Anorexia Nervosa: An eating disorder that is characterized by the lack or loss of APPETITE, known as ANOREXIA. Other features include excess fear of becoming OVERWEIGHT; BODY IMAGE disturbance; significant WEIGHT LOSS; refusal to maintain minimal normal weight; and AMENORRHEA. This disorder occurs most frequently in adolescent females. (APA, Thesaurus of Psychological Index Terms, 1994)Anorexia: The lack or loss of APPETITE accompanied by an aversion to food and the inability to eat. It is the defining characteristic of the disorder ANOREXIA NERVOSA.Bulimia: Eating an excess amount of food in a short period of time, as seen in the disorder of BULIMIA NERVOSA. It is caused by an abnormal craving for food, or insatiable hunger also known as "ox hunger".Bulimia Nervosa: An eating disorder that is characterized by a cycle of binge eating (BULIMIA or bingeing) followed by inappropriate acts (purging) to avert weight gain. Purging methods often include self-induced VOMITING, use of LAXATIVES or DIURETICS, excessive exercise, and FASTING.Eating Disorders: A group of disorders characterized by physiological and psychological disturbances in appetite or food intake.Refeeding Syndrome: A condition of metabolic imbalance that is caused by complications of initially feeding a severely malnourished patient too aggressively. Usually occurring within the first 5 days of refeeding, this syndrome is characterized by WATER-ELECTROLYTE IMBALANCE; GLUCOSE INTOLERANCE; CARDIAC ARRHYTHMIAS; and DIARRHEA.Cachexia: General ill health, malnutrition, and weight loss, usually associated with chronic disease.Eating: The consumption of edible substances.Appetite: Natural recurring desire for food. Alterations may be induced by APPETITE DEPRESSANTS or APPETITE STIMULANTS.Appetite Stimulants: Agents that are used to stimulate appetite. These drugs are frequently used to treat anorexia associated with cancer and AIDS.Amenorrhea: Absence of menstruation.Body Weight: The mass or quantity of heaviness of an individual. It is expressed by units of pounds or kilograms.Feeding Behavior: Behavioral responses or sequences associated with eating including modes of feeding, rhythmic patterns of eating, and time intervals.Appetite Depressants: Agents that are used to suppress appetite.Gastric Dilatation: Abnormal distention of the STOMACH due to accumulation of gastric contents that may reach 10 to 15 liters. Gastric dilatation may be the result of GASTRIC OUTLET OBSTRUCTION; ILEUS; GASTROPARESIS; or denervation.Family Therapy: A form of group psychotherapy. It involves treatment of more than one member of the family simultaneously in the same session.Emaciation: Clinical manifestation of excessive LEANNESS usually caused by disease or a lack of nutrition (MALNUTRITION).Body Image: Individuals' concept of their own bodies.Ghrelin: A 28-amino acid, acylated, orexigenic peptide that is a ligand for GROWTH HORMONE SECRETAGOGUE RECEPTORS. Ghrelin is widely expressed but primarily in the stomach in the adults. Ghrelin acts centrally to stimulate growth hormone secretion and food intake, and peripherally to regulate energy homeostasis. Its large precursor protein, known as appetite-regulating hormone or motilin-related peptide, contains ghrelin and obestatin.Leptin: A 16-kDa peptide hormone secreted from WHITE ADIPOCYTES. Leptin serves as a feedback signal from fat cells to the CENTRAL NERVOUS SYSTEM in regulation of food intake, energy balance, and fat storage.Appetite Regulation: Physiologic mechanisms which regulate or control the appetite and food intake.Weight Gain: Increase in BODY WEIGHT over existing weight.Body Mass Index: An indicator of body density as determined by the relationship of BODY WEIGHT to BODY HEIGHT. BMI=weight (kg)/height squared (m2). BMI correlates with body fat (ADIPOSE TISSUE). Their relationship varies with age and gender. For adults, BMI falls into these categories: below 18.5 (underweight); 18.5-24.9 (normal); 25.0-29.9 (overweight); 30.0 and above (obese). (National Center for Health Statistics, Centers for Disease Control and Prevention)Hypothalamus: Ventral part of the DIENCEPHALON extending from the region of the OPTIC CHIASM to the caudal border of the MAMMILLARY BODIES and forming the inferior and lateral walls of the THIRD VENTRICLE.Megestrol Acetate: Megestrol acetate is a progestogen with actions and uses similar to those of the progestogens in general. It also has anti-androgenic properties. It is given by mouth in the palliative treatment or as an adjunct to other therapy in endometrial carcinoma and in breast cancer. Megestrol acetate has been approved to treat anorexia and cachexia. (From Reynolds JEF(Ed): Martindale: The Extra Pharmacopoeia (electronic version). Micromedex, Inc, Englewood, CO, 1995)Hunger: The desire for FOOD generated by a sensation arising from the lack of food in the STOMACH.Hypothalamic Diseases: Neoplastic, inflammatory, infectious, and