Acyclovir
Antiviral Agents
Agents used in the prophylaxis or therapy of VIRUS DISEASES. Some of the ways they may act include preventing viral replication by inhibiting viral DNA polymerase; binding to specific cell-surface receptors and inhibiting viral penetration or uncoating; inhibiting viral protein synthesis; or blocking late stages of virus assembly.
Herpes Simplex
A group of acute infections caused by herpes simplex virus type 1 or type 2 that is characterized by the development of one or more small fluid-filled vesicles with a raised erythematous base on the skin or mucous membrane. It occurs as a primary infection or recurs due to a reactivation of a latent infection. (Dorland, 27th ed.)
Herpes Genitalis
Herpes Zoster
An acute infectious, usually self-limited, disease believed to represent activation of latent varicella-zoster virus (HERPESVIRUS 3, HUMAN) in those who have been rendered partially immune after a previous attack of CHICKENPOX. It involves the SENSORY GANGLIA and their areas of innervation and is characterized by severe neuralgic pain along the distribution of the affected nerve and crops of clustered vesicles over the area. (From Dorland, 27th ed)
Herpes Labialis
Herpesvirus 2, Human
Simplexvirus
Valine
Foscarnet
Ganciclovir
Encephalitis, Herpes Simplex
An acute (or rarely chronic) inflammatory process of the brain caused by SIMPLEXVIRUS infections which may be fatal. The majority of infections are caused by human herpesvirus 1 (HERPESVIRUS 1, HUMAN) and less often by human herpesvirus 2 (HERPESVIRUS 2, HUMAN). Clinical manifestations include FEVER; HEADACHE; SEIZURES; HALLUCINATIONS; behavioral alterations; APHASIA; hemiparesis; and COMA. Pathologically, the condition is marked by a hemorrhagic necrosis involving the medial and inferior TEMPORAL LOBE and orbital regions of the FRONTAL LOBE. (From Adams et al., Principles of Neurology, 6th ed, pp751-4)
Ointments
Herpes Zoster Ophthalmicus
Prodrugs
Stomatitis, Herpetic
Herpesvirus 3, Human
Keratitis, Dendritic
Thymidine Kinase
Herpesvirus 1, Human
The type species of SIMPLEXVIRUS causing most forms of non-genital herpes simplex in humans. Primary infection occurs mainly in infants and young children and then the virus becomes latent in the dorsal root ganglion. It then is periodically reactivated throughout life causing mostly benign conditions.
Administration, Topical
The application of drug preparations to the surfaces of the body, especially the skin (ADMINISTRATION, CUTANEOUS) or mucous membranes. This method of treatment is used to avoid systemic side effects when high doses are required at a localized area or as an alternative systemic administration route, to avoid hepatic processing for example.
Chickenpox
A highly contagious infectious disease caused by the varicella-zoster virus (HERPESVIRUS 3, HUMAN). It usually affects children, is spread by direct contact or respiratory route via droplet nuclei, and is characterized by the appearance on the skin and mucous membranes of successive crops of typical pruritic vesicular lesions that are easily broken and become scabbed. Chickenpox is relatively benign in children, but may be complicated by pneumonia and encephalitis in adults. (From Dorland, 27th ed)
Virus Shedding
Viral Plaque Assay
Method for measuring viral infectivity and multiplication in CULTURED CELLS. Clear lysed areas or plaques develop as the VIRAL PARTICLES are released from the infected cells during incubation. With some VIRUSES, the cells are killed by a cytopathic effect; with others, the infected cells are not killed but can be detected by their hemadsorptive ability. Sometimes the plaque cells contain VIRAL ANTIGENS which can be measured by IMMUNOFLUORESCENCE.
Encephalitis, Varicella Zoster
Inflammation of brain tissue caused by infection with the varicella-zoster virus (HERPESVIRUS 3, HUMAN). This condition is associated with immunocompromised states, including the ACQUIRED IMMUNODEFICIENCY SYNDROME. Pathologically, the virus tends to induce a vasculopathy and infect oligodendrocytes and ependymal cells, leading to CEREBRAL INFARCTION, multifocal regions of demyelination, and periventricular necrosis. Manifestations of varicella encephalitis usually occur 5-7 days after onset of HERPES ZOSTER and include HEADACHE; VOMITING; lethargy; focal neurologic deficits; FEVER; and COMA. (From Joynt, Clinical Neurology, 1996, Ch 26, pp29-32; Hum Pathol 1996 Sep;27(9):927-38)
Bell Palsy
A syndrome characterized by the acute onset of unilateral FACIAL PARALYSIS which progresses over a 2-5 day period. Weakness of the orbicularis oculi muscle and resulting incomplete eye closure may be associated with corneal injury. Pain behind the ear often precedes the onset of paralysis. This condition may be associated with HERPESVIRUS 1, HUMAN infection of the facial nerve. (Adams et al., Principles of Neurology, 6th ed, p1376)
Phosphonoacetic Acid
Idoxuridine
Virus Activation
The mechanism by which latent viruses, such as genetically transmitted tumor viruses (PROVIRUSES) or PROPHAGES of lysogenic bacteria, are induced to replicate and then released as infectious viruses. It may be effected by various endogenous and exogenous stimuli, including B-cell LIPOPOLYSACCHARIDES, glucocorticoid hormones, halogenated pyrimidines, IONIZING RADIATION, ultraviolet light, and superinfecting viruses.
