Albuterol
Bronchodilator Agents
Nebulizers and Vaporizers
Ipratropium
Administration, Inhalation
Inhalation Spacers
Metered Dose Inhalers
Ozone Depletion
Chlorofluorocarbons
Asthma
Aerosol Propellants
Compressed gases or vapors in a container which, upon release of pressure and expansion through a valve, carry another substance from the container. They are used for cosmetics, household cleaners, and so on. Examples are BUTANES; CARBON DIOXIDE; FLUOROCARBONS; NITROGEN; and PROPANE. (McGraw-Hill Dictionary of Scientific and Technical Terms, 4th ed)
Bronchoconstriction
Respiratory Therapy Department, Hospital
Aerosols
Forced Expiratory Volume
Measure of the maximum amount of air that can be expelled in a given number of seconds during a FORCED VITAL CAPACITY determination . It is usually given as FEV followed by a subscript indicating the number of seconds over which the measurement is made, although it is sometimes given as a percentage of forced vital capacity.
Drug Incompatibility
The quality of not being miscible with another given substance without a chemical change. One drug is not of suitable composition to be combined or mixed with another agent or substance. The incompatibility usually results in an undesirable reaction, including chemical alteration or destruction. (Dorland, 27th ed; Stedman, 25th ed)
Forced Expiratory Flow Rates
Receptors, Adrenergic, beta-2
A subclass of beta-adrenergic receptors (RECEPTORS, ADRENERGIC, BETA). The adrenergic beta-2 receptors are more sensitive to EPINEPHRINE than to NOREPINEPHRINE and have a high affinity for the agonist TERBUTALINE. They are widespread, with clinically important roles in SKELETAL MUSCLE; LIVER; and vascular, bronchial, gastrointestinal, and genitourinary SMOOTH MUSCLE.
Beclomethasone
Masks
Double-Blind Method
Models, Anatomic
Myasthenic Syndromes, Congenital
A heterogeneous group of disorders characterized by a congenital defect in neuromuscular transmission at the NEUROMUSCULAR JUNCTION. This includes presynaptic, synaptic, and postsynaptic disorders (that are not of autoimmune origin). The majority of these diseases are caused by mutations of various subunits of the nicotinic acetylcholine receptor (RECEPTORS, NICOTINIC) on the postsynaptic surface of the junction. (From Arch Neurol 1999 Feb;56(2):163-7)
Pulmonary Disease, Chronic Obstructive
Airway Resistance
Respiratory Therapy
Care of patients with deficiencies and abnormalities associated with the cardiopulmonary system. It includes the therapeutic use of medical gases and their administrative apparatus, environmental control systems, humidification, aerosols, ventilatory support, bronchopulmonary drainage and exercise, respiratory rehabilitation, assistance with cardiopulmonary resuscitation, and maintenance of natural, artificial, and mechanical airways.
Bronchial Provocation Tests
Stereoisomerism
Respiratory Function Tests
Propanolamines
Budesonide
Intubation, Intratracheal
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Treatment Outcome
Androstadienes
Respiration, Artificial
Any method of artificial breathing that employs mechanical or non-mechanical means to force the air into and out of the lungs. Artificial respiration or ventilation is used in individuals who have stopped breathing or have RESPIRATORY INSUFFICIENCY to increase their intake of oxygen (O2) and excretion of carbon dioxide (CO2).
Respiratory Sounds
Glucocorticoids
A group of CORTICOSTEROIDS that affect carbohydrate metabolism (GLUCONEOGENESIS, liver glycogen deposition, elevation of BLOOD SUGAR), inhibit ADRENOCORTICOTROPIC HORMONE secretion, and possess pronounced anti-inflammatory activity. They also play a role in fat and protein metabolism, maintenance of arterial blood pressure, alteration of the connective tissue response to injury, reduction in the number of circulating lymphocytes, and functioning of the central nervous system.
