The symptom of paroxysmal pain consequent to MYOCARDIAL ISCHEMIA usually of distinctive character, location and radiation. It is thought to be provoked by a transient stressful situation during which the oxygen requirements of the MYOCARDIUM exceed that supplied by the CORONARY CIRCULATION.
Precordial pain at rest, which may precede a MYOCARDIAL INFARCTION.
A clinical syndrome characterized by the development of CHEST PAIN at rest with concomitant transient ST segment elevation in the ELECTROCARDIOGRAM, but with preserved exercise capacity.
Persistent and reproducible chest discomfort usually precipitated by a physical exertion that dissipates upon cessation of such an activity. The symptoms are manifestations of MYOCARDIAL ISCHEMIA.
A volatile vasodilator which relieves ANGINA PECTORIS by stimulating GUANYLATE CYCLASE and lowering cytosolic calcium. It is also sometimes used for TOCOLYSIS and explosives.
A technique for assisting the circulation by decreasing the afterload of the left ventricle and augmenting the diastolic pressure. It may be achieved by intra-aortic balloon, or by implanting a special pumping device in the chest, or externally by applying a negative pressure to the lower extremities during cardiac systole.
Radiography of the vascular system of the heart muscle after injection of a contrast medium.
ANGINA PECTORIS or angina-like chest pain with a normal coronary arteriogram and positive EXERCISE TEST. The cause of the syndrome is unknown. While its recognition is of clinical importance, its prognosis is excellent. (Braunwald, Heart Disease, 4th ed, p1346; Jablonski Dictionary of Syndromes & Eponymic Diseases, 2d ed). It is different from METABOLIC SYNDROME X, a syndrome characterized by INSULIN RESISTANCE and HYPERINSULINEMIA, that has increased risk for cardiovascular disease.
NECROSIS of the MYOCARDIUM caused by an obstruction of the blood supply to the heart (CORONARY CIRCULATION).
Controlled physical activity which is performed in order to allow assessment of physiological functions, particularly cardiovascular and pulmonary, but also aerobic capacity. Maximal (most intense) exercise is usually required but submaximal exercise is also used.
An imbalance between myocardial functional requirements and the capacity of the CORONARY VESSELS to supply sufficient blood flow. It is a form of MYOCARDIAL ISCHEMIA (insufficient blood supply to the heart muscle) caused by a decreased capacity of the coronary vessels.
Recording of the moment-to-moment electromotive forces of the HEART as projected onto various sites on the body's surface, delineated as a scalar function of time. The recording is monitored by a tracing on slow moving chart paper or by observing it on a cardioscope, which is a CATHODE RAY TUBE DISPLAY.
Spasm of the large- or medium-sized coronary arteries.
A drug used in the treatment of angina pectoris, heart failure, conduction defects, and myocardial infarction. It is a partial agonist at beta adrenergic receptors and acts as a coronary vasodilator and cardiotonic agent.
Pathological processes of CORONARY ARTERIES that may derive from a congenital abnormality, atherosclerotic, or non-atherosclerotic cause.
A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (CORONARY ARTERY DISEASE), to obstruction by a thrombus (CORONARY THROMBOSIS), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (MYOCARDIAL INFARCTION).
A vasodilator used in the treatment of ANGINA PECTORIS. Its actions are similar to NITROGLYCERIN but with a slower onset of action.
An ergot alkaloid (ERGOT ALKALOIDS) with uterine and VASCULAR SMOOTH MUSCLE contractile properties.
The veins and arteries of the HEART.
Compounds possessing both a hydroxyl (-OH) and an amino group (-NH2).
Drugs that bind to but do not activate beta-adrenergic receptors thereby blocking the actions of beta-adrenergic agonists. Adrenergic beta-antagonists are used for treatment of hypertension, cardiac arrhythmias, angina pectoris, glaucoma, migraine headaches, and anxiety.
Studies in which individuals or populations are followed to assess the outcome of exposures, procedures, or effects of a characteristic, e.g., occurrence of disease.
An aspect of personal behavior or lifestyle, environmental exposure, or inborn or inherited characteristic, which, on the basis of epidemiologic evidence, is known to be associated with a health-related condition considered important to prevent.
Observation of a population for a sufficient number of persons over a sufficient number of years to generate incidence or mortality rates subsequent to the selection of the study group.
Dilation of an occluded coronary artery (or arteries) by means of a balloon catheter to restore myocardial blood supply.
Pressure, burning, or numbness in the chest.
A potent vasodilator agent with calcium antagonistic action. It is a useful anti-anginal agent that also lowers blood pressure.
Expenditure of energy during PHYSICAL ACTIVITY. Intensity of exertion may be measured by rate of OXYGEN CONSUMPTION; HEAT produced, or HEART RATE. Perceived exertion, a psychological measure of exertion, is included.
A selective adrenergic beta-1 blocking agent that is commonly used to treat ANGINA PECTORIS; HYPERTENSION; and CARDIAC ARRHYTHMIAS.
Percutaneous transluminal procedure for removing atheromatous plaque from the coronary arteries. Both directional (for removing focal atheromas) and rotational (for removing concentric atheromatous plaque) atherectomy devices have been used.
A widely used non-cardioselective beta-adrenergic antagonist. Propranolol has been used for MYOCARDIAL INFARCTION; ARRHYTHMIA; ANGINA PECTORIS; HYPERTENSION; HYPERTHYROIDISM; MIGRAINE; PHEOCHROMOCYTOMA; and ANXIETY but adverse effects instigate replacement by newer drugs.
Compounds based on N-phenylacetamide, that are similar in structure to 2-PHENYLACETAMIDES. They are precursors of many other compounds. They were formerly used as ANALGESICS and ANTIPYRETICS, but often caused lethal METHEMOGLOBINEMIA.
A method of studying a drug or procedure in which both the subjects and investigators are kept unaware of who is actually getting which specific treatment.
The restoration of blood supply to the myocardium. (From Dorland, 28th ed)
Motion pictures of the passage of contrast medium through blood vessels.
A beta-1 adrenergic antagonist that has been used in the emergency treatment of CARDIAC ARRYTHMIAS.
The number of times the HEART VENTRICLES contract per unit of time, usually per minute.
An episode of MYOCARDIAL ISCHEMIA that generally lasts longer than a transient anginal episode that ultimately may lead to MYOCARDIAL INFARCTION.
Radiography of the heart and great vessels after injection of a contrast medium.
Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.
A prediction of the probable outcome of a disease based on a individual's condition and the usual course of the disease as seen in similar situations.
Measurable and quantifiable biological parameters (e.g., specific enzyme concentration, specific hormone concentration, specific gene phenotype distribution in a population, presence of biological substances) which serve as indices for health- and physiology-related assessments, such as disease risk, psychiatric disorders, environmental exposure and its effects, disease diagnosis, metabolic processes, substance abuse, pregnancy, cell line development, epidemiologic studies, etc.
Surgical therapy of ischemic coronary artery disease achieved by grafting a section of saphenous vein, internal mammary artery, or other substitute between the aorta and the obstructed coronary artery distal to the obstructive lesion.
A beta-adrenergic antagonist used in the treatment of hypertension, angina pectoris, arrhythmias, and anxiety.
Graphic registration of the heart sounds picked up as vibrations and transformed by a piezoelectric crystal microphone into a varying electrical output according to the stresses imposed by the sound waves. The electrical output is amplified by a stethograph amplifier and recorded by a device incorporated into the electrocardiograph or by a multichannel recording machine.
The circulation of blood through the CORONARY VESSELS of the HEART.
A derivative of the NIACINAMIDE that is structurally combined with an organic nitrate. It is a potassium-channel opener that causes vasodilatation of arterioles and large coronary arteries. Its nitrate-like properties produce venous vasodilation through stimulation of guanylate cyclase.
Method in which prolonged electrocardiographic recordings are made on a portable tape recorder (Holter-type system) or solid-state device ("real-time" system), while the patient undergoes normal daily activities. It is useful in the diagnosis and management of intermittent cardiac arrhythmias and transient myocardial ischemia.
Works about pre-planned studies of the safety, efficacy, or optimum dosage schedule (if appropriate) of one or more diagnostic, therapeutic, or prophylactic drugs, devices, or techniques selected according to predetermined criteria of eligibility and observed for predefined evidence of favorable and unfavorable effects. This concept includes clinical trials conducted both in the U.S. and in other countries.
Genotypic differences observed among individuals in a population.
Drugs used to cause dilation of the blood vessels.
Drugs that inhibit the actions of the sympathetic nervous system by any mechanism. The most common of these are the ADRENERGIC ANTAGONISTS and drugs that deplete norepinephrine or reduce the release of transmitters from adrenergic postganglionic terminals (see ADRENERGIC AGENTS). Drugs that act in the central nervous system to reduce sympathetic activity (e.g., centrally acting alpha-2 adrenergic agonists, see ADRENERGIC ALPHA-AGONISTS) are included here.
Elements of limited time intervals, contributing to particular results or situations.
Lesions formed within the walls of ARTERIES.
Severe cellulitis of the submaxillary space with secondary involvement of the sublingual and submental space. It usually results from infection in the lower molar area or from a penetrating injury to the mouth floor. (From Dorland, 27th ed)
A cardioselective beta-1 adrenergic blocker possessing properties and potency similar to PROPRANOLOL, but without a negative inotropic effect.
A class of drugs that act by selective inhibition of calcium influx through cellular membranes.
A calcium channel blocker that is a class IV anti-arrhythmia agent.
The use of ultrasound to guide minimally invasive surgical procedures such as needle ASPIRATION BIOPSY; DRAINAGE; etc. Its widest application is intravascular ultrasound imaging but it is useful also in urology and intra-abdominal conditions.
PRESSURE of the BLOOD on the ARTERIES and other BLOOD VESSELS.
A plasma protein that circulates in increased amounts during inflammation and after tissue damage.
Narrowing or constriction of a coronary artery.
Any dummy medication or treatment. Although placebos originally were medicinal preparations having no specific pharmacological activity against a targeted condition, the concept has been extended to include treatments or procedures, especially those administered to control groups in clinical trials in order to provide baseline measurements for the experimental protocol.
Application of electric current to the spine for treatment of a variety of conditions involving innervation from the spinal cord.
Studies which start with the identification of persons with a disease of interest and a control (comparison, referent) group without the disease. The relationship of an attribute to the disease is examined by comparing diseased and non-diseased persons with regard to the frequency or levels of the attribute in each group.
AMINO ALCOHOLS containing the propanolamine (NH2CH2CHOHCH2) group and its derivatives.
Agents that affect the rate or intensity of cardiac contraction, blood vessel diameter, or blood volume.
In screening and diagnostic tests, the probability that a person with a positive test is a true positive (i.e., has the disease), is referred to as the predictive value of a positive test; whereas, the predictive value of a negative test is the probability that the person with a negative test does not have the disease. Predictive value is related to the sensitivity and specificity of the test.
The natural enzymatic dissolution of FIBRIN.
The movement and the forces involved in the movement of the blood through the CARDIOVASCULAR SYSTEM.
Levels within a diagnostic group which are established by various measurement criteria applied to the seriousness of a patient's disorder.
The use of photothermal effects of LASERS to coagulate, incise, vaporize, resect, dissect, or resurface tissue.
The return of a sign, symptom, or disease after a remission.
One of the three polypeptide chains that make up the TROPONIN complex. It is a cardiac-specific protein that binds to TROPOMYOSIN. It is released from damaged or injured heart muscle cells (MYOCYTES, CARDIAC). Defects in the gene encoding troponin T result in FAMILIAL HYPERTROPHIC CARDIOMYOPATHY.
Procedures in which placement of CARDIAC CATHETERS is performed for therapeutic or diagnostic procedures.
Application of electric current in treatment without the generation of perceptible heat. It includes electric stimulation of nerves or muscles, passage of current into the body, or use of interrupted current of low intensity to raise the threshold of the skin to pain.
A salicylamide derivative that is a non-cardioselective blocker of BETA-ADRENERGIC RECEPTORS and ALPHA-1 ADRENERGIC RECEPTORS.
Maintenance of blood flow to an organ despite obstruction of a principal vessel. Blood flow is maintained through small vessels.
The hollow, muscular organ that maintains the circulation of the blood.
Coagulation of blood in any of the CORONARY VESSELS. The presence of a blood clot (THROMBUS) often leads to MYOCARDIAL INFARCTION.
A pteridine derivative present in body fluids; elevated levels result from immune system activation, malignant disease, allograft rejection, and viral infections. (From Stedman, 26th ed) Neopterin also serves as a precursor in the biosynthesis of biopterin.
The exercise capacity of an individual as measured by endurance (maximal exercise duration and/or maximal attained work load) during an EXERCISE TEST.
Diseases which have one or more of the following characteristics: they are permanent, leave residual disability, are caused by nonreversible pathological alteration, require special training of the patient for rehabilitation, or may be expected to require a long period of supervision, observation, or care. (Dictionary of Health Services Management, 2d ed)
Therapy with two or more separate preparations given for a combined effect.
A moderately lipophilic beta blocker (ADRENERGIC BETA-ANTAGONISTS). It is non-cardioselective and has intrinsic sympathomimetic actions, but little membrane-stabilizing activity. (From Martindale, The Extra Pharmocopoeia, 30th ed, p638)
A vasodilator used in angina of effort or ischemic heart disease.
A benzothiazepine derivative with vasodilating action due to its antagonism of the actions of CALCIUM ion on membrane functions.
Ocular disorders attendant upon non-ocular disease or injury.
Dosage forms of a drug that act over a period of time by controlled-release processes or technology.
Methods and procedures for the diagnosis of diseases or dysfunction of the cardiovascular system or its organs or demonstration of their physiological processes.
Plasma glycoprotein clotted by thrombin, composed of a dimer of three non-identical pairs of polypeptide chains (alpha, beta, gamma) held together by disulfide bonds. Fibrinogen clotting is a sol-gel change involving complex molecular arrangements: whereas fibrinogen is cleaved by thrombin to form polypeptides A and B, the proteolytic action of other enzymes yields different fibrinogen degradation products.
A drug formerly used in the treatment of angina pectoris but superseded by less hazardous drugs. Prenylamine depletes myocardial catecholamine stores and has some calcium channel blocking activity. (From Martindale, The Extra Pharmacopoeia, 30th ed, p1406)
Procedures for finding the mathematical function which best describes the relationship between a dependent variable and one or more independent variables. In linear regression (see LINEAR MODELS) the relationship is constrained to be a straight line and LEAST-SQUARES ANALYSIS is used to determine the best fit. In logistic regression (see LOGISTIC MODELS) the dependent variable is qualitative rather than continuously variable and LIKELIHOOD FUNCTIONS are used to find the best relationship. In multiple regression, the dependent variable is considered to depend on more than a single independent variable.
An alpha-1 adrenergic antagonist that is commonly used as an antihypertensive agent.
Devices that provide support for tubular structures that are being anastomosed or for body cavities during skin grafting.
A distribution in which a variable is distributed like the sum of the squares of any given independent random variable, each of which has a normal distribution with mean of zero and variance of one. The chi-square test is a statistical test based on comparison of a test statistic to a chi-square distribution. The oldest of these tests are used to detect whether two or more population distributions differ from one another.
The abrupt cessation of all vital bodily functions, manifested by the permanent loss of total cerebral, respiratory, and cardiovascular functions.
The amount of BLOOD pumped out of the HEART per beat, not to be confused with cardiac output (volume/time). It is calculated as the difference between the end-diastolic volume and the end-systolic volume.
A clinical syndrome with intermittent abdominal pain characterized by sudden onset and cessation that is commonly seen in infants. It is usually associated with obstruction of the INTESTINES; of the CYSTIC DUCT; or of the URINARY TRACT.
A vasodilator with general properties similar to NITROGLYCERIN but with a more prolonged duration of action. (From Martindale, The Extra Pharmacopoeia, 30th ed, p1025)
Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.
Drugs or agents which antagonize or impair any mechanism leading to blood platelet aggregation, whether during the phases of activation and shape change or following the dense-granule release reaction and stimulation of the prostaglandin-thromboxane system.
The number of new cases of a given disease during a given period in a specified population. It also is used for the rate at which new events occur in a defined population. It is differentiated from PREVALENCE, which refers to all cases, new or old, in the population at a given time.
A phosphodiesterase inhibitor that blocks uptake and metabolism of adenosine by erythrocytes and vascular endothelial cells. Dipyridamole also potentiates the antiaggregating action of prostacyclin. (From AMA Drug Evaluations Annual, 1994, p752)
A single nucleotide variation in a genetic sequence that occurs at appreciable frequency in the population.
Studies in which subsets of a defined population are identified. These groups may or may not be exposed to factors hypothesized to influence the probability of the occurrence of a particular disease or other outcome. Cohorts are defined populations which, as a whole, are followed in an attempt to determine distinguishing subgroup characteristics.
Recording of the moment-to-moment electromotive forces of the heart on a plane of the body surface delineated as a vector function of time.
A set of techniques used when variation in several variables has to be studied simultaneously. In statistics, multivariate analysis is interpreted as any analytic method that allows simultaneous study of two or more dependent variables.
The probability that an event will occur. It encompasses a variety of measures of the probability of a generally unfavorable outcome.
A transferase that catalyzes formation of PHOSPHOCREATINE from ATP + CREATINE. The reaction stores ATP energy as phosphocreatine. Three cytoplasmic ISOENZYMES have been identified in human tissues: the MM type from SKELETAL MUSCLE, the MB type from myocardial tissue and the BB type from nervous tissue as well as a mitochondrial isoenzyme. Macro-creatine kinase refers to creatine kinase complexed with other serum proteins.
A method of computed tomography that uses radionuclides which emit a single photon of a given energy. The camera is rotated 180 or 360 degrees around the patient to capture images at multiple positions along the arc. The computer is then used to reconstruct the transaxial, sagittal, and coronal images from the 3-dimensional distribution of radionuclides in the organ. The advantages of SPECT are that it can be used to observe biochemical and physiological processes as well as size and volume of the organ. The disadvantage is that, unlike positron-emission tomography where the positron-electron annihilation results in the emission of 2 photons at 180 degrees from each other, SPECT requires physical collimation to line up the photons, which results in the loss of many available photons and hence degrades the image.
Inorganic or organic salts and esters of nitric acid. These compounds contain the NO3- radical.
The qualitative or quantitative estimation of the likelihood of adverse effects that may result from exposure to specified health hazards or from the absence of beneficial influences. (Last, Dictionary of Epidemiology, 1988)
Computed tomography where there is continuous X-ray exposure to the patient while being transported in a spiral or helical pattern through the beam of irradiation. This provides improved three-dimensional contrast and spatial resolution compared to conventional computed tomography, where data is obtained and computed from individual sequential exposures.
2-(2,2-Dicyclohexylethyl)piperidine. Coronary vasodilator used especially for angina of effort. It may cause neuropathy and hepatitis.
Descriptions of specific amino acid, carbohydrate, or nucleotide sequences which have appeared in the published literature and/or are deposited in and maintained by databanks such as GENBANK, European Molecular Biology Laboratory (EMBL), National Biomedical Research Foundation (NBRF), or other sequence repositories.
The genetic constitution of the individual, comprising the ALLELES present at each GENETIC LOCUS.
A characteristic symptom complex.
The lower right and left chambers of the heart. The right ventricle pumps venous BLOOD into the LUNGS and the left ventricle pumps oxygenated blood into the systemic arterial circulation.
The formation of clumps of RED BLOOD CELLS under low or non-flow conditions, resulting from the attraction forces between the red blood cells. The cells adhere to each other in rouleaux aggregates. Slight mechanical force, such as occurs in the circulation, is enough to disperse these aggregates. Stronger or weaker than normal aggregation may result from a variety of effects in the ERYTHROCYTE MEMBRANE or in BLOOD PLASMA. The degree of aggregation is affected by ERYTHROCYTE DEFORMABILITY, erythrocyte membrane sialylation, masking of negative surface charge by plasma proteins, etc. BLOOD VISCOSITY and the ERYTHROCYTE SEDIMENTATION RATE are affected by the amount of erythrocyte aggregation and are parameters used to measure the aggregation.
Progressive diminution of the susceptibility of a human or animal to the effects of a drug, resulting from its continued administration. It should be differentiated from DRUG RESISTANCE wherein an organism, disease, or tissue fails to respond to the intended effectiveness of a chemical or drug. It should also be differentiated from MAXIMUM TOLERATED DOSE and NO-OBSERVED-ADVERSE-EFFECT LEVEL.
Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues. The standard approach is transthoracic.
The process which spontaneously arrests the flow of BLOOD from vessels carrying blood under pressure. It is accomplished by contraction of the vessels, adhesion and aggregation of formed blood elements (eg. ERYTHROCYTE AGGREGATION), and the process of BLOOD COAGULATION.
The muscle tissue of the HEART. It is composed of striated, involuntary muscle cells (MYOCYTES, CARDIAC) connected to form the contractile pump to generate blood flow.
Statistical models used in survival analysis that assert that the effect of the study factors on the hazard rate in the study population is multiplicative and does not change over time.
The internal resistance of the BLOOD to shear forces. The in vitro measure of whole blood viscosity is of limited clinical utility because it bears little relationship to the actual viscosity within the circulation, but an increase in the viscosity of circulating blood can contribute to morbidity in patients suffering from disorders such as SICKLE CELL ANEMIA and POLYCYTHEMIA.
Abdominal symptoms after removal of the GALLBLADDER. The common postoperative symptoms are often the same as those present before the operation, such as COLIC, bloating, NAUSEA, and VOMITING. There is pain on palpation of the right upper quadrant and sometimes JAUNDICE. The term is often used, inaccurately, to describe such postoperative symptoms not due to gallbladder removal.
Pathological conditions involving the CARDIOVASCULAR SYSTEM including the HEART; the BLOOD VESSELS; or the PERICARDIUM.
Persistently high systemic arterial BLOOD PRESSURE. Based on multiple readings (BLOOD PRESSURE DETERMINATION), hypertension is currently defined as when SYSTOLIC PRESSURE is consistently greater than 140 mm Hg or when DIASTOLIC PRESSURE is consistently 90 mm Hg or more.

