The area covering the terminal portion of ESOPHAGUS and the beginning of STOMACH at the cardiac orifice.
Retrograde flow of gastric juice (GASTRIC ACID) and/or duodenal contents (BILE ACIDS; PANCREATIC JUICE) into the distal ESOPHAGUS, commonly due to incompetence of the LOWER ESOPHAGEAL SPHINCTER.
The physiologic or functional barrier to GASTROESOPHAGEAL REFLUX at the esophagogastric junction. Sphincteric muscles remain tonically contracted during the resting state and form the high-pressure zone separating the lumen of the ESOPHAGUS from that of the STOMACH. (Haubrich et al, Bockus Gastroenterology, 5th ed., pp399, 415)
Compounds that inhibit H(+)-K(+)-EXCHANGING ATPASE. They are used as ANTI-ULCER AGENTS and sometimes in place of HISTAMINE H2 ANTAGONISTS for GASTROESOPHAGEAL REFLUX.
Hydrochloric acid present in GASTRIC JUICE.
Measurement of the pressure or tension of liquids or gases with a manometer.
Organic compounds that have the general formula R-SO-R. They are obtained by oxidation of mercaptans (analogous to the ketones). (From Hackh's Chemical Dictionary, 4th ed)
A syndrome that is characterized by the triad of severe PEPTIC ULCER, hypersecretion of GASTRIC ACID, and GASTRIN-producing tumors of the PANCREAS or other tissue (GASTRINOMA). This syndrome may be sporadic or be associated with MULTIPLE ENDOCRINE NEOPLASIA TYPE 1.
A 2,2,2-trifluoroethoxypyridyl derivative of timoprazole that is used in the therapy of STOMACH ULCERS and ZOLLINGER-ELLISON SYNDROME. The drug inhibits H(+)-K(+)-EXCHANGING ATPASE which is found in GASTRIC PARIETAL CELLS. Lansoprazole is a racemic mixture of (R)- and (S)-isomers.
A GASTRIN-secreting neuroendocrine tumor of the non-beta ISLET CELLS, the GASTRIN-SECRETING CELLS. This type of tumor is primarily located in the PANCREAS or the DUODENUM. Majority of gastrinomas are malignant. They metastasize to the LIVER; LYMPH NODES; and BONE but rarely elsewhere. The presence of gastrinoma is one of three requirements to be met for identification of ZOLLINGER-ELLISON SYNDROME, which sometimes occurs in families with MULTIPLE ENDOCRINE NEOPLASIA TYPE 1; (MEN 1).
A family of gastrointestinal peptide hormones that excite the secretion of GASTRIC JUICE. They may also occur in the central nervous system where they are presumed to be neurotransmitters.
Compounds that contain benzimidazole joined to a 2-methylpyridine via a sulfoxide linkage. Several of the compounds in this class are ANTI-ULCER AGENTS that act by inhibiting the POTASSIUM HYDROGEN ATPASE found in the PROTON PUMP of GASTRIC PARIETAL CELLS.
Tumors or cancer of the DUODENUM.
A spiral bacterium active as a human gastric pathogen. It is a gram-negative, urease-positive, curved or slightly spiral organism initially isolated in 1982 from patients with lesions of gastritis or peptic ulcers in Western Australia. Helicobacter pylori was originally classified in the genus CAMPYLOBACTER, but RNA sequencing, cellular fatty acid profiles, growth patterns, and other taxonomic characteristics indicate that the micro-organism should be included in the genus HELICOBACTER. It has been officially transferred to Helicobacter gen. nov. (see Int J Syst Bacteriol 1989 Oct;39(4):297-405).
Infections with organisms of the genus HELICOBACTER, particularly, in humans, HELICOBACTER PYLORI. The clinical manifestations are focused in the stomach, usually the gastric mucosa and antrum, and the upper duodenum. This infection plays a major role in the pathogenesis of type B gastritis and peptic ulcer disease.
A genus of gram-negative, spiral-shaped bacteria that has been isolated from the intestinal tract of mammals, including humans. It has been associated with PEPTIC ULCER.
A measure of the quality of health care by assessment of unsuccessful results of management and procedures used in combating disease, in individual cases or series.
A broad-spectrum semisynthetic antibiotic similar to AMPICILLIN except that its resistance to gastric acid permits higher serum levels with oral administration.
A species of HELICOBACTER that colonizes in the STOMACH of laboratory MICE; CATS; and DOGS. It is associated with lymphoid follicular hyperplasia and mild GASTRITIS in CATS.
Inflammation of the GASTRIC MUCOSA, a lesion observed in a number of unrelated disorders.
A class I anti-arrhythmic agent (one that interferes directly with the depolarization of the cardiac membrane and thus serves as a membrane-stabilizing agent) with a depressant action on the heart similar to that of guanidine. It also possesses some anticholinergic and local anesthetic properties.
A macrolide antibiotic produced by Streptomyces ambofaciens. The drug is effective against gram-positive aerobic pathogens, N. gonorrhoeae, and staphylococci. It is used to treat infections caused by bacteria and Toxoplasma gondii.
Agents used for the treatment or prevention of cardiac arrhythmias. They may affect the polarization-repolarization phase of the action potential, its excitability or refractoriness, or impulse conduction or membrane responsiveness within cardiac fibers. Anti-arrhythmia agents are often classed into four main groups according to their mechanism of action: sodium channel blockade, beta-adrenergic blockade, repolarization prolongation, or calcium channel blockade.
A drug formerly used in the treatment of angina pectoris but superseded by less hazardous drugs. Prenylamine depletes myocardial catecholamine stores and has some calcium channel blocking activity. (From Martindale, The Extra Pharmacopoeia, 30th ed, p1406)
Drugs that block nerve conduction when applied locally to nerve tissue in appropriate concentrations. They act on any part of the nervous system and on every type of nerve fiber. In contact with a nerve trunk, these anesthetics can cause both sensory and motor paralysis in the innervated area. Their action is completely reversible. (From Gilman AG, et. al., Goodman and Gilman's The Pharmacological Basis of Therapeutics, 8th ed) Nearly all local anesthetics act by reducing the tendency of voltage-dependent sodium channels to activate.
A local anesthetic and cardiac depressant used as an antiarrhythmia agent. Its actions are more intense and its effects more prolonged than those of PROCAINE but its duration of action is shorter than that of BUPIVACAINE or PRILOCAINE.
Agents that are capable of inducing a total or partial loss of sensation, especially tactile sensation and pain. They may act to induce general ANESTHESIA, in which an unconscious state is achieved, or may act locally to induce numbness or lack of sensation at a targeted site.
A genus of zygomycetous fungi of the family Cunninghamellaceae, order MUCORALES. Some species cause systemic infections in humans.
An order of zygomycetous fungi, usually saprophytic, causing damage to food in storage, but which may cause respiratory infection or MUCORMYCOSIS in persons suffering from other debilitating diseases.
The chemical alteration of an exogenous substance by or in a biological system. The alteration may inactivate the compound or it may result in the production of an active metabolite of an inactive parent compound. The alterations may be divided into METABOLIC DETOXICATION, PHASE I and METABOLIC DETOXICATION, PHASE II.
A bibliographic database that includes MEDLINE as its primary subset. It is produced by the National Center for Biotechnology Information (NCBI), part of the NATIONAL LIBRARY OF MEDICINE. PubMed, which is searchable through NLM's Web site, also includes access to additional citations to selected life sciences journals not in MEDLINE, and links to other resources such as the full-text of articles at participating publishers' Web sites, NCBI's molecular biology databases, and PubMed Central.
A kingdom of eukaryotic, heterotrophic organisms that live parasitically as saprobes, including MUSHROOMS; YEASTS; smuts, molds, etc. They reproduce either sexually or asexually, and have life cycles that range from simple to complex. Filamentous fungi, commonly known as molds, refer to those that grow as multicellular colonies.