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Inosine Pranobex
Double-Blind Method
Vidarabine
A nucleoside antibiotic isolated from Streptomyces antibioticus. It has some antineoplastic properties and has broad spectrum activity against DNA viruses in cell cultures and significant antiviral activity against infections caused by a variety of viruses such as the herpes viruses, the VACCINIA VIRUS and varicella zoster virus.
Infectious complications in 126 patients treated with high-dose chemotherapy and autologous peripheral blood stem cell transplantation. (1/1121)
The effect of an extensive prophylactic antimicrobial regimen was prospectively assessed in 126 patients after high-dose chemotherapy and autologous PBSC. They received ciprofloxacin (500 mg/12 h), acyclovir (200 mg/6 h), and itraconazole (200 mg/12 h) orally until neutrophil recovery. Febrile patients received i.v. imipenem (500 mg/6 h) to which vancomycin and amikacin were added if fever persisted for 2-3 and 5 days, respectively. Amphotericin B lipid complex was further given on day 7 or 8 of fever. Median times for a neutrophil count of >0.5 x 10(9)/l and a platelet count of >20 x 10(9)/l were 9 and 11 days. Severe neutropenia (<0.1 x 10(9)/l) lasted for a median of 5 days in which 72% of febrile episodes and 50% of cases of bacteremia occurred. Gram-positive bacteria were isolated in 30 of 40 episodes of bacteremia, 25 of which were caused by Staphylococcus epidermidis. Clinical foci were the intravascular catheter in 35 cases, respiratory infection in 11, cellulitis in two, anal abscess in one, and neutropenic enterocolitis in one. The high incidence of febrile episodes (94%) and bacteremias (31%) may be due to the lack of efficacy of antimicrobial prophylaxis and the persistence of a 5-day period of severe neutropenia. (+info)B cell lymphoproliferative disorders following hematopoietic stem cell transplantation: risk factors, treatment and outcome. (2/1121)
Twenty-six cases of B cell lymphoproliferative disorder (BLPD) were identified among 2395 patients following hematopoietic stem cell transplants (HSCT) for which an overall incidence of BLPD was 1.2%. The true incidence was probably higher, since 9/26 of the diagnoses were made at autopsy. No BLPD was observed following autologous HSCT, so risk factor analyses were confined to the 1542 allogeneic HSCT. Factors assessed were HLA-mismatching (> or = 1 antigen), T cell depletion (TCD), presence of acute GvHD (grades II-IV), donor type (related vs unrelated), age of recipient and donor, and underlying disease. Factors found to be statistically significant included patients transplanted for immune deficiency and CML, donor age > or = 18 years, TCD, and HLA-mismatching, with recipients of combined TCD and HLA-mismatched grafts having the highest incidence. Factors found to be statistically significant in a multiple regression analysis were TCD, donor age and immune deficiency, although 7/8 of the patients with immunodeficiencies and BLPD received a TCD graft from a haploidentical parent. The overall mortality was 92% (24/26). One patient had a spontaneous remission, but subsequently died >1 year later of chronic GVHD. Thirteen patients received therapy for BLPD. Three patients received lymphocyte infusions without response. The only patients with responses and longterm survival received alpha interferon (alphaIFN). Of seven patients treated with alphaIFN there were four responses (one partial and three complete). These data demonstrate that alphaIFN can be an effective agent against BLPD following HSCT, if a timely diagnosis is made. (+info)Interactions of a nonpeptidic drug, valacyclovir, with the human intestinal peptide transporter (hPEPT1) expressed in a mammalian cell line. (3/1121)
The results of previous work performed in our laboratory using an in situ perfusion technique in rats and rabbit apical brush border membrane vesicles have suggested that the intestinal uptake of valacyclovir (VACV) appears to be mediated by multiple membrane transporters. Using these techniques, it is difficult to characterize the transport kinetics of VACV with each individual transporter in the presence of multiple known or unknown transporters. The purpose of this study was to characterize the interaction of VACV and the human intestinal peptide transporter using Chinese hamster ovary (CHO) cells that overexpress the human intestinal peptide transporter (hPEPT1) gene. VACV uptake was significantly greater in CHO cells transfected with hPEPT1 than in cells transfected with only the vector, pcDNA3. The optimum pH for VACV uptake was determined to occur at pH 7.5. Proton cotransport was not observed in hPEPT1/CHO cells, consistent with previously observed results in tissues and Caco-2 cells. VACV uptake was concentration dependent and saturable with a Michaelis-Menten constant and maximum velocity of 1.64 +/- 0.06 mM and 23.34 +/- 0.36 nmol/mg protein/5 min, respectively. A very similar Km value was obtained in hPEPT1/CHO cells and in rat and rabbit tissues and Caco-2 cells, suggesting that hPEPT1 dominates the intestinal transport properties of VACV in vitro. VACV uptake was markedly inhibited by various dipeptides and beta-lactam antibiotics, and Ki values of 12.8 +/- 2.7 and 9.1 +/- 1.2 mM were obtained for Gly-Sar and cefadroxil at pH 7.5, respectively. The present results demonstrate that VACV is a substrate for the human intestinal peptide transporter in hPEPT1/CHO cells and that although transport is pH dependent, proton cotransport is not apparent. Also, the results demonstrate that the hPEPT1/CHO cell system has use in investigating the transport kinetics of drugs with the human intestinal peptide transporter hPEPT1; however, the extrapolation of these transport properties to the in vivo situation requires further investigation. (+info)Management of human cytomegalovirus infection and disease after allogeneic bone marrow transplantation. (4/1121)
