MYOCARDIAL INFARCTION in which the anterior wall of the heart is involved. Anterior wall myocardial infarction is often caused by occlusion of the left anterior descending coronary artery. It can be categorized as anteroseptal or anterolateral wall myocardial infarction.
MYOCARDIAL INFARCTION in which the inferior wall of the heart is involved. It is often caused by occlusion of the right coronary artery.
NECROSIS of the MYOCARDIUM caused by an obstruction of the blood supply to the heart (CORONARY CIRCULATION).
Radiography of the vascular system of the heart muscle after injection of a contrast medium.
The hemodynamic and electrophysiological action of the left HEART VENTRICLE. Its measurement is an important aspect of the clinical evaluation of patients with heart disease to determine the effects of the disease on cardiac performance.
Recording of the moment-to-moment electromotive forces of the HEART as projected onto various sites on the body's surface, delineated as a scalar function of time. The recording is monitored by a tracing on slow moving chart paper or by observing it on a cardioscope, which is a CATHODE RAY TUBE DISPLAY.
Laceration or tearing of cardiac tissues appearing after MYOCARDIAL INFARCTION.
The lower right and left chambers of the heart. The right ventricle pumps venous BLOOD into the LUNGS and the left ventricle pumps oxygenated blood into the systemic arterial circulation.
Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues. The standard approach is transthoracic.
The veins and arteries of the HEART.
Formation of an infarct, which is NECROSIS in tissue due to local ISCHEMIA resulting from obstruction of BLOOD CIRCULATION, most commonly by a THROMBUS or EMBOLUS.
Generally, restoration of blood supply to heart tissue which is ischemic due to decrease in normal blood supply. The decrease may result from any source including atherosclerotic obstruction, narrowing of the artery, or surgical clamping. Reperfusion can be induced to treat ischemia. Methods include chemical dissolution of an occluding thrombus, administration of vasodilator drugs, angioplasty, catheterization, and artery bypass graft surgery. However, it is thought that reperfusion can itself further damage the ischemic tissue, causing MYOCARDIAL REPERFUSION INJURY.
Dilation of an occluded coronary artery (or arteries) by means of a balloon catheter to restore myocardial blood supply.
The outermost layer of a cell in most PLANTS; BACTERIA; FUNGI; and ALGAE. The cell wall is usually a rigid structure that lies external to the CELL MEMBRANE, and provides a protective barrier against physical or chemical agents.
Elements of limited time intervals, contributing to particular results or situations.
The formation of an area of NECROSIS in the CEREBRUM caused by an insufficiency of arterial or venous blood flow. Infarcts of the cerebrum are generally classified by hemisphere (i.e., left vs. right), lobe (e.g., frontal lobe infarction), arterial distribution (e.g., INFARCTION, ANTERIOR CEREBRAL ARTERY), and etiology (e.g., embolic infarction).
The geometric and structural changes that the HEART VENTRICLES undergo, usually following MYOCARDIAL INFARCTION. It comprises expansion of the infarct and dilatation of the healthy ventricle segments. While most prevalent in the left ventricle, it can also occur in the right ventricle.
Use of infusions of FIBRINOLYTIC AGENTS to destroy or dissolve thrombi in blood vessels or bypass grafts.
The circulation of blood through the CORONARY VESSELS of the HEART.
Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.
Observation of a population for a sufficient number of persons over a sufficient number of years to generate incidence or mortality rates subsequent to the selection of the study group.
The muscle tissue of the HEART. It is composed of striated, involuntary muscle cells (MYOCYTES, CARDIAC) connected to form the contractile pump to generate blood flow.
An aspect of personal behavior or lifestyle, environmental exposure, or inborn or inherited characteristic, which, on the basis of epidemiologic evidence, is known to be associated with a health-related condition considered important to prevent.
An imbalance between myocardial functional requirements and the capacity of the CORONARY VESSELS to supply sufficient blood flow. It is a form of MYOCARDIAL ISCHEMIA (insufficient blood supply to the heart muscle) caused by a decreased capacity of the coronary vessels.
A condition in which the LEFT VENTRICLE of the heart was functionally impaired. This condition usually leads to HEART FAILURE; MYOCARDIAL INFARCTION; and other cardiovascular complications. Diagnosis is made by measuring the diminished ejection fraction and a depressed level of motility of the left ventricular wall.
A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (CORONARY ARTERY DISEASE), to obstruction by a thrombus (CORONARY THROMBOSIS), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (MYOCARDIAL INFARCTION).
The hollow, muscular organ that maintains the circulation of the blood.
The amount of BLOOD pumped out of the HEART per beat, not to be confused with cardiac output (volume/time). It is calculated as the difference between the end-diastolic volume and the end-systolic volume.
In screening and diagnostic tests, the probability that a person with a positive test is a true positive (i.e., has the disease), is referred to as the predictive value of a positive test; whereas, the predictive value of a negative test is the probability that the person with a negative test does not have the disease. Predictive value is related to the sensitivity and specificity of the test.
Streptococcal fibrinolysin . An enzyme produced by hemolytic streptococci. It hydrolyzes amide linkages and serves as an activator of plasminogen. It is used in thrombolytic therapy and is used also in mixtures with streptodornase (STREPTODORNASE AND STREPTOKINASE). EC 3.4.-.
Studies in which individuals or populations are followed to assess the outcome of exposures, procedures, or effects of a characteristic, e.g., occurrence of disease.
A transferase that catalyzes formation of PHOSPHOCREATINE from ATP + CREATINE. The reaction stores ATP energy as phosphocreatine. Three cytoplasmic ISOENZYMES have been identified in human tissues: the MM type from SKELETAL MUSCLE, the MB type from myocardial tissue and the BB type from nervous tissue as well as a mitochondrial isoenzyme. Macro-creatine kinase refers to creatine kinase complexed with other serum proteins.
A prediction of the probable outcome of a disease based on a individual's condition and the usual course of the disease as seen in similar situations.
Procedures in which placement of CARDIAC CATHETERS is performed for therapeutic or diagnostic procedures.
Contractile activity of the MYOCARDIUM.
The hospital unit in which patients with acute cardiac disorders receive intensive care.
A HERNIA-like condition in which the weakened pelvic muscles cause the URINARY BLADDER to drop from its normal position. Fallen urinary bladder is more common in females with the bladder dropping into the VAGINA and less common in males with the bladder dropping into the SCROTUM.
Fibrinolysin or agents that convert plasminogen to FIBRINOLYSIN.
Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.
Surgical therapy of ischemic coronary artery disease achieved by grafting a section of saphenous vein, internal mammary artery, or other substitute between the aorta and the obstructed coronary artery distal to the obstructive lesion.
The symptom of paroxysmal pain consequent to MYOCARDIAL ISCHEMIA usually of distinctive character, location and radiation. It is thought to be provoked by a transient stressful situation during which the oxygen requirements of the MYOCARDIUM exceed that supplied by the CORONARY CIRCULATION.
A method of computed tomography that uses radionuclides which emit a single photon of a given energy. The camera is rotated 180 or 360 degrees around the patient to capture images at multiple positions along the arc. The computer is then used to reconstruct the transaxial, sagittal, and coronal images from the 3-dimensional distribution of radionuclides in the organ. The advantages of SPECT are that it can be used to observe biochemical and physiological processes as well as size and volume of the organ. The disadvantage is that, unlike positron-emission tomography where the positron-electron annihilation results in the emission of 2 photons at 180 degrees from each other, SPECT requires physical collimation to line up the photons, which results in the loss of many available photons and hence degrades the image.
Drugs or agents which antagonize or impair any mechanism leading to blood platelet aggregation, whether during the phases of activation and shape change or following the dense-granule release reaction and stimulation of the prostaglandin-thromboxane system.
Motion pictures of the passage of contrast medium through blood vessels.
Precordial pain at rest, which may precede a MYOCARDIAL INFARCTION.
The movement and the forces involved in the movement of the blood through the CARDIOVASCULAR SYSTEM.
Devices that provide support for tubular structures that are being anastomosed or for body cavities during skin grafting.
The restoration of blood supply to the myocardium. (From Dorland, 28th ed)
A family of percutaneous techniques that are used to manage CORONARY OCCLUSION, including standard balloon angioplasty (PERCUTANEOUS TRANSLUMINAL CORONARY ANGIOPLASTY), the placement of intracoronary STENTS, and atheroablative technologies (e.g., ATHERECTOMY; ENDARTERECTOMY; THROMBECTOMY; PERCUTANEOUS TRANSLUMINAL LASER ANGIOPLASTY). PTCA was the dominant form of PCI, before the widespread use of stenting.
The return of a sign, symptom, or disease after a remission.
Coagulation of blood in any of the CORONARY VESSELS. The presence of a blood clot (THROMBUS) often leads to MYOCARDIAL INFARCTION.
This structure includes the thin muscular atrial septum between the two HEART ATRIA, and the thick muscular ventricular septum between the two HEART VENTRICLES.
Pathological processes of CORONARY ARTERIES that may derive from a congenital abnormality, atherosclerotic, or non-atherosclerotic cause.
Tomography using x-ray transmission and a computer algorithm to reconstruct the image.
The qualitative or quantitative estimation of the likelihood of adverse effects that may result from exposure to specified health hazards or from the absence of beneficial influences. (Last, Dictionary of Epidemiology, 1988)
Shock resulting from diminution of cardiac output in heart disease.
A vital statistic measuring or recording the rate of death from any cause in hospitalized populations.
Downward displacement of the UTERUS. It is classified in various degrees: in the first degree the UTERINE CERVIX is within the vaginal orifice; in the second degree the cervix is outside the orifice; in the third degree the entire uterus is outside the orifice.
Damage to the MYOCARDIUM resulting from MYOCARDIAL REPERFUSION (restoration of blood flow to ischemic areas of the HEART.) Reperfusion takes place when there is spontaneous thrombolysis, THROMBOLYTIC THERAPY, collateral flow from other coronary vascular beds, or reversal of vasospasm.
Disease having a short and relatively severe course.

