An anticoagulant that acts by inhibiting the synthesis of vitamin K-dependent coagulation factors. Warfarin is indicated for the prophylaxis and/or treatment of venous thrombosis and its extension, pulmonary embolism, and atrial fibrillation with embolization. It is also used as an adjunct in the prophylaxis of systemic embolism after myocardial infarction. Warfarin is also used as a rodenticide.
Diagnostic Techniques and Procedures
The pathological process occurring in cells that are dying from irreparable injuries. It is caused by the progressive, uncontrolled action of degradative ENZYMES, leading to MITOCHONDRIAL SWELLING, nuclear flocculation, and cell lysis. It is distinct it from APOPTOSIS, which is a normal, regulated cellular process.
Prostheses and Implants
Artificial substitutes for body parts, and materials inserted into tissue for functional, cosmetic, or therapeutic purposes. Prostheses can be functional, as in the case of artificial arms and legs, or cosmetic, as in the case of an artificial eye. Implants, all surgically inserted or grafted into the body, tend to be used therapeutically. IMPLANTS, EXPERIMENTAL is available for those used experimentally.
Radio Frequency Identification Device
Molecular Motor Proteins
Any liquid or solid preparation made specifically for the growth, storage, or transport of microorganisms or other types of cells. The variety of media that exist allow for the culturing of specific microorganisms and cell types, such as differential media, selective media, test media, and defined media. Solid media consist of liquid media that have been solidified with an agent such as AGAR or GELATIN.
A thiazide diuretic often considered the prototypical member of this class. It reduces the reabsorption of electrolytes from the renal tubules. This results in increased excretion of water and electrolytes, including sodium, potassium, chloride, and magnesium. It is used in the treatment of several disorders including edema, hypertension, diabetes insipidus, and hypoparathyroidism.
Sodium Chloride Symporter Inhibitors
Drugs used in the treatment of acute or chronic vascular HYPERTENSION regardless of pharmacological mechanism. Among the antihypertensive agents are DIURETICS; (especially DIURETICS, THIAZIDE); ADRENERGIC BETA-ANTAGONISTS; ADRENERGIC ALPHA-ANTAGONISTS; ANGIOTENSIN-CONVERTING ENZYME INHIBITORS; CALCIUM CHANNEL BLOCKERS; GANGLIONIC BLOCKERS; and VASODILATOR AGENTS.
Theoretical construct used in applied mathematics to analyze certain situations in which there is an interplay between parties that may have similar, opposed, or mixed interests. In a typical game, decision-making "players," who each have their own goals, try to gain advantage over the other parties by anticipating each other's decisions; the game is finally resolved as a consequence of the players' decisions.
Medical History Taking
Interleukin-1 Receptor-Associated Kinases
A family of intracellular signaling kinases that were identified by their ability to signal from the activated INTERLEUKIN-1 RECEPTORS. Signaling from these kinases involves their interaction with SIGNAL TRANSDUCING ADAPTOR PROTEINS such as MYELOID DIFFERENTIATION FACTOR 88 and TNF RECEPTOR-ASSOCIATED FACTOR 6.
Property, such as patents, trademarks, and copyright, that results from creative effort. The Patent and Copyright Clause (Art. 1, Sec. 8, cl. 8) of the United States Constitution provides for promoting the progress of science and useful arts by securing for limited times to authors and inventors, the exclusive right to their respective writings and discoveries. (From Black's Law Dictionary, 5th ed, p1014)
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TNF Receptor-Associated Factor 6
Body of knowledge related to the use of organisms, cells or cell-derived constituents for the purpose of developing products which are technically, scientifically and clinically useful. Alteration of biologic function at the molecular level (i.e., GENETIC ENGINEERING) is a central focus; laboratory methods used include TRANSFECTION and CLONING technologies, sequence and structure analysis algorithms, computer databases, and gene and protein structure function analysis and prediction.
