Agents used in the prophylaxis or therapy of VIRUS DISEASES. Some of the ways they may act include preventing viral replication by inhibiting viral DNA polymerase; binding to specific cell-surface receptors and inhibiting viral penetration or uncoating; inhibiting viral protein synthesis; or blocking late stages of virus assembly.
A group of acute infections caused by herpes simplex virus type 1 or type 2 that is characterized by the development of one or more small fluid-filled vesicles with a raised erythematous base on the skin or mucous membrane. It occurs as a primary infection or recurs due to a reactivation of a latent infection. (Dorland, 27th ed.)
An acute infectious, usually self-limited, disease believed to represent activation of latent varicella-zoster virus (HERPESVIRUS 3, HUMAN) in those who have been rendered partially immune after a previous attack of CHICKENPOX. It involves the SENSORY GANGLIA and their areas of innervation and is characterized by severe neuralgic pain along the distribution of the affected nerve and crops of clustered vesicles over the area. (From Dorland, 27th ed)
Herpesvirus 2, Human
Encephalitis, Herpes Simplex
An acute (or rarely chronic) inflammatory process of the brain caused by SIMPLEXVIRUS infections which may be fatal. The majority of infections are caused by human herpesvirus 1 (HERPESVIRUS 1, HUMAN) and less often by human herpesvirus 2 (HERPESVIRUS 2, HUMAN). Clinical manifestations include FEVER; HEADACHE; SEIZURES; HALLUCINATIONS; behavioral alterations; APHASIA; hemiparesis; and COMA. Pathologically, the condition is marked by a hemorrhagic necrosis involving the medial and inferior TEMPORAL LOBE and orbital regions of the FRONTAL LOBE. (From Adams et al., Principles of Neurology, 6th ed, pp751-4)
Herpes Zoster Ophthalmicus
Herpesvirus 3, Human
Herpesvirus 1, Human
The type species of SIMPLEXVIRUS causing most forms of non-genital herpes simplex in humans. Primary infection occurs mainly in infants and young children and then the virus becomes latent in the dorsal root ganglion. It then is periodically reactivated throughout life causing mostly benign conditions.
The application of drug preparations to the surfaces of the body, especially the skin (ADMINISTRATION, CUTANEOUS) or mucous membranes. This method of treatment is used to avoid systemic side effects when high doses are required at a localized area or as an alternative systemic administration route, to avoid hepatic processing for example.
A highly contagious infectious disease caused by the varicella-zoster virus (HERPESVIRUS 3, HUMAN). It usually affects children, is spread by direct contact or respiratory route via droplet nuclei, and is characterized by the appearance on the skin and mucous membranes of successive crops of typical pruritic vesicular lesions that are easily broken and become scabbed. Chickenpox is relatively benign in children, but may be complicated by pneumonia and encephalitis in adults. (From Dorland, 27th ed)
Viral Plaque Assay
Method for measuring viral infectivity and multiplication in CULTURED CELLS. Clear lysed areas or plaques develop as the VIRAL PARTICLES are released from the infected cells during incubation. With some VIRUSES, the cells are killed by a cytopathic effect; with others, the infected cells are not killed but can be detected by their hemadsorptive ability. Sometimes the plaque cells contain VIRAL ANTIGENS which can be measured by IMMUNOFLUORESCENCE.
Encephalitis, Varicella Zoster
Inflammation of brain tissue caused by infection with the varicella-zoster virus (HERPESVIRUS 3, HUMAN). This condition is associated with immunocompromised states, including the ACQUIRED IMMUNODEFICIENCY SYNDROME. Pathologically, the virus tends to induce a vasculopathy and infect oligodendrocytes and ependymal cells, leading to CEREBRAL INFARCTION, multifocal regions of demyelination, and periventricular necrosis. Manifestations of varicella encephalitis usually occur 5-7 days after onset of HERPES ZOSTER and include HEADACHE; VOMITING; lethargy; focal neurologic deficits; FEVER; and COMA. (From Joynt, Clinical Neurology, 1996, Ch 26, pp29-32; Hum Pathol 1996 Sep;27(9):927-38)
A syndrome characterized by the acute onset of unilateral FACIAL PARALYSIS which progresses over a 2-5 day period. Weakness of the orbicularis oculi muscle and resulting incomplete eye closure may be associated with corneal injury. Pain behind the ear often precedes the onset of paralysis. This condition may be associated with HERPESVIRUS 1, HUMAN infection of the facial nerve. (Adams et al., Principles of Neurology, 6th ed, p1376)
The mechanism by which latent viruses, such as genetically transmitted tumor viruses (PROVIRUSES) or PROPHAGES of lysogenic bacteria, are induced to replicate and then released as infectious viruses. It may be effected by various endogenous and exogenous stimuli, including B-cell LIPOPOLYSACCHARIDES, glucocorticoid hormones, halogenated pyrimidines, IONIZING RADIATION, ultraviolet light, and superinfecting viruses.
A nucleoside antibiotic isolated from Streptomyces antibioticus. It has some antineoplastic properties and has broad spectrum activity against DNA viruses in cell cultures and significant antiviral activity against infections caused by a variety of viruses such as the herpes viruses, the VACCINIA VIRUS and varicella zoster virus.
