AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPhenomena and Processes
SulfamethoxazoleTrimethoprimTrimethoprim ResistanceTrimethoprim-Sulfamethoxazole CombinationAnti-Bacterial AgentsMicrobial Sensitivity TestsDapsoneAnti-Infective AgentsAnti-Infective Agents, UrinaryFolic Acid AntagonistsSulfadiazineSulfisoxazolePneumonia, PneumocystisDrug Resistance, BacterialIntegronsUrinary Tract InfectionsTetrahydrofolate DehydrogenaseDrug Resistance, Multiple, BacterialDrug Resistance, MicrobialDrug CombinationsDrug HypersensitivityPneumocystis jiroveciiPentamidineEscherichia coliTropherymaEnterobacteriaceaeStenotrophomonas maltophiliaR Factors4-Aminobenzoic AcidEscherichia coli InfectionsShigella sonneiDrug SynergismShigellaConjugation, GeneticNocardia InfectionsDihydropteroate SynthaseWhipple DiseaseCiprofloxacinSulfamethoxypyridazineTetracyclineAmpicillinRifampinSalmonellaPneumocystisSulfonamidesMegaloblastsDNA, BacterialNocardiaNalidixic AcidSalmonella entericaVibrio cholerae O1Gram-Negative Bacterial InfectionsAtovaquoneDysentery, BacillaryEnterobacteriaceae InfectionsFecesElectrophoresis, Gel, Pulsed-FieldHaptensStaphylococcal InfectionsCholeraSalmonella Infections, AnimalBacterial InfectionsCulture MediaPlasmidsSerotypingDisk Diffusion Antimicrobial TestsPenicillin ResistanceStaphylococcus aureusOpportunistic InfectionsDrug EruptionsGenes, BacterialAgarMethicillin-Resistant Staphylococcus aureusUrineAdministration, OralSalmonella InfectionsColony Count, MicrobialCommunity-Acquired InfectionsSwine DiseasesHydroxylaminesGram-Negative BacteriaBacteriaDrug Therapy, CombinationCross Infection
Order Sulfamethoxazole800mgAntibioticAntibioticsTabletsClindamycinSeptraPurchase SulfamethoxazoleGlomerular filtration and tubular secretionSulfonamidePrescriptionIngredients200mgAntimicrobial combinationInfectionsMetaboliteOrallyTabletInfectionFolatePediatric patientsDihydrofolic acidCombinationVitroDrugsProphylaxisMicronucleusGatifloxacinSulfanilamideUrinaryUrineDosageGastrointestinalLeast 5 metabolitesConcentrationsPurchasesDistributesEnzymeTreatmentRapidlySulfonamidesVersusPregnancySerumProtein
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Antibiotic4
Antibiotics7
  • Treatment success rates for empiric therapy were compared among commonly prescribed antibiotics in our clinic: cephalexin, trimethoprim-sulfamethoxazole, and clindamycin. (nih.gov)
  • Antibiotics with activity against community-associated MRSA, such as trimethoprim-sulfamethoxazole and clindamycin, are preferred empiric therapy for outpatients with cellulitis in the community-associated MRSA-prevalent setting. (nih.gov)
  • Using trimethoprim and sulfamethoxazole together enhances the effectiveness of both antibiotics. (merckmanuals.com)
  • Clinicians have begun to use alternative antibiotics, including trimethoprim-sulfamethoxazole (TMP-SMX) and clindamycin, as treatment options in outpatient settings because of their favorable in vitro activity, high oral bioavailability, and excellent tissue penetration. (jabfm.org)
  • Sulfamethoxazole and trimethoprim are both antibiotics that treat different types of infection caused by bacteria. (thetopmedstore.com)
  • Trisul tablets 480mg contain cotrimoxazole, a combination of two antibiotics trimethoprim and sulfamethoxazole that block two consecutive steps in the biosynthetic pathway needed for bacterial DNA and protein synthesis. (inhousepharmacy.vu)
  • Twelve different soil types that represent the soil compartments of the Czech Republic were fortified with three antibiotics (clindamycin (CLI), sulfamethoxazole (SUL), and trimethoprim (TRI)) to investigate their fate. (slu.se)
Tablets5
  • in tablets, each containing 400 mg sulfamethoxazole and 80 mg trimethoprim for oral administration. (nih.gov)
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  • Sulfamethoxazole in Trisul tablets 480mg competes with para-aminobenzoic acid (PABA) which the bacterial cells use to make an important metabolite dihydrofolic acid. (inhousepharmacy.vu)