Investigation of the most effective provocation test for patients with coronary spastic angina: usefulness of accelerated exercise following hyperventilation. (1/135)

This study sought to compare the clinical usefulness of the hyperventilation plus cold stress test or the hyperventilation combined with accelerated exercise test with other single tests in patients with coronary spastic angina. The study examined 24 patients (23 men, mean age 66 years) with angiographically confirmed coronary spastic angina and less than 50% stenosis. Moreover, none had spontaneous ST segment elevation before the study. Under no medication for at least 24 h prior, 4 procedures were performed from 09.00 h to 11.00 h: (i) a hyperventilation test for 5 min (HV(5)); (ii) HV(5) combined with a cold stress test for the last 2 min (HV(5)+CS(2)); (iii) a treadmill exercise test based on Bruce's protocol (TM(3)); and (iv) a treadmill exercise test accelerated at 1 min intervals according to Bruce's protocol immediately after HV(5) (HV(5)+TM(1)). The rate of appearance of chest pain and ischemia-induced ECG changes due to HV(5)+TM(1) were significantly higher than the other 3 tests. HV(5)+CS(2) was not superior to HV(5) alone. The incidence of provoked ST segment elevation due to HV(5)+TM(1) was higher than with the other 3 procedures. Thus, in patients with coronary spastic angina, no spontaneous ST segment elevation and near normal coronary arteries, HV(5)+CS(2) was no more useful than HV(5) alone. It is recommended that the newly designed HV(5)+TM(1) combination test be used for documenting evidence of ischemia in patients with coronary spastic angina, low disease activity and near normal coronary arteries.  (+info)

Consumption of vitamin E in coronary circulation in patients with variant angina. (2/135)

OBJECTIVES: The plasma status of vitamin E has been suggested to be linked to the activity of coronary artery spasm. This study was designed to determine whether vitamin E is actually consumed in the coronary circulation in patients with active variant angina having repetitive spasm-induced transient myocardial ischemia and reperfusion. METHODS: Blood samples were obtained simultaneously from the aortic root, coronary sinus and right atrium in 12 patients with variant angina due to spasm of the left coronary artery, nine patients with stable effort angina and nine control subjects. Plasma vitamin E (alpha- and gamma-tocopherol) concentrations were determined by use of high-performance liquid chromatography and plasma lipid peroxides were measured as thiobarbituric acid-reactive substances (TBARS). RESULTS: At baseline, both plasma alpha- (p < 0.01) and gamma- (p < 0.05) tocopherol levels were significantly lower in the coronary sinus (5.50 +/- 0.50 and 0.55 +/- 0.07 mg/l, mean +/- SEM) than in the aortic root (6.63 +/- 0.57 and 0.63 +/- 0.08 mg/l) and also in the right atrium (6.44 +/- 0.61 and 0.63 +/- 0.09 mg/l) in the variant angina group. The TBARS level was significantly (p < 0.05) higher in the coronary sinus than in the aortic in this group. In contrast, these levels were not significantly different between the samples from the coronary sinus and the aortic root or the right atrium in the control group and also in the stable effort angina group. The coronary sinus-aortic difference in plasma vitamin E levels in the variant angina group was not significantly altered after left coronary artery spasm induced by intracoronary injection of acetylcholine. Also, the plasma vitamin E levels in the aortic root, coronary sinus and right atrium all remained unchanged in the stable effort angina group after pacing-induced angina and in the control group after intracoronary administration of acetylcholine. CONCLUSIONS: Transcardiac reduction in plasma vitamin E concentrations concomitant with lipid peroxide formation was demonstrated in patients with active variant angina, suggesting actual consumption of this major endogenous antioxidant. Oxidative stress and vitamin E exhaustion may be involved in the pathogenesis of coronary artery spasm.  (+info)

Racial heterogeneity in coronary artery vasomotor reactivity: differences between Japanese and Caucasian patients. (3/135)

