Arteriosclerosis Obliterans: Common occlusive arterial disease which is caused by ATHEROSCLEROSIS. It is characterized by lesions in the innermost layer (ARTERIAL INTIMA) of arteries including the AORTA and its branches to the extremities. Risk factors include smoking, HYPERLIPIDEMIA, and HYPERTENSION.Thromboangiitis Obliterans: A non-atherosclerotic, inflammatory thrombotic disease that commonly involves small and medium-sized arteries or veins in the extremities. It is characterized by occlusive THROMBOSIS and FIBROSIS in the vascular wall leading to digital and limb ISCHEMIA and ulcerations. Thromboangiitis obliterans is highly associated with tobacco smoking.Carbonic Anhydrase III: A cytosolic carbonic anhydrase isoenzyme primarily expressed in skeletal muscle (MUSCLES, SKELETAL). EC 4.2.1.-Bronchiolitis Obliterans: Inflammation of the BRONCHIOLES leading to an obstructive lung disease. Bronchioles are characterized by fibrous granulation tissue with bronchial exudates in the lumens. Clinical features include a nonproductive cough and DYSPNEA.Leg: The inferior part of the lower extremity between the KNEE and the ANKLE.Arteriosclerosis: Thickening and loss of elasticity of the walls of ARTERIES of all sizes. There are many forms classified by the types of lesions and arteries involved, such as ATHEROSCLEROSIS with fatty lesions in the ARTERIAL INTIMA of medium and large muscular arteries.Cryptogenic Organizing Pneumonia: An interstitial lung disease of unknown etiology, occurring between 21-80 years of age. It is characterized by a dramatic onset of a "pneumonia-like" illness with cough, fever, malaise, fatigue, and weight loss. Pathological features include prominent interstitial inflammation without collagen fibrosis, diffuse fibroblastic foci, and no microscopic honeycomb change. There is excessive proliferation of granulation tissue within small airways and alveolar ducts.Monckeberg Medial Calcific Sclerosis: Thickening and loss of elasticity of the walls of muscular ARTERIES due to calcification of the TUNICA MEDIA, the concentric layers of helically arranged SMOOTH MUSCLE CELLS.Encyclopedias as Topic: Works containing information articles on subjects in every field of knowledge, usually arranged in alphabetical order, or a similar work limited to a special field or subject. (From The ALA Glossary of Library and Information Science, 1983)Arterial Occlusive Diseases: Pathological processes which result in the partial or complete obstruction of ARTERIES. They are characterized by greatly reduced or absence of blood flow through these vessels. They are also known as arterial insufficiency.ArchivesIliac Vein: A vein on either side of the body which is formed by the union of the external and internal iliac veins and passes upward to join with its fellow of the opposite side to form the inferior vena cava.Femoral Vein: The vein accompanying the femoral artery in the same sheath; it is a continuation of the popliteal vein and becomes the external iliac vein.Ischemia: A hypoperfusion of the BLOOD through an organ or tissue caused by a PATHOLOGIC CONSTRICTION or obstruction of its BLOOD VESSELS, or an absence of BLOOD CIRCULATION.Bone Marrow Transplantation: The transference of BONE MARROW from one human or animal to another for a variety of purposes including HEMATOPOIETIC STEM CELL TRANSPLANTATION or MESENCHYMAL STEM CELL TRANSPLANTATION.Transplantation, Autologous: Transplantation of an individual's own tissue from one site to another site.Ficoll: A sucrose polymer of high molecular weight.Bone Marrow Cells: Cells contained in the bone marrow including fat cells (see ADIPOCYTES); STROMAL CELLS; MEGAKARYOCYTES; and the immediate precursors of most blood cells.Lower Extremity: The region of the lower limb in animals, extending from the gluteal region to the FOOT, and including the BUTTOCKS; HIP; and LEG.Centrifugation, Density Gradient: Separation of particles according to density by employing a gradient of varying densities. At equilibrium each particle settles in the gradient at a point equal to its density. (McGraw-Hill Dictionary of Scientific and Technical Terms, 4th ed)Femoral Artery: The main artery of the thigh, a continuation of the external iliac artery.Angioplasty, Balloon: Use of a balloon catheter for dilation of an occluded artery. It is used in treatment of arterial occlusive diseases, including renal artery stenosis and arterial occlusions in the leg. For the specific technique of BALLOON DILATION in coronary arteries, ANGIOPLASTY, BALLOON, CORONARY is available.TetrazolesPopliteal Artery: The continuation of the femoral artery coursing through the popliteal fossa; it divides into the anterior and posterior tibial arteries.Stents: Devices that provide support for tubular structures that are being anastomosed or for body cavities during skin grafting.Phosphodiesterase 3 Inhibitors: Compounds that specifically inhibit PHOSPHODIESTERASE 3.Platelet Aggregation Inhibitors: Drugs or agents which antagonize or impair any mechanism leading to blood platelet aggregation, whether during the phases of activation and shape change or following the dense-granule release reaction and stimulation of the prostaglandin-thromboxane system.Organophosphonates: Carbon-containing phosphonic acid compounds. Included under this heading are compounds that have carbon bound to either OXYGEN atom or the PHOSPHOROUS atom of the (P=O)O2 structure.Adenine: A purine base and a fundamental unit of ADENINE NUCLEOTIDES.Anti-HIV Agents: Agents used to treat AIDS and/or stop the spread of the HIV infection. These do not include drugs used to treat symptoms or opportunistic infections associated with AIDS.HIV Infections: Includes the spectrum of human immunodeficiency virus infections that range from asymptomatic seropositivity, thru AIDS-related complex (ARC), to acquired immunodeficiency syndrome (AIDS).Benzoxazines: OXAZINES with a fused BENZENE ring.Reverse Transcriptase Inhibitors: Inhibitors of reverse transcriptase (RNA-DIRECTED DNA POLYMERASE), an enzyme that synthesizes DNA on an RNA template.HIV-1: The type species of LENTIVIRUS and the etiologic agent of AIDS. It is characterized by its cytopathic effect and affinity for the T4-lymphocyte.Glucose Tolerance Test: A test to determine the ability of an individual to maintain HOMEOSTASIS of BLOOD GLUCOSE. It includes measuring blood glucose levels in a fasting state, and at prescribed intervals before and after oral glucose intake (75 or 100 g) or intravenous infusion (0.5 g/kg).Hydroxymethylglutaryl-CoA Reductase Inhibitors: Compounds that inhibit HMG-CoA reductases. They have been shown to directly lower cholesterol synthesis.Diabetes Mellitus: A heterogeneous group of disorders characterized by HYPERGLYCEMIA and GLUCOSE INTOLERANCE.Diabetes Mellitus, Type 2: A subclass of DIABETES MELLITUS that is not INSULIN-responsive or dependent (NIDDM). It is characterized initially by INSULIN RESISTANCE and HYPERINSULINEMIA; and eventually by GLUCOSE INTOLERANCE; HYPERGLYCEMIA; and overt diabetes. Type II diabetes mellitus is no longer considered a disease exclusively found in adults. Patients seldom develop KETOSIS but often exhibit OBESITY.Diabetes Mellitus, Type 1: A subtype of DIABETES MELLITUS that is characterized by INSULIN deficiency. It is manifested by the sudden onset of severe HYPERGLYCEMIA, rapid progression to DIABETIC KETOACIDOSIS, and DEATH unless treated with insulin. The disease may occur at any age, but is most common in childhood or adolescence.QuinolinesBlood Glucose: Glucose in blood.Burning Mouth Syndrome: A group of painful oral symptoms associated with a burning or similar sensation. There is usually a significant organic component with a degree of functional overlay; it is not limited to the psychophysiologic group of disorders.Pruritus Vulvae: Intense itching of the external female genitals.Diagnostic Errors: Incorrect diagnoses after clinical examination or technical diagnostic procedures.Hypesthesia: Absent or reduced sensitivity to cutaneous stimulation.Sensation: The process in which specialized SENSORY RECEPTOR CELLS transduce peripheral stimuli (physical or chemical) into NERVE IMPULSES which are then transmitted to the various sensory centers in the CENTRAL NERVOUS SYSTEM.Cellulitis: An acute, diffuse, and suppurative inflammation of loose connective tissue, particularly the deep subcutaneous tissues, and sometimes muscle, which is most commonly seen as a result of infection of a wound, ulcer, or other skin lesions.Selenoprotein P: An extracellular selenoprotein that contains most of the SELENIUM in PLASMA. Selenoprotein P functions as an antioxidant and appears to transport selenium from the LIVER to peripheral tissues.Search Engine: Software used to locate data or information stored in machine-readable form locally or at a distance such as an INTERNET site.Databases, Genetic: Databases devoted to knowledge about specific genes and gene products.Genome, Human: The complete genetic complement contained in the DNA of a set of CHROMOSOMES in a HUMAN. The length of the human genome is about 3 billion base pairs.P-Selectin: Cell adhesion molecule and CD antigen that mediates the adhesion of neutrophils and monocytes to activated platelets and endothelial cells.Silk: A continuous protein fiber consisting primarily of FIBROINS. It is synthesized by a variety of INSECTS and ARACHNIDS.Antibodies: Immunoglobulin molecules having a specific amino acid sequence by virtue of which they interact only with the ANTIGEN (or a very similar shape) that induced their synthesis in cells of the lymphoid series (especially PLASMA CELLS).

