Alphaherpesvirinae: A subfamily of HERPESVIRIDAE characterized by a short replication cycle. The genera include: SIMPLEXVIRUS; VARICELLOVIRUS; MAREK'S DISEASE-LIKE VIRUSES; and ILTOVIRUS.Betaherpesvirinae: A subfamily of HERPESVIRIDAE characterized by a relatively long replication cycle. Genera include: CYTOMEGALOVIRUS; MUROMEGALOVIRUS; and ROSEOLOVIRUS.Herpesvirus 1, Bovine: A species of VARICELLOVIRUS that causes INFECTIOUS BOVINE RHINOTRACHEITIS and other associated syndromes in CATTLE.Herpesvirus 1, Suid: A species of VARICELLOVIRUS producing a respiratory infection (PSEUDORABIES) in swine, its natural host. It also produces an usually fatal ENCEPHALOMYELITIS in cattle, sheep, dogs, cats, foxes, and mink.Herpesviridae Infections: Virus diseases caused by the HERPESVIRIDAE.Viral Proteins: Proteins found in any species of virus.Molecular Sequence Data: Descriptions of specific amino acid, carbohydrate, or nucleotide sequences which have appeared in the published literature and/or are deposited in and maintained by databanks such as GENBANK, European Molecular Biology Laboratory (EMBL), National Biomedical Research Foundation (NBRF), or other sequence repositories.Cytomegalovirus Retinitis: Infection of the retina by cytomegalovirus characterized by retinal necrosis, hemorrhage, vessel sheathing, and retinal edema. Cytomegalovirus retinitis is a major opportunistic infection in AIDS patients and can cause blindness.Cytomegalovirus: A genus of the family HERPESVIRIDAE, subfamily BETAHERPESVIRINAE, infecting the salivary glands, liver, spleen, lungs, eyes, and other organs, in which they produce characteristically enlarged cells with intranuclear inclusions. Infection with Cytomegalovirus is also seen as an opportunistic infection in AIDS.Cytomegalovirus Infections: Infection with CYTOMEGALOVIRUS, characterized by enlarged cells bearing intranuclear inclusions. Infection may be in almost any organ, but the salivary glands are the most common site in children, as are the lungs in adults.Suppressor of Cytokine Signaling Proteins: A family of structurally related proteins that are induced by CYTOKINES and negatively regulate cytokine-mediated SIGNAL TRANSDUCTION PATHWAYS. SOCS proteins contain a central SH2 DOMAIN and a C-terminal region of homology known as the SOCS box.Immune Evasion: Methods used by pathogenic organisms to evade a host's immune system.Retinitis Pigmentosa: Hereditary, progressive degeneration of the neuroepithelium of the retina characterized by night blindness and progressive contraction of the visual field.Retinitis: Inflammation of the RETINA. It is rarely limited to the retina, but is commonly associated with diseases of the choroid (CHORIORETINITIS) and of the OPTIC DISK (neuroretinitis).Herpesvirus 1, Canid: A species of VARICELLOVIRUS virus that causes a disease in newborn puppies.Encyclopedias as Topic: Works containing information articles on subjects in every field of knowledge, usually arranged in alphabetical order, or a similar work limited to a special field or subject. (From The ALA Glossary of Library and Information Science, 1983)Sneezing: The sudden, forceful, involuntary expulsion of air from the NOSE and MOUTH caused by irritation to the MUCOUS MEMBRANES of the upper RESPIRATORY TRACT.Dogs: The domestic dog, Canis familiaris, comprising about 400 breeds, of the carnivore family CANIDAE. They are worldwide in distribution and live in association with people. (Walker's Mammals of the World, 5th ed, p1065)Dog Diseases: Diseases of the domestic dog (Canis familiaris). This term does not include diseases of wild dogs, WOLVES; FOXES; and other Canidae for which the heading CARNIVORA is used.Herpesviridae: A family of enveloped, linear, double-stranded DNA viruses infecting a wide variety of animals. Subfamilies, based on biological characteristics, include: ALPHAHERPESVIRINAE; BETAHERPESVIRINAE; and GAMMAHERPESVIRINAE.Terminology as Topic: The terms, expressions, designations, or symbols used in a particular science, discipline, or specialized subject area.GermanyRed Cross: International collective of humanitarian organizations led by volunteers and guided by its Congressional Charter and the Fundamental Principles of the International Red Cross Movement, to provide relief to victims of disaster and help people prevent, prepare for, and respond to emergencies.Classification: The systematic arrangement of entities in any field into categories classes based on common characteristics such as properties, morphology, subject matter, etc.Bacteria: One of the three domains of life (the others being Eukarya and ARCHAEA), also called Eubacteria. They are unicellular prokaryotic microorganisms which generally possess rigid cell walls, multiply by cell division, and exhibit three principal forms: round or coccal, rodlike or bacillary, and spiral or spirochetal. Bacteria can be classified by their response to OXYGEN: aerobic, anaerobic, or facultatively anaerobic; by the mode by which they obtain their energy: chemotrophy (via chemical reaction) or PHOTOTROPHY (via light reaction); for chemotrophs by their source of chemical energy: CHEMOLITHOTROPHY (from inorganic compounds) or chemoorganotrophy (from organic compounds); and by their source for CARBON; NITROGEN; etc.; HETEROTROPHY (from organic sources) or AUTOTROPHY (from CARBON DIOXIDE). They can also be classified by whether or not they stain (based on the structure of their CELL WALLS) with CRYSTAL VIOLET dye: gram-negative or gram-positive.Editorial Policies: The guidelines and policy statements set forth by the editor(s) or editorial board of a publication.Indigo Carmine: Indolesulfonic acid used as a dye in renal function testing for the detection of nitrates and chlorates, and in the testing of milk.BooksVanilla: A plant genus of the family ORCHIDACEAE that is the source of the familiar flavoring used in foods and medicines (FLAVORING AGENTS).Paeonia: A plant genus of the family Paeoniaceae, order Dilleniales, subclass Dilleniidae, class Magnoliopsida. These perennial herbs are up to 2 m (6') tall. Leaves are alternate and are divided into three lobes, each lobe being further divided into three smaller lobes. The large flowers are symmetrical, bisexual, have 5 sepals, 5 petals (sometimes 10), and many stamens.Herpesvirus 3, Human: The type species of VARICELLOVIRUS causing CHICKENPOX (varicella) and HERPES ZOSTER (shingles) in humans.Indonesia: A republic stretching from the Indian Ocean east to New Guinea, comprising six main islands: Java, Sumatra, Bali, Kalimantan (the Indonesian portion of the island of Borneo), Sulawesi (formerly known as the Celebes) and Irian Jaya (the western part of New Guinea). Its capital is Djakarta. The ethnic groups living there are largely Chinese, Arab, Eurasian, Indian, and Pakistani; 85% of the peoples are of the Islamic faith.Herpes Zoster: An acute infectious, usually self-limited, disease believed to represent activation of latent varicella-zoster virus (HERPESVIRUS 3, HUMAN) in those who have been rendered partially immune after a previous attack of CHICKENPOX. It involves the SENSORY GANGLIA and their areas of innervation and is characterized by severe neuralgic pain along the distribution of the affected nerve and crops of clustered vesicles over the area. (From Dorland, 27th ed)Chickenpox: A highly contagious infectious disease caused by the varicella-zoster virus (HERPESVIRUS 3, HUMAN). It usually affects children, is spread by direct contact or respiratory route via droplet nuclei, and is characterized by the appearance on the skin and mucous membranes of successive crops of typical pruritic vesicular lesions that are easily broken and become scabbed. Chickenpox is relatively benign in children, but may be complicated by pneumonia and encephalitis in adults. (From Dorland, 27th ed)RNA Viruses: Viruses whose genetic material is RNA.Vaccinia virus: The type species of ORTHOPOXVIRUS, related to COWPOX VIRUS, but whose true origin is unknown. It has been used as a live vaccine against SMALLPOX. It is also used as a vector for inserting foreign DNA into animals. Rabbitpox virus is a subspecies of VACCINIA VIRUS.Gammaherpesvirinae: A subfamily of HERPESVIRIDAE characterized by variable reproductive cycles. The genera include: LYMPHOCRYPTOVIRUS and RHADINOVIRUS.Herpesvirus 6, Human: The type species of ROSEOLOVIRUS isolated from patients with AIDS and other LYMPHOPROLIFERATIVE DISORDERS. It infects and replicates in fresh and established lines of hematopoietic cells and cells of neural origin. It also appears to alter NK cell activity. HHV-6; (HBLV) antibodies are elevated in patients with AIDS, Sjogren's syndrome, sarcoidosis, chronic fatigue syndrome, and certain malignancies. HHV-6 is the cause of EXANTHEMA SUBITUM and has been implicated in encephalitis.Herpesvirus 1, Ranid: A species of the family HERPESVIRIDAE, whose genus is so far unassigned. It is probably the causative agent of ADENOMA or ADENOCARCINOMA in the kidneys of certain frogs.DucksMink Viral Enteritis: A highly contagious parvovirus infection in mink, caused by MINK ENTERITIS VIRUS or the closely related FELINE PANLEUKOPENIA VIRUS or CANINE PARVOVIRUS. Transmission usually occurs by the fecal/oral route.GeeseEnteritis: Inflammation of any segment of the SMALL INTESTINE.Complement Factor H: An important soluble regulator of the alternative pathway of complement activation (COMPLEMENT ACTIVATION PATHWAY, ALTERNATIVE). It is a 139-kDa glycoprotein expressed by the liver and secreted into the blood. It binds to COMPLEMENT C3B and makes iC3b (inactivated complement 3b) susceptible to cleavage by COMPLEMENT FACTOR I. Complement factor H also inhibits the association of C3b with COMPLEMENT FACTOR B to form the C3bB proenzyme, and promotes the dissociation of Bb from the C3bBb complex (COMPLEMENT C3 CONVERTASE, ALTERNATIVE PATHWAY).Complement Activation: The sequential activation of serum COMPLEMENT PROTEINS to create the COMPLEMENT MEMBRANE ATTACK COMPLEX. Factors initiating complement activation include ANTIGEN-ANTIBODY COMPLEXES, microbial ANTIGENS, or cell surface POLYSACCHARIDES.Complement System Proteins: Serum glycoproteins participating in the host defense mechanism of COMPLEMENT ACTIVATION that creates the COMPLEMENT MEMBRANE ATTACK COMPLEX. Included are glycoproteins in the various pathways of complement activation (CLASSICAL COMPLEMENT PATHWAY; ALTERNATIVE COMPLEMENT PATHWAY; and LECTIN COMPLEMENT PATHWAY).Complement C3: A glycoprotein that is central in both the classical and the alternative pathway of COMPLEMENT ACTIVATION. C3 can be cleaved into COMPLEMENT C3A and COMPLEMENT C3B, spontaneously at low level or by C3 CONVERTASE at high level. The smaller fragment C3a is an ANAPHYLATOXIN and mediator of local inflammatory process. The larger fragment C3b binds with C3 convertase to form C5 convertase.Complement Inactivating Agents: Compounds that negatively regulate the cascade process of COMPLEMENT ACTIVATION. Uncontrolled complement activation and resulting cell lysis is potentially dangerous for the host.Complement Pathway, Alternative: Complement activation initiated by the interaction of microbial ANTIGENS with COMPLEMENT C3B. When COMPLEMENT FACTOR B binds to the membrane-bound C3b, COMPLEMENT FACTOR D cleaves it to form alternative C3 CONVERTASE (C3BBB) which, stabilized by COMPLEMENT FACTOR P, is able to cleave multiple COMPLEMENT C3 to form alternative C5 CONVERTASE (C3BBB3B) leading to cleavage of COMPLEMENT C5 and the assembly of COMPLEMENT MEMBRANE ATTACK COMPLEX.Sunlight: Irradiation directly from the sun.Skin Pigmentation: Coloration of the skin.Herpes Simplex: A group of acute infections caused by herpes simplex virus type 1 or type 2 that is characterized by the development of one or more small fluid-filled vesicles with a raised erythematous base on the skin or mucous membrane. It occurs as a primary infection or recurs due to a reactivation of a latent infection. (Dorland, 27th ed.)

