Acyclovir: A GUANOSINE analog that acts as an antimetabolite. Viruses are especially susceptible. Used especially against herpes.Antiviral Agents: Agents used in the prophylaxis or therapy of VIRUS DISEASES. Some of the ways they may act include preventing viral replication by inhibiting viral DNA polymerase; binding to specific cell-surface receptors and inhibiting viral penetration or uncoating; inhibiting viral protein synthesis; or blocking late stages of virus assembly.Herpes Simplex: A group of acute infections caused by herpes simplex virus type 1 or type 2 that is characterized by the development of one or more small fluid-filled vesicles with a raised erythematous base on the skin or mucous membrane. It occurs as a primary infection or recurs due to a reactivation of a latent infection. (Dorland, 27th ed.)Herpes Genitalis: Infection of the genitals (GENITALIA) with HERPES SIMPLEX VIRUS in either the males or the females.Herpes Zoster: An acute infectious, usually self-limited, disease believed to represent activation of latent varicella-zoster virus (HERPESVIRUS 3, HUMAN) in those who have been rendered partially immune after a previous attack of CHICKENPOX. It involves the SENSORY GANGLIA and their areas of innervation and is characterized by severe neuralgic pain along the distribution of the affected nerve and crops of clustered vesicles over the area. (From Dorland, 27th ed)Herpes Labialis: Herpes simplex, caused by type 1 virus, primarily spread by oral secretions and usually occurring as a concomitant of fever. It may also develop in the absence of fever or prior illness. It commonly involves the facial region, especially the lips and the nares. (Dorland, 27th ed.)Herpesvirus 2, Human: A species of SIMPLEXVIRUS associated with genital infections (HERPES GENITALIS). It is transmitted by sexual intercourse and close personal contact.Simplexvirus: A genus of the family HERPESVIRIDAE, subfamily ALPHAHERPESVIRINAE, consisting of herpes simplex-like viruses. The type species is HERPESVIRUS 1, HUMAN.Valine: A branched-chain essential amino acid that has stimulant activity. It promotes muscle growth and tissue repair. It is a precursor in the penicillin biosynthetic pathway.Foscarnet: An antiviral agent used in the treatment of cytomegalovirus retinitis. Foscarnet also shows activity against human herpesviruses and HIV.Ganciclovir: An ACYCLOVIR analog that is a potent inhibitor of the Herpesvirus family including cytomegalovirus. Ganciclovir is used to treat complications from AIDS-associated cytomegalovirus infections.Encephalitis, Herpes Simplex: An acute (or rarely chronic) inflammatory process of the brain caused by SIMPLEXVIRUS infections which may be fatal. The majority of infections are caused by human herpesvirus 1 (HERPESVIRUS 1, HUMAN) and less often by human herpesvirus 2 (HERPESVIRUS 2, HUMAN). Clinical manifestations include FEVER; HEADACHE; SEIZURES; HALLUCINATIONS; behavioral alterations; APHASIA; hemiparesis; and COMA. Pathologically, the condition is marked by a hemorrhagic necrosis involving the medial and inferior TEMPORAL LOBE and orbital regions of the FRONTAL LOBE. (From Adams et al., Principles of Neurology, 6th ed, pp751-4)Ointments: Semisolid preparations used topically for protective emollient effects or as a vehicle for local administration of medications. Ointment bases are various mixtures of fats, waxes, animal and plant oils and solid and liquid hydrocarbons.Herpes Zoster Ophthalmicus: Virus infection of the Gasserian ganglion and its nerve branches characterized by pain and vesicular eruptions with much swelling. Ocular involvement is usually heralded by a vesicle on the tip of the nose. This area is innervated by the nasociliary nerve.Prodrugs: A compound that, on administration, must undergo chemical conversion by metabolic processes before becoming the pharmacologically active drug for which it is a prodrug.GuanineStomatitis, Herpetic: Stomatitis caused by Herpesvirus hominis. It usually occurs as acute herpetic stomatitis (or gingivostomatitis), an oral manifestation of primary herpes simplex seen primarily in children and adolescents.Herpesvirus 3, Human: The type species of VARICELLOVIRUS causing CHICKENPOX (varicella) and HERPES ZOSTER (shingles) in humans.Keratitis, Dendritic: A form of herpetic keratitis characterized by the formation of small vesicles which break down and coalesce to form recurring dendritic ulcers, characteristically irregular, linear, branching, and ending in knoblike extremities. (Dictionary of Visual Science, 3d ed)Thymidine Kinase: An enzyme that catalyzes the conversion of ATP and thymidine to ADP and thymidine 5'-phosphate. Deoxyuridine can also act as an acceptor and dGTP as a donor. (From Enzyme Nomenclature, 1992) EC 2.7.1.21.Herpesvirus 1, Human: The type species of SIMPLEXVIRUS causing most forms of non-genital herpes simplex in humans. Primary infection occurs mainly in infants and young children and then the virus becomes latent in the dorsal root ganglion. It then is periodically reactivated throughout life causing mostly benign conditions.Administration, Oral: The giving of drugs, chemicals, or other substances by mouth.Administration, Topical: The application of drug preparations to the surfaces of the body, especially the skin (ADMINISTRATION, CUTANEOUS) or mucous membranes. This method of treatment is used to avoid systemic side effects when high doses are required at a localized area or as an alternative systemic administration route, to avoid hepatic processing for example.Chickenpox: A highly contagious infectious disease caused by the varicella-zoster virus (HERPESVIRUS 3, HUMAN). It usually affects children, is spread by direct contact or respiratory route via droplet nuclei, and is characterized by the appearance on the skin and mucous membranes of successive crops of typical pruritic vesicular lesions that are easily broken and become scabbed. Chickenpox is relatively benign in children, but may be complicated by pneumonia and encephalitis in adults. (From Dorland, 27th ed)Virus Shedding: The expelling of virus particles from the body. Important routes include the respiratory tract, genital tract, and intestinal tract. Virus shedding is an important means of vertical transmission (INFECTIOUS DISEASE TRANSMISSION, VERTICAL).Viral Plaque Assay: Method for measuring viral infectivity and multiplication in CULTURED CELLS. Clear lysed areas or plaques develop as the VIRAL PARTICLES are released from the infected cells during incubation. With some VIRUSES, the cells are killed by a cytopathic effect; with others, the infected cells are not killed but can be detected by their hemadsorptive ability. Sometimes the plaque cells contain VIRAL ANTIGENS which can be measured by IMMUNOFLUORESCENCE.Encephalitis, Varicella Zoster: Inflammation of brain tissue caused by infection with the varicella-zoster virus (HERPESVIRUS 3, HUMAN). This condition is associated with immunocompromised states, including the ACQUIRED IMMUNODEFICIENCY SYNDROME. Pathologically, the virus tends to induce a vasculopathy and infect oligodendrocytes and ependymal cells, leading to CEREBRAL INFARCTION, multifocal regions of demyelination, and periventricular necrosis. Manifestations of varicella encephalitis usually occur 5-7 days after onset of HERPES ZOSTER and include HEADACHE; VOMITING; lethargy; focal neurologic deficits; FEVER; and COMA. (From Joynt, Clinical Neurology, 1996, Ch 26, pp29-32; Hum Pathol 1996 Sep;27(9):927-38)Bell Palsy: A syndrome characterized by the acute onset of unilateral FACIAL PARALYSIS which progresses over a 2-5 day period. Weakness of the orbicularis oculi muscle and resulting incomplete eye closure may be associated with corneal injury. Pain behind the ear often precedes the onset of paralysis. This condition may be associated with HERPESVIRUS 1, HUMAN infection of the facial nerve. (Adams et al., Principles of Neurology, 6th ed, p1376)Arabinofuranosyluracil: A pyrimidine nucleoside formed in the body by the deamination of CYTARABINE.Skin Diseases, Viral: Skin diseases caused by viruses.Phosphonoacetic Acid: A simple organophosphorus compound that inhibits DNA polymerase, especially in viruses and is used as an antiviral agent.Idoxuridine: An analog of DEOXYURIDINE that inhibits viral DNA synthesis. The drug is used as an antiviral agent.Virus Activation: The mechanism by which latent viruses, such as genetically transmitted tumor viruses (PROVIRUSES) or PROPHAGES of lysogenic bacteria, are induced to replicate and then released as infectious viruses. It may be effected by various endogenous and exogenous stimuli, including B-cell LIPOPOLYSACCHARIDES, glucocorticoid hormones, halogenated pyrimidines, IONIZING RADIATION, ultraviolet light, and superinfecting viruses.Pharmaceutical Vehicles: A carrier or inert medium used as a solvent (or diluent) in which the medicinally active agent is formulated and or administered. (Dictionary of Pharmacy, 1986)Inosine Pranobex: An alkylamino-alcohol complex of inosine used in the treatment of a variety of viral infections. Unlike other antiviral agents, it acts by modifying or stimulating cell-mediated immune processes rather than acting on the virus directly.Double-Blind Method: A method of studying a drug or procedure in which both the subjects and investigators are kept unaware of who is actually getting which specific treatment.Recurrence: The return of a sign, symptom, or disease after a remission.Vidarabine: A nucleoside antibiotic isolated from Streptomyces antibioticus. It has some antineoplastic properties and has broad spectrum activity against DNA viruses in cell cultures and significant antiviral activity against infections caused by a variety of viruses such as the herpes viruses, the VACCINIA VIRUS and varicella zoster virus.Keratitis, Herpetic: A superficial, epithelial Herpesvirus hominis infection of the cornea, characterized by the presence of small vesicles which may break down and coalesce to form dendritic ulcers (KERATITIS, DENDRITIC). (Dictionary of Visual Science, 3d ed)Electronic Mail: Messages between computer users via COMPUTER COMMUNICATION NETWORKS. This feature duplicates most of the features of paper mail, such as forwarding, multiple copies, and attachments of images and other file types, but with a speed advantage. The term also refers to an individual message sent in this way.Food Dispensers, Automatic: Mechanical food dispensing machines.Editorial Policies: The guidelines and policy statements set forth by the editor(s) or editorial board of a publication.Authorship: The profession of writing. Also the identity of the writer as the creator of a literary production.Periodicals as Topic: A publication issued at stated, more or less regular, intervals.Postal Service: The functions and activities carried out by the U.S. Postal Service, foreign postal services, and private postal services such as Federal Express.Internet: A loose confederation of computer communication networks around the world. The networks that make up the Internet are connected through several backbone networks. The Internet grew out of the US Government ARPAnet project and was designed to facilitate information exchange.Solutions: The homogeneous mixtures formed by the mixing of a solid, liquid, or gaseous substance (solute) with a liquid (the solvent), from which the dissolved substances can be recovered by physical processes. (From Grant & Hackh's Chemical Dictionary, 5th ed)Sodium: A member of the alkali group of metals. It has the atomic symbol Na, atomic number 11, and atomic weight 23.Universal Precautions: Prudent standard preventive measures to be taken by professional and other health personnel in contact with persons afflicted with a communicable disease, to avoid contracting the disease by contagion or infection. Precautions are especially applicable in the diagnosis and care of AIDS patients.Injections: Introduction of substances into the body using a needle and syringe.Skin Cream: A water-soluble medicinal preparation applied to the skin.Ice Cream: A frozen dairy food made from cream or butterfat, milk, sugar, and flavorings. Frozen custard and French-type ice creams also contain eggs.