other diseases of the hypothalamus. Clinical manifestations include appetite disorders; AUTONOMIC NERVOUS SYSTEM DISEASES; SLEEP DISORDERS; behavioral symptoms related to dysfunction of the LIMBIC SYSTEM; and neuroendocrine disorders.Nutrition Disorders: Disorders caused by nutritional imbalance, either overnutrition or undernutrition.Vomiting: The forcible expulsion of the contents of the STOMACH through the MOUTH.Dog Diseases: Diseases of the domestic dog (Canis familiaris). This term does not include diseases of wild dogs, WOLVES; FOXES; and other Canidae for which the heading CARNIVORA is used.Weight Loss: Decrease in existing BODY WEIGHT.Energy Intake: Total number of calories taken in daily whether ingested or by parenteral routes.Melanocortins: Peptides derived from pro-opiomelanocortin (POMC) which can stimulate MELANOCYTES or CORTICOTROPHS. Melanocortins include ACTH; ALPHA-MSH; and other peptides such as BETA-MSH and GAMMA-MSH, derived from other fragments of POMC. These peptides act through a variety of MELANOCORTIN RECEPTORS to control different functions including steroidogenesis, energy homeostasis, feeding, and skin pigmentation.Body Composition: The relative amounts of various components in the body, such as percentage of body fat.Fatal Outcome: Death resulting from the presence of a disease in an individual, as shown by a single case report or a limited number of patients. This should be differentiated from DEATH, the physiological cessation of life and from MORTALITY, an epidemiological or statistical concept.Malnutrition: An imbalanced nutritional status resulted from insufficient intake of nutrients to meet normal physiological requirement.Satiation: Full gratification of a need or desire followed by a state of relative insensitivity to that particular need or desire.Chilblains: Recurrent localized itching, swelling and painful erythema on the fingers, toes or ears, produced by exposure to cold.Oxonic Acid: Antagonist of urate oxidase.Treatment Outcome: Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.Erysipelas: An acute infection of the skin caused by species of STREPTOCOCCUS. This disease most frequently affects infants, young children, and the elderly. Characteristics include pink-to-red lesions that spread rapidly and are warm to the touch. The commonest site of involvement is the face.Bone Density: The amount of mineral per square centimeter of BONE. This is the definition used in clinical practice. Actual bone density would be expressed in grams per milliliter. It is most frequently measured by X-RAY ABSORPTIOMETRY or TOMOGRAPHY, X RAY COMPUTED. Bone density is an important predictor for OSTEOPOROSIS.Peptide Hormones: Hormones synthesized from amino acids. They are distinguished from INTERCELLULAR SIGNALING PEPTIDES AND PROTEINS in that their actions are systemic.Pro-Opiomelanocortin: A 30-kDa protein synthesized primarily in the ANTERIOR PITUITARY GLAND and the HYPOTHALAMUS. It is also found in the skin and other peripheral tissues. Depending on species and tissues, POMC is cleaved by PROHORMONE CONVERTASES yielding various active peptides including ACTH; BETA-LIPOTROPIN; ENDORPHINS; MELANOCYTE-STIMULATING HORMONES; and others (GAMMA-LPH; CORTICOTROPIN-LIKE INTERMEDIATE LOBE PEPTIDE; N-terminal peptide of POMC or NPP).Nutritional Status: State of the body in relation to the consumption and utilization of nutrients.Fenfluramine: A centrally active drug that apparently both blocks serotonin uptake and provokes transport-mediated serotonin release.Neuropeptide Y: A 36-amino acid peptide present in many organs and in many sympathetic noradrenergic neurons. It has vasoconstrictor and natriuretic activity and regulates local blood flow, glandular secretion, and smooth muscle activity. The peptide also stimulates feeding and drinking behavior and influences secretion of pituitary hormones.Neoplasms: New abnormal growth of tissue. Malignant neoplasms show a greater degree of anaplasia and have the properties of invasion and metastasis, compared to benign neoplasms.Bhopal Accidental Release: 1984 accident in Bhopal, INDIA at a PESTICIDES facility, resulting when WATER entered a storage tank containing ISOCYANATES. The following accidental chemical release and uncontrolled reaction resulted in several thousand deaths.Isocyanates: Organic compounds that contain the -NCO radical.Military ScienceEthnobotany: The study of plant lore and agricultural customs of a people. In the fields of ETHNOMEDICINE and ETHNOPHARMACOLOGY, the emphasis is on traditional medicine and the existence and medicinal uses of PLANTS and PLANT EXTRACTS and their constituents, both historically and in modern times.Comprehension: The act or fact of grasping the meaning, nature, or importance of; understanding. (American Heritage Dictionary, 4th ed) Includes understanding by a patient or research subject of information disclosed orally or in writing.

