Amaurosis Fugax
Blindness
Leber Congenital Amaurosis
A rare degenerative inherited eye disease that appears at birth or in the first few months of life that results in a loss of vision. Not to be confused with LEBER HEREDITARY OPTIC NEUROPATHY, the disease is thought to be caused by abnormal development of PHOTORECEPTOR CELLS in the RETINA, or by the extremely premature degeneration of retinal cells.
Carotid Artery Diseases
Ischemic Attack, Transient
Brief reversible episodes of focal, nonconvulsive ischemic dysfunction of the brain having a duration of less than 24 hours, and usually less than one hour, caused by transient thrombotic or embolic blood vessel occlusion or stenosis. Events may be classified by arterial distribution, temporal pattern, or etiology (e.g., embolic vs. thrombotic). (From Adams et al., Principles of Neurology, 6th ed, pp814-6)
Retinal Artery Occlusion
Carotid Artery, Internal
Carotid Stenosis
Narrowing or stricture of any part of the CAROTID ARTERIES, most often due to atherosclerotic plaque formation. Ulcerations may form in atherosclerotic plaques and induce THROMBUS formation. Platelet or cholesterol emboli may arise from stenotic carotid lesions and induce a TRANSIENT ISCHEMIC ATTACK; CEREBROVASCULAR ACCIDENT; or temporary blindness (AMAUROSIS FUGAX). (From Adams et al., Principles of Neurology, 6th ed, pp 822-3)
Endarterectomy, Carotid
Endarterectomy
Retinal Artery
Constriction, Pathologic
Remission, Spontaneous
Optic Atrophy, Hereditary, Leber
A maternally linked genetic disorder that presents in mid-life as acute or subacute central vision loss leading to central scotoma and blindness. The disease has been associated with missense mutations in the mtDNA, in genes for Complex I, III, and IV polypeptides, that can act autonomously or in association with each other to cause the disease. (from Online Mendelian Inheritance in Man, http://www.ncbi.nlm.nih.gov/Omim/, MIM#535000 (April 17, 2001))
Cerebrovascular Disorders
Carotid Arteries
Ultrasonography, Doppler, Duplex
Optic Atrophies, Hereditary
Retinal Degeneration
A retrogressive pathological change in the retina, focal or generalized, caused by genetic defects, inflammation, trauma, vascular disease, or aging. Degeneration affecting predominantly the macula lutea of the retina is MACULAR DEGENERATION. (Newell, Ophthalmology: Principles and Concepts, 7th ed, p304)
Rectal Diseases
Postoperative Complications
Retinitis Pigmentosa
Photoreceptor Connecting Cilium
Retinal Dystrophies
Retrospective Studies
Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.
Axillary-to-carotid artery bypass grafting for symptomatic severe common carotid artery occlusive disease. (1/38)
PURPOSE: Revascularization of the internal or external carotid arteries is occasionally indicated for symptomatic atherosclerotic common carotid artery occlusion or long-segment high-grade stenosis beginning at its origin. I report the outcome of axillary artery-based bypass grafts to the distal common, internal, or external carotid arteries. METHODS: Between 1981 and 1997, 29 axillary-to-carotid bypass grafting procedures were performed on 28 patients, 15 men and 13 women, with a mean age of 68 years. Indications were transient ischemia in nine patients, amaurosis fugax in four patients, completed stroke in six patients, and nonlateralizing global ischemia in nine patients. Twenty-three common carotid arteries were totally occluded, and six had long-segment stenosis of 90% or greater beginning at the origin. Saphenous vein grafts were used in 25 procedures, and synthetic grafts were used in four. Grafts were placed to 13 internal, eight distal common, and eight external carotid arteries. RESULTS: There were no perioperative deaths; one stroke occurred (3.4%). No lymphatic or peripheral nerve complications occurred. In a 1- to 11-year follow-up period (mean, 4.5 years), there were no graft occlusions, one restenosis of 50% or greater, and two restenoses of 70% or greater. The 1-year stenosis-free rate for 50% or greater stenosis was 93%, and the 5- and 10-year rates were 87%. No late ipsilateral strokes occurred. The 5- and 10-year survival rates were 64% and 28%, respectively. Coronary artery disease was the major cause of late mortality. CONCLUSION: Axillary-to-carotid bypass grafting for severe symptomatic common carotid occlusive disease is safe, well tolerated, durable, and effective in stroke prevention. There is a high late mortality rate because of coronary artery disease in patients with severe proximal common carotid occlusive disease. (+info)Hemispheric symptoms and carotid plaque echomorphology. (2/38)