Infectious complications in 126 patients treated with high-dose chemotherapy and autologous peripheral blood stem cell transplantation. (1/1121)

The effect of an extensive prophylactic antimicrobial regimen was prospectively assessed in 126 patients after high-dose chemotherapy and autologous PBSC. They received ciprofloxacin (500 mg/12 h), acyclovir (200 mg/6 h), and itraconazole (200 mg/12 h) orally until neutrophil recovery. Febrile patients received i.v. imipenem (500 mg/6 h) to which vancomycin and amikacin were added if fever persisted for 2-3 and 5 days, respectively. Amphotericin B lipid complex was further given on day 7 or 8 of fever. Median times for a neutrophil count of >0.5 x 10(9)/l and a platelet count of >20 x 10(9)/l were 9 and 11 days. Severe neutropenia (<0.1 x 10(9)/l) lasted for a median of 5 days in which 72% of febrile episodes and 50% of cases of bacteremia occurred. Gram-positive bacteria were isolated in 30 of 40 episodes of bacteremia, 25 of which were caused by Staphylococcus epidermidis. Clinical foci were the intravascular catheter in 35 cases, respiratory infection in 11, cellulitis in two, anal abscess in one, and neutropenic enterocolitis in one. The high incidence of febrile episodes (94%) and bacteremias (31%) may be due to the lack of efficacy of antimicrobial prophylaxis and the persistence of a 5-day period of severe neutropenia.  (+info)

B cell lymphoproliferative disorders following hematopoietic stem cell transplantation: risk factors, treatment and outcome. (2/1121)

Twenty-six cases of B cell lymphoproliferative disorder (BLPD) were identified among 2395 patients following hematopoietic stem cell transplants (HSCT) for which an overall incidence of BLPD was 1.2%. The true incidence was probably higher, since 9/26 of the diagnoses were made at autopsy. No BLPD was observed following autologous HSCT, so risk factor analyses were confined to the 1542 allogeneic HSCT. Factors assessed were HLA-mismatching (> or = 1 antigen), T cell depletion (TCD), presence of acute GvHD (grades II-IV), donor type (related vs unrelated), age of recipient and donor, and underlying disease. Factors found to be statistically significant included patients transplanted for immune deficiency and CML, donor age > or = 18 years, TCD, and HLA-mismatching, with recipients of combined TCD and HLA-mismatched grafts having the highest incidence. Factors found to be statistically significant in a multiple regression analysis were TCD, donor age and immune deficiency, although 7/8 of the patients with immunodeficiencies and BLPD received a TCD graft from a haploidentical parent. The overall mortality was 92% (24/26). One patient had a spontaneous remission, but subsequently died >1 year later of chronic GVHD. Thirteen patients received therapy for BLPD. Three patients received lymphocyte infusions without response. The only patients with responses and longterm survival received alpha interferon (alphaIFN). Of seven patients treated with alphaIFN there were four responses (one partial and three complete). These data demonstrate that alphaIFN can be an effective agent against BLPD following HSCT, if a timely diagnosis is made.  (+info)

Interactions of a nonpeptidic drug, valacyclovir, with the human intestinal peptide transporter (hPEPT1) expressed in a mammalian cell line. (3/1121)