Japanese investigators have provided a substantial contribution in the understanding of coronary vasomotor reactivity. On occasions, their findings have been at variance with those undertaken on caucasian patients, raising speculation that vasomotor differences between races may exist. In a comparative review of the published literature, we evaluated the vasoreactive differences among Japanese and caucasian patients with variant angina or myocardial infarction. In variant angina, Japanese patients appear to have diffusely hyperreactive coronary arteries compared with caucasian people, manifested by their segmental rather than focal spasm, hyperreactive nonspastic vessels and multivessel spasm. These differences may reflect the increased basal tone among Japanese variant angina patients and may relate to controversial differences in endothelial nitric oxide production or autonomic nervous system activity. Provocative vasomotor studies of Japanese patients with a recent myocardial infarction report a higher incidence of inducible spasm than caucasian studies, an observation recently supported by a controlled study. Furthermore, the hyperreactivity was diffuse, occurring in both non-infarct- and infarct-related vessels. These observations support the existence of racial coronary vasomotor reactivity differences but require confirmation in further prospectively conducted studies.  (+info)

Alterations of autonomic nervous activity in recurrence of variant angina. (4/135)

OBJECTIVE: To investigate whether autonomic nervous activity is involved in the recurrence of spontaneous coronary spasm in variant angina. DESIGN: Retrospective analysis. SETTING: Cardiology department of a university hospital. PATIENTS: 18 patients with variant angina were divided into single attack group (SA; nine patients) and multiple attack group (MA; nine patients) according to the frequency of ischaemic episodes with ST segment elevation during 24 hour Holter monitoring. METHODS: Heart rate variability indices were calculated using MemCalc method, which is a combination of the maximum entropy method for spectral analysis and the non-linear least squares method for fitting analysis, at 30 second intervals for 30 second periods, from 40 minutes before the attack to 30 minutes after the attack. High frequency (HF; 0.04-0.15 Hz) was defined as a marker of parasympathetic activity, and the ratio of low frequency (LF; 0.15-0.40 Hz) to high frequency (LF/HF) as an indicator of sympathetic activity. The averaged value during the 40 to 30 minute period before an attack was defined as the baseline. RESULTS: Compared with baseline, the HF component decreased in both groups at two minutes before the attack (p < 0.01), and the LF/HF ratio decreased at three minutes before the attack (p < 0.01). The baseline LF/HF was lower in the MA group than in the SA group (p < 0. 01). CONCLUSIONS: A reduction of sympathetic activity may play a key role in determining the recurrence of transient ischaemic events caused by spontaneous coronary spasm in patients with variant angina.  (+info)

Vasospastic angina likely related to cisplatin-containing chemotherapy and thoracic irradiation for lung cancer. (5/135)

Vasospastic angina is rarely observed during cancer treatment. The present report describes two males with lung cancer, aged 73 and 61, who developed vasospastic angina during combination treatment of cisplatin-containing chemotherapy and thoracic irradiation. As both patients have smoked and their ages are typical for patients with coronary artery disease, such events may be incidental. However, oncologists should be aware of the possible development of myocardial ischemia during or following administration of antineoplastic agents, especially in elderly patients with pre-existing coronary risk factors or a history of thoracic radiotherapy.  (+info)

Prognostic significance of the pattern of multivessel spasm in patients with variant angina. (6/135)

Multivessel spasm in variant angina is believed to be a major prognostic factor. Three patterns of multivessel spasm have been detected: (1) spasm at different sites on different occasions (migratory spasm); (2) spasm sequentially affecting 2 different sites (sequential spasm); and (3) simultaneous spasm at more than 1 site (simultaneous spasm). The present study investigated the prognosis based on this factor for variant angina without fixed coronary stenosis and examined the influence of multivessel spasm on cardiac events. Twenty-six patients were diagnosed as having variant angina without fixed coronary stenosis using 12-lead 24-h ECG recording system and coronary cineangiography. These patients were followed up prospectively for 57.1+/-7.6 months. Of the 26 patients 13 had single-vessel spasm, 6 had migratory multivessel spasm angina, and 7 showed sequential and/or simultaneous multivessel spasm angina. The survival free of serious cardiac events and of all cardiac events was significantly lower for patients with sequential and/or simultaneous multivessel spasm than for those with migratory multivessel spasm (p<0.05, p<0.05), whereas for patients with migratory multivessel spasm the difference comparison with single-vessel spasm did not attain statistical significance (p = ns, p = ns). The results of this study suggest that there seems to be a high-risk subgroup (i.e., sequential and/or simultaneous multivessel spasm) among patients with variant angina.  (+info)

Plasma endothelin-1 elevation associated with alcohol-induced variant angina. (7/135)

Vasospastic angina as a result of alcohol ingestion has been reported, but the mechanism of alcohol-induced coronary artery spasm is presently unknown. This report presents 2 cases of alcohol-induced variant angina (VA) with elevated levels of plasma endothelin-1 after alcohol ingestion. In case 1, the plasma endothelin-1 concentration was 3.15 pg/ml before drinking (normal <2.30 pg/ml) and increased to 4.09 pg/ml when measured 5 h after alcohol ingestion. After 2 months of abstinence, the plasma endothelin-1 concentration was 2.88 pg/ml and 6 months after abstinence, it decreased to 2.03 pg/ml (normal range). In case 2, the plasma endothelin-1 concentration was 2.44 pg/ml before drinking and increased to 4.36 pg/ml when measured 5 h after alcohol ingestion. After 2 months of abstinence, the plasma endothelin-1 concentration was 3.04 pg/ml and 6 months after abstinence, it decreased to 2.09 pg/ml (normal range). These 2 cases suggest that a relationship may exist between alcohol-induced VA and elevation in the plasma endothelin-1 concentration after alcohol ingestion.  (+info)

Beware of the heart: the multiple picture of cardiac involvement in myositis. (8/135)

A 42-yr-old woman with dermatomyositis had two myocardial infarctions, episodes of acute chest pain and an acute lung oedema. These events were initially misinterpreted as atherosclerotic ischaemic heart disease accompanying the autoimmune disease. The lack of improvement of cardiac symptoms with anti-ischaemic and immunosuppressive drugs indicated other mechanisms. Intracoronary drug provocation as well as myocardial biopsy revealed a coincidence of small-vessel disease and vasospastic angina as a cause for the severe cardiac symptoms. After initiating therapy with high doses of calcium channel blockers, marked improvement of cardiac symptoms occurred. In the pathogenesis of cardiac involvement in dermatomyositis, two different mechanisms should be considered: inflammatory processes due to dermatomyositis and vasoconstriction caused by an impaired regulation of vascular tone, such as abnormal vessel reactivity or disturbed neuropeptide release. Signs of this generalized vasopathy are Raynaud's phenomenon, Prinzmetal's angina and small-vessel disease, which can coincide. In patients with severe cardiac symptoms and autoimmune diseases, Prinzmetal's angina should be excluded by intracoronary drug provocation using acetylcholine.  (+info)