BACKGROUND AND OBJECTIVE: Human cytomegalovirus (HCMV) infection and disease remain a major cause of morbidity and mortality after bone marrow transplantation. HCMV disease, especially pneumonitis, may be treated with ganciclovir and immunoglobulin but even so the outcome is poor with mortality rates of 30-70%. It is therefore imperative to treat HCMV infection before it develops into disease. The aim of this article is to describe the main strategies used to prevent HCMV infection and to improve the survival after CMV disease in bone marrow transplant recipients. INFORMATION SOURCES: In the present review, we examined personal papers in this field and articles published in journals covered by the Science Citation Index and Medline. STATE OF THE ART: Major advances have been made in preventing HCMV infection and disease through two different approaches, both of which reduce HCMV induced morbidity and mortality: In pre-emptive therapy, patients are given ganciclovir when HCMV infection is first identified and this is continued 3-4 months after transplantation; in prophylactic therapy ganciclovir is given to all patients at risk of HCMV disease from engraftment up to 3-4 months post transplantation. Each strategy has advantages and disadvantages and there is no evidence for the superiority of one over the other since the overall survival is the same and the incidence of death from HCMV disease is similar. PERSPECTIVES: The use of more sensitive tests such as HCMV PCR or antigenemia may improve the outcome but probably will not eradicate all HCMV disease. Future possible strategies could include adoptive transfer of CD8+ HCMV-specific cytotoxic T lymphocytes clones derived from the donor marrow or boosting donor or patient immunity using subunit anti-HCMV vaccines such as gB or pp65. (+info)Antiviral therapy for neonatal herpes simplex virus: a cost-effectiveness analysis. (5/1121)
Each year, about 1,600 infants in the United States are infected with neonatal herpes simplex virus. We conducted a cost-effectiveness analysis of antiviral drug therapy (acyclovir) for three forms of herpes simplex virus infection: skin, ear, and mouth (SEM), central nervous system (CNS), and disseminated multiorgan (DIS) disease. Five levels of patient outcomes were examined (normal, mild, moderate, severe, dead). We obtained information on disease occurrence and survival from clinical trials and historical reviews of untreated newborns. We considered approaches for treating all or any of the forms of the disease and compared them with no use of antiviral drugs. The main measure of effectiveness was lives saved, including those of descendants of survivors. Costs were measured from a societal perspective and included direct medical costs, institutional care, and special education. We used a discount rate of 3% and valued dollars at 1995 levels. We also considered the perspective of a managed care organization. From a societal viewpoint relative to no treatment, antiviral therapy for SEM resulted in a gain of 0.8 lives and a cost reduction of $78,601 per case. For the treatment of CNS and DIS disease, antiviral therapy saved more lives but at increased cost, with respective marginal costs per additional life saved of $75,125 and $46,619. From a managed care perspective, antiviral therapy is more cost-effective than from a societal viewpoint because costs of institutional care and special education are not the responsibility of managed care organizations. Development of at-home therapies will further improve the cost-effectiveness of antiviral therapy for neonatal herpes simplex virus infection. (+info)Intravenous penciclovir for treatment of herpes simplex infections in immunocompromised patients: results of a multicenter, acyclovir-controlled trial. The Penciclovir Immunocompromised Study Group. (6/1121)
The efficacy and safety of penciclovir (PCV) for the treatment of herpes simplex virus (HSV) infections in immunocompromised (IC) patients were studied in a double-blind, acyclovir (ACV)-controlled, multicenter study. A total of 342 patients with mucocutaneous HSV infections received 5 mg of PCV per kg every 12 or 8 h (q12h or q8h) or 5 mg of ACV per kg q8h, beginning within 72 h of lesion onset and continuing for up to 7 days. The mean age of the patients was 49 years; 94% were white and 52% were female. The main reasons for their IC states were hematologic disorder (63%) and transplant plus hematologic disorder (16%). Clinical and virological assessments were performed daily during the 7-day treatment and then every other day until lesion healing. The primary efficacy parameter addressed new lesion formation. Secondary end points focused on viral shedding, healing, and pain. Approximately 20% of patients in each treatment group developed new lesions during therapy; thus, equivalence with ACV (defined prospectively) was demonstrated for both q12h and q8h PCV regimens. For all three treatment groups, the median time to the cessation of viral shedding was 4 days and the median time to complete healing was 8 days; there were no statistically significant differences in the rates of complete healing or the cessation of viral shedding when the results for PCV q12h and q8h were compared with those for ACV q8h. In addition, there was no statistically significant difference between PCV q12h or q8h, compared with ACV q8h, for the resolution of pain. PCV was well tolerated, with an adverse event profile comparable to that of ACV. In conclusion, PCV q12h is a well-tolerated and effective therapy for mucocutaneous HSV infection in IC patients and offers a reduced frequency of dosing compared with ACV q8h. (+info)Valacyclovir for the prevention of cytomegalovirus disease after renal transplantation. International Valacyclovir Cytomegalovirus Prophylaxis Transplantation Study Group. (7/1121)