The results of previous work performed in our laboratory using an in situ perfusion technique in rats and rabbit apical brush border membrane vesicles have suggested that the intestinal uptake of valacyclovir (VACV) appears to be mediated by multiple membrane transporters. Using these techniques, it is difficult to characterize the transport kinetics of VACV with each individual transporter in the presence of multiple known or unknown transporters. The purpose of this study was to characterize the interaction of VACV and the human intestinal peptide transporter using Chinese hamster ovary (CHO) cells that overexpress the human intestinal peptide transporter (hPEPT1) gene. VACV uptake was significantly greater in CHO cells transfected with hPEPT1 than in cells transfected with only the vector, pcDNA3. The optimum pH for VACV uptake was determined to occur at pH 7.5. Proton cotransport was not observed in hPEPT1/CHO cells, consistent with previously observed results in tissues and Caco-2 cells. VACV uptake was concentration dependent and saturable with a Michaelis-Menten constant and maximum velocity of 1.64 +/- 0.06 mM and 23.34 +/- 0.36 nmol/mg protein/5 min, respectively. A very similar Km value was obtained in hPEPT1/CHO cells and in rat and rabbit tissues and Caco-2 cells, suggesting that hPEPT1 dominates the intestinal transport properties of VACV in vitro. VACV uptake was markedly inhibited by various dipeptides and beta-lactam antibiotics, and Ki values of 12.8 +/- 2.7 and 9.1 +/- 1.2 mM were obtained for Gly-Sar and cefadroxil at pH 7.5, respectively. The present results demonstrate that VACV is a substrate for the human intestinal peptide transporter in hPEPT1/CHO cells and that although transport is pH dependent, proton cotransport is not apparent. Also, the results demonstrate that the hPEPT1/CHO cell system has use in investigating the transport kinetics of drugs with the human intestinal peptide transporter hPEPT1; however, the extrapolation of these transport properties to the in vivo situation requires further investigation.  (+info)

Management of human cytomegalovirus infection and disease after allogeneic bone marrow transplantation. (4/1121)

BACKGROUND AND OBJECTIVE: Human cytomegalovirus (HCMV) infection and disease remain a major cause of morbidity and mortality after bone marrow transplantation. HCMV disease, especially pneumonitis, may be treated with ganciclovir and immunoglobulin but even so the outcome is poor with mortality rates of 30-70%. It is therefore imperative to treat HCMV infection before it develops into disease. The aim of this article is to describe the main strategies used to prevent HCMV infection and to improve the survival after CMV disease in bone marrow transplant recipients. INFORMATION SOURCES: In the present review, we examined personal papers in this field and articles published in journals covered by the Science Citation Index and Medline. STATE OF THE ART: Major advances have been made in preventing HCMV infection and disease through two different approaches, both of which reduce HCMV induced morbidity and mortality: In pre-emptive therapy, patients are given ganciclovir when HCMV infection is first identified and this is continued 3-4 months after transplantation; in prophylactic therapy ganciclovir is given to all patients at risk of HCMV disease from engraftment up to 3-4 months post transplantation. Each strategy has advantages and disadvantages and there is no evidence for the superiority of one over the other since the overall survival is the same and the incidence of death from HCMV disease is similar. PERSPECTIVES: The use of more sensitive tests such as HCMV PCR or antigenemia may improve the outcome but probably will not eradicate all HCMV disease. Future possible strategies could include adoptive transfer of CD8+ HCMV-specific cytotoxic T lymphocytes clones derived from the donor marrow or boosting donor or patient immunity using subunit anti-HCMV vaccines such as gB or pp65.  (+info)

Antiviral therapy for neonatal herpes simplex virus: a cost-effectiveness analysis. (5/1121)

Each year, about 1,600 infants in the United States are infected with neonatal herpes simplex virus. We conducted a cost-effectiveness analysis of antiviral drug therapy (acyclovir) for three forms of herpes simplex virus infection: skin, ear, and mouth (SEM), central nervous system (CNS), and disseminated multiorgan (DIS) disease. Five levels of patient outcomes were examined (normal, mild, moderate, severe, dead). We obtained information on disease occurrence and survival from clinical trials and historical reviews of untreated newborns. We considered approaches for treating all or any of the forms of the disease and compared them with no use of antiviral drugs. The main measure of effectiveness was lives saved, including those of descendants of survivors. Costs were measured from a societal perspective and included direct medical costs, institutional care, and special education. We used a discount rate of 3% and valued dollars at 1995 levels. We also considered the perspective of a managed care organization. From a societal viewpoint relative to no treatment, antiviral therapy for SEM resulted in a gain of 0.8 lives and a cost reduction of $78,601 per case. For the treatment of CNS and DIS disease, antiviral therapy saved more lives but at increased cost, with respective marginal costs per additional life saved of $75,125 and $46,619. From a managed care perspective, antiviral therapy is more cost-effective than from a societal viewpoint because costs of institutional care and special education are not the responsibility of managed care organizations. Development of at-home therapies will further improve the cost-effectiveness of antiviral therapy for neonatal herpes simplex virus infection.  (+info)

Intravenous penciclovir for treatment of herpes simplex infections in immunocompromised patients: results of a multicenter, acyclovir-controlled trial. The Penciclovir Immunocompromised Study Group. (6/1121)

The efficacy and safety of penciclovir (PCV) for the treatment of herpes simplex virus (HSV) infections in immunocompromised (IC) patients were studied in a double-blind, acyclovir (ACV)-controlled, multicenter study. A total of 342 patients with mucocutaneous HSV infections received 5 mg of PCV per kg every 12 or 8 h (q12h or q8h) or 5 mg of ACV per kg q8h, beginning within 72 h of lesion onset and continuing for up to 7 days. The mean age of the patients was 49 years; 94% were white and 52% were female. The main reasons for their IC states were hematologic disorder (63%) and transplant plus hematologic disorder (16%). Clinical and virological assessments were performed daily during the 7-day treatment and then every other day until lesion healing. The primary efficacy parameter addressed new lesion formation. Secondary end points focused on viral shedding, healing, and pain. Approximately 20% of patients in each treatment group developed new lesions during therapy; thus, equivalence with ACV (defined prospectively) was demonstrated for both q12h and q8h PCV regimens. For all three treatment groups, the median time to the cessation of viral shedding was 4 days and the median time to complete healing was 8 days; there were no statistically significant differences in the rates of complete healing or the cessation of viral shedding when the results for PCV q12h and q8h were compared with those for ACV q8h. In addition, there was no statistically significant difference between PCV q12h or q8h, compared with ACV q8h, for the resolution of pain. PCV was well tolerated, with an adverse event profile comparable to that of ACV. In conclusion, PCV q12h is a well-tolerated and effective therapy for mucocutaneous HSV infection in IC patients and offers a reduced frequency of dosing compared with ACV q8h.  (+info)

Valacyclovir for the prevention of cytomegalovirus disease after renal transplantation. International Valacyclovir Cytomegalovirus Prophylaxis Transplantation Study Group. (7/1121)

