Anterior Wall Myocardial Infarction: MYOCARDIAL INFARCTION in which the anterior wall of the heart is involved. Anterior wall myocardial infarction is often caused by occlusion of the left anterior descending coronary artery. It can be categorized as anteroseptal or anterolateral wall myocardial infarction.Inferior Wall Myocardial Infarction: MYOCARDIAL INFARCTION in which the inferior wall of the heart is involved. It is often caused by occlusion of the right coronary artery.Myocardial Infarction: NECROSIS of the MYOCARDIUM caused by an obstruction of the blood supply to the heart (CORONARY CIRCULATION).Coronary Angiography: Radiography of the vascular system of the heart muscle after injection of a contrast medium.Ventricular Function, Left: The hemodynamic and electrophysiological action of the left HEART VENTRICLE. Its measurement is an important aspect of the clinical evaluation of patients with heart disease to determine the effects of the disease on cardiac performance.Electrocardiography: Recording of the moment-to-moment electromotive forces of the HEART as projected onto various sites on the body's surface, delineated as a scalar function of time. The recording is monitored by a tracing on slow moving chart paper or by observing it on a cardioscope, which is a CATHODE RAY TUBE DISPLAY.Heart Rupture, Post-Infarction: Laceration or tearing of cardiac tissues appearing after MYOCARDIAL INFARCTION.Heart Ventricles: The lower right and left chambers of the heart. The right ventricle pumps venous BLOOD into the LUNGS and the left ventricle pumps oxygenated blood into the systemic arterial circulation.Echocardiography: Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues. The standard approach is transthoracic.Coronary Vessels: The veins and arteries of the HEART.Infarction: Formation of an infarct, which is NECROSIS in tissue due to local ISCHEMIA resulting from obstruction of BLOOD CIRCULATION, most commonly by a THROMBUS or EMBOLUS.Myocardial Reperfusion: Generally, restoration of blood supply to heart tissue which is ischemic due to decrease in normal blood supply. The decrease may result from any source including atherosclerotic obstruction, narrowing of the artery, or surgical clamping. Reperfusion can be induced to treat ischemia. Methods include chemical dissolution of an occluding thrombus, administration of vasodilator drugs, angioplasty, catheterization, and artery bypass graft surgery. However, it is thought that reperfusion can itself further damage the ischemic tissue, causing MYOCARDIAL REPERFUSION INJURY.Angioplasty, Balloon, Coronary: Dilation of an occluded coronary artery (or arteries) by means of a balloon catheter to restore myocardial blood supply.Cell Wall: The outermost layer of a cell in most PLANTS; BACTERIA; FUNGI; and ALGAE. The cell wall is usually a rigid structure that lies external to the CELL MEMBRANE, and provides a protective barrier against physical or chemical agents.Time Factors: Elements of limited time intervals, contributing to particular results or situations.Cerebral Infarction: The formation of an area of NECROSIS in the CEREBRUM caused by an insufficiency of arterial or venous blood flow. Infarcts of the cerebrum are generally classified by hemisphere (i.e., left vs. right), lobe (e.g., frontal lobe infarction), arterial distribution (e.g., INFARCTION, ANTERIOR CEREBRAL ARTERY), and etiology (e.g., embolic infarction).Ventricular Remodeling: The geometric and structural changes that the HEART VENTRICLES undergo, usually following MYOCARDIAL INFARCTION. It comprises expansion of the infarct and dilatation of the healthy ventricle segments. While most prevalent in the left ventricle, it can also occur in the right ventricle.Thrombolytic Therapy: Use of infusions of FIBRINOLYTIC AGENTS to destroy or dissolve thrombi in blood vessels or bypass grafts.Coronary Circulation: The circulation of blood through the CORONARY VESSELS of the HEART.Treatment Outcome: Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.Prospective Studies: Observation of a population for a sufficient number of persons over a sufficient number of years to generate incidence or mortality rates