Infectious complications in 126 patients treated with high-dose chemotherapy and autologous peripheral blood stem cell transplantation. (1/1121)The effect of an extensive prophylactic antimicrobial regimen was prospectively assessed in 126 patients after high-dose chemotherapy and autologous PBSC. They received ciprofloxacin (500 mg/12 h), acyclovir (200 mg/6 h), and itraconazole (200 mg/12 h) orally until neutrophil recovery. Febrile patients received i.v. imipenem (500 mg/6 h) to which vancomycin and amikacin were added if fever persisted for 2-3 and 5 days, respectively. Amphotericin B lipid complex was further given on day 7 or 8 of fever. Median times for a neutrophil count of >0.5 x 10(9)/l and a platelet count of >20 x 10(9)/l were 9 and 11 days. Severe neutropenia (<0.1 x 10(9)/l) lasted for a median of 5 days in which 72% of febrile episodes and 50% of cases of bacteremia occurred. Gram-positive bacteria were isolated in 30 of 40 episodes of bacteremia, 25 of which were caused by Staphylococcus epidermidis. Clinical foci were the intravascular catheter in 35 cases, respiratory infection in 11, cellulitis in two, anal abscess in one, and neutropenic enterocolitis in one. The high incidence of febrile episodes (94%) and bacteremias (31%) may be due to the lack of efficacy of antimicrobial prophylaxis and the persistence of a 5-day period of severe neutropenia. (+info)
B cell lymphoproliferative disorders following hematopoietic stem cell transplantation: risk factors, treatment and outcome. (2/1121)Twenty-six cases of B cell lymphoproliferative disorder (BLPD) were identified among 2395 patients following hematopoietic stem cell transplants (HSCT) for which an overall incidence of BLPD was 1.2%. The true incidence was probably higher, since 9/26 of the diagnoses were made at autopsy. No BLPD was observed following autologous HSCT, so risk factor analyses were confined to the 1542 allogeneic HSCT. Factors assessed were HLA-mismatching (> or = 1 antigen), T cell depletion (TCD), presence of acute GvHD (grades II-IV), donor type (related vs unrelated), age of recipient and donor, and underlying disease. Factors found to be statistically significant included patients transplanted for immune deficiency and CML, donor age > or = 18 years, TCD, and HLA-mismatching, with recipients of combined TCD and HLA-mismatched grafts having the highest incidence. Factors found to be statistically significant in a multiple regression analysis were TCD, donor age and immune deficiency, although 7/8 of the patients with immunodeficiencies and BLPD received a TCD graft from a haploidentical parent. The overall mortality was 92% (24/26). One patient had a spontaneous remission, but subsequently died >1 year later of chronic GVHD. Thirteen patients received therapy for BLPD. Three patients received lymphocyte infusions without response. The only patients with responses and longterm survival received alpha interferon (alphaIFN). Of seven patients treated with alphaIFN there were four responses (one partial and three complete). These data demonstrate that alphaIFN can be an effective agent against BLPD following HSCT, if a timely diagnosis is made. (+info)
Interactions of a nonpeptidic drug, valacyclovir, with the human intestinal peptide transporter (hPEPT1) expressed in a mammalian cell line. (3/1121)The results of previous work performed in our laboratory using an in situ perfusion technique in rats and rabbit apical brush border membrane vesicles have suggested that the intestinal uptake of valacyclovir (VACV) appears to be mediated by multiple membrane transporters. Using these techniques, it is difficult to characterize the transport kinetics of VACV with each individual transporter in the presence of multiple known or unknown transporters. The purpose of this study was to characterize the interaction of VACV and the human intestinal peptide transporter using Chinese hamster ovary (CHO) cells that overexpress the human intestinal peptide transporter (hPEPT1) gene. VACV uptake was significantly greater in CHO cells transfected with hPEPT1 than in cells transfected with only the vector, pcDNA3. The optimum pH for VACV uptake was determined to occur at pH 7.5. Proton cotransport was not observed in hPEPT1/CHO cells, consistent with previously observed results in tissues and Caco-2 cells. VACV uptake was concentration dependent and saturable with a Michaelis-Menten constant and maximum velocity of 1.64 +/- 0.06 mM and 23.34 +/- 0.36 nmol/mg protein/5 min, respectively. A very similar Km value was obtained in hPEPT1/CHO cells and in rat and rabbit tissues and Caco-2 cells, suggesting that hPEPT1 dominates the intestinal transport properties of VACV in vitro. VACV uptake was markedly inhibited by various dipeptides and beta-lactam antibiotics, and Ki values of 12.8 +/- 2.7 and 9.1 +/- 1.2 mM were obtained for Gly-Sar and cefadroxil at pH 7.5, respectively. The present results demonstrate that VACV is a substrate for the human intestinal peptide transporter in hPEPT1/CHO cells and that although transport is pH dependent, proton cotransport is not apparent. Also, the results demonstrate that the hPEPT1/CHO cell system has use in investigating the transport kinetics of drugs with the human intestinal peptide transporter hPEPT1; however, the extrapolation of these transport properties to the in vivo situation requires further investigation. (+info)
Management of human cytomegalovirus infection and disease after allogeneic bone marrow transplantation. (4/1121)BACKGROUND AND OBJECTIVE: Human cytomegalovirus (HCMV) infection and disease remain a major cause of morbidity and mortality after bone marrow transplantation. HCMV disease, especially pneumonitis, may be treated with ganciclovir and immunoglobulin but even so the outcome is poor with mortality rates of 30-70%. It is therefore imperative to treat HCMV infection before it develops into disease. The aim of this article is to describe the main strategies used to prevent HCMV infection and to improve the survival after CMV disease in bone marrow transplant recipients. INFORMATION SOURCES: In the present review, we examined personal papers in this field and articles published in journals covered by the Science Citation Index and Medline. STATE OF THE ART: Major advances have been made in preventing HCMV infection and disease through two different approaches, both of which reduce HCMV induced morbidity and mortality: In pre-emptive therapy, patients are given ganciclovir when HCMV infection is first identified and this is continued 3-4 months after transplantation; in prophylactic therapy ganciclovir is given to all patients at risk of HCMV disease from engraftment up to 3-4 months post transplantation. Each strategy has advantages and disadvantages and there is no evidence for the superiority of one over the other since the overall survival is the same and the incidence of death from HCMV disease is similar. PERSPECTIVES: The use of more sensitive tests such as HCMV PCR or antigenemia may improve the outcome but probably will not eradicate all HCMV disease. Future possible strategies could include adoptive transfer of CD8+ HCMV-specific cytotoxic T lymphocytes clones derived from the donor marrow or boosting donor or patient immunity using subunit anti-HCMV vaccines such as gB or pp65. (+info)
