Apoptotic gene expression in potentially fertile and subfertile men. (33/57)

Direct cytotoxicity produced by adenoviral-mediated interferon alpha gene transfer in interferon-resistant cancer cells involves ER stress and caspase 4 activation. (34/57)

c-Rel deficiency increases caspase-4 expression and leads to ER stress and necrosis in EBV-transformed cells. (35/57)

Estrogen induces apoptosis in estrogen deprivation-resistant breast cancer through stress responses as identified by global gene expression across time. (36/57)

Caspase-4 is required for activation of inflammasomes. (37/57)

Role of ER stress response in photodynamic therapy: ROS generated in different subcellular compartments trigger diverse cell death pathways. (38/57)

Apoptosis, autophagy and ER stress in mevalonate cascade inhibition-induced cell death of human atrial fibroblasts. (39/57)

A genome-wide RNA interference screen identifies caspase 4 as a factor required for tumor necrosis factor alpha signaling. (40/57)

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