Role of central melanocortins in endotoxin-induced anorexia. (1/490)

Inflammation and microbial infection produce symptoms, including fever, anorexia, and hypoactivity, that are thought to be mediated by endogenous proinflammatory cytokines. Melanocortins are known to act centrally to suppress effects on fever and other sequelae of proinflammatory cytokine actions in the central nervous system, but the roles of melanocortins in anorexia and hypoactivity occurring during the acute phase response are unknown. The present study was designed to determine the effects of exogenous and endogenous alpha-melanocyte stimulating hormone (alpha-MSH) on lipopolysaccharide (LPS)-induced anorexia in relation to their effects on fever. Rats were fasted overnight to promote feeding behavior, then injected intraperitoneally with LPS (100 micrograms/kg ip), followed 30 min later by intracerebroventricular injection of either alpha-MSH or the melanocortin receptor subtype 3/subtype 4 (MC3-R/MC4-R) antagonist SHU-9119. Food intake, locomotor activity, and body temperature (Tb) were monitored during the ensuing 24-h period. Each of two intracerebroventricular doses of alpha-MSH (30 and 300 ng) potentiated the suppressive effects of LPS on food intake and locomotion, despite the fact that the higher dose alleviated LPS-induced fever. In control rats that were not treated with LPS, only the higher dose of alpha-MSH significantly inhibited food intake, and Tb and locomotor activity were unaffected. To assess the roles of endogenous central melanocortins, LPS-treated rats received intracerebroventricular SHU-9119 (200 ng). Central MC3-R/MC4-R blockade did not affect Tb or food intake in the absence of LPS treatment, but it reversed the LPS-induced reduction in 24-h food intake and increased LPS-induced fever without altering the LPS-induced suppression of locomotion. Taken together, the results suggest that exogenous and endogenous melanocortins acting centrally exert divergent influences on different aspects of the acute phase response, suppressing LPS-induced fever but contributing to LPS-induced anorexia and hypoactivity.  (+info)

Weaning anorexia may contribute to local inflammation in the piglet small intestine. (2/490)

Compromising alterations in villus-crypt structure are common in pigs postweaning. Possible contributions of local inflammatory reactions to villus-crypt alterations during the weaning transition have not been described. This study evaluated local inflammatory responses and their relationship with morphological changes in the intestine in 21-d-old pigs (n = 112) killed either at weaning (Day 0) or 0.5, 1, 2, 4 or 7 d after weaning to either milk- or soy-based pelleted diets. Cumulative intake averaged <100 g during the first 2 d postweaning, regardless of diet. During this period of weaning anorexia, inflammatory T-cell numbers and local expression of the matrix metalloproteinase stromelysin increased while jejunal villus height, crypt depth and major histocompatibility complex (MHC) class I RNA expression decreased. Upon resumption of feed intake by the fourth d postweaning, villus height and crypt depth, CD8(+) T cell numbers, MHC class I RNA expression and local expression of stromelysin returned to Day 0 values. Together the results indicate that inadequate feed intake during the immediate postweaning period may contribute to intestinal inflammation and thereby compromise villus-crypt structure and function.  (+info)

The hypothalamic satiety peptide CART is expressed in anorectic and non-anorectic pancreatic islet tumors and in the normal islet of Langerhans. (3/490)

