Amantadine: An antiviral that is used in the prophylactic or symptomatic treatment of influenza A. It is also used as an antiparkinsonian agent, to treat extrapyramidal reactions, and for postherpetic neuralgia. The mechanisms of its effects in movement disorders are not well understood but probably reflect an increase in synthesis and release of dopamine, with perhaps some inhibition of dopamine uptake.Rimantadine: An RNA synthesis inhibitor that is used as an antiviral agent in the prophylaxis and treatment of influenza.Adamantane: A tricyclo bridged hydrocarbon.Antiviral Agents: Agents used in the prophylaxis or therapy of VIRUS DISEASES. Some of the ways they may act include preventing viral replication by inhibiting viral DNA polymerase; binding to specific cell-surface receptors and inhibiting viral penetration or uncoating; inhibiting viral protein synthesis; or blocking late stages of virus assembly.Dyskinesias: Abnormal involuntary movements which primarily affect the extremities, trunk, or jaw that occur as a manifestation of an underlying disease process. Conditions which feature recurrent or persistent episodes of dyskinesia as a primary manifestation of disease may be referred to as dyskinesia syndromes (see MOVEMENT DISORDERS). Dyskinesias are also a relatively common manifestation of BASAL GANGLIA DISEASES.Antiparkinson Agents: Agents used in the treatment of Parkinson's disease. The most commonly used drugs act on the dopaminergic system in the striatum and basal ganglia or are centrally acting muscarinic antagonists.Influenza A virus: The type species of the genus INFLUENZAVIRUS A that causes influenza and other diseases in humans and animals. Antigenic variation occurs frequently between strains, allowing classification into subtypes and variants. Transmission is usually by aerosol (human and most non-aquatic hosts) or waterborne (ducks). Infected birds shed the virus in their saliva, nasal secretions, and feces.Viral Matrix Proteins: Proteins associated with the inner surface of the lipid bilayer of the viral envelope. These proteins have been implicated in control of viral transcription and may possibly serve as the "glue" that binds the nucleocapsid to the appropriate membrane site during viral budding from the host cell.Dopamine Agents: Any drugs that are used for their effects on dopamine receptors, on the life cycle of dopamine, or on the survival of dopaminergic neurons.Oseltamivir: An acetamido cyclohexene that is a structural homolog of SIALIC ACID and inhibits NEURAMINIDASE.Coma, Post-Head Injury: Prolonged unconsciousness from which the individual cannot be aroused, associated with traumatic injuries to the BRAIN. This may be defined as unconsciousness persisting for 6 hours or longer. Coma results from injury to both cerebral hemispheres or the RETICULAR FORMATION of the BRAIN STEM. Contributing mechanisms include DIFFUSE AXONAL INJURY and BRAIN EDEMA. (From J Neurotrauma 1997 Oct;14(10):699-713)Influenza, Human: An acute viral infection in humans involving the respiratory tract. It is marked by inflammation of the NASAL MUCOSA; the PHARYNX; and conjunctiva, and by headache and severe, often generalized, myalgia.Zanamivir: A guanido-neuraminic acid that is used to inhibit NEURAMINIDASE.

Studies on the mechanism of action of amantadine. (1/382)

1 The effect of amantadine hydrochloride on various aspects of catecholamine metabolism in the rat brain has been investigated. 2 Amantadine failed to have any significant effect on brain concentrations of dopamine or noradrenaline even when administered daily for 9 days. 3 Amantadine had no effect on the rate of decline of noradrenaline and dopamine concentrations after alpha-methyl-p-tyrosine. 4 In vitro amantadine inhibited dopamine uptake into synaptosomes only at high concentrations, and caused little release of dopamine from synaptosomes. 5 There is no evidence from these results to suggest that the anti-Parkinsonian effect of amantadine is related to an action on dopaminergic mechanisms.  (+info)

An Advisory Committee Statement (ACS). National Advisory Committee on Immunization (NACI). Statement on influenza vaccination for the 1998-1999 season. (2/382)

The National Advisory Committee on Immunization (NACI) provides Health Canada with ongoing and timely medical, scientific, and public-health advice relating to immunization. Health Canada acknowledges that the advice and recommendations set out in this statement are based upon the best current available scientific knowledge, and is disseminating this document for information purposes. Persons administering or using the vaccine should also be aware of the contents of the relevant product monograph(s). Recommendations for use and other information set out herein may differ from that set out in the monograph(s) of the Canadian licensed manufacturer(s) of the vaccine(s). Manufacturer(s) have only sought approval of the vaccine(s) and provided evidence as to its safety and efficacy when used in accordance with the product monographs.  (+info)

Prevention and control of influenza: recommendations of the Advisory Committee on Immunization Practices (ACIP). (3/382)

This report updates 1998 recommendations by the Advisory Committee on Immunization Practices (ACIP) on the use of influenza vaccine and antiviral agents (MMWR 1998;47[No. RR-6]:1-26). The principal changes include a) information on the influenza virus strains included in the 1999-2000 trivalent vaccine; b) discussion of the potential expanded use of influenza vaccine; c) new background information on live-attenuated influenza vaccines (LAIVs), neuraminidase-inhibitor drugs, and rapid diagnostic tests; d) new information on the epidemiology of influenza among travelers; and e) the addition of referenced citations. This report and other information on influenza can be accessed at the website for the Influenza Branch, Division of Viral and Rickettsial Diseases, National Center for Infectious Diseases, CDC at .  (+info)

Tetraethylammonium and amantadine identify distinct organic cation transporters in rat renal cortical proximal and distal tubules. (4/382)

Tetraethylammonium (TEA) and amantadine are two organic cations that are secreted by the kidney. It appears that each cation may characterize distinct renal tubule organic cation transport pathways. To test this hypothesis, we investigated the renal proximal and distal tubule energy-dependent transport properties of TEA and amantadine. Isolated tubules were incubated at 25 degrees C in bicarbonate buffer (Krebs-Henseleit solution) and nonbicarbonate buffer (Cross-Taggart) with varying concentrations of [14C]TEA or [3H]amantadine to determine initial rates of energy-dependent uptake of TEA and amantadine, respectively. The uptake of TEA could best be described by two transport sites, a high-affinity site and a lower affinity site. TEA uptake was not influenced by the presence of bicarbonate. Consistent with our previously reported data, amantadine uptake could also be described by two transport sites, a high-affinity-capacity site that is bicarbonate-dependent and a lower-affinity-capacity transport site that is bicarbonate-independent. The renal tubule uptake of amantadine into proximal and distal tubules, in Krebs-Henseleit solution or Cross-Taggart buffers, was not inhibited by 10 to 1000 microM of TEA. However, tubule accumulation of TEA could be inhibited (>90%) by amantadine in proximal and distal tubules in Krebs-Henseleit solution and Cross-Taggart buffers. In proximal tubules, N1-methylnicotinamide was not able to inhibit amantadine uptake but it reduced TEA uptake by 60 to 70% at similar concentrations. These data support the existence of multiple renal tubule organic cation transporters that have different substrate affinity and controlling mechanisms. It is also apparent that amantadine characterizes organic cation transporters that are distinct from those characterized by TEA.  (+info)

In vivo analysis of amantadine renal clearance in the uninephrectomized rat: functional significance of in vitro bicarbonate-dependent amantadine renal tubule transport. (5/382)