Antiviral therapy for neonatal herpes simplex virus: a cost-effectiveness analysis. (5/1121)Each year, about 1,600 infants in the United States are infected with neonatal herpes simplex virus. We conducted a cost-effectiveness analysis of antiviral drug therapy (acyclovir) for three forms of herpes simplex virus infection: skin, ear, and mouth (SEM), central nervous system (CNS), and disseminated multiorgan (DIS) disease. Five levels of patient outcomes were examined (normal, mild, moderate, severe, dead). We obtained information on disease occurrence and survival from clinical trials and historical reviews of untreated newborns. We considered approaches for treating all or any of the forms of the disease and compared them with no use of antiviral drugs. The main measure of effectiveness was lives saved, including those of descendants of survivors. Costs were measured from a societal perspective and included direct medical costs, institutional care, and special education. We used a discount rate of 3% and valued dollars at 1995 levels. We also considered the perspective of a managed care organization. From a societal viewpoint relative to no treatment, antiviral therapy for SEM resulted in a gain of 0.8 lives and a cost reduction of $78,601 per case. For the treatment of CNS and DIS disease, antiviral therapy saved more lives but at increased cost, with respective marginal costs per additional life saved of $75,125 and $46,619. From a managed care perspective, antiviral therapy is more cost-effective than from a societal viewpoint because costs of institutional care and special education are not the responsibility of managed care organizations. Development of at-home therapies will further improve the cost-effectiveness of antiviral therapy for neonatal herpes simplex virus infection. (+info)
Intravenous penciclovir for treatment of herpes simplex infections in immunocompromised patients: results of a multicenter, acyclovir-controlled trial. The Penciclovir Immunocompromised Study Group. (6/1121)The efficacy and safety of penciclovir (PCV) for the treatment of herpes simplex virus (HSV) infections in immunocompromised (IC) patients were studied in a double-blind, acyclovir (ACV)-controlled, multicenter study. A total of 342 patients with mucocutaneous HSV infections received 5 mg of PCV per kg every 12 or 8 h (q12h or q8h) or 5 mg of ACV per kg q8h, beginning within 72 h of lesion onset and continuing for up to 7 days. The mean age of the patients was 49 years; 94% were white and 52% were female. The main reasons for their IC states were hematologic disorder (63%) and transplant plus hematologic disorder (16%). Clinical and virological assessments were performed daily during the 7-day treatment and then every other day until lesion healing. The primary efficacy parameter addressed new lesion formation. Secondary end points focused on viral shedding, healing, and pain. Approximately 20% of patients in each treatment group developed new lesions during therapy; thus, equivalence with ACV (defined prospectively) was demonstrated for both q12h and q8h PCV regimens. For all three treatment groups, the median time to the cessation of viral shedding was 4 days and the median time to complete healing was 8 days; there were no statistically significant differences in the rates of complete healing or the cessation of viral shedding when the results for PCV q12h and q8h were compared with those for ACV q8h. In addition, there was no statistically significant difference between PCV q12h or q8h, compared with ACV q8h, for the resolution of pain. PCV was well tolerated, with an adverse event profile comparable to that of ACV. In conclusion, PCV q12h is a well-tolerated and effective therapy for mucocutaneous HSV infection in IC patients and offers a reduced frequency of dosing compared with ACV q8h. (+info)
Valacyclovir for the prevention of cytomegalovirus disease after renal transplantation. International Valacyclovir Cytomegalovirus Prophylaxis Transplantation Study Group. (7/1121)BACKGROUND: Cytomegalovirus (CMV) disease is a major complication of organ transplantation. We hypothesized that prophylactic treatment with valacyclovir would reduce the risk of CMV disease. METHODS: A total of 208 CMV-negative recipients of a kidney from a seropositive donor and 408 CMV-positive recipients were randomly assigned to receive either 2 g of valacyclovir or placebo orally four times daily for 90 days after transplantation, with the dose adjusted according to renal function. The primary end point was laboratory-confirmed CMV disease in the first six months after transplantation. RESULTS: Treatment with valacyclovir reduced the incidence or delayed the onset of CMV disease in both the seronegative patients (P<0.001) and the seropositive patients (P=0.03). Among the seronegative patients, the incidence of CMV disease 90 days after transplantation was 45 percent among placebo recipients and 3 percent among valacyclovir recipients. Among the seropositive patients, the respective values were 6 percent and 0 percent. At six months, the incidence of CMV disease was 45 percent among seronegative recipients of placebo and 16 percent among seronegative recipients of valacyclovir; it was 6 percent among seropositive placebo recipients and 1 percent among seropositive valacyclovir recipients. At six months, the rate of biopsy-confirmed acute graft rejection in the seronegative group was 52 percent among placebo recipients and 26 percent among valacyclovir recipients (P=0.001). Treatment with valacyclovir also decreased the rates of CMV viremia and viruria, herpes simplex virus disease, and the use of inpatient medical resources. Hallucinations and confusion were more common with valacyclovir treatment, but these events were not severe or treatment-limiting. The rates of other adverse events were similar among the groups. CONCLUSIONS: Prophylactic treatment with valacyclovir is a safe and effective way to prevent CMV disease after renal transplantation. (+info)
Long term neurological outcome of herpes encephalitis. (8/1121)Twenty eight children with herpes simplex encephalitis were followed up for a mean of 5.5 years. Two children died and 26 survived, of whom 16 were left with no neurological sequelae and 10 had persistent neurological sequelae. Mean (SD) Glasgow coma score was significantly lower in the patients with neurological sequelae (7.7 (1.5)) and the patients who died (4.5 (0.7)), compared with the patients without neurological sequelae (11 (1.7)). (+info)
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The data presented below include references to peak steady-state plasma acyclovir concentrations observed in humans treated with 800 mg given orally 5 times a day (dosing appropriate for treatment of herpes zoster) or 200 mg given orally 5 times a day (dosing appropriate for treatment of genital herpes). Plasma drug concentrations in animal studies are expressed as multiples of human exposure to acyclovir at the higher and lower dosing schedules (see CLINICAL PHARMACOLOGY: Pharmacokinetics).. Acyclovir was tested in lifetime bioassays in rats and mice at single daily doses of up to 450 mg/kg administered by gavage. There was no statistically significant difference in the incidence of tumors between treated and control animals, nor did acyclovir shorten the latency of tumors. Maximum plasma concentrations were 3 to 6 times human levels in the mouse bioassay and 1 to 2 times human levels in the rat bioassay.. Acyclovir was tested in 16 in vitro and in vivo genetic toxicity assays. Acyclovir was ...
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One year acyclovir suppression of frequently recurring genital herpes: a study of efficacy, safety, virus sensitivity and...
The efficacy of oral acyclovir 400 mg twice daily in suppressing frequently relapsing genital herpes simplex was evaluated in an open multicenter study. Seventy-one patients were treated for 12 months. During treatment, 73% of the patients were completely free of symptoms when taking the tablets continuously, and another 14% had mild symptoms such as erythema and/or itching at single occasions. An accidental treatment interruption for 2-4 days led to mild but definite herpes episodes within a few days in 5 otherwise symptom free patients. Definite herpes episodes despite acyclovir medication occurred in 3 cases (4%). No noteworthy side effects were recorded during the acyclovir treatment. After withdrawal of acyclovir, herpes relapsed within 1-4 weeks in 69% of the patients. The frequency of relapses during the following months was reported to be equal to that before the treatment period in most of the patients. Acyclovir susceptibility of the isolated herpes simplex virus (HSV) strains did not change
Acyclovir treatment of varicella in otherwise healthy adolescents. The Collaborative Acyclovir Varicella Study Group.