We made continuous electrocardiographic recordings on magnetic tape during 15 episodes of ischemia in five patients with variant angina to determine the characteristics of the QRS changes. Orthogonal leads were used and the electrocardiograms were analyzed visually and by digital computer. Changes were quantified by subtracting baseline electrocardiograms from those obtained during ischemia. Large changes in the QRS occurred during ischemia but the waveform quickly returned to baseline when the episode subsided. In all patients there was prolongation of the QRS duration and an increase in QRS voltage during the terminal 40 msec of the waveform in the lead(s) showing the most marked ST displacement. The increase in the terminal QRS could be represented by a vector directed toward the ischemic zone. In a given patient the amplitude of ST displacement varied between episodes, presumably because of variation in the intensity of ischemia, but the QRS changes were directionally similar in each ...
A 24-year-old male student had three myocardial infarctions, one prior to and two following the angiographic documentation of normal coronary arteries. A spontaneous episode of variant angina prompted repeat coronary angiography, during which intravenous ergonovine caused spasm of the left anterior descending coronary artery, transient ST-segment elevation, and ischemic chest pain; the previously normal right coronary artery was found to be occluded proximally. This constellation of clinical and angiographic findings suggests that coronary spasm can cause acute myocardial infarction as well as variant angina. ...
TY - JOUR. T1 - Basal release of endothelium-derived nitric oxide at site of spasm in patients with variant angina. AU - Egashira, Kensuke. AU - Katsuda, Yousuke. AU - Mohri, Masahiro. AU - Kuga, Takeshi. AU - Tagawa, Tatuya. AU - Shimokawa, Hiroaki. AU - Takeshita, Akira. N1 - Copyright: Copyright 2017 Elsevier B.V., All rights reserved.. PY - 1996/5. Y1 - 1996/5. N2 - Objectives. The aim of this study was to investigate the basal release of nitric oxide at spastic sites in patients with variant angina. Background. We previously reported that endothelium-dependent dilator responses to acetylcholine, substance P and bradykinin are preserved at the site of coronary artery spasm. However, it is not known whether the basal release of endothelium-derived nitric oxide is altered at the spastic site. Methods. The effects of intracoronary N(G)-monomethyl-L-arginine (L-NMMA, an inhibitor of nitric oxide synthesis) at cumulative doses of 50, 100 and 200 μmol on basal coronary artery tone were ...
Coronary artery spasm is an important pathogenetic mechanism in some forms of myocardial ischemic disease. Factors that may be important in the genesis of spasm include the autonomic nervous system, prostaglandins, endoperoxides, thromboxanes, and the calcium availability to the contractile apparatus. Spasm results in myocardial ischemia with attendant chest pain and electrocardiographic and hemodynamic changes; it is the primary pathogenetic mechanism in Prinzmetals variant angina and has been found in association with classic angina pectoris and acute myocardial infarction. Diagnosis of coronary artery spasm is firmly made only by coronary angiography. Treatment includes the use of both short- and long-acting nitrates and the slow-channel blocking agents such as verapamil, nifedipine, and perhexiline. ...
Felodipine, a dihydropyridine calcium-channel blocker, is used alone or with an angiotensin-converting enzyme inhibitor, to treat hypertension, chronic stable angina pectoris, and Prinzmetals variant angina. Felodipine is similar to other peripheral vasodilators. Felodipine inhibits the influx of extra cellular calcium across the myocardial and vascular smooth muscle cell membranes blocking the calcium channels. The decrease in intracellular calcium inhibits the contractile processes of the myocardial smooth muscle cells, causing dilation of the coronary and systemic arteries, increased oxygen delivery to the myocardial tissue, decreased total peripheral resistance, decreased systemic blood pressure, and decreased afterload ...
Felodipine, a dihydropyridine calcium-channel blocker, is used alone or with an angiotensin-converting enzyme inhibitor, to treat hypertension, chronic stable angina pectoris, and Prinzmetals variant angina. Felodipine is similar to other peripheral vasodilators. Felodipine inhibits the influx of extra cellular calcium across the myocardial and vascular smooth muscle cell membranes blocking the calcium channels. The decrease in intracellular calcium inhibits the contractile processes of the myocardial smooth muscle cells, causing dilation of the coronary and systemic arteries, increased oxygen delivery to the myocardial tissue, decreased total peripheral resistance, decreased systemic blood pressure, and decreased afterload ...
Novel compounds of the general formula: ##STR1## and pharmaceutically acceptable acid addition salts thereof, wherein the compounds are useful in therapy to protect skeletal muscles against damage resulting from trauma or to protect skeletal muscles subsequent to muscle or systemic diseases such as intermittent claudication, to treat shock conditions, to preserve donor tissue and organs used in transplants, in the treatment of cardiovascular diseases including atrial and ventricular arrhythmias, Prinzmetals (variant) angina, stable angina, and exercise induced angina, congestive heart disease, and myocardial infarction.. Granted by the US Patent Office February 22, 2001 ⇒. ...
Learn more about Variant Angina: Heart Spasms at Redmond Regional Medical Center Whenever most people think of chest pain, they associate it with a heart...
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Vasospastic angina is presented by myocardial ischemia with spasm of coronary artery accompanying chest pain or discomfort. The precise mechanisms have not been established, but a reduction in NO (nitric oxide) production, an imbalance between endothelium-derived relaxing and contracting factors,or an injury of endothelium have been suggested.. Impaired FMD(flow mediated endothelium-dependent vasodilation) in the brachial artery was demonstrated in vasospastic angina,and improvement of endothelial dysfunction with treatment of statin is documented in several studies.. So, we expect that statin treatment for vasospastic angina provide additional therapeutic effects via improvement of endothelial dysfunction. ...
Her finder du det sidste nye om Prinzmetal Variant Angina og det Kardiologiske Syndrom X, samt et debatforum hvor patienter har mulighed for at komme i kontakt med andre i samme situation.
Her finder du det sidste nye om Prinzmetal Variant Angina og det Kardiologiske Syndrom X, samt et debatforum hvor patienter har mulighed for at komme i kontakt med andre i samme situation.
We conclude that 1) plasma levels of FPA, BTG, and PF4 were increased in patients with variant angina as compared with those with stable exertional angina; 2) there was a significant circadian variation in the plasma levels of FPA in parallel with that of the frequency of the attacks with the peak level occurring from midnight to early morning in patients with variant angina; and 3) elevated levels of plasma FPA are the result and not the cause of coronary spasm ...
It is believed that the vasoconstriction caused by thromboxanes plays a role in Prinzmetals angina. Omega-3 fatty acids are metabolized to produce higher levels of TxA,3 which is relatively less potent than TxA2 and PGI3; therefore, there is a balance shift toward inhibition of vasoconstriction and platelet aggregation. It is believed that this shift in balance lowers the incidence of myocardial infarction (heart attack) and stroke. Vasoconstriction and, perhaps, various proinflammatory effects exerted by TxA on tissue microvasculature, is probable reason why the TxA is pathogenic in various diseases, such as ischemia-reperfusion injury.,[2] hepatic inflammatory processes,[3] acute hepatotoxicity [4] etc. TxB2, a stable degradation product of TxA2, plays a role in acute hepatoxicity induced by acetaminophen.[5][6]. ...
It is believed that the vasoconstriction caused by thromboxanes plays a role in Prinzmetals angina. Omega-3 fatty acids are metabolized to produce higher levels of TxA,3 which is relatively less potent than TxA2 and PGI3; therefore, there is a balance shift toward inhibition of vasoconstriction and platelet aggregation. It is believed that this shift in balance lowers the incidence of myocardial infarction (heart attack) and stroke. Vasoconstriction and, perhaps, various proinflammatory effects exerted by TxA on tissue microvasculature, is probable reason why the TxA is pathogenic in various diseases, such as ischemia-reperfusion injury.,[2] hepatic inflammatory processes,[3] acute hepatotoxicity [4] etc. TxB2, a stable degradation product of TxA2, plays a role in acute hepatoxicity induced by acetaminophen.[5][6] ...
Chest pain that results when blood flow through the coronary arteries is insufficient to meet the oxygen needs of the heart. Often simply called angina. It is marked by pressure, squeezing, or general discomfort in the heart, breast, and neck, and can also spread to the back, shoulders, jaw, arm, and fingers. People experiencing angina may also feel light-headed and have an abnormally fast or irregular heartbeat. See Stable angina, Unstable angina, and Variant angina.. ...
AMLODIPINO ACCORD aa Comp. 10 mg,efectos secundarios, efectos adversos, precio (Amlodipino besilato) de ACCORD HEALTHCARE S.L.U. es indicado para Angina de pecho crónica estable,Angina de pecho vasospástica (angina de Prinzmetal, angina variante),Hipertensión arterial esencial. Incluye indicaciones de AMLODIPINO ACCORD y información detallade de Amlodipino besilato.
Cardiac syndrome X is angina (chest pain) with signs associated with decreased blood flow to heart tissue but with normal coronary arteries. Cardiac syndrome X is sometimes referred to as microvascular angina when there are findings of microvascular dysfunction. Some studies have found increased risk of other vasospastic disorders in cardiac syndrome X patients, such as migraine and Raynauds phenomenon. It is treated with beta-blockers, such as metoprolol, and usually carries a favorable prognosis. This is a distinct diagnosis from Prinzmetals angina. While there is no formal definition for cardiac syndrome X, the general consensus is that it entails all of the following: Angina: This usually does not cause dysfunction on echocardiogram and can last longer than that of heart disease. Abnormal cardiac stress test: ST changes are typically similar to those of coronary artery disease, and the opposite of those of Prinzmetals angina. Myocardial perfusion imaging can be abnormal in 30% of ...
Symptoms are often very similar to pain associated with a more typical heart attack (development of a blockage in a heart artery). Symptoms often occur at rest, and at night. This is different than typical chest pain associated with heart artery blockages which is more typically experience during exertion.. ...
Vasospastic angina is caused by sudden occlusive vasoconstriction of a segment of an epicardial artery, which can present with a wide spectrum of clinical scenario. We report the cases of two patients diagnosed with vasospastic angina, with one of which presenting with sudden cardiac arrest, while the other presenting with a relatively benign syncope. But both of them have J waves formation on ECG during active ischemia. The diagnosis and management of vasospastic angina, as well as the proposed clinical significance of J waves during coronary spasm are discussed. ...
Indications for Drugs ::. Mild to moderate hypertension, Chronic stable and vasospastic angina, Raynauds disease, Coronary Artery Disease, HTN, Stroke prevention. Drug Dose ::. Adult: PO Stable angina; HTN; Prinzmetals angina Initial: 5 mg once daily; If the desired therapeutic effect cannot be achieved within 2-4 weeks, the dose may be increased to a maximum dose of 10 mg once daily. Child: >12 yr Initially, 2.5 mg once daily, increased to 5 mg once daily if necessary. Elderly: Initially, 2.5 mg once daily. Hepatic impairment: Initially, 2.5 mg once daily.. Contraindication ::. Hypersensitivity to Amlodipine or other calcium channel antagonist, Severe hypertension.. Drug Precautions ::. Impaired liver or renal function, CHF, sick-sinus syndrome, severe ventricular dysfunction, hypertrophic cardiomyopathy, severe aortic stenosis. Caution when used in patients with idiopathic hypertrophic subaortic stenosis. Elderly, children. Pregnancy, lactation. Use in renal failure: Although Amlodipine is ...
Indications for Drugs ::. Mild to moderate hypertension, Chronic stable and vasospastic angina, Raynauds disease, Coronary Artery Disease, HTN, Stroke prevention. Drug Dose ::. Adult: PO Stable angina; HTN; Prinzmetals angina Initial: 5 mg once daily; If the desired therapeutic effect cannot be achieved within 2-4 weeks, the dose may be increased to a maximum dose of 10 mg once daily. Child: >12 yr Initially, 2.5 mg once daily, increased to 5 mg once daily if necessary. Elderly: Initially, 2.5 mg once daily. Hepatic impairment: Initially, 2.5 mg once daily.. Contraindication ::. Hypersensitivity to Amlodipine or other calcium channel antagonist, Severe hypertension.. Drug Precautions ::. Impaired liver or renal function, CHF, sick-sinus syndrome, severe ventricular dysfunction, hypertrophic cardiomyopathy, severe aortic stenosis. Caution when used in patients with idiopathic hypertrophic subaortic stenosis. Elderly, children. Pregnancy, lactation. Use in renal failure: Although Amlodipine is ...
Angina pectoris in pregnancy is unusual and Prinzmetals angina is much rarer. It accounts for 2% of all cases of angina. It is caused by vasospasm, but the mechanism of spasm is unknown but has been linked with hyperthyroidism in some studies. Patients with thyrotoxicosis-induced acute myocardial infarction are unusual and almost all reported cases have been associated with Graves disease. Human chorionic gonadotropin hormone-induced hyperthyroidism occurs in about 1.4% of pregnant women, mostly when hCG levels are above 70-80 000 IU/L. Gestational transient thyrotoxicosis is transient and generally resolves spontaneously in the latter half of pregnancy, and specific antithyroid treatment is not required. Treatment with calcium channel blockers or nitrates reduces spasm in most of these patients. Overall, the prognosis for hyperthyroidism-associated coronary vasospasm is good. We describe a very rare case of an acute myocardial infarction in a 27-year-old female, at 9 weeks of gestation due to ...
There have been rare reports of serious cardiac adverse reactions, including acute myocardial infarction, occurring within a few hours following administration of SUMAVEL® DosePro®. SUMAVEL® DosePro® may cause coronary artery vasospasm (Prinzmetals angina). These types of reactions have occurred in some patients without known CAD. For triptan-naïve patients with multiple cardiovascular risk factors who have a negative cardiovascular evaluation, consider administrating the first SUMAVEL® DosePro® dose in a medically-supervised setting and performing an electrocardiogram (ECG) immediately following SUMAVEL® DosePro® administration. For such patients, consider periodic cardiovascular evaluation in intermittent long-term users of SUMAVEL® DosePro®. Life-threatening arrhythmias have been reported within a few hours following the administration of 5-HT1 agonists. Discontinue SUMAVEL® DosePro® if these disturbances occur. Sensations of tightness, pain, pressure, and heaviness have been ...
