Arteriolosclerosis
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Arteriosclerosis Obliterans
Arteriosclerosis
beta-Thalassemia
A disorder characterized by reduced synthesis of the beta chains of hemoglobin. There is retardation of hemoglobin A synthesis in the heterozygous form (thalassemia minor), which is asymptomatic, while in the homozygous form (thalassemia major, Cooley's anemia, Mediterranean anemia, erythroblastic anemia), which can result in severe complications and even death, hemoglobin A synthesis is absent.
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Fatigue Syndrome, Chronic
A syndrome characterized by persistent or recurrent fatigue, diffuse musculoskeletal pain, sleep disturbances, and subjective cognitive impairment of 6 months duration or longer. Symptoms are not caused by ongoing exertion; are not relieved by rest; and result in a substantial reduction of previous levels of occupational, educational, social, or personal activities. Minor alterations of immune, neuroendocrine, and autonomic function may be associated with this syndrome. There is also considerable overlap between this condition and FIBROMYALGIA. (From Semin Neurol 1998;18(2):237-42; Ann Intern Med 1994 Dec 15;121(12): 953-9)
Colestipol
Pleurodynia, Epidemic
Bipolar Disorder
Attention Deficit Disorder with Hyperactivity
A behavior disorder originating in childhood in which the essential features are signs of developmentally inappropriate inattention, impulsivity, and hyperactivity. Although most individuals have symptoms of both inattention and hyperactivity-impulsivity, one or the other pattern may be predominant. The disorder is more frequent in males than females. Onset is in childhood. Symptoms often attenuate during late adolescence although a minority experience the full complement of symptoms into mid-adulthood. (From DSM-V)
Amyotrophic Lateral Sclerosis
A degenerative disorder affecting upper MOTOR NEURONS in the brain and lower motor neurons in the brain stem and SPINAL CORD. Disease onset is usually after the age of 50 and the process is usually fatal within 3 to 6 years. Clinical manifestations include progressive weakness, atrophy, FASCICULATION, hyperreflexia, DYSARTHRIA, dysphagia, and eventual paralysis of respiratory function. Pathologic features include the replacement of motor neurons with fibrous ASTROCYTES and atrophy of anterior SPINAL NERVE ROOTS and corticospinal tracts. (From Adams et al., Principles of Neurology, 6th ed, pp1089-94)
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Pharmacology, Clinical
Competitive Behavior
Cerebral Small Vessel Diseases
Video Games
CD8+ T lymphocytes regulate the arteriogenic response to ischemia by infiltrating the site of collateral vessel development and recruiting CD4+ mononuclear cells through the expression of interleukin-16. (1/10)
BACKGROUND: Previous studies have demonstrated that macrophages and CD4+ T lymphocytes play pivotal roles in collateral development. Indirect evidence suggests that CD8+ T cells also play a role. Thus, after acute cerebral ischemia, CD8+ T cells infiltrate the perivascular space and secrete interleukin-16 (IL-16), a potent chemoattractant for monocytes and CD4+ T cells. We tested whether CD8+ T lymphocytes contribute to collateral vessel development and whether the lack of circulating CD8+ T cells prevents IL-16 expression, impairs CD4+ mononuclear cell recruitment, and reduces collateral vessel growth after femoral artery ligation in CD8(-/-) mice. METHODS AND RESULTS: After surgical excision of the femoral artery, laser Doppler perfusion imaging demonstrated reduced blood flow recovery in CD8(-/-) mice compared with C57/BL6 mice (ischemic/nonischemic limb at day 28, 0.66+/-0.04 versus 0.87+/-0.04, respectively; P<0.01). This resulted in greater calf muscle atrophy (mean fiber area, 785+/-68 versus 1067+/-69 microm2, respectively; P<0.01) and increased fibrotic tissue content (10.8+/-1.2% versus 7+/-1%, respectively; P<0.01). Moreover, CD8(-/-) mice displayed reduced IL-16 expression and decreased CD4+ T-cell recruitment at the site of collateral vessel development. Exogenous CD8+ T cells, infused into CD8(-/-) mice immediately after femoral artery ligation, selectively homed to the ischemic hind limb and expressed IL-16. The restoration of IL-16 expression resulted in significant CD4+ mononuclear cell infiltration of the ischemic limb, faster blood flow recovery, and reduced hindlimb muscle atrophy/fibrosis. When exogenous CD8+ T cells deficient in IL-16 (IL-16(-/-)) were infused into CD8(-/-) mice immediately after femoral artery ligation, they selectively homed to the ischemic hind limb but were unable to recruit CD4+ mononuclear cells and did not improve blood flow recovery. CONCLUSIONS: These results demonstrate that CD8+ T cells importantly contribute to the early phase of collateral development. After femoral artery ligation, CD8+ T cells infiltrate the site of collateral vessel growth and recruit CD4+ mononuclear cells through the expression of IL-16. Our study provides further evidence of the significant role of the immune system in modulating collateral development in response to peripheral ischemia. (+info)Association between serum adiponectin levels and arteriolosclerosis in IgA nephropathy patients. (2/10)
