OrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPhenomena and ProcessesHealth Care
AcyclovirAntiviral AgentsHerpes SimplexHerpes GenitalisHerpes ZosterHerpes LabialisHerpesvirus 2, HumanSimplexvirusValineFoscarnetGanciclovirEncephalitis, Herpes SimplexOintmentsHerpes Zoster OphthalmicusProdrugsGuanineStomatitis, HerpeticHerpesvirus 3, HumanKeratitis, DendriticThymidine KinaseHerpesvirus 1, HumanAdministration, OralAdministration, TopicalChickenpoxVirus SheddingViral Plaque AssayEncephalitis, Varicella ZosterBell PalsyArabinofuranosyluracilSkin Diseases, ViralPhosphonoacetic AcidIdoxuridineVirus ActivationPharmaceutical VehiclesInosine PranobexDouble-Blind MethodRecurrenceVidarabineKeratitis, Herpetic
Acyclovir
A GUANOSINE analog that acts as an antimetabolite. Viruses are especially susceptible. Used especially against herpes.
Antiviral Agents
Agents used in the prophylaxis or therapy of VIRUS DISEASES. Some of the ways they may act include preventing viral replication by inhibiting viral DNA polymerase; binding to specific cell-surface receptors and inhibiting viral penetration or uncoating; inhibiting viral protein synthesis; or blocking late stages of virus assembly.
Herpes Simplex
A group of acute infections caused by herpes simplex virus type 1 or type 2 that is characterized by the development of one or more small fluid-filled vesicles with a raised erythematous base on the skin or mucous membrane. It occurs as a primary infection or recurs due to a reactivation of a latent infection. (Dorland, 27th ed.)
Herpes Genitalis
Infection of the genitals (GENITALIA) with HERPES SIMPLEX VIRUS in either the males or the females.
Herpes Zoster
An acute infectious, usually self-limited, disease believed to represent activation of latent varicella-zoster virus (HERPESVIRUS 3, HUMAN) in those who have been rendered partially immune after a previous attack of CHICKENPOX. It involves the SENSORY GANGLIA and their areas of innervation and is characterized by severe neuralgic pain along the distribution of the affected nerve and crops of clustered vesicles over the area. (From Dorland, 27th ed)
Herpes Labialis
Herpes simplex, caused by type 1 virus, primarily spread by oral secretions and usually occurring as a concomitant of fever. It may also develop in the absence of fever or prior illness. It commonly involves the facial region, especially the lips and the nares. (Dorland, 27th ed.)
Herpesvirus 2, Human
A species of SIMPLEXVIRUS associated with genital infections (HERPES GENITALIS). It is transmitted by sexual intercourse and close personal contact.
Simplexvirus
A genus of the family HERPESVIRIDAE, subfamily ALPHAHERPESVIRINAE, consisting of herpes simplex-like viruses. The type species is HERPESVIRUS 1, HUMAN.
Valine
A branched-chain essential amino acid that has stimulant activity. It promotes muscle growth and tissue repair. It is a precursor in the penicillin biosynthetic pathway.
Foscarnet
An antiviral agent used in the treatment of cytomegalovirus retinitis. Foscarnet also shows activity against human herpesviruses and HIV.
Ganciclovir
An ACYCLOVIR analog that is a potent inhibitor of the Herpesvirus family including cytomegalovirus. Ganciclovir is used to treat complications from AIDS-associated cytomegalovirus infections.
Encephalitis, Herpes Simplex
An acute (or rarely chronic) inflammatory process of the brain caused by SIMPLEXVIRUS infections which may be fatal. The majority of infections are caused by human herpesvirus 1 (HERPESVIRUS 1, HUMAN) and less often by human herpesvirus 2 (HERPESVIRUS 2, HUMAN). Clinical manifestations include FEVER; HEADACHE; SEIZURES; HALLUCINATIONS; behavioral alterations; APHASIA; hemiparesis; and COMA. Pathologically, the condition is marked by a hemorrhagic necrosis involving the medial and inferior TEMPORAL LOBE and orbital regions of the FRONTAL LOBE. (From Adams et al., Principles of Neurology, 6th ed, pp751-4)
Ointments
Semisolid preparations used topically for protective emollient effects or as a vehicle for local administration of medications. Ointment bases are various mixtures of fats, waxes, animal and plant oils and solid and liquid hydrocarbons.
Herpes Zoster Ophthalmicus
Virus infection of the Gasserian ganglion and its nerve branches characterized by pain and vesicular eruptions with much swelling. Ocular involvement is usually heralded by a vesicle on the tip of the nose. This area is innervated by the nasociliary nerve.
Prodrugs
A compound that, on administration, must undergo chemical conversion by metabolic processes before becoming the pharmacologically active drug for which it is a prodrug.
Guanine
Stomatitis, Herpetic
Stomatitis caused by Herpesvirus hominis. It usually occurs as acute herpetic stomatitis (or gingivostomatitis), an oral manifestation of primary herpes simplex seen primarily in children and adolescents.
Herpesvirus 3, Human
The type species of VARICELLOVIRUS causing CHICKENPOX (varicella) and HERPES ZOSTER (shingles) in humans.
Keratitis, Dendritic
A form of herpetic keratitis characterized by the formation of small vesicles which break down and coalesce to form recurring dendritic ulcers, characteristically irregular, linear, branching, and ending in knoblike extremities. (Dictionary of Visual Science, 3d ed)
Thymidine Kinase
An enzyme that catalyzes the conversion of ATP and thymidine to ADP and thymidine 5'-phosphate. Deoxyuridine can also act as an acceptor and dGTP as a donor. (From Enzyme Nomenclature, 1992) EC 2.7.1.21.
Herpesvirus 1, Human
The type species of SIMPLEXVIRUS causing most forms of non-genital herpes simplex in humans. Primary infection occurs mainly in infants and young children and then the virus becomes latent in the dorsal root ganglion. It then is periodically reactivated throughout life causing mostly benign conditions.
Administration, Oral
The giving of drugs, chemicals, or other substances by mouth.
Administration, Topical
The application of drug preparations to the surfaces of the body, especially the skin (ADMINISTRATION, CUTANEOUS) or mucous membranes. This method of treatment is used to avoid systemic side effects when high doses are required at a localized area or as an alternative systemic administration route, to avoid hepatic processing for example.
Chickenpox
A highly contagious infectious disease caused by the varicella-zoster virus (HERPESVIRUS 3, HUMAN). It usually affects children, is spread by direct contact or respiratory route via droplet nuclei, and is characterized by the appearance on the skin and mucous membranes of successive crops of typical pruritic vesicular lesions that are easily broken and become scabbed. Chickenpox is relatively benign in children, but may be complicated by pneumonia and encephalitis in adults. (From Dorland, 27th ed)
Virus Shedding
The expelling of virus particles from the body. Important routes include the respiratory tract, genital tract, and intestinal tract. Virus shedding is an important means of vertical transmission (INFECTIOUS DISEASE TRANSMISSION, VERTICAL).
Viral Plaque Assay
Method for measuring viral infectivity and multiplication in CULTURED CELLS. Clear lysed areas or plaques develop as the VIRAL PARTICLES are released from the infected cells during incubation. With some VIRUSES, the cells are killed by a cytopathic effect; with others, the infected cells are not killed but can be detected by their hemadsorptive ability. Sometimes the plaque cells contain VIRAL ANTIGENS which can be measured by IMMUNOFLUORESCENCE.
Encephalitis, Varicella Zoster
Inflammation of brain tissue caused by infection with the varicella-zoster virus (HERPESVIRUS 3, HUMAN). This condition is associated with immunocompromised states, including the ACQUIRED IMMUNODEFICIENCY SYNDROME. Pathologically, the virus tends to induce a vasculopathy and infect oligodendrocytes and ependymal cells, leading to CEREBRAL INFARCTION, multifocal regions of demyelination, and periventricular necrosis. Manifestations of varicella encephalitis usually occur 5-7 days after onset of HERPES ZOSTER and include HEADACHE; VOMITING; lethargy; focal neurologic deficits; FEVER; and COMA. (From Joynt, Clinical Neurology, 1996, Ch 26, pp29-32; Hum Pathol 1996 Sep;27(9):927-38)
Bell Palsy
A syndrome characterized by the acute onset of unilateral FACIAL PARALYSIS which progresses over a 2-5 day period. Weakness of the orbicularis oculi muscle and resulting incomplete eye closure may be associated with corneal injury. Pain behind the ear often precedes the onset of paralysis. This condition may be associated with HERPESVIRUS 1, HUMAN infection of the facial nerve. (Adams et al., Principles of Neurology, 6th ed, p1376)
Arabinofuranosyluracil
A pyrimidine nucleoside formed in the body by the deamination of CYTARABINE.
Skin Diseases, Viral
Skin diseases caused by viruses.
Phosphonoacetic Acid
A simple organophosphorus compound that inhibits DNA polymerase, especially in viruses and is used as an antiviral agent.
Idoxuridine
An analog of DEOXYURIDINE that inhibits viral DNA synthesis. The drug is used as an antiviral agent.
Virus Activation
The mechanism by which latent viruses, such as genetically transmitted tumor viruses (PROVIRUSES) or PROPHAGES of lysogenic bacteria, are induced to replicate and then released as infectious viruses. It may be effected by various endogenous and exogenous stimuli, including B-cell LIPOPOLYSACCHARIDES, glucocorticoid hormones, halogenated pyrimidines, IONIZING RADIATION, ultraviolet light, and superinfecting viruses.
Pharmaceutical Vehicles
A carrier or inert medium used as a solvent (or diluent) in which the medicinally active agent is formulated and or administered. (Dictionary of Pharmacy, 1986)
Inosine Pranobex
An alkylamino-alcohol complex of inosine used in the treatment of a variety of viral infections. Unlike other antiviral agents, it acts by modifying or stimulating cell-mediated immune processes rather than acting on the virus directly.
Double-Blind Method
A method of studying a drug or procedure in which both the subjects and investigators are kept unaware of who is actually getting which specific treatment.
Recurrence
The return of a sign, symptom, or disease after a remission.
Vidarabine
A nucleoside antibiotic isolated from Streptomyces antibioticus. It has some antineoplastic properties and has broad spectrum activity against DNA viruses in cell cultures and significant antiviral activity against infections caused by a variety of viruses such as the herpes viruses, the VACCINIA VIRUS and varicella zoster virus.
Keratitis, Herpetic
A superficial, epithelial Herpesvirus hominis infection of the cornea, characterized by the presence of small vesicles which may break down and coalesce to form dendritic ulcers (KERATITIS, DENDRITIC). (Dictionary of Visual Science, 3d ed)