Do not take Coreg if you are allergic to Coreg components.. Do not take Coreg if youre pregnant or you plan to have a baby, or you are a nursing mother.. Be careful with Coreg if you have a history of asthma, emphysema, thyroid disorder, pheochromocytoma, myasthenia gravis, low blood pressure, liver, kidney or heart disease diabetes, hyperthyroidism, depression, Prinzmetals angina, bronchitis.. Be careful using Coreg if you take monoamine oxidase inhibitors (tranylcypromine (such as Parnate), isocarboxazid (such as Marplan), selegiline (such as Zelapar, Eldepryl, Emsam), phenelzine (such as Nardil)); verapamil (such as Calan,Verelan, Covera-HS); paroxetine (such as Paxil); cimetidine (such as Tagamet); rifampin (such as Rifadin, Rimactane); clonidine (such as Catapres), cyclosporine (such as Sandimmune, Neoral); digoxin (such as Lanoxin, Lanoxicaps); quinidine; diltiazem (such as Tiazac, Cardizem); fluoxetine (such as Prozac); epinephrine (such as Epipen); oral diabetes medicines and insulin; ...
Exercise induced ST elevation in patients with previous myocardial infarction may simply indicate left ventricular wall asynergy. ST segment elevation without prior myocardial infarction is a rare condition and is commonly associated with critical coronary artery stenosis.1 Temporary ST segment elevation associated with chest pain is the hallmark of variant (Prinzmetal) angina, and is a consequence of severe coronary spasm. In a way that is not yet fully understood, exercise can induce coronary spasm in patients with variant angina, as demonstrated in small groups of patients subjected to supine bicycle exercise on the cardiac catheterisation table.2DSE is widely performed as a useful diagnostic tool in patients with known or suspected coronary artery disease. Through its inotropic and chronotropic effects, dobutamine increases the myocardial oxygen demand and may induce ischaemia and segmental wall motion abnormality in the presence of significant coronary artery disease. Myocardial ischaemia ...
In 1959 Myron Prinzmetal first described variant angina. Without the aid of selective coronary angiography he attributed the syndrome to the temporary occlusion of a large diseased artery with a narrow lumen due to a normal increase in the tonus of the vessel wall.. A 66 year old woman who had been smoking 15 cigarettes each day for 50 years and who had a long history of Raynauds syndrome presented with an episode of severe chest pain associated with pronounced ST elevation in the inferior and lateral leads (left). She was enrolled in a thrombolytic trial that involved the performance of early coronary angiography. Ninety minutes after receiving 100 mg of tissue plasminogen activator the ST segments were still elevated. Coronary angiography of the right coronary artery showed a discrete 90% stenosis that resolved following 200 μg of intracoronary nitroglycerin (top). An ECG recorded 30 minutes later demonstrated complete resolution of the ST segments (bottom). There was no subsequent enzyme ...
There are three types of angina - stable, unstable and prinzmetals. Stable angina is the most common and symptoms usually last for a few minutes before they subside, often with the help of nitroglycerine tablets.. With unstable angina the symptoms can be more severe and not so predictable. The attacks often last much longer and can occur even at times of rest. An unstable attack can be a precursor to a heart attack and thus it is taken more seriously than stable angina. Medical attention should be sought immediately at the first sign of unstable angina.. Prinzmetals, is defined as angina that occurs when the patient is at rest, rather than the result of physical exercise. Dr James Pierce Ph.D., relates that hes identified the cause of this angina. He says that it generally occurs at certain times of the day, in the early morning and late afternoon.(1) These are, as it happens, the times of day when Mg is at its lowest ebb in the body.. Dr Pierce estimates that some 50% of sudden heart attacks ...
A discrepancy in the prevalence of coronary artery spasm exists across different ethnicities; in particular, the Japanese population has been reported to have a greater prevalence of this condition compared to Caucasians.[6] In fact, according to a study involving 2251 patients, coronary artery spasm has been estimated to account for approximately 41% of Japanese patients with angina pectoris who underwent angiography.[7] In addition, coronary spasm in Japanese is characterized by a diffuse hyperreactivity as manifested by a segmental pattern of spasm as well as by a multivessel involvement compared to focal involvement in other populations.[8] Moreover, following the administration of acetylcholine early after myocardial infarction for provocation of spasm, there has been three fold higher incidence of coronary spasm and a higher incidence of multivessel spasm among Japanese compared to Caucasians.[9] These findings highlight that the vasomotor reactivity of coronary artery is not homogeneous ...
Rev Esp Cardiol. 1997 Nov;50(11):808-11. [The placement of a Wiktor stent for the treat ment of vasospastic angina: a case report]. [Article in Spanish] Rodríguez Díez S1, Lázaro R, Ruiz Nodar JM, Enero J, Romero C, Gómez Recio M, Martínez Elbal L.
Get even more testosterone 99 k24 apotik jual viagra dietary tricks too much effort and variant angina refractory to atropine may produce marked respiratory depression it is applied to the clitoral body. Preferred usage solvent misuse . See also attitude change. Discourse n. A tendency to produce peripheral vasodilatation by stimulating the uterus is primarily generated during the 1987s at the anterior spinal roots to sweat on regaining consciousness may be effective in blocking the release of histamine on isolated rabbit auricle. Mutter et al. Acquired barbiturate tolerance usually disappears at around what time he receives a call to mind, from reagain + mens mind + neuron a nerve + shuairuo weak] shen-kuei n. A cognitive style defined by such traits as imagination, curiosity, and creativity, and at least reliably report their heart rate and leads to local therapy are treated for adnexal torsion in adolescents: Update and review of high-risk factors to determine tumor diameter, called r1 ...
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In patients with angina and nonobstructive coronary-artery disease, coexistence of epicardial coronary spasm and increased microvascular resistance (IMR) is associated with worse outcomes, according to Japanese researchers.
Angina pectoris,Types as stable,unstable and even Microvascular,Signs and Symptoms,Risk factors,Diagnosis,Life Style Modification and Prinzmetal Angina
This drug is recommended to treat arterial hypertension and in the preventive treatment of crises of pectoris angina: effort pectoris angina and spontaneous pectoris angina ( of which the Prinzmetal angor ...
Prinzmetal angina olarak da bilinen vazospastik angina, bir ok fakt r n tetikleyebilece i bir damar a r duyarl l olarak d n lmektedir. T bbi tedaviye yan t s kl kla iyidir, ancak her be hastadan birinde diren li semptomlar g r lmektedir. Nadiren vazospazma ba l l mc l aritmiler g r lebilmektedir ve bu hastalarda prognoz daha k t d r. Burada, kalsiyum kanal blokeri ve nitrat tedavisine ra men semptomlar devam eden diren li bir vazospastik anginal olguyu sunduk. Hasta, hemodinamik bozuklu a yol a an ve tekrarlayan kardiyoversiyonlara diren li y ksek h zl ventrik ler ta ikardi ile ba vurdu. T bbi tedavisine ek olarak, koronerlerdeki belirgin vazospastik segmentlere stent yerle tirilerek tedavi edildi. Ayr ca, vazospazm kesin bir tedavisi olmayan yayg n bir vask ler hastal k oldu undan, ventrik l aritmilerine kar ikincil korunma ama l implante edilebilir defibrilat r yerle tirildi ...
The Baseline and Classical controllers use a classic cascaded-loop architecture with three inner P-only loops to control the angular rates p,q,r, and three outer PI loops to control the angular positions phi,alpha,beta. The six proportional gains and three integral gains are all scheduled as a function of alpha and beta. The Baseline variant contains the baseline design featured in docid:aeroblks_ug.f4-50264. Parts 2 and 3 of this series use the Classical variant to walk through the tuning process. The active variant is controlled by the workspace variable CTYPE. Set its value to 2 to activate the Classical variant of the controller.. ...
Collins, Hugh (2011) Constructive dismissal and the West Lothian question: Aberdeen City Council v McNeill. Industrial Law Journal, 40 (4). pp. 439-450. ISSN 0305-9332 ...
1. Armstrong PW. Stable ischemic syndromes. In: Topol E. Textbook of Cardiovascular Medicine. Philadelphia: Lippincott Raven Publishers 1998. 2. Chatterjee T, Juelke PD, Thum P et al. Successful brachytherapy of coronary vasospasm. Heart 2003; 89(9): 25. 3. Cheng TO. Clinical implication of the hyperventilation test in the diagnosis of coronary artery spasm. Am J Cardiol 1997; 80: 1647. 4. Gersh BJ, Braunwald E, Bonow BO. Chronic coronary artery disease. In: Braunwald E. Heart Disease. A textbook of cardiovascular medicine. 6th ed. Philadelphia: WB Saunders 2001. 5. Halawa B, Salomon P. Activity of transmembrane calcium transport and levels of endothelin-1 in patients with variant angina. Pol Arch Med Wewn 2000; 104(2): 447-453. 6. Hirano Y, Ozasa Y, Yamamoto T et al. Hyperventilation and cold pressor stress echocardiography for noninvasive diagnosis of coronary artery spasm. J Am Soc Echocardiogr 2001; 14(6): 626-633. 7. Hirano Y, Ozasa Y, Yamamoto T et al. Diagnosis of vasospastic angina by ...
Angina Pectoris Drugs Market research report provides the newest industry data and industry future trends, allowing you to identify the products and end users driving revenue growth and profitability. Angina pectoris is a clinical indication characterized by precordial heaviness or discomfort due to transient myocardial ischemia without infarction, elicited by physical exertion or psychological stress. Angina pectoris is categorized as - stable, unstable, microvascular and Prinzmetal / variant. Angina pectoris is an initial presentation of coronary heart disease (CHD) and exerts a major impact on quality of life (QOL), costs to the society and ability to work.. Chronic stable angina pectoris has a prevalence of 2.0 - 4.0% in the seven major markets (U.S., U.K., Germany, France, Italy, Spain, Japan). Heart Disease and Stroke Statistics from American Heart Association estimates indicate that over nine million adults in the U.S. have chronic angina pectoris.. Get Free Sample Copy of Report ...
In the ovarian pregnancy to dilation of causation of smell -ia indicating a condition or the hymenal tissue can be generalizable to see aphasia. Sensory modality effect, it is inactive. Thus, clinically used in control of severity of preterm birth to level of these v-shaped wedges and organ failure in normals consuming alcohol without conviction. [from greek peri around a variant angina. Table 69. Postnatal 73 102) in cardiac pain syndrome has elevated erythrocyte sedimentation rateplasma viscosity raised) calculating naming of causative pathogens involved. The history is used if the brain tumor. When this then the right to chloroform (fig. However, it in agoraphobia is an adjuvant therapy on the myometrium viagrx the incidence of the sutures; the hip. Protective positioning or surgery. Surgery for almost always a more difficult and hence collagen deposition of the liver requires percutaneous absorption of iga or with pain loss viagra side effects hearing gl is allowed to erfects or high-grade ...
rate control has more long term benefit than conversion and maintenance of sinus (NEJM 347:p. 1825, 2002 and NEJM 347:p. 1834, 2002) One study showed safe to withhold anticoagulation if less than 48 hours (Ann Intern Med 1997;126:615) Transient ST-depression with Rapid AF - Significance? Transient ST-segment depression during rapid atrial fibrillation is a common finding in the ED. Frequently, patients without known CAD exhibit such ischemic ST-segment depression during an episode of rapid AF. Clinicians often consider this to be a positive stress-test equivalent. However, a recent study indicates that in patients without a history of cardiovascular disease, there is no strong association between transient ischemic type ST-segment depression during paroxysms of AF and underlying occult CAD, i.e., they are not consistently associated with with positive stress testing or occlusions on cardiac catheterization (1). Conversely, however, if the ST-segment depression persists after the rate is ...
Introduction Oral capecitabine is an oral prodrug of 5-fluorouracil that has been integrated into the management of multiple cancer types because of the convenience of administration and its efficacy...
The angina is the medical condition in which the person faces pain in the chest, and that pain may extend towards the left arm of the person. Angina also named angina pectoris is an initial level pain that lasts for quite some time. The main reason for such pain is the inadequate supply of the blood to the heart. It wont be wrong to say that ischemia causes angina. The ischemia is the condition of the short supply of the blood and angina comes out as the result of it. Many of the expert physicians term angina pectoris as the mini heart attack as it is often perceived as the alarm of the serious heart issues. In a maximum of the cases angina is nothing more than pain, means it doesnt lead to the death of the person. Angina indicates the heart trouble at the initial level when the person may undergo the ischemia. The inadequacy of the blood leads to the issues like angina pectoris, which are not much serious if treated at initial level properly. Mainly there are two types of angina, one is ...
The term angina pectoris refers to a feeling of pain or discomfort in the chest. Angina pectoris occurs when the heart muscle does not get enough blood and as a result, not enough oxygen, to function normally. Angina pectoris is usually caused by the hardening of the arteries. When plaques largely block the coronary
Angina pectoris, also known as Angina, a symptoms of Ischemic heart disease, is defined as a condition of chest pain caused by poor blood flow through the blood vessels due to obstruction or spasm of the coronary arteries resulting of lack of blood that lead to lack of oxygen supply and waste removal. Preventions A. …. ...
Angina pectoris, also known as Angina, a symptoms of Ischemic heart disease, is defined as a condition of chest pain caused by poor blood flow through the blood vessels due to obstruction or spasm of the coronary arteries resulting of lack of blood that lead to lack of oxygen supply and waste removal. Types of …. ...
Angina pectoris - or simply angina - is chest pain or discomfort that keeps coming back. It happens when some part of your heart does not get enough blood and oxygen.
Angina pectoris - or simply angina - is chest pain or discomfort that keeps coming back. It happens when some part of your heart does not get enough blood and oxygen.
Many sufferers of chest pain have asked what is angina and the symptoms of angina attack. People want answers to the question what is angina...
Angina is the medical term for chest pain caused by the heart. Learn more about whether your chest pain is angina, as well as how to handle angina symptoms.
Study Flashcards On Angina Pectoris: Pharmacology at Cram.com. Quickly memorize the terms, phrases and much more. Cram.com makes it easy to get the grade you want!
Study Flashcards On Medsurg: Cardiovascular: Angina Pectoris at Cram.com. Quickly memorize the terms, phrases and much more. Cram.com makes it easy to get the grade you want!
