Amnesia
Amnesia, Retrograde
Amnesia, Anterograde
Amnesia, Transient Global
Korsakoff Syndrome
Memory
Alcohol Amnestic Disorder
Retention (Psychology)
Pyrithiamine
Diencephalon
Scopolamine Hydrobromide
Neuropsychological Tests
Confusion
Unconsciousness
Temporal Lobe
Autobiography as Topic
Hippocampus
Thalamic Diseases
Memory Disorders
Midazolam
Craniocerebral Trauma
Learning
Anisomycin
Paired-Associate Learning
Mamillary Bodies
Thiamine Deficiency
Preanesthetic Medication
Recognition (Psychology)
Anterior Thalamic Nuclei
Multiple Personality Disorder
Magnetic Resonance Imaging
Brain Damage, Chronic
Cognitive recovery after severe head injury. 3. WAIS verbal and performance IQs as a function of post-traumatic amnesia duration and time from injury. (1/690)
Two studies are reported are reported in which severely head-injured patients were followed up and Verbal (VIQ) and Performance (PIQ) IQs obtained on the Wechsler Adult Intelligence Scale at four intervals after injury. In the first study 51 patients were systematically followed, and results were based upon serial testing. In the second study results were based on the earliest data available from an additional 98 patients who had not been followed so systematically, in order to introduce a control for the effects of practice. Patients in both studies were categorised into four groups of the severity of head injury based upon duration of post-traumatic amnesia (PTA). In both studies, VIQ level was found to be related to PTA duration at three months after injury, while PIQ was related to PTA duration at both three and six months. No such relationships were found at 12 and 30 months after injury. Results are discussed in the context of previous studies relating the outcome of head injury to the duration of PTA. (+info)Episodic memory in transient global amnesia: encoding, storage, or retrieval deficit? (2/690)
OBJECTIVES: To assess episodic memory (especially anterograde amnesia) during the acute phase of transient global amnesia to differentiate an encoding, a storage, or a retrieval deficit. METHODS: In three patients, whose amnestic episode fulfilled all current criteria for transient global amnesia, a neuropsychological protocol was administered which included a word learning task derived from the Grober and Buschke's procedure. RESULTS: In one patient, the results suggested an encoding deficit, and in two others, a storage deficit. CONCLUSIONS: The encoding/storage impairment concerning anterograde amnesia documented in our patients stands in clear contrast with the impairment in retrieval which must underly the retrograde amnesia that also characterises transient global amnesia. This dissociation in turn favours the idea of a functional independence among the cognitive mechanisms that subserve episodic memory. (+info)Recovery from anterograde and retrograde amnesia after percutaneous drainage of a cystic craniopharyngioma. (3/690)
A case is reported of a cystic craniopharyngioma involving the floor and walls of the third ventricle. Pronounced anterograde and retrograde amnesia were documented preoperatively by formal testing. Rapid improvement in both new learning capacity and remote memory occurred after percutaneous twist drill drainage of the cystic portion of the tumour. The relevance of these observations to the amnesic syndrome and its neuropathological basis is discussed. (+info)Sequence-specific impairment of memory formation by NCAM antisense oligonucleotides. (4/690)
The functional role of NCAM gene expression in memory formation was studied in the one-trial passive avoidance task in day-old chicks by pretraining injections of one of three different 18-mer end-protected oligonucleotides corresponding to positions 190-, 207-, and 332- of the NCAM Ig1 domain. Twenty-four-hour-old chicks were trained by pecking at a bitter-tasting bead and tested for avoidance 30 min, 3, 8, or 24 hr later. Memory retention was significantly reduced only in the group of animals injected with the NCAM antisense corresponding to position 207- (AS-ODN-207), and only if given twice, both immediately after hatching and 12 hr before training. This antisense was without effect on the general behavior of the chicks, training or acquisition, and did not produce observable neurotoxic damage. Under such conditions amnesia was evident by 3 hr after training and lasted until at least 24 hr after training. The two other tested oligonucleotides were without behavioral effect. To control for nonsequence-specific effects of AS-ODN-207, brains from injected and trained animals were processed for Western blotting and probed using anti-NCAM, anti-L1, and anti-actin antibodies. NCAM antisense corresponding to position 207- significantly reduced the level of NCAM, whereas the level of L1 and actin remained unchanged. These results confirm our earlier conclusion that NCAM is necessary for longer term memory retention. (+info)Retrograde temporal order amnesia resulting from damage to the fornix. (5/690)
Some amnesic patients show an impairment of temporal order memory that cannot be accounted for by content memory deficits. The performance of an amnesic patient on memory tasks assessing the patient's content and temporal memories for remotely acquired material is described, after a lesion including the bilateral anterior fornix and adjacent anterior thalamus. The patient displayed a deficit in the temporal order tasks for remotely acquired information. Neither frontal cognitive deficits nor recognition deficits can account for this patient's poor temporal memory. This retrograde temporal order memory impairment without content memory deficits were not seen in previously reported thalamic amnesic patients. Accordingly, the present patient's poor retrograde temporal memory could hardly be explained by only a thalamic lesion. It is concluded that the patient's impairment of temporal order memory for the retrograde material is probably due to the direct disconnection between the frontal lobe and the hippocampus by disruption of the fornix. (+info)The attenuation of learning impairments induced after exposure to CO or trimethyltin in mice by sigma (sigma) receptor ligands involves both sigma1 and sigma2 sites. (6/690)
1. Sigma (sigma) receptor ligands were previously reported to alleviate learning and memory impairments on several pharmacological and pathological rodent models of amnesia. Such effect was demonstrated as involving the sigma1 subtype of sigma receptor. 2. In this study, we characterized the pharmacological effect mediated by sigma ligands on two lesional models of amnesia in mice: (1) the hypoxia-related learning and memory impairment model induced by repeated exposure to carbon monoxide (CO) gas; and (2) the intoxication with trimethyltin (1 mg kg(-1)). 3. The selective sigma1 ligand PRE-084 (1 mg kg(-1)) or the non-selective sigma1/sigma2 compounds DTG (0.1 mg kg(-1)), BD1008 (3 mg kg(-1)), and haloperidol (0.1 mg kg(-1)) reversed significantly the spontaneous alternation deficits observed 7 days after exposure to CO or 14 days after intoxication with trimethyltin. 4. The selective sigma1 receptor antagonist NE-100 (1 mg kg(-1)) was ineffective by itself, but blocked completely the PRE-084 effects, partially the DTG effects, and did not affect the effects induced by BD1008 or haloperidol. 5. A similar pharmacological profile was observed in the step-down type passive avoidance test performed 8 days after exposure to CO. 6. These results show that, in contrast to the previously reported amnesia models, the impairments induced after exposure to CO or intoxication with trimethyltin could be alleviated not only by sigma1 receptor agonists but also by sigma2 agonists. The particular pattern of neurodegeneration observed in these lesional models may explain these differences. (+info)Basal forebrain amnesia: does the nucleus accumbens contribute to human memory? (7/690)