BACKGROUND: Cytomegalovirus (CMV) disease is a major complication of organ transplantation. We hypothesized that prophylactic treatment with valacyclovir would reduce the risk of CMV disease. METHODS: A total of 208 CMV-negative recipients of a kidney from a seropositive donor and 408 CMV-positive recipients were randomly assigned to receive either 2 g of valacyclovir or placebo orally four times daily for 90 days after transplantation, with the dose adjusted according to renal function. The primary end point was laboratory-confirmed CMV disease in the first six months after transplantation. RESULTS: Treatment with valacyclovir reduced the incidence or delayed the onset of CMV disease in both the seronegative patients (P<0.001) and the seropositive patients (P=0.03). Among the seronegative patients, the incidence of CMV disease 90 days after transplantation was 45 percent among placebo recipients and 3 percent among valacyclovir recipients. Among the seropositive patients, the respective values were 6 percent and 0 percent. At six months, the incidence of CMV disease was 45 percent among seronegative recipients of placebo and 16 percent among seronegative recipients of valacyclovir; it was 6 percent among seropositive placebo recipients and 1 percent among seropositive valacyclovir recipients. At six months, the rate of biopsy-confirmed acute graft rejection in the seronegative group was 52 percent among placebo recipients and 26 percent among valacyclovir recipients (P=0.001). Treatment with valacyclovir also decreased the rates of CMV viremia and viruria, herpes simplex virus disease, and the use of inpatient medical resources. Hallucinations and confusion were more common with valacyclovir treatment, but these events were not severe or treatment-limiting. The rates of other adverse events were similar among the groups. CONCLUSIONS: Prophylactic treatment with valacyclovir is a safe and effective way to prevent CMV disease after renal transplantation.  (+info)

Long term neurological outcome of herpes encephalitis. (8/1121)

Twenty eight children with herpes simplex encephalitis were followed up for a mean of 5.5 years. Two children died and 26 survived, of whom 16 were left with no neurological sequelae and 10 had persistent neurological sequelae. Mean (SD) Glasgow coma score was significantly lower in the patients with neurological sequelae (7.7 (1.5)) and the patients who died (4.5 (0.7)), compared with the patients without neurological sequelae (11 (1.7)).  (+info)