Amantadine transport into renal proximal and distal tubules is bicarbonate dependent. In the present study, we addressed the effects of bicarbonate on renal clearance and urinary excretion of amantadine. Renal clearance of kynurenic acid was also studied to determine whether bicarbonate effects are specific for organic base transport by the kidney. After a moderate diuresis was established, animals received i.v. [(3)H]amantadine or [(3)H]kynurenic acid followed by an acute dose of sodium bicarbonate or physiological saline. Urine and blood samples were analyzed for [(3)H]amantadine or [(3)H]kynurenic acid, blood gases, and pH. Amantadine and kynurenic acid were excreted by the kidneys, and both compounds underwent renal tubular secretion. Amantadine metabolism occurred, and one metabolite was detected in the urine. In the bicarbonate-treated rats, the total amount of amantadine excreted in the urine was decreased, whereas the amount of metabolite recovered was similar in both groups. Bicarbonate treatment caused a sustained increase in blood bicarbonate levels, a mild increase in blood pH, and a decrease in amantadine renal clearance and in the amantadine/creatinine clearance ratio. Only a transient decrease in the renal clearance of kynurenic acid and the kynurenic acid/creatinine clearance ratio was observed. This study demonstrates that short-term changes in bicarbonate concentration may have significant effects on renal organic cation elimination. Coupled with our previous in vitro demonstration of bicarbonate-dependent organic cation transport, the present study suggests that bicarbonate inhibition of renal tubule organic cation secretion may explain the previous observation that bicarbonate dosing decreases amantadine excretion by the kidney.  (+info)

New experimental and clinical data on leukaemia immunotherapy. (6/382)

The present results of our treatment of acute lymphoid leukaemia patients are summarized: 7 out of 20 randomized patients given active immunotherapy after chemoradiotherapy are still in complete remission after periods varying from seven to ten years (compared to none in the control group). The actuarial results on 100 patients show remission and survival curves presenting a plateau between three and five years for a certain percentage, suggesting a possible cure. Several parameters studied in 200 patients indicate that the factors affecting this percentage are age, cytological type, volume of the tumour, and the localization of leukaemic cells in certain areas. Experiments with L1210 leukaemia show that immunotherapy enhances the effect of chemotherapy when administered after chemotherapy but decreases it when administered before, which is in favour of the use of the sequence chemotherapy-immunotherapy clinically.  (+info)

Amantadine and rimantadine have no direct inhibitory effects against hepatitis C viral protease, helicase, ATPase, polymerase, and internal ribosomal entry site-mediated translation. (7/382)

Amantadine, a drug known to inhibit influenza A viral matrix (M2) protein function, was reported to be an effective treatment in some patients with chronic hepatitis C virus (HCV) infection. Sequence comparison shows no homology between M2 and any of the HCV proteins. The effects of amantadine and a related analogue, rimantadine, on viral protease, helicase, ATPase, RNA-dependent RNA polymerase, and HCV internal ribosomal entry site (IRES) translation were tested by established in vitro biochemical assays. No inhibition (>15%) of HCV protease, helicase, ATPase, and polymerase was observed with concentrations up to 400 microgram/mL. IRES-specific inhibition was not observed at clinically relevant concentrations, but both cap and IRES reporter genes were suppressed at higher levels, suggesting nonspecific translation inhibition. In conclusion, amantadine and rimantadine have no direct and specific inhibitory effects against HCV protease, helicase, ATPase, polymerase, and IRES in vitro.  (+info)

Laboratory characteristics of an attenuated influenza type A (H3N2) virus ('Alice' strain). (8/382)

The Alice strain of live attenuated influenza virus was obtained by selection of a gamma inhibitor-resistant strain from a virus recombinant between A/PR/8/34 (HON1) and A/England/42/72 (H3N2). Its behaviour in vitro and in vivo was studied. Three marker systems were investigated: resistance to serum inhibitors, growth capacity at high temperature and low sensitivity to amantadine hydrochloride. In ferrets the strain was found to be attenuated and immunogenic. Passages in man, animals and eggs have not affected its resistance to gamma inhibitors.  (+info)

*Amantadine

... has been associated with several central nervous system (CNS) side effects, likely due to amantadine's dopaminergic ... The mechanisms for amantadine's antiviral and antiparkinsonian effects are unrelated. The mechanism of amantadine's antiviral ... "Amantadine in Parkinson's disease". Cochrane Database of Systematic Reviews. doi:10.1002/14651858.CD003468. "Amantadine - FDA ... dog treats because the product has the potential to be contaminated with amantadine. Tromantadine "SYMMETREL® (amantadine ...

*DMOZ - Health: Pharmacy: Drugs and Medications: A: Amantadine

Features information about dosage, usage, warnings, and side effects. ...

*Brazilian Controlled Drugs and Substances Act

AMANTADINE 4. AMINEPTINE 5. AMISSULPRIDA 6. AMITRIPTILINE 7. AMOXAPINE 8. AZACICLONOL 9. BECLAMIDE 10. BENACTIZIN 11. ...

*List of dopaminergic drugs

Adamantanes: Amantadine • Memantine • Rimantadine Aminotetralins: 7-OH-DPAT • 8-OH-PBZI • Rotigotine • UH-232 Benzazepines: 6- ...

*Multiple sclerosis signs and symptoms

"Amantadine for fatigue in multiple sclerosis". Cochrane Database of Systematic Reviews (1): CD002818. doi:10.1002/14651858. ... There are also different medications used to treat fatigue; such as amantadine, or pemoline; as well as psychological ... CS1 maint: Multiple names: authors list (link) Amantadine. US National Library of Medicine (Medline) (2003-04-01). Retrieved on ...

*Antigenic shift

Aoki, FY; Sitar, DS (January 1988). "Clinical pharmacokinetics of amantadine hydrochloride". Clinical Pharmacokinetics. 14 (1 ...

*Management of multiple sclerosis

A few medications have been studied to treat MS-related fatigue, such as amantadine pemoline (which is a psychostimulant also ... "Amantadine for fatigue in multiple sclerosis". Cochrane Database of Systematic Reviews (1): CD002818. doi:10.1002/14651858. ... CS1 maint: Multiple names: authors list (link) Amantadine. US National Library of Medicine (Medline) (1 April 2003). Retrieved ...

*Benzatropine

... benztropine versus amantadine". Clinical Psychopharmacology. 9 (3): 180-5. doi:10.1097/00004714-198906000-00004. PMID 2661606. ... "A controlled trial of amantadine in drug-induced extrapyramidal disorders". Archives of General Psychiatry. 33 (5): 599-602. ...

*Anorgasmia

Limited data has shown that the drug amantadine may help to relieve SSRI-induced sexual dysfunction. Cyproheptadine, buspirone ... Balogh S, Hendricks SE, Kang J (1992). "Treatment of fluoxetine-induced anorgasmia with amantadine". The Journal of Clinical ... "Amantadine in the treatment of sexual dysfunction associated with selective serotonin reuptake inhibitors". Journal of clinical ...

*Bifemelane

Moryl E, Danysz W, Quack G (June 1993). "Potential antidepressive properties of amantadine, memantine and bifemelane". ...

*Pneumonia

Influenza A may be treated with rimantadine or amantadine, while influenza A or B may be treated with oseltamivir, zanamivir or ... Jefferson, T; Demicheli, V; Di Pietrantonj, C; Rivetti, D (19 April 2006). "Amantadine and rimantadine for influenza A in ... When influenza outbreaks occur, medications such as amantadine or rimantadine may help prevent the condition; however are ... have shown resistance to rimantadine and amantadine. The use of antibiotics in viral pneumonia is recommended by some experts, ...

*Antiviral drug

Amantadine and rimantadine have been introduced to combat influenza. These agents act on penetration and uncoating. Pleconaril ...

*Ritter reaction

... and synthesis of Amantadine, an antiviral and antiparkinsonian drug. Other applications of the Ritter reaction include ...