subsequent to the selection of the study group.Myocardium: The muscle tissue of the HEART. It is composed of striated, involuntary muscle cells (MYOCYTES, CARDIAC) connected to form the contractile pump to generate blood flow.Risk Factors: An aspect of personal behavior or lifestyle, environmental exposure, or inborn or inherited characteristic, which, on the basis of epidemiologic evidence, is known to be associated with a health-related condition considered important to prevent.Coronary Disease: An imbalance between myocardial functional requirements and the capacity of the CORONARY VESSELS to supply sufficient blood flow. It is a form of MYOCARDIAL ISCHEMIA (insufficient blood supply to the heart muscle) caused by a decreased capacity of the coronary vessels.Ventricular Dysfunction, Left: A condition in which the LEFT VENTRICLE of the heart was functionally impaired. This condition usually leads to HEART FAILURE; MYOCARDIAL INFARCTION; and other cardiovascular complications. Diagnosis is made by measuring the diminished ejection fraction and a depressed level of motility of the left ventricular wall.Myocardial Ischemia: A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (CORONARY ARTERY DISEASE), to obstruction by a thrombus (CORONARY THROMBOSIS), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (MYOCARDIAL INFARCTION).Heart: The hollow, muscular organ that maintains the circulation of the blood.Stroke Volume: The amount of BLOOD pumped out of the HEART per beat, not to be confused with cardiac output (volume/time). It is calculated as the difference between the end-diastolic volume and the end-systolic volume.Predictive Value of Tests: In screening and diagnostic tests, the probability that a person with a positive test is a true positive (i.e., has the disease), is referred to as the predictive value of a positive test; whereas, the predictive value of a negative test is the probability that the person with a negative test does not have the disease. Predictive value is related to the sensitivity and specificity of the test.Streptokinase: Streptococcal fibrinolysin . An enzyme produced by hemolytic streptococci. It hydrolyzes amide linkages and serves as an activator of plasminogen. It is used in thrombolytic therapy and is used also in mixtures with streptodornase (STREPTODORNASE AND STREPTOKINASE). EC 3.4.-.Follow-Up Studies: Studies in which individuals or populations are followed to assess the outcome of exposures, procedures, or effects of a characteristic, e.g., occurrence of disease.Creatine Kinase: A transferase that catalyzes formation of PHOSPHOCREATINE from ATP + CREATINE. The reaction stores ATP energy as phosphocreatine. Three cytoplasmic ISOENZYMES have been identified in human tissues: the MM type from SKELETAL MUSCLE, the MB type from myocardial tissue and the BB type from nervous tissue as well as a mitochondrial isoenzyme. Macro-creatine kinase refers to creatine kinase complexed with other serum proteins.Prognosis: A prediction of the probable outcome of a disease based on a individual's condition and the usual course of the disease as seen in similar situations.Cardiac Catheterization: Procedures in which placement of CARDIAC CATHETERS is performed for therapeutic or diagnostic procedures.Myocardial Contraction: Contractile activity of the MYOCARDIUM.Coronary Care Units: The hospital unit in which patients with acute cardiac disorders receive intensive care.Cystocele: A HERNIA-like condition in which the weakened pelvic muscles cause the URINARY BLADDER to drop from its normal position. Fallen urinary bladder is more common in females with the bladder dropping into the VAGINA and less common in males with the bladder dropping into the SCROTUM.Fibrinolytic Agents: Fibrinolysin or agents that convert plasminogen to FIBRINOLYSIN.Retrospective Studies: Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.Coronary Artery Bypass: Surgical therapy of ischemic coronary artery disease achieved by grafting a section of saphenous vein, internal mammary artery, or other substitute between the aorta and the obstructed coronary artery distal to the obstructive lesion.Angina Pectoris: The symptom of