Amitriptyline: Tricyclic antidepressant with anticholinergic and sedative properties. It appears to prevent the re-uptake of norepinephrine and serotonin at nerve terminals, thus potentiating the action of these neurotransmitters. Amitriptyline also appears to antagonize cholinergic and alpha-1 adrenergic responses to bioactive amines.Antidepressive Agents, Tricyclic: Substances that contain a fused three-ring moiety and are used in the treatment of depression. These drugs block the uptake of norepinephrine and serotonin into axon terminals and may block some subtypes of serotonin, adrenergic, and histamine receptors. However the mechanism of their antidepressant effects is not clear because the therapeutic effects usually take weeks to develop and may reflect compensatory changes in the central nervous system.Nortriptyline: A metabolite of AMITRIPTYLINE that is also used as an antidepressive agent. Nortriptyline is used in major depression, dysthymia, and atypical depressions.Maprotiline: A bridged-ring tetracyclic antidepressant that is both mechanistically and functionally similar to the tricyclic antidepressants, including side effects associated with its use.Dibenzazepines: Compounds with two BENZENE rings fused to AZEPINES.Mianserin: A tetracyclic compound with antidepressant effects. It may cause drowsiness and hematological problems. Its mechanism of therapeutic action is not well understood, although it apparently blocks alpha-adrenergic, histamine H1, and some types of serotonin receptors.Analgesics, Non-Narcotic: A subclass of analgesic agents that typically do not bind to OPIOID RECEPTORS and are not addictive. Many non-narcotic analgesics are offered as NONPRESCRIPTION DRUGS.Imipramine: The prototypical tricyclic antidepressant. It has been used in major depression, dysthymia, bipolar depression, attention-deficit disorders, agoraphobia, and panic disorders. It has less sedative effect than some other members of this therapeutic group.Protriptyline: Tricyclic antidepressant similar in action and side effects to IMIPRAMINE. It may produce excitation.Antidepressive Agents: Mood-stimulating drugs used primarily in the treatment of affective disorders and related conditions. Several MONOAMINE OXIDASE INHIBITORS are useful as antidepressants apparently as a long-term consequence of their modulation of catecholamine levels. The tricyclic compounds useful as antidepressive agents (ANTIDEPRESSIVE AGENTS, TRICYCLIC) also appear to act through brain catecholamine systems. A third group (ANTIDEPRESSIVE AGENTS, SECOND-GENERATION) is a diverse group of drugs including some that act specifically on serotonergic systems.Clomipramine: A tricyclic antidepressant similar to IMIPRAMINE that selectively inhibits the uptake of serotonin in the brain. It is readily absorbed from the gastrointestinal tract and demethylated in the liver to form its primary active metabolite, desmethylclomipramine.Nomifensine: An isoquinoline derivative that prevents dopamine reuptake into synaptosomes. The maleate was formerly used in the treatment of depression. It was withdrawn worldwide in 1986 due to the risk of acute hemolytic anemia with intravascular hemolysis resulting from its use. In some cases, renal failure also developed. (From Martindale, The Extra Pharmacopoeia, 30th ed, p266)Fluoxetine: The first highly specific serotonin uptake inhibitor. It is used as an antidepressant and often has a more acceptable side-effects profile than traditional antidepressants.Trimipramine: Tricyclic antidepressant similar to IMIPRAMINE, but with more antihistaminic and sedative properties.Desipramine: A tricyclic dibenzazepine compound that potentiates neurotransmission. Desipramine selectively blocks reuptake of norepinephrine from the neural synapse, and also appears to impair serotonin transport. This compound also possesses minor anticholinergic activity, through its affinity to muscarinic receptors.

The novel analgesic compound OT-7100 (5-n-butyl-7-(3,4,5-trimethoxybenzoylamino)pyrazolo[1,5-a]pyrimid ine) attenuates mechanical nociceptive responses in animal models of acute and peripheral neuropathic hyperalgesia. (1/363)

We investigated the effects of OT-7100, a novel analgesic compound (5-n-butyl-7-(3,4,5-trimethoxybenzoylamino)pyrazolo[1,5-a]pyrimidi ne), on prostaglandin E2 biosynthesis in vitro, acute hyperalgesia induced by yeast and substance P in rats and hyperalgesia in rats with a chronic constriction injury to the sciatic nerve (Bennett model), which is a model for peripheral neuropathic pain. OT-7100 did not inhibit prostaglandin E2 biosynthesis at 10(-8)-10(-4) M. Single oral doses of 3 and 10 mg/kg OT-7100 were effective on the hyperalgesia induced by yeast. Single oral doses of 0.1, 0.3, 1 and 3 mg/kg OT-7100 were effective on the hyperalgesia induced by substance P in which indomethacin had no effect. Repeated oral administration of OT-7100 (10 and 30 mg/kg) was effective in normalizing the mechanical nociceptive threshold in the injured paw without affecting the nociceptive threshold in the uninjured paw in the Bennett model. Indomethacin had no effect in this model. While amitriptyline (10 and 30 mg/kg) and clonazepam (3 and 10 mg/kg) significantly normalized the nociceptive threshold in the injured paw, they also increased the nociceptive threshold in the uninjured paw. These results suggest that OT-7100 is a new type of analgesic with the effect of normalizing the nociceptive threshold in peripheral neuropathic hyperalgesia.  (+info)

Amitriptyline and procainamide inhibition of cocaine and cocaethylene degradation in human serum in vitro. (2/363)

Amitriptyline (AMI) and procainamide (PA) have been reported to inhibit the activity of human plasma butyrylcholinesterase, an enzyme important in the metabolic degradation of cocaine (COC) and its ethyl analogue cocaethylene (CE). Because both AMI and PA may be used in the treatment of COC intoxication and abuse, the effect of high pharmacological concentrations of these compounds on the degradation of COC and CE in pooled human serum was studied. AMI (1.8 micromol/L) modestly inhibited the degradation of COC by 4.2% and of CE by 4.0%. PA (42.5 micromol/L) profoundly inhibited degradation of COC by 42.7% and of CE by 47.2%. In contrast, lithium carbonate (1 mmol/L, control) showed no inhibition of degradation of either COC or CE. These results suggest that AMI and PA may prolong the half-life of COC and CE in human serum.  (+info)

Determination of amitriptyline and nortriptyline in human liver microsomes with reversed-phase HPLC in vitro. (3/363)

AIM: To develop a method for simultaneous determinations of amitriptyline (Ami) and its metabolite nortriptyline (Nor) in human liver microsomes. METHODS: An incubation buffer containing microsomes, NADPH-generating system, and Ami, after termination of enzyme reaction and desipramine (Des) as internal standard (IS), was extracted with diethy ether and separated on a reversed-phase ODS column. Detection was achieved at 242 nm by ultraviolet detector. RESULTS: No potential interfering peaks were found. Ami and Nor gave rapid elution and baseline resolution. The linear curves of both analyses ranged 0.02-10 nmol and the limit of detection was 0.01 nmol. The recovery (94%-101%) had good precision with relative s of < 8.3%. CONCLUSION: This method is rapid, sensitive, and simple for studying the metabolism of Ami and Nor.  (+info)