*Lysosome

... and amantadine. However, high tissue concentrations and long elimination half-lives are explained also by lipophilicity and ... "Therapeutic brain concentration of the NMDA receptor antagonist amantadine". Neuropharmacology. 34 (7): 713-21. doi:10.1016/ ...

*Oseltamivir

"Amantadine, oseltamivir and zanamivir for the treatment of influenza". www.nice.org.uk. NICE. 25 February 2009. Archived from ... Amantadine and rimantadine - M2 inhibitors, other medications used for influenza treatment "Tamiflu - Drugs.com". www.drugs.com ... zanamivir and amantadine in the prevention of influenza: a systematic review". The Journal of infection. 62 (1): 14-25. doi: ...

*Adamantane

The first adamantane derivative used as a drug was amantadine - first (1967) as an antiviral drug against various strains of ... An important nitrogen-substituted drug amantadine can be prepared by reacting adamantane with bromine or nitric acid to give ... Moiseev, I. K.; Doroshenko, R. I.; Ivanova, V. I. (1976). "Synthesis of amantadine via the nitrate of 1-adamantanol". ... Adapalene Adapromine Amantadine Bromantane Memantine Rimantadine Saxagliptin Tromantadine Vildagliptin Adamantane was recently ...

*Levodopa-induced dyskinesia

Amongst pharmacological treatments, N-methyl-D-aspartate (NMDA) antagonist, (a glutamate receptor), amantadine, has been proven ... "Long-term antidyskinetic efficacy of amantadine in Parkinson's Disease". Movement Disorders. 25 (10): 1357-1363. doi:10.1002/ ...

*Memantine

Adapromine Amantadine Bromantane Neramexane Nitromemantine Rimantadine "International brands for memantine". Drugs.com. ... "Involvement of the sigma 1 receptor in the modulation of dopaminergic transmission by amantadine". Eur J Neurosci. 19 (8): 2212 ...

*Viroporin

Amantadine, which was discovered in the 1960s and has been in clinical use against influenza A for some time, is an example of ... Alves Galvão, MG; Rocha Crispino Santos, MA; Alves da Cunha, AJ (21 November 2014). "Amantadine and rimantadine for influenza A ... Oxford, JS; Galbraith, A (1980). "Antiviral activity of amantadine: a review of laboratory and clinical data". Pharmacology & ...

*Influenza

"CDC Recommends against the Use of Amantadine and Rimantadine for the Treatment or Prophylaxis of Influenza in the United States ... Ilyushina NA, Govorkova EA, Webster RG (October 2005). "Detection of amantadine-resistant variants among avian influenza ... The M2 ion channel is blocked by amantadine drugs, preventing infection. These core proteins and vRNA form a complex that is ...

*Apimostinel

Included here are the aminoadamantanes, memantine and amantadine (Sani et al., 2012); the NR2B-selective antagonists, ...

*Tromantadine

Like rimantadine, amantadine, and adapromine, tromantadine is a derivative of adamantane. Tromantadine inhibits the early and ...

*Rimantadine

Both rimantadine and the similar drug amantadine are derivates of adamantane. Rimantadine was approved by the Food and Drug ... August 2008). "Functional studies indicate amantadine binds to the pore of the influenza A virus M2 proton-selective ion ... Rimantadine, like its antiviral cousin amantadine, possesses some NMDA antagonistic properties and is used as an ... antiparkinsonic drug (i.e., in the treatment of Parkinson's disease). However, in general, neither rimantadine nor amantadine ...

*NMDA receptor antagonist

Minocycline Amantadine: used for treating Parkinson's disease and influenza and Alzheimer's. Atomoxetine: a norepinephrine ... Amantadine." MedlinePlus website Accessed May 29, 2007 Ludolph, Andrea G; Udvardi, Patrick T; Schaz, Ulrike; Henes, Carolin; ...