Usefulness of the index of microcirculatory resistance for invasively assessing myocardial viability immediately after primary angioplasty for anterior myocardial infarction. (1/56)

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Dual left anterior descending artery distribution. (2/56)

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Low adiponectin blood concentration predicts left ventricular remodeling after ST-segment elevation myocardial infarction treated with primary percutaneous coronary intervention. (3/56)

BACKGROUND: Left ventricular remodeling (LVR), an increase in left ventricular end-diastolic volume index > or = 20%, is an adverse consequence of myocardial infarction. The aim of this study was to assess the association between LVR and adiponectin, which has been shown to protect against myocardial ischemia-reperfusion injury. METHODS: In 75 patients echocardiographic examination was performed one year after ST-segment elevation myocardial infarction, successfully treated with primary percutaneous coronary intervention (pPCI). Two groups of patients were analyzed: those with LVR (n = 15) and those without LVR (n = 60). RESULTS: The predictors of LVR were: anterior myocardial infarction, glucose at admission, baseline C-reactive protein, adiponectin, and echocardiographic parameters: left ventricular end-diastolic and end-systolic volume indices, ejection fraction < 40% and left ventricular wall motion score index (WMSI) at discharge. On multivariable regression analysis, lower adiponectin level (OR = 0.67, 95% CI 0.49-0.91, p < 0.05) and higher WMSI (OR = 20.14, 95% CI 2.62-154.82, p < 0.01) were the only independent negative predictors of LVR. The optimal cut-off for adiponectin for predicting LVR was < or = 4.7 mg/mL (sensitivity: 73%, specificity: 85%) and this level increased the risk of LVR 15-fold (95% CI 4.05-59.87, p = 0.0001). CONCLUSIONS: Baseline low blood adiponectin concentration, along with WMSI, can be considered as a predictor of the LVR in male patients one year after myocardial infarction and pPCI.  (+info)

Dor procedure for dyskinetic anteroapical myocardial infarction fails to improve contractility in the border zone. (4/56)

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Right ventricular involvement in anterior myocardial infarction: a translational approach. (5/56)

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Retrograde approach to a totally occluded right coronary artery via a septal perforator artery: the tale of a long and winding wire. (6/56)

Retrograde recannalization of chronic total occlusions has developed as a viable alternative to restore coronary patency. Techniques continue to evolve and complications described. We present a new complication related to equipment developed to improve outcomes via a retrograde approach.  (+info)

Complete atrioventricular block complicating acute anterior myocardial infarction can be reversed with acute coronary angioplasty. (7/56)

INTRODUCTION: A retrospective case series of acute anterior myocardial infarction (MI) patients complicated by complete atrioventricular block (AVB) treated with acute percutaneous transluminal coronary angioplasty (PTCA). CLINICAL PICTURE: Eight patients with anterior MI and complete AVB underwent acute PTCA between 2000 and 2005. Mean onset of complete AVB was 16.6 +/- 16.9 hours from chest pain onset. TREATMENT: All patients underwent successful PTCA to the left anterior descending artery. OUTCOME: Complete AVB resolved with PTCA in 88%; mean time of resolution was 89 +/- 144 minutes after revascularisation. One patient had permanent pacemaker implanted at Day 12 after developing an 8-second ventricular standstill during hospitalisation but not pacing-dependent on follow-up. The rhythm on discharge for the other surviving patients was normal sinus rhythm. CONCLUSION: This case series suggests that complete AVB complicating anterior MI is reversible with acute PTCA and survivors are not at increased risk of recurrent AVB. Nevertheless, this condition is associated with extensive myocardial damage and high mortality during the acute hospitalisation was not improved with correction of AVB with temporary pacing.  (+info)

Coronary flow velocity pattern and recovery of regional left ventricular function: the relationship observed in patients with reperfused acute myocardial infarction. (8/56)

Coronary flow velocity pattern (CFVP) recorded within 3 days of percutaneous coronary intervention (PCI) has been reported to be useful in predicting left ventricular (LV) function. The aim of this prospective study was to investigate, via transthoracic Doppler echocardiography, whether the relationship between CFVP and recovery of LV function persists. Our study group comprised 37 patients with 1st anterior-wall acute myocardial infarction who underwent successful PCI for lesions in the left anterior descending coronary artery (LAD). The CFVP in the LAD was recorded at 24-48 hours, 7 days, and 4 weeks after PCI. Myocardial contrast echocardiography was performed at 24-48 hours after PCI. The diastolic deceleration time (DDT) at each stage correlated significantly with the regional LV wall-motion score index at 6-month follow-up (r=-0.58 at 24-48 hr, -0.57 at day 7, and -0.50 at week 4; P <0.01 for all). The mean DDT increased over time. Optimal cutoff values for DDT to predict regional LV wall-motion score indices of <2.0 were 327 ms at 24-48 hours (sensitivity, 0.78; specificity, 0.64), 495 ms at day 7 (sensitivity, 0.75; specificity, 0.69), and 525 ms at week 4 (sensitivity, 0.83; specificity, 0.69). The DDT at 24-48 hours significantly correlated, better than the peak creatine kinase value, with reperfusion (r=0.68, P <0.01) as defined by myocardial contrast echocardiography. In conclusion, CFVP in the LAD can be used, within 4 weeks after PCI, to predict the recovery of regional LV function in patients with reperfused anterior-wall acute myocardial infarction.  (+info)

Example sentences:

1. The patient was diagnosed with an anterior wall myocardial infarction after experiencing chest pain and shortness of breath.
2. The anterior wall myocardial infarction was caused by a blockage in the left anterior descending coronary artery, which supplies blood to the front wall of the heart.
3. The patient underwent urgent angioplasty to open up the blocked artery and restore blood flow to the affected area, reducing the risk of further damage to the heart muscle.

Symptoms of Inferior Wall Myocardial Infarction:

* Chest pain or discomfort that may radiate to the arm, neck, jaw, or back
* Shortness of breath
* Fatigue
* Lightheadedness or dizziness
* Palpitations

Diagnosis of Inferior Wall Myocardial Infarction:

* Electrocardiogram (ECG) to detect abnormal heart rhythms and determine the location of the infarction
* Blood tests to check for cardiac enzymes, such as troponin, which are released when the heart muscle is damaged
* Echocardiogram or cardiac magnetic resonance imaging (MRI) to visualize the heart and detect any damage to the heart muscle

Treatment of Inferior Wall Myocardial Infarction:

* Medications to dissolve blood clots, reduce inflammation, and manage pain
* Angiography or angioplasty to open up the blocked coronary artery
* Cardiac rehabilitation to improve cardiovascular health and prevent future heart problems.

There are different types of myocardial infarctions, including:

1. ST-segment elevation myocardial infarction (STEMI): This is the most severe type of heart attack, where a large area of the heart muscle is damaged. It is characterized by a specific pattern on an electrocardiogram (ECG) called the ST segment.
2. Non-ST-segment elevation myocardial infarction (NSTEMI): This type of heart attack is less severe than STEMI, and the damage to the heart muscle may not be as extensive. It is characterized by a smaller area of damage or a different pattern on an ECG.
3. Incomplete myocardial infarction: This type of heart attack is when there is some damage to the heart muscle but not a complete blockage of blood flow.
4. Collateral circulation myocardial infarction: This type of heart attack occurs when there are existing collateral vessels that bypass the blocked coronary artery, which reduces the amount of damage to the heart muscle.

Symptoms of a myocardial infarction can include chest pain or discomfort, shortness of breath, lightheadedness, and fatigue. These symptoms may be accompanied by anxiety, fear, and a sense of impending doom. In some cases, there may be no noticeable symptoms at all.

Diagnosis of myocardial infarction is typically made based on a combination of physical examination findings, medical history, and diagnostic tests such as an electrocardiogram (ECG), cardiac enzyme tests, and imaging studies like echocardiography or cardiac magnetic resonance imaging.

Treatment of myocardial infarction usually involves medications to relieve pain, reduce the amount of work the heart has to do, and prevent further damage to the heart muscle. These may include aspirin, beta blockers, ACE inhibitors or angiotensin receptor blockers, and statins. In some cases, a procedure such as angioplasty or coronary artery bypass surgery may be necessary to restore blood flow to the affected area.

Prevention of myocardial infarction involves managing risk factors such as high blood pressure, high cholesterol, smoking, diabetes, and obesity. This can include lifestyle changes such as a healthy diet, regular exercise, and stress reduction, as well as medications to control these conditions. Early detection and treatment of heart disease can help prevent myocardial infarction from occurring in the first place.

The risk of post-infarction heart rupture is highest in individuals who have had a large heart attack, particularly if it involves the left ventricle, which is the main pumping chamber of the heart. Other risk factors include older age, diabetes, high blood pressure, and prior history of heart disease.

Symptoms of post-infarction heart rupture can include chest pain, shortness of breath, fatigue, and swelling of the legs. In some cases, there may be no symptoms at all, and the condition is diagnosed incidentally on a medical imaging test.

Post-infarction heart rupture is typically diagnosed using echocardiography, a non-invasive imaging test that uses sound waves to visualize the heart. Other diagnostic tests that may be used include electrocardiogram (ECG), chest X-ray, and cardiac magnetic resonance imaging (MRI).

Treatment of post-infarction heart rupture usually involves supportive care, such as oxygen therapy, pain management, and medication to manage symptoms. In some cases, surgery may be necessary to repair the damaged heart tissue or to insert a device to help the heart pump more effectively.

Overall, post-infarction heart rupture is a serious complication of heart attacks that can have significant morbidity and mortality. Prompt recognition and treatment are essential to improve outcomes for patients with this condition.

The term "infarction" is derived from the Latin words "in" meaning "into" and "farcire" meaning "to stuff", which refers to the idea that the tissue becomes "stuffed" with blood, leading to cell death and necrosis.

Infarction can be caused by a variety of factors, including atherosclerosis (the buildup of plaque in the blood vessels), embolism (a blood clot or other foreign material that blocks the flow of blood), and vasospasm (constriction of the blood vessels).

The symptoms of infarction vary depending on the location and severity of the blockage, but can include chest pain or discomfort, shortness of breath, numbness or weakness in the affected limbs, and confusion or difficulty speaking or understanding speech.

Diagnosis of infarction typically involves imaging tests such as electrocardiograms (ECGs), echocardiograms, or computerized tomography (CT) scans to confirm the presence of a blockage and assess the extent of the damage. Treatment options for infarction include medications to dissolve blood clots, surgery to restore blood flow, and other interventions to manage symptoms and prevent complications.

Prevention of infarction involves managing risk factors such as high blood pressure, high cholesterol, smoking, and obesity, as well as maintaining a healthy diet and exercise routine. Early detection and treatment of blockages can help reduce the risk of infarction and minimize the damage to affected tissues.

Cerebral infarction can result in a range of symptoms, including sudden weakness or numbness in the face, arm, or leg on one side of the body, difficulty speaking or understanding speech, sudden vision loss, dizziness, and confusion. Depending on the location and severity of the infarction, it can lead to long-term disability or even death.

