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OxygenOxygen ConsumptionCoronary CirculationMyocardiumDogsHemodynamicsHeartReactive Oxygen SpeciesHeart RateMyocardial ContractionCoronary VesselsCardiac OutputDobutamineBlood PressureVascular ResistanceEnergy MetabolismCardiotonic AgentsLactatesPartial PressureLactic AcidVentricular Function, LeftPrenylamineIsothiuroniumCardiac Pacing, ArtificialMyocardial IschemiaPhysical ExertionOxygen Inhalation TherapyAdenosinePropranololSinglet OxygenFelodipineVentricular FunctionHydrazonesCarbon RadioisotopesCoronary DiseaseCounterpulsationMyocardial StunningAngina PectorisTomography, Emission-ComputedBlood VolumeExtracorporeal CirculationStroke VolumeHyperemiaDipyridamoleHeart VentriclesNitroglycerinAdrenergic beta-AntagonistsAcetatesCarbon DioxideMitochondria, HeartHeart Arrest, InducedNitric OxidePyridazinesVasomotor SystemPerfusionVasodilator AgentsHalothaneBlood Flow VelocityRegional Blood FlowAmrinoneVasodilationSwineBlood Gas AnalysisVeratrinePhosphocreatineVentricular PressureNorepinephrineExercise TestCalcium ChlorideCardiac VolumeModels, CardiovascularMyocardial ReperfusionHeart FailureAnesthesiaIsoproterenolElectrocardiographyOxygen IsotopesPurinergic P1 Receptor AntagonistsDepression, ChemicalDiastoleGlucoseCardiomyopathy, DilatedOxidation-ReductionFentanylMyocardial Reperfusion InjuryNifedipineTime FactorsCardiac CatheterizationDose-Response Relationship, DrugIsofluraneVasoconstrictionPhysical Conditioning, AnimalEchocardiographyAnesthesia, GeneralAdrenergic beta-AgonistsVeinsTheophyllineBradykininCitric Acid CycleMyocardial Infarction
InfarctionIschemiaCardiacReactive OxygenConsumptionChest PainPerfusionIncreasesDysfunctionFree RadicalsCardiogenicPatientsReperfusion InjurySymptomsCoronary arteryHyperbaric oxygenHeartTherapyMortalityAortic pressureCerebralInfarctionAcuteSTEMIDeprivationChanges in myocardialReperfusionConsumptionUtilizationProduction of reactive oxygenReactiveCarbon dioxideTissueHeartArterialIschaemiaFibresCardiovascularHypoxiaMitochondrialMyocardiumBiopsyExertionViability2016Benefit of supplementalMetabolismAntioxidantBloodExperimentalVascularInhalationIschaemicOxidative
Infarction56
Ischemia23
  • One systematic review found no benefit from oxygen therapy in the management of myocardial ischemia ( Nicholson 2004 ). (wustl.edu)
  • While effective early reperfusion of the criminal coronary artery after a confirmed AMI is the typical treatment at present, collateral myocardial ischemia-reperfusion injury (MIRI) and pertinent cardioprotection are still challenging to address and have inadequately understood mechanisms. (hindawi.com)
  • This coupled comorbidity of pathological ischemia and therapeutic reinjury of infarcted myocardium, namely, myocardial ischemia-reperfusion injury (MIRI), is particularly refractory to treatment [ 4 , 5 ]. (hindawi.com)
  • Conceptual diagram of the development and unknown mechanisms of myocardial ischemia-reperfusion injury. (hindawi.com)
  • The pathophysiological nature of MIRI is the short-term disturbance of myocardial energy and metabolism caused by reflow after ischemia and hypoxia in the coronary artery and the dynamic changes in apoptosis and the prosurvival signaling pathways in response to related injury factors. (hindawi.com)
  • Acute treatment with troglitazone increases susceptibility to ventricular fibrillation during myocardial ischemia and reperfusion. (diabetesjournals.org)
  • Myocardial ischemia occurs when blood flow to your heart is reduced, preventing the heart muscle from receiving enough oxygen. (sparrow.org)
  • Myocardial ischemia, also called cardiac ischemia, reduces the heart muscle's ability to pump blood. (sparrow.org)
  • Myocardial ischemia might also cause serious abnormal heart rhythms. (sparrow.org)
  • Treatment for myocardial ischemia involves improving blood flow to the heart muscle. (sparrow.org)
  • Making heart-healthy lifestyle choices is important in treating and preventing myocardial ischemia. (sparrow.org)
  • Some people who have myocardial ischemia don't have any signs or symptoms (silent ischemia). (sparrow.org)
  • Myocardial ischemia occurs when the blood flow through one or more of your coronary arteries is decreased. (sparrow.org)
  • Myocardial ischemia can develop slowly as arteries become blocked over time. (sparrow.org)
  • Atherosclerosis is the most common cause of myocardial ischemia. (sparrow.org)
  • The clot might block an artery and lead to sudden, severe myocardial ischemia, resulting in a heart attack. (sparrow.org)