Anesthesia, Inhalation
Respiratory Mechanics
Drug Administration Schedule
Bronchi
Trachea
Vital Capacity
Lung
Kinetic analysis of drug-receptor interactions of long-acting beta2 sympathomimetics in isolated receptor membranes: evidence against prolonged effects of salmeterol and formoterol on receptor-coupled adenylyl cyclase. (1/1501)
The long-acting beta2 sympathomimetics salmeterol and formoterol have been presumed to exert their prolonged action either by binding to an accessory binding site ("exo-site") near the beta2 adrenoceptor or by their high affinity for beta2 adrenoceptors and correspondingly slow dissociation. Whereas most studies with salmeterol had been done in intact tissues, which have slow diffusion and compartmentation of drugs in lipophilic phases, that restrict drug access to the receptor biophase, we used purified receptor membranes from rat lung and disaggregated calf tracheal myocytes as model systems. Binding experiments were designed to measure the slow dissociation of agonists by means of delayed association of (-)-[125I]iodopindolol. Rat lung membranes were pretreated with high concentrations of agonists (salmeterol, formoterol, isoprenaline) before dissociation was induced by 50-fold dilution. Half-times of association of (-)-[125I]iodopindolol remained unchanged compared with untreated controls, indicating that dissociation of agonists occurred in less than 2 min. Adenylyl cyclase experiments were designed to determine the on and off kinetics of agonists to beta2 adrenoceptors by measuring the rate of receptor-induced cyclic AMP (cAMP) formation. Experiments were performed in tracheal membranes characterized by high Vmax values of cAMP formation. Adenylyl cyclase activation occurred simultaneously with the addition of the agonist, continued linearly with time for 60 min, and ceased immediately after the antagonist was added. Similarly, when receptor membranes were preincubated in a small volume with high salmeterol concentrations, there was a linear increase in cAMP formation, which was immediately interrupted by a 100-fold dilution of the reaction mixture. This militates against the exo-site hypothesis. On the other hand, dissociation by dilution was much less when membranes were preincubated with a large volume of salmeterol at the same concentration, indicating that physicochemical effects, and not exo-site binding, underlie its prolonged mode of action. (+info)Reversal of severe pulmonary hypertension with beta blockade in a patient with end stage left ventricular failure. (2/1501)
A 52 year old man with severe chronic left ventricular failure (New York Heart Association class IV) was considered unsuitable for cardiac transplantation because of high and irreversible pulmonary vascular resistance (PVR). In an attempt to produce symptomatic improvement, metoprolol was cautiously introduced, initially at 6.25 mg twice daily. This was slowly increased to 50 mg twice daily over a two month period and continued thereafter. After four months of treatment the patient's symptoms had improved dramatically. His exercise tolerance had increased and diuretic requirements reduced to frusemide 160 mg/day only. Assessment of right heart pressures was repeated and, other than a drop in resting heart rate, there was little change in his pulmonary artery pressure or PVR. His right heart pressures were reassessed showing a pronounced reduction in pulmonary artery pressure and a significant reduction in PVR, which fell further with inhaled oxygen and sublingual nitrates. He was then accepted onto the active waiting list for cardiac transplantation. A possible mechanism of action was investigated by assessing responses to beta agonists during treatment. Not only was there pronounced improvement in PVR but it was also demonstrated that beta receptor subtype cross-regulation may have contributed to the mechanism of benefit. (+info)As-required versus regular nebulized salbutamol for the treatment of acute severe asthma. (3/1501)