Evaluation of the effect of azapropazone on neutrophil migration in anaesthetized swine using a multichamber blister suction technique. (1/14)1. The purpose of this study was to determine the in vivo inhibitory efficacy of azapropazone on neutrophil migration. The effects of azapropazone given at a dose of 100 mg kg-1 i.v. every 2 h on the neutrophil migration into skin inflammation sites (blister fluid) as well as into an autologous serum (+/- chemoattractant) placed above the blister surface (2nd chamber) were determined. 2. Azapropazone treatment schedule maintained blood levels at 70-100 micrograms ml-1 throughout the time course (360 min) of the experiment. 3. Azapropazone significantly inhibited (48 +/- 6%) neutrophil migration into the blister fluid (as evident from the decrease in myeloperoxidase activity). 4. Azapropazone significantly inhibited the neutrophil migration into the autologous serum either with (65 +/- 5%) or (35 +/- 6%) without the chemoattractant, formyl-methionyl-leucyl-phenylalanine (FMLP). The chemoattractant, FMLP, markedly increased neutrophil migration into the autologous serum by approximately 1.5 to 2 times the non-FMLP treated group. Azapropazone was more efficacious in inhibiting the neutrophil migration in the presence of FMLP than in its absence. 5. We conclude that azapropazone is an effective inhibitor of neutrophil migration into topically inflamed sites in anaesthetized swine. (+info)
Evaluation of the effect of azapropazone on neutrophil migration in regional myocardial ischaemia/reperfusion injury in rabbits. (2/14)1. The purpose of the present study was to determine the myocardial cytoprotective efficacy of azapropazone (AZA) and its potential site of action on neutrophil infiltration into reperfused/ischaemic myocardium with or without in vivo activation of neutrophils in rabbits. 2. AZA, 100 mg kg-1, was administered i.v. 10 min after occlusion of the left circumflex (LCX) artery in rabbits with and without pretreatment with phorbol myristate acetate ester (PMA). The LCX occlusion was then released at 10 min after AZA administration. Haemodynamic parameters (heart rate, LV pressure, mean arterial blood pressure and dp/dt) were monitored throughout the experiment. After 60 min reperfusion, the area at risk was delineated and the heart was then excised and divided into epi- and endocardial pieces for analysis of myeloperoxidase activity. 3. AZA inhibited neutrophil infiltration into the reperfused/ischaemic rabbit myocardium with and without PMA treatment. The inhibition of neutrophil infiltration was more apparent in the epicardium than in the endocardium. Additionally, AZA inhibited to a similar extent the in vivo PMA-stimulated neutrophil migration into the epicardium and endocardium area at risk. AZA had no significant effect on the haemodynamic parameters as compared to control. 4. AZA administered in an anaesthetized rabbit model of LCX occlusion/reperfusion resulted in the reduction of infarct size. 5. It is concluded that AZA has significant inhibitory effects on neutrophil migration which might contribute to its myocardial cytoprotective effect. (+info)
Effect of treatment on erythrocyte phosphoribosyl pyrophosphate synthetase and glutathione reductase activity in patients with primary gout. (3/14)The activities of erythrocyte phosphoribosyl pyrophosphate (PRPP) synthetase and glutathione reductase (GTR) were studied in 26 patients with primary gout who were receiving no treatment or treatment with either allopurinol or azapropazone, and compared with the activity in a group of healthy controls. The activity of PRPP synthetase was significantly higher in the gout group and was not influenced by either drug. No significant difference in the activity of GTR was observed. The failure of either drug to suppress the increased activity of PRPP synthetase associated with gout is discussed. (+info)
Comparative trial of azapropazone and indomethacin plus allopurinol in acute gout and hyperuricaemia. (4/14)This study compared the effects of azapropazone and indomethacin plus allopurinol in the management of acute gout and hyperuricaemia. A group of 93 patients predominantly based in general practice were randomly allocated to the two treatment regimens (azapropazone (days 1-225) or indomethacin (1-28) followed by allopurinol (29-225)) on a double-blind double dummy basis. Azapropazone produced a substantial reduction in serum uric acid levels by day 4 compared with day 1 (P<0.002) and was superior to indomethacin with regard to recorded levels of serum uric acid at day 4 (P<0.01) and day 28 (P<0.05). From day 28 onwards allopurinol produced and azapropazone maintained similar reductions in serum uric acid. Both treatments rapidly controlled the initial acute attacks of gout and both produced side effects similar in frequency and nature. Fewer breakthrough attacks of gout occurred in the azapropazone group (12) than the indomethacin/allopurinol group (21).Although the results achieved in both treatment groups were similar it has been shown that azapropazone is effective monotherapy for controlling both acute attacks of gout and hyperuricaemia. (+info)
Drug-binding properties of rat alpha 1-foetoprotein. Binding of warfarin, phenylbutazone, azapropazone, diazepam, digitoxin and cholic acid. (5/14)As part of an investigation into whether alpha 1-foetoprotein (alpha 1-FP) plays the same transport role in foetal serum as albumin does in the adult, the binding properties of both proteins were compared with respect to the binding of a series of compounds known to be bound by albumin's specific drug-binding sites. The binding of warfarin, phenylbutazone, azapropazone, diazepam, digitoxin and cholic acid by rat alpha 1-FP and serum albumin was studied by equilibrium dialysis at 4 degrees C. Rat alpha 1-FP was shown to have neither albumin's high-affinity site II (diazepam as marker) nor its site III (digitoxin and cholic acid as markers). High-affinity binding by alpha 1-FP was found for the specific markers (warfarin, phenylbutazone, azapropazone) of albumin's drug-binding site I. However, instead of albumin's one high-affinity site/molecule, a mean value of 0.5 site/molecule was obtained with rat alpha 1-FP. Charcoal treatment at neutral pH of rat serum albumin did not affect its measured binding properties, but treatment of the alpha 1-FP led to an increased affinity for warfarin, phenylbutazone and azapropazone without a change in the measured number of sites, indicating competition for binding at this site by (an) endogenous ligand(s). These results are discussed in terms of the structures of the two proteins and with respect to the physiological implications of the differences found. (+info)
