Air Pollutants
Air Pollution
Nitrogen Dioxide
Ozone
Sulfur Dioxide
Particulate Matter
Air
Vehicle Emissions
Environmental Monitoring
Environmental Exposure
Environmental Pollutants
Oxidants, Photochemical
Epidemiological Monitoring
Water Pollutants
Carbon Monoxide
United States Environmental Protection Agency
Air Pollutants, Occupational
Amyloid Precursor Protein Secretases
Atmosphere Exposure Chambers
Water Pollutants, Chemical
Meteorological Concepts
Polycyclic Hydrocarbons, Aromatic
Acid Rain
Gases
Nitric Acid
Motor Vehicles
Asthma
Hazardous Substances
Seasons
Organic Chemicals
Environmental Health
Benzene
Maternal Exposure
Soil Pollutants
Volatilization
Fossil Fuels
Sulfuric Acids
Air Microbiology
Respiration Disorders
Embolism, Air
Nutritional Sciences
Volatile Organic Compounds
Maximum Allowable Concentration
Epidemiologic Studies
Poisson Distribution
Hydrocarbons, Chlorinated
Extraction and Processing Industry
Polychlorinated Biphenyls
Soot
Barrett Esophagus
Complex Mixtures
Geographic Information Systems
Lung
Industry
Aerosols
Respiratory System
Risk Assessment
Formaldehyde
Petroleum
Models, Theoretical
Air Conditioning
Biodegradation, Environmental
Environmental Pollution
Ethylene Oxide
Respiratory Sounds
Air Sacs
Public Health
Allergens
Peak Expiratory Flow Rate
Benz(a)Anthracenes
DNA Adducts
Regression Analysis
Climate
Czech Republic
Occupational Exposure
Hypersensitivity
Respiratory Function Tests
Cross-Over Studies
Greenhouse Effect
Pregnancy
Atmosphere
Risk Factors
Metals, Heavy
Emergency Service, Hospital
Mutagens
Nasal Mucosa
Carbon
Respiratory Hypersensitivity
Toxicogenetics
Cardiovascular Diseases
Prenatal Exposure Delayed Effects
Pesticides
Child Welfare
Dioxins
Linear Models
Cohort Studies
Air Ionization
Spain
Air Bags
Confounding Factors (Epidemiology)
Oxidative Stress
Geography
Confidence Intervals
Pentachlorophenol
Bronchitis
Industrial Waste
Temperature
Models, Statistical
Odds Ratio
Premature Birth
Hong Kong
Methodological issues in biomonitoring of low level exposure to benzene. (1/1747)
Data from a pilot study on unmetabolized benzene and trans,trans muconic acid (t,t-MA) excretion in filling station attendants and unexposed controls were used to afford methodological issues in the biomonitoring of low benzene exposures (around 0.1 ppm). Urinary concentrations of benzene and t,t-MA were measured by dynamic head-space capillary GC/FID and HPLC, respectively. The accuracy of the HPLC determination of t,t-MA was assessed in terms of inter- and intra-method reliability. The adequacy of urinary t,t-MA and benzene as biological markers of low benzene exposure was evaluated by analysing the relationship between personal exposure to benzene and biomarker excretion. Filling station attendants excreted significantly higher amounts of benzene, but not of t,t-MA, than controls. Adjusting for occupational benzene exposure, smokers excreted significantly higher amounts of t,t-MA, but not of unmetabolized benzene, than nonsmokers. A comparative analysis of the present and previously published biomonitoring surveys showed a good inter-study agreement regarding the amount of t,t-MA and unmetabolized benzene excreted (about 0.1-0.2 mg/l and 1-2 micrograms/l, respectively) per unit of exposure (0.1 ppm). For each biomarker, based on the distribution of parameters observed in the pilot study, we calculated the minimum sample size required to estimate the population mean with given confidence and precision. (+info)Mechanisms and mediators in coal dust induced toxicity: a review. (2/1747)
Chronic inhalation of coal dust can cause several lung disorders, including simple coal workers pneumoconiosis (CWP), progressive massive fibrosis (PMF), chronic bronchitis, lung function loss, and emphysema. This review focuses on the cellular actions and interactions of key inflammatory cells and target cells in coal dust toxicity and related lung disorders, i.e. macrophages and neutrophils, epithelial cells, and fibroblasts. Factors released from or affecting these cells are outlined in separate sections, i.e. (1) reactive oxygen species (ROS) and related antioxidant protection mechanisms, and (2) cytokines, growth factors and related proteins. Furthermore, (3) components of the extracellular matrix (ECM), including the modifying role of ROS, cytokines, proteases and antiproteases are discussed in relation to tissue damage and remodelling in the respiratory tract. It is recognised that inhaled coal dust particles are important non-cellular and cellular sources of ROS in the lung, and may be significantly involved in the damage of lung target cells as well as important macromolecules including alpha-1-antitrypsin and DNA. In vitro and in vivo studies with coal dusts showed the up-regulation of important leukocyte recruiting factors, e.g. Leukotriene-B4 (LTB4), Platelet Derived Growth Factor (PDGF), Monocyte Chemotactic Protein-1 (MCP-1), and Tumor Necrosis Factor-alpha (TNF alpha), as well as the neutrophil adhesion factor Intercellular Adhesion Molecule-1 (ICAM-1). Coal dust particles are also known to stimulate the (macrophage) production of various factors with potential capacity to modulate lung cells and/or extracellular matrix, including O2-., H2O2, and NO, fibroblast chemoattractants (e.g. Transforming Growth Factor-beta (TGF beta), PDGF, and fibronectin) and a number of factors that have been shown to stimulate and/or inhibit fibroblast growth or collagen production such as (TNF alpha, TGF beta, PDGF, Insulin Like Growth Factor, and Prostaglandin-E2). Further studies are needed to clarify the in vivo kinetics and relative impact of these factors. (+info)Personal exposure to dust, endotoxin and crystalline silica in California agriculture. (3/1747)
AIMS: The aim of this study was to measure personal exposure to dust, endotoxin and crystalline silica during various agricultural operations in California over a period of one year. METHODS: Ten farms were randomly selected in Yolo and Solano counties and workers were invited to wear personal sampling equipment to measure inhalable and respirable dust levels during various operations. The samples were analysed for endotoxin using the Limulus Amebocyte Lysate assay and crystalline silica content using X-ray diffraction. In total 142 inhalable samples and 144 respirable samples were collected. RESULTS: The measurements showed considerable difference in exposure levels between various operations, in particular for the inhalable fraction of the dust and the endotoxin. Machine harvesting of tree crops (Geometric mean (GM) = 45.1 mg/m3) and vegetables (GM = 7.9 mg/m3), and cleaning of poultry houses (GM = 6.7 mg/m3) showed the highest inhalable dust levels. Cleaning of poultry houses also showed the highest inhalable endotoxin levels (GM = 1861 EU/m3). Respirable dust levels were generally low, except for machine harvesting of tree crops (GM = 2.8 mg/m3) and vegetables (GM = 0.9 mg/m3). Respirable endotoxin levels were also low. For the inhalable dust fraction, levels were reduced considerably when an enclosed cabin was present. The percentage of crystalline silica was overall higher in the respirable dust samples than the inhalable dust samples. CONCLUSIONS: Considerable differences exist in personal exposure levels to dust, endotoxin and crystalline silica during various agricultural operations in California agriculture with some operations showing very high levels. (+info)Hierarchical cluster analysis applied to workers' exposures in fiberglass insulation manufacturing. (4/1747)