STUDY OBJECTIVE: To determine whether orally administered acyclovir is of therapeutic benefit for varicella in otherwise healthy adolescents, and to compare the severity of the disease in adolescents with that in younger children. DESIGN: Multicenter, randomized, placebo-controlled, double-blind trial. SETTING: Patients homes and university hospital clinics. PATIENTS: Sixty-eight adolescents between 13 and 18 years of age with varicella entered the study. Of the 62 adolescents with laboratory-confirmed varicella who were included in the final analysis, 31 received acyclovir and 31 received placebo. INTERVENTIONS: Placebo or an 800 mg acyclovir tablet was given orally four times daily for 5 days, beginning within 24 hours of onset of rash. MEASUREMENTS AND MAIN RESULTS: Acyclovir recipients had significant reductions in times to cessation of new lesion formation (p less than 0.001), maximum number of lesions (p = 0.019), and defervescence (p = 0.045). Mean constitutional illness score was ...
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Episodic Acyclovir Therapy for Genital Ulcers - Full Text View - ClinicalTrials.gov
Background and Objectives: Herpes simplex virus type 2 (HSV-2) is the primary cause of genital ulcer and one of the most prevalent sexually transmitted infections (STI) worldwide. HSV-2 has been recognized as a risk factor for HIV in multiple studies. A substantial shift in the aetiology of genital ulcer disease (GUD) towards genital herpes has been noted in many countries in Africa, especially those with mature HIV epidemics. Some countries guided by the predominance of HSV-2 as the aetiology of GUD in their country, are changing syndromic guidelines to include acyclovir as part of the treatment for GUD. Little data is available to support this decision in terms of its effect on clinical course and its cost-effectiveness. Yet, substantial investment would be needed in poor countries to add acyclovir to their essential drug list. Studies to determine the appropriateness of episodic acyclovir therapy for HSV-2 in the developing world are needed.. Episodic therapy with acyclovir both as a ...
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Carcinogenesis, Mutagenesis, Impairment of Fertility: The data presented below include references to peak steady-state plasma acyclovir concentrations observed in humans treated with 800 mg given orally 5 times a day (dosing appropriate for treatment of herpes zoster) or 200 mg given orally 5 times a day (dosing appropriate for treatment of genital herpes). Plasma drug concentrations in animal studies are expressed as multiples of human exposure to acyclovir at the higher and lower dosing schedules (see CLINICAL PHARMACOLOGY: Pharmacokinetics). Acyclovir was tested in lifetime bioassays in rats and mice at single daily doses of up to 450 mg/kg administered by gavage. There was no statistically significant difference in the incidence of tumors between treated and control animals, nor did acyclovir shorten the latency of tumors. Maximum plasma concentrations were 3 to 6 times human levels in the mouse bioassay and 1 to 2 times human levels in the rat bioassay. Acyclovir was tested in 16 in vitro ...
An Open, Multicenter Study of Foscarnet Treatment of Acyclovir-Resistant Herpes Simplex Virus in Patients With the Acquired...
Patients must have AIDS as defined by the CDC, or be patients with other immunocompromised states (transplant patients; other hematologic malignancies with recognized cell-mediated immune deficiency), and have herpes simplex virus (HSV) infection documented by culture and in vitro resistance to acyclovir by standard laboratory susceptibility or suspected resistance to acyclovir after an acyclovir treatment failure in which acyclovir was administered for at least 2 weeks intravenously at doses of 30 mg/kg/day (or a comparative dose adjusted for renal insufficiency). In vitro susceptibility data must be pending in the latter case.. ...
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Preferred Name: Valacyclovir Definition: The hydrochloride salt of the L-valyl ester of the antiviral drug acyclovir. Orally administered, valacyclovir is rapidly converted to acyclovir which inhibits viral DNA replication after further conversion to the nucleotide analog acyclovir triphosphate by viral thymidine kinase, cellular guanyl cyclase, and a number of other cellular enzymes. Acyclovir triphosphate competitively inhibits viral DNA polymerase; incorporates into and terminates the growing viral DNA chain; and inactivates viral DNA polymerase. The greater antiviral activity of acyclovir against herpes simplex virus (HSV) compared with varicella-zoster virus (VZV) is due to its more efficient phosphorylation by HSV thymidine kinase. NCI-GLOSS Definition: A substance that is being studied in the prevention of fungal, bacterial, and viral infections in patients undergoing donor stem cell transplantation with cells that are infected with cytomegalovirus. It belongs to the family of drugs ...
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A randomised, double-blind trial of valaciclovir prophylaxis for cytomegalovirus disease in patients with advanced human...
Cytomegalovirus (CMV) disease is a common complication of advanced human immunodeficiency virus (HIV) infection. Administration of oral valaciclovir, a valine ester of acyclovir, achieves sufficient plasma acyclovir levels to inhibit many clinical isolates. Acyclovir has been associated with enhanced survival in AIDS but not with CMV disease prevention. CMV-seropositive patients (1227) with CD4 cell counts ,100/mm3 were enrolled in a randomized, double-blind trial. Valaciclovir, 8 g/day, was compared with acyclovir, 3.2 or 0.8 g/day, for CMV prevention; all three arms were compared for survival. The confirmed CMV disease rate was 11.7% among valaciclovir recipients and 17.5% in the pooled acyclovir arms, a 33% reduction in risk. Time to confirmed CMV disease was significantly longer for the valaciclovir group (P = .03). A trend toward earlier mortality for valaciclovir recipients was seen (P = .06). Toxicity and earlier medication discontinuation were more common in this group. Valaciclovir ...
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Postexposure prophylaxis of varicella in family contact by oral acyclovir<...
TY - JOUR. T1 - Postexposure prophylaxis of varicella in family contact by oral acyclovir. AU - Asano, Y.. AU - Yoshikawa, T.. AU - Suga, S.. AU - Kobayashi, I.. AU - Nakashima, T.. AU - Yazaki, T.. AU - Ozaki, T.. AU - Yamada, A.. AU - Imanishi, J.. PY - 1993/1/1. Y1 - 1993/1/1. N2 - Objective. To determine whether varicella can be prevented by administration of oral acyclovir (ACV) during the incubation period of the disease. Subjects and methods. ACV (40 or 80 mg/kg daily in four divided doses) was given orally to 25 exposed infants and children for 7 days, starting 7 to 9 days after exposure from the index case in their families. Their clinical features were compared with those of 25 age-matched control subjects who had been exposed in their families but did not receive ACV. A fluorescent antibody to membrane antigen assay was used for determination of the antibody to varicella-zoster virus, and a nested polymerase chain reaction method was used for detection of viremia. Results. Among the ...