Macropodid herpesviruses 1 and 2 occupy unexpected molecular phylogenic positions within the Alphaherpesvirinae. (1/106)

The molecular phylogeny of macropodid herpesviruses 1 and 2 (MaHV-1 and -2) has been investigated by cloning and sequencing the genes encoding glycoprotein B from both viruses. Phylogenetic reconstructions based on the putative amino acid sequences of glycoprotein B indicate that MaHV-1 and -2 are most closely related to the subfamily Alphaherpesvirinae. Within the Alphaherpesvirinae, MaHV-1 and -2 are closely associated with those herpesviruses that infect primates. This phylogenetic relationship does not fit the constraints of the proposed co-evolution theory described for other members of the Alphaherpesvirinae which have mammalian hosts.  (+info)

Cellular elongation factor 1delta is modified in cells infected with representative alpha-, beta-, or gammaherpesviruses. (2/106)

Earlier reports (Y. Kawaguchi, R. Bruni, and B. Roizman, J. Virol. 71:1019-1024, 1997; Y. Kawaguchi, C. Van Sant, and B. Roizman, J. Virol. 72:1731-1736, 1998) showed that herpes simplex virus 1 (HSV-1) infection causes the hyperphosphorylation of translation elongation factor 1delta (EF-1delta) and that the modification of EF-1delta is the consequence of direct phosphorylation by a viral protein kinase encoded by the UL13 gene of HSV-1. The UL13 gene is conserved in members of all herpesvirus subfamilies. Here we report the following. (i) In various mammalian cells, accumulation of the hyperphosphorylated form of EF-1delta is observed after infection with alpha-, beta-, and gammaherpesviruses, including HSV-2, feline herpesvirus 1, pseudorabiesvirus, bovine herpesvirus 1, human cytomegalovirus (HCMV), and equine herpesvirus 2. (ii) In human lung fibroblast cells infected with recombinant HSV-1 lacking the UL13 gene, the hypophosphorylated form of EF-1delta is a minor species, whereas the amount of the hyperphosphorylated form of EF-1delta significantly increases in cells infected with the recombinant HSV-1 in which UL13 had been replaced by HCMV UL97, a homologue of UL13. These results indicate that the posttranslational modification of EF-1delta is conserved herpesvirus function and the UL13 homologues may be responsible for the universal modification of the translation factor.  (+info)