Infectious complications in 126 patients treated with high-dose chemotherapy and autologous peripheral blood stem cell transplantation. (1/1121)

The effect of an extensive prophylactic antimicrobial regimen was prospectively assessed in 126 patients after high-dose chemotherapy and autologous PBSC. They received ciprofloxacin (500 mg/12 h), acyclovir (200 mg/6 h), and itraconazole (200 mg/12 h) orally until neutrophil recovery. Febrile patients received i.v. imipenem (500 mg/6 h) to which vancomycin and amikacin were added if fever persisted for 2-3 and 5 days, respectively. Amphotericin B lipid complex was further given on day 7 or 8 of fever. Median times for a neutrophil count of >0.5 x 10(9)/l and a platelet count of >20 x 10(9)/l were 9 and 11 days. Severe neutropenia (<0.1 x 10(9)/l) lasted for a median of 5 days in which 72% of febrile episodes and 50% of cases of bacteremia occurred. Gram-positive bacteria were isolated in 30 of 40 episodes of bacteremia, 25 of which were caused by Staphylococcus epidermidis. Clinical foci were the intravascular catheter in 35 cases, respiratory infection in 11, cellulitis in two, anal abscess in one, and neutropenic enterocolitis in one. The high incidence of febrile episodes (94%) and bacteremias (31%) may be due to the lack of efficacy of antimicrobial prophylaxis and the persistence of a 5-day period of severe neutropenia.  (+info)

B cell lymphoproliferative disorders following hematopoietic stem cell transplantation: risk factors, treatment and outcome. (2/1121)

Twenty-six cases of B cell lymphoproliferative disorder (BLPD) were identified among 2395 patients following hematopoietic stem cell transplants (HSCT) for which an overall incidence of BLPD was 1.2%. The true incidence was probably higher, since 9/26 of the diagnoses were made at autopsy. No BLPD was observed following autologous HSCT, so risk factor analyses were confined to the 1542 allogeneic HSCT. Factors assessed were HLA-mismatching (> or = 1 antigen), T cell depletion (TCD), presence of acute GvHD (grades II-IV), donor type (related vs unrelated), age of recipient and donor, and underlying disease. Factors found to be statistically significant included patients transplanted for immune deficiency and CML, donor age > or = 18 years, TCD, and HLA-mismatching, with recipients of combined TCD and HLA-mismatched grafts having the highest incidence. Factors found to be statistically significant in a multiple regression analysis were TCD, donor age and immune deficiency, although 7/8 of the patients with immunodeficiencies and BLPD received a TCD graft from a haploidentical parent. The overall mortality was 92% (24/26). One patient had a spontaneous remission, but subsequently died >1 year later of chronic GVHD. Thirteen patients received therapy for BLPD. Three patients received lymphocyte infusions without response. The only patients with responses and longterm survival received alpha interferon (alphaIFN). Of seven patients treated with alphaIFN there were four responses (one partial and three complete). These data demonstrate that alphaIFN can be an effective agent against BLPD following HSCT, if a timely diagnosis is made.  (+info)

Interactions of a nonpeptidic drug, valacyclovir, with the human intestinal peptide transporter (hPEPT1) expressed in a mammalian cell line. (3/1121)