PURPOSE: In patients with carotid bifurcation disease, the risk of stroke mainly depends on the severity of the stenosis, the presenting hemispheric symptom, and, as recently suggested, on plaque echodensity. We tested the hypothesis that asymptomatic carotid plaques and plaques of patients who present with different hemispheric symptoms are related to different plaque structure in terms of echodensity and the degree of stenosis. METHODS: Two hundred sixty-four patients with 295 carotid bifurcation plaques (146 symptomatic, 149 asymptomatic) causing more than 50% stenosis were examined with duplex scanning. Thirty-six plaques were associated with amaurosis fugax (AF), 68 plaques were associated with transient ischemic attacks (TIAs), and 42 plaques were associated with stroke. B-mode images were digitized and normalized using linear scaling and two reference points, blood and adventitia. The gray scale median (GSM) of blood was set to 0, and the GSM of the adventitia was set to 190 (gray scale range, black = 0; white = 255). The GSM of the plaque in the normalized image was used as the objective measurement of echodensity. RESULTS: The mean GSM and the mean degree of stenosis, with 95% confidence intervals, for plaques associated with hemispheric symptoms were 13.3 (10.6 to 16) and 80.5 (78.3 to 82.7), respectively; and for asymptomatic plaques, the mean GSM and the mean degree of stenosis were 30.5 (26.2 to 34.7) and 72. 2 (69.8 to 74.5), respectively. Furthermore, in plaques related to AF, the mean GSM and the mean degree of stenosis were 7.4 (1.9 to 12. 9) and 85.6 (82 to 89.2), respectively; in those related to TIA, the mean GSM and the mean degree of stenosis were 14.9 (11.2 to 18.6) and 79.3 (76.1 to 82.4), respectively; and in those related to stroke, the mean GSM and the mean degree of stenosis were 15.8 (10.2 to 21.3) and 78.1 (73.4 to 82.8), respectively. CONCLUSION: Plaques associated with hemispheric symptoms are more hypoechoic and more stenotic than those associated with no symptoms. Plaques associated with AF are more hypoechoic and more stenotic than those associated with TIA or stroke or those without symptoms. Plaques causing TIA and stroke have the same echodensity and the same degree of stenosis. These findings confirm previous suggestions that hypoechoic plaques are more likely to be symptomatic than hyperechoic ones. They support the hypothesis that the pathophysiologic mechanism for AF is different from that for TIA and stroke. (+info)Clinical and pathophysiological features of amaurosis fugax in Japanese stroke patients. (3/38)
OBJECTIVE: It has been emphasized that amaurosis fugax (AmF) is caused by thromboembolism due to atheromatous lesions of the extracranial carotid artery (EC-CA) in Caucasian populations. However, there have been few studies of AmF in Japan. We analyzed the clinical and pathophysiologic features of AmF in 43 Japanese AmF patients. SUBJECTS AND METHODS: Forty-three patients presented with AmF from a group of 2,056 Japanese patients with acute ischemic stroke. We investigated angiographic and transcranial Doppler findings, precipitating factors, medical treatment and prognosis, to elucidate the pathogenetic mechanism of AmF. RESULTS: Angiographic findings revealed an intracranial lesion in 22 patients (51%), extracranial lesion in 16 (37%), and no abnormality in 5 (12%). Blood flow in the ophthalmic artery (OA) examined by the transcranial Doppler ultrasonography (TCD) showed normal antegrade flow in 24 patients and reversed flow in 7. Precipitating factors for AmF were seen in 7 out of 43 patients. Regarding the pathogenesis of AmF, the micro-thromboembolism originated from the internal carotid artery (ICA) in 25 patients, the thromboembolism was via the external carotid artery (ECA) in 7, the hemodynamic retinal vascular insufficiency in 6 patients showed various atheromatous changes in the intracranial carotid artery (IC-CA) or EC-CA, and the cause was unknown in 5. CONCLUSION: In this series of patients, AmF was mainly caused by thromboembolism from IC-CA atheromatous lesions. Micro-thromboemboli from the ECA or hemodynamic retinal vascular insufficiency, although less frequent, should also be considered as possible etiologies for AmF. (+info)Echomorphologic and histopathologic characteristics of unstable carotid plaques. (4/38)
BACKGROUND AND PURPOSE: Our hypothesis was that the carotid plaques associated with retinal and cerebrovascular symptomatology and asymptomatic presentation may be differ from each other. The aim of this study was to identify the sonographic and histopathologic characteristics of plaques that corresponded to these three clinical manifestations. METHODS: The echo process involved duplex preoperative imaging of 71 plaques (67 patients, 21 plaques were associated with retinal, 25 with cerebrovascular symptoms, and 25 were asymptomatic), which was performed in a longitudinal fashion. Appropriate frames were captured and digitized via S-video signal in a computer and digitized sonograms were normalized by two echo-anatomic reference points: the gray scale median (GSM) of the blood and that of the adventitia. The GSM of the plaques was evaluated to distinguish dark (low-GSM) from bright (high-GSM) plaques. Subsequent