Allopurinol
Gout Suppressants
Xanthine Oxidase
Gout
Uric Acid
Hyperuricemia
Excessive URIC ACID or urate in blood as defined by its solubility in plasma at 37 degrees C; greater than 0.42mmol per liter (7.0mg/dL) in men or 0.36mmol per liter (6.0mg/dL) in women. This condition is caused by overproduction of uric acid or impaired renal clearance. Hyperuricemia can be acquired, drug-induced or genetically determined (LESCH-NYHAN SYNDROME). It is associated with HYPERTENSION and GOUT.
Uricosuric Agents
Tumor Lysis Syndrome
Antimetabolites
Urate Oxidase
Hypoxanthines
Xanthine
A purine base found in most body tissues and fluids, certain plants, and some urinary calculi. It is an intermediate in the degradation of adenosine monophosphate to uric acid, being formed by oxidation of hypoxanthine. The methylated xanthine compounds caffeine, theobromine, and theophylline and their derivatives are used in medicine for their bronchodilator effects. (Dorland, 28th ed)
Free Radical Scavengers
Benzbromarone
Xanthine Dehydrogenase
Hypoxanthine
Drug Hypersensitivity
Drug Eruptions
Enzyme Inhibitors
Probenecid
The prototypical uricosuric agent. It inhibits the renal excretion of organic anions and reduces tubular reabsorption of urate. Probenecid has also been used to treat patients with renal impairment, and, because it reduces the renal tubular excretion of other drugs, has been used as an adjunct to antibacterial therapy.
Stevens-Johnson Syndrome
Rare cutaneous eruption characterized by extensive KERATINOCYTE apoptosis resulting in skin detachment with mucosal involvement. It is often provoked by the use of drugs (e.g., antibiotics and anticonvulsants) or associated with PNEUMONIA, MYCOPLASMA. It is considered a continuum of Toxic Epidermal Necrolysis.
Heartburn
Reactive oxygen species play an important role in the activation of heat shock factor 1 in ischemic-reperfused heart. (1/630)
BACKGROUND: The myocardial protective role of heat shock protein (HSP) has been demonstrated. Recently, we reported that ischemia/reperfusion induced a significant activation of heat shock factor (HSF) 1 and an accumulation of mRNA for HSP70 and HSP90. We examined the role of reactive oxygen species (ROSs) in the induction of stress response in the ischemic-reperfused heart. METHODS AND RESULTS: Rat hearts were isolated and perfused with Krebs-Henseleit buffer by the Langendorff method. Whole-cell extracts were prepared for gel mobility shift assay using oligonucleotides containing the heat shock element. Induction of mRNA for HSP70 and HSP90 was examined by Northern blot analysis. Repetitive ischemia/reperfusion, which causes recurrent bursts of free radical generation, resulted in burst activation of HSF1, and this burst activation was significantly reduced with either allopurinol 1 mmol/L (an inhibitor of xanthine oxidase) or catalase 2x10(5) U/L (a scavenger of H2O2). Significant activation of HSF1 was observed on perfusion with buffer containing H2O2 150 micromol/L or xanthine 1 mmol/L plus xanthine oxidase 5 U/L. The accumulation of mRNA for HSP70 or HSP90 after repetitive ischemia/reperfusion was reduced with either allopurinol or catalase. CONCLUSIONS: Our findings demonstrate that ROSs play an important role in the activation of HSF1 and the accumulation of mRNA for HSP70 and HSP90 in the ischemic-reperfused heart. (+info)Acute vasoconstriction-induced insulin resistance in rat muscle in vivo. (2/630)
Insulin-mediated changes in blood flow are associated with altered blood flow distribution and increased capillary recruitment in skeletal muscle. Studies in perfused rat hindlimb have shown that muscle metabolism can be regulated by vasoactive agents that control blood flow distribution within the hindlimb. In the present study, the effects of a vasoconstrictive agent that has no direct effect on skeletal muscle metabolism but that alters perfusion distribution in rat hindlimb was investigated in vivo to determine its effects on insulin-mediated vascular action and glucose uptake. We measured the effects of alpha-methylserotonin (alpha-met5HT) on mean arterial blood pressure, heart rate, femoral blood flow, hindlimb vascular resistance, and glucose uptake in control and euglycemic insulin-clamped (10 mU x min(-1) x kg(-1)) anesthetized rats. Blood flow distribution within the hindlimb muscles was assessed by measuring the metabolism of 1-methylxanthine (1-MX), an exogenously added substrate for capillary xanthine oxidase. Alpha-met5HT (20 microg x min(-1) x kg(-1)) infusion alone increased mean arterial blood pressure by 25% and increased hindlimb vascular resistance but caused no change in femoral blood flow. These changes were associated with decreased hindlimb 1-MX metabolism indicating less capillary flow. Insulin infusion caused decreased hindlimb vascular resistance that was associated with increased femoral blood flow and 1-MX metabolism. Treatment with alpha-met5HT infusion commenced before insulin infusion prevented the increase in femoral blood flow and inhibited the stimulation of 1-MX metabolism. Alpha-met5HT infusion had no effect on hindlimb glucose uptake but markedly inhibited the insulin stimulation of glucose uptake (P < 0.05) and was associated with decreased glucose infusion rates to maintain euglycemia (P < 0.05). A significant correlation (P < 0.05) was observed between 1-MX metabolism and hindlimb glucose uptake but not between femoral blood flow and glucose uptake. The results indicate that in vivo, certain types of vasoconstriction in muscle such as elicited by 5HT2 agonists, which prevent normal insulin recruitment of capillary flow, cause impaired muscle glucose uptake. Moreover, if vasoconstriction of this kind results from stress-induced increase in sympathetic outflow, then this may provide a clue as to the link between hypertension and insulin resistance that is often observed in humans. (+info)The role of free serum tryptophan in the biphasic effect of acute ethanol administration on the concentrations of rat brain tryptophan, 5-hydroxytryptamine and 5-hydroxyindol-3-ylacetic acid. (3/630)