BACKGROUND: Cytomegalovirus (CMV) disease is a major complication of organ transplantation. We hypothesized that prophylactic treatment with valacyclovir would reduce the risk of CMV disease. METHODS: A total of 208 CMV-negative recipients of a kidney from a seropositive donor and 408 CMV-positive recipients were randomly assigned to receive either 2 g of valacyclovir or placebo orally four times daily for 90 days after transplantation, with the dose adjusted according to renal function. The primary end point was laboratory-confirmed CMV disease in the first six months after transplantation. RESULTS: Treatment with valacyclovir reduced the incidence or delayed the onset of CMV disease in both the seronegative patients (P<0.001) and the seropositive patients (P=0.03). Among the seronegative patients, the incidence of CMV disease 90 days after transplantation was 45 percent among placebo recipients and 3 percent among valacyclovir recipients. Among the seropositive patients, the respective values were 6 percent and 0 percent. At six months, the incidence of CMV disease was 45 percent among seronegative recipients of placebo and 16 percent among seronegative recipients of valacyclovir; it was 6 percent among seropositive placebo recipients and 1 percent among seropositive valacyclovir recipients. At six months, the rate of biopsy-confirmed acute graft rejection in the seronegative group was 52 percent among placebo recipients and 26 percent among valacyclovir recipients (P=0.001). Treatment with valacyclovir also decreased the rates of CMV viremia and viruria, herpes simplex virus disease, and the use of inpatient medical resources. Hallucinations and confusion were more common with valacyclovir treatment, but these events were not severe or treatment-limiting. The rates of other adverse events were similar among the groups. CONCLUSIONS: Prophylactic treatment with valacyclovir is a safe and effective way to prevent CMV disease after renal transplantation. (+info)Long term neurological outcome of herpes encephalitis. (8/1121)
Twenty eight children with herpes simplex encephalitis were followed up for a mean of 5.5 years. Two children died and 26 survived, of whom 16 were left with no neurological sequelae and 10 had persistent neurological sequelae. Mean (SD) Glasgow coma score was significantly lower in the patients with neurological sequelae (7.7 (1.5)) and the patients who died (4.5 (0.7)), compared with the patients without neurological sequelae (11 (1.7)). (+info)
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Aciclovir
... (ACV), also known as acyclovir, is an antiviral medication. It is primarily used for the treatment of herpes simplex ... Aciclovir is a nucleoside analogue that mimics guanosine. It works by decreasing the production of the virus's DNA. Aciclovir ... In those with herpes of the eye, aciclovir may be more effective and safer than idoxuridine. It is not clear if aciclovir eye ... aciclovir may be used. For severe HSV infections (especially disseminated HSV), IV aciclovir may also be used. Studies in mice ...
Neuritis
Antiviral medications, including acyclovir, are effective at controlling viral reactivation. Management of ensuing neuropathic ... Herpes reactivation is often treated with acyclovir, although evidence for its efficacy in controlling peripheral neurological ... Taylor M, Gerriets V (2020). Acyclovir. StatPearls. StatPearls Publishing. PMID 31194337. Johnson RW, Alvarez-Pasquin MJ, Bijl ...
List of antiviral drugs
"Acyclovir". The American Society of Health-System Pharmacists. Archived from the original on 2015-01-05. Retrieved Jan 1, 2015 ...
Prototype drug
"Acyclovir". DrugBank. 16 October 2018. Retrieved 16 October 2018. "Aspirin - MeSH". NCBI. 16 October 2018. Retrieved 16 October ... is the prototype dihydropyridine calcium channel blocker Chloroquine is the prototypical antimalarial agent Acyclovir is the ...
Amikacin
Such drugs include other aminoglycosides; the antiviral acyclovir; the antifungal amphotericin B; the antibiotics bacitracin, ...
Gertrude B. Elion
Acyclovir (Zovirax), for viral herpes. Nelarabine for cancer treatment. Elion GB; Hitchins GH; Vanderwerff H (1951). " ... acyclovir, which was the first selective and effective drug of its kind). Most of Elion's early work came from the use and ... acyclovir (ACV), used in the treatment of herpes infection. Elion was born in New York City on January 23, 1918, to parents ... "Developing the Purine Nucleoside Analogue Acyclovir: the Work of Gertrude B. Elion". J. Biol. Chem. 283 (19): e11. doi:10.1016/ ...
Herpetic gingivostomatitis
Nasser M, Fedorowicz Z, Khoshnevisan MH, Shahiri Tabarestani M (October 2008). Nasser M (ed.). "Acyclovir for treating primary ... Antiviral drugs are used to treat herpetic gingivostomatitis such as aciclovir, valaciclovir, famciclovir, and in resistance ... Goldman RD (May 2016). "Acyclovir for herpetic gingivostomatitis in children". Canadian Family Physician. 62 (5): 403-4. PMC ...
Herpes simplex
aciclovir trials in the late 1980s. The lower toxicity and ease of administration over vidarabine has led to aciclovir becoming ... However, aciclovir seems to inhibit antibody response, and newborns on aciclovir antiviral treatment experienced a slower rise ... Several antiviral drugs are effective for treating herpes, including aciclovir (acyclovir), valaciclovir, famciclovir, and ... Aciclovir was the first discovered and is now available in generic. Valaciclovir is also available as a generic and is slightly ...
Lemelson-MIT Prize
acyclovir (Zovirax), for herpes simplex virus infection. 1996 Stanley Norman Cohen (Co-recipient, Lemelson-MIT Prize) for the ...
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Trifluridine Acyclovir Foscarnet Prusoff, W. H. (1959). "Synthesis and biological activities of iododeoxyuridine, an analog of ...