The results of previous work performed in our laboratory using an in situ perfusion technique in rats and rabbit apical brush border membrane vesicles have suggested that the intestinal uptake of valacyclovir (VACV) appears to be mediated by multiple membrane transporters. Using these techniques, it is difficult to characterize the transport kinetics of VACV with each individual transporter in the presence of multiple known or unknown transporters. The purpose of this study was to characterize the interaction of VACV and the human intestinal peptide transporter using Chinese hamster ovary (CHO) cells that overexpress the human intestinal peptide transporter (hPEPT1) gene. VACV uptake was significantly greater in CHO cells transfected with hPEPT1 than in cells transfected with only the vector, pcDNA3. The optimum pH for VACV uptake was determined to occur at pH 7.5. Proton cotransport was not observed in hPEPT1/CHO cells, consistent with previously observed results in tissues and Caco-2 cells. VACV uptake was concentration dependent and saturable with a Michaelis-Menten constant and maximum velocity of 1.64 +/- 0.06 mM and 23.34 +/- 0.36 nmol/mg protein/5 min, respectively. A very similar Km value was obtained in hPEPT1/CHO cells and in rat and rabbit tissues and Caco-2 cells, suggesting that hPEPT1 dominates the intestinal transport properties of VACV in vitro. VACV uptake was markedly inhibited by various dipeptides and beta-lactam antibiotics, and Ki values of 12.8 +/- 2.7 and 9.1 +/- 1.2 mM were obtained for Gly-Sar and cefadroxil at pH 7.5, respectively. The present results demonstrate that VACV is a substrate for the human intestinal peptide transporter in hPEPT1/CHO cells and that although transport is pH dependent, proton cotransport is not apparent. Also, the results demonstrate that the hPEPT1/CHO cell system has use in investigating the transport kinetics of drugs with the human intestinal peptide transporter hPEPT1; however, the extrapolation of these transport properties to the in vivo situation requires further investigation.  (+info)

Management of human cytomegalovirus infection and disease after allogeneic bone marrow transplantation. (4/1121)

BACKGROUND AND OBJECTIVE: Human cytomegalovirus (HCMV) infection and disease remain a major cause of morbidity and mortality after bone marrow transplantation. HCMV disease, especially pneumonitis, may be treated with ganciclovir and immunoglobulin but even so the outcome is poor with mortality rates of 30-70%. It is therefore imperative to treat HCMV infection before it develops into disease. The aim of this article is to describe the main strategies used to prevent HCMV infection and to improve the survival after CMV disease in bone marrow transplant recipients. INFORMATION SOURCES: In the present review, we examined personal papers in this field and articles published in journals covered by the Science Citation Index and Medline. STATE OF THE ART: Major advances have been made in preventing HCMV infection and disease through two different approaches, both of which reduce HCMV induced morbidity and mortality: In pre-emptive therapy, patients are given ganciclovir when HCMV infection is first identified and this is continued 3-4 months after transplantation; in prophylactic therapy ganciclovir is given to all patients at risk of HCMV disease from engraftment up to 3-4 months post transplantation. Each strategy has advantages and disadvantages and there is no evidence for the superiority of one over the other since the overall survival is the same and the incidence of death from HCMV disease is similar. PERSPECTIVES: The use of more sensitive tests such as HCMV PCR or antigenemia may improve the outcome but probably will not eradicate all HCMV disease. Future possible strategies could include adoptive transfer of CD8+ HCMV-specific cytotoxic T lymphocytes clones derived from the donor marrow or boosting donor or patient immunity using subunit anti-HCMV vaccines such as gB or pp65.  (+info)

Antiviral therapy for neonatal herpes simplex virus: a cost-effectiveness analysis. (5/1121)

Each year, about 1,600 infants in the United States are infected with neonatal herpes simplex virus. We conducted a cost-effectiveness analysis of antiviral drug therapy (acyclovir) for three forms of herpes simplex virus infection: skin, ear, and mouth (SEM), central nervous system (CNS), and disseminated multiorgan (DIS) disease. Five levels of patient outcomes were examined (normal, mild, moderate, severe, dead). We obtained information on disease occurrence and survival from clinical trials and historical reviews of untreated newborns. We considered approaches for treating all or any of the forms of the disease and compared them with no use of antiviral drugs. The main measure of effectiveness was lives saved, including those of descendants of survivors. Costs were measured from a societal perspective and included direct medical costs, institutional care, and special education. We used a discount rate of 3% and valued dollars at 1995 levels. We also considered the perspective of a managed care organization. From a societal viewpoint relative to no treatment, antiviral therapy for SEM resulted in a gain of 0.8 lives and a cost reduction of $78,601 per case. For the treatment of CNS and DIS disease, antiviral therapy saved more lives but at increased cost, with respective marginal costs per additional life saved of $75,125 and $46,619. From a managed care perspective, antiviral therapy is more cost-effective than from a societal viewpoint because costs of institutional care and special education are not the responsibility of managed care organizations. Development of at-home therapies will further improve the cost-effectiveness of antiviral therapy for neonatal herpes simplex virus infection.  (+info)

Intravenous penciclovir for treatment of herpes simplex infections in immunocompromised patients: results of a multicenter, acyclovir-controlled trial. The Penciclovir Immunocompromised Study Group. (6/1121)

The efficacy and safety of penciclovir (PCV) for the treatment of herpes simplex virus (HSV) infections in immunocompromised (IC) patients were studied in a double-blind, acyclovir (ACV)-controlled, multicenter study. A total of 342 patients with mucocutaneous HSV infections received 5 mg of PCV per kg every 12 or 8 h (q12h or q8h) or 5 mg of ACV per kg q8h, beginning within 72 h of lesion onset and continuing for up to 7 days. The mean age of the patients was 49 years; 94% were white and 52% were female. The main reasons for their IC states were hematologic disorder (63%) and transplant plus hematologic disorder (16%). Clinical and virological assessments were performed daily during the 7-day treatment and then every other day until lesion healing. The primary efficacy parameter addressed new lesion formation. Secondary end points focused on viral shedding, healing, and pain. Approximately 20% of patients in each treatment group developed new lesions during therapy; thus, equivalence with ACV (defined prospectively) was demonstrated for both q12h and q8h PCV regimens. For all three treatment groups, the median time to the cessation of viral shedding was 4 days and the median time to complete healing was 8 days; there were no statistically significant differences in the rates of complete healing or the cessation of viral shedding when the results for PCV q12h and q8h were compared with those for ACV q8h. In addition, there was no statistically significant difference between PCV q12h or q8h, compared with ACV q8h, for the resolution of pain. PCV was well tolerated, with an adverse event profile comparable to that of ACV. In conclusion, PCV q12h is a well-tolerated and effective therapy for mucocutaneous HSV infection in IC patients and offers a reduced frequency of dosing compared with ACV q8h.  (+info)

Valacyclovir for the prevention of cytomegalovirus disease after renal transplantation. International Valacyclovir Cytomegalovirus Prophylaxis Transplantation Study Group. (7/1121)

BACKGROUND: Cytomegalovirus (CMV) disease is a major complication of organ transplantation. We hypothesized that prophylactic treatment with valacyclovir would reduce the risk of CMV disease. METHODS: A total of 208 CMV-negative recipients of a kidney from a seropositive donor and 408 CMV-positive recipients were randomly assigned to receive either 2 g of valacyclovir or placebo orally four times daily for 90 days after transplantation, with the dose adjusted according to renal function. The primary end point was laboratory-confirmed CMV disease in the first six months after transplantation. RESULTS: Treatment with valacyclovir reduced the incidence or delayed the onset of CMV disease in both the seronegative patients (P<0.001) and the seropositive patients (P=0.03). Among the seronegative patients, the incidence of CMV disease 90 days after transplantation was 45 percent among placebo recipients and 3 percent among valacyclovir recipients. Among the seropositive patients, the respective values were 6 percent and 0 percent. At six months, the incidence of CMV disease was 45 percent among seronegative recipients of placebo and 16 percent among seronegative recipients of valacyclovir; it was 6 percent among seropositive placebo recipients and 1 percent among seropositive valacyclovir recipients. At six months, the rate of biopsy-confirmed acute graft rejection in the seronegative group was 52 percent among placebo recipients and 26 percent among valacyclovir recipients (P=0.001). Treatment with valacyclovir also decreased the rates of CMV viremia and viruria, herpes simplex virus disease, and the use of inpatient medical resources. Hallucinations and confusion were more common with valacyclovir treatment, but these events were not severe or treatment-limiting. The rates of other adverse events were similar among the groups. CONCLUSIONS: Prophylactic treatment with valacyclovir is a safe and effective way to prevent CMV disease after renal transplantation.  (+info)