The objectives of this study were to explore the application of cluster analysis to the characterization of multiple exposures in industrial hygiene practice and to compare exposure groupings based on the result from cluster analysis with that based on non-measurement-based approaches commonly used in epidemiology. Cluster analysis was performed for 37 workers simultaneously exposed to three agents (endotoxin, phenolic compounds and formaldehyde) in fiberglass insulation manufacturing. Different clustering algorithms, including complete-linkage (or farthest-neighbor), single-linkage (or nearest-neighbor), group-average and model-based clustering approaches, were used to construct the tree structures from which clusters can be formed. Differences were observed between the exposure clusters constructed by these different clustering algorithms. When contrasting the exposure classification based on tree structures with that based on non-measurement-based information, the results indicate that the exposure clusters identified from the tree structures had little in common with the classification results from either the traditional exposure zone or the work group classification approach. In terms of the defining homogeneous exposure groups or from the standpoint of health risk, some toxicological normalization in the components of the exposure vector appears to be required in order to form meaningful exposure groupings from cluster analysis. Finally, it remains important to see if the lack of correspondence between exposure groups based on epidemiological classification and measurement data is a peculiarity of the data or a more general problem in multivariate exposure analysis. (+info)Modeling breathing-zone concentrations of airborne contaminants generated during compressed air spray painting. (5/1747)
This paper presents a mathematical model to predict breathing-zone concentrations of airborne contaminants generated during compressed air spray painting in cross-flow ventilated booths. The model focuses on characterizing the generation and transport of overspray mist. It extends previous work on conventional spray guns to include exposures generated by HVLP guns. Dimensional analysis and scale model wind-tunnel studies are employed using non-volatile oils, instead of paint, to produce empirical equations for estimating exposure to total mass. Results indicate that a dimensionless breathing zone concentration is a nonlinear function of the ratio of momentum flux of air from the spray gun to the momentum flux of air passing through the projected area of the worker's body. The orientation of the spraying operation within the booth is also very significant. The exposure model requires an estimate of the contaminant generation rate, which is approximated by a simple impactor model. The results represent an initial step in the construction of more realistic models capable of predicting exposure as a mathematical function of the governing parameters. (+info)Causes of nitrous oxide contamination in operating rooms. (6/1747)
BACKGROUND: To reduce the ambient concentration of waste anesthetic agents, exhaust gas scavenging systems are standard in almost all operating rooms. The incidence of contamination and the factors that may increase the concentrations of ambient anesthetic gases have not been evaluated fully during routine circumstances, however. METHODS: Concentrations of nitrous oxide (N2O) in ambient air were monitored automatically in 10 operating rooms in Kagoshima University Hospital from January to March 1997. Ambient air was sampled automatically from each operating room, and the concentrations of N2O were analyzed every 22 min by an infrared spectrophotometer. The output of the N2O analyzer was integrated electronically regarding time, and data were displayed on a monitor in the administrative office for anesthesia supervisors. A concentration of N2O > 50 parts per million was regarded as abnormally high and was displayed with an alarm signal. The cause of the high concentration of N2O was then sought. RESULTS: During the 3-month investigation, N2O was used in 402 cases. Abnormally high concentrations of N2O were detected at some time during 104 (25.9%) of those cases. The causes were mask ventilation (42 cases, 40.4% of detected cases), unconnected scavenging systems (20 cases, 19.2%), leak around uncuffed pediatric endotracheal tube (13 cases, 12.5%), equipment leakage (12 cases, 11.5%), and others (17 cases, 16.4%). CONCLUSIONS: N2O contamination was common during routine circumstances in our operating rooms. An unconnected scavenging system led to the highest concentrations of N2O recorded. Proper use of scavenging systems is necessary if contamination by anesthetic gas is to be limited. (+info)Exposure of medical personnel to methylmethacrylate vapor during percutaneous vertebroplasty. (7/1747)
The occupational exposure to methylmethacrylate (MMA) vapor during percutaneous vertebroplasty was determined. During five vertebroplasty procedures, air-sampling pumps were attached to medical personnel. MMA vapor levels in the samples were then quantified using gas chromatography. The samples collected yielded MMA vapor levels of less than five parts per million (ppm). The MMA vapor concentrations measured were well below the recommended maximum exposure of 100 ppm over the course of an 8-hour workday. (+info)Potential effects of gas hydrate on human welfare. (8/1747)
For almost 30 years. serious interest has been directed toward natural gas hydrate, a crystalline solid composed of water and methane, as a potential (i) energy resource, (ii) factor in global climate change, and (iii) submarine geohazard. Although each of these issues can affect human welfare, only (iii) is considered to be of immediate importance. Assessments of gas hydrate as an energy resource have often been overly optimistic, based in part on its very high methane content and on its worldwide occurrence in continental margins. Although these attributes are attractive, geologic settings, reservoir properties, and phase-equilibria considerations diminish the energy resource potential of natural gas hydrate. The possible role of gas hydrate in global climate change has been often overstated. Although methane is a "greenhouse" gas in the atmosphere, much methane from dissociated gas hydrate may never reach the atmosphere, but rather may be converted to carbon dioxide and sequestered by the hydrosphere/biosphere before reaching the atmosphere. Thus, methane from gas hydrate may have little opportunity to affect global climate change. However, submarine geohazards (such as sediment instabilities and slope failures on local and regional scales, leading to debris flows, slumps, slides, and possible tsunamis) caused by gas-hydrate dissociation are of immediate and increasing importance as humankind moves to exploit seabed resources in ever-deepening waters of coastal oceans. The vulnerability of gas hydrate to temperature and sea level changes enhances the instability of deep-water oceanic sediments, and thus human activities and installations in this setting can be affected. (+info)Some common examples of respiratory tract diseases include:
1. Pneumonia: An infection of the lungs that can be caused by bacteria, viruses, or fungi.
2. Bronchitis: Inflammation of the airways (bronchi) that can cause coughing, wheezing, and difficulty breathing.
3. Asthma: A chronic condition that causes inflammation and narrowing of the airways, leading to symptoms such as wheezing, coughing, and shortness of breath.
4. Chronic obstructive pulmonary disease (COPD): A progressive condition that makes it difficult to breathe due to damage to the lungs over time.
5. Tuberculosis: An infectious disease caused by the bacteria Mycobacterium tuberculosis that primarily affects the lungs.
6. Laryngitis: Inflammation of the voice box (larynx) that can cause hoarseness and difficulty speaking.
7. Tracheitis: Inflammation of the trachea, or windpipe, that can cause coughing, fever, and difficulty breathing.
8. Croup: An infection of the throat and lungs that can cause a barky cough and difficulty breathing.
9. Pleurisy: Inflammation of the lining around the lungs (pleura) that can cause chest pain, fever, and difficulty breathing.
10. Pertussis (whooping cough): An infectious disease caused by the bacteria Bordetella pertussis that can cause coughing fits and difficulty breathing.
These are just a few examples of the many different types of respiratory tract diseases that exist. Each one has its own unique symptoms, causes, and treatment options.
Asthma can cause recurring episodes of wheezing, coughing, chest tightness, and shortness of breath. These symptoms occur when the muscles surrounding the airways contract, causing the airways to narrow and swell. This can be triggered by exposure to environmental allergens or irritants such as pollen, dust mites, pet dander, or respiratory infections.
There is no cure for asthma, but it can be managed with medication and lifestyle changes. Treatment typically includes inhaled corticosteroids to reduce inflammation, bronchodilators to open up the airways, and rescue medications to relieve symptoms during an asthma attack.
Asthma is a common condition that affects people of all ages, but it is most commonly diagnosed in children. According to the American Lung Association, more than 25 million Americans have asthma, and it is the third leading cause of hospitalization for children under the age of 18.
While there is no cure for asthma, early diagnosis and proper treatment can help manage symptoms and improve quality of life for those affected by the condition.
Some common examples of respiration disorders include:
1. Asthma: A chronic condition that causes inflammation and narrowing of the airways, leading to wheezing, coughing, and shortness of breath.
2. Chronic obstructive pulmonary disease (COPD): A progressive lung disease that makes it difficult to breathe, caused by exposure to pollutants such as cigarette smoke.
3. Pneumonia: An infection of the lungs that can cause fever, chills, and difficulty breathing.
4. Bronchitis: Inflammation of the airways that can cause coughing and difficulty breathing.
5. Emphysema: A condition where the air sacs in the lungs are damaged, making it difficult to breathe.
6. Sleep apnea: A sleep disorder that causes a person to stop breathing for short periods during sleep, leading to fatigue and other symptoms.
7. Cystic fibrosis: A genetic disorder that affects the respiratory system and digestive system, causing thick mucus buildup and difficulty breathing.
8. Pulmonary fibrosis: A condition where the lungs become scarred and stiff, making it difficult to breathe.
9. Tuberculosis (TB): A bacterial infection that primarily affects the lungs and can cause coughing, fever, and difficulty breathing.
10. Lung cancer: A type of cancer that originates in the lungs and can cause symptoms such as coughing, chest pain, and difficulty breathing.
These are just a few examples of respiration disorders, and there are many other conditions that can affect the respiratory system and cause breathing difficulties. If you are experiencing any symptoms of respiration disorders, it is important to seek medical attention to receive an accurate diagnosis and appropriate treatment.
A blockage caused by air bubbles in the bloodstream, which can occur after a sudden change in atmospheric pressure (e.g., during an airplane flight or scuba diving). Air embolism can cause a variety of symptoms, including shortness of breath, chest pain, and stroke. It is a potentially life-threatening condition that requires prompt medical attention.
Note: Air embolism can also occur in the venous system, causing a pulmonary embolism (blockage of an artery in the lungs). This is a more common condition and is discussed separately.
The condition is named after Dr. Norman Barrett, who first described it in 1956. It is a precancerous condition, meaning that if left untreated, it can progress to esophageal cancer over time. The exact cause of Barrett esophagus is not fully understood, but chronic acid reflux is thought to play a role in its development.
The symptoms of Barrett esophagus are similar to those of GERD and may include heartburn, difficulty swallowing, chest pain, and regurgitation of food. The condition can be diagnosed through an endoscopy, which involves inserting a flexible tube with a camera into the esophagus to visualize the cells lining the esophagus.
Treatment for Barrett esophagus typically involves controlling the underlying acid reflux through lifestyle changes and medications. In some cases, surgery may be necessary to repair any damage to the esophageal lining or to strengthen the lower esophageal sphincter (LES), which is the muscle that separates the esophagus from the stomach and prevents acid reflux.
It's important for individuals with chronic acid reflux to be screened regularly for Barrett esophagus, as early detection and treatment can help prevent the development of esophageal cancer.
Some common types of lung diseases include:
1. Asthma: A chronic condition characterized by inflammation and narrowing of the airways, leading to wheezing, coughing, and shortness of breath.
2. Chronic Obstructive Pulmonary Disease (COPD): A progressive condition that causes chronic inflammation and damage to the airways and lungs, making it difficult to breathe.
3. Pneumonia: An infection of the lungs that can be caused by bacteria, viruses, or fungi, leading to fever, chills, coughing, and difficulty breathing.
4. Bronchiectasis: A condition where the airways are damaged and widened, leading to chronic infections and inflammation.