Current British guidelines for the administration of beta2-agonists in acute severe asthma recommend regular nebulized therapy in hospitalized patients, followed by as-required (p.r.n.) use via hand-held devices after discharge. Since beta2-agonists do not possess anti-inflammatory activity in vivo, and are thus unlikely to influence the rate of recovery from an asthma exacerbation, it was hypothesized that patients given the short-acting beta2-agonist salbutamol on an as-required basis after admission to hospital would recover as quickly as those on regular treatment, but with potential reductions in the total dose delivered. Forty-six patients with acute severe asthma were randomly assigned to either regular prescriptions of nebulized salbutamol or to usage on a p.r.n. basis, from 24 h after hospital admission. The primary outcome measures were length of hospital stay, time to recovery, and frequency of salbutamol nebulization from 24 h after admission to discharge. Secondary outcome measures were treatment side-effects (tremor, palpitations), and patient satisfaction. Length of hospital stay was reduced in those patients allocated to p.r.n. salbutamol (geometric mean (GM) 3.7 days) versus regular salbutamol (GM 4.7 days). Time taken for peak expiratory flow to reach 75% of recent best was the same in both groups. There was a highly significant reduction in the number of times nebulized therapy was delivered to the p.r.n. group (GM 7.0, range 1-30) compared with the regular treatment group (GM 14.0, range 4-57; p=0.003; 95% confidence interval for ratio of GMs 1.29-3.09). In addition, patients reported less tremor (p=0.062) and fewer palpitations (p=0.049) in the p.r.n. group. Of the patients in the p.r.n. group who had received regular nebulized therapy on previous admissions (n=12), all preferred the p.r.n. regimen. Prescribing beta2-agonists on a p.r.n. basis from 24 h after hospital admission is associated with reduced amount of drug delivered, incidence of side-effects, and possibly length of hospital stay. This has implications for the efficient use of healthcare resources. (+info)The contribution of the swallowed fraction of an inhaled dose of salmeterol to it systemic effects. (4/1501)
Salmeterol is approximately eight times as potent as salbutamol for systemic effects. This may be because the drug is eight times more potent on receptors or there may be differences in systemic bioavailability. The systemic effects of salbutamol are limited by its fairly high first-pass metabolism, but the oral bioavailability of salmeterol is unknown. The contribution of the swallowed fraction of an inhaled dose of salmeterol to its systemic effects were analysed in a randomized, double-blind, crossover study. Twelve healthy subjects were given inhaled salmeterol 400 microg, inhaled salmeterol 400 microg plus oral activated charcoal or inhaled placebo plus oral activated charcoal on three separate days. Cardiac frequency (fC), Q-T interval corrected for heart rate (QTc), plasma potassium and glucose concentrations were measured for 4 h following the inhaled drug. Salmeterol with and without oral charcoal produced significant changes for all measures compared to placebo. The magnitude of effect following salmeterol alone was significantly greater than that following salmeterol plus charcoal for fC and glucose (mean (95% confidence interval) differences 8 (2-13) beats x min(-1), 0.59 (0.04, 1.13) mmol x L(-1), respectively) and nonsignificantly greater for QTc interval and potassium concentration. The differences between salmeterol given with and without charcoal suggest that 28-36% of the systemic response to salmeterol administered from a metered-dose inhaler are due to drug absorbed from the gastrointestinal tract. Thus, most of the systemic effects are due to the inhaled fraction of the drug. (+info)beta2-adrenoceptor agonists reduce the decline of rat diaphragm twitch force during severe hypoxia. (5/1501)