OBJECTIVE: To analyse amnesia caused by basal forebrain lesions. METHODS: A single case study of a patient with amnesia after bleeding into the anterior portion of the left basal ganglia. Neuropsychological examination included tests of attention, executive function, working memory, recall, and recognition of verbal and non-verbal material, and recall from remote semantic and autobiographical memory. The patient's MRI and those of other published cases of basal forebrain amnesia were reviewed to specify which structures within the basal forebrain are crucial for amnesia. RESULTS: Attention and executive function were largely intact. There was anterograde amnesia for verbal material which affected free recall and recognition. With both modes of testing the patient produced many false positive responses and intrusions when lists of unrelated words had been memorised. However, he confabulated neither on story recall nor in day to day memory, nor in recall from remote memory. The lesion affected mainly the nucleus accumbens, but encroached on the inferior limb of the capsula interna and the most ventral portion of the nucleus caudatus and globus pallidus, and there was evidence of some atrophy of the head of the caudate nucleus. The lesion spared the nucleus basalis Meynert, the diagnonal band, and the septum, which are the sites of cholinergic cell concentrations. CONCLUSIONS: It seems unlikely that false positive responses were caused by insufficient strategic control of memory retrieval. This speaks against a major role of the capsular lesion which might disconnect the prefrontal cortex from the thalamus. It is proposed that the lesion of the nucleus accumbens caused amnesia. (+info)Different training procedures recruit either one or two critical periods for contextual memory consolidation, each of which requires protein synthesis and PKA. (8/690)
We have used a combined genetic and pharmacological approach to define the time course of the requirement for protein kinase A (PKA) and protein synthesis in long-term memory for contextual fear conditioning in mice. The time course of amnesia in transgenic mice that express R(AB) and have genetically reduced PKA activity in the hippocampus parallels that observed both in mice treated with inhibitors of PKA and mice treated with inhibitors of protein synthesis. This PKA- and protein synthesis-dependent memory develops between 1 hr and 3 hr after training. By injecting the protein synthesis inhibitor anisomycin or the PKA inhibitor Rp-cAMPs at various times after training, we find that depending on the nature of training, contextual memory has either one or two brief consolidation periods requiring synthesis of new proteins, and each of these also requires PKA. Weak training shows two time periods of sensitivity to inhibitors of protein synthesis and PKA, whereas stronger training exhibits only one. These studies underscore the parallel dependence of long-term contextual memory on protein synthesis and PKA and suggest that different training protocols may recruit a common signaling pathway in distinct ways. (+info)Amnesia is a condition characterized by memory loss, which can be temporary or permanent. It may result from brain damage or disease, and it can affect various aspects of memory, such as the ability to recall past events (retrograde amnesia), the ability to form new memories (anterograde amnesia), or both. Amnesia can also affect a person's sense of identity and their ability to learn new skills.
There are several types of amnesia, including:
1. Anterograde amnesia: This type of amnesia affects the ability to form new memories after an injury or trauma. People with anterograde amnesia may have difficulty learning new information and remembering recent events.
2. Retrograde amnesia: Retrograde amnesia affects the ability to recall memories that were formed before an injury or trauma. People with retrograde amnesia may have trouble remembering events, people, or facts from their past.
3. Transient global amnesia: This is a temporary form of amnesia that usually lasts for less than 24 hours. It is often caused by a lack of blood flow to the brain, and it can be triggered by emotional stress, physical exertion, or other factors.
4. Korsakoff's syndrome: This is a type of amnesia that is caused by alcohol abuse and malnutrition. It is characterized by severe memory loss, confusion, and disorientation.
5. Dissociative amnesia: This type of amnesia is caused by psychological factors, such as trauma or stress. People with dissociative amnesia may have trouble remembering important personal information or events that are emotionally charged.
The treatment for amnesia depends on the underlying cause. In some cases, memory may improve over time, while in other cases, it may be permanent. Treatment may involve medication, therapy, or rehabilitation to help people with amnesia cope with their memory loss and develop new skills to compensate for their memory impairments.
Retrograde amnesia is a form of memory loss where an individual cannot recall information, events, or facts from their personal past before a specific point in time. This type of amnesia is caused by damage to the brain, often as a result of head injury, stroke, infection, or certain medical conditions. The extent and duration of retrograde amnesia can vary widely, depending on the severity and location of the brain injury. In some cases, memory function may return over time as the brain heals, while in other cases the memory loss may be permanent.
Anterograde amnesia is a specific type of memory loss where a person has difficulty forming new memories or learning and retaining new information after the onset of the amnesia. It is often caused by damage to certain parts of the brain, such as the hippocampus and surrounding structures, which play a crucial role in the formation of new memories.
In anterograde amnesia, people may have trouble remembering events that occurred after the onset of their memory impairment, while their ability to recall remote memories or those that were formed before the onset of the amnesia is typically preserved. The severity of anterograde amnesia can vary widely, from mild difficulty with learning new information to a complete inability to form any new memories.
Anterograde amnesia can be caused by various factors, including brain injury, infection, stroke, alcohol or drug abuse, seizures, and certain medical conditions such as Wernicke-Korsakoff syndrome, which is often associated with chronic alcoholism. Treatment for anterograde amnesia depends on the underlying cause and may involve medication, rehabilitation, or other interventions to help improve memory function.
Transient global amnesia (TGA) is a sudden, temporary episode of memory loss that cannot be explained by a more common neurological or medical condition. It is characterized by an inability to form new memories (anterograde amnesia) and to recall past events or personal information (retrograde amnesia). These memory gaps typically last for several hours, but may persist for up to 24 hours.
TGA usually affects people over the age of 50 and is more common in men than women. The exact cause of TGA is not known, although it has been associated with various triggers such as emotional stress, physical exertion, sudden immersion in cold water, sexual activity, and medical procedures.