OBJECTIVE: Adiponectin has attracted great attention because of its anti-atherogenic properties; however, to date the relationship between serum adiponectin and arteriolosclerosis has not been reported. In our study, we aimed to examine whether or not serum adiponectin levels are associated with arteriolosclerosis in patients with IgA nephropathy which is the most common form of chronic glomerulonephritis. MATERIALS AND METHODS: We enrolled 35 patients aged 35.0+/-14.6, who underwent renal biopsy from August 2004 to February 2006 in our hospital, and were confirmed to have IgA nephropathy. We examined serum adiponectin, high-sensitive C-reactive protein, total cholesterol and triglyceride level, urinary protein excretion, body mass index (BMI), and the presence of arteriolosclerosis in the renal specimens. Since the serum adiponectin level is strongly affected by renal function, we classified the patients by creatinine clearance. RESULTS: Multiple regression analysis showed the associations of adiponectin with creatinine clearance (p<0.001), BMI (p<0.001), serum triglyceride (p=0.001) and urinary protein excretion (p=0.001). We observed a positive relation of adiponectin with urinary protein excretion and an inverse relation of adiponectin with creatinine clearance, serum triglyceride, and BMI. We could not detect any relation between the presence of arteriolosclerosis and adiponectin in the IgA nephropathy patients as a whole; however, in patients whose creatinine clearance was 90-120 ml/min/1.73 m2, the serum adiponectin level of patients with arteriolosclerosis was lower than in those without arteriolosclerosis (p=0.025). CONCLUSION: The serum level of adiponectin was related to arteriolosclerosis in IgA nephropathy patients whose renal function was almost normal. Adiponectin may prevent renal arteriolosclerosis. (+info)Lead, at low levels, accelerates arteriolopathy and tubulointerstitial injury in chronic kidney disease. (3/10)
Chronic lead exposure has been epidemiologically linked with hypertension and renal disease. Clinical studies suggest that low lead levels may contribute to renal progression. However, experimental studies have not examined whether low levels of lead accelerate progression in experimental chronic renal disease. Sprague-Dawley rats were administered lead (L; 150 ppm in drinking water, n = 16) for 4 wk, followed by remnant kidney (RK) surgery with continuation of lead for an additional 12 wk; control rats (n = 9) were treated similarly but did not receive lead. Lead treatment was well tolerated and resulted in modest elevations in whole blood lead levels (26.4 +/- 4.5 vs. 1 +/- 0 mug/dl, week 16, P < 0.001). Lead treatment was associated with higher systolic blood pressure (P < 0.05) and worse renal function (creatinine clearance 1.4 +/- 0.4 vs. 1.8 +/- 0.5 ml/min, RK+L vs. RK, P < 0.05), and with a tendency for greater proteinuria (6.6 +/- 6.1 vs. 3.6 +/- 1.5 mg protein/mg creatinine, RK+L vs. RK, P = 0.08). While glomerulosclerosis tended to be worse in lead-treated rats (37.6 +/- 11 vs. 28.8 +/- 2.3%, RK+L vs. RK, P = 0.06), the most striking finding was the development of worse arteriolar disease (P < 0.05), peritubular capillary loss (P < 0.05), tubulointerstitial damage, and macrophage infiltration (P < 0.05) in association with significantly increased renal expression of monocyte chemoattractant protein-1 mRNA. In conclusion, lead accelerates chronic renal disease, primarily by raising blood pressure and accelerating microvascular and tubulointerstitial injury. (+info)Apo B/Apo A-I ratio in central and peripheral arterial diseases. (4/10)
BACKGROUND: The apo B/apo A-I ratio represents the balance between atherogenic particles, rich in apo B, and the antiatherogenic ones, apo A-I rich. This study investigated the association between atherosclerotic diseases in different anatomical sites and apo B/apo A-I ratio. METHODS: Lipids, lipoproteins, and apolipoproteins A-I and B were assessed in 30 subjects with coronary artery disease (CAD), 26 with ischemic stroke (IS), 30 with peripheral arterial obstructive disease (PAOD), and 38 healthy subjects (controls). RESULTS: HDLc and Apo A-I were significantly lower in PAOD and CAD groups, respectively, than in other groups. Significantly higher levels of triglycerides were observed for CAD and PAOD groups than for controls. Apo B was significantly higher in IS group than in control and PAOD groups. The apo B/apo A-I ratio showed significantly higher in CAD and IS groups when compared to control and PAOD groups (p < 0.001). CONCLUSION: The apo B/apo A-I ratio was important for identifying an increased trend for coronary and cerebral atherosclerosis. In spite of the increased trend for apo B/apo A-I ratio in IS and CAD groups, the studied variables cannot be considered in an isolated way, given as those parameters were analyzed together by a binary logistic regression, no association has been demonstrated. (+info)Cholesterol-lowering effect of kori-tofu protein and its high-molecular-weight fraction content. (5/10)
The serum total cholesterol concentration was significantly lower in the kori-tofu feeding group than in the soy protein isolate (SPI) group, except on the 28th day of the experiment. The high-molecular-weight fraction (HMF) content of the kori-tofu protein was significantly higher than that of SPI. This difference in the HMF content may have influenced the cholesterol-lowering effect of the protein. (+info)Chronic kidney disease, severe arterial and arteriolar sclerosis and kidney neoplasia: on the spectrum of kidney involvement in MELAS syndrome. (6/10)
(+info)Serum complement C3 predicts renal arteriolosclerosis in non-diabetic chronic kidney disease. (7/10)
