The chemokine CXCL13 is a key regulator of B cell recruitment to the cerebrospinal fluid in acute Lyme neuroborreliosis. (49/80)

 (+info)

Mapping of hormones and cortisol responses in patients after Lyme neuroborreliosis. (50/80)

 (+info)

Humoral immune responses in patients with Lyme neuroborreliosis. (51/80)

 (+info)

Neuroinflammation in Lyme neuroborreliosis affects amyloid metabolism. (52/80)

 (+info)

Delayed diagnosis of neuroborreliosis presenting as bell palsy and meningitis. (53/80)

Lyme disease is most prevalent in the northeast and upper Midwest regions of the United States. While early symptoms may be mild (eg, rash, flu-like symptoms, joint pain), late or persistent infection can cause chronic neurologic impairments. Because of this range of symptoms, physicians can have difficulty diagnosing Lyme disease, especially in the absence of erythema chronicum migrans. We report a case of a woman who initially presented with severe vertigo and vomiting and later with fever, headache, and facial droop. After more than 3 weeks of misdiagnosis, the patient tested positive for Lyme disease and was diagnosed as having neuroborreliosis presenting as Bell palsy and meningitis. The authors review the history, diagnosis, and management of Lyme disease.  (+info)

A twist on Lyme: the challenge of diagnosing European Lyme neuroborreliosis. (54/80)

 (+info)

Severe course of Lyme neuroborreliosis in an HIV-1 positive patient; case report and review of the literature. (55/80)

 (+info)

Lyme neuroborreliosis in 2 horses. (56/80)

 (+info)