During a TGA episode, individuals often repeat the same questions or statements due to their inability to form new memories. They may also appear confused or disoriented, but they typically remain conscious and maintain their ability to perform familiar tasks. Most people with TGA do not experience any long-term memory loss or other neurological symptoms after the episode resolves.
The diagnosis of TGA is based on the characteristic symptoms, a normal physical examination, and the exclusion of other potential causes of amnesia through medical testing. Treatment for TGA is generally supportive, focusing on reassuring the individual and providing a safe environment during the episode. Recurrent episodes of TGA are uncommon but can occur in some individuals.
Korsakoff syndrome is a neuropsychiatric disorder typically caused by alcohol abuse, specifically thiamine (vitamin B1) deficiency in the brain. It's often associated with Wernicke encephalopathy, and the two together are referred to as Wernicke-Korsakoff syndrome.
The main features of Korsakoff syndrome include severe memory impairment, particularly anterograde amnesia (inability to form new memories), confabulation (making up stories due to gaps in memory), and a lack of insight into their condition. Other cognitive functions like intelligence and perception are usually preserved.
The syndrome is believed to result from damage to the mammillary bodies and other structures in the diencephalon, particularly the thalamus. Treatment involves abstinence from alcohol, thiamine replacement, and a balanced diet. The prognosis varies but often includes some degree of permanent memory impairment.
In the context of medical and clinical neuroscience, memory is defined as the brain's ability to encode, store, retain, and recall information or experiences. Memory is a complex cognitive process that involves several interconnected regions of the brain and can be categorized into different types based on various factors such as duration and the nature of the information being remembered.
The major types of memory include:
1. Sensory memory: The shortest form of memory, responsible for holding incoming sensory information for a brief period (less than a second to several seconds) before it is either transferred to short-term memory or discarded.
2. Short-term memory (also called working memory): A temporary storage system that allows the brain to hold and manipulate information for approximately 20-30 seconds, although this duration can be extended through rehearsal strategies. Short-term memory has a limited capacity, typically thought to be around 7±2 items.
3. Long-term memory: The memory system responsible for storing large amounts of information over extended periods, ranging from minutes to a lifetime. Long-term memory has a much larger capacity compared to short-term memory and is divided into two main categories: explicit (declarative) memory and implicit (non-declarative) memory.
Explicit (declarative) memory can be further divided into episodic memory, which involves the recollection of specific events or episodes, including their temporal and spatial contexts, and semantic memory, which refers to the storage and retrieval of general knowledge, facts, concepts, and vocabulary, independent of personal experience or context.
Implicit (non-declarative) memory encompasses various forms of learning that do not require conscious awareness or intention, such as procedural memory (skills and habits), priming (facilitated processing of related stimuli), classical conditioning (associative learning), and habituation (reduced responsiveness to repeated stimuli).
Memory is a crucial aspect of human cognition and plays a significant role in various aspects of daily life, including learning, problem-solving, decision-making, social interactions, and personal identity. Memory dysfunction can result from various neurological and psychiatric conditions, such as dementia, Alzheimer's disease, stroke, traumatic brain injury, and depression.
Alcohol Amnestic Disorder is not listed as a separate disorder in the current edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-5), which is used by mental health professionals to diagnose mental conditions. However, it was previously included in earlier editions as a subtype of Amnestic Disorder due to the effects of substance use or exposure to toxins.
Alcohol Amnestic Disorder is characterized by significant memory impairment that is directly caused by alcohol consumption. This disorder can result in anterograde amnesia, which is the inability to form new memories after drinking, and/or retrograde amnesia, which involves forgetting previously learned information or personal experiences.
The diagnosis of Alcohol Amnestic Disorder typically requires a comprehensive medical and neuropsychological evaluation to determine the extent and nature of memory impairment, as well as to rule out other potential causes for cognitive decline. Treatment usually involves a combination of abstinence from alcohol, pharmacotherapy, and psychosocial interventions to address substance use disorder and any co-occurring mental health conditions.
"Mental recall," also known as "memory recall," refers to the ability to retrieve or bring information from your memory storage into your conscious mind, so you can think about, use, or apply it. This process involves accessing and retrieving stored memories in response to certain cues or prompts. It is a fundamental cognitive function that allows individuals to remember and recognize people, places, events, facts, and experiences.
In the context of medical terminology, mental recall may be used to assess an individual's cognitive abilities, particularly in relation to memory function. Impairments in memory recall can be indicative of various neurological or psychological conditions, such as dementia, Alzheimer's disease, or amnesia.
Avoidance learning is a type of conditioning in which an individual learns to act in a certain way to avoid experiencing an unpleasant or aversive stimulus. It is a form of learning that occurs when an organism changes its behavior to avoid a negative outcome or situation. This can be seen in both animals and humans, and it is often studied in the field of psychology and neuroscience.
In avoidance learning, the individual learns to associate a particular cue or stimulus with the unpleasant experience. Over time, they learn to perform an action to escape or avoid the cue, thereby preventing the negative outcome from occurring. For example, if a rat receives an electric shock every time it hears a certain tone, it may eventually learn to press a lever to turn off the tone and avoid the shock.
Avoidance learning can be adaptive in some situations, as it allows individuals to avoid dangerous or harmful stimuli. However, it can also become maladaptive if it leads to excessive fear or anxiety, or if it interferes with an individual's ability to function in daily life. For example, a person who has been attacked may develop a phobia of public places and avoid them altogether, even though this limits their ability to engage in social activities and live a normal life.
In summary, avoidance learning is a type of conditioning in which an individual learns to act in a certain way to avoid experiencing an unpleasant or aversive stimulus. It can be adaptive in some situations but can also become maladaptive if it leads to excessive fear or anxiety or interferes with daily functioning.
Pyrithiamine is not typically considered a medical term, but it is a chemical compound that has been used in scientific research. It's an antivitamin, specifically an analog of thiamine (vitamin B1), which means it can interfere with the metabolism of thiamine in the body.
Here's a more specific definition from a biochemical perspective:
Pyrithiamine is a synthetic organic compound with the formula C6H7N2O2S. It is an analog of thiamine, where the aminomethyl group of thiamine is replaced by a pyridine ring. This structural modification makes pyrithiamine unable to act as a vitamin, but it can still interact with the enzymes and transport proteins involved in thiamine metabolism. As a result, pyrithiamine has been used as a tool to study thiamine deficiency and its effects on various organisms, including mammals.