Angina is not a heart attack, but it does increase your risk of having a heart attack. Know the ways using which you can cope with Angina.
I saw a cardiologist a few months ago. She said my symptoms didnt sound like angina because angina pain is usually the same each time it happens. Does anyone have any views on her comment about ang...
Angina can be a painful chronic condition but with angina medications & treatments those with angina can reduce pain. Get more info & browse our Rx discounts.
Angina Symptoms and what they mean Angina is caused by the narrowing of the Coronary arteries, these are the vessels that supply your heart with blood. The
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Read about angina (chest pain) and the reasons why you should not take this symptom lightly. Angina may be a precursor to a heart attack.
Learn more about Angina at TriStar Southern Hills DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
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A variant form of angina pectoris. Preliminary report. Am Heart J 1959; 27:375. For example Henry Dudeney noted in his 1917 ... Prinzmetal angina: also known as variant angina, referring to angina (chest pain) caused by vasospasm of the coronary arteries ... Electrocardiographic study during a paroxysm of angina pectoris. Am Heart J 1933; 9:259. Prinzmetal, M, Kennamer, R, Merliss, R ... Electrocardiographic changes during brief attacks of angina pectoris. Lancet 1931; 1:15. Brow, GR, Holman, DV. ...
"Comparison of Plasma Concentrations of Thromboxane B-2 and Prinzmetal's Variant Angina and Classical Angina Pectoris". Clinical ... "Detection of thromboxane B2 in peripheral blood of patients with Prinzmetal's angina". Prostaglandines and Medicine. 2 (4): 243 ...
"Angina pectoris I. A variant form of angina pectoris". The American Journal of Medicine. 27 (3): 375-388. doi:10.1016/0002-9343 ... Cheng, Tsung O. (1972-05-01). "Variant Angina of Printzmetal with Normal Coronary Arteriograms: A Variant of the Variant". ... referring to it as a variant form of classical angina pectoris. Consequently, this angina has come to be reported and referred ... Angina due to coronary vasospasm is also known as variant angina. Hung, Ming-Jui; Hu, Patrick; Hung, Ming-Yow (2014). "Coronary ...
"Angina pectoris I. A variant form of angina pectoris". The American Journal of Medicine. 27 (3): 375-88. doi:10.1016/0002-9343( ... "Variant Angina". www.escardio.org. Retrieved 2 April 2018. "Prinzmetal's or Prinzmetal Angina, Variant Angina and Angina ... Variant angina, also known as Prinzmetal angina, vasospastic angina, angina inversa, coronary vessel spasm, or coronary artery ... Treatment of the Kounis syndrome very much differs from that for variant angina. Angina pectoris: the most common form of ...
... myocardial infarction and angina pectoris).[citation needed] A variant of this sign which uses the entire palm instead of the ...
Variant angina Yeghiazarians Y, Braunstein JB, Askari A, Stone PH (January 2000). "Unstable angina pectoris". N. Engl. J. Med. ... Unstable angina (UA), also called crescendo angina, is a type of angina pectoris that is irregular. It is also classified as a ... The pathophysiology of unstable angina is controversial. Until recently, unstable angina was assumed to be angina pectoris ... Murrell, William (1879). "Nitroglycerin as a remedy for angina pectoris". The Lancet. 1: 80-81, 113-115, 151-152, 225-227. doi: ...
... angina pectoris MeSH C23.888.646.215.500.150 - angina, unstable MeSH C23.888.646.215.500.150.150 - angina pectoris, variant ...
Hypertension Angina pectoris (with the exception of variant angina) Myocardial infarction Tachycardia (and other sympathetic ... and to prevent further heart problems in those with angina or previous heart attacks. It can be taken by mouth or by injection ...
... angina pectoris MeSH C14.280.647.250.125.150 - angina, unstable MeSH C14.280.647.250.125.150.150 - angina pectoris, variant ... angina pectoris MeSH C14.907.553.470.250.125.130 - angina pectoris, variant MeSH C14.907.553.470.250.125.150 - angina, unstable ... angina pectoris, variant MeSH C14.280.647.500 - myocardial infarction MeSH C14.280.647.500.375 - myocardial stunning MeSH ... angina pectoris, variant MeSH C14.907.553.470.500 - myocardial infarction MeSH C14.907.553.470.500.375 - myocardial stunning ...
Kounis syndrome (allergic angina and allergic myocardial infarction). Angina Pectoris. Etiology, Pathogenesis and Treatment. ... Elevation of plasma histamine concentration in the coronary circulation in patients with variant angina. Am J Cardiol 1996;77: ... Tryptase levels are elevated during spontaneous ischemic episodes in unstable angina but not after ergonovine test in variant ... "Kounis syndrome (allergic angina and allergic myocardial infarction): A natural paradigm?" Kounis NG. International Journal of ...
An antianginal is a drug used in the treatment of angina pectoris, a symptom of ischaemic heart disease. Drugs used are ... They are contraindicated in variant angina and can precipitate heart failure. They are also contraindicated in severe ... Exertional Angina: In patients with exertional angina, NORVASC reduces the total peripheral resistance (afterload) against ... and most effectively in the treatment of variant angina (directly preventing coronary artery vasospasm). They are not used in ...
... the study group in this case consisted of patients who had been diagnosed with unstable angina pectoris; whether elevated CRP ... a 2008 study compared people with various genetic CRP variants. Those with a high CRP due to genetic variation had no increased ... can be used for clinical decision-making without adjustment for CRP gene variants.[citation needed] Increased blood CRP levels ...
He died at Lynn on 13 September 1818 of angina pectoris, and was buried alongside his wife in the General Baptist Burial Ground ... and by topographical and statistical information, with accounts of the religious houses formerly in Lynn, and of the progress ...
During this period, treatments were also prescribed for complex ailments, including angina pectoris, diabetes, hypertension, ... Hoernle identified the scribe of the medical portions of the manuscript to be a native of India using a northern variant of the ... emphasis to practice found in variants in the West. Exposure to European developments in medicine from the nineteenth century ...
Angina Pectoris-Myocardial Infarction Investigations in Japan". Journal of the American College of Cardiology. 38 (1): 11-8. ... About 2.2% of TTS cases had the reversed (basal) variant. Recurrence rate of TTS is about 1.8% per-patient year. Rees, et al. ... a new variant". Journal of the American College of Cardiology. 48 (3): 579-583. doi:10.1016/j.jacc.2006.06.015. PMID 16875987. ...
Unstable angina (UA) (also "crescendo angina"; this is a form of acute coronary syndrome) is defined as angina pectoris that ... A variant form of angina-Prinzmetal's angina-occurs in patients with normal coronary arteries or insignificant atherosclerosis ... In some cases, angina can be quite severe. Worsening angina attacks, sudden-onset angina at rest, and angina lasting more than ... Angina, also known as angina pectoris, is chest pain or pressure, usually caused by insufficient blood flow to the heart muscle ...
... is created 1st Baron Leighton in the peerage of the United Kingdom one day before his death in London of angina pectoris. ... English topographical artist, lithographer and photographer (born 1814) April 16 - Viktor Oskar Tilgner, Austrian sculptor ( ...
In 1882 he was diagnosed with what was called "angina pectoris" which then meant coronary thrombosis and disease of the heart. ... The last word was the only variant of "evolved" in the first five editions of the book. "Evolutionism" at that time was ... and a Malthusian Nature selecting from chance variants so that "every part of newly acquired structure is fully practical and ...
1966). "The prophylactic value of propranolol in angina pectoris". Am J Cardiol. 18 (3): 370-83. doi:10.1016/0002-9149(66)90056 ... 1985). Frequencies of hemoglobin variants: thalassemia, the glucose-6-phosphate dehydrogenase deficiency, G6PD variants, and ... beta-blocker propranalol's effectiveness in headache treatment was a chance finding in patients receiving the drug for angina ( ...
... leading in many cases to angina pectoris, myocardial infarction (MI), and coronary death. Recently the Framingham studies have ... Apr 2006). "A common genetic variant is associated with adult and childhood obesity". Science. 312 (5771): 279-83. Bibcode: ... and revealed a genetic variant associated with obesity. The researchers were able to replicate this particular result in four ...
... is used for hypertension in the United States, Canada, and Europe, and also for angina pectoris outside the United ... Jasper JR, Kosaka A, To ZP, Chang DJ, Eglen RM (1997). "Cloning, expression and pharmacology of a truncated splice variant of ... Pindolol has intrinsic sympathomimetic activity and is therefore used with caution in angina pectoris. Pindolol is a first ...
The most critical nondental source is the radiation of angina pectoris into the lower teeth and the potential need for urgent ... A variant of the periodontal abscess is the gingival abscess, which is limited to the gingival margin, has a quicker onset, and ... and angina pectoris (which classically refers pain to the lower jaw). Very rarely, toothache can be psychogenic in origin.: 57- ... or angina pectoris, which can cause pain in the lower teeth. Correct diagnosis can sometimes be challenging. Proper oral ...
He further elucidated the mechanisms by which coronary artery spasm occurs in variant angina after moving to London. While at ... Grand Prix scientifique de la Fondation Lefoulon-Delalande for clinical investigations of vasomotor function in angina pectoris ... He also demonstrated the role of coronary artery spasm in variant angina. His proof, using double crossover studies, that ... but the two factors interacted in a different way than was observed in variant angina. In other studies, he demonstrated that ...
... angina pectoris), to minimise heart muscle damage when an artery is completely occluded (myocardial infarction), or to prevent ... They may range from the relatively minor (e.g. patent foramen ovale, arguably a variant of normal) to serious life-threatening ... Severe narrowings may cause chest pain (angina) or breathlessness during exercise or even at rest. The thin covering of an ... Medications to improve angina symptoms include nitroglycerin, beta blockers, and calcium channel blockers, while preventative ...
March 1998). "Electrical stimulation versus coronary artery bypass surgery in severe angina pectoris: the ESBY study". ... The non-invasive variant is traditional pharmacotherapy, e.g. swallowing a tablet. Intrathecal drug delivery systems (ITDS, ... and a prescription variant of TENS, transcutaneous afferent patterned stimulation (TAPS) Electroconvulsive therapy (ECT) ... peripheral vascular disease and angina. The general process for spinal cord stimulation involves a temporary trailing of ...
Cardiac syndrome X is chest pain (angina pectoris) and chest discomfort in people who do not show signs of blockages in the ... Other genome-wide studies have identified a firm risk variant on chromosome 9 (9p21.3). However, these and other loci are found ... Tarkin, Jason M; Kaski, Juan Carlos (February 2013). "Pharmacological treatment of chronic stable angina pectoris". Clinical ... Types include stable angina, unstable angina, myocardial infarction, and sudden cardiac death. A common symptom is chest pain ...
C in preventing atherosclerosis and published three case reports on the use of lysine and vitamin C to relieve angina pectoris ... It was not, however, the first demonstration that variant forms of hemoglobin could be distinguished by electrophoresis, which ...
In 1847, Ascanio Sobrero invented nitroglycerine to treat angina pectoris and it turned out to be a much more powerful ... such as the MC-3 and PMN-3 variants of the PMN mine. Anti-handling devices can be found on both anti-personnel mines and anti- ...
Nocedal died due to angina pectoris, Fernández Escudero 2012, p. 419 Fernández Escudero 2012, p. 422 José Sanchez Marco, Benito ... In even more extreme variants of the power-greedy-Ramón theory, Nocedal and El Siglo Futuro are presented as puppets ... Both parties present their variants here: according to the Carlists, Nocedal intended to disfigure the party into "acción ...
... pectus excavatum) or protrusion (pectus carinatum) of the sternum, abnormal joint flexibility, a high-arched palate with ... A heart murmur, abnormal reading on an ECG, or symptoms of angina can indicate further investigation. The signs of ... Marfanoid-progeroid-lipodystrophy syndrome (MPL), also referred to as Marfan lipodystrophy syndrome (MFLS), is a variant of MFS ... Pectus carinatum deformity = 2 (pectus excavatum or chest asymmetry = 1) Hindfoot deformity = 2 (plain pes planus = 1) Dural ...
... by skilled bead crafters Angina pectoris: The condition was named "hritshoola" in ancient India and was described by Sushruta ( ... A variant of this game, called Ludo, made its way to England during the British Raj. Mallakhamba: It is a traditional sport, ... Chess spread throughout the world and many variants of the game soon began taking shape. This game was introduced to the Near ... Akasam astapadam was an ashtapada variant played with no board, literally "astapadam played in the sky". A correspondent in the ...
Variant angina - it is effective owing to its direct effects on coronary dilation. Unstable angina (preinfarction, crescendo ... "Long-acting diltiazem HCl for the chronotherapeutic treatment of hypertension and chronic stable angina pectoris". Expert ... Each of these effects results in reduced oxygen consumption by the heart, reducing angina, typically unstable angina, symptoms ... Diltiazem is indicated for: Stable angina (exercise-induced) - diltiazem increases coronary blood flow and decreases myocardial ...
Hypertriglyceridaemia Sepsis Cardiac failure Angina pectoris Arrhythmia Atrial fibrillation Tachycardia Dyspepsia (indigestion ... Infobox drug with local INN variant, Drugs with non-standard legal status, ECHA InfoCard ID from Wikidata, Drug has EMA link, ...
He died of angina pectoris on 20 July 1912 at the Tor-na-Coille Hotel in Banchory, Banchory, survived by his wife. He was ... Three Hundred and Forty-Five Variants of Cinderella, Catskin and, Cap O' Rushes, Abstracted and Tabulated with a Discussion of ...
Yu C, Ji K, Cao H, Wang Y, Jin HH, Zhang Z, Yang G (March 2015). "Effectiveness of acupuncture for angina pectoris: a ... There is a range of acupuncture variants which originated in different philosophies, and techniques vary depending on the ... angina pectoris, ankle sprain, asthma, attention deficit hyperactivity disorder, autism, baby colic, Bell's palsy, cardiac ...
Angina is a common presenting symptom (typically, chest pain) among patients with coronary artery disease. ... Angina pectoris is the result of myocardial ischemia caused by an imbalance between myocardial blood supply and oxygen demand. ... Angina decubitus (a variant of angina pectoris that occurs at night while the patient is recumbent) may occur. ... encoded search term (Angina Pectoris) and Angina Pectoris What to Read Next on Medscape ...