Pharmacokinetic and pharmacodynamic characterization of OROS and immediate-release amitriptyline. (4/363)

AIMS: To characterize the pharmacokinetics of amitriptyline and its metabolite nortriptyline following OROS and IR treatments, and to correlate them with anticholinergic side-effects. METHODS: The pharmacokinetics and safety of amitriptyline following administration of an osmotic controlled release tablet (OROS and an immediate release (IR) tablet were evaluated in 14 healthy subjects. In this randomized, open label, three-way crossover feasibility study, the subjects received a single 75 mg OROS tablet, three 25 mg IR tablets administered every 8 h, or 3x25 mg IR tablets administered at nighttime. In each treatment arm serial blood samples were collected for a period of 84 h after dosing. The plasma samples were analysed by gas chromatography for amitriptyline and its metabolite nortriptyline. Anticholinergic effects such as saliva output, visual acuity, and subject-rated drowsiness and dry mouth were measured on a continuous scale during each treatment period. RESULTS: Following dosing with OROS (amitriptyline hydrochloride), the mean maximal plasma amitriptyline concentration Cmax (15.3 ng ml-1 ) was lower and the mean tmax (25.7 h) was longer than that associated with the equivalent IR dose administered at nighttime (26.8 ng ml-1 and 6.3 h, respectively). The bioavailability of amitriptyline following OROS dosing was 95% relative to IR every 8 h dosing, and 89% relative to IR nighttime dosing. The metabolite-to-drug ratios after the three treatment periods were similar, suggesting no change in metabolism between treatments. The relationships between plasma amitriptyline concentration and anticholinergic effects (e.g. reduced saliva weight, dry mouth, and drowsiness) were similar with all three treatments. Of the anticholinergic effects, only decreased saliva weight and dry mouth correlated well with plasma amitriptyline concentrations; drowsiness did not. There was no apparent correlation between anticholinergic effects and the plasma nortriptyline concentration. CONCLUSIONS: The bioavailability of OROS (amitriptyline hydrochloride) was similar to that of the IR treatments and the pharmacokinetics of amitriptyline after OROS dosing may decrease the incidence of anticholinergic effects compared with that seen with nighttime dosing of the IR formulation. Therefore, this controlled-release formulation of amitriptyline may be appropriate for single daily administration.  (+info)

Relaxant effects of antidepressants on human isolated mesenteric arteries. (5/363)

AIMS: The therapeutic action of tricyclic agents may be accompanied by unwanted effects on the cardiovascular system. The evidence for the effects on vascular and nonvascular smooth muscle comes from animal studies. Whether these studies can be extrapolated to human vessels remains to be determined. Therefore, the present study was designed to investigate the influence of amitriptyline, nortriptyline and sertraline on the contractile responses of human isolated mesenteric arteries to electrical field stimulation, noradrenaline and potassium chloride. METHODS: Arterial segments (lumen diameter 0.8-1.2 mm) were obtained from portions of the human omentum during the course of 41 abdominal operations (22 men and 19 women), and rings 3 mm long were mounted in organ baths for isometric recording of tension. In some artery rings the endothelium was removed mechanically. RESULTS: In precontracted artery rings amitriptyline, nortriptyline and sertraline (3x10(-7)-10(-4) m ) produced concentration-dependent relaxation that was independent of the presence or absence of vascular endothelium. Incubation with indomethacin (3x10(-6) m ) reduced the pD2 values thus indicating the participation of dilating prostanoid substances in this response. Amitriptyline and nortriptyline inhibited both the neurogenic-and noradrenaline-induced contractions. In contrast, only the highest concentration of sertraline reduced the adrenergic responses. Amitriptyline, nortriptyline and sertraline inhibited contractions elicited by KCl and produced rightward shifts of the concentration-response curve to CaCl2 following incubation in calcium-free solution. CONCLUSIONS: These results indicate that amitriptyline and nortriptyline could act as adrenoceptor antagonists and direct inhibitors of smooth muscle contraction of human mesenteric arteries, whereas sertraline might principally exert its action only as direct inhibitor of smooth muscle contraction. This relaxant mechanism involves an interference with the entry of calcium.  (+info)

Inhibition of the high affinity myo-inositol transport system: a common mechanism of action of antibipolar drugs? (6/363)

The mechanism of action of antibipolar drugs like lithium, carbamazepine, and valproate that are used in the treatment of manic-depressive illness, is unknown. Lithium is believed to act through uncompetitive inhibition of inositolmonophosphatase, which results in a depletion of neural cells of inositol and a concomitant modulation of phosphoinositol signaling. Here, we show that lithium ions, carbamazepine, and valproate, but not the tricyclic antidepressant amitriptyline, inhibit at therapeutically relevant concentrations and with a time course similar to their clinical actions the high affinity myo-inositol transport in astrocyte-like cells and downregulate the level of the respective mRNA. Inhibition of inositol uptake could thus represent an additional pathway for inositol depletion, which might be relevant in the mechanism of action of all three antibipolar drugs.  (+info)

Inhibition of the current of heterologously expressed HERG potassium channels by imipramine and amitriptyline. (7/363)

1 Tricyclic antidepressants (TCAs) are associated with cardiovascular side effects including prolongation of the QT interval of the ECG. In this report we studied the effects of two TCAs (imipramine and amitriptyline) on ionic current mediated by cloned HERG potassium channels. 2 Voltage clamp measurements of HERG currents were made from CHO cells transiently transfected with HERG cDNA. HERG-encoded potassium channels were inhibited in a reversible manner by both imipramine and amitriptyline. HERG tail currents (IHERG) following test pulses to +20 mV were inhibited by imipramine with an IC50 of 3.4+/-0.4 microM (mean+/-s.e.mean) and a Hill coefficient of 1.17+/-0.03 (n = 5). 3 microM amitriptyline inhibited IHERG by 34+/-6% (n = 3). The inhibition showed only weak voltage dependence. 3 Using an 'envelope of tails' comprised of pulses to +20 mV of varying durations, the tau of activation was found to be 155+/-30 ms for control and 132+/-26 ms for 3 microM imipramine (n = 5). Once maximal channel activation was achieved after 320 ms (as demonstrated by maximal tail currents), further prolongation of depolarization did not increase imipramine-mediated HERG channel inhibition. 4 Taking current measurements every second during a 10 s depolarizing pulse from -80 mV to 0 mV, block was observed during the first pulse in the presence of imipramine and the level of IHERG block was similar throughout the pulse (n=5). 5 A three pulse protocol (two depolarizing pulses to +20 mV separated by 20 ms at -80 mV) revealed that imipramine did not significantly alter the kinetics of IHERG inactivation. The tau of inactivation was 8+/-2 ms and 5.6+/-0.4 ms (n = 5) in the absence and presence of 3 microM imipramine, respectively, and currents inactivated to a similar extent. 6 Our data are consistent with TCAs causing components of block of the HERG channel in both the closed and open states. Any component of open channel block occurs rapidly upon depolarization. Inhibition of IHERG by the prototype TCAs imipramine and amitriptyline may suggest a mechanism for QT prolongation associated with risks of arrhythmia and sudden death that accompany high concentrations of TCAs following overdose.  (+info)

