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Kindling, NeurologicEpilepsySeizuresStatus EpilepticusConvulsantsAnticonvulsantsEpilepsy, Post-TraumaticPentylenetetrazoleHippocampusEpilepsy, Temporal LobePilocarpineAmygdalaKainic AcidMossy Fibers, HippocampalDisease Models, AnimalElectroencephalographyDentate GyrusAdenosine KinaseMalformations of Cortical DevelopmentPerforant PathwayElectric StimulationRats, Sprague-DawleyLimbic SystemSeizures, FebrileNeurologic ExaminationNervous System DiseasesAlcohol Withdrawal DeliriumEthosuximideEpilepsy, Partial, SensorySclerosisBrainRats, WistarFlurothylDantroleneNeuronal PlasticityDihydroxytryptaminesNeuronsPyramidal CellsElectrodes, ImplantedChannelopathiesReceptors, GABA-ANeocortexEpilepsies, PartialMuscarinic Agonistsgamma-Aminobutyric AcidDiazepamEpilepsy, GeneralizedTime FactorsBehavior, AnimalReceptor, trkBSynaptic TransmissionExcitatory Amino Acid AgonistsGangliogliomaExcitatory Postsynaptic PotentialsDisease Susceptibility2-Amino-5-phosphonovalerateVideo RecordingGliosisBrain InjuriesSynapsesEpilepsy, Complex PartialExcitatory Amino Acid AntagonistsPhenylenediaminesTuberous SclerosisNeural InhibitionOrganic ChemicalsReceptors, N-Methyl-D-AspartateHyperpolarization-Activated Cyclic Nucleotide-Gated ChannelsD-Aspartic AcidAnimals, NewbornCA3 Region, HippocampalLong-Term PotentiationMultiple Chemical SensitivityGABA Antagonists
EpilepsyBrain
Epilepsy2
  • Taken together, these findings suggest that DMF, through the activation of Nrf2, has the potential to serve as a therapeutic target for preventing epileptogenesis and modifying epilepsy. (biomedcentral.com)
  • We propose that various shared mechanisms of stress and epilepsy present multiple avenues for the development of new symptomatic and preventative treatments, including disease modifying therapies aimed at reducing epileptogenesis. (bvsalud.org)
Brain1
  • Epileptogenesis has been described in remote brain regions without any detectable lesions, giving rise to dual epileptogenic foci. (medscape.com)