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Clindamycin3
  • Trimethoprim-sulfamethoxazole (TMP-SMX) and clindamycin are often used as first-line treatment options, but clinical data are lacking. (jabfm.org)
  • Trimethoprim/sulfamethoxazole or clindamycin should be added for this coverage. (medscape.com)
  • Five metabolites (clindamycin sulfoxide (CSO), hydroxy clindamycin sulfoxide (HCSO), S-(SDC) and N-demethyl clindamycin (NDC), N-4-acetyl sulfamethoxazole (N(4)AS), and hydroxy trimethoprim (HTR)) were detected and identified using HPLC/HRMS and HRPS in the soil matrix in this study. (slu.se)
Septra1
  • Trimethoprim-sulfamethoxazole (TMP-SMX) , sold under the trade names Bactrim*, Septra*, and Cotrim*, is the medication of choice for Cystoisospora infection. (cdc.gov)
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Glomerular filtration and tubular secretion2
  • 1 Excretion of sulfamethoxazole and trimethoprim is primarily by the kidneys through both glomerular filtration and tubular secretion. (nih.gov)
  • Excretion Sulfamethoxazole is primarily renally excreted via glomerular filtration and tubular secretion. (wikipedia.org)
Sulfonamide3
  • The average percentage of the dose recovered in urine from 0 to 72 hours after a single oral dose of sulfamethoxazole and trimethoprim is 84.5% for total sulfonamide and 66.8% for free trimethoprim. (nih.gov)
  • Thirty percent of the total sulfonamide is excreted as free sulfamethoxazole, with the remaining as N 4 -acetylated metabolite. (nih.gov)
  • If you have ever had a low platelet count when using trimethoprim or a sulfa (sulfonamide) drug. (aarppharmacy.com)
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Ingredients2
200mg1
  • CO-TRIMOXAZOLE 40mg/200mg - 5ml - Trimethoprim + Sulfamethoxazole is supplied for Tenders/ Emergency imports/ Un - licensed, Specials, Orphan drug/ Name patient line/ RLD supplies/ Reference listed drugs/ Comparator Drug/ Bio-Similar/ Innovator samples For Clinical trials. (gnhindia.com)
Antimicrobial combination2
  • The antimicrobial combination of trimethoprim and sulfamethoxazole is active in vitro against various gram-positive and gram-negative bacteria. (nih.gov)
  • Trimethoprim (TMP)-sulfamethoxazole (SMX) is a widely used synergistic antimicrobial combination to treat a variety of bacterial and certain fungal infections. (umn.edu)
Infections4
  • To reduce the development of drug-resistant bacteria and maintain the effectiveness of sulfamethoxazole and trimethoprim oral suspension and other antibacterial drugs, sulfamethoxazole and trimethoprim oral suspension should be used only to treat or prevent infections that are proven or strongly suspected to be caused by bacteria. (nih.gov)
  • Trimethoprim eliminates bacteria that cause urinary tract infections. (nih.gov)
  • Trimethoprim-sulfamethoxazole by itself or in combination with other antimicrobial agents is indicated for most Nocardia asteroides infections and is the antimicrobial agent of choice for Pneumocystis carinii pneumonia. (nih.gov)
  • trimethoprim (80mg) and sulfamethoxazole (400mg) that are used in combination to treat a range of bacterial infections. (inhousepharmacy.vu)
Metabolite1
  • About 20% of the sulfamethoxazole in urine is the unchanged drug, about 15-20% is the N-glucuronide conjugate, and about 50-70 % is the acetylated metabolite. (wikipedia.org)
Orally1
  • The two compounds studied most extensively for prophylaxis against PCP have been trimethoprim-sulfamethoxazole, given orally, and pentamidine, given as an aerosol. (cdc.gov)
Tablet3
Infection3
  • Trimethoprim-sulfamethoxazole compared with ciprofloxacin for treatment and prophylaxis of Isospora belli and Cyclospora cayetanensis infection in HIV-infected patients: a randomized, controlled trial. (cdc.gov)