Fluoxetine and amitriptyline inhibit nitric oxide, prostaglandin E2, and hyaluronic acid production in human synovial cells and synovial tissue cultures. (8/363)

OBJECTIVE: To evaluate the effects of fluoxetine and amitriptyline on nitric oxide (NO), prostaglandin E2 (PGE2), and hyaluronic acid (HA) production in human synovial cells and synovial tissue cultures. METHODS: Human synovial cells, synovial tissue, and cartilage were cultured in the presence or absence of cytokines, lipopolysaccharides (LPS), fluoxetine, or amitriptyline. Production of NO, PGE2, and HA was determined in culture media. Sulfated glycosaminoglycan (S-GAG) synthesis was evaluated in cartilage by 35S incorporation. RESULTS: Fluoxetine (0.3 microg/ml, 1 microg/ml, and 3 microg/ml) inhibited NO release by 56%, 62%, and 71%, respectively, in the media of synovial cells stimulated by interleukin-1alpha (IL-1alpha; 1 ng/ml) plus tumor necrosis factor alpha (TNFalpha; 30 ng/ml). Amitriptyline (0.3 microg/ml, 1 microg/ml, and 3 microg/ml) caused a 16%, 27.3%, and 51.4% inhibition of NO release. Fluoxetine and amitriptyline (0.3 microg/ml, 1 microg/ml, and 3 microg/ml) significantly (P<0.05) inhibited PGE2 release in the media of human synovial cells in the presence of IL-1alpha plus TNFalpha, in a dose-dependent manner (up to 88% inhibition). Fluoxetine (0.3 microg/ml, 1 microg/ml, and 3 microg/ml) and amitriptyline (1 microg/ml and 3 microg/ml) significantly (P<0.05) inhibited PGE2 release in the media of human synovial tissue in the presence of LPS. Fluoxetine and amitriptyline (0.3 microg/ml, 1 microg/ml, and 3 microg/ml) also significantly (P<0.05) inhibited HA production by human synovial cells in the presence of IL-1beta plus TNFalpha. Fluoxetine and amitriptyline (1 microg/ml) partially reversed IL-1beta-induced inhibition of 35S-GAG synthesis by human cartilage cultures (P<0.05). Neither fluoxetine nor amitriptyline had a toxic effect on cells in the concentrations used. CONCLUSION: Inhibition of NO and PGE2 production by connective tissue cells is a mechanism by which some antidepressant medications may affect pain, articular inflammation, and joint damage.  (+info)

*Benzocycloheptene

Antidepressants and Anticholinergics Amineptine Amitriptyline Nortriptyline Noxiptyline Octriptyline Protriptyline Various ...

*DMOZ - Health: Pharmacy: Drugs and Medications: A: Amitriptyline

Full chemical and drug information. Includes indication, pharmacology and contraindications. ...

*Analgesic adjuvant

Bryson, HM; Wilde, MI (1 June 1996). "Amitriptyline. A review of its pharmacological properties and therapeutic use in chronic ... Examples include: amitriptyline antihistamines (e.g. hydroxyzine, promethazine) carisoprodol cyclobenzaprine dextromethorphan ...

*Management of multiple sclerosis

Amitriptyline. US National Library of Medicine (Medline) (1 August 2007). Retrieved on 2 September 2007. Moulin DE, Foley KM, ... Both Lhermitte's sign and painful dysesthesias usually respond to treatment with carbamazepine, clonazepam, or amitriptyline. ...

*Major depressive disorder

"Amitriptyline versus placebo for major depressive disorder". Cochrane Database of Systematic Reviews. 12: CD009138. doi:10.1002 ... Guaiana G, Barbui C, Hotopf M (2007). "Amitriptyline for depression". Cochrane Database of Systematic Reviews. 18 (3): 11-7. ... Similarly, a Cochrane systematic review of clinical trials of the generic tricyclic antidepressant amitriptyline concluded that ...

*Tricyclic antidepressant

B. denied suicidal ideation or intent but did admit to taking over 800 mg of amitriptyline per day for the past 3 years after ... She clearly described a euphoria associated with amitriptyline, noting that it gave her a "buzz" and that she felt "numbed up" ... Several cases of the misuse of amitriptyline alone or together with methadone or in other drug dependent patients and of ... Delisle JD (October 1990). "A case of amitriptyline abuse". Am J Psychiatry. 147 (10): 1377-8. PMID 2400006. Ms. A, a 24-year- ...

*Amitriptylinoxide

ISBN 3-88763-075-0. Rapp W (September 1978). "Comparative trial of amitriptyline-N-oxide and amitriptyline in the treatment of ... Godt HH, Fredslund-Andersen K, Edlund AH (1971). "[Amitriptyline N-oxide. A new antidepressant. A clinical double-blind trial ... Amitriptylinoxide is both an analogue and metabolite of amitriptyline, and has similar effects as well as equivalent efficacy ... Maj J, Vetulani J, Michaluk J, Rogóz Z, Skuza G (November 1982). "Central action of amitriptyline N-oxide". Pharmacopsychiatria ...

*Migraine treatment

Amitriptyline has been more frequently studied of the antidepressants and is the only antidepressant with fairly consistent ... The main two side effects that occur from taking amitriptyline are drowsiness and a dry mouth. Other common side effects of ... "Amitriptyline: Side Effects, Dosage, Uses". healthline.com. Klapper J (1997). "Divalproex sodium in migraine prophylaxis: a ... using amitriptyline are mostly due to its anticholinergic activity, including: weight gain, changes in appetite, muscle ...

*Fibromyalgia

... such as amitriptyline. However, many people experience more adverse effects than benefits. While amitriptyline has been used as ... It can take up to three months to derive benefit from the antidepressant amitriptyline and between three and six months to gain ... Moore, RA; Derry, S; Aldington, D; Cole, P; Wiffen, PJ (31 July 2015). "Amitriptyline for fibromyalgia in adults". The Cochrane ...

*Cunninghamella elegans

Zhang, D.; Evans, F. E.; Freeman, J. P.; Duhart Jr, B.; Cerniglia, C. E. (1995). "Biotransformation of amitriptyline by ... C. elegans is able to transform the tricyclic antidepressants amitriptyline and doxepin, the tetracyclic antidepressant ...

*Ritanserin

A double-blind study vs amitriptyline". Headache. 30 (7): 439-44. doi:10.1111/j.1526-4610.1990.hed3007439.x. PMID 2119355. ...