Studies of genetic relationships between bovine, caprine, cervine, and rangiferine alphaherpesviruses and improved molecular methods for virus detection and identification. (3/106)

The glycoprotein B (gB) and D (gD) genes from five ruminant alphaherpesviruses, bovine herpesvirus 1 (BHV-1), bovine herpesvirus 5 (BHV-5), caprine herpesvirus 1 (CapHV-1), cervine herpesvirus 1, and rangiferine herpesvirus 1, were partially sequenced. The nucleotide sequence alignments revealed a highly conserved gB gene, with homologies ranging between 87.2 and 99.6%, and a more variable gD gene, with homologies ranging between 71.3 and 98.9%. The phylogenetic analysis of the gB and gD nucleotide and deduced amino acid sequences revealed that BHV-5 is the most closely related virus to the BHV-1 subtype 1 and BHV-1 subtype 2 cluster and that CapHV-1 is the most distantly related virus. The phylogenetic data showed a close relationship of all the studied viruses with suid herpesvirus 1. On the basis of sequence data for the gB gene, a nested PCR combined with restriction enzyme analysis (REA) of the PCR products was developed for the simultaneous detection and identification of the viruses that were studied. Nested primers from highly conserved sequence stretches were selected in order to amplify a region of 294 bp in all five viruses, and a subsequent REA of the PCR products allowed specific identification. A mimic molecule that served as an internal standard of the amplification efficiency was constructed. The practical diagnostic applicability of the assay was evaluated with clinical samples consisting of semen and organ specimens from experimentally infected animals.  (+info)

Complementation of a gl-deficient feline herpesvirus recombinant by allotopic expression of truncated gl derivatives. (4/106)

The alphaherpesvirus glycoproteins gE and gI form a hetero-oligomeric complex involved in cell-to-cell transmission. The gI-deficient recombinant feline herpesvirus (FHV), FHVdeltagI-LZ, produces plaques that are only 15% the size of those of wild-type FHV. Here, we have complemented FHV(delta)gI-LZ allotopically by expressing intact gI and C-terminally truncated gI derivatives from the thymidine kinase locus. The effect on gE-gI-mediated cell-to-cell spread was assessed by plaque assay employing computer-assisted image analysis (software available at http://www.androclus.vet.uu.nl/spotter/spotter.htm+ ++). Allotopic complementation with intact gI fully restored plaque size. Deletion of the C-terminal 11 residues of gI did not affect cell-to-cell spread, whereas deletion of the complete cytoplasmic tail reduced plaque size by only 35%. Mutants expressing gI166, roughly corresponding to the N-terminal half of the ectodomain, displayed a small-plaque phenotype. Nevertheless, their plaques were reproducibly larger than those of matched gI-deficient controls, indicating that the gE-gI166 hetero-oligomer, though crippled, is still able to mediate cell-to-cell spread. Our data demonstrate that plaque analysis provides a reliable and convenient tool to measure and quantitate gE-gI function in vitro.  (+info)

Mutations in the conserved carboxy-terminal hydrophobic region of glycoprotein gB affect infectivity of herpes simplex virus. (5/106)

Glycoprotein gB is the most highly conserved glycoprotein in the herpesvirus family and plays a critical role in virus entry and fusion. Glycoprotein gB of herpes simplex virus type 1 contains a hydrophobic stretch of 69 aa near the carboxy terminus that is essential for its biological activity. To determine the role(s) of specific amino acids in the carboxy-terminal hydrophobic region, a number of amino acids were mutagenized that are highly conserved in this region within the gB homologues of the family HERPESVIRIDAE: Three conserved residues in the membrane anchor domain, namely A786, A790 and A791, as well as amino acids G743, G746, G766, G770 and P774, that are non-variant in Herpesviridae, were mutagenized. The ability of the mutant proteins to rescue the infectivity of the gB-null virus, K082, in trans was measured by a complementation assay. All of the mutant proteins formed dimers and were incorporated in virion particles produced in the complementation assay. Mutants G746N, G766N, F770S and P774L showed negligible complementation of K082, whereas mutant G743R showed a reduced activity. Virion particles containing these four mutant glycoproteins also showed a markedly reduced rate of entry compared to the wild-type. The results suggest that non-variant residues in the carboxy-terminal hydrophobic region of the gB protein may be important in virus infectivity.  (+info)

Identification and characterization of bovine herpesvirus type 5 glycoprotein H gene and gene products. (6/106)

Bovine herpesvirus type 5 (BHV-5) is the causative agent of a fatal meningo-encephalitis in calves and is closely related to BHV-1 which causes infectious bovine rhinotracheitis. The gene encoding BHV-5 glycoprotein gH was sequenced. A high degree of conservation was found between BHV-1 and BHV-5 deduced gH amino acid sequences (86. 4%), which is also observed for all alphaherpesvirus gH sequences. Transcriptional analysis revealed a 3.1 kb mRNA as the specific gH transcript which was detected 2 h post-infection (p.i.). Twelve out of twenty-one MAbs directed against BHV-1 gH immunoprecipitated a 108-110 kDa glycoprotein, which was then designated BHV-5 gH. Synthesis and intracellular processing of BHV- 5 gH was analysed in infected MDBK cells using gH cross-reacting MAbs. Glycoprotein gH was expressed as a beta-gamma protein, detected by radioimmunoprecipitation as early as 3 h p.i. Glycosylation studies indicated that BHV-5 gH contains N-linked carbohydrates which are essential for the recognition of the protein by the MAbs. This suggests that N-linked glycans are involved in protein folding or are targets for the gH cross-reacting MAbs. Plaque- reduction neutralization assays showed that at least one BHV-1 gH antigenic domain is lacking in BHV-5 which may possibly relate to in vivo differences in virus tropism.  (+info)

Bovine herpesvirus 5 glycoprotein E is important for neuroinvasiveness and neurovirulence in the olfactory pathway of the rabbit. (7/106)

Glycoprotein E (gE) is important for full virulence potential of the alphaherpesviruses in both natural and laboratory hosts. The gE sequence of the neurovirulent bovine herpesvirus 5 (BHV-5) was determined and compared with that of the nonneurovirulent BHV-1. Alignment of the predicted amino acid sequences of BHV-1 and BHV-5 gE open reading frames showed that they had 72% identity and 77% similarity. To determine the role of gE in the differential neuropathogenesis of BHV-1 and BHV-5, we have constructed BHV-1 and BHV-5 recombinants: gE-deleted BHV-5 (BHV-5gEDelta), BHV-5 expressing BHV-1 gE (BHV-5gE1), and BHV-1 expressing BHV-5 gE (BHV-1gE5). Neurovirulence properties of these recombinant viruses were analyzed using a rabbit seizure model (S. I. Chowdhury et al., J. Comp. Pathol. 117:295-310, 1997) that distinguished wild-type BHV-1 and -5 based on their differential neuropathogenesis. Intranasal inoculation of BHV-5 gEDelta and BHV-5gE1 produced significantly reduced neurological signs that affected only 10% of the infected rabbits. The recombinant BHV-1gE5 did not invade the central nervous system (CNS). Virus isolation and immunohistochemistry data suggest that these recombinants replicate and spread significantly less efficiently in the brain than BHV-5 gE revertant or wild-type BHV-5, which produced severe neurological signs in 70 to 80% rabbits. Taken together, the results of neurological signs, brain lesions, virus isolation, and immunohistochemistry indicate that BHV-5 gE is important for efficient neural spread and neurovirulence within the CNS and could not be replaced by BHV-1 gE. However, BHV-5 gE is not required for initial viral entry into olfactory pathway.  (+info)