  • Coronary artery spasm is an uncommon cause of myocardial ischemia. (sparrow.org)
  • Type 1 and type 2 diabetes are linked to an increased risk of myocardial ischemia, heart attack and other heart problems. (sparrow.org)
  • People who get regular aerobic exercise have better heart health, which is associated with a lower risk of myocardial ischemia and heart attack. (sparrow.org)
  • Myocardial ischemia (not enough blood and oxygen getting to your heart muscle). (clevelandclinic.org)
  • Epigastric or chest symptoms from myocardial ischemia may incorrectly be attributed to a gastrointestinal (GI) source. (medscape.com)
  • Additionally, patients with myocardial ischemia may report relief or improvement with GI remedies (eg, antacids). (medscape.com)
  • The discomfort of myocardial ischemia may also erroneously be attributed to a musculoskeletal etiology. (medscape.com)
Cardiac7
  • The effect on cardiac biomarkers was mixed, with one study showing a significant increase in maximum AST level with oxygen therapy, one showing a significant decrease in maximum MB-CPK level, and one study showing no significant difference in troponin T or NT-proBNP levels. (wustl.edu)
  • A slightly larger trial is currently underway ( AVOID Study ) with a primary outcome measure of myocardial infarct size assessed by measuring cardiac troponin I and creatine kinase (CK). (wustl.edu)
  • At the same time, by increasing the oxygen demand, increases in heart rate also shorten diastolic duration and thus the time interval of the cardiac cycle, in which almost all of the coronary blood flow occurs. (onteenstoday.com)
  • HupA significantly diminished the infarct size and inhibited the activities of myocardial enzymes, including creatine kinase (CK), the MB isoenzyme of creatine kinase (CK-MB), lactate dehydrogenase (LDH) and cardiac troponin T (cTnT). (spandidos-publications.com)
  • Patients with cocaine toxicity should receive initial evaluation and stabilization, including attention to ABCs, oxygen, intravenous access, and cardiac and pulse oximetry monitoring. (medscape.com)
  • Although CP-BOLD looks similar to standard CINE, changes in myocardial intensity patterns and shape across cardiac phases, due to the heart's motion, BOLD effect and artifacts affect the underlying mechanisms of fully supervised segmentation techniques resulting in a significant drop in segmentation accuracy. (springer.com)
  • Through balloon inflations and deflations synchronized with the natural heartbeat the IABP increases diastolic aortic pressure, which enhances diastolic blood flow to the coronary arteries and vital organs, as well as reduces systolic aortic pressure, which reduces afterload and oxygen consumption of the myocardium and increases cardiac output. (cochrane.org)
Reactive Oxygen3
Consumption2
  • Patients underwent exercise echocardiography and also completed a symptom-limited cardiopulmonary ramp test to determine peak exercise capacity, maximal heart rate, heart rate (HR) at the anaerobic threshold (AT), peak oxygen uptake (VO2peak) VO2 consumption at AT, and exercise capacity at AT. (nih.gov)
  • Epinephrine can also have adverse effects including increased myocardial oxygen consumption and necrosis, postresuscitation myocardial dysfunction and ventricular arrhythmias. (medscape.com)
Chest Pain7
  • You are receiving a patient from EMS that is having chest pain and you notice that the patient is not on oxygen. (wustl.edu)
  • Remembering from medical school that everyone gets ABCs and IV, O2, monitor (especially with chest pain), you ask the paramedics why they aren't on oxygen. (wustl.edu)
  • In fact, they point out, that the patient's oxygen saturation is 96% and according to their protocols, written by their Medical Directors, they are not supposed to place a chest pain patient on oxygen when the SpO2 is greater than 92% (unless the patient appears to be in respiratory distress). (wustl.edu)
  • Angina is chest pain or discomfort that occurs if an area of your heart muscle doesn't get enough oxygen-rich blood. (cdc.gov)
  • The most common symptom of an MI is chest pain, resulting from deprivation of oxygen to the heart. (proprofs.com)
  • Approximately one-quarter of all myocardial infarctions are "silent", that is without chest pain or other symptoms. (medicalxpress.com)
  • Chest pain, recent myocardial infraction. (dentalcare.com)
Perfusion6