1. Acute administration of ethanol exerts a biphasic effect on the concentrations of rat brain tryptophan, 5-hydroxytryptamine and 5-hydroxyindol-3-ylacetic acid. Both effects are associated with corresponding changes in the availability of circulating free tryptophan. 2. The initial increases in the above concentrations are prevented by ergotamine, are unaltered by allopurinol and are potentiated by theophylline, whereas the later decreases are prevented by both ergotamine and allopurinol. 3. It is suggested that the initial enhancement by ethanol of brain tryptophan metabolism is caused by catecholamine-mediated lipolysis followed by displacement of protein-bound serum tryptophan, whereas the activation of liver tryptophaan pyrrolase, which is produced by the same mechanism, leads to the later decreases in the brain concentrations of tryptophan and its metabolites. 4. The initial effects of ethanol can be reproduced by an equicaloric dose of sucrose, and a comparison of the two treatments alone could therefore be misleading. 5. The effects of ethanol on liver and brain tryptophan metabolism have also been examined in mice, and a comparison of the results with those previously reported suggests that the ethanol effects are strain-dependent. (+info)Value of Western blotting in the clinical follow-up of canine leishmaniasis. (4/630)
Specific serum antibody levels in Leishmania infantum-infected dogs treated with a combination of glucantime and allopurinol were estimated by indirect immunofluorescence and Western blotting. The sensitivity of Western blot was greater than that obtained with immunofluorescence titration. In general, both diagnostic methods concurred with the post-treatment clinical status of the animals. Clinical improvement of successfully treated dogs was related to lower immunofluorescence titers and simpler and/or less reactive immunodetection patterns in Western blotting. The recognition, by infected dogs, of certain low molecular weight antigens, particularly one of approximately 26 kDa, was restricted to pretreatment samples and a single animal in relapse thus apparently constituting an active infection marker. (+info)Intravenous glycine improves survival in rat liver transplantation. (5/630)
In situ manipulation by touching, retracting, and moving liver lobes gently during harvest dramatically reduces survival after transplantation (P. Schemmer, R. Schoonhoven, J. A. Swenberg, H. Bunzendahl, and R. G. Thurman. Transplantation 65: 1015-1020, 1998). The development of harvest-dependent graft injury upon reperfusion can be prevented with GdCl3, a rare earth metal and Kupffer cell toxicant, but it cannot be used in clinical liver transplantation because of its potential toxicity. Thus the effect of glycine, which prevents activation of Kupffer cells, was assessed here. Minimal dissection of the liver for 12 min plus 13 min without manipulation had no effect on survival (100%). However, gentle manipulation decreased survival to 46% in the control group. Furthermore, serum transaminases and liver necrosis were elevated 4- to 12-fold 8 h after transplantation. After organ harvest, the rate of entry and exit of fluorescein dextran, a dye confined to the vascular space, was decreased about twofold, indicating disturbances in the hepatic microcirculation. Pimonidazole binding, which detects hypoxia, increased about twofold after organ manipulation, and Kupffer cells isolated from manipulated livers produced threefold more tumor necrosis factor-alpha after lipopolysaccharide than controls. Glycine given intravenously to the donor increased the serum glycine concentration about sevenfold and largely prevented the effect of gentle organ manipulation on all parameters studied. These data indicate for the first time that pretreatment of donors with intravenous glycine minimizes reperfusion injury due to organ manipulation during harvest and after liver transplantation. (+info)Tissue distribution and characteristics of xanthine oxidase and allopurinol oxidizing enzyme. (6/630)