paroxysmal pain consequent to MYOCARDIAL ISCHEMIA usually of distinctive character, location and radiation. It is thought to be provoked by a transient stressful situation during which the oxygen requirements of the MYOCARDIUM exceed that supplied by the CORONARY CIRCULATION.Tomography, Emission-Computed, Single-Photon: A method of computed tomography that uses radionuclides which emit a single photon of a given energy. The camera is rotated 180 or 360 degrees around the patient to capture images at multiple positions along the arc. The computer is then used to reconstruct the transaxial, sagittal, and coronal images from the 3-dimensional distribution of radionuclides in the organ. The advantages of SPECT are that it can be used to observe biochemical and physiological processes as well as size and volume of the organ. The disadvantage is that, unlike positron-emission tomography where the positron-electron annihilation results in the emission of 2 photons at 180 degrees from each other, SPECT requires physical collimation to line up the photons, which results in the loss of many available photons and hence degrades the image.Platelet Aggregation Inhibitors: Drugs or agents which antagonize or impair any mechanism leading to blood platelet aggregation, whether during the phases of activation and shape change or following the dense-granule release reaction and stimulation of the prostaglandin-thromboxane system.Cineangiography: Motion pictures of the passage of contrast medium through blood vessels.Angina, Unstable: Precordial pain at rest, which may precede a MYOCARDIAL INFARCTION.Hemodynamics: The movement and the forces involved in the movement of the blood through the CARDIOVASCULAR SYSTEM.Stents: Devices that provide support for tubular structures that are being anastomosed or for body cavities during skin grafting.Myocardial Revascularization: The restoration of blood supply to the myocardium. (From Dorland, 28th ed)Percutaneous Coronary Intervention: A family of percutaneous techniques that are used to manage CORONARY OCCLUSION, including standard balloon angioplasty (PERCUTANEOUS TRANSLUMINAL CORONARY ANGIOPLASTY), the placement of intracoronary STENTS, and atheroablative technologies (e.g., ATHERECTOMY; ENDARTERECTOMY; THROMBECTOMY; PERCUTANEOUS TRANSLUMINAL LASER ANGIOPLASTY). PTCA was the dominant form of PCI, before the widespread use of stenting.Recurrence: The return of a sign, symptom, or disease after a remission.Coronary Thrombosis: Coagulation of blood in any of the CORONARY VESSELS. The presence of a blood clot (THROMBUS) often leads to MYOCARDIAL INFARCTION.Heart Septum: This structure includes the thin muscular atrial septum between the two HEART ATRIA, and the thick muscular ventricular septum between the two HEART VENTRICLES.Coronary Artery Disease: Pathological processes of CORONARY ARTERIES that may derive from a congenital abnormality, atherosclerotic, or non-atherosclerotic cause.Tomography, X-Ray Computed: Tomography using x-ray transmission and a computer algorithm to reconstruct the image.Risk Assessment: The qualitative or quantitative estimation of the likelihood of adverse effects that may result from exposure to specified health hazards or from the absence of beneficial influences. (Last, Dictionary of Epidemiology, 1988)Shock, Cardiogenic: Shock resulting from diminution of cardiac output in heart disease.Hospital Mortality: A vital statistic measuring or recording the rate of death from any cause in hospitalized populations.Uterine Prolapse: Downward displacement of the UTERUS. It is classified in various degrees: in the first degree the UTERINE CERVIX is within the vaginal orifice; in the second degree the cervix is outside the orifice; in the third degree the entire uterus is outside the orifice.Myocardial Reperfusion Injury: Damage to the MYOCARDIUM resulting from MYOCARDIAL REPERFUSION (restoration of blood flow to ischemic areas of the HEART.) Reperfusion takes place when there is spontaneous thrombolysis, THROMBOLYTIC THERAPY, collateral flow from other coronary vascular beds, or reversal of vasospasm.Acute Disease: Disease having a short and relatively severe course.