The hypothalamic satiety peptide CART (cocaine and amphetamine regulated transcript) is expressed at high levels in anorectic rat glucagonomas but not in hypoglycemic insulinomas. However, a non-anorectic metastasis derived from the glucagonoma retained high CART expression levels and produced circulating CART levels comparable to that of the anorectic tumors. Moreover, distinct glucagonoma lines derived by stable HES-1 transfection of the insulinoma caused severe anorexia but retained low circulating levels of CART comparable to that of insulinoma bearing or control rats. Islet tumor associated anorexia and circulating CART levels are thus not correlated, and in line with this peripheral administration of CART (5-50 mg/kg) produced no effect on feeding behavior. In the rat two alternatively spliced forms of CART mRNA exist and quantitative PCR revealed expression of both forms in the hypothalamus, in the different islet tumors, and in the islets of Langerhans. Immunocytochemistry as well as in situ hybridization localized CART expression to the somatostatin producing islet D cell. A potential endocrine/paracrine role of islet CART remains to be clarified.  (+info)

Evidence of splanchnic-brain signaling in inhibition of ingestive behavior by middle molecules. (4/490)

Anorexia, nausea, and vomiting are common symptoms of uremic intoxication. Fractions in the middle molecule weight range, isolated from normal urine and uremic plasma ultrafiltrate, inhibit ingestive behavior in the rat. To investigate their site of action and specificity, male rats were injected intraperitoneally, intravenously, or intracerebroventricularly with concentrated fractions of uremic plasma ultrafiltrate or normal urine (molecular weight range: 1.0 to 5.0 kD) and tested for ingestive and sexual behavior. An intraperitoneal injection of 0.5 ml of urine fraction (10:1) or 2.0 ml of uremic plasma ultrafiltrate fraction (25:1) inhibited carbohydrate intake by 76.3 and 45.9%, respectively, but an intravenous injection had no effect. However, intravenous injection of higher doses inhibited carbohydrate ingestion. An intracerebroventricular injection of 5 or 10 microl of urine (20:1) middle molecule fraction inhibited carbohydrate intake by 13.4 and 41.6%, respectively. An injection of 5 or 10 microl of uremic plasma ultrafiltrate (125:1) middle molecule fraction inhibited carbohydrate intake by 22.6 and 49.5%, respectively. Injections of the corresponding fraction from normal plasma ultrafiltrate had no effect. Injection of urine or uremic plasma ultrafiltrate middle molecule fractions did not affect the display of sexual behavior. These results suggest that middle molecule fractions from uremic plasma ultrafiltrate or normal urine act in the splanchnic region and/or brain to inhibit food intake and that the effect is specific for ingestive behavior.  (+info)

Capsaicin-sensitive fibers are required for the anorexic action of systemic but not central bombesin. (5/490)

Bombesin (BN) suppresses food intake in rats whether given centrally or systemically. Although the brain BN-sensitive receptors are known to be essential for the anorexic effect of systemic BN, the mode of communication between the gut and the brain remains unclear. This study assessed whether the anorexic effect of systemic BN is mediated humorally or via neural circuits. Afferent neurons were lesioned using capsaicin (50 mg/kg sc) on postnatal day 2, and responses to BN were assessed during adulthood. Capsaicin treatment decreased body weight gain significantly from postnatal age 4-7 wk. Peripheral BN (4-16 micrograms/kg ip) dose dependently suppressed food intake in control animals. However, this effect was completely blocked in capsaicin-treated rats. In contrast to systemic effects, feeding-suppressant effects of centrally administered BN (0.01-0.5 microgram icv) were not affected by capsaicin treatment. This research suggests that peripheral BN communicates with the brain via a neuronal system(s) whose afferent arm is constituted of capsaicin-sensitive C and/or Adelta-fibers, whereas the efferent arm of this satiety- and/or anorexia-mediating circuitry is capsaicin resistant.  (+info)

Role of cholecystokinin in the anorexia produced by duodenal delivery of peptone in rats. (6/490)

We used the cholecystokinin receptor antagonist devazepide to assess the importance of CCK in mediating the anorexia produced by 2-h duodenal infusions of peptone, a protein digest, at dark onset in nonfasted rats. Peptone alone (0.14-2.24 g/h) suppressed food intake dose dependently by 18-96%, with an approximate half-maximal dose of 1 g/h. Peptone-induced reductions in caloric ingestion were comparable to the caloric loads infused. Devazepide alone (30-1,000 microgram/kg) stimulated food intake dose dependently by 30-73%, with a minimal effective dose of 100 micrograms/kg. Devazepide appeared to reverse the anorexic response to peptone (1.1 g/h) dose dependently by 29-65%, with a minimal effective dose of 30 micrograms/kg. The magnitudes of these devazepide-induced effects were similar to, and in some cases were larger than, those produced when the same doses of devazepide were administered alone. Coadministration of devazepide (1,000 micrograms/kg) and a lower peptone dose (0.8 g/h) produced similar results. These results suggest that an essential CCK mechanism plays a significant role in mediating the satiety response to duodenal delivery of protein.  (+info)