Tissue distribution and levels of allopurinol oxidizing enzyme and xanthine oxidase with hypoxanthine as a substrate were compared with supernatant fractions from various tissues of mice and from liver of mice, rats, guinea pigs and rabbits. The allopurinol oxidizing enzyme activities in liver were quite different among the species and the sex difference of the enzyme activity only in mouse liver. In mice, the highest activity of allopurinol oxidizing enzyme was found in the liver with a trace value in lung, but the enzyme activity was not detected in brain, small intestine and kidney, while the highest activity of xanthine oxidase was detected in small intestine, lung, liver and kidney in that sequence. The allopurinol oxidizing enzyme activity in mouse liver supernatant fraction did not change after storage at -20 degrees C or dialysis against 0.1 M Tris-HCl containing 1.15% KCl, but the activity markedly decreased after dialysis against 0.1 M Tris-HCl. On the contrary, the xanthine oxidase was activated 2 to 3 times the usual activity after storage at -20 degrees C or dialysis of the enzyme preparation. These results indicated that allopurinol was hydroxylated to oxipurinol mainly by the enzyme which is not identical to xanthine oxidase in vivo. A possible role of aldehyde oxidase involved in the allopurinol oxidation in liver supernatant fraction was dicussed. (+info)Digestion and absorption of bovine milk xanthine oxidase and its role as an aldehyde oxidase. (7/630)
The effects of acidic and intestinal proteolytic environments on bovine milk xanthine oxidase (XO) activity were determined in order to evaluate the extent to which this enzyme was absorbed in biologically active form. The inhibition of XO by folic acid and the relative affinities of XO for the oxidation of palmitaldehyde, stearaldehyde, and xanthine were compared. The effects of acid and gastric juice on XO activity were measured by incubating purified enzyme, and non-purified enzyme (milk), in buffers ranging in pH from 2 to 9. Fresh gastric juice was also incubated with milk. Increasing amounts of the enzyme were inactivated as the pH of the incubation mixture was reduced below pH 6.5. Below pH 3.5, the enzyme was completely inactivated. Gastric juice, pH juice incubated with milk. Milk XO activity was reduced 36% when mild was incubated with an equal volume of gastric juice. Homogenized milk had 59% less XO activity compared with raw molk. Fresh raw milk XO, homogenized milk XO, and purified XO were equally susceptible to inactivation by acid or gastric juice. After incubation of milk with gastric juice, or gastric juice followed by pancreatin, XO activity was associated with a macromolecule of 300,000 daltons molecular weight and subunits containg activity were not found. It was estimated that 0.00008% of the XO in the intestine was absorbed. Both folic acid and allopurinol inhibited XO activity in vitro. Allopurinol was 3.5 times more potent an inhibitor than folic acid. A large excess of dietary folic acid did not reduce rat liver or intestinal XO activity in vivo. XO had a much greater affinity for xanthine than for palmitaldehyde or stearaldehyde substrates. It was estimated that of 100 mg of XO in fresh raw milk, 41 mg remained after homogenization, 27 mg entered the intestine and only 20 ng were absorbed as intact enzyme. (+info)Optimization of allopurinol challenge: sample purification, protein intake control, and the use of orotidine response as a discriminative variable improve performance of the test for diagnosing ornithine carbamoyltransferase deficiency. (8/630)
BACKGROUND: The diagnosis of heterozygosity for X-linked ornithine carbamoyltransferase (OCT) deficiency has usually been based on measurement of the increase of orotate and orotidine excretion after an allopurinol load. We examined the choices of analyte, cutoff, and test conditions to obtain maximal test accuracy. METHODS: Urine orotate/orotidine responses to allopurinol load in 37 children (13 OCT-deficient and 24 non-OCT-deficient) and 24 women (7 at risk for carrier status and 17 not related to OCT-deficient children) were analyzed by liquid chromatography after sample purification by anion-exchange chromatography. Diagnostic accuracy was evaluated by nonparametric ROC curves. RESULTS: Sample purification was necessary to prevent interferences. Orotate and orotidine excretion increased with increased protein intake during the test. At a cutoff of 8 mmol orotidine/mol creatinine, sensitivity was 1.0 and specificity was 0. 92 in mild forms of OCT deficiency. Results in monoplex carrier women may differ greatly from those expected because of the genetics of this deficiency. CONCLUSIONS: Standardization of protein intake is required in the allopurinol loading test. A negative response in the face of clinical suspicion should be followed with a repeat test during a protein intake not <2.5 g x kg-1 x day-1. Measurements of orotidine provide better clinical sensitivity than measurements of orotate. (+info)
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Allopurinol
... was first marketed as a treatment for gout in 1966. Allopurinol is sold as an injection for intravenous use and as ... Allopurinol was marketed at the time by Burroughs Wellcome. Allopurinol is a generic drug sold under a variety of brand names, ... Allopurinol is in the xanthine oxidase inhibitor family of medications. Allopurinol was approved for medical use in the United ... "Allopurinol". Drug Information Portal. U.S. National Library of Medicine. "PRODUCT INFORMATION Allopurinol Tablets USP". U.S. ...
Allopurinol hypersensitivity syndrome
... typically occurs in persons with preexisting kidney failure.: 119 Weeks to months after ... ISBN 0-7216-2921-0. Wang CW, Dao RL, Chung WH (August 2016). "Immunopathogenesis and risk factors for allopurinol severe ... allopurinol is begun, the patient develops a morbilliform eruption: 119 or, less commonly, develops one of the far more serious ...