Usefulness of the index of microcirculatory resistance for invasively assessing myocardial viability immediately after primary angioplasty for anterior myocardial infarction. (1/56)

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Dual left anterior descending artery distribution. (2/56)

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Low adiponectin blood concentration predicts left ventricular remodeling after ST-segment elevation myocardial infarction treated with primary percutaneous coronary intervention. (3/56)

BACKGROUND: Left ventricular remodeling (LVR), an increase in left ventricular end-diastolic volume index > or = 20%, is an adverse consequence of myocardial infarction. The aim of this study was to assess the association between LVR and adiponectin, which has been shown to protect against myocardial ischemia-reperfusion injury. METHODS: In 75 patients echocardiographic examination was performed one year after ST-segment elevation myocardial infarction, successfully treated with primary percutaneous coronary intervention (pPCI). Two groups of patients were analyzed: those with LVR (n = 15) and those without LVR (n = 60). RESULTS: The predictors of LVR were: anterior myocardial infarction, glucose at admission, baseline C-reactive protein, adiponectin, and echocardiographic parameters: left ventricular end-diastolic and end-systolic volume indices, ejection fraction < 40% and left ventricular wall motion score index (WMSI) at discharge. On multivariable regression analysis, lower adiponectin level (OR = 0.67, 95% CI 0.49-0.91, p < 0.05) and higher WMSI (OR = 20.14, 95% CI 2.62-154.82, p < 0.01) were the only independent negative predictors of LVR. The optimal cut-off for adiponectin for predicting LVR was < or = 4.7 mg/mL (sensitivity: 73%, specificity: 85%) and this level increased the risk of LVR 15-fold (95% CI 4.05-59.87, p = 0.0001). CONCLUSIONS: Baseline low blood adiponectin concentration, along with WMSI, can be considered as a predictor of the LVR in male patients one year after myocardial infarction and pPCI.  (+info)

Dor procedure for dyskinetic anteroapical myocardial infarction fails to improve contractility in the border zone. (4/56)

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Right ventricular involvement in anterior myocardial infarction: a translational approach. (5/56)

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Retrograde approach to a totally occluded right coronary artery via a septal perforator artery: the tale of a long and winding wire. (6/56)

Retrograde recannalization of chronic total occlusions has developed as a viable alternative to restore coronary patency. Techniques continue to evolve and complications described. We present a new complication related to equipment developed to improve outcomes via a retrograde approach.  (+info)

Complete atrioventricular block complicating acute anterior myocardial infarction can be reversed with acute coronary angioplasty. (7/56)

INTRODUCTION: A retrospective case series of acute anterior myocardial infarction (MI) patients complicated by complete atrioventricular block (AVB) treated with acute percutaneous transluminal coronary angioplasty (PTCA). CLINICAL PICTURE: Eight patients with anterior MI and complete AVB underwent acute PTCA between 2000 and 2005. Mean onset of complete AVB was 16.6 +/- 16.9 hours from chest pain onset. TREATMENT: All patients underwent successful PTCA to the left anterior descending artery. OUTCOME: Complete AVB resolved with PTCA in 88%; mean time of resolution was 89 +/- 144 minutes after revascularisation. One patient had permanent pacemaker implanted at Day 12 after developing an 8-second ventricular standstill during hospitalisation but not pacing-dependent on follow-up. The rhythm on discharge for the other surviving patients was normal sinus rhythm. CONCLUSION: This case series suggests that complete AVB complicating anterior MI is reversible with acute PTCA and survivors are not at increased risk of recurrent AVB. Nevertheless, this condition is associated with extensive myocardial damage and high mortality during the acute hospitalisation was not improved with correction of AVB with temporary pacing.  (+info)

Coronary flow velocity pattern and recovery of regional left ventricular function: the relationship observed in patients with reperfused acute myocardial infarction. (8/56)