5. Pulmonary Fibrosis: A condition where the lungs become scarred and stiff, making it difficult to breathe.
6. Lung Cancer: A malignant tumor that develops in the lungs, often caused by smoking or exposure to carcinogens.
7. Cystic Fibrosis: A genetic disorder that affects the respiratory and digestive systems, leading to chronic infections and inflammation in the lungs.
8. Tuberculosis (TB): An infectious disease caused by Mycobacterium Tuberculosis, which primarily affects the lungs but can also affect other parts of the body.
9. Pulmonary Embolism: A blockage in one of the arteries in the lungs, often caused by a blood clot that has traveled from another part of the body.
10. Sarcoidosis: An inflammatory disease that affects various organs in the body, including the lungs, leading to the formation of granulomas and scarring.
These are just a few examples of conditions that can affect the lungs and respiratory system. It's important to note that many of these conditions can be treated with medication, therapy, or surgery, but early detection is key to successful treatment outcomes.
Clinical Significance:
Respiratory sounds can help healthcare providers diagnose and manage respiratory conditions, such as asthma, chronic obstructive pulmonary disease (COPD), and pneumonia. By listening to the sounds of a patient's breathing, healthcare providers can identify abnormalities in lung function, airway obstruction, or inflammation.
Types of Respiratory Sounds:
1. Vesicular Sounds:
a. Inspiratory wheeze: A high-pitched whistling sound heard during inspiration, usually indicative of bronchial asthma or COPD.
b. Expiratory wheeze: A low-pitched whistling sound heard during expiration, typically seen in patients with chronic bronchitis or emphysema.
c. Decreased vocal fremitus: A decrease in the normal vibratory sounds heard over the lung fields during breathing, which can indicate fluid or consolidation in the lungs.
2. Adventitious Sounds:
a. Crackles (rales): High-pitched, bubbly sounds heard during inspiration and expiration, indicating fluid or air in the alveoli.
b. Rhonchi: Low-pitched, harsh sounds heard during inspiration and expiration, often indicative of bronchitis, pneumonia, or COPD.
c. Stridors: High-pitched, squeaky sounds heard during breathing, commonly seen in patients with inflammatory conditions such as pneumonia or tuberculosis.
It's important to note that the interpretation of lung sounds requires a thorough understanding of respiratory physiology and pathophysiology, as well as clinical experience and expertise. A healthcare professional, such as a nurse or respiratory therapist, should always be consulted for an accurate diagnosis and treatment plan.
There are several types of hypersensitivity reactions, including:
1. Type I hypersensitivity: This is also known as immediate hypersensitivity and occurs within minutes to hours after exposure to the allergen. It is characterized by the release of histamine and other chemical mediators from immune cells, leading to symptoms such as hives, itching, swelling, and difficulty breathing. Examples of Type I hypersensitivity reactions include allergies to pollen, dust mites, or certain foods.
2. Type II hypersensitivity: This is also known as cytotoxic hypersensitivity and occurs within days to weeks after exposure to the allergen. It is characterized by the immune system producing antibodies against specific proteins on the surface of cells, leading to their destruction. Examples of Type II hypersensitivity reactions include blood transfusion reactions and serum sickness.
3. Type III hypersensitivity: This is also known as immune complex hypersensitivity and occurs when antigens bind to immune complexes, leading to the formation of deposits in tissues. Examples of Type III hypersensitivity reactions include rheumatoid arthritis and systemic lupus erythematosus.
4. Type IV hypersensitivity: This is also known as delayed-type hypersensitivity and occurs within weeks to months after exposure to the allergen. It is characterized by the activation of T cells, leading to inflammation and tissue damage. Examples of Type IV hypersensitivity reactions include contact dermatitis and toxic epidermal necrolysis.
The diagnosis of hypersensitivity often involves a combination of medical history, physical examination, laboratory tests, and elimination diets or challenges. Treatment depends on the specific type of hypersensitivity reaction and may include avoidance of the allergen, medications such as antihistamines or corticosteroids, and immunomodulatory therapy.
Respiratory hypersensitivity can be diagnosed through medical history, physical examination, and allergy testing. Treatment options include avoidance of allergens, medication, such as antihistamines or corticosteroids, and immunotherapy, which involves exposing the person to small amounts of the allergen over time to build up their tolerance.
Some people with respiratory hypersensitivity may experience more severe symptoms, such as asthma, which can be life-threatening if left untreated. It is important for individuals with respiratory hypersensitivity to work closely with their healthcare provider to manage their condition and prevent complications.
1. Coronary artery disease: The narrowing or blockage of the coronary arteries, which supply blood to the heart.
2. Heart failure: A condition in which the heart is unable to pump enough blood to meet the body's needs.
3. Arrhythmias: Abnormal heart rhythms that can be too fast, too slow, or irregular.
4. Heart valve disease: Problems with the heart valves that control blood flow through the heart.
5. Heart muscle disease (cardiomyopathy): Disease of the heart muscle that can lead to heart failure.
6. Congenital heart disease: Defects in the heart's structure and function that are present at birth.
7. Peripheral artery disease: The narrowing or blockage of blood vessels that supply oxygen and nutrients to the arms, legs, and other organs.
8. Deep vein thrombosis (DVT): A blood clot that forms in a deep vein, usually in the leg.
9. Pulmonary embolism: A blockage in one of the arteries in the lungs, which can be caused by a blood clot or other debris.
10. Stroke: A condition in which there is a lack of oxygen to the brain due to a blockage or rupture of blood vessels.
Prenatal Exposure Delayed Effects can affect various aspects of the child's development, including:
1. Physical growth and development: PDEDs can lead to changes in the child's physical growth patterns, such as reduced birth weight, short stature, or delayed puberty.
2. Brain development: Prenatal exposure to certain substances can affect brain development, leading to learning disabilities, memory problems, and cognitive delays.
3. Behavioral and emotional development: Children exposed to PDEDs may exhibit behavioral and emotional difficulties, such as anxiety, depression, or attention deficit hyperactivity disorder (ADHD).
4. Immune system functioning: Prenatal exposure to certain substances can affect the immune system's development, making children more susceptible to infections and autoimmune diseases.
5. Reproductive health: Exposure to certain chemicals during fetal development may disrupt the reproductive system, leading to fertility problems or an increased risk of infertility later in life.