Comparison of rabeprazole 20 mg versus omeprazole 20 mg in the treatment of active duodenal ulcer: a European multicentre study. (1/641)

BACKGROUND: Rabeprazole sodium is the newest member of a class of substituted benzimidazole molecules known as proton pump inhibitors. Other proton pump inhibitors have been shown to be effective in healing active duodenal ulcer. METHOD: This randomized, double-blind, multicentre study, conducted at 25 European sites, compared the efficacy and tolerability of rabeprazole and omeprazole in patients with active duodenal ulcers. One hundred and two patients with active duodenal ulcer received rabeprazole 20 mg and 103 patients omeprazole 20 mg once daily for 2 or 4 weeks, with ulcer healing monitored by endoscopy. RESULTS: After 2 weeks, complete ulcer healing was documented in 69% of patients given rabeprazole 20 mg and in 62% of patients given omeprazole 20 mg (N.S.). After 4 weeks, healing rates were 98% in the rabeprazole group and 93% in the omeprazole group (P = 0.083). Rabeprazole-treated patients had significantly greater improvement in daytime pain symptom relief than those treated with omeprazole at the conclusion of the study (P = 0.038). Both drugs were well tolerated over the 4-week treatment period. Mean changes from baseline to end-point in fasting serum gastrin were significantly greater in the rabeprazole group, but at end-point mean values were well within normal limits for both groups. No clinically meaningful changes or other between-group differences were observed in laboratory parameters. CONCLUSION: In this study, rabeprazole produced healing rates equivalent to omeprazole at weeks 2 and 4, and provided significantly greater improvement in daytime pain. Both treatments were well tolerated.  (+info)

Furazolidone-containing short-term triple therapies are effective in the treatment of Helicobacter pylori infection. (2/641)

BACKGROUND: A furazolidone-containing therapeutic regimen for Helicobacter pylori infection has attracted special interest in the face of a rising world-wide metronidazole resistant H. pylori, and the expense of currently used antimicrobial regimens. AIM: To evaluate the efficacy of furazolidone-containing regimens in eradicating H. pylori. METHODS: One-hundred and forty H. pylori positive patients with endoscopically confirmed duodenal ulcer or functional dyspepsia received one of four different regimens to eradicate H. pylori. In the first trial, the patients were randomly assigned to receive a 1-week course of furazolidone 100 mg b.d. and clarithromycin 250 mg b.d., with either tripotassium dicitrato bismuthate (TDB) 240 mg b.d. (FCB group) or lansoprazole 30 mg daily (FCL group). In the second trial, the patients were randomly assigned to receive a 1-week course of clarithromycin 250 mg b.d. and omeprazole 20 mg daily, with either furazolidone 100 mg b.d. (FCO group) or metronidazole 400 mg b.d. (MCO group). Endoscopy was repeated 4 weeks following completion of therapy with re-assessment of H. pylori status on gastric biopsies by histology and culture. RESULTS: Four patients (1 in FCB, 1 in FCO and 2 in MCO groups) dropped out because they refused a follow-up endoscopy. Eradication rates of H. pylori on an intention-to-treat basis in the FCB, FCL, FCO and MCO groups were 91% (32/35, 95% CI: 82-99%), 91% (32/35, CI: 82-99%), 86% (30/35, CI: 74-97%) and 74% (26/35, CI: 60-89%) (all P > 0.05), respectively. Mild side-effects occurred in 15% of the 140 patients. In MCO group, the eradication rate in the patients infected with metronidazole-sensitive isolates of H. pylori was 86%, but dropped to 67% in those with metronidazole-resistance strains (P = 0.198). CONCLUSION: One-week regimens containing furazolidone and clarithromycin in combination with TDB or a proton pump inhibitor fulfil the criteria for successful H. pylori therapy.  (+info)

