alcoholic cirrhosis may develop ascites early in the course of liver disease. (
  • Those who have other forms of cirrhosis, such as that caused by viral hepatitis, may develop ascites much later. (
  • Chylous ascites has a milky appearance caused by lymph that has leaked into the abdominal cavity. (
  • What Is Chylous Ascites? (
  • Chylous ascites is when chyle collects in the belly. (
  • Depending the cause, doctors can treat chylous ascites (KYE-lus uh-SYE-teez) and often cure it. (
  • What Happens in Chylous Ascites? (
  • In chylous ascites, it leaks into the belly. (
  • While there's only a little fluid in the belly, chylous ascites may not cause any symptoms. (
  • Kids with chylous ascites also can have trouble fighting infections and may get blood clots. (
  • How Is Chylous Ascites Diagnosed? (
  • Before a baby is born, doctors might suspect chylous ascites when a routine prenatal ultrasound shows fluid in the baby's belly. (
  • How Is Chylous Ascites Treated? (
  • If a fetus has chylous ascites, doctors will watch the mother's pregnancy closely. (
  • Chylous ascites caused by a leak in the lymphatic system may heal on its own. (
  • Chylous ascites is the extravasation of milky chyle rich in triglycerides into the peritoneal cavity, which can occur de novo as a result of trauma or obstruction of the lymphatic system. (
  • True chylous ascites is defined as the presence of ascitic fluid with high fat (triglyceride) content, usually higher than 110 mg/dL. (
  • Based on animal experiments, Blalock concluded that obstruction of the thoracic duct alone was not sufficient to cause chylous ascites. (
  • Chylous ascites is an uncommon clinical condition that occurs as a result of disruption of the abdominal lymphatics. (
  • Multiple causes have been described, with the most common causes being malignancy (hepatoma, small bowel lymphoma, small bowel angiosarcoma, and retroperitoneal lymphoma), cirrhosis (≤0.5% of patients with ascites from cirrhosis may have chylous ascites), and trauma after abdominal surgery. (
  • In adults, chylous ascites is associated most frequently with malignant conditions. (
  • [ 6 ] can cause chylous ascites. (
  • In children, the most common causes of chylous ascites are congenital abnormalities, such as lymphangiectasia, mesenteric cyst, and idiopathic "leaky lymphatics. (
  • [ 6 ] Neoplasia is an uncommon cause of pediatric chylous ascites. (
  • [ 10 ] Lymphatics may spontaneously rupture in patients with cirrhosis as a result of higher than typical flow, with the formation of chylous ascites. (
  • Chylous ascites has been reported in patients with polycythemia vera and resulting hepatic vein thrombosis. (
  • Abdominal surgery is a common cause of chylous ascites. (
  • In a series of 329 patients with testicular cancer who underwent postchemotherapy retroperitoneal lymph node dissection, 7% of patients developed chylous ascites. (
  • Neonatal ascites is usually biliary, urinary, or chylous. (
  • Neonatal chylous ascites is almost always idiopathic, but a congenital lymphatic abnormality is thought to be the usual underlying cause. (
  • Elevated fluid triglyceride levels in chylous Ascites should prompt an evaluation for malignancy or tuberculosis. (
  • Chylous ascites as a complication of laparoscopic colorectal surg. (
  • Until now, no data have been available on the incidence of chylous ascites after laparoscopic colorectal surgery. (
  • The clinical data of the patients with postoperative chylous ascites were compared with those of the patients without chylous ascites. (
  • Blood loss, operative time, tumor location, and extent of lymph node dissection were not associated with postoperative chylous ascites. (
  • Chylous ascites are not a rare complication of laparoscopic colorectal surgery. (
  • Pediatric chylous ascites treatment with combined ultrasound and fluoroscopy-guided intranodal lymphangiography. (
  • Chylous ascites is a form of ascites resulting from the leakage of lymph into the peritoneal cavity, which is particularly rare in children, most common etiology being an iatrogenic injury to lymphatics during surgery. (
  • This study reports a case of a 7-year-old child with refractory chylous ascites to demonstrate a minimally invasive technique of intranodal lymphangiography with lipiodol as a viable treatment option for chylous ascites in children, particularly in cases of minor and undetectable leaks. (
  • Most experts recommend a diagnostic paracentesis be performed if the ascites is new or if the patient with ascites is being admitted to the hospital. (
  • The initial diagnostic approach to a patient with ascites requires the combination of a thorough history and physical examination followed by abdominal paracentesis with lab analysis of the fluid. (
  • Care and management of the patient with Ascites. (
  • A careful history of the patient with ascites should include information that places a patient at risk for liver disease, including quantity and duration of alcohol use, transfusion of blood products, and personal or family history of autoimmune or hepatic diseases. (