The aim of the present study was to investigate the in vitro effects of the short-acting beta2-adrenoceptor agonist salbutamol and the long-acting beta2-adrenoceptor agonist salmeterol on hypoxia-induced rat diaphragm force reduction. In vitro diaphragm twitch force (Pt) and maximal tetanic force (Po) of isolated diaphragm muscle strips were measured for 90 min during hyperoxia (tissue bath PO2 83.8 +/- 0.9 kPa and PCO2 3.9 +/- 0.1 kPa) or severe hypoxia (PO2 7.1 +/- 0.3 kPa and PCO2 3.9 +/- 0.1 kPa) in the presence and absence of 1 microM salbutamol or 1 microM salmeterol. During hyperoxia, salbutamol and salmeterol did not significantly alter the time-related decreases in Pt and Po (to approximately 50% of initial values). Salbutamol had no effects on Po or the Pt-to-Po ratio. Salmeterol treatment significantly reduced Po and increased the Pt-to-Po ratio during hyperoxia (P < 0.05 compared with control value). Hypoxia resulted in a severe decrease in Pt (to approximately 30% of initial value) and Po after 90 min. Both salbutamol and salmeterol significantly reduced the decline in Pt during hypoxia (P < 0.05). The reduction in Po was not prevented. Salbutamol increased Pt rapidly but transiently. Salmeterol had a delayed onset of effect and a longer duration of action. Addition of 1 microM propranolol (a nonselective beta-adrenoceptor antagonist) did not alter Pt, Po, or the Pt-to-Po ratio during hypoxia but completely blocked the inotropic effects of salbutamol and salmeterol, indicating that these effects are dependent on beta2-adrenoceptor agonist-related processes. (+info)Incorporating quality of life data into managed care formulary decisions: a case study with salmeterol. (6/1501)
Pharmacy and Therapeutics committees of managed care organizations have traditionally developed formularies by limiting the numbers and kinds of pharmaceuticals they purchase, with the goal of cutting costs. These attempts to manage pharmaceutical costs do not take into account the interrelationship of the costs of various components of care; thus, drug costs may decrease, but expenditures for utilization of other resources may increase. Cost-minimization and basic cost-effectiveness studies, on which many prior- authorization and formulary programs are based, only evaluate only the cost of the drug and its effectiveness. However, with the heightened competition in the healthcare market, emphasis is increasingly being laid on patient satisfaction and outcomes. Cost-utility analysis is a potentially superior pharmacoeconomic tool because it evaluate the effect of drug therapy on quality of life; however, data from such analyses are seldom readily available to the committees that evaluate a drug's potential effects on the entire healthcare system. The purpose of this review is to stress the importance of approaching formulary management from a wider perspective and to emphasize that the results of cost-utility studies should be proactively evaluated and incorporated into decisions regarding formularies. This is especially important for symptom-intensive diseases, such as asthma, in which the quality of life can be notably impaired. Cost-utility analyses should be conducted for all newer therapies, such as salmeterol, which are highly effective and which have a positive impact on quality of life, to determine the overall effect on the managed care plan's budget. (+info)Evaluation of the effect on heart rate variability of a beta2-adrenoceptor agonist and antagonist using non-linear scatterplot and sequence methods. (7/1501)
AIMS: To examine the impact on heart rate variability (HRV), of agonism or antagonism at the cardiac beta2-adrenoceptor in healthy volunteers, using standard time-domain summary statistics and non-linear methods (scatterplot and quadrant analysis). METHODS: Under double-blind and randomised conditions (Latin square design), 17 normal volunteers received placebo, salbutamol (beta2-adrenoceptor partial agonist), ICI 118,551 (specific beta2-adrenoceptor antagonist), or salbutamol plus ICI 118,551. Single oral doses of medication (at weekly intervals) were administered at 22.30 h, with HRV assessed from the sleeping heart rates. RESULTS: Salbutamol reduced the long-term (SDNN: 135 ms [120, 156], SDANN: 107 ms [89, 