  • An IABP may be placed in the emergency department (ED) as a bridge to percutaneous coronary intervention (PCI) or coronary artery bypass grafting (CABG), to decrease myocardial workload, and to improve end-organ perfusion. (medscape.com)
  • A myocardial perfusion scan uses a tiny amount of a radioactive substance, called a radioactive tracer. (onteenstoday.com)
  • 5. In the absence of coronary stenosis and myocardial hypertrophy, coronary blood flow increases proportionally as diastolic perfusion time decreases during stress tests. (onteenstoday.com)
  • Myocardial perfusion scans help us diagnose: Coronary artery disease. (onteenstoday.com)
  • How would heart rate influence coronary artery perfusion and myocardial oxygen delivery? (onteenstoday.com)
  • As the heart rate increases, the myocardial oxygen requirement increases, yet there is a concomitant decrease in diastolic filling period, which consequently decreases the available time for perfusion. (onteenstoday.com)
Increases5
  • Calcium channel blockers like verapamil dilate the coronary blood vessels, which increases the supply of blood and oxygen to the heart. (wikipedia.org)
  • Vasopressin, on the other hand, increases coronary blood flow and myocardial oxygen availability. (medscape.com)
  • Imaging data suggests that dialysis increases myocardial blood flow (BF) heterogeneity, but its causes remain poorly understood. (frontiersin.org)
  • Lie inside a cozy pressurized chamber and begin to experience the therapeutic benefits of oxygen as it increases your body's ability to regenerate. (powershow.com)
  • The rapid heartbeat increases the workload and oxygen demand of the heart. (bartleby.com)
Dysfunction2
  • Summary: Postischaemic myocardial dysfunction (stunning) induced by partial occlusion of the left anterior descending coronary artery and its relation to lactate production during reperfusion were studied in nine swine. (elsevierpure.com)
  • A specific mtDNA deletion of 4977 bp is associated to aging, myocardial dysfunction, and bioenergetic deficit. (astm.org)
Free Radicals2
  • When amiodarone is reduced, iodine is cleaved from the molecule which generated oxygen-free radicals. (bartleby.com)
  • 2011). Larger quantities of free radicals (reactive disease, diabetes, chronic respiratory diseases, and oxygen species [ROS]) present in the cigarette cancer (WHO, 2012). (bvsalud.org)
Cardiogenic1
  • Cardiogenic shock is a severe condition in which a suddenly weakened heart is not able to pump enough blood to meet the body's energy needs, so not enough oxygen will reach the body's organs. (cochrane.org)
Patients1
  • Preliminary studies have indicated that systemic treatment of diabetic foot ulcer patients with hyperbaric oxygen therapy have beneficial effects on wound healing, risk of amputation, glycaemic control, atherosclerosis, inflammatory markers and other clinical and laboratory parameters. (bmj.com)
Reperfusion Injury1
Symptoms1
Coronary artery2
  • There has been some indirect evidence that supplemental oxygen may cause harm, with proposed mechanisms including coronary artery vasoconstriction ( Momen 2009 ), decreased coronary blood flow ( McNulty 2005 , Farquhar 2009 ), increased peripheral vascular resistance ( Milone 1999 ), and oxygen free radial damage ( Tatarkova 2012 ). (wustl.edu)
  • If a coronary artery becomes completely blocked, the lack of blood and oxygen can lead to a heart attack that destroys part of the heart muscle. (sparrow.org)
Hyperbaric oxygen8
  • This protocol for a systematic review aims at identifying the beneficial and harmful effects of adding hyperbaric oxygen therapy to standard wound care for diabetic foot ulcers. (bmj.com)
  • We plan to include all relevant randomised clinical trials assessing the effects of hyperbaric oxygen therapy in the treatment of diabetic foot ulcer versus any control group with any intervention defined as standard wound care or similar, together with sham interventions. (bmj.com)
  • Hyperbaric Oxygen Therapy - Hyperbaric Oxygen Therapy also known as hyperbaric oxygen therapy (HBOT) is the medical use of oxygen at a higher than atmospheric pressure. (powershow.com)
  • Hyperbaric Oxygen Therapy - Hyperbaric Oxygen Therapy (HBOT) is a powerful medical therapy producing phenomenal positive results for a broad range of health conditions. (powershow.com)
  • Hyperbaric Oxygen Therapy HBOT Devices Equipment Market 2019 - The worldwide market for Hyperbaric Oxygen Therapy (HBOT) Devices/Equipment is expected to grow at a CAGR of roughly 3.8% over the next five years, will reach 240 million US$ in 2024, from 200 million US$ in 2019, according to our study. (powershow.com)