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AVIRAX Fabrigen Acyclovir Antiviral Action And Clinical Pharmacology: Acyclovir, a synthetic acyclic nucleoside analog, is a substrate with a high degree of specificity for herpes simplex and varicella-zoster specified thymidine kinase. Acyclovir is a poor substrate for host cell-specified thymidine kinase. Herpes simplex and varicella-zoster specified thymidine kinase transform acyclovir to its monophosphate which is then transformed by a number of cellular enzymes to acyclovir diphosphate and acyclovir triphosphate. Acyclovir triphosphate is both an inhibitor of, and a substrate for, herpesvirus-specified DNA polymerase. Although the cellular a-DNA polymerase in infected cells may also be inhibited by acyclovir triphosphate, this occurs only at concentrations of acyclovir triphosphate which are higher than those which inhibit the herpesvirus-specified DNA polymerase. Acyclovir is selectively converted to its active form in herpesvirus-infected cells and is thus preferentially taken up by these ...
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The efficacy of oral acyclovir 400 mg twice daily in suppressing frequently relapsing genital herpes simplex was evaluated in an open multicenter study. Seventy-one patients were treated for 12 months. During treatment, 73% of the patients were completely free of symptoms when taking the tablets continuously, and another 14% had mild symptoms such as erythema and/or itching at single occasions. An accidental treatment interruption for 2-4 days led to mild but definite herpes episodes within a few days in 5 otherwise symptom free patients. Definite herpes episodes despite acyclovir medication occurred in 3 cases (4%). No noteworthy side effects were recorded during the acyclovir treatment. After withdrawal of acyclovir, herpes relapsed within 1-4 weeks in 69% of the patients. The frequency of relapses during the following months was reported to be equal to that before the treatment period in most of the patients. Acyclovir susceptibility of the isolated herpes simplex virus (HSV) strains did not change
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Background and Objectives: Herpes simplex virus type 2 (HSV-2) is the primary cause of genital ulcer and one of the most prevalent sexually transmitted infections (STI) worldwide. HSV-2 has been recognized as a risk factor for HIV in multiple studies. A substantial shift in the aetiology of genital ulcer disease (GUD) towards genital herpes has been noted in many countries in Africa, especially those with mature HIV epidemics. Some countries guided by the predominance of HSV-2 as the aetiology of GUD in their country, are changing syndromic guidelines to include acyclovir as part of the treatment for GUD. Little data is available to support this decision in terms of its effect on clinical course and its cost-effectiveness. Yet, substantial investment would be needed in poor countries to add acyclovir to their essential drug list. Studies to determine the appropriateness of episodic acyclovir therapy for HSV-2 in the developing world are needed.. Episodic therapy with acyclovir both as a ...
You can buy the drug in varying doses: 200 mg, 400 mg, 800 mg. It is also used to prevent cytomegalovirus following a kidney transplant in high risk cases. Whether you suffer from genital herpes or cold sores Acyclovir 800 mg tablets will alleviate the symptoms associated with the herpes virus. Zovirax acyclovir tablets online. Bệnh nhân nhiễm HIV dùng 20 mg/kg (tối đa 800 mg mỗi liều) uống 4 lần một ngày trong 7-10 ngày. Famciclovir 500 mg po tid for 7 days and valacyclovir 1 g po tid for 7 days have better bioavailability with oral dosing than acyclovir, and therefore for herpes zoster, they are generally preferred to oral acyclovir 800 mg 5 times/day for 7 to 10 days. Typical dosage for prevention of recurrent herpes: 400 mg twice per day, every day for up to 12 months. The usual adult intravenous dose is 5-10 mg/kg every 8 hours for 7-10 days. Acyclovir cream apply 5 times per day for 4 days; Sitavig buccal one tablet per episode placed in upper gum on same side of mouth ...
Carcinogenesis, Mutagenesis, Impairment of Fertility: The data presented below include references to peak steady-state plasma acyclovir concentrations observed in humans treated with 800 mg given orally 5 times a day (dosing appropriate for treatment of herpes zoster) or 200 mg given orally 5 times a day (dosing appropriate for treatment of genital herpes). Plasma drug concentrations in animal studies are expressed as multiples of human exposure to acyclovir at the higher and lower dosing schedules (see CLINICAL PHARMACOLOGY: Pharmacokinetics). Acyclovir was tested in lifetime bioassays in rats and mice at single daily doses of up to 450 mg/kg administered by gavage. There was no statistically significant difference in the incidence of tumors between treated and control animals, nor did acyclovir shorten the latency of tumors. Maximum plasma concentrations were 3 to 6 times human levels in the mouse bioassay and 1 to 2 times human levels in the rat bioassay. Acyclovir was tested in 16 in vitro ...
Patients must have AIDS as defined by the CDC, or be patients with other immunocompromised states (transplant patients; other hematologic malignancies with recognized cell-mediated immune deficiency), and have herpes simplex virus (HSV) infection documented by culture and in vitro resistance to acyclovir by standard laboratory susceptibility or suspected resistance to acyclovir after an acyclovir treatment failure in which acyclovir was administered for at least 2 weeks intravenously at doses of 30 mg/kg/day (or a comparative dose adjusted for renal insufficiency). In vitro susceptibility data must be pending in the latter case.. ...
Acyclovir IV Infusion. A multicenter trial of acyclovir injection at a dose of 500 mg/m 2 every 8. dosing appropriate for treatment of herpes zoster or."Acyclovir"? is an antiviral drug that is used in most cases for the treatment of herpes infections. It is available in various dosage forms and may be administered.Overpaying For Valtrex? - With Blink - You Dont Have To Ad Buy Generic Valtrex Online. Pick Up At Any Pharmacy. Pay $21-With $5 Offer. Shingles (Herpes zoster.Cases reported • Herpes Zoster; Shingles;. despite initial high-dose intravenous therapy with acyclovir and in one case with additional VZV-immunoglobulin.Cream webmd dose herpes simplex zovirax tabletki 800 mg herpes during pregnancy. Is spelling oral acyclovir dosage for oral herpes preparations oral for cold ...
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Preferred Name: Valacyclovir Definition: The hydrochloride salt of the L-valyl ester of the antiviral drug acyclovir. Orally administered, valacyclovir is rapidly converted to acyclovir which inhibits viral DNA replication after further conversion to the nucleotide analog acyclovir triphosphate by viral thymidine kinase, cellular guanyl cyclase, and a number of other cellular enzymes. Acyclovir triphosphate competitively inhibits viral DNA polymerase; incorporates into and terminates the growing viral DNA chain; and inactivates viral DNA polymerase. The greater antiviral activity of acyclovir against herpes simplex virus (HSV) compared with varicella-zoster virus (VZV) is due to its more efficient phosphorylation by HSV thymidine kinase. NCI-GLOSS Definition: A substance that is being studied in the prevention of fungal, bacterial, and viral infections in patients undergoing donor stem cell transplantation with cells that are infected with cytomegalovirus. It belongs to the family of drugs ...
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Patients with renal impairment need Acyclovir dosage adjustments. Caution should also be exercised to patients receiving potentially nephrotoxic agents (this may increase the risk of renal dysfunction and/or the risk of reversible central nervous system symptoms such as those that have been reported in patients treated with intravenous acyclovir). Patients should consult with their physician if any adverse reactions occurs, also if they become pregnant or intend to become pregnant, they intend to breastfeed while taking orally administered Acyclovir, or they have any other questions.. Herpes Zoster: There are no data on treatment initiated more than 72 hours after onset of the zoster rash. Patients should be advised to initiate treatment as soon as possible after a diagnosis of herpes zoster.. Genital Herpes Infections: Acyclovir is not a cure for genital herpes ...
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Acyclovir is an antiviral drug. It slows the growth and spread of the herpes virus in the body. Acyclovir will not cure herpes, but it can lessen the symptoms of the infection. Acyclovir is used to treat infections caused by herpes viruses, such as genital herpes, cold sores, shingles, and chicken pox. Acyclovir may...
Acyclovir is an antiviral drug. It slows the growth and spread of the herpes virus in the body. Acyclovir will not cure herpes, but it can lessen the symptoms of the infection. Acyclovir is used to treat infections caused by herpes viruses, such as genital herpes, cold sores, shingles, and chicken pox. Acyclovir may...