Quantitative sacroiliac scintiscanning: a sensitive and objective method for assessing efficacy of nonsteroidal, anti-inflammatory drugs in patients with sacroiliitis. (6/14)Serial computer assisted quantitative sacroiliac scintiscanning (SI joint/sacrum ratios) 3 hours after low dosage (5 mCi) 99mTc methylene diphosphonate has been used as an objective index of sacroiliitis in a single blind 14-day cross-over comparison of azapropazone 600 mg b.d. and naproxen 500 mg b.d. in 18 patients with active sacroiliitis. Clinical assessments included visual analogue scales for measurement of pain and early morning stiffness, chest expansion, a modified Schober test, and goniometric measurement of thoracolumbar spinal flexion by means of an inclinometer. Statistically significant decreases in pain (p less than 0.001) and early morning stiffness (p less than 0.001) followed treatment with each NSAID, but there was no significant difference in the fall in these parameters, although 15 out of 18 patients expressed a preference for naproxen. Chest expansion and thoracolumbar flexion were not significantly affected by either drug. Serial quantitative scintigraphy showed a mean fall in joint sacrum ratios following each treatment which was statistically significant (p less than 0.02) only after naproxen. Serial quantitative scintigraphy can be used as an objective method of assessing sacroiliitis and was sufficiently sensitive to reflect the patients' subjective preference in a short-term comparison of 2 NSAID. (+info)
The uricosuric action of azapropazone: dose-response and comparison with probenecid. (7/14)Azapropazone is an anti-inflammatory agent with reported uricosuric properties. The aim of the present study was to extend these observations, by examining the dose-response and to compare the uricosuric effect of azapropazone with that of probenecid. Patients were given varying doses of azapropazone from 900-2400 mg daily for 4-day periods at separated intervals. Plasma uric acid levels were measured before and at the end of each treatment period. Three other patients maintained on low purine diets were given a 4-day course of 1200 mg azapropazone daily followed at an interval by a 4-day period of probenecid 1 g daily. Plasma uric acid levels and 24 h urinary uric acid excretion were compared. The mean fall in plasma uric acid level after four days of 900 mg azapropazone daily was 31.4% (n = 9) compared with 33.9% (n = 12) on 1200 mg daily; 42.3% (n = 10) on 1800 mg daily; and 46% (n = 6) on 2400 mg daily, indicating a graded dosage response. In the three patients on low purine diets the falls in plasma uric acid levels on probenecid 1 g daily were 50.5%, 46% and 29% compared with 33.5%, 32% and 20% respectively on azapropazone 1200 mg daily. Similarly the total amount of uric acid excreted in the urine by each patient during the 4-day period on probenecid 1 g daily was 14.01; 13.03 and 8.97 mmol compared with 23.53, 10.9 and 7.69 mmol on azapropazone 1200 mg daily.(ABSTRACT TRUNCATED AT 250 WORDS) (+info)
A study of the potential interactions between azapropazone and frusemide in man. (8/14)Ten healthy individuals received frusemide 40 mg orally for 7 days. Following a drug free period of 7 days they received azapropazone 600 mg twice daily for 7 days and then both treatments for a further 7 days. Sodium excretion fell from 141 +/- 16.8 mmol/day to 84.3 +/- 6.8 mmol/day (P less than 0.01) on initiation of azapropazone treatment. The natriuretic response to frusemide was unchanged by premedication with azapropazone. Urate excretion rose from 3.35 +/- 0.249 mmol/day to 4.98 +/- 0.365 mmol/day on initiation of azapropazone therapy but subsequently returned to baseline values. Plasma uric acid fell from 0.289 +/- 0.024 mmol/l to 0.167 +/- 0.0125 mmol/l (P less than 0.001) on azapropazone but rose to 0.186 +/- 0.0116 mmol/l (P less than 0.001) with the addition of frusemide. Azapropazone may cause sodium retention but after repeated administration frusemide still has a marked diuretic action. The hypouricaemic effect of azapropazone is only slightly antagonised by frusemide at the doses studied. (+info)
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The police officer, who was not identified, was exposed to the Soviet-developed nerve agent that was used to attack a former Russian spy last year.
IL-17 induces hyperalgesia via TNF-dependent neutrophil infiltration. - Nuffield Department of Orthopaedics, Rheumatology and...
Interleukin-17 (IL-17) and tumour necrosis factor-α (TNF) are critical in the pathogenesis of arthritis but their relationship during inflammatory pain has received limited attention. We aimed to establish whether IL-17 can induce hyperalgesia in acute conditions, and investigated the role of TNF in mediating the pain response. Hyperalgesia was elicited in C57BL/6 mice by injection of recombinant IL-17, TNF or vehicle into the plantar tissue. Elevated pain was measured by the Hargreaves test for thermal hyperalgesia and Linton incapacitance tester for weight-bearing change. Cellular infiltration during hyperalgesia was determined by histological analysis and myeloperoxidase assay. IL-17 was found to induce hyperalgesia, but this was dependent on neutrophil migration and TNF binding to TNF receptor 1 (TNFR1). Because TNF-induced hyperalgesia was also dependent on neutrophil migration, the relationship between the resident fibroblasts, the cytokines and the migrating neutrophils was further investigated.