*Antidepressant

"Amitriptyline versus placebo for major depressive disorder". Cochrane Database of Systematic Reviews. 12: CD009138. doi:10.1002 ... The same group reviewed data for amitriptyline in the treatment of neuropathic pain and found limited useful randomized ... The group was concerned about the potential for overestimating the amount of pain relief provided by amitriptyline, and ... Antidepressants including amitriptyline, fluoxetine, duloxetine, milnacipran, moclobemide, and pirlindole are recommended by ...

*Chronic pain

Moore RA, Derry S, Aldington D, Cole P, Wiffen PJ (2015). "Amitriptyline for neuropathic pain in adults". Cochrane Database ...

*Neuropathic pain

While amitriptyline and desipramine have been used as first-line treatments, the quality of evidence to support their use is ... Moore, R. Andrew; Derry, Sheena; Aldington, Dominic; Cole, Peter; Wiffen, Philip J. (2015-07-06). "Amitriptyline for ... Other topical agents such as amitriptyline, gabapentin, Citrullus colocynthis extract, nifedipine, and pentoxifylline are also ... as well as tricyclic antidepressants such as amitriptyline, nortriptyline, and desipramine are considered first-line ...

*Minitran

It contains Amitriptyline hydrochloride and Perphenazine. It is sold in the following forms: Minitran 2-10: 2 mg Perphenazine ... Minitran 2-25: 2 mg Perphenazine and 25 mg Amitriptyline hydrochloride in each tablet. Minitran 4-10: 4 mg Perphenazine and 10 ... Minitran 4-25: 4 mg Perphenazine and 25 mg Amitriptyline hydrochloride in each tablet. Minitran is also a pharmaceutical drug ... and 10 mg Amitriptyline hydrochloride in each tablet. ...

*Anxiolytic

Examples include imipramine, amitriptyline, nortriptyline and desipramine. Mirtazapine has demonstrated anxiolytic effects with ...

*Diabetic neuropathy

Among the TCAs, amitriptyline is most widely used for this condition, but desipramine and nortriptyline have fewer side effects ... TCAs include imipramine, amitriptyline, desipramine, and nortriptyline. They are generally regarded as first or second-line ... and amitriptyline were more effective than placebo, but that comparative effectiveness between each agent is unclear. The only ...

*Migraine

Amitriptyline and venlafaxine are probably also effective. Angiotensin inhibition by either an angiotensin-converting enzyme ...

*Noxiptiline

Of the TCAs, noxiptiline has been described as one of the most effective, rivaling amitriptyline in clinical efficacy. Swiss ... multicentre comparison with amitriptyline". Pharmakopsychiatrie, Neuro-Psychopharmakologie. 8 (1): 26-35. doi:10.1055/s-0028- ...

*Butriptyline

... is an analogue of amitriptyline with an isobutyl side chain instead of a propylidene side chain. It is a tertiary ... Butriptyline is closely related to amitriptyline, and produces similar effects as other TCAs, but its side effects like ... However, in small clinical trials, using similar doses, butriptyline was found to be similarly effective to amitriptyline and ... Other tertiary amine TCAs include amitriptyline, imipramine, clomipramine, dosulepin (dothiepin), doxepin, and trimipramine. ...

*Medifoxamine

... notably greater affinity relative to amitriptyline and imipramine). It is known to produce two active metabolites during first- ...

*Pseudobulbar affect

Traditionally, antidepressants such as sertraline, fluoxetine,citalopram, nortriptyline and amitriptyline have been prescribed ... "Treatment of Pathologic Laughing and Weeping with Amitriptyline". The New England Journal of Medicine. 312 (23): 1480-2. doi: ...

*Ciguatera

Some medications such as amitriptyline may reduce some symptoms, such as fatigue and paresthesia, although benefit does not ... ISBN 3-540-94069-3. Davis R, Villar L (1986). "Symptomatic improvement with amitriptyline in ciguatera fish poisoning". N. Engl ...

*Phenoxypropazine

ROSE JT, LEAHY MR, PLOWMAN R (October 1963). "A comparison of phenoxypropazine and amitriptyline in depression". The American ...