AIM: Complement C3 (C3) is one of the major mediators of inflammation. Serum C3 has been shown to be correlated with the presence of atherosclerosis. We examined whether the serum C3 level might be correlated with the severity of renal arteriolosclerosis in patients with chronic kidney disease (CKD). METHODS: Non-diabetic CKD (stages 1-3) patients who underwent renal biopsy were enrolled in this study. Renal arteriolosclerosis was defined by the presence of hyaline changes and vessel wall thickening in the renal biopsy specimens. We examined whether the serum C3 level might be correlated with the severity of renal arteriolosclerosis in CKD patients. RESULTS: A total of 208 CKD patients (age 36.0+/-13.6 years; 94 male) who underwent renal biopsy were included. Univariate analysis showed that the serum C3 level was positively correlated with age, body mass index, blood pressure and the serum triglyceride, LDL cholesterol and CRP (p<0.001). The serum C3 level was also inversely correlated with serum HDL cholesterol (p<0.001). Multiple regression analysis identified that the serum C3 (p=0.043) as well as age (p<0.001), serum uric acid (p=0.009) and eGFR (p= 0.025) were independently associated with the severity of renal arteriolosclerosis. CONCLUSION: Our results suggest that the serum C3 level is a reliable marker of renal arteriolosclerosis. Components of metabolic syndrome were also correlated with the serum C3 level. Inflammation or metabolic syndrome may contribute to CKD through influencing the rate of progression of renal arteriolosclerosis. (+info)Relationship of red splenic arteriolar hyaline with rapid death: a clinicopathological study of 82 autopsy cases. (8/10)
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Arteriolosclerosis synonyms, arteriolosclerosis antonyms - FreeThesaurus.com
Arteriolosclerosis - Wikipedia
arteriolosclerosis | Tabers Medical Dictionary
Hypertensive kidney disease - Wikipedia
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Driver mutations in cancer genome of Nothobranchius furzeri: from tumor biology to concept of experimental model of spontaneous...
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Arteriosclerosis
Arteriolosclerosis, unlike atherosclerosis, is a sclerosis that only affects small arteries and arterioles, which carry ... "hyaline arteriolosclerosis". Retrieved 2015-06-19. "Angioplasty: MedlinePlus". www.nlm.nih.gov. Retrieved 2015-06-19. "Coronary ...
Arteriosclerosis - Simple English Wikipedia, the free encyclopedia
Arteriolosclerosis. This affects the body's smaller arteries.[3]. The effects of arteriosclerosis can lead to lack of regular ...
Arteriole
Arteriolosclerosis is the term specifically used for the hardening of arteriole walls. This can be due to decreased elastic ... An increase in the media to lumenal diameter ratio has been observed in hypertensive arterioles (arteriolosclerosis) as the ...
Atherosclerosis
... arteriolosclerosis is any hardening (and loss of elasticity) of arterioles (small arteries); atherosclerosis is a hardening of ... arteriolosclerosis, and atherosclerosis. Arteriosclerosis is a general term describing any hardening (and loss of elasticity) ...
Coronary arteries
... or arteriolosclerosis. This occurs when plaques (made up of deposits of cholesterol and other substances) build up over time in ...
Benign nephrosclerosis
... hyaline arteriolosclerosis). Under a microscope, this appears as a homogeneous, pink hyaline thickening at the expense of the ...
Hyaline
... arteriolosclerosis Hyaloid canal, which passes through the eye Hyalopilitic Hyaloserositis Infant respiratory distress ...
Hypertensive kidney disease
The changes in small arterioles include hyaline arteriolosclerosis (deposition of hyaline, collagenous material),[citation ... a process known as arteriolosclerosis. The resulting inadequate blood flow produces tubular atrophy, interstitial fibrosis, and ...
Outline of cardiology
... arteriosclerosis and arteriolosclerosis - to narrow which arteries are diseased and can easily be confused due to similar ...
List of MeSH codes (C14)
... arteriolosclerosis MeSH C14.907.137.126.114 - arteriosclerosis obliterans MeSH C14.907.137.126.307 - atherosclerosis MeSH ...
Arteriolosclerosis
Types include hyaline arteriolosclerosis and hyperplastic arteriolosclerosis, both involved with vessel wall thickening and ... Arteriolosclerosis is a form of cardiovascular disease involving hardening and loss of elasticity of arterioles or small ... It is a type of arteriolosclerosis, which refers to thickening of the arteriolar wall and is part of the ageing process. ... This is a type of arteriolosclerosis involving a narrowed lumen. The term "onion-skin" is sometimes used to describe this form ...
Intermittent claudication
Exercise can improve symptoms, as can revascularization.[4] Both together may be better than one intervention of its own.[4] Pharmacological options exist, as well. Medicines that control lipid profile, diabetes, and hypertension may increase blood flow to the affected muscles and allow for increased activity levels. Angiotensin converting enzyme inhibitors, adrenergic agents such as alpha-1 blockers and beta-blockers and alpha-2 agonists, antiplatelet agents (aspirin and clopidogrel), naftidrofuryl, pentoxifylline, and cilostazol (selective PDE3 inhibitor) are used for the treatment of intermittent claudication.[5] However, medications will not remove the blockages from the body. Instead, they simply improve blood flow to the affected area.[6] Catheter-based intervention is also an option. Atherectomy, stenting, and angioplasty to remove or push aside the arterial blockages are the most common procedures for catheter-based intervention. These procedures can be performed by interventional ...