Acyclovir is an antiviral drug. It slows the growth and spread of the herpes virus in the body. Acyclovir will not cure herpes, but it can lessen the symptoms of the infection. Acyclovir is used to treat infections caused by herpes viruses, such as genital herpes, cold sores, shingles, and chicken pox. Acyclovir may...
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Acyclovir (ACV) is the most commonly used drug for herpes simplex virus (HSV) infection therapy. Prolonged antiviral therapy or prophylaxis in immunocompromised patients may promote the development of drug-resistant strains. Due to the high polymorphism in genes involved in drug resistance, phenotypic methods, although work-intensive, are still required to test drug susceptibility. Real-time cell analysis (RTCA) based methods could offer a rapid and less labor-intensive alternative for phenotypic testing of ACV resistance ...
To the editor: We read the report of Wade and Meyers (1) with interest as we have also seen a case of transient neurologic deterioration associated with intravenous acyclovir.. A 56-year-old black woman was diagnosed as having hypereosinophilic syndrome in May 1982 when she presented with cough and dyspnea. Results of transbronchial and skin biopsies showed eosinophilic infiltrates. Prednisone and hydroxyurea were started. In June 1982, the patient had dysphagia. An esophagoscopic examination showed herpes simplex. On hospital day 8, intravenous acyclovir therapy was initiated at a dose of 250 mg/m2 body surface area every 8 hours. On the following ...
The sodium salt form of acyclovir, a synthetic analog of the purine nucleoside, guanosine, with potent antiviral activity against herpes simplex viruses type 1 and 2, varicella-zoster virus and other viruses. After conversion in vivo to the active metabolite acyclovir triphosphate, acyclovir competitively inhibits viral DNA polymerase, incorporates into and terminates the growing viral DNA chain, and inactivates viral DNA polymerase.
Acyclovir is a potent and selective antiviral agent. Unfortunately, drug-resistant (acyclovir-resistant) mutants have already been reported in herpes simplex virus type 1 (HSV-1) orofacial infections. We have developed a laboratory acyclovir-resistant HSV-1 mutant. The natural course of acyclovir-resistant HSV-1 keratitis was found to be less virulent than that observed in wild type HSV-1 keratitis, but the rate of ganglionic latency was as high as that induced by the parental strain. In vitro studies and in vivo observation of rabbit corneas infected with acyclovir-resistant HSV-1 both demonstrated a significant sensitivity to vidarabine and bromovinyldeoxyuridine ([E]-5-[2-bromovinyl]-2-deoxyuridine). The thymidine kinase activity of the acyclovir-resistant mutant was 69% of that of the wild type HSV-1 ...
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Such drugs include other aminoglycosides; the antiviral acyclovir; the antifungal amphotericin B; the antibiotics bacitracin, ...
Aciclovir. Antiretrovirals[edit]. Nucleoside/nucleotide reverse transcriptase inhibitors[edit]. *Abacavir (ABC). *Lamivudine ( ...
aciclovir trials in the late 1980s.[106] The lower toxicity and ease of administration over vidarabine has led to aciclovir ... Several antiviral drugs are effective for treating herpes, including aciclovir (acyclovir), valaciclovir, famciclovir, and ... aciclovir seems to inhibit antibody response, and newborns on aciclovir antiviral treatment experienced a slower rise in ... Aciclovir was the first discovered and is now available in generic.[61] Valaciclovir is also available as a generic[62] and is ...
Related antiviral drugs Aciclovir Valacyclovir, a prodrug form of aciclovir Famciclovir, an analogue of Penciclovir with ... It is taken orally once daily, in contrast to aciclovir, valaciclovir and other antivirals. A study has found that it is more ... 5246-8. ISBN 978-3-85200-181-4. "Brivudin (Zostex) besser als Aciclovir (Zovirax a.a.)?". Arznei-telegramm (in German). 5/2007 ... effective than aciclovir, but this has been disputed because of a possible conflict of interest on part of the study authors. ...
Acyclovir, steroids and therapeutic plasma exchange have been tried; one report suggests that the latter is more effective than ...
Systemic aciclovir facilitates the early control of HZO lesions. James, William D.; Berger, Timothy G.; et al. (2006). Andrews ... Treatment is usually with antivirals such as aciclovir orally. Topical steroids and cycloplegics for several months. ...
One review found that antivirals (such as aciclovir) are ineffective in improving recovery from Bell's palsy beyond steroids ... prednisone and acyclovir). Other research, however, identifies HSV-1 in only 31 cases (18 percent), herpes zoster (zoster sine ... "Early treatment with prednisolone or acyclovir in Bell's palsy". New England Journal of Medicine. 357 (16): 1598-607. doi ...
For herpes the treatment of choice is aciclovir.[23]. Surgical management is indicated where there is extremely increased ...
"Acyclovir". The American Society of Health-System Pharmacists. Archived from the original on 2015-01-05. Retrieved Jan 1, 2015. ... The antiviral drug acyclovir, often used in herpes treatment, and the anti-HIV drug abacavir, are structurally similar to ...
... is a prodrug, an esterified version of aciclovir that has greater oral bioavailability (about 55%) than aciclovir ... Aciclovir is selectively converted into a monophosphate form by viral thymidine kinase, which is far more effective (3000 times ... Aciclovir, the active metabolite of valaciclovir, is active against most species in the herpesvirus family. In descending order ... Common adverse drug reactions (≥1% of people) associated with valaciclovir are the same as for aciclovir, its active metabolite ...
S01AD03 Aciclovir. S01AD05 Interferon. S01AD06 Vidarabine. S01AD07 Famciclovir. S01AD08 Fomivirsen. S01AD09 Ganciclovir. S01AX ...
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... acyclovir, which was the first selective and effective drug of its kind).[25] Most of Elion's early work came from the use and ... acyclovir (ACV), used in the treatment of herpes infection.[3] ... Acyclovir (Zovirax), for viral herpes.[19]. *Nelarabine for ... "Developing the Purine Nucleoside Analogue Acyclovir: the Work of Gertrude B. Elion". J. Biol. Chem. 283 (19): e11. Retrieved ...
Multimedia: Aciclovir. *Identifikadoreak. *LCCN: sh88002652. *Hiztegiak eta entziklopediak. *Britannica: url. *Medikuntzako ...
acyclovir trials in the late 1980s.[101] The lower toxicity and ease of administration over vidarabine has led to acyclovir ... acyclovir seems to inhibit antibody response, and newborns on acyclovir antiviral treatment experienced a slower rise in ... Acyclovir was the first discovered and is now available in generic.[56] Valacyclovir is also available as a generic[57] and is ... "Acyclovir for treating primary herpetic gingivostomatitis". Cochrane Database Syst Rev (4): CD006700. doi:10.1002/14651858. ...
Intravenous aciclovir, can be used to prevent viral replication, and in the event of persistent lymphocytic pleocytosis, higher ... Since herpes infections often lead to pleocytosis, aciclovir and valacyclovir are commonly prescribed. When pleocytosis is the ... doses of aciclovir can also be taken. Studies have shown this treatment combined with valacyclovir to be effective in combating ...
acyclovir (Zovirax), for viral herpes. 1996 Stanley Norman Cohen (Co-recipient, Lemelson-MIT Prize) for the development of ...
Aciclovir is one of the oldest and most frequently prescribed antiviral drugs.[205] Other antiviral drugs in use target ... This is called DNA chain termination.[204] Examples of nucleoside analogues are aciclovir for Herpes simplex virus infections ... Mindel A, Sutherland S. Genital herpes - the disease and its treatment including intravenous acyclovir. Journal of ... The structure of the DNA base guanosine and the antiviral drug acyclovir ...
acyclovir trials in the late 1980s. The lower toxicity and ease of administration over vidarabine has led to acyclovir becoming ... However, acyclovir seems to inhibit antibody response, and newborns on acyclovir antiviral treatment experienced a slower rise ... Acyclovir was the first discovered and is now available in generic. Valacyclovir is also available as a generic and is slightly ... Acyclovir is the recommended antiviral for herpes suppressive therapy during the last months of pregnancy. The use of ...
Antiviral drugs such as acyclovir have been tried during the terminal phase of the disease. Cyclophosphamide and prednisone ...
Trifluridine Acyclovir Foscarnet Prusoff, W. H. (1959). "Synthesis and biological activities of iododeoxyuridine, an analog of ...
... has also shown efficacy in the treatment of aciclovir-resistant HSV infections. Cidofovir has also been investigated ... Chilukuri, S; Rosen, T (Apr 2003). "Management of acyclovir-resistant herpes simplex virus". Dermatologic clinics. 21 (2): 311- ... likely due to the abundance of safer alternatives such as aciclovir. Cidofovir shows anti-BK virus activity in a subgroup of ... aciclovir, aminosalicylic acid, etc.) are also withheld. Its active metabolite, cidofovir diphosphate, inhibits viral ...
Elion GB (1993). "Acyclovir: discovery, mechanism of action, and selectivity". J. Med. Virol. Suppl 1: 2-6. PMID 8245887. ... acyclovir for herpes virus infection, and nelarabine for cancer treatment. Since 2010, GlaxoSmithKline has several times ranked ...