Distinct patterns of neuropeptide gene expression in the lateral hypothalamic area and arcuate nucleus are associated with dehydration-induced anorexia. (7/490)

We have investigated the hormonal and hypothalamic neuropeptidergic substrates of dehydration-associated anorexia. In situ hybridization and hormone analyses of anorexic and paired food-restricted rats revealed two distinct profiles. First, both groups had the characteristic gene expression and endocrine signatures usually associated with starvation: increased neuropeptide Y and decreased proopiomelanocortin and neurotensin mRNAs in the arcuate nucleus (ARH); increased circulating glucocorticoid but reduced leptin and insulin. Dehydrated animals are strongly anorexic despite these attributes, showing that the output of leptin- and insulin-sensitive ARH neurons that ordinarily stimulate eating must be inhibited. The second pattern occurred only in anorexic animals and had two components: (1) reduced corticotropin-releasing hormone (CRH) mRNA in the neuroendocrine paraventricular nucleus (PVH) and (2) increased CRH and neurotensin mRNAs in the lateral hypothalamic (LHA) and retrochiasmatic areas. However, neither corticosterone nor suppressed PVH CRH gene expression is required for anorexia after dehydration because PVH CRH mRNA in dehydrated adrenalectomized animals is unchanged from euhydrated adrenalectomized controls. We also showed that LHA CRH mRNA was strongly correlated with the intensity of anorexia, increased LHA CRH gene expression preceded the onset of anorexia, and dehydrated adrenalectomized animals (which also develop anorexia) had elevated LHA CRH gene expression with a distribution pattern similar to intact animals. Finally, we identified specific efferents from the CRH-containing region of the LHA to the PVH, thereby providing a neuroanatomical framework for the integration by the PVH of neuropeptidergic signals from the ARH and the LHA. Together, these observations suggest that CRH and neurotensin neurons in the LHA constitute a novel anatomical substrate for their well known anorexic effects.  (+info)

Changes in neuropeptide Y receptors and pro-opiomelanocortin in the anorexia (anx/anx) mouse hypothalamus. (8/490)

The pro-opiomelanocortinergic (POMCergic) system originating in the hypothalamic arcuate nucleus extends projections widely over the brain and has been shown to be intricately linked and parallel to the arcuate neuropeptide Y (NPY) system. Both NPY and POMC-derived peptides (melanocortins) have been strongly implicated in the control of feeding behavior, with the former exerting orexigenic effects and the latter having anorexigenic properties. Mice homozygous for the lethal anorexia (anx) mutation are hypophagic, emaciated, and exhibit anomalous processing of NPY exclusively in the arcuate nucleus, providing an interesting model to study NPY-POMC interactions. In the present study, several morphological markers were used to investigate the histochemistry and morphology of the POMC system in anx/anx mice. In situ hybridization demonstrated decreased numbers of POMC mRNA-expressing neurons in the anx/anx arcuate nucleus. In parallel, mRNA levels for both the NPY Y1 and Y5 receptors, which are expressed in POMC neurons, were decreased. Also, expression of the NPY Y2 autoreceptor was attenuated. Immunohistochemistry using antibodies against adrenocorticotropic hormone to demonstrate POMC cell bodies, against alpha-melanocyte-stimulating hormone to demonstrate axonal projections and against the NPY Y1 receptor to demonstrate dendritic arborizations, showed strikingly decreased immunoreactivities for all these markers. The present data suggest that degeneration of the arcuate POMC system is a feature characteristic of the anx/anx mouse. The possible relationship to the NPYergic phenotype of this animal is discussed.  (+info)