Cellular expression of alphaherpesvirus gD interferes with entry of homologous and heterologous alphaherpesviruses by blocking access to a shared gD receptor. (8/106)

Several human and animal alphaherpesviruses can enter cells via human herpesvirus entry mediator C (HveC), a receptor for viral glycoprotein D (gD). In previous studies with cells expressing unknown entry mediators, cellular expression of alphaherpesvirus gD was shown to inhibit entry of the homologous virus and sometimes also of heterologous alphaherpesviruses. To investigate the mechanism of gD-mediated interference and the basis for cross-interference among alphaherpesviruses, HveC was expressed in cells as the sole entry mediator, in the presence or absence of one of the gDs encoded by herpes simplex virus type 1, pseudorabies virus, or bovine herpesvirus type 1. Cells expressing HveC alone were highly susceptible to entry of all three viruses, whereas cells coexpressing HveC and any one of the gDs were at least partially resistant to infection by each virus. Coexpression of gD with HveC did not cause reduced levels of cell-surface HveC but the HveC had reduced ability to bind to exogenous gD. Coimmunoprecipitation experiments revealed that HveC was complexed with gD in lysates of cells expressing both. Thus, cellular expression of gD can interfere with alphaherpesvirus entry by blocking ligand-binding sites of the gD receptor(s) used for entry and cross-interference can occur because different forms of alphaherpesvirus gD can compete for shared entry receptors.  (+info)

