Chemical eye injuries: presentation and management difficulties.
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BACKGROUND/PURPOSE: To review the pattern of presentation and management difficulty of patients with ocular chemical bums. METHOD: Retrospective study of all patients with ocular burns seen over 8-year period (March 1998-April 2006) in the eye clinic as well as the plastic and reconstructive surgery unit of our hospital. Case files were retrieved and relevant information extracted and analyzed. RESULTS: There were a total of 23 patients (n=33 eyes) seen in the period under review consisting of 19 males and 4 females. The age ranged from 6-52 years with mean age of 31.87years. The chemical injuries were accidental in 73.9% of the cases and of these 76.5% were work related. The remaining 26.1% resulting from assault were bilateral and led to blindness in all the patients. Main injurious agents were acid and alkali. CONCLUSION: Major cause of chemical injury was work related; although blindness and disability occurred more in the cases of assault. Occupational health issues need a revisit, preparedness of health institutions to take adequate care of people with ocular burns is presently poor. (+info)
Suppression of alkali burn-induced corneal neovascularization by dendritic cell vaccination targeting VEGF receptor 2.
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HF is a corrosive mineral acid. It has extremely harmful systemic effects through any route of exposure. In dilute solutions the onset of symptoms is latent. It is important for caregivers to remember that HF has distinct clinical signs and a specific antidote, namely calcium gluconate. The industrial use of HF heightens the importance of being prepared for possible exposures. The emergency medical teams should be familiar with its symptoms, should possess the proper protective means and should be ready to respond properly in case of emergency. (+info)
Temporary sutureless amniotic membrane patch for acute alkaline burns.
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