*Protriptyline

... is a secondary amine TCA, with its N-methylated relative amitriptyline being a tertiary amine. Other secondary ... Other dibenzocycloheptadiene TCAs include amitriptyline, nortriptyline, and butriptyline. ...
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Amitriptyline Side Effects Nerve Pain. Amitriptyline for neuropathic pain in adults Cochrane Amitriptyline is an antidepressant, and antidepressants are widely recommended for treating neuropathic pain. Amitriptyline is commonly used to treat neuropathic pain conditions, but an earlier review found no good quality evidence to support its use. Most studies were small, relatively old, and used nbsp; Amitriptyline User Reviews for Pain at when used in the treatment of pain. 178 reviews submitted. quot;Have been prescribed Amitriptyline for sun damaged nerves on my scalp start with 10mg and I was so scared to take as don 39;t want to become dependant on them. It has helped a little and Dr wants me to increase to 20mg. Amitriptyline Pain Concern Conventional painkillers such as codeine and ibuprofen are used for nociceptive pain. They are often not effective for neuropathic pain. Most of the drugs used for the relief of neuropathic pain were originally developed to treat different conditions. For ...
Amitriptyline was introduced at night, so I take 20mg of them as well, to aid nerve pain, not depression. However, my back pain became worse and the Tramadol was increased again to mg SR at night and mornings. This I was fine with until recently. My doctor did not want to increase the tramadol further Can I take co-codamol while on Amitriptyline? , Amitriptyline. Youre more likely to feel sleepy if you take other medicines that can cause drowsiness, such as the following, while youre taking amitriptyline: antihistamines that can cause drowsiness, eg chlorphenamine, promethazine; baclofen (severe muscle weakness may also occur with this combination).. A Occupy Drug Interaction costs between Elavil and tramadol. Crown detailed information regarding this drug interaction. TraMADol may then cause seizures, and combining it with other patients that can also comes does tramadol and amitriptyline mix well such as amitriptyline may find that risk. You may be more anxious if you are elderly. Can you ...
Eight double-blind controlled clinical studies were performed in 642 patients comparing cyclobenzaprine hydrochloride 10 mg, diazepam, and placebo. Muscle spasm, local pain and tenderness, limitation of motion, and restriction in activities of daily living were evaluated. In three of these studies there was a significantly greater improvement with cyclobenzaprine than with diazepam, while in the other studies the improvement following both treatments was comparable.. Although the frequency and severity of adverse reactions observed in patients treated with cyclobenzaprine were comparable to those observed in patients treated with diazepam, dry mouth was observed more frequently in patients treated with cyclobenzaprine and dizziness more frequently in those treated with diazepam. The incidence of drowsiness, the most frequent adverse reaction, was similar with both drugs.. The efficacy of cyclobenzaprine hydrochloride tablets 5 mg was demonstrated in two seven-day, double-blind, controlled ...
Although overdoses of tricyclic antidepressant are known to produce both sinus tachycardia and ventricular tachyarrhythmias in man, these have been assumed to occur by independent mechanisms. This study was designed to evaluate the relationship of ventricular activation frequency to the cardiotoxic effects of amitriptyline. When amitriptyline was infused into dogs with formalin-induced atrioventricular (AV) block to evaluate a broad range of pacing frequencies, the drug produced dose-related QRS prolongation that was markedly frequency dependent. Similar frequency-dependent depression of the maximum rate of depolarization (Vmax) was noted for canine Purkinje fibers superfused with amitriptyline in vitro. The time constant of recovery from amitriptyline-induced block was dose independent and averaged 228 msec in vivo and 216 msec in vitro. When amitriptyline was infused into dogs with intact AV conduction, sinus tachycardia occurred within 15 min, followed by progressive QRS prolongation and ...
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The main side effects seen with amitriptyline are sedation (extreme drowsiness), followed by dry mouth. Amitriptyline can cause a dog to salivate less, which will cause him to lick his lips a lot, feel thirstier, pant, have dry eyes and increase his water intake. A dog may have an upset stomach or a loss of appetite while taking this medication. Amitriptyline can also cause a dogs blood pressure to lower, increase his heart rate, or alter his blood-sugar levels. Urine retention, constipation, headaches, slight behavior changes, nausea and weight gain have also been reported in dogs using amitriptyline. In rare cases, a dog will faint after exercising or standing up, and can experience muscle weakness or stiffness while being treated with amitriptyline. Other serious side effects include seizures and heart problems. Dogs with a liver condition may not tolerate amitriptyline well as this medicine is filtered and removed through this organ. Periodic blood tests will need to be done to check the ...
Amitriptyline Codeine Naproxen. Amitriptyline: interactions with other medicines -…28 Apr 2016 Find out if its safe to take over-the-counter medicines with amitriptyline and what effect it can have on other medicines you may already be taking. benzodiazepines, eg diazepam, temazepam; sleeping tablets, eg zopiclone; strong opioid painkillers, such as morphine, codeine, co-codamol, tramadol.Been taking Amitriptyline for 2 weeks, side effects…17 Jan 2015 When I started taking Amitriptyline, the same dose as you I did wake up with headaches but after about 4 weeks it settled down and now I dont have any side effects fortunately. I also take codeine and Naproxen as I have Rheumatoid arthritis. Im not sure what could be causing your other symptoms. I would The UKFibromyalgia Forums • View topic - Naproxen and…9 Nov 2012 Dont know whether to persevere in the hope that the amitriptyline might build up and eventually make the naproxen work or whether to give up and head back to my I am ...
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Twenty-eight elderly inpatients suffering from major depressive episodes (diagnosed according to DSM III) received randomly, on a double-blind basis, amitriptyline (75 mg/die) or fluoxetine (20 mg/die) for five weeks. There were four drop-outs in the amitriptyline group and two drop-outs in the fluoxetine group. Both groups showed a significant amelioration at the end point for Hamilton Rating Scale of Depression scores compared to the baseline value. Anticholinergic side-effects were significantly more severe in the amitriptyline group. Weight gain was detected only in patients receiving amitriptyline.
Doctors give trusted answers on uses, effects, side-effects, and cautions: Dr. Newton on amitriptyline hydrochloride overdose: Is a Tricyclic Antidepressant, used for Major depression, Enuresis, ADHD, Headache prophylaxis, among others.
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Amitriptyline and breastfeeding - My child just turned 3 months. I am exclusively breastfeeding But I started taking Elavil (amitriptyline) 10mg for bith my deppression and insomnia. I really do not stop breastfeedin n decided to continue. Okey???? Not the best. Elavil (amitriptyline) is one of the medications that will go into the breast milk if you are taking it. That means breast feeding while taking Elavil (amitriptyline) is like feeding tiny doses of Elvail to your baby. Please talk to your doctor and consider stopping wither the Elavil (amitriptyline) (and switching to something that does not go into breast milk) or breastfeeding. It is in your babys best interest.
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Background: This study was designed to compare the efficacy, safety, tolerability profiles, and effects on quality of life of the serotonin selective reuptake inhibitor antidepressant sertraline versus the nonselective tricyclic antidepressant amitriptyline and placebo in patients with major depression. Method: Outpatients with DSM-III-R major depression were randomly assigned to double-blind treatment for 8 weeks with sertraline (50_200 mg daily), amitriptyline (50_150 mg daily), or matching placebo. Assessments included the Hamilton Rating Scale for Depression, Montgomery-Asberg Depression Rating Scale, Clinical Global Impressions-Severity of Illness scale, Clinical Global Impressions-Improvement scale, Global Assessment Scale, Profile of Mood States, Beck Depression Inventory, Quality of Life Enjoyment and Satisfaction Questionnaire, and Health-Related Quality of Life battery. Results: All treatment groups demonstrated statistically significant improvement from baseline in depression ratings ...