124]) time-domain indicators of HRV compared with placebo (SDNN: 39 [24, 55], SDANN 42 [29, 56], [mean difference [95% confidence intervals of difference]]). Alone, ICI 118,551 did not effect HRV, but in combination blocked the actions of salbutamol. Scatterplot length (944 ms [869, 1019]) and area (222*10(3) ms2 [191, 253]) were reduced by salbutamol compared with placebo; (length difference (164 [98, 230]) and area difference 59 [36, 83]). Scatterplot width (dispersion) was lower at both low (width RR-1 25% salbutamol 277 ms [261, 293]: salbutamol minus placebo 14 ms [0, 28]) and high (width 75% salbutamol 417 [391, 443]: salbutamol minus placebo 41 [20, 62]) heart rates. ICI 118,551 alone did not alter scatterplot parameters but in combination blocked the effect of salbutamol. Cardiac acceleration episodes (i.e. consecutive deltaRR and deltaRRn+1 shorten) were increased following salbutamol 7288 [6089, 8486] compared with placebo -1890 [-2600, -1179]; the beat-to beat difference (deltaRRn+1) was reduced after salbutamol compared with the other treatments. ICI 118,551 did not effect acceleration episodes but reduced the effect of salbutamol when used in combination. CONCLUSIONS: Agonism at the cardiac beta2-adrenoceptor in healthy volunteers with salbutamol altered autonomic balance towards sympathetic dominance; this re-balancing was blocked by ICI 118,551 given in combination with salbutamol. However antagonism at the beta2-adrenoceptor with ICI 118,551 alone did not significantly alter the HRV. The beta2-adrenoceptor modulates HRV in healthy volunteers; the implications of agonism and antagonism at the beta2-adrenoceptor in cardiovascular disease states warrants further investigation. (+info)Determination of salbutamol and detection of other beta-agonists in human postmortem whole blood and urine by GC-MS-SIM. (8/1501)
This paper details a sensitive and quantitative method for the determination of salbutamol and the detection of terbutaline, clenbuterol, fenoterol, and isoprenaline in postmortem human whole blood and urine. It describes solid-phase extraction using a XtrackT XRDAH515 column, formation of trimethylsilyl derivatives, and analysis by gas chromatography-mass spectrometry-selective ion monitoring. (+info)
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Salbutamol
"Albuterol Sulfate". Rx List: The Internet Drug Index. Archived from the original on 18 July 2014. Retrieved 13 July 2014. ... "Albuterol". Drug Information Portal. U.S. National Library of Medicine. Salbutamol at The Periodic Table of Videos Portal: ... Papich MG (2007). "Albuterol Sulfate". Saunders Handbook of Veterinary Drugs (2nd ed.). St. Louis, Mo: Saunders/Elsevier. pp. ... "Albuterol". Drugs.com. The American Society of Health-System Pharmacists. Archived from the original on 8 December 2015. ...
Racemic mixture
Ameredes, Bill T.; Calhoun, William J. (November 2006). "(R)-Albuterol for Asthma: Pro [a.k.a. (S)-Albuterol for Asthma: Con ...
Asthma trigger
Examples include albuterol and levalbuterol. Commonly they are used with a portable inhaler which allow the patient to ...
Inhaler
Hendeles L, Colice GL, Meyer RJ (March 2007). "Withdrawal of albuterol inhalers containing chlorofluorocarbon propellants". N. ...
Isoetarine
Orciprenaline in turn was replaced by salbutamol (albuterol). Some practitioners still believe that these later aerosolized ...
Facioscapulohumeral muscular dystrophy
Oral albuterol, a β2 agonist, on the basis of its anabolic properties. Although it improved muscle mass and certain measures of ... 23 October 2001). "Randomized, double-blind, placebo-controlled trial of albuterol in facioscapulohumeral dystrophy". Neurology ... "Pilot trial of albuterol in facioscapulohumeral muscular dystrophy. FSH-DY Group". Neurology. 50 (5): 1402-6. doi:10.1212/wnl. ... "Strength training and albuterol in facioscapulohumeral muscular dystrophy". Neurology. 63 (4): 702-8. doi:10.1212/01.wnl. ...
Adrenergic agonist
β2 selective: salbutamol/albuterol, terbutaline, salmeterol, formoterol, pirbuterol, clenbuterol. These are agents that ...