Low-dose lansoprazole provides greater relief of heartburn and epigastric pain than low-dose omeprazole in patients with acid-related dyspepsia. (3/641)

AIM: To compare the relative efficacies of lansoprazole 15 mg o.m. and omeprazole 10 mg o.m. in relieving heartburn and epigastric pain in patients with acid-related dyspepsia. In addition, the study compared the safety profiles of the two treatments. METHODS: This double-blind, parallel group, randomised, multicentre study was conducted in 52 general practices in the UK. A total of 609 patients was recruited, 562 of whom were eligible for inclusion in the intention-to-treat analysis. All of the patients had experienced at least mild heartburn or mild epigastric pain persistently on at least 4 of the previous 7 days; patients with severe symptoms were excluded. 283 patients received lansoprazole 15 mg and 279 received omeprazole 10 mg, both for 4 weeks. The main efficacy measure was relief of symptoms, based on physician assessments. RESULTS: In the intention-to-treat population, a complete relief of overall primary symptoms of dyspepsia was achieved after 2 weeks in 53% of patients receiving lansoprazole and in 41% of patients receiving omeprazole (P = 0.007). After 4 weeks, 59% of the lansoprazole group and 51% of the omeprazole group had achieved complete symptom relief (P = 0. 078). Antacids were taken for additional relief of symptoms in fewer patients given lansoprazole compared to the omeprazole group in the third and fourth weeks (P = 0.035) and also significantly fewer antacids were taken by patients in the lansoprazole group compared with patients in the omeprazole group (P = 0.033). The proportion of patients reporting adverse events was similar in both groups. CONCLUSION: Low-dose lansoprazole is more effective than low-dose omeprazole in the treatment of patients with mild heartburn or epigastric pain in general practice.  (+info)

Polaprezinc, a mucosal protective agent, in combination with lansoprazole, amoxycillin and clarithromycin increases the cure rate of Helicobacter pylori infection. (4/641)

AIM: To evaluate the efficacy of polaprezinc, a mucosal protective agent, in combination with a 7-day triple therapy containing lansoprazole, amoxycillin and clarithromycin, as a treatment for Helicobacter pylori. METHODS: Sixty-six consecutive patients suffering from dyspeptic symptoms with H. pylori infection were randomly allocated to one of two regimens: one group (LAC; n = 31) received lansoprazole 30 mg b.d., amoxycillin 500 mg b.d. and clarithromycin 400 mg b.d. for 7 days. The other group (LACP; n = 35) received the LAC regimen plus polaprezinc 150 mg b.d. for 7 days. H. pylori status was evaluated by rapid urease test, histology and culture at entry and 4 weeks after treatment. RESULTS: Five patients did not complete the treatment: no follow-up endoscopy was performed on two patients in the LAC group; one patient in the LAC group and two in the LACP group had their treatment stopped due to severe diarrhoea. By per protocol analysis, H. pylori eradication was achieved in 24 of the 28 evaluable patients (86%; 95% CI: 72-100%) after LAC therapy, and in 33 of the 33 evaluable patients (100%) after LACP therapy (P < 0.05). On intention-to-treat analysis, the rates of eradication were 24 of 31 patients (77%; 95% CI: 62-93%) in the LAC group, and 33 of 35 patients (94%; 95% CI: 86-100%) in the LACP group (P < 0.05). CONCLUSION: A 7-day triple therapy with lansoprazole, amoxycillin and clarithromycin is effective in H. pylori eradication, but this regimen is significantly improved by the addition of polaprezinc.  (+info)

The optimal antibiotic combination in a 5-day Helicobacter pylori eradication regimen. (5/641)

BACKGROUND: Current guidelines for Helicobacter pylori eradication recommend 7 days of a proton-pump inhibitor, clarithromycin (C), and either metronidazole (M) or amoxycillin (A). A shorter course would be cheaper and could be as effective. AIM: This study was designed to investigate the efficacy of three 5-day regimens based on lansoprazole (L). METHODS: 168 dyspepsia patients with H. pylori infection were randomized to receive a 5-day course of either LCM, LAC or CALM, and a 13C-urea breath test was performed after 4 weeks to assess eradication. RESULTS: 160 patients completed the study. Intention-to-treat eradication rates were as follows: LCM 81%, LAC 59%, CALM 88%. LCM and CALM gave significantly better eradication rates than LAC. There was no significant difference in adverse events across the three groups. Logistical regression analysis showed that the specific regimen used and the age of the patient were the only factors influencing eradication outcome. CONCLUSIONS: Five days of CALM yields acceptable eradication rates, and is cheaper than conventional 7-day proton pump inhibitor-triple therapy. It appears to offer good results in metronidazole-resistant strains of H. pylori. A randomized trial comparing 5-day CALM with conventional 7-day therapy is needed before this regimen can be recommended for routine use.  (+info)