Hypertensive emergency
The resulting ischemia prompts further release of vasoactive substances including prostaglandins, free radicals, and thrombotic/mitotic growth factors, completing a vicious cycle of inflammatory changes.[9] If the process is not stopped, homeostatic failure begins, leading to loss of cerebral and local autoregulation, organ system ischemia and dysfunction, and myocardial infarction.[10] Single-organ involvement is found in approximately 83% of hypertensive emergency patients, two-organ involvement in about 14% of patients, and multi-organ failure (failure of at least 3 organ systems) in about 3% of patients. In the brain, hypertensive encephalopathy - characterized by hypertension, altered mental status, and swelling of the optic disc - is a manifestation of the dysfunction of cerebral autoregulation.[7] Cerebral autoregulation is the ability of the blood vessels in the brain to maintain a constant blood flow. People who suffer from chronic hypertension can tolerate higher arterial pressure ...
Secondary hypertension
Yet another related disorder causing hypertension is glucocorticoid remediable aldosteronism, which is an autosomal dominant disorder in which the increase in aldosterone secretion produced by ACTH is no longer transient, causing of primary hyperaldosteronism, the Gene mutated will result in an aldosterone synthase that is ACTH-sensitive, which is normally not.[22][23][24][25][26] GRA appears to be the most common monogenic form of human hypertension.[27] Compare these effects to those seen in Conn's disease, an adrenocortical tumor which causes excess release of aldosterone,[28] that leads to hypertension.[29][30][31] Another adrenal related cause is Cushing's syndrome which is a disorder caused by high levels of cortisol. Cortisol is a hormone secreted by the cortex of the adrenal glands. Cushing's syndrome can be caused by taking glucocorticoid drugs, or by tumors that produce cortisol or adrenocorticotropic hormone (ACTH).[32] More than 80% of patients with Cushing's syndrome develop ...
Vertebral artery dissection
The vertebral arteries arise from the subclavian artery, and run through the transverse foramen of the upper six vertebrae of the neck. After exiting at the level of the first cervical vertebra, its course changes from vertical to horizontal, and then enters the skull through the foramen magnum. Inside the skull, the arteries merge to form the basilar artery, which joins the circle of Willis. In total, three quarters of the artery are outside the skull; it has a high mobility in this area due to rotational movement in the neck and is therefore vulnerable to trauma. Most dissections happen at the level of the first and second vertebrae. The vertebral artery supplies a number of vital structures in the posterior cranial fossa, such as the brainstem, the cerebellum and the occipital lobes. The brainstem harbors a number of vital functions (such as respiration) and controls the nerves of the face and neck. The cerebellum is part of the diffuse system that coordinates movement. Finally, the occipital ...
Intracranial aneurysm
Aneurysm means an outpouching of a blood vessel wall that is filled with blood. Aneurysms occur at a point of weakness in the vessel wall. This can be because of acquired disease or hereditary factors. The repeated trauma of blood flow against the vessel wall presses against the point of weakness and causes the aneurysm to enlarge.[12] As described by the Law of Young-Laplace, the increasing area increases tension against the aneurysmal walls, leading to enlargement.. Both high and low wall shear stress of flowing blood can cause aneurysm and rupture. However, the mechanism of action is still unknown. It is speculated that low shear stress causes growth and rupture of large aneurysms through inflammatory response while high shear stress causes growth and rupture of small aneurysm through mural response (response from the blood vessel wall). Other risk factors that contributes to the formation of aneurysm are: cigarette smoking, hypertension, female gender, family history of cerebral aneurysm, ...
Pulmonary hypertension
The pathogenesis of pulmonary arterial hypertension (WHO Group I) involves the narrowing of blood vessels connected to and within the lungs. This makes it harder for the heart to pump blood through the lungs, as it is much harder to make water flow through a narrow pipe as opposed to a wide one. Over time, the affected blood vessels become stiffer and thicker, in a process known as fibrosis. The mechanisms involved in this narrowing process include vasoconstriction, thrombosis, and vascular remodeling (excessive cellular proliferation, fibrosis, and reduced apoptosis/programmed cell death in the vessel walls, caused by inflammation, disordered metabolism and dysregulation of certain growth factors).[26][27] This further increases the blood pressure within the lungs and impairs their blood flow. In common with other types of pulmonary hypertension, these changes result in an increased workload for the right side of the heart.[12][28] The right ventricle is normally part of a low pressure system, ...
Pituitary apoplexy
The first priority in suspected or confirmed pituitary apoplexy is stabilization of the circulatory system. Cortisol deficiency can cause severe low blood pressure.[1][6] Depending on the severity of the illness, admission to a high dependency unit (HDU) may be required.[1] Treatment for acute adrenal insufficiency requires the administration of intravenous saline or dextrose solution; volumes of over two liters may be required in an adult.[6] This is followed by the administration of hydrocortisone, which is pharmaceutical grade cortisol, intravenously or into a muscle.[4][6] The drug dexamethasone has similar properties,[6] but its use is not recommended unless it is required to reduce swelling in the brain around the area of hemorrhage.[1] Some are well enough not to require immediate cortisol replacement; in this case, blood levels of cortisol are determined at 9:00 AM (as cortisol levels vary over the day). A level below 550 nmol/l indicates a need for replacement.[1] The decision on ...