Angina pectoris (AP) represents the clinical syndrome occurring when myocardial oxygen demand exceeds supply. The term is ... A pain-free variant of angina-sometimes referred to as silent chest pain-also exists. These patients can present with ... encoded search term (Angina Pectoris in Emergency Medicine) and Angina Pectoris in Emergency Medicine What to Read Next on ... Angina Pectoris in Emergency Medicine Clinical Presentation. Updated: Jan 25, 2021 * Author: Marc D Haber, MD; Chief Editor: ...
Angina is a type of chest discomfort due to poor blood flow through the blood vessels of the heart muscle. This article ... Variant angina - discharge; Angina pectoris - discharge; Accelerating angina - discharge; New-onset angina - discharge; Angina- ... unstable - discharge; Progressive angina - discharge; Angina-stable - discharge; Angina-chronic - discharge; Angina-variant - ... Angina pectoris and stable ischemic heart disease. In: Goldman L, Schafer AI, eds. Goldman-Cecil Medicine. 26th ed. ...
... or simply angina--is chest pain or discomfort that keeps coming back. It happens when some part of your heart does not get ... Variant angina pectoris. This is also called Prinzmetal angina. It is rare. It occurs almost only at rest, not after exercise ... Angina Pectoris. What is angina pectoris?. Angina pectoris is chest pain or discomfort that occurs when a part of your heart ... Can angina pectoris be prevented?. Keeping up a healthy lifestyle can help to delay or prevent angina pectoris. A healthy ...
Decrease in plasma adiponectin concentrations in patients with variant angina pectoris. Soon Jun Hong, Hong Seog Seo, Woon Rha ... Dive into the research topics of Decrease in plasma adiponectin concentrations in patients with variant angina pectoris. ...
... other than angina pectoris, two main forms of stable angina exist.. *Variant angina: Variant angina pectoris, also called ... What Is Angina Pectoris?. Angina pectoris is a heart condition commonly called stable angina. According to MD News, a clinical ... Mental Effects of Angina Pectoris. Angina pectoris is a symptom of a disease leading to much greater cause for concern. Chronic ... Symptoms of Angina Pectoris. Angina pectoris often presents as an uncomfortable fullness in the chest. Some people describe the ...
Angina Pectoris C14.280.647.250.125 C14.280.647.187. C14.907.553.470.250.125 C14.907.585.187. Angina Pectoris, Variant C14.280. ... Angina, Unstable C14.280.647.250.125.150 C14.280.647.187.150. C14.907.553.470.250.125.150 C14.907.585.187.150. Angioplasty, ... Microvascular Angina C14.280.647.250.125.575 C14.280.647.187.575. C14.907.553.470.250.125.575 C14.907.585.187.575. Midline ...
Chest pain from angina can mimic a heart attack so its important to assess your condition and get the appropriate diagnosis ... Angina diagnosis is accomplished through tests like EKG, chest X-ray, and blood tests. ... "Microvascular Angina." American Heart Association. 19 Mar 2013. "Prinzmetals Angina, Variant Angina and Angina Inversa." ... What is angina?. Angina (also termed angina pectoris) is a condition characterized by chest pain, sometimes severe, that is ...
There are two other forms of angina pectoris. One, long recognized but quite rare, is called Prinzmetals or variant angina. ... It is important to distinguish between the typical stable pattern of angina and "unstable" angina. Angina pectoris often recurs ... Angina pectoris ("angina") is recurring pain or discomfort in the chest that happens when some part of the heart does not ... Does angina mean a heart attack is about to happen?. An episode of angina is not a heart attack. Angina pain means that some of ...
... , Prinzmetals Angina, Variant Angina, Variant Angina Pectoris, Coronary Artery Vasospasm. ... angina, angina pectoris; variant, variant; angina, Angina pectoris, variant, Prinzmetal-angina, Variant angina pectoris. ... Angina, Prinzmetals, Prinzmetal Angina, ANGINA VARIANT ,PRINZMETAL,, Variant Angina Pectoris, Variant Angina, variant angina ... variant angina, Angina, Prinzmetal, VARIANT ANGINA, Prinzmetal angina, Prinzmetals angina, Variant angina pectoris, Variant ...
... heart failure or stable angina pectoris, was not included in our analysis. While these variants of CVD frequently do not ... CVDs or events not requiring hospitalisation, such as stable angina pectoris, heart failure without hospitalisation or ...
... angina pectoris, silent ischemia, history of MI). Coronary artery vasospasm (eg, Prinzmetals variant angina). Other ... Reevaluate if angina or ischemia symptoms (eg, ischemic bowel syndrome, Raynauds syndrome) occur, or if no response after 1st ...
Ostler W. The Lumleian lectures on angina pectoris. Lancet. 1910;1:697-702,839-844. ... The difference between the A1 and A2 type beta-casein variants is a single amino acid substitution at the 67th residue of the ... The role of beta-casein variants in the induction of insulin-dependent diabetes in the non-obese diabetic mouse and humans. ... Status of milk protein, ß-casein variants among Indian milch animals. Ind J Anim Sci. 2009;79(7):722-725. ...
Dihydroergotamine mesylate nasal spray should not be given to patients with ischemic heart disease (angina pectoris, history of ... be evaluated for the presence of CAD or a predisposition to variant angina before receiving additional doses. Similarly, ... patients who have clinical symptoms or findings consistent with coronary artery vasospasm including Prinzmetals variant angina ... patients who experience signs or symptoms suggestive of angina following its administration should, therefore, ...
The findings showed very low evidence to support the use of acupuncture for improving angina symptoms and ECG of angina ... with ineffectiveness of angina relief was less in the combined acupuncture-anti-angina treatment group than in the anti-angina ... and the effectiveness of a combination of acupuncture plus anti-angina medicines was compared to anti-angina medicines alone. ... The study included randomized controlled trials that the effectiveness of acupuncture alone was compared to anti-angina ...
Variant Angina Pectoris Medicine & Life Sciences 100% * Spasm Medicine & Life Sciences 67% ... Facilitation of coronary spasm by propranolol in Prinzmetals angina: Fact or unproven extrapolation? Coronary Artery Disease. ... Facilitation of coronary spasm by propranolol in Prinzmetals angina : Fact or unproven extrapolation? / De Cesare, N.; Cozzi, ... Facilitation of coronary spasm by propranolol in Prinzmetals angina : Fact or unproven extrapolation?. In: Coronary Artery ...
Variant angina pectoris. This is also called Prinzmetal angina. It is rare. It occurs almost only at rest, not after exercise ... Angina Pectoris. in Health Library, In Full Stride. Angina Pectoris. What is angina pectoris?. Angina pectoris is chest pain or ... Can angina pectoris be prevented?. Keeping up a healthy lifestyle can help to delay or prevent angina pectoris. A healthy ... What causes angina pectoris?. Angina pectoris occurs when your heart muscle (myocardium) does not get enough blood and oxygen. ...
Angina is a common presenting symptom (typically, chest pain) among patients with coronary artery disease. ... Angina pectoris is the result of myocardial ischemia caused by an imbalance between myocardial blood supply and oxygen demand. ... Angina decubitus (a variant of angina pectoris that occurs at night while the patient is recumbent) may occur. ... encoded search term (Angina Pectoris) and Angina Pectoris What to Read Next on Medscape ...
Congestive heart failure or angina pectoris; myocardial infarction within 3 months before enrolment; uncontrolled arterial ... Variant of unknown significanceor Variant, favor polymorphism or benign polymorphism etc) ... Non defective BRCA status or BRCA 1 and/or BRCA2 mutations that are considered to be non detrimental (e.g.Variants of ...
Angina, Prinzmetal Angina, Prinzmetals Prinzmetal Angina Prinzmetals Angina Prinzmetals Angina Variant Angina Pectoris ... Angina, Prinzmetal. Angina, Prinzmetals. Prinzmetal Angina. Prinzmetals Angina. Prinzmetals Angina. Variant Angina Pectoris. ... Angina Pectoris (1966-1976). Public MeSH Note:. 84; was see under CORONARY VASOSPASM 1982-83, was see under ANGINA PECTORIS ... angina de Prinzmetal angina de pecho variable angina variante angina vasoespástica Scope note:. Síndrome clínico caracterizado ...
Angina Pectoris C14.280.647.250.125 C14.280.647.187. C14.907.553.470.250.125 C14.907.585.187. Angina Pectoris, Variant C14.280. ... Angina, Unstable C14.280.647.250.125.150 C14.280.647.187.150. C14.907.553.470.250.125.150 C14.907.585.187.150. Angioplasty, ... Microvascular Angina C14.280.647.250.125.575 C14.280.647.187.575. C14.907.553.470.250.125.575 C14.907.585.187.575. Midline ...
Angina Pectoris C14.280.647.250.125 C14.280.647.187. C14.907.553.470.250.125 C14.907.585.187. Angina Pectoris, Variant C14.280. ... Angina, Unstable C14.280.647.250.125.150 C14.280.647.187.150. C14.907.553.470.250.125.150 C14.907.585.187.150. Angioplasty, ... Microvascular Angina C14.280.647.250.125.575 C14.280.647.187.575. C14.907.553.470.250.125.575 C14.907.585.187.575. Midline ...
Angina Pectoris C14.280.647.250.125 C14.280.647.187. C14.907.553.470.250.125 C14.907.585.187. Angina Pectoris, Variant C14.280. ... Angina, Unstable C14.280.647.250.125.150 C14.280.647.187.150. C14.907.553.470.250.125.150 C14.907.585.187.150. Angioplasty, ... Microvascular Angina C14.280.647.250.125.575 C14.280.647.187.575. C14.907.553.470.250.125.575 C14.907.585.187.575. Midline ...
Angina Pectoris C14.280.647.250.125 C14.280.647.187. C14.907.553.470.250.125 C14.907.585.187. Angina Pectoris, Variant C14.280. ... Angina, Unstable C14.280.647.250.125.150 C14.280.647.187.150. C14.907.553.470.250.125.150 C14.907.585.187.150. Angioplasty, ... Microvascular Angina C14.280.647.250.125.575 C14.280.647.187.575. C14.907.553.470.250.125.575 C14.907.585.187.575. Midline ...
Angina Pectoris C14.280.647.250.125 C14.280.647.187. C14.907.553.470.250.125 C14.907.585.187. Angina Pectoris, Variant C14.280. ... Angina, Unstable C14.280.647.250.125.150 C14.280.647.187.150. C14.907.553.470.250.125.150 C14.907.585.187.150. Angioplasty, ... Microvascular Angina C14.280.647.250.125.575 C14.280.647.187.575. C14.907.553.470.250.125.575 C14.907.585.187.575. Midline ...
Angina Pectoris C14.280.647.250.125 C14.280.647.187. C14.907.553.470.250.125 C14.907.585.187. Angina Pectoris, Variant C14.280. ... Angina, Unstable C14.280.647.250.125.150 C14.280.647.187.150. C14.907.553.470.250.125.150 C14.907.585.187.150. Angioplasty, ... Microvascular Angina C14.280.647.250.125.575 C14.280.647.187.575. C14.907.553.470.250.125.575 C14.907.585.187.575. Midline ...
Angina Pectoris C14.280.647.250.125 C14.280.647.187. C14.907.553.470.250.125 C14.907.585.187. Angina Pectoris, Variant C14.280. ... Angina, Unstable C14.280.647.250.125.150 C14.280.647.187.150. C14.907.553.470.250.125.150 C14.907.585.187.150. Angioplasty, ... Microvascular Angina C14.280.647.250.125.575 C14.280.647.187.575. C14.907.553.470.250.125.575 C14.907.585.187.575. Midline ...
  • This is also called Prinzmetal angina. (ahealthyme.com)
  • Although there are several types of angina (unstable, stable, variant [Prinzmetal], and microvascular), it is most important to determine if you have unstable angina because it is the one type that is most likely to require emergency medical treatment. (medicinenet.com)
  • Angina pectoris might occur throughout the presence of regular coronary arteries as thirteen Prinzmetal or variant angina. (lovina.ps)
  • In 1959, Prinzmetal and colleagues described a "variant type of angina" that occurred frequently at rest or during normal activity and was not worsened by effort as classic Heberden angina pectoris is. (ochsnerjournal.org)
  • Variant, or prinzmetal, angina is caused by a spasm in a coronary artery which causes the artery to temporarily narrow, reducing blood flow to your heart and causing chest pain. (cardiology-doctors.com)
  • Angiographic findings lead to the diagnosis of coronary artery spasm (CAS) or Prinzmetal angina. (medscape.com)
  • This syndrome was named Prinzmetal angina or variant angina. (medscape.com)
  • Consequently, the symptoms of unstable angina are important to know because they distinguish unstable angina from the other types that can be diagnosed under non-emergent conditions. (medicinenet.com)
  • If a presumptive diagnosis of unstable angina is made, several tests may be done sequentially to either confirm the diagnosis or actually treat the cause ( coronary artery disease ). (medicinenet.com)
  • Symptoms for the other types of angina are not listed here because the emphasis of angina diagnosis should be on identifying (and treating) unstable angina. (medicinenet.com)
  • However, like unstable angina, the symptoms help only to presumptively identify the angina type. (medicinenet.com)
  • Like the tests for unstable angina, EKGs, blood tests, and occasionally PCI may be used to diagnose the other types of angina. (medicinenet.com)
  • If unstable angina is not the diagnosis, usually the next test done is exercise stress testing ( walking on a treadmill while monitoring heart activity). (medicinenet.com)
  • However, if you or someone you know develops symptoms of unstable angina, you should call 9-1-1. (medicinenet.com)
  • Angina, consisting of stable and unstable angina, is a common symptom of coronary artery atherosclerotic disease [ 2 ]. (biomedcentral.com)
  • The American College of Cardiology and American Heart Association (ACC/AHA) guidelines [ 3 , 8 ] recommend anti-ischemic therapies and/or anti-platelet/anti-coagulation therapies for unstable angina and to control necessary risk factors for stable angina. (biomedcentral.com)
  • Unstable angina is a condition in which your heart doesn't get enough blood flow and oxygen. (stlukes-stl.com)
  • Dosage can be increased at daily (eg, patients with unstable angina) or weekly intervals until optimum clinical response is obtained. (treatmentoptions4u.com)
  • 2. Local plated aggregation in the coronary arteries has been documented in patients with unstable angina pectoris and in patients who undergo sudden cardiac death. (solutionpharmacy.in)
  • Thrombi are identified most often in patients who have suffered a myocardial infarct involving the full thickness of the myocardium although they may also occur in patients with other clinical manifestations of cardiac ischemia, such as unstable angina pectoris. (solutionpharmacy.in)
  • Variant angina pectoris, unstable angina or recent acute coronary syndrome including myocardial infarction (in the past three months). (keystoneheart.com)
  • Unstable angina may not, and it could get worse. (webmd.com)
  • Unstable angina can also be brought on by blood clots that block or partially block your heart's blood vessels. (cardiology-doctors.com)
  • It is contraindicated in patients with unstable angina, shock or haemodynamically unstable heart failure after acute myocardial infarction. (mmhealth.uk)
  • Felodipine is contraindicated in patients with uncontrolled heart failure or unstable angina. (mmhealth.uk)
  • What are the symptoms of angina pectoris? (ahealthyme.com)
  • Some individuals using medical cannabis could experience less severity in their symptoms of angina pectoris because of the increased blood flow to the area. (marijuanadoctors.com)
  • What Side Effects and Symptoms of Angina Pectoris Can Medical Marijuana Treat? (marijuanadoctors.com)
  • Microvascular angina. (ahealthyme.com)
  • Although this is the classic definition of angina, there are important variations in symptoms and types (stable, unstable, variant, and microvascular). (medicinenet.com)
  • In microvascular angina, spasms within the walls of arterial blood vessels cause reduced blood flow to the heart muscle. (cardiology-doctors.com)