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... has also shown efficacy in the treatment of aciclovir-resistant HSV infections. Cidofovir has also been investigated ... Chilukuri S, Rosen T (April 2003). "Management of acyclovir-resistant herpes simplex virus". Dermatologic Clinics. 21 (2): 311- ... likely due to the abundance of safer alternatives such as aciclovir. Cidofovir shows anti-BK virus activity in a subgroup of ... aciclovir, aminosalicylic acid, etc.) are also withheld. Its active metabolite, cidofovir diphosphate, inhibits viral ...
Herpes labialis
Antivirals creams include acyclovir and penciclovir, which can speed healing by as much as 10%. Oral antivirals include ... Treatment with oral antivirals such as acyclovir in children within 72 hours of illness onset has shown to shorten duration of ... therefore use of zinc-based sunscreen or topical and oral therapeutics such as acyclovir and valacyclovir may prove helpful. ... acyclovir, valaciclovir, and famciclovir. Famciclovir or valacyclovir, taken in pill form, can be effective using a single day ...
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Elion GB (1993). "Acyclovir: discovery, mechanism of action, and selectivity". J. Med. Virol. Suppl 1: 2-6. doi:10.1002/jmv. ... acyclovir for herpes virus infection, and nelarabine for cancer treatment. Since 2010, GlaxoSmithKline has several times ranked ...
Chronic fatigue syndrome treatment
December 1988). "Acyclovir treatment of the chronic fatigue syndrome. Lack of efficacy in a placebo-controlled trial". The New ... A small 1988 RCT compared acyclovir against placebo and found that an equal proportion of patients improved from placebo and ... Nucleosidic class drugs such as acyclovir, valacyclovir and ganciclovir are inhibitors of viral replication during DNA (for DNA ... Three people withdrew from acyclovir treatment due to reversible renal failure. Antidepressants are often prescribed to CFS ...
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Jabs DA (1998). "Acyclovir for recurrent herpes simplex virus ocular disease". N Engl J Med. 339 (5): 340-341. doi:10.1056/ ...
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Roy S, Sukla S, De A, Biswas S (January 2022). "Non-cytopathic herpes simplex virus type-1 isolated from acyclovir-treated ... Studies of vulnerable patient populations have indicated that daily use of antivirals such as aciclovir and valaciclovir can ... Hussin A, Md Nor NS, Ibrahim N (November 2013). "Phenotypic and genotypic characterization of induced acyclovir-resistant ... "Oral acyclovir suppression and neurodevelopment after neonatal herpes". N. Engl. J. Med. 365 (14): 1284-92. doi:10.1056/ ...
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There is little evidence to support the use of antivirals such as aciclovir and valacyclovir although they may reduce initial ... Torre D, Tambini R; Tambini (1999). "Acyclovir for treatment of infectious mononucleosis: a meta-analysis". Scand. J. Infect. ...
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Treatment is generally with antiviral pills such as acyclovir. Steroid eye drops and drops to dilate the pupil may also be used ... Treatment is usually with antivirals such as acyclovir, valacyclovir, or famcyclovir by mouth. There is uncertainty as to the ... "Valacyclovir versus acyclovir for the treatment of herpes zoster ophthalmicus in immunocompetent patients". The Cochrane ...
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... intravenous and oral formulations of acyclovir in classic studies performed between 1980 and 1984. Acyclovir was the first ... Intravenous acyclovir for the treatment of primary genital herpes. Ann Intern Med. 1983 Jun;98(6):914-21. Coombs RW, Collier AC ... Intravenous acyclovir for the treatment of primary genital herpes. Ann Intern Med. 1983;98:914-21. Douglas JM, Critchlow C, ... Acyclovir and its derivatives valacyclovir and famciclovir are the most prescribed antivirals in the world: over 400 million ...
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Treatment with acyclovir was successful, indicating that a herpes virus was the cause of his symptoms. Acyclovir is the ... The IHMF recommends that patients with benign recurrent lymphocytic meningitis receive intravenous acyclovir in the amount of ...
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Diezel, W.; Michel, G.; Görtelmeyer, R.; Ostheimer, K. E. (April 1993). "Efficacy of tromantadine and aciclovir in the topical ... Its performance is similar to aciclovir. Like rimantadine, amantadine, and adapromine, tromantadine is a derivative of ... "Randomized double-blind trial of tromantadine versus aciclovir in recurrent herpes orofacialis". Arzneimittel-Forschung. 39 (9 ...
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Chickenpox
If aciclovir by mouth is started within 24 hours of rash onset, it decreases symptoms by one day but has no effect on ... Use of aciclovir therefore is not currently recommended for individuals with normal immune function. Children younger than 12 ... There was speculation in 2005 that continuous dosing of aciclovir by mouth for a period of time could eradicate VZV from the ... Treatment with antiviral drugs (e.g. aciclovir or valaciclovir) is generally advised, as long as it is started within 24-48 ...
Virus
Aciclovir is one of the oldest and most frequently prescribed antiviral drugs. Other antiviral drugs in use target different ... Examples of nucleoside analogues are aciclovir for Herpes simplex virus infections and lamivudine for HIV and hepatitis B virus ... Mindel A, Sutherland S (September 1983). "Genital herpes - the disease and its treatment including intravenous acyclovir". The ...
Pediatric Trials Network
January 2014). "Population pharmacokinetics of intravenous acyclovir in preterm and term infants". The Pediatric Infectious ...