  • SBP is suspected if a patient with ascites also has abdominal pain, fever, or unexplained deterioration. (
  • 1.1 g/dL) indicates the ascites is due to portal hypertension. (
  • Ascites results from high pressure in the blood vessels of the liver (portal hypertension) and low levels of a protein called albumin . (
  • A classic cause of transudative ascites would be portal hypertension secondary to cirrhosis and congestive heart failure. (
  • Although the exact mechanism of ascites development is not completely understood, most theories suggest portal hypertension (increased pressure in the liver blood flow to the liver) as the main contributor. (
  • Ascites occurs when portal hypertension develops. (
  • A radiologist can place a shunt directly through the liver, thereby relieving portal hypertension and diminishing ascites. (
  • Ascites resulting from hydrostatic disease (portal hypertension, congestive heart failure) has serum Ascites albumin difference greater than 1.1g/dL, whereas inflammatory or neoplastic disease produces a serum Ascites albumin difference less than 1.1g/dL. (
  • Ascites, another important complication of advanced cirrhosis and severe portal hypertension, is sometimes refractory to treatment and is complicated by spontaneous bacterial peritonitis and hepatorenal syndrome. (
  • We describe the pathophysiology of portal hypertension and the current management of its complications, with emphasis on the prophylaxis and treatment of variceal bleeding and ascites. (
  • The most recent theory of cirrhotic ascites formation is the peripheral arterial vasodilation hypothesis, which proposes that portal hypertension develops prior to the development of ascites (see Figure 1). (
  • The main outcome measure was the prediction of portal hypertension (PHT) or non-portal hypertension related causes of ascites from SAAG. (
  • The most common cause of ascites is portal hypertension secondary to cirrhosis. (
  • As discussed earlier, the most common cause of ascites is cirrhosis and portal hypertension. (
  • Serum-ascites albumin concentration gradient, a parameter of oncotic pressure gradient reflecting presence or absence of portal hypertension, was compared with the usual parameters of ascitic fluid analysis in the differential diagnosis of ascites. (
  • Quantitative modeling of the physiology of ascites in portal hypertension. (
  • Although the factors involved in cirrhotic ascites have been studied for a century, a number of observations are not understood, including the action of diuretics in the treatment of ascites and the ability of the plasma-ascitic albumin gradient to diagnose portal hypertension. (
  • IV medication to treat the persistent infection in the liver cysts, she also had dreadful ascites due increasing portal hypertension as her portal vein had become clotted. (
  • of liver disease, such as portal hypertension and ascites (abdominal swelling), as well as a nutritional assessment. (
  • Many disorders can cause ascites, but the most common is high blood pressure in the veins that bring blood to the liver ( portal hypertension ), which is usually due to cirrhosis . (
  • Ascites , accumulation of fluid in the peritoneal cavity , between the membrane lining the abdominal wall and the membrane covering the abdominal organs. (
  • The most common causes of ascites are cirrhosis of the liver, heart failure , tumours of the peritoneal membranes, and escape of chyle (lymph laden with emulsified fats) into the peritoneal cavity. (
  • The medical definition of ascites is an abnormal accumulation of fluid within the (peritoneal) cavity. (
  • To diagnose the cause of ascites, your doctor and other healthcare professionals will examine you around the belly to determine how much fluid is in the peritoneal cavity. (
  • In medicine ( gastroenterology ), ascites (also known as peritoneal cavity fluid , peritoneal fluid excess , hydroperitoneum or more archaically as abdominal dropsy ) is an accumulation of fluid in the peritoneal cavity. (
  • Ascites happens in the peritoneal cavity when the body makes more fluid than it can remove. (
  • Ascites is most commonly caused by liver damage and other medical terms for this condition include peritoneal cavity fluid, hydroperitoneum or abdominal dropsy. (
  • Ascites is an accumulation of fluid in the peritoneal cavity. (
  • Ascites is the term used to denote increased fluid in the peritoneal cavity, a situation that is not normal. (
  • This water overload can cause leakage into the peritoneal cavity and the formation of ascites. (
  • Ascites is the pathologic accumulation of fluid within the peritoneal cavity. (
  • Ascites is the accumulation of fluid in the peritoneal cavity, usually resulting from cirrhosis. (
  • Cancer and infections may lead to ascites because of the increased fluid production in the peritoneal cavity exceeding resorptive capacity. (
  • Ascites is the accumulation of fluid in the peritoneal cavity. (
  • This communication presents an explanation of ascites based solely on pathophysiological alterations within the peritoneal cavity. (
  • This focus on renal and systemic effects has led investigators to lose sight of the local factors within the peritoneal cavity that actually are responsible for the accumulations of ascites. (