  • Pro-anorexia websites are website suggesting that anorexia nervosa is a conscious lifestyle choice rather than a disorder. (
  • Visiting pro-anorexia websites was associated with higher drive for thinness, worse perception of appearance, more perfectionism and more fear of gaining weight, all potential predictors of developing anorexia nervosa. (
  • If women are increasingly pressured to 'have it all', then we predict a rise in the number of successful women suffering from anorexia," explains Dr Ian Frampton, senior lecturer in development neuropsychology at Exeter University. (
  • That inside voice was flirting with me but it hadn't become the full-blown "every single decision I make is based on how to avoid food at all costs" Anorexia voice, yet. (
  • Treatment of anorexia involves restoring a healthy weight, treating the underlying psychological problems, and addressing behaviors that promote the problem. (
  • You don't mention whether your friend has been diagnosed with anorexia or whether you've noticed behaviors that are indicative of disordered eating. (
  • Typically, people with anorexia use food and other unhealthy behaviors, such as exercising excessively, to cope with painful emotions. (
  • Also, many sufferers think of the behaviors associated with anorexias as a useful way to cope. (
  • In regards to food, people with anorexia can have obsessive thoughts and compulsive behaviors. (
  • Women in the study who had the most difficulty recognizing distorted positive emotions displayed anorexia -type behaviors -- such as calorie restriction, excessive exercise , laxative use, vomiting , and frequent body fat and weight checks -- more often. (
  • This study was the first to test the hypothesis that the behaviors in anorexia nervosa meet the cognitive neuroscience definition of 'habit',' Steinglass told Reuters Health by email. (
  • The scientists who led this study, published in The Lancet Psychiatry journal on Friday, said their results suggest deep brain stimulation - which involves implanting electrodes to stimulate brain areas that control dysfunctional behaviors - might alter the brain circuits that drive anorexia. (
  • There are different types of anorexia including restricting and binge-purge types. (
  • do chemical changes cause anorexia, or does anorexia cause chemical changes? (
  • Many factors combine to cause anorexia. (
  • Whatever its cause, anorexia can have a severe impact on a mature cat's health if it persists for as little as 24 hours. (
  • Contrary to her claim, there is no scientific evidence that families cause anorexia nervosa. (
  • Can Deep Brain Stimulation Help Anorexia? (
  • Researchers and doctors have begun using deep brain stimulation (DBS) to treat anorexia nervosa. (
  • Awake the whole time, Adrianna Boot underwent deep brain stimulation to treat her anorexia nervosa more than three years ago. (
  • The treatment, called deep brain stimulation, or DBS, has been used to treat a number of other conditions but had never before been tried with anorexia. (
  • Knowing the physical and emotional complications can also make it easier to help yourself or someone else recover from anorexia. (
  • Along with physical complications, a person with anorexia can also have emotional complications. (
  • Overcome anorexia for good by building emotional tools such as self-confidence, acceptance and motivation which will empower you to stay in control of your health. (
  • With workbook style exercises, this book will help you to identify the underlying causes of your anorexia, focusing on building your emotional wellbeing and confidence before giving tips on how you can make positive changes to your thinking and behaviour. (
  • Because anorexia is both a physical and emotional problem, you may work with a doctor, a dietitian , and a counsellor. (
  • Women with anorexia are often in complex emotional places, that is why it is important to understand all we can about what they are getting out of this experience," Selby said. (
  • Anorexia often stems from a distorted body image, which may result from emotional trauma, depression , or anxiety . (
  • Anorexia also includes emotional and behavioral issues involving an unrealistic perception of body weight and an extremely strong fear of gaining weight or becoming fat. (
  • While men do succumb to anorexia nervosa, (5 percent to 10 percent of sufferers) women are more likely to develop the disorder in adolescence or young adulthood. (
  • WOMEN in their 30s, 40s and even older are the new breed of anorexia nervosa sufferers, with a study showing that the disease is not only a condition of the young. (
  • Professor Touyz said this was because 30 per cent of anorexia sufferers who were not diagnosed in adolescence went on to suffer throughout their life. (
  • Many anorexia sufferers experience major changes in personality, going from happy and energetic to listless. (
  • Some anorexia sufferers develop a condition doctors call lanugo - soft, downy hair on their arms and legs. (