in Archives of Virology (2014), 159. Herpesviruses have mainly co-evolved with their hosts for millions of years. However, bovine herpesvirus 1 (BoHV1) and related ruminant alphaherpesviruses have been reported to cross the species barrier ... [more ▼]. Herpesviruses have mainly co-evolved with their hosts for millions of years. However, bovine herpesvirus 1 (BoHV1) and related ruminant alphaherpesviruses have been reported to cross the species barrier. Bubaline herpesvirus 1 (BuHV1) is an alphaherpesvirus closely related to BoHV1 and BoHV5. According to the serological cross-relationships between ruminant alphaherpesviruses, several surveys have studied the occurrence of BoHV1-related virus infection in wild and domestic ruminant species. Recent studies in Argentina showed an increase in serological prevalence against BoHV1 related viruses in water buffaloes (Bubalus bubalis) population. The aim of this study was to investigate the presence of related ruminant alphaherpesvirus in the ...
Looking for Feline herpesvirus? Find out information about Feline herpesvirus. of, relating to, or belonging to the Felidae , a family of predatory mammals, including cats, lions, leopards, and cheetahs, typically having a round head... Explanation of Feline herpesvirus
Symptoms, diagnosis, treatment and prevention of Feline Herpesvirus. Watery eyes, sneezing, a runny nose, seems like symptoms of the common cold; however, in our feline friends it can be indicative of Feline herpesvirus.
Previous work with HSV and the related alphaherpesviruses PrV and varicella-zoster virus has clearly demonstrated that gE-gI mediates or facilitates cell-to-cell spread, especially in solid tissues such as epithelium, in the nervous system, and with certain cultured cells, e.g., normal fibroblasts, epithelial cells, and neurons, cells that form extensive cell junctions (2, 24, 25). Other HSV glycoproteins function similarly in cell-to-cell spread, but unlike these glycoproteins, gE-gI functions in cell-to-cell spread but not in the entry of extracellular virions. Therefore, interactions between gE-gI and cell junctions may provide molecular details of how a herpesvirus moves directly from cell to cell.. When gE-gI was expressed in HEC-1A cells or in other epithelial cells by recombinant Ad vectors, the protein accumulated specifically along the lateral surfaces of cells and was not found on either the apical or basal surfaces or at tight junctions. Sorting of gE-gI to the basolateral surfaces of ...
The Herpesviridae are a family of viruses widely spread in nature that can infect a wide variety of species. After the primary infection, the human alphaherpesvirinae sub-family remains quiescent in the nerve ganglia from which it can periodically reactivate, causing clinical manifestations. Although spontaneous recurrences are possible, a wide variety of internal and external triggers may lead to transformation of the Herpes Simplex and Varicella-Zoster Viruses from a dormant to a proliferative state. Sunlight is a potent stimulus for the alphaherpesvirinae reactivation. The purpose of this paper is to analyze various features of this correlation and several steps you can take to lower your risk of triggering a herpes outbreak after sun exposure. Learning how to reduce the recurrence is extremely important and it is necessary: to perform a gradual and progressive sun exposure; to know what garments to wear; to know the environmental conditions of exposure; to know each skin phototype; to use a
Enquist LW, Scherer J. Alphaherpesviruses: parasites of theperipheral nervous system. Future Virology [Internet]. 2017;12 (10) :555-559.
Attenuated alphaherpesvirus vaccines are used widely in production and companion animals to help control disease. These vaccines help to prevent clini
Indirect immunofluorescence staining of MDBK cells infected with either BoHV-1, BoHV-5, CpHV-1, CvHV-1, CvHV-2, or the Anlier isolate. Cells were incubated unti
List of all the English words with 18 letters containing letter V. alphabaculoviruses, alphacoronaviruses, alphaherpesviruses, alumoklyuchevskite, anarcho-primitivism, anarchoprimitivism, anarcho-primitivist, anhydrovinblastine, anthroconservatism
PATEL, J R e DIDLICK, S. Epidemiology, disease and control of infections in ruminants by herpesviruses - An overview. J. S. Afr. Vet. Assoc. [online]. 2008, vol.79, n.1, pp.8-14. ISSN 2224-9435.. There are at least 16 recognised herpesviruses that naturally infect cattle, sheep, goats and various species of deer and antelopes. Six of the viruses are recognised as distinct alphaherpesviruses and 9 as gammaherpesviruses. Buffalo herpesvirus (BflHV) and ovine herpesvirus-1 (OvHV-1) remain officially unclassified. The prevalence of ruminant herpesviruses varies from worldwide to geographically restricted in distribution. Viruses in both subfamilies Alphaherpesvirinae and Gammaherpesvirinae cause mild to moderate and severe disease in respective natural or secondary ruminant hosts. Accordingly, the economic and ecological impact of the viruses is also variable. The molecular characteristics of some members have been investigated in detail. This has led to the identification of virulence-associated ...
The Herpesvirales naming system originated in 1973 and has been elaborated considerably since. All herpesviruses described since this system was adopted have been named in accordance with it. The recommended naming system specifies that each species name consists of three parts: a first word, a second word, and finally a number. The first word should be derived from the taxon (family or subfamily) to which its primary natural host belongs. The subfamily name is used for viruses from members of the family Bovidae or from primates (the virus name ending in -ine, e.g. bovine), and the host family name for other viruses (ending in -id, e.g. equid). Human herpesviruses have been treated as an exception (human rather than hominid). Following the host-derived term, species in the family Herpesviridae, which are divided into subfamilies Alphaherpesvirinae, Betaherpesvirinae, and Gammaherpesvirinae, will have the word alphaherpesvirus, betaherpesvirus, or gammaherpesvirus added, respectively. Species in ...
Severe tracheitis and bronchitis were identified in two fatal cases of respiratory disease affecting a flock of Gouldian finches (Erythrura [Chloebia] gouldiae). Intranuclear inclusion bodies in epithelial cells of the upper respiratory tract were identified in samples from two birds. Electron microscopic examination showed that the inclusions consisted of viral particles consistent in appearance with Herpesviridae. Degenerate PCR primers targeting a conserved region of the herpesviral-DNA-dependent DNA polymerase were used to amplify a region of DNA isolated from tissues with lesions from each animal. Nucleotide sequencing of the PCR products yielded identical viral sequences that were distinct from known herpesviruses. An analysis of sequence homology indicated that these gene segments appear to belong to a member of the subfamily Alphaherpesvirinae.
American Journal of Veterinary ResearchJanuary 2009, Vol. 70, No. 1, Pages 99-104Assessment of viremia associated with experimental primary feline herpesvirus infection or presumed herpetic recrudescence in catsHans D. Westermeyer, DVM, Sara M. Thomasy, DVM, PhD, Helen Kado-Fong, MS, David J. Maggs, BVScObjective-To detect feline herpesvirus type 1 (FHV-1) in blood of cats undergoing experimental primary…
ICP4 is an important factor regulating the life cycle of HSV1. This conserved protein has several molecular functions, including activation of expression of viral late gene transcripts and inhibition of immediate early genes. Although ICP4 and its Alphaherpesvirinae homologs (eg.: IE62 of VZV) have been subjects of various molecular studies, a complete view of their molecular function is lacking. Here we present the results of fold recognition and molecular modelling of ICP4 functional domains. The performed state-of-the-art bioinformatic fold recognition analysis identified a dual helix-turn-helix motif as a binding module of repressor activities (so called region 2 domain). The mapping of distant homology identified that a segment responsible for activation of late gene promoters (region 4) exhibits folding of uracil DNA glycosylase (UDG), but seems to be a non-functional homolog of UDG. Potential implications of the results are discussed ...