Role of Site-Specific Binding to Plasma Albumin in Drug Availability to Brain | Journal of Pharmacology and Experimental...
Serum albumins from mammalian species differ in amino acid sequence and protein structure. Consistent with this, binding-site affinity and selectivity vary from species to species. Site I on human serum albumin consists of two overlapping subregions, one for warfarin and one for azapropazone (Fehske et al., 1982). These subregions overlap in rat and are separate in bovine and absent in dog (Kosa et al., 1997). We observed differences in binding affinity of warfarin with rat, bovine, and human albumin that correlated with reported KD constants (Seller et al., 1977). Because of these differences, albumins from separate species should not be considered interchangeable. Nonetheless, no statistically significant differences were observed between predicted and measured up-take Kin with any of the three albumins.. A number of studies have been published suggesting enhanced dissociation of ligands that bind fairly selectively to Sudlow site II of albumin (e.g., benzodiazepines and tryptophan) (Jones et ...
Indomethacin Plus Biological Therapy in Treating Patients With Advanced Melanoma - Full Text View - ClinicalTrials.gov
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Neutrophil migration across monolayers of cytokine-prestimulated endothelial cells: a role for platelet-activating factor and...
In a previous study we observed that neutrophils respond with a rapid rise in [Ca2+]i during adherence to cytokine-activated endothelial cells (EC), caused by EC membrane-associated platelet-activating factor (PAF). In the present study, we investigated whether this form of PAF was important in neutrophil adherence and migration across monolayers of rIL-1 beta- or rTNF alpha-prestimulated EC. PAF receptor antagonists prevented neutrophil migration across cytokine-pretreated EC by approximately 60% (P less than 0.005) without interfering with the process of adherence. The antagonists WEB 2086 and L-652,731 had no effect on neutrophil migration across resting EC induced by formylmethionyl-leucyl-phenylalanine (FMLP). A murine anti-IL-8 antiserum was found to also partially inhibit the neutrophil transmigration across cytokine-activated EC. When the anti-IL-8 antiserum was used in combination with a PAF receptor antagonist, neutrophil migration across cytokine-pretreated monolayers of EC was ...
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This paper presents subjective and objective methods for evaluating transient vehicle dynamics characteristics in four sections: (1) Definition of transient behavior in terms of four traits-agility, stability, precision, and roll support; (2) Description of subjective evaluation methods; (3) Implementation of Design for Six Sigma principles to the development of a steering robot controlled objective test for transient performance; (4) The final section of this paper uses data from simulation and road tests to demonstrate how chassis design parameters can affect transient handling performance ...
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The aim of the present study was to evaluate the safety and efficacy of the combination of indomethacin and statin compared with indomethacin plus placebo in patients with a first episode of pericarditis. A total of 55 consecutive patients with acute pericarditis were randomized in a double-blind manner into two groups: group 1 (statin group) was treated with 150 mg of indomethacin plus 10 mg of rosuvastatin, and group 2 (placebo group) was treated with 150 mg of indomethacin plus placebo. Both groups received treatment up to the normalization of inflammation markers and for the following week. Clinical and laboratory assessments [white cell count, ESR (erythrocyte sedimentation rate) and CRP (C-reactive protein), troponin I, creatine kinase and brain natriuretic peptide plasma levels], ECG and echocardiogram were performed at baseline and daily up to discharge. All of the patients were followed as outpatients for 3 months to evaluate any recurrence of pericarditis. The two groups were similar ...
Helianthus tuberosus agglutinin directly induces neutrophil migration, which can be modulated/inhibited by resident mast cells.
We investigated the effect of Helianthus tuberosus agglutinin (HTA) on neutrophil migration in vivo and in vitro. The role of resident cells in this effect was analyzed. Peritonitis was induced by injecting stimuli into rat (150-200 g) peritoneal cav
Granulocyte-Macrophage Colony-Stimulating Factor Is a Chemoattractant Cytokine for Human Neutrophils: Involvement of the...
These studies demonstrate that GM-CSF is a neutrophil chemotactic agent. The concentration of GM-CSF needed to achieve maximal chemotaxis is comparable to that of the potent neutrophil chemoattractant IL-8 and less than the other known chemoattractants that we studied. We believe that GM-CSF induction of neutrophil chemotaxis has been previously unrecognized because the stimulatory effect occurs in a narrow range of concentrations (Fig. 3⇑A) and requires an extended incubation interval of at least 30 min. Results reported by Harakawa et al. (44) agreed with our finding that GM-CSF produces an early stimulation of neutrophil chemokinesis, but observations were not reported past 15 min, which may have been insufficient to recognize an effect on chemotaxis. Other earlier studies provided contradictory data on the effect of GM-CSF on neutrophil migration. In a checkerboard assay using polycarbonate filters, Wang et al. (45) demonstrated that GM-CSF induced chemotaxis in neutrophils, while Kharazmi ...
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EEG measurements offer objective method of assessing pain
A team of researchers has developed a new method that is claimed to objectively measure pain levels. The system uses electroencephalography (EEG) to identify oscillations in certain brainwaves that have been found to clearly correlate with commonly used pain assessment tools.