Please note that pyrithiamine is not a term commonly used in clinical medicine or patient care. If you have any concerns about vitamins, nutrition, or health-related topics, it's best to consult a healthcare professional for accurate information and advice tailored to your specific situation.
The diencephalon is a term used in anatomy to refer to the part of the brain that lies between the cerebrum and the midbrain. It includes several important structures, such as the thalamus, hypothalamus, epithalamus, and subthalamus.
The thalamus is a major relay station for sensory information, receiving input from all senses except smell and sending it to the appropriate areas of the cerebral cortex. The hypothalamus plays a crucial role in regulating various bodily functions, including hunger, thirst, body temperature, and sleep-wake cycles. It also produces hormones that regulate mood, growth, and development.
The epithalamus contains the pineal gland, which produces melatonin, a hormone that helps regulate sleep-wake cycles. The subthalamus is involved in motor control and coordination.
Overall, the diencephalon plays a critical role in integrating sensory information, regulating autonomic functions, and modulating behavior and emotion.
Scopolamine hydrobromide is a synthetic anticholinergic drug, which means it blocks the action of acetylcholine, a neurotransmitter in the nervous system. It is primarily used for its anti-motion sickness and anti-nausea effects. It can also be used to help with symptoms of Parkinson's disease, such as muscle stiffness and tremors.
In medical settings, scopolamine hydrobromide may be administered as a transdermal patch, which is placed behind the ear to allow for slow release into the body over several days. It can also be given as an injection or taken orally in the form of tablets or liquid solutions.
It's important to note that scopolamine hydrobromide can have various side effects, including dry mouth, blurred vision, dizziness, and drowsiness. It may also cause confusion, especially in older adults, and should be used with caution in patients with glaucoma, enlarged prostate, or certain heart conditions.
Neuropsychological tests are a type of psychological assessment that measures cognitive functions, such as attention, memory, language, problem-solving, and perception. These tests are used to help diagnose and understand the cognitive impact of neurological conditions, including dementia, traumatic brain injury, stroke, Parkinson's disease, and other disorders that affect the brain.
The tests are typically administered by a trained neuropsychologist and can take several hours to complete. They may involve paper-and-pencil tasks, computerized tasks, or interactive activities. The results of the tests are compared to normative data to help identify any areas of cognitive weakness or strength.
Neuropsychological testing can provide valuable information for treatment planning, rehabilitation, and assessing response to treatment. It can also be used in research to better understand the neural basis of cognition and the impact of neurological conditions on cognitive function.
Confusion is a state of bewilderment or disorientation in which a person has difficulty processing information, understanding their surroundings, and making clear decisions. It can be caused by various medical conditions such as infections, brain injury, stroke, dementia, alcohol or drug intoxication or withdrawal, and certain medications. Confusion can also occur in older adults due to age-related changes in the brain.
In medical terms, confusion is often referred to as "acute confusional state" or "delirium." It is characterized by symptoms such as difficulty paying attention, memory loss, disorientation, hallucinations, and delusions. Confusion can be a serious medical condition that requires immediate evaluation and treatment by a healthcare professional.
Unconsciousness is a state of complete awareness where a person is not responsive to stimuli and cannot be awakened. It is often caused by severe trauma, illness, or lack of oxygen supply to the brain. In medical terms, it is defined as a lack of response to verbal commands, pain, or other stimuli, indicating that the person's brain is not functioning at a level necessary to maintain wakefulness and awareness.
Unconsciousness can be described as having different levels, ranging from drowsiness to deep coma. The causes of unconsciousness can vary widely, including head injury, seizure, stroke, infection, drug overdose, or lack of oxygen supply to the brain. Depending on the cause and severity, unconsciousness may last for a few seconds or continue for an extended period, requiring medical intervention and treatment.
The temporal lobe is one of the four main lobes of the cerebral cortex in the brain, located on each side of the head roughly level with the ears. It plays a major role in auditory processing, memory, and emotion. The temporal lobe contains several key structures including the primary auditory cortex, which is responsible for analyzing sounds, and the hippocampus, which is crucial for forming new memories. Damage to the temporal lobe can result in various neurological symptoms such as hearing loss, memory impairment, and changes in emotional behavior.
An autobiography is a type of literature that describes the personal life experiences of an individual, written by that individual. It typically includes details about their upbringing, education, career, relationships, and other significant events in their life. The author may also reflect on their thoughts, feelings, and motivations during these experiences, providing insight into their personality and character.
Autobiographies can serve various purposes, such as sharing one's story with others, leaving a legacy for future generations, or exploring one's personal growth and development. They can be written in different styles, from straightforward and factual to introspective and reflective.
It is important to note that autobiographies are not always entirely accurate, as memory can be selective or distorted. Additionally, some individuals may choose to embellish or exaggerate certain aspects of their lives for dramatic effect or to protect the privacy of others. Nonetheless, autobiographies remain a valuable source of information about an individual's life and experiences.
The hippocampus is a complex, curved formation in the brain that resembles a seahorse (hence its name, from the Greek word "hippos" meaning horse and "kampos" meaning sea monster). It's part of the limbic system and plays crucial roles in the formation of memories, particularly long-term ones.
This region is involved in spatial navigation and cognitive maps, allowing us to recognize locations and remember how to get to them. Additionally, it's one of the first areas affected by Alzheimer's disease, which often results in memory loss as an early symptom.
Anatomically, it consists of two main parts: the Ammon's horn (or cornu ammonis) and the dentate gyrus. These structures are made up of distinct types of neurons that contribute to different aspects of learning and memory.
Thalamic diseases refer to conditions that affect the thalamus, which is a part of the brain that acts as a relay station for sensory and motor signals to the cerebral cortex. The thalamus plays a crucial role in regulating consciousness, sleep, and alertness. Thalamic diseases can cause a variety of symptoms depending on the specific area of the thalamus that is affected. These symptoms may include sensory disturbances, motor impairment, cognitive changes, and altered levels of consciousness. Examples of thalamic diseases include stroke, tumors, multiple sclerosis, infections, and degenerative disorders such as dementia and Parkinson's disease. Treatment for thalamic diseases depends on the underlying cause and may include medications, surgery, or rehabilitation therapy.