*Musical hallucinations

Although the patient was taking other medications at the same time, the timing of onset and offset suggested that amantadine ... 2010, a seventy-seven-year-old woman with Parkinson's disease (PD) was administered amantadine after a year of various other ... "Transient amantadine‑induced musical hallucinations in a patient with Parkinson's disease". Movement Disorders. Wiley-Blackwell ...
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In an attempt to clarify the effect of amantadine hydrochloride therapy in Parkinsons disease, dopa metabolites were measured in the urine of 15 patients who were taking this medication. The results indicated that patients on amantadine therapy had lower urinary levels of epinephrine plus norepinephrine than either normal individuals or parkinsonian patients not receiving amantadine. Patients who developed livedo reticularis during amantadine therapy showed a small but significant increase in urinary dopamine levels and a similar decrease in dopac levels, when compared to other patients on amantadine who did not develop livedo reticularis.
Amantadine hydrochloride is well absorbed orally. Maximum plasma concentrations are directly related to dose for doses up to 200 mg/day. Doses above 200 mg/day may result in a greater than proportional increase in maximum plasma concentrations. It is primarily excreted unchanged in the urine by glomerular filtration and tubular secretion. Eight metabolites of amantadine have been identified in human urine. One metabolite, an N-acetylated compound, was quantified in human urine and accounted for 5-15% of the administered dose. Plasma acetylamantadine accounted for up to 80% of the concurrent amantadine plasma concentration in 5 of 12 healthy volunteers following the ingestion of a 200 mg dose of amantadine. Acetylamantadine was not detected in the plasma of the remaining seven volunteers. The contribution of this metabolite to efficacy or toxicity is not known. There appears to be a relationship between plasma amantadine concentrations and toxicity. As concentration increases, toxicity seems to ...
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Do not use FluMist nasal influenza "live vaccine" while you are being treated with amantadine and for at least 48 hours after you stop taking amantadine. The nasal vaccine may not be as effective if you receive it while you are taking amantadine. Before taking amantadine, tell your doctor if you have received a nasal flu vaccine within the past 14 days.. Before taking amantadine, tell your doctor if you have epilepsy or other seizure disorder, congestive heart failure, kidney or liver disease, low blood pressure, eczema, glaucoma, or a history of mental illness, suicide attempt, or drug/alcohol addiction.. Amantadine can cause side effects that may impair your thinking, vision, or reactions. Be careful if you drive or do anything that requires you to be alert and able to see clearly. If you are taking amantadine to treat influenza A, take the medication for the entire length of time prescribed by your doctor. Your symptoms may get better before the infection is completely treated. If you are ...
Symmetrel (Amantadine Hydrochloride) is used to treat a variety of conditions. It is often prescribed to treat the symptoms of Parkinsons disease, including shaking, and stiffness. This drug also works as an antiviral, and is used to prevent and treat influenza A.
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Amantadine HCl administration has resulted in accelerated resolution of influenza A illness. Prolonged abnormalities in pulmonary function have been described in uncomplicated influenza A. To study the effect of amantadine on these changes, we evaluated young adults with documented natural influenza A with clear chest examinations and X rays. Subjects received placebo or amantadine in random, doubleblind fashion. Physiologic studies included maximal expiratory flow volume curves with air and helium-oxygen mixtures. Air flow rates were unchanged in all subjects throughout. Initially, both groups showed comparable decreases in mean helium-oxygen maximal expiratory flow rates. The amantadine group showed accelerated physiologic improvement: significant increase in helium-oxygen flow rates occurred within 7 days (P , 0.05). The rate of improvement in the helium-oxygen flow rates in the placebo group was not statistically significant. These studies confirm peripheral airways dysfunction after ...
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Abstract: A double-blind crossover trial of amantadine vs. placebo was carried out involving 42 patients with Parkinsons disease: 64 per cent of the patients on amantadine experienced subjective improvement compared to 21 per cent on placebo. A comprehensive battery of qualitative and quantitative tests was carried out on each patient on entry to the study, after previous standard treatment was discontinued or reduced to a minimal tolerable dose, while on placebo, and while on amantadine, at 3 week intervals. Almost all relevant symptoms and physical signs improved, and the neurologists judged amantadine superior to placebo in 74 per cent of the patients. Quantitative measurement revealed significant improvement in 10 of 19 tests of simulated activities of daily living, in several tests of strength and station, and in all tests of coordination and gait. When the amantadine scores were compared to the placebo scores, an average improvement of 29 per cent occurred in the simulated activities of ...
This is a randomized, placebo-controlled, crossover, 4-sequence, 4-period, double-blind (participants and investigators), multicenter trial of 3 commonly used medications for treatment of MS-related fatigue (amantadine, modafinil, methylphenidate) versus placebo in fatigued subjects with MS defined by McDonald Criteria.. Using a balanced Latin-square crossover design, subjects will be allocated, in a double-blind, randomized fashion, to one of the four treatment sequences (Figure 1): 1) amantadine, placebo, modafinil, methylphenidate; 2) placebo, methylphenidate, amantadine, modafinil; 3) modafinil, amantadine, methylphenidate, placebo; and 4) methylphenidate, modafinil, placebo and amantadine. Each medication will be titrated over four weeks to the participants highest tolerated dose or the pre-defined highest dose. The dosing and titration schedule of the study medications are depicted in Figure 2. Each treatment period will be 6 weeks and there will be a 2-week washout period between each ...
We studied the cardiac effects of amantadine, an antiviral and anti-Parkinson drug. Amantadine hydrochloride (100--800 microM) produced significant changes in the electrophysiological properties of isolated ventricular muscle preparations from frog, rabbit, cat, dog, and calf. At relatively low concentrations (100--300 microM), the drug increased action potential duration, decreased action potential amplitude and maximum diastolic potential, and induced phase 4 depolarization. Amantadine also caused subthreshold diastolic depolarizations, apparent upon cessation of stimulation in all preparations studied. The amplitude of the diastolic depolarizations increased as a function of time and/or concentration of drug, eventually reached threshold, and spontaneous activity ensued. In the steady state, amantadine-induced spontaneous activity was rather stable, and the rate was dependent upon the amantadine and external potassium concentrations, as well as the membrane potential. In the absence of ...
The M2 protein is a 97-residue protein that is made up of 23 amino acids; it is a single-pass membrane protein that has all of the N-terminuss orientated to the outside of the cell. Inside of the cell is a 19 residue transmembrane domain (M2TM) and a 54-residue tail. It is the M2TM, which is the target of the anti-influenza drugs amantadine and rimantadine. The structure of these residues was solved using a combination of X-ray crystallography and NMR techniques; both techniques support a semi-symmetric tetramer proton channel, the section inside the cell membrane coiling in a leftward orientation. The structural determination of this channel differs between these two methods in regards to the bound inhibitor position. Even though both methods suggest similar hot-spots for possible binding sites inside and on the outside of the channel, X-ray crystallography indicates a amantadine binding site in the central pore of the channel whereas NMR indicates four rimantadine binding sites on the surface ...
The clinical efficacy of promising cocaine anti-craving medications was examined in combination with buprenorphine. Twenty-one opioid-dependent cocaine abusers were enrolled in a double-blind, 12-week trial in which they received on a daily basis buprenorphine (8 mg, s.l.) plus either desipramine (150 mg, p.o.), amantadine (300 mg, p.o.), or fluoxetine (60 mg, p.o.). Urine samples and self-reported drug use were obtained 1-3 times/week. The order of greatest patient retention across the 12 weeks was desipramine (83.3%) > amantadine (66.7%) > fluoxetine (20.0%). The desipramine and amantadine groups appeared to have greater increases in opioid- and cocaine-free urines than the fluoxetine group. These results suggest that desipramine and amantadine may facilitate greater opioid and cocaine abstinence than fluoxetine. ...
Amantadine hydrochloride is one of the drugs given at rehabilitation programs to people who suffered Acquired Brain Injury in order to expedite recovery and improve functioning.. A previous study examined the spatially asymmetric allocation of attention in patients with traumatic brain injury (TBI). Patients demonstrated significantly worse performance with leftward than with rightward cross-hemi field shifts of attention. This is reminiscence of neglect patients. This difference was significantly reduced during and following treatment. Our objective is to investigate whether Amantadine Hydrochloride is effective in improving allocation of spatial attention and improving function in people with Traumatic Brain Injury. ...
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A 61-year-old man with Parkinsons disease (PD) developed sudden-onset visual impairment after initiation of amantadine treatment. Ophthalmologic examination revealed corneal endothelial edema. Discontinuation of amantadine resulted in rapid improvem
New Delhi: Drug firm Alembic Pharmaceuticals has received US health regulators nod for Amantadine Hydrochloride capsules used to treat symptoms of infection caused by influenza virus.. "The company has received approval from the United States Food and Drug Administration (USFDA) for its ANDA for Amantadine Hydrochloride capsules USP 100 mg," Alembic Pharmaceuticals said in a BSE filing.. The approved Abbreviated New Drug Application (ANDA) is equivalent to Symmetrel capsules of Endo Pharmaceuticals. ...
Hello everyone, So its been almost 6weeks since Winstons decompression surgery for CM & SM and hes doing ok. Surgery wounds have healed nicely. He had a recheck with the regular neurologist yesterday & I explained to her that hes still scratching his head & rubbing his head in the floor periodically. She did say its not even 6 weeks & it is going to take a long time for him to feel a lot better. She also said she would like to try using Amantadine on him. Its a anti flu drug that theyve
Testing of jerky pet treats from China has also revealed the presence of the drug amantadine in some samples containing chicken. These samples were from jerky pet treats that were sold a year or more ago. Amantadine is an antiviral that is FDA-approved for use in people. It has also been used in an extra-label manner (using an approved drug in a way that isnt listed on the label) in dogs for pain control, but FDA prohibited its use in poultry in 2006.. FDA does not believe that amantadine contributed to the illnesses because the known side effects or adverse events associated with amantadine do not seem to correlate with the symptoms seen in the jerky pet treat-related cases. However, amantadine should not be present at all in jerky pet treats, and the agency has notified the Chinese authorities that the presence of amantadine in these products is an adulterant. Chinese authorities have also assured us that they will perform additional screening and will follow up with jerky pet treat ...
The mechanisms for amantadines antiviral and antiparkinsonian effects are unrelated. The mechanism of amantadines antiviral activity involves interference with the viral protein, M2, a proton channel.[14][15] After entry of the virus into cells via endocytosis, it is localized in acidic vacuoles; the M2 channel functions in transporting protons with the gradient from the vacuolar space into the interior of the virion. Acidification of the interior results in disassociation of ribonucleoproteins, and the initiation of viral replication. Amantadine and rimantadine function in a mechanistically identical fashion in entering the barrel of the tetrameric M2 channel, and blocking pore function (i.e., proton translocation). Resistance to the drug class is a consequence of mutations to the pore-lining residues of the channel, leading to the inability of the sterically bulky adamantane ring that both amantadine and rimantadine share, in entering in their usual way, into the channel.[citation ...
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PURPOSE OF PROJECT: To study the effect of amantadine 100 mg administered twice daily compared to placebo on irritability from baseline to treatment Day 28.. SUMMARY OF PROJECT: It is anticipated that 168 subjects with 168 corresponding subject informants will be recruited for the study. Carolinas Rehabilitation, the lead center, and 5 collaborating centers will enroll approximately 28 subjects each.. Subjects will be recruited primarily from the clinics. Also, letters will be sent to patients in our data base. If the first encounter with research personnel is by telephone, the research assistant will obtain verbal (telephone) consent from the subjects informant for the Neuropsychiatric Inventory (NPI) for subject irritability. The score on this questionnaire must be ≥ 6 for qualification. This allows pre-screening to take place and avoid an unnecessary clinic visit.. Subjects who consent and qualify will be randomized in a 1:1 ratio, amantadine to placebo. Stratification to randomization ...
