AnatomyDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPhenomena and ProcessesInformation Science
von Willebrand Diseasesvon Willebrand Factorvon Willebrand Disease, Type 2von Willebrand Disease, Type 1von Willebrand Disease, Type 3RistocetinFactor VIIIDeamino Arginine VasopressinBleeding TimePlatelet Glycoprotein GPIb-IX ComplexGlycogen Storage Disease Type IBlood PlateletsCrotalid VenomsPlatelet Membrane GlycoproteinsHemostaticsWeibel-Palade BodiesPlatelet AdhesivenessPlatelet AggregationBlood Coagulation TestsHemophilia ACharcot-Marie-Tooth DiseaseHemostasisHemorrhagic DisordersPedigreePoint MutationHemorrhageGlycogen Storage Disease Type IIADAM ProteinsMutation, MissenseGlycogen Storage Disease Type IIIPregnancy Complications, HematologicMutationBlood Platelet DisordersNoxaeThrombopoiesisPhenotypeGlycogen Storage Disease Type IVRecombinant ProteinsBlood Coagulation FactorsMolecular Sequence DataHeterozygote

Gain-of-function GPIb ELISA assay for VWF activity in the Zimmerman Program for the Molecular and Clinical Biology of VWD. (1/5)

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Pathologic shear triggers shedding of vascular receptors: a novel mechanism for down-regulation of platelet glycoprotein VI in stenosed coronary vessels. (2/5)

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The genetics of Canadian type 3 von Willebrand disease: further evidence for co-dominant inheritance of mutant alleles. (3/5)

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The molecular characterization of von Willebrand disease: good in parts. (4/5)

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Terminal platelet production is regulated by von Willebrand factor. (5/5)

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