Coronary flow velocity pattern (CFVP) recorded within 3 days of percutaneous coronary intervention (PCI) has been reported to be useful in predicting left ventricular (LV) function. The aim of this prospective study was to investigate, via transthoracic Doppler echocardiography, whether the relationship between CFVP and recovery of LV function persists. Our study group comprised 37 patients with 1st anterior-wall acute myocardial infarction who underwent successful PCI for lesions in the left anterior descending coronary artery (LAD). The CFVP in the LAD was recorded at 24-48 hours, 7 days, and 4 weeks after PCI. Myocardial contrast echocardiography was performed at 24-48 hours after PCI. The diastolic deceleration time (DDT) at each stage correlated significantly with the regional LV wall-motion score index at 6-month follow-up (r=-0.58 at 24-48 hr, -0.57 at day 7, and -0.50 at week 4; P <0.01 for all). The mean DDT increased over time. Optimal cutoff values for DDT to predict regional LV wall-motion score indices of <2.0 were 327 ms at 24-48 hours (sensitivity, 0.78; specificity, 0.64), 495 ms at day 7 (sensitivity, 0.75; specificity, 0.69), and 525 ms at week 4 (sensitivity, 0.83; specificity, 0.69). The DDT at 24-48 hours significantly correlated, better than the peak creatine kinase value, with reperfusion (r=0.68, P <0.01) as defined by myocardial contrast echocardiography. In conclusion, CFVP in the LAD can be used, within 4 weeks after PCI, to predict the recovery of regional LV function in patients with reperfused anterior-wall acute myocardial infarction.  (+info)