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Patients with renal impairment need Acyclovir dosage adjustments. Caution should also be exercised to patients receiving potentially nephrotoxic agents (this may increase the risk of renal dysfunction and/or the risk of reversible central nervous system symptoms such as those that have been reported in patients treated with intravenous acyclovir). Patients should consult with their physician if any adverse reactions occurs, also if they become pregnant or intend to become pregnant, they intend to breastfeed while taking orally administered Acyclovir, or they have any other questions.. Herpes Zoster: There are no data on treatment initiated more than 72 hours after onset of the zoster rash. Patients should be advised to initiate treatment as soon as possible after a diagnosis of herpes zoster.. Genital Herpes Infections: Acyclovir is not a cure for genital herpes ...
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Acyclovir is an antiviral drug. It slows the growth and spread of the herpes virus in the body. Acyclovir will not cure herpes, but it can lessen the symptoms of the infection. Acyclovir is used to treat infections caused by herpes viruses, such as genital herpes, cold sores, shingles, and chicken pox. Acyclovir may...
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Herpes Simplex Virus Encephalitis. Herpes Zoster (Shingles) Dose adjustment based on renal clearance and normal dosage regimen. Detailed Acyclovir dosage information for adults and children. Apply 50 mg (1 buccal tablet) as a single-dose to the upper gum region (canine fossa) Comments: Tablet should be applied within 1 hour after the onset of prodromal symptoms and before the appearance of any signs of herpes labialis lesions. -Tablet should.. Shake the oral dosing (liquid) well just before you measure a short. Measure liquid cherry with the dosing syringe or, or with a special education-measuring spoon or cherry cup. If you do not have a headache-measuring device, ask your pharmacist for one. To take the acyclovir resistant tablet (Sitavig). Keep the tablet. Poised Dosing. Dosage great: CAP: mg; TAB: mg, mg; SUSP: mg per 5 mL; INJ: composed. HSV tab acyclovir dose. [10 mg/kg IV q8h x21 days]: Info: use IBW in healthy pts. genital/mucocutaneous HSV, transmural pts. [1st tab acyclovir dose, ...
Phenotypic testing of patient herpes simplex virus type 1 and 2 isolates for acyclovir resistance by a novel method based on...
Acyclovir (ACV) is the most commonly used drug for herpes simplex virus (HSV) infection therapy. Prolonged antiviral therapy or prophylaxis in immunocompromised patients may promote the development of drug-resistant strains. Due to the high polymorphism in genes involved in drug resistance, phenotypic methods, although work-intensive, are still required to test drug susceptibility. Real-time cell analysis (RTCA) based methods could offer a rapid and less labor-intensive alternative for phenotypic testing of ACV resistance ...
Intravenous Acyclovir and Neurologic Effects | Annals of Internal Medicine | American College of Physicians
To the editor: We read the report of Wade and Meyers (1) with interest as we have also seen a case of transient neurologic deterioration associated with intravenous acyclovir.. A 56-year-old black woman was diagnosed as having hypereosinophilic syndrome in May 1982 when she presented with cough and dyspnea. Results of transbronchial and skin biopsies showed eosinophilic infiltrates. Prednisone and hydroxyurea were started. In June 1982, the patient had dysphagia. An esophagoscopic examination showed herpes simplex. On hospital day 8, intravenous acyclovir therapy was initiated at a dose of 250 mg/m2 body surface area every 8 hours. On the following ...
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The sodium salt form of acyclovir, a synthetic analog of the purine nucleoside, guanosine, with potent antiviral activity against herpes simplex viruses type 1 and 2, varicella-zoster virus and other viruses. After conversion in vivo to the active metabolite acyclovir triphosphate, acyclovir competitively inhibits viral DNA polymerase, incorporates into and terminates the growing viral DNA chain, and inactivates viral DNA polymerase.
Lirias: Herpes simplex acyclovir-resistant mutant in experimental keratouveitis
Acyclovir is a potent and selective antiviral agent. Unfortunately, drug-resistant (acyclovir-resistant) mutants have already been reported in herpes simplex virus type 1 (HSV-1) orofacial infections. We have developed a laboratory acyclovir-resistant HSV-1 mutant. The natural course of acyclovir-resistant HSV-1 keratitis was found to be less virulent than that observed in wild type HSV-1 keratitis, but the rate of ganglionic latency was as high as that induced by the parental strain. In vitro studies and in vivo observation of rabbit corneas infected with acyclovir-resistant HSV-1 both demonstrated a significant sensitivity to vidarabine and bromovinyldeoxyuridine ([E]-5-[2-bromovinyl]-2-deoxyuridine). The thymidine kinase activity of the acyclovir-resistant mutant was 69% of that of the wild type HSV-1 ...
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Lirias: Acyclovir-resistant herpes simplex encephalitis in a patient treated with anti-tumor necrosis factor-α monoclonal...
Herpes simplex virus is the most common cause of severe sporadic encephalitis. We report a case of herpes simplex type 1-encephalitis in a 50-year-old woman receiving anti-tumor necrosis factor-α monoclonal antibodies adalimumab. Although she was an acyclovir naïve patient, a mixed viral population (wild-type and acyclovir-resistant bearing a thymidine-kinase mutation) was identified in the cerebrospinal fluid. The virus in cerebrospinal fluid evolved and a second thymidine-kinase mutant virus emerged. Combined foscavir and acyclovir treatment resolved the herpes simplex encephalitis. To our knowledge, this is the first report of acyclovir-resistant herpes simplex encephalitis in a patient treated with adalimumab ...
Controlled Trial Comparing Vidarabine With Acyclovir In Neonatal Herpes Simplex Virus Infection
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5-Year Follow-Up on the Effect of Oral Acyclovir After Penetrating Keratoplasty for Herpetic Keratitis | IOVS | ARVO Journals
Introduction: : In October 2003 we published the results of a double-blind randomised trial on the effect of oral acyclovir after penetrating keratoplasty (PK)for herpetic eye disease (HED) (1). During the 2 years follow up, 3 herpes-related (PCR or culture proven) recurrences occurred after oral acyclovir 400 mg twice daily (ACY group) as opposed to 9 recurrences after placebo (PLAC) (p = 0.026, log-rank test). Purpose: : This study was performed to determine the 5 year follow-up of the same patient population. Methods: : All patients were followed up according to the national keratoplasty follow-up register (at 6, 12, 24, 36 and 60 months). Patient records were used to score HSV recurrences and rejection episodes on clinical grounds: dendritic HED or kerato-uveitis. Rejection episodes and anterior chamber reactions were excluded. Results: : : After the 6 months acyclovir prophylaxis until 5 years after the operation 8 recurrences occurred in the ACY group as opposed to 16 in the PLAC group ...