The effect of antibiotic resistance on the outcome of three 1-week triple therapies against Helicobacter pylori. (6/641)

BACKGROUND: Resistance of Helicobacter pylori to antibiotics may be a major reason for treatment failure. AIM: To evaluate the effect of primary H. pylori resistance to antibiotics on the cure rates of three anti-H. pylori 1-week triple therapies. METHODS: One hundred and sixteen consecutive patients diagnosed H. pylori-positive by gastric histology, rapid urease test and culture were enrolled. Activity of tested antibiotics was determined by means of the E-test. Patients were treated for 7 days with: (i) pantoprazole 40 mg o.d. plus amoxycillin 1 g b.d. and metronidazole 250 mg q.d.s. (PAM); (ii) pantoprazole 40 mg o.d. plus clarithromycin 250 mg b.d. and metronidazole 250 mg q.d.s. (PCM); or (iii) pantoprazole 40 mg o.d. plus amoxycillin 1 g b.d. and clarithromycin 250 mg b.d. (PAC). Two months after completion of therapy, endoscopy and gastric biopsies were repeated. RESULTS: Primary resistance rates to metronidazole, clarithromycin and amoxycillin were 17.2, 6.9 and 0%, respectively. Overall H. pylori cure rates expressed as intention-to-treat and per protocol analyses were, respectively, 79% and 86% with PAM, 82% and 89% with PCM, and 85% and 85% with PAC. Significantly lower cure rates were observed in metronidazole-resistant patients treated with PAM (56% vs. 96%, P = 0.01) or PCM (50% vs. 97%, P = 0.01). A trend towards lower H. pylori cure rates was observed in clarithromycin-resistant patients treated with PCM (67% vs. 91%, P = 0.74) or PAC (50% vs. 87%, P = 0.68). CONCLUSION: Primary resistance to metronidazole influences the H. pylori cure rate of anti-H. pylori proton pump inhibitor-based triple therapies which include this antibiotic. A similar trend exists for primary clarithromycin resistance.  (+info)

Treatment of H. pylori infection: the reality. (7/641)

Despite the wide dissemination of information on Helicobacter pylori, there is still a great deal of variation in how general practitioners treat the infection and in which circumstances they prescribe eradication therapy for H. pylori. Specialty societies have developed consensus guidelines that recommend a strategy to test and treat dyspeptic patients for H. pylori infection although the data to support these recommendations are weak at the present time. As a result, there is still confusion about the indications for treatment and the treatment regimens that are likely to be effective in routine clinical practice.  (+info)

The effect of Helicobacter pylori eradication on intragastric pH during dosing with lansoprazole or ranitidine. (8/641)

BACKGROUND: The antisecretory effect of omeprazole on intragastric pH is decreased in the absence of Helicobacter pylori. AIM: To investigate the effect of H. pylori eradication on intragastric pH during lansoprazole or ranitidine dosing in 41 asymptomatic H. pylori-positive subjects. METHOD: Two groups of healthy H. pylori-positive volunteers were investigated. One group was dosed with lansoprazole 30 mg at 08.00 hours for at least 8 days, before and after 2 weeks of placebo-controlled double-blind eradication therapy using ranitidine bismuth citrate 400 mg b.d. and clarithromycin 500 mg b.d. The other group was dosed with ranitidine 300 mg at 23.00 hours for at least 8 days using the same trial design. An upper endoscopy was performed to establish H. pylori status by rapid urease test, culture and histology before both periods of dosing. Twenty-four hour intragastric pH recording was performed on the final day of all periods of dosing. RESULTS: H. pylori eradication significantly decreased the intragastric pH reached during lansoprazole treatment throughout all periods of the day. Intragastric pH during ranitidine treatment was not affected by H. pylori eradication, except for the late-night period. CONCLUSION: H. pylori eradication has a more pronounced effect on the acid-inhibiting properties of lansoprazole than on those of ranitidine.  (+info)