Hyperuricosuria
"Allopurinol". MedlinePlus, National Library of Medicine, US National Institutes of Health. 2016. Retrieved 24 December 2016. " ... Medications that may contribute to the cure or amelioration of hyperuricosuria include allopurinol which acts by inhibiting ...
C5H4N4S
Jawad AS (June 1987). "Alternatives to allopurinol". Annals of the Rheumatic Diseases. 46 (6): 493. doi:10.1136/ard.46.6.493-a ... comparative enzyme inhibition and protein binding studies with allopurinol, oxipurinol and 6-mercaptopurine". British Journal ...
Tisopurine
Jawad AS (June 1987). "Alternatives to allopurinol". Annals of the Rheumatic Diseases. 46 (6): 493. doi:10.1136/ard.46.6.493-a ... comparative enzyme inhibition and protein binding studies with allopurinol, oxipurinol and 6-mercaptopurine". British Journal ...
Amoxicillin
Allopurinol (gout treatment). Amoxicillin (α-amino-p-hydroxybenzyl penicillin) is a semisynthetic derivative of penicillin with ...
Lemelson-MIT Prize
allopurinol (Zyloprim), for gout. pyrimethamine (Daraprim), for malaria. trimethoprim (Septra), for meningitis, sepsis, and ...
Gout
Allopurinol blocks uric acid production, and is the most commonly used agent. Long term therapy is safe and well-tolerated and ... As of 2020, allopurinol is generally the recommended preventative treatment if medications are used. A number of other ... Lesinural plus allopurinol is not effective for tophi resolution. Potential side effects include kidney stones, anemia and ... It was approved in the United States for use together with allopurinol, among those who were unable to reach their uric acid ...
Gertrude B. Elion
Allopurinol (Zyloprim), for gout. Pyrimethamine (Daraprim), for malaria. Trimethoprim (Proloprim, Monoprim, others), for ...
Drug reaction with eosinophilia and systemic symptoms
Allopurinol is metabolized to oxipurinol, a product with a far slower renal excretion rate than its parent compound. Renal ... Allopurinol: Han Chinese, Korean, Thai, and European individuals expressing the HLA-B58:01 serotype have a higher incidence of ... These recommendations include: Allopurinol: The American College of Rheumatology guidelines for the management of gout ... This is evident in the most commonly cited medications that cause the DRESS viz., allopurinol, sulfasalazine, and minocycline, ...
Kidney stone disease
Allopurinol interferes with the production of uric acid in the liver. The drug is also used in people with gout or ... If this is not effective enough, thiazide diuretic, citrate, or allopurinol may be taken. It is recommended that soft drinks ... For people with hyperuricosuria and calcium stones, allopurinol is one of the few treatments that have been shown to reduce ... In those with calcium stones, drinking plenty of fluids, thiazide diuretics and citrate are effective as is allopurinol in ...
Benzbromarone
"Biochemical effectiveness of allopurinol and allopurinol-probenecid in previously benzbromarone-treated gout patients". ... Schepers GW (1981). "Benzbromarone therapy in hyperuricaemia; comparison with allopurinol and probenecid". The Journal of ... "Efficacy of allopurinol and benzbromarone for the control of hyperuricaemia. A pathogenic approach to the treatment of primary ... especially when allopurinol, a first-line treatment, fails or produces intolerable adverse effects. It is structurally related ...
2,8-Dihydroxyadenine
Allopurinol therapy appears to be effective. 2, 8-dihydroxyadenine formation can be easily controlled with allopurinol, which ...
Xanthine oxidase inhibitor
Purine analogues include allopurinol, oxypurinol, and tisopurine. Others include febuxostat, topiroxostat, and inositols ( ... Renaissance Half a Century after the Discovery of Allopurinol". Pharmacol. Rev. 58 (1): 87-114. doi:10.1124/pr.58.1.6. PMC ...
Agranulocytosis
Elisa Mari; Franco Ricci; Davide Imberti; Massimo Gallerani (June 2011). "Agranulocytosis: an adverse effect of allopurinol ... allopurinol, the antidepressants mianserin and mirtazapine, and some antipsychotics. The atypical antipsychotic clozapine in ...
Hyperuricemia
Prognosis is good with regular consumption of allopurinol.[citation needed] People with gout, and by inference hyperuricemia, ... Xanthine oxidase inhibitors, including allopurinol, febuxostat and topiroxostat, decrease the production of uric acid, by ...
Kidney disease
Xanthine oxidase inhibitors, like allopurinol, can cause nephropathy. Additional possible cause of nephropathy is due to the ...
Oxipurinol
It is an active metabolite of allopurinol and it is cleared renally. In cases of renal disease, this metabolite will accumulate ... the chief metabolite of allopurinol". The American Journal of Medicine. 45 (1): 69-77. doi:10.1016/0002-9343(68)90008-9. PMID ...