During infection of neurons by alphaherpesviruses including Pseudorabies virus (PRV) and Herpes simplex virus type 1 (HSV-1) viral nucleocapsids assemble in the cell nucleus, become enveloped in the cell body then traffic into and down axons to nerve termini for spread to adjacent epithelia. The viral membrane protein US9p and the membrane glycoprotein heterodimer gE/gI play critical roles in anterograde spread of both HSV-1 and PRV, and several models exist to explain their function. Biochemical studies suggest that PRV US9p associates with the kinesin-3 motor KIF1A in a gE/gI-stimulated manner, and the gE/gI-US9p complex has been proposed to recruit KIF1A to PRV for microtubule-mediated anterograde trafficking into or along the axon. However, as loss of gE/gI-US9p essentially abolishes delivery of alphaherpesviruses to the axon it is difficult to determine the microtubule-dependent trafficking properties and motor-composition of Δ(gE/gI-US9p) particles. Alternatively, studies in HSV-1 have ...
Elsa always had runny eyes. The two-year-old cat had been rescued as a kitten by Penn Vet student Jennifer Bortree. Elsa was treated at the time for feline herpesvirus (FHV-1), one of the most common causes of upper respiratory infections in cats. Among other symptoms, FHV-1 infection often impacts the eyes.. ...
Canine herpesvirus (CHV) is in the order Herpesvirales, family Herpesviridae, subfamily Alphaherpesvirinae, genus Varicellovirus as the species Canid alphaherpesvirus 1. As a typical alphaherpesvirus, it shares many of the properties common to this group of viruses. As such, most infections are subclinical and the virus establishes latency in the host for the life of the animal. Virus will recrudesce for unknown reasons at unpredictable intervals in many instances with no overt signs. It is through this mechanism that the virus spreads and persists in the canine population. The most significant clinical findings linked to CHV infections are respiratory signs, ocular lesions and neonatal mortality which is by far the issue of most concern. Neonatal mortalities most frequently occur within the first two weeks of life. The etiology of this syndrome is the infection of a sero-negative pregnant dog late in gestation so that there is infectious virus in the environment and with too little time for ...
Research data were analyzed by Pearson Correlation Coefficient, and stepwise regression procedures. Does the use of antibiotics in food-producing animals contribute to the problem? The diagnosis of Pachecos disease was based on the finding of characteristic histologic lesions and in many cases on the detection of herpesvirus virions by electron microscopy or the detection of herpesvirus-infected cells in impression smears of spleen or liver by use of a fluorescent antibody conjugate prepared against a PsHV isolated from a bird with Pachecos disease. Regular monitoring for treatment-emergent PN, sensible detection and intervention are relevant to prevent the development of more severe PN (Table 3).74,78 The NCI CTC definition of PN is commonly used in clinical routine, but should be used with neuropathy-specific patient-completed questionnaires, such as the whole or the reduced Total Neuropathy Score (Table 4).79 However, a need remains for more sensitive assessment tools that focus on MM ...
matrin 3 an acidic protein that is a component of the nuclear matrix. May play a role in transcription or may interact with other nuclear matrix proteins to form the internal fibrogranular network. Note: This description may include information from UniProtKB ...
The feline herpesvirus (FHV) is responsible for nasty respiratory infections in cats, and the disease can become chronic just like its human-specific counterpart. You arent at any risk of ...
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Reindeer are way more profound than Santas sleigh-pullers! Theyve been fascinating humans since prehistory! Learn more about rich symbolic reindeer meaning here.
This page includes the following topics and synonyms: Herpesviridae, Herpesviridae Infection, Herpes Family of Viruses, Herpesvirus, Human Herpes Virus, Alphaherpesvirus, Alphaherpesvirinae, Betaherpesvirinae, Betaherpesvirus, Gammaherpesvirinae.
This page includes the following topics and synonyms: Herpesviridae, Herpesviridae Infection, Herpes Family of Viruses, Herpesvirus, Human Herpes Virus, Alphaherpesvirus, Alphaherpesvirinae, Betaherpesvirinae, Betaherpesvirus, Gammaherpesvirinae.
Footnotes:. 1 Fraunfelder FW. Ocular side effects associated with dietary supplements and herbal medicines. Drugs of Today. 2005;41(8):537-545.. 2 West AL, Oren GA, and Moroi SE. Evidence for the use of nutritional supplements and herbal medicines in common eye diseases. American Journal of Ophthalmology. 2006;141(1):157-166.. 3 Bartlett H and Eperjesi F. Possible contraindications and adverse reactions associated with the use of ocular nutritional supplements. Ophthal Physiol Opt. 2005;25:179-194.. 4 Schwartz SG and Chavis PS. Dietary supplements and the ophthalmologist. Comp Ophthalmol Update. 2005;6(3):153-159.. 5 Maggs DJ, Collins BK, Thorne JG, and Nasisse MP. Effects of L-lysine and L-arginine on in vitro replication of feline herpesvirus type-1. AJVR. 2000;61(12):1474-1478.. 6 Maggs DJ, Nasisse MP, and Kass PH. Efficacy of oral supplementation with l-lysine in cats latently infected with feline herpesvirus. AJVR. 2003;64(1):37-42.. 7 Maggs DJ, Nasisse MP, and Kass PH. Efficacy of oral ...
Veterinary Microbiology and Microbial Disease - Google Books ResultP. J. Quinn, B. K. Markey, F. C. Leonard - ‎2011 - 400 pages - MedicalDetection of canine herpesvirus DNA in the ganglionic neurons and the lymph ... Protection of puppies against canine herpesvirus by vaccination of the dams. ...
Early lesions in these cultures (3-10 hours after virus inoculation) consisted of nucleolar sweating and disruption; there was loss of RNA from the nucleo-lus and segregation of nueleolar components (demonstrated by electron microscopy). During the middle phase (10-34 hours) three types of eosinophilic intranuclear inclusion appeared; the inclusions seemed to be formed by progressive transformations. Viral antigen was demonstrated in the nucleus by immuno-fluorescent staining, with viral Show moreEarly lesions in these cultures (3-10 hours after virus inoculation) consisted of nucleolar sweating and disruption; there was loss of RNA from the nucleo-lus and segregation of nueleolar components (demonstrated by electron microscopy). During the middle phase (10-34 hours) three types of eosinophilic intranuclear inclusion appeared; the inclusions seemed to be formed by progressive transformations. Viral antigen was demonstrated in the nucleus by immuno-fluorescent staining, with viral replication ...
4/29/13: Jordan Pacheco crushes a ground-rule double to left center, scoring Josh Rutledge and Tyler Chatwood in the top of the fourth
ANSWER: Canine herpesvirus is another common cause of fading puppy syndrome. This virus can be caught from the mother in utero or from infected fluids after birth. Sometimes puppies appear to be healthy at birth but die suddenly in the first few weeks of life.
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Introduction: : In man, latent herpes simplex virus-1 has been detected in ciliary ganglia. Infection of bulbar conjunctiva and cornea with feline herpesvirus-1 (FHV-1) would allow FHV-1 access to the ciliary ganglia. Purpose: : Determine if active FHV-1 can be isolated from the ciliary ganglia of experimentally infected cats. Methods: : Twelve, juvenile, specific pathogen free (SPF) cats were inoculated with 1x105 TCID50 of the C27 strain of FHV-1 via the ocular and nasal routes. Three, juvenile, SPF cats served as controls. Four infected and 1 control cat were euthanized on day 6 (all animals demonstrating clinical signs), 10 (peak of ocular and respiratory signs), and 30 (virus latent with resolution of clinical signs). The right ciliary ganglia were harvested, placed in 800µl Bovarnicks solution, and homogenized. Cultures were performed in duplicate. A 100µl aliquot of filtered tissue extract was inoculated onto monolayers of Crandall Reese feline kidney cells. The cultures were examined ...