Venous thrombosis
Evidence supports the use of heparin in people following surgery who have a high risk of thrombosis to reduce the risk of DVTs; however, the effect on PEs or overall mortality is not known.[27] In hospitalized non-surgical patients, mortality does not appear to change.[28][29][30] It does not appear, however, to decrease the rate of symptomatic DVTs.[28] Using both heparin and compression stockings appears better than either one alone in reducing the rate of DVT.[31] In hospitalized people who have had a stroke and not had surgery, mechanical measures (compression stockings) resulted in skin damage and no clinical improvement.[28] Data on the effectiveness of compression stockings among hospitalized non-surgical patients without stroke is scarce.[28] The American College of Physicians (ACP) gave three strong recommendations with moderate quality evidence on VTE prevention in non-surgical patients: that hospitalized patients be assessed for their risk of thromboembolism and bleeding before ...
Hypertensive heart disease
... includes a number of complications of high blood pressure that affect the heart. While there are several definitions of hypertensive heart disease in the medical literature,[1][2][3] the term is most widely used in the context of the International Classification of Diseases (ICD) coding categories. The definition includes heart failure and other cardiac complications of hypertension when a causal relationship between the heart disease and hypertension is stated or implied on the death certificate. In 2013 hypertensive heart disease resulted in 1.07 million deaths as compared with 630,000 deaths in 1990.[4] According to ICD-10, hypertensive heart disease (I11), and its subcategories: hypertensive heart disease with heart failure (I11.0) and hypertensive heart disease without heart failure (I11.9) are distinguished from chronic rheumatic heart diseases (I05-I09), other forms of heart disease (I30-I52) and ischemic heart diseases (I20-I25). However, since high blood ...
Thrombosis
Fibrinolysis is the physiological breakdown of blood clots by enzymes such as plasmin. Organisation: following the thrombotic event, residual vascular thrombus will be re-organised histologically with several possible outcomes. For an occlusive thrombus (defined as thrombosis within a small vessel that leads to complete occlusion), wound healing will reorganise the occlusive thrombus into collagenous scar tissue, where the scar tissue will either permanently obstruct the vessel, or contract down with myofibroblastic activity to unblock the lumen. For a mural thrombus (defined as a thrombus in a large vessel that restricts the blood flow but does not occlude completely), histological reorganisation of the thrombus does not occur via the classic wound healing mechanism. Instead, the platelet-derived growth factor degranulated by the clotted platelets will attract a layer of smooth muscle cells to cover the clot, and this layer of mural smooth muscle will be vascularised by the blood inside the ...
ICD-10 Chapter IX: Diseases of the circulatory system
arteriolosclerosis. *arteriosclerosis. *arteriosclerotic vascular disease. *atheroma. *(I71) Aortic aneurysm and dissection *( ...
Venous ulcer
The exact cause of venous ulcers is not certain, but they are thought to arise when venous valves that exist to prevent backflow of blood do not function properly, causing the pressure in veins to increase.[6][7][8][9] The body needs the pressure gradient between arteries and veins in order for the heart to pump blood forward through arteries and into veins. When venous hypertension exists, arteries no longer have significantly higher pressure than veins, and blood is not pumped as effectively into or out of the area.[6][7][8][9] Venous hypertension may also stretch veins and allow blood proteins to leak into the extravascular space, isolating extracellular matrix (ECM) molecules and growth factors, preventing them from helping to heal the wound.[6][9] Leakage of fibrinogen from veins as well as deficiencies in fibrinolysis may also cause fibrin to build up around the vessels, preventing oxygen and nutrients from reaching cells.[6] Venous insufficiency may also cause white blood cells ...
Systolic hypertension
Based on these studies, treating to a systolic blood pressure of 140, as long as the diastolic blood pressure is 68 or more, seems safe.[citation needed] Corroborating this, a reanalysis of the SHEP data suggests allowing the diastolic to go below 70 may increase adverse effects.[4] A meta-analysis of individual patient data from randomized controlled trials found the lowest diastolic blood pressure for which cardiovascular outcomes improve is 85 mm Hg for untreated hypertensives and 80 mm Hg for treated hypertensives.[5] The authors concluded "poor health conditions leading to low blood pressure and an increased risk for death probably explain the J-shaped curve".[5] Interpreting the meta-analysis is difficult, but avoiding a diastolic blood pressure below 68-70 mm Hg seems reasonable because: ...
Mondor's disease
... (also known as "Mondor's syndrome of superficial thrombophlebitis"[2]) is a rare condition which involves thrombophlebitis of the superficial veins of the breast and anterior chest wall. It sometimes occurs in the arm or penis.[3] In axilla, this condition is known as axillary web syndrome.[4]. Patients with this disease often have abrupt onset of superficial pain, with possible swelling and redness of a limited area of their anterior chest wall or breast. There is usually a lump present, which may be somewhat linear and tender. Because of the possibility of the lump being from another cause, patients are often referred for mammogram and/or breast ultrasound.[5]. Mondor's disease is self-limiting and generally benign. A cause is often not identified, but when found includes trauma, surgery, or inflammation such as infection. There have been occasional cases of associated cancer.[6] Management is with warm compresses and pain relievers, most commonly NSAIDS such as ibuprofen. ...