  • What is HBOT Hyperbaric Oxygen Therapy: Why it's done? (powershow.com)
  • 1) - Hyperbaric oxygen therapy involves breathing pure oxygen in a pressurized room or tube. (powershow.com)
  • Hyperbaric Oxygen Therapy What it is and why you should care. (powershow.com)
Heart8
  • Myocardial contusion is a bruise of the heart muscle. (medlineplus.gov)
  • Arteries are blood vessels that carry oxygen-rich blood to your heart and other parts of your body. (cdc.gov)
  • These arteries supply oxygen-rich blood to your heart muscle. (cdc.gov)
  • The pain of angina is caused by a deficit in oxygen supply to the heart. (wikipedia.org)
  • Myocardial oxygen demand is governed by three principal factors: heart rate, contractility, and wall tension. (onteenstoday.com)
  • Heart rate influences myocardial oxygen demand, coronary blood flow, and myocardial function. (nih.gov)
  • This review summarizes the specific influence of heart rate on vascular morphology and function as well as on myocardial lesions leading from early impact on vascular homeostasis to myocardial hemodynamics in chronic heart failure. (nih.gov)
  • The low blood flow decreases the amount of oxygen your heart muscle receives. (sparrow.org)
Therapy1
  • Although non-invasive interventions may not affect existing large vessel structural defects such as stenosis, it is thought that adenosine stress and dialysate cooling therapy may improve myocardial BF by vasodilation of the smaller blood vessels. (frontiersin.org)
Mortality2
  • Two of the randomized controlled trials and the meta-analysis demonstrated a trend towards increased mortality with the routine use of supplemental oxygen in AMI, but none was sufficiently powered to show a statistically significant difference. (wustl.edu)
  • Only the PGY-4 study demonstrated a decrease in mortality with oxygen use, though this was also statistically non-significant. (wustl.edu)
Aortic pressure1
  • Steady state simulations were performed to investigate the effects of altered aortic pressure and blood vessel diameters on myocardial BF heterogeneity. (frontiersin.org)
Cerebral1
  • Noninvasive, infrared monitoring of cerebral and myocardial oxygen sufficiency and circulatory parameters. (bvsalud.org)
Infarction32
Acute1
  • Since there are degenerative changes in myocardial fibres in acute poisoning due to massive doses of fluoride, the effect of persistent minute doses of fluoride cannot be ignored. (fluoridealert.org)
STEMI6
  • Oxygen therapy is commonly used within the STEMI patient population. (wikidoc.org)
  • 1. It is reasonable to administer supplemental oxygen to all patients with uncomplicated STEMI during the first 6 hours. (wikidoc.org)
  • In 2015 the same group of investigators published a study in Circulation showing that air instead of oxygen supplementation in STEMI led to improved outco. (bmj.com)
  • In 2015 the same group of investigators published a study in Circulation showing that air instead of oxygen supplementation in STEMI led to improved outcome. (bmj.com)
  • The conclusions of these two studies seem to be that supplemental oxygen therapy in patients with STEMI but without hypoxia may increase early myocardial injury and is associated with larger myocardial infarct size assessed at 6 months. (bmj.com)
  • Conclusion: The present findings indicate no harm or benefit of supplemental O 2 on myocardial function in STEMI patients. (lu.se)
Deprivation2
  • 6. Activation of SphK1 by K6PC-5 Inhibits Oxygen-Glucose Deprivation/Reoxygenation-Induced Myocardial Cell Death. (nih.gov)
  • They saw changes that indicated oxygen deprivation and abnormal heart rhythms. (harvard.edu)
Changes in myocardial2
  • CP-BOLD MRI is a new contrast agent- and stress-free approach for examining changes in myocardial oxygenation in response to coronary artery disease. (imtlucca.it)
  • It is believed that fluoride toxicity is brought about by oxidative stress and, thus, the probably interaction between fluoride and the myocardial metabolism can be responsible for histopathological and biochemical changes in myocardial tissue. (fluoridealert.org)
Reperfusion9
  • Background: Valvular heart surgery (VHS) utilizing cardiopulmonary bypass (CPB) is inevitably associated with ischemic-perfusion injury, which is known to depend on oxygen tension during reperfusion. (elsevierpure.com)