Herpes esophagitis
Antivirals such as acyclovir, famciclovir, or valacyclovir may be used. Intravenous acyclovir is reserved for individuals who ... Antiviral prophylaxis such as daily acyclovir in immunocompromised individuals may be considered. ...
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Ribavirin
Another study found that ribavirin potentiated the antiviral effect of acyclovir. Some interest has been seen in its possible ... Pancheva SN (September 1991). "Potentiating effect of ribavirin on the anti-herpes activity of acyclovir". Antiviral Research. ...
Acyclovir: MedlinePlus Drug Information
Acyclovir: learn about side effects, dosage, special precautions, and more on MedlinePlus ... Take or use acyclovir until you finish the prescription, even if you feel better. If you stop taking acyclovir too soon or skip ... Before taking acyclovir,. *tell your doctor and pharmacist if you are allergic to acyclovir, valacyclovir (Valtrex), any other ... If you become pregnant while taking acyclovir, call your doctor.. *if you are taking acyclovir to treat genital herpes, you ...
Prolonged Oral Acyclovir Improves Neurodevelopment of Infants With CNS HSV
Neonates with CNS herpes simplex virus had better neurodevelopment at 1 year if they received 6 months of oral acyclovir after ... All patients received intravenous acyclovir. Once parenteral acyclovir treatment was completed, the infants were randomly ... He said any baby who develops neonatal CNS or SEM herpes disease "should be treated with intravenous acyclovir...regardless of ... Once theyve completed that course of [intravenous] acyclovir therapy, I believe that our data would strongly support that they ...
acyclovir (CHEBI:2453)
Pregnancy Outcomes Following Systemic Prenatal Acyclovir Exposure -
- June 1, 1984-June 30, 1993
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acyclovir, Zovirax: Drug Facts, Side Effects and Dosing
Explains the medication acyclovir (Zovirax), a drug used to treat initial genital herpes, shingles, and chickenpox infections ... may increase acyclovir serum levels by decreasing renal excretion of acyclovir. Acyclovir may increase serum levels of ... The acyclovir is converted to an active form by the virus itself, and the virus then uses the active form of acyclovir rather ... There are no adequate studies of acyclovir in pregnant women. In a patient registry of women who used acyclovir during the ...
Aciclovir - Oxford Reference
A nucleoside analogue (hydroxyethoxymethylguanine) with antiviral properties against both type 1 and 2 herpes virus and varicella-zoster virus. The drug is converted to the monophosphate by viral thymidine kinase and to the triphosphate by cellular enzymes. The triphosphate competitively inhibits viral DNA polymerase and can be incorporated into viral DNA where it blocks further replication. TN Zovirax.
Acyclovir - HealthLibrary
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Zovirax (Acyclovir): Uses, Dosage, Side Effects, Interactions, Warning
Acyclovir) may treat, side effects, dosage, drug interactions, warnings, patient labeling, reviews, and related medications ... Table 8 : Acyclovir Concentrations in a Teratology Study in Rats. Dose mg/kg b.i.d., s.c.. Plasma fag/mL). Acyclovir ... Table 9 : Acyclovir Concentrations in a Teratology Study in Rabbits. Dose mg/kg b.i.d., s.c.. Plasma fag/mL). Acyclovir ... Acyclovir was tested in 16 in vitro and in vivo genetic toxicity assays. Acyclovir was positive in 5 of the assays. ...
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Drug Information on acyclovir (injection) includes side effects, uses, drug interactions, dosage, drug pictures, overdose ... Acyclovir is an antiviral drug. It slows the growth and spread of the herpes virus in the body. Acyclovir will not cure herpes ... You may need to use acyclovir injection for up to 21 days. Use this medicine for the full prescribed length of time, even if ... Acyclovir can harm your kidneys, especially if you also use certain medicines for infections, cancer, osteoporosis, organ ...