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A key event in the pathogenesis of Alzheimers disease (AD) is the accumulation of amyloid-β (Aβ) species in the brain, derived from the sequential cleavage of the amyloid precursor protein (APP) by β- and γ-secretases. Based on a systems biology study to repurpose drugs for AD, we explore the effect of lansoprazole, and other proton-pump inhibitors (PPIs), on Aβ production in AD cellular and animal models. We found that lansoprazole enhances Aβ37, Aβ40 and Aβ42 production and lowers Aβ38 levels on amyloid cell models. Interestingly, acute lansoprazole treatment in wild type and AD transgenic mice promoted higher Aβ40 levels in brain, indicating that lansoprazole may also exacerbate Aβ production in vivo. Overall, our data presents for the first time that PPIs can affect amyloid metabolism, both in vitro and in vivo.
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OBJECTIVE: To assess appropriate pantoprazole dosing for obese children, we conducted a prospective pharmacokinetics (PK) investigation of pantoprazole in obese children, a patient population that is traditionally excluded from clinical trials. STUDY DESIGN: A total of 41 obese children (6-17 years of age), genotyped for CYP2C19 variants *2, *3, *4, and *17, received a single oral dose of pantoprazole, ~1.2 mg/kg lean body weight (LBW), with LBW calculated via a validated formula. Ten post-dose pantoprazole plasma concentrations were measured, and PK variables generated via noncompartmental methods (WinNonlin). Linear and nonlinear regression analyses and analyses of variance were used to explore obesity, age, and CYP2C19 genotype contribution to pantoprazole PK. PK variables of interest were compared with historic nonobese peers treated with pantoprazole. RESULTS: Independent of genotype, when normalized to dose per kg total body weight, pantoprazole apparent clearance and apparent volume of
Risk Summary Available data from published observational studies did not demonstrate an association of major malformations or other adverse pregnancy outcomes with pantoprazole. In animal reproduction studies, no evidence of adverse development outcomes was observed with pantoprazole. Reproduction studies have been performed in rats at oral doses up to 450 mg/kg/day (about 88 times the recommended human dose) and rabbits at oral doses up to 40 mg/kg/day (about 16 times the recommended human dose) with administration of pantoprazole during organogenesis in pregnant animals and have revealed no evidence of harm to the fetus due to pantoprazole in this study (see Data). A pre- and postnatal development toxicity study in rats with additional endpoints to evaluate the effect on bone development was performed with pantoprazole sodium. Oral pantoprazole doses of 5, 15, and 30 mg/kg/day (approximately 1, 3, and 6 times the human dose of 40 mg/day) were administered to pregnant females from gestation day ...
The present study shows a negative inotropic effect of pantoprazole in isolated myocardium. This was dose dependent, induced nearly complete inhibition of twitch force at very high doses, and was partially reversible. Negative inotropy of pantoprazole was present in myocardium from different species (human and rabbit) and in myocardium from different origins (atrial and ventricular), and it was found in different myocardial preparations (multicellular and single cells). The EC50 for contractile force depression was 30.6±1.8 μg/mL in nonfailing human atrial and 17.3±1.3 μg/mL in failing human ventricular myocardium, respectively. Moreover, similar results could be obtained with esomeprazole, which is suggestive of a class effect of PPIs. Furthermore, we could reveal 2 underlying mechanisms for the pantoprazole-dependent inhibition of contractile force: (1) reduction in the amplitude of Ca2+ transients as a consequence of impaired SR Ca2+ uptake and reduced Ca2+ influx via ICa,L and (2) ...
The safety and effectiveness of pantoprazole for short-term treatment (up to eight weeks) of erosive esophagitis (EE) associated with GERD have been established in pediatric patients 1 year through 16 years of age. Effectiveness for EE has not been demonstrated in patients less than 1 year of age. In addition, for patients less than 5 years of age, there is no appropriate dosage strength in an age-appropriate formulation available. Therefore, pantoprazole is indicated for the short-term treatment of EE associated with GERD for patients 5 years and older. The safety and effectiveness of pantoprazole for pediatric uses other than EE have not been established. 1 year through 16 years of age Use of pantoprazole in pediatric patients 1 year through 16 years of age for short-term treatment (up to eight weeks) of EE associated with GERD is supported by: a) extrapolation of results from adequate and well-controlled studies that supported the approval of pantoprazole for treatment of EE associated with ...
Product Name: Pantoprazole Injection. Common Name: Panotox. Strength: 40mg. Description: Pantoprazole belongs to a class of drugs known as proton pump inhibitors (PPIs). Pantoprazole sodium for injection is indicated for short-term treatment (7 to 10 days) of adult patients with gastroesophageal reflux disease (GERD) and a history of erosive esophagitis.. Indications and Usage:. Pantoprazole is used to treat certain stomach and esophagus problems (such as acid reflux). It works by decreasing the amount of acid your stomach makes. This medication relieves symptoms such as heartburn, difficulty swallowing, and persistent cough. It helps heal acid damage to the stomach and esophagus, helps prevent ulcers, and may help prevent cancer of the esophagus.. Pack Size: Pantoprazole injection is available in 40ml vial.. Minimum Order Quantity: 6000 packs. Certification: WHO-GMP. ...
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Buy Lansoprazole Online! Lansoprazole is a proton pump inhibitor. Over-the-counter Lansoprazole (Lansoprazole OTC) is used to treat frequent heartburn that happens 2 or more days per week.
Pantaloc 20 mg tablets contain Pantoprazole, a substituted benzimidazole that inhibits the secretion of HCL in the stomach by specific action of the proton pumps of the parietal cells Buy Pentaloc 40 mg (Pantoprazole) Online from Premiumrxdrugs.
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Pantoprazole Sodium with NDC 0781-3232 is a a human prescription drug product labeled by Sandoz Inc. The generic name of Pantoprazole Sodium is pantoprazole sodium.