Therapeutic angiogenesis induced by human recombinant hepatocyte growth factor in rabbit hind limb ischemia model as cytokine supplement therapy. (1/58)

Hepatocyte growth factor (HGF) exclusively stimulates the growth of endothelial cells without replication of vascular smooth muscle cells and acts as a survival factor against endothelial cell death. Therefore we hypothesized that a decrease in local vascular HGF might be related to the pathogenesis of peripheral arterial disease. We initially evaluated vascular HGF concentration in the vessels of patients with arteriosclerosis obliterans. Consistent with in vitro findings that hypoxia downregulated vascular HGF production, vascular HGF concentration in the diseased segments of vessels from patients with arteriosclerosis obliterans was significantly decreased as compared with disease-free segments from the same patients (P<0.05), accompanied by a marked reduction in HGF mRNA. On the other hand, a novel therapeutic strategy for ischemic diseases that uses angiogenic growth factors to expedite and/or augment collateral artery development has recently been proposed. Thus in view of the decreased endogenous vascular HGF, rhHGF (500 micrograms/animal) was intra-arterially administered through the internal iliac artery of rabbits in which the femoral artery was excised to induce unilateral hind limb ischemia, to evaluate the angiogenic activity of HGF, which could potentially have a beneficial effect in hypoxia. Administration of rhHGF twice on days 10 and 12 after surgery produced significant augmentation of collateral vessel development on day 30 in the ischemic model as assessed by angiography (P<0.01). Serial angiograms revealed progressive linear extension of collateral arteries from the origin stem artery to the distal point of the reconstituted parent vessel in HGF-treated animals. In addition, we examined the feasibility of intravenous administration of rhHGF in a moderate ischemia model. Importantly, intravenous administration of rhHGF also resulted in a significant increase in angiographic score as compared with vehicle (P<0.01). Overall, a decrease in vascular HGF might be related to the pathogenesis of peripheral arterial disease. In the presence of decreased endogenous HGF, administration of rhHGF induced therapeutic angiogenesis in the rabbit ischemic hind limb model, as potential cytokine supplement therapy for peripheral arterial disease.  (+info)

Interleukin-10 (IL-10) augments allograft arterial disease: paradoxical effects of IL-10 in vivo. (2/58)

Interleukin-10 (IL-10) is an anti-inflammatory helper T cell type 2 (Th2) cytokine that modulates Th1-type cytokine production. Graft arterial disease (GAD) is a vascular obliterative process mediated via the Th1 cytokine interferon-gamma (IFN-gamma); allografts in IFN-gamma-deficient animals do not develop GAD. We investigated the effect of IL-10 and anti-IL-10 on GAD in murine heart transplants and whether anti-IL-10 reestablishes GAD in IFN-gamma-deficient hosts. Major histocompatibility complex class II-mismatched hearts were transplanted for 8 weeks into wild-type or IFN-gamma-deficient mice. In one set of experiments, wild-type hosts received daily administration of phosphate-buffered saline (PBS) or increasing IL-10; in a subsequent set of experiments, wild-type hosts received weekly PBS, rat IgG, or anti-IL-10 monoclonal antibody; IFN-gamma-deficient recipients received weekly PBS or anti-IL-10 monoclonal antibody. Explanted allografts were assessed for parenchymal rejection and GAD, cytokine profiles, and adhesion/costimulatory-molecule expression. Exogenous IL-10 resulted in increased Th2-like cytokine production; nevertheless, it exacerbated parenchymal rejection and GAD and increased CD8(+) infiltration. Anti-IL-10 did not significantly affect the extent of rejection or GAD, cytokine profiles, or immunohistology of the allografts in wild-type hosts. Adhesion molecule (CD54 and CD106) expression was not diminished by IL-10 treatment, and costimulatory-molecule (CD80 and CD86) expression was augmented by administration of exogenous IL-10. Allografts in IFN-gamma-deficient recipients showed mild rejection and no GAD, regardless of anti-IL-10 treatment. IL-10 in vivo thus has markedly different effects than predicted from in vitro experience. Although allografts develop Th2-like cytokine profiles treatment with IL-10 causes exacerbated rejection and GAD.  (+info)

Ultrasonographic demonstration of manipulation-related aortic injuries after cardiac surgery. (3/58)

OBJECTIVES: This study was performed to evaluate the frequency and risk factors associated with new aortal lesions induced by surgical manipulation and their correlation with postoperative stroke. BACKGROUND: Little is known about the causative mechanism of intraoperative atheroembolism after cardiac surgery. METHODS: Epiaortic echocardiography was performed before cannulation and after decannulation in 472 patients undergoing cardiac surgery with extracorporeal circulation. RESULTS: A new lesion in the ascending aortal intima was identified in 16 patients (3.4%) after decannulation. New lesions were severe, with mobile lesions or disruption of the intima in 10 patients. Six of the severe lesions were related to aortic damping and the other four to aortic cannulation. Three patients in this group had postoperative stroke. Univariate analysis identified only the maximal thickness of the atheroma near the aorta manipulation site as a predictor of new lesions. The incidence of new lesions was 11.8% if the atheroma was approximately 3 to 4 mm thick and as high as 33.3% if the atheroma was >4 mm, but only 0.8% when it was <3 mm. Total 10 patients (2.1%) sustained neurological complications. Arteriosclerosis obliterans, atherosclerosis of the aorta and new mobile lesions were identified as predictors of strokes. CONCLUSIONS: This study demonstrated an association between new lesions created by surgical maneuvers and postoperative stroke. Embolic strokes were more likely to occur if new lesions were complicated with intimal disruption, especially of the mobile type. Modifications in surgical procedures will be needed if thick plaque (especially >4 mm) is noted near the manipulation site.  (+info)

Postoperative delirium in patients with chronic lower limb ischaemia: what are the specific markers? (4/58)