Long term neurological outcome of herpes encephalitis. (8/1121)

Twenty eight children with herpes simplex encephalitis were followed up for a mean of 5.5 years. Two children died and 26 survived, of whom 16 were left with no neurological sequelae and 10 had persistent neurological sequelae. Mean (SD) Glasgow coma score was significantly lower in the patients with neurological sequelae (7.7 (1.5)) and the patients who died (4.5 (0.7)), compared with the patients without neurological sequelae (11 (1.7)).  (+info)

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One year acyclovir suppression of frequently recurring genital herpes: a study of efficacy, safety, virus sensitivity and...One year acyclovir suppression of frequently recurring genital herpes: a study of efficacy, safety, virus sensitivity and...
The efficacy of oral acyclovir 400 mg twice daily in suppressing frequently relapsing genital herpes simplex was evaluated in an open multicenter study. Seventy-one patients were treated for 12 months. During treatment, 73% of the patients were completely free of symptoms when taking the tablets continuously, and another 14% had mild symptoms such as erythema and/or itching at single occasions. An accidental treatment interruption for 2-4 days led to mild but definite herpes episodes within a few days in 5 otherwise symptom free patients. Definite herpes episodes despite acyclovir medication occurred in 3 cases (4%). No noteworthy side effects were recorded during the acyclovir treatment. After withdrawal of acyclovir, herpes relapsed within 1-4 weeks in 69% of the patients. The frequency of relapses during the following months was reported to be equal to that before the treatment period in most of the patients. Acyclovir susceptibility of the isolated herpes simplex virus (HSV) strains did not change
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Acyclovir treatment of varicella in otherwise healthy adolescents. The Collaborative Acyclovir Varicella Study Group.Acyclovir treatment of varicella in otherwise healthy adolescents. The Collaborative Acyclovir Varicella Study Group.
STUDY OBJECTIVE: To determine whether orally administered acyclovir is of therapeutic benefit for varicella in otherwise healthy adolescents, and to compare the severity of the disease in adolescents with that in younger children. DESIGN: Multicenter, randomized, placebo-controlled, double-blind trial. SETTING: Patients homes and university hospital clinics. PATIENTS: Sixty-eight adolescents between 13 and 18 years of age with varicella entered the study. Of the 62 adolescents with laboratory-confirmed varicella who were included in the final analysis, 31 received acyclovir and 31 received placebo. INTERVENTIONS: Placebo or an 800 mg acyclovir tablet was given orally four times daily for 5 days, beginning within 24 hours of onset of rash. MEASUREMENTS AND MAIN RESULTS: Acyclovir recipients had significant reductions in times to cessation of new lesion formation (p less than 0.001), maximum number of lesions (p = 0.019), and defervescence (p = 0.045). Mean constitutional illness score was ...
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Background and Objectives: Herpes simplex virus type 2 (HSV-2) is the primary cause of genital ulcer and one of the most prevalent sexually transmitted infections (STI) worldwide. HSV-2 has been recognized as a risk factor for HIV in multiple studies. A substantial shift in the aetiology of genital ulcer disease (GUD) towards genital herpes has been noted in many countries in Africa, especially those with mature HIV epidemics. Some countries guided by the predominance of HSV-2 as the aetiology of GUD in their country, are changing syndromic guidelines to include acyclovir as part of the treatment for GUD. Little data is available to support this decision in terms of its effect on clinical course and its cost-effectiveness. Yet, substantial investment would be needed in poor countries to add acyclovir to their essential drug list. Studies to determine the appropriateness of episodic acyclovir therapy for HSV-2 in the developing world are needed.. Episodic therapy with acyclovir both as a ...
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https://dailymed.nlm.nih.gov/dailymed/archives/fdaDrugInfo.cfm?archiveid=302784
An Open, Multicenter Study of Foscarnet Treatment of Acyclovir-Resistant Herpes Simplex Virus in Patients With the Acquired...An Open, Multicenter Study of Foscarnet Treatment of Acyclovir-Resistant Herpes Simplex Virus in Patients With the Acquired...
Patients must have AIDS as defined by the CDC, or be patients with other immunocompromised states (transplant patients; other hematologic malignancies with recognized cell-mediated immune deficiency), and have herpes simplex virus (HSV) infection documented by culture and in vitro resistance to acyclovir by standard laboratory susceptibility or suspected resistance to acyclovir after an acyclovir treatment failure in which acyclovir was administered for at least 2 weeks intravenously at doses of 30 mg/kg/day (or a comparative dose adjusted for renal insufficiency). In vitro susceptibility data must be pending in the latter case.. ...
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A randomised, double-blind trial of valaciclovir prophylaxis for cytomegalovirus disease in patients with advanced human...A randomised, double-blind trial of valaciclovir prophylaxis for cytomegalovirus disease in patients with advanced human...
Cytomegalovirus (CMV) disease is a common complication of advanced human immunodeficiency virus (HIV) infection. Administration of oral valaciclovir, a valine ester of acyclovir, achieves sufficient plasma acyclovir levels to inhibit many clinical isolates. Acyclovir has been associated with enhanced survival in AIDS but not with CMV disease prevention. CMV-seropositive patients (1227) with CD4 cell counts ,100/mm3 were enrolled in a randomized, double-blind trial. Valaciclovir, 8 g/day, was compared with acyclovir, 3.2 or 0.8 g/day, for CMV prevention; all three arms were compared for survival. The confirmed CMV disease rate was 11.7% among valaciclovir recipients and 17.5% in the pooled acyclovir arms, a 33% reduction in risk. Time to confirmed CMV disease was significantly longer for the valaciclovir group (P = .03). A trend toward earlier mortality for valaciclovir recipients was seen (P = .06). Toxicity and earlier medication discontinuation were more common in this group. Valaciclovir ...
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Postexposure prophylaxis of varicella in family contact by oral acyclovir<...Postexposure prophylaxis of varicella in family contact by oral acyclovir<...
TY - JOUR. T1 - Postexposure prophylaxis of varicella in family contact by oral acyclovir. AU - Asano, Y.. AU - Yoshikawa, T.. AU - Suga, S.. AU - Kobayashi, I.. AU - Nakashima, T.. AU - Yazaki, T.. AU - Ozaki, T.. AU - Yamada, A.. AU - Imanishi, J.. PY - 1993/1/1. Y1 - 1993/1/1. N2 - Objective. To determine whether varicella can be prevented by administration of oral acyclovir (ACV) during the incubation period of the disease. Subjects and methods. ACV (40 or 80 mg/kg daily in four divided doses) was given orally to 25 exposed infants and children for 7 days, starting 7 to 9 days after exposure from the index case in their families. Their clinical features were compared with those of 25 age-matched control subjects who had been exposed in their families but did not receive ACV. A fluorescent antibody to membrane antigen assay was used for determination of the antibody to varicella-zoster virus, and a nested polymerase chain reaction method was used for detection of viremia. Results. Among the ...
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Phenotypic testing of patient herpes simplex virus type 1 and 2 isolates for acyclovir resistance by a novel method based on...Phenotypic testing of patient herpes simplex virus type 1 and 2 isolates for acyclovir resistance by a novel method based on...