The diagnosis of Prenatal Exposure Delayed Effects often requires a comprehensive medical history and physical examination, as well as specialized tests such as imaging studies or laboratory assessments. Treatment for PDEDs typically involves addressing the underlying cause of exposure and providing appropriate interventions to manage any associated symptoms or developmental delays.
In summary, Prenatal Exposure Delayed Effects can have a profound impact on a child's growth, development, and overall health later in life. It is essential for healthcare providers to be aware of the potential risks and to monitor children exposed to substances during fetal development for any signs of PDEDs. With early diagnosis and appropriate interventions, it may be possible to mitigate or prevent some of these effects and improve outcomes for affected children.
Acute bronchitis is a short-term infection that is usually caused by a virus or bacteria, and can be treated with antibiotics and supportive care such as rest, hydration, and over-the-counter pain relievers. Chronic bronchitis, on the other hand, is a long-term condition that is often associated with smoking and can lead to chronic obstructive pulmonary disease (COPD).
Bronchitis can cause a range of symptoms including:
* Persistent cough, which may be dry or produce mucus
* Chest tightness or discomfort
* Shortness of breath or wheezing
* Fatigue and fever
* Headache and body aches
The diagnosis of bronchitis is usually made based on a physical examination, medical history, and results of diagnostic tests such as chest X-rays and pulmonary function tests. Treatment for bronchitis typically focuses on relieving symptoms and managing the underlying cause, such as a bacterial infection or smoking cessation.
Bronchitis can be caused by a variety of factors, including:
* Viral infections, such as the common cold or flu
* Bacterial infections, such as pneumonia
* Smoking and exposure to environmental pollutants
* Asthma and other allergic conditions
* Chronic lung diseases, such as COPD
Preventive measures for bronchitis include:
* Quitting smoking and avoiding exposure to secondhand smoke
* Getting vaccinated against flu and pneumonia
* Practicing good hygiene, such as washing hands frequently
* Avoiding exposure to environmental pollutants
* Managing underlying conditions such as asthma and allergies.
Premature birth can be classified into several categories based on gestational age at birth:
1. Extreme prematurity: Born before 24 weeks of gestation.
2. Very preterm: Born between 24-27 weeks of gestation.
3. Moderate to severe preterm: Born between 28-32 weeks of gestation.
4. Late preterm: Born between 34-36 weeks of gestation.
The causes of premature birth are not fully understood, but several factors have been identified as increasing the risk of premature birth. These include:
1. Previous premature birth
2. Multiple gestations (twins, triplets etc.)
3. History of cervical surgery or cervical incompetence
4. Chronic medical conditions such as hypertension and diabetes
5. Infections such as group B strep or urinary tract infections
6. Pregnancy-related complications such as preeclampsia and placenta previa
7. Stress and poor social support
8. Smoking, alcohol and drug use during pregnancy
9. Poor nutrition and lack of prenatal care.
Premature birth can have significant short-term and long-term health consequences for the baby, including respiratory distress syndrome, bronchopulmonary dysplasia, intraventricular hemorrhage, retinopathy of prematurity and necrotizing enterocolitis. Children who are born prematurely may also have developmental delays, learning disabilities and behavioral problems later in life.
There is no single test that can predict premature birth with certainty, but several screening tests are available to identify women at risk. These include ultrasound examination, maternal serum screening for estriol and pregnancy-associated plasma protein A (PAPP-A), and cervical length measurement.
While there is no proven way to prevent premature birth entirely, several strategies have been shown to reduce the risk, including:
1. Progesterone supplementation: Progesterone appears to help prevent preterm labor in some women with a history of previous preterm birth or other risk factors.
2. Corticosteroids: Corticosteroids given to mothers at risk of preterm birth can help mature the baby's lungs and reduce the risk of respiratory distress syndrome.
3. Calcium supplementation: Calcium may help improve fetal bone development and reduce the risk of premature birth.
4. Good prenatal care: Regular prenatal check-ups, proper nutrition and avoiding smoking, alcohol and drug use during pregnancy can help reduce the risk of premature birth.
5. Avoiding stress: Stress can increase the risk of premature birth, so finding ways to manage stress during pregnancy is important.
6. Preventing infections: Infections such as group B strep and urinary tract infections can increase the risk of premature birth, so it's important to take steps to prevent them.
7. Maintaining a healthy weight gain during pregnancy: Excessive weight gain during pregnancy can increase the risk of premature birth.
8. Avoiding preterm contractions: Preterm contractions can be a sign of impending preterm labor, so it's important to be aware of them and seek medical attention if they occur.
9. Prolonged gestation: Prolonging pregnancy beyond 37 weeks may reduce the risk of premature birth.
10. Cervical cerclage: A cervical cerclage is a stitch used to close the cervix and prevent preterm birth in women with a short cervix or other risk factors.
It's important to note that not all of these strategies will be appropriate or effective for every woman, so it's important to discuss your individual risk factors and any concerns you may have with your healthcare provider.
Indoor air quality
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Nikolai Izmerov
Cannabis cultivation
Environmental impact of cannabis cultivation
Patricia Silveyra
Nitrogen dioxide poisoning
Sick building syndrome
Polycyclic aromatic hydrocarbon
Health equity
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Tapered element oscillating microbalance
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Environmental impact of fracking in the United States
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Environmental issues in India
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Analytical thermal desorption
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Mercury regulation in the United States
Ozone
1,1,1-Trichloroethane
Lame-duck session
List of Statutory Instruments of the United Kingdom, 2019
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WHO EMRO | Regional Committee decisions | Volume 20, issue 10 | EMHJ volume 20, 2014
Characterization of PM, PAHs and Gaseous Pollutants Emitted from Sintering Process and Electric Arc Furnace - Aerosol and Air...