  • Hybridoma cells will be injected into the mouse peritoneal cavity to commence ascites production. (
  • Symptoms may develop slowly or suddenly depending on the cause of ascites. (
  • In this article, we will explore the causes of ascites, as well as the symptoms and treatment options. (
  • Abdominal swelling and associated weight gain are typical symptoms of ascites. (
  • What are the symptoms of ascites? (
  • What causes signs and symptoms of ascites? (
  • Signs and symptoms of ascites include abdominal pain and bloating , shortness of breath , and liver failure. (
  • Symptoms of ascites can vary depending on their cause and other factors. (
  • They relieve the symptoms of ascites or treat the cancer that is causing them. (
  • If you experience ascites symptoms, talk to your doctor as soon as possible. (
  • Symptoms of ascites can appear either slowly or suddenly, depending on the cause of the fluid buildup. (
  • Keep in mind that ascites symptoms may be caused by other conditions. (
  • Tell your doctor if you develop these problems or if you have been treated for ascites and these symptoms have come back. (
  • If you suffer from a mild case of ascites, the symptoms can be quite impossible to pinpoint. (
  • Salt and water retention can also contribute to the severity of ascites symptoms. (
  • This book provides research on the aetiology, symptoms and treatment of ascites. (
  • These symptoms could be signs of Ascites. (
  • If you have a fever, while also suffering from a swollen or distended stomach, or if you are having difficulties breathing, then you may be experiencing symptoms of Ascites, and should seek medical attention. (
  • Because the symptoms of Ascites can easily be attributed to other conditions, many of which are not serious, you should talk to your doctor if you feel you may have Ascites and have him run the appropriate tests for the condition. (
  • Mild Ascites often does not produce any symptoms, therefore you need to get tested from a doctor to determine whether you have the illness and to discuss what treatment is best for you. (
  • As ascites progresses, patients may complain of compressive symptoms such as abdominal discomfort, early satiety, or nausea. (
  • The delay in the presentation until the patient began to experience the symptoms due to urinary ascites, the diagnostic dilemma because of the rarity of the condition, and the possible aetiologies are discussed. (
  • Background: Malignant ascites is common in ovarian cancer and often causes symptoms such as abdominal pressure and shortness of breath, resulting in an decreased quality of life for the patient. (
  • Though ascites is usually a secondary symptom of another potentially serious issue, it can bring symptoms of its own. (
  • If you see the symptoms of ascites in your dog, you should consult your veterinarian so they can diagnose the underlying cause and begin treatment. (
  • Here is what you should know about the symptoms, causes, and treatments for ascites in dogs. (
  • There are several possible symptoms of ascites in dogs that result from the accumulation of fluid in the abdominal cavity and the pressure it puts on organs. (
  • If you see any of the following symptoms in your dog, consult your veterinarian immediately, as ascites is often, itself, a secondary symptom of a serious medical issue. (
  • Treatment of ascites in dogs may provide relief from the symptoms of fluid build up, but treatment for the underlying cause is necessary, or ascites is likely to return. (
  • Differential diagnosis of benign and malignant ascites can rely on certain precise findings such as appropriate ascites fluid serum analysis, cytological examination, laparoscopy, and the symptoms of the patients. (
  • The underlying conditions that cause ascites are often serious illnesses linked to reduced life expectancy. (
  • Cancer that spreads to the liver can also cause ascites. (
  • For example, metastatic cancer can cause ascites, and this abdominal fluid collection will never be fully resolved until the cancer is treated. (
  • Many diseases can cause ascites, including liver disease and some cancers. (
  • conditions unrelated to the liver can also cause ascites. (
  • Portal vein thrombosis does not usually cause ascites unless hepatocellular damage is also present. (
  • Here are a few possible medical problems that are known to cause ascites. (
  • Pancreatic ascites develops when a cyst that has thick, fibrous walls (pseudocyst) bursts and permits pancreatic juices to enter the abdominal cavity. (
  • This type of ascites is typically a manifestation of advanced cancers of the organs in the abdominal cavity, such as, colon cancer , pancreatic cancer , stomach cancer , breast cancer , lymphoma , lung cancer , or ovarian cancer . (
  • Pancreatic ascites can be seen in people with chronic (long standing) pancreatitis or inflammation of the pancreas. (
  • Pancreatic ascites (either from trauma or pancreatitis) may occur in children. (