  • The aim of this study was to evaluate the effect of arterial oxygen tension during reperfusion on myocardial recovery in patients undergoing VHS. (elsevierpure.com)
  • Methods: Fifty-six patients undergoing isolated VHS were randomly exposed to an oxygen fraction of 0.7 (hyperoxic group, n = 28) or 0.5 (normoxic group, n = 28) during reperfusion. (elsevierpure.com)
  • Conclusions: Normoxic reperfusion from the last cardioplegia administration to 1 minute after aortic unclamping in patients undergoing VHS resulted in significantly less frequent use of vasopressin and inotropics during weaning from CPB than hyperoxic reperfusion, although it did not affect the post-operative myocardial enzyme release or short-term prognosis. (elsevierpure.com)
  • These involve the activation of Jak/STAT3 and PI3K/Akt, which subsequently decreases mitochondrial permeability transition pore (mPTP) opening and increases mitochondrial K ATP (Mito K ATP ) channel opening, which attenuates myocardial ischaemia reperfusion injury. (researchgate.net)
  • Myocardial ischaemia reperfusion injury is the leading cause of death in patients with cardiovascular disease. (researchgate.net)
  • Interventions such as ischaemic pre and postconditioning protect against myocardial ischaemia reperfusion injury. (researchgate.net)
  • as a feasible method for reduction of myocardial ischaemia-reperfusion (IR) injury. (researchgate.net)
  • AIMS: Reactive oxygen species (ROS) are generated in the ischaemic myocardium especially during early reperfusion and affect myocardial function and viability. (ca.gov)
Consumption9
  • Myocyte contraction is the primary factor determining myocardial oxygen consumption (MVO 2 ) above basal levels. (cvphysiology.com)
  • Increasing inotropy also increases MVO 2 because the rate of tension development is increased as well as the magnitude of tension, both of which result in increased ATP hydrolysis and oxygen consumption. (cvphysiology.com)
  • The following parameters were measured: sinusal coronary blood flow (SCBF), total coronary vascular resistance (TCVR), aortic blood pressure, heart rate, left dP/dt max, amplitude of right ventricular contraction, double product, myocardial oxygen consumption (MVO2) and arterial and coronary venous blood gases (pO2 and pCO2) and pH. (eurekamag.com)
  • We tested the proposition that linear length-dependence of twitch duration underlies the well-characterised linear dependence of oxygen consumption (VO[sub]2[/sub]) on pressure-volume area (PVA) in the heart. (auckland.ac.nz)
  • Potential associations were sought between beta1AR genotypes and the primary outcome variables of peak oxygen consumption (VO2), heart rate response, and exercise time.The major determinants of exercise capacity were the polymorphisms at position 389, where patients homozygous for Gly389 had significantly lower peak VO2 compared with those with Arg389 (14.5 +/- 0.6 vs 17.7 +/- 0.4 mL/kg/min, P =.006), despite similar clinical characteristics including left ventricular ejection fraction. (nih.gov)
  • Physiological responses were examined via fatty acid utilization, oxygen consumption, mitochondrial mass, and Western blot analysis. (nih.gov)
  • The functional outcome was an increase in myocyte fatty acid-substrate utilization, oxygen consumption, mitochondrial mass, PPARα (peroxisome proliferator-activated receptor α) protein expression, and extracellular acidosis. (nih.gov)
  • Cardiac external work decreased, as did myocardial oxygen consumption, causing increased cardiac efficiency at higher concentrations of sevoflurane. (avma.org)
  • Poor exercise capacity and low maximal oxygen consumption (8 ml/kg/min) were shown by treadmill exercise testing. (bmj.com)
Utilization1
  • 2. In DC, adrenergic stimulus (CPT) impairs the myocardial utilization of NEFA. (nih.gov)
Production of reactive oxygen1
Reactive4
  • Our findings have important clinical implications because chronic dependence on fatty acids is associated with an accumulation in lipid intermediates, lactate, protons, and reactive oxygen species. (nih.gov)
  • Elevated matrix Ca 2+ concentrations and reactive oxygen species (ROS) are the primary activators of the MPTP during I/R. Inhibition of ion exchangers that influence cytosolic Ca 2+ levels, such as the Na + /H + exchanger (NHE) and Na + /Ca 2+ exchanger (NCX), have been shown to reduce I/R injury. (tocris.com)
  • Differences between reactive oxygen species and antioxidant defense system unbalance the redox status. (bvsalud.org)