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Herpes simplex encephalitis (HSE) in children sometimes exacerbates after successful treatment; yet the frequency, etiology, and clinical features of exacerbation remain unclear. We report data for 27 children with HSE confirmed by polymerase chain reaction (PCR) analysis; all were successfully treated with acyclovir, but 7 (26%) had a relapse of encephalitic illness. In 2 of those 7, serial examination with a PCR assay showed that herpes simplex virus (HSV) DNA reappeared temporarily in the cerebrospinal fluid (CSF). For 5 of the 7 patients, a second course of acyclovir therapy was effective. Coxsackievirus A9 was isolated from CSF of 1 case patient during subsequent exacerbation. The total dose during initial acyclovir therapy was significantly lower in the relapse group than in the control group (P = .027). In conclusion, exacerbation of HSE in children may be more common than previously recognized. It is suggested that the replication of HSV or another viral pathogen caused a second ...
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Acyclovir downregulate TNF-alpha in human limbal fibroblasts infected in vitro with Fusarium solani isolated from patient with...
Purpose: Keratomycosis can cause damage with risk the loss of the eye. The treatment requires early and aggressive approaches based on the evolution of the lesion and the type of etiologic agent.1 In a first approach, we found the Acyclovir of inhibiting growth of Fusarium solani and Aspergillus fumigatus2 and significant decrease was ejected in MICs by amphotericin B and Natamicyn in presence of Acyclovir.3,br /, The aim of this work was to assess the expression of IL-6 and TNF-α from human limbal fibroblasts (HLF), in the presence of Fusarium solani, treated with Acyclovir, in an in vitro model.. Methods: Human limbal fibroblast from cadaveric donors were obtained and were infected with conidia of a Fusarium solani strain isolated from a patient with keratomycosis. Some were treated with Acyclovir. The expression of IL-6, TNF-α and β-actin by means of RT PCR and 2-ΔCt was evaluated.. Results: IL-6 is expressed at similar concentrations like HLF without Acyclovir, also, infection of HLF ...
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TY - JOUR. T1 - Effect of acyclovir on the radiation-induced micronuclei and cell death. AU - Jagetia, Ganesh Chandra. AU - Aruna, R.. PY - 2000/8/21. Y1 - 2000/8/21. N2 - Treatment of HeLa cells with 0.1 μM Acyclovir [9-(2-hydroxyethoxymethyl)guanine] (ACV) before exposure to 0, 0.25, 0.5, 1, 2 and 3 Gy of γ-radiation resulted in a dose-dependent decline in the growth kinetics and cell proliferation indices at 20, 30 and 40 h post-irradiation when compared with the PBS + irradiation group. These results were reflected in the cell survival, which declined in a dose-dependent manner and the surviving fraction of cells was significantly lower in ACV + irradiation group than that of PBS + irradiation group. The effect of ACV + 1 Gy irradiation was almost similar to PBS + 3 Gy irradiation suggesting an enhancement of the radiation effect by ACV pretreatment. The frequency of micronuclei increased in a dose-dependent manner at all the post-irradiation time periods in both PBS + irradiation and ACV ...
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Pediatrics. 2001 Aug;108(2):223-9. Comparative Study; Multicenter Study; Research Support, Non-U.S. Govt; Research Support, U.S. Govt, P.H.S.
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We previously showed that long-term oral acyclovir therapy could reduce the prevalence of recurrent uveitis in patients who are having their recurrent uveitis on the basis of herpes simplex virus. (Reference: Rodriguez A, Power WJ, Neves RA, Foster CS: 1994. Long-Term Systemic Acyclovir in the Management of Recurrent Herpes Simplex Keratouveitis. In:Advances in Ocular Immunology (R.B. Nussenblatt, S.M. Whitcup, R.R. Caspi and I. Gery, eds., pp 465-468, Elsevier Science B.V.) This study, however, was not randomized, placebo-controlled, double masked. And while we are convinced, on the basis of this experience that, just as in the case of recurrent herpes simplex keratitis, so too in recurrent herpes simplex uveitis long-term chronic oral prophylactic maintenance anti-viral therapy is safe and effective in reducing recurrences of inflammation, we feel obligated now to subject this belief to rigorous scientific testing through a randomized, double masked, placebo-controlled clinical trial. We have ...
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Acyclovir (Zovirax), for viral herpes. Nelarabine for cancer treatment. Elion GB; Hitchins GH; Vanderwerff H (1951). " ... acyclovir, which was the first selective and effective drug of its kind). Most of Elion's early work came from the use and ... acyclovir (ACV), used in the treatment of herpes infection. Elion was born in New York City on January 23, 1918, to parents ... "Developing the Purine Nucleoside Analogue Acyclovir: the Work of Gertrude B. Elion". J. Biol. Chem. 283 (19): e11. doi:10.1016/ ...
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Jabs DA (1998). "Acyclovir for recurrent herpes simplex virus ocular disease". N Engl J Med. 339 (5): 340-341. doi:10.1056/ ...
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Roy S, Sukla S, De A, Biswas S (January 2022). "Non-cytopathic herpes simplex virus type-1 isolated from acyclovir-treated ... Studies of vulnerable patient populations have indicated that daily use of antivirals such as aciclovir and valaciclovir can ... Hussin A, Md Nor NS, Ibrahim N (November 2013). "Phenotypic and genotypic characterization of induced acyclovir-resistant ... "Oral acyclovir suppression and neurodevelopment after neonatal herpes". N. Engl. J. Med. 365 (14): 1284-92. doi:10.1056/ ...
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There is little evidence to support the use of antivirals such as aciclovir and valacyclovir although they may reduce initial ... Torre D, Tambini R; Tambini (1999). "Acyclovir for treatment of infectious mononucleosis: a meta-analysis". Scand. J. Infect. ...
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... intravenous and oral formulations of acyclovir in classic studies performed between 1980 and 1984. Acyclovir was the first ... Intravenous acyclovir for the treatment of primary genital herpes. Ann Intern Med. 1983 Jun;98(6):914-21. Coombs RW, Collier AC ... Intravenous acyclovir for the treatment of primary genital herpes. Ann Intern Med. 1983;98:914-21. Douglas JM, Critchlow C, ... Acyclovir and its derivatives valacyclovir and famciclovir are the most prescribed antivirals in the world: over 400 million ...
Treatment with acyclovir was successful, indicating that a herpes virus was the cause of his symptoms. Acyclovir is the ... The IHMF recommends that patients with benign recurrent lymphocytic meningitis receive intravenous acyclovir in the amount of ...