There are several types of cerebral infarction, including:

1. Ischemic stroke: This is the most common type of cerebral infarction, accounting for around 87% of all cases. It occurs when a blood clot blocks the flow of blood to the brain, leading to cell death and tissue damage.
2. Hemorrhagic stroke: This type of cerebral infarction occurs when a blood vessel in the brain ruptures, leading to bleeding and cell death.
3. Lacunar infarction: This type of cerebral infarction affects the deep structures of the brain, particularly the basal ganglia, and is often caused by small blockages or stenosis (narrowing) in the blood vessels.
4. Territorial infarction: This type of cerebral infarction occurs when there is a complete blockage of a blood vessel that supplies a specific area of the brain, leading to cell death and tissue damage in that area.

Diagnosis of cerebral infarction typically involves a combination of physical examination, medical history, and imaging tests such as CT or MRI scans. Treatment options vary depending on the cause and location of the infarction, but may include medication to dissolve blood clots, surgery to remove blockages, or supportive care to manage symptoms and prevent complications.

During ventricular remodeling, the heart muscle becomes thicker and less flexible, leading to a decrease in the heart's ability to fill with blood and pump it out to the body. This can lead to shortness of breath, fatigue, and swelling in the legs and feet.

Ventricular remodeling is a natural response to injury, but it can also be exacerbated by factors such as high blood pressure, diabetes, and obesity. Treatment for ventricular remodeling typically involves medications and lifestyle changes, such as exercise and a healthy diet, to help manage symptoms and slow the progression of the condition. In some cases, surgery or other procedures may be necessary to repair or replace damaged heart tissue.

The process of ventricular remodeling is complex and involves multiple cellular and molecular mechanisms. It is thought to be driven by a variety of factors, including changes in gene expression, inflammation, and the activity of various signaling pathways.

Overall, ventricular remodeling is an important condition that can have significant consequences for patients with heart disease. Understanding its causes and mechanisms is crucial for developing effective treatments and improving outcomes for those affected by this condition.

Coronary disease is often caused by a combination of genetic and lifestyle factors, such as high blood pressure, high cholesterol levels, smoking, obesity, and a lack of physical activity. It can also be triggered by other medical conditions, such as diabetes and kidney disease.

The symptoms of coronary disease can vary depending on the severity of the condition, but may include:

* Chest pain or discomfort (angina)
* Shortness of breath
* Fatigue
* Swelling of the legs and feet
* Pain in the arms and back

Coronary disease is typically diagnosed through a combination of physical examination, medical history, and diagnostic tests such as electrocardiograms (ECGs), stress tests, and cardiac imaging. Treatment for coronary disease may include lifestyle changes, medications to control symptoms, and surgical procedures such as angioplasty or bypass surgery to improve blood flow to the heart.

Preventative measures for coronary disease include:

* Maintaining a healthy diet and exercise routine
* Quitting smoking and limiting alcohol consumption
* Managing high blood pressure, high cholesterol levels, and other underlying medical conditions
* Reducing stress through relaxation techniques or therapy.

There are several potential causes of LVD, including:

1. Coronary artery disease: The buildup of plaque in the coronary arteries can lead to a heart attack, which can damage the left ventricle and impair its ability to function properly.
2. Heart failure: When the heart is unable to pump enough blood to meet the body's needs, it can lead to LVD.
3. Cardiomyopathy: This is a condition where the heart muscle becomes weakened or enlarged, leading to impaired function of the left ventricle.
4. Heart valve disease: Problems with the heart valves can disrupt the normal flow of blood and cause LVD.
5. Hypertension: High blood pressure can cause damage to the heart muscle and lead to LVD.
6. Genetic factors: Some people may be born with genetic mutations that predispose them to developing LVD.
7. Viral infections: Certain viral infections, such as myocarditis, can inflame and damage the heart muscle, leading to LVD.
8. Alcohol or drug abuse: Substance abuse can damage the heart muscle and lead to LVD.
9. Nutritional deficiencies: A diet lacking essential nutrients can lead to damage to the heart muscle and increase the risk of LVD.

Diagnosis of LVD typically involves a physical exam, medical history, and results of diagnostic tests such as electrocardiograms (ECGs), echocardiograms, and stress tests. Treatment options for LVD depend on the underlying cause, but may include medications to improve cardiac function, lifestyle changes, and in severe cases, surgery or other procedures.

Preventing LVD involves taking steps to maintain a healthy heart and reducing risk factors such as high blood pressure, smoking, and obesity. This can be achieved through a balanced diet, regular exercise, stress management, and avoiding substance abuse. Early detection and treatment of underlying conditions that increase the risk of LVD can also help prevent the condition from developing.

Myocardial ischemia can be caused by a variety of factors, including coronary artery disease, high blood pressure, diabetes, and smoking. It can also be triggered by physical exertion or stress.

There are several types of myocardial ischemia, including:

1. Stable angina: This is the most common type of myocardial ischemia, and it is characterized by a predictable pattern of chest pain that occurs during physical activity or emotional stress.
2. Unstable angina: This is a more severe type of myocardial ischemia that can occur without any identifiable trigger, and can be accompanied by other symptoms such as shortness of breath or vomiting.
3. Acute coronary syndrome (ACS): This is a condition that includes both stable angina and unstable angina, and it is characterized by a sudden reduction in blood flow to the heart muscle.
4. Heart attack (myocardial infarction): This is a type of myocardial ischemia that occurs when the blood flow to the heart muscle is completely blocked, resulting in damage or death of the cardiac tissue.

Myocardial ischemia can be diagnosed through a variety of tests, including electrocardiograms (ECGs), stress tests, and imaging studies such as echocardiography or cardiac magnetic resonance imaging (MRI). Treatment options for myocardial ischemia include medications such as nitrates, beta blockers, and calcium channel blockers, as well as lifestyle changes such as quitting smoking, losing weight, and exercising regularly. In severe cases, surgical procedures such as coronary artery bypass grafting or angioplasty may be necessary.

Symptoms of cystocele may include:

* A bulge in the vagina that may be felt through the skin
* Pain or discomfort during sexual activity
* Difficulty starting a stream of urine
* Frequent urination
* Increased urgency to urinate
* Leaking of urine

Diagnosis of cystocele is typically made through a physical exam and may also involve imaging tests such as ultrasound or MRI. Treatment for cystocele depends on the severity of the condition and may include:

* Kegel exercises to strengthen the muscles that support the bladder
* A pessary, which is a device inserted into the vagina to support the bladder
* Surgery to repair or remove the damaged tissue

It's important for individuals experiencing symptoms of cystocele to consult with a healthcare provider for proper diagnosis and treatment.

Angina pectoris is a medical condition that is characterized by recurring chest pain or discomfort due to reduced blood flow and oxygen supply to the heart muscle, specifically the myocardium. It is also known as stable angina or effort angina. The symptoms of angina pectoris typically occur during physical activity or emotional stress and are relieved by rest.

The term "angina" comes from the Latin word for "strangulation," which refers to the feeling of tightness or constriction in the chest that is associated with the condition. Angina pectoris can be caused by atherosclerosis, or the buildup of plaque in the coronary arteries, which supply blood to the heart muscle. This buildup can lead to the formation of atherosclerotic plaques that can narrow the coronary arteries and reduce blood flow to the heart muscle, causing chest pain.

There are several types of angina pectoris, including:

1. Stable angina: This is the most common type of angina and is characterized by predictable and reproducible symptoms that occur during specific situations or activities, such as exercise or emotional stress.
2. Unstable angina: This type of angina is characterized by unpredictable and changing symptoms that can occur at rest or with minimal exertion. It is often a sign of a more severe underlying condition, such as a heart attack.
3. Variant angina: This type of angina occurs during physical activity, but the symptoms are not relieved by rest.
4. Prinzmetal's angina: This is a rare type of angina that occurs at rest and is characterized by a feeling of tightness or constriction in the chest.

The diagnosis of angina pectoris is typically made based on a combination of physical examination, medical history, and diagnostic tests such as electrocardiogram (ECG), stress test, and imaging studies. Treatment for angina pectoris usually involves lifestyle modifications, such as regular exercise, a healthy diet, and stress management, as well as medications to relieve symptoms and reduce the risk of complications. In some cases, surgery or other procedures may be necessary to treat the underlying condition causing the angina.

Word origin: Greek "anginos" meaning "pain in the neck".

Recurrence can also refer to the re-emergence of symptoms in a previously treated condition, such as a chronic pain condition that returns after a period of remission.

In medical research, recurrence is often studied to understand the underlying causes of disease progression and to develop new treatments and interventions to prevent or delay its return.

Coronary Thrombosis can cause a range of symptoms including chest pain, shortness of breath, lightheadedness and fatigue. The severity of the symptoms depends on the location and size of the clot. In some cases, the condition may be asymptomatic and diagnosed incidentally during a medical examination or imaging test.

Diagnosis of Coronary Thrombosis is typically made using electrocardiogram (ECG), blood tests and imaging studies such as angiography or echocardiography. Treatment options include medications to dissolve the clot, surgery to open or bypass the blocked artery or other interventional procedures such as angioplasty or stenting.

Prevention of Coronary Thrombosis includes managing risk factors such as high blood pressure, high cholesterol levels, smoking and diabetes through lifestyle changes and medications. Early detection and treatment can help reduce the risk of complications and improve outcomes for patients with this condition.

The buildup of plaque in the coronary arteries is often caused by high levels of low-density lipoprotein (LDL) cholesterol, smoking, high blood pressure, diabetes, and a family history of heart disease. The plaque can also rupture, causing a blood clot to form, which can completely block the flow of blood to the heart muscle, leading to a heart attack.

CAD is the most common type of heart disease and is often asymptomatic until a serious event occurs. Risk factors for CAD include:

* Age (men over 45 and women over 55)
* Gender (men are at greater risk than women, but women are more likely to die from CAD)
* Family history of heart disease
* High blood pressure
* High cholesterol
* Diabetes
* Smoking
* Obesity
* Lack of exercise

Diagnosis of CAD typically involves a physical exam, medical history, and results of diagnostic tests such as:

* Electrocardiogram (ECG or EKG)
* Stress test
* Echocardiogram
* Coronary angiography

Treatment for CAD may include lifestyle changes such as a healthy diet, regular exercise, stress management, and quitting smoking. Medications such as beta blockers, ACE inhibitors, and statins may also be prescribed to manage symptoms and slow the progression of the disease. In severe cases, surgical intervention such as coronary artery bypass grafting (CABG) or percutaneous coronary intervention (PCI) may be necessary.

Prevention of CAD includes managing risk factors such as high blood pressure, high cholesterol, and diabetes, quitting smoking, maintaining a healthy weight, and getting regular exercise. Early detection and treatment of CAD can help to reduce the risk of complications and improve quality of life for those affected by the disease.