  • Thrombi that do not develop rapidly allow a collateral blood supply to develop, whilst rapidly developing thrombi that last for 6 hours or more result in transmural infarction of the myocardium supplied by that vessel. (forensicmed.co.uk)
  • Coronary artery disease remains the most common CVD and is caused by atherosclerotic plaque accumulation in the epicardial arteries supplying the myocardium 2 , which can restrict blood flow leading to myocardial infarction (MI) 1 , 3 . (nature.com)
  • Lack of adequate oxygen and insufficient metabolite delivery to the myocardium diminish the force of muscular contraction and decrease systolic wall motion in the affected territory. (scribd.com)
  • Even brief deprivation of oxygen and the requisite metabolites to the myocardium diminishes diastolic relaxation and causes abnormal regional systolic contractile function, wall thickening, and abnormal wall motion. (scribd.com)
  • The present study was designed to investigate whether microvascular remodeling could occur in hibernating myocardium and infarction regions distal to a total occluded coronary artery after acute myocardial infarction. (ovid.com)
  • Apical wall motion partitions would have enabled all patients with LV thrombus to be appropriately referred for DE-CMR testing (100% sensitivity and negative predictive value), while avoiding further testing in more than one-half (56% to 63%) of patients. (onlinejacc.org)
  • Although DE-CMR yielded improved overall thrombus detection, apical wall motion on a noncontrast echocardiogram can be an effective stratification tool to identify patients in whom DE-CMR thrombus assessment is most warranted. (onlinejacc.org)
  • CGX1321 improved cardiac function, reduced myocardial infarct size, and fibrosis of post-MI hearts. (clinsci.org)
  • Although the majority of animal and preliminary human studies of cell-based therapy shows an overall improvement in cardiac function when administered to hearts after acute infarction, the effects generally are modest, and the mechanisms underlying such an observed improvement are far from clear ( 3 ). (pnas.org)
  • We recently have reported that intracardiac implantation of genetically engineered MSCs overexpressing the Akt gene (Akt-MSCs) yielded dramatic diminution of infarct size and restoration of cardiac function in rodent hearts after myocardial injury ( 7 ). (pnas.org)
  • Adverse remodeling after myocardial infarction (MI) has a significant impact on global cardiac function. (ahajournals.org)
  • Accordingly, we investigated whether LVEDP could be predicted using a new parameter, ie, LA dimension at LV end-systole (LADs) corrected by LV diastolic myocardial strain (%thinning from end systole). (ahajournals.org)
  • Myocardial infarction may lead to impairment of systolic or diastolic function and to increased predisposition to arrhythmias and other longterm complications. (scribd.com)
  • We examined whether intracoronary injection of autologous mononuclear bone marrow cells (mBMC) influenced regional myocardial function or LV twist. (biomedcentral.com)
  • Myocardial infraction is a result of a blockage in any one of the coronary arteries leading to lack of oxygen in the heart. (sbwire.com)
  • The anatomical basis of myocardial infarction is in 98 percent of the cases atherosclerosis of the coronary arteries. (thefreedictionary.com)
  • however, with anomalous vessels originating from the proximal right and left anterior descending coronary arteries, draining into the pulmonary artery through a plexus of small vessels. (bvsalud.org)
  • Copper stents were implanted in the left descending coronary arteries of 64 pigs to induce anterior wall myocardial infarction. (ovid.com)
  • The wall area (WA) and lumen area (LA) of small intramyocardial coronary arteries (SIMCA) distal to occlusions were measured and the ratios of WA/LA and LA/total vessel area (%L) were calculated. (ovid.com)
  • Periods of low GMA showed a related increase (negative correlation) in in-hospital non-myocardial infarction-related cardiovascular deaths. (kup.at)
  • Albany, NY -- ( SBWIRE ) -- 12/09/2016 -- The global myocardial infraction treatment market is expected to register promising growth owing to the growing number of cases of cardiovascular diseases across the globe, says Transparency Market Research (TMR) in a research report. (sbwire.com)
  • Cardiovascular disease accounts for 30% of deaths globally, and of these, nearly one-half are due to coronary heart disease ( CHD ) [ 1 ] with acute myocardial infarction ( AMI ) occurring every 44 seconds [ 1 ] in the United States. (thno.org)
  • SSO 2 Therapy has been shown in multiple randomized prospective clinical trials to consistently and safely reduce infarct size in anterior AMI patients and thereby improve outcomes. (mpo-mag.com)
  • We have repeatedly demonstrated that SSO 2 Therapy significantly reduces infarct size among patients who suffer from large anterior AMIs, which are the most serious heart attacks," said Kevin T. Larkin, president and chief executive officer of TherOx. (mpo-mag.com)
  • Myocardial infarct size (percentage of total heart weight +/- SEM) did not significantly differ between MRL and C57/Bl6 controls (18.9 +/- 1.8% for MRL vs. 15.7 + 1.3% for C57/Bl6, p = 0.20). (nih.gov)
