Oral lichen planus in childhood: A rare case report. (65/123)

Oral lichen planus is a cell-mediated immune condition and is infrequently encountered in children, with a prevalence of about 0.03 percent in childhood. Reports of oral lichen planus affecting children are scarce in the literature. The purpose of this article is to present a rare case of oral lichen planus affecting a seven-year-old child without concomitant cutaneous lesions. Intraoral lesions and associated mild discomfort treated with topical corticosteroid therapy and a plaque control regime resulted in a favorable result.  (+info)

Oral mucosal alterations among the institutionalized elderly in Brazil. (66/123)

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Schwannoma of the tongue: an unusual presentation in a child. (67/123)

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Familial ankyloglossia (tongue-tie): a case report. (68/123)

Ankyloglossia (tongue-tie) is a congenital anomaly with a prevalence of 4-5% and characterized by an abnormally short lingual frenulum. For unknown reasons the abnormality seems to be more common in males. The pathogenesis of ankyloglossia is not known. The authors report a family with isolated ankyloglossia inherited as an autosomal dominant or recessive trait. The identification of the defective gene(s) in these patients might reveal novel information on the pathogenesis of this disorder.  (+info)

Prenatal sonographic diagnosis of a congenital oral fibrovascular hamartoma. (69/123)

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Tongue lesions in a Jordanian population. Prevalence, symptoms, subject's knowledge and treatment provided. (70/123)

Tongue lesions constitute a considerable proportion of oral mucosal lesions, and are health concern to both oral health care providers and public. OBJECTIVES: The aim of this study was to determine the prevalence of tongue lesions and conditions among a group of Jordanian population attending dental clinics, in addition to assessment of their symptoms, knowledge, and treatment provided for their tongue lesions. STUDY DESIGN: A total of 2000 dental out-patients were screened for tongue lesions. RESULTS: Fissured tongue was the most common tongue lesion diagnosed in 11.5% of the subjects, followed by coated tongue (8.2%), geographic tongue (4.8%), hairy tongue (2.4%) and median rhomboid glossitis (0.5%). Symptoms were reported by 28% of the subjects with geographic tongue and 23% with fissured tongue. The majority of the subjects (ranging from 61.1%-86.4%) were neither aware about their tongue lesions nor worried about their prognosis. Less than 50% of those who were aware of their tongue lesions sought medical advice. CONCLUSION: The high prevalence of tongue lesions necessitates higher awareness of the general dental practitioner by their etiology, diagnosis and management.  (+info)

Immunolocalization of heat shock proteins 27 and 47 during repair of induced oral ulcers. (71/123)

Heat shock proteins (Hsps) 27 and 47 are involved in the control of apoptosis, cell migration, and collagen synthesis. There is some understanding of the immunolocalization of these proteins during the repair process in skin and gastrointestinal mucosa, but their expressions in normal and injured oral mucosa are unknown. The aim of this study was to analyze the immunolocalization and intensity of these proteins in oral ulcers induced in rats and to compare these expression levels with those reported in skin and gastric mucosa. Ulcers were induced on the ventral surface of the tongues of rats. The rats were then euthanized at 0, 24, 48, 72, and 120 h. Hsp27 expression remained low in the first hours of repair, but was higher at 72 h, mainly in the migrating epithelium. Expression of Hsp47 was high at 48 h, mainly in fibroblasts, cells of the vascular wall, and basal keratinocytes of migrating epithelium. In the control group, expressions of these proteins were low, which indicates that these Hsps are constitutive proteins in oral mucosa. Expression levels were similar to those reported in the healing of skin lesions and gastric ulcer, suggesting a common mechanism of Hsp activation in the repair of these tissues.  (+info)

Localized tongue Amyloidosis in a Patient with Neurofibromatosis type II. (72/123)

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