  • Pancreatic cancer patients may have ascites at any time, but it is especially common in patients whose cancer has metastasized, or spread. (
  • For more information on ascites or for free, in-depth and personalized resources and information on pancreatic cancer, contact a Patient Central Associate . (
  • The most common cancers that lead to ascites are pancreatic and ovarian followed by colon and other abdominal cancers, lymphoma, uterine, and breast cancer. (
  • Pancreatic ascites can occur with acute or chronic pancreatitis, from a leaking pseudocyst, or after pancreatic trauma. (
  • However, ascites is more often associated with liver disease and other long-lasting (chronic) conditions. (
  • In patients having liver disease , the onset of ascites is usually preceded by accumulation of fluid in the ankles. (
  • Liver disease is the most common cause of ascites. (
  • Similarly, if the ascites is due to liver disease , the health of the liver should be addressed in order to cure the ascites. (
  • Ascites can cause liver disease and cirrhosis, and death. (
  • Most commonly, ascites is due to liver disease and the inability of that organ to produce enough protein to retain fluid in the bloodstream as well as an obstruction to flow through the scarred cirrhotic liver . (
  • Significant mortality is still being observed in cirrhotic patients with ascites and relatively preserved liver and renal function, as indicated by a lower Model for End-Stage Liver Disease (MELD) score. (
  • Ascites is usually associated with more severe conditions, such as liver disease, cirrhosis, congestive heart failure, liver cancer, ovarian cancer and pancreatitis. (
  • Development of ascites is predictive of mortality, especially in liver disease. (
  • This condition will evolve in overt fluid retention and ascites, as the liver disease progresses. (
  • Renal ascites develops when blood levels of albumin dip below normal. (
  • The increase in portal blood pressure and decrease in albumin (a protein that is carried in the blood) may be responsible in forming the pressure gradient and resulting in abdominal ascites. (
  • 1.1 g/dL) (see the Ascites Albumin Gradient calculator). (
  • Another method to categorize the type of ascites is by measuring the amount of albumin in the fluid and compare this to the serum albumin found in the blood. (
  • It is believed that the increase in pressure in the liver blood flow along with a decrease in the albumin content in the blood may be responsible for the development of abdominal ascites. (
  • To show the interest of the serum-ascites concentration gradient in albumin (SAAG) in the etiological diagnosis of ascites in African black. (
  • Our study therefore aimed to clarify the interest of the ascites serum albumin gradient (SAAG) in the etiological diagnosis of ascites by assessing its threshold value in the diagnosis of cirrhosis and peritoneal tuberculosis in Black Africans. (
  • Also, I have seen where trials have been done using albumin to treat cirrhosis related ascites which represents about 80% of all ascites. (
  • Mike's ascites got pretty bad at the end, where they had to tap him about 2 times per week and give him the Albumin. (
  • If the physical examination is equivocal, ultrasound confirms the diagnosis of Ascites. (
  • The present study was to create and evaluate a new bioscore combined with PCT, sNFI and dCHC in the diagnosis of ascites infection in cirrhotic patients. (
  • A bioscore of ≥3.40 was considered to be statistically significant in making a positive diagnosis of ascites infection. (
  • Describe the advantages and disadvantages of this technique for diagnosis of Ascites. (
  • V. Describe the advantages and disadvantages of the alternative techniques for diagnosis of Ascites. (
  • The formation of ascites in cirrhosis depends on the presence of unfavorable Starling forces within the hepatic sinusoid and on some degree of renal dysfunction. (
  • The circulating blood volume may be perceived as low by the sensors in the kidneys as the formation of ascites may deplete some volume from the blood. (
  • 14. Alterations of Hepatic and Splanchnic Microvascular Exchange in Cirrhosis: Local Factors in the Formation of Ascites (Jens H. Henriksen and Søren Møller). (
  • The fundamental factors involved in the formation of ascites were established over a century ago when Starling [ 1 ] used observations of thoracic duct lymph flow and fluid absorption from the peritoneal space to support his classic description of the forces that determine capillary fluid balance. (
  • Ascites is one of the most common liver diseases that is caused by cirrhosis of the liver. (
  • Complications of ascites include bleeding from the lower intestine, kidney failure , and other complications of cirrhosis of the liver. (
  • Common risk factors for the development of ascites are any diseases that can cause cirrhosis of the liver. (
  • His family was surprised to learn that he had cirrhosis of the liver and that was what was causing his ascites. (
  • The most common cause of ascites is cirrhosis of the liver, which in turn is most often caused by excessive alcohol intake. (