OBJECTIVES: we determined the incidence and specific markers of postoperative delirium in elderly patients with chronic lower limb ischaemia. PATIENTS AND METHODS: since April, 1995, 110 patients aged 60 years or older (mean: 71.6+/-6.6 years) who underwent bypass surgery were assessed regarding aetiological factors of delirium: age, sex, dementia, body-mass index, hypertension, diabetes, cerebral disease, laboratory test results, severity of limb ischaemia, type of arterial occlusion, operative time, and blood transfusion. RESULTS: discriminant analysis showed statistical significance in the following five variables: age >/=70 years; critical limb ischaemia (and/or ankle pressure <40 mmHg); dementia; duration of operation >/=7 hours; low serum albumin. The overall percentage of cases correctly classified was 78.2% (Wilks>> Lambda=0.695, p<0.001); the standardized regression coefficients of the five variables were 0.648, 0.500, 0.329, 0.218, and 0.200, respectively. In logistic regression, the regression coefficients for old age and critical limb ischaemia were 2.646 (14.1 of odds ratio; 95% confidence interval, 2.7-72.0) and 1.337 (3.8; 1.3-10.9), respectively. CONCLUSIONS: the incidence of postoperative delirium in elderly patients with chronic lower-limb ischaemia was as high as 42.3%, and an age of over 70 years and critical limb ischaemia were identified as specific markers, with 14.1 times and 3.8 times the odds of suffering from delirium after bypass surgery.  (+info)

Usefulness of the Symphony Nitinol Stent for arteriosclerosis obliterans. (5/58)

The Symphony Peripheral Stent is a self-expanding stent made of thermal memory Nitinol wire. Stents were implanted in 39 lesions of 32 patients (26 men, 6 women) with atherosclerosis obliterans (ASO). The ankle-arm index (AAI), and vessel diameters evaluated by quantitative angiography were compared before and 6 months after treatment. Symphony Peripheral Stent implantation significantly improved the AAI from 0.50+/-0.4 to 0.9+/-0.2 (p<0.01), the minimum lumen diameter (MLD) from 2+/-1.5 to 5+/-1.4 mm (p<0.01) and percent diameter stenosis (% DS) from 69+/-20% to 16.5+/-8% (p<0.01). Re-evaluation of 33 of the 39 lesions 6 months after treatment revealed a low restenosis rate of 15%, an AAI of 0.8+/-0.3, MLD of 4.5+/-2 mm and %DS of 30+/-22%, so the Symphony Peripheral Stent is thus a promising choice for patients with ASO.  (+info)

Infrainguinal arterial reconstruction for limb salvage in patients with end-stage renal disease. (6/58)

OBJECTIVES: to evaluate the efficacy of infrainguinal bypass for limb-threatening ischaemia in patients with end-stage renal disease (ESRD). MATERIALS AND METHODS: from 1991 through 2000, 28 limbs in 22 patients with ESRD received 33 infrainguinal bypasses, while 65 limbs in 57 patients with functioning kidneys underwent 77 bypasses for limb salvage. The prevalence of diabetes is higher in the ESRD group (p = 0.03). RESULTS: perioperative mortality and patient survival rate in the follow-up period were significantly poorer in patients with ESRD (18% vs 0%; p = 0.001, and 45% vs 85%, p < 0.001, respectively). Most causes of death were related to atherosclerosis or respiratory diseases. In spite of no significant difference in 2-year primary and secondary graft patency rates and limb salvage between the ESRD and non-ESRD groups (76% vs 83%; p = 0.12, 85% vs 91%; p = 0.06, and 83% vs 93%; p = 0.06, respectively), two cases of early limb loss occurred as a result of uncontrolled infection in the ESRD group. In contrast to autogenous conduits, nonautogenous conduits revealed a poorer outcome in ESRD patients (p = 0.03). CONCLUSIONS: perioperative mortality and patient survival rate were significantly poorer in the ESRD group. Preoperative full evaluation of myocardial and brain ischaemia, revascularisation with autogenous conduits, appropriate treatment of wound infection, and strict follow-up for accompanying diseases may be needed in these patients.  (+info)

The effects of Chinese medicines on micro-circulation of nail folds and blood flow velocity of limbs in the patients with arteriosclerotic obliteration. (7/58)

The effects of Chinese medicines on micro-circulation and blood flow velocity in arteries of the lower limbs were observed in 33 patients with arteriosclerotic obliteration (ASO). The results showed that the integral values of micro-circulation after treatment were obviously higher than those before treatment (P < 0.05 or P < 0.01). Blood flow velocity in arteries of the lower limbs determined with the color Doppler ultrasound detecting method after treatment were also obviously higher than those before treatment (P < 0.05 or P < 0.01). In this series of 33 ASO patients, the markedly effective rate was 36.36%, and the total effective rate was 63.64%. The observation indicates that the recipe prescribed according to the principle of supplementing qi and activating blood circulation can effectively improve micro-circulation in ASO patients, and accelerate blood flow in arteries of the lower limbs.  (+info)

Serum concentrations of vascular endothelial growth factor and monocyte-colony stimulating factor in peripheral arterial disease. (8/58)

Vascular endothelial growth factor (VEGF) strongly promotes angiogenesis, and monocyte-colony stimulating factor (M-CSF) regulates the differentiation, proliferation, and survival of monocytes. Both VEGF and M-CSF are expressed in atherosclerotic lesions. The present study was performed to clarify the role of VEGF and M-CSF in the development of peripheral artery disease (PAD). The serum VEGF and M-CSF concentrations were determined in patients with arteriosclerosis obliterans (ASO) and thromboangitis obliterans (TAO). In both patient groups the serum VEGF concentrations were significantly higher than those in the control subjects. In contrast, the serum M-CSF concentrations in the ASO patients were significantly higher than those in both the TAO patients and control subjects, but there were no differences in the M-CSF concentrations between the TAO patients and control subjects. There was no correlation between the serum concentrations of VEGF and M-CSF. In conclusion, the serum VEGF concentration was increased in ASO and TAO patients, but increased concentration of M-CSF was seen only in ASO patients. These results may reflect a difference between ASO and TAO in disease pathogenesis.  (+info)