Memory disorders are a category of cognitive impairments that affect an individual's ability to acquire, store, retain, and retrieve memories. These disorders can be caused by various underlying medical conditions, including neurological disorders, psychiatric illnesses, substance abuse, or even normal aging processes. Some common memory disorders include:
1. Alzheimer's disease: A progressive neurodegenerative disorder that primarily affects older adults and is characterized by a decline in cognitive abilities, including memory, language, problem-solving, and decision-making skills.
2. Dementia: A broader term used to describe a group of symptoms associated with a decline in cognitive function severe enough to interfere with daily life. Alzheimer's disease is the most common cause of dementia, but other causes include vascular dementia, Lewy body dementia, and frontotemporal dementia.
3. Amnesia: A memory disorder characterized by difficulties in forming new memories or recalling previously learned information due to brain damage or disease. Amnesia can be temporary or permanent and may result from head trauma, stroke, infection, or substance abuse.
4. Mild cognitive impairment (MCI): A condition where an individual experiences mild but noticeable memory or cognitive difficulties that are greater than expected for their age and education level. While some individuals with MCI may progress to dementia, others may remain stable or even improve over time.
5. Korsakoff's syndrome: A memory disorder often caused by alcohol abuse and thiamine deficiency, characterized by severe short-term memory loss, confabulation (making up stories to fill in memory gaps), and disorientation.
It is essential to consult a healthcare professional if you or someone you know experiences persistent memory difficulties, as early diagnosis and intervention can help manage symptoms and improve quality of life.
Midazolam is a medication from the class of drugs known as benzodiazepines. It works by enhancing the effect of a neurotransmitter called gamma-aminobutyric acid (GABA), which has a calming effect on the brain and nervous system. Midazolam is often used for its sedative, hypnotic, anxiolytic, anticonvulsant, and muscle relaxant properties.
Medically, midazolam is used for various purposes, including:
1. Preoperative medication (sedation before surgery)
2. Procedural sedation (for minor surgical or diagnostic procedures)
3. Treatment of seizures (status epilepticus)
4. Sedation in critically ill patients
5. As an adjunct to anesthesia during surgeries
6. Treatment of alcohol withdrawal symptoms
7. To induce amnesia for certain medical or dental procedures
Midazolam is available in various forms, such as tablets, intravenous (IV) solutions, and intranasal sprays. It has a rapid onset of action and a short duration, making it suitable for brief, intermittent procedures. However, midazolam can cause side effects like drowsiness, confusion, respiratory depression, and memory impairment. Therefore, its use should be carefully monitored by healthcare professionals.
Craniocerebral trauma, also known as traumatic brain injury (TBI), is a type of injury that occurs to the head and brain. It can result from a variety of causes, including motor vehicle accidents, falls, sports injuries, violence, or other types of trauma. Craniocerebral trauma can range in severity from mild concussions to severe injuries that cause permanent disability or death.
The injury typically occurs when there is a sudden impact to the head, causing the brain to move within the skull and collide with the inside of the skull. This can result in bruising, bleeding, swelling, or tearing of brain tissue, as well as damage to blood vessels and nerves. In severe cases, the skull may be fractured or penetrated, leading to direct injury to the brain.
Symptoms of craniocerebral trauma can vary widely depending on the severity and location of the injury. They may include headache, dizziness, confusion, memory loss, difficulty speaking or understanding speech, changes in vision or hearing, weakness or numbness in the limbs, balance problems, and behavioral or emotional changes. In severe cases, the person may lose consciousness or fall into a coma.
Treatment for craniocerebral trauma depends on the severity of the injury. Mild injuries may be treated with rest, pain medication, and close monitoring, while more severe injuries may require surgery, intensive care, and rehabilitation. Prevention is key to reducing the incidence of craniocerebral trauma, including measures such as wearing seat belts and helmets, preventing falls, and avoiding violent situations.
In the context of medicine and healthcare, learning is often discussed in relation to learning abilities or disabilities that may impact an individual's capacity to acquire, process, retain, and apply new information or skills. Learning can be defined as the process of acquiring knowledge, understanding, behaviors, and skills through experience, instruction, or observation.
Learning disorders, also known as learning disabilities, are a type of neurodevelopmental disorder that affects an individual's ability to learn and process information in one or more areas, such as reading, writing, mathematics, or reasoning. These disorders are not related to intelligence or motivation but rather result from differences in the way the brain processes information.
It is important to note that learning can also be influenced by various factors, including age, cognitive abilities, physical and mental health status, cultural background, and educational experiences. Therefore, a comprehensive assessment of an individual's learning abilities and needs should take into account these various factors to provide appropriate support and interventions.
Anisomycin is an antibiotic derived from the bacterium Streptomyces griseolus. It is a potent inhibitor of protein synthesis and has been found to have antitumor, antiviral, and immunosuppressive properties. In medicine, it has been used experimentally in the treatment of some types of cancer, but its use is limited due to its significant side effects, including neurotoxicity.
In a medical or scientific context, 'anisomycin' refers specifically to this antibiotic compound and not to any general concept related to aniso- (meaning "unequal" or "asymmetrical") or -mycin (suffix indicating a bacterial antibiotic).
Paired-associate learning is a form of implicit or non-declarative memory task that involves learning and remembering the association between two unrelated items, such as a word and an object, or a taste and a sound. In this type of learning, the individual learns to respond appropriately when presented with one member of the pair, due to its association with the other member. This process is often used in various fields including cognitive psychology, neuroscience, and education to study memory, learning, and brain function.
The mamillary bodies are a pair of small, round structures located in the hypothalamus region of the brain. They play a crucial role in the limbic system, which is involved in emotions, memory, and learning. Specifically, the mamillary bodies are part of the circuit that forms the Papez circuit, a neural network responsible for memory and cognitive functions.
The mamillary bodies receive inputs from several brain regions, including the hippocampus, anterior thalamic nuclei, and cingulate gyrus. They then project this information to the thalamus, which in turn sends it to the cerebral cortex for further processing.
Damage to the mamillary bodies can result in memory impairment, as seen in patients with Korsakoff's syndrome, a condition often associated with chronic alcohol abuse.
Thiamine deficiency, also known as beriberi, is a condition that results from inadequate intake or impaired absorption of thiamine (vitamin B1), which is essential for energy metabolism and nerve function. This deficiency can lead to various symptoms such as peripheral neuropathy, muscle weakness, heart failure, and in severe cases, Wernicke-Korsakoff syndrome, a neurological disorder associated with alcoholism. Thiamine deficiency is commonly found in populations with poor nutrition, alcohol dependence, and gastrointestinal disorders affecting nutrient absorption.