Diezel, W.; Michel, G.; Görtelmeyer, R.; Ostheimer, K. E. (April 1993). "Efficacy of tromantadine and aciclovir in the topical ... Its performance is similar to aciclovir. Like rimantadine, amantadine, and adapromine, tromantadine is a derivative of ... "Randomized double-blind trial of tromantadine versus aciclovir in recurrent herpes orofacialis". Arzneimittel-Forschung. 39 (9 ...
This means that acyclovir cannot be used to cure the virus infection and infected birds will remain infected and become a ... Acyclovir is a synthetic acyclic purine nucleoside that has strong activity against certain members of the herpesvirus group. ... Acyclovir has decreased morbidity and mortality during multiple Pacheco's disease outbreaks. However, it cannot prevent the ... Smith, C. Gregory (1987). "Use of Acyclovir in an Outbreak of Pacheco's Parrot Disease". AAV Today. 1 (2): 55-56. doi:10.2307/ ...
If aciclovir by mouth is started within 24 hours of rash onset, it decreases symptoms by one day but has no effect on ... Use of aciclovir therefore is not currently recommended for individuals with normal immune function. Children younger than 12 ... There was speculation in 2005 that continuous dosing of aciclovir by mouth for a period of time could eradicate VZV from the ... Treatment with antiviral drugs (e.g. aciclovir or valaciclovir) is generally advised, as long as it is started within 24-48 ...
Aciclovir is one of the oldest and most frequently prescribed antiviral drugs. Other antiviral drugs in use target different ... Examples of nucleoside analogues are aciclovir for Herpes simplex virus infections and lamivudine for HIV and hepatitis B virus ... Mindel A, Sutherland S (September 1983). "Genital herpes - the disease and its treatment including intravenous acyclovir". The ...
Pediatric Trials Network
January 2014). "Population pharmacokinetics of intravenous acyclovir in preterm and term infants". The Pediatric Infectious ...
Antivirals such as acyclovir, famciclovir, or valacyclovir may be used. Intravenous acyclovir is reserved for individuals who ... Antiviral prophylaxis such as daily acyclovir in immunocompromised individuals may be considered. ...
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Another study found that ribavirin potentiated the antiviral effect of acyclovir. Some interest has been seen in its possible ... Pancheva SN (September 1991). "Potentiating effect of ribavirin on the anti-herpes activity of acyclovir". Antiviral Research. ...
Acyclovir: MedlinePlus Drug Information
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Prolonged Oral Acyclovir Improves Neurodevelopment of Infants With CNS HSV
Neonates with CNS herpes simplex virus had better neurodevelopment at 1 year if they received 6 months of oral acyclovir after ... All patients received intravenous acyclovir. Once parenteral acyclovir treatment was completed, the infants were randomly ... He said any baby who develops neonatal CNS or SEM herpes disease "should be treated with intravenous acyclovir...regardless of ... Once theyve completed that course of [intravenous] acyclovir therapy, I believe that our data would strongly support that they ...
Pregnancy Outcomes Following Systemic Prenatal Acyclovir Exposure - - June 1, 1984-June 30, 1993
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acyclovir, Zovirax: Drug Facts, Side Effects and Dosing
Explains the medication acyclovir (Zovirax), a drug used to treat initial genital herpes, shingles, and chickenpox infections ... may increase acyclovir serum levels by decreasing renal excretion of acyclovir. Acyclovir may increase serum levels of ... The acyclovir is converted to an active form by the virus itself, and the virus then uses the active form of acyclovir rather ... There are no adequate studies of acyclovir in pregnant women. In a patient registry of women who used acyclovir during the ...
Aciclovir - Oxford Reference
A nucleoside analogue (hydroxyethoxymethylguanine) with antiviral properties against both type 1 and 2 herpes virus and varicella-zoster virus. The drug is converted to the monophosphate by viral thymidine kinase and to the triphosphate by cellular enzymes. The triphosphate competitively inhibits viral DNA polymerase and can be incorporated into viral DNA where it blocks further replication. TN Zovirax.
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Zovirax (Acyclovir): Uses, Dosage, Side Effects, Interactions, Warning
Acyclovir) may treat, side effects, dosage, drug interactions, warnings, patient labeling, reviews, and related medications ... Table 8 : Acyclovir Concentrations in a Teratology Study in Rats. Dose mg/kg b.i.d., s.c.. Plasma fag/mL). Acyclovir ... Table 9 : Acyclovir Concentrations in a Teratology Study in Rabbits. Dose mg/kg b.i.d., s.c.. Plasma fag/mL). Acyclovir ... Acyclovir was tested in 16 in vitro and in vivo genetic toxicity assays. Acyclovir was positive in 5 of the assays. ...
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Drug Information on acyclovir (injection) includes side effects, uses, drug interactions, dosage, drug pictures, overdose ... Acyclovir is an antiviral drug. It slows the growth and spread of the herpes virus in the body. Acyclovir will not cure herpes ... You may need to use acyclovir injection for up to 21 days. Use this medicine for the full prescribed length of time, even if ... Acyclovir can harm your kidneys, especially if you also use certain medicines for infections, cancer, osteoporosis, organ ...