Arteriosclerosis obliterans is an occlusive arterial disease most prominently affecting the abdominal aorta and the small- and medium-sized arteries of the lower extremities, which may lead to absent dorsalis pedis, posterior tibial, and/or popliteal artery pulses. It is characterized by fibrosis of the tunica intima and calcification of the tunica media. Arteriosclerosis Monckebergs arteriosclerosis Skin lesion James, William D.; Berger, Timothy G.; et al. (2006). Andrews Diseases of the Skin: clinical Dermatology. Saunders Elsevier. ISBN 0-7216-2921-0 ...
Semantic Scholar extracted view of Follow-up studies of iliofemoral arterial reconstruction in arteriosclerosis obliterans. by Sarah Hoye et al.
An increase of IMT of the common carotid artery is related to increased prevalence of myocardial infarction, angina, cerebrovascular disease, and peripheral vascular disease19 20 and is also associated with an increased risk of stroke and myocardial infarction.21 22 Previous studies have suggested that DM2 23 and insulin24 are related to increased stiffness of the arterial wall in humans. DM and mean fasting glucose level have been found to be positively associated with increased IMT of the common carotid artery but not IMT of the internal carotid artery. This pattern is different from hypercholesterolemia, which is related to plaque in the internal carotid arteries.4 DM is one of the most important risk factors for coronary artery disease, arteriosclerosis obliterans, and stroke.1 Many studies are focused on the prevention of coronary artery disease, arteriosclerosis obliterans, and stroke modulating IM thickening and plaques.12 13 25 Touboul et al26 reported that the risk of brain infarction ...
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The histopathologic process of CTO formation has not been well understood. It is generally considered that CTO starts with occlusion of the artery with thrombus, followed by replacement with collagen formation and deposition of calcium.4 Although this theory is widely believed, it is difficult to prove in vivo, and the previous histologic study reported no fresh thrombus in CTO lesions.1 Angioscopy enables direct visualization of the lumen,2 which makes it useful for the diagnosis of thrombus, but it cannot reach the distal side of CTO lesions in coronary arteries. Therefore, we decided to examine it in the femoral artery of patients with arteriosclerosis obliterans, and to our knowledge, this is the first reported angioscopic finding of the distal side of CTO lesions. In both of our cases, multiple thrombi of variable nature were identified with patchy distribution on the distal side, but not on the proximal side, of the CTO lesions. Those thrombi could not be identified with angiography. There ...
The stiffening of the arteries does not take place all of a sudden. It develops over a period of time. High Blood Pressure, High Cholesterol, Obesity, chronic Kidney Disease and Diabetes tend to cause Arteriosclerosis. Irritants like nicotine and similar drugs and allergic reactions also facilitate the clogging of arteries. Besides, increasing age and a family history of heart disease can be held responsible for developing blockage in the arteries, especially the coronary arteries. Excessive consumption of alcohol and smoking can also cause Arteriosclerosis.. Symptoms of Arteriosclerosis. The symptoms of the disease are generally not dominant until the arteries become so clogged that they fail to supply sufficient amount of blood to the essential organs. The nature of symptoms depends on the location of the clogged arteries. Blockage of coronary arteries tends to result in chest pain or the symptoms of heart attack. Blockage in the carotid artery in the neck may lead to sudden weakness, ...
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Arteriosclerosis is the hardening, loss of elasticity and thickening of the walls of the arteries, leading to the gradual restriction of the flow of blood to the tissues and organs, and this can cause serious health risks through atherosclerosis, which is a type of arteriosclerosis which takes place through the buildup of cholesterol, fatty plaques, and some other substances on and in the artery walls. This is the forum for discussing anything related to this health condition
Arteriosclerosis is an actual hardening, thickening, or loss of walls of the blood vessels. Atherosclerosis is an actual type of arteriosclerosis. Fatty substances, cholesterol, cellular waste, calcium & fibrin build up in the inner lining of an artery. These substances actually make up plaque.
With the help of this arteriosclerosis model with cross section of artery doctors can explain changes in the blood vessels due to arteriosclerosis.
Different forms of arteriosclerosis are the primary causes of heart disease and strokes. In fact, some studies show that in the United States and many other Westernized societies, arteriosclerosis is considered to be the underlying ... Read More ...
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Clínica e Investigación en Arteriosclerosis is the official journal of the Spanish Society of Arteriosclerosis, the Latinoamerican...
The Arteriosclerosis, Thrombosis, and Vascular Biology document template can be used to prepare manuscripts according to the citation style and authoring guidelines of Arteriosclerosis, Thrombosis, and Vascular Biology
Arteriosclerosis of peripheral extremities (native arteries) with ulceration of the ankle and gangrene. Is the correct way to code 440.20, 440.24
[toc]Sometimes the arteries become thick and hard due to the deposition of too much plaque around the walls of the artery. Such a condition is known as
... is the result of thickening and hardening of the walls of the arteries in the brain. Symptoms of cerebral arteriosclerosi...
Arteriosclerosis is due to the fat accumulated in the artery walls. You can find best priced medications at our pharmacy for this slow disease.
Læs om International Symposium: State of Prevention and Therapy in Human Arteriosclerosis and in Animal Models (Abhandlungen Der Rheinisch westfalischen Akademie Der Wissenschaften) - Unter der Schirmherrschaft der Rheinisch-Westfalischen Akademie der Wissenschaften. Udgivet af Vs Verlag Fur Sozialwissenschaften. E-bogens ISBN er 9783663067542, køb den her
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div class=citation vocab=http://schema.org/,,i class=fa fa-external-link-square fa-fw,,/i, Data from ,span resource=http://link.hays.selco.info/resource/FakJafNGU1w/ typeof=Enumeration http://bibfra.me/vocab/lite/Topic,,span property=name http://bibfra.me/vocab/lite/label,,a href=http://link.hays.selco.info/resource/FakJafNGU1w/,Arteriosclerosis,/a,,/span, - ,span property=offers typeOf=Offer,,span property=offeredBy typeof=Library ll:Library resource=http://link.hays.selco.info/,,span property=name http://bibfra.me/vocab/lite/label,,a property=url href=http://link.hays.selco.info/,Hayfield Community School,/a,,/span,,/span,,/span,,/span,,/div ...
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Eberhard Roeseler, Ulrich Julius, Franz Heigl, Ralf Spitthoever, Dennis Heutling, Paul Breitenberger, Josef Leebmann, Walter Lehmacher, Pia R. Kamstrup, Børge G. Nordestgaard, Winfried Maerz, Asma Noureen, Konrad Schmidt, Florian Kronenberg, Andreas Heibges, Reinhard Klingel and for the Pro(a)LiFe-Study Group ...
Magnesium is undoubtedly the most essential element in the prevention and treatment of heart disease and strokes. It has been established that people suffering from heart problems have very low… Read more ». ...
Sclerosis refers to any fibrous degeneration of tissue or organ. With age, the tissues that make up the arteries lose their elasticity and become more rigid.
Cavin K. Ward-Caviness, William E. Kraus, Colette Blach, Carol S. Haynes, Elaine Dowdy, Marie Lynn Miranda, Robert Devlin, David Diaz-Sanchez, Wayne E. Cascio, Shaibal Mukerjee, Casson Stallings, Luther A. Smith, Simon G. Gregory, Svati H. Shah, Lucas M. Neas and Elizabeth R. Hauser ...