Signs and symptoms of cardiogenic shock may include:

* Shortness of breath
* Chest pain or discomfort
* Confusion or altered mental status
* Cool, clammy skin
* Weak or absent pulse in the arms and legs
* Rapid or irregular heartbeat
* Low blood pressure

Treatment of cardiogenic shock typically involves supportive care to help the heart pump more effectively, as well as medications to help improve blood flow and reduce inflammation. In some cases, a procedure called extracorporeal membrane oxygenation (ECMO) may be used to take over the work of the heart and lungs.

Cardiogenic shock can be caused by a variety of factors, including:

* Heart attack or myocardial infarction
* Heart failure or ventricular dysfunction
* Cardiac tamponade or fluid accumulation in the space around the heart
* Myocarditis or inflammation of the heart muscle
* Coronary artery disease or blockages in the blood vessels that supply the heart
* Other conditions that can cause damage to the heart, such as aortic dissection or endocarditis.

Treatment options for uterine prolapse include lifestyle changes such as exercise, weight loss, and pelvic floor exercises, as well as surgical procedures such as hysterectomy or vaginal repair. The choice of treatment depends on the severity of the condition and the individual's overall health status.

It is important to seek medical advice if symptoms persist or worsen over time, as uterine prolapse can lead to complications such as urinary incontinence, kidney damage, and bowel problems if left untreated. Early diagnosis and treatment can help prevent these complications and improve quality of life for individuals affected by the condition.

MRI can occur in various cardiovascular conditions, such as myocardial infarction (heart attack), cardiac arrest, and cardiac surgery. The severity of MRI can range from mild to severe, depending on the extent and duration of the ischemic event.

The pathophysiology of MRI involves a complex interplay of various cellular and molecular mechanisms. During ischemia, the heart muscle cells undergo changes in energy metabolism, electrolyte balance, and cell membrane function. When blood flow is restored, these changes can lead to an influx of calcium ions into the cells, activation of enzymes, and production of reactive oxygen species (ROS), which can damage the cells and their membranes.

The clinical presentation of MRI can vary depending on the severity of the injury. Some patients may experience chest pain, shortness of breath, and fatigue. Others may have more severe symptoms, such as cardiogenic shock or ventricular arrhythmias. The diagnosis of MRI is based on a combination of clinical findings, electrocardiography (ECG), echocardiography, and cardiac biomarkers.

The treatment of MRI is focused on addressing the underlying cause of the injury and managing its symptoms. For example, in patients with myocardial infarction, thrombolysis or percutaneous coronary intervention may be used to restore blood flow to the affected area. In patients with cardiac arrest, cardiopulmonary resuscitation (CPR) and other life-saving interventions may be necessary.

Prevention of MRI is crucial in reducing its incidence and severity. This involves aggressive risk factor management, such as controlling hypertension, diabetes, and dyslipidemia, as well as smoking cessation and stress reduction. Additionally, patients with a history of MI should adhere to their medication regimen, which may include beta blockers, ACE inhibitors or ARBs, statins, and aspirin.

In conclusion, myocardial injury with ST-segment elevation (MRI) is a life-threatening condition that requires prompt recognition and treatment. While the clinical presentation can vary depending on the severity of the injury, early diagnosis and management are crucial in reducing morbidity and mortality. Prevention through aggressive risk factor management and adherence to medication regimens is also essential in preventing MRI.

Examples of acute diseases include:

1. Common cold and flu
2. Pneumonia and bronchitis
3. Appendicitis and other abdominal emergencies
4. Heart attacks and strokes
5. Asthma attacks and allergic reactions
6. Skin infections and cellulitis
7. Urinary tract infections
8. Sinusitis and meningitis
9. Gastroenteritis and food poisoning
10. Sprains, strains, and fractures.

Acute diseases can be treated effectively with antibiotics, medications, or other therapies. However, if left untreated, they can lead to chronic conditions or complications that may require long-term care. Therefore, it is important to seek medical attention promptly if symptoms persist or worsen over time.