  • Politicians say the new law, which follows a failed attempt to crack down on the hiring of excessively thin fashion models, 'will not call into question the idea of freedom of expression on the Internet,' the New York Daily News reports, and will not apply to websites set up to help anorexia sufferers. (
  • In particular, feelings of stress, anxiety and low self-esteem are thought to trigger anorexia. (
  • Psychotherapy with adjunctive low-dose olanzapine may be useful for anorexia nervosa during inpatient treatment, especially in the context of anxiety, obsessive eating-related ruminations, and treatment resistance due to failure to engage. (
  • Women who have had a certain type of anorexia nervosa show an alteration of the activity of a chemical in their brain that is widely associated with anxiety and other affective disorders more than one year after recovery, according to a study in the September issue of Archives of General Psychiatry, one of the JAMA/Archives journals. (
  • Anxiety Induced Anorexia? (
  • Yet less than half of 1 percent of all women develop anorexia nervosa, which indicates to us that societal pressure alone isn't enough to cause someone to develop this disease," said Walter H. Kaye, M.D., professor of psychiatry, University of Pittsburgh School of Medicine. (
  • Many women with anorexia develop amenorrhea , or the absence of her menstrual period, but this is no longer a required criteria in the updated 2013 DSM-5 to receive an anorexia diagnosis. (
  • A new study by University of North Carolina at Chapel Hill and Norwegian researchers has found that women with anorexia nervosa are much more likely to have both unplanned pregnancies and induced abortions than women who don't have the serious eating disorder. (
  • Some women with anorexia take laxatives to help them lose weight. (
  • I'm very worried about one of my friends who suffers from anorexia. (
  • If you have a loved one who suffers from anorexia it is important to be there as a support system to them. (
  • If a child suffers from anorexia, family members should not let the eating disorder become a hidden, secret problem. (
  • When I received the news it had little effect on me-I was so entrenched in the desire to lose weight and stay inside the "safe" place that anorexia had provided that I couldn't see beyond the next meal, let alone consider my prognosis. (
  • Anorexia is an eating disorder that causes people lose more weight than is considered healthy for their age and height. (
  • As a general rule, the weight of an individual with anorexia nervosa is less than 85% of what is considered average when normalized for their age, height and gender. (
  • Anorexia nervosa is a complex condition, but the main sign is usually severe weight loss. (
  • People with anorexia nervosa purposely avoid food to prevent weight gain. (
  • Anorexia is a serious eating disorder that is associated with an intense fear of food and weight gain. (
  • While anorexia revolves around food and weight, it rarely has anything to do with those things. (
  • Anorexia nervosa (AN) is a potentially life-threatening eating disorder characterized by the inability to maintain a minimally normal weight, a devastating fear of weight gain, relentless dietary habits that prevent weight gain, and a disturbance in the way in which body weight and shape are perceived. (
  • Davis 1 Anorexia nervosa is defined as a refusal to maintain a minimally normal body weight, in addition to a disturbance in perception of body shape and weight( DSM-IV-TR, 2000). (
  • I'm recovering from anorexia and have been for over a year, I am now a healthy weight and have been for about 8 months, but I still haven't got my periods although I do get discharge in my underwear and have for about a year. (
  • WEDNESDAY, Aug. 6, 2014 (HealthDay News) -- Women with the eating disorder anorexia nervosa feel a sense of pride about their weight loss , and this positive emotion may play a major role in the deadly condition, according to a new study. (
  • In 2011, a study from the University of Haifa of 248 girls ages 12 to 19 found that the "more time girls spend on Facebook, the more they suffered conditions of bulimia, anorexia, physical dissatisfaction, negative physical self-image, negative approach to eating and more of an urge to be on a weight-loss diet. (
  • People with anorexia place a high value on controlling their weight and shape, using extreme efforts that tend to significantly interfere with their lives. (
  • In their recent newsletter, reveals that people with COPD (chronic obstructive pulmonary disease) are at increased risk for developing anorexia which results in unwanted weight loss. (
  • We present a model of weight choice and dieting applicable to anorexia nervosa, and the sometimes-associated purging behavior. (
  • Anorexia nervosa (AN) is an eating disorder associated with underweight, fear of weight gain, and disturbance in the way in which one's body weight or shape is experienced, undue influence of body weight on self-evaluation, or persistent lack of recognition of the seriousness of the low weight. (