  • These combined actions of trimethoprim and sulfamethoxazole block bacterial folate biosynthesis, needed for making nucleic acids and proteins, preventing replication of the bacteria by bacteriostasis, which limits the growth of the bacteria and stops the spread of infection. (inhousepharmacy.vu)
  • Prevalence and Risk Factor Analysis of Trimethoprim-Sulfamethoxazole- and Fluoroquinolone-Resistant Escherichia coli Infection among Emergency Department Patients with Pyelonephritis. (wustl.edu)
Folate1
Pediatric patients1
  • A simulation conducted with data from a pharmacokinetic study in 153 infants and children demonstrated that mean steady state AUC and maximum plasma concentration of trimethoprim and sulfamethoxazole would be comparable between pediatric patients 2 months to 18 years receiving 8/40 (trimethoprim/sulfamethoxazole) mg/kg/day divided every 12 hours and adult patients receiving 320/1600 (trimethoprim/sulfamethoxazole) mg/day. (nih.gov)
Dihydrofolic acid2
  • One of the components, Sulfamethoxazole holds back bacterial synthesis of dihydrofolic acid by competing with para-aminobenzoic acid. (thetopmedstore.com)
  • Trimethoprim works by blocking the tetrahydrofolic acid production from dihydrofolic acid. (thetopmedstore.com)
Combination4
Vitro3
  • Trimethoprim is metabolized in vitro to 11 different metabolites, of which, five are glutathione adducts and six are oxidative metabolites, including the major metabolites, 1- and 3-oxides and the 3- and 4-hydroxy derivatives. (nih.gov)
  • In vitro studies suggest that trimethoprim is a substrate of P-glycoprotein, OCT1 and OCT2, and that sulfamethoxazole is not a substrate of P-glycoprotein. (nih.gov)
  • In vitro studies suggest sulfamethoxazole is not a substrate of the P-glycoprotein transporter. (wikipedia.org)
Drugs2
Prophylaxis2
  • There are limited alternative therapeutic choices to trimethoprim-sulfamethoxazole (TMP-SMX) which is the standard first line therapy/prophylaxis for PcP. (unl.pt)
  • The efficacy of trimethoprim-sulfamethoxazole for prophylaxis against PCP has been clearly demonstrated among pediatric cancer patients (7-8). (cdc.gov)
Micronucleus1
Gatifloxacin1
  • Trimethoprim/sulfamethoxazole (TMP/SMX) resist- gatifloxacin (14.1%) ( 3 ). (cdc.gov)
Sulfanilamide1
  • Sulfamethoxazole, a sulfanilamide, is a structural analog of para-aminobenzoic acid (PABA). (wikipedia.org)
Urinary1
Urine2
  • Urine concentrations of both sulfamethoxazole and trimethoprim are considerably higher than are the concentrations in the blood. (nih.gov)
  • The native compounds and the metabolites of trimethoprim and sulfamethoxazole are excreted primarily in the urine. (nih.gov)
Dosage1
  • When the creatinine clearance is less than 30 ml/min, the dosage of trimethoprim-sulfamethoxazole should be adjusted. (nih.gov)
Gastrointestinal1
  • The most common side effects of sulfamethoxazole are gastrointestinal disturbances (nausea, vomiting, anorexia) and allergic skin reactions (such as rash and urticaria). (wikipedia.org)
Least 5 metabolites2
  • Sulfamethoxazole is metabolized in humans to at least 5 metabolites: the N 4 -acetyl-, N 4 -hydroxy-, 5-methylhydroxy-, N 4 -acetyl-5methylhydroxy- sulfamethoxazole metabolites, and an N-glucuronide conjugate. (nih.gov)
  • Metabolism Sulfamethoxazole is metabolized in the human liver to at least 5 metabolites. (wikipedia.org)
Concentrations1
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Distributes2
  • trimethoprim also distributes to bronchial secretion, and both pass the placental barrier and are excreted in human milk. (nih.gov)
  • Distribution Sulfamethoxazole distributes into most body tissues as well as into sputum, vaginal fluid, and middle ear fluid. (wikipedia.org)
Enzyme1
Treatment2
Rapidly1
Sulfonamides1
Versus1
  • A controlled trial of trimethoprim-sulfamethoxazole versus trimethoprim-dapsone. (nih.gov)
Pregnancy1
  • Trimethoprim-sulfamethoxazole (TMP-SMX) is in pregnancy category C. TMP-SMX should be used during pregnancy only if the potential benefit justifies the potential risk to the fetus. (cdc.gov)
Serum3
Protein1