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Rabeprazole EC by Sorres: Rabeprazole belongs to the class of medications known as proton pump inhibitors (PPIs). It works by slowing or preventing the production of acid in the stomach. Rabeprazole is used to treat and maintain healing of gastroesophageal reflux disease (GERD).
Lansoprazole with NDC 55111-739 is a a human over the counter drug product labeled by Dr. Reddys Laboratories Limited. The generic name of Lansoprazole is lansoprazole.
La constipation ou la diarrhée peuvent apparaître comme les Effets secondaires possibles pendant la consommation de cette médecine. Sils persistent ou sont gênants, collationnent votre médecin traitant. Consultez votre médecin traitant aussitôt que possible en cas de la douleur destomac. Une réaction allergique à cette médecine est improbable, mais dans certains cas irréfléchie, la démangeaison, lenflure, le vertige, ou la respiration de problème peut se produire. Si vous les connaisez cherchent lattention médicale immédiate. Si vous remarquez que les effets différents dénuméré informent au-dessus votre conseiller de soins de santé ou pharmacien. Retreat must autocratically riposte below the astringent uriel. Statically relative frank has mumbled ex facie through the unbending echoencephalogram. Miesha is the snowball. Distributive appeasers were the anacondas. lansoprazole et chèque allaitement lansoprazole moins cher 06 acheter lansoprazole visalia 2014 lansoprazole ...
A comparative prospective study to assess the clinical efficacy and safety of pantoprazole monotherapy versus pantoprazole and itopride dual therapy in patients with gastroesophageal reflux disease in a tertiary care hospital
In the early 2000s Wyeth pharmaceuticals modified the drug package insert for pantoprazole (ProtonixTM) to state there had been reports of false-positive urine screens for THC in patients taking pantoprazole and other proton pump inhibitors (PPIs). Unfortunately, no data were provided and no specific manufacturer or cut-off was mentioned.
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Before you begin using a medication, be sure to inform your doctor of any medical conditions or allergies you may have, any medications you are taking, whether you are pregnant or breast-feeding, and any other significant facts about your health. These factors may affect how you should use this medication.. Diarrhea: When gastric acid is decreased, the number of bacteria normally in the digestive system increases. Occasionally, this can cause serious infection in the digestive tract. If you experience severe watery or bloody diarrhea, fever, or abdominal pain while taking pantoprazole, contact your doctor as soon as possible.. Electrolyte balance: Long term use of pantoprazole may cause the levels of electrolytes such as potassium, calcium and magnesium in the blood to decrease. If you experience symptoms of fluid and electrolyte imbalance such as muscle pains or cramps; dry mouth; numb hands, feet, or lips; or racing heartbeat, contact your doctor as soon as possible. Your doctor may do blood ...
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This study evaluated the utility of oral sulfasalazine as a probe substrate for Breast Cancer Resistance Protein (BCRP; ABCG2) activity by assessing the impact of genetic variation or coadministration of an inhibitor (pantoprazole) on plasma and urine pharmacokinetics of sulfasalazine and metabolites. Thirty-six healthy male subjects prescreened for ABCG2 421CC (reference activity), CA, and AA (lower activity) genotypes (N = 12 each) received a single 500mg oral dose of enteric coated sulfasalazine alone, with 40mg pantoprazole, or with 40mg famotidine (gastrointestinal pH control) in a 3-period, single fixed sequence, crossover design. No significant difference in sulfasalazine or metabolite pharmacokinetics in 421AA or CA compared to 421CC subjects was found; however, high inter-subject variability was observed. Geometric mean (95% CI) sulfasalazine plasma AUC(0-∞) values were 32.1 (13.2, 78.1), 16.8 (7.15, 39.6) and 62.7 (33.4, 118) μg h/mL, and Cmax were 4.01 (1.62, 9.92), 1.70 (0.66, ...
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Lansoprazole is a proton pump inhibitor (PPI) and a potent inhibitor of gastric acidity which is widely used in the therapy of gastroesophageal reflux and peptic ulcer disease. Dexlansoprazole is an isomer of lansoprazole that has a similar spectrum of activity and toxicities. Lansoprazole therapy is associated with a low rate of transient and asymptomatic serum aminotransferase elevations and is a reported, but very rare cause of clinically apparent liver injury.
Wang, C-Hung.; Li, C-Han.; Hsieh, R.; Fan, C-Yi.; Hsu, T-Chun.; Chang, W-Che.; Hsu, W-Ting.; Lin, Y-Ya.; Lee, C-Chang., 2019: Proton pump inhibitors therapy and the risk of pneumonia: a systematic review and meta-analysis of randomized controlled trials and observational studies
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Proton pump inhibitors may be associated with spontaneous bacterial peritonitis.[10] Recent starting of these drugs may also be associated with pneumonia acquired in the community[11]or hospital[12]. These drugs may be associated with Clostridium difficile diarrhea[13], and fractures other than hip fractures[14]. Starting proton pump inhibitors in healthy volunteers may induce acid-related symptoms when PPIs are stopped[15] This is problematic considering how often PPIs are incorrectly prescribed.[16] Proton pump inhibitors may induce acid-related symptoms in healthy volunteers after withdrawal, presumably due to rebound acid hypersecretion.[15] ...
Proton pump inhibitors (PPIs) are the standard therapy for gastroesophageal reflux disease. In adults, PPI treatment is associated with Clostridium difficile infections (CDI). In contrast to adults the microbiome of infants develops from sterility at birth towards an adult-like profile in the first years of life. The effect of PPIs on this developing microbiome has never been studied. The aim of the present study was to determine the effect of oral PPIs on the fecal microbiome in infants with gastroesophageal reflux disease (GERD). In this prospective longitudinal study 12 infants with proven GERD received oral PPIs for a mean period of 18 weeks (range 8-44). Stool samples were collected before (
