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  • neovascularization
  • The resident population of endothelial cells that is competent to respond to an available level of angiogenic growth factors, however, may potentially limit the extent to which cytokine supplementation enhances tissue neovascularization. (pnas.org)
  • ligand
  • 15 , 16 The precise mechanism underlying recruitment of EPC to tumor sites remains poorly understood, but recent reports suggested the involvement of c-Kit ligand expression by endothelial cells. (haematologica.org)
  • vivo
  • Unlike other species, human corneal endothelial cells (HCEC) are notorious for their limited proliferative capacity in vivo [ 4 ] due the mitotic block at the G1 phase in the cell cycle [ 5 ]. (medsci.org)
  • cell
  • Our hypothesis is that immobilization of the endothelial-cell specific lectin Ulex europa eus-I (UEL-I) into the nanofibrous graft matrix will provide anchor sites in order to enhance cell-specific attachment and stabilize these cells the nanofibrous surface that will be under shear forces, thereby promoting cellular growth across the materia l surface. (sbir.gov)
  • Given the potential of such a progenitor cell to treat a variety of diseases, many studies in the last fifteen years have aimed to isolate and characterize them. (stemcell.com)
  • Change in endothelial progenitor cell (EPC) levels in fenofibrate-treated vs placebo-treated patients over 12 weeks. (clinicaltrials.gov)
  • These findings motivated many researchers to equivalently search for and prove the existence of an endothelial progenitor cell. (creativebiomart.net)
  • pression, but the differing characteristics of the various cell types all previously called 'endothelial progenitors' have become more clear in recent years. (creativebiomart.net)
  • After overnight incubation of CD31+ MNCs in noncoated plates, nonadhesive cells (NA/CD31+ MNCs) were harvested as the endothelial progenitor cell-enriched fraction. (ahajournals.org)
  • nitric
  • 21-27 CRP potently downregulates endothelial nitric oxide synthase (eNOS) transcription and destabilizes eNOS mRNA, which decreases both basal and stimulated nitric oxide (NO) release. (ahajournals.org)