Endotoxin-induced autocrine ATP signaling inhibits neutrophil chemotaxis through enhancing myosin light chain phosphorylation |...
To gain further insight into the effects of calcium influx on neutrophil chemotaxis, we depleted the calcium in the under-agarose chemotaxis models using a calcium-free solution and EGTA. We observed that the inhibitory effects of LPS on neutrophil chemotaxis were impaired in the presence of calcium-depleted medium (Fig. 3C). Moreover, using a calcium ionophore ionomycin to stimulate neutrophils, a sustained calcium influx was observed and chemoattractant-induced neutrophil chemotaxis was dramatically inhibited (SI Appendix, Fig. S7A). The sustained calcium influx appeared to be required for initiating stop signal of neutrophil chemotaxis. Previous reports displayed that intracellular calcium was necessary for neutrophil migration (19). Therefore, we further clarified the effects of calcium on neutrophil migration. Different chemoattractant-induced calcium mobilization patterns were found to be inconsistent (20, 21). We noted that different from stimulation of IL-8, a rapid increase in ...
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The Index of Activities of Daily Living (ADL) encompasses six basic human functions: bathing, dressing, toileting, transfer, continence, and feeding. It provides an objective method of classifying heterogeneous groups of people with chronic illnesses, disabilities and impairments, and of describing their health needs and outcomes ...
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Dangerous Product Report » Sagent Pharmaceuticals Initiates a Nationwide Voluntary Recall of Three Lots of Ketorolac...
Sagent Pharmaceuticals, Inc. today announced the voluntary nationwide recall of three lots of Ketorolac Tromethamine Injection, USP, 30mg/mL single-dose vials (NDC numbers 25021-701-01 and 25021-701-02) manufactured by Cadila Healthcare Limited and distributed by Sagent. Sagent has initiated this voluntary recall of Ketorolac Tromethamine Injection, USP, 30mg/mL to the user level due to labeling the product with the incorrect expiration date ...
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Torasic information about active ingredients, pharmaceutical forms and doses by Kalbe Farma, Torasic indications, usages and related health products lists
Ketorolac tromethamine - routes and clinical implications - NEMA Research
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Mix Ranvets Flexi-Joint thoroughly through a dry feed. A measure is provided. One level measure holds 30g Flexi-Joint. Not advisable to be fed to pregnant or lactating animals or in conjunction with phenylbutazone (bute), as clotting factors may be affected.. ...
OMEPRAZOLE AUROBINDO 20mg 7TK ⋆ MediKeep
Mõned ravimid põhjustavad tugevaid kõrvaltoimeid või ei ole nende toime ootuspärane, kuna mõjuvad kas liiga tugevalt või on nende toime liiga nõrk - põhjus on inimese omapäras ja geneetikas. Ravimite omastamist mõjutab veel mikrobioom. Teadust, mis uurib ravimi toimeainete sobivust nimetatakse farmakogenoomikaks ja sellel on väga oluline roll personaalmeditsiinis. Geenitest võib näidata kätte sobiva toimeaine või anda vastust, miks mõni ravim ei ole sobinud.. Päring aitab selgitada, kas teadusuuringud on leidnud seose ravimi sobivuse ja geneetika vahel ...
Acupain Tablet | 10 mg Ketorolac Tromethamine (Oral / Injection / Nasal) | Uses, Doses, Price, Side Effects | Drugs used for...
Ketorolac is a nonsteroidal anti-inflammatory drug (NSAID) chemically related to indomethacin and tolmetin. Ketorolac tromethamine is a racemic mixture of [-]S- and [+]R-enantiomeric forms, with the S-form having analgesic activity. Its antiinflammatory effects are believed to be due to inhibition of both cylooxygenase-1 (COX-1) and cylooxygenase-2 (COX-2) which leads to the inhibition of prostaglandin synthesis leading to decreased formation of precursors of prostaglandins and thromboxanes from arachidonic acid. The resultant reduction in prostaglandin synthesis and activity may be at least partially responsible for many of the adverse, as well as the therapeutic, effects of these medications. Analgesia is probably produced via a peripheral action in which blockade of pain impulse generation results from decreased prostaglandin activity. However, inhibition of the synthesis or actions of other substances that sensitize pain receptors to mechanical or chemical stimulation may also contribute to ...
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SCINTISCANNING SYSTEM FOR STUDY OF REGIONAL DISTRIBUTION OF BLOOD FLOW.
The effect of changes in the direction and magnitude of the gravitational-inertial force environment on the regional distribution of impacted 35-micron diameter microspheres has been measured in the lungs of six anesthetized chimpanzees. These distributions were determined by two computer-controlled scintiscans at 780 sites covering the dorsal and ventral surfaces of the thorax at 1 G subsequent to four injections of differentially isotope-tagged microspheres into the right ventricular outflow tract. Pulse-height analysis at each site allowed separation of count values for the isotopes, and, after correction for collimator distortion, these values were assumed to be proportional to the respective blood flows which were present below each site at the respective time of injections. Computer-generated 3-dimensional and contour map displays of the scintiscan and related physiologic data indicate that pulmonary blood flow tended to redistribute toward the midthoracic region during acceleration exposures
Elevated monocyte interleukin-6 (IL-6) production in immunosppressed t by Gyongyi Szabo, Karen Kodys et al.