Acyclovir (ACV) is the most commonly used drug for herpes simplex virus (HSV) infection therapy. Prolonged antiviral therapy or prophylaxis in immunocompromised patients may promote the development of drug-resistant strains. Due to the high polymorphism in genes involved in drug resistance, phenotypic methods, although work-intensive, are still required to test drug susceptibility. Real-time cell analysis (RTCA) based methods could offer a rapid and less labor-intensive alternative for phenotypic testing of ACV resistance ...
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Intravenous Acyclovir and Neurologic Effects | Annals of Internal Medicine | American College of PhysiciansIntravenous Acyclovir and Neurologic Effects | Annals of Internal Medicine | American College of Physicians
To the editor: We read the report of Wade and Meyers (1) with interest as we have also seen a case of transient neurologic deterioration associated with intravenous acyclovir.. A 56-year-old black woman was diagnosed as having hypereosinophilic syndrome in May 1982 when she presented with cough and dyspnea. Results of transbronchial and skin biopsies showed eosinophilic infiltrates. Prednisone and hydroxyurea were started. In June 1982, the patient had dysphagia. An esophagoscopic examination showed herpes simplex. On hospital day 8, intravenous acyclovir therapy was initiated at a dose of 250 mg/m2 body surface area every 8 hours. On the following ...
http://annals.org/aim/article-abstract/697087/intravenous-acyclovir-neurologic-effects
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Lirias: Herpes simplex acyclovir-resistant mutant in experimental keratouveitisLirias: Herpes simplex acyclovir-resistant mutant in experimental keratouveitis
Acyclovir is a potent and selective antiviral agent. Unfortunately, drug-resistant (acyclovir-resistant) mutants have already been reported in herpes simplex virus type 1 (HSV-1) orofacial infections. We have developed a laboratory acyclovir-resistant HSV-1 mutant. The natural course of acyclovir-resistant HSV-1 keratitis was found to be less virulent than that observed in wild type HSV-1 keratitis, but the rate of ganglionic latency was as high as that induced by the parental strain. In vitro studies and in vivo observation of rabbit corneas infected with acyclovir-resistant HSV-1 both demonstrated a significant sensitivity to vidarabine and bromovinyldeoxyuridine ([E]-5-[2-bromovinyl]-2-deoxyuridine). The thymidine kinase activity of the acyclovir-resistant mutant was 69% of that of the wild type HSV-1 ...
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Lirias: Acyclovir-resistant herpes simplex encephalitis in a patient treated with anti-tumor necrosis factor-α monoclonal...Lirias: Acyclovir-resistant herpes simplex encephalitis in a patient treated with anti-tumor necrosis factor-α monoclonal...
Herpes simplex virus is the most common cause of severe sporadic encephalitis. We report a case of herpes simplex type 1-encephalitis in a 50-year-old woman receiving anti-tumor necrosis factor-α monoclonal antibodies adalimumab. Although she was an acyclovir naïve patient, a mixed viral population (wild-type and acyclovir-resistant bearing a thymidine-kinase mutation) was identified in the cerebrospinal fluid. The virus in cerebrospinal fluid evolved and a second thymidine-kinase mutant virus emerged. Combined foscavir and acyclovir treatment resolved the herpes simplex encephalitis. To our knowledge, this is the first report of acyclovir-resistant herpes simplex encephalitis in a patient treated with adalimumab ...
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Introduction: : In October 2003 we published the results of a double-blind randomised trial on the effect of oral acyclovir after penetrating keratoplasty (PK)for herpetic eye disease (HED) (1). During the 2 years follow up, 3 herpes-related (PCR or culture proven) recurrences occurred after oral acyclovir 400 mg twice daily (ACY group) as opposed to 9 recurrences after placebo (PLAC) (p = 0.026, log-rank test). Purpose: : This study was performed to determine the 5 year follow-up of the same patient population. Methods: : All patients were followed up according to the national keratoplasty follow-up register (at 6, 12, 24, 36 and 60 months). Patient records were used to score HSV recurrences and rejection episodes on clinical grounds: dendritic HED or kerato-uveitis. Rejection episodes and anterior chamber reactions were excluded. Results: : : After the 6 months acyclovir prophylaxis until 5 years after the operation 8 recurrences occurred in the ACY group as opposed to 16 in the PLAC group ...
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Herpes simplex encephalitis (HSE) in children sometimes exacerbates after successful treatment; yet the frequency, etiology, and clinical features of exacerbation remain unclear. We report data for 27 children with HSE confirmed by polymerase chain reaction (PCR) analysis; all were successfully treated with acyclovir, but 7 (26%) had a relapse of encephalitic illness. In 2 of those 7, serial examination with a PCR assay showed that herpes simplex virus (HSV) DNA reappeared temporarily in the cerebrospinal fluid (CSF). For 5 of the 7 patients, a second course of acyclovir therapy was effective. Coxsackievirus A9 was isolated from CSF of 1 case patient during subsequent exacerbation. The total dose during initial acyclovir therapy was significantly lower in the relapse group than in the control group (P = .027). In conclusion, exacerbation of HSE in children may be more common than previously recognized. It is suggested that the replication of HSV or another viral pathogen caused a second ...
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We previously showed that long-term oral acyclovir therapy could reduce the prevalence of recurrent uveitis in patients who are having their recurrent uveitis on the basis of herpes simplex virus. (Reference: Rodriguez A, Power WJ, Neves RA, Foster CS: 1994. Long-Term Systemic Acyclovir in the Management of Recurrent Herpes Simplex Keratouveitis. In:Advances in Ocular Immunology (R.B. Nussenblatt, S.M. Whitcup, R.R. Caspi and I. Gery, eds., pp 465-468, Elsevier Science B.V.) This study, however, was not randomized, placebo-controlled, double masked. And while we are convinced, on the basis of this experience that, just as in the case of recurrent herpes simplex keratitis, so too in recurrent herpes simplex uveitis long-term chronic oral prophylactic maintenance anti-viral therapy is safe and effective in reducing recurrences of inflammation, we feel obligated now to subject this belief to rigorous scientific testing through a randomized, double masked, placebo-controlled clinical trial. We have ...
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Self-collected genital swabs compared with cervicovaginal lavage for measuring HIV-1 and HSV-2 and the effect of acyclovir on...Self-collected genital swabs compared with cervicovaginal lavage for measuring HIV-1 and HSV-2 and the effect of acyclovir on...
HIV-1 and HSV-2 are frequent genital co-infections in women. To determine how self-collected genital swabs compare to provider-collected cervicovaginal lavage, paired self-collected genital swabs and cervicovaginal lavage from women co-infected with HIV-1 and HSV-2 were evaluated. Women were in an acyclovir clinical trial and their samples were tested for HIV-1 RNA (361 samples) and HSV-2 DNA (378 samples). Virus shedding, quantity and acyclovir effect were compared. HIV-1 and HSV-2 were more frequently detected in self-collected genital swabs: 74.5% of self-collected genital swabs and 63.6% of cervicovaginal lavage had detectable HIV-1 (p ≤ 0.001, Fishers exact test) and 29.7% of self-collected genital swabs and 19.3% of cervicovaginal lavage had detectable HSV-2 (p ≤ 0.001) in the placebo month. Cervicovaginal lavage and self-collected genital swabs virus levels were correlated (Spearmans rho, 0.68 for HIV; 0.61 for HSV-2) and self-collected genital swabs levels were generally higher. In ...