December 1988). "Acyclovir treatment of the chronic fatigue syndrome. Lack of efficacy in a placebo-controlled trial". N. Engl ... A small 1988 RCT compared acyclovir against placebo and found that an equal proportion of patients improved from placebo and ... Nucleosidic class drugs such as acyclovir, valacyclovir and ganciclovir are inhibitors of viral replication during DNA (for DNA ... Three people withdrew from acyclovir treatment due to reversible renal failure. Antidepressants are often prescribed to CFS ...
The drug of choice is acyclovir.[4] Classification. Herpes simplex encephalitis may be classified according to origin of ... The drug of choice is acyclovir.[4] Supportive therapy for herpes simplex encephalitis includes breathing assistance, ...
Aciclovir. Famciclovir. Other. Comprehensive Review of Market Growth, Future Prospects, and Applications: ...
Valacyclovir, a prodrug of acyclovir, produces serum acyclovir levels that are three to five times as high as those achieved ... Oral acyclovir in the treatment of herpes zoster in general practice. N Z Med J 1989;102:93-95. [ISI][Medline] ... Acyclovir, valacyclovir, and famciclovir are approved for the treatment of herpes zoster. These drugs are well tolerated and ... Wood MJ, Johnson RW, McKendrick MW, Taylor J, Mandal BK, Crooks J. A randomized trial of acyclovir for 7 days and 21 days with ...
There was low quality evidence from 1 RCT that long-term use of oral aciclovir reduced clinical recurrences (1.80 versus 0.85 ... One RCT found significantly fewer research-diagnosed recurrences of HSL when on aciclovir cream treatment than on placebo (P , ... There was moderate quality evidence from 2 RCTs that topical aciclovir 5% cream probably has little effect on preventing ... use of oral aciclovir in preventing recurrent HSL was inconsistent across the doses used in the studies: 2 RCTs showed low ...
All patients were treated with intravenous acyclovir 10 mg/kg 3 times daily or oral valacyclovir 3 g/day for 7 to 10 days. ... A good recovery of HZO optic neuritis with systemic acyclovir and steroid has been also reported [18]. ...
The systemic component of this disorder is best treated with oral acyclovir, (Zovirax), 600 to 800mg five times a day for seven ... has been shown to be as effective in treating herpes zoster ophthalmicus as acyclovir 800mg fives times per day. Timing is ...
Acyclovir: learn about side effects, dosage, special precautions, and more on MedlinePlus ... Take or use acyclovir until you finish the prescription, even if you feel better. If you stop taking acyclovir too soon or skip ... Before taking acyclovir,. *tell your doctor and pharmacist if you are allergic to acyclovir, valacyclovir (Valtrex), any other ... If you become pregnant while taking acyclovir, call your doctor.. *if you are taking acyclovir to treat genital herpes, you ...
Acyclovir Topical: learn about side effects, dosage, special precautions, and more on MedlinePlus ... Acyclovir cream is usually applied five times a day for 4 days. Acyclovir cream may be applied at any time during a cold sore ... Acyclovir cream and ointment are for use only on the skin. Do not let acyclovir cream or ointment get into your eyes, or inside ... Acyclovir cream should only be applied to skin where a cold sore has formed or seems likely to form. Do not apply acyclovir ...
Congenital Abnormality Transmit Disease Advisory Committee Acyclovir Disease Treatment These keywords were added by machine and ...
Acyclovir definition, a crystalline compound, C18H11N5O3, used as an antiviral drug in the treatment of herpes infections. See ... acyclovir. 1980-85; perhaps acyclo(guanosine) an earlier name for the compound (a-6 + cyclo- + guanosine a nucleotide) + vir(us ...
The acyclovir oral tablet is used to treat viral infections such as shingles, genital herpes, and chickenpox. This form is ... Acyclovir comes in several prescription forms, including an oral tablet. ... Highlights for acyclovir. *Acyclovir oral tablet is available as both a generic and a brand-name drug. Brand name: Zovirax. ... How to take acyclovir. This dosage information is for acyclovir oral tablet. All possible dosages and drug forms may not be ...
... acyclovir, herpes - Answer: The first outbreak is usually the worst. It will take a few days before it ... ... How fast should Acyclovir.... How fast should Acyclovir work?. Asked. 1 Aug 2014 by helpp_30. Updated. 24 November 2018 (8 ... pain, acyclovir, herpes. Details:. I was told that I have herpes yesterday, but I feel like I waited too long to go to the dr, ... Acyclovir - So I got diagnosed with genital herpes I am to take 5 pills a day but Im confused?. Updated 3 Oct 2017 • 2 answers ...
Aciclovir-induced acute kidney injury. Medicina Clinica 154: 36, No. 1, 10 Jan 2020. Available from: URL: http://doi.org/ ... Aciclovir. Reactions Weekly 1790, 12 (2020). https://doi.org/10.1007/s40278-020-74631-y ...
Do not use Acyclovir Cream if you are:. •. allergic to Acyclovir Cream or any of the ingredients in Acyclovir Cream. See the ... Each gram of Acyclovir Cream 5% contains 50 mg acyclovir in an aqueous cream base. Acyclovir Cream is supplied as follows:. •. ... The pKas of acyclovir are 2.27 and 9.25.. Each gram of Acyclovir Cream 5% contains 50 mg of acyclovir and the following ... Systemic absorption of acyclovir from Acyclovir Cream is minimal in adults.. The systemic absorption of acyclovir following ...
Acyclovir capsule is a formulation for oral administration. Each capsule of acyclovir contains 200 mg of acyclovir and the ... Acyclovir pharmacokinetic parameters are summarized in Table 1.. Table 1. Acyclovir Pharmacokinetic Characteristics (Range) ... Acyclovir was positive in 5 of the assays.. Acyclovir did not impair fertility or reproduction in mice (450 mg/kg/day, p.o.) or ... The pkas of acyclovir are 2.27 and 9.25.. The chemical name of acyclovir is 2-amino-1,9-dihydro-9-[(2-hydroxyethoxy)methyl]-6H ...
Renal function after acyclovir intravenous injection.. Brigden D, Rosling AE, Woods NC. ... Plasma urea or creatinine was noted to be raised in 58 of 354 patients treated with intravenous acyclovir. This occurred after ... Animal studies indicate that the impairment of renal function associated with high bolus injections of acyclovir is due to ... intravenous bolus injection of high dosages of acyclovir but the risk was considerably reduced by slow intravenous infusion of ...
Acyclovir Suspension). Includes indications, proper use, special instructions, precautions, and possible side effects. ... Cold Sores acyclovir, Valtrex, valacyclovir, famciclovir, tetracaine topical, More.... Herpes Simplex acyclovir, Abreva, ... Herpes Simplex - Congenital acyclovir, More.... Herpes Simplex, Suppression acyclovir, Valtrex, valacyclovir, lysine, ... If you are taking Zovirax (acyclovir suspension) for an infection that came back, start Zovirax (acyclovir suspension) as soon ...
Acyclovir will not cure herpes, but it can lessen the symptoms of the infection. Acyclovir is used to treat infections caused ... Acyclovir is an antiviral drug. It slows the growth and spread of the herpes virus in the body. ... What is acyclovir?. Acyclovir is an antiviral drug. It slows the growth and spread of the herpes virus in the body. Acyclovir ... What other drugs will affect acyclovir?. Acyclovir can harm your kidneys. This effect is increased when you also use certain ...
Acyclovir for uncomplicated chickenpox?. BMJ 1993; 306 doi: https://doi.org/10.1136/bmj.306.6890.1478-c (Published 29 May 1993) ...
... acyclovir sodium injection, solution), including warnings and precautions, directions, and the names of other drugs and ...
Find treatment reviews for Acyclovir from other patients. Learn from their experiences about effectiveness, side effects and ... Showing 3 of 15 patient evaluations for Acyclovir Previous page 1 2 3 4 5 Next page ...
... Learn about the reported side effects, related class drugs, and ... ACYCLOVIR (ay SYE kloe veer) is an antiviral medicine. It is used to treat or prevent infections caused by certain kinds of ...
Acyclovir Oral capsule drug summary. Find medication information including related drug classes, side effects, patient ... ACYCLOVIR (ay SYE kloe veer) is an antiviral medicine. It is used to treat or prevent infections caused by certain kinds of ... an unusual or allergic reaction to acyclovir, ganciclovir, valacyclovir, other medicines, foods, dyes, or preservatives. - ...
Virasorb cold sore cream contains the active ingredient aciclovir, which is an antiviral medicine used to treat infections with ... Virasorb cold sore cream (aciclovir). Virasorb cold sore cream contains the active ingredient aciclovir, which is an antiviral ... Aciclovir is converted to a more active form inside cells of the body that are infected with herpes simplex. The activated ... This aciclovir cream is designed for the treatment of herpes simplex infections of the lips (cold sores). The cream should be ...
Aciclovir (ACV), also known as acyclovir, is an antiviral medication. It is primarily used for the treatment of herpes simplex ... In those with herpes of the eye, aciclovir may be more effective and safer than idoxuridine. It is not clear if aciclovir eye ... aciclovir may be used. For severe HSV infections (especially disseminated HSV), IV aciclovir may also be used. Studies in mice ... Aciclovir trials show that this agent has no role in preventing HIV transmission, but it can help slow HIV disease progression ...