Preanesthetic medication, also known as premedication, refers to the administration of medications before anesthesia to help prepare the patient for the upcoming procedure. These medications can serve various purposes, such as:
1. Anxiolysis: Reducing anxiety and promoting relaxation in patients before surgery.
2. Amnesia: Causing temporary memory loss to help patients forget the events leading up to the surgery.
3. Analgesia: Providing pain relief to minimize discomfort during and after the procedure.
4. Antisialagogue: Decreasing saliva production to reduce the risk of aspiration during intubation.
5. Bronchodilation: Relaxing bronchial smooth muscles, which can help improve respiratory function in patients with obstructive lung diseases.
6. Antiemetic: Preventing or reducing the likelihood of postoperative nausea and vomiting.
7. Sedation: Inducing a state of calmness and drowsiness to facilitate a smooth induction of anesthesia.
Common preanesthetic medications include benzodiazepines (e.g., midazolam), opioids (e.g., fentanyl), anticholinergics (e.g., glycopyrrolate), and H1-antihistamines (e.g., diphenhydramine). The choice of preanesthetic medication depends on the patient's medical history, comorbidities, and the type of anesthesia to be administered.
Verbal learning is a type of learning that involves the acquisition, processing, and retrieval of information presented in a verbal or written form. It is often assessed through tasks such as list learning, where an individual is asked to remember a list of words or sentences after a single presentation or multiple repetitions. Verbal learning is an important aspect of cognitive functioning and is commonly evaluated in neuropsychological assessments to help identify any memory or learning impairments.
The anterior thalamic nuclei are a group of nuclei in the thalamus, which is a part of the brain. The thalamus serves as a relay station for sensory and motor signals to the cerebral cortex. The anterior thalamic nuclei, specifically, are involved in various functions such as memory, navigation, and arousal. They receive inputs from the hippocampus and other limbic structures and project to the cingulate gyrus and other areas of the cerebral cortex. The anterior thalamic nuclei have been implicated in several neurological and psychiatric conditions, including epilepsy, Alzheimer's disease, and schizophrenia.
Multiple Personality Disorder (MPD) is currently referred to as Dissociative Identity Disorder (DID) in the Diagnostic and Statistical Manual of Mental Disorders, Fifth Edition (DSM-5). It's a complex dissociative disorder involving the presence of two or more distinct identities or personality states that recurrently take control of an individual's behavior.
In DID, each identity, or 'alter', may have its own unique name, personal history, characteristics, and mannerisms. The switching between these identities is often triggered by stressful situations or traumatic memories.
This disorder typically develops as a coping mechanism in response to severe, prolonged trauma during early childhood, such as emotional, physical, or sexual abuse. It's important to note that this condition should be diagnosed and managed by mental health professionals with expertise in dissociative disorders.
Medical Definition:
Magnetic Resonance Imaging (MRI) is a non-invasive diagnostic imaging technique that uses a strong magnetic field and radio waves to create detailed cross-sectional or three-dimensional images of the internal structures of the body. The patient lies within a large, cylindrical magnet, and the scanner detects changes in the direction of the magnetic field caused by protons in the body. These changes are then converted into detailed images that help medical professionals to diagnose and monitor various medical conditions, such as tumors, injuries, or diseases affecting the brain, spinal cord, heart, blood vessels, joints, and other internal organs. MRI does not use radiation like computed tomography (CT) scans.
Chronic brain damage is a condition characterized by long-term, persistent injury to the brain that results in cognitive, physical, and behavioral impairments. It can be caused by various factors such as trauma, hypoxia (lack of oxygen), infection, toxic exposure, or degenerative diseases. The effects of chronic brain damage may not be immediately apparent and can worsen over time, leading to significant disability and reduced quality of life.
The symptoms of chronic brain damage can vary widely depending on the severity and location of the injury. They may include:
* Cognitive impairments such as memory loss, difficulty concentrating, trouble with problem-solving and decision-making, and decreased learning ability
* Motor impairments such as weakness, tremors, poor coordination, and balance problems
* Sensory impairments such as hearing or vision loss, numbness, tingling, or altered sense of touch
* Speech and language difficulties such as aphasia (problems with understanding or producing speech) or dysarthria (slurred or slow speech)
* Behavioral changes such as irritability, mood swings, depression, anxiety, and personality changes
Chronic brain damage can be diagnosed through a combination of medical history, physical examination, neurological evaluation, and imaging studies such as MRI or CT scans. Treatment typically focuses on managing symptoms and maximizing function through rehabilitation therapies such as occupational therapy, speech therapy, and physical therapy. In some cases, medication or surgery may be necessary to address specific symptoms or underlying causes of the brain damage.
