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  • serotype
  • The results showed that a new serotype, combined with resistance to beta-lactams, could emerge in a susceptible strain via a single transformation event. (harvard.edu)
  • Ceftriaxone
  • Antimicrobial susceptibility testing was performed and interpreted according to Clinical Laboratory Standards Institute (CLSI) criteria using the disk diffusion methodology against penicillin, ceftriaxone, tetracycline and ofloxacin. (hindawi.com)
  • Ceftriaxone resistance was not observed. (hindawi.com)
  • Using Clinical and Laboratory Standards Institute (CLSI) breakpoints, the correlation between the zones of inhibition of the ODD test and the MICs of penicillin and ceftriaxone was determined. (vanopijnenlab.com)
  • Conclusions There was good correlation between ODD zones of inhibition and penicillin meningeal breakpoints but weak correlation between the ODD results and non-meningeal breakpoints for both penicillin and ceftriaxone. (vanopijnenlab.com)
  • binding protein
  • Carapito R, Chesnel L, Vernet T et al (2006) Pneumococcal β-lactam resistance due to a conformational change in penicillin-binding protein 2x. (springer.com)
  • Marrec-Fairley M, Piette A, Gallet X et al (2000) Differential functionalities of amphiphilic peptide segments of the cell-septation penicillin-binding protein 3 of Escherichia coli . (springer.com)
  • The B-lactam ring stops PBP (penicillin-binding protein) from creating an amino acid bridge connecting the walls, making the cell resistant to osmotic pressure. (prezi.com)
  • Witherden, EA 2014 , 'Phenotypic and genotypic characterisation of altered penicillin-binding protein 3 (PBP3) mediated resistance in Haemophilus influenzae and Haemophilus haemolyticus', PhD thesis, University of Tasmania. (edu.au)
  • inhibition
  • Comparison of the ODD test zones of inhibition and the penicillin MICs, using the CLSI meningeal breakpoints, showed good correlation (Cohen's kappa coefficient = 0.8239). (vanopijnenlab.com)
  • locus
  • Resistance to these agents is most commonly due to a single base pair deletion in the regulatory region of the mtrCDE locus that results in increased expression of the energy-dependent Mtr efflux pump. (asm.org)