https://www.physiciansweekly.com/self-collected-genital-swabs-compared-with-cervicovaginal-lavage-for-measuring-hiv-1-and-hsv-2-and-the-effect-of-acyclovir-on-viral-shedding/
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Will Zovirax Work On Genital Herpes. 3 Ways to Treat Genital Herpes Naturally - wikiHowHow to Treat Genital Herpes Naturally. Genital herpes is very common. In those 12 and older, there are about 45 million cases in the US. According to the US Center Genital Herpes Treatment: 2015s Top OptionsLearn about the best Treatment for Genital Herpes to Cure and Get rid of Herpes outbreaks quickly and naturally.acyclovir, Zovirax: Drug Facts, Side Effects and Dosing4/16/2015 · Explains the medication acyclovir (Zovirax), a drug used to how long before viagra takes effect treat initial genital herpes, shingles, and chickenpox infections and to treat patients with Understanding Genital Herpes - TheBody.comGenital herpes is a sexually transmitted infection (STI). According to the Centers for Disease Control and Prevention, in the United States, one of every Genital infections , Genital herpes - Embarrassing …, How men and women can catch genital herpes, symptoms, and what you can doGenital Herpes - Home ...
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AciclovirAciclovir
... (ACV), also known as acyclovir, is an antiviral medication. It is primarily used for the treatment of herpes simplex ... Aciclovir is a nucleoside analogue that mimics guanosine. It works by decreasing the production of the virus's DNA. Aciclovir ... In those with herpes of the eye, aciclovir may be more effective and safer than idoxuridine. It is not clear if aciclovir eye ... aciclovir may be used. For severe HSV infections (especially disseminated HSV), IV aciclovir may also be used. Studies in mice ...
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NeuritisNeuritis
Antiviral medications, including acyclovir, are effective at controlling viral reactivation. Management of ensuing neuropathic ... Herpes reactivation is often treated with acyclovir, although evidence for its efficacy in controlling peripheral neurological ... Taylor M, Gerriets V (2020). Acyclovir. StatPearls. StatPearls Publishing. PMID 31194337. Johnson RW, Alvarez-Pasquin MJ, Bijl ...
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List of antiviral drugsList of antiviral drugs
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Prototype drugPrototype drug
"Acyclovir". DrugBank. 16 October 2018. Retrieved 16 October 2018. "Aspirin - MeSH". NCBI. 16 October 2018. Retrieved 16 October ... is the prototype dihydropyridine calcium channel blocker Chloroquine is the prototypical antimalarial agent Acyclovir is the ...
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Such drugs include other aminoglycosides; the antiviral acyclovir; the antifungal amphotericin B; the antibiotics bacitracin, ...
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Gertrude B. ElionGertrude B. Elion
Acyclovir (Zovirax), for viral herpes. Nelarabine for cancer treatment. Elion GB; Hitchins GH; Vanderwerff H (1951). " ... acyclovir, which was the first selective and effective drug of its kind). Most of Elion's early work came from the use and ... acyclovir (ACV), used in the treatment of herpes infection. Elion was born in New York City on January 23, 1918, to parents ... "Developing the Purine Nucleoside Analogue Acyclovir: the Work of Gertrude B. Elion". J. Biol. Chem. 283 (19): e11. doi:10.1016/ ...
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Nasser M, Fedorowicz Z, Khoshnevisan MH, Shahiri Tabarestani M (October 2008). Nasser M (ed.). "Acyclovir for treating primary ... Antiviral drugs are used to treat herpetic gingivostomatitis such as aciclovir, valaciclovir, famciclovir, and in resistance ... Goldman RD (May 2016). "Acyclovir for herpetic gingivostomatitis in children". Canadian Family Physician. 62 (5): 403-4. PMC ...
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Herpes simplexHerpes simplex
aciclovir trials in the late 1980s. The lower toxicity and ease of administration over vidarabine has led to aciclovir becoming ... However, aciclovir seems to inhibit antibody response, and newborns on aciclovir antiviral treatment experienced a slower rise ... Several antiviral drugs are effective for treating herpes, including aciclovir (acyclovir), valaciclovir, famciclovir, and ... Aciclovir was the first discovered and is now available in generic. Valaciclovir is also available as a generic and is slightly ...
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Lemelson-MIT PrizeLemelson-MIT Prize
acyclovir (Zovirax), for herpes simplex virus infection. 1996 Stanley Norman Cohen (Co-recipient, Lemelson-MIT Prize) for the ...
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IdoxuridineIdoxuridine
Trifluridine Acyclovir Foscarnet Prusoff, W. H. (1959). "Synthesis and biological activities of iododeoxyuridine, an analog of ...
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CidofovirCidofovir
... has also shown efficacy in the treatment of aciclovir-resistant HSV infections. Cidofovir has also been investigated ... Chilukuri S, Rosen T (April 2003). "Management of acyclovir-resistant herpes simplex virus". Dermatologic Clinics. 21 (2): 311- ... likely due to the abundance of safer alternatives such as aciclovir. Cidofovir shows anti-BK virus activity in a subgroup of ... aciclovir, aminosalicylic acid, etc.) are also withheld. Its active metabolite, cidofovir diphosphate, inhibits viral ...
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Antivirals creams include acyclovir and penciclovir, which can speed healing by as much as 10%. Oral antivirals include ... Treatment with oral antivirals such as acyclovir in children within 72 hours of illness onset has shown to shorten duration of ... therefore use of zinc-based sunscreen or topical and oral therapeutics such as acyclovir and valacyclovir may prove helpful. ... acyclovir, valaciclovir, and famciclovir. Famciclovir or valacyclovir, taken in pill form, can be effective using a single day ...
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Elion GB (1993). "Acyclovir: discovery, mechanism of action, and selectivity". J. Med. Virol. Suppl 1: 2-6. doi:10.1002/jmv. ... acyclovir for herpes virus infection, and nelarabine for cancer treatment. Since 2010, GlaxoSmithKline has several times ranked ...
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Chronic fatigue syndrome treatmentChronic fatigue syndrome treatment
December 1988). "Acyclovir treatment of the chronic fatigue syndrome. Lack of efficacy in a placebo-controlled trial". The New ... A small 1988 RCT compared acyclovir against placebo and found that an equal proportion of patients improved from placebo and ... Nucleosidic class drugs such as acyclovir, valacyclovir and ganciclovir are inhibitors of viral replication during DNA (for DNA ... Three people withdrew from acyclovir treatment due to reversible renal failure. Antidepressants are often prescribed to CFS ...
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Roy S, Sukla S, De A, Biswas S (January 2022). "Non-cytopathic herpes simplex virus type-1 isolated from acyclovir-treated ... Studies of vulnerable patient populations have indicated that daily use of antivirals such as aciclovir and valaciclovir can ... Hussin A, Md Nor NS, Ibrahim N (November 2013). "Phenotypic and genotypic characterization of induced acyclovir-resistant ... "Oral acyclovir suppression and neurodevelopment after neonatal herpes". N. Engl. J. Med. 365 (14): 1284-92. doi:10.1056/ ...
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Diezel, W.; Michel, G.; Görtelmeyer, R.; Ostheimer, K. E. (April 1993). "Efficacy of tromantadine and aciclovir in the topical ... Its performance is similar to aciclovir. Like rimantadine, amantadine, and adapromine, tromantadine is a derivative of ... "Randomized double-blind trial of tromantadine versus aciclovir in recurrent herpes orofacialis". Arzneimittel-Forschung. 39 (9 ...