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- Acyclovir is an antiviral drug, a synthetic nucleoside analogue, that has inhibitory activity (interferes with viral replication) against the herpes viruses , including herpes simplex 1 and 2 ( cold sores and genital herpes ), varicella -zoster ( shingles and chickenpox ), and Epstein-Barr virus ( mononucleosis ). (medicinenet.com)
- Acyclovir (Zovirax) is an antiviral drug prescribed to treat genital herpes, shingles, and chickenpox. (medicinenet.com)
- Acyclovir injection is used to treat severe infections caused by herpes viruses , including severe forms of genital herpes , shingles , herpes encephalitis (swelling of the brain), and herpes infections in people with other diseases that weaken the immune system. (emedicinehealth.com)
- Patients with frequently recurring genital herpes were enrolled in a double-blind placebo-controlled trial comparing 200-mg acyclovir capsules, given five or two times daily, with placebo. (nih.gov)
- We conclude that oral acyclovir given for four months markedly reduces but does not completely prevent recurrences of genital herpes and does not influence the long-term natural history of the disease. (nih.gov)
- Parenteral aciclovir has been shown to be effective in herpes simplex virus infections such as encephalitis, 1 primary genital herpes, 2 and herpes neonatorum. (bmj.com)
- 3 Oral aciclovir has been used successfully to treat genital herpes 4 5 and recurrent herpes labialis. (bmj.com)
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Nucleoside6
- Acyclovir is in a class of antiviral medications called synthetic nucleoside analogues. (medlineplus.gov)
- Acyclovir is a synthetic nucleoside analogue active against herpes viruses. (nih.gov)
- Acyclovir is a synthetic purine nucleoside analogue with in vitro and in vivo inhibitory activity against herpes simplex virus types 1 (HSV-1), 2 (HSV-2), and varicella-zoster virus (VZV). (nih.gov)
- The acyclovir is converted to an active form by the virus itself, and the virus then uses the active form of acyclovir rather than the nucleoside it normally uses to manufacture DNA, a critical component of viral replication. (medicinenet.com)
- Aciclovir is a synthetic purine nucleoside analogue structurally related to guanine. (epharmacyke.com)
- Aciclovir is an antiviral drug, it is a synthetic ingredient with a similar molecular structure to purine nucleoside. (pcostracker.app)
Viral14
- This viral enzyme converts acyclovir into acyclovir monophosphate, a nucleotide analogue. (nih.gov)
- In vitro, acyclovir triphosphate stops replication of herpes viral DNA. (nih.gov)
- The greater antiviral activity of acyclovir against HSV compared to VZV is due to its more efficient phosphorylation by the viral TK. (nih.gov)
- Resistance of HSV and VZV to acyclovir can result from qualitative and quantitative changes in the viral TK and/or DNA polymerase. (nih.gov)
- While most of the acyclovir-resistant mutants isolated thus far from immunocompromised patients have been found to be TK-deficient mutants, other mutants involving the viral TK gene (TK partial and TK altered) and DNA polymerase have been isolated. (nih.gov)
- The possibility of viral resistance to acyclovir should be considered in patients who show poor clinical response during therapy. (nih.gov)
- Incorporation of active acyclovir into new viral DNA stops the production of the DNA. (medicinenet.com)
- Breakthrough recurrences in acyclovir recipients were of shorter duration and associated with a lower frequency of viral shedding than recurrences in placebo recipients. (nih.gov)
- Aciclovir AL is a medicinal product for the treatment of viral infections (antiviral medicine). (medicine-one.org)
- Aciclovir AL is not recommended for the treatment of herpes simplex, viral infections in neonates and severe herpes simplex infection in immunocompromised children. (medicine-one.org)
- Acyclovir/Zovirax is the Original aciclovir in Kenya is used mainly for the treatment of viral infections due to herpes simplex virus (types 1 and 2) and varicella-zoster virus (herpes zoster and chickenpox). (epharmacyke.com)
- Phosphorylation by either HSV-TK or VZV-TK converts acyclovir into acyclovir triphosphate, which is then incorporated into viral DNA, thereby blocking further DNA synthesis. (britannica.com)
- Aciclovir is used to treat viral infections such as cold sores, to stop the growth of Herpes simplex virus, Varicella zoster virus, etc. (pcostracker.app)
- Aciclovir is used to treat viral infections such as cold sores, to stop the growth of Herpes simplex virus, Varicella zoster virus (caused by chickenpox and shingles), Epstein Barr Virus (caused by glandular fever), and to a lesser extent Cytomegalovirus (CMV). (pcostracker.app)
Drugs4
- Which drugs or supplements interact with acyclovir? (medicinenet.com)
- Combining acyclovir with cidofovir ( Vistide ), amphotericin B ( Fungizone ) or other drugs that reduce kidney function may increase harmful effects on the kidney. (medicinenet.com)
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- [ 31 ] Valacyclovir (1000 mg 3 times a day) and famciclovir (500 mg 3 times a day) are newer antiviral drugs with higher oral bioavailability than acyclovir and can be dosed less often. (medscape.com)
Intravenous7
- All patients received intravenous acyclovir. (medscape.com)
- He said any baby who develops neonatal CNS or SEM herpes disease "should be treated with intravenous acyclovir. (medscape.com)
- Once they've completed that course of [intravenous] acyclovir therapy, I believe that our data would strongly support that they should then be started on 6 months of oral acyclovir suppressive therapy with periodic monitoring of the white blood cell count through just a regular routine blood test. (medscape.com)
- A prenatal exposure to acyclovir is defined as inadvertent or intentional use of oral or intravenous acyclovir at any time during pregnancy. (cdc.gov)
- Aciclovir/Herperax in Kenya is administered by intravenous infusion as the sodium salt over 1 hour. (epharmacyke.com)
- Even if the final diagnosis of HSE has not been established, intravenous (IV) acyclovir should be initiated immediately. (medscape.com)
- The patient was treated with intravenous (IV) acyclovir at 5 mg/kg followed by antiviral therapy containing 5% cidofovir administered as oral gel. (cdc.gov)
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Valacyclovir3
- tell your doctor and pharmacist if you are allergic to acyclovir, valacyclovir (Valtrex), any other medications, milk proteins, or any of the ingredients in acyclovir products. (medlineplus.gov)
- You should not use this medication if you are allergic to acyclovir or valacyclovir ( Valtrex ). (emedicinehealth.com)