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Those who were treated without angiography will develop anterior wall myocardial infarction in a mean period of 9 days. An ... are highly suggestive of myocardial ischaemia. Other ECG changes associate with myocardial ischaemia are: ST segment depression ... The two main causes of these waves are myocardial ischemia and hypokalemia. Ischemic T waves rise and then fall below the ... Inversion of T waves in most of the ECG leads except aVR indicates many causes most commonly myocardial ischaemia and ...
Complications may include myocardial infarction and stroke. Relative to the general population the risk for certain types of ... anterior chest wall deformities as well as scoliosis. Patients are at a much greater risk for fractures than the general ...
An anterior wall myocardial infarction may damage the distal conduction system of the heart, causing third-degree heart block. ... In addition, acute myocardial infarction may present with third-degree AV block. An inferior wall myocardial infarction may ... Studies have shown that third-degree heart block in the setting of an inferior wall myocardial infarction typically resolves ... "Advanced early and late atrioventricular block in acute inferior wall myocardial infarction". American Heart Journal. 108 (1): ...
... of anterior-wall myocardial infarctions, compared with only 5% of inferior ones. Some other risk factors are poor ejection ... It is a major cause of infarction (tissue death from blockage of the blood supply). An embolus lodging in the brain from either ...
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The clinical significance of this is that a myocardial infarction involving the PDA is more likely to cause mitral ... The larger anterior interventricular artery, also known as the left anterior descending artery (LAD), is the second major ... There are three aortic sinuses (dilations) in the wall of the aorta just superior to the aortic semilunar valve. Two of these, ... Severe ischemia can cause the heart muscle to die from hypoxia, such as during a myocardial infarction. Chronic moderate ...
Deep to sternum and anterior chest wall. The right phrenic nerve passes to the right of the pericardium. The left phrenic nerve ... but may also occur following a myocardial infarction. Pericarditis is usually a short-lived condition that can be successfully ... The pericardium, also called pericardial sac, is a double-walled sac containing the heart and the roots of the great vessels. ...
These monocytes, upon moving to injured tissue (such as the heart after myocardial infarction), turn into dendritic cells and ... The other side of the spleen is divided by a ridge into two regions: an anterior gastric portion, and a posterior renal portion ... and is in contact with the posterior wall of the stomach. Below this it is in contact with the tail of the pancreas. The renal ... Release of neutrophils following myocardial infarction. Creation of red blood cells. While the bone marrow is the primary site ...
... and any abnormal motion of the heart walls as they beat that may indicate a myocardial infarction. The flow of blood can be ... Anterior infarcts, persistent ventricular tachycardia or fibrillation, development of heart blocks, and left ventricular ... Many risk factors of myocardial infarction are shared with coronary artery disease, the primary cause of myocardial infarction ... it may be classified as an ST elevation myocardial infarction (STEMI) or Non-ST elevation myocardial infarction (NSTEMI) based ...
The condition is characterized by tenderness and painful swelling of the anterior (front) chest wall at the costochondral (rib ... Due to its presentation, Tietze syndrome can be misdiagnosed as a number of conditions, including myocardial infarction (heart ... Tietze syndrome typically presents unilaterally at a single joint of the anterior chest wall, with 70% of patients having ... Though Tietze syndrome can be misdiagnosed, life-threatening conditions with similar symptoms such as myocardial infarction ( ...
Myocardial infarction can rarely present as referred pain and this usually occurs in people with diabetes or older age. Also, ... The CNS does not clearly discern whether the pain is coming from the body wall or from the viscera, but it perceives the pain ... For example, stimulated local pain in the anterior tibial muscle causes referred pain in the ventral portion of the ankle; ... An example is the case of angina pectoris brought on by a myocardial infarction (heart attack), where pain is often felt in the ...
Mc Loughlin, MJ (2020). "Precordial bipolar leads: A new method to study anterior acute myocardial infarction". J ... The left anterior descending (LAD) artery supplies the anterior wall of the heart, and therefore causes ST elevations in ... Chest pain or suspected myocardial infarction (heart attack), such as ST elevated myocardial infarction (STEMI) or non-ST ... ST elevation and ST depression High Frequency QRS changes Myocardial infarction (heart attack) Non-Q wave myocardial infarction ...
Mc Loughlin, MJ (2020). "Precordial bipolar leads: A new method to study anterior acute myocardial infarction". J ... Man, S.; Rahmattulla, C.; Maan, A.; Holman, E.; Bax, J.; van der Wall, E.; et al. (2012). "Role of the vectorcardiogram-derived ... Stein, Paul & Simon, Armando P. vectorcardiographic diagnosis of diaphragmatic myocardial infarction. The American Journal of ... infarction, coronary interventions) and total mortality. Independently, the SA was a stronger risk indicator of cardiac ...
... ischemia Non Q-wave myocardial infarction Reciprocal changes in acute Q-wave myocardial infarction (e.g., ST depression in ... The positive electrodes on the anterior chest wall detect the movement of positive charge away from the electrode and record it ... aVL with acute inferior myocardial infarction) ST segment depression and T-wave changes may be seen in patients with unstable ... It is often a sign of myocardial ischemia, of which coronary insufficiency is a major cause. Other ischemic heart diseases ...
Myocardial imaging usually demonstrates left ventricular dilation, severe ventricular dysfunction, and multiple infarctions. ... Some argue that only left main- or proximal-left anterior descending artery disease is relevant to the diagnostic criteria for ... 1225-. ISBN 978-0-323-29064-7. E. van der Wall; K.J. Lie (6 December 2012). Recent Views on Hypertrophic Cardiomyopathy. ... Typically, patients with ischemic cardiomyopathy have a history of acute myocardial infarction, however, it may occur in ...
Commonly a result of myocardial infarction that weakens the wall sufficiently to result in frank rupture and is typically seen ... Cerebral aneurysm - Aneurysms of the arteries in the brain most commonly affect the anterior cerebral artery. Rupture of the ... Myocardial infarction (a.k.a. heart attack) - A myocardial infarction is the death of a part of the heart which is typically ... The top three causes of ACS are ST elevation myocardial infarction (STEMI, 30%), non ST elevation myocardial infarction (NSTEMI ...
Many cases of myocardial infarction in young adults have now been attributed to Kawasaki disease that went undiagnosed during ... Anterior uveitis may be present under slit-lamp examination. Iritis can occur, too. Keratic precipitates are another eye ... Narrowing of the coronary artery, which occurs as a result of the healing process of the vessel wall, often leads to ... Death can occur either due to myocardial infarction secondary to blood clot formation in a coronary artery aneurysm or to ...
... causes of chest pain similar to that produced by costochondritis may include but are not limited to myocardial infarction, ... Pain is typically widespread and reproducible with palpation of the anterior (front) chest at the affected joints.: 171 Pain ... doi:10.1016/S0197-0070(81)80056-3. Ahmed HS, Shah KB, Pal DJ (2021). Atypical Chest Wall Pain. Interventional Management of ... Costochondritis, also known as chest wall pain syndrome or costosternal syndrome, is a benign inflammation of the upper ...
A small (20-40%) increased risk of acute myocardial infarction has also been described. Pulmonary arterial hypertension occurs ... The combination of anterior uveitis, parotitis, VII cranial nerve paralysis and fever is called uveoparotid fever or Heerfordt ... Joseph Rago, Pulitzer Prize-winning writer known for his work at The Wall Street Journal, died of sarcoidosis complications in ... "Risk of acute myocardial infarction in sarcoidosis: A population-based cohort study from Sweden". Respiratory Medicine. 188: ...
Stress-related factors include physical, exercise, hypoglycemia, myocardial infarction, and surgery. Coitus and sleep can also ... Prolactin (PRL) is a peptide hormone produced by lactotroph cells in the anterior pituitary gland. PRL is involved in lactation ... Physiological (i.e., non-pathological) causes include: ovulation, pregnancy, breastfeeding, chest wall injury, stress, stress- ...
The anterior thoracic wall, the airways and the pulmonary vessels anterior to the root of the lung have been digitally removed ... Chest pain may be a symptom of myocardial infarctions ('heart attack'). If this condition is present in the body, discomfort ... Anterior view. Thorax. Anterior view. Clearly visible thorax of an artistic gymnast. Pectus carinatum Pectus excavatum Thoracic ... Chest wall pain can be experienced after an increase in activity. Persons who add exercise to their daily routine generally ...
A dilated left ventricle is generally due to the effects of a myocardial infarction. An occlusion, or blockage, results in ... To begin a basic remodeling, the surgeon makes an incision at the center of the depressed area on the LV wall and removes blood ... Athanasuleas CL, Buckberg GD, Menicanti L, Gharib M (2001). "Optimizing ventricular shape in anterior restoration". Semin. ... Geometric derangement induced by nonviable myocardium (see myocardial infarction) is exponentially impacted and proportional to ...
These gap junctions can close to isolate damaged or dying tissue, as in a myocardial infarction (heart attack). Embryologic ... Although the ventricular stimulus originates from the AV node in the wall separating the atria and ventricles, the Bundle of ... The left bundle branch is short, splitting into the left anterior fascicle and the left posterior fascicle. The left posterior ... Like a neuron, a given myocardial cell has a negative membrane potential when at rest. Stimulation above a threshold value ...
If the myocardial infarction is treated with thrombolytic therapy, the mortality increases to over 70%, mostly due to bleeding ... A pleural effusion (fluid collection in the space between the lungs and the chest wall or diaphragm) can be due to either blood ... Anterior chest pain is associated with dissections involving the ascending aorta, while interscapular back pain is associated ... Mechanic O, Gavin M, Grossman S (9 March 2021). "Acute Myocardial Infarction". National Center for Biotechnology Information, U ...
Diagram of a myocardial infarction. Aorta and coronary arteries at autopsy. The proximal portion of the RCA and its ostium can ... The PDA supplies the inferior wall, ventricular septum, and the posteromedial papillary muscle. The RCA also supplies the SA ... "Anterior view of the heart." (Articles with TA98 identifiers, Arteries of the thorax). ...
"Left ventricular free wall rupture after reperfusion therapy for acute myocardial infarction". Am J Cardiol. 92 (3): 282-4. doi ... Within the left ventricle, these areas are adjacent to both anterior and posterior papillary muscles (regardless of whether the ... The incidence of myocardial rupture if PCI is performed in the setting of an acute myocardial infarction is about 1 percent. ... The most common cause of myocardial rupture is a recent myocardial infarction, with the rupture typically occurring three to ...
For example, chest pain described as extreme heaviness may indicate myocardial infarction, while chest pain described as ... Wall PD, Melzack R (1996). The challenge of pain (2nd ed.). New York: Penguin Books. ISBN 9780140256703. Ramachandran VS, ... These spinal cord fibers then cross the cord via the anterior white commissure and ascend in the spinothalamic tract. Before ... Is this patient having a myocardial infarction?". JAMA. 280 (14): 1256-63. doi:10.1001/jama.280.14.1256. PMID 9786377. Slater ...
... to improve delineation of left ventricle for visualizing contractibility of heart muscle after a myocardial infarction. Finally ... B-lines can occur in a healthy lung; however, the presence of 3 or more in the anterior or lateral lung regions is always ... Lung sliding indicates both that the lung is present at the chest wall and that the lung is functioning. § A-lines: When the ... The most common site for cranial ultrasound is the anterior fontanelle. The smaller the fontanelle, the more the image is ...
Diaphoresis is also seen in an acute myocardial infarction (heart attack), from the increased firing of the sympathetic nervous ... Sweating is controlled from a center in the preoptic and anterior regions of the brain's hypothalamus, where thermosensitive ... In addition, pneumothorax can cause diaphoresis with splinting of the chest wall. Neuroleptic malignant syndrome and other ...