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This means that acyclovir cannot be used to cure the virus infection and infected birds will remain infected and become a ... Acyclovir is a synthetic acyclic purine nucleoside that has strong activity against certain members of the herpesvirus group. ... Acyclovir has decreased morbidity and mortality during multiple Pacheco's disease outbreaks. However, it cannot prevent the ... Smith, C. Gregory (1987). "Use of Acyclovir in an Outbreak of Pacheco's Parrot Disease". AAV Today. 1 (2): 55-56. doi:10.2307/ ...
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VirusVirus
Aciclovir is one of the oldest and most frequently prescribed antiviral drugs. Other antiviral drugs in use target different ... Examples of nucleoside analogues are aciclovir for Herpes simplex virus infections and lamivudine for HIV and hepatitis B virus ... Mindel A, Sutherland S (September 1983). "Genital herpes - the disease and its treatment including intravenous acyclovir". The ...
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Pediatric Trials NetworkPediatric Trials Network
January 2014). "Population pharmacokinetics of intravenous acyclovir in preterm and term infants". The Pediatric Infectious ...
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RibavirinRibavirin
Another study found that ribavirin potentiated the antiviral effect of acyclovir. Some interest has been seen in its possible ... Pancheva SN (September 1991). "Potentiating effect of ribavirin on the anti-herpes activity of acyclovir". Antiviral Research. ...
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Prolonged Oral Acyclovir Improves Neurodevelopment of Infants With CNS HSVProlonged Oral Acyclovir Improves Neurodevelopment of Infants With CNS HSV
Neonates with CNS herpes simplex virus had better neurodevelopment at 1 year if they received 6 months of oral acyclovir after ... All patients received intravenous acyclovir. Once parenteral acyclovir treatment was completed, the infants were randomly ... He said any baby who develops neonatal CNS or SEM herpes disease "should be treated with intravenous acyclovir...regardless of ... Once theyve completed that course of [intravenous] acyclovir therapy, I believe that our data would strongly support that they ...
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Pregnancy Outcomes Following Systemic Prenatal Acyclovir Exposure - - June 1, 1984-June 30, 1993Pregnancy Outcomes Following Systemic Prenatal Acyclovir Exposure - - June 1, 1984-June 30, 1993
A prenatal exposure to acyclovir is defined as inadvertent or intentional use of oral or intravenous acyclovir at any time ... To assess the outcomes of pregnancies exposed to acyclovir, the Acyclovir in Pregnancy Registry was established on June 1, 1984 ... acyclovir administered intravenously is indicated. Among pregnant women without life-threatening disease, systemic acyclovir ... Acyclovir is available as a prescription medication and is used most commonly in its oral form to treat genital herpes; it is ...
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Aciclovir - Oxford ReferenceAciclovir - Oxford Reference
A nucleoside analogue (hydroxyethoxymethylguanine) with antiviral properties against both type 1 and 2 herpes virus and varicella-zoster virus. The drug is converted to the monophosphate by viral thymidine kinase and to the triphosphate by cellular enzymes. The triphosphate competitively inhibits viral DNA polymerase and can be incorporated into viral DNA where it blocks further replication. TN Zovirax.
https://www.oxfordreference.com/view/10.1093/oi/authority.20110803095347270
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Take or use acyclovir until you finish the prescription, even if you feel better. If you stop taking acyclovir too soon or skip ... Before taking acyclovir,. *tell your doctor and pharmacist if you are allergic to acyclovir, valacyclovir (Valtrex), any other ... If you become pregnant while taking acyclovir, call your doctor.. *if you are taking acyclovir to treat genital herpes, you ... Acyclovir may cause other side effects. Call your doctor if you have any unusual problems while you are taking or using this ...
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Zovirax (Acyclovir): Uses, Dosage, Side Effects, Interactions, WarningZovirax (Acyclovir): Uses, Dosage, Side Effects, Interactions, Warning
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Acyclovir 5% Topical OintmentAcyclovir 5% Topical Ointment
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Comparative in Vitro Release Testing (IVRT) of Acyclovir ProductsComparative in Vitro Release Testing (IVRT) of Acyclovir Products
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A double-blind study of oral acyclovir for suppression of recurrences of genital herpes simplex virus infectionA double-blind study of oral acyclovir for suppression of recurrences of genital herpes simplex virus infection
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Acyclovir - Patient | NIHAcyclovir - Patient | NIH
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  • Acyclovir is an antiviral drug, a synthetic nucleoside analogue, that has inhibitory activity (interferes with viral replication) against the herpes viruses , including herpes simplex 1 and 2 ( cold sores and genital herpes ), varicella -zoster ( shingles and chickenpox ), and Epstein-Barr virus ( mononucleosis ). (medicinenet.com)
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  • Patients with frequently recurring genital herpes were enrolled in a double-blind placebo-controlled trial comparing 200-mg acyclovir capsules, given five or two times daily, with placebo. (nih.gov)
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  • The acyclovir is converted to an active form by the virus itself, and the virus then uses the active form of acyclovir rather than the nucleoside it normally uses to manufacture DNA, a critical component of viral replication. (medicinenet.com)
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  • In vitro, acyclovir triphosphate stops replication of herpes viral DNA. (nih.gov)
  • The greater antiviral activity of acyclovir against HSV compared to VZV is due to its more efficient phosphorylation by the viral TK. (nih.gov)
  • Resistance of HSV and VZV to acyclovir can result from qualitative and quantitative changes in the viral TK and/or DNA polymerase. (nih.gov)
  • While most of the acyclovir-resistant mutants isolated thus far from immunocompromised patients have been found to be TK-deficient mutants, other mutants involving the viral TK gene (TK partial and TK altered) and DNA polymerase have been isolated. (nih.gov)
  • The possibility of viral resistance to acyclovir should be considered in patients who show poor clinical response during therapy. (nih.gov)
  • Incorporation of active acyclovir into new viral DNA stops the production of the DNA. (medicinenet.com)
  • Breakthrough recurrences in acyclovir recipients were of shorter duration and associated with a lower frequency of viral shedding than recurrences in placebo recipients. (nih.gov)
  • Aciclovir AL is a medicinal product for the treatment of viral infections (antiviral medicine). (medicine-one.org)
  • Aciclovir AL is not recommended for the treatment of herpes simplex, viral infections in neonates and severe herpes simplex infection in immunocompromised children. (medicine-one.org)
  • Acyclovir/Zovirax is the Original aciclovir in Kenya is used mainly for the treatment of viral infections due to herpes simplex virus (types 1 and 2) and varicella-zoster virus (herpes zoster and chickenpox). (epharmacyke.com)
  • Phosphorylation by either HSV-TK or VZV-TK converts acyclovir into acyclovir triphosphate, which is then incorporated into viral DNA, thereby blocking further DNA synthesis. (britannica.com)
  • Aciclovir is used to treat viral infections such as cold sores, to stop the growth of Herpes simplex virus, Varicella zoster virus, etc. (pcostracker.app)