Depressive disorders are more common among persons with chronic diseases such as inflammatory bowel disease and anti-inflammatory effect of some antidepressants such as amitriptyline has been reported. Acetic acid colitis was induced in both reserpinised (depressed) and non-reserpinised (normal) rats. Reserpinised groups received reserpine (6 mg/kg, i.p.) one hour prior to colitis induction. Then Amitriptyline (5, 10, 20 mg/kg, i.p.) was administered to separate groups of male Wistar rats. All treatments were carried out two hours after colitis induction and continued daily for four days. Dexamethasone (1 mg/kg) and normal saline (1 ml/kg) were used in reference and control groups, respectively. At day five, animals were euthanized and colonic tissue injuries were assessed macroscopically and pathologically. Myeloperoxidase activity as a marker of neutrophil infiltration was also measured in colonic tissues. Results showed that reserpine (6 mg/kg, i.p.) intensified colitic condition. Compared to control
Amitriptyline is a tricyclic antidepressant. Amitriptyline affects chemicals in the brain that may be unbalanced in people with depression. Amitriptyline is used to treat symptoms of depression. Amitriptyline may also be used for purposes not listed in this medication guide.
We studied 145 people who take Citalopram hydrobromide and Amitriptyline hydrochloride from FDA. Drug interactions are found. See what they are, when they happen and for whom.
Drugs may be covered by multiple patents or regulatory protections. All trademarks and applicant names are the property of their respective owners or licensors. Although great care is taken in the proper and correct provision of this service, thinkBiotech LLC does not accept any responsibility for possible consequences of errors or omissions in the provided data. The data presented herein is for information purposes only. There is no warranty that the data contained herein is error free. thinkBiotech performs no independent verifification of facts as provided by public sources nor are attempts made to provide legal or investing advice. Any reliance on data provided herein is done solely at the discretion of the user. Users of this service are advised to seek professional advice and independent confirmation before considering acting on any of the provided information. thinkBiotech LLC reserves the right to amend, extend or withdraw any part or all of the offered service without notice. ...
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Suicidal or agitated behaviour: People taking antidepressants such as amitriptyline may feel agitated (restless, anxious, aggressive, emotional, trouble sleeping, and feeling not like themselves), or they may want to hurt themselves or others. If you notice any changes in mood, behaviours, thoughts, or feelings in yourself or someone who is taking this medication, contact a doctor immediately. Your doctor will monitor you closely for behaviour changes, especially at the start of treatment or when your dose is increased or decreased.. Surgery: Using amitriptyline before, during, and after surgery may increase the risk of developing abnormal heart rhythms. The risks and benefits of continuing amitriptyline during elective surgery should be discussed with your doctor. Your doctor may recommend to stop or reduce the dose of amitriptyline several days prior to the scheduled surgery.. Thyroid disease: Patients who have an overactive thyroid (hyperthyroidism) or are taking thyroid medication should be ...
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Is Vomiting a common side effect of Cyclobenzaprine? View Vomiting Cyclobenzaprine side effect risks. Female, 41 years of age, was diagnosed with ligament sprain and took Cyclobenzaprine . Patient was hospitalized.
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Amitriptyline is an antidepressant drug belonging to class Tricyclic Antidepressant. It is used for the treatment of depression and other chemical imbalances in the brain. It was discovered in 1960 and was approved by US Food and Drug Administration in 1961. The brand name under which amitriptyline is sold is Vanattip, Elavil, etc. It is chemically known as 3-(10,11-Dihydro-5H-dibenzo[a,d]cycloheptene-5-ylidene)-N,N-dimethylpropan-1-amine. Its generic is also available in the US market and it is also included in the WHO essential medicine list. The recommended route of administration is oral. It is available in various dosage forms i.e oral tablets, capsule, oral suspension, oral solution and transdermal gel. The recommended dose for amitriptyline is 75 mg orally per day and has divided doses with a maximum of 150 mg per day. The drug is basically used for the treatment of number of mental illnesses which may include depressive disorder and anxiety disorder and bipolar disorder. Other than ...
We report the case of 9 years old child who was referto us for intermittent exotropia of the right eye. The XTonset according to the parents was the age 6 months.. He was treated with prism glasses. No other treatmentshave been used.Surgery was proposed but initially parents refused it.. The first examination was done in our clinic in June2012 and revealed:VA OD=0, 9 with -1, 50 cyl ax 180; VAOS=0, 8 with +1,50 cyl ax 90; 5 PD base-in were included in each lensof his glasses.. Fusion at distance was intermittently present with bettercontrol at near (Figure 1).. Re-evaluation was done 3 months and 9 months laterafter prisms-in removal and new correction prescriptionaccording to cycloplegic (cyclopentolate 1%)measurements.. Maximum deviation found at distance and near was:-35 PD in primary position, -40 PD in up-gaze, -30 PDin down-gaze. Good adduction and acceptable convergenceamplitude on both eyes were present.. The surgical treatment was provided in June 2013: ODRight Lateral Rectus Muscle ...
BACKGROUND: Coma blisters are most commonly associated with barbiturate and benzodiazepine overdose; however, they have also been described in association with many other substances, including amitriptyline. OBJECTIVE: To review the literature on the
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Cyclobenzaprine is used to help relax certain muscles in your body. It helps relieve pain, stiffness, and discomfort caused by strains, sprains, or injuries to your muscles. However, this medicine does not take the place of rest, exercise or physical therapy, or other treatment that your doctor may recommend for your medical problem. Cyclobenzaprine acts on the central nervous system (CNS) to produce its muscle relaxant effects. Its actions on the CNS may also cause some of this medicines side effects. ...
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Amitriptyline hydrochloride is a highly permeable active pharmaceutical ingredient (API). The function of these drugs is to block the reuptake of the neurotransmitters, norepinephrine and serotonin in the central nervous system. The nano-sized Amitriptyline (AT) imprinted polymer particles were synthesized successfully. The nanoparticles were characterized by Fourier transform infrared spectroscopy (FT-IR), scanning electron microscopy (SEM) and thermal gravimetric (TG) methods. AT-imprinted polymer was prepared using suspension polymerization in silicon oil with AT as template, Methacrylic acid (MAA) as functional monomer and ethylene glycol dimethacrylate (EGDMA) as cross-linker. As illustrated in SEM images, it is possible to obtain real nano-sized molecular imprinted polymer particles (around 80 nm) with approximately spherical shapes, through the methods and techniques presented and discussed in this study. Thermal analyzes indicated that, an abrupt weight loss for nano-sized MIP was observed at
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The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our disclaimer for details ...
Which Muscle Relaxant? Submitted by Henry Resnick, MS, PharmD, RPh, CPh A patients medication history upon admission stated that he had an allergy to Triavil (perphenazine + amitriptyline), which is used for anxiety, agitation, or depression. Two days after admission, a physician ordered Flexeril (cyclobenzaprine), which is indicated for herniated discs, lower back strain, or muscle spasms. An alert pharmacist saw that cyclobenzaprine labeling has the following warning, per CliniSphere 2.0: "Cyclobenzaprine is closely related [has a similar chemical structure] to the tricyclic antidepressants (eg, amitriptyline, imipramine). In shortterm studies for indications other than muscle spasm associated with acute musculoskeletal conditions, and usually at doses somewhat greater than those recommended for skeletal muscle spasm, some of the more serious CNS [central nervous system] reactions noted with the tricyclic antidepressants have occurred. Tricyclic antidepressants have been reported to produce ...