AmnesiA - Wikipedia
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Taylor Swift fans believe post-concert amnesia is a real phenomenon | Marca
20232
- Amnesia: The Bunker is described as a sandbox semi-open world game and is set for release in 2023. (shacknews.com)
- Amnesia: The Bunker will come to PC, Xbox, and PlayStation platforms when it launches in 2023. (shacknews.com)
Retrograde11
- D.D.B.'s trampoline injury caused "skull fracture, head lacerations, a concussion, and retrograde and anteriograde amnesia," the lawsuit states. (findlaw.com)
- Retrograde amnesia affects memories around the time of the incident, while anteriograde amnesia affects recent memories, Seattle Weekly explains. (findlaw.com)
- In a fugue state, people not only suffer from retrograde amnesia (an inability to recall the past), but they also have erased all biographical information from their memory. (psychologytoday.com)
- Retrograde amnesia occurs when one cannot recall events from one's own past. (psychologytoday.com)
- Sometimes, retrograde amnesia occurs following a blow to the head that results in the loss of events that occurred just prior to the injury, but the memory loss can be much more extensive and debilitating if there has been severe brain damage. (psychologytoday.com)
- There are many well-known cases of individuals who have experienced these types of amnesia, with one of the most famous being that of Clive Wearing , a gifted British musician who developed both profound anterograde and retrograde amnesia, resulting in a desperate existence. (psychologytoday.com)
- It seems poor Samantha was the victim of a hit-and-run driver eight days earlier, leaving her with "retrograde amnesia," a scientific term for not being able to remember almost a blessed thing. (jpost.com)
- Is it possible that Christie suffered from what is called retrograde amnesia as a result of an automobile accident and was no longer capable of remembering the event? (scientificamerican.com)
- Although affected persons may be as attentive and awake as ever, their access to previously acquired memories may be disturbed (retrograde amnesia). (scientificamerican.com)
- Transient global amnesia is characterized by sudden-onset anterograde and retrograde amnesia that typically lasts up to 24 hours. (msdmanuals.com)
- But a less severe retrograde amnesia may be the presenting symptom. (msdmanuals.com)
Frictional-games4
- Frictional Games has unveiled a new sandbox game in the Amnesia franchise. (shacknews.com)
- Now, developer Frictional Games will look to offer a new brand of scares with Amnesia: The Bunker, the latest installment in the franchise. (shacknews.com)
- Frictional Games shared early details about Amnesia: The Bunker with Shacknews ahead of its official announcement. (shacknews.com)
- Despite a couple of delays , Frictional Games finally released Amnesia: The Bunker in June, representing a return to form in the iconic Amnesia series. (destructoid.com)
Dark Descent5
- Amnesia: The Dark Descent changed the face of horror games 13 years ago, and 2020's Amnesia: Rebirth seemed to bring the story seeds it planted to a bone-chilling and climactic final bloom. (ign.com)
- The award winning Amnesia: The Dark Descent has been updated and found a new home on Desura! (moddb.com)
- Amnesia: The Dark Descent, a first person survival horror. (moddb.com)
- Enjoy Amnesia: The Dark Descent and many other great games on Desura. (moddb.com)
- You see, dear reader, I was repurposing the theme song of 1980s children's television show The Trap Door as a hook for a post about The Trapdoor, an Amnesia: The Dark Descent mod inspired by the claymation creepshow. (rockpapershotgun.com)
Bunker8
- So Amnesia: The Bunker, a smaller and more self-contained chapter, has its work cut out for it in getting me excited about this frightening franchise again. (ign.com)
- The first and biggest shake-up to the usual Amnesia routine is that the entire bunker, which feels bigger than it looks on paper, is available to explore from fairly early on. (ign.com)
- Whereas most enemies in other Amnesia games are scripted to patrol a specific area in a specific way, The Bunker takes a refreshing page out of Alien: Isolation's book and features a single, ever-present threat called the Stalker that lives behind the walls and above the ceiling of your concrete prison. (ign.com)
- Amnesia made a name for itself as a horror game with no weapons, so it seemed odd to me at first that The Bunker hands you a pistol and even sometimes grenades. (ign.com)
- The developer also provided some brief gameplay details to go alongside the newly released trailer for Amnesia: The Bunker . (shacknews.com)
- Unlike their previous Amnesia games, Amnesia: The Bunker will feature a new kind of horror survival adventure that incorporates emergent gameplay and sandbox elements into the mix. (shacknews.com)
- Armed with a revolver gun and a noisy dynamo flashlight, players will walk the course of an ill-lighted WW1 bunker on the shoes of a French soldier troubled by his amnesia and hunted by an otherworldly creature. (shacknews.com)
- The update for Amnesia: The Bunker will also be introducing "new and randomized locations of corpses, dog tags and bunker rats. (destructoid.com)
Transient global amnesia2
- The precise pathophysiology of transient global amnesia is not clear. (medscape.com)
- When diagnostic criteria are properly applied, transient global amnesia is not difficult to diagnose because it is a unique entity. (aafp.org)
Cases of transient global1
- Most cases of transient global amnesia are preceded by an emotional stress, intense pain or cold, or strenuous physical activity. (aafp.org)
Fortnight2
- So we created Amnesia Fortnight. (doublefine.com)
- Day seven of 2 Player Productions' extensive video coverage of the 2012 Amnesia Fortnight event at Double Fine. (doublefine.com)
Rock Paper Shotgun1
- According to Creative Lead Fredrik Olsson (as spotted by Rock Paper Shotgun ), the latest Amnesia game is getting a Halloween update, which includes a "shell shocked mode. (destructoid.com)
Sudden1
- Transient global amnesia involves the sudden loss of memory of recent events and a transient inability to retain new information. (aafp.org)
Forgetfulness1
- Oh yes, I was saying that older people are not the main offenders when it comes to mental oblivion, general amnesia and chronic forgetfulness. (koreatimes.co.kr)
Severe2
- This type of amnesia may be caused by severe emotional stress, but in such cases, people almost always retain knowledge of who they are and of those with whom they are close. (scientificamerican.com)
- These tests can help rule out other possible causes of the amnesia (eg, severe anemia, clotting disorders). (msdmanuals.com)
Dissociative fugue3
- The type that perhaps comes to mind most quickly is the amnesia that accompanies a dissociative fugue state . (psychologytoday.com)
- These include dissociative amnesia (DA), dissociative identity disorder (DID), dissociative fugue, depersonalization/derealization disorder, and dissociative disorder not otherwise specified (DDNOS). (medscape.com)
- [ 1 ] The previous entity of dissociative fugue has been incorporated into dissociative amnesia and is no longer a separate diagnosis. (medscape.com)
Memories5
- Amnesia: Memories was my first foray into Visual Novels and has left me wanting more of from the genre, its unique story and interesting cast will keep you entertained and hooked on the game, with multiple ending and several unlockable bonuses Amnesia: Memories is a worthwhile addition to your Vita collection. (metacritic.com)
- While its story is clever, well-constructed, and filled with some great moments, Amnesia: Memories encourages players to not question or challenge the mistreatment they experience at the hands of their romantic interests, ultimately undercutting a lot of its standout moments. (metacritic.com)
- Cute" just about sums up Amnesia: Memories. (metacritic.com)
- Amnesia: Memories is really well made otome visual novel. (metacritic.com)