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A visual score shot in image by image format with a single Super 8 reel, the film is an evocation of the arteriosclerosis of the authors grandfather, the decay of a loved one.. ...
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Workshop 5: Arteriosclerosis: basic mechanisms and clinical consequences 122 The gap between evidence based medicine and clinical practice in ACS - the Swedish…
Looking for online definition of hypertensive arteriosclerosis in the Medical Dictionary? hypertensive arteriosclerosis explanation free. What is hypertensive arteriosclerosis? Meaning of hypertensive arteriosclerosis medical term. What does hypertensive arteriosclerosis mean?
... - Arteriosclerosis is usually preceded by atherosclerosis, a kind of fatty infiltration in the inner lining of the blood vessel walls. The most risky places for such degeneration are the coronary vessels of the heart and the arteries of the brain. Arteriosclerosis results in the loss of elasticity of the blood vessels and a narrowing of the smaller arteries, which interferes with the free circulation of the blood.
The effects of arteriosclerosis can lead to lack of regular blood flow. Low blood flow to the brain can make you dizzy, numb, have slurred speech, and have other symptoms. Thromboses can further block the blood flow. This causes you to be at risk of getting stroke, or a heart attack.. Arteriosclerosis is a major health problem. Arteriosclerosis can be prevented by reducing risks. Risks are smoking, obesity, and low amounts of exercise. Doctors recommend dieting and exercise. Diets consist of low amounts of cholesterol and saturated fats. Some patients may take drugs that lower the amount of cholesterol in their blood.[4]. ...
Synonym of ARTERIOSCLEROSIS: ARTERIOSCLEROSIS Synonyme from Moby Thesaurus Moby Thesaurus arteriosclerosis Synonyms and related words:angina, angina pectoris, aortic insufficiency, aortic stenosis, apoplectic stroke, apoplexy, arrhythmia, atherosclerosis, atrial fibrillation, auricular fibrillation, beriberi heart, calcification, callusing, cardiac arrest, cardiac insufficiency, cardiac shock, cardiac stenos
Get information, facts, and pictures about arteriosclerosis at Encyclopedia.com. Make research projects and school reports about arteriosclerosis easy with credible articles from our FREE, online encyclopedia and dictionary.
Buergers disease, also called thromboangitis obliterans, is a rare form of vasculitis characterized by acute inflammation and clotting of the arteries and veins of the hands and feet. The inflammation can lead to blockages of the arteries of the lower portions of the arms and legs. These blockages can cause critical limb ischemia and/or claudication, rest pain and non-healing sores or ulcers, in the hands and/or feet.. Buergers disease, which occurs almost exclusively in heavy smokers, is different from peripheral arterial disease (PAD) in that it is not caused by atherosclerosis (plaque buildup in the arteries). The disease can cause pain in the hands and feet in response to exercise due to reduced blood-flow.. ...
Atherosclerosis or arteriosclerosis is hardening and narrowing of the arteries where plaque builds up inside your arteries. Find out more.
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Arteriosclerosis Cardiovascular Disease by William Ophuls available in Trade Paperback on Powells.com, also read synopsis and reviews.
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Atherosclerosis (arteriosclerosis) is a term used to describe a thickening, hardening and loss of elasticity in arterial walls. Atherosclerosis is the most common form of arteriosclerosis and involves progressive degeneration of the inner lining of the arteries (especially where arteries bifurcate) … Continue reading →. ...
So what is the best exercise following a stent? Answering the question what is the best exercise following a stent really. Read more ...
Fatigue is a term used to describe an overall feeling of tiredness or lack of energy. It isnt the same as simply feeling drowsy or sleepy. When youre fatigued, you have no motivation and no energy. Being sleepy may be a symptom of fatigue, but i ...
Our mission is to build healthier lives, free of cardiovascular diseases and stroke. That single purpose drives all we do. The need for our work is beyond question. Find Out More about the American Heart Association. ...
Our mission is to build healthier lives, free of cardiovascular diseases and stroke. That single purpose drives all we do. The need for our work is beyond question. Find Out More about the American Heart Association. ...
A method for guiding replacement cells to diseased vascular segments using nanoparticles. The scientists demonstrated in mice that the fresh cells actually exert their curative effect in these segments.
Sbb dia banyak masa terluang dia tolong Aso kt Biology Club.. Sejak tu la dia rapat. Aso ni pn sedih gak sbb kehilangan abang dia. Abang dia lemas masa dorg sama² memancing. Ayah Aso seorg doktor & dia nk Aso jd doktor gak. Tp Aso ni jenis keras kepala, x macam abg dia. Bila Aso kawan ngan Aya, ayah Aso x setuju sbb dia rase Aso ni terlalu muda & x matang. Die xnak nnt, bila saat Aso da bosan jaga Aya (sbb keadaan dia meneruk), Aso tinggalkan Aya, pasti yg rasa terluka tu nnt Aya sdr. Tapi Aso x peduli sbb dia mmg kawan yg baik... Sebab Aya, Aso pilih nk jadi doktor w/pn die masih rasa usaha doktor tu semua sia². Sbb xde doktor berjaya ubati Aya pn.. Aso la kawan yg sentiasa ade di sisi Aya. ...
Sbb dia banyak masa terluang dia tolong Aso kt Biology Club.. Sejak tu la dia rapat. Aso ni pn sedih gak sbb kehilangan abang dia. Abang dia lemas masa dorg sama² memancing. Ayah Aso seorg doktor & dia nk Aso jd doktor gak. Tp Aso ni jenis keras kepala, x macam abg dia. Bila Aso kawan ngan Aya, ayah Aso x setuju sbb dia rase Aso ni terlalu muda & x matang. Die xnak nnt, bila saat Aso da bosan jaga Aya (sbb keadaan dia meneruk), Aso tinggalkan Aya, pasti yg rasa terluka tu nnt Aya sdr. Tapi Aso x peduli sbb dia mmg kawan yg baik... Sebab Aya, Aso pilih nk jadi doktor w/pn die masih rasa usaha doktor tu semua sia². Sbb xde doktor berjaya ubati Aya pn.. Aso la kawan yg sentiasa ade di sisi Aya. ...
Hypertension and/or arteriosclerosis are often called the silent killers, as most people dont realize that they have a problem
Occasionally, in an aorta diseased by arteriosclerosis, a tear de-velops in the intima or the media degenerates, resulting in a dis-section .
If an shop beitrag zur kritik der verfahren vorbestimmter is endothelial or unique to establish the energy, the injury must do stored by a rapid safety. If the reliability causes no Arteriosclerosis or clinical ©, highly the collagen may avoid concerned by any
Arteriosclerosis is a type of coronary artery disease that causes hardening of the arteries. It causes the arteries to experience a loss in elasticity and an increase in stiffness. Hardened arterial walls will cause the blood to move slower through the arteries, while also increasing the chance of damage by high blood pressure. Serious cases of arteriosclerosis may require heart surgery. If left untreated, arteriosclerosis may cause heart attacks and death. Often times, arteriosclerosis is used interchangeably with the term atherosclerosis. However, these are two very different types of coronary artery disease. By definition, atherosclerosis is simply the buildup of plaque in the arteries. Just like arteriosclerosis causes the arterial walls to harden and become stiff, plaque buildups from atherosclerosis also cause a loss of elastin, stiffness, and thickening in the walls of the arteries.. While it is possible for a person to have hardened arteries (arteriolosclerosis) without having arterial ...
Nicolle, A P and Sampedrano, C C and Fontaine, J J and Tessier-Vetzel, D and Goumi, V and Pelligand, L and Pouchelon, J L and Chetboul, V (2005) Longitudinal left ventricular myocardial dysfunction assessed by 2D colour tissue Doppler imaging in a dog with systemic hypertension and severe arteriosclerosis. JOURNAL OF VETERINARY MEDICINE SERIES A-PHYSIOLOGY PATHOLOGY CLINICAL MEDICINE, 52 (2). pp. 83-87. Full text not available from this repository ...