  • Doing an initial paracentesis for diagnostic purposes is an important aspect of the treatment for ascites. (
  • Hepatic hydrothorax is best treated by removing the abdominal ascites by paracentesis. (
  • Patients with refractory ascites require second-line treatments of repeat large-volume paracentesis (LVP) or the insertion of a transjugular intrahepatic portosystemic shunt (TIPS), and assessment for liver transplantation. (
  • A one-time paracentesis treatment may relieve the discomfort of tense ascites before you begin treatment with one or more diuretics . (
  • A diagnostic paracentesis of at least 30 mL should always be performed to elucidate the cause of ascites. (
  • ALFApump® System Versus Large Volume Paracentesis in the Treatment of Refractory Ascites. (
  • The model's predictions accurately mimic clinical observations in ascites, including the magnitude and time course of changes observed following paracentesis or diuretic therapy. (
  • The ATLANTIS-study was designed to determine the safety of a full paracentesis in patients with malignant ascites due to ovarian cancer. (
  • Half of the patients will receive a full paracentesis, while the other half will receive fractioned paracentesis with clamping of the drain after 3 liters of ascites was evacuated. (
  • This can be used in medium to serious cases of ascites. (
  • Nearly eighty percent of all reported cases of ascites can be traced back to advanced liver damage. (
  • We knew several stage IV survivors his Oncologist was working with that had extreme cases of ascites and had been given no hope by other doctors. (
  • What are the risk factors for development of ascites? (
  • This complicated pathophysiology ultimately leads to the development of ascites. (
  • Hypoalbuminemia is often a contributing factor in the development of ascites in cirrhosis as well. (
  • Two years after the development of ascites associated with alcoholic cirrhosis, an affected individual has a 50% chance of survival. (
  • Development of ascites associated with chronic hepatitis C suggests a 5-year survival time (Saadeh & Davis, 2004). (
  • Disease caused by this mold, so-called "brooder pneumonia", forms mould colonies in the lungs, and produces hard nodular areas leading to air sac infection and subsequently to the development of ascites. (
  • Other treatments for ascites include giving medications called diuretics , maintaining a low- sodium diet, and restricting daily fluid intake. (
  • What are the best treatments for ascites? (
  • Ascites caused by cirrhosis or heart failure generally has a SAAG score greater than 1.1 whereas the SAAG score of ascites caused by other reasons is generally lower than 1.1. (
  • The simple technical SAAG, allowing to distinguish the ascites linked to the PHT and the ascites not bound to the PHT, can be used like a method of diagnosis of the etiologies of the ascites especially in the peritoneal tuberculosis in the African countries low income. (
  • It is therefore questionable whether SAAG has the same diagnostic value in the etiological research of ascites in Africans as in Caucasians. (
  • Many underlying diseases can be responsible for causing ascites, including tuberculosis , kidney disease, pancreatitis , and an underactive thyroid. (
  • Ascites may also result from other systemic diseases, such as heart failure (cardiac Ascites) and nephritic syndrome, or from primary peritoneal pathology, such as peritoneal carcinomatosis or tuberculosis. (
  • The causes of ascites are multiple, nevertheless two etiologies predominate, liver cirrhosis for transudative ascites and peritoneal tuberculosis for exudative ascites in our contexts. (
  • If cirrhosis has caused ascites, an individual may benefit from reducing salt and taking water tablets (diuretics). (
  • While this is an effective approach in many situations, some types of ascites are resistant to diuretics. (
  • Part of the treatment for ascites often involves supplying the patient with medications called diuretics. (
  • Often two diuretics, called spironolactone and furosemide , are given on a daily basis to treat ascites. (
  • The treatment for ascites may include diuretics to reduce water retention and remove excess fluid as well as antibiotics in case there is an infection. (
  • Often, patients will require diuretics ('water pills') to treat ascites. (
  • Diuretics are not useful for malignant ascites. (
  • The treatment of ascites requires dietary sodium restriction and the judicious use of distal and loop diuretics, sequential at an earlier stage of ascites, and a combination at a later stage of ascites. (
  • About 90 out of 100 people with ascites caused by cirrhosis respond to treatment with a low-salt diet and medicines ( diuretics ) that eliminate extra fluid from the body. (
  • Treatment is of the underlying condition, but diuretics and draining the fluid can be used to reduce the ascites. (
  • Once ascites is present, most therapeutic modalities are directed on maintaining negative sodium balance, including salt restriction, bed rest and diuretics. (
  • Have severe ascites that is causing extreme discomfort, including abdominal pain and difficulty breathing (tense ascites). (