- Anterograde amnesia is the inability to form new memories, and it may result from damage to the hippocampus that has occurred through accident or disease. (psychologytoday.com)
Benign1
- The authors conclude that transient global amnesia is a benign syndrome with a low risk of subsequent neurologic or vascular disease. (aafp.org)
Scanner6
- Amnesia Scanner find a kindred spirit with French-born NYC culture jammer Freeka Tet, who helps them reposition their confrontational and chaotic cross-genre experimentation in the post-NFT era. (boomkat.com)
- For a while, Berlin-based Finnish duo Amnesia Scanner sounded not like they were channeling the future, as many thought, but making a salient comment about the present's deluge of information and distraction. (boomkat.com)
- Amnesia Scanner is now living in the world it has built,' the duo state, somewhat conceitedly. (boomkat.com)
- As always, Amnesia Scanner are able to hold a mirror up to the wider scene with music that's, this time around at least, subtly bombastic. (boomkat.com)
- To be fair, Amnesia Scanner have form on this. (loudandquiet.com)
- Ville Haimala and Martti Kalliala, the surprisingly unassuming-looking Finnish pair behind Amnesia Scanner, are notoriously press-shy and reluctant to comment on the specific themes and processes of their work, but the record's pervasive senses of unease, atomisation and hyper-connected neurosis lay their general thesis pretty bare: we've never wielded so much power, nor have we ever been so vulnerable. (loudandquiet.com)
Diagnosis2
- If any lateralizing or focal findings are noted on the examination, then the diagnosis of transient global amnesia should be questioned. (medscape.com)
- Brown conducted a literature review and presents criteria for emergency diagnosis of patients with transient global amnesia. (aafp.org)
Etiology1
- Transient global amnesia (TGA) is a well-described syndrome, but one whose exact etiology is not yet completely understood. (medscape.com)
Episode1
- There is no increased incidence of vascular deaths or epilepsy in patients with an episode of transient global amnesia, but a significantly greater proportion of persons with recurrent episodes of transient global amnesia go on to experience epilepsy than do control subjects. (aafp.org)
Syndrome1
- As the syndrome resolves, the amnesia improves, but the patient may be left with a distinct lapse of recollection for events during the attack. (medscape.com)
Onset3
- Acute-onset amnesia: Transient global amnesia and other causes. (msdmanuals.com)
- A 29-year-old female experienced the onset of dissociative amnesia during an academic trip to China. (medscape.com)
- The amnesia persisted for nearly 10 months, until the feeling of blood on the woman's fingers triggered the recollection of events from the night of onset of dissociative amnesia, and, subsequently, other facts and events. (medscape.com)
Occur2
- This argument, however, is less than cogent because we know today that partial amnesia can occur and that it can affect certain memory systems such as episodic memory (of events) while leaving semantic (factual) memory intact. (scientificamerican.com)
- In addition, short-term memory lapses such as transient global amnesia lasting no more than 24 hours are known to occur. (scientificamerican.com)
Profound1
- Though the surgery was a success as far as controlling the epilepsy went, Molaison was left with profound amnesia. (listverse.com)
Unclear1
- The exact mechanism that produces transient global amnesia is unclear. (medscape.com)
Confusion1
- Although "Strawberry Amnesia" didn't start out like this, the track quickly began to share a mood of hurt and confusion. (prweb.com)
Traumatic2
Occasional1
- Despite these occasional flaws, Amnesia as a whole, is still a very good game - definitely one of the most tense psychological horrors we've played in recent years. (bit-tech.net)
Clinical1
- Detailed work-ups are unproductive in patients who have all of the features of transient global amnesia unless other clinical indications are present. (aafp.org)
Game4
- What is your favorite past Amnesia game? (ign.com)
- The goal of Amnesia is simply communicated in a letter that lays nearby your character at the start of the game. (bit-tech.net)
- Unfortunately, the only retort to these issues is to shrug them off, reminding yourself that Amnesia is just a game after all. (bit-tech.net)
- The thought occurred to me that Amnesia (the first one) was every bit as much a VR game before VR headsets came out. (shacknews.com)
Mods4
- making Amnesia mods together. (indiedb.com)
- Amnesia 1.2 update is out (new stories) + mods! (moddb.com)
- Post news RSS Amnesia 1.2 update is out (new stories) + mods! (moddb.com)
- We are always on the look out for awesome mods so if you know of or have a great Amnesia mod get in contact and get your work shown on Desura! (moddb.com)
Physical1
- Amnesia resulting from physical problems with the brain are more common. (psychologytoday.com)
Childhood1
- Inspired by concepts from the Philosophy of Difference - Schizoanalysis proposed by the philosophers Deleuze and Guattari, in ethnic-racial studies and in the artwork "Amnesia", by Flávio Cerqueira, we will present some threads of analysis on the diagrams of power that imprison the black child´s childhood experience in lines of subjection and the resistances that are half-opened from art as a powerful intercessor. (bvsalud.org)
Games1
- It's a shame I already own most games that get a 'delayed' Desura release :( It's good to see Amnesia got a Desura release anyway. (moddb.com)
Disorders1
- However, several disorders may be misdiagnosed as transient global amnesia. (aafp.org)
Memory4
- Amnesia (or amnaesia in Commonwealth English) is a condition in which memory is disturbed. (sciencedaily.com)
- These include acute confusional states such as toxin-induced memory loss, psychogenic amnesia and transient complex partial seizures that present in the postictal state. (aafp.org)
- But for people with neurological conditions like amnesia, the loss of memory can prove utterly devastating and leave them with no clue as to the person that they are. (listverse.com)
- Liljefors, M 2008, The Interplay of Memory and Amnesia: Sites of Memory in Europe and Africa . (lu.se)
Years3
- Transient global amnesia (TGA) has been a well-described phenomenon for more than 40 years. (medscape.com)
- Stroke morbidity is not increased in patients with transient global amnesia, and the risk of a recurrent attack of transient global amnesia within five years ranges from 3 to 20 percent. (aafp.org)
- Amnesia Brewing celebrates 10 years of craft brewing in the Pacific Northwest with special barrel-aged barleywine. (brewpublic.com)
Artist1
- The first electronic release for Amnesia Jones' 'Polaroid' is an exciting change of direction for the artist. (musicglue.com)
Time and place1
- Most patients with transient global amnesia are disoriented to time and place and exhibit repetitive questioning that may last throughout the attack. (aafp.org)
Early1
- in regards to them killing him so early) only to get amnesia, turn into a good guy, turn into a bad guy, then turn back into a good guy and sacrifice himself in a fairly weak scene? (superherohype.com)
Found2
- Researchers Maryam Kouchaki and Francesca Gino found evidence of unethical amnesia across nine experimental studies involving 2,109 diverse sample populations. (forbes.com)
- Amnesia," can be found on You Only Live 2wice , which is well worth a listen or several. (hotnewhiphop.com)
Life1
- My passion for photography has evolved throughout my life, particularly from an accident where I suffered amnesia. (edu.au)
Story2
- Not only is "Strawberry Amnesia" sonically captivating, but the meaning behind the song is also interesting enough to make you want to understand the story. (prweb.com)
- NEW- Amnesia custom story created by both Shyrtexx and Secretial. (indiedb.com)
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