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- Acyclovir is an antiviral drug, a synthetic nucleoside analogue, that has inhibitory activity (interferes with viral replication) against the herpes viruses , including herpes simplex 1 and 2 ( cold sores and genital herpes ), varicella -zoster ( shingles and chickenpox ), and Epstein-Barr virus ( mononucleosis ). (medicinenet.com)
- Acyclovir (Zovirax) is an antiviral drug prescribed to treat genital herpes, shingles, and chickenpox. (medicinenet.com)
- Acyclovir injection is used to treat severe infections caused by herpes viruses , including severe forms of genital herpes , shingles , herpes encephalitis (swelling of the brain), and herpes infections in people with other diseases that weaken the immune system. (emedicinehealth.com)
- Patients with frequently recurring genital herpes were enrolled in a double-blind placebo-controlled trial comparing 200-mg acyclovir capsules, given five or two times daily, with placebo. (nih.gov)
- We conclude that oral acyclovir given for four months markedly reduces but does not completely prevent recurrences of genital herpes and does not influence the long-term natural history of the disease. (nih.gov)
- Parenteral aciclovir has been shown to be effective in herpes simplex virus infections such as encephalitis, 1 primary genital herpes, 2 and herpes neonatorum. (bmj.com)
- 3 Oral aciclovir has been used successfully to treat genital herpes 4 5 and recurrent herpes labialis. (bmj.com)
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- Acyclovir is in a class of antiviral medications called synthetic nucleoside analogues. (medlineplus.gov)
- Acyclovir is a synthetic nucleoside analogue active against herpes viruses. (nih.gov)
- Acyclovir is a synthetic purine nucleoside analogue with in vitro and in vivo inhibitory activity against herpes simplex virus types 1 (HSV-1), 2 (HSV-2), and varicella-zoster virus (VZV). (nih.gov)
- The acyclovir is converted to an active form by the virus itself, and the virus then uses the active form of acyclovir rather than the nucleoside it normally uses to manufacture DNA, a critical component of viral replication. (medicinenet.com)
- Aciclovir is a synthetic purine nucleoside analogue structurally related to guanine. (epharmacyke.com)
- Aciclovir is an antiviral drug, it is a synthetic ingredient with a similar molecular structure to purine nucleoside. (pcostracker.app)
- This viral enzyme converts acyclovir into acyclovir monophosphate, a nucleotide analogue. (nih.gov)
- In vitro, acyclovir triphosphate stops replication of herpes viral DNA. (nih.gov)
- The greater antiviral activity of acyclovir against HSV compared to VZV is due to its more efficient phosphorylation by the viral TK. (nih.gov)
- Resistance of HSV and VZV to acyclovir can result from qualitative and quantitative changes in the viral TK and/or DNA polymerase. (nih.gov)
- While most of the acyclovir-resistant mutants isolated thus far from immunocompromised patients have been found to be TK-deficient mutants, other mutants involving the viral TK gene (TK partial and TK altered) and DNA polymerase have been isolated. (nih.gov)
- The possibility of viral resistance to acyclovir should be considered in patients who show poor clinical response during therapy. (nih.gov)
- Incorporation of active acyclovir into new viral DNA stops the production of the DNA. (medicinenet.com)
- Breakthrough recurrences in acyclovir recipients were of shorter duration and associated with a lower frequency of viral shedding than recurrences in placebo recipients. (nih.gov)
- Aciclovir AL is a medicinal product for the treatment of viral infections (antiviral medicine). (medicine-one.org)
- Aciclovir AL is not recommended for the treatment of herpes simplex, viral infections in neonates and severe herpes simplex infection in immunocompromised children. (medicine-one.org)
- Acyclovir/Zovirax is the Original aciclovir in Kenya is used mainly for the treatment of viral infections due to herpes simplex virus (types 1 and 2) and varicella-zoster virus (herpes zoster and chickenpox). (epharmacyke.com)
- Phosphorylation by either HSV-TK or VZV-TK converts acyclovir into acyclovir triphosphate, which is then incorporated into viral DNA, thereby blocking further DNA synthesis. (britannica.com)
- Aciclovir is used to treat viral infections such as cold sores, to stop the growth of Herpes simplex virus, Varicella zoster virus, etc. (pcostracker.app)
- Aciclovir is used to treat viral infections such as cold sores, to stop the growth of Herpes simplex virus, Varicella zoster virus (caused by chickenpox and shingles), Epstein Barr Virus (caused by glandular fever), and to a lesser extent Cytomegalovirus (CMV). (pcostracker.app)
- Which drugs or supplements interact with acyclovir? (medicinenet.com)
- Combining acyclovir with cidofovir ( Vistide ), amphotericin B ( Fungizone ) or other drugs that reduce kidney function may increase harmful effects on the kidney. (medicinenet.com)
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- [ 31 ] Valacyclovir (1000 mg 3 times a day) and famciclovir (500 mg 3 times a day) are newer antiviral drugs with higher oral bioavailability than acyclovir and can be dosed less often. (medscape.com)
- All patients received intravenous acyclovir. (medscape.com)
- He said any baby who develops neonatal CNS or SEM herpes disease "should be treated with intravenous acyclovir. (medscape.com)
- Once they've completed that course of [intravenous] acyclovir therapy, I believe that our data would strongly support that they should then be started on 6 months of oral acyclovir suppressive therapy with periodic monitoring of the white blood cell count through just a regular routine blood test. (medscape.com)
- A prenatal exposure to acyclovir is defined as inadvertent or intentional use of oral or intravenous acyclovir at any time during pregnancy. (cdc.gov)
- Aciclovir/Herperax in Kenya is administered by intravenous infusion as the sodium salt over 1 hour. (epharmacyke.com)
- Even if the final diagnosis of HSE has not been established, intravenous (IV) acyclovir should be initiated immediately. (medscape.com)
- The patient was treated with intravenous (IV) acyclovir at 5 mg/kg followed by antiviral therapy containing 5% cidofovir administered as oral gel. (cdc.gov)
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- tell your doctor and pharmacist if you are allergic to acyclovir, valacyclovir (Valtrex), any other medications, milk proteins, or any of the ingredients in acyclovir products. (medlineplus.gov)
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- Acyclovir is also the drug of choice for varicella-zoster virus (VZV) encephalitis, although ganciclovir is also considered an alternative option. (medscape.com)
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- Therefore, the Collaborative Antiviral Study Group (CASG) of the National Institute of Allergy and Infectious Diseases assessed the efficacy of oral acyclovir suppressive therapy in modifying these outcomes. (medscape.com)
- Once parenteral acyclovir treatment was completed, the infants were randomly assigned to receive either placebo or oral acyclovir 300 mg/m 2 3 times a day for 6 months. (medscape.com)
- Infants on oral acyclovir stayed on blinded study medication 2 months longer before discontinuation because of recurrent skin lesions compared with infants receiving placebo ( P = .009). (medscape.com)
- Daily oral acyclovir was well tolerated. (nih.gov)
- Effect of oral acyclovir against primary and secondary viraemia in incubation period of varicella. (bmj.com)
- These data indicate that oral acyclovir more effectively inhibits replication of VZV in secondary viraemia than that of the primary viraemia. (bmj.com)
- Prophylaxis or modification of varicella by oral acyclovir after household exposure. (bmj.com)
- Acyclovir may also be used for other purposes not listed in this medication guide. (emedicinehealth.com)
- The mean monthly recurrence rate during the medication period was 0.86 in placebo recipients, compared with 0.13 in patients treated with acyclovir five times daily and 0.14 in patients treated with acyclovir twice daily (P less than 0.001 for both groups compared with placebo). (nih.gov)
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- Clinical isolates of HSV and VZV with reduced susceptibility to acyclovir have been recovered from immunocompromised patients, especially with advanced HIV infection. (nih.gov)
- Acyclovir will not cure herpes, but it can lessen the symptoms of the infection. (emedicinehealth.com)
- The infection was confirmed by a fluorescent antibody to membrane antigen assay in 11 (85%) of 13 children who received acyclovir for the first seven days after exposure to VZV and in 11 (79%) of 14 who received acyclovir for the next seven days. (bmj.com)
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