Well, I have taking amantadine for two weeks now. When I first started taking the drug I think I was going through a flair up and was extremely tired all the time. Immedialtly up taking amantadine I ...
There is a company in the process of bringing time release amantadine to market. Just wondering if anyone taking amantadine needs or wants a time release version. It would be priced at a premium to...
The report generally describes amantadine sulphate, examines its uses, production methods, patents. Amantadine sulphate market situation is overviewed;
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Amantadine can be used for treatment of arthritis pain, neurologic pain (disk disease etc.), pain associated with cancer, especially osteosarcoma and chronic pain associated with declaw surgery.
JS Scott.; J deSchoolmeester.; E Kilgour. Novel Acidic 11β-Hydroxysteroid Dehydrogenase Type 1 (11β-HSD1) Inhibitor with Reduced Acyl Glucuronide Liability: The Discovery of 4-[4-(2-Adamantylcarbamoyl)-5-tert-butyl-pyrazol-1-yl]benzoic Acid (AZD8329). J. Med. Chem.201255 (22), 10136-10147.. SW Kwon.; SK Kang.; JH Lee.; JH Bok.; CH Kim. Synthesis and 11β hydroxysteroid dehydrogenase 1 inhibition of thiazolidine derivatives with an adamantyl group. Bioorganic & Medicinal Chemistry Letters. 201121 (1), 435-439.. ...
Such data are valuable to the chemical and pharmaceutical industries for getting the highest production yields and shelf life for the medication.. Our research results are not directly related to the medical application of this multifunctional drug, although I am really fascinated by the range of its pharmacological activity, NIST research chemist Ala Bazyleva said.. We studied its thermodynamic properties and decomposition, Bazyleva said. It is surprising, given the long history of amantadine-based drugs, that there is almost no information like this in the literature for many of them. Chemical engineers often have to rely on estimates and predictions based on similar compounds. Collating this information and developing these types of recommendations is at the core of what our group at NIST does.. Amantadine hydrochloride belongs to a diamondoid class, a family of compounds whose structure is based on a cage of carbon atoms similar to diamond. Amantadine has a single carbon cage with a ...
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Amantadine is a weak antagonist of the NMDA-type glutamate receptor, increases dopamine release, and blocks dopamine reuptake. This makes it therapy for Parkinsons disease (PD). In 2013, PD was present in 53 million people and resulted in about 103,000 deaths globally. Parkinsons disease typically occurs in people over the age of 60, of which about one percent is affected. Amantadine is also an anti-influenza drug, effectively inhibits the replication of all human subtypes of influenza A virus (H1N1, H2N2 and H3N2) both in laboratory studies and in a variety of clinical situations in young and old persons. Worldwide, influenza A (H3N2) virus is predominant. This review article provides an evaluation of the literature on the concomitant use of amantadine available. The efficacy, safety, tolerability, and patient quality of life of this regimen are discussed. A summary of the Mechanism of action of amantadine, as well as its effects during pregnancy are also reviewed. ...
Co-administration of memantine and amantadine with sub/suprathreshold doses of L-Dopa restores motor behviour of MPTP-treated mice. ...
Generic Symmetrel is an antiviral. It is used to prevent or treat certain influenza (flu) infections (type A). It may be given alone or along with flu shots. Generic Symmetrel will not work for colds, other types of flu or other virus infections. This medicine is also used to treat Parkinsons disease and other movement disorders.. Generic Symmetrel (Amantadine 100mg) $ 1.36 pill - Alzheimers And Parkinsons, Antivirals @ KupTania.
Buy Symmetrel is an antiviral. It is used to prevent or treat certain influenza (flu) infections (type A). It may be given alone or along with flu shots. Symmetrel will not work for colds, other types of flu or other virus infections. This medicine is also used to treat Parkinsons disease and other movement disorders.. Symmetrel (Amantadine 100mg) $1.11 pill - Alzheimers And Parkinsons, Antivirals @ Ohne Rezept Medikamente : Rezeptfrei Kaufen : Apotheke Online. Medikamente Ohne Rezept Im Ausland Kaufen. Rezeptfrei Medikamente Bestellen., Apotheke Preisvergleich.
Amantadine oral capsule (Gocovri) is used to treat Parkinsons disease, movement problems, and influenza A. Learn about side effects, warnings, dosage, and more.
Amantadine is an antiviral medicine that blocks the actions of viruses in your body. It is used to treat and prevent influenza A (a viral infection). It may not
... Symmetrel is prescribed to treat or prevent certain types of the flu virus, treat Parkinsons disease, and treat certain side effects of other medications.
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Out of 13 pertinent publications, 5 trials met the criteria for inclusion in this review: one study was a parallel arms study, and 4 were crossover trials. The number of randomised participants ranged between 10 and 115, and a total of 272 MS patients were studied. Overall the quality of the studies considered was poor and all trials were open to bias. All studies reported small and inconsistent improvements in fatigue, whereas the clinical relevance of these findings and the impact on patients functioning and health related quality of life remained undetermined. The number of participants reporting side effects during amantadine therapy ranged from 10% to 57%.. ...
In this international, multicenter, randomized, controlled trial involving patients with post-traumatic disorders of consciousness, we found that the administration of amantadine between 4 and 16 weeks after injury significantly improved the rate of functional recovery over the 4-week period of treatment, as compared with placebo. In keeping with evidence on the rate of change during inpatient rehabilitation,10,18 both groups had improvement during the 4-week period. However, the rate of recovery was more rapid in the amantadine group, affecting functionally meaningful behaviors such as consistent responses to commands, intelligible speech, reliable yes-or-no communication, and functional-object use.. The benefit of amantadine appeared to be consistent, regardless of the interval since injury or whether patients were in a vegetative state or a minimally conscious state at enrollment. Although gains were generally well maintained in the amantadine group after the washout period, the rate of ...
In this international, multicenter, randomized, controlled trial involving patients with post-traumatic disorders of consciousness, we found that the administration of amantadine between 4 and 16 weeks after injury significantly improved the rate of functional recovery over the 4-week period of treatment, as compared with placebo. In keeping with evidence on the rate of change during inpatient rehabilitation,10,18 both groups had improvement during the 4-week period. However, the rate of recovery was more rapid in the amantadine group, affecting functionally meaningful behaviors such as consistent responses to commands, intelligible speech, reliable yes-or-no communication, and functional-object use.. The benefit of amantadine appeared to be consistent, regardless of the interval since injury or whether patients were in a vegetative state or a minimally conscious state at enrollment. Although gains were generally well maintained in the amantadine group after the washout period, the rate of ...
In this international, multicenter, randomized, controlled trial involving patients with post-traumatic disorders of consciousness, we found that the administration of amantadine between 4 and 16 weeks after injury significantly improved the rate of functional recovery over the 4-week period of treatment, as compared with placebo. In keeping with evidence on the rate of change during inpatient rehabilitation,10,18 both groups had improvement during the 4-week period. However, the rate of recovery was more rapid in the amantadine group, affecting functionally meaningful behaviors such as consistent responses to commands, intelligible speech, reliable yes-or-no communication, and functional-object use.. The benefit of amantadine appeared to be consistent, regardless of the interval since injury or whether patients were in a vegetative state or a minimally conscious state at enrollment. Although gains were generally well maintained in the amantadine group after the washout period, the rate of ...
WFTV.COM in FLORIDA reports on Oct. 1, 2012 that Amantadine, the Flu drug, has been effective with improvement in recovery of cognitive skills amongst Traumatic Brain Injury (TBI) victims. TBI is a brain injury caused by a sudden trauma to the brain. This usually is caused by a sports or automobile accident but…
Seek medical attention right away if any of these severe side effects occur:. Severe allergic reactions (rash; hives; itching; difficulty breathing; tightness in the chest; swelling of the mouth, face, lips, or tongue); aggression; agitation; confusion; depression; fainting; fast or irregular heartbeat; fever; hallucinations; memory loss; mental or mood changes; muscle problems (eg, spasms, uncontrolled movements); paranoid thoughts; personality changes; seizures; severe or persistent drowsiness or trouble sleeping; shortness of breath; sore throat; swelling of hands, legs, feet, or ankles; thoughts of suicide; trouble urinating; unusual anxiety or irritability; vision changes ...
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Answers from specialists on amantadine along with combo therapy. First: Insomnia is caused by many many things. Thinking about biological and psychological interventions is often helpful. Anxiety meds are helpful in the short run but maybe not long term. Cbtcan be very helpful to look at various cognitive and behavioral components which are contributory.
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Tromantadine. Molecular model of the antiviral drug tromantadine (C16.H28.N2.O2), used to treat infections with the herpes simplex virus. Atoms are represented as spheres and are colour-coded: carbon (grey), hydrogen (white), nitrogen (blue) and oxygen (red). Illustration. - Stock Image F017/0094
Around 2005 the Centers for Disease Control and Prevention (CDC) issued an alert instructing doctors to avoid using specific anti-flu compounds such as amantadine and rimantadine during the flu season because amantadine resistance has been increased in one of the influenza A (H3N2) viruses, which were the cause of flu. This was one of the…
RESULTS: Amantadine had antidepressant-like effects in animal models and appeared to potentiate the antidepressant effects of other antidepressants. These preclinical findings have received some support from the results of small open-label clinical trials, suggesting that amantadine can reduce depressive symptomatology and potentiate the antidepressant effects of monoaminergic drugs. In addition to its glutamatergic and dopaminergic effects, the potential antidepressant-like effects of amantadine have been linked to molecular and cellular actions, such as increased expression of neurotrophic factors (e.g., brain-derived neurotrophic factor), activation of σ1 receptors, decreased corticosterone levels, and decreased inflammatory response to stress ...
Generic Symmetrel (amantadine Hydrochloride) is an antiviral medication. It is a very unique medication used for numerous reasons. For effective relief of various symptoms order Generic Symmetrel t...
Bernd Sebastian Kamps and Christian Hoffmann Amantadine inhibits the replication of influenza A viruses by interfering with the uncoating of the virus inside the cell. Like rimantadine, it is an M2 inhibitor which blocks the ion channel formed by the M2 protein that spans the viral membrane (Hay 1985, Sugrue 1991). The influenza virus enters its host cell by receptor-mediated endocytosis. Thereafter, acidification of the endocytotic vesicles is required for the dissociation of the M1 protein from the ribonucleoprotein complexes. Only then are the ribonucleoprotein particles imported into the nucleus via the nuclear pores. The hydrogen ions needed for acidification pass through the M2 channel. Amantadine blocks the channel (Bui 1996). Amantadine is effective against all influenza A subtypes that have previously caused disease in humans (H1N1, H2N2 and H3N2), but not against influenza B virus, because the protein M2 is unique to influenza A viruses. For both the prevention and treatment of ...
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The tetramer is not exactly symmetric. Two crystal structures (3bkd and 3c9j) are close to each other (CA atom rmsd of 1.1 A) but strongly differ from NMR structure, 2rlf (rmsd of 3.8-3.9 A). This structure is similar to amantadine-bound solid state NMR model 2h95 (rmsd of 2.4A ...