It is probable that the process characteristic of thrombo-angiitis obliterans involves vessels in areas other than the extremities more commonly than is believed. Such involvement may only be recognized by careful clinical and pathologic investigation. We have not had an opportunity to examine cases of thrombo-angiitis obliterans after death. Necropsy was performed in seven cases recorded in the literature. Evidence of disease of the coronary arteries was found in four. A summary of these four cases follows:. Case reported by Buerger.-A man aged twenty-four years with thrombo-angiitis obliterans died suddenly following a Gritti-Stokes amputation of the right thigh. He had ...
Buergers Disease is the inflammation of blood vessels. There are many other names for Buergers Disease. Read all about Buergers Disease at patient.co.uk
Adhesion molecules play a crucial role in transplant rejection in regulating the interaction of inflammatory cells with cells in the vascular wall. In an aortic transplantation model, we have previously analysed the early adhesion process (7.5 min to 24 h) and the impact of cold ischaemia time (1-24 h) upon transplant arteriosclerosis during the first 2 months after transplantation in the rat. The aim of this investigation was to study adhesion molecules in accelerated transplant arteriosclerosis in a rat model by analysing the immunohistochemical expression of CD11b and ICAM-1 up to 2 months and followed by a semiquantitative evaluation and multivariant analysis. Antigen expression of CD11b and ICAM-1 adhesion molecules was stronger in the aortic allografts than in the ischaemia-induced syngeneic aortic grafts in the whole vessel wall. Neither ICAM-1 nor CD11b antigen expression correlated significantly with time periods of ischaemia/reperfusion injury in allogeneic or syngeneic aortic ...
Atherosclerosis Diagnosis. Atherosclerosis, also known as arteriosclerotic vascular disease, is a medical condition in which arteries begin to narrow because of an excessive amount of plaque surrounding the artery wall. This accumulation can interrupt the bodys usual blood flow, leading to dangerous possibilities in an individuals cardiovascular system. Though atherosclerosis can affect a variety of arteries, the bigger high-pressure arteries are most often influenced by this vascular condition.. Due to sometimes similar definitions, atherosclerosis and arteriosclerosis are often used interchangeably. There is a subtle difference between the two terms. Arteriosclerosis refers to the artery walls becoming hardened while atherosclerosis is defined as the artery becoming narrow due to an accumulation of plaque. Those who have atherosclerosis also have arteriosclerosis, but those with arteriosclerosis do not always have atherosclerosis.. Possible Signs of Atherosclerosis. Initial signs of ...
Budding Winter Press Release (Draft V1). So last two posts were all about healing arteriosclerosis and atherosclerosis with some Chinese recipe of some very convenient food. The extra chapter will be some habits to heal and prevent these diseases.. 1. Avoid animal fat. Even vegetarian should avoid any food that are very spicy. Of course, veggie diet would be more healthy. Over-weight patients should avoid fine rice, meat and high carbohydrates food / high sugar food. Reduce food assumption when necessary.. 2. Garlic contains vitamin B that helps reduce cholesterol and prevent heart attacks. Or eat some carrots or carrot juice before sleep.. ...
The main cause of the disease is lack of physical exercise. Excessive intake of white sugar, refined foods and a high fat diet rich in cholesterol are also causes of arteriosclerosis. The hardening of the arteries may also be caused by other diseases such as high blood pressure , obesity and diabetes. Emotional stress also plays an important role. Heart attacks are common when mental and emotional disturbances occurs , and in people who live sedentary lifestyle. Hereditary is a cause for this disease too.. It is very essential that the patient consults the doctor in this condition before adopting any home cure methods.. ...
Anti-cancer, lower blood sugar, lower cholesterol, improve arteriosclerosis, improve immunity, prevent cardiovascular disease, bone marrow hematopoiesis
Arteriosclerosis is a general term for the hardening of the arteries and, according to medical records, the primary cause of death in the western world.
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arteriosclerosis answers are found in the Tabers Medical Dictionary powered by Unbound Medicine. Available for iPhone, iPad, Android, and Web.
TY - JOUR. T1 - The inhibitory effect of cacao liquor crude polyphenols (CLP) on experimental arteriosclerosis with calcification in rat soft tissue.. AU - M., Horiuchi. AU - N, Osakabe. AU - T., Takizawa. AU - Y., Seyama. AU - Osakabe, Naomi. PY - 2001/3/1. Y1 - 2001/3/1. M3 - Article. VL - 47. SP - 208. EP - 212. JO - J. Health Sci.. JF - J. Health Sci.. ER - ...
Arteriosclerosis is a disease of the arteries. The arteries transport blood that has high oxygen content from the heart to the other organs. Today scientists assume that the artery walls are aggravated chemically and small excrescences, so-called atheromatous plaque, are formed. This plaque will constrict the arteries more and more until it becomes impossible to supply the organs with enough oxygen. High LDL cholesterol levels can be responsible for the chemical aggravation, for example. In the worst case scenario the consequence is a cardiac infarction or a stroke. Arteriosclerosis in very small arteries is often a result of a long-standing diabetes. Overweight and high blood pressure also contribute to the development of arteriosclerosis. The development of atheromatous plaque in the arteries is, however, reversible. It was discovered in studies with patients that endurance sports can lead to regression of the mutations in the arteries.. ...
Arteriosclerosis or "hardening of the arteries" occurs when the inside walls of an artery become thicker and less elastic. The thickening reduces the area available for blood flow. Although arteriosclerosis reveals few symptoms at first, as plaque grows, it narrows the artery and in some cases blocks it entirely. This can lead to angina and heart attacks.. Congestive Heart Failure (CHF) is often encountered in the elder care field. It occurs when the heart cannot pump out enough blood to meet the needs of the body. All forms of heart disease may eventually lead to CHF. CHF is the most common cause of death for people over the age of 65. Approximately 75% of our clients are over the age of 65, making this disease very important in the senior health care arena.. ...
Arteriosclerosis or "hardening of the arteries" occurs when the inside walls of an artery become thicker and less elastic. The thickening reduces the area available for blood flow. Although arteriosclerosis reveals few symptoms at first, as plaque grows, it narrows the artery and in some cases blocks it entirely. This can lead to angina and heart attacks.. Congestive Heart Failure (CHF) is often encountered in the elder care field. It occurs when the heart cannot pump out enough blood to meet the needs of the body. All forms of heart disease may eventually lead to CHF. CHF is the most common cause of death for people over the age of 65. Approximately 75% of our clients are over the age of 65, making this disease very important in the senior health care arena.. ...
Common. Unless you die from an accident or truma, if you die of natural causes, the most common cause of death are cardiac event or stroke or cancer. Heart attacks and strokes are secondary to arteriosclerosis. Dying during sleep when you are 100 y/o still a cardiac event or a stroke. Of about 3000 autopsies done on young soldiers, approx. 20% already showed signs of early arteriosclerosis. Read more... ...
|em||strong|Asthma is related to various cardiovascular risks and there is abundant evidence that adulthood asthma has links with cardiovascular disease. Dr Dianjianyi Sun, at Tulane Universitys School of Public |/em|...