... as well as ventricular aneurysms that follow transmural myocardial infarctions (aneurysms that involve all layers of the ... The wall strength of ruptured aneurysmal aortic wall was also found to be 54.2 N/cm2, which is much lower than that of a ... Examples include: Berry aneurysms of the anterior communicating artery of the circle of Willis, associated with autosomal ... The neck of the aneurysm is the most at risk due to the combination of a small wall thickness and high wall shear stress. When ...
Rare side effects are congestive heart failure, myocardial infarction, and hepatotoxicity. Diltiazem is one of the most common ... Diltiazem works by relaxing the smooth muscle in the walls of arteries, resulting in them opening and allowing blood to flow ... local anaesthetics and quinidine injected into the posterior and the anterior septal artery of the atrio-ventricular node ... Myocardial bridge Dysmenorrhea For supraventricular tachycardias (PSVT), diltiazem appears to be as effective as verapamil in ...
... a condition known as myocardial infarction or heart attack, this can cause heart failure or cardiac arrest and eventually death ... The smooth muscle lining the ureter walls continuously tighten and relax through a process called peristalsis to force urine ... This causes the anterior pituitary gland to release follicle-stimulating hormone (FSH) and luteinizing hormone (LH). FSH ...
... while anterior in the image also is the patient's anterior and vice versa. This left-right interchange corresponds to the view ... Bronchial wall thickening can be seen on lung CTs and generally (but not always) implies inflammation of the bronchi. An ... CT scanning of the head is typically used to detect infarction (stroke), tumors, calcifications, haemorrhage, and bone trauma. ... Williams, M.C.; Newby, D.E. (2016-08-01). "CT myocardial perfusion imaging: current status and future directions". Clinical ...
... is more commonly associated with anterior infarction because of left ventricular impairment; inferior infarction is ... An estimated 33% of persons with myocardial infarction in the United States do not present with chest pain, and carry a ... Marx JA, Hockberger RS, Walls RM, Biros MH, Danzl DF, Gausche-Hill M, Jagoda A, Ling L, Newton E, Zink BJ, Rosen P (2014). ... Other clues in the history can help lower the suspicion for myocardial infarction. These include chest pain described as "sharp ...
Cardiac disorders such as coronary heart disease, including myocardial infarction, heart failure, cardiomyopathy, and ... The problems may involve the interior walls of the heart, the heart valves, or the large blood vessels that lead to and from ... These include the great cardiac vein, the middle cardiac vein, the small cardiac vein and the anterior cardiac veins. As the ... myocardial infarction, and is one of the causes of sudden cardiac death. It is within the group of cardiovascular diseases of ...
At the neural tube stage, the anterior portion undergoes encephalization to generate or 'pattern' the basic form of the brain. ... Winslow R, Mundy A (23 January 2009). "First Embryonic Stem-Cell Trial Gets Approval from the FDA". The Wall Street Journal. " ... January 2003). "Autologous bone-marrow stem-cell transplantation for myocardial regeneration". Lancet. 361 (9351): 45-46. doi: ... and traumatic brain injury repair Learning disability due to congenital disorder Spinal cord injury repair Heart infarction ...
... or myocardial thickness (strength training). Ventricular hypertrophy, the thickening of the ventricular walls, is generally ... Persons who modify their behavior after myocardial infarction to include regular exercise have improved rates of survival. ... anterior cingulate cortex, supplementary motor area, posterior middle frontal gyrus, and left superior temporal lobe) and white ... including decreased activation of the anterior cingulated cortex. One of the possible mechanisms by which physical activity may ...
CVD includes coronary artery diseases (CAD) such as angina and myocardial infarction (commonly known as a heart attack). Other ... is a paired muscle running vertically on each side of the anterior wall of the human abdomen, as well as that of some other ... Anterior tibial artery - The anterior tibial artery of the leg carries blood to the anterior compartment of the leg and dorsal ... of the three flat muscles of the lateral anterior abdominal wall. Abdominal internal oblique muscle - A muscle of the abdominal ...
... myocardial infarction, heart failure, intermittent claudication, Raynaud's phenomenon, chronic kidney disease, and diabetes ... Because PDE5 is also present in the smooth muscle of the walls of the arterioles within the lungs, two PDE5 inhibitors, ... Since 2007 there has been evidence to suggest that PDE5 inhibitors can cause an anterior optic neuropathy, although the ... Rao AR, Thwaini A, Ahmed HU, Shergill IS, Minhas S (July 2007). "The phosphodiesterase inhibitors and non-arteritic anterior ...
Axford-Gatley R. A. and Wilson G. J. "The "border zone" in myocardial infarction: an ultrastructural study in the dog using an ... Anterior of the aorta to become the four pre-aortic ganglia: (celiac ganglion, superior mesenteric ganglion, inferior ... The cardiac neural crest cells have a number of functions including creation of the muscle and connective tissue walls of large ... Tamura's study of 2011 examined the fate of CNCCs after a heart attack (myocardial infarction) in young mice. The CNCCs in the ...
The most worrisome type is the proximal left anterior descending (LAD) MI, often referred to as the widow-maker infarction, ... Diagonals run along the surface of the heart and supply the lateral wall of the left ventricle and the anterolateral papillary ... and is therefore considered the most critical vessel in terms of myocardial blood supply. Widow maker is an alternative name ... The left anterior descending artery (also LAD, anterior interventricular branch of left coronary artery, or anterior descending ...
Non-ST-Segment Elevation Myocardial Infarction and Unstable Angina) Chapter 269: ST-Segment Elevation Myocardial Infarction ... Physiology of Anterior Pituitary Hormones Chapter 372: Hypopituitarism Chapter 373: Pituitary Tumor Syndromes Chapter 374: ... Disorders of Platelets and Vessel Wall Chapter 112: Coagulation Disorders Chapter 113: Arterial and Venous Thrombosis Chapter ...
... recent myocardial infarction, chronic myocardial infarction together with ejection fraction ... the stroke episode is classified as total anterior circulation infarct (TACI), partial anterior circulation infarct (PACI), ... various inflammatory diseases of the blood vessel wall (Takayasu arteritis, giant cell arteritis, vasculitis), noninflammatory ...
A VSD can also form a few days after a myocardial infarction (heart attack) due to mechanical tearing of the septal wall, ... Can be sub classified again based on the location into anterior, apical, posterior and mid Type: Gerbode also known as left ... A ventricular septal defect (VSD) is a defect in the ventricular septum, the wall dividing the left and right ventricles of the ...
... block associated with inferior wall myocardial infarction (MI), His-Purkinje block associated with anterior wall MI (see below) ... Myocardial infarction. Anterior wall MI can be associated with an infranodal complete AV block; this is an ominous finding. ... for the Other Korea Acute Myocardial Infarction Registry, Korea Working Group on Myocardial Infarction Investigators. Influence ... Data from the Harmonizing Outcomes with Revascularization and Stents in Acute Myocardial Infarction (HORIZONS-AMI) Trial, in ...
The electrocardiogram showed changes to the heart consistent with an acute anterior wall myocardial infarction (heart attack). ... Large recent anterior myocardial infarction. *Coronary artery disease *- Left anterior descending coronary artery: proximally ... 1992]. Modifiers of timing and possible triggers of acute myocardial infarction in the Thrombolysis in Myocardial Infarction ... The death certificate and the autopsy, completed by the City Medical Examiner, listed "acute myocardial infarction" due to " ...
The current status of stem cell therapies for patients with myocardial infarction is discussed from a bioengineering and ... trends in bioengineering to promote stem cell therapies for patients with myocardial infarction. We explain why the number of ... One million CSCs were infused into anterior wall infarcts, and 0.5 million cells were infused into infarcts in the left ... Stem cell therapies for myocardial infarction in clinical trials: bioengineering and biomaterial aspects. *Akon Higuchi. 1,2,3, ...
It is the result of defective fusion of the anterior chest wall. Depending on the location of the heart, ectopia cordis can be ... MYOCARDIAL INFARCTION; and SUDDEN CARDIAC DEATH. HN - 2008 MH - Ectopia Cordis UI - D054083 MN - C14.240.400.422 MN - C16.131. ... transient anginal episode but that does not usually result in MYOCARDIAL INFARCTION. HN - 2008 MH - Coronary Occlusion UI - ... ISCHEMIA and is associated with MYOCARDIAL INFARCTION and changes in microvasculature. HN - 2008 MH - Livedo Reticularis UI - ...
Original article: Galectin-3 predicts left ventricular remodelling after anterior-wall myocardial infarction treated by primary ... Assessment and classification of patients with myocardial injury and infarction in clinical practice (2 November, 2016) Andrew ...
... in patients with acute inferior wall MI. Methods: In 159 patients with I-STEMI, 127 (80%) had RCA occlusion and 32 (20%) had ... aVL helps to diagnose left circumflex artery as a culprit IRA in an acute inferior wall MI. ... study was to assess the role of ST segment depression in the limb leads aVR and aVL for the diagnosis of acute posterior wall ... infarction and the identification of infarct related artery (IRA) ...
... "acute anterior wall myocardial infarction" due to "coronary artery disease" (CAD) as the cause of death. No autopsy was ... As emergency angioplasty and stent placement of his left anterior descending coronary artery was started, the FF had an ...
Cardiac: myocardial ischaemia, coronary artery spasm, acute myocardial infarction (MI), atherosclerosis, myocarditis, ... the anterior wall is involved in most cases (77%) of cocaine induced MI.31 Chest pain and ECG changes are very common in ... Cocaine-induced myocardial infarction: an analysis and review of the literature. J Emerg Med1992;10:169-77. ... Myocardial infarction after cocaine use involves several mechanisms. It is related to the block of the re-uptake of ...
A 70-year-old man was admitted with a history of anterior wall Myocardial Infarction. He had been thrombolysed from a local ...
The most common site of rupture is the anterior wall. Myocardial rupture presents similar to cardiac tamponade, most frequently ... Myocardial rupture is a rare complication of acute myocardial infarction (MI), usually presenting with chest pain. ... Free Wall Myocardial Rupture and Ventricular Septal Defect in an Ambulatory Patient. ... Transthoracic echocardiography revealed an anterior, anterolateral akinesis, a ventricular septal defect, and free wall rupture ...
Frequency of Complications of anterior wall myocardial infarction. Pak Heart Journal 50(03), 190-193. ... Frequency and severity of pericardial effusion after acute myocardial infarction. Ali Z, Ahmad I, Sheikh S, Hameed S, Naveed T ... Pathology of acute myocardial infarction with particular reference to occlusive coronary thrombi. British Heart Journal 38, 659 ... Frequency and severity of pericardial effusion after acute myocardial infarction. Abid Ullah Shah, Muhammad Imran, Jafar Iqbal ...
Anterior Wall Myocardial Infarction 1 genes has been reported with Anterior Wall Myocardial Infarction ...
Infarctions, Anterolateral Myocardial. Infarctions, Anteroseptal Myocardial. Myocardial Infarction, Anterior Wall. Myocardial ... Myocardial Infarction, Anteroseptal Myocardial Infarctions, Anteroseptal Acute Anterior Wall Myocardial Infarction - Narrower ... MYOCARDIAL INFARCTION in which the anterior wall of the heart is involved. Anterior wall myocardial infarction is often caused ... MYOCARDIAL INFARCTION in which the anterior wall of the heart is involved. Anterior wall myocardial infarction is often caused ...
anterior wall myocardial infarction (3) * antidromic AV reentry tachycardia (1) * antitachycardia pacing (2) ...
The diagnosis is atrial tachycardia, ST elevation myocardial infarction (STEMI) of the anterior wall, QRS (electrical) ... consistent with an acute anterior wall STEMI. There is also beat-to-beat variation in the QRS complex amplitude (+,*), known as ...
... years old female without any conventional risk factors except obesity presented with acute anterior wall myocardial infarction ... Acute Myocardial Infarction in a Young Lady due to Vitamin B12 Deficiency Induced Hyperhomocysteinemia. Warkaa Al Shamkani, ... MI). Her echocardiography showed anterior wall hypokinesia with moderate left ventricular dysfunction. Angiography showed tight ... The use of balloon tip perfusion catheter presumably provides the initial insult with local vessel wall hypoxia. Once the ...
... tachycardia and tachyarrhythmia occurred more commonly with anterior wall myocardial infarction, while S. bradycardia and ... their nature and relation to site of infarction as well as prognostic value in patients of acute myocardial infarction proper ... Background: Myocardial infarction is one of the major causes of sudden death in present era and cardiac arrhythmias are common ... Study of various cardiac arrhythmias in patients of acute myocardial infarction.. Authors: Rathod, Sangita. Parmar, Pragnesh. ...