Take Symmetrel exactly as prescribed by your doctor. The recommended dose for treating or preventing the flu in adults is 100 mg twice daily or 200 mg once daily. Taking it twice a day may decrease the chance of side effects. A lower dose (100 mg once daily) is recommended for people age 65 years or older. For children age 9 and older, the recommended dose is 100 mg twice daily. For flu treatment, Symmetrel should be started as soon as possible within 24 to 48 hours of the start of flu symptoms, and it should be continued for 24 to 48 hours after the symptoms go away. For treating Parkinsons disease the recommended dose is 100 mg twice a day, if no other Parkinsons medications are used. Your doctor may increase your dose up to a maximum of 400 mg total per day. The recommended dose for treating extrapyramidal symptoms caused by other medications is 100 mg twice daily. Your doctor may increase your dose up to a total of 300 mg per day. If you have liver or kidney disease, your doctor may ...
Once I started taking it, I knew the first day I did not get the placebo. It caused the effect with my dyskinesia identical to the first day I started Amantadine last June. It spread my peak dyskinesia of around 30 minutes to last most of the 6 hour dose. Only since it was a higher amount--originally was on 200 mg/day, the trial dosage was either 250 mg/day or 350 mg/day, never found out which one I was on--the dyskinesia was more intense. As the study progressed, that seemed to increase. Likewise, my side-effects were there, but became worse. For example, the treatment I used to offset the constipation from the regular Amantadine was failing me a few weeks into the study. However, the peak dyskinesia was reduced significant and the left-arm stiffness was non-existent, as was any off times on the levadopa. My leg swelling seemed to be worse, and I was concerned it would get bad enough Id have to stop participating in the study ...
Dom-Amantadine: Amantadine belongs to the family of medications called antivirals. It is used to prevent and treat influenza A infections. It is believed to work by preventing the virus from reproducing. If used to prevent an infection, amantadine must be taken before exposure to the flu virus.
Phl-Amantadine: Amantadine belongs to the family of medications called antivirals. It is used to prevent and treat influenza A infections. It is believed to work by preventing the virus from reproducing. If used to prevent an infection, amantadine must be taken before exposure to the flu virus.
Amantadine belongs to the family of medications called antivirals. It is used to prevent and treat influenza A infections. It is believed to work by preventing the virus from reproducing. If used to prevent an infection, amantadine must be taken before exposure to the flu virus.
Not clearly understood. Prevent the release of infectious viral nucleic acid into the host cell by interfering with the function of the transmembrane domain of the viral M2 protein. In certain cases, prevent virus assembly during virus replication ...
Some side effects may occur that usually do not need medical attention. These side effects may go away during treatment as your body adjusts to the medicine. Also, your health care professional may be able to tell you about ways to prevent or reduce some of these side effects. Check with your health care professional if any of the following side effects continue or are bothersome or if you have any questions about them:. ...
He is a very difficult dog to read as doesnt present any obvious signs of pain and never "complains". We have primarily managed any pain in his life using holistic methods but he has been on an nsaid since summer. We are supporting his system with some supplements and slippery elm for his stomach. There were however some minor things like shifting a lot, groans while sleeping and some minor habit changes that led me to believe he was still uncomfortable. We recently added amantadine to his metacam and that seems to have made him a lot more comfortable and he is bouncing like a crazy puppy. Amantadine is a human anti-viral drug and often used in Parkinsons disease. It doesnt do a lot for dogs by itself but it helps boosts the effectiveness of other pain medications by resetting spinal cord receptors. He might feel better but he still has a limit of about 15-20 minutes on exercise before his body starts to rebel and his back end gets all wonky. He is a mind-over-matter dog and would just keep ...
共有13 個適合的文獻(來自5 個試驗資料)符合納入條件。1個試驗為平行設計,其他四個有交換治療。各組隨機分配的人數為10 到115人,共納入272位多發性硬化病患。整體品質不佳,都有可能有偏差。所有試驗結果都顯示只有些微不一致的改進疲勞效果,但此結果與治療的臨床相關性不明,對病患官能和相關的生活品質的影響也不明。治療期間發生副作用的人數比例為10%-57%. ...
Hi all, Hope you are all hanging in there you wonderful people x My OH has been doing fine, well more than fine for the past 18months. He has been on Stavelo, and Azilect and Neuro patches but the...
This is a beta test version of the Everyone Healthy Database.Any medical information published or self diagnosed conditions on this website is not intended as a substitute for professional medical advice and you should not take any action or change your current treatment before consulting with a certified health care professional ...
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It seems that in the three months since I have been on Amantadine, Trihexphenidyl and Azilect for Parkensons, I have lost considerable weight(16 lbs).. I have very little appetite and get filled quickl...
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Learn about the drug Amantadine or Rimantadine for Influenza (Flu). Includes generic and brand name examples, dosage, how the drug works, why it is used and side effects.
Evidence-based recommendations on amantadine (Lysovir, Symmetrel), oseltamivir (Tamiflu) and zanamivir (Relenza) for treating influenza (flu)
Data can be acquired at frequencies up to % of the excitation frequency and╘ by CRC Press LLCexcitation frequency varies from Hz to kHz, although higher frequencies are attainable Linearpotentiometers can also measure displacement; they are considerably less expensive, approximately %of the cost of an LVDT. Dystonia is similar to weight lifting and athetosis is similar to run-ning. Further interventions in this patient may not be indicated, and thephysician may decide that doing more procedures on the patient would be unethical; how-ever, it would be more appropriate to have a discussion with the family and to educatethem about the condition and prognosis. She also relates thatshe believes she is being watched carefully by the FBI and that your conversation with her is probablybeing monitored. A -year-old man presents to a hospital with fatigue and fever of weeksр duration. With respect to injury,this effort is compromised by the absence of adequate nationalsurveillance information from all ...
Gay N. Inter-pandemic surveillance, international spread & impact of control measures. GHSAG Flu Modeling Workshop. London, 2005. Longini IM, Jr., Koopman JS, Monto AS, Fox JP. Estimating household and community transmission parameters for influenza. Am J Epidemiol 1982;115:736-51. Longini IM, Jr., Koopman JS. Household and community transmission parameters from final distributions of infections in households. Biometrics 1982;38:115-26. Fritz RS, Hayden FG, Calfee DP, Cass LM, Peng AW, Alvord WG, et al. Nasal cytokine and chemokine responses in experimental influenza A virus infection: results of a placebo-controlled trial of intravenous zanamivir treatment. J Infect Dis 1999;180:586-93. Reuman PD, Bernstein DI, Keefer MC, Young EC, Sherwood JR, Schiff GM. Efficacy and safety of low dosage amantadine hydrochloride as prophylaxis for influenza A. Antiviral Res 1989;11:27-40. Hayden FG, Treanor JJ, Fritz RS, Lobo M, Betts RF, Miller M, et al. Use of the oral neuraminidase inhibitor oseltamivir in ...
The Traumatic Brain Injury Model System (TBIMS) program was created and funded by the National Institute on Disability and Rehabilitation Research (NIDRR) in 1987 to demonstrate the benefits of a coordinated system of neurotrauma and rehabilitation care and conduct innovative research on all aspects of care for those who sustain traumatic brain injuries. Each Center systematically collects important data about each individual who meets criteria for inclusion in the TBI National Database and sends this information to the TBI National Data Center at KMRREC. The Centers are currently located at 16 sites throughout the United States that provide comprehensive systems of brain injury care to individuals who sustain a traumatic brain injury, from acute care through community re-entry. The mission of the TBIMS is to improve the lives of persons who experience traumatic brain injury, their families and communities by creating and disseminating new knowledge about the course, treatment and outcomes relating to
The Hepatitis C Virus is the most common cause of chronic liver disease. Current therapy is only partially effective and fraught with side effects. A greater understanding of viral replication and new virus particle formation is thus important for developing new therapeutic targets. The HCV p7 protein is critical for virus production and an attractive antiviral target. p7 is an ion channel when reconstituted in artificial lipid bilayers, but channel function has not been demonstrated in vivo and it is unknown whether p7 channel activity plays a critical role in virus production. To evaluate the contribution of p7 to organelle pH regulation and virus production, a fluorescent pH sensor was incorporated within native, intracellular vesicles in the presence or absence of p7. p7 increased proton conductance in vesicles and was able to rapidly equilibrate H+ gradients. This conductance was blocked by the viroporin inhibitors amantadine, rimantadine and hexamethylene amiloride. Fluorescence microscopy ...
Influenza causes significant morbidity and mortality and is responsible for considerable medical expenditures. Vaccination is the most effective public health measure to combat this illness. Amantadine and rimantadine are older antiviral agents that have been important adjuncts in the prevention and treatment of influenza A outbreaks. Zanamivir and oseltamivir are newer agents indicated for the treatment of both influenza A and B. For antiviral agents to be effective, they must be used within 48 hours of the onset of influenza symptoms. Antiviral agents reduce the duration of fever and illness by one to two and one-half days and also reduce the severity of some symptoms. Use of amantadine or rimantadine is appropriate if influenza virus A is known to be the predominant agent in a particular year or location. Data need to be evaluated on the development of resistance and use of the newer antiviral agents in geriatric patients, high-risk patients and children. For optimal use of antiviral agents, patients
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Results For irritability, SSRI was first choice of 58%, an antipsychotic was first choice for 22%, a mood stabilizing anticonvulsant 14%, and benzodiazepine 2%. For perseverative behaviors, SSRI was first choice of 75%, an antipsychotic choice of 4%, a mood stabilizing anticonvulsant choice of 6%, and clomipramine 2%. The remaining 13% chose to qualify the response to include 2 first choices. For chorea, an antipsychotic was first choice for 56%, tetrabenazine 15%, amantadine 6%, and benzodiazepines 4%. The remaining 13% chose to qualify the response to include 2 first choices. Drug choice for use as adjunctive therapy was widely variable. ...
The Southeast Asian outbreak of the highly lethal avian influenza A H5N1 infection in humans is unlikely to abate because of the enormous number of backyard farms providing poultry as the main source of food protein in developing countries. This increases the risk of the emergence of a reassortant pandemic influenza virus with improved human-to-human transmissibility. Currently triage of suspected cases by epidemiological risk factors remains the only practical way of case identification for laboratory investigation and infection control. The clinical usefulness of rapid diagnostic laboratory tests requires more vigorous evaluation. The lethality of this disease may reflect systemic viral dissemination, cytokine storm, or alveolar flooding due to inhibition of cellular sodium channels. The present circulating genotype Z is intrinsically resistant to amantadine and rimantadine. Prognosis may be improved by early treatment with a neuraminidase inhibitor with good systemic drug levels, and ...
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5-HT receptor (1) acetylation (1) Acetylcarnitine (1) acetylcholine (1) ACTH (1) Acute myeloid leukemia (1) adaptive immune response (2) Adaptive immune system (3) ADHD (107) Adipocyte (1) Aflatoxin (1) Ageing (18) Agent Orange (1) Aggression (1) AIDS (48) Air pollution (7) alcohol (2) Algal bloom (2) Allergen (2) allergies (21) Allergy (9) ALS (25) Aluminium (4) Alzheimers disease (803) Alzheimers disease (22) Amantadine (1) Androstadienone (1) Anemia (2) angiogenesis (1) Anno Domini (1) anorexia (10) Anti Trafficking (1) Antibacterial (14) Antibiotic resistance (9) antibiotics (25) Anticholinergic (2) Antidepressant (18) Antimicrobial peptides (7) Antipsychotic (2) antiviral (19) Antiviral drug (5) anxiety (2) APOE (32) Apoptosis (2) Appendicitis (1) arsenic (5) arthritis (18) artificial sweeteners (9) aryl hydrocarbon receptor (1) aspartame (14) Aspergillus (2) aspirin (7) asthma (44) asthma. (2) asthma. mold (4) Astrocyte (5) atherosclerosis (149) atopy (10) atrazine (1) Attention deficit ...
Background: About 25% of patients with chronic hepatitis C virus (HCV) infection have persistently normal alanine aminotransferase (ALT) values (1). No agreement about management of these patients exists (2). The standard of care for chronic HCV infection is the combination of peginterferon plus ribavirin, and the addition of amantadine may increase the virologic response (3, 4). However, optimal treatment in patients with persistently normal ALT levels has not been investigated ...
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Amantadine-induced diastolic depolarization and automaticity in ventricular muscle. | Circulation ResearchAmantadine-induced diastolic depolarization and automaticity in ventricular muscle. | Circulation Research