Ipratropium bromide
"Ipratropium Bromide 0.5 mg/Albuterol Sulfate 3.0 mg" (PDF). FDA. 2004. Retrieved 4 January 2021. "Atrovent Nasal Spray". Drugs. ... albuterol - USAN) under the trade names Combivent (a non-aerosol metered-dose inhaler or MDI) and Duoneb (nebulizer) for the ... albuterol) as a rescue medication. "Ipratropium Bromide". The American Society of Health-System Pharmacists. Archived from the ...
Shock (circulatory)
Albuterol, normal saline, and steroids are also commonly given. Intra-aortic balloon pump (IABP) - a device inserted into the ...
Levosalbutamol
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Discovery and development of beta2 agonists
In the mid-1960s, albuterol or salbutamol was discovered, followed by tributalin and fenoterol a few years later. Albuterol and ... Barberich, T.J. "Method for treating asthma using optically pure (R)-albuterol". Google Patents. Retrieved 27 October 2014. ...
Bronchiole
Bronchospasm is commonly treated by oxygen therapy and bronchodilators such as albuterol. Diseases of the bronchioles include ...
Chronic obstructive pulmonary disease
Several short-acting β2 agonists are available, including salbutamol (albuterol) and terbutaline. They provide some relief of ...
Löffler's syndrome
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Chlorofluorocarbon
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Electrolyte imbalance
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Heliox
Lee DL, Hsu CW, Lee H, Chang HW, Huang YC (September 2005). "Beneficial effects of albuterol therapy driven by heliox versus by ...
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Hyperkalemia
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Bronchodilatation
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Paramedics in the United States
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Gentech Pharmaceutical
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Salmeterol
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Procaterol
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Salbutamol12
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- Drugs such as albuterol (also known as salbutamol) are widely prescribed for the treatment of these spells. (internationaldrugmart.com)
- Those with prior allergies to salbutamol-based bronchodilators must take added care while consuming albuterol. (internationaldrugmart.com)
- In addition, you can always purchase Salbutamol - see Ventolin or Pro Air HFA Inhaler, the generic alternative, instead of Albuterol Inhaler. (qualityprescriptiondrugs.com)
- Salbutamol, also known as albuterol and sold under the brand name Ventolin among others,[1] is a medication that opens up the medium and large airways in the lungs. (medschat.com)
- The World Health Organization recommended name for albuterol base is salbutamol. (medlibrary.org)
- Albuterol (also known as salbutamol) is used to prevent and treat wheezing and shortness of breath caused by breathing problems (such as asthma, chronic obstructive pulmonary disease). (americaspharmacy.com)
- Comparison of the effects of salbutamol and clenbuterol on skeletal muscle mass and carcass composition in senescent rats Long-term use of steroids may lead to bone loss (osteoporosis), especially if you smoke, if you do not exercise, if you do not get enough vitamin D or calcium in your diet, or if you have a family history of osteoporosis, albuterol clenbuterol difference. (necpad.org)
- Albuterol (also known as salbutamol) is used to treat wheezing and shortness of breath caused by breathing problems such as asthma. (ndclist.com)
- More popularly known as Salbutamol, and prescribed under the brand name Ventolin, Albuterol is a medication is a short-acting bronchodilator, which opens up the medium and large airways, preventing bronchial spasms in patients who experience lung conditions such as bronchitis, emphysema, Chronic Obstructive Pulmonary Disease (COPD) and is most widely prescribed as a short-term relief for people with symptoms of asthma. (drhouse.com)
- Albuterol sulfate , also known as salbutamol (brand names: Proventil® and Ventolin®), is a bronchodilator medication that relaxes the muscles of the airways and improves breathing. (countrygrovevet.ca)
- A meta-analysis of the influence of ADRB2 genetic polymorphisms on albuterol (salbutamol) therapy in patients with asthma. (cdc.gov)