  • The severe weight loss anorexia triggers can put an end to menstrual periods. (
  • That's an anorexia-related disorder in which severe weight loss damages the stomach muscles and nerves to the point that the process of digestion is affected. (
  • The weight loss may delay the onset of menstruation or stop it once it has started, Anorexia nervosa rarely occurs before puberty or after age 40. (
  • Anorexia is an eating disorder that makes people extremely afraid of gaining weight and gives them a distorted view of their body shape and size. (
  • Anorexia Nervosa is a troublesome disorder characterized by an obsession with weight and food. (
  • Anorexia nervosa is one of the most common psychiatric diagnoses in young women (Hus, 1996). (
  • Anorexia nervosa originates from a combination of psychiatric and metabolic factors, suggests a new genome-wide association study. (
  • Detailed findings appeared June 15 in the journal Nature Genetics , in an article titled, " Genome-wide association study identifies eight risk loci and implicates metabo-psychiatric origins for anorexia nervosa . (
  • Anorexia remains the psychiatric disorder with the highest mortality rate, and there is an urgent need to develop safe, effective, evidence-driven treatments that are informed by a growing understanding of brain circuitry,' he said. (
Anorexia Nervosa | Case Files: Psychiatry 5e | AccessMedicine | McGraw-Hill Medical
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Anorexia nervosa - Wikipedia
Anorexia nervosa - Wikipedia (
Epidemiology of Eating Disorders: Incidence, Prevalence and Mortality Rates | SpringerLink
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Mental Disorder (Illness) (Stanford Encyclopedia of Philosophy)
Mental Disorder (Illness) (Stanford Encyclopedia of Philosophy) (
Binge eating |
Binge eating | (
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Anorexia Nervosa : Judy's Story
Anorexia Nervosa : Judy's Story (
NEWSDAY | Preteen eating disorders on the rise | CBC News
NEWSDAY | Preteen eating disorders on the rise | CBC News (
FDA Approved Drugs by Medical Condition | CenterWatch
FDA Approved Drugs by Medical Condition | CenterWatch (
Anorexia by londun howard on Prezi
Anorexia by londun howard on Prezi (
Cold intolerance: Causes, Symptoms and Diagnosis
Cold intolerance: Causes, Symptoms and Diagnosis (
Anorexia | Cornell University College of Veterinary Medicine
Anorexia | Cornell University College of Veterinary Medicine (
'Do not say anything': Threat to career forced ex-Olympian Allison Forsyth to stay mum on Bertrand Charest assault | National...
'Do not say anything': Threat to career forced ex-Olympian Allison Forsyth to stay mum on Bertrand Charest assault | National... (
Cancer Cachexia and Anorexia | SpringerLink
Cancer Cachexia and Anorexia | SpringerLink ( Search results : fat-thin - Nemours KidsHealth Search results : fat-thin - Nemours KidsHealth (
Anorexia Nervosa | National Eating Disorders Association
Anorexia Nervosa | National Eating Disorders Association (
Anorexia Nervosa: Practice Essentials, Background, Pathophysiology
Anorexia Nervosa: Practice Essentials, Background, Pathophysiology (
Diagnosing an Eating Disorder
Diagnosing an Eating Disorder (
The Psychobiology of Anorexia Nervosa | SpringerLink
The Psychobiology of Anorexia Nervosa | SpringerLink (
Taste Responsiveness in Anorexia Nervosa | The British Journal of Psychiatry | Cambridge Core
Taste Responsiveness in Anorexia Nervosa | The British Journal of Psychiatry | Cambridge Core (
You snooze, you lose? Think again | National Post
You snooze, you lose? Think again | National Post (
Sex hormones for gender transitioning hike risk of stroke, heart attacks, study finds | National Post
Sex hormones for gender transitioning hike risk of stroke, heart attacks, study finds | National Post (
Anxiety disorder associated with a two times higher risk for cancer death - but only in men, study finds | National Post
Anxiety disorder associated with a two times higher risk for cancer death - but only in men, study finds | National Post (
On a Diet? Learn How Distraction Reduces Temptation | Psychology Today
On a Diet? Learn How Distraction Reduces Temptation | Psychology Today (
6 Common Types of Eating Disorders (and Their Symptoms)
6 Common Types of Eating Disorders (and Their Symptoms) (
Eating disorders: key facts
Eating disorders: key facts (
Signs of Anorexia?
Signs of Anorexia? (
Wannarexia: What Parents Can Do
Wannarexia: What Parents Can Do (
Anorexia Nervosa Causes
Anorexia Nervosa Causes (
Anorexia+nervosa | Origin and meaning of phrase anorexia+nervosa by Online Etymology Dictionary
Anorexia+nervosa | Origin and meaning of phrase anorexia+nervosa by Online Etymology Dictionary (
Bulimia | Psych Central
Bulimia | Psych Central (
Eating Disorders Help - Forums and Discussions - MedHelp
Eating Disorders Help - Forums and Discussions - MedHelp (
Hormone boost 'may help infertility' - BBC News
Hormone boost 'may help infertility' - BBC News (
Mesothelioma Treatment Side Effects | Chemo Brain, Anorexia & More
Mesothelioma Treatment Side Effects | Chemo Brain, Anorexia & More (