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  • saccharin
  • The researchers used saccharin for subsequent experiments as this artificial sweetener showed the most pronounced effect on glucose levels in preliminary trials. (the-scientist.com)
  • Further, the team exposed seven young, healthy volunteers who did not have a history of artificial sweetener consumption to one week of the FDA's maximum acceptable daily saccharin intake, and continuously monitored their glucose levels. (the-scientist.com)
  • ARTIFICIAL
  • In a cohort of 381 non-diabetic volunteers who answered diet questionnaires, those who regularly consumed artificial sweeteners-particularly those who consumed the highest amounts-showed higher fasting glucose levels, poorer glucose tolerance, and different gut microbe profiles compared to those who did not consume such sweeteners. (the-scientist.com)
  • plasma glucose
  • OBJECTIVE We tested the hypotheses that in nondiabetic individuals, partial inhibition of insulin secretion with the ATP-sensitive K + channel agonist (opener) diazoxide, compared with placebo, results in higher plasma glucose and higher plasma glucagon concentrations after a mixed meal and after administration of the sulfonylurea glimepiride. (diabetesjournals.org)
  • However, intravenous and oral administration of diazoxide, an ATP-sensitive K + (K ATP ) channel agonist (opener), partially inhibited insulin secretion and raised postabsorptive plasma glucose concentrations in nondiabetic humans ( 3 ). (diabetesjournals.org)
  • impairment
  • CONCLUSIONS Partial inhibition of insulin secretion results in impairment of glucose tolerance after a mixed meal and after glimepiride administration in the absence of a difference in glucagon secretion. (diabetesjournals.org)
  • Exercise
  • In patients with diabetes, this may be caused by several factors, such as too much or incorrectly timed insulin, too much or incorrectly timed exercise (exercise decreases insulin requirements) or not enough food (specifically glucose containing carbohydrates). (wikipedia.org)
  • liver
  • Expression of human CTRP7 in the liver is positively correlated with glucose-6-phosphatase (G6PC) ( E ) and phosphoenolpyruvate carboxykinase (PCK1) ( F ). Data shown are expressed as means ± SE, and statistical analyses represent Mann-Whitney U -tests. (nih.gov)
  • Furthermore, it reduces the level of plasma leptin, mitigates liver damage and alleviates glucose intolerance. (wikipedia.org)