... ejection fraction recovering from supine exercise with resting function in anterior wall healed myocardial infarction (273 ... Effects of exercise training started within 2 weeks after acute myocardial infarction on myocardial perfusion and left ... Tetrofosmin imaging in the detection of myocardial viability in patients with previous myocardial infarction: Comparison with ... Technetium 99m furifosmin regional myocardial uptake in patients with previous myocardial infarction: Relation to thallium-201 ...
Heart attack or myocardial infarction (MI) is broadly of 2 types: anterior wall and inferior wall, depending on the wall of ... Newest myocardial-infarction questions feed Subscribe to RSS Newest myocardial-infarction questions feed To subscribe to this ... Cardiac events/Myocardial infarction from 1 clinic or 1 large city daily data Requesting help in finding a DAILY data about ... What would happen if you administered endothelin during a myocardial infarction? I have been going through the Rhodes medical ...
Two dimension echocardiographic visualization of free ventricular wall rupture in acute anterior myocardial infarction. Am. ... MANN, J. M. & ROBERTS, W. C. - Rupture of the left ventricular free wall during acute myocardial infarction: analysis of 138 ... TWINDALE, N.: HENRY, L.; MORPHETT, A.; TONKIN, A. M. - Hemorrhagic myocardial infarction complicated by free wall-rupture: a ... 1. AKASAKA, T.; YOSHIKAWA, J.; YOSHIDA, K. - Ventricular free wall rupture following acute myocardial infarction: a two ...
... with a first acute anterior myocardial infarction (AMI) treated with trombolytic agents. Twelve pts who were treated with ... Wall motion indices (WMI) and LV diastolic volume indices (DVI) were echocardiographically determined at 5th and 10th days, and ... The Effect of L-Carnitine on Left Ventricular Function in Patients with Acute Myocardial Infarction Treated with Streptokinase ... The Effect of L-Carnitine on Left Ventricular Function in Patients with Acute Myocardial Infarction Treated with Streptokinase ...
Clinical and echocardiographic predictors of left ventricular remodeling following anterior acute myocardial infarction ... anterior wall STEMI who underwent primary percutaneous intervention in the acute phase and at 6 months after the infarction. ... Ventricular remodeling is associated with worse prognosis following ST-segment elevation myocardial infarction (STEMI) and is a ... METHODS: This prospective, observational, and longitudinal study included 50 patients with anterior wall STEMI who were ...
Acute myocardial infarction (AMI) is one of the leading causes for death in both developed and developing countries and it is ... Intra-aortic balloon pump insertion through the right subclavian artery in a patient of anterior wall myocardial infarction ... acute myocardial infarction; STEMI, ST-segment elevation myocardial infarction; NSTEMI, non-ST-segment elevation myocardial ... including ST-elevation myocardial infarction (STEMI), Non ST-elevation myocardial infarction (NSTEMI), and unstable angina (UA ...
... thrombus in a patient presenting with an acute ST-elevation myocardial infarction is a rare complication of myocardial ... The patient underwent coronary artery bypass grafting (CABG), thrombectomy, and lateral wall repair. CONCLUSIONS:Left ... with an acute syncopal episode and intermittent chest pain for 7 days was found to have an anterolateral myocardial infarction ... free-wall rupture, is often a lethal complication, and if not followed by sudden death, the rupture may be contained by the ...
The coronary arteries were visualized with smooth walls and clear edges. The ligated vessels, with the diameter of about three ... MI was surgically induced by ligating left anterior descending artery. Imaging was performed 24 hours post-infarct, with ... The treatment of microcirculatory impairment will have great impact if it can be applied to myocardial infarction (MI) patients ... The two rats were established as models of acute myocardial infarction. 24 h after infarction, we started the imaging test. The ...
Rudd is having ST elevations in leads II, III and aVF suggesting acute inferior wall myocardial infarction. This explains the ... Tachycardia is seen in anterior and lateral wall MI where the circumflex and/or LAD coronary arteries are blocked. The nurse ... Acute myocardial infarction (AMI) is the most common cause and early recognition of cardiogenic shock is essential to saving ... In response to heart failure from a lateral or anterior AMI, the patient will exhibit increases in heart rate and/or stroke ...
LGE MRI (left panel) demonstrates myocardial infarction scar of the lateral wall. On FDG PET (middle panel), an intense FDG ... The inferior wall and the basal part of the anterior wall demonstrate no late gadolinium enhancement (LGE) and a normal FDG ... Imaging of myocardial viability. Repetitive or chronic ischemia (i.e., continuous hypoperfusion) may cause myocardial wall ... Hybrid 18F-FDG PET/MRI (suppressed myocardial glucose metabolism) early after ST-elevation myocardial infarction. ...
... was given a diagnosis of acute myocardial infarction. Emergency coronary angiography revealed mid-left anterior descending ... Sutureless Treatment for Blow-out Type Left Ventricular Free Wall Rupture (Department of Cardiovascular Surgery, Kikuna ... A Case of Acute Myocardial Infarction after Thrombolytic Therapy for Mechanical Valve Dysfunction in the Late Postoperative ... Acute myocardial infarction due to a free-floating thrombus was suspected, and emergency cardiac catheterization was performed ...
Linseed oil in ischemia include heart diet to be able to prevent the development of myocardial infarction Myocardial infarction ... Ischemia of the anterior wall of the heart has the chance of a favorable outcome when blood flow is restored to the damaged ... which can develop in myocardial infarction Myocardial infarction - the most formidable diagnosis or with severe cardiac ... Myocardial that localizes at the rear wall, occurs in the form painless embodiment. It is for this reason, has a value of ...
  • ST segment elevation of the ECG is often used in determining the treatment protocol (see also ST Elevation Myocardial Infarction ). (nih.gov)
  • ST segment elevation in the ECG is often used in determining the treatment protocol (see also NON-ST ELEVATION MYOCARDIAL INFARCTION ). (nih.gov)
  • Fothergill RT, Watson LR, Virdi GK, Moore FP, Whitbread M. Survival of resuscitated cardiac arrest patients with st-elevation myocardial infarction (STEMI) conveyed directly to a heart attack centre by ambulance clinicians. (smw.ch)
  • ESC guidelines for the management of acute myocardial infarction in patients presenting with st-segment elevation. (smw.ch)
  • 2013 ACCF/AHA guideline for the management of st-elevation myocardial infarction: A report of the american college of cardiology foundation/american heart association task force on practice guidelines. (smw.ch)
  • 1. Circulating Endothelial Cells and Endothelial Function Predict Major Adverse Cardiac Events and Early Adverse Left Ventricular Remodeling in Patients With ST-Segment Elevation Myocardial Infarction. (nih.gov)
  • 2. Prognosis-based definition of left ventricular remodeling after ST-elevation myocardial infarction. (nih.gov)
  • 3. Combined assessment of left ventricular end-diastolic pressure and ejection fraction by left ventriculography predicts long-term outcomes of patients with ST-segment elevation myocardial infarction. (nih.gov)
  • 5. Adverse diastolic remodeling after reperfused ST-elevation myocardial infarction: An important prognostic indicator. (nih.gov)
  • 7. Prognostic value of left ventricular global function index in patients after ST-segment elevation myocardial infarction. (nih.gov)
  • 9. Relation of plasma brain natriuretic peptide levels on admission for ST-elevation myocardial infarction to left ventricular end-diastolic volume six months later measured by both echocardiography and cardiac magnetic resonance. (nih.gov)
  • 10. Plasma angiopoietin-1 level, left ventricular ejection fraction, and multivessel disease predict development of 1-year major adverse cardiovascular events in patients with acute ST elevation myocardial infarction - a pilot study. (nih.gov)
  • 11. Sustained endothelial dysfunction in the infarct-related coronary artery is associated with left ventricular adverse remodeling in survivors of ST-segment elevation myocardial infarction. (nih.gov)
  • 14. Left ventricular global function index assessed by cardiovascular magnetic resonance for the prediction of cardiovascular events in ST-elevation myocardial infarction. (nih.gov)
  • 15. Intracoronary administration of bone marrow-derived progenitor cells improves left ventricular function in patients at risk for adverse remodeling after acute ST-segment elevation myocardial infarction: results of the Reinfusion of Enriched Progenitor cells And Infarct Remodeling in Acute Myocardial Infarction study (REPAIR-AMI) cardiac magnetic resonance imaging substudy. (nih.gov)
  • Cases with Inferior ST elevation Myocardial Infarction. (scirp.org)
  • Andersen, H.R., Nielsen, D. and Falk, E. (1989) Right Ventricular Infarction: Diagnostic Value of ST Elevation in Lead III Exceeding That of Lead II during Inferior/Posterior Infarction and Comparison with Right-Chest Leads V3R to V7R. (scirp.org)
  • 2000) Usefulness of ST Elevation II/III Ratio and ST Deviation in Lead I for Identifying the Culprit Artery in Inferior Wall Acute Myocardial Infarction. (scirp.org)
  • 2010) ST-Segment Depression in aVR as a Predictor of Culprit Artery and Infarct size in Acute Inferior Wall ST-Segment Elevation Myocardial Infarction. (scirp.org)
  • 2009) Predicting the Culprit Artery in Acute ST-Elevation Myocardial Infarction and Introducing a New Algorithm to Predict Infarct-Related Artery in Inferior ST-Elevation Myocardial Infarction: Correlation with Coronary Anatomy in the HAAMU Trial. (scirp.org)
  • This was initially unrecognised by the emergency department as unstable coronary syndrome and she subsequently progressed to an anterior non-ST elevation MI (NSTEMI). (bmj.com)
  • The diagnosis is atrial tachycardia , ST elevation myocardial infarction (STEMI) of the anterior wall, QRS (electrical) alternans, and T-wave alternans. (medscape.com)
  • ST segment elevation is seen in leads V3-V5 (↓), consistent with an acute anterior wall STEMI. (medscape.com)
  • Electrocardiographic ST-segment elevation in lead aVR was previously described in association with left main, left anterior descending, and triple-vessel coronary artery disease as well as Takotsubo cardiomyopathy. (radcliffecardiology.com)
  • is large enough, it can produce ST elevation in the posterior leads (not performed in this case), and ST depression in the anterior leads, especially V1, V2, and V3. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • Patients with inferior ST elevation myocardial infarction (STEMI), associated with right ventricular infarction, are thought to be at higher risk of developing hypotension when administered nitroglycerin (NTG). (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • However, current basic life support (BLS) protocols do not differentiate location of STEM … ST elevation, developing Q waves and T wave inversion may all be present depending on the timing of the ECG relative to the onset of myocardial infarction. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • The ST depression in V2 suggests posterior wall injury, and would normally be seen in V1 as well, unless something else is causing ST elevation in V1 at the same time. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • IMSEAR at SEARO: Multivessel Percutaneous Coronary Intervention in Patients with Acute ST-segment Elevation Myocardial Infarction in Same Sitting. (who.int)
  • Conclusion: In the current prospective non randomized study, we found that the multivessel primary PCI for ST elevation myocardial infarction with non-culprit vessel are suitable for PCI at the same sitting with better in-hospital and 1 yr survival outcome. (who.int)
  • Nomograms referenced by cardiac magnetic resonance in the prediction of cardiac injuries in patients with ST-elevation myocardial infarction. (amedeo.com)
  • Reply to Machine learning-based prediction of infarct size in patients with ST-segment elevation myocardial infarction: Misinterpretation. (amedeo.com)
  • Disparities in the management of non-ST-segment elevation myocardial infarction in the United States. (amedeo.com)
  • Speckle-tracking echocardiography has emerged as a unique technique for accurately evaluating myocardial function by analyzing the motion of speckles identified. (who.int)
  • Dobutamine stress echocardiography was performed 4 days post-infarction accompanied with automated functional imaging analysis of left ventricle during rest and then during low dose stress. (who.int)
  • Her echocardiography showed anterior wall hypokinesia with moderate left ventricular dysfunction. (heartviews.org)
  • 6. Extent of RV dysfunction and myocardial infarction assessed by CMR are independent outcome predictors early after STEMI treated with primary angioplasty. (nih.gov)
  • The aim of the current study was to evaluate stress speckle tracking to detect myocardial viability in comparison to cardiac MRI in post-STEMI patients. (who.int)
  • Strain rate obtained from speckle tracking during stress is a novel method of detecting myocardial viability after STEMI. (who.int)
  • 2002) Impact of Normalized Myocardial Perfusion after Successful Angioplasty in Acute Myocardial Infarction. (scirp.org)