  • Aciclovir is used to treat viral infections such as cold sores, to stop the growth of Herpes simplex virus, Varicella zoster virus (caused by chickenpox and shingles), Epstein Barr Virus (caused by glandular fever), and to a lesser extent Cytomegalovirus (CMV). (pcostracker.app)
Drugs4
  • Which drugs or supplements interact with acyclovir? (medicinenet.com)
  • Combining acyclovir with cidofovir ( Vistide ), amphotericin B ( Fungizone ) or other drugs that reduce kidney function may increase harmful effects on the kidney. (medicinenet.com)
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  • [ 31 ] Valacyclovir (1000 mg 3 times a day) and famciclovir (500 mg 3 times a day) are newer antiviral drugs with higher oral bioavailability than acyclovir and can be dosed less often. (medscape.com)
Intravenous7
  • All patients received intravenous acyclovir. (medscape.com)
  • He said any baby who develops neonatal CNS or SEM herpes disease "should be treated with intravenous acyclovir. (medscape.com)
  • Once they've completed that course of [intravenous] acyclovir therapy, I believe that our data would strongly support that they should then be started on 6 months of oral acyclovir suppressive therapy with periodic monitoring of the white blood cell count through just a regular routine blood test. (medscape.com)
  • A prenatal exposure to acyclovir is defined as inadvertent or intentional use of oral or intravenous acyclovir at any time during pregnancy. (cdc.gov)
  • Aciclovir/Herperax in Kenya is administered by intravenous infusion as the sodium salt over 1 hour. (epharmacyke.com)
  • Even if the final diagnosis of HSE has not been established, intravenous (IV) acyclovir should be initiated immediately. (medscape.com)
  • The patient was treated with intravenous (IV) acyclovir at 5 mg/kg followed by antiviral therapy containing 5% cidofovir administered as oral gel. (cdc.gov)
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Valacyclovir3
  • tell your doctor and pharmacist if you are allergic to acyclovir, valacyclovir (Valtrex), any other medications, milk proteins, or any of the ingredients in acyclovir products. (medlineplus.gov)
  • You should not use this medication if you are allergic to acyclovir or valacyclovir ( Valtrex ). (emedicinehealth.com)
  • IMSEAR at SEARO: Valacyclovir-induced urticaria without acyclovir hypersensitivity. (who.int)
Varicella-zoste1
  • Acyclovir is also the drug of choice for varicella-zoster virus (VZV) encephalitis, although ganciclovir is also considered an alternative option. (medscape.com)
Patients with impaired renal function1
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Oral acyclovir8
  • November 3, 2010 (Vancouver, British Columbia) - Infants with neonatal herpes simplex virus (HSV) infection with central nervous system (CNS) involvement have better neurodevelopmental outcomes at 1 year if they are started on oral acyclovir suppressive therapy immediately after parenteral acyclovir than if oral therapy is delayed. (medscape.com)
  • Therefore, the Collaborative Antiviral Study Group (CASG) of the National Institute of Allergy and Infectious Diseases assessed the efficacy of oral acyclovir suppressive therapy in modifying these outcomes. (medscape.com)
  • Once parenteral acyclovir treatment was completed, the infants were randomly assigned to receive either placebo or oral acyclovir 300 mg/m 2 3 times a day for 6 months. (medscape.com)
  • Infants on oral acyclovir stayed on blinded study medication 2 months longer before discontinuation because of recurrent skin lesions compared with infants receiving placebo ( P = .009). (medscape.com)
  • Daily oral acyclovir was well tolerated. (nih.gov)
  • Effect of oral acyclovir against primary and secondary viraemia in incubation period of varicella. (bmj.com)
  • These data indicate that oral acyclovir more effectively inhibits replication of VZV in secondary viraemia than that of the primary viraemia. (bmj.com)
  • Prophylaxis or modification of varicella by oral acyclovir after household exposure. (bmj.com)
Medication2
  • Acyclovir may also be used for other purposes not listed in this medication guide. (emedicinehealth.com)
  • The mean monthly recurrence rate during the medication period was 0.86 in placebo recipients, compared with 0.13 in patients treated with acyclovir five times daily and 0.14 in patients treated with acyclovir twice daily (P less than 0.001 for both groups compared with placebo). (nih.gov)
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Renal2
  • Probenecid (Benemid) may increase acyclovir serum levels by decreasing renal excretion of acyclovir. (medicinenet.com)
  • You should talk with your healthcare provider prior to taking Aciclovir if you have kidney disease, including kidney failure (renal failure), any allergies, including allergies to food, dyes, or preservatives. (pcostracker.app)
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Infection8
  • Acyclovir is also sometimes used to treat eczema herpeticum (a skin infection caused by the herpes virus) to treat and prevent herpes infections of the skin, eyes, nose, and mouth in patients with human immunodeficiency virus (HIV), and to treat oral hairy leukoplakia (condition that causes hairy white or gray-colored patches on the tongue or inside of the cheek). (medlineplus.gov)
  • Acyclovir (Zovirax Registered *), an antiviral drug effective in the treatment of herpes simplex infection, was approved by the Food and Drug Administration (FDA) in 1984. (cdc.gov)
  • Some physicians have reported intentional use of acyclovir during pregnancy for treatment of life-threatening herpes simplex infection. (cdc.gov)
  • Clinical isolates of HSV and VZV with reduced susceptibility to acyclovir have been recovered from immunocompromised patients, especially with advanced HIV infection. (nih.gov)
  • Acyclovir will not cure herpes, but it can lessen the symptoms of the infection. (emedicinehealth.com)
  • The infection was confirmed by a fluorescent antibody to membrane antigen assay in 11 (85%) of 13 children who received acyclovir for the first seven days after exposure to VZV and in 11 (79%) of 14 who received acyclovir for the next seven days. (bmj.com)
  • Aciclovir or acyclovir (USAN, former BAN) is a guanine analogue antiviral drug used most commonly for the treatment of herpes simplex virus infection. (loinc.org)
  • In order to achieve good results, treatment with Aciclovir AL should begin immediately with the onset of the first symptoms of infection. (medicine-one.org)
Hypersensitivity1
  • Aciclovir Injection is contraindicated for patients who develop hypersensitivity to Acitop or valaciclovir. (pcostracker.app)
Herpes zoster1
  • For herpes zoster: 800 mg aciclovir AL 5 times a day (corresponding to 4000 mg aciclovir / day) every 4 hours during the day. (medicine-one.org)
Inhibitory activity2
  • The inhibitory activity of acyclovir is highly selective due to its affinity for the enzyme thymidine kinase (TK) encoded by HSV and VZV. (nih.gov)
  • Acyclovir has demonstrated inhibitory activity against both herpes simplex virus type 1 (HSV-1) and herpes simplex virus type 2 (HSV-2) and is taken up selectively by infected cells. (medscape.com)
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Sodium1
  • Aciclovir sodium 1.1 g is approximately equivalent to 1 g of aciclovir. (epharmacyke.com)
Chickenpox1
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Placebo3
  • Of 47 placebo recipients, 44 (94 per cent) had recurrences during the 120-day treatment period, compared with 13 (29 per cent) of 45 patients treated with acyclovir five times daily and 18 of 51 (35 per cent) treated with acyclovir twice daily (P less than 0.001 for each regimen compared with placebo). (nih.gov)
  • The median time to the first clinical recurrence was 18 days in placebo recipients, compared with over 120 days in both acyclovir-treated groups (P less than 0.001 for both groups compared with placebo). (nih.gov)
  • Aciclovir suspension 15 mg/kg five times a day for seven days, or placebo. (bmj.com)