to endarterectomy, the plaques were sectioned transversely, and a slice at the level of the largest plaque area was examined for the relative size of necrotic core and presence of calcification and hemorrhage. RESULTS: Retinal symptomatology was associated with a hypoechoic plaque appearance (median GSM: 0), asymptomatic status with a hyperechoic plaque appearance (median GSM: 34), and cerebrovascular symptomatology with an intermediate plaque appearance (median GSM: 16) (P = .001). The histopathologic characteristics did not disclose differences between the three clinical groups. The hypoechoic plaque appearance was associated only with the presence of hemorrhage (median GSM for the hemorrhagic plaques, 6, and for the non-hemorrhagic ones, 20 [P = .04]). The relative necrotic core size and the presence of calcification did not show any echomorphologic predilection. CONCLUSION: Our results showed that distinct echomorphologic characteristics of plaques were associated with retinal and cerebrovascular symptomatology and asymptomatic status. Histopathologically, only the presence of hemorrhage proved to have an echomorphologic predilection. (+info)Types of neurovascular symptoms and carotid plaque ultrasonic textural characteristics. (5/38)
The aim of this study was to identify the echo morphology and stenosis of carotid plaques that corresponded to ipsilateral asymptomatic status, amaurosis fugax, hemispheric transient ischemic attack, and stroke. One hundred ninety-two plaques (150 patients), producing stenosis in the range of 50% to 99% and associated with various neurovascular manifestations, were studied. These plaques were imaged on duplex scans, and a series of textural features was produced in a computer to distinguish quantitatively their various echo patterns. Amaurosis fugax corresponded to dark, severely stenosed atheromas (90%); hemispheric transient ischemic attack and stroke corresponded to plaques with intermediate echoic characteristics and intermediate stenosis (80%); and asymptomatic status corresponded to bright, moderately stenosed plaques (70%; P < .05). The significance of these findings is discussed. (+info)Delayed onset of amaurosis fugax in a patient with type A aortic dissection post surgical repair. (6/38)
Stroke is an important complication for the surgical treatment of type A aortic dissection and it occurs immediately post operation. Many surgical techniques such as deep hypothermic circulatory arrest and retrograde cerebral perfusion have been reported to ameliorate this complication. We report here a male Taiwanese patient with type A aortic dissection involving the arch who underwent surgical repair. Amaurosis fugax appeared on the 4th day post operation. Funduscopic findings demonstrated multi focal embolization and carotid sonography revealed normal carotid arteries. The symptoms and signs improved after anticoagulation therapy. This is a rare case of delayed onset of amaurosis fugax in a patient with type A aortic dissection post surgical repair. The thromboemboli might have originated from the internal surface of the sawing area. (+info)Reoperation for recurrent carotid stenosis: early results and late outcome in 199 patients. (7/38)
PURPOSE: This study was undertaken to determine the safety and efficacy of reoperations for recurrent carotid stenosis (REDOCEA) at the Cleveland Clinic. MATERIALS AND METHODS: From 1989 to 1999, 206 consecutive REDOCEAs were performed in 199 patients (131 men, 68 women) with a mean age of 68 years (median, 69 years; range, 47-86 years). A total of 119 procedures (57%) were performed for severe asymptomatic stenosis, 55 (27%) for hemispheric transient ischemic attacks or amaurosis fugax, 26 (13%) for prior stroke, and 6 (3%) for vertebrobasilar symptoms. Eleven REDOCEAs (5%) were combined with myocardial revascularization, and another 19 (9%) represented multiple carotid reoperations (17 second reoperations and 2 third reoperations). Three REDOCEAs (1%) were closed primarily, and nine (4%) required interposition grafts, whereas the remaining 194 (95%) were repaired with either vein patch angioplasty (139 [68%]) or synthetic patches (55 [27%]). Three patients (2%) were lost to follow-up, but late information was available for 196 patients (203 operations) at a mean interval of 4.3 years (median, 3.9 years; maximum, 10.2 years). RESULTS: Considering all 206 procedures, there were 7 early (< 30 days) postoperative neurologic events (3.4%), including 6 perioperative strokes (2.9%) and 1 occipital hemorrhage (0.5%) on the 12th postoperative day. Seventeen additional neurologic events occurred during the late follow-up period, consisting of eight strokes (3.9%) and nine transient ischemic attacks (4.4 %). With the Kaplan-Meier method, the estimated 5-year freedom from stroke was 92% (95% CI, 88%-96%). There were two early postoperative deaths (1%), both from cardiac complications after REDOCEAs combined