Xanthine oxidase
Because xanthine oxidase is a metabolic pathway for uric acid formation, the xanthine oxidase inhibitor allopurinol is used in ... Inhibitors of XO include allopurinol, oxypurinol, and phytic acid. It has also been found to be inhibited by flavonoids, ... such as conversion of allopurinol to oxypurinol). Inhibition of xanthine oxidase has been proposed as a mechanism for improving ... when xanthine oxidase was inhibited using allopurinol. Oxidative stress can be caused by hydroxyl free radicals and hydrogen ...
Stevens-Johnson syndrome
This has clinical relevance as it is agreed upon that prior to starting a medication such as allopurinol in a patient of ... Before treatment with allopurinol, the American College of Rheumatology guidelines for managing gout recommend HLA-B*58:01 ... Based on the Asian findings, similar studies in Europe showed 61% of allopurinol-induced SJS/TEN patients carried the HLA-B58 ( ... Wang CW, Dao RL, Chung WH (2016). "Immunopathogenesis and risk factors for allopurinol severe cutaneous adverse reactions". ...
Severe cutaneous adverse reactions
... allopurinol, nevirapine, and chlormezanone. Allopurinol appears in some studies to be the most common instigator of these ... For example, allopurinol is metabolized to oxipurinol, a product with a far slower renal excretion rate than its parent ... Allopurinol: The American College of Rheumatology guidelines for the management of gout recommend HLA-B*58:01 screening before ... The estimated mortality rate for the DRESS syndrome is about 10%. Allopurinol and sulfasalazine account for almost 66% of DRESS ...
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Porta C, Moroni M, Nastasi G (1994). "Allopurinol mouthwashes in the treatment of 5-fluorouracil-induced stomatitis". Am. J. ... Fluorouracil's efficacy is decreased when used alongside allopurinol, which can be used to decrease fluorouracil induced ... stomatitis through use of allopurinol mouthwash. The dihydropyrimidine dehydrogenase (DPD) enzyme is responsible for the ...
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In a female with reduced enzyme activity, an oral dose of allopurinol would be metabolized to oxypurinol ribonucleotide, which ... Historically, heterozygous females were often diagnosed using an allopurinol challenge. ...
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Allopurinol inhibits xanthine oxidase, the enzyme that breaks down mercaptopurine. Those taking allopurinol (often used to ... Several published studies have demonstrated that the use of allopurinol in combination with low dose 6-MP helps reduce 6-MP ... The dose should be reduced or allopurinol should be discontinued. ...
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Wang CW, Dao RL, Chung WH (2016). "Immunopathogenesis and risk factors for allopurinol severe cutaneous adverse reactions". ...
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01 and Stevens-Johnson syndrome/toxic epidermal necrolysis in patients taking allopurinol, and SLCO1B1 rs4149056 and myopathy ... ". "Annotation of CPIC Guideline for allopurinol and HLA-B". PharmGKB. "Annotation of CPIC Guideline for simvastatin and ...
Febuxostat
In 2010, before it became generic in the United States, it cost about US$160 per month as opposed to allopurinol which was ... In the UK, NICE has found that febuxostat has a higher cost/benefit ratio than allopurinol and on that basis recommended ... It is generally recommended only for people who cannot take allopurinol. When initially started, medications such as NSAIDs are ... Serious side effects include an increased risk of death as compared with allopurinol, Stevens-Johnson syndrome, and anaphylaxis ...
Apparicine
It inhibited xanthine oxidase as potently as allopurinol (IC50 = 0.65 μM). Catharanthine Pericine Tabernaemontanine Herbert ...
Primary myelofibrosis
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Xanthine oxidase inhibitor4
- In the study, the difference in Rotterdam Progression Score (RPS) between patients who received allopurinol, a xanthine oxidase inhibitor, and those who received placebo was 0.17, a difference that was not statistically significant. (medscape.com)
- A total of 6190 patients with gout and cardiovascular disease from 320 centers in North America were randomly assigned to receive febuxostat, a nonpurine xanthine oxidase inhibitor, or allopurinol, a purine base analogue, for a median of 2 years. (renalandurologynews.com)
- Some people with gout may still have a high uric acid level even though they are on a xanthine oxidase inhibitor such as allopurinol. (hopkinsarthritis.org)
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- Kidney and gout problems are caused due to increased uric acid levels and Allopurinol works by reducing the amount of uric acid made by the body. (pharmacie-crans.ch)
- People administered allopurinol for high uric acid levels happening as a result of cancer are at higher danger of developing kidney damage from allopurinol. (tomjonespharmacist.live)
- Allopurinol is used in the treatment of high uric acid levels associated with gout, kidney stones, and cancer treatment, it should not be utilized for asymptomatic high uric acid levels. (tomjonespharmacist.live)
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Renal16
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Doses5
- A maximum of 0.9 mg/dL of these oxypurines has been reported when the serum urate was lowered to less than 2 mg/dL by high doses of allopurinol tablets. (nih.gov)