  • In patients with large and tense ascites, the respiratory function and physical activity may be impaired. (
  • Emergency measures are required for large, tense ascites that causes dyspnea, hypoxia and/or severe abdominal discomfort. (
  • People with cirrhotic ascites have a two-year. (
  • People with cirrhotic ascites have a two-year survival rate of approximately 50 percent . (
  • Ascites that is caused by liver damage is referred to as cirrhotic ascites. (
  • Chapter Four reports information about the management of malignant ascites in ovarian cancer and discusses the involvement of malignant ascites in dissemination, angiogenesis, migration, invasion and apoptosis of ovarian cancer cells. (
  • Options for late-stage ovarian cancer are limited and invasive, especially once malignant ascites develops. (
  • Malignant ascites, a complication observed in terminal ovarian cancer, significantly contributes to poor quality of life and to mortality. (
  • Here we applied non-invasive magnetic resonance imaging (MRI) and spectroscopic imaging (MRSI) of syngeneic mouse tumors in vivo , and high-resolution 1 H MRS of mouse tumor extracts, to characterize the relationship between ascites volumes and the vasculature and metabolism of an experimental model of ovarian cancer. (
  • Therapeutic options for advanced stage ovarian cancer are extremely limited and very invasive, especially once malignant ascites develops ( 2 ). (
  • 38% of malignant ascites occurring in women are associated with ovarian cancer. (
  • But no guidelines exist on the management of ascites drainage in ovarian cancer. (
  • Ascites and spontaneous bacterial peritonitis. (
  • Patients with known ascites should be questioned regarding compliance with salt restriction and diuretic therapy, and for any past complications related to the ascites such as spontaneous bacterial peritonitis (SBP). (
  • It can result in severe complications, including bleeding of esophagogastric varices as well as spontaneous bacterial peritonitis or hepatorenal syndrome as complications of ascites. (
  • Patients with ascites and spontaneous bacterial peritonitis (SBP) can present with fever, chills, abdominal pain, hepatic encephalopathy, and rebound abdominal tenderness. (
  • The case is reported of a patient presenting with ascites and acute renal failure resulting from spontaneous rupture of the urinary bladder, 30 years after the successful initial treatment of childhood rhabdomyosarcoma. (
  • Additionally, the sonographer can make an estimation of the amount of ascitic fluid, and difficult-to-drain ascites may be drained under ultrasound guidance. (
  • Furthermore, the time and availability of ascites are not constant at all-time [ 7 ], therefore, is essential to develop sensitive, accurate and rapid methods to diagnose ascitic fluid infection. (
  • This book addresses the increasing incidence and significance of diseases, such as ascites, renal dysfunction, cirrhosis and hypertension where both organs are involved. (
  • Soft and stable" handling of parenchymal organs could be achieved using "bellows" type suckers in combination with artificial ascites. (
  • Other causes of ascites related to portal HTN include heart failure, acute liver failure or alcoholic hepatitis, hepatic veno-occlusive disease (Budd-Chiari syndrome), constrictive pericarditis, and end-stage renal disease (ESRD). (
  • What are the complications of ascites? (
  • The diagnosis of refractory ascites requires the demonstration of diuretic non-responsiveness, despite dietary sodium restriction, or the presence of diuretic-related complications. (
  • What other diseases, conditions, or complications should I look for in patients with ascites? (
  • Diseases that can cause severe liver damage can lead to ascites. (
  • Hypoalbuminemia, with resultant decrease in oncotic pressure, may lead to ascites in protein-losing enteropathy, malnutrition, and nephrotic syndrome. (
  • In the case of ascites caused by genetics, feed restriction might reduce the effect of the disease. (
  • In the case of ascites caused by microorganisms, recent studies investigating the effect of feed supplementation with acidifiers have shown promising results. (
  • The etiology of ascites may differ among neonates and older children. (
  • The etiology of ascites is very conflicting. (
  • The precise incidence of ascites in children is unknown, but the condition is rare. (
  • Since several seemingly independent factors contribute to the overall incidence of ascites, attempts to manage ascites can be confusing unless we are able to focus on a unifying strategy. (
  • For example, respiratory exposure to E. coli can amplify the incidence of ascites five-fold in broilers. (
  • Feed restriction is only of economic benefit when the incidence of ascites is very severe. (
  • Those with ascites due to cancer (peritoneal carcinomatosis) may complain of chronic fatigue or weight loss. (
  • Increased portal venous pressure, Hypoproteinaemia, Chronic peritoneal inflammation and infection, Leakage of lymphatic fluid , can be the major causes of ascites. (