Exposures11
- If asbestos exposure estimates based upon PCM methods were strictly proportional to total exposure to all etiologically-relevant asbestos fibers then a policy that regulated asbestos exposure as measured by PCM might suffice to control etiologically-relevant occupational asbestos exposures. (cdc.gov)
- The project applied innovative epidemiological methods to estimation of associations between occupational exposures and disease in settings of complex, protracted occupational exposure histories, and potential bias due to health-related selection out of employment. (cdc.gov)
- Parental exposures to occupational asthmagens and risk of autism spectrum disorder in a Danish population-based case-control study. (nih.gov)
- It's also important to recognize occupational exposures that can be both seen in an urban or rural environment [can] contribute to asthma-COPD overlap. (medscape.com)
- Fatalities from respiratory diseases and cancers caused by inhalation exposures account for approximately 70% of all occupational disease deaths, according to the National Institute for Occupational Safety and Health (NIOSH). (nih.gov)
- Inhalation exposures to toxic compounds tend to occur more frequently in occupational settings, although the general population can also be exposed to similar hazards through inhalation in the environment or consumer use. (nih.gov)
- Across these independent studies, shared risk factors for RA and airway disease were determined to include older age, cigarette smoking, lower socioeconomic status, infections and microbial dysbiosis, occupational exposures and air pollutants, and poor nutrition [6, 7]. (nih.gov)
- However, of these shared risk factors, only older age, cigarette smoking, and occupational exposures have been specifically investigated as risk factors for airway disease among patients with RA. (nih.gov)
- Travelers should be mindful of, and limit exposures to, outdoor and indoor air pollution and carbon monoxide ( Table 4-02 ). (cdc.gov)
- The researchers note that the study results are "parallel" to findings regarding "other environmental toxicants" and "air pollutants," suggesting a need to inspect the impact of occupational and environmental exposures on the severity of COVID-19. (personalinjurylawcal.com)
- Dr. Bailey has investigated exposures to alternating current, direct current, and radiofrequency electromagnetic fields, 'stray voltage', and electrical shock, as well as to a variety of chemical agents and air pollutants. (exponent.com)
Hazardous8
- Indoor air pollution and exposure to hazardous substances in the home are risks we can do something about. (cdc.gov)
- Information on levels of exposure to these compounds as measured by their levels in blood is essential to determine the need for regulatory mechanisms to reduce the levels of hazardous air pollutants to which the general population is exposed. (cdc.gov)
- Action to manage these risks is cost-effective and can result in major reductions in the burden of disease associated with air pollution, chemical toxins, radiation, hazardous wastes and traffic accidents. (who.int)
- Other products used by janitorial staff such as floor strippers also impact indoor air quality and are hazardous to workers. (turi.org)
- Styrene is one of many substances selected for TRI reporting as a hazardous air pollutant. (styrene.org)
- The U.S. Occupational Safety and Health Administration regulates styrene as a hazardous substance because of short-term reversible central nervous system effects such as drowsiness and delayed reaction time that may be experienced from exposure to styrene in the workplace. (styrene.org)
- Zimmetry Environmental offers testing and consulting services to identify hazardous materials and mitigate occupational exposure risks at recycling and reuse facilities. (webwire.com)
- Protecting workers and the environment from hazardous materials and airborne pollutants are the professionals at Zimmetry Environmental. (webwire.com)
Particulate matter3
- The common air pollutants emitted with engine exhaust include particulate matter, carbon monoxide, sulfur dioxide, and nitrogen oxides. (dieselnet.com)
- Motor vehicles emit a variety of air pollutants including nitrogen oxides, elemental carbon (EC), particulate matter ≤2.5 μm in aerodynamic diameter (PM 2.5 ), ultrafine particles (UFP), heavy metals, polynuclear aromatic hydrocarbons, and volatile organic compounds (VOC). (dieselnet.com)
- The association between long-term exposure to air pollutants, including nitrogen dioxide (NO 2 ), carbon monoxide (CO), sulfur dioxide (SO 2 ), ozone (O 3 ), and particulate matter 10 μm or less in diameter (PM 10 ), and mortality by ischemic heart disease (IHD), cerebrovascular disease (CVD), pneumonia (PN), and chronic lower respiratory disease (CLRD) is unclear. (biomedcentral.com)
Airborne pollutants1
- The lung is the most common site of injury by airborne pollutants. (cpsc.gov)
PAHs2
- The samples were then processed and analyzed to measure concentrations of pollutants known as polycyclic aromatic hydrocarbons (PAHs), which are linked with poor health outcomes, including different types of cancer. (eurekalert.org)
- PAHs also attach themselves to particles in the air, meaning once they settle, they can be moved around by flood waters. (eurekalert.org)
Respiratory7
- We attempted to determine the association between occupational and air pollutant exposure with the development of adult asthma through the analysis of a standardized respiratory questionnaire administered to a cohort of 3914 nonsmoking adults in 1977 and again in 1987. (nih.gov)
- Effects of long-term exposure to traffic-related air pollution on respiratory and cardiovascular mortality in the Netherlands: the NLCS-AIR study. (nih.gov)
- Urban-rural-occupational air pollutants or respiratory sensitizers impact asthma and the asthma-COPD overlap features," Jill A. Poole, MD, division chief of allergy and immunology at the University of Nebraska Medical Center, Omaha, said in her presentation. (medscape.com)
- According to the Canadian Centre for Occupational Health and Safety , even very low concentrations of ozone can be harmful to the upper respiratory tract and the lungs. (thestar.com)
- The health outcomes associated with the most common air pollutants (NO 2 , EC, and PM 2.5 ) with a confidence assessment rated 'high' or 'moderate-to-high' included: asthma onset in children and adults, acute lower respiratory infection in children, and mortality-from all-causes and circulatory, from lung cancer, and from ischemic heart disease. (dieselnet.com)
- Because a few prominent aspects of indoor air pollution, notably environmental tobacco smoke and "sick building syndrome," have been brought to public attention, individuals may volunteer suggestions of a connection between respiratory or other symptoms and conditions in the home or, especially, the workplace. (cpsc.gov)
- Inhalation exposure could result in occupational asthma, allergies, hypersensitivity pneumonitis (HP) and other respiratory diseases. (webwire.com)
Pollution22
- Ambient air pollution concentrations were estimated over a 20-year period using monthly interpolations from fixed-site monitoring stations applied to zip code locations by month of residence and work site. (nih.gov)
- Thus, for many people, the risks to health from exposure to indoor air pollution may be greater than risks from outdoor pollution. (cdc.gov)