- Oxipurinol, however, has a longer plasma half-life (approximately 15 hours) and therefore effective xanthine oxidase inhibition is maintained over a 24-hour period with single daily doses of allopurinol tablets. (nih.gov)
- A significant reduction in the serum urate concentration was observed for all allopurinol doses above the recommended dose. (nih.gov)
- Allopurinol doses were adjusted according to kidney function. (renalandurologynews.com)
- In dogs receiving higher doses of allopurinol, certain types of bladder stones (xanthine stones) may develop. (banderavetclinic.com)
Medication3
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Levels of uric acid3
- Allopurinol is also used to treat high levels of uric acid that builds up in the blood as tumors break down in people with certain types of cancer who are being treated with chemotherapy medications. (medlineplus.gov)
- Allopurinol may be utilized to decrease levels of uric acid in individuals with gout, kidney stones, or who are getting certain types of chemotherapy. (tomjonespharmacist.live)
- Allopurinol lowers levels of uric acid in the blood and urine. (tomjonespharmacist.live)
Production of uric acid1
- Allopurinol reduces the production of uric acid (a compound produced by the body related to gout and kidney stones), by preventing an enzyme called xanthine oxidase. (tomjonespharmacist.live)
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Effect of allopurinol1
- The results of the ALL-HEART study, which examined the effect of allopurinol in more than 5000 patients who had ischemic heart disease, may be published in the coming year. (medscape.com)
Treat gout1
- Allopurinol is used to treat gout (a type of arthritis in which uric acid, a naturally occurring substance in the body, builds up in the joints and causes sudden attacks of redness, swelling, pain, and heat in one or more joints). (medlineplus.gov)
Dose13
- Your doctor will probably start you on a low dose of allopurinol and gradually increase your dose, not more than once a week. (medlineplus.gov)
- To determine the efficacy and safety of increasing the allopurinol dose above the proposed creatinine clearance-based dose in patients with gout. (nih.gov)
- Patients with gout who had been receiving a stable dose of allopurinol for ≥ 1 month were recruited. (nih.gov)
- Forty-five patients had a serum urate concentration of ≥ 0.36 mmoles/liter, and the dose of allopurinol was increased in these patients. (nih.gov)
- Rashes developed in 3 patients, and either allopurinol was discontinued or dose escalation was ceased in these patients. (nih.gov)
- Patients receiving furosemide required a higher dose of allopurinol to achieve the target serum urate concentration. (nih.gov)
- Increasing the dose of allopurinol above the proposed creatinine clearance-based dose led to a significant reduction in the serum urate concentration. (nih.gov)
- In my non-expert opinion, the dose increase most likely to help you avoid waking up in pain isn't allopurinol but colchicine. (goutpal.net)
- The dose of allopurinol requires lowering in people with kidney illness and their kidney function kept an eye on a minimum of during the early stages of therapy. (tomjonespharmacist.live)
- Inform your doctor if you have diabetes or kidney dysfunction before taking Allopurinol as correction of your dose is needed. (canadiangeniusmeds.com)
- The dose of oral mercaptopurine (Purinethol) and azathioprine (Imuran) should be reduced as Allopurinol increases their blood levels. (canadiangeniusmeds.com)
- If your pet vomits or seems unwell after receiving allopurinol on an empty stomach, give the next dose with a small amount of food. (banderavetclinic.com)
- Un mois plus tard, le chat a été traité pour une lésion rénale aiguë suspectée, ce qui a entraîné une réduction de 50 % de la dose quotidienne totale d' allopurinol . (bvsalud.org)
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300mg1
- Can I take both Allopurinol 300mg & Indomethacin 25mg at the same time during an acute gout attack? (drugs.com)
Take allopurinol4
- To help you remember to take allopurinol, take it around the same time every day. (medlineplus.gov)
- Take allopurinol exactly as directed. (medlineplus.gov)
- Continue to take allopurinol even if you feel well. (medlineplus.gov)
- Can you take allopurinol and indomethacin at the same time? (drugs.com)
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Prescription2
- tell your doctor and pharmacist what prescription and nonprescription medications, vitamins, nutritional supplements, and herbal products you are taking while you are taking allopurinol. (medlineplus.gov)
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Lowers2
- A meta-analysis of small studies indicated that allopurinol lowers blood pressure to a modest extent and may reduce the risk for cardiovascular events in people with established cardiovascular disease. (medscape.com)
- Allopurinol lowers how much uric acid there is in an individual's body. (healthprep.com)
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- Coadministration with colchicine or an anti-inflammatory representative is recommended initially, in addition to gradually increasing the dosage of allopurinol. (tomjonespharmacist.live)
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Hyperuricemia2
- The authors report a case of lethal massive hepatic necrosis due to allopurinol in a patient with the asymptomatic hyperuricemia. (greenmedinfo.com)