with myocardial revascularization procedures. With the Kaplan-Meier method, the estimated 5-year survival was 81% (range, 75%-88%). A univariate Cox regression model yielded the presence of coronary artery disease as the only variable that was significantly associated with survival (P =.024). The presence of pulmonary disease (P =.036), diabetes (P =.01), and advancing age (P =.006) was found to be significantly associated with stroke after REDOCEA. Causes of 53 late deaths were cardiovascular problems in 25 patients (47%), unknown in 14 (26%), renal failure in 4 (8%), stroke in 3 (6%), and miscellaneous in 7 (13%). CONCLUSIONS: We conclude that REDOCEA may be safely performed in selected patients with recurrent carotid stenosis and that most of these patients enjoy long-term freedom from stroke. (+info)Clinical features of transient monocular blindness and the likelihood of atherosclerotic lesions of the internal carotid artery. (8/38)
To assess which features of transient monocular blindness (TMB) are associated with atherosclerotic changes in the ipsilateral internal carotid artery (ICA), 337 patients with sudden, transient monocular loss of vision were prospectively studied. History characteristics of the attack were compared with the presence of atherosclerotic lesions of the ipsilateral ICA. All patients were directly interviewed by a single investigator. Of all patients, 159 had a normal ICA on the relevant side, 33 had a stenosis between 0%-69%, 100 had a stenosis of 70%-99%, and 45 had an ICA occlusion. An altitudinal onset or disappearance of symptoms was associated with atherosclerotic lesions of the ipsilateral ICA. A severe (70%-99%) stenosis was also associated with a duration between 1 and 10 minutes, and with a speed of onset in seconds. An ICA occlusion was associated with attacks being provoked by bright light, an altitudinal onset, and the occurrence of more than 10 attacks. Patients who could not remember details about the mode of onset, disappearance, or duration of the attack were likely to have a normal ICA. Our findings may facilitate the clinical decision whether or not to perform ancillary investigations in these patients. (+info)
Clinical features of transient monocular blindness and the likelihood of atherosclerotic lesions of the internal carotid artery...
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Amaurosis fugax
The Amaurosis Fugax Study Group (February 1990). "Current management of amaurosis fugax. The Amaurosis Fugax Study Group". ... Amaurosis fugax (Greek amaurosis meaning darkening, dark, or obscure, Latin fugax meaning fleeting) is a painless temporary ... the causes of amaurosis fugax were better refined by the Amaurosis Fugax Study Group, which has defined five distinct classes ... leading to decreased blood flow manifesting as amaurosis fugax. Commonly, amaurosis fugax caused by giant cell arteritis may be ...
Ocular ischemic syndrome
Amaurosis fugax is a form of acute vision loss caused by reduced blood flow to the eye; it may be a warning sign of an ... Kaiboriboon K, Piriyawat P, Selhorst JB (May 2001). "Light-induced amaurosis fugax". American Journal of Ophthalmology. 131 (5 ...
Amaurosis
It was first described by Theodore Leber in the 19th century.[citation needed] Amaurosis fugax (Latin: fugax meaning fleeting) ... "Hersh: What causes amaurosis fugax, a temporary loss of vision?", metrowestdailynews.com, Dec 4, 2012 v t e (All articles with ... Quinidine toxicity can lead to cinchonism and also to quinine amaurosis.[citation needed] Those experiencing amaurosis are ... Amaurosis (Greek meaning darkening, dark, or obscure) is vision loss or weakness that occurs without an apparent lesion ...
List of medical symptoms
... pelvic pain vaginal discharge Ocular amaurosis fugax (G45.3) and amaurosis blurred vision Dalrymple's sign double vision (H53.2 ... It is rare that a person would visit a doctor and complain as follows: "Doctor, I have amaurosis fugax." They are more likely ... odynophagia proctalgia fugax pyrosis (R12) Rectal tenesmus steatorrhea vomiting (R11) Integumentary Hair: alopecia hirsutism ...
Multiple sclerosis
His symptoms began at age 28 with a sudden transient visual loss (amaurosis fugax) after the funeral of a friend. During his ...
Visual impairment
In addition to the various permanent conditions, fleeting temporary vision impairment, amaurosis fugax, may occur, and may ... known as Leber's congenital amaurosis or LCA. Leber's Congenital Amaurosis damages the light receptors in the retina and ... Leber congenital amaurosis can cause total blindness or severe sight loss from birth or early childhood. Retinitis pigmentosa ... May 2008). "Effect of gene therapy on visual function in Leber's congenital amaurosis" (PDF). The New England Journal of ...
Cholesterol embolism
... known as amaurosis fugax). Emboli to the eye can be seen by ophthalmoscopy and are known as plaques of Hollenhorst. Emboli to ...