- Numerous forms of indoor air pollution are possible in the modern home. (cdc.gov)
- While air pollution did not increase the risk of developing asthma-COPD overlap, there was an association between body mass index, low level of education, living in a rural area, and smoking status. (medscape.com)
- Epidemiological studies have shown that exposure to traffic-related air pollution causes increased morbidity and mortality, such as from cardiovascular and pulmonary disease. (dieselnet.com)
- Motor vehicles are a significant source of air pollution and adverse health effects, particularly in large urban centers. (dieselnet.com)
- Traffic-related air pollution (TRAP) is a complex mixture of gases and particles resulting from the use of engines and vehicles. (dieselnet.com)
- In addition, air pollution from other sources-such as industry, oil, coal, and wood burning, and agricultural sources as well as atmospheric transport of pollutants from distant sources-contributes to the overall air quality. (dieselnet.com)
- The task of quantifying the health effects of air pollution and correlating them with the exposure to particular pollutants is challenging. (dieselnet.com)
- Therefore, it is difficult to ascertain which pollutant is chiefly responsible for the adverse effects of pollution, or whether they are attributable to the mixture as such. (dieselnet.com)
- Air pollution studies not adequately adjusting for traffic noise exposure may overestimate the cardiovascular disease burden of air pollution. (dieselnet.com)
- Following the advice given will not necessarily provide complete protection in all situations or against all health hazards that may be caused by indoor air pollution. (cpsc.gov)
- Indoor air pollution poses many challenges to the health professional. (cpsc.gov)
- This booklet addresses the indoor air pollution problems that may be caused by contaminants encountered in the daily lives of persons in their homes and offices. (cpsc.gov)
- The health professional should use this booklet as a tool in diagnosing an individual's signs and symptoms that could be related to an indoor air pollution problem. (cpsc.gov)
- Although air pollution has decreased in many parts of the world, it represents a major and growing health problem for the residents of some cities in certain industrializing countries. (cdc.gov)
- Conversely, those with preexisting heart and lung disease, children, and older adults have an increased risk for adverse health effects from even short-term exposure to air pollution. (cdc.gov)
- 80 countries around the world, and the World Health Organization posts historical data on outdoor air pollution in urban areas. (cdc.gov)
- Secondhand smoke from smoking tobacco is a primary contributor to indoor air pollution. (cdc.gov)
- We investigated whether living in an administrative district with heavy air pollution is associated with an increased risk of mortality by the diseases through an ecological study using South Korean administrative data over 19 years. (biomedcentral.com)
- This may be partly because collecting and analyzing long-term air pollution and cardiopulmonary mortality data together are relatively difficult than collecting and analyzing short-term data together. (biomedcentral.com)
- To gain a better understanding of how flooding mobilized pollution in the area, a research team led by Garett Sansom, DrPH, research assistant professor in the Department of Environmental and Occupational Health at the Texas A&M University School of Public Health, analyzed samples of soil from the Manchester neighborhood collected immediately after Hurricane Harvey. (eurekalert.org)
Ambient air5
- The method described in this paper could also be extended to setting limits for ambient air contaminants where risk perception plays a dominant role in whether the public views the exposure as being reasonable or safe. (cdc.gov)
- In most jurisdictions, these pollutants are also subject to ambient air quality standards and occupational health exposure limits. (dieselnet.com)
- Engine and vehicle emissions undergo dilution and transformation in the ambient air. (dieselnet.com)
- Opines that sam 'ih' sampler, an industrial hygienist, set up a project to take 5 personal samples and one area sample of airborne contaminants to obtain information and track the level of contaminate in the ambient air. (123helpme.com)
- Recent observations have suggested that radon in the ambient air may cause cancers at sites other than the lung, but the evidence is indirect. (ox.ac.uk)
Contaminants1
- Respirators are specifically designed to remove contaminants from the air or to provide clean respirable air from another source. (cdc.gov)
Asthma5
- Some of the known triggers of intrinsic asthma are infections, such as cold and flu viruses, exercise and cold air, industrial and occupational pollutants, food additives and preservatives, drugs such as aspirin, and emotional stress. (cdc.gov)
- Parental occupational asthmagen exposure was estimated by linking DISCO-88 codes to an asthma-specific job-exposure matrix.Our maternal analyses included 6706 case mothers and 29,359 control mothers employed during the pregnancy period. (nih.gov)
- The development and worsening of overlapping asthma and chronic obstructive pulmonary disease (COPD) can be affected by pollutants found in rural and urban environments, according to a recent presentation at the annual meeting of the American Academy of Allergy, Asthma, and Immunology, held virtually this year. (medscape.com)
- Traditional cleaning products contain chemicals that can cause short term ailments such skin, eye, lung irritation, and longer-term health problems such as occupational asthma, organ damage, or potentially cancer. (turi.org)
- According to Massachusetts Department of Public Health's SENSOR Work-Related Asthma surveillance project, indoor air pollutants (21%) and cleaning products (17%) accounted for the most frequently reported cases of occupational asthma between 1993 and 2002. (turi.org)
Emissions3
- Air pollutant levels in the home increase if not enough outdoor air is brought in to dilute emissions from indoor sources and to carry indoor air pollutants out of the home. (cdc.gov)
- When emitted through vehicle exhaust, these pollutants are called exhaust or tailpipe emissions. (dieselnet.com)
- The results of these emissions are elevated concentrations of air pollutants through primary emissions and through the formation of secondary pollutants, such as secondary PM and ozone. (dieselnet.com)
Dust2
- Increased pesticide concentrations in house dust in agricultural areas have been attributed to several exposure pathways, including agricultural drift, para-occupational, and residential use. (nih.gov)
- Dust masks, surgical masks, and bandanas offer limited protection against severely polluted air. (cdc.gov)
OSHA3
- President Barack Obama, in conjunction with the U.S. Department of Labor's Occupational Safety and Health Administration (OSHA), proclaimed April 28, 2010 to be Worker's Memorial Day to celebrate and preserve the memory "of those who have been killed due to unsafe working conditions. (rapoportlaw.com)
- Three different companies in Wisconsin were hit with proposed fines by the Occupational Safety and Health Administration (OSHA) during the months of March and April for alleged violations of federal workplace health and safety regulations. (rapoportlaw.com)