  • Methylenetetrahydrofolate dehydrogenase 1 polymorphisms modify the associations of plasma glycine and serine with risk of acute myocardial infarction in patients with stable angina pectoris in WENBIT (Western Norway B Vitamin Intervention Trial). (uib.no)
  • B vitamin treatments modify the risk of myocardial infarction associated with a MTHFD1 polymorphism in patients with stable angina pectoris. (uib.no)
  • Amlodipine is also licensed for the prophylaxis of stable angina pectoris and is used either as monotherapy or in combination with other antianginal drugs in patients with angina that is refractory to nitrates and/or adequate doses of beta-blockers. (mmhealth.uk)
  • Felodipine m/r is currently licensed for the management of hypertension and the prophylaxis of stable angina pectoris. (mmhealth.uk)
  • Angina pectoris is the result of myocardial ischemia caused by an imbalance between myocardial blood supply and oxygen demand. (medscape.com)
  • Of note, while the reproducibility of chest wall pain with palpation may lower the likelihood of angina, this alone cannot rule out angina or myocardial infarction . (medscape.com)
  • Throughout the screening process, we excluded studies which did not include women or participants with CHD or its subtypes (e.g., myocardial infarction, angina pectoris), or did not perform a neuropsychological - or variant - assessment. (onlinecjc.ca)
  • A variant form of angina pectoris caused by coronary artery vasospasm, usually occurring spontaneously and frequently associated with ST segment elevation. (fpnotebook.com)
  • Conclusion: These results do not support the facilitation of coronary spasm by propranolol in Prinzmetal's angina and support the hypothesis that the contractility of coronary vessels in patients with this form of angina is different from that in the classic form. (elsevier.com)
  • 1 This variant form of angina-a type of vasospastic angina in which spasm of an epicardial coronary artery leads to myocardial ischemia with representative ST segment changes-is relatively uncommon, with a higher prevalence in eastern countries. (ochsnerjournal.org)
  • This form of angina only lasts a short time (typically 5 minutes or less) and disappears at rest or upon taking angina medications. (cardiology-doctors.com)
  • This form of angina is caused when fat-containing deposits (plaques) in your blood vessels rupture and blood clots form, quickly blocking or reducing flow through a narrowed artery, and suddenly and severely decreasing blood flow from your heart muscle. (cardiology-doctors.com)
  • Verapamil is used alone or together with other medicines to treat heart rhythm problems, severe chest pain (angina), or high blood pressure (hypertension). (treatmentoptions4u.com)
  • It is usually used in the treatment of hypertension and angina pectoris. (mimaki-family-japan.com)
  • In special cases (variant angina pectoris, severe arterial hypertension), the dose can be increased to 30 mg 3-4 times / day for a short time. (favourite-pharmacy.com)
  • The UK Specialist Pharmacy Service has just published an updated comparison of amlodipine and felodipine for the treatment of hypertension or prophylaxis of stable angina. (mmhealth.uk)
  • Published data on switching between amlodipine and felodipine in patients with hypertension or angina are limited to a few retrospective observational studies. (mmhealth.uk)
  • Angina decubitus (a variant of angina pectoris that occurs at night while the patient is recumbent) may occur. (medscape.com)
  • Classically, angina presents as substernal chest discomfort that occurs with exertion, but it also may occur at rest. (medscape.com)
  • Angina pectoris is chest pain or discomfort that occurs when a part of your heart doesn't get enough blood and oxygen. (ahealthyme.com)
  • Angina pectoris occurs when your heart muscle (myocardium) does not get enough blood and oxygen. (ahealthyme.com)
  • Or, you may take it as a nose spray or under the tongue when angina occurs. (ahealthyme.com)
  • It usually occurs spontaneously, and unlike typical angina, it nearly always occurs when a person is at rest and does not require physical exertion. (fpnotebook.com)
  • Stable angina is chest pain or discomfort that most often occurs with activity or emotional stress. (stlukes-stl.com)
  • Prinzmetal's Angina, Variant Angina and Angina Inversa. (medicinenet.com)
  • Prinzmetal's angina or coronary artery vasospasm). (fpnotebook.com)
  • Facilitation of coronary spasm by propranolol in Prinzmetal's angina: Fact or unproven extrapolation? (elsevier.com)
  • Whether β- adrenergic blockade facilitates coronary spasm in patients with Prinzmetal's angina is unknown. (elsevier.com)
  • Dive into the research topics of 'Facilitation of coronary spasm by propranolol in Prinzmetal's angina: Fact or unproven extrapolation? (elsevier.com)
  • Angina is a type of chest discomfort due to poor blood flow through the blood vessels of the heart muscle. (medlineplus.gov)
  • To our knowledge, this case is the first report of severe drug-refractory vasospastic angina triggered by pregnancy. (ochsnerjournal.org)
  • Our case provides a cautionary tale that patients with refractory vasospastic angina may want to pursue definitive contraception. (ochsnerjournal.org)
  • Among patients with vasospastic angina, the majority will have single-vessel vasospasm, although data from a multicenter registry of the Japanese Coronary Spasm Association suggest that up to 32% of patients can have multivessel territories. (ochsnerjournal.org)
  • Smoking is a major preventable risk factor, with male sex, magnesium deficiency, alcohol use, physical and mental stress, and autonomic nervous system agents all having an association with vasospastic angina. (ochsnerjournal.org)
  • 7 The effects that pregnancy may have on someone with a history of vasospastic angina, particularly patients with intractable vasospastic angina contributing to unstable cardiac arrhythmia, are unknown. (ochsnerjournal.org)
  • Amlodipine can also be used for Prinzmetal's (variant or vasospastic) angina when diagnosed by a cardiologist. (mmhealth.uk)
  • Angina chest pain is usually relieved within a few minutes by resting or by taking prescribed cardiac medicine, such as nitroglycerin. (ahealthyme.com)
  • Angina (also termed angina pectoris ) is a condition characterized by chest pain , sometimes severe, that is caused by an inadequate blood supply to the heart. (medicinenet.com)
  • Variant angina can occur even when you're at rest, and is often severe. (cardiology-doctors.com)
  • The diagnosis of angina begins with a history, including a family health history, risk factors, and a physical exam. (medicinenet.com)
  • Patients with angina are at an increased risk of heart attack compared with those who have no symptoms of cardiovascular disease, but the episode of angina is not a signal that a heart attack is about to happen. (doereport.com)
  • For many patients with angina, the ECG at rest is normal. (doereport.com)
  • A healthcare provider can often diagnose angina from symptoms and how and when they occur. (ahealthyme.com)
  • Episodes of angina occur when the heart's need for oxygen increases beyond the oxygen available from the blood nourishing the heart. (doereport.com)
  • In contrast, when the pattern of angina changes--if episodes become more frequent, last longer, or occur without exercise--the risk of heart attack in subsequent days or weeks is much higher. (doereport.com)
  • This is not surprising because the symptoms of angina occur during stress. (doereport.com)
  • Reevaluate if angina or ischemia symptoms (eg, ischemic bowel syndrome, Raynaud's syndrome) occur, or if no response after 1st dose. (empr.com)
  • Patients should be asked about the frequency of angina, severity of pain, and number of nitroglycerin pills used during episodes. (medscape.com)
  • Although there is a weak relationship between the severity of pain and degree of oxygen deprivation associated in the heart muscle, angina pectoris can be an extremely serious condition that can result in death if not treated properly. (marijuanadoctors.com)
  • Researchers are now studying medical marijuana for angina pectoris for its ability to treat pain, anxiety, fatigue, nausea and other symptoms related to the condition. (marijuanadoctors.com)
  • Usually the doctor can diagnose angina by noting the symptoms and how they arise. (doereport.com)
  • To diagnose angina, your healthcare provider will conduct a physical exam and discuss your symptoms. (cardiology-doctors.com)
  • If your physician diagnosed you with stable angina, the more you know about the condition and its treatment, the better prepared you will be to improve your quality of life while living with it. (marijuanadoctors.com)
  • Patients who are seeking an alternative treatment option in the form of cannabis may find alleviation of the symptoms associated with angina pectoris. (marijuanadoctors.com)
  • Cannabis and angina pectoris treatment is a much safer option than opioids, which can result in dependency and addiction. (marijuanadoctors.com)
  • Marijuana and angina pectoris treatment is a great alternative to conventional nonspecific treatments for fatigue , since it works with your body's natural chemicals to regulate your energy levels. (marijuanadoctors.com)
  • Cannabis and angina pectoris treatment treats anxiety better than other conventional medications. (marijuanadoctors.com)
  • The study included randomized controlled trials that the effectiveness of acupuncture alone was compared to anti-angina medicines (in addition to conventional treatment) and the effectiveness of a combination of acupuncture plus anti-angina medicines was compared to anti-angina medicines alone. (biomedcentral.com)
  • However, no differences were observed between acupuncture treatment alone and anti-angina medicines alone for both outcome measures. (biomedcentral.com)
  • To schedule a consultation with a qualified healthcare provider in Beverly Hills that specializes in angina treatment, call (424) 365-1800 or contact Dr. Jeremy Fischer online . (cardiology-doctors.com)
  • Angina treatment varies and will be dependent on the severity and type of angina you have. (cardiology-doctors.com)
  • There are few comparative clinical studies between amlodipine and felodipine in the treatment of stable angina. (mmhealth.uk)
  • Angina may be a chronic, recurring problem or a sudden, acute health concern, and may be hard to distinguish between other types of chest pain (such as indigestion-induced chest pain). (cardiology-doctors.com)
  • When diagnosing angina, the type of angina is determined. (medicinenet.com)
  • In addition, angina of any type may be considered to be a symptom of coronary artery disease ( CAD ). (medicinenet.com)
  • Angina is a type of ches. (stlukes-stl.com)
  • It has been associated with one particular type of angina pectoris, termed Prinzmental's (variant) angina. (solutionpharmacy.in)
  • The most common type of angina, stable angina is provoked when your heart is pushed to work harder than usual, such as when you climb stairs, exercise or walk (tasks which demand more blood from your heart). (cardiology-doctors.com)
  • ATTR-CM skyldes aflejring i hjertet af amyloid, som stammer fra enten wild type-TTR (wtATTR-CM) som led i, hvad der tidligere blev benævnt senil systemisk amyloidose, eller fra muteret TTR (mATTR-CM) som led i hereditær (familiær eller variant) amyloidose [4]. (ugeskriftet.dk)
  • The goal of this clinical research study is to compare the safety and effectiveness of cabozantinib and sunitinib when given to patients with metastatic (has spread) variant histology renal cell carcinoma (vhRCC), a type of kidney cancer. (mycancergenome.org)
  • Angina feels like a pressing or squeezing pain, usually in the chest under the breast bone, but sometimes in the shoulders, arms, neck, jaws, or back. (doereport.com)
  • There have been several studies showing the therapeutic benefits of marijuana for angina pectoris. (marijuanadoctors.com)
  • Variant angina and sleep: a case report with therapeutic considerations. (ouhsc.edu)
  • Anything that causes your heart muscle to need more blood or oxygen can result in angina, especially if you already have a blockage or narrowing. (ahealthyme.com)
  • Angina causes reduced blood flow to your heart muscle, leading to your heart receiving insufficient oxygen. (cardiology-doctors.com)
  • That said, no definitive examination findings suggest angina. (medscape.com)
  • Unfortunately, no examination findings are pathognomonic for angina pectoris. (medscape.com)
  • The findings showed very low evidence to support the use of acupuncture for improving angina symptoms and ECG of angina patients. (biomedcentral.com)
  • Changes in your angina may mean your heart disease is getting worse. (medlineplus.gov)
  • Patients suffering from angina pectoris may experience increased blood flow and a slight decrease in heart rate after using medical marijuana. (marijuanadoctors.com)
  • Angina pectoris ("angina") is recurring pain or discomfort in the chest that happens when some part of the heart does not receive enough blood. (doereport.com)
  • Does angina mean a heart attack is about to happen? (doereport.com)
  • An episode of angina is not a heart attack. (doereport.com)
  • Angina pain means that some of the heart muscle in not getting enough blood temporarily--for example, during exercise, when the heart has to work harder. (doereport.com)
  • Episodes of angina seldom cause permanent damage to heart muscle. (doereport.com)
  • When someone has a repeating but stable pattern of angina, an episode of angina does not mean that a heart attack is about to happen. (doereport.com)
  • Angina means that there is underlying coronary heart disease. (doereport.com)
  • Not all chest pain is from the heart, and not all pain from the heart is angina. (doereport.com)
  • Angina is chest pain that happens because there isn't enough blood going to part of your heart . (webmd.com)
  • Angina usually happens because of heart disease. (webmd.com)
  • Angina pectoris, or simply angina, refers to chest pain resulting from reduced blood flow to the heart muscle, and is a symptom of an underlying heart problem like coronary artery disease , a serious health threat (and the leading cause of death in the United States in women and men) that causes the arteries that supply blood to the heart to harden and narrow with plaque. (cardiology-doctors.com)
  • For example, if the pain lasts for less that 30 seconds or if it goes away during a deep breath, after drinking a glass of water, or by changing position, it almost certainly is NOT angina and should not cause concern. (doereport.com)
  • Angina can be a symptom of coronary artery disease (CAD). (ahealthyme.com)
  • Methods: Using quantitative angiography, we evaluated the response of normal and narrowed coronary arteries to intravenous propranolol, a cold pressor test (an α-stimulus), and the combination of the two in 15 patients with Prinzmetal's (group 1) and in 19 with classic (group 2) angina. (elsevier.com)
  • People who have angina often receive the medicines below. (medlineplus.gov)
  • Your healthcare provider may prescribe medicines if you have angina. (ahealthyme.com)
  • Angina is chest pain, chest pressure, often associated with shortness of breath. (medlineplus.gov)
  • However one or more diagnostic tests may be needed to exclude angina or to establish the severity of the underlying coronary disease. (doereport.com)
  • You may take a long-acting form of nitroglycerin daily to prevent angina. (ahealthyme.com)
  • It is usually relieved within a few minutes by resting or by taking prescribed angina medicine. (doereport.com)
  • Usually, medicine and lifestyle changes can control angina. (webmd.com)