Transient ischemic attack
Symptoms such as unilateral weakness, amaurosis fugax, and double vision have higher odds of representing TIA compared to ... Amaurosis fugax (painless, temporary loss of vision) One-sided facial droop One-sided motor weakness Diplopia (double vision) ...
Ophthalmic artery
Amaurosis fugax is a temporary loss of vision that occurs in two conditions which cause a temporary reduction in ophthalmic ... "Blood pressure and pressure amaurosis." Investigative Ophthalmology and Visual Science 1975 Mar;14(3):237-40. PMID 1116922 A ...
Central retinal artery occlusion
Central retinal vein occlusion Branch retinal artery occlusion Branch retinal vein occlusion Amaurosis fugax Ocular ischemic ...
List of MeSH codes (C23)
... amaurosis fugax MeSH C23.888.592.763.941.162.250 - blindness, cortical MeSH C23.888.592.763.941.256 - color vision defects MeSH ...
List of neurological conditions and disorders
Alien hand syndrome Allan-Herndon-Dudley syndrome Alternating hemiplegia of childhood Alzheimer's disease Amaurosis fugax ...
Temporary blindness
... , a type of non-permanent vision loss, may refer to: Amaurosis fugax, or fleeting blindness Conversion ...
List of MeSH codes (C10)
... amaurosis fugax MeSH C10.597.751.941.162.250 - blindness, cortical MeSH C10.597.751.941.256 - color vision defects MeSH C10.597 ...
Eclampsia
... either temporary due to amaurosis fugax or potentially permanent due to retinal detachment), or cortical blindness, which ...
Carotid artery stenosis
... amaurosis fugax) in one eye. Less common symptoms are artery sounds (bruits), or ringing in the ears (tinnitus). In ...
Polycythemia
... amaurosis fugax), other signs of a transient ischemic attack (TIA) or stroke Dizziness, fatigue Unusual bleeding, nosebleeds ...
List of MeSH codes (C11)
... amaurosis fugax MeSH C11.966.075.250 - blindness, cortical MeSH C11.966.075.500 - hemianopsia MeSH C11.966.256 - color vision ...
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The 2014 Ju-Jitsu World Championship were the 12th edition of the Ju-Jitsu World Championships, and were held in Paris, France from November 28 to November 30, 2014. 28.11.2014 - Men's and Women's Fighting System, Men's and Women's Jiu-Jitsu (ne-waza), Men's Duo System - Classic 29.11.2014 - Men's and Women's Fighting System, Men's and Women's Jiu-Jitsu (ne-waza), Women's Duo System - Classic 30.11.2014 - Men's Jiu-Jitsu (ne-waza), Mixed Duo System - Classic, Team event Vincent MATCZAK (2014-09-30). "4TH INVITAION TO WORLD CHAMPIONSHIP 2014" (PDF). Retrieved 2019-11-28.[dead link] Online results Official results (PDF) Mixed team event results (PDF) (All articles with dead external links, Articles with dead external links from April 2022, Ju-Jitsu World Championships, 2014 in French sport ...
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Maria Margaret Pollen
Maria Margaret La Primaudaye Pollen (10 April 1838 - c. 1919), known as Minnie, was a decorative arts collector. As Mrs John Hungerford Pollen, she became known during the early-twentieth century as an authority on the history of textiles, publishing Seven Centuries of Lace in 1908. Maria Margaret La Primaudaye was born into a Huguenot family on 10 April 1838, the third child of the Revd Charles John La Primaudaye, a descendant of Pierre de La Primaudaye. She was educated in Italy. Her family converted to Catholicism in 1851, and it was in Rome that her father met another recent English convert, John Hungerford Pollen, previously an Anglican priest and a decorative artist. She became engaged to Pollen, who was then seventeen years her senior, in the summer of 1854, and was married in the church of Woodchester monastery, near Stroud, Gloucester, on 18 September 1855. The Pollens initially settled in Dublin, where John Hungerford Pollen had been offered the professorship of fine arts at the ...
Ronald Fogleman
Ronald Robert Fogleman (born January 27, 1942) is a retired United States Air Force general who served as the 15th Chief of Staff of the Air Force from 1994 to 1997 and as Commanding General of the United States Transportation Command from 1992 to 1994. A 1963 graduate from the United States Air Force Academy, he holds a master's degree in military history and political science from Duke University. A command pilot and a parachutist, he amassed more than 6,800 flying hours in fighter, transport, tanker and rotary wing aircraft. He flew 315 combat missions and logged 806 hours of combat flying in fighter aircraft. Eighty of his missions during the Vietnam War were as a "Misty FAC" in the F-100F Super Sabre at Phù Cát Air Base, South Vietnam between 25 December 1968 and 23 April 1969. Fogleman was shot down in Vietnam in 1968, while piloting an F-100. He was rescued by clinging to an AH-1 Cobra attack helicopter that landed at the crash site. In early assignments he instructed student pilots, ...