- The Occupational Safety and Health Administration (OSHA) reports that recycling can be dangerous for workers since certain materials that are recycled or reused, such as scrap metal, electronics, batteries, and used oil and other chemicals, have materials that directly pose hazards to workers. (webwire.com)
Ozone3
- The prevalence of work-related symptoms among mild steel welders was studied, and the occupational exposure to welding fumes was quantified by repeated measurements of RD, respirable Mn, and ozone. (nih.gov)
- The perennial household nuisance actually purifies the air by neutralizing ozone that can harm our lungs. (thestar.com)
- However, there is a knowledge gap regarding whether exposure to higher concentrations of air pollutants, including carbon monoxide (CO), sulfur dioxide (SO 2 ), nitrogen dioxide (NO 2 ), ozone (O 3 ), and PM 10 in a residential district in South Korea over a long term, such as 19 years, would be associated with higher cardiopulmonary mortality. (biomedcentral.com)
Mitigate1
- Operating out of Puerto Rico, their environmental and air quality experts offer testing and consulting services to identify, mitigate and eliminate hazards. (webwire.com)
Breathe2
- But when it's down here, closer to us in the air that we breathe, it's a pollutant. (thestar.com)
- Most residents are not worried about what pollutants are filling the air that they breathe, nor should they have to be concerned about this. (rapoportlaw.com)
Toxic3
- For example, experience has shown that chemicals with a low odor threshold often require low OELs even though many are not toxic or do not cause irrita tion at those air concentrations. (cdc.gov)
- Toxic air-borne matter related to work performed They are usually produced by the specific nature of the occupation. (nih.gov)
- We know that when we reduce the use of toxic materials in the workplace our costs, represented by what we pay for training, air monitoring, elaborate ventilation systems, personal protective equipment, and ultimately potential health impact, decreases. (cdc.gov)
Chemical1
- Indoor pollutants can be placed into two groups, biologic and chemical. (cdc.gov)
Asbestos3
- Occupational standards for asbestos exposure in the US are based on visual counting of particles by phase contrast microscopy (PCM). (cdc.gov)
- However, in most occupational settings the majority of asbestos fibers are too small to count by PCM. (cdc.gov)
- The professionals at Zimmetry offer environmental compliance, indoor air quality, asbestos, lead-based paint, Phase I ESAs and general environmental consulting services. (webwire.com)
Hazards1
- Heavy industry-related occupational hazards are generally regulated and likely to be dealt with by an on-site or company physician or other health personnel2. (cpsc.gov)
Symptoms2
Mortality3
- Multivariate beta-regression analysis was performed to estimate the associations between air pollutant concentrations and mortality rates, after adjusting for confounding factors including altitude, population density, higher education rate, smoking rate, obesity rate, and gross regional domestic product per capita. (biomedcentral.com)
- For IHD, higher SO 2 concentrations were significantly associated with a higher mortality rate, whereas other air pollutants had null associations. (biomedcentral.com)
- In the subgroup analysis, positive associations between SO 2 concentrations and IHD mortality were consistently observed in all subgroups, while other pollutant-disease pairs showed null, or mixed associations. (biomedcentral.com)
Environmental health1
- The department employs environmental health specialists who work to ensure that the air, water, and food supply is safe for the public. (kinsburg.ru)
NIEHS1
- Much of the work carried out by DTT is in support of the National Toxicology Program (NTP), an interagency partnership of the Food and Drug Administration, National Institute for Occupational Safety and Health, and NIEHS. (nih.gov)
Injuries4
- For example, an estimated 20% of vehicle crashes are occupational injuries ( 1 ), and each year, approximately linked to drowsy driving ( 3 ). (cdc.gov)
- Overall, 30.0% of civilian departments for occupational injuries and illnesses, employed U.S. adults (approximately 40.6 million workers) and approximately 110,000 were hospitalized (CDC, reported an average sleep duration of 6 hours per day. (cdc.gov)
- prevalence of short sleep duration (6 hours per day) varied by Economists are working to calculate the costs associated industry of employment (range: 24.1%-41.6%), with a sig- with occupational injuries and illnesses in the United nificantly higher rate of short sleep duration among workers in States. (cdc.gov)
- Fatal occupational injuries by industry and selected event or exposure, 2010 (preliminary). (cdc.gov)
Risk1
- Driving in cars, flying in airplanes, engaging in recreational activities, and being exposed to environmental pollutants all pose varying degrees of risk. (cdc.gov)
Data1
- More data on baseline pollutant concentrations and improved analysis methods will help researchers, policy makers and community leaders assess the risks people living in environmental justice communities face, and possibly find ways to limit the health risks residents of neighborhoods like Manchester face in the future. (eurekalert.org)
Levels1
- In addition, high temperature and humidity levels can increase the concentration of some pollutants. (cdc.gov)
Health effects2
Allergies2
- Although pollen allergies can be bothersome, asthmatic responses to pollutants can be life threatening. (cdc.gov)
- The challenge is further compounded by the similar manifestations of many of the pollutants and by the similarity of those effects, in turn, to those that may be associated with allergies, influenza, and the common cold. (cpsc.gov)
Chemicals1
- OBJECTIVES: Setting occupational exposure limits (OELs) for odorous or irrita ting chemicals is a global occupational health challenge. (cdc.gov)
Include1
- Other potential sources of indoor air pollutants include cooking or combustion sources (e.g., kerosene, coal, wood, animal dung). (cdc.gov)
Urban5
- A patient's interaction with urban, rural, and occupational environments may additionally impact their disease, Poole explained. (medscape.com)
- 1. Urinary Nickel and Progesterone in Workers Exposed to Urban Pollutants. (nih.gov)
- 6. Immune parameters in female workers exposed to urban pollutants. (nih.gov)
- 7. Occupational exposure to urban pollutants and plasma insulin. (nih.gov)
- 8. Occupational exposure to urban pollutants and urinary 5-hydroxy-3-indoleacetic acid. (nih.gov)
Duration1
- The analysis compared sleep duration by nonfatal occupational injury or illness ( 3 ). (cdc.gov)
Safety2
- The National Institute for Occupational Safety and Health (NIOSH) provides a testing, approval, and certification program for respirators . (cdc.gov)
- Ms. Montgomery is an Occupational Health and Safety Specialist at the Center for Research on Occupational and Environmental Toxicology (CROET) at Oregon Health and Science University. (cdc.gov)
Water3
- The international, national, and state regulations and guidelines regarding ethion in air, water, and other media are summarized in Table 7-1. (cdc.gov)
- air and water pollutant. (nih.gov)
- Styrene dissipates rapidly when released into the air or water. (styrene.org)