Aberrant monocyte mediator production is pivotal in the development of posttrauma immunosuppression. We have previously shown that immunodepressed trauma patients monocytes produce elevated interleukin-6, suggesting their in vivo preactivation. This study confirms that preactivated patients Mo produce greater levels of IL-6 than normals Mo to the same in the in vitro Fc gamma RI stimulation. We also demonstrate the capacity of interleukin-4 to downregulate the elevated interleukin-6 production of trauma patients in vivo preactivated monocytes. Monocyte interleukin-6 downregulation by interleukin-4 is dose dependent and occurs whether Fc gamma RI cross-linking, muramyl dipeptide, indomethacin plus muramyl dipeptide, or interferon-gamma plus muramyl dipeptide is the interleukin-6 inducing stimulus. Furthermore, interleukin-4-dependent downregulation of monocyte interleukin-6 expression is confirmed at both the supernatant and the mRNA levels. Simultaneous downregulation of posttrauma elevated
Ketorolac (Tromethamine) - Drug Monograph - DrugInfoSys.com
Information provided on this Web site is neither intended nor implied to be a substitute for professional medical advice. You should not use this information on this web site or the information on links from this site to diagnose or treat a health problem or disease without consulting with a qualified healthcare provider. Pharma Professional Services advises you to always seek the advice of your physician or other qualified health provider prior to starting any new treatment or with any questions you may have regarding a medical condition.. You should check with your physician/health care provider before using any of the means or methods presented on this site. If you undertake any treatment methods displayed on this site without such supervision, you are solely and entirely responsible for its outcome. Pharma Professional Services nor anyone connected with this site cannot be held responsible for your actions nor any conditions resulting thereof.. ...
SPRIX® | Official Site for HCPs
Learn about SPRIX (ketorolac tromethamine). Please see the full Prescribing Information, including Boxed Warning and Patient Medication Guide.
Tromethamine (Professional Patient Advice) - Drugs.com
Professional guide for Tromethamine. Includes: pharmacology, pharmacokinetics, contraindications, interactions, adverse reactions and more.
Disease & Pests - California Apple Commission
The second phase of this project will be to begin designing an objective method to test and compare Granny Smith starch iodine results. In basic terms, this testing method would include crushing the apples into a slurry type mixture and measuring any light that is refracted through the apple blend. The testing phase of tis study is just about complete. Upon completion of the project, the Commission anticipates having an objective test that will measure starch iodine in California Granny Smith apples ...
Tobacco cheats caught by smoking gun of VOCs - Ezine - separationsNOW.com
Smokers have particular VOCs in their breath Smoking is a major cause of serious ill health and early death. Unfortunately, patients are not always entirely honest about whether or not they are smoking, so objective methods are required to determine smoking habits one technique is to examine the VOCs in smokers breath. The breath of smokers tends...
Prednisone for gout
The goal of treatment during an acute gout attack is suppression of inflammation and control of pain. It is important to note, that if a patient is not on
Tolerability, Safety and Pharmacokinetics of Four Formulations of Ketorolac Tromethamine in Healthy Volunteers - Tabular View -...
This was a phase 1, double-blind, 4-way crossover study in healthy male and female volunteers. Subjects received 4 formulations of intranasal ketorolac tromethamine 30 mg. There was a wash-out period of 3-7 days between each dose. On Day 1 of each period subjects were randomised to receive either a single intranasal dose of 30 mg ketorolac tromethamine alone or single intranasal dose of 30 mg ketorolac tromethamine with 4%, 5% or 6% lidocaine hydrochloride. At the end of the study each subject had received all 4 treatments.. The primary objective of this study in healthy volunteers was to compare the safety, tolerability, and pharmacokinetics of 4 formulations of ketorolac tromethamine. A secondary objective was to monitor lidocaine hydrochloride plasma levels. ...
Search results detail | Stomach Acid Vision Esarquite Ctonicas
In post-marketing feel, postoperative hematomas and other signs of wound bleeding have already been reported in colaboration with the perioperative use of IV or IM dosing of Ketorolac tromethamine. NSAIDs should be given carefully to patients with a history of inflammatory bowel sickness (ulcerative colitis, Crohns sickness) as their condition could be exacerbated. Ketorolac tromethamine tablets are not indicated for used in pediatric patients. The total combined duration of use of Ketorolac tromethamine-IV/IM and Ketorolac tromethamine pills isnt to exceed 5 times in adults. Ketorolac tromethamine is certainly contraindicated in patients currently acquiring aspirin or NSAIDs due to the cumulative hazards of inducing critical NSAID-related adverse events.. Stopping the medicine instantly could make your stomach produce a many more acid, and generate your symptoms return. Some people wont need to take omeprazole every day and take it only once they have symptoms. People might have differences ...