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Peachtree Street" is a 1950 song co-written and recorded by Frank Sinatra in a duet with Rosemary Clooney. The song was released as a Columbia Records single. Frank Sinatra co-wrote the song with Leni Mason and Jimmy Saunders. Mason composed the music while Sinatra and Saunders wrote the lyrics. The song was arranged by George Siravo The song was released as an A side Columbia 10" 78 single, Catalog Number 38853, Matrix Number CO-43100-1 and as a 7" 33, 1-669. The B side was the re-issued "This Is the Night." Neither of the songs charted. The subject of the song is a stroll down the street in Atlanta, Georgia of the same name. Sinatra originally intended Dinah Shore to sing the duet with him. When Shore declined, Clooney was asked. The song was recorded on April 8, 1950. The song features spoken asides by Sinatra and Clooney. Rosemary Clooney asks: "Say, Frank, you wanna take a walk?" Frank Sinatra replies: "Sure, sweetie, just pick a street." He noted how there were no peach trees on the ...
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At least 33[failed verification] people were killed by a fuel tanker explosion in Tleil, Akkar District, Lebanon on 15 August 2021. The disaster was reportedly exacerbated by the ongoing Lebanese liquidity crisis; in which the Lebanese pound has plummeted and fuel has been in short supply. The survivors were evacuated by the Lebanese Red Cross. An investigation is underway. The fuel tanker had been confiscated by the Lebanese Armed Forces from black marketeers, the fuel was then distributed/taken by the locals. The son of the man whose land the fuel tanker was located on, was later arrested, accused of deliberately causing the explosion. Agencies (2021-08-15). "At least 20 killed and 79 injured in fuel tank explosion in Lebanon". the Guardian. Retrieved 2021-08-15. "Lebanon fuel explosion kills 22 and injures dozens more". The Independent. 2021-08-15. Archived from the original on 2021-08-15. Retrieved 2021-08-15. "Lebanon: At least 20 dead and dozens injured after fuel tank explodes as ...
Amaurosis fugax: MedlinePlus Medical Encyclopedia
Amaurosis fugax is a temporary loss of vision in one or both eyes due to a lack of blood flow to the retina. The retina is the ... Treatment of amaurosis fugax depends on its cause. When amaurosis fugax is due to a blood clot or plaque, the concern is to ... Amaurosis fugax is not itself a disease. Instead, it is a sign of other disorders. Amaurosis fugax can occur from different ... Amaurosis fugax is a temporary loss of vision in one or both eyes due to a lack of blood flow to the retina. The retina is the ...
Transient Vision Loss (TVL) and Amaurosis Fugax: Overview, Pathophysiology, Etiology
encoded search term (Transient Vision Loss (TVL) and Amaurosis Fugax) and Transient Vision Loss (TVL) and Amaurosis Fugax What ... Amaurosis fugax and ocular infarction in adolescents and young adults. Ann Neurol. 1989 Jul. 26(1):69-77. [QxMD MEDLINE Link]. ... Transient Vision Loss (TVL) and Amaurosis Fugax. Updated: Dec 22, 2022 * Author: Donny W Suh, MD, MBA, FAAP, FACS; Chief Editor ... Amaurosis fugax and a visual TIA are similar in several respects: Both are of sudden onset, last 2-30 minutes, and resolve ...
Amaurosis fugax - Defining Anything
amaurosis fugax. amaurosis fugax amaurosis fu·gax (fōōgāks, fyōō-). n.. a temporary blindness that may result from transient ... Disclaimer: Amaurosis fugax definition / meaning should not be considered complete, up to date, and is not intended to be used ... historical examples amaurotic (amaurosis, blindness) family idiocy is hereditary, and the child dies at about two years of age ...
Transient Vision Loss (TVL) and Amaurosis Fugax: Overview, Pathophysiology, Etiology
encoded search term (Transient Vision Loss (TVL) and Amaurosis Fugax) and Transient Vision Loss (TVL) and Amaurosis Fugax What ... Amaurosis fugax and ocular infarction in adolescents and young adults. Ann Neurol. 1989 Jul. 26(1):69-77. [QxMD MEDLINE Link]. ... Transient Vision Loss (TVL) and Amaurosis Fugax. Updated: Dec 22, 2022 * Author: Donny W Suh, MD, MBA, FAAP, FACS; Chief Editor ... Amaurosis fugax and a visual TIA are similar in several respects: Both are of sudden onset, last 2-30 minutes, and resolve ...