Vascular disease
Hirsch, Alan T.; Haskal, Ziv J.; Hertzer, Norman R.; Bakal, Curtis W.; Creager, Mark A.; Halperin, Jonathan L.; Hiratzka, Loren F.; Murphy, William R.C.; Olin, Jeffrey W.; Puschett, Jules B.; Rosenfield, Kenneth A.; Sacks, David; Stanley, James C.; Taylor, Lloyd M.; White, Christopher J.; White, John; White, Rodney A.; Antman, Elliott M.; Smith, Sidney C.; Adams, Cynthia D.; Anderson, Jeffrey L.; Faxon, David P.; Fuster, Valentin; Gibbons, Raymond J.; Halperin, Jonathan L.; Hiratzka, Loren F.; Hunt, Sharon A.; Jacobs, Alice K.; Nishimura, Rick; et al. (2006). "ACC/AHA 2005 Guidelines for the Management of Patients With Peripheral Arterial Disease (Lower Extremity, Renal, Mesenteric, and Abdominal Aortic): A Collaborative Report from the American Association for Vascular Surgery/Society for Vascular Surgery,⁎ Society for Cardiovascular Angiography and Interventions, Society for Vascular Medicine and Biology, Society of Interventional Radiology, and the ACC/AHA Task Force on Practice Guidelines ...
Venous thrombosis
Evidence supports the use of heparin in people following surgery who have a high risk of thrombosis to reduce the risk of DVTs; however, the effect on PEs or overall mortality is not known.[22] In hospitalized non-surgical patients, mortality decreased but not statistically significant.[23][24] It does not appear however to decrease the rate of symptomatic DVTs.[23] Using both heparin and compression stockings appears better than either one alone in reducing the rate of DVT.[25]. In hospitalized people who have had a stroke and not had surgery, mechanical measures (compression stockings) resulted in skin damage and no clinical improvement.[23] Data on the effectiveness of compression stockings among hospitalized non-surgical patients without stroke is scarce.[23]. The American College of Physicians (ACP) gave three strong recommendations with moderate quality evidence on VTE prevention in non-surgical patients: that hospitalized patients be assessed for their risk of thromboembolism and bleeding ...
Trans fat
Hydrogenation of an unsaturated fatty acid refers to the addition of hydrogen atoms to the acid, causing double bonds to become single ones, as carbon atoms acquire new hydrogen partners (to maintain four bonds per carbon atom). Full hydrogenation results in a molecule containing the maximum amount of hydrogen (in other words, the conversion of an unsaturated fatty acid into a saturated one). Partial hydrogenation results in the addition of hydrogen atoms at some of the empty positions, with a corresponding reduction in the number of double bonds. Typical commercial hydrogenation is partial to obtain a malleable mixture of fats that is solid at room temperature, but melts during baking, or consumption. In most naturally occurring unsaturated fatty acids, the hydrogen atoms are on the same side of the double bonds of the carbon chain (cis configuration - from the Latin, meaning "on the same side"). However, partial hydrogenation reconfigures most of the double bonds that do not become chemically ...
Orthostatic hypotension
... , also known as postural hypotension,[2] is a medical condition wherein a person's blood pressure falls when standing or sitting. The drop in blood pressure may be sudden (vasovagal orthostatic hypotension), within 3 minutes (classic orthostatic hypotension) or gradual (delayed orthostatic hypotension).[3] It is defined as a fall in systolic blood pressure of at least 20 mm Hg or diastolic blood pressure of at least 10 mm Hg when a person assumes a standing position. It occurs predominantly by delayed (or absent) constriction of the lower body blood vessels, which is normally required to maintain an adequate blood pressure when changing position to standing. As a result, blood pools in the blood vessels of the legs for a longer period and less is returned to the heart, thereby leading to a reduced cardiac output and inadequate blood flow to the brain. Very mild occasional orthostatic hypotension is common and can occur briefly in anyone, although it is prevalent in ...
Arteriolosclerosis - Wikipedia
Types include hyaline arteriolosclerosis and hyperplastic arteriolosclerosis, both involved with vessel wall thickening and ... Arteriolosclerosis is a form of cardiovascular disease involving hardening and loss of elasticity of arterioles or small ... It is a type of arteriolosclerosis, which refers to thickening of the arteriolar wall and is part of the ageing process. ... This is a type of arteriolosclerosis involving a narrowed lumen. The term "onion-skin" is sometimes used to describe this form ...
arteriolosclerosis | Taber's Medical Dictionary
arteriolosclerosis answers are found in the Tabers Medical Dictionary powered by Unbound Medicine. Available for iPhone, iPad ... arteriolosclerosis is a topic covered in the Tabers Medical Dictionary. To view the entire topic, please sign in or purchase a ... "Arteriolosclerosis." Tabers Medical Dictionary, 23rd ed., F.A. Davis Company, 2017. Tabers Online, www.tabers.com/ ... tabersonline/view/Tabers-Dictionary/752483/all/arteriolosclerosis. Arteriolosclerosis. In: Venes D, ed. Tabers Medical ...
Arteriolosclerosis synonyms, arteriolosclerosis antonyms - FreeThesaurus.com
Antonyms for arteriolosclerosis. 2 words related to arteriolosclerosis: induration, sclerosis. What are synonyms for ... arteriolosclerosis. Also found in: Dictionary, Medical, Encyclopedia.. Related to arteriolosclerosis: hypertension, stroke ... Arteriolosclerosis synonyms, arteriolosclerosis antonyms - FreeThesaurus.com https://www.freethesaurus.com/arteriolosclerosis. ... Hyaline arteriolosclerosis is more pronounced in patients with diabetes than it is in patients with any other disease, except ...
arteriolosclerosis | Taber's Medical Dictionary
arteriolosclerosis answers are found in the Tabers Medical Dictionary powered by Unbound Medicine. Available for iPhone, iPad ... arteriolosclerosis is a topic covered in the Tabers Medical Dictionary. To view the entire topic, please sign in or purchase a ... "Arteriolosclerosis." Tabers Medical Dictionary, 23rd ed., F.A. Davis Company, 2017. Nursing Central, nursing.unboundmedicine. ... com/nursingcentral/view/Tabers-Dictionary/752483/all/arteriolosclerosis. Arteriolosclerosis. In: Venes D, ed. Tabers Medical ...