Clenbuterol for weight loss8
- This may also occur in people who have inflammatory conditions, such as psoriasis or rheumatoid arthritis, albuterol or clenbuterol for weight loss. (harrykroner.com)
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- This method is known as 'n-butanol,' the same as that used to treat alcohol poisoning, albuterol vs clenbuterol for weight loss. (acehrproyectos.com)
- A doctor administering n-butanol (for example, for a liver condition) could prescribe clenbuterol (for example, for sexual dysfunction, high blood pressure, or obesity), albuterol vs clenbuterol for weight loss. (goldmongsite.com)
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Asthma30
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- Regular vs ad-lib albuterol for patients hospitalized with acute asthma. (druglib.com)
- CONCLUSIONS: In the management of asthma exacerbation, ad-lib administration of albuterol is therapeutically as effective as regular, scheduled administration. (druglib.com)
- According to the Centers for Disease Control and Prevention, cheap albuterol coughing, sneezing, wheezing and constrained breathing are the main symptoms of Asthma. (nephew.dk)
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- Albuterol is a drug used in the treatment of asthma. (healthhearty.com)
- Albuterol in inhaler, drug or syrup form, should not be administered, if an asthma patient also suffers from any of the following conditions. (healthhearty.com)
- Albuterol comes with loads of side effects, but it's a lifesaving drug for asthma patients and is widely used throughout the world. (healthhearty.com)
- Albuterol is a bronchodilator medication prescribed to manage symptoms of asthma and other lung conditions. (sesamecare.com)
- It is important to note that albuterol does not cure asthma or lung disease. (sesamecare.com)
- Albuterol is prescribed to treat and prevent the symptoms of bronchospasm (the tightening of muscles that line the airways of your lungs) in patients with asthma and other lung conditions. (sesamecare.com)
- Albuterol does not cure patients of asthma or lung disease, but it can improve quality of life and prevent potentially severe breathing problems. (sesamecare.com)
- Albuterol Sulfate is prescribed for asthma. (medschat.com)
- albuterol and clenbuterol are used to treat asthma and copd. (necpad.org)
- Q: Can albuterol hurt a child if they do not have asthma? (azasthma.org)
- Albuterol sulfate syrup (oral solution), is a bronchodilator used to treat or prevent the symptoms of asthma, emphysema, and other breathing conditions. (californiapetpharmacy.com)
- Albuterol 2mg theophylline 100mg tablets is a combination of medicines used for treating asthma. (daynightdrugs.com)
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- Because albuterol has a stimulating effect, some people try to use this asthma medication to promote weight or fat loss . (answersdrive.com)
- Albuterol is a prescription medication used to treat breathing problems, especially in people with asthma or COPD. (answersdrive.com)
- A group of patients using an effective albuterol asthma inhaler was compared to 2 placebo groups (a placebo inhaler group and a sham acupuncture group), and to a group that got no treatment at all. (sciencebasedmedicine.org)
- The authors say that many pediatric hospitals use this kind of albuterol when preparing solutions for continuous nebulized albuterol for asthma patients. (talkingaboutthescience.com)
- Standardized acute asthma management with score-based, respiratory therapist (RT)-driven pathways and protocols improves outcomes including decreased length of stay (LOS) and time on continuous albuterol therapy. (rcjournal.com)
- The safety and effectiveness of using PAS to initiate/stop continuous albuterol as part of a score-based, RT-driven asthma pathway were evaluated. (rcjournal.com)
- A retrospective review of children ≥ 2 y admitted for asthma exacerbation to the PICU and step-down unit who received continuous albuterol as part of the asthma pathway during 2017-2019 was completed. (rcjournal.com)
- Use of an RT-driven, score-based pathway for initiation and discontinuation of continuous albuterol for treatment of pediatric asthma exacerbation was safe and effective in the PICU and step-down unit. (rcjournal.com)