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N2 - A twenty-four-year-old woman with Takayasus disease was admitted in June 1992 to the hospital because of amaurosis fugax ... AB - A twenty-four-year-old woman with Takayasus disease was admitted in June 1992 to the hospital because of amaurosis fugax ... A twenty-four-year-old woman with Takayasus disease was admitted in June 1992 to the hospital because of amaurosis fugax. ... "A twenty-four-year-old woman with Takayasus disease was admitted in June 1992 to the hospital because of amaurosis fugax. ...
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Amaurosis Fugax - Preferred Concept UI. M0328094. Scope note. Transient complete or partial monocular blindness due to retinal ... Amaurosis Fugax Entry term(s). Blindness, Monocular, Transient Blindness, Transient Monocular Monocular Blindness, Transient ... Amaurosis Fugax Spanish from Spain Descriptor. amaurosis fugaz. Entry term(s). ceguera monocular transitoria ceguera ...
DeCS - New terms
Carotid1
- amaurosis fugax amaurosis fu·gax (fōō'gāks', fyōō'-) n. a temporary blindness that may result from transient ischemia caused by an insufficiency of the carotid artery or exposure to centrifugal force. (definithing.com)
Transient vision loss2
- Some authors refer to ischemic transient vision loss as amaurosis fugax syndrome, so transient vision loss can be a symptom of a serious vision or life-threatening condition, requiring urgent investigation and treatment, or it may have a more benign origin (eg, migraine, dry eye). (medscape.com)
- Gaze-evoked amaurosis (compression) is transient vision loss occurring when looking in a particular direction. (medscape.com)
Ocular1
- Amaurosis fugax in ocular vascular occlusive disorders: prevalence and pathogeneses. (medscape.com)
Retina4
- Amaurosis fugax is a temporary loss of vision in one or both eyes due to a lack of blood flow to the retina . (medlineplus.gov)
- Puede estar causada por EMBOLIA procedente de la ARTERIA CARÓTIDA (por lo general asociada con ESTENOSIS CAROTÍDEA) y de otras localizaciones que entran en la ARTERIA CENTRAL DE LA RETINA. (bvsalud.org)
- In case of Leber Congenital Amaurosis, rods and cones do not develop properly and thereby retina ceases to function effectively causing visual impairment. (targetwoman.com)
- There are 14 types of genes that are associated with Leber Congenital Amaurosis and mutations in any of these genes hinder the development and effective function of the retina. (targetwoman.com)
Stroke4
- When amaurosis fugax is due to a blood clot or plaque, the concern is to prevent a stroke. (medlineplus.gov)
- Amaurosis fugax increases your risk for stroke. (medlineplus.gov)
- But double vision or temporary loss of vision that feels like a shade being drawn over your eye (called amaurosis fugax) can also be a sign of a stroke or TIA about to happen. (sharecare.com)
- While the majority experiencing amaurosis have a complete symptom abeyance within a few minutes, there is a minority who experience a stroke or a vision loss as a result of amaurosis. (targetwoman.com)
Depends2
- Treatment of amaurosis fugax depends on its cause. (medlineplus.gov)
- Treatment of amaurosis depends mainly upon identifying the source of blood clots and cholesterol that have caused this block in the artery. (targetwoman.com)
Temporary1
- Amaurosis fugax is a temporary loss of vision in one eye. (targetwoman.com)
Occur1
- Amaurosis fugax can occur from different causes. (medlineplus.gov)
Loss of vis1
- Leber Congenital Amaurosis or LCA is an inherited retinal degenerative disease that causes severe loss of vision in infants. (targetwoman.com)
Treatment1
- No effective treatment is currently available for Leber Congenital Amaurosis. (targetwoman.com)
Definition1
- Disclaimer: Amaurosis fugax definition / meaning should not be considered complete, up to date, and is not intended to be used in place of a visit, consultation, or advice of a legal, medical, or any other professional. (definithing.com)
Risk1
- Amaurosis fugax: Risk factors include presence of bone marrow toxicity and tubular formations. (mylifestyle.us)
Term1
- The term Amaurosis is taken from Greek meaning dark or obscure. (targetwoman.com)
Monocular3
- Subsequent neurology reports concurred with this monocular designation, 2 and it was re-emphasised in the recommendations of the Amaurosis Fugax Study Group. (avehjournal.org)
- The symptoms of amaurosis fugax (transient monocular loss of vision) indicate not only atherosclerotic disease of the ocular or extra cranial cerebral vessels, but also that of the systemic circulation. (elitmed.hu)
- 1. What should be done for a patient with monocular vision loss, not due to nonarteritic anterior ischemic optic neuropathy, arteritic anterior ischemic optic neuropathy, or other ophthalmologic disease, but with branch or central retinal artery occlusions or amaurosis fugax? (stanford.edu)
Retinal4