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Arteriosclerosis - Wikipedia
Supplementary Material for: Genetic Susceptibility for Ischemic Infarction and Arteriolosclerosis Based on Neuropathologic...
Genetic Susceptibility for Ischemic Infarction and Arteriolosclerosis Based on Neuropathologic Evaluations ... Supplementary Material for: Genetic Susceptibility for Ischemic Infarction and Arteriolosclerosis Based on Neuropathologic ... The diabetes risk variant rs864745 within JAZF1 was associated with arteriolosclerosis (OR = 0.80, p = 0.014). We observed ... or macroscopic infarcts and with arteriolosclerosis. Methods: Measures of neuropathology and genotyping were available from 755 ...
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Arteriolosclerosis. Arteriolosclerosis is characterized by thickening of the walls of small arteries and arterioles. It is most ... hyperplastic arteriolosclerosis. Hyaline arteriolosclerosis. Seen in the elderly, hypertension, diabetic microangiopathy and ... Hyperplastic arteriolosclerosis. Seen usually in malignant hypertension. Onion-skin laminated concentric thickening of ...
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Early Punch Grafting in Arteriolopathy Ulcers in the Elderly
Introduction: Posttraumatic ulcers secondary to age-related arteriolosclerosis may be included in the clinic-histopathological ... Histologically, they both present occlusive subcutaneous arteriolosclerosis. Considering these similarities, they could benefit ... both types of wounds present occlusive subcutaneous arteriolosclerosis. The results of a retrospective study comparing leg ... biopsy findings of patients with and without Martorell ulcers concluded that arteriolosclerosis, which is present in both ...
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Free Medical Flashcards about Vascular Terms
hyaline arteriolosclerosis. hyaline thickening, benign nephrosclerosis. hyperplastic arteriolosclerosis. onion-skin appearance ... Arteriolosclerosis. affects small arteries and arterioles; hylaine and hyperplastic; assoc with hypertension and DM. ... Mnckebergs arteriosclerosis, Arteriolosclerosis, Atherosclerosis. Monckebergs arteriosclerosis. medial calcific sclerosis; ...
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Arteriosclerosis4
- A statistically significant rise in creatinine level was observed for different degrees of arteriosclerosis and arteriolosclerosis from preoperative to postoperative period with a mean follow-up ranging from 45. (freethesaurus.com)
- Arteriolosclerosis is one of the three subcategories of arteriosclerosis. (marham.pk)
- While arteriosclerosis refers to the unusual hardening or thickening of arteries, arteriolosclerosis is especially about the thickening of arterioles and small arteries. (marham.pk)
- Arteriosclerosis "comprises three distinct forms: atherosclerosis, Monckeberg's arteriosclerosis, and arteriolosclerosis. (google.ca)
Atherosclerosis4
- Arteriolosclerosis , unlike atherosclerosis, is a sclerosis that only affects small arteries and arterioles, which carry nutrients and blood to the cells. (wikipedia.org)
- At the time, there was no known link between atherosclerosis/arteriolosclerosis and this arteritis. (aao.org)
- In short, atherosclerosis of the main renal artery, and/or intrarenal arteriolosclerosis, resulted in the production of renin. (sciencechatforum.com)
- The basic pathology of intraparenchymal hemorrhage is arteriolosclerosis, characterized by angionecrosis (destruction) of smooth muscle cells in intracerebral small arteries, as opposed to atherosclerosis (plaque formation) in medium to large arteries. (ahajournals.org)
Arteries4
- Arteriolosclerosis is a form of cardiovascular disease involving hardening and loss of elasticity of arterioles or small arteries and is most often associated with hypertension and diabetes mellitus. (wikipedia.org)
- Arteriolosclerosis is characterized by thickening of the walls of small arteries and arterioles. (radiopaedia.org)
- SVD initially manifests as lipohyalinosis and arteriolosclerosis in vessels of the basal ganglia, that is, the putamen and globus pallidus, and then in leptomeningeal arteries. (biomedcentral.com)
- In the kidneys, as a result of benign arterial hypertension , hyaline (pink, amorphous, homogeneous material) accumulates in the walls of small arteries and arterioles, producing the thickening of their walls and the narrowing of the arterial openings , a process known as arteriolosclerosis . (wikipedia.org)
Hyperplastic2
- Types include hyaline arteriolosclerosis and hyperplastic arteriolosclerosis, both involved with vessel wall thickening and luminal narrowing that may cause downstream ischemic injury. (wikipedia.org)
- There are two classifications of arteriolosclerosis: Hyaline arteriolosclerosis and Hyperplastic arteriolosclerosis. (marham.pk)
Arterioles1
- Hyaline arteriolosclerosis refers to thickening of the walls of arterioles by the deposition of hyaline material. (doctorbhatia.com)
Arteriolar3
- It is a type of arteriolosclerosis, which refers to thickening of the arteriolar wall and is part of the ageing process. (wikipedia.org)