  • 2011). Larger quantities of free radicals (reactive disease, diabetes, chronic respiratory diseases, and oxygen species [ROS]) present in the cigarette cancer (WHO, 2012). (bvsalud.org)
Carbon dioxide3
Tissue4
  • prolonged ingestion of fluoride through drinking water, particularly with high doses, induced significant histopathological and biochemical changes leading to myocardial tissue damage. (fluoridealert.org)
  • Lanoxin Tablets (digoxin) is a cardiac glycoside that has specific effects on the myocardial ( heart muscle ) tissue and is used to treat heart failure by increasing left ventricular ejection fractions and arrhythmias such as atrial fibrillation by controlling the ventricular response rate. (rxlist.com)
  • The obstruction usually takes the form of a blood clot that prevents part of the heart tissue being supplied with oxygen. (news-medical.net)
  • This lack of blood supply and oxygen can cause injury to the heart muscle and if supply is prevented for more than 20 minutes, the part of the muscle tissue failing to receive blood may die. (news-medical.net)
Heart21
Arterial1
  • Administration of oxygen to these patients should be judicious and guided by periodic arterial blood gas values. (wikidoc.org)
Ischaemia1
  • Myocardial fibrosis can develop as a consequence of myocardial ischaemia and is present in 16-55% of sudden cardiac deaths (Davies and Popple 1979). (forensicmed.co.uk)
Fibres1
  • 1- 3 This idiopathic cardiomyopathy is characterised by an altered structure of the myocardial wall as a result of intrauterine arrest of compaction of the myocardial fibres in the absence of any coexisting congenital lesion. (bmj.com)
Cardiovascular2
  • Conclusions and Clinical Relevance -Sevoflurane caused minimal and predictable cardiovascular effects in ferrets without increasing myocardial metabolic demands. (avma.org)
  • Improves the capacity of the cardiovascular system to uptake and transport oxygen. (physio-pedia.com)
Hypoxia1
  • A decrease in the myocardial extraction or excretion of lactates indicates its hypoxia. (nih.gov)
Mitochondrial1
  • Several mechanisms are involved in the process of DOX-related toxicities including mitochondrial dysfunction, oxidative stress, inhibition of autophagy, and myocardial apoptosis [ 4 ]. (hindawi.com)
Myocardium1
  • To supply the myocardium with oxygen, the blood flows from the epicardial vessels to the endocardial vessels and then returns to the right atrium via the coronary sinus. (mhmedical.com)
Biopsy1
  • Myocardial biopsy had excluded active myocarditis two years previously. (bmj.com)
Exertion1
Viability1
  • Assessment of myocardial viability is often needed in patients with chest pain and reduced ejection fraction. (nih.gov)
20161
  • However, magnetic resonance imaging on May 2, 2016, revealed anoxic brain damage due to lack of oxygen. (cdc.gov)
Benefit of supplemental1
  • A recent review of available trial data indicated no benefit of supplemental oxygen, and in fact there was signs of a hazard [3] . (wikidoc.org)
Metabolism1
Antioxidant1
  • 2011). Chronic fluoride toxicity and myocardial damage: antioxidant offered protection in second generation rats. (fluoridealert.org)
Blood5
  • This paper aims to identify approaches that generate appropriate synthetic data (computer generated) for cardiac phase-resolved blood-oxygen-level-dependent (CP-BOLD) MRI. (imtlucca.it)
  • The right ventricle gets blood from the right atrium and pumps it to the lungs to load it with oxygen. (medicinenet.com)
  • It pumps oxygen-rich blood to the rest of the body. (medicinenet.com)
  • HF is a progressive condition in which cardiac muscle weakens and becomes inefficient to meet the body's demand for blood and oxygen supply. (biorxiv.org)
  • Myocardial blood flow was determined by use of colored microsphere technology. (avma.org)
Experimental1
Vascular1
  • Antiplatelet therapy has been shown to reduce mortality rates by reducing the risk of fatal myocardial infarctions, fatal strokes, and vascular death. (medscape.com)
Inhalation1
  • 3D stack-of-spirals ultrashort-TE imaging in a healthy volunteer during inhalation of room air versus 100% oxygen. (nih.gov)
Ischaemic1
Oxidative2
  • DOX-induced oxidative damage, inflammation accumulation, and myocardial apoptosis were largely attenuated by the treatment of Mul. (hindawi.com)
  • DOX-dependent oxidative damage induced the release of cytochrome C and subsequent activation of caspase 3, causing myocardial apoptosis [ 9 ]. (hindawi.com)