  • This occurred after intravenous bolus injection of high dosages of acyclovir but the risk was considerably reduced by slow intravenous infusion of the same dosage over one hour, with adequate hydration of the patient and adjustment of dosage in patients with preexisting impaired renal function. (nih.gov)
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  • What is the dosage of Acyclovir And Hydrocortisone? (medindia.net)
  • However, if it is almost time for the acyclovir dosage ointment next dose, skip the ointment missed dose and continue your regular dosing schedule. (sportsrubbish.com)
  • This viral enzyme converts acyclovir into acyclovir monophosphate, a nucleotide analogue. (nih.gov)
  • In vitro, acyclovir triphosphate stops replication of herpes viral DNA. (nih.gov)
  • The greater antiviral activity of acyclovir against HSV compared to VZV is due to its more efficient phosphorylation by the viral TK. (nih.gov)
  • Resistance of HSV and VZV to acyclovir can result from qualitative and quantitative changes in the viral TK and/or DNA polymerase. (nih.gov)
  • While most of the acyclovir-resistant mutants isolated thus far from immunocompromised patients have been found to be TK-deficient mutants, other mutants involving the viral TK gene (TK partial and TK altered) and DNA polymerase have been isolated. (nih.gov)
  • The possibility of viral resistance to acyclovir should be considered in patients who show poor clinical response during therapy. (nih.gov)
  • The activated aciclovir works by blocking the action of a viral enzyme called DNA polymerase. (netdoctor.co.uk)
  • After conversion in vivo to the active metabolite acyclovir triphosphate by viral thymidine kinase, acyclovir competitively inhibits viral DNA polymerase by incorporating into the growing viral DNA chain and terminating further polymerization. (cancer.gov)
  • Acyclovir will not treat a viral infection such as the common cold or flu. (adventisthealthcare.com)
  • To achieve maximal benefit from antiviral therapy, acyclovir must be initiated before widespread viral dissemination throughout the body or to significant replication within the central nervous system (CNS). (aappublications.org)
  • Although clinically significant viral resistance associated with the use of acyclovir ointment 5 has not been observed, this possibility exists. (sportsrubbish.com)
  • As prednisone has a limited role in improving the recovery of incomplete Bell's palsy and medical literature postulates a viral etiology in Bell's palsy, you wonder whether acyclovir would improve the outcome for this patient. (bestbets.org)
  • It is not likely that other drugs you take orally or inject will have an effect on topically applied acyclovir. (everydayhealth.com)
  • Orally administered acyclovir in children less than 2 years of age has not yet been fully studied. (rxmed.com)
  • Some people use Acyclovir in the form of an ointment, and with this form of the drug, dizziness is less likely to be a problem, so drinking alcohol will have less of an effect than if the drug is taken orally. (accessrx.com)
  • Episodic (Intermittent) Therapy: Effective treatment requires therapy initiation acyclovir 400 mg within 1 day acyclovir of lesion onset or during the prodrome preceding an episode/recurrence 400 mg orally 3 times a day for 5 to 14 days. (silentbobspeaks.com)
  • Acyclovir belongs to a class of drugs called antivirals. (medicalnewstoday.com)
  • Clinical experience has identified no interactions resulting from topical or systemic administration of other drugs concomitantly with Acyclovir Cream. (nih.gov)
  • Aglaia Koutsodimou and Giovanni Natile, "Reaction of cis - and trans -Dichlorotetra(Dimethylsulfoxide)Ruthenium(II) With the Antiviral Drug Acyclovir," Metal-Based Drugs , vol. 7, no. 6, pp. 325-334, 2000. (hindawi.com)
  • NC NOT covered Drugs that acyclovir acyclovir are not covered by the plan. (sportsrubbish.com)
  • Since caffeine, alcohol, the nicotine from cigarettes, or street drugs can affect the action of many medications, you should let acyclovir your prescriber know if you use them. (sportsrubbish.com)
  • Los fármacos aprobados en Europa para el tratamiento sistémico del herpes zóster son aciclovir, valaciclovir. (cysers.info)
  • Valaciclovir er i sammenligning med aciclovir vist at have signifikant hurtigere smertelindrindende effekt på zosterrelaterede smerter og at være associeret med signifikant lavere forekomst af postherpetisk neuralgi. (medicin.dk)
  • Methods: We plan an individually randomized double blind placebo-control trial of the WHO and US CDC recommended dose of 3-times daily acyclovir for a 5-day treatment course. (clinicaltrials.gov)
  • In 1 of these studies, which included 16 inpatients, the complete ointment or its vehicle were randomly administered in a dose of 1-cm strips (25 mg acyclovir) 4 times a day for 7 days to an intact skin surface area of 4.5 square inches. (bioportfolio.com)
  • Coombs RW, Collier AC, Chaloupka K, Corey L. Decreased HIV plasma titer in response to combined low-dose zidovudine and acyclovir therapy in CDC class IVA patients. (clinicaltrials.gov)
  • On hospital day 8, intravenous acyclovir therapy was initiated at a dose of 250 mg/m 2 body surface area every 8 hours. (annals.org)
  • Idiopathic Facial Paralysis: A randomized, prospective and controlled study using single-dose prednisone versus acyclovir three times daily. (bestbets.org)
  • Viagra for sale in us How much does a prescription of phenergan cost torsemide cost buy acyclovir 5 cream medrol generic name womens viagra for sale uk. (tsrocks.com)
  • The global Acyclovir market is valued at xx million US$ in 2017 and will reach xx million US$ by the end of 2025, growing at a CAGR of xx% during 2018-2025. (qyresearchreports.com)
  • 2) Why don't doctors in the field suggest that people take Acyclovir to see if they can delay the starting of treatments that might have strong side effects? (thebody.com)
  • Read user comments about the side effects, benefits, and effectiveness of acyclovir sodium intravenous. (webmd.com)
  • It is created by eHealthMe generated on aciclovir tratamiento paras zoster of 42, semester who have side effects when. (cysers.info)
  • Aciclovir: Side Effects, Drug Information - Medical News Today! (google.com)
  • Other side effects of Acyclovir that should be reported to a physician right away include extreme fatigue, changes in heart rate, easy bruising or bleeding, dark or bloody urine, severe gastric pain, vision changes, seizures, or yellowing of the skin or the whites of the eyes. (accessrx.com)
  • The most common problem that can occur when a person taking Acyclovir drinks alcohol is an amplification of the dizziness and drowsiness that frequently occur as side effects of the drug. (accessrx.com)
  • People who drink while taking Acyclovir are more likely to experience side effects such as fainting or changes in vision. (accessrx.com)
  • Please note, like all medicine, Aciclovir tablets can cause side effects although this does not apply to everyone who has taken them. (chemistdirect.co.uk)
  • Acyclovir Cream is a herpes simplex virus (HSV) nucleoside analogue DNA polymerase inhibitor indicated for the treatment of recurrent herpes labialis (cold sores) in immunocompetent adults and adolescents 12 years of age and older. (nih.gov)
  • By blocking the action of DNA polymerase, aciclovir prevents the herpes virus from multiplying. (netdoctor.co.uk)
  • Aciclovir virker selektivt i herpesinficerede celler, da stoffet fosforyleres af den herpesvirusspecifikke thymidinkinase til aciclovirmonofosfat, der hæmmer den herpesvirusspecifikke DNA-polymerase, hvorved virusproduktionen ophører. (medicin.dk)
  • Reviews of research dating from the 1980s show there is some effect in reducing the number and duration of lesions if aciclovir is applied at an early stage of an outbreak. (wikipedia.org)
  • In both parts, the duration of virus shedding and the time to crusting and healing of lesions were shorter among acyclovir recipients than among placebo recipients. (nih.gov)
  • Two 5-ounce glasses of wine a day are generally thought to be safe for people taking Acyclovir. (accessrx.com)
  • What Happens When You Drink Alcohol While Taking Acyclovir? (accessrx.com)
  • Heat, tiredness, stress, and hunger tend to worsen dizziness when taking Acyclovir, and alcohol consumption can amplify the effect even more. (accessrx.com)
  • Taking Acyclovir , Words sort of fail me when I go to describe the character that is Marmaduke (it's unclear so far how he got his name. (unsoughtinput.com)
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  • A few reported cases of skin flushing, allergic inflammation, itching, stinging or burning sensations and skin drying has been associated with the use of Acyclovir. (coldsoreproof.com)
  • Consenting participants will be randomized to receive either acyclovir plus syndromic management or placebo plus syndromic management. (clinicaltrials.gov)
  • therefore, 1 placebo subject and 6 acyclovir subjects are included in the analysis. (clinicaltrials.gov)
  • A large PRCT with a real placebo control group is needed to clarify the effectiveness of acyclovir in the treatment of Bell's palsy. (bestbets.org)