- Hyaline arteriolosclerosis is a major morphologic characteristic of benign nephrosclerosis, in which the arteriolar narrowing causes diffuse impairment of renal blood supply, with loss of nephrons. (wikipedia.org)
- arteriolar sclerosis arteriolosclerosis . (thefreedictionary.com)
Arterial1
- People with higher arterial calcium levels are more likely to develop arteriolosclerosis , stroke and other heart ailments later in life than those with normal levels. (freethesaurus.com)
Taber's Medical Dictionary1
- arteriolosclerosis is a topic covered in the Taber's Medical Dictionary . (tabers.com)
Subcutaneous3
- Histologically, they both present occlusive subcutaneous arteriolosclerosis. (medscape.com)
- Skin biopsy does not assist in differentiating them because, histopathologically, both types of wounds present occlusive subcutaneous arteriolosclerosis. (medscape.com)
- Martorell hypertensive ischemic leg ulcer: a model of ischemic subcutaneous arteriolosclerosis. (uzh.ch)
Lipohyalinosis1
- All donated brains were examined by a board-certified neuropathologist using standardized protocol for the presence of microscopic infarct, macroscopic infarct and arteriolosclerosis (lipohyalinosis). (figshare.com)
Hypertensive1
- Posttraumatic ulcers secondary to age-related arteriolosclerosis may be included in the clinic-histopathological spectrum of the Martorell hypertensive ischemic ulcer. (medscape.com)
Renal2
- The histologic lesion termed "gouty nephropathy" consists of glomerulosclerosis, interstitial fibrosis, and renal arteriolosclerosis, often with focal interstitial urate crystal deposition ( 2 , 3 ). (asnjournals.org)
- A majority of papers at that time questioned the minority renal arteriolosclerosis/ischaemia hypothesis, with the result that the older hypothesis appears to have maintained sway to this day. (sciencechatforum.com)
Dementia2
- Among former National Football League players who died with CTE, tau tangles, crumbling white matter, and arteriolosclerosis independently contributed to dementia. (alzforum.org)
- Association of White Matter Rarefaction, Arteriolosclerosis, and Tau With Dementia in Chronic Traumatic Encephalopathy. (bu.edu)
Diabetes3
- Hyaline arteriolosclerosis is more pronounced in patients with diabetes than it is in patients with any other disease, except those associated with chronic transplant glomerulopathy. (freethesaurus.com)
- The diabetes risk variant rs864745 within JAZF1 was associated with arteriolosclerosis (OR = 0.80, p = 0.014). (figshare.com)
- White matter hyperintensities (WMHs) on T2-weighted magnetic resonance imaging (MRI) ( 7 ) have been associated with arteriolosclerosis, arising as consequences of aging, diabetes, and other cardiovascular risk factors ( 8 , 9 ). (diabetesjournals.org)
Affects2
- A second study, "Arteriolosclerosis that affects multiple brain regions," appears in a recent issue of the journal Brain, and looks at small blood vessels in patients with HS-Aging and describes a specific change, called "arteriolosclerosis," which is present in patients with HS-Aging. (mindzilla.com)
- Arteriolosclerosis that affects multiple brain regions. (mindzilla.com)
Infarcts3
- An association between global parkinsonian score and arteriolosclerosis was no longer statistically significant after accounting for infarcts, the investigators noted. (freethesaurus.com)
- In this study, we tested whether known single nucleotide polymorphisms (SNPs) associated with stroke or putative stroke risk factors are associated with neuropathologically defined micro- or macroscopic infarcts and with arteriolosclerosis. (figshare.com)
- Both arteriolosclerosis and microatheromatosis-the two main types of small vessel disease-feature periventricular hypodensity, according to an August 8, 2002, article on "Lacunar Infarcts" published by the American Heart Association. (ehow.co.uk)
Moderate1
- A moderate degree of diffuse arteriolosclerosis was observed. (freethesaurus.com)
Deposition1
- Weight should remain with the extracellular deposition of homogenous eosinophilic mate- rial hyaline arteriolosclerosis. (norfolkspca.com)
Stages1
- In 1953, Scheie classified the changes of hypertension and arteriolosclerosis separately into five stages ranging from normal to the most severe changes in the retina (Table 1). (freethesaurus.com)
Disease1
- This mechanism of enteric hyperoxaluria is manifested in several ways, including with orlistat therapy [ 7 , 8 ], Roux-en-Y gastric bypass surgery [ 9 ], celiac disease, and Crohn's disease. (hindawi.com)
Patients1
- The results of a retrospective study comparing leg biopsy findings of patients with and without Martorell ulcers concluded that arteriolosclerosis, which is present in both groups, was associated with age. (medscape.com)
View2
- Taber's Online , www.tabers.com/tabersonline/view/Tabers-Dictionary/752483/all/arteriolosclerosis. (tabers.com)
- Nursing Central , nursing.unboundmedicine.com/nursingcentral/view/Tabers-Dictionary/752483/all/arteriolosclerosis. (unboundmedicine.com)
Form1
- Huchard H: Arteriolosclerosis: including its cardiac form. (springer.com)
Easily1
- The following two terms whilst similar, are distinct in both spelling and meaning and may easily be confused with arteriolosclerosis. (wikipedia.org)
Type1
- This is a type of arteriolosclerosis involving a narrowed lumen. (wikipedia.org)