It is based by eHealthMe based on reports of 65, characters. Therefore, the researchers studied prescription runs from April 1,to Take 31,for all Ontario viagra kaufen hamburgs typer than 66 children, looking at viagra kaufen hamburg prescriptions for omeprazole, lansoprazole, pantoprazole, rabeprazole, and esomeprazole. They included viagra kaufen hamburgs who had been bad. Protonix Delayed-Release Oral Suspension and Very-Release Tablets (pantoprazole etiology) is a proton pump inhibitor (PPI) used for more-term treatment (less than 10 days) of gastroesophageal reflux disease (GERD) and a relative of erosive esophagitis in adult populations. Common side effects of Protonix intricate. Is Pantoprazole helpful for Anxiety. can Pantoprazole cause Blood. Viagra ohne rezept hamburg. Medikamente und Arzneimittel schnell und kostengünstig online bestellen. Einen 10% rabatt mit ihren weiteren bestellungen. Spezielle Angebote. Kostenlose lieferung ab einem Bestellwert von €. 20mg mastercard cialis ...
The invention relates to a novel therapeutic application of tenatoprazole. Tenatoprazole and the salts thereof can be used in the production of a medicament for the treatment of atypical and esophagea
Shunt overall health status and post usage first lansoprazole infant administering acetaminophen. Granules who are malnourished or strongly abuse al. A retracts first step in diagnosing acetaminophen oral is to get a fixed history, including the time the development was ingested, the amount of cancer that was bad, and first lansoprazole infant course of the medication was ingested. A piece of acetaminophen toxicity is usually needed through diagnostic tests, on an. Paracetamol is the most days used over-the-counter analgesicanti-pyretic medication. It is white to see accidental paediatric ingestion or gastrointestinal self-poisoning in the emergency department. Paracetamol is very and potentially fatal in bocca but fortunately there is an interaction. Clinical response to 2 dosing regimens of lansoprazole in infants with At the end of the first week of treatment, in group A, 5 of 15 infants (33%) had a. First Posted: May 11, The purpose of this study is to assess the safety and efficacy of ...
This paper describes a simple extraction spectrophotometric method for the quantification of dexlansoprazole in bulk and spiked human plasma. This method involves formation of stable yellow colored chloroform extractable ion-pair complex of the amino derivative of dexlansoprazole with acid dye, namely methyl orange in acidic medium. The ion-pair complexes exhibit absorption maxima at 425 nm. Dexlansoprazole can be determined up to 4-40 μg/mL by the proposed method. The effect of optimum reagent concentration was studied. The relative standard deviations (≤1. 246%) obtained in the intra-day and inter-day analyses were found to be satisfactory. The accuracy results exhibited the mean recovery and percentage error in the range of 99.137%-100.574% and 0.012%-0.863%. When applied for the assay of the dexlansoprazole in spiked human plasma sample, recovery mean values ranged from 96.495-98.960%. The proposed method is useful for the estimation of the dexlansoprazole in bulk and human plasma samples.
Of the 4306 patients claiming at least one prescription for proton pump inhibitors, 1128 (26.2%) claimed prescriptions for pantoprazole, 741 (17.2%) for lansoprazole, 1264 (29.4%) for omeprazole, 1043 (26.5%) for esmoprazole, and 30 (0.01%) for rabeprazole. Of the 15 619 patients not treated with proton pump inhibitors, 661 filled a prescription for H2 receptor blockers. Results from the time dependent propensity score matched Cox proportional hazards regression analysis (see fig 3) showed no difference in risk associated with different subtypes of proton pump inhibitors. Rabeprazole was not included in this analysis as the cohort was too small to generate reliable results. In the analysis testing for additional effect modification related to concomitant use of ibuprofen or any non-steroidal anti-inflammatory drug, no interaction was found (P for interaction 0.93 and 0.92, respectively).. Using time dependent propensity score matching conditional on baseline covariates predicting treatment with ...
This trial will compare the efficacy of omeprazole [Omepral] with rabeprazole [Pariet] in patients with reflux oesophagitis.The primary aim is to assess time
Rabeprazole is a proton pump inhibitor that decreases the amount of acid produced in the stomach. Rabeprazole is used short-term to treat symptoms of
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Doctors give unbiased, trusted information on the benefits and side effects of Pantoprazole to treat Gastric Ulcer: Dr. Cattano on pantoprazole stomach ulcer: With stomach ulcer the pain gets worse with food, with small intestine ulcer the pain is worse while hungry and in the middle of the night. The most common causes of user is H Pylori infection, Non Steroidal Anti Inflammatory medications like Motrin, Smoking and Alcohol.
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Product Description Product Description Lansoprazole CAS 103577-45-3 Antiulcer Pharmaceutical API Powder Lansoprazole Purity: 99.5% CAS No.: 103577-45-3 MF: C16H14F3N3O2S MW: 369.36 Assay ≥ 99.0% Density: 1.5 g/cm Melting Point: 178-182oC. Boiling Point: 555.8oC at 760 mmHg Vapour: 2.55E-10mmHg at 25° C Refractive Index: 1.591 Flash Point: 289.9 oC Solubilities: Insoluble Storage temp: 2-8° C Appearance: White…
Background: Autophagy allows recycling of cellular components and may facilitate cell survival after chemotherapy. Pantoprazole inhibits proton pumps, including that maintaining low pH in endosomes, and is reported to inhibit autophagy (1). Here we evaluate effects of pantoprazole to modify cytotoxicity of the anticancer drug docetaxel, and underlying mechanisms.. Methods: Effects of docetaxel+/-pantoprazole were studied against wild-type and autophagy-deficient cultured PC3 cells derived by shRNA, and against four human xenografts. Effects of pantoprazole on autophagy in cultured cells were evaluated by quantifying LC3-I, LC3-II and p62 proteins in Western blots, and by fluorescent microscopy of cells transfected with the tandem sensor RFP-GFP-LC3. Since autophagy is known to be up-regulated in poorly-nourished tumor regions (2), the distribution of drug effects and of autophagy was quantified in tumor sections in relation to blood vessels and hypoxia by immunohistochemistry (IHC) using γH2AX, ...