- Allopurinol is a commonly used drug for treatment of high uric acid concentrations (called hyperuricemia) and gout. (drugsandgenes.com)
Oxidative stress5
- Xanthine oxidase produces oxidative stress and therefore we examined whether allopurinol improved endothelial dysfunction in chronic heart failure. (dundee.ac.uk)
- P=0.03), consistent with reduced oxidative stress with allopurinol therapy. (dundee.ac.uk)
- Accelerated tissue aging and increased oxidative stress in broiler chickens fed allopurinol. (cdc.gov)
- Study 1 was designed to quantitatively manipulate the plasma concentrations of uric acid using hemin and allopurinol while study 2 determined their effects on skin pentosidine, the shear force value of Pectoralis major muscle, plasma glucose, body weight and chemiluminescence monitored oxidative stress in broiler chickens. (cdc.gov)
- Hemin was hypothesized to raise uric acid concentrations thereby lowering oxidative stress whereas allopurinol was hypothesized to lower uric acid concentrations and raise measures of oxidative stress. (cdc.gov)
Hypersensitivity3
- Allopurinol hypersensitivity syndrome (AHS) is a severe reaction which is potentially lethal. (greenmedinfo.com)
- May increase risk for allergic or hypersensitivity reactions to allopurinol. (medscape.com)
- May increase risk for allergic or hypersensitivity reactions to allopurinol Monitor for symptoms of hypersensitivty reactions if both drugs must be used together. (medscape.com)
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- Cialis works faster than other ED drugs allopurinol acute gout flare . (beltstl.com)
- Might happen anywhere from 6 weeks to years after administration of allopurinol and the risk is biggest in those receiving other drugs that likewise have the prospective to trigger bone marrow depression. (tomjonespharmacist.live)
- Caution must be taken when allopurinol is used in conjunction with certain antibiotics (e.g., amoxicillin, ampicillin), immunosuppressive drugs, diuretics, and some other medications. (banderavetclinic.com)
- SJS, depending upon the severity is also known as Toxic Epidermal Necrolysis (TEN) - a violent reaction that comes after exposure to some drugs like allopurinol, sulfonamide antibiotics, lamotrigine, carbamazepine, nevirapine and phenytoin. (targetwoman.com)
Allergic2
- May cause a skin rash which may suggest an allergic reaction to allopurinol. (tomjonespharmacist.live)
- In rare cases Allopurinol can cause damage to nerve, kidney, and bone marrow as well as be a reason to a serious and even fatal allergic liver toxicity. (canadiangeniusmeds.com)
Skin rash1
- Risk of skin rash increases in patients taking Allopurinol with penicillins. (canadiangeniusmeds.com)
Generic2
- Generic allopurinol is offered. (tomjonespharmacist.live)
- Silagra is a generic version of the brand name drug called Viagra amoxicillin allopurinol drug interactions . (psicoterapeutaonline.es)
Medications4
- Allopurinol is in a class of medications called xanthine oxidase inhibitors. (medlineplus.gov)
- Celexa Online application consisting of Regional acheter dapoxetine en pharmacie Information.pharmacy is a secure and trustworthy top-level domain where consumers around the globe can be sure the medications they buy online are safe. (bz.it)
- Allopurinol belongs to the class of medications called xanthine oxidase inhibitors. (tomjonespharmacist.live)
- Reported that previously serviced on an eye care center and diagnosed with conjunctivitis and was under use of the following medications: allopurinol,nimesulide, simvastatin, omeprazole and front. (bvsalud.org)
Purine base1
- Allopurinol is a structural analogue of the natural purine base, hypoxanthine. (nih.gov)
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- Keep in mind that allopurinol does not treat severe attacks of gout, although as soon as started, it ought to be continued throughout an acute attack. (tomjonespharmacist.live)
- Muwakkit S, Saab R, Yazbeck N, Samia L, Abboud MR. L-asparaginase-induced pancreatitis in children with acute lymphoblastic leukemia: is allopurinol protective? (musc.edu)
Hypoxanthine3
- It has been shown that reutilization of both hypoxanthine and xanthine for nucleotide and nucleic acid synthesis is markedly enhanced when their oxidations are inhibited by allopurinol and oxipurinol. (nih.gov)
- Allopurinol is an arthrifuge which inhibits xanthine oxidase and prevents formation of uric acid, from xanthine and hypoxanthine. (canadiangeniusmeds.com)
- Allopurinol is also considered a structural isomer of hypoxanthine, one of the natural purines in the body. (healthprep.com)
Azathioprine1
- allopurinol increases levels of azathioprine by decreasing metabolism. (medscape.com)
Purines1
- Allopurinol acts on purine catabolism, without disrupting the biosynthesis of purines. (nih.gov)
Kidney stones2
- Some natural remedies for gout pain and kidney stones may help reduce the patient's reliance on allopurinol. (healthprep.com)
- Allopurinol is most commonly used for the treatment of gout and kidney stones . (healthprep.com)
Bladder stones2
- Allopurinol is used to prevent urate bladder stones. (cfspharmacy.pharmacy)
- Because bladder stones can develop with long-term use, dogs should also be monitored for urinary problems (e.g., straining to urinate, blood in the urine, pain while urinating) while taking allopurinol. (banderavetclinic.com)
Occur1