All actions of amantadine were rapidly reversed upon washout. Propranolol had no effect on the actions of the drug. Amantadine- ... In the steady state, amantadine-induced spontaneous activity was rather stable, and the rate was dependent upon the amantadine ... We studied the cardiac effects of amantadine, an antiviral and anti-Parkinson drug. Amantadine hydrochloride (100--800 microM) ... Amantadine-induced diastolic depolarization and automaticity in ventricular muscle.. J J Salata, J Jalife, J L Megna, G ...
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Visual impairment in Parkinsons disease treated with amantadine: case report and review of the literature.Visual impairment in Parkinson's disease treated with amantadine: case report and review of the literature.

... developed sudden-onset visual impairment after initiation of amantadine treatment. Ophthalmologic examination revealed corneal ... Amantadine / therapeutic use*. Antiparkinson Agents / therapeutic use*. Cornea / pathology. Endothelium, Corneal / pathology. ... Discontinuation of amantadine resulted in rapid improvement of visual acuity. A review of the literature indicated only a few ... Amantadine-associated visual impairment in PD could be possibly overlooked, since PD mainly affects elderly people who often ...
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Pharmacokinetics of amantadine hydrochloride - Generic Online Pharmacy.Pharmacokinetics of amantadine hydrochloride - Generic Online Pharmacy.