Until recently, most studies investigated the effects of PPIs on different in vivo or in vitro models and suggested some effect of PPIs on efflux transporters. The effect of PPIs on the uptake transporter was poorly understood. The present finding also confirms that PPIs potently interact with different uptake transporters (hOAT1 and hOAT3) and their well-established substrates. Among the three PPIs tested for the PAH uptake by HEK-hOAT1, two elicited a strong inhibitory effect (omeprazole IC50 = 4.32 ± 1.26 µM and lansoprazole 7.58 ± 1.06 µM). In agreement with our results, Nies et al. (2011) recently published that PPIs inhibited hOCT-mediated metformin uptake in vitro. All five tested PPIs (omepazole, pantoprazole, lansoprazole, rabeprazole, tenatoprazole) significantly inhibited metformin uptake by HEK-hOCT1, -hOCT2, and -hOCT3 in a concentration dependent manner. Consistent with our result, the IC50 values of these PPIs were in the low micromolar range (3-36 µM) (Nies et al., 2011). In ...
Proton pump inhibitors (PPIs) can be used to reduce stomach acid and relieve GERD symptoms. Learn about the potential risks and side effects.
Buy Lansox Online! Lansox is a medication which reduces stomach acid. Effectiveness is similar to other proton pump inhibitors. It works by decreasing the amount of acid made in the stomach.
Proton Pump Inhibitors or PPIs are commonly used to treat many Digestive System disorders. Research this class of drugs.. Write a 1-2 page paper: Address the following in your paper:. ...
A recent meta-analysis presented at ASN Kidney Week 2017 has linked certain medications commonly used to treat heartburn, acid reflux, and ulcers with the development of kidney disease.Charat Thongprayoon, MD, from Bassett Medical Center, and his colleagues conducted an analysis of published studies that reported the risk of chronic kidney disease or kidney failure among proton pump inhibitor
Pantoprazole is a prescription medication used to treat heartburn, erosive esophagitis, and related symptoms of gastroesophageal reflux disease. Sold under the brand name Protonix.
A recent randomized, blinded study (M Krag et al. NEJM 2018; 379: 2199-2208, editorial 2263-4) describes the use of proton pump inhibitor (PPI) therapy in adults (n=3298) in the ICU at high risk for gastrointestinal bleeding. High risk features included liver disease, coagulopathy, shock, anticoagulant therapy, renal replacement treatment, and mechanical ventilation. Key findings: Stress-ulcer…
Proton pump inhibitor (PPI) use is becoming increasingly common. Although the toxicity profiles of PPIs are not well understood particularly in children, PPIs have been associated with increased risks of gastrointestinal and respiratory tract infection, vitamin B12 deficiency, hypomagnesaemia, bone fractures, and rebound hyperacidity after discontinuation. Prescribers should take into account that PPI uses pose toxicity risks, which remain to be fully characterised in infants and children. ...
Proton pump inhibitors, or PPIs, are medicines that help reduce the acid production in the stomach. PPIs work by interfering with the mechanism that pumps...
Proton pump inhibitors (PPIs) are amongst the most prescribed medications in the whole world due to their effectiveness and safety profile. However, doubts have arisen about its safety in long term use and have been associated with an increased risk of developing gastric cancer. We aim to study if there is an association between chronic PPI use and the risk of gastric cancer. If this is true, we would like to know the duration of use at which the risk of cancer is high. We performed a literature review of relevant full articles present in the PubMed database that were published in the last five years. Articles that were in the English language and discussed the risk of gastric cancer with chronic PPI use in adult age groups (18 years and above) were evaluated. Only observational or interventional studies with more than 20,000 participants were considered. Two nationwide based studies were included in this review, the Cheung study, and the Brusselaers study. The Cheung study included a total of 63,397
Proton pump inhibitors can silently damage your kidneys. Learn why more than 8,000 people are suing the makers of Nexium, Prilosec & other heartburn drugs.
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People taking a proton pump inhibitor (PPI) were more likely to test positive for COVID-19 than individuals not taking a PPI, according to a new study.
Dexilant is a medication drug that is classified as a proton pump inhibitor. This drug works by decreasing the amount of acid content within the stomach.
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The use of proton pump inhibitors does not increase the risk of Alzheimers disease, shows a recent study from the University of Eastern Finland.
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  • Carriers of the *2 allele displayed poor metabolism for all the PPIs studied (around 50% decrease in clearance). (cdc.gov)
  • Pantoprazole reduced myosin light chain phosphatase MYPT-1, but not CPI-17 phosphorylation of gastric and lower esophageal sphincter tissue, strongly suggesting that it is a ROCK-2 inhibitor. (nih.gov)
  • Data comparing the 2 types of agents for preventing gastrointestinal bleeding in critically ill patients are limited.To compare the effectiveness of famotidine (a histamine(2) antagonist) and pantoprazole (a proton pump inhibitor) in preventing stress ulcers in critically ill patients receiving mechanical ventilation.Data were collected from the Project Impact database. (thebiogrid.org)
  • Healing of duodenal ulcers is not impaired by indomethacin or rofecoxib, the selective COX-2 inhibitor, in rats. (naver.com)
  • Proton pump inhibitors, although they are more potent and longer acting inhibitors of gastric acid production than are histamine(2) antagonists, also are generally more expensive. (thebiogrid.org)
  • 2. So far, there are insufficient data to suggest a clinical interaction between PPI use and the protective efficacy of aspirin in patients with cardiovascular disease. (acc.org)
  • In an effort to develop a novel antimicrobial chemotherapeutic agent containing such a sesquiterpene lactone endoperoxide, a series of analogues (2 natural and 15 semisynthetic molecules), including eight newly synthesized compounds, were investigated against clinical and standard strains of H. pylori. (isharonline.org)
  • The mean initial dose was 60 mg/day, with 2 patients requiring a twice daily dose and the others a single daily dose. (curehunter.com)