- Vision loss with negative scotoma may be seen with amaurosis fugax or retinal transient ischemic attack. (medscape.com)
- Central retinal artery occlusion is inextricably linked to embolic disease, of which amaurosis fugax is a classic symptom. (scientificarchives.com)
- Anterior ischemic optic neuropathy (AION), amaurosis fugax and retinal Tubacin inhibition vessels occlusion are the most often ophthalmic symptoms in this disorder. (health-media.net)
- The incidence of ophthalmic complications in diagnostic angiography is extremely low, and differential diagnosis should always include contrast induced transient cortical blindness, retinal artery occlusion, amaurosis fugax and hysterical blindness [1]. (biomedres.us)
Carotid3
- The first example most likely represents classic migraine with visual aura, the second amaurosis fugax (AF) associated with carotid artery occlusive disease (CAOD), and the third is probably related to late-onset acephalgic migraine. (avehjournal.org)
- 1. Carotid system involvement: Amaurosis fugax, homonymous hemianopsia, unilateral weakness, unilateral numbness or tingling. (berea.edu)
- Depending on the extent of stenosis, ischemia in the carotid perfusion territory can result in amaurosis fugax , transient ischemic attack ( TIA ), or stroke . (amboss.com)
Occlusion1
- Central artery occlusion is one cause of amaurosis fugax. (benwhite.com)
Symptom2
- Some authors refer to ischemic transient vision loss as amaurosis fugax syndrome, so transient vision loss can be a symptom of a serious vision or life-threatening condition, requiring urgent investigation and treatment, or it may have a more benign origin (eg, migraine, dry eye). (medscape.com)
- This symptom is called amaurosis fugax (from the Greek amaurosis , meaning dark, and the Latin fugax , meaning fleeting). (healthyeatslife.com)
Ischemic2
- In a 12-member, 3-generation kindred with conjoint inheritance of G1691A factor V Leiden (FVL) and G20210A prothrombin gene (PTG) mutations, identified through a proband with amaurosis fugax and his father with nonarteritic ischemic optic neuropathy (NAION), the authors' hypothesis was that ocular thrombosis was a diagnostic window to familial thrombophilia-thrombosis. (nih.gov)
- The 39-year-old white male proband, with amaurosis fugax and transient ischemic attacks (TIA), was found to be a compound heterozygote for FVL and PTG mutations. (nih.gov)
Blindness2
- Instead of complete blindness, many people suffer blurring or dimming of vision (usually in one eye) like a shade or veil slowly descending across their field of vision (the medical term for this is amaurosis fugax ). (medbroadcast.com)
- Their lack of blood supply can cause permanent damage, including amaurosis fugax, or blindness. (vapevep.com)
Patients2
- According to the mortality data, patients presenting with symptoms suggestive of amaurosis fugax will die due to heart disease, primarily myocardial infarct. (elitmed.hu)
- Therapy of the whole disease and not just the symptoms will influence life expectancy in patients with amaurosis fugax. (elitmed.hu)
Ocular1
- The differential diagnosis of amaurosis fugax is extensive and can be divided into circulatory, ocular, and neurologic etiologies (Table 1 ). (thoracickey.com)
Greek1
- Amaurosis is the Greek word for darkening, dark, or obscure. (rxlist.com)
Headache2
- Rarely, a patient harboring a cavernous angioma may describe gaze-evoked amaurosis fugax or headache. (medscape.com)
- A 49-year-old woman presented to her primary care physician after a 5-minute episode of left amaurosis fugax, followed by a headache. (thoracickey.com)
Cerebrovascular1
- At 10-year follow-up, the cumulative incidence of cerebrovascular events was similar between both groups, though the GCA group had a higher incidence of amaurosis fugax compared with controls. (neurologyadvisor.com)
Vision4
- Amaurosis fugax therefore means a fleeting loss of vision. (rxlist.com)
- Amaurosis fugax is a temporary loss of vision in one or both eyes due to a lack of blood flow to the retina . (medlineplus.gov)
- A 60-year-old girl who had experienced two shows of amaurosis fugax in her best eye offered vision reduction. (lcl-161.com)
- A 60-year-old woman who had experienced two episodes of amaurosis fugax in her right eye presented with vision loss. (nih.gov)
Central1
- Puede estar causada por EMBOLIA procedente de la ARTERIA CARÓTIDA (por lo general asociada con ESTENOSIS CAROTÍDEA) y de otras localizaciones que entran en la ARTERIA CENTRAL DE LA RETINA. (bvsalud.org)
Brain1
- The prognostic significance of amaurosis fugax developing after the age of 50-55 years is similar to that of transient ischaemic attack of the brain: it could be a harbinger of atherosclerotic disease of the extra- and intracranial brain vessels as well as the coronary arteries. (elitmed.hu)
Medical1
- PubMed is a searchable database of medical literature and lists journal articles that discuss Amaurosis fugax. (rarerheumatologynews.com)