  • Chronic pancreatitis can result in malnutrition, leading to decreased total body protein, loss of oncotic pressure, and ascites. (
  • Ascites tends to occur in long-standing (chronic) rather than in short-lived (acute) liver disorders. (
  • Ascites may occur in other liver disorders, such as severe alcoholic hepatitis without cirrhosis, chronic hepatitis, and obstruction of the hepatic vein ( Budd-Chiari syndrome ). (
  • Mild ascites is hard to notice, but severe ascites leads to abdominal distension . (
  • A very hard drinker, was attacked with a severe hæmoptoe, which was followed by ascites and anasarca. (
  • This treatment is used most commonly when the ascites is severe or recurrent. (
  • Cardiac abnormalities (eg, congestive failure, physiologic right-side heart obstruction, severe valvular regurgitation) may result in ascites. (
  • In the case of severe ascites, where the individual does not respond to medical treatment, more drastic measures may be necessary. (
  • But for severe ascites the fluid may have to be removed with a needle. (
  • It is difficult to diagnose ascites infection early in cirrhotic patients. (
  • The efficacy of procalcitonin(PCT), c-reactive protein (CRP), white blood cell (WBC), mean fluorescence intensity of mature neutrophils(sNFI) and difference in hemoglobin concentration between newly formed and mature red blood cells(dCHC) for diagnosing ascites infection was examined. (
  • The scoring system was analyzed by logistic regression analysis to determine which parameters were statistically different between ascites infection and non-ascites infection patients. (
  • Receiver operating characteristic curve (ROC) was used to analyze the diagnostic ability of bioscore for ascites infection. (
  • The composite markers of combining PCT, dCHC and sNFI could be a valuable diagnostic score to early diagnose ascites infection in patients with cirrhosis. (
  • However, in some studies lesions of salmonellosis were reported for 4 to 6 week old broilers with E. coli co-infection consequentially leading to ascites (Ganapathy et al. (
  • This infection is common among people with ascites and cirrhosis, especially alcoholics. (
  • Sometimes the ascites can be due to a bacterial infection, a condition called septic ascites. (
  • Chapter Three addresses the mechanism of malignant ascites production and covers all clinical aspects of tunneled catheters and tunneled ports including indication, contraindication, device design, technical procedure considerations and, complication. (
  • Ascites is a common complication of liver cirrhosis associated with a poor prognosis. (
  • Ascites is a condition that refers to the accumulation of fluid in the peritoneal or abdominal cavity. (
  • This increase in pulmonary arterial pressure (hypertension) triggers the accumulation of fluid in the abdominal cavity (ascites). (
  • Ascites in chickens, commonly referred to as water belly, represents a spectrum of physiological and metabolic changes leading to the excess accumulation of fluid in abdominal cavity. (
  • Ascites is defined as the physiological disorder, which is characterized by the accumulation of fluid in the body, especially in abdominal cavity. (
  • Definition and diagnostic criteria of refractory ascites and hepatorenal syndrome in cirrhosis. (
  • Combined versus sequential diuretic treatment of ascites in non-azotaemic patients with cirrhosis: results of an open randomised clinical trial. (
  • Treatment for ascites includes curing or managing the disease causing ascites, limit your fluid intake, avoid alcohol, limit the amount salt in your diet , and other lifestyle changes. (
  • Treatment for ascites depends on what's causing your condition. (
  • Your doctor may recommend liver transplant if ascites does not respond to treatment. (
  • Below is an overview about ascites in cats followed by in-depth information about the causes, diagnosis and treatment of this condition. (
  • These new observations offer the opportunity for development of new, more targeted therapies for the treatment of malignant ascites. (
  • What Is the Treatment for Ascites? (
  • The treatment for ascites can depend on the underlying reason why the fluid buildup occurred, as this should be addressed. (
  • Another aspect of treatment for ascites is altering the affected patient's diet. (
  • A low-sodium diet is a critical aspect of treatment because eating excess salt can lead to fluid retention , and thus worsening of the ascites. (
  • Mangoes are said to be beneficial in the treatment of ascites. (
  • The presence of Ascites is often suspected based on the history and physical examination- which will often give clues to the etiology and therefore the treatment. (
  • This textbook is essential, even for confirmed practitioners and contains current information regarding treatment and therapy for patients with cirrhosis and ascites, clearly and effectively presented by the top international experts within this field. (
  • 18. Medical Treatment of Ascites in Cirrhosis (Paolo Angeli and Angelo Gatta). (
  • Careful patient selection is paramount for TIPS to be successful as a treatment for ascites. (