- On each visit, the patients received 1 of 4 different interventions: an inhaler with albuterol (the standard treatment for relieving sudden asthma symptoms), a placebo inhaler, sham acupuncture or no treatment. (nih.gov)
- Also, asthma would respond to Albuterol but anxiety would not. (sutterhealth.org)
- When Meredith first visited the doctor for her breathing problems, doctors told her she had asthma and gave her albuterol (an inhaler that relaxes and opens the airways to the lungs). (medlineplus.gov)
Bronchodilator12
- Albuterol Sulfate is used primarily as a bronchodilator. (chewy.com)
- Like other bronchodilator meds, albuterol is likely to cause a few side effects. (internationaldrugmart.com)
- Albuterol is a bronchodilator used to relax muscles in the airways and encourage airflow to and from the lungs. (sesamecare.com)
- id":6572170575985,"title":"Albuterol Sulfate Inhalation Solution","handle":"albuterol-sulfate-inhalation-solution","description":"\u003ch2\u003eAlbuterol Sulfate Inhalation Solution, 0.5%\u003c\/h2\u003e \u003cp\u003eAlbuterol is a bronchodilator that relaxes muscles in the airways and increases air flow to the lungs. (agtechinc.com)
- Albuterol is a bronchodilator that relaxes muscles in the airways and increases air flow to the lungs. (agtechinc.com)
- Albuterol sulfate inhalation solution is a relatively selective beta2-adrenergic bronchodilator (see CLINICAL PHARMACOLOGY section below). (medlibrary.org)
- Albuterol is a prescribed bronchodilator planned for clients with constricted air passages that prevent them from working out and appreciating their everyday life as a whole. (albuterol.company)
- Albuterol is an effective medication which is classified as a bronchodilator. (advicarehealth.com)
- Albuterol is a bronchodilator dilator that helps relax the muscles in a patient's airways resulting in an increase of air flow to the lungs. (lifepolicyshopper.com)
- Combivent contains a combination of ipratropium and albuterol, a bronchodilator, is used with other bronchodilators to prevent wheezing, shortness of breath, and troubled breathing caused by chronic obstructive pulmonary disease (COPD). (globalpharmacyplus.com)
- Ventolin HFA ( albuterol sulfate inhalation aerosol) is a bronchodilator used to treat or prevent bronchospasm in people with reversible obstructive airway disease. (validanswer.com)
- however, it may have bronchodilator effects more potent than those of albuterol. (medscape.com)
Inhalers12
- The FDA released a draft guidance in March 2020 specific to proposed generic albuterol sulfate metered-dose inhalers, including drug products referencing Proventil HFA. (medscape.com)
- Albuterol Albuterol is the generic form of many metered-dose inhalers, and also comes in tablet form and as a solution for nebulizers. (nephew.dk)
- Albuterol HFA inhalers will have brand names such as ProAir HFA, Proventil HFA and Ventolin HFA while Xopenex HFA is the same as levalbuterol HFA. (drsamrobbins.com)
- The Albuterol HFA inhalers have a basic strength of 108mcg/actuation while the Xopenex has about 45mcg/actuation, so the Albuterol is stronger. (drsamrobbins.com)
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- 82.MacDorman MF, Declercq albuterol inhalers over the counter E, Menacker F, Malloy MH. (ifaqeer.com)
- Generally, albuterol inhalers should be stored at room temperature. (azasthma.org)
- I am allergic to albuterol sulfate, it causes numbing of my mouth/nose if I use the HFA inhalers more than 3-4 times in a week. (drugs.com)
- Public drugs plans in Canada currently list opioids, stimulants, benzodiazepines, and opioid substitute therapies, side effects of albuterol sulfate inhalers although some have quantity and refill restrictions. (bpoxyintl.com)
- Generic albuterol inhalers and the branded versions require a prescription in order to be dispensed by a pharmacy. (validanswer.com)
- At this time, over-the-counter (OTC) albuterol is not available and one cannot simply buy albuterol inhalers online. (validanswer.com)
- Xopenex is an alternative for patients who experience unwanted side effects such as increased heart rate from the albuterol inhalers (Ventolin HFA, Proair HFA, and Proventil HFA). (validanswer.com)
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Bronchospasm7
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