In adult dogs, the virus infects the reproductive tract, which allows it to be sexually transmitted or passed to puppies during birth. The disease can cause abortion, stillbirths, and infertility. It is also an infrequent cause of kennel cough. However, sexual contact is not the primary cause of transmission. Most adult dogs become infected by inhaling the virus via airborne particles spread by coughs or sneezes. It can also be contracted by drinking from a contaminated water bowl, or even just by sniffing or licking another dog that is shedding the virus.. Like other types of herpesvirus, previously infected dogs can from time to time release the virus in vaginal secretions, penile secretions, and discharge from the nose. Raised sores in the vagina or on the penis may be seen during these times. Spread of the disease is controlled by not breeding dogs known to have it. Serology can show what dogs have been exposed (although not all of them will be releasing the virus at that time). Serological ...
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In addition to Matrin3 and Raver1, we found a number of other interesting factors that bound to PTB RRM2 and were sensitive to the Y247Q mutation (Joshi et al, 2011), suggesting that their interactions might also be mediated by PRI motifs (Fig 1). These included splicing regulators, proteins involved in the co‐transcriptional‐dependent regulation of splicing and 3′ end processing factors. We did not identify a number of previously identified PTB interactors, including hnRNP‐L, PSF, hnRNPA1, hnRNPA2B1, hnRNPC and MRG15 (Patton et al, 1993; Hahm et al, 1998; Luco et al, 2010; King et al, 2014), although we did identify the helicase DDX3X (King et al, 2014). The lack of overlap could be because we focused on proteins that interact primarily via RRM2. Interesting novel PTB interactors with possible relevance for PTBs splicing regulatory activities include KIAA1967/DBC1/CCAR2 and its paralogue CCAR1, both of which bound strongly to PTB RRM2 in a manner that was sensitive to the PTB Y247Q ...
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Hi! I just found your blog too via some other blog that I cant remember right now. Even tho its April I had to read this headless reindeer post cuz I thought maybe you did what I did about 23 years ago ... to my son... who was 3 or 4 at the time. I had a buffet table display set up with a Santa, sleigh and a couple of plastic reindeer for the holidays. One of the reindeers heads became mysteriously disconnected from his body but if you were real careful you could set the head on the body just so and it would stay. Unless you bumped the table or touched it. For some reason my son was scared to death of the headless reindeer. Which struck me as insanely funny. I know, Im demented and really really mean. But my son made the cutest funniest little screechy noises when I even reached for the headless reindeer. I couldnt resist. I tried (not very hard) to resist but I just couldnt. I am demented and mean but I dont have a blog so nobody knows - except my son. And I like to remind him about that ...
Hi! I just found your blog too via some other blog that I cant remember right now. Even tho its April I had to read this headless reindeer post cuz I thought maybe you did what I did about 23 years ago ... to my son... who was 3 or 4 at the time. I had a buffet table display set up with a Santa, sleigh and a couple of plastic reindeer for the holidays. One of the reindeers heads became mysteriously disconnected from his body but if you were real careful you could set the head on the body just so and it would stay. Unless you bumped the table or touched it. For some reason my son was scared to death of the headless reindeer. Which struck me as insanely funny. I know, Im demented and really really mean. But my son made the cutest funniest little screechy noises when I even reached for the headless reindeer. I couldnt resist. I tried (not very hard) to resist but I just couldnt. I am demented and mean but I dont have a blog so nobody knows - except my son. And I like to remind him about that ...
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Alphaherpesvirinae. Genus:. Varicellovirus. Species:. Canine herpesvirus 1 (CHV-1). Canine herpesvirus (CHV) is a virus of the ...
Subfamily Alphaherpesvirinae Genus Simplexvirus Ateline herpesvirus 1, spider monkey herpesvirus. Bovine herpesvirus 2 causes ... These are zoonotic infections: In animal virology, the best known herpesviruses belong to the subfamily Alphaherpesvirinae. ... Group: dsDNA Order: Herpesvirales Family: Herpesviridae Sub-Family: Alphaherpesvirinae Genus: Iltovirus Gallid herpesvirus 1 ... In this report the family Herpesviridae was divided into 3 subfamilies (Alphaherpesvirinae, Betaherpesvirinae and ...
McGeoch DJ, Cook S (1994). "Molecular phylogeny of the alphaherpesvirinae subfamily and a proposed evolutionary timescale". J ...
The alphaherpesvirinae separated from the branch leading to the betaherpesvirinae and gammaherpesvirinae about 180 million ... McGeoch DJ, Cook S (1994). "Molecular phylogeny of the alphaherpesvirinae subfamily and a proposed evolutionary timescale". J ...
Unterfamilie Alphaherpesvirinae. *Genus Iltovirus. *Genus Mardivirus. *Genus Scutavirus. *Genus Simplexvirus. *Genus ...
Anatid herpesvirus 1 is under the Herpesvirales order, the Herpesviridae family, the Alphaherpesvirinae subfamily. The genus ...
... is a virus species of the Varicellovirus genus and Alphaherpesvirinae subfamily. It causes ...
Virus varicella-zoster adalah virus penyebab cacar air dan cacar ular (herper zoster).[1] Inang dari virus ini hanya terbatas pada manusia dan primata (simian). Stuktur partikel virus (virion) varicella-zoster berukuran 120-300 nm.[2] Genom virus ini berukuran 125 kb (kilo-basa) dan mengandung sedikitnya 69 daerah yang mengkodekan gen-gen tertentu.[1] Virion terdiri dari glikoprotein, kapsid, amplop (selubung) virus, dan nukleokapsid yang melindungi bagian inti berisi DNA genom utas ganda.[1] Bagian nukleokapsid berbentuk ikosahedral, berdiameter 100-110 nm, dan terdiri dari 162 protein yang disebut kapsomer.[2] Virus ini akan mengalami inaktivasi pada suhu 56-60 °C dan menjadi tidak berbahaya apabila bagian amplop (selubung) dari virus ini rusak.[1] Penyebaran virus ini dapat terjadi melalui pernapasan.[1]. ...
The stages of replication are similar to other viruses in Alphaherpesvirinae, but there are a few differences. After the virus ... Mardivirus is a genus of viruses in the order Herpesvirales, in the family Herpesviridae, in the subfamily Alphaherpesvirinae. ... Group: dsDNA Order: Herpesvirales Family: Herpesviridae Sub-Family: Alphaherpesvirinae Genus: Mardivirus Anatid herpesvirus 1 ...
... is a genus of viruses in the order Herpesvirales, in the family Herpesviridae, in the subfamily Alphaherpesvirinae ... Group: dsDNA Order: Herpesvirales Family: Herpesviridae Sub-Family: Alphaherpesvirinae Genus: Simplexvirus Ateline herpesvirus ...
... is a genus of viruses in the order Herpesvirales, in the family Herpesviridae, in the subfamily Alphaherpesvirinae. ... Group: dsDNA Order: Herpesvirales Family: Herpesviridae Sub-Family: Alphaherpesvirinae Genus: Scutavirus Chelonid herpesvirus 5 ...
... is a genus of viruses in the order Herpesvirales, in the family Herpesviridae, in the subfamily Alphaherpesvirinae. ... Group: dsDNA Order: Herpesvirales Family: Herpesviridae Sub-Family: Alphaherpesvirinae Genus: Iltovirus Gallid herpesvirus 1 ...
In this report, the family Herpesviridae was divided into 3 subfamilies (alphaherpesvirinae, betaherpesvirinae and ...
... (var′i-sel′ō-vi′rŭs) is a genus of viruses belonging to subfamily Alphaherpesvirinae, a member of family ...
Simian varicella virus (SVV) is a varicellovirus that, like HSV-1 and HSV-2, belongs to the alphaherpesvirinae subfamily of ...
However, infection with human herpes simplex virus type-1, a member of the alphaherpesvirinae subfamily, did not provide ...
In this report the family Herpesviridae was divided into 3 subfamilies (Alphaherpesvirinae, Betaherpesvirinae and ...
This is different from Alphaherpesvirinae, which establish latency in neurons, and Gammaherpesvirinae, which establish latency ...
... subfamily Alphaherpesvirinae, and genus Varicellovirus. VZV is in Group I of the Baltimore Classification because it is a dsDNA ...
... is caused by a virus with icosahedral symmetry and belongs to the subfamily Alphaherpesvirinae within the family Herpesviridae ...