  • Potential new treatment options for ascites include the use of various vasoconstrictors, vasopressin V(2) receptor antagonists, or the insertion of a peritoneo-vesical shunt, all of which could possibly improve the management of ascites. (
  • Treatment for ascites depends on the cause. (
  • I have heard no good news as to treatment of the ascites fluid and wondered if chemo will help assuming he is strong enough for more chemo. (
  • However, because there are so many possible causes for ascites, it is important for a veterinarian to determine the underlying cause and provide treatment accordingly. (
  • Determine the effectiveness of laparoscopic hyperthermic perfusion of mitomycin C in preventing relapse at 4 weeks post-treatment in patients with malignant ascites. (
  • However, the distinction between the two types of ascites fluid is not only the basis of diagnosis but also a prerequisite for formulating a treatment plan. (
  • Some other causes of ascites related to increased pressure gradient are congestive heart failure and advanced kidney failure due to generalized retention of fluid in the body. (
  • Ascites is the buildup of an abnormal amount of fluid inside the belly. (
  • For example, in patients with ascites, improved kidney function helps rid the body of excess sodium (salt) and prevent the buildup of fluids. (
  • Of course, optimal management practices are also very important for reducing the problem of ascites and maximising performance of broilers. (
  • Endocrine and renal ascites are rare disorders. (
  • Factors Involved in the Pathogenesis of Renal Dysfunction and Ascites in Cirrhosis. (
  • These result in renal function compromise, sodium and fluid retention, and eventual ascites (Feldman, 2006). (
  • drain ascites continually if patient has no hemodynamic instability or pre-renal failure. (
  • Over the past 30 years, ascites research largely has focused on associated systemic abnormalities such as increases in cardiac output, blood volume, renal sodium retention and reduction in total systemic vascular resistance. (
  • Rocco and Ware [ 2 ] review the two older competing hypotheses: 1) the "underfill" theory in which ascites formation is the primary event causing the systemic changes, versus 2) the "overflow" theory in which renal sodium retention is the primary event. (
  • E. W. Haeffner, C. J. K. Hoffmann, M. Stoehr and H. Scherf, Cholesterol induced growth stimulation, cell aggregation and membrane properties of ascites tumor cells in culture, Cancer Res. (
  • E. W. Haeffner, K. Kolbe, D. Schroeter and N. Paweletz, Plasma membrane heterogeneity in ascites tumor cells. (
  • E. W. Haeffner, A. Holl and D. Schroeter, Preparation of two plasma membrane fractions from ascites tumor cells by gel chromatography on Sephacryl S-1000, J. Chromatogr. (
  • Eosinophilic ascites (EA) is generally a rare finding in clinical practice. (
  • This article uses a clinical case to highlight the problem, then reviews these new concepts in the pathophysiology of malignant ascites formation. (
  • Ascites Global Clinical Trials Review, H1, 2016″ provides an overview of Ascites clinical trials scenario. (
  • This report provides top line data relating to the clinical trials on Ascites. (
  • 1. Determine presence of ascites by clinical evaluation via history and physical examination. (
  • This clinical trial report, "Ascites Global Clinical Trials Review, H2, 2018" provides an overview of Ascites clinical trials scenario. (
  • Ascites commonly is regarded as a clinical condition that can be understood in terms of classic physiological principals. (
  • This research aimed to develop a user-friendly predictive model to discriminate malignant ascites from non-malignant ascites through easy-to-obtain clinical parameters. (
  • Ascites fluid is a common clinical syndrome, which can be divided into benign and malignant ascites for various reasons. (
  • Malignant ascites is the result of cancer and account for nearly 10% of all ascites cases occurring in related to various tumors, especially breast, ovary, stomach, pancreas, and colon cancer, and presents a challenging clinical problem in some cases [ 2 ]. (
  • PURPOSE: This phase II trial is studying how well mitomycin works when given as a hyperthermic peritoneal perfusion in treating patients with malignant ascites. (
  • Ascites usually occurs when the liver stops working properly. (
  • There are a variety of diseases that can cause the fluid to accumulate and the reasons that the ascites occurs may be different for each disease. (
  • Peritoneal effusion, or ascites, occurs when fluid builds up in the abdominal cavity. (
  • Ascites occurs in many different conditions, in this case it is due to liver cirrhosis. (
  • Certain diagnostic tests must be performed for a definitive diagnosis of the underlying cause of ascites. (
  • The mathematical diagnostic model based on the five markers is a user-friendly method to differentiate malignant ascites from benign ascites with high efficiency. (
  • If the ascites develops quickly, it is often associated with profound weakness or shock. (
  • Ascites usually develops insidiously over the course of weeks. (