  • TherOx Inc., a privately held medical device company focused on improving treatment of acute myocardial infarction (AMI), announced that the U.S. Food and Drug Administration (FDA) granted premarket approval for its SuperSaturated Oxygen (SSO 2 ) Therapy. (mpo-mag.com)
Remote myocardial dysfunction after acute anterior myocardial infarction: impact of left ventricular shape on regional function...
Remote myocardial dysfunction after acute anterior myocardial infarction: impact of left ventricular shape on regional function... (onlinejacc.org)
Cardiology Lectures 1 and 2 -- EKG Flashcards by Jodeci Malixi | Brainscape
Cardiology Lectures 1 and 2 -- EKG Flashcards by Jodeci Malixi | Brainscape (brainscape.com)
Cardiovascular molecular imaging of apoptosis | SpringerLink
Cardiovascular molecular imaging of apoptosis | SpringerLink (link.springer.com)
Publications - Mayo Clinic
Publications - Mayo Clinic (mayo.edu)
ICD-10: Coding Snapshot
ICD-10: Coding Snapshot (aapc.com)
The American Academy of Insurance Medicine
The American Academy of Insurance Medicine (aaimedicine.org)
The dilemma of refractory hypoxemia after inferior wall myocardial infarction.
The dilemma of refractory hypoxemia after inferior wall myocardial infarction. (medworm.com)
Postinfarction Ventricular Septal Rupture: Practice Essentials, Pathophysiology, Epidemiology
Postinfarction Ventricular Septal Rupture: Practice Essentials, Pathophysiology, Epidemiology (emedicine.medscape.com)
Summary: Effect of geomagnetic activity on cardiovascular parameters
Summary: Effect of geomagnetic activity on cardiovascular parameters (kup.at)
Validation of an algorithm to reveal the U wave in atrial fibrillation | Scientific Reports
Validation of an algorithm to reveal the U wave in atrial fibrillation | Scientific Reports (nature.com)
Practice Cardiovascular System Questions for the Physician Assistant Exam - dummies
Practice Cardiovascular System Questions for the Physician Assistant Exam - dummies (dummies.com)
Frontiers | Interplay Between Phosphorylation and O-GlcNAcylation of Sarcomeric Proteins in Ischemic Heart Failure |...
Frontiers | Interplay Between Phosphorylation and O-GlcNAcylation of Sarcomeric Proteins in Ischemic Heart Failure |... (frontiersin.org)
Molecular Imaging of Interstitial Alterations in Remodeling Myocardium After Myocardial Infarction | JACC: Journal of the...
Molecular Imaging of Interstitial Alterations in Remodeling Myocardium After Myocardial Infarction | JACC: Journal of the... (onlinejacc.org)
What is the morbidity associated with arterial thrombosis in protein C deficiency?
What is the morbidity associated with arterial thrombosis in protein C deficiency? (medscape.com)
Pathology of Acute Myocardial Infarction: Overview, Pathophysiology, Etiology
Pathology of Acute Myocardial Infarction: Overview, Pathophysiology, Etiology (emedicine.medscape.com)
Numerical Simulation and Clinical Implications of Stenosis in Coronary Blood Flow
Numerical Simulation and Clinical Implications of Stenosis in Coronary Blood Flow (hindawi.com)
What is the role of electrocardiography in the workup of Wellens syndrome?
What is the role of electrocardiography in the workup of Wellens syndrome? (medscape.com)
Impact of intracoronary aspiration thrombectomy during primary angioplasty on left ventricular remodelling in patients with...
Impact of intracoronary aspiration thrombectomy during primary angioplasty on left ventricular remodelling in patients with... (heart.bmj.com)
Sudden cardiac death and coronary artery disease :: www.forensicmed.co.uk
Sudden cardiac death and coronary artery disease :: www.forensicmed.co.uk (forensicmed.co.uk)
Valbuena-Lopez, S., Hinojar, R. and Puntmann, V.O. (2016) Cardiovascular Magnetic Resonance in Cardiology Practice A Concise...
Valbuena-Lopez, S., Hinojar, R. and Puntmann, V.O. (2016) Cardiovascular Magnetic Resonance in Cardiology Practice A Concise... (scirp.org)
Percutaneous Intramyocardial Delivery of Mesenchymal Stem Cells Induces Superior Improvement in Regional Left Ventricular...
Percutaneous Intramyocardial Delivery of Mesenchymal Stem Cells Induces Superior Improvement in Regional Left Ventricular... (thno.org)
Current Pacemaker and Defibrillator Therapy (27.05.2011)
Current Pacemaker and Defibrillator Therapy (27.05.2011) (aerzteblatt.de)
Cocaine Toxicity: Practice Essentials, Background, Pathophysiology
Cocaine Toxicity: Practice Essentials, Background, Pathophysiology (emedicine.medscape.com)
Internet Scientific Publications
Internet Scientific Publications (ispub.com)
Applying the new STEMI guidelines: 1. Reperfusion in acute ST-segment elevation myocardial infarction | CMAJ
Applying the new STEMI guidelines: 1. Reperfusion in acute ST-segment elevation myocardial infarction | CMAJ (cmaj.ca)
Fire Fighter Fatality Investigation Report F2007-34 | NIOSH | CDC
Fire Fighter Fatality Investigation Report F2007-34 | NIOSH | CDC (cdc.gov)
Injectable human recombinant collagen matrices limit adverse remodeling and improve cardiac function after myocardial...
Injectable human recombinant collagen matrices limit adverse remodeling and improve cardiac function after myocardial... (nature.com)