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Desipramine ( Norpramin) , amantadine ( Symmetrel ), or fluoxetine ( Prozac ) for  cocaine users on buprenorphine ( Temgesic ,...Desipramine ( Norpramin) , amantadine ( Symmetrel ), or fluoxetine ( Prozac ) for cocaine users on buprenorphine ( Temgesic ,...

Desipramine, amantadine, or fluoxetine. in buprenorphine-maintained cocaine users. by. Oliveto A, Kosten TR, Schottenfeld R, ... These results suggest that desipramine and amantadine may facilitate greater opioid and cocaine abstinence than fluoxetine. ... The order of greatest patient retention across the 12 weeks was desipramine (83.3%) > amantadine (66.7%) > fluoxetine (20.0%). ... amantadine (300 mg, p.o.), or fluoxetine (60 mg, p.o.). Urine samples and self-reported drug use were obtained 1-3 times/week. ...
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Parkinsons Disease, Amantadine Hydrochloride Therapy and Dopa Metabol by Dee Edward Silver, J. I. Routh et al."Parkinson's Disease, Amantadine Hydrochloride Therapy and Dopa Metabol" by Dee Edward Silver, J. I. Routh et al.

The results indicated that patients on amantadine therapy had lower urinary levels of epinephrine plus norepinephrine than ... Patients who developed livedo reticularis during amantadine therapy showed a small but significant increase in urinary dopamine ... levels and a similar decrease in dopac levels, when compared to other patients on amantadine who did not develop livedo ... In an attempt to clarify the effect of amantadine hydrochloride therapy in Parkinsons disease, dopa metabolites were measured ...
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Use of amantadine or rimantidine is not recommended. Additional information regarding influenza viruses, influenza surveillance ... amantadine and rimantidine). During the 2009--10 influenza season, testing of the 2009 pandemic H1N1 viruses found that 1.1% of ...
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J. Med. 307, 580 (1982). Comparative toxicity of rimantadine and amantadine in healthy adults: F. G. Hayden et al., Antimicrob ...
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Isolation of amantadine-resistant influenza A viruses (H3N2) from patients following administration of amantadine in japan. J ... Yavarian, J., Mokhtari Azad, T., Zheng, X., Gregory, V., Lin, P., Hay, A. (2010) Amantadine resistance in relation to the ... Higgins, R.R., Eshaghi, A., Burton, L., Mazzulli, T., Drews, S.J. (2009) Differential patterns of amantadine-resistance in ... Saito, R., Oshitani, H., Masuda, H., Suzuki, H. (2002) Detection of amantadine-resistant influenza A virus strains in nursing ...
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  • Plasma acetylamantadine accounted for up to 80% of the concurrent amantadine plasma concentration in 5 of 12 healthy volunteers following the ingestion of a 200 mg dose of amantadine. (nih.gov)
  • Sporadic cases of possible Neuroleptic Malignant Syndrome (NMS) have been reported in association with dose reduction or withdrawal of amantadine therapy. (healthyplace.com)
  • The dose of amantadine should be reduced in patients with renal impairment and in patients who are 65 years of age or older. (healthyplace.com)
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  • The recommended adult dose of amantadine varies according to the condition being treated. (medbroadcast.com)
  • Amantadine is effective in reducing severity or abolishing drug-induced extrapyramidal reactions including parkinsonism syndrome, dystonia and akathisia. (healthyplace.com)
  • To treat influenza A infections, amantadine should be taken within 24 to 48 hours of the start of flu symptoms, and continued for 24 to 48 hours after the symptoms disappear, up to 5 days. (medbroadcast.com)
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  • Each tablet intended for oral administration contains 100 mg amantadine hydrochloride and has the following inactive ingredients: microcrystalline cellulose, povidone, sodium starch glycolate, magnesium stearate, colloidal silicon dioxide, FD&C Yellow No. 6 Aluminum Lake. (nih.gov)
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  • If used to prevent an infection, amantadine must be taken before exposure to the flu virus. (medbroadcast.com)
  • These effects can also be caused by the administration of anticholinesterases or antihistamines while taking amantadine. (britannica.com)
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  • The safety and efficacy of use of amantadine in infants less than 1 year old have not been established. (healthyplace.com)
  • cheap amantadine blue pill amantadine order singapore amantadine com cheap He verified my observations from the point of view of the latest in medical research regarding viruses along with his own experience. (lanyrd.com)
  • Following completion of baseline assessments, randomization to amantadine or placebo will occur. (knowcancer.com)
  • If you have blurry vision or other confusion after taking amantadine, you should not drive or work in a situation in which you need to be alert or be able to move well. (healthline.com)
  • The mechanism by which amantadine exerts its antiviral activity is not clearly understood. (nih.gov)