- IMSEAR at SEARO: Valacyclovir-induced urticaria without acyclovir hypersensitivity. (who.int)
Varicella-zoste1
- Acyclovir is also the drug of choice for varicella-zoster virus (VZV) encephalitis, although ganciclovir is also considered an alternative option. (medscape.com)
Patients with impaired renal function1
- Increased fluid levels should be taken with the use of Aciclovir AL tablets, especially in patients with impaired renal function. (medicine-one.org)
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Oral acyclovir8
- November 3, 2010 (Vancouver, British Columbia) - Infants with neonatal herpes simplex virus (HSV) infection with central nervous system (CNS) involvement have better neurodevelopmental outcomes at 1 year if they are started on oral acyclovir suppressive therapy immediately after parenteral acyclovir than if oral therapy is delayed. (medscape.com)
- Therefore, the Collaborative Antiviral Study Group (CASG) of the National Institute of Allergy and Infectious Diseases assessed the efficacy of oral acyclovir suppressive therapy in modifying these outcomes. (medscape.com)
- Once parenteral acyclovir treatment was completed, the infants were randomly assigned to receive either placebo or oral acyclovir 300 mg/m 2 3 times a day for 6 months. (medscape.com)
- Infants on oral acyclovir stayed on blinded study medication 2 months longer before discontinuation because of recurrent skin lesions compared with infants receiving placebo ( P = .009). (medscape.com)
- Daily oral acyclovir was well tolerated. (nih.gov)
- Effect of oral acyclovir against primary and secondary viraemia in incubation period of varicella. (bmj.com)
- These data indicate that oral acyclovir more effectively inhibits replication of VZV in secondary viraemia than that of the primary viraemia. (bmj.com)
- Prophylaxis or modification of varicella by oral acyclovir after household exposure. (bmj.com)
Medication2
- Acyclovir may also be used for other purposes not listed in this medication guide. (emedicinehealth.com)
- The mean monthly recurrence rate during the medication period was 0.86 in placebo recipients, compared with 0.13 in patients treated with acyclovir five times daily and 0.14 in patients treated with acyclovir twice daily (P less than 0.001 for both groups compared with placebo). (nih.gov)
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Renal2
- Probenecid (Benemid) may increase acyclovir serum levels by decreasing renal excretion of acyclovir. (medicinenet.com)
- You should talk with your healthcare provider prior to taking Aciclovir if you have kidney disease, including kidney failure (renal failure), any allergies, including allergies to food, dyes, or preservatives. (pcostracker.app)
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Efficacy of oral1
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Infection8
- Acyclovir is also sometimes used to treat eczema herpeticum (a skin infection caused by the herpes virus) to treat and prevent herpes infections of the skin, eyes, nose, and mouth in patients with human immunodeficiency virus (HIV), and to treat oral hairy leukoplakia (condition that causes hairy white or gray-colored patches on the tongue or inside of the cheek). (medlineplus.gov)
- Acyclovir (Zovirax Registered *), an antiviral drug effective in the treatment of herpes simplex infection, was approved by the Food and Drug Administration (FDA) in 1984. (cdc.gov)
- Some physicians have reported intentional use of acyclovir during pregnancy for treatment of life-threatening herpes simplex infection. (cdc.gov)
- Clinical isolates of HSV and VZV with reduced susceptibility to acyclovir have been recovered from immunocompromised patients, especially with advanced HIV infection. (nih.gov)
- Acyclovir will not cure herpes, but it can lessen the symptoms of the infection. (emedicinehealth.com)
- The infection was confirmed by a fluorescent antibody to membrane antigen assay in 11 (85%) of 13 children who received acyclovir for the first seven days after exposure to VZV and in 11 (79%) of 14 who received acyclovir for the next seven days. (bmj.com)
- Aciclovir or acyclovir (USAN, former BAN) is a guanine analogue antiviral drug used most commonly for the treatment of herpes simplex virus infection. (loinc.org)
- In order to achieve good results, treatment with Aciclovir AL should begin immediately with the onset of the first symptoms of infection. (medicine-one.org)
Hypersensitivity1
- Aciclovir Injection is contraindicated for patients who develop hypersensitivity to Acitop or valaciclovir. (pcostracker.app)
Herpes zoster1
- For herpes zoster: 800 mg aciclovir AL 5 times a day (corresponding to 4000 mg aciclovir / day) every 4 hours during the day. (medicine-one.org)
Inhibitory activity2
- The inhibitory activity of acyclovir is highly selective due to its affinity for the enzyme thymidine kinase (TK) encoded by HSV and VZV. (nih.gov)
- Acyclovir has demonstrated inhibitory activity against both herpes simplex virus type 1 (HSV-1) and herpes simplex virus type 2 (HSV-2) and is taken up selectively by infected cells. (medscape.com)
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Sodium1
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Chickenpox1
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Injection7
- What are the possible side effects of acyclovir injection? (emedicinehealth.com)
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- What should I discuss with my healthcare provider before using acyclovir injection? (emedicinehealth.com)
- How should I use acyclovir injection? (emedicinehealth.com)
- You may need to use acyclovir injection for up to 21 days. (emedicinehealth.com)
- Store acyclovir injection at room temperature away from moisture and heat. (emedicinehealth.com)
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Allergic1
- if you are allergic to aciclovir or valaciclovir or to any of the other ingredients of the medicine (listed in section 6). (medicine-one.org)
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Placebo3
- Of 47 placebo recipients, 44 (94 per cent) had recurrences during the 120-day treatment period, compared with 13 (29 per cent) of 45 patients treated with acyclovir five times daily and 18 of 51 (35 per cent) treated with acyclovir twice daily (P less than 0.001 for each regimen compared with placebo). (nih.gov)
- The median time to the first clinical recurrence was 18 days in placebo recipients, compared with over 120 days in both acyclovir-treated groups (P less than 0.001 for both groups compared with placebo). (nih.gov)
- Aciclovir suspension 15 mg/kg five times a day for seven days, or placebo. (bmj.com)