  • Echocardiographic findings in cardiogenic shock due to acute myocardial infarction versus heart failure. (amedeo.com)
  • Inferior, posterior and lateral wall myocardial infarction Inferior, posterior and lateral wall myocardial infarction. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • This prospective study included a total of 74 revascularization using M4S probe in left lateral myocardial infarction patients diagnosed with ST position. (who.int)
  • These conditions are (1) spinal cord injury, (2) diabetes, (3) acute myocardial infarction (AMI), and (4) macular degeneration (namely Stargardt macular dystrophy and age-related macular degeneration). (nature.com)
  • The knowledge of the incidence of LVT may therefore be of importance to guide antiplatelet and antithrombotic therapy after acute myocardial infarction (AMI). (smw.ch)
  • Jugdutt BI, Sivaram CA. Prospective two-dimensional echocardiographic evaluation of left ventricular thrombus and embolism after acute myocardial infarction. (smw.ch)
  • Left ventricular thrombus formation after acute myocardial infarction as assessed by cardiovascular magnetic resonance imaging. (smw.ch)
  • Left ventricular mural thrombi complicating acute myocardial infarction. (smw.ch)
  • 13. Effect of intensive vs standard statin therapy on endothelial progenitor cells and left ventricular function in patients with acute myocardial infarction: Statins for regeneration after acute myocardial infarction and PCI (STRAP) trial. (nih.gov)
  • Sclarovsky, S. (1999) Electrocardiography of Acute Myocardial Ischaemic Syndromes. (scirp.org)
  • Zimetbaum, P.J. and Josephson, M.E. (2003) Use of the Electrocardiogram in Acute Myocardial Infarction. (scirp.org)
  • The death certificate listed "acute myocardial infarction" due to "atherosclerotic coronary artery disease" as the immediate cause of death. (cdc.gov)
  • The autopsy listed "coronary atherosclerosis" followed by "stenosis, thrombosis, chronic myocardial infarct, subacute myocardial infarcts, acute myocardial infarct, and acute ischemic change" as the cause(s) of death. (cdc.gov)
  • Pericardial Effusion (PE) is a complication after acute myocardial infarction (AMI), the objectives of this study was to determine the frequency and severity of PE after AMI and its association with other AMI complications. (innspub.net)
  • Pericardial effusion in acute myocardial infarction: frequency and in-hospital course. (innspub.net)
  • Pericardial Effusion Early in Acute Myocardial Infarction. (innspub.net)
  • 1985. Review Plaque fissuring-the cause of acute myocardial infarction, sudden ischaemic death, and crescendo angina. (innspub.net)
  • 1976. Pathology of acute myocardial infarction with particular reference to occlusive coronary thrombi. (innspub.net)
  • Pericardial Effusion as a Consequence of Acute Myocardial Infarction. (innspub.net)
  • Myocardial infarction (MI) or acute myocardial infarction (AMI) is the death of heart muscle from the sudden blockage of a coronary artery by a blood clot. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • Myocardial infarction 0 Myocardial infarction (MI) or acute myocardial infarction (AMI), commonly known as a heart attack 0 Typical symptoms of … Critical Cases in Electrocardiography. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • RESULTS: We found 72 cases of acute myocardial rupture with a mortality rate of 98.6% and 24 cases of sub-acute myocardial rupture with 41.6% of deaths. (bjcvs.org)
  • YOSHIDA, K. - Ventricular free wall rupture following acute myocardial infarction: a two dimensional echocar-diographic assessment. (bjcvs.org)
  • Acute inferior MI Leads II, III and aVF reflect electrocardiogram changes associated with acute infarction of the inferior aspect of the heart. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • On recording the right-sided ECG for those patients, there was ST … Electrocardiography in suspected myocardial infarction has the main purpose of detecting ischemia or acute coronary injury in emergency department populations coming for symptoms of myocardial infarction (MI). (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • Objectives: The aim of the study was to assess the role of ST segment depression in the limb leads aVR and aVL for the diagnosis of acute posterior wall infarction and the identification of infarct related artery (IRA) in patients with acute inferior wall MI. (scirp.org)
  • Further work has shown that CMR can recently emerged as a quantitative ultrasound technique differentiate between the densely bright infarct core and for accurately evaluating myocardial function by the adjacent peri-infarct zone, which appears with lower analyzing the motion of speckles identified on routine signal intensity because of the admixture of infarct and 2-dimensional sonograms (2, 3, 4). (who.int)
  • From these patients 1.05% had cardiac rupture as an ischaemic complication of the myocardium infarction. (bjcvs.org)
  • MYOCARDIAL INFARCTION in which the inferior wall of the heart is involved. (nih.gov)
  • ST segment depression in limb leads aVR and aVL with avR ≥ aVL helps to diagnose left circumflex artery as a culprit IRA in an acute inferior wall MI. (scirp.org)
  • ventricular MI (RVMI) is typically a conse-quent of large inferior wall MI and is pre- account for 40% to 55% of cases of MI. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • Right Ventricular AMIs Right Ventricular Infarcts (RVI) are most often due to an occlusion of the RCA and almost always occur in conjunction with an inferior wall MI (Kinch & Ryan 1994). (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • Up to 40% of inferior wall Myocardial infarctions have associated right ventricular involvement. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • An inferior myocardial infarction is a problem with the heart where cells along the inferior wall of the heart die in response to oxygen deprivation. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • Inferior wall MI (IWMI) patients remain quite common and. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • 1. To compare ivabradine with metoprolol in acute inferior wall MI in terms of feasibility, tolerability, and efficacy. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • Heart attack or myocardial infarction (MI) is broadly of 2 types: anterior wall and inferior wall, depending on the wall of heart involved. (stackexchange.com)
  • Those showing reciprocal changes had higher (65% vs. 15.5%) incidence of complications such as dysrhythmias, conduction defects, hypotension, left ventricular failure which was more conspicuous in inferior infarction. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • Asinger RW, Mikell FL, Elsperger J, Hodges M. Incidence of left-ventricular thrombosis after acute transmural myocardial infarction. (smw.ch)
  • Furthermore, a clear inverse relationship was found between the segmental strain and the transmural extent of infarction in each segment. (who.int)
  • Meanwhile it provided 81.82% sensitivity and 82.6% specificity to detect transmural from non-transmural infarction at a cut-off value of -10.15. (who.int)
  • Twelve pts who were treated with streptokinase (SK) within 6 hours of MI were given L 9 g/day IV for 5 days followed by 3 g/day orally for 3 months (group A). Eighteen pts treated with SK received no LC (group B). Wall motion indices (WMI) and LV diastolic volume indices (DVI) were echocardiographically determined at 5th and 10th days, and 1st and 3rd months. (archivestsc.com)
  • The aim of this study was to investigate the effect of sacubitril/ valsartan (Sal/Val) on left ventricular (LV) remodeling in patients with LV systolic dysfunction following acute anterior wall myocardial infarction (AAMI). (bvsalud.org)
  • The current status of stem cell therapies for patients with myocardial infarction is discussed from a bioengineering and biomaterial perspective in this review. (nature.com)
  • We describe (a) the current status of clinical trials of human pluripotent stem cells (hPSCs) compared with clinical trials of human adult or fetal stem cells, (b) the gap between fundamental research and application of human stem cells, (c) the use of biomaterials in clinical and pre-clinical studies of stem cells, and finally (d) trends in bioengineering to promote stem cell therapies for patients with myocardial infarction. (nature.com)
  • Here, we have described the current status of stem cell therapies using hPSCs for patients with myocardial infarction (MI), focusing on the bioengineering aspects of these therapies. (nature.com)
  • METHODS: In 177 patients with large, mainly anterior AMI, standard cardiac magnetic resonance imaging (CMR) including cine and late gadolinium enhancement (LGE) imaging was performed shortly after AMI as per protocol. (smw.ch)
  • Background: Aim of the study was to evaluate the primary procedural success of Multivessel Percutaneous coronary intervention in patients with acute ST-segment elevated myocardial infarction at the same sitting. (who.int)
  • We examined the effect of l-carnitine (LC) on left ventricular (LV) function in patients (pts) with a first acute anterior myocardial infarction (AMI) treated with trombolytic agents. (archivestsc.com)
  • Effects of sacubitril/valsartan on ventricular remodeling in patents with left ventricular systolic dysfunction following acute anterior wall myocardial infarction. (bvsalud.org)
  • 354). Dr. Stanley S. Josef, of Central Arkansas Cardiovascular Institute (CACI), reported that Plaintiff had a history of angina, status post PTCA (percutaneous transluminal coronary angioplasty), and stenting of the LAD (left anterior descending) in 2003, and the risk factors included hypertension and hyperlipidemia.2 He stated that plaintiff also suffered from paroxysmal atrial fibrillation3 and was on Coumadin and Rythmol therapy. (justia.com)
  • Association of anxiety or depression with risk of recurrent cardiovascular events and death after myocardial infarction: A nationwide registry study. (amedeo.com)
  • Cardiovascular Diseases: Increased risk of myocardial infarction, sudden cardiac death and stroke has been reported in association with use of GnRH analogs in men. (nih.gov)
  • A myocardial infarction that does not produce elevations in the ST segments of the ELECTROCARDIOGRAM. (nih.gov)
  • 2008) Value of the 12-Lead Electrocardiogram to Define the Level of Obstruction in Acute Anterior Wall Myocardial Infarction: Correlation to Coronary Angiography and Clinical Outcome in the DANAMI-2 Trial. (scirp.org)
  • inferior myocardial infarction: infarction in which the inferior or diaphragmatic wall of the heart is involved, producing indicative changes in leads II, III, and aVF in the electrocardiogram. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • Pulmonary: pulmonary oedema, pulmonary infarction, and haemoptysis. (bmj.com)
  • Embolic potential, prevention and management of mural thrombus complicating anterior myocardial infarction: A meta-analysis. (smw.ch)
  • Incidence of early left ventricular thrombus after acute anterior wall myocardial infarction in the primary coronary intervention era. (smw.ch)
  • As shown in the examples below, myocardial infarction diagnosis in right bundle branch block is not very different from normal MI diagnosis. (xn--22c0bihcbb7dg4lnac3am9zla.com)
  • As emergency angioplasty and stent placement of his left anterior descending coronary artery was started, the FF had an arrhythmia and cardiac arrest. (cdc.gov)
  • A 32 years old female without any conventional risk factors except obesity presented with acute anterior wall myocardial infarction (MI). (heartviews.org)
  • Wellens' syndrome refers to specific ECG abnormalities in the precordial T-wave segment, which are associated with critical stenosis of the proximal left anterior descending (LAD) coronary artery culminating in an acute anterior wall myocardial infarction (MI) if the patient is not urgently revascularised. (bmj.com)
  • Anterior wall myocardial infarction is often caused by occlusion of the left anterior descending coronary artery. (nih.gov)
  • Myocardial infarction in the absence of obstructive coronary artery disease. (nih.gov)
  • The death certificate (completed by the coroner) listed "cardiac arrest with electromechanical dissociation" due to "acute anterior wall myocardial infarction" due to "coronary artery disease" (CAD) as the cause of death. (cdc.gov)
  • Angiography showed tight stenosis of the proximal left anterior descending (LAD) and borderline lesion in left circumflex coronary artery (LCX). (heartviews.org)
  • 1. Anterior descending coronary artery flow. (nih.gov)
  • Pericardial effusion in the course of myocardial infarction: incidence, natural history, and clinical relevance. (innspub.net)
  • Myocardial blood flow (MBF) is the critical determinant of cardiac function. (snmjournals.org)
  • Moreover, it carries a promising role in post-myocardial infarction risk stratification with a reasonable prediction of reversible cardiac-related hospital re-admission. (who.int)
  • Polygenic risk scores point toward potential genetic mechanisms of type 2 myocardial infarction in people with HIV. (amedeo.com)
  • Symptoms develop from lesions in the CORPUS CALLOSUM or medial frontal cortex, stroke, infarction, and neurodegenerative diseases (e.g. (nih.gov)
  • Investigating strain rate obtained with stress speckle tracking after revascularization predicted the extent of myocardial scar, determined by contrast-enhanced magnetic resonance imaging. (who.int)