- Allopurinol is used to prevent gout attacks, not to treat them once they occur. (medlineplus.gov)
Febuxostat9
- All-cause and cardiovascular mortality risks were 22% and 34% higher among febuxostat-treated patients vs allopurinol recipients. (renalandurologynews.com)
- In patients with gout and co-existing cardiovascular disease, febuxostat is noninferior to allopurinol in the rates of major cardiovascular events, but febuxostat is associated with higher risks for all-cause and cardiovascular mortality. (renalandurologynews.com)
- At the request of the FDA , William B. White, MD, of the University of Connecticut School of Medicine in Farmington, and colleagues conducted a double-blind, randomized trial to assess the Cardiovascular Safety of Febuxostat and Allopurinol in Patients with Gout and Cardiovascular Morbidities (CARES, NCT01101035). (renalandurologynews.com)
- The study's main endpoint - cardiovascular death, nonfatal myocardial infarction, nonfatal stroke, or unstable angina with urgent revascularization - occurred in 10.8% of the febuxostat group and 10.4% of the allopurinol group. (renalandurologynews.com)
- The confidence interval of the hazard ratio did not cross a prespecified threshold, demonstrating that febuxostat is noninferior to allopurinol. (renalandurologynews.com)
- The all-cause and cardiovascular mortality risks were 22% and 34% higher in the febuxostat group, respectively, compared with the allopurinol group. (renalandurologynews.com)
- In addition, the rates of adjudicated nonfatal events, including myocardial infarction, coronary revascularization, arrhythmias, and hospitalization for heart failure, were similar in the febuxostat group and the allopurinol group. (renalandurologynews.com)
- This was more likely in people who took lesinurad without taking allopurinol or febuxostat with it. (hopkinsarthritis.org)
- You must take lesinurad with your allopurinol or febuxostat. (hopkinsarthritis.org)
Oxypurinol2
- Two of the 5 patients who had a serum urate level ≥ 0.36 mmoles/liter had undetectable levels of plasma oxypurinol, indicating noncompliance with allopurinol treatment. (nih.gov)
- Allopurinol and oxypurinol are dialyzable. (medilib.ir)
Treatment6
- There were no significant differences in the forearm blood flow changes between the placebo and allopurinol treatment arms with regard to sodium nitroprusside or verapamil. (dundee.ac.uk)
- In this study, we investigated the effect of antenatal allopurinol (ALLO) treatment on levels and expression of plasticity markers in the dorsal hippocampus of low (LBW) and normal (NBW) birth weight piglets. (maastrichtuniversity.nl)
- The acid treatment of gout, and allopurinol 30 0mg cost walmart the other hand, is medical insurance, and it was really tough to pick our top three as there were still insolvable problems, she says, including the grand opening of its allopurinol and alcohol 4D Health Approach, the Center also provides a safe and responsible way. (revertalloysandmetals.com)
- When allopurinol is prescribed in the treatment of leishmaniasis or for use in birds and reptiles, it is referred to as off-label use. (banderavetclinic.com)
- During treatment with allopurinol, patients could experience side effects. (healthprep.com)
- To our knowledge , this represents a rare case of successful treatment of FeL with a suspected nephrotoxic effect associated with long-term use of allopurinol . (bvsalud.org)
Enalapril1
- Allopurinol - Not to be confused with Enalapril. (en-academic.com)
Severe6
- This is thought about a severe event and allopurinol should be discontinued right away and urgent medical advice looked for. (tomjonespharmacist.live)
- However, allopurinol has proven to be a cause of severe side effects on your skin that require medical attention. (drugsandgenes.com)
- While the development of a severe skin reaction due to allopurinol use is rare, the condition is very serious and can cause death in up to 25% of patients that experience one. (drugsandgenes.com)
- Allopurinol should not be prescribed to those who test positive for the HLA-B*5801 allele since they are at relatively high risk of developing an allopurinol-induced severe skin reaction. (drugsandgenes.com)
- HLA-B*5801 allele as a genetic marker for severe cutaneous adverse reactions caused by allopurinol. (drugsandgenes.com)
- Before prescribing allopurinol, consider testing for the HLA-B*5801 allele in patients at elevated risk for developing severe cutaneous adverse reactions (SCAR) (including Koreans with CKD ≥ stage 3 and all patients of Han Chinese or Thai descent). (medilib.ir)
58015
- The specific gene form (allele), HLA-B*5801, interacts with allopurinol and causes a significant increase in the likelihood of developing a serious skin reaction. (drugsandgenes.com)
- Those that test negative for the allele can be given allopurinol according to normal dosing recommendations because these individuals do not possess the HLA-B*5801 allele that predisposes them to an increased risk of an adverse skin reaction. (drugsandgenes.com)
- The patient agrees and upon receiving results it is recognized that the patient has the HLA-B*5801 allele and is immediately taken off allopurinol. (drugsandgenes.com)
- Dean L. Allopurinol therapy and HLA-B*5801 genotype. (drugsandgenes.com)
- Strong association between HLA-B*5801 and allopurinol- induced Stevens-Johnson syndrome and toxic epidermal necrolysis in a Thai population. (drugsandgenes.com)
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