Looking for Atheromae? Find out information about Atheromae. : see arteriosclerosis arteriosclerosis , general term for a condition characterized by thickening, hardening, and loss of elasticity of the walls of the... Explanation of Atheromae
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... is an exact form of arteriosclerosis in which an artery wall thickens as a consequence of attack and accumulation of white blood cells. The
Biomedical illustration of a cross sectional view of an artery with a stent in place due the occlusion due to plaque build up (arteriosclerosis). - Stock Image C022/4289
Arteriosclerotic Plaque development is actually a typical procedure and is contrary to the belief of several science procedures and programs. Arteriosclerotic factually known as hardening of artery. As humans age, the walls of the blood vessel also grow a bit harder and stiffer, with less flexibility. One of the very common problems of arteriosclerosis is […]. ...
Differential regulation of homocysteine transport in vascular endothelial and smooth muscle cells. Arteriosclerosis, Thrombosis, and Vascular Biology. 2007 ...
Hello, I am not strong in cardio :). I am trying understand what constitutes the use of this code. If patient has ASHD, Arteriosclerosis, Cardiomyopathy...
A new study published in the journal Arteriosclerosis, Thrombosis and Vascular Biology suggests they can also pose hazards to your health.
It is used in peripheral vascular diseases, like arteriosclerosis obliterans, Raynaud's disease, thromboangiitis obliterans ( ... Richter IH, Fogel M, Fabricant H (1951). "An evaluation of roniacol tartrate in arteriosclerosis obliterans". New York State ...
"Serum adiponectin and markers of endothelial injury in hemodialysis patients with arteriosclerosis obliterans". Clin. Exp. ...
CLBS12 (autologous CD34+ cells) for critical limb ischemia (CLI) due to arteriosclerosis obliterans (ASO). As of January 2016[ ...
Arteriosclerosis obliterans has been postulated as the cause, along with errors of the clotting and fibrinolytic pathways such ... Arteriography reveals large and medium vessel arteriosclerosis occasionally with dye within the area of tissue infarction . ...
... arteriosclerosis obliterans, thromboangiitis obliterans, diabetic microangiopathy and Raynaud's phenomenon). Ancrod has been ... forms of peripheral arterial circulatory disorders such as those resulting from years of heavy smoking and/or arteriosclerosis ...
Causes include: Thrombosis (approximately 40% of cases) Arterial embolism (approximately 40%) arteriosclerosis obliterans ... angiography can distinguish between cases caused by arteriosclerosis obliterans (displaying abnormalities in other vessels and ...
Arteriosclerosis obliterans Bier spots Blueberry muffin baby Bonnet-Dechaume-Blanc syndrome (Wyburn-Mason syndrome) Bullous ... Thromboangiitis obliterans (Buerger's disease) Thrombotic thrombocytopenic purpura (Moschcowitz syndrome) Traumatic purpura ...
... arteriosclerosis MeSH C14.907.137.126.056 --- arteriolosclerosis MeSH C14.907.137.126.114 --- arteriosclerosis obliterans MeSH ... Coronary Arteriosclerosis MeSH C14.280.647.250.285 --- coronary stenosis MeSH C14.280.647.250.285.200 --- coronary restenosis ... Coronary Arteriosclerosis MeSH C14.907.553.470.250.285 --- coronary stenosis MeSH C14.907.553.470.250.285.200 --- coronary ... thromboangiitis obliterans MeSH C14.907.940.907 --- vasculitis, central nervous system MeSH C14.907.940.907.175 --- aids ...
... is an occlusive arterial disease most prominently affecting the abdominal aorta and the small- and ... Arteriosclerosis Monckeberg's arteriosclerosis Skin lesion James, William D.; Berger, Timothy G.; et al. (2006). Andrews' ...
Anemia Cardiovascular diseases Arteriosclerosis Hypertension Pre-eclampsia (toxemia of pregnancy) Occlusive vascular disease ( ... Thromboangiitis obliterans Hereditary telangiectasia (Rendu-Osler-Weber syndrome) Cushing's disease Addison's disease Diabetes ...
Atherosclerosis arteriolosclerosis arteriosclerosis arteriosclerotic vascular disease atheroma (I71) Aortic aneurysm and ... Thromboangiitis obliterans (Buerger) (I73.8) Other specified peripheral vascular diseases Acrocyanosis Acroparaesthesia ...
"Arteriosclerosis, Thrombosis, and Vascular Biology. 23 (2): 168-175. PMID 12588755.. *^ Talhout R, Opperhuizen A, van Amsterdam ... Joyce JW (May 1990). "Buerger's disease (thromboangiitis obliterans)". Rheumatic Diseases Clinics of North America. 16 (2): 463 ... "Arteriosclerosis, Thrombosis, and Vascular Biology. 34 (3): 509-515. doi:10.1161/ATVBAHA.113.300156. PMID 24554606.. ... The usage of tobacco has also been linked to Buerger's disease (thromboangiitis obliterans) the acute inflammation and ...
IP receptor agonists have been used to treat Thromboangiitis obliterans, a disease involving blood clotting and inflammation of ... Ricciotti E, FitzGerald GA (May 2011). "Prostaglandins and inflammation". Arteriosclerosis, Thrombosis, and Vascular Biology. ...
"Arteriosclerosis, Thrombosis, and Vascular Biology. 31 (5): 986-1000. doi:10.1161/ATVBAHA.110.207449. PMC 3081099. PMID ... IP receptor agonists have been used to treat Thromboangiitis obliterans, a disease involving blood clotting and inflammation of ...
"Thromboangiitis obliterans: MedlinePlus Medical Encyclopedia". www.nlm.nih.gov. Retrieved 2015-06-23.. ... Arteriosclerosis. *Atherosclerosis *Foam cell. *Fatty streak. *Atheroma. *Intermittent claudication. *Critical limb ischemia ...
Thromboangiitis obliterans - Affects small- & medium-sized vessels that is strongly associated with tobacco products. Pain, ... arteriosclerosis and arteriolosclerosis - to narrow which arteries are diseased and can easily be confused due to similar ...
en:Arteriosclerosis (55) → 동맥경화증 *en:Asbestosis (33) → 석면폐 *en:Atherosclerosis (39). *en:Attenuated vaccine (14) → 생백신 ... en:Bronchiolitis obliterans (12). *en:Bulimia nervosa (29). *en:Cachexia (35). *en:Candidiasis (47) → 칸디다증 ...
Other uncommon causes are Trousseau disease,[medical citation needed] Beurger's disease (Thromboangiitis obliterans),[medical ... Arteriosclerosis. *Atherosclerosis *Foam cell. *Fatty streak. *Atheroma. *Intermittent claudication. *Critical limb ischemia ...
Arteriosclerosis obliterans is an occlusive arterial disease most prominently affecting the abdominal aorta and the small- and ... Arteriosclerosis Monckebergs arteriosclerosis Skin lesion James, William D.; Berger, Timothy G.; et al. (2006). Andrews ...
MediSavvy has been developed as an educational platform and learning resource for health professionals, as well as patients that are keen to learn more about their conditions. ...
Follow-up studies of iliofemoral arterial reconstruction in arteriosclerosis obliterans. by Sarah Hoye et al. ... Follow-up studies of iliofemoral arterial reconstruction in arteriosclerosis obliterans.. *. Sarah Hoye. , Roland Warren ... article{Hoye1956FollowupSO, title={Follow-up studies of iliofemoral arterial reconstruction in arteriosclerosis obliterans.}, ...
In cases of long standing, without adequate treatment, arteriosclerosis is common. It develops early in life and to a more ... THROMBOANGIITIS OBLITERANS OF PATIENTS WITH DIABETES(THROMBOANGIITIS OBLITERANS OF PATIENTS WITH DIABETES*) BAYARD T. HORTON, M ... THROMBOANGIITIS OBLITERANS OF PATIENTS WITH DIABETES(THROMBOANGIITIS OBLITERANS OF PATIENTS WITH DIABETES*). Ann Intern Med. ... The present report, with the exception of a case reported by Adams in 1930, includes all cases of thromboangiitis obliterans ...
Diabetes mellitus (DM) is an important risk factor for coronary artery disease, arteriosclerosis obliterans, and stroke.1 An ... arteriosclerosis obliterans, and stroke modulating IM thickening and plaques.12 13 25 Touboul et al26 reported that the risk of ... arteriosclerosis obliterans, and stroke.1 Many studies are focused on the prevention of coronary artery disease, ... arteriosclerosis obliterans, or stroke), or medications received (Ca2+ channel blocker, insulin, sulfonylurea, acarbose). No ...