Ketorolac Tromethamine - Nonsteroidal Anti-inflammatory Agent
Ketorolac Tromethamine ophthalmic solutions and tablets are indicated for the temporary relief of ocular itching due to seasonal allergic conjunctivitis and for the treatment of postoperative inflammation in patients who have undergone cataract extraction.
Ocular Discomfort : Ketorolac Tromethamine Vs. Travoprost Drugs - DrugInformer
Information on drugs commonly used to treat ocular discomfort : ketorolac tromethamine vs. travoprost. Compare user review scores, and side effect occurrence rates for similar drugs side-by-side.
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Colchicine dose for acute gout : Clomid taux grossesse
the optimum dose? Colchicine in acute gouty. to treat and prevent recurrence of acute gout . The maximum dose of colchicine for treating an acute.Colchicine. - Acute attack of gout,. 4.2 Dosage and delivery system Acute attack of gout: 1st day: 3 tablets (1 in the morning,.Moins de fibres plus de maladies cardiovasculaires. 30-10-2013 Lu:5062 Maladies Santé TN. Moins de fibres plus de maladies cardiovasculaires Les fibres alimentaires.Terkeltaub RA, et al. High versus low dosing of oral colchicine for early acute gout flare:. et la colchicine à dose élevée (répartie sur 6 heures).Attack of gout Colchicine is indicated to prevent. Colchicine Dosage. The usual recommended dosage of Colchicine for Acute gouty arthritis attacks is 1.Allopurinol during acute gout attacks did not. indomethacin and colchicine within 7 days of onset of an acute attack. efficacité aux doses reconnues.Thiazide diuretic with a history of gout. PPI for peptic ulcer disease at full therapeutic dosage for,8 weeks. ...
Serum uric acid in acute gout | Annals of the Rheumatic Diseases
The relation between gout and uric acid is such that in general clinical practice there is a tendency (diminishing) to misdiagnose gout in the presence of hyperuricaemia. Conversely the diagnosis of gout may be rejected when a normal serum uric acid (SUA) value is found. Given that a high proportion of estimations are made at the time of the acute episode a correct diagnosis may depend on a practitioners knowledge of the fact that the SUA may be within the normal range at this time. Most, if not all rheumatologists, are aware of this fact, although the emphasis in current general and rheumatological publications and text books is that this is an unusual occurrence.1 2 We have conducted a prospective study to determine the frequency of normal SUA values in acute gout and also to compare acute and intercritical values.. Over a period of three years we observed 38 consecutive patients during 42 episodes of acute gout and who had the following characteristics: 34 men, four women, age 40-80 years ...
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Indian Pharmacy: Toradol is used for the short-term treatment of moderate to severe pain in adults., Toradol (generic name: Ketorolac) belongs to a group of medicines known as non-steroidal anti-inflammatory drugs (NSAIDs). Toradol is used for the short-term treatment of moderate to severe pain in adults, usually after surgery.
DailyMed - KETOROLAC TROMETHAMINE injection, solution
Clinical trials of several COX-2 selective and nonselective NSAIDs of up to three years duration have shown an increased risk of serious cardiovascular (CV) thrombotic events, including myocardial infarction (MI) and stroke, which can be fatal. Based on available data, it is unclear that the risk for CV thrombotic events is similar for all NSAIDs. The relative increase in serious CV thrombotic events over baseline conferred by NSAID use appears to be similar in those with and without known CV disease or risk factors for CV disease. However, patients with known CV disease or risk factors had a higher absolute incidence of excess serious CV thrombotic events, due to their increased baseline rate. Some observational studies found that this increased risk of serious CV thrombotic events began as early as the first weeks of treatment. The increase in CV thrombotic risk has been observed most consistently at higher doses. To minimize the potential risk for an adverse CV event in NSAID-treated ...
Acute Gout Attack in Cameroonians and Oxidative Stress: Cause and Effect? | Abstract
Aim: Hyperuricemia and urates crystals deposition which lead to acute gout are responsible for inflammatory processes and possibly oxidative stress. H..
Ketorolac tromethamine is a prescription medication used for short-term relief of moderate to severe pain. This eMedTV Web page discusses ketorolac, including information about its uses, its strengths, and some of its potential side effects.
tromethamine | Ligand page | IUPHAR/BPS Guide to PHARMACOLOGY
The IUPHAR/BPS Guide to Pharmacology. tromethamine ligand page. Quantitative data and detailed annnotation of the targets of licensed and experimental drugs.
Replacement For Colchicine | Acute Gout Remedy Recommendations to Aid You Beat Gout Naturally
The problem with this is that the drugs do not address the underlying causes and only perform while becoming taken. They cant do something for you when they are stopped. This is why so numerous much more gout victims tend to be enjoying the rewards of all-natural remedies for gout. And an essential element of this approach is to adhere to straightforward gout therapy tips that recognize the effect that some underlying issues have. For example, the chemical compounds in out body (purines), that create uric acid during the metabolizing procedure, also occur in our foods. Other problems are things like your weight position, common health, drugs you might be taking, loved ones history of arthritis / gout, high blood pressure, as well much alcohol, poor kidney function, also way of life troubles ...