Peripheral neuropathic pain is characterized by an increased activation of afferent nociceptors and sensitized afferent information through spinal processing [4, 24]. Mechanical allodynia, a central sensitization caused by peripheral noxious barrage to the spinal cord, is characterized by a painful sensation evoked by light touch, a normally innocuous stimulation. Touch-evoked pain is a hallmark of neuropathic pain and is triggered by spontaneous ectopic discharges from injured peripheral nerves to sensitized spinal dorsal horn cells. Although mechanisms of allodynia are not entirely understood, they may involve A-beta myelinated afferents [25], activated microglia and astrocytes [26-30], and dorsal horn neuron cells [28]. It has been shown that sodium channel blockers applied to the injured site [31], DRG [32] or the spinal cord [33] all effectively decrease neuropathic pain and attenuate hyperalgesia and allodynia. The present study demonstrates that intrathecal pretreatment with ...
In rats treated with cyclobenzaprine for up to 67 weeks at doses of approximately 5 to 40 times the maximum recommended human dose, pale, sometimes enlarged, livers were noted and there was a dose-related hepatocyte vacuolation with lipidosis. In the higher dose groups, this microscopic change was seen after 26 weeks and even earlier in rats that died prior to 26 weeks; at lower doses, the change was not seen until after 26 weeks. Cyclobenzaprine did not affect the onset, incidence, or distribution of neoplasia in an 81-week study in the mouse or in a 105-week study in the rat. At oral doses of up to 10 times the human dose, cyclobenzaprine did not adversely affect the reproductive performance or fertility of male or female rats. Cyclobenzaprine did not demonstrate mutagenic activity in the male mouse at dose levels of up to 20 times the human dose.. A battery of mutagenicity tests using bacterial and mammalian systems for point mutations and cytogenic effects have provided no evidence for a ...
f_id sub_id field_name field_val 49 1 page /embedded-figures-test 50 1 Gender Female 51 1 Age 30 - 39 52 1 Occupation housewife, full-time carer to two autistic children 53 1 New Activities Cooking,Meditation,Break Up 54 1 I Enjoy My Job Agree 55 1 My Job Is Well Defined Strongly Disagree 56 1 My Co-workers Are Co-operative Strongly Disagree 57 1 My Workplace Is Stressful Strongly Agree 58 1 My Job Is Stressful Strongly Agree 59 1 Morale Is Good Where I Work Disagree 60 1 I Get Frustrated At Work Strongly Agree 61 1 Prescription Meds Benzodiazepines (Valium) 62 1 Other Meds tricyclic antidepressant (amitriptyline) 63 1 Non-Prescription Drugs Tobacco 64 1 Other Drugs diphenhydramine, melatonin, paracetamol 65 1 Before 2284 66 1 After 1989 67 5 page /embedded-figures-test 68 5 Gender Male 69 5 Age 40 - 49 70 5 Occupation Software Architect 71 5 New Activities Evening Walks,Cooking,Meditation,Change Routes,See Old Friends,Get More Sleep 72 5 I Enjoy My Job Strongly Agree 73 5 My Job Is Well Defined ...
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We thank Dr. Sawynok for the interest in our study1 and for correcting an error in the conversion of our doses from mM to nmol. Fortunately, the experimental protocol, preparation and dosing of drug, and results and conclusions of our article remain unchanged, as this error was only one of unit conversion for the reader. The error does not affect the experimental paradigm or significance of the results. We also thank Dr. Sawynok for comparing the dose used in several studies of amitriptyline in their table, as this helps place our experiments in context. The range of dose evaluated in our study was in accordance with the doses previously used in studies that reported a prolongation of sciatic nerve blockade2 (i.e. , 5-0.625 mg or 16,000-2,000 nmol; each dose administered in a 0.2-ml volume adjacent to rat sciatic nerve). Our data clearly demonstrate that amitriptyline causes a dose-related neuropathologic change if it is administered in the immediate vicinity of a nerve, and suggests that it ...
We found eligible studies of treatment for fibromyalgia with amitriptyline, nortriptyline, citalopram, fluoxetine, paroxetine, cyclobenzaprine, pregabalin, gabapentin, milnacipran, and duloxetine.. We found no eligible studies with the other included drugs and no eligible studies of included interventions when used as adjunctive therapy.. Head-to-head trials were few, and provided low-strength evidence that short-term treatment with immediate-release paroxetine is superior to amitriptyline in reducing pain and sleep disturbance and provided low-strength evidence there are no significant differences between amitriptyline as compared with cyclobenzaprine and nortriptyline.. Although there were some significant differences between drugs in overall adverse events, they did not produce any differences in withdrawals due to adverse events.. Additionally, based on indirect comparison meta-analysis, we found low evidence that duloxetine was superior to milnacipran on outcomes of pain, sleep disturbance, ...
Both amitriptyline, an antidepressant, and acupuncture, a Chinese medical approach that uses needles to relieve pain, have been used successfully to reduce pain in some people. It is not known how effectively these approaches relieve or reduce pain in patients with peripheral neuropathy secondary to HIV infection.. Patients are randomized to receive either standardized point acupuncture or alternate point acupuncture treatment twice weekly for the first 6 weeks, then once weekly for the next 8 weeks, plus either oral amitriptyline or placebo daily for the entire 14 weeks. Acupuncture points are located on the lower leg. Patients are evaluated at weeks 6 and 14 and are asked to keep a daily pain diary. ...
Vingt-et-une études (1 437 participants) ont été incluses ; elles portaient individuellement sur 15 à 235 participants, seules quatre portaient sur plus de 100 participants et la taille médiane des études était de 44 participants. La durée médiane était de six semaines. Dix études avaient un plan d'étude croisé. Les doses d'amitriptyline étaient généralement situées entre 25 mg et 125 mg et l'utilisation de doses croissantes était courante.. Il n'y avait pas de preuves de niveau supérieur concernant l'amitriptyline pour le traitement de la douleur neuropathique ou de la fibromyalgie.. Les preuves du second niveau ne démontraient aucun effet pour la douleur neuropathique liée au cancer ou la douleur neuropathique liée au VIH, mais certaines preuves indiquaient un effet sur la neuropathie diabétique douloureuse (NDD), la douleur neuropathique mixte et la fibromyalgie. En combinant les douleurs neuropathiques classiques de la NDD, la névralgie ...
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I had bowel resection surgery two years ago. They removed most of my colon and 18 of small bowel. Since surgery, I have severe diarrhea. The colon absorbs water so without the colon, there is nothing to absorb the water which results in diarrhea. Ive tried imodium, questran, keopectate etc. My doc has recently prescribed the anti-depressant Amitriptyline. Im told one of the side affects is constipation so it should help to bulk up my stool. Ive been very reluctant to try this new med as I dont want to feel out of it, groggy etc. Is anyone taking this med and has it helped with diarrhea? My doc also prescribed codiene which is an addictive drug? Im told it should also help with the diarrhea. I havent tried either drug yet and am hoping to get some feedback before I decide. My doc says to try the amitriptyline first and see how that goes. Any advice is greatly appreciated ...
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All medicines may cause side effects, but many people have no, or minor, side effects.. Check with your doctor if any of these most common side effects persist or become bothersome:. Blurred vision; change in sexual desire or ability; constipation; diarrhea; dizziness; drowsiness; dry mouth; headache; loss of appetite; nausea; tiredness; trouble sleeping; weakness.. Seek medical attention right away if any of these severe side effects occur:. Severe allergic reactions (rash; hives; itching; difficulty breathing; tightness in the chest; swelling of the mouth, face, lips, or tongue); chest pain; confusion; dark urine; delusions; difficulty speaking or swallowing; fainting; fast or irregular heartbeat; fever, chills, or sore throat; hallucinations; new or worsening agitation, anxiety, panic attacks, aggressiveness, impulsiveness, irritability, hostility, exaggerated feeling of well-being, restlessness, or inability to sit still; numbness or tingling in an arm or leg; one-sided weakness; seizures; ...