Thromboangiitis Obliterans is a vasculitis of small and medium-sized arteries which can extend into contiguous veins and nerves ... Thromboangiitis Obliterans is characterized by an initial infiltration of the vascular wall with neutrophils. Over time, the ... Thromboangiitis Obliterans is characterized by claudication of the extremities which can be highly painful, even at rest, ... Thromboangiitis Obliterans typically occurs in smokers, especially men below the age of 40. ...
Arteriosclerosis obliterans..La Arteriosclerosis obliterante..Arteriosclerose obliterante..闭塞性动脉硬化.... ... Diabetic Obliterans..Diabéticos Obliterante..Diabéticos Obliterante..糖尿病的闭塞性脉管
Arteriosclerosis obliterans; its surgical repair. The Journal of the American Osteopathic Association, October 1958, Vol. 58, ... Arteriosclerosis obliterans; its surgical repair You will receive an email whenever this article is corrected, updated, or ... PEASE G. Arteriosclerosis obliterans; its surgical repair. J Am Osteopath Assoc 1958;58(2):103-105. doi: https://doi.org/. ...
Failure of Vasodilator Drugs in Arteriosclerosis Obliterans JAY D. COFFMAN, M.D., F.A.C.P.; JOHN A. MANNICK, M.D. ... Failure of Vasodilator Drugs in Arteriosclerosis Obliterans. Ann Intern Med. 1972;76:35-39. doi: 10.7326/0003-4819-76-1-35 ... in patients with intermittent claudication caused by aortoiliac or femoropopliteal arteriosclerosis obliterans. All drugs were ... Drug-Coated Balloon Angioplasty versus Drug-Coated Balloon Angioplasty Alone for Femoropopliteal Arteriosclerosis Obliterans. ...
Eight patients with rest pain in the lower extremity due to arteriosclerosis obliterans were given sub-dissociative doses of ... Chronic ischemic pain due to lower extremity arteriosclerosis obliterans often responds poorly to analgesics, and the pain- ... analgesic effect of racemic ketamine in patients with chronic ischemic pain due to lower extremity arteriosclerosis obliterans. ... Arteriosclerosis Obliterans/complications*. *Arteriosclerosis Obliterans/physiopathology. *Chromatography, High Pressure Liquid ...
miR-21 regulates vascular smooth muscle cell function in arteriosclerosis obliterans of lower extremities through AKT and ERK1/ ... miR-21 regulates vascular smooth muscle cell function in arteriosclerosis obliterans of lower extremities through AKT and ERK1/ ... "miR-21 regulates vascular smooth muscle cell function in arteriosclerosis obliterans of lower extremities through AKT and ERK1/ ... "miR-21 regulates vascular smooth muscle cell function in arteriosclerosis obliterans of lower extremities through AKT and ERK1/ ...
Introduction Arteriosclerosis obliterans (ASO) is a disease that affects the lower extremities. The mechanism of ASO is ... Arteriosclerosis obliterans (ASO) is a disease that affects the lower extremities. The mechanism of ASO is associated with the ... miR-21 regulates vascular smooth muscle cell function in arteriosclerosis obliterans of lower extremities through AKT and ERK1/ ... miR-21 regulates vascular smooth muscle cell function in arteriosclerosis obliterans of lower extremities through AKT and ERK1/ ...
MalaCards integrated aliases for Arteriosclerosis Obliterans:. Name: Arteriosclerosis Obliterans 12 38 56 45 15 17 74 ... MalaCards based summary : Arteriosclerosis Obliterans is related to arteriosclerosis and intermittent claudication. An ... MalaCards organs/tissues related to Arteriosclerosis Obliterans:. 42 Heart, Bone, Bone Marrow, Smooth Muscle, Skin, Spinal Cord ... Articles related to Arteriosclerosis Obliterans:. (show top 50) (show all 257) #. Title. Authors. Year. ...
Detecting vulnerable plaque of arteriosclerosis obliterans by multidetector-row computed tomography--comparing with VH (virtual ... Detecting vulnerable plaque of arteriosclerosis obliterans by multidetector-row computed tomography--comparing with VH (virtual ...
... - 11 Studies Found. Status. Study Recruiting. Study Name: A Safety/Efficacy Study of Alprostadil ... Liposomes for Injection to Treat Lower Extremity Arteriosclerosis Obliterans. Condition: Arteriosclerosis Obliterans. Date: ... Condition: Arteriosclerosis Obliterans. Date: 2008-07-08. Completed. Study Name: Study of Cilostazol and Probucol to Assess ... Condition: Arteriosclerosis Obliterans. Date: 2009-06-02. Interventions: Drug: cilostazol 200 mg/day BID. ...
Main-Services-Treatment of arteriosclerosis obliterans.. Treatment of arteriosclerosis obliterans.. RFA is a new method of ... Treatment of arteriosclerosis obliterans. Interventional cardiology Coronary artery bypass surgery Laser coagulation of the ...
Arteriosclerosis Obliterans. On-line free medical diagnosis assistant. Ranked list of possible diseases from either several ... Ruptured distal aortic arch aneurysm associated with arteriosclerosis obliterans. A 73-year-old man with a ruptured distal ... IV was performed in 4 dialysis patients against the complication of arteriosclerosis obliterans (ASO) but the result was ... Percutaneous transluminal angioplasty against arteriosclerosis obliterans in dialysis patients. The incidence of peripheral ...
Arteriosclerosis obliterans (ASO) - Image answers are found in the Evidence-Based Medicine Guidelines powered by Unbound ... Arteriosclerosis obliterans (ASO) - Image is a topic covered in the Evidence-Based Medicine Guidelines. To view the entire ... Arteriosclerosis Obliterans (ASO) - Image [Internet]. In: Evidence-Based Medicine Guidelines. Duodecim Medical Publications ... "Arteriosclerosis Obliterans (ASO) - Image." Evidence-Based Medicine Guidelines, Duodecim Medical Publications Limited, 2019. ...
PAD; Peripheral Vascular Disease; PVD; Arteriosclerosis Obliterans). by Sonja Lyons. More InDepth Information on This Condition ...
PAD; Peripheral Vascular Disease; PVD; Arteriosclerosis Obliterans). by Sonja Lyons. More InDepth Information on This Condition ...
PAD; Peripheral Vascular Disease; PVD; Arteriosclerosis Obliterans). by Sonja Lyons. Related Media: The Benefits of Regular ...
Full text: Available Index: LILACS (Americas) Main subject: Arteriosclerosis Obliterans / Lipids Limits: Animals Language: ... Animals , Rats , Arteriosclerosis Obliterans , Lipids , Inflammation , Lipids/blood , Lower Extremity , Rats , Simvastatin ... Effects of puerarin on blood lipids and inflammatory factors in rats with lower limb arteriosclerosis obliterans ... on blood lipid and inflammatory factor levels in rats with lower limb arteriosclerosis obliterans (ASO). Sixty rats were ...
a. Thromboangiitis obliterans. b. Arteriosclerosis. c. Embolism. d. Thrombosis. 4. Dysglobulinemia. a. Cold hemagglutination ...
... obliterans) (of) (senile) (with calcification) within the ICD-10 Index to Diseases and Injuries. ... Arteriosclerosis, arteriosclerotic (diffuse) (obliterans) (of) (senile) (with calcification) - I70.90 Unspecified ... peripheral (of extremities) - See: Arteriosclerosis, extremities;. *pulmonary (idiopathic) - I27.0 Primary pulmonary ...
  • The pathophysiologic effects of CRE include mucosal ulceration, epithelial atypia, ileitis cystic profunda, serosal thickening, endarteritis obliterans , intestinal fibrosis and lymphatic congestion. (thefreedictionary.com)
  • To explore the effect of long non-coding ribonucleic acid (lncRNA) H19 on the apoptosis of vascular endothelial cells in arteriosclerosis obliterans (ASO) via the nuclear factor kappa-light-chain-enhancer of activated B cells (NF-κB) pathway. (europeanreview.org)
  • In addition to evaluating the symptoms described above, angiography can distinguish between cases caused by arteriosclerosis obliterans (displaying abnormalities in other vessels and collateral circulations) from those caused by emboli. (wikipedia.org)
  • Arteriosclerosis obliterans has been postulated as the cause, along with errors of the clotting and fibrinolytic pathways such as antiphospholipid syndrome. (wikipedia.org)
  • We experienced two cases whose low back pain was improved after vascular reconstructive surgery for arteriosclerosis obliterans in the abdominal cavity . (lookfordiagnosis.com)