... namely Alphaherpesvirinae, Betaherpesvirinae and Gammaherpesvirinae. Gammaherpesviruses belong to four separate genera: the ...
Alphaherpesvirinae Genus: Iltovirus Gallid herpesvirus 1 Psittacid herpesvirus 1 Genus: Mardivirus Anatid herpesvirus 1 ...
... 1 of subfamily Alphaherpesvirinae and genus Varicellovirus Bovine herpesvirus 2 of subfamily ... Alphaherpesvirinae and genus Simplexvirus Bovine herpesvirus 3 Bovine herpesvirus 4 of subfamily Gammaherpesvirinae and genus ... Rhadinovirus Bovine herpesvirus 5 of subfamily Alphaherpesvirinae and genus Varicellovirus Bovine herpesvirus 6 of subfamily ...
... alphaherpesvirinae MeSH B04.909.204.382.100.374 --- infectious laryngotracheitis-like viruses MeSH B04.909.204.382.100.374.450 ... alphaherpesvirinae MeSH B04.280.382.100.374 --- infectious laryngotracheitis-like viruses MeSH B04.280.382.100.374.450 --- ...
... 1 of the subfamily Alphaherpesvirinae and genus Varicellovirus Equine herpesvirus 2 of the subfamily ... 3 of the subfamily Alphaherpesvirinae and genus Varicellovirus Equine herpesvirus 4 of the subfamily Alphaherpesvirinae and ... 8 of the subfamily Alphaherpesvirinae and genus Varicellovirus Equine herpesvirus 9 of the subfamily Alphaherpesvirinae and ... 5 of the subfamily Gammaherpesvirinae and genus Rhadinovirus Equine herpesvirus 6 of the subfamily Alphaherpesvirinae and genus ...
Alphaherpesvirinae at the US National Library of Medicine Medical Subject Headings (MeSH) Viralzone: Alphaherpesvirinae ICTV ... Alphaherpesvirinae is a subfamily of Herpesviridae, primarily distinguished by reproducing more quickly than other subfamilies ... In animal virology the most important herpesviruses belong to the Alphaherpesvirinae. Pseudorabies virus is the causative agent ... Group: dsDNA Order: Herpesvirales Family: Herpesviridae Sub-Family: Alphaherpesvirinae Genus: Iltovirus Gallid herpesvirus 1 ...
Alphaherpesvirinae. The α-herpesviruses are characterized by their ability to establish latency in neurons, to infect a variety ... The Herpesviridae family contains three subfamilies: Alphaherpesvirinae, Betaherpesvirinae, and Gammaherpesvirinae. Members of ... The genus Mardivirus of the Alphaherpesvirinae subfamily contains gallid alphaherpesvirus 2 (GaHV-2), or MDV, which infects ...
Alphaherpesvirinae. Genus:. Varicellovirus. Species:. Canine herpesvirus 1 (CHV-1). Canine herpesvirus (CHV) is a virus of the ...
Infectious Pustular Vulvovaginitis (IPV) (Bovine Herpesvirus type 1) (Herpesviridae; Alphaherpesvirinae; Varicellovirus) - ... Infectious Pustular Vulvovaginitis (IPV) (Bovine Herpesvirus type 1) (Herpesviridae; Alphaherpesvirinae; Varicellovirus) - ...
Unterfamilie Alphaherpesvirinae. *Genus Iltovirus. *Genus Mardivirus. *Genus Scutavirus. *Genus Simplexvirus. *Genus ...
Alphaherpesvirinae. Genus. Iltovirus. Genus. Mardivirus. Genus. Simplexvirus. Genus. Varicellovirus. Subfamily. ...
Alphaherpesvirinae, genus: Simplexvirus). This designation is based on virologic characteristics and serologic cross-reactivity ...
Virus varicella-zoster adalah virus penyebab cacar air dan cacar ular (herper zoster).[1] Inang dari virus ini hanya terbatas pada manusia dan primata (simian). Stuktur partikel virus (virion) varicella-zoster berukuran 120-300 nm.[2] Genom virus ini berukuran 125 kb (kilo-basa) dan mengandung sedikitnya 69 daerah yang mengkodekan gen-gen tertentu.[1] Virion terdiri dari glikoprotein, kapsid, amplop (selubung) virus, dan nukleokapsid yang melindungi bagian inti berisi DNA genom utas ganda.[1] Bagian nukleokapsid berbentuk ikosahedral, berdiameter 100-110 nm, dan terdiri dari 162 protein yang disebut kapsomer.[2] Virus ini akan mengalami inaktivasi pada suhu 56-60 °C dan menjadi tidak berbahaya apabila bagian amplop (selubung) dari virus ini rusak.[1] Penyebaran virus ini dapat terjadi melalui pernapasan.[1]. ...
Alphaherpesvirinae:. Simplexvirus. human herpesvirus 1. Vertebrates. Varicellovirus. human herpesvirus 3. Vertebrates. ...
Cloning of the DNA of Alphaherpesvirinae Andrew Davison, Frazer Rixon. Pages 103-124 ...
Alphaherpesvirinae, Betaherpesvirinae, Betaherpesvirus, Gammaherpesvirinae. ...
Subfamily Alphaherpesvirinae (organism), Subfamily Alphaherpesvirinae, Subfamily alphaherpesvirinae, NOS. Swedish. ... Alphaherpesvirinae. Spanish. subfamilia: alphaherpesvirinae, subfamilia: virus del grupo herpes simplex, subfamilia ... Alphaherpesvirinae (C0206557) Definition (CSP). a subfamily of Herpesviridae; replicate rapidly and latent infection is often ... Types: Alphaherpesvirinae. *Simplexvirus (Herpes Simplex Virus). *Human Herpes Virus 1: Oral Herpes ...
CHARACTERISTICS: Herpesviridae, Alphaherpesvirinae; dsDNA, 100nm diameter, enveloped, icosahedral capsid SECTION II - HEALTH ...
In this study, egress of EHV-1, HSV-1, and ILTV was analyzed by EM and compared with previous results with PrV (19) to obtain a comprehensive picture of similarities and differences in alphaherpesvirus replication. Although cell lines of different origins (porcine, equine, chicken, and primate) were analyzed, no cell-specific effects on the ultrastructure of virus morphogenesis were detected; rather, morphogenesis generally proved to be very similar. Exit of capsids from the nucleus started by budding at the inner nuclear membrane, resulting in acquisition of a primary envelope. This event was observed for each of the four virus species investigated. Since virions apparently did not accumulate in the perinuclear cisterna, we assume that the passage through the two leaflets of the nucleus is a rapid process. Comparison of micrographs of the four viruses analyzed showed that, under identical preparative conditions, all virus particles within the perinuclear cisterna exhibit the same morphology. ...
Anatid herpesvirus 1 is under the Herpesvirales order, the Herpesviridae family, the Alphaherpesvirinae subfamily. The genus ...
Belongs to the alphaherpesvirinae HHV-1 UL4 family.Curated. Family and domain databases. Integrated resource of protein ... Viruses › dsDNA viruses, no RNA stage › Herpesvirales › Herpesviridae › Alphaherpesvirinae › Simplexvirus › Human herpesvirus 1 ...
Subfamily Alphaherpesvirinae Genus Simplexvirus Ateline herpesvirus 1, spider monkey herpesvirus. Bovine herpesvirus 2 causes ... These are zoonotic infections: In animal virology, the best known herpesviruses belong to the subfamily Alphaherpesvirinae. ... Group: dsDNA Order: Herpesvirales Family: Herpesviridae Sub-Family: Alphaherpesvirinae Genus: Iltovirus Gallid herpesvirus 1 ... In this report the family Herpesviridae was divided into 3 subfamilies (Alphaherpesvirinae, Betaherpesvirinae and ...
Categories: Alphaherpesvirinae Image Types: Photo, Illustrations, Video, Color, Black&White, PublicDomain, CopyrightRestricted ...
Alphaherpesvirinae. Genus: Simplexvirus. Species Herpes simplex virus 1 (HSV-1). Herpes simplex virus 2 (HSV-2). ...
The members of Alphaherpesvirinae encode gC that target complement. The gCs of HSV type 1 (gC-1) as well as type 2 (gC-2) have ... They are classified into three subfamilies - Alphaherpesvirinae, Betaherpesvirinae, and Gammaherpesvirinae - and all the ...
Subfamily Alphaherpesvirinae*Molecular analysis shows it to be a distinct lineage within the family ...
Sunlight is a potent stimulus for the alphaherpesvirinae reactivation. The purpose of this paper is to analyze various features ... After the primary infection, the human alphaherpesvirinae sub-family remains quiescent in the nerve ganglia from which it can ... After the primary infection, the human alphaherpesvirinae sub-family remains quiescent in the nerve ganglia from which it can ... Sunlight is a potent stimulus for the alphaherpesvirinae reactivation. The purpose of this paper is to analyze various features ...
1994) Molecular phylogeny of the alphaherpesvirinae subfamily and a proposed evolutionary timescale. J. Mol. Biol. 238:9-22. ...
Human herpesvirus 2 (alphaherpesvirinae) simplex 2 Human herpesvirus 3 (alphaherpesvirinae) Varicella-zoster Human herpesvirus ... Subfamily: Alphaherpesvirinae) Classification basis: biologic properties Betaherpesvirinae) Gammaherpsvirinae) Genus: several ...
  • In general, members of the Alphaherpesvirinae use the epithelium of the upper respiratory and/or genital tract as a preferential site for primary replication. (ugent.be)