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In this study we have evaluated the postmortem pharmacokinetics of amitriptyline (Ami) and metabolites in pigs after oral and intravenous administration, and the results are compared with previous studies in rats and humans. In addition a meticulous investigation of blood and tissue concentrations after postmortem intravenous infusion of Ami was undertaken. Of a total of 9 over-night fasted pigs, 3 were given 25 mg/Kg Ami orally, and another 3 pigs received an intravenous infusion lasting 1 h of 3.3 mg/Kg Ami prior to death. The final 3 pigs were sacrificed and then given the intravenous infusion after death. After ∼5 h at room temperature, all carcasses were subsequently stored at 4-5°C. Postmortem blood samples were collected at 0.25, 1, 2, 4, 8, 24, 48, and 96 h through an indwelling intracardial needle. Postmortem examination with blood and tissue sampling was performed 96 h after death. Analysis was carried out by high performance liquid chromatography with ultraviolet detection. ...
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Since my new rheumy thinks I have Fibro, not lupus, Ive been weaning off the prednisone and started a very low dose of amitriptyline (just 5Mg daily - Im already on medication for bipolar disorder and have to be careful with levels.) I was only on 5Mg of pred anyway so not a lot to come off, just dropping 1Mg every 2 weeks a advised. Around the time I started this whole med change I have developed a type of numbness at the front of my head, sometimes reaching down to my eyebrows. The skin
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OBJECTIVES: Although the tricyclic antidepressant amitriptyline is extensively used in the prophylactic treatment of chronic tension-type headache, only few studies have investigated the efficacy of this treatment and the results are contradictory. In addition, the new selective serotonin reuptake inhibiting antidepressants, which are widely used in depression and of potential value in pain management, have never been investigated in a placebo controlled study of tension-type headache. The aim was to evaluate the efficacy of amitriptyline and of the selective serotonin reuptake inhibitor citalopram in chronic tension-type headache. METHODS: Forty non-depressed patients with chronic tension type headache were included in a 32 week, double blind, placebo controlled, three-way crossover study. RESULTS: Thirty four patients completed the trial. Amitriptyline reduced area under the headache curve by 30% compared with placebo (P = 0.002), whereas citalopram had no significant effect (P = 0.68). ...
The results from this study support the efficacy and tolerability of melatonin 3 mg as a preventive therapy for migraine. Melatonin was more effective than placebo in the primary end point and all other end points studied. Although melatonin was as effective as amitriptyline 25 mg in the primary end point, in the secondary end point, proportion of patients who improved ,50% in headache frequency, melatonin was superior to both placebo and amitriptyline. The placebo rate in our study was 20.4%, similar to the rates reported in the meta-analysis of pharmacological migraine prevention studies 22% (0.17% to 0.28%).. A pilot open-label study was published in 2004,9 showing efficacy and good tolerability of the same melatonin dose as was studied in this trial. A placebo controlled study comparing a different melatonin compound and dose (extended-release melatonin 2.0 mg, Circadin (Neurim Pharmaceuticals, Tel-Aviv, Israel)) with placebo showed negative results.10 Several methodological issues including ...
Amitriptyline pills at dosages ranging from 25mg to 150 mg is the best studied tricyclic antidepressant in diabetic peripheral neuropathic pain. The tricyclic antidepressants are widely used to tret chronic pain syndromes including diabetic peripheral neuropathic pain. In high doses, antidepressants are most pronounced true antidepressant medications. But in lower doses, the antidepressants such as Amitriptyline and Desipramine have a more potent analgesic (painkiller) effect. Amitriptyline pills inhibit serotonin and norepinephrine reuptake in people with diabetic peripheral neuropathic pain. Curiously enough, none of the tricyclic antidepressants has been approved by the FDA for treatment of diabetic peripheral neuropathic pain despite their widespread use in diabetic pain management and treatment.. » Read more. .huge-it-share-buttons { border:0px solid #0FB5D6; border-radius:5px; background:#3BD8FF; text-align:right; } #huge-it-share-buttons-top {margin-bottom:0px;} ...
OBJECTIVE To describe a case of cross hepatotoxicity between tricyclic antidepressants and phenothiazines. PATIENT A woman who developed three episodes of drug-induced hepatitis within 3 years as a result of successive treatment with two tricyclic antidepressants, trimipramine and desipramine, and one neuroleptic derivative, cyamemazine. INTERVENTIONS The drugs were withdrawn after the patient experienced liver dysfunction, although bromazepam was later administered with no side effects. RESULTS After three episodes of drug-induced hepatitis the patients serum aspartate aminotransferase and alanine aminotransferase levels returned to normal when the tricyclic antidepressants and cyamemazine were withdrawn. CONCLUSIONS Trimipramine, desipramine and cyamemazine are related by their chemical structures which include a tricyclic ring. This suggests that this chemical moiety might be involved in the hepatotoxicity of tricyclic antidepressants and phenothiazine derivatives.
We studied the safety and efficacy of 0 U, 50 U, 100 U, 150 U (five sites), 86 Usub and 100 Usub (three sites) botulinum toxin type A (BoNTA; BOTOX®; Allergan, Inc., Irvine, CA, USA) for the prophylaxis of chronic tension-type headache (CTTH). Three hundred patients (62.3% female; mean age 42.6 years) enrolled. For the primary endpoint, the mean change from baseline in the number of TTH-free days per month, there was no statistically significant difference between placebo and four BoNTA groups, but a significant difference favouring placebo vs. BoNTA 150 was observed (4.5 vs. 2.8 tension headache-free days/month; P = 0.007). All treatment groups improved at day 60. Although efficacy was not demonstrated for the primary endpoint, at day 90, more patients in three BoNTA groups had ≥50% decrease in tension headache days than did placebo (P ≤ 0.024). Most treatment-related adverse events were mild or moderate, and transient. BoNTA was safe and well-tolerated in the study. ...
Benzocycloheptenes are cycloheptenes with additional benzene rings attached. Most have two benzene rings, and are called "dibenzocycloheptenes". Some benzocycloheptenes and substituted benzocycloheptenes have medical uses as antihistamines, anticholinergics, antidepressants, antiserotonergics. Examples include: Antihistamines and Antiserotonergics Azatadine Desloratadine Loratadine Rupatadine Cyproheptadine Ketotifen Pizotifen Anticholinergics Deptropine Anticonvulsants Oxitriptyline Antidepressants and Anticholinergics Amineptine Amitriptyline Nortriptyline Noxiptyline Octriptyline Protriptyline Various Cyclobenzaprine Intriptyline Toll-like receptor 4 investigating probable antagonistic (antiinflammatory) property of several TCA based molecules Benzocycloheptenes at the US National Library of Medicine Medical Subject Headings (MeSH ...
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Antidepressants and Phototherapy. By Marcello Lanares, MD, PhD. Antidepressants are used commonly in medical and psychiatric practice. As a class, antidepressants have in common their ability to treat major depressive illness. Most antidepressants are also effective in the treatment of panic disorder and other anxiety disorders. Some antidepressants effectively treat obsessive-compulsive disorder (OCD) and a variety of other conditions (see indications below). The most commonly prescribed antidepressants are listed in Table 12-1. Antidepressants are subdivided into groups based on structure or prominent functional activity: selective serotonin reuptake inhibitors (SSRls), tricyclic antidepressants (TCAs), monoamine oxidase inhibitors (MAOls), and other antidepressant compounds with a variety of mechanisms of action. Antidepressants are typically thought to act on either the serotonin or norepinephrine systems, or both. Choice of medications typically depends on diagnosis, history of response (in ...
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