Comparison of oral prednisolone and oral indomethacin in the treatment of acute gout: a double-blind, randomised, controlled...
Optimal breeding with robotics - WUR
Food production relies on the efficiency of plants and animals. To find and select the best performing variety in a nursery, greenhouse, barn and on the field, automation and large scale testing is key. Wageningen University & Research is specialised in sensing & plant/animal phenotyping with non-destructive, fast and robust applications. In co-development with machine builders and breeding companies we develop objective methods to collect data for describing variety performances in all stages of (re)production.
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Phenytoin Intravenous Advanced Patient Information - Drugs.com
Warfarin (Oral Route) Proper Use - Mayo Clinic
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Phenytoin (Injection Route) Precautions - Mayo Clinic
Using this medicine while you are pregnant can harm your unborn baby. Use an effective form of birth control to keep from getting pregnant. If you think you have become pregnant while using the medicine, tell your doctor right away. Do not receive this medicine while you or your child are also taking delavirdine (Rescriptor®). Using these medicines together may cause unwanted effects. This medicine may cause some people to become dizzy, lightheaded, faint, or less alert than they are normally. Make sure you know how you react to this medicine before you drive, use machines, or do anything else that could be dangerous if you are dizzy or are not alert and able to see well. Do not stop using this medicine without first checking with your doctor. Stopping the medicine suddenly may cause your seizures to return or to occur more often. If you or your child develop a skin rash, hives, or any allergic reaction to this medicine, tell your doctor or nurse as soon as possible. Tell your doctor right away ...
Study to Evaluate the Urate-Lowering Activity and Safety of Oral BCX4208 Administered in Subjects With Gout
Survey of toxicological investigations of azapropazone | SpringerLink
Chemical Compound - AstraZeneca AB
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N-glycan microheterogeneity regulates interactions of plasma proteins | PNAS
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OSTEOCONDUCTIVE MATRICES COMPRISING CALCIUM PHOSPHATE PARTICLES AND STATINS AND METHODS OF USING THE SAME - WARSAW ORTHOPEDIC,...
WO 2008/109462 A2 - Sustained Release Compositions Using Wax-like Materials - The Lens - Free & Open Patent and Scholarly...
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Pindolol - Aesthetic Clinic Malaysia
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Tinzaparin - Aesthetic Clinic Malaysia
It is very important that your doctor check your progress at regular visits to make sure this medicine is working properly. Blood tests will be needed to check for unwanted effects. Be sure to keep all appointments. This medicine may cause a serious type of allergic reaction called anaphylaxis. Anaphylaxis can be life-threatening and requires immediate medical attention. Tell your doctor right away if you have a rash; itching; swelling of the face, tongue, and throat; trouble breathing; or chest pain after you receive this medicine. Make sure your doctor knows if you have had an allergic reaction to heparin, sulfites, benzyl alcohol, or pork products. Tinzaparin may increase your chance of bleeding or bruising. This risk is higher if you have a catheter in your back for pain medicine or anesthesia (sometimes called an "epidural"), or if you have kidney problems. The risk of bleeding increases as the severity of your kidney problems increases. Check with your doctor right away if you notice any ...
Methods Using Lipoprotein-Associated Phospholipase A2 in an Acute Care Setting - Patent application
Warfarin (Intravenous route) - Loyola University Health System
It is very important that your doctor check your progress at regular visits to see if the medicine is working properly. Blood tests, such as an INR, are needed to check for proper dosage and unwanted side effects. Be sure to keep all appointments. Using this medicine while you are pregnant can harm your unborn baby. Use an effective form of birth control to keep from getting pregnant during treatment and for at least 1 month after the last dose. If you think you have become pregnant while using the medicine, tell your doctor right away. Make sure any doctor or dentist who treats you knows that you are using this medicine. You may need to stop using this medicine several days before having surgery or medical tests. Check with your doctor immediately if you have diarrhea, fever, or any symptoms of an infection. This medicine may cause skin necrosis or gangrene. Call your doctor right away if you have pain, a color change, or a temperature change to any area of your body. Call your doctor right ...
Combination of probenecid-sulphadoxine-pyrimethamine for intermittent preventive treatment in pregnancy | Malaria Journal |...
The antifolate sulphadoxine-pyrimethamine (SP) has been used in the intermittent prevention of malaria in pregnancy (IPTp). SP is an ideal choice for IPTp, however, as resistance of Plasmodium falciparum to SP increases, data are accumulating that SP may no longer provide benefit in areas of high-level resistance. Probenecid was initially used as an adjunctive therapy to increase the blood concentration of penicillin; it has since been used to augment concentrations of other drugs, including antifolates. The addition of probenecid has been shown to increase the treatment efficacy of SP against malaria, suggesting that the combination of probenecid plus SP may prolong the useful lifespan of SP as an effective agent for IPTp. Here, the literature on the pharmacokinetics, adverse reactions, interactions and available data on the use of these drugs in pregnancy is reviewed, and the possible utility of an SP-probenecid combination is discussed. This article concludes by calling for further research into this